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You searched for subject:(Synthetic lethality). Showing records 1 – 30 of 52 total matches.

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University of Saskatchewan

1. Sumi, Sharmin Sultana. Characterization of PIK3CD as a Cancer Drug Target.

Degree: 2018, University of Saskatchewan

 Peroxisome proliferator activated Receptors (PPARs) belong to the nuclear receptor super family and are ligand activated transcription factors regulating the expression of a wide variety… (more)

Subjects/Keywords: PPARγ; PIK3CD; hTERT; Synthetic Dosages Lethality

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APA (6th Edition):

Sumi, S. S. (2018). Characterization of PIK3CD as a Cancer Drug Target. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/11491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sumi, Sharmin Sultana. “Characterization of PIK3CD as a Cancer Drug Target.” 2018. Thesis, University of Saskatchewan. Accessed April 14, 2021. http://hdl.handle.net/10388/11491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sumi, Sharmin Sultana. “Characterization of PIK3CD as a Cancer Drug Target.” 2018. Web. 14 Apr 2021.

Vancouver:

Sumi SS. Characterization of PIK3CD as a Cancer Drug Target. [Internet] [Thesis]. University of Saskatchewan; 2018. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10388/11491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sumi SS. Characterization of PIK3CD as a Cancer Drug Target. [Thesis]. University of Saskatchewan; 2018. Available from: http://hdl.handle.net/10388/11491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

2. Mereniuk, Todd. Synthetic lethal targeting of polynucleotide kinase/phosphatase and its potential role in directed cancer therapies.

Degree: PhD, Department of Oncology, 2012, University of Alberta

Synthetic lethality arises when simultaneous disruption of two non-essential, non-allelic genes in the same cell causes lethality. This phenomenon has been shown to occur between… (more)

Subjects/Keywords: polynucleotide kinase/phosphatase; PTEN; synthetic lethality; SHP-1

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APA (6th Edition):

Mereniuk, T. (2012). Synthetic lethal targeting of polynucleotide kinase/phosphatase and its potential role in directed cancer therapies. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/c7p88cg67w

Chicago Manual of Style (16th Edition):

Mereniuk, Todd. “Synthetic lethal targeting of polynucleotide kinase/phosphatase and its potential role in directed cancer therapies.” 2012. Doctoral Dissertation, University of Alberta. Accessed April 14, 2021. https://era.library.ualberta.ca/files/c7p88cg67w.

MLA Handbook (7th Edition):

Mereniuk, Todd. “Synthetic lethal targeting of polynucleotide kinase/phosphatase and its potential role in directed cancer therapies.” 2012. Web. 14 Apr 2021.

Vancouver:

Mereniuk T. Synthetic lethal targeting of polynucleotide kinase/phosphatase and its potential role in directed cancer therapies. [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2021 Apr 14]. Available from: https://era.library.ualberta.ca/files/c7p88cg67w.

Council of Science Editors:

Mereniuk T. Synthetic lethal targeting of polynucleotide kinase/phosphatase and its potential role in directed cancer therapies. [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/c7p88cg67w


Temple University

3. Reed, Katherine Sullivan. Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers.

Degree: PhD, 2018, Temple University

Biomedical Sciences

PARP inhibitors (PARPi) have been used to induce synthetic lethality in BRCA-deficient tumors in clinical trials with limited success due to the development… (more)

Subjects/Keywords: Oncology; Cellular biology;

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APA (6th Edition):

Reed, K. S. (2018). Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,507327

Chicago Manual of Style (16th Edition):

Reed, Katherine Sullivan. “Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers.” 2018. Doctoral Dissertation, Temple University. Accessed April 14, 2021. http://digital.library.temple.edu/u?/p245801coll10,507327.

MLA Handbook (7th Edition):

Reed, Katherine Sullivan. “Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers.” 2018. Web. 14 Apr 2021.

Vancouver:

Reed KS. Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2021 Apr 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,507327.

Council of Science Editors:

Reed KS. Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,507327


University of Saskatchewan

4. Paul, James 1986-. Synthetic lethal interactions of EPHB6 in breast cancer cells.

Degree: 2016, University of Saskatchewan

 Sequencing of tumor genomes has shown that many loss-of-function alterations exist in cancer cells. Some of these alterations are a product of the cancerous progression… (more)

Subjects/Keywords: breast cancer; genetic interaction; synthetic lethality; EPHB6; SRC kinase; KX2-391

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APA (6th Edition):

Paul, J. 1. (2016). Synthetic lethal interactions of EPHB6 in breast cancer cells. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/7663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Paul, James 1986-. “Synthetic lethal interactions of EPHB6 in breast cancer cells.” 2016. Thesis, University of Saskatchewan. Accessed April 14, 2021. http://hdl.handle.net/10388/7663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Paul, James 1986-. “Synthetic lethal interactions of EPHB6 in breast cancer cells.” 2016. Web. 14 Apr 2021.

Vancouver:

Paul J1. Synthetic lethal interactions of EPHB6 in breast cancer cells. [Internet] [Thesis]. University of Saskatchewan; 2016. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10388/7663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Paul J1. Synthetic lethal interactions of EPHB6 in breast cancer cells. [Thesis]. University of Saskatchewan; 2016. Available from: http://hdl.handle.net/10388/7663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wesleyan University

5. Zhang, Shu. Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment.

Degree: Chemistry, 2014, Wesleyan University

  Certain types of cancer have defective repair mechanisms, which can cause the accumulation of damaged DNA. The persistent DNA damage can stall replication process… (more)

Subjects/Keywords: DNA damage; DNA damage repair; Synthetic Lethality; SSB; DSB

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APA (6th Edition):

Zhang, S. (2014). Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment. (Masters Thesis). Wesleyan University. Retrieved from https://wesscholar.wesleyan.edu/etd_mas_theses/78

Chicago Manual of Style (16th Edition):

Zhang, Shu. “Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment.” 2014. Masters Thesis, Wesleyan University. Accessed April 14, 2021. https://wesscholar.wesleyan.edu/etd_mas_theses/78.

MLA Handbook (7th Edition):

Zhang, Shu. “Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment.” 2014. Web. 14 Apr 2021.

Vancouver:

Zhang S. Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment. [Internet] [Masters thesis]. Wesleyan University; 2014. [cited 2021 Apr 14]. Available from: https://wesscholar.wesleyan.edu/etd_mas_theses/78.

Council of Science Editors:

Zhang S. Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment. [Masters Thesis]. Wesleyan University; 2014. Available from: https://wesscholar.wesleyan.edu/etd_mas_theses/78


University of Toronto

6. Lian, Huan. Exploration of Essential Biological Processes by Construction of Conditional Alleles of Non-essential Genes in S. cerevisiae.

Degree: 2017, University of Toronto

Conditional mutants in S. cerevisiae have been useful tools in studying essential gene functions. In this study, I describe a new method that makes use… (more)

Subjects/Keywords: Budding yeast; Dosage suppression; Genetic interactions; Mutagenesis; Synthetic lethality; 0369

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APA (6th Edition):

Lian, H. (2017). Exploration of Essential Biological Processes by Construction of Conditional Alleles of Non-essential Genes in S. cerevisiae. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/77842

Chicago Manual of Style (16th Edition):

Lian, Huan. “Exploration of Essential Biological Processes by Construction of Conditional Alleles of Non-essential Genes in S. cerevisiae.” 2017. Masters Thesis, University of Toronto. Accessed April 14, 2021. http://hdl.handle.net/1807/77842.

MLA Handbook (7th Edition):

Lian, Huan. “Exploration of Essential Biological Processes by Construction of Conditional Alleles of Non-essential Genes in S. cerevisiae.” 2017. Web. 14 Apr 2021.

Vancouver:

Lian H. Exploration of Essential Biological Processes by Construction of Conditional Alleles of Non-essential Genes in S. cerevisiae. [Internet] [Masters thesis]. University of Toronto; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1807/77842.

Council of Science Editors:

Lian H. Exploration of Essential Biological Processes by Construction of Conditional Alleles of Non-essential Genes in S. cerevisiae. [Masters Thesis]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/77842


Texas Medical Center

7. Huang, Shaoyi. Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy.

Degree: PhD, 2013, Texas Medical Center

  Lung cancer is the leading cause of cancer death in both men and women in the United States and worldwide. Despite improvement in treatment… (more)

Subjects/Keywords: NSCLC; KRAS; Synthetic lethality; Chemoprevention; Medicine and Health Sciences

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APA (6th Edition):

Huang, S. (2013). Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/396

Chicago Manual of Style (16th Edition):

Huang, Shaoyi. “Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy.” 2013. Doctoral Dissertation, Texas Medical Center. Accessed April 14, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/396.

MLA Handbook (7th Edition):

Huang, Shaoyi. “Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy.” 2013. Web. 14 Apr 2021.

Vancouver:

Huang S. Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy. [Internet] [Doctoral dissertation]. Texas Medical Center; 2013. [cited 2021 Apr 14]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/396.

Council of Science Editors:

Huang S. Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy. [Doctoral Dissertation]. Texas Medical Center; 2013. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/396


University of California – Irvine

8. Thompson, Jordan Michael. Identifying Synthetic Lethal Interactions in VHL-Deficient CC-RCC.

Degree: Biological Sciences, 2017, University of California – Irvine

 Clear Cell Renal Cell Carcinoma (CC-RCC) is a devastating disease in its metastatic manifestation with a 5-year survival rate of 11.7%. The loss of the… (more)

Subjects/Keywords: Molecular biology; Biochemistry; CC-RCC; Rho Kinase; Statin; Synthetic Lethality; VHL

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APA (6th Edition):

Thompson, J. M. (2017). Identifying Synthetic Lethal Interactions in VHL-Deficient CC-RCC. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/9fw83435

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thompson, Jordan Michael. “Identifying Synthetic Lethal Interactions in VHL-Deficient CC-RCC.” 2017. Thesis, University of California – Irvine. Accessed April 14, 2021. http://www.escholarship.org/uc/item/9fw83435.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thompson, Jordan Michael. “Identifying Synthetic Lethal Interactions in VHL-Deficient CC-RCC.” 2017. Web. 14 Apr 2021.

Vancouver:

Thompson JM. Identifying Synthetic Lethal Interactions in VHL-Deficient CC-RCC. [Internet] [Thesis]. University of California – Irvine; 2017. [cited 2021 Apr 14]. Available from: http://www.escholarship.org/uc/item/9fw83435.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thompson JM. Identifying Synthetic Lethal Interactions in VHL-Deficient CC-RCC. [Thesis]. University of California – Irvine; 2017. Available from: http://www.escholarship.org/uc/item/9fw83435

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

9. O'Connor, Kevin William. Molecular determinants of sensitivity to poly(ADP-ribose) polymerase inhibitors in epithelial ovarian cancer.

Degree: MS, Medical Sciences, 2016, Boston University

 Less than half of patients with epithelial ovarian cancer (EOC) survive five years following diagnosis, underscoring the imperative need for improved treatment. Many patients, including… (more)

Subjects/Keywords: Oncology; PARP inhibitors; Homologous recombination; Ovarian cancer; Synthetic lethality

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APA (6th Edition):

O'Connor, K. W. (2016). Molecular determinants of sensitivity to poly(ADP-ribose) polymerase inhibitors in epithelial ovarian cancer. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16827

Chicago Manual of Style (16th Edition):

O'Connor, Kevin William. “Molecular determinants of sensitivity to poly(ADP-ribose) polymerase inhibitors in epithelial ovarian cancer.” 2016. Masters Thesis, Boston University. Accessed April 14, 2021. http://hdl.handle.net/2144/16827.

MLA Handbook (7th Edition):

O'Connor, Kevin William. “Molecular determinants of sensitivity to poly(ADP-ribose) polymerase inhibitors in epithelial ovarian cancer.” 2016. Web. 14 Apr 2021.

Vancouver:

O'Connor KW. Molecular determinants of sensitivity to poly(ADP-ribose) polymerase inhibitors in epithelial ovarian cancer. [Internet] [Masters thesis]. Boston University; 2016. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2144/16827.

Council of Science Editors:

O'Connor KW. Molecular determinants of sensitivity to poly(ADP-ribose) polymerase inhibitors in epithelial ovarian cancer. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16827


University of Manitoba

10. Guppy, Brent. Characterizing and selectively targeting RNF20 defects within colorectal cancer cells.

Degree: Biochemistry and Medical Genetics, 2016, University of Manitoba

 By 2030, the global colorectal cancer burden is projected to approximately double. This highlights the immediate need to expand our understanding of the etiological origins… (more)

Subjects/Keywords: RNF20; H2Bub1; H3K79me2; H3K4me2; CRISPR-Cas9; Synthetic lethality; Colorectal cancer

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APA (6th Edition):

Guppy, B. (2016). Characterizing and selectively targeting RNF20 defects within colorectal cancer cells. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31860

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guppy, Brent. “Characterizing and selectively targeting RNF20 defects within colorectal cancer cells.” 2016. Thesis, University of Manitoba. Accessed April 14, 2021. http://hdl.handle.net/1993/31860.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guppy, Brent. “Characterizing and selectively targeting RNF20 defects within colorectal cancer cells.” 2016. Web. 14 Apr 2021.

Vancouver:

Guppy B. Characterizing and selectively targeting RNF20 defects within colorectal cancer cells. [Internet] [Thesis]. University of Manitoba; 2016. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1993/31860.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guppy B. Characterizing and selectively targeting RNF20 defects within colorectal cancer cells. [Thesis]. University of Manitoba; 2016. Available from: http://hdl.handle.net/1993/31860

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

11. Wedge, Marie-Ève. Tailoring Oncolytic Viruses for the Treatment of Pancreatic Cancer .

Degree: 2020, University of Ottawa

 Pancreatic cancer (PC) is a highly aggressive disease with unmet therapeutic needs. Recent advances in the use of oncolytic viruses (OVs) as cancer therapeutic agents… (more)

Subjects/Keywords: Oncolytic virus; Pancreatic cancer; Extracellular vesicles; microRNA; Synthetic lethality; Bystander effect

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APA (6th Edition):

Wedge, M. (2020). Tailoring Oncolytic Viruses for the Treatment of Pancreatic Cancer . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/40384

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wedge, Marie-Ève. “Tailoring Oncolytic Viruses for the Treatment of Pancreatic Cancer .” 2020. Thesis, University of Ottawa. Accessed April 14, 2021. http://hdl.handle.net/10393/40384.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wedge, Marie-Ève. “Tailoring Oncolytic Viruses for the Treatment of Pancreatic Cancer .” 2020. Web. 14 Apr 2021.

Vancouver:

Wedge M. Tailoring Oncolytic Viruses for the Treatment of Pancreatic Cancer . [Internet] [Thesis]. University of Ottawa; 2020. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10393/40384.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wedge M. Tailoring Oncolytic Viruses for the Treatment of Pancreatic Cancer . [Thesis]. University of Ottawa; 2020. Available from: http://hdl.handle.net/10393/40384

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universitat de Valencia

12. Llorca Cardeñosa, Marta Jessica. Genetic determinants of ATR inhibitor sensitivity and resistance in Gastric Cancer .

Degree: 2019, Universitat de Valencia

Synthetic lethal approaches in identifying genetic determinants of drug response is a powerful method in selecting patents for targeted cancer therapies. Ataxia-Telangiectasia Mutated (ATM) and… (more)

Subjects/Keywords: ATR inhibitors; synthetic lethality; ATR; ARID1A; GW CRISPR screen; gastric cancer

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APA (6th Edition):

Llorca Cardeñosa, M. J. (2019). Genetic determinants of ATR inhibitor sensitivity and resistance in Gastric Cancer . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/70728

Chicago Manual of Style (16th Edition):

Llorca Cardeñosa, Marta Jessica. “Genetic determinants of ATR inhibitor sensitivity and resistance in Gastric Cancer .” 2019. Doctoral Dissertation, Universitat de Valencia. Accessed April 14, 2021. http://hdl.handle.net/10550/70728.

MLA Handbook (7th Edition):

Llorca Cardeñosa, Marta Jessica. “Genetic determinants of ATR inhibitor sensitivity and resistance in Gastric Cancer .” 2019. Web. 14 Apr 2021.

Vancouver:

Llorca Cardeñosa MJ. Genetic determinants of ATR inhibitor sensitivity and resistance in Gastric Cancer . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2019. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10550/70728.

Council of Science Editors:

Llorca Cardeñosa MJ. Genetic determinants of ATR inhibitor sensitivity and resistance in Gastric Cancer . [Doctoral Dissertation]. Universitat de Valencia; 2019. Available from: http://hdl.handle.net/10550/70728


University of Oklahoma

13. Bhowmik, Bijit Kumar. Chromosome Organization and Segregation in Pseudomonas Aeruginosa.

Degree: Dr.P.H, 2017, University of Oklahoma

 To determine the segregation pattern in the P. aeruginosa strain PAO1, a fluorescent repressor-operator system was used. The data indicate that the PAO1 chromosome is… (more)

Subjects/Keywords: Pseudomonas aeruginosa; Two condensins; Asymmetric chromosome; Chromosome segregation; ParB; Synthetic lethality

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APA (6th Edition):

Bhowmik, B. K. (2017). Chromosome Organization and Segregation in Pseudomonas Aeruginosa. (Doctoral Dissertation). University of Oklahoma. Retrieved from http://hdl.handle.net/11244/52950

Chicago Manual of Style (16th Edition):

Bhowmik, Bijit Kumar. “Chromosome Organization and Segregation in Pseudomonas Aeruginosa.” 2017. Doctoral Dissertation, University of Oklahoma. Accessed April 14, 2021. http://hdl.handle.net/11244/52950.

MLA Handbook (7th Edition):

Bhowmik, Bijit Kumar. “Chromosome Organization and Segregation in Pseudomonas Aeruginosa.” 2017. Web. 14 Apr 2021.

Vancouver:

Bhowmik BK. Chromosome Organization and Segregation in Pseudomonas Aeruginosa. [Internet] [Doctoral dissertation]. University of Oklahoma; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/11244/52950.

Council of Science Editors:

Bhowmik BK. Chromosome Organization and Segregation in Pseudomonas Aeruginosa. [Doctoral Dissertation]. University of Oklahoma; 2017. Available from: http://hdl.handle.net/11244/52950

14. Walason da Silva Abjaude. Estudo de letalidade sintética em células transformadas por papilomavírus humano (HPV).

Degree: 2016, University of São Paulo

Os Papilomavírus Humanos (HPV) são vírus de DNA, não envelopados que infectam as células epiteliais. A infecção persistente por alguns tipos de HPV é o… (more)

Subjects/Keywords: HPV; Letalidade sintética; Reparo de DNA; DNA repair; HPV; Synthetic lethality

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APA (6th Edition):

Abjaude, W. d. S. (2016). Estudo de letalidade sintética em células transformadas por papilomavírus humano (HPV). (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42132/tde-15052017-144525/

Chicago Manual of Style (16th Edition):

Abjaude, Walason da Silva. “Estudo de letalidade sintética em células transformadas por papilomavírus humano (HPV).” 2016. Doctoral Dissertation, University of São Paulo. Accessed April 14, 2021. http://www.teses.usp.br/teses/disponiveis/42/42132/tde-15052017-144525/.

MLA Handbook (7th Edition):

Abjaude, Walason da Silva. “Estudo de letalidade sintética em células transformadas por papilomavírus humano (HPV).” 2016. Web. 14 Apr 2021.

Vancouver:

Abjaude WdS. Estudo de letalidade sintética em células transformadas por papilomavírus humano (HPV). [Internet] [Doctoral dissertation]. University of São Paulo; 2016. [cited 2021 Apr 14]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42132/tde-15052017-144525/.

Council of Science Editors:

Abjaude WdS. Estudo de letalidade sintética em células transformadas por papilomavírus humano (HPV). [Doctoral Dissertation]. University of São Paulo; 2016. Available from: http://www.teses.usp.br/teses/disponiveis/42/42132/tde-15052017-144525/


Harvard University

15. Vallurupalli, Mounica. Identifying Targetable Liabilities in Ewing Sarcoma.

Degree: Doctor of Medicine, 2014, Harvard University

 Background: Despite multi-modality therapy, the majority of patients with metastatic or recurrent Ewing sarcoma (ES), the second most common pediatric bone malignancy, will die of… (more)

Subjects/Keywords: Ewing sarcoma; synthetic lethality; cancer genomics; NF-kB; Cell cycle; chemical biology

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APA (6th Edition):

Vallurupalli, M. (2014). Identifying Targetable Liabilities in Ewing Sarcoma. (Doctoral Dissertation). Harvard University. Retrieved from http://etds.lib.harvard.edu/hms/admin/view/62 ; http://nrs.harvard.edu/urn-3:HUL.InstRepos:12407620

Chicago Manual of Style (16th Edition):

Vallurupalli, Mounica. “Identifying Targetable Liabilities in Ewing Sarcoma.” 2014. Doctoral Dissertation, Harvard University. Accessed April 14, 2021. http://etds.lib.harvard.edu/hms/admin/view/62 ; http://nrs.harvard.edu/urn-3:HUL.InstRepos:12407620.

MLA Handbook (7th Edition):

Vallurupalli, Mounica. “Identifying Targetable Liabilities in Ewing Sarcoma.” 2014. Web. 14 Apr 2021.

Vancouver:

Vallurupalli M. Identifying Targetable Liabilities in Ewing Sarcoma. [Internet] [Doctoral dissertation]. Harvard University; 2014. [cited 2021 Apr 14]. Available from: http://etds.lib.harvard.edu/hms/admin/view/62 ; http://nrs.harvard.edu/urn-3:HUL.InstRepos:12407620.

Council of Science Editors:

Vallurupalli M. Identifying Targetable Liabilities in Ewing Sarcoma. [Doctoral Dissertation]. Harvard University; 2014. Available from: http://etds.lib.harvard.edu/hms/admin/view/62 ; http://nrs.harvard.edu/urn-3:HUL.InstRepos:12407620

16. O. An. ROLE OF SOMATIC COPY NUMBER VARIATIONS IN CANCER.

Degree: 2016, Università degli Studi di Milano

 Genetic variation is the main reason of the phenotypic differences among individuals, as well as of many human genetic diseases. Recent advances in the methods… (more)

Subjects/Keywords: somatic CNV; gene expression; synthetic lethality; cancer genes; Settore BIO/11 - Biologia Molecolare

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

An, O. (2016). ROLE OF SOMATIC COPY NUMBER VARIATIONS IN CANCER. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/361604

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

An, O.. “ROLE OF SOMATIC COPY NUMBER VARIATIONS IN CANCER.” 2016. Thesis, Università degli Studi di Milano. Accessed April 14, 2021. http://hdl.handle.net/2434/361604.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

An, O.. “ROLE OF SOMATIC COPY NUMBER VARIATIONS IN CANCER.” 2016. Web. 14 Apr 2021.

Vancouver:

An O. ROLE OF SOMATIC COPY NUMBER VARIATIONS IN CANCER. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2434/361604.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

An O. ROLE OF SOMATIC COPY NUMBER VARIATIONS IN CANCER. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/361604

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

17. Thompson, Nicola Anne. Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening.

Degree: PhD, 2019, University of Cambridge

 Abstract As our understanding of the cancer genome has progressed, traditional chemotherapeutic agents are being replaced, in part, by targeted therapies. Development of these therapies… (more)

Subjects/Keywords: CRISPR screen; synthetic lethality; paralog; FAM50A; FAM50B; paired screening; melanoma; cancer genetics; target discovery; paralogue

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thompson, N. A. (2019). Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/291485https://www.repository.cam.ac.uk/bitstream/1810/291485/1/1.1%20Multiplex%20library%20content.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/2/1.2%20Oligonucleotides.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/3/1.3%20Gene%20pair%20output%20of%20the%20paired%20library.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/4/1.4%20Single%20gene%20output%20of%20the%20paired%20library.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/5/1.5%20Genomics%20of%20the%20cell%20lines%20used.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/6/1.6%20Mass%20spectrometry%20data.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/7/1.7%20Differential%20expression%20analysis.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/8/1.8%20RNA%20splicing%20analysis.xlsx

Chicago Manual of Style (16th Edition):

Thompson, Nicola Anne. “Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening.” 2019. Doctoral Dissertation, University of Cambridge. Accessed April 14, 2021. https://www.repository.cam.ac.uk/handle/1810/291485https://www.repository.cam.ac.uk/bitstream/1810/291485/1/1.1%20Multiplex%20library%20content.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/2/1.2%20Oligonucleotides.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/3/1.3%20Gene%20pair%20output%20of%20the%20paired%20library.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/4/1.4%20Single%20gene%20output%20of%20the%20paired%20library.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/5/1.5%20Genomics%20of%20the%20cell%20lines%20used.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/6/1.6%20Mass%20spectrometry%20data.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/7/1.7%20Differential%20expression%20analysis.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/8/1.8%20RNA%20splicing%20analysis.xlsx.

MLA Handbook (7th Edition):

Thompson, Nicola Anne. “Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening.” 2019. Web. 14 Apr 2021.

Vancouver:

Thompson NA. Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Apr 14]. Available from: https://www.repository.cam.ac.uk/handle/1810/291485https://www.repository.cam.ac.uk/bitstream/1810/291485/1/1.1%20Multiplex%20library%20content.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/2/1.2%20Oligonucleotides.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/3/1.3%20Gene%20pair%20output%20of%20the%20paired%20library.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/4/1.4%20Single%20gene%20output%20of%20the%20paired%20library.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/5/1.5%20Genomics%20of%20the%20cell%20lines%20used.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/6/1.6%20Mass%20spectrometry%20data.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/7/1.7%20Differential%20expression%20analysis.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/8/1.8%20RNA%20splicing%20analysis.xlsx.

Council of Science Editors:

Thompson NA. Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/291485https://www.repository.cam.ac.uk/bitstream/1810/291485/1/1.1%20Multiplex%20library%20content.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/2/1.2%20Oligonucleotides.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/3/1.3%20Gene%20pair%20output%20of%20the%20paired%20library.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/4/1.4%20Single%20gene%20output%20of%20the%20paired%20library.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/5/1.5%20Genomics%20of%20the%20cell%20lines%20used.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/6/1.6%20Mass%20spectrometry%20data.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/7/1.7%20Differential%20expression%20analysis.xlsx ; https://www.repository.cam.ac.uk/bitstream/1810/291485/8/1.8%20RNA%20splicing%20analysis.xlsx


University of California – San Francisco

18. Ku, Angel Alejandro. Systems Biology Approaches for Identifying Synthetic Lethal Targets in Cancer.

Degree: Pharmaceutical Sciences and Pharmacogenomics, 2019, University of California – San Francisco

 The development of therapeutic agent against cancer is based on targeting key signaling proteins that cancer highjacks and uses to survive. Although progress has been… (more)

Subjects/Keywords: Biology; Cellular biology; Bioinformatics; Cancer Biology; DNA Repair; Functional Genomics; KRAS; Synthetic Lethality; Systems Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ku, A. A. (2019). Systems Biology Approaches for Identifying Synthetic Lethal Targets in Cancer. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/0gj863n2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ku, Angel Alejandro. “Systems Biology Approaches for Identifying Synthetic Lethal Targets in Cancer.” 2019. Thesis, University of California – San Francisco. Accessed April 14, 2021. http://www.escholarship.org/uc/item/0gj863n2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ku, Angel Alejandro. “Systems Biology Approaches for Identifying Synthetic Lethal Targets in Cancer.” 2019. Web. 14 Apr 2021.

Vancouver:

Ku AA. Systems Biology Approaches for Identifying Synthetic Lethal Targets in Cancer. [Internet] [Thesis]. University of California – San Francisco; 2019. [cited 2021 Apr 14]. Available from: http://www.escholarship.org/uc/item/0gj863n2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ku AA. Systems Biology Approaches for Identifying Synthetic Lethal Targets in Cancer. [Thesis]. University of California – San Francisco; 2019. Available from: http://www.escholarship.org/uc/item/0gj863n2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

19. Srinivasan, Gayathri. MicroRNA Regulation of DNA Repair.

Degree: PhD, Medical Sciences - Genetics (IDP), 2017, University of Florida

Subjects/Keywords: damage; dna; lethality; microrna; repair; synthetic

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Srinivasan, G. (2017). MicroRNA Regulation of DNA Repair. (Doctoral Dissertation). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0051684

Chicago Manual of Style (16th Edition):

Srinivasan, Gayathri. “MicroRNA Regulation of DNA Repair.” 2017. Doctoral Dissertation, University of Florida. Accessed April 14, 2021. https://ufdc.ufl.edu/UFE0051684.

MLA Handbook (7th Edition):

Srinivasan, Gayathri. “MicroRNA Regulation of DNA Repair.” 2017. Web. 14 Apr 2021.

Vancouver:

Srinivasan G. MicroRNA Regulation of DNA Repair. [Internet] [Doctoral dissertation]. University of Florida; 2017. [cited 2021 Apr 14]. Available from: https://ufdc.ufl.edu/UFE0051684.

Council of Science Editors:

Srinivasan G. MicroRNA Regulation of DNA Repair. [Doctoral Dissertation]. University of Florida; 2017. Available from: https://ufdc.ufl.edu/UFE0051684

20. Albrecht, Delphine. Etude des mutants synthétiques létaux avec l'AICAR chez la levure et conservation chez l'Homme : Chemo-genetic interactions between histone modification and the antiproliferation drug aicar are conserved in yeast and humans.

Degree: Docteur es, Génétique, 2016, Bordeaux

L’identification d’interactions synthétiques létales (SL) apparait aujourd’hui comme une approche prometteuse, qui permet de cibler directement les cellules cancéreuses. Dans cette étude, nous avons utilisé… (more)

Subjects/Keywords: Levure; Génétique; Modification d’histones; Cancer; Létalité synthétique; Yeast; Histone Modifications; Cancer; Synthetic Lethality; Genetics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Albrecht, D. (2016). Etude des mutants synthétiques létaux avec l'AICAR chez la levure et conservation chez l'Homme : Chemo-genetic interactions between histone modification and the antiproliferation drug aicar are conserved in yeast and humans. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2016BORD0134

Chicago Manual of Style (16th Edition):

Albrecht, Delphine. “Etude des mutants synthétiques létaux avec l'AICAR chez la levure et conservation chez l'Homme : Chemo-genetic interactions between histone modification and the antiproliferation drug aicar are conserved in yeast and humans.” 2016. Doctoral Dissertation, Bordeaux. Accessed April 14, 2021. http://www.theses.fr/2016BORD0134.

MLA Handbook (7th Edition):

Albrecht, Delphine. “Etude des mutants synthétiques létaux avec l'AICAR chez la levure et conservation chez l'Homme : Chemo-genetic interactions between histone modification and the antiproliferation drug aicar are conserved in yeast and humans.” 2016. Web. 14 Apr 2021.

Vancouver:

Albrecht D. Etude des mutants synthétiques létaux avec l'AICAR chez la levure et conservation chez l'Homme : Chemo-genetic interactions between histone modification and the antiproliferation drug aicar are conserved in yeast and humans. [Internet] [Doctoral dissertation]. Bordeaux; 2016. [cited 2021 Apr 14]. Available from: http://www.theses.fr/2016BORD0134.

Council of Science Editors:

Albrecht D. Etude des mutants synthétiques létaux avec l'AICAR chez la levure et conservation chez l'Homme : Chemo-genetic interactions between histone modification and the antiproliferation drug aicar are conserved in yeast and humans. [Doctoral Dissertation]. Bordeaux; 2016. Available from: http://www.theses.fr/2016BORD0134


University of Cambridge

21. Thompson, Nicola Anne. Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening.

Degree: PhD, 2019, University of Cambridge

 As our understanding of the cancer genome has progressed, traditional chemotherapeutic agents are being replaced, in part, by targeted therapies. Development of these therapies is… (more)

Subjects/Keywords: CRISPR screen; synthetic lethality; paralog; FAM50A; FAM50B; paired screening; melanoma; cancer genetics; target discovery; paralogue

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thompson, N. A. (2019). Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.38647 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774707

Chicago Manual of Style (16th Edition):

Thompson, Nicola Anne. “Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening.” 2019. Doctoral Dissertation, University of Cambridge. Accessed April 14, 2021. https://doi.org/10.17863/CAM.38647 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774707.

MLA Handbook (7th Edition):

Thompson, Nicola Anne. “Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening.” 2019. Web. 14 Apr 2021.

Vancouver:

Thompson NA. Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Apr 14]. Available from: https://doi.org/10.17863/CAM.38647 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774707.

Council of Science Editors:

Thompson NA. Identification of novel synthetic lethal interactions using multiplexed CRISPR-Cas9 screening. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://doi.org/10.17863/CAM.38647 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.774707


University of Cincinnati

22. Michel, Daniel R. Cytoskeletal Architecture and Cell Motility Remain Unperturbed in Mouse Embryonic Fibroblasts from <i>Plk3</i> Knockout Mice.

Degree: PhD, Medicine: Molecular Genetics, Biochemistry, and Microbiology, 2015, University of Cincinnati

 Polo-like kinase 3 (Plk3) is a member of a conserved family of serine/threonine kinases that primarily regulate cell cycle progression and mitotic events. In response… (more)

Subjects/Keywords: Molecular Biology; Polo-like kinase 3; Synthetic lethality; Plk3; Cell motility; Cytoskeleton; Polo-like kinases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Michel, D. R. (2015). Cytoskeletal Architecture and Cell Motility Remain Unperturbed in Mouse Embryonic Fibroblasts from <i>Plk3</i> Knockout Mice. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1446546516

Chicago Manual of Style (16th Edition):

Michel, Daniel R. “Cytoskeletal Architecture and Cell Motility Remain Unperturbed in Mouse Embryonic Fibroblasts from <i>Plk3</i> Knockout Mice.” 2015. Doctoral Dissertation, University of Cincinnati. Accessed April 14, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1446546516.

MLA Handbook (7th Edition):

Michel, Daniel R. “Cytoskeletal Architecture and Cell Motility Remain Unperturbed in Mouse Embryonic Fibroblasts from <i>Plk3</i> Knockout Mice.” 2015. Web. 14 Apr 2021.

Vancouver:

Michel DR. Cytoskeletal Architecture and Cell Motility Remain Unperturbed in Mouse Embryonic Fibroblasts from <i>Plk3</i> Knockout Mice. [Internet] [Doctoral dissertation]. University of Cincinnati; 2015. [cited 2021 Apr 14]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1446546516.

Council of Science Editors:

Michel DR. Cytoskeletal Architecture and Cell Motility Remain Unperturbed in Mouse Embryonic Fibroblasts from <i>Plk3</i> Knockout Mice. [Doctoral Dissertation]. University of Cincinnati; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1446546516


University of Cambridge

23. Mulhearn, Darcie Sinead. Exploring genetic interactions with G-quadruplex structures.

Degree: PhD, 2019, University of Cambridge

 G-quadruplexes are non-canonical nucleic acid secondary structures of increasing biological and medicinal interest due to their proposed physiological functions in transcription, replication, translation and telomere… (more)

Subjects/Keywords: 572.8; synthetic lethality; G-quadruplex; secondary structures; shRNA screening; screening; RNAi; genomic instability; small molecule

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mulhearn, D. S. (2019). Exploring genetic interactions with G-quadruplex structures. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.35269 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763883

Chicago Manual of Style (16th Edition):

Mulhearn, Darcie Sinead. “Exploring genetic interactions with G-quadruplex structures.” 2019. Doctoral Dissertation, University of Cambridge. Accessed April 14, 2021. https://doi.org/10.17863/CAM.35269 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763883.

MLA Handbook (7th Edition):

Mulhearn, Darcie Sinead. “Exploring genetic interactions with G-quadruplex structures.” 2019. Web. 14 Apr 2021.

Vancouver:

Mulhearn DS. Exploring genetic interactions with G-quadruplex structures. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Apr 14]. Available from: https://doi.org/10.17863/CAM.35269 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763883.

Council of Science Editors:

Mulhearn DS. Exploring genetic interactions with G-quadruplex structures. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://doi.org/10.17863/CAM.35269 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763883

24. Kirzinger, Morgan 1988-. Humanized yeast genetic interaction mapping predicts synthetic lethal interactions of FBXW7 in breast cancer.

Degree: 2018, University of Saskatchewan

 A Synthetic lethal (SL) interaction involves a pair of genes (geneA and geneB) where inhibition of either geneA or geneB individually has no effect on… (more)

Subjects/Keywords: Breast Cancer; Synthetic Lethality; Genetic Interactions

…Diagram of Synthetic Lethality and Synthetic Dosage Lethality . . . . . . . Diagram of the… …Mechanisms of Synthetic Lethality . . . . . . . . . . . . . . . 10 13 14 4.1 Steps in the… …palindromic repeats data mining synthetic lethality database of genomic variation and phenotype in… …lethality short hairpin RNA small interfering RNA S-phase kinase associated protein 1 synthetic… …lethality survival of the fittest synthetic lethal database T-cell acute lymphoblastic leukaemia… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kirzinger, M. 1. (2018). Humanized yeast genetic interaction mapping predicts synthetic lethal interactions of FBXW7 in breast cancer. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/11057

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kirzinger, Morgan 1988-. “Humanized yeast genetic interaction mapping predicts synthetic lethal interactions of FBXW7 in breast cancer.” 2018. Thesis, University of Saskatchewan. Accessed April 14, 2021. http://hdl.handle.net/10388/11057.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kirzinger, Morgan 1988-. “Humanized yeast genetic interaction mapping predicts synthetic lethal interactions of FBXW7 in breast cancer.” 2018. Web. 14 Apr 2021.

Vancouver:

Kirzinger M1. Humanized yeast genetic interaction mapping predicts synthetic lethal interactions of FBXW7 in breast cancer. [Internet] [Thesis]. University of Saskatchewan; 2018. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10388/11057.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kirzinger M1. Humanized yeast genetic interaction mapping predicts synthetic lethal interactions of FBXW7 in breast cancer. [Thesis]. University of Saskatchewan; 2018. Available from: http://hdl.handle.net/10388/11057

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Otago

25. Chin, Chue Vin. Identification of synthetic lethal compounds targeting cancers with cohesin mutations .

Degree: University of Otago

 The cohesin complex, comprising four core subunits, RAD21, SMC3, SMC1A, and STAG1/2, is important for the regulation of many biological processes, including sister chromatid cohesion,… (more)

Subjects/Keywords: cohesin; synthetic lethality; high throughput screening; MCF10A

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chin, C. V. (n.d.). Identification of synthetic lethal compounds targeting cancers with cohesin mutations . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/9832

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Chin, Chue Vin. “Identification of synthetic lethal compounds targeting cancers with cohesin mutations .” Doctoral Dissertation, University of Otago. Accessed April 14, 2021. http://hdl.handle.net/10523/9832.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Chin, Chue Vin. “Identification of synthetic lethal compounds targeting cancers with cohesin mutations .” Web. 14 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Chin CV. Identification of synthetic lethal compounds targeting cancers with cohesin mutations . [Internet] [Doctoral dissertation]. University of Otago; [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10523/9832.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Chin CV. Identification of synthetic lethal compounds targeting cancers with cohesin mutations . [Doctoral Dissertation]. University of Otago; Available from: http://hdl.handle.net/10523/9832

Note: this citation may be lacking information needed for this citation format:
No year of publication.


University of Queensland

26. Bokhari, Fawzi Faisal A. siRNA screening of the kinome identified Aurora A kinase as a therapeutic target gene in cervical cancer.

Degree: UQ Diamantina Institute, 2014, University of Queensland

 HPV oncogenes disable a number of tumour suppressor pathways, including p53 and Rb, contributing to the transformed phenotype. Loss of these critical host cell functions… (more)

Subjects/Keywords: siRNA; AURKA; AURKB; Haspin; GSG2; cervical cancer; HPV; synthetic lethality; 0601 Biochemistry and Cell Biology; 0604 Genetics; 1112 Oncology and Carcinogenesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bokhari, F. F. A. (2014). siRNA screening of the kinome identified Aurora A kinase as a therapeutic target gene in cervical cancer. (Thesis). University of Queensland. Retrieved from http://espace.library.uq.edu.au/view/UQ:341649

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bokhari, Fawzi Faisal A. “siRNA screening of the kinome identified Aurora A kinase as a therapeutic target gene in cervical cancer.” 2014. Thesis, University of Queensland. Accessed April 14, 2021. http://espace.library.uq.edu.au/view/UQ:341649.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bokhari, Fawzi Faisal A. “siRNA screening of the kinome identified Aurora A kinase as a therapeutic target gene in cervical cancer.” 2014. Web. 14 Apr 2021.

Vancouver:

Bokhari FFA. siRNA screening of the kinome identified Aurora A kinase as a therapeutic target gene in cervical cancer. [Internet] [Thesis]. University of Queensland; 2014. [cited 2021 Apr 14]. Available from: http://espace.library.uq.edu.au/view/UQ:341649.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bokhari FFA. siRNA screening of the kinome identified Aurora A kinase as a therapeutic target gene in cervical cancer. [Thesis]. University of Queensland; 2014. Available from: http://espace.library.uq.edu.au/view/UQ:341649

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Blomen, Vincent A. Studying disease-linked phenotypes using haploid genetics.

Degree: 2017, University Utrecht

 Although genes are unequivocally important for the development of both common and rare human diseases, the connection between the genotype (an individual’s genetic makeup) and… (more)

Subjects/Keywords: Haploid genetics; Genetic Interactions; Mutagenesis; Synthetic lethality; Genetic suppressors

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Blomen, V. A. (2017). Studying disease-linked phenotypes using haploid genetics. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/355941 ; URN:NBN:NL:UI:10-1874-355941 ; urn:isbn:978-94-6295-782-4 ; URN:NBN:NL:UI:10-1874-355941 ; http://dspace.library.uu.nl/handle/1874/355941

Chicago Manual of Style (16th Edition):

Blomen, Vincent A. “Studying disease-linked phenotypes using haploid genetics.” 2017. Doctoral Dissertation, University Utrecht. Accessed April 14, 2021. http://dspace.library.uu.nl/handle/1874/355941 ; URN:NBN:NL:UI:10-1874-355941 ; urn:isbn:978-94-6295-782-4 ; URN:NBN:NL:UI:10-1874-355941 ; http://dspace.library.uu.nl/handle/1874/355941.

MLA Handbook (7th Edition):

Blomen, Vincent A. “Studying disease-linked phenotypes using haploid genetics.” 2017. Web. 14 Apr 2021.

Vancouver:

Blomen VA. Studying disease-linked phenotypes using haploid genetics. [Internet] [Doctoral dissertation]. University Utrecht; 2017. [cited 2021 Apr 14]. Available from: http://dspace.library.uu.nl/handle/1874/355941 ; URN:NBN:NL:UI:10-1874-355941 ; urn:isbn:978-94-6295-782-4 ; URN:NBN:NL:UI:10-1874-355941 ; http://dspace.library.uu.nl/handle/1874/355941.

Council of Science Editors:

Blomen VA. Studying disease-linked phenotypes using haploid genetics. [Doctoral Dissertation]. University Utrecht; 2017. Available from: http://dspace.library.uu.nl/handle/1874/355941 ; URN:NBN:NL:UI:10-1874-355941 ; urn:isbn:978-94-6295-782-4 ; URN:NBN:NL:UI:10-1874-355941 ; http://dspace.library.uu.nl/handle/1874/355941


Universitat Pompeu Fabra

28. Serrat Farran, Xènia, 1993-. Unraveling the functional roles of sftb-1/SF3B1 and prpf-4/PRPF4B in Caenorhabditis elegans splicing and human disease.

Degree: Departament de Ciències Experimentals i de la Salut, 2019, Universitat Pompeu Fabra

 El nematode Caenorhabditis elegans és un sistema experimental que pot ajudar a resoldre qüestions fonamentals en recerca biomèdica, incloses les conseqüències d’alteracions en el processament… (more)

Subjects/Keywords: Synthetic lethality; Splicing inhibitors; Spliceosome; SF3B1; RNA sequencing; RNA interference; PRP4 kinase; Pre-mRNA splicing; CRISPR; Caenorhabditis elegans; 577

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Serrat Farran, Xènia, 1. (2019). Unraveling the functional roles of sftb-1/SF3B1 and prpf-4/PRPF4B in Caenorhabditis elegans splicing and human disease. (Thesis). Universitat Pompeu Fabra. Retrieved from http://hdl.handle.net/10803/668152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Serrat Farran, Xènia, 1993-. “Unraveling the functional roles of sftb-1/SF3B1 and prpf-4/PRPF4B in Caenorhabditis elegans splicing and human disease.” 2019. Thesis, Universitat Pompeu Fabra. Accessed April 14, 2021. http://hdl.handle.net/10803/668152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Serrat Farran, Xènia, 1993-. “Unraveling the functional roles of sftb-1/SF3B1 and prpf-4/PRPF4B in Caenorhabditis elegans splicing and human disease.” 2019. Web. 14 Apr 2021.

Vancouver:

Serrat Farran, Xènia 1. Unraveling the functional roles of sftb-1/SF3B1 and prpf-4/PRPF4B in Caenorhabditis elegans splicing and human disease. [Internet] [Thesis]. Universitat Pompeu Fabra; 2019. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10803/668152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Serrat Farran, Xènia 1. Unraveling the functional roles of sftb-1/SF3B1 and prpf-4/PRPF4B in Caenorhabditis elegans splicing and human disease. [Thesis]. Universitat Pompeu Fabra; 2019. Available from: http://hdl.handle.net/10803/668152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Blomen, Vincent A. Studying disease-linked phenotypes using haploid genetics.

Degree: 2017, University Utrecht

 Although genes are unequivocally important for the development of both common and rare human diseases, the connection between the genotype (an individual’s genetic makeup) and… (more)

Subjects/Keywords: Haploid genetics; Genetic Interactions; Mutagenesis; Synthetic lethality; Genetic suppressors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Blomen, V. A. (2017). Studying disease-linked phenotypes using haploid genetics. (Doctoral Dissertation). University Utrecht. Retrieved from https://dspace.library.uu.nl/handle/1874/355941 ; URN:NBN:NL:UI:10-1874-355941 ; urn:isbn:978-94-6295-782-4 ; URN:NBN:NL:UI:10-1874-355941 ; https://dspace.library.uu.nl/handle/1874/355941

Chicago Manual of Style (16th Edition):

Blomen, Vincent A. “Studying disease-linked phenotypes using haploid genetics.” 2017. Doctoral Dissertation, University Utrecht. Accessed April 14, 2021. https://dspace.library.uu.nl/handle/1874/355941 ; URN:NBN:NL:UI:10-1874-355941 ; urn:isbn:978-94-6295-782-4 ; URN:NBN:NL:UI:10-1874-355941 ; https://dspace.library.uu.nl/handle/1874/355941.

MLA Handbook (7th Edition):

Blomen, Vincent A. “Studying disease-linked phenotypes using haploid genetics.” 2017. Web. 14 Apr 2021.

Vancouver:

Blomen VA. Studying disease-linked phenotypes using haploid genetics. [Internet] [Doctoral dissertation]. University Utrecht; 2017. [cited 2021 Apr 14]. Available from: https://dspace.library.uu.nl/handle/1874/355941 ; URN:NBN:NL:UI:10-1874-355941 ; urn:isbn:978-94-6295-782-4 ; URN:NBN:NL:UI:10-1874-355941 ; https://dspace.library.uu.nl/handle/1874/355941.

Council of Science Editors:

Blomen VA. Studying disease-linked phenotypes using haploid genetics. [Doctoral Dissertation]. University Utrecht; 2017. Available from: https://dspace.library.uu.nl/handle/1874/355941 ; URN:NBN:NL:UI:10-1874-355941 ; urn:isbn:978-94-6295-782-4 ; URN:NBN:NL:UI:10-1874-355941 ; https://dspace.library.uu.nl/handle/1874/355941

30. Silva Evangelista, Cláudia. Molecular Characterization of Pediatric Brainstem Gliomas (DIPG) and Identification of New Therapeutic Targets : Caractérisation moléculaire des gliomes malins pédiatriques du tronc cérébral (DIPG) et identification de nouvelles stratégies thérapeutiques.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2018, Université Paris-Saclay (ComUE)

Les DIPG représentent les tumeurs cérébrales pédiatriques les plus sévères. Aucun progrès dans leur prise en charge n’a été accompli au cours des 50 dernières… (more)

Subjects/Keywords: Dipg; Crible d’ARN interférence; Létalité synthétique; Histone H3-K27M; Radioresistance; Dipg; RNA interference screening; Synthetic lethality; H3-K27M Histone; Radioresistance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Silva Evangelista, C. (2018). Molecular Characterization of Pediatric Brainstem Gliomas (DIPG) and Identification of New Therapeutic Targets : Caractérisation moléculaire des gliomes malins pédiatriques du tronc cérébral (DIPG) et identification de nouvelles stratégies thérapeutiques. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2018SACLS269

Chicago Manual of Style (16th Edition):

Silva Evangelista, Cláudia. “Molecular Characterization of Pediatric Brainstem Gliomas (DIPG) and Identification of New Therapeutic Targets : Caractérisation moléculaire des gliomes malins pédiatriques du tronc cérébral (DIPG) et identification de nouvelles stratégies thérapeutiques.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed April 14, 2021. http://www.theses.fr/2018SACLS269.

MLA Handbook (7th Edition):

Silva Evangelista, Cláudia. “Molecular Characterization of Pediatric Brainstem Gliomas (DIPG) and Identification of New Therapeutic Targets : Caractérisation moléculaire des gliomes malins pédiatriques du tronc cérébral (DIPG) et identification de nouvelles stratégies thérapeutiques.” 2018. Web. 14 Apr 2021.

Vancouver:

Silva Evangelista C. Molecular Characterization of Pediatric Brainstem Gliomas (DIPG) and Identification of New Therapeutic Targets : Caractérisation moléculaire des gliomes malins pédiatriques du tronc cérébral (DIPG) et identification de nouvelles stratégies thérapeutiques. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2018. [cited 2021 Apr 14]. Available from: http://www.theses.fr/2018SACLS269.

Council of Science Editors:

Silva Evangelista C. Molecular Characterization of Pediatric Brainstem Gliomas (DIPG) and Identification of New Therapeutic Targets : Caractérisation moléculaire des gliomes malins pédiatriques du tronc cérébral (DIPG) et identification de nouvelles stratégies thérapeutiques. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2018. Available from: http://www.theses.fr/2018SACLS269

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