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You searched for subject:(T Cell ALL). Showing records 1 – 15 of 15 total matches.

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University of Iowa

1. Vrieze, Katherine Elna. Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia.

Degree: PhD, Molecular and Cellular Biology, 2011, University of Iowa

T-cell acute lymphocytic leukemia (T-ALL) affects approximately 1,500 people per year in the United States, many of them children. The overall survival rate of… (more)

Subjects/Keywords: ETP-ALL; NOTCH1; T-ALL; T-cell Acute Lymphocytic Leukemia; Cell Biology

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APA (6th Edition):

Vrieze, K. E. (2011). Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia. (Doctoral Dissertation). University of Iowa. Retrieved from http://ir.uiowa.edu/etd/2785

Chicago Manual of Style (16th Edition):

Vrieze, Katherine Elna. “Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia.” 2011. Doctoral Dissertation, University of Iowa. Accessed June 21, 2018. http://ir.uiowa.edu/etd/2785.

MLA Handbook (7th Edition):

Vrieze, Katherine Elna. “Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia.” 2011. Web. 21 Jun 2018.

Vancouver:

Vrieze KE. Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia. [Internet] [Doctoral dissertation]. University of Iowa; 2011. [cited 2018 Jun 21]. Available from: http://ir.uiowa.edu/etd/2785.

Council of Science Editors:

Vrieze KE. Using mouse models to investigate the genetics of T-cell acute lymphocytic leukemia. [Doctoral Dissertation]. University of Iowa; 2011. Available from: http://ir.uiowa.edu/etd/2785


University of Kansas

2. Mitchell, Julie Lynne. Multi-Parameter Flow Cytometric Analysis of Ikaros Family Expression in Human T Cell Development and T-ALL.

Degree: PhD, Microbiology, Molecular Genetics & Immunology, 2016, University of Kansas

 The Ikaros family transcription factors are critical regulators of T cell development and leukemogenesis. Loss of function of all five family members leads to an… (more)

Subjects/Keywords: Immunology; Aiolos; Helios; Ikaros; T-ALL; T cell development

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APA (6th Edition):

Mitchell, J. L. (2016). Multi-Parameter Flow Cytometric Analysis of Ikaros Family Expression in Human T Cell Development and T-ALL. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/23987

Chicago Manual of Style (16th Edition):

Mitchell, Julie Lynne. “Multi-Parameter Flow Cytometric Analysis of Ikaros Family Expression in Human T Cell Development and T-ALL.” 2016. Doctoral Dissertation, University of Kansas. Accessed June 21, 2018. http://hdl.handle.net/1808/23987.

MLA Handbook (7th Edition):

Mitchell, Julie Lynne. “Multi-Parameter Flow Cytometric Analysis of Ikaros Family Expression in Human T Cell Development and T-ALL.” 2016. Web. 21 Jun 2018.

Vancouver:

Mitchell JL. Multi-Parameter Flow Cytometric Analysis of Ikaros Family Expression in Human T Cell Development and T-ALL. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2018 Jun 21]. Available from: http://hdl.handle.net/1808/23987.

Council of Science Editors:

Mitchell JL. Multi-Parameter Flow Cytometric Analysis of Ikaros Family Expression in Human T Cell Development and T-ALL. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/23987


Delft University of Technology

3. Gouw, M.R. Gene set–based decomposition of T-ALL with respect to normal T-cell implies two distinct T-ALL progression pathways :.

Degree: 2008, Delft University of Technology

 In this investigation a gene set–based approach is used to compare T-ALL samples to a series of chronologically ordered T-cell development stages. These results revealed… (more)

Subjects/Keywords: T-ALL; T-cell Acute Lymphoblastic leukemia; gene sets; tumorigenesis; stemness

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APA (6th Edition):

Gouw, M. R. (2008). Gene set–based decomposition of T-ALL with respect to normal T-cell implies two distinct T-ALL progression pathways :. (Masters Thesis). Delft University of Technology. Retrieved from http://resolver.tudelft.nl/uuid:567ffb5a-2ba2-46eb-a383-d645848fa477

Chicago Manual of Style (16th Edition):

Gouw, M R. “Gene set–based decomposition of T-ALL with respect to normal T-cell implies two distinct T-ALL progression pathways :.” 2008. Masters Thesis, Delft University of Technology. Accessed June 21, 2018. http://resolver.tudelft.nl/uuid:567ffb5a-2ba2-46eb-a383-d645848fa477.

MLA Handbook (7th Edition):

Gouw, M R. “Gene set–based decomposition of T-ALL with respect to normal T-cell implies two distinct T-ALL progression pathways :.” 2008. Web. 21 Jun 2018.

Vancouver:

Gouw MR. Gene set–based decomposition of T-ALL with respect to normal T-cell implies two distinct T-ALL progression pathways :. [Internet] [Masters thesis]. Delft University of Technology; 2008. [cited 2018 Jun 21]. Available from: http://resolver.tudelft.nl/uuid:567ffb5a-2ba2-46eb-a383-d645848fa477.

Council of Science Editors:

Gouw MR. Gene set–based decomposition of T-ALL with respect to normal T-cell implies two distinct T-ALL progression pathways :. [Masters Thesis]. Delft University of Technology; 2008. Available from: http://resolver.tudelft.nl/uuid:567ffb5a-2ba2-46eb-a383-d645848fa477


Universitat Pompeu Fabra

4. Colomer-Lahiguera, Sara, 1981-. The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia.

Degree: Departament de Ciències Experimentals i de la Salut, 2015, Universitat Pompeu Fabra

 La leucemia linfoblástica aguda de células T (LLA-T) representa el 10 15% de las leucemias pediátricas. La transformación oncogénica de precursores de células T es… (more)

Subjects/Keywords: CDKN1B-p27kip1; T-ALL; Lymphoblastic; MEF2C; SKP2; T-cell; Leukemia; Pediatric; Leucemia; Pediátrica; 616

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APA (6th Edition):

Colomer-Lahiguera, Sara, 1. (2015). The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia. (Thesis). Universitat Pompeu Fabra. Retrieved from http://hdl.handle.net/10803/523487

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Colomer-Lahiguera, Sara, 1981-. “The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia.” 2015. Thesis, Universitat Pompeu Fabra. Accessed June 21, 2018. http://hdl.handle.net/10803/523487.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Colomer-Lahiguera, Sara, 1981-. “The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia.” 2015. Web. 21 Jun 2018.

Vancouver:

Colomer-Lahiguera, Sara 1. The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia. [Internet] [Thesis]. Universitat Pompeu Fabra; 2015. [cited 2018 Jun 21]. Available from: http://hdl.handle.net/10803/523487.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Colomer-Lahiguera, Sara 1. The role of CDKN1B-p27kip1 deregulation in the pathogenesis of pediatric T-cell acute lymphoblastic leukemia. [Thesis]. Universitat Pompeu Fabra; 2015. Available from: http://hdl.handle.net/10803/523487

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

5. Bailis, Will Harrison. Non-instructional roles for Notch signaling in T cell development.

Degree: 2014, University of Pennsylvania

 Mammalian immunity requires the presence of a broad and diverse repertoire of antigen receptors that can recognize the virtually infinite number of pathogenic epitopes encountered… (more)

Subjects/Keywords: Helper T cell; Notch; Notch signaling; T-ALL; T cell; Thymocyte; Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology

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APA (6th Edition):

Bailis, W. H. (2014). Non-instructional roles for Notch signaling in T cell development. (Thesis). University of Pennsylvania. Retrieved from http://repository.upenn.edu/edissertations/1198

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bailis, Will Harrison. “Non-instructional roles for Notch signaling in T cell development.” 2014. Thesis, University of Pennsylvania. Accessed June 21, 2018. http://repository.upenn.edu/edissertations/1198.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bailis, Will Harrison. “Non-instructional roles for Notch signaling in T cell development.” 2014. Web. 21 Jun 2018.

Vancouver:

Bailis WH. Non-instructional roles for Notch signaling in T cell development. [Internet] [Thesis]. University of Pennsylvania; 2014. [cited 2018 Jun 21]. Available from: http://repository.upenn.edu/edissertations/1198.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bailis WH. Non-instructional roles for Notch signaling in T cell development. [Thesis]. University of Pennsylvania; 2014. Available from: http://repository.upenn.edu/edissertations/1198

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

6. Moradi Manesh, Donya. Molecular determinants of T-cell acute lymphoblastic leukaemia sensitivity to the pre-prodrug PR-104.

Degree: Biotechnology & Biomolecular Sciences, 2014, University of New South Wales

 Despite major improvements in the treatment of childhood acute lymphoblastic leukaemia (ALL), curing those patients who relapse remains a significant clinical challenge. Cellular drug resistance… (more)

Subjects/Keywords: T-cell; Acute lymphoblastic leukaemia (ALL); Pre-prodrug PR-104; Childhood; Sensitivity; Bone marrow; Hypoxic; Molecular determinants; Aldo-keto reductase 1C3 (AKR1C3); Cytochrome P450 oxidoreductase (CYPOR); Early T-cell precursor (ETP); T-cell lineage ALL (T-ALL); Xenografts

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APA (6th Edition):

Moradi Manesh, D. (2014). Molecular determinants of T-cell acute lymphoblastic leukaemia sensitivity to the pre-prodrug PR-104. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/53877

Chicago Manual of Style (16th Edition):

Moradi Manesh, Donya. “Molecular determinants of T-cell acute lymphoblastic leukaemia sensitivity to the pre-prodrug PR-104.” 2014. Doctoral Dissertation, University of New South Wales. Accessed June 21, 2018. http://handle.unsw.edu.au/1959.4/53877.

MLA Handbook (7th Edition):

Moradi Manesh, Donya. “Molecular determinants of T-cell acute lymphoblastic leukaemia sensitivity to the pre-prodrug PR-104.” 2014. Web. 21 Jun 2018.

Vancouver:

Moradi Manesh D. Molecular determinants of T-cell acute lymphoblastic leukaemia sensitivity to the pre-prodrug PR-104. [Internet] [Doctoral dissertation]. University of New South Wales; 2014. [cited 2018 Jun 21]. Available from: http://handle.unsw.edu.au/1959.4/53877.

Council of Science Editors:

Moradi Manesh D. Molecular determinants of T-cell acute lymphoblastic leukaemia sensitivity to the pre-prodrug PR-104. [Doctoral Dissertation]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/53877


Université de Montréal

7. Assaker, Gloria. Étude de la régulation de l'activité du ligand Delta dans le cadre de la signalisation Notch .

Degree: 2015, Université de Montréal

 La voie de signalisation Notch est conservée au cours de l'évolution. Elle joue un rôle clé dans le développement, et elle est impliquée dans de… (more)

Subjects/Keywords: Notch; Delta-like; Crible; Screen; Inhibiteurs de protéases; Protease inhibitors; Tmem128; Cdc37; Différenciation des cellules T; T-cell differentiation; LAL-T; T-ALL

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APA (6th Edition):

Assaker, G. (2015). Étude de la régulation de l'activité du ligand Delta dans le cadre de la signalisation Notch . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/13025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Assaker, Gloria. “Étude de la régulation de l'activité du ligand Delta dans le cadre de la signalisation Notch .” 2015. Thesis, Université de Montréal. Accessed June 21, 2018. http://hdl.handle.net/1866/13025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Assaker, Gloria. “Étude de la régulation de l'activité du ligand Delta dans le cadre de la signalisation Notch .” 2015. Web. 21 Jun 2018.

Vancouver:

Assaker G. Étude de la régulation de l'activité du ligand Delta dans le cadre de la signalisation Notch . [Internet] [Thesis]. Université de Montréal; 2015. [cited 2018 Jun 21]. Available from: http://hdl.handle.net/1866/13025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Assaker G. Étude de la régulation de l'activité du ligand Delta dans le cadre de la signalisation Notch . [Thesis]. Université de Montréal; 2015. Available from: http://hdl.handle.net/1866/13025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

8. Zimmerman, Grant Robert. T-ALL LEUKEMIA DYSREGULATES STROMAL BONE MARROW ENVIRONMENT AND DISRUPTS NICHE-STEM CELL SIGNALING AXIS.

Degree: MSs, Pathology, 2015, Case Western Reserve University

 Recent characterization of the bone marrow microenvironment has pointed to comprehensive dysregulation of hematopoietic-stromal interactions in the context of leukemia. We used common oncogenic mutations… (more)

Subjects/Keywords: Pathology; Medicine; Molecular Biology; Bone Marrow; Stromal Cells; Acute Lymphoblastic Leukemia; T-ALL; CXCR4; Notch; Leukemic Stem Cell

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APA (6th Edition):

Zimmerman, G. R. (2015). T-ALL LEUKEMIA DYSREGULATES STROMAL BONE MARROW ENVIRONMENT AND DISRUPTS NICHE-STEM CELL SIGNALING AXIS. (Masters Thesis). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1436293859

Chicago Manual of Style (16th Edition):

Zimmerman, Grant Robert. “T-ALL LEUKEMIA DYSREGULATES STROMAL BONE MARROW ENVIRONMENT AND DISRUPTS NICHE-STEM CELL SIGNALING AXIS.” 2015. Masters Thesis, Case Western Reserve University. Accessed June 21, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1436293859.

MLA Handbook (7th Edition):

Zimmerman, Grant Robert. “T-ALL LEUKEMIA DYSREGULATES STROMAL BONE MARROW ENVIRONMENT AND DISRUPTS NICHE-STEM CELL SIGNALING AXIS.” 2015. Web. 21 Jun 2018.

Vancouver:

Zimmerman GR. T-ALL LEUKEMIA DYSREGULATES STROMAL BONE MARROW ENVIRONMENT AND DISRUPTS NICHE-STEM CELL SIGNALING AXIS. [Internet] [Masters thesis]. Case Western Reserve University; 2015. [cited 2018 Jun 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1436293859.

Council of Science Editors:

Zimmerman GR. T-ALL LEUKEMIA DYSREGULATES STROMAL BONE MARROW ENVIRONMENT AND DISRUPTS NICHE-STEM CELL SIGNALING AXIS. [Masters Thesis]. Case Western Reserve University; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1436293859


University of Western Australia

9. Gottardo, Nicholas G. Oncogenes and prognosis in childhood T-cell acute lymphoblastic leukaemia.

Degree: PhD, 2008, University of Western Australia

 [Truncated abstract] The treatment of childhood acute lymphoblastic leukaemia (ALL) is one of the great success stories of paediatric oncology, transforming a universally fatal disease… (more)

Subjects/Keywords: Acute leukemia in children; Acute leukemia in children; Leukemia in children; Lymphoblastic leukemia in children; Lymphoblastic leukemia in children; Oncogenes; T-cell acute lymphoblastic leukaemia (T-ALL); Oncogenes; Prognosis; Children

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APA (6th Edition):

Gottardo, N. G. (2008). Oncogenes and prognosis in childhood T-cell acute lymphoblastic leukaemia. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=5895&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Gottardo, Nicholas G. “Oncogenes and prognosis in childhood T-cell acute lymphoblastic leukaemia.” 2008. Doctoral Dissertation, University of Western Australia. Accessed June 21, 2018. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=5895&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Gottardo, Nicholas G. “Oncogenes and prognosis in childhood T-cell acute lymphoblastic leukaemia.” 2008. Web. 21 Jun 2018.

Vancouver:

Gottardo NG. Oncogenes and prognosis in childhood T-cell acute lymphoblastic leukaemia. [Internet] [Doctoral dissertation]. University of Western Australia; 2008. [cited 2018 Jun 21]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=5895&local_base=GEN01-INS01.

Council of Science Editors:

Gottardo NG. Oncogenes and prognosis in childhood T-cell acute lymphoblastic leukaemia. [Doctoral Dissertation]. University of Western Australia; 2008. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=5895&local_base=GEN01-INS01


Université de Montréal

10. Tardif, Magalie. Les oncogènes NUP98-PHF23 et NUP98-HOXD13 confèrent un potentiel aberrant d’auto-renouvellement aux progéniteurs thymiques.

Degree: 2016, Université de Montréal

 L’initiation de la leucémogénèse dans la leucémie aigue lymphoblastique (LAL)-T résulte de l’activation aberrante de facteurs de transcription de la lignée lymphocytaire T. Nous démontrons… (more)

Subjects/Keywords: leucémie aigue lymphoblastique-T (LAL-T); thymocytes; cellule souche pré-leucémique (CS-préL); NUP98-PHF23 (NP23); NUP98-HOXD13 (NHD13); auto-renouvellement; reprogrammation; souris transgénique; T-cell acute lymphoblastic leukemia (T-ALL); preleukemic stem cell (pre-LSC); self-renewal; transgenic mouse model

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APA (6th Edition):

Tardif, M. (2016). Les oncogènes NUP98-PHF23 et NUP98-HOXD13 confèrent un potentiel aberrant d’auto-renouvellement aux progéniteurs thymiques. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/13858

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tardif, Magalie. “Les oncogènes NUP98-PHF23 et NUP98-HOXD13 confèrent un potentiel aberrant d’auto-renouvellement aux progéniteurs thymiques. ” 2016. Thesis, Université de Montréal. Accessed June 21, 2018. http://hdl.handle.net/1866/13858.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tardif, Magalie. “Les oncogènes NUP98-PHF23 et NUP98-HOXD13 confèrent un potentiel aberrant d’auto-renouvellement aux progéniteurs thymiques. ” 2016. Web. 21 Jun 2018.

Vancouver:

Tardif M. Les oncogènes NUP98-PHF23 et NUP98-HOXD13 confèrent un potentiel aberrant d’auto-renouvellement aux progéniteurs thymiques. [Internet] [Thesis]. Université de Montréal; 2016. [cited 2018 Jun 21]. Available from: http://hdl.handle.net/1866/13858.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tardif M. Les oncogènes NUP98-PHF23 et NUP98-HOXD13 confèrent un potentiel aberrant d’auto-renouvellement aux progéniteurs thymiques. [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/13858

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

11. Hurton, Lenka. TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY.

Degree: PhD, 2014, Texas Medical Center

  Tethered IL-15 to augment the therapeutic potential of T cells expressing chimeric antigen receptor: Maintaining memory potential, persistence, and antitumor activity Adoptive immunotherapy can… (more)

Subjects/Keywords: immunotherapy; cytokines; IL-15; chimeric antigen receptor; T-cell persistence; memory; memory stem cell; leukemia; B-ALL; Medical Biotechnology; Medical Immunology; Medicine and Health Sciences; Other Immunology and Infectious Disease

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APA (6th Edition):

Hurton, L. (2014). TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/421

Chicago Manual of Style (16th Edition):

Hurton, Lenka. “TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY.” 2014. Doctoral Dissertation, Texas Medical Center. Accessed June 21, 2018. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/421.

MLA Handbook (7th Edition):

Hurton, Lenka. “TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY.” 2014. Web. 21 Jun 2018.

Vancouver:

Hurton L. TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY. [Internet] [Doctoral dissertation]. Texas Medical Center; 2014. [cited 2018 Jun 21]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/421.

Council of Science Editors:

Hurton L. TETHERED IL-15 TO AUGMENT THE THERAPEUTIC POTENTIAL OF T CELLS EXPRESSING CHIMERIC ANTIGEN RECEPTOR: MAINTAINING MEMORY POTENTIAL, PERSISTENCE, AND ANTITUMOR ACTIVITY. [Doctoral Dissertation]. Texas Medical Center; 2014. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/421

12. Tanguy Schmidt, Aline. Les leucémies aiguës lymphoblastiques en 2015 : contribution des facteurs de risque cytogénétiques et moléculaires à une thérapeutique adaptée : Acute leukemia lymphoblastic in 2015 : contribution of the oncogenic and molecular risk factors to an adapted treatment.

Degree: Docteur es, Medecine, 2015, Angers

Les leucémies aiguës (LA) sont un groupe hétérogène d'hémopathies malignes dues à latransformation oncogénique clonale des cellules souches hématopoïétiques (CSH). On distingue les LA myéloblastiques… (more)

Subjects/Keywords: Leucémie aiguë lymphoblastique; LAL à Ph1; Lal t; Résistance aux inhibiteurs de tyrosine kinase; Mutations Notch, FBXW7, RAS et PTEN; Groupe pronostique; Acute lymphoblastic leukaemia; Ph + ALL; T-Cell ALL; Resistance to tyrosine kinase inhibitors; Notch, FBXW7, RAS, and PTEN mutations; Prognostic groups; 616.994

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APA (6th Edition):

Tanguy Schmidt, A. (2015). Les leucémies aiguës lymphoblastiques en 2015 : contribution des facteurs de risque cytogénétiques et moléculaires à une thérapeutique adaptée : Acute leukemia lymphoblastic in 2015 : contribution of the oncogenic and molecular risk factors to an adapted treatment. (Doctoral Dissertation). Angers. Retrieved from http://www.theses.fr/2015ANGE0041

Chicago Manual of Style (16th Edition):

Tanguy Schmidt, Aline. “Les leucémies aiguës lymphoblastiques en 2015 : contribution des facteurs de risque cytogénétiques et moléculaires à une thérapeutique adaptée : Acute leukemia lymphoblastic in 2015 : contribution of the oncogenic and molecular risk factors to an adapted treatment.” 2015. Doctoral Dissertation, Angers. Accessed June 21, 2018. http://www.theses.fr/2015ANGE0041.

MLA Handbook (7th Edition):

Tanguy Schmidt, Aline. “Les leucémies aiguës lymphoblastiques en 2015 : contribution des facteurs de risque cytogénétiques et moléculaires à une thérapeutique adaptée : Acute leukemia lymphoblastic in 2015 : contribution of the oncogenic and molecular risk factors to an adapted treatment.” 2015. Web. 21 Jun 2018.

Vancouver:

Tanguy Schmidt A. Les leucémies aiguës lymphoblastiques en 2015 : contribution des facteurs de risque cytogénétiques et moléculaires à une thérapeutique adaptée : Acute leukemia lymphoblastic in 2015 : contribution of the oncogenic and molecular risk factors to an adapted treatment. [Internet] [Doctoral dissertation]. Angers; 2015. [cited 2018 Jun 21]. Available from: http://www.theses.fr/2015ANGE0041.

Council of Science Editors:

Tanguy Schmidt A. Les leucémies aiguës lymphoblastiques en 2015 : contribution des facteurs de risque cytogénétiques et moléculaires à une thérapeutique adaptée : Acute leukemia lymphoblastic in 2015 : contribution of the oncogenic and molecular risk factors to an adapted treatment. [Doctoral Dissertation]. Angers; 2015. Available from: http://www.theses.fr/2015ANGE0041

13. Kazheunikava, Larysa. Impact fonctionnel de l' oncogène TLX3 sur la thymopoïse dans les leucémies aiguës lymphoblastiques T . : Functional impact of the TLX3 oncogene on T-cell development in T-cell acute lymphoblastic leukemia.

Degree: Docteur es, Immunologie, 2012, Aix Marseille Université

Les membres de la famille Homeobox jouent un rôle critique dans le développement hématopoïétique normal. L'expression ectopique des gènes Homeobox provoque des désordres dans l'hématopoïèse… (more)

Subjects/Keywords: Lal-t; TLX3 proto-oncogène; Développement lymphocytaire T; Facteur de transcription; Ets1; Runx1; Enhancers; Éléments cis-régulateurs; T-all; TLX3 proto-oncogene; T-cell development; Transcription factor; Ets1; Runx1; Enhancers; Cis-regulatory elements

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kazheunikava, L. (2012). Impact fonctionnel de l' oncogène TLX3 sur la thymopoïse dans les leucémies aiguës lymphoblastiques T . : Functional impact of the TLX3 oncogene on T-cell development in T-cell acute lymphoblastic leukemia. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2012AIXM4046

Chicago Manual of Style (16th Edition):

Kazheunikava, Larysa. “Impact fonctionnel de l' oncogène TLX3 sur la thymopoïse dans les leucémies aiguës lymphoblastiques T . : Functional impact of the TLX3 oncogene on T-cell development in T-cell acute lymphoblastic leukemia.” 2012. Doctoral Dissertation, Aix Marseille Université. Accessed June 21, 2018. http://www.theses.fr/2012AIXM4046.

MLA Handbook (7th Edition):

Kazheunikava, Larysa. “Impact fonctionnel de l' oncogène TLX3 sur la thymopoïse dans les leucémies aiguës lymphoblastiques T . : Functional impact of the TLX3 oncogene on T-cell development in T-cell acute lymphoblastic leukemia.” 2012. Web. 21 Jun 2018.

Vancouver:

Kazheunikava L. Impact fonctionnel de l' oncogène TLX3 sur la thymopoïse dans les leucémies aiguës lymphoblastiques T . : Functional impact of the TLX3 oncogene on T-cell development in T-cell acute lymphoblastic leukemia. [Internet] [Doctoral dissertation]. Aix Marseille Université 2012. [cited 2018 Jun 21]. Available from: http://www.theses.fr/2012AIXM4046.

Council of Science Editors:

Kazheunikava L. Impact fonctionnel de l' oncogène TLX3 sur la thymopoïse dans les leucémies aiguës lymphoblastiques T . : Functional impact of the TLX3 oncogene on T-cell development in T-cell acute lymphoblastic leukemia. [Doctoral Dissertation]. Aix Marseille Université 2012. Available from: http://www.theses.fr/2012AIXM4046


Université Catholique de Louvain

14. Grau, Carlos. Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic.

Degree: 2008, Université Catholique de Louvain

 La leucémie aiguë lymphoblastique T (LAL-T) est une maladie néoplasique qui se développe à partir d'un précurseur hématopoiétique T ayant accumulé des anomalies oncogéniques. C'est… (more)

Subjects/Keywords: T-cell acute lymphoblastic leukemia; LAL-T; Chimeric protein; Protéine chimérique; T-ALL; Episome; Leucémie aiguë lymphoblastique T; Gene amplification; Tyrosine kinase; Leucémie; Leukemia; Hsr; Fusion gene

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Grau, C. (2008). Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/21344

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Grau, Carlos. “Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic.” 2008. Thesis, Université Catholique de Louvain. Accessed June 21, 2018. http://hdl.handle.net/2078.1/21344.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Grau, Carlos. “Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic.” 2008. Web. 21 Jun 2018.

Vancouver:

Grau C. Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic. [Internet] [Thesis]. Université Catholique de Louvain; 2008. [cited 2018 Jun 21]. Available from: http://hdl.handle.net/2078.1/21344.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Grau C. Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic. [Thesis]. Université Catholique de Louvain; 2008. Available from: http://hdl.handle.net/2078.1/21344

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Catholique de Louvain

15. Graux, Carlos. Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic.

Degree: 2008, Université Catholique de Louvain

T-cell acute lymphoblastic leukemia (T-ALL) is a neoplastic disorder that develops from a single hematopoietic T-cell precursor that acquired oncogenic anomalies. T-ALL is a heterogeneous… (more)

Subjects/Keywords: Leucémie aiguë lymphoblastique T; LAL-T; Protéine chimérique; Chimeric protein; Fusion gene; T-cell acute lymphoblastic leukemia; Episome; Gene amplification; Tyrosine kinase; Leucémie; Hsr; Leukemia; T-ALL

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Graux, C. (2008). Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/30478

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Graux, Carlos. “Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic.” 2008. Thesis, Université Catholique de Louvain. Accessed June 21, 2018. http://hdl.handle.net/2078.1/30478.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Graux, Carlos. “Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic.” 2008. Web. 21 Jun 2018.

Vancouver:

Graux C. Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic. [Internet] [Thesis]. Université Catholique de Louvain; 2008. [cited 2018 Jun 21]. Available from: http://hdl.handle.net/2078.1/30478.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Graux C. Rearrangements and amplification of the ABL1 gene as an example of kinase activation in T-cell acute lymphoblastic. [Thesis]. Université Catholique de Louvain; 2008. Available from: http://hdl.handle.net/2078.1/30478

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.