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You searched for subject:(Sulforaphane). Showing records 1 – 30 of 42 total matches.

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Oregon State University

1. Clarke, John D. Prostate cancer prevention with broccoli : from cellular to human studies.

Degree: PhD, Molecular and Cellular Biology, 2011, Oregon State University

 Prostate cancer is the second leading cause of cancer related death in American men. Epidemiologic studies suggest that cruciferous vegetable intake may lower the risk… (more)

Subjects/Keywords: Sulforaphane

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APA (6th Edition):

Clarke, J. D. (2011). Prostate cancer prevention with broccoli : from cellular to human studies. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/21193

Chicago Manual of Style (16th Edition):

Clarke, John D. “Prostate cancer prevention with broccoli : from cellular to human studies.” 2011. Doctoral Dissertation, Oregon State University. Accessed December 06, 2019. http://hdl.handle.net/1957/21193.

MLA Handbook (7th Edition):

Clarke, John D. “Prostate cancer prevention with broccoli : from cellular to human studies.” 2011. Web. 06 Dec 2019.

Vancouver:

Clarke JD. Prostate cancer prevention with broccoli : from cellular to human studies. [Internet] [Doctoral dissertation]. Oregon State University; 2011. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/1957/21193.

Council of Science Editors:

Clarke JD. Prostate cancer prevention with broccoli : from cellular to human studies. [Doctoral Dissertation]. Oregon State University; 2011. Available from: http://hdl.handle.net/1957/21193


University of Louisville

2. Schmidt, Robin H., 1984-. Sulforaphane prevents acetaminophen-induced hepatic injury in mice.

Degree: MS, 2011, University of Louisville

Sulforaphane (SFN), an isothiocyanate found in cruciferous vegetables, is known to confer antioxidant protection in vivo. Rather than directly reacting with free radicals, however, SFN… (more)

Subjects/Keywords: Acetaminophen; Liver; Sulforaphane; Hepatology; Antioxidant

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APA (6th Edition):

Schmidt, Robin H., 1. (2011). Sulforaphane prevents acetaminophen-induced hepatic injury in mice. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/1273 ; https://ir.library.louisville.edu/etd/1273

Chicago Manual of Style (16th Edition):

Schmidt, Robin H., 1984-. “Sulforaphane prevents acetaminophen-induced hepatic injury in mice.” 2011. Masters Thesis, University of Louisville. Accessed December 06, 2019. 10.18297/etd/1273 ; https://ir.library.louisville.edu/etd/1273.

MLA Handbook (7th Edition):

Schmidt, Robin H., 1984-. “Sulforaphane prevents acetaminophen-induced hepatic injury in mice.” 2011. Web. 06 Dec 2019.

Vancouver:

Schmidt, Robin H. 1. Sulforaphane prevents acetaminophen-induced hepatic injury in mice. [Internet] [Masters thesis]. University of Louisville; 2011. [cited 2019 Dec 06]. Available from: 10.18297/etd/1273 ; https://ir.library.louisville.edu/etd/1273.

Council of Science Editors:

Schmidt, Robin H. 1. Sulforaphane prevents acetaminophen-induced hepatic injury in mice. [Masters Thesis]. University of Louisville; 2011. Available from: 10.18297/etd/1273 ; https://ir.library.louisville.edu/etd/1273


University of Southern California

3. Lee, Jo. Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells.

Degree: MS, Biochemistry and Molecular Biology, 2014, University of Southern California

 Alcoholism is one of the leading causes of liver disease in the United States. The liver metabolizes 80% of ethanol consumed. Ethanol and its metabolite,… (more)

Subjects/Keywords: r-sulforaphane; heme oxygenase-1

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APA (6th Edition):

Lee, J. (2014). Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1983

Chicago Manual of Style (16th Edition):

Lee, Jo. “Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells.” 2014. Masters Thesis, University of Southern California. Accessed December 06, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1983.

MLA Handbook (7th Edition):

Lee, Jo. “Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells.” 2014. Web. 06 Dec 2019.

Vancouver:

Lee J. Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2019 Dec 06]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1983.

Council of Science Editors:

Lee J. Differential effect of ethanol and r-sulforaphane on regulation of heme oxygenase-1 in endothelial cells. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/121882/rec/1983


Virginia Tech

4. Luo, Jing. Investigating the potential anti-diabetic effect of sulforaphane.

Degree: MS, Human Nutrition, Foods, and Exercise, 2014, Virginia Tech

 Type 2 diabetes (T2D) is a major public health issue worldwide and it currently affects nearly 26 million people in the United States. It is… (more)

Subjects/Keywords: Sulforaphane; Obesity; T2D; Insulin Sensitivity

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APA (6th Edition):

Luo, J. (2014). Investigating the potential anti-diabetic effect of sulforaphane. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/49264

Chicago Manual of Style (16th Edition):

Luo, Jing. “Investigating the potential anti-diabetic effect of sulforaphane.” 2014. Masters Thesis, Virginia Tech. Accessed December 06, 2019. http://hdl.handle.net/10919/49264.

MLA Handbook (7th Edition):

Luo, Jing. “Investigating the potential anti-diabetic effect of sulforaphane.” 2014. Web. 06 Dec 2019.

Vancouver:

Luo J. Investigating the potential anti-diabetic effect of sulforaphane. [Internet] [Masters thesis]. Virginia Tech; 2014. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/10919/49264.

Council of Science Editors:

Luo J. Investigating the potential anti-diabetic effect of sulforaphane. [Masters Thesis]. Virginia Tech; 2014. Available from: http://hdl.handle.net/10919/49264


University of Alberta

5. Yoon, Jenny J. Neuroprotective Effects of Sulforaphane on Oxygen/Glucose Deprived Neurons and Astrocytes.

Degree: MS, Medical Sciences-Paediatrics, 2016, University of Alberta

 Cerebral Palsy (CP) constitutes the primary outcome of premature nerve injury. Impairment of oxygen and glucose supply during pregnancy to the fetus can induce neuron… (more)

Subjects/Keywords: natural health products; cerebral palsy; sulforaphane

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APA (6th Edition):

Yoon, J. J. (2016). Neuroprotective Effects of Sulforaphane on Oxygen/Glucose Deprived Neurons and Astrocytes. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cnc580m69p

Chicago Manual of Style (16th Edition):

Yoon, Jenny J. “Neuroprotective Effects of Sulforaphane on Oxygen/Glucose Deprived Neurons and Astrocytes.” 2016. Masters Thesis, University of Alberta. Accessed December 06, 2019. https://era.library.ualberta.ca/files/cnc580m69p.

MLA Handbook (7th Edition):

Yoon, Jenny J. “Neuroprotective Effects of Sulforaphane on Oxygen/Glucose Deprived Neurons and Astrocytes.” 2016. Web. 06 Dec 2019.

Vancouver:

Yoon JJ. Neuroprotective Effects of Sulforaphane on Oxygen/Glucose Deprived Neurons and Astrocytes. [Internet] [Masters thesis]. University of Alberta; 2016. [cited 2019 Dec 06]. Available from: https://era.library.ualberta.ca/files/cnc580m69p.

Council of Science Editors:

Yoon JJ. Neuroprotective Effects of Sulforaphane on Oxygen/Glucose Deprived Neurons and Astrocytes. [Masters Thesis]. University of Alberta; 2016. Available from: https://era.library.ualberta.ca/files/cnc580m69p


University of Saskatchewan

6. Banigesh, Ali. Antihypertensive effects of sulforaphane.

Degree: 2011, University of Saskatchewan

 Persistent hypertension is associated with a greater incidence of organ damage and conditions such as stroke, heart failure and end–stage kidney disease, which results in… (more)

Subjects/Keywords: SHRsp; sulforaphane; hypertension; phase2 protein inducer

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APA (6th Edition):

Banigesh, A. (2011). Antihypertensive effects of sulforaphane. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/etd-03292011-144232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Banigesh, Ali. “Antihypertensive effects of sulforaphane.” 2011. Thesis, University of Saskatchewan. Accessed December 06, 2019. http://hdl.handle.net/10388/etd-03292011-144232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Banigesh, Ali. “Antihypertensive effects of sulforaphane.” 2011. Web. 06 Dec 2019.

Vancouver:

Banigesh A. Antihypertensive effects of sulforaphane. [Internet] [Thesis]. University of Saskatchewan; 2011. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/10388/etd-03292011-144232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Banigesh A. Antihypertensive effects of sulforaphane. [Thesis]. University of Saskatchewan; 2011. Available from: http://hdl.handle.net/10388/etd-03292011-144232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

7. Townsend, Brigitte Elyse. Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior.

Degree: PhD, Nutritional Sciences, 2015, University of Illinois – Urbana-Champaign

 Innate immune cells provide critical protection against pathogens and produce immune factors that drive adaptive sickness behaviors to facilitate recovery from acute illness and injury.… (more)

Subjects/Keywords: Aging; Broccoli; Neuroinflammation; Sickness Behavior; Sulforaphane

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APA (6th Edition):

Townsend, B. E. (2015). Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/78770

Chicago Manual of Style (16th Edition):

Townsend, Brigitte Elyse. “Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior.” 2015. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed December 06, 2019. http://hdl.handle.net/2142/78770.

MLA Handbook (7th Edition):

Townsend, Brigitte Elyse. “Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior.” 2015. Web. 06 Dec 2019.

Vancouver:

Townsend BE. Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2015. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/2142/78770.

Council of Science Editors:

Townsend BE. Sulforaphane as a potential nutritional intervention to reduce neuroinflammation associated with aging and sickness behavior. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2015. Available from: http://hdl.handle.net/2142/78770


Queens University

8. Philbrook, Nicola. Investigating the Mechanisms of Toxicity of Benzene and its Metabolite Benzoquinone and the Role of Sulforaphane as a Potential Protective Agent in CD-1 Mouse Development .

Degree: Pharmacology and Toxicology, 2015, Queens University

 Benzene is an environmental pollutant and a known human leukemogen. It is also suspected to be associated with in utero-initiated leukemia. However, the mechanisms of… (more)

Subjects/Keywords: benzene; sulforaphane; developmental toxicity; CD-1 mice

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APA (6th Edition):

Philbrook, N. (2015). Investigating the Mechanisms of Toxicity of Benzene and its Metabolite Benzoquinone and the Role of Sulforaphane as a Potential Protective Agent in CD-1 Mouse Development . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/13377

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Philbrook, Nicola. “Investigating the Mechanisms of Toxicity of Benzene and its Metabolite Benzoquinone and the Role of Sulforaphane as a Potential Protective Agent in CD-1 Mouse Development .” 2015. Thesis, Queens University. Accessed December 06, 2019. http://hdl.handle.net/1974/13377.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Philbrook, Nicola. “Investigating the Mechanisms of Toxicity of Benzene and its Metabolite Benzoquinone and the Role of Sulforaphane as a Potential Protective Agent in CD-1 Mouse Development .” 2015. Web. 06 Dec 2019.

Vancouver:

Philbrook N. Investigating the Mechanisms of Toxicity of Benzene and its Metabolite Benzoquinone and the Role of Sulforaphane as a Potential Protective Agent in CD-1 Mouse Development . [Internet] [Thesis]. Queens University; 2015. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/1974/13377.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Philbrook N. Investigating the Mechanisms of Toxicity of Benzene and its Metabolite Benzoquinone and the Role of Sulforaphane as a Potential Protective Agent in CD-1 Mouse Development . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/13377

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. 김, 희수. Sulforaphane sensitizes TRAIL-resistant hepatoma cells to TRAIL-induced apoptosis through reactive oxygen species-mediated upregulation of DR5.

Degree: 2006, Ajou University

"Sulforaphane (SFN) is a chemopreventive agent present in various cruciferous vegetables, including broccoli. Here, we show that treatment with tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)… (more)

Subjects/Keywords: TRAIL; sulforaphane; apoptosis; hepatoma; DR5

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APA (6th Edition):

김, . (2006). Sulforaphane sensitizes TRAIL-resistant hepatoma cells to TRAIL-induced apoptosis through reactive oxygen species-mediated upregulation of DR5. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/1331 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000001250

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

김, 희수. “Sulforaphane sensitizes TRAIL-resistant hepatoma cells to TRAIL-induced apoptosis through reactive oxygen species-mediated upregulation of DR5.” 2006. Thesis, Ajou University. Accessed December 06, 2019. http://repository.ajou.ac.kr/handle/201003/1331 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000001250.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

김, 희수. “Sulforaphane sensitizes TRAIL-resistant hepatoma cells to TRAIL-induced apoptosis through reactive oxygen species-mediated upregulation of DR5.” 2006. Web. 06 Dec 2019.

Vancouver:

김 . Sulforaphane sensitizes TRAIL-resistant hepatoma cells to TRAIL-induced apoptosis through reactive oxygen species-mediated upregulation of DR5. [Internet] [Thesis]. Ajou University; 2006. [cited 2019 Dec 06]. Available from: http://repository.ajou.ac.kr/handle/201003/1331 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000001250.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

김 . Sulforaphane sensitizes TRAIL-resistant hepatoma cells to TRAIL-induced apoptosis through reactive oxygen species-mediated upregulation of DR5. [Thesis]. Ajou University; 2006. Available from: http://repository.ajou.ac.kr/handle/201003/1331 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000001250

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

10. Chi, Po-An. Development of broccoli powder using vacuum belt drying.

Degree: MS, Food Science, 2018, University of Georgia

 The study determined if high quality broccoli powder could be produced by vacuum drying. The effect of temperature, maltodextrin and pretreatment on drying kinetics, physical… (more)

Subjects/Keywords: Broccoli; Vacuum belt drying; Drying model; Vitamin C; Sulforaphane

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APA (6th Edition):

Chi, P. (2018). Development of broccoli powder using vacuum belt drying. (Masters Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/38136

Chicago Manual of Style (16th Edition):

Chi, Po-An. “Development of broccoli powder using vacuum belt drying.” 2018. Masters Thesis, University of Georgia. Accessed December 06, 2019. http://hdl.handle.net/10724/38136.

MLA Handbook (7th Edition):

Chi, Po-An. “Development of broccoli powder using vacuum belt drying.” 2018. Web. 06 Dec 2019.

Vancouver:

Chi P. Development of broccoli powder using vacuum belt drying. [Internet] [Masters thesis]. University of Georgia; 2018. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/10724/38136.

Council of Science Editors:

Chi P. Development of broccoli powder using vacuum belt drying. [Masters Thesis]. University of Georgia; 2018. Available from: http://hdl.handle.net/10724/38136


Université du Québec à Montréal

11. Rojas-Sutterlin, Shanti. Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral.

Degree: 2008, Université du Québec à Montréal

 Le traitement des tumeurs cérébrales représente un défi d'envergure, puisqu'elles sont isolées du reste du corps par la barrière hémato-encéphalique. Il est donc impératif d'accroître… (more)

Subjects/Keywords: Cellule souche; Cancer; Endothélium vasculaire; Tumeur du cerveau; Sulforaphane; Gelatinase B

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APA (6th Edition):

Rojas-Sutterlin, S. (2008). Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral. (Thesis). Université du Québec à Montréal. Retrieved from http://www.archipel.uqam.ca/1500/1/M10553.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rojas-Sutterlin, Shanti. “Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral.” 2008. Thesis, Université du Québec à Montréal. Accessed December 06, 2019. http://www.archipel.uqam.ca/1500/1/M10553.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rojas-Sutterlin, Shanti. “Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral.” 2008. Web. 06 Dec 2019.

Vancouver:

Rojas-Sutterlin S. Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral. [Internet] [Thesis]. Université du Québec à Montréal; 2008. [cited 2019 Dec 06]. Available from: http://www.archipel.uqam.ca/1500/1/M10553.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rojas-Sutterlin S. Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral. [Thesis]. Université du Québec à Montréal; 2008. Available from: http://www.archipel.uqam.ca/1500/1/M10553.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Watson, Gregory (Gregory W.). The Response to Sulforaphane in Metastatic Prostate Cancer Cells.

Degree: PhD, Molecular and Cellular Biology, 2015, Oregon State University

Sulforaphane is an isothiocyanate derived from cruciferous vegetables and has been under investigation as a cancer chemopreventive agent for over two decades. The compound is… (more)

Subjects/Keywords: sulforaphane; Prostate  – Cancer  – Treatment

…Introduction …. 2 1.3 Metabolism and Bioactivity of sulforaphane and Indole… …3Carbinol 4 1.3.1 Sulforaphane … 4 1.3.2 Indole-3… …Sulforaphane …. 8 1.4.1.2 Indole-3-Carbinol ….. 9 1.4.2 Post… …Initiation Suppressive Activity ….. 10 1.4.2.1 Sulforaphane …. 10… …Activity ... 14 1.4.3.1 Sulforaphane …. 14 1.4.3.2 Indole-3… 

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APA (6th Edition):

Watson, G. (. W. ). (2015). The Response to Sulforaphane in Metastatic Prostate Cancer Cells. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/56125

Chicago Manual of Style (16th Edition):

Watson, Gregory (Gregory W ). “The Response to Sulforaphane in Metastatic Prostate Cancer Cells.” 2015. Doctoral Dissertation, Oregon State University. Accessed December 06, 2019. http://hdl.handle.net/1957/56125.

MLA Handbook (7th Edition):

Watson, Gregory (Gregory W ). “The Response to Sulforaphane in Metastatic Prostate Cancer Cells.” 2015. Web. 06 Dec 2019.

Vancouver:

Watson G(W). The Response to Sulforaphane in Metastatic Prostate Cancer Cells. [Internet] [Doctoral dissertation]. Oregon State University; 2015. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/1957/56125.

Council of Science Editors:

Watson G(W). The Response to Sulforaphane in Metastatic Prostate Cancer Cells. [Doctoral Dissertation]. Oregon State University; 2015. Available from: http://hdl.handle.net/1957/56125


Colorado State University

13. Acerbo, Evan Robert. PROTECTIVE EFFECTS OF SULFORAPHANE ON NITRIC OXIDE INDUCED MITOCHONDRIAL DYSFUNCTION.

Degree: MS(M.S.), Environmental and Radiological Health Sciences, 2019, Colorado State University

Sulforaphane (SFN), an isothiocyanate compound that is formed in the breakdown process of cruciferous vegetables, has demonstrated the ability to interfere with dynamin-related protein 1… (more)

Subjects/Keywords: mitochondrial fission; Nitric oxide; Dynamin-related protein 1; Sulforaphane; neurodegeneration

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APA (6th Edition):

Acerbo, E. R. (2019). PROTECTIVE EFFECTS OF SULFORAPHANE ON NITRIC OXIDE INDUCED MITOCHONDRIAL DYSFUNCTION. (Masters Thesis). Colorado State University. Retrieved from http://hdl.handle.net/10217/195379

Chicago Manual of Style (16th Edition):

Acerbo, Evan Robert. “PROTECTIVE EFFECTS OF SULFORAPHANE ON NITRIC OXIDE INDUCED MITOCHONDRIAL DYSFUNCTION.” 2019. Masters Thesis, Colorado State University. Accessed December 06, 2019. http://hdl.handle.net/10217/195379.

MLA Handbook (7th Edition):

Acerbo, Evan Robert. “PROTECTIVE EFFECTS OF SULFORAPHANE ON NITRIC OXIDE INDUCED MITOCHONDRIAL DYSFUNCTION.” 2019. Web. 06 Dec 2019.

Vancouver:

Acerbo ER. PROTECTIVE EFFECTS OF SULFORAPHANE ON NITRIC OXIDE INDUCED MITOCHONDRIAL DYSFUNCTION. [Internet] [Masters thesis]. Colorado State University; 2019. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/10217/195379.

Council of Science Editors:

Acerbo ER. PROTECTIVE EFFECTS OF SULFORAPHANE ON NITRIC OXIDE INDUCED MITOCHONDRIAL DYSFUNCTION. [Masters Thesis]. Colorado State University; 2019. Available from: http://hdl.handle.net/10217/195379


University of Illinois – Urbana-Champaign

14. Dosz, Edward. Improving the health benefits of broccoli through myrosinase maintenance.

Degree: PhD, 0037, 2014, University of Illinois – Urbana-Champaign

 Broccoli (Brassica oleracea L.) is the most significant source of sulforaphane (SF), a potent cancer-preventative agent in the Western diet. Because SF is unstable; the… (more)

Subjects/Keywords: broccoli: myrosinase; sulforaphane; thermal stability of brassica phytochemicals

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APA (6th Edition):

Dosz, E. (2014). Improving the health benefits of broccoli through myrosinase maintenance. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/49815

Chicago Manual of Style (16th Edition):

Dosz, Edward. “Improving the health benefits of broccoli through myrosinase maintenance.” 2014. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed December 06, 2019. http://hdl.handle.net/2142/49815.

MLA Handbook (7th Edition):

Dosz, Edward. “Improving the health benefits of broccoli through myrosinase maintenance.” 2014. Web. 06 Dec 2019.

Vancouver:

Dosz E. Improving the health benefits of broccoli through myrosinase maintenance. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2014. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/2142/49815.

Council of Science Editors:

Dosz E. Improving the health benefits of broccoli through myrosinase maintenance. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2014. Available from: http://hdl.handle.net/2142/49815


University of Illinois – Urbana-Champaign

15. Wang, Yanling. Improving sulforaphane bioavailability from cooked broccoli.

Degree: PhD, Food Science & Human Nutrition, 2019, University of Illinois – Urbana-Champaign

 Broccoli is a nutritious food containing many macro- and micro-nutrients. Most consumers prefer cooked broccoli (CB); unfortunately, the enzyme myrosinase is inactivated by heat, resulting… (more)

Subjects/Keywords: sulforaphane; cooked broccoli; microbiota; inflammation; quercetin-3-O-sophoroside

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APA (6th Edition):

Wang, Y. (2019). Improving sulforaphane bioavailability from cooked broccoli. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/104733

Chicago Manual of Style (16th Edition):

Wang, Yanling. “Improving sulforaphane bioavailability from cooked broccoli.” 2019. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed December 06, 2019. http://hdl.handle.net/2142/104733.

MLA Handbook (7th Edition):

Wang, Yanling. “Improving sulforaphane bioavailability from cooked broccoli.” 2019. Web. 06 Dec 2019.

Vancouver:

Wang Y. Improving sulforaphane bioavailability from cooked broccoli. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2019. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/2142/104733.

Council of Science Editors:

Wang Y. Improving sulforaphane bioavailability from cooked broccoli. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2019. Available from: http://hdl.handle.net/2142/104733


Universidade do Rio Grande do Sul

16. Bonetto, Jéssica Hellen Poletto. Efeitos do sulforanato sobre a função mecânica e parâmetros de estresse oxidativo de corações isolados de ratos submetidos à isquemia e reperfusão.

Degree: 2015, Universidade do Rio Grande do Sul

 A isquemia seguida de reperfusão está associada com a ativação de uma cascata de eventos danosos intimamente relacionados ao estresse oxidativo. Durante a isquemia, ocorre… (more)

Subjects/Keywords: Estresse oxidativo; Mechanical function; Sulforaphane; Isquemia; Reperfusão miocárdica; Oxidative stress; Isotiocianatos; Antioxidantes; Coração

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APA (6th Edition):

Bonetto, J. H. P. (2015). Efeitos do sulforanato sobre a função mecânica e parâmetros de estresse oxidativo de corações isolados de ratos submetidos à isquemia e reperfusão. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/117556

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bonetto, Jéssica Hellen Poletto. “Efeitos do sulforanato sobre a função mecânica e parâmetros de estresse oxidativo de corações isolados de ratos submetidos à isquemia e reperfusão.” 2015. Thesis, Universidade do Rio Grande do Sul. Accessed December 06, 2019. http://hdl.handle.net/10183/117556.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bonetto, Jéssica Hellen Poletto. “Efeitos do sulforanato sobre a função mecânica e parâmetros de estresse oxidativo de corações isolados de ratos submetidos à isquemia e reperfusão.” 2015. Web. 06 Dec 2019.

Vancouver:

Bonetto JHP. Efeitos do sulforanato sobre a função mecânica e parâmetros de estresse oxidativo de corações isolados de ratos submetidos à isquemia e reperfusão. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2015. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/10183/117556.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bonetto JHP. Efeitos do sulforanato sobre a função mecânica e parâmetros de estresse oxidativo de corações isolados de ratos submetidos à isquemia e reperfusão. [Thesis]. Universidade do Rio Grande do Sul; 2015. Available from: http://hdl.handle.net/10183/117556

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Atwell, Lauren Lee, 1984-. Characterizing the Roles of Dietary Sulforaphane in Human Health.

Degree: PhD, Nutrition, 2015, Oregon State University

 The consumption of cruciferous vegetables is associated with several health benefits, including cancer prevention. Many of these benefits are attributed to the phytochemical, sulforaphane (SFN),… (more)

Subjects/Keywords: sulforaphane; Organosulfur compounds  – Health aspects

…TABLE OF CONTENTS Page 1 Epigenetic Regulation by Sulforaphane: Opportunities for Breast and… …1 2 Sulforaphane Bioavailability and Chemopreventive Activity in Women Scheduled for… …18 3 Absorption and Chemopreventive Targets of Sulforaphane in Humans following… …50 3.2 Sulforaphane (SFN) metabolite levels in plasma and urine following… …Distribution of individual sulforaphane (SFN) metabolites in plasma and urine at peak… 

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APA (6th Edition):

Atwell, Lauren Lee, 1. (2015). Characterizing the Roles of Dietary Sulforaphane in Human Health. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/56126

Chicago Manual of Style (16th Edition):

Atwell, Lauren Lee, 1984-. “Characterizing the Roles of Dietary Sulforaphane in Human Health.” 2015. Doctoral Dissertation, Oregon State University. Accessed December 06, 2019. http://hdl.handle.net/1957/56126.

MLA Handbook (7th Edition):

Atwell, Lauren Lee, 1984-. “Characterizing the Roles of Dietary Sulforaphane in Human Health.” 2015. Web. 06 Dec 2019.

Vancouver:

Atwell, Lauren Lee 1. Characterizing the Roles of Dietary Sulforaphane in Human Health. [Internet] [Doctoral dissertation]. Oregon State University; 2015. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/1957/56126.

Council of Science Editors:

Atwell, Lauren Lee 1. Characterizing the Roles of Dietary Sulforaphane in Human Health. [Doctoral Dissertation]. Oregon State University; 2015. Available from: http://hdl.handle.net/1957/56126


Virginia Commonwealth University

18. Russell, Nicholas H. Heme Oxygenase 1 expression after traumatic brain injury and effect of pharmacological manipulation on functional recovery.

Degree: PhD, Neuroscience, 2017, Virginia Commonwealth University

  Traumatic Brain Injury (TBI) is an increasingly diagnosed constellation of injuries derived from acute mechanical trauma to the brain. With the rise of advanced… (more)

Subjects/Keywords: Traumatic Brain Injury; TBI; Heme Oxygenase 1; HO-1; Hemin; Sulforaphane; Medical Neurobiology

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APA (6th Edition):

Russell, N. H. (2017). Heme Oxygenase 1 expression after traumatic brain injury and effect of pharmacological manipulation on functional recovery. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/5525

Chicago Manual of Style (16th Edition):

Russell, Nicholas H. “Heme Oxygenase 1 expression after traumatic brain injury and effect of pharmacological manipulation on functional recovery.” 2017. Doctoral Dissertation, Virginia Commonwealth University. Accessed December 06, 2019. https://scholarscompass.vcu.edu/etd/5525.

MLA Handbook (7th Edition):

Russell, Nicholas H. “Heme Oxygenase 1 expression after traumatic brain injury and effect of pharmacological manipulation on functional recovery.” 2017. Web. 06 Dec 2019.

Vancouver:

Russell NH. Heme Oxygenase 1 expression after traumatic brain injury and effect of pharmacological manipulation on functional recovery. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2017. [cited 2019 Dec 06]. Available from: https://scholarscompass.vcu.edu/etd/5525.

Council of Science Editors:

Russell NH. Heme Oxygenase 1 expression after traumatic brain injury and effect of pharmacological manipulation on functional recovery. [Doctoral Dissertation]. Virginia Commonwealth University; 2017. Available from: https://scholarscompass.vcu.edu/etd/5525

19. Ahn, Eunjee. Histone Deacetylase Inhibitors Trichostatin A (tsa) And Sulforaphane (sfn) Modulate Vitamin D Responsive Cyp24 Gene Expression in 3t3-l1 Preadipocytes.

Degree: MS, Nutrition, 2013, University of Massachusetts

  Vitamin D plays an important role in preserving healthy bones, and has additional roles in the body, including modulation of cell growth, differentiation, neuromuscular… (more)

Subjects/Keywords: HISTONE DEACETYLASE INHIBITORS; TRICHOSTATIN A (TSA); SULFORAPHANE (SFN); CYP24; 3T3-L1 PREADIPOCYTES; Nutrition

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APA (6th Edition):

Ahn, E. (2013). Histone Deacetylase Inhibitors Trichostatin A (tsa) And Sulforaphane (sfn) Modulate Vitamin D Responsive Cyp24 Gene Expression in 3t3-l1 Preadipocytes. (Masters Thesis). University of Massachusetts. Retrieved from https://scholarworks.umass.edu/theses/1102

Chicago Manual of Style (16th Edition):

Ahn, Eunjee. “Histone Deacetylase Inhibitors Trichostatin A (tsa) And Sulforaphane (sfn) Modulate Vitamin D Responsive Cyp24 Gene Expression in 3t3-l1 Preadipocytes.” 2013. Masters Thesis, University of Massachusetts. Accessed December 06, 2019. https://scholarworks.umass.edu/theses/1102.

MLA Handbook (7th Edition):

Ahn, Eunjee. “Histone Deacetylase Inhibitors Trichostatin A (tsa) And Sulforaphane (sfn) Modulate Vitamin D Responsive Cyp24 Gene Expression in 3t3-l1 Preadipocytes.” 2013. Web. 06 Dec 2019.

Vancouver:

Ahn E. Histone Deacetylase Inhibitors Trichostatin A (tsa) And Sulforaphane (sfn) Modulate Vitamin D Responsive Cyp24 Gene Expression in 3t3-l1 Preadipocytes. [Internet] [Masters thesis]. University of Massachusetts; 2013. [cited 2019 Dec 06]. Available from: https://scholarworks.umass.edu/theses/1102.

Council of Science Editors:

Ahn E. Histone Deacetylase Inhibitors Trichostatin A (tsa) And Sulforaphane (sfn) Modulate Vitamin D Responsive Cyp24 Gene Expression in 3t3-l1 Preadipocytes. [Masters Thesis]. University of Massachusetts; 2013. Available from: https://scholarworks.umass.edu/theses/1102


University of Toronto

20. Mokhtari, Reza Bayat. Dietary Chemoprevention Agent Sulforaphane Inhibits Growth, Survival and Tumorigenic Activity in Human Neuroblastoma.

Degree: 2010, University of Toronto

Objective: To evaluate the anti-tumor and histone deacetylase (HDAC) inhibitory activity of the dietary isothiocyanate, sulforaphane (SFN) in the paediatric cancer,neuroblastoma (NB). Materials and Methods:… (more)

Subjects/Keywords: Neuroblastoma; Sulforaphane; Apotosis; Cell cycle arrest; Differentiation; Dietary Compond; chemoprevention; Tumorigenic activity; 0564

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APA (6th Edition):

Mokhtari, R. B. (2010). Dietary Chemoprevention Agent Sulforaphane Inhibits Growth, Survival and Tumorigenic Activity in Human Neuroblastoma. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25424

Chicago Manual of Style (16th Edition):

Mokhtari, Reza Bayat. “Dietary Chemoprevention Agent Sulforaphane Inhibits Growth, Survival and Tumorigenic Activity in Human Neuroblastoma.” 2010. Masters Thesis, University of Toronto. Accessed December 06, 2019. http://hdl.handle.net/1807/25424.

MLA Handbook (7th Edition):

Mokhtari, Reza Bayat. “Dietary Chemoprevention Agent Sulforaphane Inhibits Growth, Survival and Tumorigenic Activity in Human Neuroblastoma.” 2010. Web. 06 Dec 2019.

Vancouver:

Mokhtari RB. Dietary Chemoprevention Agent Sulforaphane Inhibits Growth, Survival and Tumorigenic Activity in Human Neuroblastoma. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/1807/25424.

Council of Science Editors:

Mokhtari RB. Dietary Chemoprevention Agent Sulforaphane Inhibits Growth, Survival and Tumorigenic Activity in Human Neuroblastoma. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25424


Queens University

21. Harris, Christopher. Modification of Nucleotide Excision Repair Activity by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and Sulforaphane .

Degree: Biomedical and Molecular Sciences, Queens University

 The studies described in this thesis investigated the relationship between the DNA repair pathway nucleotide excision repair (NER, which is critical for protecting against carcinogenesis),… (more)

Subjects/Keywords: Nucleotide Excision Repair; Sulforaphane; NNK; nrf2

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APA (6th Edition):

Harris, C. (n.d.). Modification of Nucleotide Excision Repair Activity by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and Sulforaphane . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/15338

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Harris, Christopher. “Modification of Nucleotide Excision Repair Activity by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and Sulforaphane .” Thesis, Queens University. Accessed December 06, 2019. http://hdl.handle.net/1974/15338.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Harris, Christopher. “Modification of Nucleotide Excision Repair Activity by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and Sulforaphane .” Web. 06 Dec 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Harris C. Modification of Nucleotide Excision Repair Activity by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and Sulforaphane . [Internet] [Thesis]. Queens University; [cited 2019 Dec 06]. Available from: http://hdl.handle.net/1974/15338.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Harris C. Modification of Nucleotide Excision Repair Activity by 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) and Sulforaphane . [Thesis]. Queens University; Available from: http://hdl.handle.net/1974/15338

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Universidade do Rio Grande do Sul

22. Conzatti, Adriana. Efeitos do sulforafano sobre o remodelamento do ventrículo direito e estado redox em modelo de hipertensão arterial pulmonar.

Degree: 2016, Universidade do Rio Grande do Sul

A hipertensão arterial pulmonar (HAP) é uma doença grave, caracterizada pelo aumento na resistência vascular pulmonar, elevando a pós-carga imposta ao ventrículo direito. Na tentativa… (more)

Subjects/Keywords: Pulmonary arterial hypertension; Hipertensão pulmonar; Monocrotaline; Remodelação ventricular; Espécies de oxigênio reativas; Sulforaphane; Monocrotalina; Oxidative stress; Isotiocianatos

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APA (6th Edition):

Conzatti, A. (2016). Efeitos do sulforafano sobre o remodelamento do ventrículo direito e estado redox em modelo de hipertensão arterial pulmonar. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/143610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Conzatti, Adriana. “Efeitos do sulforafano sobre o remodelamento do ventrículo direito e estado redox em modelo de hipertensão arterial pulmonar.” 2016. Thesis, Universidade do Rio Grande do Sul. Accessed December 06, 2019. http://hdl.handle.net/10183/143610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Conzatti, Adriana. “Efeitos do sulforafano sobre o remodelamento do ventrículo direito e estado redox em modelo de hipertensão arterial pulmonar.” 2016. Web. 06 Dec 2019.

Vancouver:

Conzatti A. Efeitos do sulforafano sobre o remodelamento do ventrículo direito e estado redox em modelo de hipertensão arterial pulmonar. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2016. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/10183/143610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Conzatti A. Efeitos do sulforafano sobre o remodelamento do ventrículo direito e estado redox em modelo de hipertensão arterial pulmonar. [Thesis]. Universidade do Rio Grande do Sul; 2016. Available from: http://hdl.handle.net/10183/143610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Schmidt, Robin H., 1984-. Olanzapine-induced liver injury : direct metabolic effects, exacerbation by high-fat diet, and protection with sulforaphane.

Degree: PhD, 2013, University of Louisville

 Olanzapine (OLZ) is an effective first-line treatment for schizophrenia and bipolar disorder. The benefits of OLZ are countermanded by side effects such as weight gain,… (more)

Subjects/Keywords: Antipsychotics; Pharmacology; Olanzapine; Glutamine; Hepatology; Sulforaphane

…or OLZ alone. Lastly, sulforaphane (SFN) was tested as a possible preventive… …in fatty liver diseases?...........5 3. Can Nrf2 activation with sulforaphane protect… …19 L. Sulforaphane administration . …19 III. NEW MECHANISTIC INSIGHT INTO… …sulforaphane protect against OLZ-induced liver injury? The formation of electrophilic and oxidative… …sulforaphane (SFN) have neither reducing nor oxidizing capability.69,70 SFN instead works… 

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APA (6th Edition):

Schmidt, Robin H., 1. (2013). Olanzapine-induced liver injury : direct metabolic effects, exacerbation by high-fat diet, and protection with sulforaphane. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1272 ; https://ir.library.louisville.edu/etd/1272

Chicago Manual of Style (16th Edition):

Schmidt, Robin H., 1984-. “Olanzapine-induced liver injury : direct metabolic effects, exacerbation by high-fat diet, and protection with sulforaphane.” 2013. Doctoral Dissertation, University of Louisville. Accessed December 06, 2019. 10.18297/etd/1272 ; https://ir.library.louisville.edu/etd/1272.

MLA Handbook (7th Edition):

Schmidt, Robin H., 1984-. “Olanzapine-induced liver injury : direct metabolic effects, exacerbation by high-fat diet, and protection with sulforaphane.” 2013. Web. 06 Dec 2019.

Vancouver:

Schmidt, Robin H. 1. Olanzapine-induced liver injury : direct metabolic effects, exacerbation by high-fat diet, and protection with sulforaphane. [Internet] [Doctoral dissertation]. University of Louisville; 2013. [cited 2019 Dec 06]. Available from: 10.18297/etd/1272 ; https://ir.library.louisville.edu/etd/1272.

Council of Science Editors:

Schmidt, Robin H. 1. Olanzapine-induced liver injury : direct metabolic effects, exacerbation by high-fat diet, and protection with sulforaphane. [Doctoral Dissertation]. University of Louisville; 2013. Available from: 10.18297/etd/1272 ; https://ir.library.louisville.edu/etd/1272


North-West University

24. Crous, Ané. The effect of sulforaphane on oxidative stress and biotransformation in HepaRG cells / A. Crous.

Degree: 2013, North-West University

Sulforaphane is an isothiocyanate found in high concentrations in cruciferous vegetables like broccoli. Sulforaphane has received much attention due to the evidence that it inhibits… (more)

Subjects/Keywords: Sulforaphane; oxidative stress; t-BHP; MTT; phase I and phase II; biotransformation; LC-ESI-MS/MS; HepaRG cells

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APA (6th Edition):

Crous, A. (2013). The effect of sulforaphane on oxidative stress and biotransformation in HepaRG cells / A. Crous. (Thesis). North-West University. Retrieved from http://hdl.handle.net/10394/9642

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Crous, Ané. “The effect of sulforaphane on oxidative stress and biotransformation in HepaRG cells / A. Crous. ” 2013. Thesis, North-West University. Accessed December 06, 2019. http://hdl.handle.net/10394/9642.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Crous, Ané. “The effect of sulforaphane on oxidative stress and biotransformation in HepaRG cells / A. Crous. ” 2013. Web. 06 Dec 2019.

Vancouver:

Crous A. The effect of sulforaphane on oxidative stress and biotransformation in HepaRG cells / A. Crous. [Internet] [Thesis]. North-West University; 2013. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/10394/9642.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Crous A. The effect of sulforaphane on oxidative stress and biotransformation in HepaRG cells / A. Crous. [Thesis]. North-West University; 2013. Available from: http://hdl.handle.net/10394/9642

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

25. Huang, Ching-Yu. Effect of broccoli bioactives on prostate cancer cell invasion in vitro.

Degree: MS, 0037, 2014, University of Illinois – Urbana-Champaign

 Previous studies have shown that broccoli bioactives may have anti-cancer activity. In this work, we hypothesized that broccoli bioactive components inhibit invasion of human prostate… (more)

Subjects/Keywords: Human prostate cancer cell; DU145; invasion; epithelial-mesenchymal transition; broccoli bioactives; sulforaphane; indole-3-carbinol; neoglucobrassicin; quercetin; 3,3'-indolylmethane

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APA (6th Edition):

Huang, C. (2014). Effect of broccoli bioactives on prostate cancer cell invasion in vitro. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/50403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Ching-Yu. “Effect of broccoli bioactives on prostate cancer cell invasion in vitro.” 2014. Thesis, University of Illinois – Urbana-Champaign. Accessed December 06, 2019. http://hdl.handle.net/2142/50403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Ching-Yu. “Effect of broccoli bioactives on prostate cancer cell invasion in vitro.” 2014. Web. 06 Dec 2019.

Vancouver:

Huang C. Effect of broccoli bioactives on prostate cancer cell invasion in vitro. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2014. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/2142/50403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang C. Effect of broccoli bioactives on prostate cancer cell invasion in vitro. [Thesis]. University of Illinois – Urbana-Champaign; 2014. Available from: http://hdl.handle.net/2142/50403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Burnett, Joseph. The Natural Product Sulforaphane Inhibits Breast Cancer Stem Cell Targets in Triple Negative and Trastuzumab-resistant Breast Cancers.

Degree: PhD, Pharmaceutical Sciences, 2015, University of Michigan

 The cancer stem cell (CSC) hypothesis provides a hierarchical model which explains the observed heterogeneity of cancers cells within a tumor. CSCs sit at the… (more)

Subjects/Keywords: Sulforaphane; Triple Negative Breast Cancer; Trastuzumab Resistant Breast Cancer; Cancer Stem Cells; Pharmacy and Pharmacology; Health Sciences

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APA (6th Edition):

Burnett, J. (2015). The Natural Product Sulforaphane Inhibits Breast Cancer Stem Cell Targets in Triple Negative and Trastuzumab-resistant Breast Cancers. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/111599

Chicago Manual of Style (16th Edition):

Burnett, Joseph. “The Natural Product Sulforaphane Inhibits Breast Cancer Stem Cell Targets in Triple Negative and Trastuzumab-resistant Breast Cancers.” 2015. Doctoral Dissertation, University of Michigan. Accessed December 06, 2019. http://hdl.handle.net/2027.42/111599.

MLA Handbook (7th Edition):

Burnett, Joseph. “The Natural Product Sulforaphane Inhibits Breast Cancer Stem Cell Targets in Triple Negative and Trastuzumab-resistant Breast Cancers.” 2015. Web. 06 Dec 2019.

Vancouver:

Burnett J. The Natural Product Sulforaphane Inhibits Breast Cancer Stem Cell Targets in Triple Negative and Trastuzumab-resistant Breast Cancers. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/2027.42/111599.

Council of Science Editors:

Burnett J. The Natural Product Sulforaphane Inhibits Breast Cancer Stem Cell Targets in Triple Negative and Trastuzumab-resistant Breast Cancers. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/111599


Universidade do Rio Grande do Sul

27. Corssac, Giana Blume. Efeitos do sulforafano em parâmetros de estresse oxidativo em cultura de cardiomiócitos adultos.

Degree: 2017, Universidade do Rio Grande do Sul

 O sulforafano (SFN) é um composto natural que possui propriedades antioxidantes, estimulando, principalmente, o sistema antioxidante endógeno celular. Este composto está associado a uma via… (more)

Subjects/Keywords: Primary culture; Estresse oxidativo; Miócitos cardíacos; Oxidative stress; Compostos fitoquímicos; PGC-1α; Nrf2; Fator 2 relacionado a NF-E2; Sequência rica em GC; Sulforaphane; Adult cardiomyocyte

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Corssac, G. B. (2017). Efeitos do sulforafano em parâmetros de estresse oxidativo em cultura de cardiomiócitos adultos. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/165287

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Corssac, Giana Blume. “Efeitos do sulforafano em parâmetros de estresse oxidativo em cultura de cardiomiócitos adultos.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed December 06, 2019. http://hdl.handle.net/10183/165287.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Corssac, Giana Blume. “Efeitos do sulforafano em parâmetros de estresse oxidativo em cultura de cardiomiócitos adultos.” 2017. Web. 06 Dec 2019.

Vancouver:

Corssac GB. Efeitos do sulforafano em parâmetros de estresse oxidativo em cultura de cardiomiócitos adultos. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/10183/165287.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Corssac GB. Efeitos do sulforafano em parâmetros de estresse oxidativo em cultura de cardiomiócitos adultos. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/165287

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

28. Boorgula, Smitha. Assessing Hepatic Gene Expression in Response to Xenobiotic Exposure in Mice.

Degree: MS, Animal and Poultry Sciences, 2007, Virginia Tech

 Xenobiotics are plant derived compounds metabolized by phase I and II liver enzymes. Phase I enzymes increase, and phase II enzymes decrease, xenobiotic toxicity. Xenobiotics… (more)

Subjects/Keywords: Ergotamine; Gene expression; Hepatic phase I and II enzymes; Sulforaphane

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Boorgula, S. (2007). Assessing Hepatic Gene Expression in Response to Xenobiotic Exposure in Mice. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/32186

Chicago Manual of Style (16th Edition):

Boorgula, Smitha. “Assessing Hepatic Gene Expression in Response to Xenobiotic Exposure in Mice.” 2007. Masters Thesis, Virginia Tech. Accessed December 06, 2019. http://hdl.handle.net/10919/32186.

MLA Handbook (7th Edition):

Boorgula, Smitha. “Assessing Hepatic Gene Expression in Response to Xenobiotic Exposure in Mice.” 2007. Web. 06 Dec 2019.

Vancouver:

Boorgula S. Assessing Hepatic Gene Expression in Response to Xenobiotic Exposure in Mice. [Internet] [Masters thesis]. Virginia Tech; 2007. [cited 2019 Dec 06]. Available from: http://hdl.handle.net/10919/32186.

Council of Science Editors:

Boorgula S. Assessing Hepatic Gene Expression in Response to Xenobiotic Exposure in Mice. [Masters Thesis]. Virginia Tech; 2007. Available from: http://hdl.handle.net/10919/32186

29. SHRIDHIVYA REDDY AMARR. MECHANISTIC INVESTIGATION OF THE ANTITUMOR, ANTI-INFLAMMATORY AND CYTOPROTECTIVE POTENTIAL OF ISOTHIOCYANATE ANALOGS.

Degree: 2013, National University of Singapore

Subjects/Keywords: ISOTHIOCYANATES; SULFORAPHANE; anti-NFkapppaB drugs; MITOTIC INHIBITORS; Nrf2 inducers; drug discovery

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

AMARR, S. R. (2013). MECHANISTIC INVESTIGATION OF THE ANTITUMOR, ANTI-INFLAMMATORY AND CYTOPROTECTIVE POTENTIAL OF ISOTHIOCYANATE ANALOGS. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/49305

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

AMARR, SHRIDHIVYA REDDY. “MECHANISTIC INVESTIGATION OF THE ANTITUMOR, ANTI-INFLAMMATORY AND CYTOPROTECTIVE POTENTIAL OF ISOTHIOCYANATE ANALOGS.” 2013. Thesis, National University of Singapore. Accessed December 06, 2019. http://scholarbank.nus.edu.sg/handle/10635/49305.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

AMARR, SHRIDHIVYA REDDY. “MECHANISTIC INVESTIGATION OF THE ANTITUMOR, ANTI-INFLAMMATORY AND CYTOPROTECTIVE POTENTIAL OF ISOTHIOCYANATE ANALOGS.” 2013. Web. 06 Dec 2019.

Vancouver:

AMARR SR. MECHANISTIC INVESTIGATION OF THE ANTITUMOR, ANTI-INFLAMMATORY AND CYTOPROTECTIVE POTENTIAL OF ISOTHIOCYANATE ANALOGS. [Internet] [Thesis]. National University of Singapore; 2013. [cited 2019 Dec 06]. Available from: http://scholarbank.nus.edu.sg/handle/10635/49305.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

AMARR SR. MECHANISTIC INVESTIGATION OF THE ANTITUMOR, ANTI-INFLAMMATORY AND CYTOPROTECTIVE POTENTIAL OF ISOTHIOCYANATE ANALOGS. [Thesis]. National University of Singapore; 2013. Available from: http://scholarbank.nus.edu.sg/handle/10635/49305

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université du Québec à Montréal

30. Rojas-Sutterlin, Shanti. Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral.

Degree: 2008, Université du Québec à Montréal

 Le traitement des tumeurs cérébrales représente un défi d'envergure, puisqu'elles sont isolées du reste du corps par la barrière hémato-encéphalique. Il est donc impératif d'accroître… (more)

Subjects/Keywords: Cellule souche; Cancer; Endothélium vasculaire; Tumeur du cerveau; Sulforaphane; Gelatinase B

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rojas-Sutterlin, S. (2008). Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral. (Thesis). Université du Québec à Montréal. Retrieved from http://archipel.uqam.ca/1500/1/M10553.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rojas-Sutterlin, Shanti. “Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral.” 2008. Thesis, Université du Québec à Montréal. Accessed December 06, 2019. http://archipel.uqam.ca/1500/1/M10553.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rojas-Sutterlin, Shanti. “Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral.” 2008. Web. 06 Dec 2019.

Vancouver:

Rojas-Sutterlin S. Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral. [Internet] [Thesis]. Université du Québec à Montréal; 2008. [cited 2019 Dec 06]. Available from: http://archipel.uqam.ca/1500/1/M10553.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rojas-Sutterlin S. Implication de MMP-9 dans le phénotype invasif des cellules souches cancéreuses et dans l'endothélium vasculaire cérébral tumoral. [Thesis]. Université du Québec à Montréal; 2008. Available from: http://archipel.uqam.ca/1500/1/M10553.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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