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You searched for subject:(Syk). Showing records 1 – 23 of 23 total matches.

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University of California – San Francisco

1. Graham, Michelle T. Constitutive Syk Activity Deregulates Cell Signaling and Hematopoiesis.

Degree: Biomedical Sciences, 2012, University of California – San Francisco

 Non-receptor protein tyrosine kinase Syk mediates signal transduction pathways downstream of immunoreceptors, integrins, and C-type lectins and is expressed in immune cells. Overexpression and aberrant… (more)

Subjects/Keywords: Oncology; Immunology; autoinhibition; Hematopoiesis; myeloproliferative neoplasm; signal transduction; Syk; TEL-Syk

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APA (6th Edition):

Graham, M. T. (2012). Constitutive Syk Activity Deregulates Cell Signaling and Hematopoiesis. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/4pp198gj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Graham, Michelle T. “Constitutive Syk Activity Deregulates Cell Signaling and Hematopoiesis.” 2012. Thesis, University of California – San Francisco. Accessed September 15, 2019. http://www.escholarship.org/uc/item/4pp198gj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Graham, Michelle T. “Constitutive Syk Activity Deregulates Cell Signaling and Hematopoiesis.” 2012. Web. 15 Sep 2019.

Vancouver:

Graham MT. Constitutive Syk Activity Deregulates Cell Signaling and Hematopoiesis. [Internet] [Thesis]. University of California – San Francisco; 2012. [cited 2019 Sep 15]. Available from: http://www.escholarship.org/uc/item/4pp198gj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Graham MT. Constitutive Syk Activity Deregulates Cell Signaling and Hematopoiesis. [Thesis]. University of California – San Francisco; 2012. Available from: http://www.escholarship.org/uc/item/4pp198gj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

2. Penton, Patricia Castellanos. Syk Inhibition Attenuates Airway Hyperresponsiveness in a Murine Model of Asthma and Exacerbation by Air Pollution.

Degree: 2012, University of Toronto

Airway hyperresponsiveness (AHR) is a cardinal feature of asthma that is aggravated by environmental air pollution (EAP). Splenocyte tyrosine kinase Syk has been associated with… (more)

Subjects/Keywords: asthma; airways hyperresponsiveness; Syk; pollution; 0564; 0768

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APA (6th Edition):

Penton, P. C. (2012). Syk Inhibition Attenuates Airway Hyperresponsiveness in a Murine Model of Asthma and Exacerbation by Air Pollution. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33357

Chicago Manual of Style (16th Edition):

Penton, Patricia Castellanos. “Syk Inhibition Attenuates Airway Hyperresponsiveness in a Murine Model of Asthma and Exacerbation by Air Pollution.” 2012. Masters Thesis, University of Toronto. Accessed September 15, 2019. http://hdl.handle.net/1807/33357.

MLA Handbook (7th Edition):

Penton, Patricia Castellanos. “Syk Inhibition Attenuates Airway Hyperresponsiveness in a Murine Model of Asthma and Exacerbation by Air Pollution.” 2012. Web. 15 Sep 2019.

Vancouver:

Penton PC. Syk Inhibition Attenuates Airway Hyperresponsiveness in a Murine Model of Asthma and Exacerbation by Air Pollution. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2019 Sep 15]. Available from: http://hdl.handle.net/1807/33357.

Council of Science Editors:

Penton PC. Syk Inhibition Attenuates Airway Hyperresponsiveness in a Murine Model of Asthma and Exacerbation by Air Pollution. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33357


Harvard University

3. Fu, Wenbo. The Sachdev-Ye-Kitaev Model and Matter Without Quasiparticles.

Degree: PhD, 2018, Harvard University

This thesis is devoted to the study of Sachdev-Ye-Kitaev (SYK) models, which describe matter without quasiparticles. The SYK model is an exactly solvable model that… (more)

Subjects/Keywords: SYK; non-Fermi liquid; quantum chaos

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APA (6th Edition):

Fu, W. (2018). The Sachdev-Ye-Kitaev Model and Matter Without Quasiparticles. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:41128513

Chicago Manual of Style (16th Edition):

Fu, Wenbo. “The Sachdev-Ye-Kitaev Model and Matter Without Quasiparticles.” 2018. Doctoral Dissertation, Harvard University. Accessed September 15, 2019. http://nrs.harvard.edu/urn-3:HUL.InstRepos:41128513.

MLA Handbook (7th Edition):

Fu, Wenbo. “The Sachdev-Ye-Kitaev Model and Matter Without Quasiparticles.” 2018. Web. 15 Sep 2019.

Vancouver:

Fu W. The Sachdev-Ye-Kitaev Model and Matter Without Quasiparticles. [Internet] [Doctoral dissertation]. Harvard University; 2018. [cited 2019 Sep 15]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:41128513.

Council of Science Editors:

Fu W. The Sachdev-Ye-Kitaev Model and Matter Without Quasiparticles. [Doctoral Dissertation]. Harvard University; 2018. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:41128513


Temple University

4. Thomas, Dafydd Huw. Regulation of Syk activity in GPVI-mediated platelet activation.

Degree: PhD, 2010, Temple University

Pharmacology

Activation of platelets is essential for hemostasis. Following damage to the vascular endothelium collagen is exposed, to which platelets stably adhere. After adhesion on… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Collagen; GPVI; Phosphatase; Platelets; Syk; TULA

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APA (6th Edition):

Thomas, D. H. (2010). Regulation of Syk activity in GPVI-mediated platelet activation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,92028

Chicago Manual of Style (16th Edition):

Thomas, Dafydd Huw. “Regulation of Syk activity in GPVI-mediated platelet activation.” 2010. Doctoral Dissertation, Temple University. Accessed September 15, 2019. http://digital.library.temple.edu/u?/p245801coll10,92028.

MLA Handbook (7th Edition):

Thomas, Dafydd Huw. “Regulation of Syk activity in GPVI-mediated platelet activation.” 2010. Web. 15 Sep 2019.

Vancouver:

Thomas DH. Regulation of Syk activity in GPVI-mediated platelet activation. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Sep 15]. Available from: http://digital.library.temple.edu/u?/p245801coll10,92028.

Council of Science Editors:

Thomas DH. Regulation of Syk activity in GPVI-mediated platelet activation. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,92028

5. Furukawa, Kazuo. Advanced glycation end products induce brain-derived neurotrophic factor release from human platelets through the Src-family kinase activation : 最終糖化産物(AGEs)はヒト血小板において、Srcファミリーキナーゼの活性化を介して脳由来神経栄養因子(BDNF)の放出を促進させる.

Degree: 博士(医学), 2017, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第4357号. 学位記番号: 新大院博(医)甲第770号. 学位授与年月日: 平成29年9月20日

Cardiovascular Diabetology. 2017, 16, 20.

Background: Brain-derived neurotrophic factor (BDNF) exerts beneficial effects not only on diabetic… (more)

Subjects/Keywords: AGE; BDNF; Platelets; Release; Ca^<; 2+>;

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APA (6th Edition):

Furukawa, K. (2017). Advanced glycation end products induce brain-derived neurotrophic factor release from human platelets through the Src-family kinase activation : 最終糖化産物(AGEs)はヒト血小板において、Srcファミリーキナーゼの活性化を介して脳由来神経栄養因子(BDNF)の放出を促進させる. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/48151

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Furukawa, Kazuo. “Advanced glycation end products induce brain-derived neurotrophic factor release from human platelets through the Src-family kinase activation : 最終糖化産物(AGEs)はヒト血小板において、Srcファミリーキナーゼの活性化を介して脳由来神経栄養因子(BDNF)の放出を促進させる.” 2017. Thesis, Niigata University / 新潟大学. Accessed September 15, 2019. http://hdl.handle.net/10191/48151.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Furukawa, Kazuo. “Advanced glycation end products induce brain-derived neurotrophic factor release from human platelets through the Src-family kinase activation : 最終糖化産物(AGEs)はヒト血小板において、Srcファミリーキナーゼの活性化を介して脳由来神経栄養因子(BDNF)の放出を促進させる.” 2017. Web. 15 Sep 2019.

Vancouver:

Furukawa K. Advanced glycation end products induce brain-derived neurotrophic factor release from human platelets through the Src-family kinase activation : 最終糖化産物(AGEs)はヒト血小板において、Srcファミリーキナーゼの活性化を介して脳由来神経栄養因子(BDNF)の放出を促進させる. [Internet] [Thesis]. Niigata University / 新潟大学; 2017. [cited 2019 Sep 15]. Available from: http://hdl.handle.net/10191/48151.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Furukawa K. Advanced glycation end products induce brain-derived neurotrophic factor release from human platelets through the Src-family kinase activation : 最終糖化産物(AGEs)はヒト血小板において、Srcファミリーキナーゼの活性化を介して脳由来神経栄養因子(BDNF)の放出を促進させる. [Thesis]. Niigata University / 新潟大学; 2017. Available from: http://hdl.handle.net/10191/48151

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Purdue University

6. Hardy, Shana D. SYK promotes TGF-beta-induced P-body clearance in breast cancer cells through the enhancement of autophagy.

Degree: PhD, PULSe, 2016, Purdue University

SYK is a protein tyrosine kinase that plays an essential role in the development and activation of immune cells. Its expression, however, is not… (more)

Subjects/Keywords: Biological sciences; Autophagy; EMT; P-bodies; SYK; Molecular Biology

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APA (6th Edition):

Hardy, S. D. (2016). SYK promotes TGF-beta-induced P-body clearance in breast cancer cells through the enhancement of autophagy. (Doctoral Dissertation). Purdue University. Retrieved from http://docs.lib.purdue.edu/open_access_dissertations/931

Chicago Manual of Style (16th Edition):

Hardy, Shana D. “SYK promotes TGF-beta-induced P-body clearance in breast cancer cells through the enhancement of autophagy.” 2016. Doctoral Dissertation, Purdue University. Accessed September 15, 2019. http://docs.lib.purdue.edu/open_access_dissertations/931.

MLA Handbook (7th Edition):

Hardy, Shana D. “SYK promotes TGF-beta-induced P-body clearance in breast cancer cells through the enhancement of autophagy.” 2016. Web. 15 Sep 2019.

Vancouver:

Hardy SD. SYK promotes TGF-beta-induced P-body clearance in breast cancer cells through the enhancement of autophagy. [Internet] [Doctoral dissertation]. Purdue University; 2016. [cited 2019 Sep 15]. Available from: http://docs.lib.purdue.edu/open_access_dissertations/931.

Council of Science Editors:

Hardy SD. SYK promotes TGF-beta-induced P-body clearance in breast cancer cells through the enhancement of autophagy. [Doctoral Dissertation]. Purdue University; 2016. Available from: http://docs.lib.purdue.edu/open_access_dissertations/931


Cleveland State University

7. Elsori, Deena H. Regulation of MONOCYTE NADPH OXIDASE:Role of Pattern Recognition Receptors.

Degree: PhD, College of Science, 2009, Cleveland State University

  Activation of the NADPH oxidase enzyme complex results in the production of the oxygen free radical, superoxide anion (O2.-). Superoxide anion is critical for… (more)

Subjects/Keywords: Dectin-1; MONOCYTE; Zymosan; Syk; NADPH OXIDASE; NADPH; human monocytes

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APA (6th Edition):

Elsori, D. H. (2009). Regulation of MONOCYTE NADPH OXIDASE:Role of Pattern Recognition Receptors. (Doctoral Dissertation). Cleveland State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=csu1251125658

Chicago Manual of Style (16th Edition):

Elsori, Deena H. “Regulation of MONOCYTE NADPH OXIDASE:Role of Pattern Recognition Receptors.” 2009. Doctoral Dissertation, Cleveland State University. Accessed September 15, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=csu1251125658.

MLA Handbook (7th Edition):

Elsori, Deena H. “Regulation of MONOCYTE NADPH OXIDASE:Role of Pattern Recognition Receptors.” 2009. Web. 15 Sep 2019.

Vancouver:

Elsori DH. Regulation of MONOCYTE NADPH OXIDASE:Role of Pattern Recognition Receptors. [Internet] [Doctoral dissertation]. Cleveland State University; 2009. [cited 2019 Sep 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1251125658.

Council of Science Editors:

Elsori DH. Regulation of MONOCYTE NADPH OXIDASE:Role of Pattern Recognition Receptors. [Doctoral Dissertation]. Cleveland State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1251125658


University of California – San Francisco

8. Elliott, Emily R. Neutrophils as the key mediators of antibody-mediated arthritis and edema: The central role of Syk.

Degree: Biomedical Sciences, 2012, University of California – San Francisco

 The K/BxN serum transfer model of arthritis is critically dependent on FcγR signaling events mediated by the Syk tyrosine kinase. However, the specific cell types… (more)

Subjects/Keywords: Immunology; Edema; Fc receptors; K/BxN; Neutrophil; Rheumatoid arthritis; Syk

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APA (6th Edition):

Elliott, E. R. (2012). Neutrophils as the key mediators of antibody-mediated arthritis and edema: The central role of Syk. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/08v509nc

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Elliott, Emily R. “Neutrophils as the key mediators of antibody-mediated arthritis and edema: The central role of Syk.” 2012. Thesis, University of California – San Francisco. Accessed September 15, 2019. http://www.escholarship.org/uc/item/08v509nc.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Elliott, Emily R. “Neutrophils as the key mediators of antibody-mediated arthritis and edema: The central role of Syk.” 2012. Web. 15 Sep 2019.

Vancouver:

Elliott ER. Neutrophils as the key mediators of antibody-mediated arthritis and edema: The central role of Syk. [Internet] [Thesis]. University of California – San Francisco; 2012. [cited 2019 Sep 15]. Available from: http://www.escholarship.org/uc/item/08v509nc.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Elliott ER. Neutrophils as the key mediators of antibody-mediated arthritis and edema: The central role of Syk. [Thesis]. University of California – San Francisco; 2012. Available from: http://www.escholarship.org/uc/item/08v509nc

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Roders, Nathalie. Régulation de l'activation de lymphocytes B / cellules plasmatiques pendant le rejet chronique : Le rôle de SYK dans la modulation de Mcl-1 : Regulating the activation of B lymphocytes during chronic antibody mediated rejection : The role of SYK in modulating Mcl-1.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2017, Paris Saclay

L'insuffisance rénale est un problème majeur de santé publique et la transplantation rénale est l’option thérapeutique principale, mais elle comporte le risque de rejet d'organe.… (more)

Subjects/Keywords: Lymphocytes B; Rejet d'organes chronique; Inhibition SYK; Apoptose; Mcl-1; Anticorps; B lymphocytes; Antibody mediated rejection; SYK inhibition; Apoptose; Mcl-1; Antibodies

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APA (6th Edition):

Roders, N. (2017). Régulation de l'activation de lymphocytes B / cellules plasmatiques pendant le rejet chronique : Le rôle de SYK dans la modulation de Mcl-1 : Regulating the activation of B lymphocytes during chronic antibody mediated rejection : The role of SYK in modulating Mcl-1. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2017SACLS439

Chicago Manual of Style (16th Edition):

Roders, Nathalie. “Régulation de l'activation de lymphocytes B / cellules plasmatiques pendant le rejet chronique : Le rôle de SYK dans la modulation de Mcl-1 : Regulating the activation of B lymphocytes during chronic antibody mediated rejection : The role of SYK in modulating Mcl-1.” 2017. Doctoral Dissertation, Paris Saclay. Accessed September 15, 2019. http://www.theses.fr/2017SACLS439.

MLA Handbook (7th Edition):

Roders, Nathalie. “Régulation de l'activation de lymphocytes B / cellules plasmatiques pendant le rejet chronique : Le rôle de SYK dans la modulation de Mcl-1 : Regulating the activation of B lymphocytes during chronic antibody mediated rejection : The role of SYK in modulating Mcl-1.” 2017. Web. 15 Sep 2019.

Vancouver:

Roders N. Régulation de l'activation de lymphocytes B / cellules plasmatiques pendant le rejet chronique : Le rôle de SYK dans la modulation de Mcl-1 : Regulating the activation of B lymphocytes during chronic antibody mediated rejection : The role of SYK in modulating Mcl-1. [Internet] [Doctoral dissertation]. Paris Saclay; 2017. [cited 2019 Sep 15]. Available from: http://www.theses.fr/2017SACLS439.

Council of Science Editors:

Roders N. Régulation de l'activation de lymphocytes B / cellules plasmatiques pendant le rejet chronique : Le rôle de SYK dans la modulation de Mcl-1 : Regulating the activation of B lymphocytes during chronic antibody mediated rejection : The role of SYK in modulating Mcl-1. [Doctoral Dissertation]. Paris Saclay; 2017. Available from: http://www.theses.fr/2017SACLS439

10. Quettier, Maude. Etudes des altérations fonctionnelles de la signalisation dépendante du récepteur à l’antigène dans les cellules B de la Leucémie Lymphoïde Chronique : Functional alterations of antigen-induced B cell Receptor signaling in B cells from Chronic Lymphocytic Leukemia patients.

Degree: Docteur es, Biologie moléculaire, 2015, Sorbonne Paris Cité

Des altérations de la signalisation en aval du récepteur à l’antigène (BCR) jouent un rôle clé dans la physiopathologie de la Leucémie Lymphoïde Chronique (LLC).… (more)

Subjects/Keywords: Récepteur à l'antigène des cellules B; Migration; Syk; PLC gamma 2; PKD; Phosphorylation

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APA (6th Edition):

Quettier, M. (2015). Etudes des altérations fonctionnelles de la signalisation dépendante du récepteur à l’antigène dans les cellules B de la Leucémie Lymphoïde Chronique : Functional alterations of antigen-induced B cell Receptor signaling in B cells from Chronic Lymphocytic Leukemia patients. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2015USPCD055

Chicago Manual of Style (16th Edition):

Quettier, Maude. “Etudes des altérations fonctionnelles de la signalisation dépendante du récepteur à l’antigène dans les cellules B de la Leucémie Lymphoïde Chronique : Functional alterations of antigen-induced B cell Receptor signaling in B cells from Chronic Lymphocytic Leukemia patients.” 2015. Doctoral Dissertation, Sorbonne Paris Cité. Accessed September 15, 2019. http://www.theses.fr/2015USPCD055.

MLA Handbook (7th Edition):

Quettier, Maude. “Etudes des altérations fonctionnelles de la signalisation dépendante du récepteur à l’antigène dans les cellules B de la Leucémie Lymphoïde Chronique : Functional alterations of antigen-induced B cell Receptor signaling in B cells from Chronic Lymphocytic Leukemia patients.” 2015. Web. 15 Sep 2019.

Vancouver:

Quettier M. Etudes des altérations fonctionnelles de la signalisation dépendante du récepteur à l’antigène dans les cellules B de la Leucémie Lymphoïde Chronique : Functional alterations of antigen-induced B cell Receptor signaling in B cells from Chronic Lymphocytic Leukemia patients. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2015. [cited 2019 Sep 15]. Available from: http://www.theses.fr/2015USPCD055.

Council of Science Editors:

Quettier M. Etudes des altérations fonctionnelles de la signalisation dépendante du récepteur à l’antigène dans les cellules B de la Leucémie Lymphoïde Chronique : Functional alterations of antigen-induced B cell Receptor signaling in B cells from Chronic Lymphocytic Leukemia patients. [Doctoral Dissertation]. Sorbonne Paris Cité; 2015. Available from: http://www.theses.fr/2015USPCD055


Universitat Autònoma de Barcelona

11. Carabia Juberó, Júlia. Paper de la via de senyalització del receptor de cèl·lules B (BCR) en la interacció entre les cèl·lules de leucèmia limfàtica crònica i el microambient tumoral.

Degree: Departament de Bioquímica i Biologia Molecular, 2018, Universitat Autònoma de Barcelona

 Chronic lymphocytic leukemia (CLL) is the most common leukemia found in adults in Western countries. The clinical course is heterogeneous and survival can vary from… (more)

Subjects/Keywords: Leucèmia limfàtica crònica; Leucemia linfática crónica; Chronic lymphocytic leukemia; Syk; ZAP-70; Ciències Experimentals; 577

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APA (6th Edition):

Carabia Juberó, J. (2018). Paper de la via de senyalització del receptor de cèl·lules B (BCR) en la interacció entre les cèl·lules de leucèmia limfàtica crònica i el microambient tumoral. (Thesis). Universitat Autònoma de Barcelona. Retrieved from http://hdl.handle.net/10803/572069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carabia Juberó, Júlia. “Paper de la via de senyalització del receptor de cèl·lules B (BCR) en la interacció entre les cèl·lules de leucèmia limfàtica crònica i el microambient tumoral.” 2018. Thesis, Universitat Autònoma de Barcelona. Accessed September 15, 2019. http://hdl.handle.net/10803/572069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carabia Juberó, Júlia. “Paper de la via de senyalització del receptor de cèl·lules B (BCR) en la interacció entre les cèl·lules de leucèmia limfàtica crònica i el microambient tumoral.” 2018. Web. 15 Sep 2019.

Vancouver:

Carabia Juberó J. Paper de la via de senyalització del receptor de cèl·lules B (BCR) en la interacció entre les cèl·lules de leucèmia limfàtica crònica i el microambient tumoral. [Internet] [Thesis]. Universitat Autònoma de Barcelona; 2018. [cited 2019 Sep 15]. Available from: http://hdl.handle.net/10803/572069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carabia Juberó J. Paper de la via de senyalització del receptor de cèl·lules B (BCR) en la interacció entre les cèl·lules de leucèmia limfàtica crònica i el microambient tumoral. [Thesis]. Universitat Autònoma de Barcelona; 2018. Available from: http://hdl.handle.net/10803/572069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

12. Mayanglambam, Azad. Regulation of Protein Kinases (Syk and PKC zeta) in platelets.

Degree: PhD, 2010, Temple University

Physiology

Platelets are crucial components of the hemostatic machinery of the body. When the endothelial continuity is disrupted due to injury or atherosclerotic plaque rupture,… (more)

Subjects/Keywords: Biology, Physiology; Biology, Molecular; Hemostasis; Platelets; Protein Kinase C; Signaling; Syk; Thrombosis

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APA (6th Edition):

Mayanglambam, A. (2010). Regulation of Protein Kinases (Syk and PKC zeta) in platelets. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,91635

Chicago Manual of Style (16th Edition):

Mayanglambam, Azad. “Regulation of Protein Kinases (Syk and PKC zeta) in platelets.” 2010. Doctoral Dissertation, Temple University. Accessed September 15, 2019. http://digital.library.temple.edu/u?/p245801coll10,91635.

MLA Handbook (7th Edition):

Mayanglambam, Azad. “Regulation of Protein Kinases (Syk and PKC zeta) in platelets.” 2010. Web. 15 Sep 2019.

Vancouver:

Mayanglambam A. Regulation of Protein Kinases (Syk and PKC zeta) in platelets. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Sep 15]. Available from: http://digital.library.temple.edu/u?/p245801coll10,91635.

Council of Science Editors:

Mayanglambam A. Regulation of Protein Kinases (Syk and PKC zeta) in platelets. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,91635


Temple University

13. Getz, Todd Michael. The Regulation of Phosphorylation Events in Platelets.

Degree: PhD, 2012, Temple University

Physiology

Platelets play a vital role in processes of hemostasis and thrombosis under physiological and pathological conditions. Following vascular damage, platelets will accumulate and stably… (more)

Subjects/Keywords: Physiology; Biology; Biochemistry; Dextran sulfate; Myosin light chain; Phosphorylation; Platelets; signal transduction; Syk kinase

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APA (6th Edition):

Getz, T. M. (2012). The Regulation of Phosphorylation Events in Platelets. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,213114

Chicago Manual of Style (16th Edition):

Getz, Todd Michael. “The Regulation of Phosphorylation Events in Platelets.” 2012. Doctoral Dissertation, Temple University. Accessed September 15, 2019. http://digital.library.temple.edu/u?/p245801coll10,213114.

MLA Handbook (7th Edition):

Getz, Todd Michael. “The Regulation of Phosphorylation Events in Platelets.” 2012. Web. 15 Sep 2019.

Vancouver:

Getz TM. The Regulation of Phosphorylation Events in Platelets. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2019 Sep 15]. Available from: http://digital.library.temple.edu/u?/p245801coll10,213114.

Council of Science Editors:

Getz TM. The Regulation of Phosphorylation Events in Platelets. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,213114


Temple University

14. Back, Steven. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.

Degree: PhD, 2014, Temple University

Cell Biology

The human skeleton is a dynamic organ that serves multiple functions to maintain normal physiology and health. It protects vital organs, provides support… (more)

Subjects/Keywords: Cellular biology; Biochemistry; Molecular biology;

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APA (6th Edition):

Back, S. (2014). TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,254886

Chicago Manual of Style (16th Edition):

Back, Steven. “TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.” 2014. Doctoral Dissertation, Temple University. Accessed September 15, 2019. http://digital.library.temple.edu/u?/p245801coll10,254886.

MLA Handbook (7th Edition):

Back, Steven. “TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.” 2014. Web. 15 Sep 2019.

Vancouver:

Back S. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Sep 15]. Available from: http://digital.library.temple.edu/u?/p245801coll10,254886.

Council of Science Editors:

Back S. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,254886


Universidade de Lisboa

15. Romeiras, Francisco Maria de Sousa de Macedo Malta. Phosphorylation of CFTR and other membrane proteins: a role in biogenesis and traffic?.

Degree: 2009, Universidade de Lisboa

Tese de mestrado, Bioquímica (Bioquímica Médica), Universidade de Lisboa, Faculdade de Ciências, 2009

Cystic Fibrosis (CF), the most common lethal autosomal recessive disorder among Caucasians,… (more)

Subjects/Keywords: CFTR; Fibrose quística; Fosforilação; CK2; SYK; AMPK; Metformina; Teses de mestrado - 2009

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APA (6th Edition):

Romeiras, F. M. d. S. d. M. M. (2009). Phosphorylation of CFTR and other membrane proteins: a role in biogenesis and traffic?. (Thesis). Universidade de Lisboa. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/3944

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Romeiras, Francisco Maria de Sousa de Macedo Malta. “Phosphorylation of CFTR and other membrane proteins: a role in biogenesis and traffic?.” 2009. Thesis, Universidade de Lisboa. Accessed September 15, 2019. http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/3944.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Romeiras, Francisco Maria de Sousa de Macedo Malta. “Phosphorylation of CFTR and other membrane proteins: a role in biogenesis and traffic?.” 2009. Web. 15 Sep 2019.

Vancouver:

Romeiras FMdSdMM. Phosphorylation of CFTR and other membrane proteins: a role in biogenesis and traffic?. [Internet] [Thesis]. Universidade de Lisboa; 2009. [cited 2019 Sep 15]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/3944.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Romeiras FMdSdMM. Phosphorylation of CFTR and other membrane proteins: a role in biogenesis and traffic?. [Thesis]. Universidade de Lisboa; 2009. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/3944

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade de Lisboa

16. Loureiro, Cláudia Almeida. Reguação dos níveis de transportadores de cloreto na membrana plasmática de células renais pela nova via de sinalização WNK4/SYK.

Degree: 2012, Universidade de Lisboa

Tese de mestrado em Bioquímica (Bioquímica Médica), apresentada à Universidade de Lisboa, através da Faculdade de Ciências, 2012

A hipertensão arterial afeta, em países ocidentais,… (more)

Subjects/Keywords: Cotransportadores; NKCC2; KCC3; Proteína cinase; WNK4; SYK; Fosforilação; Teses de mestrado - 2012

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APA (6th Edition):

Loureiro, C. A. (2012). Reguação dos níveis de transportadores de cloreto na membrana plasmática de células renais pela nova via de sinalização WNK4/SYK. (Thesis). Universidade de Lisboa. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/9263

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Loureiro, Cláudia Almeida. “Reguação dos níveis de transportadores de cloreto na membrana plasmática de células renais pela nova via de sinalização WNK4/SYK.” 2012. Thesis, Universidade de Lisboa. Accessed September 15, 2019. http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/9263.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Loureiro, Cláudia Almeida. “Reguação dos níveis de transportadores de cloreto na membrana plasmática de células renais pela nova via de sinalização WNK4/SYK.” 2012. Web. 15 Sep 2019.

Vancouver:

Loureiro CA. Reguação dos níveis de transportadores de cloreto na membrana plasmática de células renais pela nova via de sinalização WNK4/SYK. [Internet] [Thesis]. Universidade de Lisboa; 2012. [cited 2019 Sep 15]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/9263.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Loureiro CA. Reguação dos níveis de transportadores de cloreto na membrana plasmática de células renais pela nova via de sinalização WNK4/SYK. [Thesis]. Universidade de Lisboa; 2012. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/9263

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. MCCLUSKEY, SEAN. Size regulates particulate adjuvant induced IL-10 and IL-12p70 production.

Degree: School of Biochemistry & Immunology. Discipline of Biochemistry, 2018, Trinity College Dublin

 Until recently, vaccines were largely comprised of whole cell killed and attenuated pathogens. However, over recent decades, there has been a shift towards subunit vaccine… (more)

Subjects/Keywords: IL-10; IL-12p70; particulate adjuvants; PLGA; size; membrane fluidity; stress; p38; Syk; dendritic cells; vaccines

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

MCCLUSKEY, S. (2018). Size regulates particulate adjuvant induced IL-10 and IL-12p70 production. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/82631

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

MCCLUSKEY, SEAN. “Size regulates particulate adjuvant induced IL-10 and IL-12p70 production.” 2018. Thesis, Trinity College Dublin. Accessed September 15, 2019. http://hdl.handle.net/2262/82631.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

MCCLUSKEY, SEAN. “Size regulates particulate adjuvant induced IL-10 and IL-12p70 production.” 2018. Web. 15 Sep 2019.

Vancouver:

MCCLUSKEY S. Size regulates particulate adjuvant induced IL-10 and IL-12p70 production. [Internet] [Thesis]. Trinity College Dublin; 2018. [cited 2019 Sep 15]. Available from: http://hdl.handle.net/2262/82631.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

MCCLUSKEY S. Size regulates particulate adjuvant induced IL-10 and IL-12p70 production. [Thesis]. Trinity College Dublin; 2018. Available from: http://hdl.handle.net/2262/82631

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New Mexico

18. Relich, Peter K. Single Particle Tracking: Analysis Techniques for Live Cell Nanoscopy.

Degree: Physics & Astronomy, 2016, University of New Mexico

  Single molecule experiments are a set of experiments designed specifically to study the properties of individual molecules. It has only been in the last… (more)

Subjects/Keywords: Single Particle Tracking; Gain Calibration; Diffusion; Information; Syk; Fisher; Bayes; MLE; MAP; Nanoscopy; Single Molecule; Astrophysics and Astronomy; Physics

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APA (6th Edition):

Relich, P. K. (2016). Single Particle Tracking: Analysis Techniques for Live Cell Nanoscopy. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/phyc_etds/142

Chicago Manual of Style (16th Edition):

Relich, Peter K. “Single Particle Tracking: Analysis Techniques for Live Cell Nanoscopy.” 2016. Doctoral Dissertation, University of New Mexico. Accessed September 15, 2019. https://digitalrepository.unm.edu/phyc_etds/142.

MLA Handbook (7th Edition):

Relich, Peter K. “Single Particle Tracking: Analysis Techniques for Live Cell Nanoscopy.” 2016. Web. 15 Sep 2019.

Vancouver:

Relich PK. Single Particle Tracking: Analysis Techniques for Live Cell Nanoscopy. [Internet] [Doctoral dissertation]. University of New Mexico; 2016. [cited 2019 Sep 15]. Available from: https://digitalrepository.unm.edu/phyc_etds/142.

Council of Science Editors:

Relich PK. Single Particle Tracking: Analysis Techniques for Live Cell Nanoscopy. [Doctoral Dissertation]. University of New Mexico; 2016. Available from: https://digitalrepository.unm.edu/phyc_etds/142


Uppsala University

19. Gardell, Fredrik. Holography of SYK model.

Degree: Theoretical Physics, 2018, Uppsala University

  The aim of the thesis is to study the AdS/CFT correspondence and the AdS2/SYK connection as a very special example of the duality. While… (more)

Subjects/Keywords: SYK; Holography; AdS; CFT; AdS/CFT Correspondence; AdS2; Nearly AdS2; NAdS2; Other Physics Topics; Annan fysik

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APA (6th Edition):

Gardell, F. (2018). Holography of SYK model. (Thesis). Uppsala University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-365188

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gardell, Fredrik. “Holography of SYK model.” 2018. Thesis, Uppsala University. Accessed September 15, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-365188.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gardell, Fredrik. “Holography of SYK model.” 2018. Web. 15 Sep 2019.

Vancouver:

Gardell F. Holography of SYK model. [Internet] [Thesis]. Uppsala University; 2018. [cited 2019 Sep 15]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-365188.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gardell F. Holography of SYK model. [Thesis]. Uppsala University; 2018. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-365188

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Τασιούλας, Ιωάννης. Ο ρόλος των κυτταροκινών (IL-10 και IFN-α) στην παθογένεια χρόνιων φλεγμονωδών αυτοάνοσων παθήσεων.

Degree: 2006, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Subjects/Keywords: Κυτταροκίνες; Αυτοανοσία; Φλεγμονές; Μακροφάγα; Υποδοχείς ανοσοσφαιρίνης γ; Κινάση SYK; Μόρια - προσαρμοστές FcRγ / DAP12; Inflammation; Autoimmunity; Cytokines; JAK - STAT signalung; Macrophages; FcγR; SyK; FcRγ / DAP12 adaptor molecules

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Τασιούλας, . . (2006). Ο ρόλος των κυτταροκινών (IL-10 και IFN-α) στην παθογένεια χρόνιων φλεγμονωδών αυτοάνοσων παθήσεων. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/16119

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Τασιούλας, Ιωάννης. “Ο ρόλος των κυτταροκινών (IL-10 και IFN-α) στην παθογένεια χρόνιων φλεγμονωδών αυτοάνοσων παθήσεων.” 2006. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed September 15, 2019. http://hdl.handle.net/10442/hedi/16119.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Τασιούλας, Ιωάννης. “Ο ρόλος των κυτταροκινών (IL-10 και IFN-α) στην παθογένεια χρόνιων φλεγμονωδών αυτοάνοσων παθήσεων.” 2006. Web. 15 Sep 2019.

Vancouver:

Τασιούλας . Ο ρόλος των κυτταροκινών (IL-10 και IFN-α) στην παθογένεια χρόνιων φλεγμονωδών αυτοάνοσων παθήσεων. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2006. [cited 2019 Sep 15]. Available from: http://hdl.handle.net/10442/hedi/16119.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Τασιούλας . Ο ρόλος των κυτταροκινών (IL-10 και IFN-α) στην παθογένεια χρόνιων φλεγμονωδών αυτοάνοσων παθήσεων. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2006. Available from: http://hdl.handle.net/10442/hedi/16119

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

21. Hao, Shengli. Regulation of serum response factor activation by the B cell antigen receptor.

Degree: PhD, Integrative Biosciences, 2003, Penn State University

 ABSTRACT How B cell antigen receptor (BCR) signals lead to different cell responses is one of the central questions in immunology. In this thesis, we… (more)

Subjects/Keywords: BCR; NFAT; Ca2+; DAG; actin; IP3R; Lyn; Syk; Btk; PLCg; ERK; BCR signal; SRF

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hao, S. (2003). Regulation of serum response factor activation by the B cell antigen receptor. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/6189

Chicago Manual of Style (16th Edition):

Hao, Shengli. “Regulation of serum response factor activation by the B cell antigen receptor.” 2003. Doctoral Dissertation, Penn State University. Accessed September 15, 2019. https://etda.libraries.psu.edu/catalog/6189.

MLA Handbook (7th Edition):

Hao, Shengli. “Regulation of serum response factor activation by the B cell antigen receptor.” 2003. Web. 15 Sep 2019.

Vancouver:

Hao S. Regulation of serum response factor activation by the B cell antigen receptor. [Internet] [Doctoral dissertation]. Penn State University; 2003. [cited 2019 Sep 15]. Available from: https://etda.libraries.psu.edu/catalog/6189.

Council of Science Editors:

Hao S. Regulation of serum response factor activation by the B cell antigen receptor. [Doctoral Dissertation]. Penn State University; 2003. Available from: https://etda.libraries.psu.edu/catalog/6189

22. 北井,真貴. 自己免疫性慢性糸球体腎炎に対するSpleen tyrosine kinase阻害薬の効果 : Effet of Spleen tyrosine kinase inhibitor on autoimmune glomerulonephritis.

Degree: 博士(医学), 2014, Nihon University / 日本大学

Subjects/Keywords: 自己免疫性糸球体腎炎; autoimmune glomerulonephritis; ループス腎炎; lupus nephritis; Syk阻害薬; spleen tyrosine kinase inhibitor

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APA (6th Edition):

北井,真貴. (2014). 自己免疫性慢性糸球体腎炎に対するSpleen tyrosine kinase阻害薬の効果 : Effet of Spleen tyrosine kinase inhibitor on autoimmune glomerulonephritis. (Thesis). Nihon University / 日本大学. Retrieved from http://repository.nihon-u.ac.jp/xmlui/handle/11263/160 ; http://dx.doi.org/10.15006/32665A4766

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

北井,真貴. “自己免疫性慢性糸球体腎炎に対するSpleen tyrosine kinase阻害薬の効果 : Effet of Spleen tyrosine kinase inhibitor on autoimmune glomerulonephritis.” 2014. Thesis, Nihon University / 日本大学. Accessed September 15, 2019. http://repository.nihon-u.ac.jp/xmlui/handle/11263/160 ; http://dx.doi.org/10.15006/32665A4766.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

北井,真貴. “自己免疫性慢性糸球体腎炎に対するSpleen tyrosine kinase阻害薬の効果 : Effet of Spleen tyrosine kinase inhibitor on autoimmune glomerulonephritis.” 2014. Web. 15 Sep 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

北井,真貴. 自己免疫性慢性糸球体腎炎に対するSpleen tyrosine kinase阻害薬の効果 : Effet of Spleen tyrosine kinase inhibitor on autoimmune glomerulonephritis. [Internet] [Thesis]. Nihon University / 日本大学; 2014. [cited 2019 Sep 15]. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/160 ; http://dx.doi.org/10.15006/32665A4766.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

北井,真貴. 自己免疫性慢性糸球体腎炎に対するSpleen tyrosine kinase阻害薬の効果 : Effet of Spleen tyrosine kinase inhibitor on autoimmune glomerulonephritis. [Thesis]. Nihon University / 日本大学; 2014. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/160 ; http://dx.doi.org/10.15006/32665A4766

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

23. Ρεπανά, Αικατερίνη. SYK κινάση της τυροσίνης. Έκφραση και ρόλος της στον καρκίνο του μαστού.

Degree: 2005, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

In the present work protein tyrosine kinase SYK (Spleen Tyrosine Kinase) was studied. Its expression and role was investigated in breast cancer patients as well… (more)

Subjects/Keywords: Κινάση τυροσίνης (JAK); Καρκίνος μαστού; Έκφραση; Ρόλοι; Ασθενείς καρκίνου μαστού; Καρκινικές κυτταρικές σειρές; Ουβικουϊτινιλίωση; Μεταστάσεις; SyK; Tyrosine kinases; Breast cancer; Expression; Roles; Breast cancer patients; Cancer cell lines; Ubiquitimation; Metastasis

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APA (6th Edition):

Ρεπανά, . . (2005). SYK κινάση της τυροσίνης. Έκφραση και ρόλος της στον καρκίνο του μαστού. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/18996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ρεπανά, Αικατερίνη. “SYK κινάση της τυροσίνης. Έκφραση και ρόλος της στον καρκίνο του μαστού.” 2005. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed September 15, 2019. http://hdl.handle.net/10442/hedi/18996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ρεπανά, Αικατερίνη. “SYK κινάση της τυροσίνης. Έκφραση και ρόλος της στον καρκίνο του μαστού.” 2005. Web. 15 Sep 2019.

Vancouver:

Ρεπανά . SYK κινάση της τυροσίνης. Έκφραση και ρόλος της στον καρκίνο του μαστού. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2005. [cited 2019 Sep 15]. Available from: http://hdl.handle.net/10442/hedi/18996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ρεπανά . SYK κινάση της τυροσίνης. Έκφραση και ρόλος της στον καρκίνο του μαστού. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2005. Available from: http://hdl.handle.net/10442/hedi/18996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.