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You searched for subject:(T cell). Showing records 1 – 30 of 1930 total matches.

[1] [2] [3] [4] [5] … [65]

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University of Minnesota

1. Kotov, Dmitri. The role of T cell receptor affinity in CD4+ T cell differentiation.

Degree: PhD, Microbiology, Immunology and Cancer Biology, 2019, University of Minnesota

 Naïve helper T cells become activated when their T cell receptor (TCR) recognizes a microbial peptide presented on MHCII (p:MHCII) by dendritic cells (DCs). During… (more)

Subjects/Keywords: CD4+ T cell; Dendritic Cell; Helper T cell; T cell receptor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kotov, D. (2019). The role of T cell receptor affinity in CD4+ T cell differentiation. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/202415

Chicago Manual of Style (16th Edition):

Kotov, Dmitri. “The role of T cell receptor affinity in CD4+ T cell differentiation.” 2019. Doctoral Dissertation, University of Minnesota. Accessed April 17, 2021. http://hdl.handle.net/11299/202415.

MLA Handbook (7th Edition):

Kotov, Dmitri. “The role of T cell receptor affinity in CD4+ T cell differentiation.” 2019. Web. 17 Apr 2021.

Vancouver:

Kotov D. The role of T cell receptor affinity in CD4+ T cell differentiation. [Internet] [Doctoral dissertation]. University of Minnesota; 2019. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/11299/202415.

Council of Science Editors:

Kotov D. The role of T cell receptor affinity in CD4+ T cell differentiation. [Doctoral Dissertation]. University of Minnesota; 2019. Available from: http://hdl.handle.net/11299/202415


University of California – San Diego

2. Shaw, Laura Ann. The role of Id proteins in T cell immunity.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 Upon infection, naive T lymphocytes proliferate and differentiate into highly specialized cell types to combat the pathogen: CD4+ T cells into specialized helper subsets and… (more)

Subjects/Keywords: Immunology; T cell

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APA (6th Edition):

Shaw, L. A. (2016). The role of Id proteins in T cell immunity. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/1d22p4pg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shaw, Laura Ann. “The role of Id proteins in T cell immunity.” 2016. Thesis, University of California – San Diego. Accessed April 17, 2021. http://www.escholarship.org/uc/item/1d22p4pg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shaw, Laura Ann. “The role of Id proteins in T cell immunity.” 2016. Web. 17 Apr 2021.

Vancouver:

Shaw LA. The role of Id proteins in T cell immunity. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2021 Apr 17]. Available from: http://www.escholarship.org/uc/item/1d22p4pg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shaw LA. The role of Id proteins in T cell immunity. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/1d22p4pg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Wilson, Douglas C. Interleukin-12: A Critical Regulator of Protective CD8+ T Cell Immunity During Toxoplasma gondii Infection.

Degree: PhD, Division of Biology and Medicine. Pathobiology, 2009, Brown University

 Immunity to Toxoplasma gondii infection is principally mediated by the activation of CD8+ T cells producing the protective cytokine IFN-?. To characterize primary CTL activation,… (more)

Subjects/Keywords: CD8+ T cell

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APA (6th Edition):

Wilson, D. C. (2009). Interleukin-12: A Critical Regulator of Protective CD8+ T Cell Immunity During Toxoplasma gondii Infection. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:75/

Chicago Manual of Style (16th Edition):

Wilson, Douglas C. “Interleukin-12: A Critical Regulator of Protective CD8+ T Cell Immunity During Toxoplasma gondii Infection.” 2009. Doctoral Dissertation, Brown University. Accessed April 17, 2021. https://repository.library.brown.edu/studio/item/bdr:75/.

MLA Handbook (7th Edition):

Wilson, Douglas C. “Interleukin-12: A Critical Regulator of Protective CD8+ T Cell Immunity During Toxoplasma gondii Infection.” 2009. Web. 17 Apr 2021.

Vancouver:

Wilson DC. Interleukin-12: A Critical Regulator of Protective CD8+ T Cell Immunity During Toxoplasma gondii Infection. [Internet] [Doctoral dissertation]. Brown University; 2009. [cited 2021 Apr 17]. Available from: https://repository.library.brown.edu/studio/item/bdr:75/.

Council of Science Editors:

Wilson DC. Interleukin-12: A Critical Regulator of Protective CD8+ T Cell Immunity During Toxoplasma gondii Infection. [Doctoral Dissertation]. Brown University; 2009. Available from: https://repository.library.brown.edu/studio/item/bdr:75/


Harvard University

4. Smith, Neal P. The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals.

Degree: 2019, Harvard University

The induction and persistence of peanut allergies is fundamentally dependent on T cells’ ability to recognize allergenic antigens. Peanut-specificity of a T cell is conferred… (more)

Subjects/Keywords: T cell; allergy

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APA (6th Edition):

Smith, N. P. (2019). The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals. (Thesis). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004117

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Neal P. “The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals.” 2019. Thesis, Harvard University. Accessed April 17, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004117.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Neal P. “The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals.” 2019. Web. 17 Apr 2021.

Vancouver:

Smith NP. The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals. [Internet] [Thesis]. Harvard University; 2019. [cited 2021 Apr 17]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004117.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith NP. The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals. [Thesis]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004117

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Harvard University

5. Smith, Neal P. The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals.

Degree: ALM, 2019, Harvard University

The induction and persistence of peanut allergies is fundamentally dependent on T cells’ ability to recognize allergenic antigens. Peanut-specificity of a T cell is conferred… (more)

Subjects/Keywords: T cell; allergy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Smith, N. P. (2019). The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals. (Masters Thesis). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004158

Chicago Manual of Style (16th Edition):

Smith, Neal P. “The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals.” 2019. Masters Thesis, Harvard University. Accessed April 17, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004158.

MLA Handbook (7th Edition):

Smith, Neal P. “The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals.” 2019. Web. 17 Apr 2021.

Vancouver:

Smith NP. The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals. [Internet] [Masters thesis]. Harvard University; 2019. [cited 2021 Apr 17]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004158.

Council of Science Editors:

Smith NP. The Peanut-Specific TCR Repertoire: A Statistical Approach to Define Homology Among CDR3β Sequences in Peanut-Allergic Individuals. [Masters Thesis]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42004158


University of Texas – Austin

6. -5682-1161. High throughput methods for measuring TCR-pMHC affinity and specificity.

Degree: PhD, Chemical engineering, 2017, University of Texas – Austin

T cells are a crucial component of the human immune response. I developed two technology platforms that enable high-throughput measurement of TCR-pMHC affinity, sequence, and… (more)

Subjects/Keywords: Immunology; T cell

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APA (6th Edition):

-5682-1161. (2017). High throughput methods for measuring TCR-pMHC affinity and specificity. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://dx.doi.org/10.26153/tsw/7530

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-5682-1161. “High throughput methods for measuring TCR-pMHC affinity and specificity.” 2017. Doctoral Dissertation, University of Texas – Austin. Accessed April 17, 2021. http://dx.doi.org/10.26153/tsw/7530.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-5682-1161. “High throughput methods for measuring TCR-pMHC affinity and specificity.” 2017. Web. 17 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-5682-1161. High throughput methods for measuring TCR-pMHC affinity and specificity. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2017. [cited 2021 Apr 17]. Available from: http://dx.doi.org/10.26153/tsw/7530.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-5682-1161. High throughput methods for measuring TCR-pMHC affinity and specificity. [Doctoral Dissertation]. University of Texas – Austin; 2017. Available from: http://dx.doi.org/10.26153/tsw/7530

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

7. Fairbairn, Camilla Jayne. Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL): Surplus to requirements or a protagonist in lymphomagenesis?.

Degree: PhD, 2018, University of Cambridge

 Anaplastic Large Cell Lymphoma (ALCL) is a peripheral T cell lymphoma divided into three distinct entities: ALCL, Anaplastic Lymphoma Kinase (ALK)+, ALCL ALK- and cutaneous… (more)

Subjects/Keywords: Analplastic Large Cell Lymphoma; T Cell; Lyphoma; T Cell Receptor; T Cell Signaling

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APA (6th Edition):

Fairbairn, C. J. (2018). Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL): Surplus to requirements or a protagonist in lymphomagenesis?. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/273245

Chicago Manual of Style (16th Edition):

Fairbairn, Camilla Jayne. “Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL): Surplus to requirements or a protagonist in lymphomagenesis?.” 2018. Doctoral Dissertation, University of Cambridge. Accessed April 17, 2021. https://www.repository.cam.ac.uk/handle/1810/273245.

MLA Handbook (7th Edition):

Fairbairn, Camilla Jayne. “Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL): Surplus to requirements or a protagonist in lymphomagenesis?.” 2018. Web. 17 Apr 2021.

Vancouver:

Fairbairn CJ. Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL): Surplus to requirements or a protagonist in lymphomagenesis?. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2021 Apr 17]. Available from: https://www.repository.cam.ac.uk/handle/1810/273245.

Council of Science Editors:

Fairbairn CJ. Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL): Surplus to requirements or a protagonist in lymphomagenesis?. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/273245

8. Fairbairn, Camilla Jayne. Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL) : surplus to requirements or a protagonist in lymphomagenesis?.

Degree: PhD, 2018, University of Cambridge

 Anaplastic Large Cell Lymphoma (ALCL) is a peripheral T cell lymphoma divided into three distinct entities: ALCL, Anaplastic Lymphoma Kinase (ALK)+, ALCL ALK- and cutaneous… (more)

Subjects/Keywords: 616.07; Analplastic Large Cell Lymphoma; T Cell; Lyphoma; T Cell Receptor; T Cell Signaling

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APA (6th Edition):

Fairbairn, C. J. (2018). Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL) : surplus to requirements or a protagonist in lymphomagenesis?. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.20257 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744544

Chicago Manual of Style (16th Edition):

Fairbairn, Camilla Jayne. “Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL) : surplus to requirements or a protagonist in lymphomagenesis?.” 2018. Doctoral Dissertation, University of Cambridge. Accessed April 17, 2021. https://doi.org/10.17863/CAM.20257 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744544.

MLA Handbook (7th Edition):

Fairbairn, Camilla Jayne. “Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL) : surplus to requirements or a protagonist in lymphomagenesis?.” 2018. Web. 17 Apr 2021.

Vancouver:

Fairbairn CJ. Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL) : surplus to requirements or a protagonist in lymphomagenesis?. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2021 Apr 17]. Available from: https://doi.org/10.17863/CAM.20257 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744544.

Council of Science Editors:

Fairbairn CJ. Searching for the missing T Cell Receptor (TCR) in Anaplastic Large Cell Lymphoma (ALCL) : surplus to requirements or a protagonist in lymphomagenesis?. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://doi.org/10.17863/CAM.20257 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744544


University of Melbourne

9. CUKALAC, TANIA. Analysis of factors influencing CD8+ T cell responses after influenza A virus infection.

Degree: 2013, University of Melbourne

 CD8+ T cell responses to pathogens are characterised by the clonal expansion of cells expressing T cell receptors (TCRs) of unique specificity for antigenic peptide… (more)

Subjects/Keywords: immunology; CD8+ T cell; T cell receptors; cellular immunity; cell mediated immunity; virus infection; T cell receptor repertoires; T cell immunodominance

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APA (6th Edition):

CUKALAC, T. (2013). Analysis of factors influencing CD8+ T cell responses after influenza A virus infection. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38114

Chicago Manual of Style (16th Edition):

CUKALAC, TANIA. “Analysis of factors influencing CD8+ T cell responses after influenza A virus infection.” 2013. Doctoral Dissertation, University of Melbourne. Accessed April 17, 2021. http://hdl.handle.net/11343/38114.

MLA Handbook (7th Edition):

CUKALAC, TANIA. “Analysis of factors influencing CD8+ T cell responses after influenza A virus infection.” 2013. Web. 17 Apr 2021.

Vancouver:

CUKALAC T. Analysis of factors influencing CD8+ T cell responses after influenza A virus infection. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/11343/38114.

Council of Science Editors:

CUKALAC T. Analysis of factors influencing CD8+ T cell responses after influenza A virus infection. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/38114


Queen Mary, University of London

10. Kishore, Madhav. The role of co-stimulatory receptors in the regulation of regulatory T cell migration.

Degree: PhD, 2016, Queen Mary, University of London

 Once an immune response is initiated, a combination of several mechanisms coordinates and directs the homing of T cells to their target tissue. Co-stimulatory receptors… (more)

Subjects/Keywords: Medicine; regulatory T cells; T cell migration

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APA (6th Edition):

Kishore, M. (2016). The role of co-stimulatory receptors in the regulation of regulatory T cell migration. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/12869 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775244

Chicago Manual of Style (16th Edition):

Kishore, Madhav. “The role of co-stimulatory receptors in the regulation of regulatory T cell migration.” 2016. Doctoral Dissertation, Queen Mary, University of London. Accessed April 17, 2021. http://qmro.qmul.ac.uk/xmlui/handle/123456789/12869 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775244.

MLA Handbook (7th Edition):

Kishore, Madhav. “The role of co-stimulatory receptors in the regulation of regulatory T cell migration.” 2016. Web. 17 Apr 2021.

Vancouver:

Kishore M. The role of co-stimulatory receptors in the regulation of regulatory T cell migration. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2016. [cited 2021 Apr 17]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/12869 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775244.

Council of Science Editors:

Kishore M. The role of co-stimulatory receptors in the regulation of regulatory T cell migration. [Doctoral Dissertation]. Queen Mary, University of London; 2016. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/12869 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775244


University of Manchester

11. Dookie, Rebecca. Dissecting the molecular mechanisms of CD4+ T cell exhaustion during malaria.

Degree: 2019, University of Manchester

Malaria is a global life-threatening disease responsible for 400,000 deaths each year. Chronic infection with Plasmodium species drives CD4+ T cell exhaustion, which is characterised… (more)

Subjects/Keywords: CD4+ T cells; Malaria; T cell exhaustion

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APA (6th Edition):

Dookie, R. (2019). Dissecting the molecular mechanisms of CD4+ T cell exhaustion during malaria. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319548

Chicago Manual of Style (16th Edition):

Dookie, Rebecca. “Dissecting the molecular mechanisms of CD4+ T cell exhaustion during malaria.” 2019. Doctoral Dissertation, University of Manchester. Accessed April 17, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319548.

MLA Handbook (7th Edition):

Dookie, Rebecca. “Dissecting the molecular mechanisms of CD4+ T cell exhaustion during malaria.” 2019. Web. 17 Apr 2021.

Vancouver:

Dookie R. Dissecting the molecular mechanisms of CD4+ T cell exhaustion during malaria. [Internet] [Doctoral dissertation]. University of Manchester; 2019. [cited 2021 Apr 17]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319548.

Council of Science Editors:

Dookie R. Dissecting the molecular mechanisms of CD4+ T cell exhaustion during malaria. [Doctoral Dissertation]. University of Manchester; 2019. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319548


Freie Universität Berlin

12. Soll, Dominik. Regulierung humaner T-Zelleigenschaften durch das Mikromilieu.

Degree: 2020, Freie Universität Berlin

T-Zellen stellen einen zentralen Bestandteil des Immunsystems des menschlichen Körpers dar. Man unterscheidet CD4+ T-Helferzellen und CD8+ zytotoxische T-Zellen, deren jeweilige Funktionen vom jeweils umgebenden… (more)

Subjects/Keywords: sodium chloride; T cell; Th17 cell; cytotoxic T cell; ddc:610

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APA (6th Edition):

Soll, D. (2020). Regulierung humaner T-Zelleigenschaften durch das Mikromilieu. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-27581

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Soll, Dominik. “Regulierung humaner T-Zelleigenschaften durch das Mikromilieu.” 2020. Thesis, Freie Universität Berlin. Accessed April 17, 2021. http://dx.doi.org/10.17169/refubium-27581.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Soll, Dominik. “Regulierung humaner T-Zelleigenschaften durch das Mikromilieu.” 2020. Web. 17 Apr 2021.

Vancouver:

Soll D. Regulierung humaner T-Zelleigenschaften durch das Mikromilieu. [Internet] [Thesis]. Freie Universität Berlin; 2020. [cited 2021 Apr 17]. Available from: http://dx.doi.org/10.17169/refubium-27581.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Soll D. Regulierung humaner T-Zelleigenschaften durch das Mikromilieu. [Thesis]. Freie Universität Berlin; 2020. Available from: http://dx.doi.org/10.17169/refubium-27581

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

13. Villanueva, Jeanette Elizabeth. TRAF proteins and their role in T cell effector function and homeostasis.

Degree: Clinical School - St Vincent's Hospital, 2014, University of New South Wales

 TNF-receptor associated factor (TRAF) 2 integrates multiple TNF-family signaling pathways in T cells making it a unifying target for immunomodulation based on co-stimulation. This thesis… (more)

Subjects/Keywords: T cell; TRAF2; Transplantation; CD8 T cell; NKT cell; IL-15

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APA (6th Edition):

Villanueva, J. E. (2014). TRAF proteins and their role in T cell effector function and homeostasis. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/53661 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12356/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Villanueva, Jeanette Elizabeth. “TRAF proteins and their role in T cell effector function and homeostasis.” 2014. Doctoral Dissertation, University of New South Wales. Accessed April 17, 2021. http://handle.unsw.edu.au/1959.4/53661 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12356/SOURCE02?view=true.

MLA Handbook (7th Edition):

Villanueva, Jeanette Elizabeth. “TRAF proteins and their role in T cell effector function and homeostasis.” 2014. Web. 17 Apr 2021.

Vancouver:

Villanueva JE. TRAF proteins and their role in T cell effector function and homeostasis. [Internet] [Doctoral dissertation]. University of New South Wales; 2014. [cited 2021 Apr 17]. Available from: http://handle.unsw.edu.au/1959.4/53661 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12356/SOURCE02?view=true.

Council of Science Editors:

Villanueva JE. TRAF proteins and their role in T cell effector function and homeostasis. [Doctoral Dissertation]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/53661 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12356/SOURCE02?view=true


University of Cincinnati

14. Kurtulus, Sema. Mechanisms Regulating Survival of Effector and Memory CD8+ T Cells.

Degree: PhD, Medicine: Immunology, 2013, University of Cincinnati

 Naive T cells recruited to respond to an infection often undergo 15-20 rounds of cell division. After the infection is cleared, most of these T(more)

Subjects/Keywords: Immunology; CD8+ T cell; Bim; Bcl-2; effector T cell; memory T cell; apoptosis

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APA (6th Edition):

Kurtulus, S. (2013). Mechanisms Regulating Survival of Effector and Memory CD8+ T Cells. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1368026339

Chicago Manual of Style (16th Edition):

Kurtulus, Sema. “Mechanisms Regulating Survival of Effector and Memory CD8+ T Cells.” 2013. Doctoral Dissertation, University of Cincinnati. Accessed April 17, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1368026339.

MLA Handbook (7th Edition):

Kurtulus, Sema. “Mechanisms Regulating Survival of Effector and Memory CD8+ T Cells.” 2013. Web. 17 Apr 2021.

Vancouver:

Kurtulus S. Mechanisms Regulating Survival of Effector and Memory CD8+ T Cells. [Internet] [Doctoral dissertation]. University of Cincinnati; 2013. [cited 2021 Apr 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1368026339.

Council of Science Editors:

Kurtulus S. Mechanisms Regulating Survival of Effector and Memory CD8+ T Cells. [Doctoral Dissertation]. University of Cincinnati; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1368026339


Freie Universität Berlin

15. Wendering, Désirée Jacqueline. Regulatorische T-Zellen in der adoptiven Zelltherapie: Von der Charakterisierung von Subpopulationen bis zur funktionellen Testung.

Degree: 2021, Freie Universität Berlin

 Die aus dem Thymus stammenden regulatorischen T-Zellen (tTREG) spielen eine wichtige Rolle bei der Unterdrückung unerwünschter Immunantworten in vivo. Immuntherapien mit humanen tTREG sind daher… (more)

Subjects/Keywords: Regulatory T cell subsets; Adoptive T cell therapy; Regulatory T cell functional tests; ddc:610

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APA (6th Edition):

Wendering, D. J. (2021). Regulatorische T-Zellen in der adoptiven Zelltherapie: Von der Charakterisierung von Subpopulationen bis zur funktionellen Testung. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-28898

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wendering, Désirée Jacqueline. “Regulatorische T-Zellen in der adoptiven Zelltherapie: Von der Charakterisierung von Subpopulationen bis zur funktionellen Testung.” 2021. Thesis, Freie Universität Berlin. Accessed April 17, 2021. http://dx.doi.org/10.17169/refubium-28898.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wendering, Désirée Jacqueline. “Regulatorische T-Zellen in der adoptiven Zelltherapie: Von der Charakterisierung von Subpopulationen bis zur funktionellen Testung.” 2021. Web. 17 Apr 2021.

Vancouver:

Wendering DJ. Regulatorische T-Zellen in der adoptiven Zelltherapie: Von der Charakterisierung von Subpopulationen bis zur funktionellen Testung. [Internet] [Thesis]. Freie Universität Berlin; 2021. [cited 2021 Apr 17]. Available from: http://dx.doi.org/10.17169/refubium-28898.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wendering DJ. Regulatorische T-Zellen in der adoptiven Zelltherapie: Von der Charakterisierung von Subpopulationen bis zur funktionellen Testung. [Thesis]. Freie Universität Berlin; 2021. Available from: http://dx.doi.org/10.17169/refubium-28898

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas – Austin

16. -6996-7733. Role of TCR affinity in function, proliferation, memory generation, and exhaustion during human viral infection.

Degree: PhD, Biomedical Engineering, 2019, University of Texas – Austin

 TCR affinity is a strong predictor of T cell function, proliferation, exhaustion, and generation of immune memory. 2D TCR affinity quantification is derived from direct… (more)

Subjects/Keywords: T cell; TCR affinity; T cell function; T cell exhaustion; Human viral infection; Cytomegalovirus

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APA (6th Edition):

-6996-7733. (2019). Role of TCR affinity in function, proliferation, memory generation, and exhaustion during human viral infection. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://dx.doi.org/10.26153/tsw/12039

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-6996-7733. “Role of TCR affinity in function, proliferation, memory generation, and exhaustion during human viral infection.” 2019. Doctoral Dissertation, University of Texas – Austin. Accessed April 17, 2021. http://dx.doi.org/10.26153/tsw/12039.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-6996-7733. “Role of TCR affinity in function, proliferation, memory generation, and exhaustion during human viral infection.” 2019. Web. 17 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-6996-7733. Role of TCR affinity in function, proliferation, memory generation, and exhaustion during human viral infection. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2019. [cited 2021 Apr 17]. Available from: http://dx.doi.org/10.26153/tsw/12039.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-6996-7733. Role of TCR affinity in function, proliferation, memory generation, and exhaustion during human viral infection. [Doctoral Dissertation]. University of Texas – Austin; 2019. Available from: http://dx.doi.org/10.26153/tsw/12039

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Rochester

17. Chapman, Lesley Maraina. MicroRNA-451 Regulates T Helper Cell Responses.

Degree: PhD, 2015, University of Rochester

 Background: Malaria parasite evasion of host defense mechanisms leads to uncontrolled parasite growth, anemia, morbidity, and often mortality. A number of studies have shown that… (more)

Subjects/Keywords: CD4 T Cell; Plasmodium; MYC

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APA (6th Edition):

Chapman, L. M. (2015). MicroRNA-451 Regulates T Helper Cell Responses. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30109

Chicago Manual of Style (16th Edition):

Chapman, Lesley Maraina. “MicroRNA-451 Regulates T Helper Cell Responses.” 2015. Doctoral Dissertation, University of Rochester. Accessed April 17, 2021. http://hdl.handle.net/1802/30109.

MLA Handbook (7th Edition):

Chapman, Lesley Maraina. “MicroRNA-451 Regulates T Helper Cell Responses.” 2015. Web. 17 Apr 2021.

Vancouver:

Chapman LM. MicroRNA-451 Regulates T Helper Cell Responses. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1802/30109.

Council of Science Editors:

Chapman LM. MicroRNA-451 Regulates T Helper Cell Responses. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/30109

18. Purushothaman, Divya. Regulation of activated T cell death; -.

Degree: Chemical and Biotechnology, 2014, SASTRA University

Abstract included

Reference p116-134, List of publications p114-115, List of abbreviations p135-137, Summary included

Advisors/Committee Members: Sarin, Apurva.

Subjects/Keywords: Immune System; T cell death

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APA (6th Edition):

Purushothaman, D. (2014). Regulation of activated T cell death; -. (Thesis). SASTRA University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/22846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Purushothaman, Divya. “Regulation of activated T cell death; -.” 2014. Thesis, SASTRA University. Accessed April 17, 2021. http://shodhganga.inflibnet.ac.in/handle/10603/22846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Purushothaman, Divya. “Regulation of activated T cell death; -.” 2014. Web. 17 Apr 2021.

Vancouver:

Purushothaman D. Regulation of activated T cell death; -. [Internet] [Thesis]. SASTRA University; 2014. [cited 2021 Apr 17]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/22846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Purushothaman D. Regulation of activated T cell death; -. [Thesis]. SASTRA University; 2014. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/22846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

19. Brignall, Ruth. The single-cell and gene expression analysis of T cell activation and signalling.

Degree: 2016, University of Manchester

 Our immune system must be able to rapidly fight against pathogens, but at the same time be tightly regulated to prevent harmful autoimmune and inflammatory… (more)

Subjects/Keywords: T cell; Signalling; NFAT

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APA (6th Edition):

Brignall, R. (2016). The single-cell and gene expression analysis of T cell activation and signalling. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305715

Chicago Manual of Style (16th Edition):

Brignall, Ruth. “The single-cell and gene expression analysis of T cell activation and signalling.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 17, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305715.

MLA Handbook (7th Edition):

Brignall, Ruth. “The single-cell and gene expression analysis of T cell activation and signalling.” 2016. Web. 17 Apr 2021.

Vancouver:

Brignall R. The single-cell and gene expression analysis of T cell activation and signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 17]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305715.

Council of Science Editors:

Brignall R. The single-cell and gene expression analysis of T cell activation and signalling. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305715


Vanderbilt University

20. Conrad, Joseph Allen. Clonotypic Dominance within the HIV-epitope-specific T cell receptor repertoire correlates with phenotypic and functional impairment.

Degree: PhD, Microbiology and Immunology, 2011, Vanderbilt University

 HIV-epitope-specific T cell responses are critical components of the natural immune response to HIV infection, but these cells often become dysfunctional in chronic infection. Structural… (more)

Subjects/Keywords: TCR; HIV; T cell; CTL

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APA (6th Edition):

Conrad, J. A. (2011). Clonotypic Dominance within the HIV-epitope-specific T cell receptor repertoire correlates with phenotypic and functional impairment. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12236

Chicago Manual of Style (16th Edition):

Conrad, Joseph Allen. “Clonotypic Dominance within the HIV-epitope-specific T cell receptor repertoire correlates with phenotypic and functional impairment.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed April 17, 2021. http://hdl.handle.net/1803/12236.

MLA Handbook (7th Edition):

Conrad, Joseph Allen. “Clonotypic Dominance within the HIV-epitope-specific T cell receptor repertoire correlates with phenotypic and functional impairment.” 2011. Web. 17 Apr 2021.

Vancouver:

Conrad JA. Clonotypic Dominance within the HIV-epitope-specific T cell receptor repertoire correlates with phenotypic and functional impairment. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1803/12236.

Council of Science Editors:

Conrad JA. Clonotypic Dominance within the HIV-epitope-specific T cell receptor repertoire correlates with phenotypic and functional impairment. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/12236


University of Saskatchewan

21. Rudulier, Christopher. CD4+ T cell interactions through CD28/B7 molecules affects their Th1/Th2 phenotype.

Degree: 2011, University of Saskatchewan

 The Th1/Th2 phenotype of the immune response generated against a pathogen or disease can have a profound impact upon the survival of the host. Thus,… (more)

Subjects/Keywords: CD4 T cell differentiation

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APA (6th Edition):

Rudulier, C. (2011). CD4+ T cell interactions through CD28/B7 molecules affects their Th1/Th2 phenotype. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/ETD-2011-12-282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rudulier, Christopher. “CD4+ T cell interactions through CD28/B7 molecules affects their Th1/Th2 phenotype.” 2011. Thesis, University of Saskatchewan. Accessed April 17, 2021. http://hdl.handle.net/10388/ETD-2011-12-282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rudulier, Christopher. “CD4+ T cell interactions through CD28/B7 molecules affects their Th1/Th2 phenotype.” 2011. Web. 17 Apr 2021.

Vancouver:

Rudulier C. CD4+ T cell interactions through CD28/B7 molecules affects their Th1/Th2 phenotype. [Internet] [Thesis]. University of Saskatchewan; 2011. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10388/ETD-2011-12-282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rudulier C. CD4+ T cell interactions through CD28/B7 molecules affects their Th1/Th2 phenotype. [Thesis]. University of Saskatchewan; 2011. Available from: http://hdl.handle.net/10388/ETD-2011-12-282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

22. Afsahi, Arya. Development of novel chimeric receptors for delivery of costimulation to tumor-reactive engineered T cells.

Degree: MSc, 2015, McMaster University

Introduction: Manipulation of the immune system to eliminate cancer, known as cancer immunotherapy, is an emerging field that has shown impressive clinical success and promise.… (more)

Subjects/Keywords: Immunology; T cell; Cancer; Immunotherapy

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APA (6th Edition):

Afsahi, A. (2015). Development of novel chimeric receptors for delivery of costimulation to tumor-reactive engineered T cells. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/18413

Chicago Manual of Style (16th Edition):

Afsahi, Arya. “Development of novel chimeric receptors for delivery of costimulation to tumor-reactive engineered T cells.” 2015. Masters Thesis, McMaster University. Accessed April 17, 2021. http://hdl.handle.net/11375/18413.

MLA Handbook (7th Edition):

Afsahi, Arya. “Development of novel chimeric receptors for delivery of costimulation to tumor-reactive engineered T cells.” 2015. Web. 17 Apr 2021.

Vancouver:

Afsahi A. Development of novel chimeric receptors for delivery of costimulation to tumor-reactive engineered T cells. [Internet] [Masters thesis]. McMaster University; 2015. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/11375/18413.

Council of Science Editors:

Afsahi A. Development of novel chimeric receptors for delivery of costimulation to tumor-reactive engineered T cells. [Masters Thesis]. McMaster University; 2015. Available from: http://hdl.handle.net/11375/18413

23. Helou, Ynes. Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics.

Degree: PhD, Molecular Pharmacology, Physiology, and Biotechnology, 2015, Brown University

 Activation of T cells via the T cell receptor (TCR) is a crucial event in the adaptive immune response, and is mediated by a delicate… (more)

Subjects/Keywords: T cell receptor signaling

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APA (6th Edition):

Helou, Y. (2015). Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:419500/

Chicago Manual of Style (16th Edition):

Helou, Ynes. “Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics.” 2015. Doctoral Dissertation, Brown University. Accessed April 17, 2021. https://repository.library.brown.edu/studio/item/bdr:419500/.

MLA Handbook (7th Edition):

Helou, Ynes. “Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics.” 2015. Web. 17 Apr 2021.

Vancouver:

Helou Y. Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics. [Internet] [Doctoral dissertation]. Brown University; 2015. [cited 2021 Apr 17]. Available from: https://repository.library.brown.edu/studio/item/bdr:419500/.

Council of Science Editors:

Helou Y. Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics. [Doctoral Dissertation]. Brown University; 2015. Available from: https://repository.library.brown.edu/studio/item/bdr:419500/


Addis Ababa University

24. Haile, Benti. Lost opportunities to complete CD4+ T cell testing among HIV positive patients attending selected health centers in Afar region Northeast Ethiopia, 2014.

Degree: 2014, Addis Ababa University

 Background: In resource limited settings CD4+ T cell count (CD4 testing) facility is not available in peripheral areas and often either patients need to travel… (more)

Subjects/Keywords: CD4+ T cell count; HIV

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APA (6th Edition):

Haile, B. (2014). Lost opportunities to complete CD4+ T cell testing among HIV positive patients attending selected health centers in Afar region Northeast Ethiopia, 2014. (Thesis). Addis Ababa University. Retrieved from http://etd.aau.edu.et/dspace/handle/123456789/5655

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Haile, Benti. “Lost opportunities to complete CD4+ T cell testing among HIV positive patients attending selected health centers in Afar region Northeast Ethiopia, 2014. ” 2014. Thesis, Addis Ababa University. Accessed April 17, 2021. http://etd.aau.edu.et/dspace/handle/123456789/5655.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Haile, Benti. “Lost opportunities to complete CD4+ T cell testing among HIV positive patients attending selected health centers in Afar region Northeast Ethiopia, 2014. ” 2014. Web. 17 Apr 2021.

Vancouver:

Haile B. Lost opportunities to complete CD4+ T cell testing among HIV positive patients attending selected health centers in Afar region Northeast Ethiopia, 2014. [Internet] [Thesis]. Addis Ababa University; 2014. [cited 2021 Apr 17]. Available from: http://etd.aau.edu.et/dspace/handle/123456789/5655.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Haile B. Lost opportunities to complete CD4+ T cell testing among HIV positive patients attending selected health centers in Afar region Northeast Ethiopia, 2014. [Thesis]. Addis Ababa University; 2014. Available from: http://etd.aau.edu.et/dspace/handle/123456789/5655

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

25. Rich, Kevin Robert. The Role of Transforming Growth Factor-Beta in T-Cell Signalling.

Degree: 2016, University of Manchester

Transforming Growth Factor beta (TGFβ) is a pivotal cytokine in regulating our immune responses. TGFβ exerts many effects through T-cells, which are an important cell(more)

Subjects/Keywords: TGFb; T-cell; Smad

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APA (6th Edition):

Rich, K. R. (2016). The Role of Transforming Growth Factor-Beta in T-Cell Signalling. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:301779

Chicago Manual of Style (16th Edition):

Rich, Kevin Robert. “The Role of Transforming Growth Factor-Beta in T-Cell Signalling.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 17, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:301779.

MLA Handbook (7th Edition):

Rich, Kevin Robert. “The Role of Transforming Growth Factor-Beta in T-Cell Signalling.” 2016. Web. 17 Apr 2021.

Vancouver:

Rich KR. The Role of Transforming Growth Factor-Beta in T-Cell Signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 17]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:301779.

Council of Science Editors:

Rich KR. The Role of Transforming Growth Factor-Beta in T-Cell Signalling. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:301779


University of Illinois – Chicago

26. Ryan Payseur, Bridgett K. Host T Cell Responses to Malaria, Acquired Immunodeficiency Virus, and Listeria Infections.

Degree: 2012, University of Illinois – Chicago

 Understanding of T cell responses during infections will be vital to rational vaccine design. Malaria and AIDS represent two leading causes of death from infectious… (more)

Subjects/Keywords: T cell; malaria; listeria; AIDS

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APA (6th Edition):

Ryan Payseur, B. K. (2012). Host T Cell Responses to Malaria, Acquired Immunodeficiency Virus, and Listeria Infections. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ryan Payseur, Bridgett K. “Host T Cell Responses to Malaria, Acquired Immunodeficiency Virus, and Listeria Infections.” 2012. Thesis, University of Illinois – Chicago. Accessed April 17, 2021. http://hdl.handle.net/10027/9078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ryan Payseur, Bridgett K. “Host T Cell Responses to Malaria, Acquired Immunodeficiency Virus, and Listeria Infections.” 2012. Web. 17 Apr 2021.

Vancouver:

Ryan Payseur BK. Host T Cell Responses to Malaria, Acquired Immunodeficiency Virus, and Listeria Infections. [Internet] [Thesis]. University of Illinois – Chicago; 2012. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10027/9078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ryan Payseur BK. Host T Cell Responses to Malaria, Acquired Immunodeficiency Virus, and Listeria Infections. [Thesis]. University of Illinois – Chicago; 2012. Available from: http://hdl.handle.net/10027/9078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

27. Rich, Kevin. The role of Transforming Growth Factor-beta in T-cell signalling.

Degree: PhD, 2016, University of Manchester

 Transforming Growth Factor beta (TGFβ) is a pivotal cytokine in regulating our immune responses. TGFβ exerts many effects through T-cells, which are an important cell(more)

Subjects/Keywords: TGFb; T-cell; Smad

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APA (6th Edition):

Rich, K. (2016). The role of Transforming Growth Factor-beta in T-cell signalling. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-transforming-growth-factorbeta-in-tcell-signalling(a04e7d47-b6f7-4524-8535-29e0a5ee0d5d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771322

Chicago Manual of Style (16th Edition):

Rich, Kevin. “The role of Transforming Growth Factor-beta in T-cell signalling.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 17, 2021. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-transforming-growth-factorbeta-in-tcell-signalling(a04e7d47-b6f7-4524-8535-29e0a5ee0d5d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771322.

MLA Handbook (7th Edition):

Rich, Kevin. “The role of Transforming Growth Factor-beta in T-cell signalling.” 2016. Web. 17 Apr 2021.

Vancouver:

Rich K. The role of Transforming Growth Factor-beta in T-cell signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 17]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-transforming-growth-factorbeta-in-tcell-signalling(a04e7d47-b6f7-4524-8535-29e0a5ee0d5d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771322.

Council of Science Editors:

Rich K. The role of Transforming Growth Factor-beta in T-cell signalling. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-transforming-growth-factorbeta-in-tcell-signalling(a04e7d47-b6f7-4524-8535-29e0a5ee0d5d).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771322


University of Manchester

28. Brignall, Ruth. The single-cell and gene expression analysis of T cell activation and signalling.

Degree: PhD, 2016, University of Manchester

 Our immune system must be able to rapidly fight against pathogens, but at the same time be tightly regulated to prevent harmful autoimmune and inflammatory… (more)

Subjects/Keywords: 616.07; T cell; Signalling; NFAT

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APA (6th Edition):

Brignall, R. (2016). The single-cell and gene expression analysis of T cell activation and signalling. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-singlecell-and-gene-expression-analysis-of-t-cell-activation-and-signalling(d4f814bf-b4f7-4e23-8130-5ba4b9f38c13).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697799

Chicago Manual of Style (16th Edition):

Brignall, Ruth. “The single-cell and gene expression analysis of T cell activation and signalling.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 17, 2021. https://www.research.manchester.ac.uk/portal/en/theses/the-singlecell-and-gene-expression-analysis-of-t-cell-activation-and-signalling(d4f814bf-b4f7-4e23-8130-5ba4b9f38c13).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697799.

MLA Handbook (7th Edition):

Brignall, Ruth. “The single-cell and gene expression analysis of T cell activation and signalling.” 2016. Web. 17 Apr 2021.

Vancouver:

Brignall R. The single-cell and gene expression analysis of T cell activation and signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 17]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-singlecell-and-gene-expression-analysis-of-t-cell-activation-and-signalling(d4f814bf-b4f7-4e23-8130-5ba4b9f38c13).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697799.

Council of Science Editors:

Brignall R. The single-cell and gene expression analysis of T cell activation and signalling. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-singlecell-and-gene-expression-analysis-of-t-cell-activation-and-signalling(d4f814bf-b4f7-4e23-8130-5ba4b9f38c13).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697799


University of Minnesota

29. Tucker, Christopher. The Divergent Role of Co-receptors and Cytokines in the Immune Response to Cancer and Self.

Degree: PhD, Microbiology, Immunology and Cancer Biology, 2020, University of Minnesota

 The mammalian immune system has evolved over millennia to respond to countless biologic factors and environmental cues. This exquisite system has been shaped over time,… (more)

Subjects/Keywords: Cancer; Immunology; Immunotherapy; T-cell

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tucker, C. (2020). The Divergent Role of Co-receptors and Cytokines in the Immune Response to Cancer and Self. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/216816

Chicago Manual of Style (16th Edition):

Tucker, Christopher. “The Divergent Role of Co-receptors and Cytokines in the Immune Response to Cancer and Self.” 2020. Doctoral Dissertation, University of Minnesota. Accessed April 17, 2021. http://hdl.handle.net/11299/216816.

MLA Handbook (7th Edition):

Tucker, Christopher. “The Divergent Role of Co-receptors and Cytokines in the Immune Response to Cancer and Self.” 2020. Web. 17 Apr 2021.

Vancouver:

Tucker C. The Divergent Role of Co-receptors and Cytokines in the Immune Response to Cancer and Self. [Internet] [Doctoral dissertation]. University of Minnesota; 2020. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/11299/216816.

Council of Science Editors:

Tucker C. The Divergent Role of Co-receptors and Cytokines in the Immune Response to Cancer and Self. [Doctoral Dissertation]. University of Minnesota; 2020. Available from: http://hdl.handle.net/11299/216816


University of Illinois – Urbana-Champaign

30. Harris, Daniel Thomas. Engineering and characterizing human T cell receptors for cancer immunotherapies.

Degree: PhD, Biochemistry, 2016, University of Illinois – Urbana-Champaign

 The T cell receptor (TCR) is an heterodimer that binds to a short peptide bound to a product of the major histocompatibility complex (MHC). The… (more)

Subjects/Keywords: Immunotherapy; T Cell Receptor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Harris, D. T. (2016). Engineering and characterizing human T cell receptors for cancer immunotherapies. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/95531

Chicago Manual of Style (16th Edition):

Harris, Daniel Thomas. “Engineering and characterizing human T cell receptors for cancer immunotherapies.” 2016. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed April 17, 2021. http://hdl.handle.net/2142/95531.

MLA Handbook (7th Edition):

Harris, Daniel Thomas. “Engineering and characterizing human T cell receptors for cancer immunotherapies.” 2016. Web. 17 Apr 2021.

Vancouver:

Harris DT. Engineering and characterizing human T cell receptors for cancer immunotherapies. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2016. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/2142/95531.

Council of Science Editors:

Harris DT. Engineering and characterizing human T cell receptors for cancer immunotherapies. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2016. Available from: http://hdl.handle.net/2142/95531

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