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You searched for subject:(T type Calcium Channels). Showing records 1 – 30 of 22414 total matches.

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Temple University

1. Jaleel, Naser. Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium.

Degree: PhD, 2010, Temple University

Physiology

The role of T-type calcium channels (TTCCs) in the heart is unclear. TTCCs are transiently expressed throughout the neonatal heart during a period of… (more)

Subjects/Keywords: Biology, Physiology; Biology, General; Cardiac Electrophysiology; Cardiac Hypertrophy; Excitation Contraction Coupling; L-Type Calcium Channels; T-type Calcium Channels

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jaleel, N. (2010). Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,83093

Chicago Manual of Style (16th Edition):

Jaleel, Naser. “Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium.” 2010. Doctoral Dissertation, Temple University. Accessed August 03, 2020. http://digital.library.temple.edu/u?/p245801coll10,83093.

MLA Handbook (7th Edition):

Jaleel, Naser. “Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium.” 2010. Web. 03 Aug 2020.

Vancouver:

Jaleel N. Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Aug 03]. Available from: http://digital.library.temple.edu/u?/p245801coll10,83093.

Council of Science Editors:

Jaleel N. Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,83093


University of Melbourne

2. ALI, IDRISH. Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy.

Degree: 2013, University of Melbourne

 Introduction: In both humans and in animal models, stress during early life has been reported to have adverse consequences for various neuropsychiatric disorders in adulthood,… (more)

Subjects/Keywords: early life stress; neuronal firing; limbic epilepsy; thalamic reticular nucleus; hippocampus; T-type calcium channels

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APA (6th Edition):

ALI, I. (2013). Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38021

Chicago Manual of Style (16th Edition):

ALI, IDRISH. “Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy.” 2013. Doctoral Dissertation, University of Melbourne. Accessed August 03, 2020. http://hdl.handle.net/11343/38021.

MLA Handbook (7th Edition):

ALI, IDRISH. “Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy.” 2013. Web. 03 Aug 2020.

Vancouver:

ALI I. Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/11343/38021.

Council of Science Editors:

ALI I. Enduring electrophysiological effects of early life stress in brain: implications for limbic epilepsy. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/38021


University of Melbourne

3. Casillas Espinosa, Pablo Miguel. Genetic and molecular determinants of acquired and genetic epilepsy.

Degree: 2016, University of Melbourne

 Introduction: Despite the differences in the pathophysiological mechanisms and clinical features of both genetic and acquired epilepsies, dysfunction of both voltage- and ligand-gated ion channels(more)

Subjects/Keywords: epileptogenesis; epilepsy; acquired epilepsy; genetic epilepsy; T-type calcium channels; TARPs; AMPA receptors; whole genome sequencing; GAERS; Z944

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APA (6th Edition):

Casillas Espinosa, P. M. (2016). Genetic and molecular determinants of acquired and genetic epilepsy. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/108875

Chicago Manual of Style (16th Edition):

Casillas Espinosa, Pablo Miguel. “Genetic and molecular determinants of acquired and genetic epilepsy.” 2016. Doctoral Dissertation, University of Melbourne. Accessed August 03, 2020. http://hdl.handle.net/11343/108875.

MLA Handbook (7th Edition):

Casillas Espinosa, Pablo Miguel. “Genetic and molecular determinants of acquired and genetic epilepsy.” 2016. Web. 03 Aug 2020.

Vancouver:

Casillas Espinosa PM. Genetic and molecular determinants of acquired and genetic epilepsy. [Internet] [Doctoral dissertation]. University of Melbourne; 2016. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/11343/108875.

Council of Science Editors:

Casillas Espinosa PM. Genetic and molecular determinants of acquired and genetic epilepsy. [Doctoral Dissertation]. University of Melbourne; 2016. Available from: http://hdl.handle.net/11343/108875

4. El Alaoui, Chaymae. Étude des effets modulateurs des plantes médicinales méditerranéennes sur les canaux calciques de type T et l’évaluation de leurs effets anticonvulsivants et antiépileptiques : Study of the modulatory effects of mediterranean plant extracts on T-type calcium channels and the evaluation of their anticonvulsant and antiepileptic activities.

Degree: Docteur es, Biologie Santé, 2015, Montpellier; Université Hassan II (Mohammedia, Maroc). Faculté des sciences et techniques

Les plantes médicinales constituent un réservoir important de substances naturelles pour la découverte de nouvelles molécules thérapeutiques. L’intérêt de ce travail est d'explorer le potentiel… (more)

Subjects/Keywords: Plantes médicinales; Canaux calciques de type T; Patch-Clamp; Epilepsie; Poisson Zèbre; Medicinal plants; T-Type calcium channels; Patch-Clamp; Epilepsy; Zebrafish

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APA (6th Edition):

El Alaoui, C. (2015). Étude des effets modulateurs des plantes médicinales méditerranéennes sur les canaux calciques de type T et l’évaluation de leurs effets anticonvulsivants et antiépileptiques : Study of the modulatory effects of mediterranean plant extracts on T-type calcium channels and the evaluation of their anticonvulsant and antiepileptic activities. (Doctoral Dissertation). Montpellier; Université Hassan II (Mohammedia, Maroc). Faculté des sciences et techniques. Retrieved from http://www.theses.fr/2015MONTT014

Chicago Manual of Style (16th Edition):

El Alaoui, Chaymae. “Étude des effets modulateurs des plantes médicinales méditerranéennes sur les canaux calciques de type T et l’évaluation de leurs effets anticonvulsivants et antiépileptiques : Study of the modulatory effects of mediterranean plant extracts on T-type calcium channels and the evaluation of their anticonvulsant and antiepileptic activities.” 2015. Doctoral Dissertation, Montpellier; Université Hassan II (Mohammedia, Maroc). Faculté des sciences et techniques. Accessed August 03, 2020. http://www.theses.fr/2015MONTT014.

MLA Handbook (7th Edition):

El Alaoui, Chaymae. “Étude des effets modulateurs des plantes médicinales méditerranéennes sur les canaux calciques de type T et l’évaluation de leurs effets anticonvulsivants et antiépileptiques : Study of the modulatory effects of mediterranean plant extracts on T-type calcium channels and the evaluation of their anticonvulsant and antiepileptic activities.” 2015. Web. 03 Aug 2020.

Vancouver:

El Alaoui C. Étude des effets modulateurs des plantes médicinales méditerranéennes sur les canaux calciques de type T et l’évaluation de leurs effets anticonvulsivants et antiépileptiques : Study of the modulatory effects of mediterranean plant extracts on T-type calcium channels and the evaluation of their anticonvulsant and antiepileptic activities. [Internet] [Doctoral dissertation]. Montpellier; Université Hassan II (Mohammedia, Maroc). Faculté des sciences et techniques; 2015. [cited 2020 Aug 03]. Available from: http://www.theses.fr/2015MONTT014.

Council of Science Editors:

El Alaoui C. Étude des effets modulateurs des plantes médicinales méditerranéennes sur les canaux calciques de type T et l’évaluation de leurs effets anticonvulsivants et antiépileptiques : Study of the modulatory effects of mediterranean plant extracts on T-type calcium channels and the evaluation of their anticonvulsant and antiepileptic activities. [Doctoral Dissertation]. Montpellier; Université Hassan II (Mohammedia, Maroc). Faculté des sciences et techniques; 2015. Available from: http://www.theses.fr/2015MONTT014

5. Sakkaki, Sophie. Etude pharmacologique des canaux calciques de type T dans des modèles murins de convulsion et d'épileptogenèse. : Pharmacological study of T-type calcium channels in mice models of convulsion and epileptogenesis.

Degree: Docteur es, Neurosciences, 2011, Université Montpellier I

De nombreuses études expérimentales montrent que les canaux calciques activés par la dépolarisation membranaire, tout particulièrement les canaux calciques de type T (canaux T), jouent… (more)

Subjects/Keywords: Canaux calciques de type T; Epilepsie; Convulsion; Crise tonico-clonique; Crise partielle; T-type calcium channels; Epilepsy; Seizure; Tonic-clonic seizure; Partial seizure

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APA (6th Edition):

Sakkaki, S. (2011). Etude pharmacologique des canaux calciques de type T dans des modèles murins de convulsion et d'épileptogenèse. : Pharmacological study of T-type calcium channels in mice models of convulsion and epileptogenesis. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2011MON1T020

Chicago Manual of Style (16th Edition):

Sakkaki, Sophie. “Etude pharmacologique des canaux calciques de type T dans des modèles murins de convulsion et d'épileptogenèse. : Pharmacological study of T-type calcium channels in mice models of convulsion and epileptogenesis.” 2011. Doctoral Dissertation, Université Montpellier I. Accessed August 03, 2020. http://www.theses.fr/2011MON1T020.

MLA Handbook (7th Edition):

Sakkaki, Sophie. “Etude pharmacologique des canaux calciques de type T dans des modèles murins de convulsion et d'épileptogenèse. : Pharmacological study of T-type calcium channels in mice models of convulsion and epileptogenesis.” 2011. Web. 03 Aug 2020.

Vancouver:

Sakkaki S. Etude pharmacologique des canaux calciques de type T dans des modèles murins de convulsion et d'épileptogenèse. : Pharmacological study of T-type calcium channels in mice models of convulsion and epileptogenesis. [Internet] [Doctoral dissertation]. Université Montpellier I; 2011. [cited 2020 Aug 03]. Available from: http://www.theses.fr/2011MON1T020.

Council of Science Editors:

Sakkaki S. Etude pharmacologique des canaux calciques de type T dans des modèles murins de convulsion et d'épileptogenèse. : Pharmacological study of T-type calcium channels in mice models of convulsion and epileptogenesis. [Doctoral Dissertation]. Université Montpellier I; 2011. Available from: http://www.theses.fr/2011MON1T020

6. Bray, Jennifer Grace. Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus.

Degree: 2010, Marquette University

 During the early postnatal period, the inhibitory neurotransmitter γ-aminobutyric acid (GABA) facilitates current through voltage-dependent L-type calcium channels by activating metabotropic GABAB receptors in the… (more)

Subjects/Keywords: GABA  – Receptors; Calcium channels; Biology; GABAB Receptors; KCC2; L-Type Calcium Channels; NKCC1; PKC; Biology

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APA (6th Edition):

Bray, J. G. (2010). Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus. (Thesis). Marquette University. Retrieved from https://epublications.marquette.edu/dissertations_mu/39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bray, Jennifer Grace. “Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus.” 2010. Thesis, Marquette University. Accessed August 03, 2020. https://epublications.marquette.edu/dissertations_mu/39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bray, Jennifer Grace. “Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus.” 2010. Web. 03 Aug 2020.

Vancouver:

Bray JG. Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus. [Internet] [Thesis]. Marquette University; 2010. [cited 2020 Aug 03]. Available from: https://epublications.marquette.edu/dissertations_mu/39.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bray JG. Modulation of L-type Calcium Current by GABA-B Receptor Activation in the Neonatal Rat Hippocampus. [Thesis]. Marquette University; 2010. Available from: https://epublications.marquette.edu/dissertations_mu/39

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Guan, Wendy. Domain II (S5-P) region in Lymnaea T-type calcium channels and its role in determining biophysical properties, ion selectivity and drug sensitivity.

Degree: 2014, University of Waterloo

 Invertebrate T-type calcium channels cloned from the great pond snail, Lymnaea Stagnalis (LCav3) possess highly sodium permeant ion channel currents by means of alternative splicing… (more)

Subjects/Keywords: Ion channels; Electrophysiology; T-type calcium channel; Cav3; Invertebrate; Evolution

…119 4.10 Possible structural similarities between K2P and KIR and T-type calcium channels… …Structural model of T-type calcium channels A common feature of calcium channels is the pore… …primary pore forming subunit of T-type calcium channels, the α1 subunit. The α1 subunit is a… …gating kinetics in T-type calcium channels and stabilizes the channel in a closed state, in a… …inactivation and window currents T-type calcium channels are distinguished from other calcium… 

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APA (6th Edition):

Guan, W. (2014). Domain II (S5-P) region in Lymnaea T-type calcium channels and its role in determining biophysical properties, ion selectivity and drug sensitivity. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/8507

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guan, Wendy. “Domain II (S5-P) region in Lymnaea T-type calcium channels and its role in determining biophysical properties, ion selectivity and drug sensitivity.” 2014. Thesis, University of Waterloo. Accessed August 03, 2020. http://hdl.handle.net/10012/8507.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guan, Wendy. “Domain II (S5-P) region in Lymnaea T-type calcium channels and its role in determining biophysical properties, ion selectivity and drug sensitivity.” 2014. Web. 03 Aug 2020.

Vancouver:

Guan W. Domain II (S5-P) region in Lymnaea T-type calcium channels and its role in determining biophysical properties, ion selectivity and drug sensitivity. [Internet] [Thesis]. University of Waterloo; 2014. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/10012/8507.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guan W. Domain II (S5-P) region in Lymnaea T-type calcium channels and its role in determining biophysical properties, ion selectivity and drug sensitivity. [Thesis]. University of Waterloo; 2014. Available from: http://hdl.handle.net/10012/8507

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

8. Crandall, Shane. Dendritic properties of inhibitory thalamic neurons: implications in sub-cortical sensory processing.

Degree: PhD, 0323, 2012, University of Illinois – Urbana-Champaign

 This dissertation is focused on understanding the cellular mechanisms underlying thalamocortical network activities. Specifically, I am interested in how inhibitory neurons of the thalamus process… (more)

Subjects/Keywords: dorsal lateral geniculate nucleus; thalamic reticular nucleus; T-type calcium channels; presynaptic dendrite; local inhibition; whole cell recording; glutamate uncaging; two photon imaging; dendrite; glutamate receptor; feedforward inhibition; thalamus; interneuron

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APA (6th Edition):

Crandall, S. (2012). Dendritic properties of inhibitory thalamic neurons: implications in sub-cortical sensory processing. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/31947

Chicago Manual of Style (16th Edition):

Crandall, Shane. “Dendritic properties of inhibitory thalamic neurons: implications in sub-cortical sensory processing.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed August 03, 2020. http://hdl.handle.net/2142/31947.

MLA Handbook (7th Edition):

Crandall, Shane. “Dendritic properties of inhibitory thalamic neurons: implications in sub-cortical sensory processing.” 2012. Web. 03 Aug 2020.

Vancouver:

Crandall S. Dendritic properties of inhibitory thalamic neurons: implications in sub-cortical sensory processing. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2142/31947.

Council of Science Editors:

Crandall S. Dendritic properties of inhibitory thalamic neurons: implications in sub-cortical sensory processing. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/31947


University of New Mexico

9. Zhang, Bojun. CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA.

Degree: Biomedical Sciences Graduate Program, 2017, University of New Mexico

  Chronic hypoxia (CH)-induced pulmonary hypertension (PH) is associated with diminished ATP-induced endothelial Ca2+ entry as well as membrane cholesterol in pulmonary arteries. Store-operated Ca2+… (more)

Subjects/Keywords: pulmonary hypertension; T-type calcium channels; ATP; store-operated Ca2+ entry; depolarization-induced Ca2+ entry; STIM1; Cellular and Molecular Physiology; Medicine and Health Sciences

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APA (6th Edition):

Zhang, B. (2017). CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/180

Chicago Manual of Style (16th Edition):

Zhang, Bojun. “CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA.” 2017. Doctoral Dissertation, University of New Mexico. Accessed August 03, 2020. https://digitalrepository.unm.edu/biom_etds/180.

MLA Handbook (7th Edition):

Zhang, Bojun. “CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA.” 2017. Web. 03 Aug 2020.

Vancouver:

Zhang B. CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA. [Internet] [Doctoral dissertation]. University of New Mexico; 2017. [cited 2020 Aug 03]. Available from: https://digitalrepository.unm.edu/biom_etds/180.

Council of Science Editors:

Zhang B. CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA. [Doctoral Dissertation]. University of New Mexico; 2017. Available from: https://digitalrepository.unm.edu/biom_etds/180


University of Sydney

10. Perera, Naomi Tessa. ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx .

Degree: 2014, University of Sydney

 ZnT-1 expression in the pre-implantation mouse embryo and its effect on calcium influx Pre-implantation embryos develop into 9 stages over the first 5 days post-fertilisation.… (more)

Subjects/Keywords: preimplantation; mouse; embryo; ZnT-1; L-type; T-type; calcium channel

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APA (6th Edition):

Perera, N. T. (2014). ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/13519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perera, Naomi Tessa. “ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx .” 2014. Thesis, University of Sydney. Accessed August 03, 2020. http://hdl.handle.net/2123/13519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perera, Naomi Tessa. “ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx .” 2014. Web. 03 Aug 2020.

Vancouver:

Perera NT. ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx . [Internet] [Thesis]. University of Sydney; 2014. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2123/13519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perera NT. ZnT‐1 expression in the preimplantation mouse embryo and its effect on calcium influx . [Thesis]. University of Sydney; 2014. Available from: http://hdl.handle.net/2123/13519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Picard, Elodie. Implication physiopathologique et pharmacologique des canaux calciques Cav 3.2 dans la douleur chronique : Pathophysiological and pharmacological involvement of Cav 3.2 calcium channels in chronic pain.

Degree: Docteur es, Sciences de la vie et de la sante, 2017, Clermont Auvergne

 La douleur chronique occupe une place centrale dans les préoccupations de santé publique. En France, elle touche environ 20% de la population et a un… (more)

Subjects/Keywords: Polyarthrite rhumatoïde; Douleur viscérale; Maladie inflammatoire de l'intestin; Syndrome de l’intestin irritable; Douleur inflammatoire; Canaux calciques de type T; Douleur chronique; T type calcium channels,,; Visceral pain; Inflammatory bowel disease; Irritable bowel syndrome; Inflammatory pain; Rheumatoid arthritis; Chronic pain; 616.722

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APA (6th Edition):

Picard, E. (2017). Implication physiopathologique et pharmacologique des canaux calciques Cav 3.2 dans la douleur chronique : Pathophysiological and pharmacological involvement of Cav 3.2 calcium channels in chronic pain. (Doctoral Dissertation). Clermont Auvergne. Retrieved from http://www.theses.fr/2017CLFAS008

Chicago Manual of Style (16th Edition):

Picard, Elodie. “Implication physiopathologique et pharmacologique des canaux calciques Cav 3.2 dans la douleur chronique : Pathophysiological and pharmacological involvement of Cav 3.2 calcium channels in chronic pain.” 2017. Doctoral Dissertation, Clermont Auvergne. Accessed August 03, 2020. http://www.theses.fr/2017CLFAS008.

MLA Handbook (7th Edition):

Picard, Elodie. “Implication physiopathologique et pharmacologique des canaux calciques Cav 3.2 dans la douleur chronique : Pathophysiological and pharmacological involvement of Cav 3.2 calcium channels in chronic pain.” 2017. Web. 03 Aug 2020.

Vancouver:

Picard E. Implication physiopathologique et pharmacologique des canaux calciques Cav 3.2 dans la douleur chronique : Pathophysiological and pharmacological involvement of Cav 3.2 calcium channels in chronic pain. [Internet] [Doctoral dissertation]. Clermont Auvergne; 2017. [cited 2020 Aug 03]. Available from: http://www.theses.fr/2017CLFAS008.

Council of Science Editors:

Picard E. Implication physiopathologique et pharmacologique des canaux calciques Cav 3.2 dans la douleur chronique : Pathophysiological and pharmacological involvement of Cav 3.2 calcium channels in chronic pain. [Doctoral Dissertation]. Clermont Auvergne; 2017. Available from: http://www.theses.fr/2017CLFAS008


Louisiana State University

12. Tekmen, Merve. Calcium-dependent mechanisms in the chicken retina.

Degree: PhD, 2011, Louisiana State University

 Multiple classes of retinal amacrine cells use L-type Ca2+ channels to mediate synaptic transmission. I have used whole cell voltage clamp recordings from primary cultures… (more)

Subjects/Keywords: calcium; amacrine; L-type Channels; chicken retina; NO; NOS

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APA (6th Edition):

Tekmen, M. (2011). Calcium-dependent mechanisms in the chicken retina. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-06282011-112408 ; https://digitalcommons.lsu.edu/gradschool_dissertations/909

Chicago Manual of Style (16th Edition):

Tekmen, Merve. “Calcium-dependent mechanisms in the chicken retina.” 2011. Doctoral Dissertation, Louisiana State University. Accessed August 03, 2020. etd-06282011-112408 ; https://digitalcommons.lsu.edu/gradschool_dissertations/909.

MLA Handbook (7th Edition):

Tekmen, Merve. “Calcium-dependent mechanisms in the chicken retina.” 2011. Web. 03 Aug 2020.

Vancouver:

Tekmen M. Calcium-dependent mechanisms in the chicken retina. [Internet] [Doctoral dissertation]. Louisiana State University; 2011. [cited 2020 Aug 03]. Available from: etd-06282011-112408 ; https://digitalcommons.lsu.edu/gradschool_dissertations/909.

Council of Science Editors:

Tekmen M. Calcium-dependent mechanisms in the chicken retina. [Doctoral Dissertation]. Louisiana State University; 2011. Available from: etd-06282011-112408 ; https://digitalcommons.lsu.edu/gradschool_dissertations/909


University of Texas Southwestern Medical Center

13. Kjalarsdottir, Lilja. Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function.

Degree: 2012, University of Texas Southwestern Medical Center

 1,25-dihydroxyvitamin D3 (VitD) is a ligand for the Vitamin D Receptor (VDR, NR1I1), which is a member of the family of Nuclear Hormone Receptors (NHR).… (more)

Subjects/Keywords: Receptors, Calcitrol; Calcium Channels; Diabetes Mellitus, Type 2

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APA (6th Edition):

Kjalarsdottir, L. (2012). Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1028

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kjalarsdottir, Lilja. “Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed August 03, 2020. http://hdl.handle.net/2152.5/1028.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kjalarsdottir, Lilja. “Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function.” 2012. Web. 03 Aug 2020.

Vancouver:

Kjalarsdottir L. Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2152.5/1028.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kjalarsdottir L. Role of the Vitamin D Receptor in Insulin Secretion and Beta Cell Function. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1028

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

14. Venugopal, Jessica Dawn. Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype.

Degree: PhD, Molecular & Integrative Physiology, 2016, University of Kansas

 Autosomal dominant polycystic kidney disease (ADPKD) is a genetic disorder, caused by mutations in either of the PKD1 or PKD2 genes, which leads to the… (more)

Subjects/Keywords: Physiology; Apoptosis; Calcium; EMT; L-type calcium channels; Na; K-ATPase; Polycystins

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APA (6th Edition):

Venugopal, J. D. (2016). Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/24813

Chicago Manual of Style (16th Edition):

Venugopal, Jessica Dawn. “Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype.” 2016. Doctoral Dissertation, University of Kansas. Accessed August 03, 2020. http://hdl.handle.net/1808/24813.

MLA Handbook (7th Edition):

Venugopal, Jessica Dawn. “Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype.” 2016. Web. 03 Aug 2020.

Vancouver:

Venugopal JD. Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/1808/24813.

Council of Science Editors:

Venugopal JD. Contributions of Ouabain to the Autosomal Dominant Polycystic Kidney Disease Phenotype. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/24813

15. Crespí Sallán, Marta. Implicació dels canals de calci de tipus T en la regulació de l’autofàgia en el glioblastoma multiforme.

Degree: Departament de Medicina Experimental, 2018, Universitat de Lleida

 The focus of this doctoral thesis has been to study the role of the T-type calcium channels (TT-CC) in the modulation of autophagy in glioblastoma… (more)

Subjects/Keywords: Canals de calci de tipus T; p62/SQSTM1; Autofàgia; Estrés de reticle; Quimioresistencia; Glioblastoma; Normòxia; Hipòxia; NNC55-0369; Canales de calci de tipo T; Autofagia; Estrés de retículo; Normóxia; Hipóxia; T-type calcium channels; Autophagy; Reticulum stress; Chemoresistance; Glioblastoma; Tnormoxia; Hypoxia; Biologia Cel·lular; 576

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APA (6th Edition):

Crespí Sallán, M. (2018). Implicació dels canals de calci de tipus T en la regulació de l’autofàgia en el glioblastoma multiforme. (Thesis). Universitat de Lleida. Retrieved from http://hdl.handle.net/10803/460804

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Crespí Sallán, Marta. “Implicació dels canals de calci de tipus T en la regulació de l’autofàgia en el glioblastoma multiforme.” 2018. Thesis, Universitat de Lleida. Accessed August 03, 2020. http://hdl.handle.net/10803/460804.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Crespí Sallán, Marta. “Implicació dels canals de calci de tipus T en la regulació de l’autofàgia en el glioblastoma multiforme.” 2018. Web. 03 Aug 2020.

Vancouver:

Crespí Sallán M. Implicació dels canals de calci de tipus T en la regulació de l’autofàgia en el glioblastoma multiforme. [Internet] [Thesis]. Universitat de Lleida; 2018. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/10803/460804.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Crespí Sallán M. Implicació dels canals de calci de tipus T en la regulació de l’autofàgia en el glioblastoma multiforme. [Thesis]. Universitat de Lleida; 2018. Available from: http://hdl.handle.net/10803/460804

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

16. Keeling, Ginny Marie. Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties.

Degree: MS, Neuroscience, 2012, Loyola University Chicago

  Hyperglycemia can cause altered excitability due to increased CaV3.2 T-type calcium channel function, bestowing diabetics an increased neuropathy risk. The objective of this study… (more)

Subjects/Keywords: calcium channel; diabetes; ganglioside; glycosylation; T-type; Neurosciences

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APA (6th Edition):

Keeling, G. M. (2012). Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties. (Thesis). Loyola University Chicago. Retrieved from http://ecommons.luc.edu/luc_theses/841

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keeling, Ginny Marie. “Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties.” 2012. Thesis, Loyola University Chicago. Accessed August 03, 2020. http://ecommons.luc.edu/luc_theses/841.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keeling, Ginny Marie. “Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties.” 2012. Web. 03 Aug 2020.

Vancouver:

Keeling GM. Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties. [Internet] [Thesis]. Loyola University Chicago; 2012. [cited 2020 Aug 03]. Available from: http://ecommons.luc.edu/luc_theses/841.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keeling GM. Determination of the Molecular Mechanisms of Hyperglycemia-Induced Changes in Cav3.2 Calcium Channel Properties. [Thesis]. Loyola University Chicago; 2012. Available from: http://ecommons.luc.edu/luc_theses/841

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Stephens, Robert. The Role of Domain II and Domain IV Extracellular Turrets in Determining Ion Selectivity in the T-type Calcium Channel from Lymnaea stagnalis.

Degree: 2016, University of Waterloo

 The T-type calcium channel from the pond snail Lymnaea stagnalis, LCav3, undergoes alternative splicing in exon 12. Exon 12 codes for the L5 structure (S5-P)… (more)

Subjects/Keywords: Calcium Channels; T-type Calcium Channel; Giant Pond Snail; Electrophysiology; Membrane Biophysics; Ion Selectivity; Ion Permeability; Calcium Permeability; Sodium Permeability

…140 IV V Structural and Functional Asymmetry in T-type Calcium Channels… …41 Figure 1.14 Calcium versus Sodium Currents Through T-type Calcium Channels from Humans… …Channels, Delta-Cysteine mutant T-type Channels, and chimeric T-type Calcium Channels… …T-type Calcium Channels LCav3-12b DIVα1hL, LCav3-12a DIVα1hL, and LCav3-DIIα1hL DIVα1hL… …structural features of T-type calcium channels which allows them to alter their ion selectivity to… 

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APA (6th Edition):

Stephens, R. (2016). The Role of Domain II and Domain IV Extracellular Turrets in Determining Ion Selectivity in the T-type Calcium Channel from Lymnaea stagnalis. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/10590

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stephens, Robert. “The Role of Domain II and Domain IV Extracellular Turrets in Determining Ion Selectivity in the T-type Calcium Channel from Lymnaea stagnalis.” 2016. Thesis, University of Waterloo. Accessed August 03, 2020. http://hdl.handle.net/10012/10590.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stephens, Robert. “The Role of Domain II and Domain IV Extracellular Turrets in Determining Ion Selectivity in the T-type Calcium Channel from Lymnaea stagnalis.” 2016. Web. 03 Aug 2020.

Vancouver:

Stephens R. The Role of Domain II and Domain IV Extracellular Turrets in Determining Ion Selectivity in the T-type Calcium Channel from Lymnaea stagnalis. [Internet] [Thesis]. University of Waterloo; 2016. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/10012/10590.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stephens R. The Role of Domain II and Domain IV Extracellular Turrets in Determining Ion Selectivity in the T-type Calcium Channel from Lymnaea stagnalis. [Thesis]. University of Waterloo; 2016. Available from: http://hdl.handle.net/10012/10590

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

18. Yao, Jian. Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells.

Degree: 2011, University of Texas Southwestern Medical Center

Calcium is the most versatile second messenger and plays fundamental roles in orchestrating enzyme secretion in exocrine acinar cells. Previous studies in excitable cells demonstrated… (more)

Subjects/Keywords: Calcium; Calcium Channels; Portoid Gland

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APA (6th Edition):

Yao, J. (2011). Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/864

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yao, Jian. “Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed August 03, 2020. http://hdl.handle.net/2152.5/864.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yao, Jian. “Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells.” 2011. Web. 03 Aug 2020.

Vancouver:

Yao J. Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2152.5/864.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yao J. Ca2+ Signaling in the Near Plasma Membrane Microdomain of Non-Excitable Cells. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/864

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Schneider, Eve Rebecca. Electrogenic Dendrites of the Deep Cerebellar Nuclei .

Degree: PhD, 2012, Princeton University

 The purpose of this thesis is to investigate whether neurons of the deep cerebellar nuclei (DCN) have active dendrites. DCN neurons are the critical output… (more)

Subjects/Keywords: Active dendrites; Cerebellum; Deep cerebellar nuclei; Patch clamp; T-type calcium channels; two-photon microscopy

…Molineux et al., 2006; Molineux et al., 2008). Current through T-type calcium channels (… …type Ca2+ channels in other neuronal types: relevance for DCN. T-type calcium channels are… …primary dendrites, T-type calcium channels have been found to 19 amplify PC IPSCs in DCN… …type calcium channels. These channels are activated at moderate voltages but require… …Studies of cultured DCN neurons did not observe T-type calcium current, though it is present in… 

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APA (6th Edition):

Schneider, E. R. (2012). Electrogenic Dendrites of the Deep Cerebellar Nuclei . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp011g05fb640

Chicago Manual of Style (16th Edition):

Schneider, Eve Rebecca. “Electrogenic Dendrites of the Deep Cerebellar Nuclei .” 2012. Doctoral Dissertation, Princeton University. Accessed August 03, 2020. http://arks.princeton.edu/ark:/88435/dsp011g05fb640.

MLA Handbook (7th Edition):

Schneider, Eve Rebecca. “Electrogenic Dendrites of the Deep Cerebellar Nuclei .” 2012. Web. 03 Aug 2020.

Vancouver:

Schneider ER. Electrogenic Dendrites of the Deep Cerebellar Nuclei . [Internet] [Doctoral dissertation]. Princeton University; 2012. [cited 2020 Aug 03]. Available from: http://arks.princeton.edu/ark:/88435/dsp011g05fb640.

Council of Science Editors:

Schneider ER. Electrogenic Dendrites of the Deep Cerebellar Nuclei . [Doctoral Dissertation]. Princeton University; 2012. Available from: http://arks.princeton.edu/ark:/88435/dsp011g05fb640

20. Lopin, Kyle V. Divalent Effects on Permeatin and Gating of T-type calcium channels.

Degree: PhD, Physiology and Biophysics, 2013, Case Western Reserve University School of Graduate Studies

 Cells carefully regulate their intracellular ion composition and their membrane potential. Voltage gated calcium channels change both of these properties by allowing Ca2+ to enter… (more)

Subjects/Keywords: Biophysics; Physiology; T-Type; Calcium Channels; iron; cadmium; Erying rate theory; Permeation

…Permeation and Gating of T-type Calcium Channels Abstract By KYLE VITAUTAS LOPIN Cells carefully… …collected from α1G T-type calcium channels. Using this model of permeation we were able to fit… …ions (Ca2+, Ba2+, Mg2+, Na+). T-type calcium channels are expressed in a wide range… …coordinates vesicular release or the contraction cycle. 1.4: T-TYPE CALCIUM CHANNELS Early… …channels which are actived at higher voltages (~ -20 mV). Three T-type calcium channels… 

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APA (6th Edition):

Lopin, K. V. (2013). Divalent Effects on Permeatin and Gating of T-type calcium channels. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1364042187

Chicago Manual of Style (16th Edition):

Lopin, Kyle V. “Divalent Effects on Permeatin and Gating of T-type calcium channels.” 2013. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed August 03, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1364042187.

MLA Handbook (7th Edition):

Lopin, Kyle V. “Divalent Effects on Permeatin and Gating of T-type calcium channels.” 2013. Web. 03 Aug 2020.

Vancouver:

Lopin KV. Divalent Effects on Permeatin and Gating of T-type calcium channels. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2013. [cited 2020 Aug 03]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1364042187.

Council of Science Editors:

Lopin KV. Divalent Effects on Permeatin and Gating of T-type calcium channels. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1364042187


Université de Sherbrooke

21. Savard, Alexandre. Caractérisation de la protéine Gimap5 chez les lymphocytes T.

Degree: 2011, Université de Sherbrooke

 Le diabète de Type 1 est une maladie auto-immune caractérisée par la perte de production d'insuline par les îlots [béta] du pancréas. Les causes menant… (more)

Subjects/Keywords: Auto-immunité; Diabète de type 1; Homéostasie; Calcium; Survie; Lymphocytes T; GIMAP5

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APA (6th Edition):

Savard, A. (2011). Caractérisation de la protéine Gimap5 chez les lymphocytes T. (Masters Thesis). Université de Sherbrooke. Retrieved from http://savoirs.usherbrooke.ca/handle/11143/4070

Chicago Manual of Style (16th Edition):

Savard, Alexandre. “Caractérisation de la protéine Gimap5 chez les lymphocytes T.” 2011. Masters Thesis, Université de Sherbrooke. Accessed August 03, 2020. http://savoirs.usherbrooke.ca/handle/11143/4070.

MLA Handbook (7th Edition):

Savard, Alexandre. “Caractérisation de la protéine Gimap5 chez les lymphocytes T.” 2011. Web. 03 Aug 2020.

Vancouver:

Savard A. Caractérisation de la protéine Gimap5 chez les lymphocytes T. [Internet] [Masters thesis]. Université de Sherbrooke; 2011. [cited 2020 Aug 03]. Available from: http://savoirs.usherbrooke.ca/handle/11143/4070.

Council of Science Editors:

Savard A. Caractérisation de la protéine Gimap5 chez les lymphocytes T. [Masters Thesis]. Université de Sherbrooke; 2011. Available from: http://savoirs.usherbrooke.ca/handle/11143/4070


The Ohio State University

22. Hopp, Sarah Christine. Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences.

Degree: PhD, Neuroscience Graduate Studies Program, 2014, The Ohio State University

 Chronic neuroinflammation and aging are key contributors to the pathogenesis and progression of age-associated neurodegenerative diseases. Microglia dysregulation and calcium dysregulation are two shared features… (more)

Subjects/Keywords: Neurosciences; microglia; aging; calcium; calcium dysregulation; ryanodine receptors; L-type voltage dependent calcium channels; neuroinflammation; memory; substantia nigra; locus coeruleus; hippocampus; cytokines; neuroimmunology; behavioral neuroscience

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APA (6th Edition):

Hopp, S. C. (2014). Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1414416679

Chicago Manual of Style (16th Edition):

Hopp, Sarah Christine. “Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences.” 2014. Doctoral Dissertation, The Ohio State University. Accessed August 03, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1414416679.

MLA Handbook (7th Edition):

Hopp, Sarah Christine. “Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences.” 2014. Web. 03 Aug 2020.

Vancouver:

Hopp SC. Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences. [Internet] [Doctoral dissertation]. The Ohio State University; 2014. [cited 2020 Aug 03]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1414416679.

Council of Science Editors:

Hopp SC. Microglia and calcium dysregulation during chronic neuroinflammation and aging:causes and consequences. [Doctoral Dissertation]. The Ohio State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1414416679


Temple University

23. Wang, Fang. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.

Degree: PhD, 2012, Temple University

Physiology

Cardiovascular disease remains the number one cause or mortally in the western world. Heart failure is the most rapidly growing cardiovascular disease (Hobbs, 2004;… (more)

Subjects/Keywords: Physiology; Calcium Current; Cardiomyocyte Proliferation; Electrophysiology; Flow Cytometry; Neonatal Mice Ventricular Cardiomyocyte; T-type calcium channel

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APA (6th Edition):

Wang, F. (2012). DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214814

Chicago Manual of Style (16th Edition):

Wang, Fang. “DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.” 2012. Doctoral Dissertation, Temple University. Accessed August 03, 2020. http://digital.library.temple.edu/u?/p245801coll10,214814.

MLA Handbook (7th Edition):

Wang, Fang. “DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.” 2012. Web. 03 Aug 2020.

Vancouver:

Wang F. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 03]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214814.

Council of Science Editors:

Wang F. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,214814


University of Michigan

24. McKinney, Brandon Chad. L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology.

Degree: PhD, Neuroscience, 2010, University of Michigan

 Fear conditioning is a form of learning in which an initially neutral stimulus (e.g., tone or context) comes to be feared after being paired with… (more)

Subjects/Keywords: Fear Conditioning; L-type Voltage-gated Calcium Channels; CaV1.2; CaV1.3; Fear Extinction; Fear Consolidation; Neurosciences; Health Sciences; Science

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APA (6th Edition):

McKinney, B. C. (2010). L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/75966

Chicago Manual of Style (16th Edition):

McKinney, Brandon Chad. “L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology.” 2010. Doctoral Dissertation, University of Michigan. Accessed August 03, 2020. http://hdl.handle.net/2027.42/75966.

MLA Handbook (7th Edition):

McKinney, Brandon Chad. “L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology.” 2010. Web. 03 Aug 2020.

Vancouver:

McKinney BC. L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology. [Internet] [Doctoral dissertation]. University of Michigan; 2010. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2027.42/75966.

Council of Science Editors:

McKinney BC. L-type Voltage-Gated Calcium Channels in Fear Conditioning, Fear Extinction, and Amygdala Neurophysiology. [Doctoral Dissertation]. University of Michigan; 2010. Available from: http://hdl.handle.net/2027.42/75966


University of Texas Southwestern Medical Center

25. Tandan, Samvit. Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel.

Degree: 2008, University of Texas Southwestern Medical Center

 The L-type Ca2+ channel is the major mediator of Ca2+ influx in cardiomyocytes leading to both mechanical contraction and activation of signaling cascades. Among these… (more)

Subjects/Keywords: Calcineurin; Calcium Channels, L-Type; Cardiac Myocytes

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APA (6th Edition):

Tandan, S. (2008). Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tandan, Samvit. “Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel.” 2008. Thesis, University of Texas Southwestern Medical Center. Accessed August 03, 2020. http://hdl.handle.net/2152.5/491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tandan, Samvit. “Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel.” 2008. Web. 03 Aug 2020.

Vancouver:

Tandan S. Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2008. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2152.5/491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tandan S. Physical and Functional Interaction Between Calcineurin and the Cardiac L-Type Calcium Channel. [Thesis]. University of Texas Southwestern Medical Center; 2008. Available from: http://hdl.handle.net/2152.5/491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

26. Robinson, Philip. Targeting of Voltage-Gated Calcium Channels to Lipid Rafts: The Role of Auxiliary Alpha2/Delta-1 Subunits.

Degree: 2011, University of Manchester

 This abstract pertains to a thesis submitted to the University of Manchester for the degree of Doctor of Philosophy in the Faculty of Life Sciences.Ca2+… (more)

Subjects/Keywords: calcium channels; lipid rafts

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Robinson, P. (2011). Targeting of Voltage-Gated Calcium Channels to Lipid Rafts: The Role of Auxiliary Alpha2/Delta-1 Subunits. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:127411

Chicago Manual of Style (16th Edition):

Robinson, Philip. “Targeting of Voltage-Gated Calcium Channels to Lipid Rafts: The Role of Auxiliary Alpha2/Delta-1 Subunits.” 2011. Doctoral Dissertation, University of Manchester. Accessed August 03, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:127411.

MLA Handbook (7th Edition):

Robinson, Philip. “Targeting of Voltage-Gated Calcium Channels to Lipid Rafts: The Role of Auxiliary Alpha2/Delta-1 Subunits.” 2011. Web. 03 Aug 2020.

Vancouver:

Robinson P. Targeting of Voltage-Gated Calcium Channels to Lipid Rafts: The Role of Auxiliary Alpha2/Delta-1 Subunits. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2020 Aug 03]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:127411.

Council of Science Editors:

Robinson P. Targeting of Voltage-Gated Calcium Channels to Lipid Rafts: The Role of Auxiliary Alpha2/Delta-1 Subunits. [Doctoral Dissertation]. University of Manchester; 2011. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:127411


University of Manchester

27. Robinson, Philip. Targeting of voltage-gated calcium channels to lipid rafts : the role of auxiliary alpha2/delta-1 subunits.

Degree: PhD, 2011, University of Manchester

 Ca2+ entry through voltage-gated calcium channels (CaVs) triggers a range of physiological events, including synaptic neurotransmission and muscular excito-contraction coupling. CaVs are often localised to… (more)

Subjects/Keywords: 615.7; calcium channels; lipid rafts

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Robinson, P. (2011). Targeting of voltage-gated calcium channels to lipid rafts : the role of auxiliary alpha2/delta-1 subunits. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/targeting-of-voltagegated-calcium-channels-to-lipid-rafts-the-role-of-auxiliary-alpha2delta1-subunits(db84049c-1445-4486-9e31-34fa4110daf6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632144

Chicago Manual of Style (16th Edition):

Robinson, Philip. “Targeting of voltage-gated calcium channels to lipid rafts : the role of auxiliary alpha2/delta-1 subunits.” 2011. Doctoral Dissertation, University of Manchester. Accessed August 03, 2020. https://www.research.manchester.ac.uk/portal/en/theses/targeting-of-voltagegated-calcium-channels-to-lipid-rafts-the-role-of-auxiliary-alpha2delta1-subunits(db84049c-1445-4486-9e31-34fa4110daf6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632144.

MLA Handbook (7th Edition):

Robinson, Philip. “Targeting of voltage-gated calcium channels to lipid rafts : the role of auxiliary alpha2/delta-1 subunits.” 2011. Web. 03 Aug 2020.

Vancouver:

Robinson P. Targeting of voltage-gated calcium channels to lipid rafts : the role of auxiliary alpha2/delta-1 subunits. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2020 Aug 03]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/targeting-of-voltagegated-calcium-channels-to-lipid-rafts-the-role-of-auxiliary-alpha2delta1-subunits(db84049c-1445-4486-9e31-34fa4110daf6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632144.

Council of Science Editors:

Robinson P. Targeting of voltage-gated calcium channels to lipid rafts : the role of auxiliary alpha2/delta-1 subunits. [Doctoral Dissertation]. University of Manchester; 2011. Available from: https://www.research.manchester.ac.uk/portal/en/theses/targeting-of-voltagegated-calcium-channels-to-lipid-rafts-the-role-of-auxiliary-alpha2delta1-subunits(db84049c-1445-4486-9e31-34fa4110daf6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632144


University of Texas Southwestern Medical Center

28. Nguyen, Nam Xuan. Structural Basis of Calcium Transport and Selectivity in the Mitochondrial Calcium Uniporter.

Degree: 2018, University of Texas Southwestern Medical Center

 The mitochondrial calcium uniporter is a highly selective calcium channel localized to the inner mitochondrial membrane. Here, I describe the structure of an MCU ortholog… (more)

Subjects/Keywords: Calcium Channels; Cryoelectron Microscopy; Neosartorya

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APA (6th Edition):

Nguyen, N. X. (2018). Structural Basis of Calcium Transport and Selectivity in the Mitochondrial Calcium Uniporter. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/8316

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nguyen, Nam Xuan. “Structural Basis of Calcium Transport and Selectivity in the Mitochondrial Calcium Uniporter.” 2018. Thesis, University of Texas Southwestern Medical Center. Accessed August 03, 2020. http://hdl.handle.net/2152.5/8316.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nguyen, Nam Xuan. “Structural Basis of Calcium Transport and Selectivity in the Mitochondrial Calcium Uniporter.” 2018. Web. 03 Aug 2020.

Vancouver:

Nguyen NX. Structural Basis of Calcium Transport and Selectivity in the Mitochondrial Calcium Uniporter. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2018. [cited 2020 Aug 03]. Available from: http://hdl.handle.net/2152.5/8316.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nguyen NX. Structural Basis of Calcium Transport and Selectivity in the Mitochondrial Calcium Uniporter. [Thesis]. University of Texas Southwestern Medical Center; 2018. Available from: http://hdl.handle.net/2152.5/8316

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. TAN BAO ZHEN. Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel.

Degree: 2011, National University of Singapore

Subjects/Keywords: L-type calcium channels; Cav1.3 calcium channels; RNA editing; IQ domain; Alternative splicing; Calcium-dependent inactivation

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APA (6th Edition):

ZHEN, T. B. (2011). Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/27562

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

ZHEN, TAN BAO. “Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel.” 2011. Thesis, National University of Singapore. Accessed August 03, 2020. http://scholarbank.nus.edu.sg/handle/10635/27562.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

ZHEN, TAN BAO. “Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel.” 2011. Web. 03 Aug 2020.

Vancouver:

ZHEN TB. Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel. [Internet] [Thesis]. National University of Singapore; 2011. [cited 2020 Aug 03]. Available from: http://scholarbank.nus.edu.sg/handle/10635/27562.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

ZHEN TB. Functional Characterization of RNA Editing and Alternative Splicing in the Carboxyl-Terminus of Cav 1.3 Calcium Channel. [Thesis]. National University of Singapore; 2011. Available from: http://scholarbank.nus.edu.sg/handle/10635/27562

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Voisin, Tiphaine. Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways.

Degree: Docteur es, Neurosciences, 2015, Montpellier

Les canaux de type T Cav3.2 sont des canaux calciques activés pour de faibles dépolarisations membranaires. Ils ont un rôle important dans la régulation de… (more)

Subjects/Keywords: Canaux calciques de type T; Douleur; Souris génétiquement modifiée; Cav3.2; Neurones de DRG; T-Type calcium channel; Pain; Genetically modified mouse; Cav3.2; DRG neurons

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Voisin, T. (2015). Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2015MONTT037

Chicago Manual of Style (16th Edition):

Voisin, Tiphaine. “Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways.” 2015. Doctoral Dissertation, Montpellier. Accessed August 03, 2020. http://www.theses.fr/2015MONTT037.

MLA Handbook (7th Edition):

Voisin, Tiphaine. “Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways.” 2015. Web. 03 Aug 2020.

Vancouver:

Voisin T. Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways. [Internet] [Doctoral dissertation]. Montpellier; 2015. [cited 2020 Aug 03]. Available from: http://www.theses.fr/2015MONTT037.

Council of Science Editors:

Voisin T. Développement d’une souris modèle pour l’étude de la modulation metal/redox du canal calcique Cav3.2 dans l’excitabilité neuronale et dans les voies de la douleur : Development of a mouse model to study the metal/redox modulation of Cav3.2 calcium channels in neuronal excitability and in the pain pathways. [Doctoral Dissertation]. Montpellier; 2015. Available from: http://www.theses.fr/2015MONTT037

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