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You searched for subject:(TGF ). Showing records 1 – 30 of 625 total matches.

[1] [2] [3] [4] [5] … [21]

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1. Lantero García, Aquilino. Participación de la familia de factores TGF-beta en la percepción del dolor.

Degree: 2013, Universidad de Cantabria

 El pseudorreceptor de TGF-beta, BAMBI, se localiza en regiones del sistema nervioso relevantes en la transmisión del dolor. La ausencia de BAMBI condiciona un fenotipo… (more)

Subjects/Keywords: TGF-beta

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lantero García, A. (2013). Participación de la familia de factores TGF-beta en la percepción del dolor. (Doctoral Dissertation). Universidad de Cantabria. Retrieved from http://hdl.handle.net/10902/3123

Chicago Manual of Style (16th Edition):

Lantero García, Aquilino. “Participación de la familia de factores TGF-beta en la percepción del dolor.” 2013. Doctoral Dissertation, Universidad de Cantabria. Accessed October 29, 2020. http://hdl.handle.net/10902/3123.

MLA Handbook (7th Edition):

Lantero García, Aquilino. “Participación de la familia de factores TGF-beta en la percepción del dolor.” 2013. Web. 29 Oct 2020.

Vancouver:

Lantero García A. Participación de la familia de factores TGF-beta en la percepción del dolor. [Internet] [Doctoral dissertation]. Universidad de Cantabria; 2013. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/10902/3123.

Council of Science Editors:

Lantero García A. Participación de la familia de factores TGF-beta en la percepción del dolor. [Doctoral Dissertation]. Universidad de Cantabria; 2013. Available from: http://hdl.handle.net/10902/3123


NSYSU

2. Huang, Sz-yang. Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance.

Degree: Master, Institute of Biomedical Sciences, 2010, NSYSU

 Pancreatic ductal adenocarcinoma (PDAC) is one of the most insidious forms of cancer whose incidence nearly equals its death rate. Despite extensive research studies, no… (more)

Subjects/Keywords: TGF; PDAC; HIF-1

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APA (6th Edition):

Huang, S. (2010). Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Sz-yang. “Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance.” 2010. Thesis, NSYSU. Accessed October 29, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Sz-yang. “Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance.” 2010. Web. 29 Oct 2020.

Vancouver:

Huang S. Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance. [Internet] [Thesis]. NSYSU; 2010. [cited 2020 Oct 29]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang S. Identification of target genes of SMAD4 signaling network inhibit pancreatic tumor metastasis and chemoresistance. [Thesis]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708110-141641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

3. Shen, Jie; Chen, Di (1955 - ). Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis.

Degree: PhD, 2012, University of Rochester

 Osteoarthritis (OA) is a common degenerative joint disease affecting more than 20% of American adults. The pathogenesis of OA is poorly understood and currently there… (more)

Subjects/Keywords: TGF-B; Mmp13; Osteoarthritis; Adamts5

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APA (6th Edition):

Shen, Jie; Chen, D. (. -. ). (2012). Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/25525

Chicago Manual of Style (16th Edition):

Shen, Jie; Chen, Di (1955 - ). “Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis.” 2012. Doctoral Dissertation, University of Rochester. Accessed October 29, 2020. http://hdl.handle.net/1802/25525.

MLA Handbook (7th Edition):

Shen, Jie; Chen, Di (1955 - ). “Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis.” 2012. Web. 29 Oct 2020.

Vancouver:

Shen, Jie; Chen D(-). Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1802/25525.

Council of Science Editors:

Shen, Jie; Chen D(-). Loss of TGF-B Signaling Leads to Up-Regulation of Mmp13 and Adamts5 and the Development of Osteoarthritis. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/25525


University of Guelph

4. Avery-Cooper, Geordon James. The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis.

Degree: MS, Department of Biomedical Sciences, 2011, University of Guelph

 The Par6-polarity pathway regulates breast cancer metastasis, and more recently has been shown to regulate transforming growth factor β (TGFβ)-induced apoptosis. Integrins may mediate the… (more)

Subjects/Keywords: Par6; Polarity; apoptosis; TGF-beta

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APA (6th Edition):

Avery-Cooper, G. J. (2011). The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2874

Chicago Manual of Style (16th Edition):

Avery-Cooper, Geordon James. “The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis.” 2011. Masters Thesis, University of Guelph. Accessed October 29, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2874.

MLA Handbook (7th Edition):

Avery-Cooper, Geordon James. “The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis.” 2011. Web. 29 Oct 2020.

Vancouver:

Avery-Cooper GJ. The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis. [Internet] [Masters thesis]. University of Guelph; 2011. [cited 2020 Oct 29]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2874.

Council of Science Editors:

Avery-Cooper GJ. The Potential Role of Integrin Regulation by Par6 in TGF-beta-induced Apoptosis. [Masters Thesis]. University of Guelph; 2011. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2874


University of Toronto

5. Deheshi, Benjamin Michael. Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines.

Degree: 2010, University of Toronto

Osteosarcoma is a mesenchymal tumour of bone common among children and young adults. Prognosis is poor if metastases are present at diagnosis. The transforming growth… (more)

Subjects/Keywords: Osteosarcoma; TGF-β; 0379

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APA (6th Edition):

Deheshi, B. M. (2010). Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25551

Chicago Manual of Style (16th Edition):

Deheshi, Benjamin Michael. “Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines.” 2010. Masters Thesis, University of Toronto. Accessed October 29, 2020. http://hdl.handle.net/1807/25551.

MLA Handbook (7th Edition):

Deheshi, Benjamin Michael. “Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines.” 2010. Web. 29 Oct 2020.

Vancouver:

Deheshi BM. Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1807/25551.

Council of Science Editors:

Deheshi BM. Alterations in Tgf-β Signaling Mediate the Biologic Behaviour of Osteosarcoma Cell Lines. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25551

6. ZAIATZ BITTENCOURT, VANESSA. Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function.

Degree: School of Biochemistry & Immunology. Discipline of Biochemistry, 2019, Trinity College Dublin

 It is now known that metabolism, in addition to providing energy and biochemical building blocks, also regulates immune cell function. Over the last few years,… (more)

Subjects/Keywords: immunometabolism; nk cells; tgf beta

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APA (6th Edition):

ZAIATZ BITTENCOURT, V. (2019). Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/85996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

ZAIATZ BITTENCOURT, VANESSA. “Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function.” 2019. Thesis, Trinity College Dublin. Accessed October 29, 2020. http://hdl.handle.net/2262/85996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

ZAIATZ BITTENCOURT, VANESSA. “Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function.” 2019. Web. 29 Oct 2020.

Vancouver:

ZAIATZ BITTENCOURT V. Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function. [Internet] [Thesis]. Trinity College Dublin; 2019. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/2262/85996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

ZAIATZ BITTENCOURT V. Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function. [Thesis]. Trinity College Dublin; 2019. Available from: http://hdl.handle.net/2262/85996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

7. Klingberg, Franco. Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin.

Degree: PhD, 2014, University of Toronto

Fibrosis is a detrimental disease that causes organ failure with no effective therapy available to date. Activated fibroblasts -myofibroblasts- are responsible for the excessive secretion… (more)

Subjects/Keywords: ECM; Myofibroblast; TGF-1; 0306

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APA (6th Edition):

Klingberg, F. (2014). Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/74490

Chicago Manual of Style (16th Edition):

Klingberg, Franco. “Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin.” 2014. Doctoral Dissertation, University of Toronto. Accessed October 29, 2020. http://hdl.handle.net/1807/74490.

MLA Handbook (7th Edition):

Klingberg, Franco. “Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin.” 2014. Web. 29 Oct 2020.

Vancouver:

Klingberg F. Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1807/74490.

Council of Science Editors:

Klingberg F. Availability of TGF-1 in the Myofibroblast Matrix: A Matter of Strain and Fibronectin. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/74490

8. Donato, Tatiani Ayako Goto. Estudo da influência do fator de transformação de crescimento - Beta 1 (TGF-b1) em cultura de células osteogênicas induzidas com fatores mineralizantes.

Degree: Mestrado, Biologia Celular e Tecidual, 2009, University of São Paulo

O estudo investigou a influência do fator de transformação de crescimento beta1 (TGFb1) sobre células osteogênicas induzidas com fatores mineralizantes (dexametasona Dex), comparando a viabilidade… (more)

Subjects/Keywords: Cells Osteo; Células osteoprogenitoras; Dexametasona; Dexamethasone; TGF-B1; TGF-B1

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APA (6th Edition):

Donato, T. A. G. (2009). Estudo da influência do fator de transformação de crescimento - Beta 1 (TGF-b1) em cultura de células osteogênicas induzidas com fatores mineralizantes. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42134/tde-09022010-110931/ ;

Chicago Manual of Style (16th Edition):

Donato, Tatiani Ayako Goto. “Estudo da influência do fator de transformação de crescimento - Beta 1 (TGF-b1) em cultura de células osteogênicas induzidas com fatores mineralizantes.” 2009. Masters Thesis, University of São Paulo. Accessed October 29, 2020. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-09022010-110931/ ;.

MLA Handbook (7th Edition):

Donato, Tatiani Ayako Goto. “Estudo da influência do fator de transformação de crescimento - Beta 1 (TGF-b1) em cultura de células osteogênicas induzidas com fatores mineralizantes.” 2009. Web. 29 Oct 2020.

Vancouver:

Donato TAG. Estudo da influência do fator de transformação de crescimento - Beta 1 (TGF-b1) em cultura de células osteogênicas induzidas com fatores mineralizantes. [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2020 Oct 29]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-09022010-110931/ ;.

Council of Science Editors:

Donato TAG. Estudo da influência do fator de transformação de crescimento - Beta 1 (TGF-b1) em cultura de células osteogênicas induzidas com fatores mineralizantes. [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-09022010-110931/ ;


Université Catholique de Louvain

9. Lienart, Stéphanie. Blocking immunosuppression by human Tregs with antibodies against GArP/latent TGF-β1 complexes.

Degree: 2018, Université Catholique de Louvain

Production of active TGF-β is regulated at a post-translational level and implies the release of the mature cytokine from the inactive latent TGF-β precursor. Several… (more)

Subjects/Keywords: Tregs; TGF-β1; TGF-β activation; GARP; LRRC32; Cancer immunotherapy

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APA (6th Edition):

Lienart, S. (2018). Blocking immunosuppression by human Tregs with antibodies against GArP/latent TGF-β1 complexes. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/204065

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lienart, Stéphanie. “Blocking immunosuppression by human Tregs with antibodies against GArP/latent TGF-β1 complexes.” 2018. Thesis, Université Catholique de Louvain. Accessed October 29, 2020. http://hdl.handle.net/2078.1/204065.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lienart, Stéphanie. “Blocking immunosuppression by human Tregs with antibodies against GArP/latent TGF-β1 complexes.” 2018. Web. 29 Oct 2020.

Vancouver:

Lienart S. Blocking immunosuppression by human Tregs with antibodies against GArP/latent TGF-β1 complexes. [Internet] [Thesis]. Université Catholique de Louvain; 2018. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/2078.1/204065.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lienart S. Blocking immunosuppression by human Tregs with antibodies against GArP/latent TGF-β1 complexes. [Thesis]. Université Catholique de Louvain; 2018. Available from: http://hdl.handle.net/2078.1/204065

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

10. Wu, Yi-Chen. Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis.

Degree: Master, Biological Sciences, 2010, NSYSU

 Hepatic injuries due to viral infection and alcoholic abuse frequently lead to activation and proliferation of hepatic stellate cells (HSCs), concomitantly with tissue repairing and… (more)

Subjects/Keywords: BMP-2; Hepatic; fibrogenesis; TGF-β1

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APA (6th Edition):

Wu, Y. (2010). Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0824110-131729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Yi-Chen. “Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis.” 2010. Thesis, NSYSU. Accessed October 29, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0824110-131729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Yi-Chen. “Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis.” 2010. Web. 29 Oct 2020.

Vancouver:

Wu Y. Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis. [Internet] [Thesis]. NSYSU; 2010. [cited 2020 Oct 29]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0824110-131729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu Y. Bone Morphogenetic Protein-2 Plays an Antagonistic Role in Hepatic Fibrogenesis. [Thesis]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0824110-131729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Otago

11. Kaur, Gagandeep. Regulation of human PAX2 in cancer cells by Transforming Growth Factor-beta (TGF-β) .

Degree: 2012, University of Otago

 The PAired boX (PAX) family is a group of related genes that are highly conserved throughout evolution and are known to play important roles in… (more)

Subjects/Keywords: PAX2; RCC; TGF-beta; SMAD2/3

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APA (6th Edition):

Kaur, G. (2012). Regulation of human PAX2 in cancer cells by Transforming Growth Factor-beta (TGF-β) . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/2428

Chicago Manual of Style (16th Edition):

Kaur, Gagandeep. “Regulation of human PAX2 in cancer cells by Transforming Growth Factor-beta (TGF-β) .” 2012. Doctoral Dissertation, University of Otago. Accessed October 29, 2020. http://hdl.handle.net/10523/2428.

MLA Handbook (7th Edition):

Kaur, Gagandeep. “Regulation of human PAX2 in cancer cells by Transforming Growth Factor-beta (TGF-β) .” 2012. Web. 29 Oct 2020.

Vancouver:

Kaur G. Regulation of human PAX2 in cancer cells by Transforming Growth Factor-beta (TGF-β) . [Internet] [Doctoral dissertation]. University of Otago; 2012. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/10523/2428.

Council of Science Editors:

Kaur G. Regulation of human PAX2 in cancer cells by Transforming Growth Factor-beta (TGF-β) . [Doctoral Dissertation]. University of Otago; 2012. Available from: http://hdl.handle.net/10523/2428


NSYSU

12. Chen, Ying-Pin. Study the mechanisms of Euphol in the modulation of TGF-β responsiveness.

Degree: Master, Biological Sciences, 2015, NSYSU

 Gastric cancer is a high prevalent carcinoma and the leading cause of cancer-related mortality in Taiwan. Transforming growth factor-β is one of growth factors family,… (more)

Subjects/Keywords: cholesterol; lipid-raft; triterpene; Euphol; TGF-β

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APA (6th Edition):

Chen, Y. (2015). Study the mechanisms of Euphol in the modulation of TGF-β responsiveness. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714115-141234

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Ying-Pin. “Study the mechanisms of Euphol in the modulation of TGF-β responsiveness.” 2015. Thesis, NSYSU. Accessed October 29, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714115-141234.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Ying-Pin. “Study the mechanisms of Euphol in the modulation of TGF-β responsiveness.” 2015. Web. 29 Oct 2020.

Vancouver:

Chen Y. Study the mechanisms of Euphol in the modulation of TGF-β responsiveness. [Internet] [Thesis]. NSYSU; 2015. [cited 2020 Oct 29]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714115-141234.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen Y. Study the mechanisms of Euphol in the modulation of TGF-β responsiveness. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714115-141234

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Otago

13. Samalia, Priya Darshni. Transgelin: Discovering its Role in Prostate Cancer Progression .

Degree: 2013, University of Otago

 Prostate cancer is the most commonly diagnosed cancer and the second highest cancer causing mortality amongst men in the Western world. The best possible prognosis… (more)

Subjects/Keywords: transgelin; prostate; cancer; TGF-beta; actin

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APA (6th Edition):

Samalia, P. D. (2013). Transgelin: Discovering its Role in Prostate Cancer Progression . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/4477

Chicago Manual of Style (16th Edition):

Samalia, Priya Darshni. “Transgelin: Discovering its Role in Prostate Cancer Progression .” 2013. Doctoral Dissertation, University of Otago. Accessed October 29, 2020. http://hdl.handle.net/10523/4477.

MLA Handbook (7th Edition):

Samalia, Priya Darshni. “Transgelin: Discovering its Role in Prostate Cancer Progression .” 2013. Web. 29 Oct 2020.

Vancouver:

Samalia PD. Transgelin: Discovering its Role in Prostate Cancer Progression . [Internet] [Doctoral dissertation]. University of Otago; 2013. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/10523/4477.

Council of Science Editors:

Samalia PD. Transgelin: Discovering its Role in Prostate Cancer Progression . [Doctoral Dissertation]. University of Otago; 2013. Available from: http://hdl.handle.net/10523/4477


Universiteit Utrecht

14. Mullenders, J. Big screens with small RNAs : loss of function genetic screens to identify novel cancer genes.

Degree: 2009, Universiteit Utrecht

 This thesis described the construction and screening of one of the first large scale RNAi libraries for use in human cells. Functional genetic screens with… (more)

Subjects/Keywords: Geneeskunde; Cancer; RNAi; p53; TGF-beta

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APA (6th Edition):

Mullenders, J. (2009). Big screens with small RNAs : loss of function genetic screens to identify novel cancer genes. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/39504

Chicago Manual of Style (16th Edition):

Mullenders, J. “Big screens with small RNAs : loss of function genetic screens to identify novel cancer genes.” 2009. Doctoral Dissertation, Universiteit Utrecht. Accessed October 29, 2020. http://dspace.library.uu.nl:8080/handle/1874/39504.

MLA Handbook (7th Edition):

Mullenders, J. “Big screens with small RNAs : loss of function genetic screens to identify novel cancer genes.” 2009. Web. 29 Oct 2020.

Vancouver:

Mullenders J. Big screens with small RNAs : loss of function genetic screens to identify novel cancer genes. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2009. [cited 2020 Oct 29]. Available from: http://dspace.library.uu.nl:8080/handle/1874/39504.

Council of Science Editors:

Mullenders J. Big screens with small RNAs : loss of function genetic screens to identify novel cancer genes. [Doctoral Dissertation]. Universiteit Utrecht; 2009. Available from: http://dspace.library.uu.nl:8080/handle/1874/39504


NSYSU

15. Yang, Pei-hua. Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling.

Degree: Master, Biological Sciences, 2017, NSYSU

 Pentabromophenol (PBP), a brominated flame retardant (BFR), is widely used in various consumer products. BFRs exert adverse health effects such as neurotoxic and endocrine-disrupting effects.… (more)

Subjects/Keywords: BFR; EMT; Smad; TGF-β; PBP

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APA (6th Edition):

Yang, P. (2017). Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715117-084334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Pei-hua. “Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling.” 2017. Thesis, NSYSU. Accessed October 29, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715117-084334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Pei-hua. “Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling.” 2017. Web. 29 Oct 2020.

Vancouver:

Yang P. Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling. [Internet] [Thesis]. NSYSU; 2017. [cited 2020 Oct 29]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715117-084334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang P. Study the mechanisms of Pentabromophenol in the modulation of TGF-β signaling. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715117-084334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

16. Sanchez, Nora Sylvia. The Role of The Type III Transforming Growth Factor–beta Receptor in Epicardial Cell Behavior and Coronary Vessel Development.

Degree: PhD, Pharmacology, 2011, Vanderbilt University

 Coronary artery disease accounts for 54% of all cardiovascular disease in the United States. To provide novel therapeutic targets for the prevention and treatment of… (more)

Subjects/Keywords: TGF-beta; BMP2; coronary vessels; epicardium

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sanchez, N. S. (2011). The Role of The Type III Transforming Growth Factor–beta Receptor in Epicardial Cell Behavior and Coronary Vessel Development. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14307

Chicago Manual of Style (16th Edition):

Sanchez, Nora Sylvia. “The Role of The Type III Transforming Growth Factor–beta Receptor in Epicardial Cell Behavior and Coronary Vessel Development.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed October 29, 2020. http://hdl.handle.net/1803/14307.

MLA Handbook (7th Edition):

Sanchez, Nora Sylvia. “The Role of The Type III Transforming Growth Factor–beta Receptor in Epicardial Cell Behavior and Coronary Vessel Development.” 2011. Web. 29 Oct 2020.

Vancouver:

Sanchez NS. The Role of The Type III Transforming Growth Factor–beta Receptor in Epicardial Cell Behavior and Coronary Vessel Development. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1803/14307.

Council of Science Editors:

Sanchez NS. The Role of The Type III Transforming Growth Factor–beta Receptor in Epicardial Cell Behavior and Coronary Vessel Development. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/14307


Vanderbilt University

17. Pickup, Michael William. Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression.

Degree: PhD, Cancer Biology, 2013, Vanderbilt University

 Dissertation under the direction of Professor Harold L. Moses Transforming Growth Factor Beta (TGF-β) is acts as both a tumor suppressor and promoter in the… (more)

Subjects/Keywords: Breast Cancer; TGF-Beta; Fibroblast; Tumor Microenvironment

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APA (6th Edition):

Pickup, M. W. (2013). Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14291

Chicago Manual of Style (16th Edition):

Pickup, Michael William. “Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed October 29, 2020. http://hdl.handle.net/1803/14291.

MLA Handbook (7th Edition):

Pickup, Michael William. “Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression.” 2013. Web. 29 Oct 2020.

Vancouver:

Pickup MW. Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1803/14291.

Council of Science Editors:

Pickup MW. Effects of alterations to the tumor microenvironment driven by transforming growth factor beta on tumor progression. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/14291


Queens University

18. Sondy, Yvonne. The Effect of Prenatal Ethanol Exposure on DNA Methylation and TGF-β1, SHH and Wnt3a Transcription Regulating Factors Within the Developing Hippocampus of the Guinea Pig .

Degree: Pharmacology and Toxicology, 2012, Queens University

 One of the most frequently reported deficits seen in individuals with Fetal Alcohol Spectrum Disorder (FASD) is impairments in learning and memory, which is likely… (more)

Subjects/Keywords: Wnt3a ; TGF-β1 ; DNA Methylation ; FASD ; SHH

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APA (6th Edition):

Sondy, Y. (2012). The Effect of Prenatal Ethanol Exposure on DNA Methylation and TGF-β1, SHH and Wnt3a Transcription Regulating Factors Within the Developing Hippocampus of the Guinea Pig . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sondy, Yvonne. “The Effect of Prenatal Ethanol Exposure on DNA Methylation and TGF-β1, SHH and Wnt3a Transcription Regulating Factors Within the Developing Hippocampus of the Guinea Pig .” 2012. Thesis, Queens University. Accessed October 29, 2020. http://hdl.handle.net/1974/7673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sondy, Yvonne. “The Effect of Prenatal Ethanol Exposure on DNA Methylation and TGF-β1, SHH and Wnt3a Transcription Regulating Factors Within the Developing Hippocampus of the Guinea Pig .” 2012. Web. 29 Oct 2020.

Vancouver:

Sondy Y. The Effect of Prenatal Ethanol Exposure on DNA Methylation and TGF-β1, SHH and Wnt3a Transcription Regulating Factors Within the Developing Hippocampus of the Guinea Pig . [Internet] [Thesis]. Queens University; 2012. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1974/7673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sondy Y. The Effect of Prenatal Ethanol Exposure on DNA Methylation and TGF-β1, SHH and Wnt3a Transcription Regulating Factors Within the Developing Hippocampus of the Guinea Pig . [Thesis]. Queens University; 2012. Available from: http://hdl.handle.net/1974/7673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


North Carolina State University

19. Sriperumbudur, Rajagopal. Final.

Degree: PhD, Comparative Biomedical Sciences, 2008, North Carolina State University

Subjects/Keywords: TGF-beta; luteinization

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APA (6th Edition):

Sriperumbudur, R. (2008). Final. (Doctoral Dissertation). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/5363

Chicago Manual of Style (16th Edition):

Sriperumbudur, Rajagopal. “Final.” 2008. Doctoral Dissertation, North Carolina State University. Accessed October 29, 2020. http://www.lib.ncsu.edu/resolver/1840.16/5363.

MLA Handbook (7th Edition):

Sriperumbudur, Rajagopal. “Final.” 2008. Web. 29 Oct 2020.

Vancouver:

Sriperumbudur R. Final. [Internet] [Doctoral dissertation]. North Carolina State University; 2008. [cited 2020 Oct 29]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/5363.

Council of Science Editors:

Sriperumbudur R. Final. [Doctoral Dissertation]. North Carolina State University; 2008. Available from: http://www.lib.ncsu.edu/resolver/1840.16/5363


University of Toronto

20. Cambridge, Elizabeth. Cadherin-11 Mediated Macrophage Adhesion Promotes Myofibroblast Persistence.

Degree: 2014, University of Toronto

Chronic pulmonary fibrosis is characterized by the co-existence of macrophages, which produce pro-fibrotic transforming growth factor beta-1 (TGF-β1) and myofibroblasts, which excessively secrete and contract… (more)

Subjects/Keywords: Macrophage; Fibrosis; TGF-B; 0306; 0982

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APA (6th Edition):

Cambridge, E. (2014). Cadherin-11 Mediated Macrophage Adhesion Promotes Myofibroblast Persistence. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/72562

Chicago Manual of Style (16th Edition):

Cambridge, Elizabeth. “Cadherin-11 Mediated Macrophage Adhesion Promotes Myofibroblast Persistence.” 2014. Masters Thesis, University of Toronto. Accessed October 29, 2020. http://hdl.handle.net/1807/72562.

MLA Handbook (7th Edition):

Cambridge, Elizabeth. “Cadherin-11 Mediated Macrophage Adhesion Promotes Myofibroblast Persistence.” 2014. Web. 29 Oct 2020.

Vancouver:

Cambridge E. Cadherin-11 Mediated Macrophage Adhesion Promotes Myofibroblast Persistence. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1807/72562.

Council of Science Editors:

Cambridge E. Cadherin-11 Mediated Macrophage Adhesion Promotes Myofibroblast Persistence. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/72562


Universidad de Valladolid

21. Benito Almazán, María Jesús. Papel del factor de crecimiento transformante Beta (TGF-B) y eficacia de moléculas inhibidoras en la respuesta inflamatoria de la superficie ocular.

Degree: 2012, Universidad de Valladolid

 En la presente Tesis se ha evaluado la potencial relevancia del TGF-b en los procesos inflamatorios de la superficie ocular; concretamente, se ha estudiado el… (more)

Subjects/Keywords: Ojo-Inflamación; Córnea; Conjuntivitis; TGF-b (Proteina)

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APA (6th Edition):

Benito Almazán, M. J. (2012). Papel del factor de crecimiento transformante Beta (TGF-B) y eficacia de moléculas inhibidoras en la respuesta inflamatoria de la superficie ocular. (Thesis). Universidad de Valladolid. Retrieved from http://uvadoc.uva.es/handle/10324/1902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Benito Almazán, María Jesús. “Papel del factor de crecimiento transformante Beta (TGF-B) y eficacia de moléculas inhibidoras en la respuesta inflamatoria de la superficie ocular.” 2012. Thesis, Universidad de Valladolid. Accessed October 29, 2020. http://uvadoc.uva.es/handle/10324/1902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Benito Almazán, María Jesús. “Papel del factor de crecimiento transformante Beta (TGF-B) y eficacia de moléculas inhibidoras en la respuesta inflamatoria de la superficie ocular.” 2012. Web. 29 Oct 2020.

Vancouver:

Benito Almazán MJ. Papel del factor de crecimiento transformante Beta (TGF-B) y eficacia de moléculas inhibidoras en la respuesta inflamatoria de la superficie ocular. [Internet] [Thesis]. Universidad de Valladolid; 2012. [cited 2020 Oct 29]. Available from: http://uvadoc.uva.es/handle/10324/1902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Benito Almazán MJ. Papel del factor de crecimiento transformante Beta (TGF-B) y eficacia de moléculas inhibidoras en la respuesta inflamatoria de la superficie ocular. [Thesis]. Universidad de Valladolid; 2012. Available from: http://uvadoc.uva.es/handle/10324/1902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Miami

22. Besser, Alexandra. p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program.

Degree: PhD, Cancer Biology (Medicine), 2015, University of Miami

  In normal cells, p27 regulates cell cycle and functions as an atypical tumor suppressor. Unlike typical tumor suppressors such as p16 and pRb, p27… (more)

Subjects/Keywords: p27; Metastasis; EMT; PI3K; STAT3; TGF-B2

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APA (6th Edition):

Besser, A. (2015). p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/1545

Chicago Manual of Style (16th Edition):

Besser, Alexandra. “p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program.” 2015. Doctoral Dissertation, University of Miami. Accessed October 29, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/1545.

MLA Handbook (7th Edition):

Besser, Alexandra. “p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program.” 2015. Web. 29 Oct 2020.

Vancouver:

Besser A. p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program. [Internet] [Doctoral dissertation]. University of Miami; 2015. [cited 2020 Oct 29]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1545.

Council of Science Editors:

Besser A. p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program. [Doctoral Dissertation]. University of Miami; 2015. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1545


Boston University

23. Burke, Elaine. The effects of age or sex on chondrogenesis of human MSCs.

Degree: MS, Medical Sciences, 2017, Boston University

 INTRODUCTION: Stem cells have become promising treatments for osteoarthritis due to the cells’ ability to regenerate cartilage and availability from bone marrow. Various studies have… (more)

Subjects/Keywords: Cellular biology; MSC; TGF; Age; Chondrogenesis; Differentiation

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APA (6th Edition):

Burke, E. (2017). The effects of age or sex on chondrogenesis of human MSCs. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/23744

Chicago Manual of Style (16th Edition):

Burke, Elaine. “The effects of age or sex on chondrogenesis of human MSCs.” 2017. Masters Thesis, Boston University. Accessed October 29, 2020. http://hdl.handle.net/2144/23744.

MLA Handbook (7th Edition):

Burke, Elaine. “The effects of age or sex on chondrogenesis of human MSCs.” 2017. Web. 29 Oct 2020.

Vancouver:

Burke E. The effects of age or sex on chondrogenesis of human MSCs. [Internet] [Masters thesis]. Boston University; 2017. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/2144/23744.

Council of Science Editors:

Burke E. The effects of age or sex on chondrogenesis of human MSCs. [Masters Thesis]. Boston University; 2017. Available from: http://hdl.handle.net/2144/23744


Université Catholique de Louvain

24. Stockis, Julie. Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells.

Degree: 2011, Université Catholique de Louvain

 Regulatory T cells (Tregs) are a subset of CD4+ T cells specialized in the inhibition of immune responses. Tregs are required for the maintenance of… (more)

Subjects/Keywords: Regulatory T cells; TGF-beta; Cancer immunotherapy

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APA (6th Edition):

Stockis, J. (2011). Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/76134

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stockis, Julie. “Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells.” 2011. Thesis, Université Catholique de Louvain. Accessed October 29, 2020. http://hdl.handle.net/2078.1/76134.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stockis, Julie. “Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells.” 2011. Web. 29 Oct 2020.

Vancouver:

Stockis J. Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells. [Internet] [Thesis]. Université Catholique de Louvain; 2011. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/2078.1/76134.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stockis J. Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells. [Thesis]. Université Catholique de Louvain; 2011. Available from: http://hdl.handle.net/2078.1/76134

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Catholique de Louvain

25. Huygens, Caroline. Identification of proteins regulating TGF-ß1 production in human regulatory T lymphocytes.

Degree: 2015, Université Catholique de Louvain

Human regulatory T lymphocytes (Tregs) are a subset of CD4+ T cells that inhibit other T cells by producing TGF-β1, an immunosuppressive cytokine. All T… (more)

Subjects/Keywords: TGF-ß; Lymphocytes T régulateurs; GARP; LAPTM4B

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APA (6th Edition):

Huygens, C. (2015). Identification of proteins regulating TGF-ß1 production in human regulatory T lymphocytes. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/156438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huygens, Caroline. “Identification of proteins regulating TGF-ß1 production in human regulatory T lymphocytes.” 2015. Thesis, Université Catholique de Louvain. Accessed October 29, 2020. http://hdl.handle.net/2078.1/156438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huygens, Caroline. “Identification of proteins regulating TGF-ß1 production in human regulatory T lymphocytes.” 2015. Web. 29 Oct 2020.

Vancouver:

Huygens C. Identification of proteins regulating TGF-ß1 production in human regulatory T lymphocytes. [Internet] [Thesis]. Université Catholique de Louvain; 2015. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/2078.1/156438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huygens C. Identification of proteins regulating TGF-ß1 production in human regulatory T lymphocytes. [Thesis]. Université Catholique de Louvain; 2015. Available from: http://hdl.handle.net/2078.1/156438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Bristol

26. Marcolino De Assis Junior, Eudmar. The regulation of PRH/HHEX by transforming growth factor β.

Degree: PhD, 2019, University of Bristol

 The transcription factor PRH/HHEX (Proline-Rich Homeodomain/Haematopoietically Expressed Homeobox) controls cell proliferation, cell differentiation and cell migration/invasion in a diverse range of cell types. Here I… (more)

Subjects/Keywords: Prostate Cancer; PRH/HHEX; TGF-ß; EMT

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APA (6th Edition):

Marcolino De Assis Junior, E. (2019). The regulation of PRH/HHEX by transforming growth factor β. (Doctoral Dissertation). University of Bristol. Retrieved from http://hdl.handle.net/1983/f66f42ba-a735-4397-9367-bdd63e2e519d

Chicago Manual of Style (16th Edition):

Marcolino De Assis Junior, Eudmar. “The regulation of PRH/HHEX by transforming growth factor β.” 2019. Doctoral Dissertation, University of Bristol. Accessed October 29, 2020. http://hdl.handle.net/1983/f66f42ba-a735-4397-9367-bdd63e2e519d.

MLA Handbook (7th Edition):

Marcolino De Assis Junior, Eudmar. “The regulation of PRH/HHEX by transforming growth factor β.” 2019. Web. 29 Oct 2020.

Vancouver:

Marcolino De Assis Junior E. The regulation of PRH/HHEX by transforming growth factor β. [Internet] [Doctoral dissertation]. University of Bristol; 2019. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1983/f66f42ba-a735-4397-9367-bdd63e2e519d.

Council of Science Editors:

Marcolino De Assis Junior E. The regulation of PRH/HHEX by transforming growth factor β. [Doctoral Dissertation]. University of Bristol; 2019. Available from: http://hdl.handle.net/1983/f66f42ba-a735-4397-9367-bdd63e2e519d


University of Manchester

27. Shuttleworth, Elinor. The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis.

Degree: PhD, 2018, University of Manchester

 Intestinal immune cells remain tolerant to the trillions of commensal bacteria present in the gut, with perturbations of this process implicated in development of inflammatory… (more)

Subjects/Keywords: Macrophage; IBD; Integrin avß8; Monocyte; TGF-ß

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APA (6th Edition):

Shuttleworth, E. (2018). The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-integrin-alpha-v-beta-8-on-human-monocytes-and-macrophages-in-intestinal-immune-homeostasis(b1f62522-19ba-49d7-8b09-b9d8e1bbbf6b).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764696

Chicago Manual of Style (16th Edition):

Shuttleworth, Elinor. “The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis.” 2018. Doctoral Dissertation, University of Manchester. Accessed October 29, 2020. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-integrin-alpha-v-beta-8-on-human-monocytes-and-macrophages-in-intestinal-immune-homeostasis(b1f62522-19ba-49d7-8b09-b9d8e1bbbf6b).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764696.

MLA Handbook (7th Edition):

Shuttleworth, Elinor. “The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis.” 2018. Web. 29 Oct 2020.

Vancouver:

Shuttleworth E. The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis. [Internet] [Doctoral dissertation]. University of Manchester; 2018. [cited 2020 Oct 29]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-integrin-alpha-v-beta-8-on-human-monocytes-and-macrophages-in-intestinal-immune-homeostasis(b1f62522-19ba-49d7-8b09-b9d8e1bbbf6b).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764696.

Council of Science Editors:

Shuttleworth E. The role of integrin αvβ8 on human monocytes and macrophages in intestinal immune homeostasis. [Doctoral Dissertation]. University of Manchester; 2018. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-integrin-alpha-v-beta-8-on-human-monocytes-and-macrophages-in-intestinal-immune-homeostasis(b1f62522-19ba-49d7-8b09-b9d8e1bbbf6b).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764696


Université de Sherbrooke

28. Sathoud, Yannick. Étude sur l'inhibition du phénotype des gliomes malins par la voie de signalisation du TGF.

Degree: 2013, Université de Sherbrooke

 Les gliomes malins prolifèrent d’une manière très anarchique et elles s’infiltrent dans le parenchyme cérébral. Plusieurs facteurs de croissance, tels que le Transforming Growth Factor… (more)

Subjects/Keywords: Prolifération; Métabolisme; Avasimibe™; Chloroquine; TGF-ß; Gliomes

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APA (6th Edition):

Sathoud, Y. (2013). Étude sur l'inhibition du phénotype des gliomes malins par la voie de signalisation du TGF-ß. (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/6360

Chicago Manual of Style (16th Edition):

Sathoud, Yannick. “Étude sur l'inhibition du phénotype des gliomes malins par la voie de signalisation du TGF-ß.” 2013. Masters Thesis, Université de Sherbrooke. Accessed October 29, 2020. http://hdl.handle.net/11143/6360.

MLA Handbook (7th Edition):

Sathoud, Yannick. “Étude sur l'inhibition du phénotype des gliomes malins par la voie de signalisation du TGF-ß.” 2013. Web. 29 Oct 2020.

Vancouver:

Sathoud Y. Étude sur l'inhibition du phénotype des gliomes malins par la voie de signalisation du TGF-ß. [Internet] [Masters thesis]. Université de Sherbrooke; 2013. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/11143/6360.

Council of Science Editors:

Sathoud Y. Étude sur l'inhibition du phénotype des gliomes malins par la voie de signalisation du TGF-ß. [Masters Thesis]. Université de Sherbrooke; 2013. Available from: http://hdl.handle.net/11143/6360


University of Toronto

29. Shathasivam, Premalatha. VEPH1 Regulation of TGF-beta Signalling.

Degree: 2013, University of Toronto

Epithelial ovarian cancer is the leading cause of gynecologic cancer-related mortality. Altered transforming growth factor-β (TGF-β) and wingless and integration 1 (Wnt) signalling are associated… (more)

Subjects/Keywords: VEPH1; ovarian cancer; TGF-beta; 0719

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shathasivam, P. (2013). VEPH1 Regulation of TGF-beta Signalling. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/68964

Chicago Manual of Style (16th Edition):

Shathasivam, Premalatha. “VEPH1 Regulation of TGF-beta Signalling.” 2013. Doctoral Dissertation, University of Toronto. Accessed October 29, 2020. http://hdl.handle.net/1807/68964.

MLA Handbook (7th Edition):

Shathasivam, Premalatha. “VEPH1 Regulation of TGF-beta Signalling.” 2013. Web. 29 Oct 2020.

Vancouver:

Shathasivam P. VEPH1 Regulation of TGF-beta Signalling. [Internet] [Doctoral dissertation]. University of Toronto; 2013. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1807/68964.

Council of Science Editors:

Shathasivam P. VEPH1 Regulation of TGF-beta Signalling. [Doctoral Dissertation]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/68964


University of Melbourne

30. Sheng, Jingyi. The immune suppressive function of Transforming Growth Factor-β (TGF-β) in human diseases.

Degree: 2016, University of Melbourne

 he complex immune system has evolved to defend against pathogens while maintaining self-tolerance. Transforming growth factor-β (TGF-β) is a cytokine employed by many immune cells… (more)

Subjects/Keywords: TGF-β; autoimmune disease; cancer; vaccination

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sheng, J. (2016). The immune suppressive function of Transforming Growth Factor-β (TGF-β) in human diseases. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/116141

Chicago Manual of Style (16th Edition):

Sheng, Jingyi. “The immune suppressive function of Transforming Growth Factor-β (TGF-β) in human diseases.” 2016. Doctoral Dissertation, University of Melbourne. Accessed October 29, 2020. http://hdl.handle.net/11343/116141.

MLA Handbook (7th Edition):

Sheng, Jingyi. “The immune suppressive function of Transforming Growth Factor-β (TGF-β) in human diseases.” 2016. Web. 29 Oct 2020.

Vancouver:

Sheng J. The immune suppressive function of Transforming Growth Factor-β (TGF-β) in human diseases. [Internet] [Doctoral dissertation]. University of Melbourne; 2016. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/11343/116141.

Council of Science Editors:

Sheng J. The immune suppressive function of Transforming Growth Factor-β (TGF-β) in human diseases. [Doctoral Dissertation]. University of Melbourne; 2016. Available from: http://hdl.handle.net/11343/116141

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