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You searched for subject:(Steatosis). Showing records 1 – 30 of 164 total matches.

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University of Florida

1. Kim, Min Hyun. Regulation and Function of Zinc and Zinc Transporters during ER Stress.

Degree: PhD, Nutritional Sciences, 2017, University of Florida

 Extensive endoplasmic reticulum (ER) stress damages the liver causing apoptosis and steatosis, despite the activation of the unfolded protein response (UPR). Restriction of zinc from… (more)

Subjects/Keywords: apoptosis  – steatosis  – upr  – zinc  – zip14

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APA (6th Edition):

Kim, M. H. (2017). Regulation and Function of Zinc and Zinc Transporters during ER Stress. (Doctoral Dissertation). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0051263

Chicago Manual of Style (16th Edition):

Kim, Min Hyun. “Regulation and Function of Zinc and Zinc Transporters during ER Stress.” 2017. Doctoral Dissertation, University of Florida. Accessed January 26, 2021. https://ufdc.ufl.edu/UFE0051263.

MLA Handbook (7th Edition):

Kim, Min Hyun. “Regulation and Function of Zinc and Zinc Transporters during ER Stress.” 2017. Web. 26 Jan 2021.

Vancouver:

Kim MH. Regulation and Function of Zinc and Zinc Transporters during ER Stress. [Internet] [Doctoral dissertation]. University of Florida; 2017. [cited 2021 Jan 26]. Available from: https://ufdc.ufl.edu/UFE0051263.

Council of Science Editors:

Kim MH. Regulation and Function of Zinc and Zinc Transporters during ER Stress. [Doctoral Dissertation]. University of Florida; 2017. Available from: https://ufdc.ufl.edu/UFE0051263

2. Hendrikx, T. Intracellular traffic jam : cholesterol accumulation as cause for chronic inflammatory diseases.

Degree: 2015, Maastricht University

 A rise in obesity has brought with it a rise in the incidences of fatty liver disease. Some people go on to develop liver inflammation,… (more)

Subjects/Keywords: hepatitis; steatosis; macrophages

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APA (6th Edition):

Hendrikx, T. (2015). Intracellular traffic jam : cholesterol accumulation as cause for chronic inflammatory diseases. (Doctoral Dissertation). Maastricht University. Retrieved from https://cris.maastrichtuniversity.nl/en/publications/418fc04e-a720-4470-92a8-da5b284bd821 ; urn:nbn:nl:ui:27-418fc04e-a720-4470-92a8-da5b284bd821 ; 418fc04e-a720-4470-92a8-da5b284bd821 ; urn:isbn:9789462950603 ; urn:nbn:nl:ui:27-418fc04e-a720-4470-92a8-da5b284bd821 ; https://cris.maastrichtuniversity.nl/en/publications/418fc04e-a720-4470-92a8-da5b284bd821

Chicago Manual of Style (16th Edition):

Hendrikx, T. “Intracellular traffic jam : cholesterol accumulation as cause for chronic inflammatory diseases.” 2015. Doctoral Dissertation, Maastricht University. Accessed January 26, 2021. https://cris.maastrichtuniversity.nl/en/publications/418fc04e-a720-4470-92a8-da5b284bd821 ; urn:nbn:nl:ui:27-418fc04e-a720-4470-92a8-da5b284bd821 ; 418fc04e-a720-4470-92a8-da5b284bd821 ; urn:isbn:9789462950603 ; urn:nbn:nl:ui:27-418fc04e-a720-4470-92a8-da5b284bd821 ; https://cris.maastrichtuniversity.nl/en/publications/418fc04e-a720-4470-92a8-da5b284bd821.

MLA Handbook (7th Edition):

Hendrikx, T. “Intracellular traffic jam : cholesterol accumulation as cause for chronic inflammatory diseases.” 2015. Web. 26 Jan 2021.

Vancouver:

Hendrikx T. Intracellular traffic jam : cholesterol accumulation as cause for chronic inflammatory diseases. [Internet] [Doctoral dissertation]. Maastricht University; 2015. [cited 2021 Jan 26]. Available from: https://cris.maastrichtuniversity.nl/en/publications/418fc04e-a720-4470-92a8-da5b284bd821 ; urn:nbn:nl:ui:27-418fc04e-a720-4470-92a8-da5b284bd821 ; 418fc04e-a720-4470-92a8-da5b284bd821 ; urn:isbn:9789462950603 ; urn:nbn:nl:ui:27-418fc04e-a720-4470-92a8-da5b284bd821 ; https://cris.maastrichtuniversity.nl/en/publications/418fc04e-a720-4470-92a8-da5b284bd821.

Council of Science Editors:

Hendrikx T. Intracellular traffic jam : cholesterol accumulation as cause for chronic inflammatory diseases. [Doctoral Dissertation]. Maastricht University; 2015. Available from: https://cris.maastrichtuniversity.nl/en/publications/418fc04e-a720-4470-92a8-da5b284bd821 ; urn:nbn:nl:ui:27-418fc04e-a720-4470-92a8-da5b284bd821 ; 418fc04e-a720-4470-92a8-da5b284bd821 ; urn:isbn:9789462950603 ; urn:nbn:nl:ui:27-418fc04e-a720-4470-92a8-da5b284bd821 ; https://cris.maastrichtuniversity.nl/en/publications/418fc04e-a720-4470-92a8-da5b284bd821


Université Paris-Sud – Paris XI

3. Degli Esposti, Davide. Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis.

Degree: Docteur es, Physiopathologie moléculaire et cellulaire, 2011, Université Paris-Sud – Paris XI

 Le foie est un organe essentiel à la vie chez tous les mammifères. C’est un organe central du métabolisme énergétique et de la détoxification des… (more)

Subjects/Keywords: Autophagie; Steatosis; Autophagy; Hepatocellular carcinoma; Cancer; Liver

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APA (6th Edition):

Degli Esposti, D. (2011). Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA114809

Chicago Manual of Style (16th Edition):

Degli Esposti, Davide. “Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed January 26, 2021. http://www.theses.fr/2011PA114809.

MLA Handbook (7th Edition):

Degli Esposti, Davide. “Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis.” 2011. Web. 26 Jan 2021.

Vancouver:

Degli Esposti D. Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2021 Jan 26]. Available from: http://www.theses.fr/2011PA114809.

Council of Science Editors:

Degli Esposti D. Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA114809


University of Adelaide

4. Tse, Edmund. Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon.

Degree: 2015, University of Adelaide

 Hepatitis C Virus (HCV) is a significant global health issue that leads to the development of chronic liver inflammation, and subsequent establishment of cirrhosis and… (more)

Subjects/Keywords: HCV; hepatitis C; steatosis; interferon; hepatocyte transcriptome

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APA (6th Edition):

Tse, E. (2015). Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/100718

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tse, Edmund. “Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon.” 2015. Thesis, University of Adelaide. Accessed January 26, 2021. http://hdl.handle.net/2440/100718.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tse, Edmund. “Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon.” 2015. Web. 26 Jan 2021.

Vancouver:

Tse E. Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon. [Internet] [Thesis]. University of Adelaide; 2015. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/2440/100718.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tse E. Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon. [Thesis]. University of Adelaide; 2015. Available from: http://hdl.handle.net/2440/100718

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

5. Ivanović, Nevena Đ., 1983-. Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre.

Degree: Farmaceutski fakultet, 2016, Univerzitet u Beogradu

Farmacija - Hemija hrane i dijetetskih proizvoda / Pharmacy - Chemistry of food and dietary products

Prema definiciji Svetske Zdravstvene Organizacije, probiotici su živi mikroorganizmi… (more)

Subjects/Keywords: probiotics; Lactobacillus; high fat diet; NAFLD; steatosis

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APA (6th Edition):

Ivanović, Nevena Đ., 1. (2016). Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:13471/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ivanović, Nevena Đ., 1983-. “Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre.” 2016. Thesis, Univerzitet u Beogradu. Accessed January 26, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:13471/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ivanović, Nevena Đ., 1983-. “Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre.” 2016. Web. 26 Jan 2021.

Vancouver:

Ivanović, Nevena Đ. 1. Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2021 Jan 26]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:13471/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ivanović, Nevena Đ. 1. Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:13471/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

6. Bagley, Bradford. The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats.

Degree: PhD, Toxicology, 2017, University of Minnesota

 Perfluorooctane sulfonate (PFOS) is bioaccumulative and prevalent in the human population. PFOS induces hepatic steatosis in male rats at dietary exposures of 100 ppm via… (more)

Subjects/Keywords: Choline; Liver; Perfluorooctane sulfonate; PFOS; Steatosis

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APA (6th Edition):

Bagley, B. (2017). The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/198403

Chicago Manual of Style (16th Edition):

Bagley, Bradford. “The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats.” 2017. Doctoral Dissertation, University of Minnesota. Accessed January 26, 2021. http://hdl.handle.net/11299/198403.

MLA Handbook (7th Edition):

Bagley, Bradford. “The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats.” 2017. Web. 26 Jan 2021.

Vancouver:

Bagley B. The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats. [Internet] [Doctoral dissertation]. University of Minnesota; 2017. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/11299/198403.

Council of Science Editors:

Bagley B. The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats. [Doctoral Dissertation]. University of Minnesota; 2017. Available from: http://hdl.handle.net/11299/198403


University of Sydney

7. d'Avigdor, William Mark Henry. Differential gene expression in HCV liver injury .

Degree: 2015, University of Sydney

 Chronic hepatitis C virus (HCV) infection causes liver disease that can lead to cirrhosis and hepatocellular carcinoma (HCC). The predominant HCV genotypes in Australia (G1… (more)

Subjects/Keywords: HCV; genotype; steatosis; HCC; gene expression; miRNA

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APA (6th Edition):

d'Avigdor, W. M. H. (2015). Differential gene expression in HCV liver injury . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/13896

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

d'Avigdor, William Mark Henry. “Differential gene expression in HCV liver injury .” 2015. Thesis, University of Sydney. Accessed January 26, 2021. http://hdl.handle.net/2123/13896.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

d'Avigdor, William Mark Henry. “Differential gene expression in HCV liver injury .” 2015. Web. 26 Jan 2021.

Vancouver:

d'Avigdor WMH. Differential gene expression in HCV liver injury . [Internet] [Thesis]. University of Sydney; 2015. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/2123/13896.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

d'Avigdor WMH. Differential gene expression in HCV liver injury . [Thesis]. University of Sydney; 2015. Available from: http://hdl.handle.net/2123/13896

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Borges, Nádia Juliana Beraldo Goulart. Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar.

Degree: Mestrado, Clínica Médica, 2008, University of São Paulo

A Doença Hepática Gordurosa Não Alcoólica (DHGNA) é uma condição clínicopatológica comum, caracterizada por depósito de lipídeos no hepatócito do parênquima hepático. A esteatose hepática… (more)

Subjects/Keywords: choline; colina; esteatose hepática; fructoologosaccharide; frutooligossacarídeo; liver steatosis

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APA (6th Edition):

Borges, N. J. B. G. (2008). Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17138/tde-28032012-085320/ ;

Chicago Manual of Style (16th Edition):

Borges, Nádia Juliana Beraldo Goulart. “Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar.” 2008. Masters Thesis, University of São Paulo. Accessed January 26, 2021. http://www.teses.usp.br/teses/disponiveis/17/17138/tde-28032012-085320/ ;.

MLA Handbook (7th Edition):

Borges, Nádia Juliana Beraldo Goulart. “Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar.” 2008. Web. 26 Jan 2021.

Vancouver:

Borges NJBG. Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2021 Jan 26]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17138/tde-28032012-085320/ ;.

Council of Science Editors:

Borges NJBG. Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/17/17138/tde-28032012-085320/ ;

9. Saviani, Gisele. Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758).

Degree: PhD, Anatomia dos Animais Domésticos e Silvestres, 2009, University of São Paulo

Hoje a criação de avestruz ((Struthio camelus, Linnaeus 1758)) é uma atividade de grande potencial, porém não existem padrões definidos sobre a histologia do seu… (more)

Subjects/Keywords: Avestruzes; Colágeno; Collagen; Esteatose; Fígado; Glicogenio; Glicogênio; Liver; Ostriches; Steatosis

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APA (6th Edition):

Saviani, G. (2009). Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758). (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/10/10132/tde-20072009-095137/ ;

Chicago Manual of Style (16th Edition):

Saviani, Gisele. “Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758).” 2009. Doctoral Dissertation, University of São Paulo. Accessed January 26, 2021. http://www.teses.usp.br/teses/disponiveis/10/10132/tde-20072009-095137/ ;.

MLA Handbook (7th Edition):

Saviani, Gisele. “Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758).” 2009. Web. 26 Jan 2021.

Vancouver:

Saviani G. Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758). [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2021 Jan 26]. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-20072009-095137/ ;.

Council of Science Editors:

Saviani G. Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758). [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-20072009-095137/ ;


University of Louisville

10. Donde, Hridgandh. Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury.

Degree: MS, 2013, University of Louisville

  Highly Active Antiretroviral Therapy (HAART) has led to a significant increase in the life expectancy of HIV patients; however, there are significant side effects… (more)

Subjects/Keywords: HAART; Hepatic injury; Ethanol; Steatosis; Pharmacy and Pharmaceutical Sciences

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APA (6th Edition):

Donde, H. (2013). Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/363 ; https://ir.library.louisville.edu/etd/363

Chicago Manual of Style (16th Edition):

Donde, Hridgandh. “Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury.” 2013. Masters Thesis, University of Louisville. Accessed January 26, 2021. 10.18297/etd/363 ; https://ir.library.louisville.edu/etd/363.

MLA Handbook (7th Edition):

Donde, Hridgandh. “Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury.” 2013. Web. 26 Jan 2021.

Vancouver:

Donde H. Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury. [Internet] [Masters thesis]. University of Louisville; 2013. [cited 2021 Jan 26]. Available from: 10.18297/etd/363 ; https://ir.library.louisville.edu/etd/363.

Council of Science Editors:

Donde H. Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury. [Masters Thesis]. University of Louisville; 2013. Available from: 10.18297/etd/363 ; https://ir.library.louisville.edu/etd/363

11. Rafiei, Hossein 1981-. Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease.

Degree: 2017, University of Saskatchewan

 Non-alcoholic fatty liver disease (NAFLD) is a public health burden. Steatosis as the “first hit”, and oxidative stress, inflammation, mitochondrial dysfunction, and endoplasmic reticulum stress… (more)

Subjects/Keywords: Non-alcoholic fatty liver disease; Mitochondrial dysfunction; endoplasmic reticulum stress; Steatosis

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APA (6th Edition):

Rafiei, H. 1. (2017). Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/7946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rafiei, Hossein 1981-. “Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease.” 2017. Thesis, University of Saskatchewan. Accessed January 26, 2021. http://hdl.handle.net/10388/7946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rafiei, Hossein 1981-. “Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease.” 2017. Web. 26 Jan 2021.

Vancouver:

Rafiei H1. Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease. [Internet] [Thesis]. University of Saskatchewan; 2017. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/10388/7946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rafiei H1. Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease. [Thesis]. University of Saskatchewan; 2017. Available from: http://hdl.handle.net/10388/7946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

12. Huang, Kuan-Hsun. Exogenous Lipids Regulate the Development of Hepatic Steatosis in a Lean NAFLD model-fed a High Carbohydrate Diet.

Degree: 2016, Penn State University

 Non-alcoholic fatty liver disease (NAFLD), an umbrella term that encompasses hepatic steatosis, steatohepatitis, fibrosis, and cirrhosis, has become the most common chronic liver disease in… (more)

Subjects/Keywords: Lean NAFLD; Glucose metabolism; lipidmetabolism; hepatic steatosis; metabolomics; high carbohydrate diet

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APA (6th Edition):

Huang, K. (2016). Exogenous Lipids Regulate the Development of Hepatic Steatosis in a Lean NAFLD model-fed a High Carbohydrate Diet. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13342kxh359

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Kuan-Hsun. “Exogenous Lipids Regulate the Development of Hepatic Steatosis in a Lean NAFLD model-fed a High Carbohydrate Diet.” 2016. Thesis, Penn State University. Accessed January 26, 2021. https://submit-etda.libraries.psu.edu/catalog/13342kxh359.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Kuan-Hsun. “Exogenous Lipids Regulate the Development of Hepatic Steatosis in a Lean NAFLD model-fed a High Carbohydrate Diet.” 2016. Web. 26 Jan 2021.

Vancouver:

Huang K. Exogenous Lipids Regulate the Development of Hepatic Steatosis in a Lean NAFLD model-fed a High Carbohydrate Diet. [Internet] [Thesis]. Penn State University; 2016. [cited 2021 Jan 26]. Available from: https://submit-etda.libraries.psu.edu/catalog/13342kxh359.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang K. Exogenous Lipids Regulate the Development of Hepatic Steatosis in a Lean NAFLD model-fed a High Carbohydrate Diet. [Thesis]. Penn State University; 2016. Available from: https://submit-etda.libraries.psu.edu/catalog/13342kxh359

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Castro benitez, Carlos. Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation.

Degree: Docteur es, Toxicologie, 2019, Université Paris-Saclay (ComUE)

La préservation statique à froid (SCS) est l’étalon-or de la préservation des organes après une greffe. En raison de la pénurie d’organes et de l’augmentation… (more)

Subjects/Keywords: Transplantation Hépatique; Solution de préservation; Stéatose; Liver Transplant; Preservation Solution; Steatosis

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APA (6th Edition):

Castro benitez, C. (2019). Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLS068

Chicago Manual of Style (16th Edition):

Castro benitez, Carlos. “Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed January 26, 2021. http://www.theses.fr/2019SACLS068.

MLA Handbook (7th Edition):

Castro benitez, Carlos. “Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation.” 2019. Web. 26 Jan 2021.

Vancouver:

Castro benitez C. Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2021 Jan 26]. Available from: http://www.theses.fr/2019SACLS068.

Council of Science Editors:

Castro benitez C. Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLS068

14. Groebner, Jennifer. Alcohol-induced tubulin hyperacetylation impairs microtubule dynamics and motor function: contributions to hepatic injury.

Degree: 2018, The Catholic University of America

Although the progression of alcoholic liver disease (ALD) is clinically well described, there are currently no treatments to relieve or reverse the disease state. Our… (more)

Subjects/Keywords: alcoholic liver disease; dynein; lipid droplets; microtubule acetylation; steatosis; tubulin acetyltransferase

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APA (6th Edition):

Groebner, J. (2018). Alcohol-induced tubulin hyperacetylation impairs microtubule dynamics and motor function: contributions to hepatic injury. (Thesis). The Catholic University of America. Retrieved from http://hdl.handle.net/1961/cuislandora:213557

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Groebner, Jennifer. “Alcohol-induced tubulin hyperacetylation impairs microtubule dynamics and motor function: contributions to hepatic injury.” 2018. Thesis, The Catholic University of America. Accessed January 26, 2021. http://hdl.handle.net/1961/cuislandora:213557.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Groebner, Jennifer. “Alcohol-induced tubulin hyperacetylation impairs microtubule dynamics and motor function: contributions to hepatic injury.” 2018. Web. 26 Jan 2021.

Vancouver:

Groebner J. Alcohol-induced tubulin hyperacetylation impairs microtubule dynamics and motor function: contributions to hepatic injury. [Internet] [Thesis]. The Catholic University of America; 2018. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1961/cuislandora:213557.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Groebner J. Alcohol-induced tubulin hyperacetylation impairs microtubule dynamics and motor function: contributions to hepatic injury. [Thesis]. The Catholic University of America; 2018. Available from: http://hdl.handle.net/1961/cuislandora:213557

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Colorado State University

15. Nivala, Angela Marie. Novel therapies for NAFLD.

Degree: PhD, Food Science and Human Nutrition, 2011, Colorado State University

 Background/Aims Non Alcoholic Fatty Liver Disease (NAFLD) is a chronic liver disease often associated with metabolic disorders like type 2 diabetes, cardiovascular disease, obesity, and… (more)

Subjects/Keywords: hepatic steatosis; insulin resistance; lipotoxicity; NAFLD; root exudates; taurine

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APA (6th Edition):

Nivala, A. M. (2011). Novel therapies for NAFLD. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/49853

Chicago Manual of Style (16th Edition):

Nivala, Angela Marie. “Novel therapies for NAFLD.” 2011. Doctoral Dissertation, Colorado State University. Accessed January 26, 2021. http://hdl.handle.net/10217/49853.

MLA Handbook (7th Edition):

Nivala, Angela Marie. “Novel therapies for NAFLD.” 2011. Web. 26 Jan 2021.

Vancouver:

Nivala AM. Novel therapies for NAFLD. [Internet] [Doctoral dissertation]. Colorado State University; 2011. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/10217/49853.

Council of Science Editors:

Nivala AM. Novel therapies for NAFLD. [Doctoral Dissertation]. Colorado State University; 2011. Available from: http://hdl.handle.net/10217/49853


University of Texas Medical Branch – Galveston

16. [No author]. Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice.

Degree: University of Texas Medical Branch – Galveston

 The aryl hydrocarbon receptor (AhR) is a cytosolic, ligand activated transcription factor commonly known for its role in environmental toxicant metabolism. However, the generation of… (more)

Subjects/Keywords: AhR FGF21 Steatosis Obesity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

author], [. (n.d.). Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice. (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/11212

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice. ” Thesis, University of Texas Medical Branch – Galveston. Accessed January 26, 2021. http://hdl.handle.net/2152.3/11212.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice. ” Web. 26 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

author] [. Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice. [Internet] [Thesis]. University of Texas Medical Branch – Galveston; [cited 2021 Jan 26]. Available from: http://hdl.handle.net/2152.3/11212.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

author] [. Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice. [Thesis]. University of Texas Medical Branch – Galveston; Available from: http://hdl.handle.net/2152.3/11212

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Toronto

17. Tersigni, Claudia Ariana. Effect of Autophagy Induction via TRPML1 Activation on Hepatic Steatosis.

Degree: 2019, University of Toronto

Discrepancies between the number of liver transplant candidates and available organs has led to an increased use of marginal grafts, including steatotic liver grafts. Steatotic… (more)

Subjects/Keywords: autophagy; fatty liver; hepatic steatosis; mucolipin 1; transplant; TRPML1; 0306

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APA (6th Edition):

Tersigni, C. A. (2019). Effect of Autophagy Induction via TRPML1 Activation on Hepatic Steatosis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/98408

Chicago Manual of Style (16th Edition):

Tersigni, Claudia Ariana. “Effect of Autophagy Induction via TRPML1 Activation on Hepatic Steatosis.” 2019. Masters Thesis, University of Toronto. Accessed January 26, 2021. http://hdl.handle.net/1807/98408.

MLA Handbook (7th Edition):

Tersigni, Claudia Ariana. “Effect of Autophagy Induction via TRPML1 Activation on Hepatic Steatosis.” 2019. Web. 26 Jan 2021.

Vancouver:

Tersigni CA. Effect of Autophagy Induction via TRPML1 Activation on Hepatic Steatosis. [Internet] [Masters thesis]. University of Toronto; 2019. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1807/98408.

Council of Science Editors:

Tersigni CA. Effect of Autophagy Induction via TRPML1 Activation on Hepatic Steatosis. [Masters Thesis]. University of Toronto; 2019. Available from: http://hdl.handle.net/1807/98408


University of Toronto

18. Brandt, Sydney Laura. The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism.

Degree: 2017, University of Toronto

Elevation of the fish oil metabolite 3-carboxy-4-methyl-5-propyl-2-furanpropanoic acid (CMPF) induces metabolic remodeling within the -cell to a preferential use of fatty acids. Although detrimental to… (more)

Subjects/Keywords: ACC Inhibition; CMPF; FGF21; Lipid metabolism; NAFLD; Steatosis; 0719

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APA (6th Edition):

Brandt, S. L. (2017). The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/88616

Chicago Manual of Style (16th Edition):

Brandt, Sydney Laura. “The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism.” 2017. Masters Thesis, University of Toronto. Accessed January 26, 2021. http://hdl.handle.net/1807/88616.

MLA Handbook (7th Edition):

Brandt, Sydney Laura. “The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism.” 2017. Web. 26 Jan 2021.

Vancouver:

Brandt SL. The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism. [Internet] [Masters thesis]. University of Toronto; 2017. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1807/88616.

Council of Science Editors:

Brandt SL. The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism. [Masters Thesis]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/88616


Boston University

19. Yengul, Sanjay S. Shear wave rheometry with applications in elastography.

Degree: PhD, Mechanical Engineering, 2019, Boston University

 The goal of elastography is to map the mechanical properties of soft tissues associated with health and disease. The mechanical property of interest in this… (more)

Subjects/Keywords: Acoustics; Elastic wave; Liver fibrosis; Medical imaging; NAFLD; Steatosis; Viscoelasticity

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APA (6th Edition):

Yengul, S. S. (2019). Shear wave rheometry with applications in elastography. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/34923

Chicago Manual of Style (16th Edition):

Yengul, Sanjay S. “Shear wave rheometry with applications in elastography.” 2019. Doctoral Dissertation, Boston University. Accessed January 26, 2021. http://hdl.handle.net/2144/34923.

MLA Handbook (7th Edition):

Yengul, Sanjay S. “Shear wave rheometry with applications in elastography.” 2019. Web. 26 Jan 2021.

Vancouver:

Yengul SS. Shear wave rheometry with applications in elastography. [Internet] [Doctoral dissertation]. Boston University; 2019. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/2144/34923.

Council of Science Editors:

Yengul SS. Shear wave rheometry with applications in elastography. [Doctoral Dissertation]. Boston University; 2019. Available from: http://hdl.handle.net/2144/34923


University of Dundee

20. Garbacz, Wojciech G. Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice.

Degree: PhD, 2012, University of Dundee

 Peroxisome proliferator-activated receptor alpha (PPARa) and delta (PPARd) belong to the nuclear receptor superfamily. PPARa is a target of lipid-lowering drugs and PPARd promotes fatty… (more)

Subjects/Keywords: 616.3; Peroxisome Proliferator Activator Receptor (PPAR); Liver; Steatosis

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APA (6th Edition):

Garbacz, W. G. (2012). Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/9b2509f1-3391-45fa-91eb-9b5364a9b6d9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578897

Chicago Manual of Style (16th Edition):

Garbacz, Wojciech G. “Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice.” 2012. Doctoral Dissertation, University of Dundee. Accessed January 26, 2021. https://discovery.dundee.ac.uk/en/studentTheses/9b2509f1-3391-45fa-91eb-9b5364a9b6d9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578897.

MLA Handbook (7th Edition):

Garbacz, Wojciech G. “Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice.” 2012. Web. 26 Jan 2021.

Vancouver:

Garbacz WG. Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice. [Internet] [Doctoral dissertation]. University of Dundee; 2012. [cited 2021 Jan 26]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/9b2509f1-3391-45fa-91eb-9b5364a9b6d9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578897.

Council of Science Editors:

Garbacz WG. Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice. [Doctoral Dissertation]. University of Dundee; 2012. Available from: https://discovery.dundee.ac.uk/en/studentTheses/9b2509f1-3391-45fa-91eb-9b5364a9b6d9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578897

21. GENG WEI. Roles of the microRNAs in fatty liver lipid accumulation.

Degree: 2012, National University of Singapore

Subjects/Keywords: microRNA; NAFLD; lipid; steatosis

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APA (6th Edition):

WEI, G. (2012). Roles of the microRNAs in fatty liver lipid accumulation. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/33282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

WEI, GENG. “Roles of the microRNAs in fatty liver lipid accumulation.” 2012. Thesis, National University of Singapore. Accessed January 26, 2021. http://scholarbank.nus.edu.sg/handle/10635/33282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

WEI, GENG. “Roles of the microRNAs in fatty liver lipid accumulation.” 2012. Web. 26 Jan 2021.

Vancouver:

WEI G. Roles of the microRNAs in fatty liver lipid accumulation. [Internet] [Thesis]. National University of Singapore; 2012. [cited 2021 Jan 26]. Available from: http://scholarbank.nus.edu.sg/handle/10635/33282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

WEI G. Roles of the microRNAs in fatty liver lipid accumulation. [Thesis]. National University of Singapore; 2012. Available from: http://scholarbank.nus.edu.sg/handle/10635/33282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Arizona State University

22. Crawford, Meli'sa Shaunte. Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats.

Degree: Biology, 2019, Arizona State University

 The prevalence of obesity and obesity-related disorders have increased world-wide. In the last decade, the intestinal microbiome has become a major indicator of metabolic and… (more)

Subjects/Keywords: Physiology; High Fat Diet; Inflammation; Metabolic Syndrome; Microbiome; Obesity; Steatosis

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APA (6th Edition):

Crawford, M. S. (2019). Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats. (Doctoral Dissertation). Arizona State University. Retrieved from http://repository.asu.edu/items/53625

Chicago Manual of Style (16th Edition):

Crawford, Meli'sa Shaunte. “Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats.” 2019. Doctoral Dissertation, Arizona State University. Accessed January 26, 2021. http://repository.asu.edu/items/53625.

MLA Handbook (7th Edition):

Crawford, Meli'sa Shaunte. “Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats.” 2019. Web. 26 Jan 2021.

Vancouver:

Crawford MS. Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats. [Internet] [Doctoral dissertation]. Arizona State University; 2019. [cited 2021 Jan 26]. Available from: http://repository.asu.edu/items/53625.

Council of Science Editors:

Crawford MS. Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats. [Doctoral Dissertation]. Arizona State University; 2019. Available from: http://repository.asu.edu/items/53625

23. Villemain Le Hagre, Laure. Le récepteur Sigma 1 : implication dans la prolifération et la stéatose des hépatocytes : Sigma 1 Receptor : Role in Hepatocyte Proliferation and Steatosis.

Degree: Docteur es, Sciences de la vie et de la santé, 2019, Université Paris-Saclay (ComUE)

Le récepteur Sigma 1 (SigR1) est une protéine transmembranaire du RE, enrichie dans les MAMs, qui agirait comme une chaperonne. Ubiquitaire, SigR1 est très exprimé… (more)

Subjects/Keywords: Adénome hépatocellulaire; Prolifération; Stéatose; Hepatocellular adenoma; Proliferation; Steatosis

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APA (6th Edition):

Villemain Le Hagre, L. (2019). Le récepteur Sigma 1 : implication dans la prolifération et la stéatose des hépatocytes : Sigma 1 Receptor : Role in Hepatocyte Proliferation and Steatosis. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLS370

Chicago Manual of Style (16th Edition):

Villemain Le Hagre, Laure. “Le récepteur Sigma 1 : implication dans la prolifération et la stéatose des hépatocytes : Sigma 1 Receptor : Role in Hepatocyte Proliferation and Steatosis.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed January 26, 2021. http://www.theses.fr/2019SACLS370.

MLA Handbook (7th Edition):

Villemain Le Hagre, Laure. “Le récepteur Sigma 1 : implication dans la prolifération et la stéatose des hépatocytes : Sigma 1 Receptor : Role in Hepatocyte Proliferation and Steatosis.” 2019. Web. 26 Jan 2021.

Vancouver:

Villemain Le Hagre L. Le récepteur Sigma 1 : implication dans la prolifération et la stéatose des hépatocytes : Sigma 1 Receptor : Role in Hepatocyte Proliferation and Steatosis. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2021 Jan 26]. Available from: http://www.theses.fr/2019SACLS370.

Council of Science Editors:

Villemain Le Hagre L. Le récepteur Sigma 1 : implication dans la prolifération et la stéatose des hépatocytes : Sigma 1 Receptor : Role in Hepatocyte Proliferation and Steatosis. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLS370


University of Toronto

24. Prentice, Kacey June. Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes.

Degree: PhD, 2015, University of Toronto

Both gestational diabetes (GDM) and type 2 diabetes (T2D) are caused by a failure of the pancreatic beta cell to compensate for increased insulin demand.… (more)

Subjects/Keywords: Beta Cell; Diabetes; Gestational Diabetes; Insulin; Metabolomics; Steatosis; 0719

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APA (6th Edition):

Prentice, K. J. (2015). Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/75688

Chicago Manual of Style (16th Edition):

Prentice, Kacey June. “Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes.” 2015. Doctoral Dissertation, University of Toronto. Accessed January 26, 2021. http://hdl.handle.net/1807/75688.

MLA Handbook (7th Edition):

Prentice, Kacey June. “Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes.” 2015. Web. 26 Jan 2021.

Vancouver:

Prentice KJ. Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/1807/75688.

Council of Science Editors:

Prentice KJ. Identification and Characterization of the Role of the Circulating Metabolite 3-Carboxy-4-Methyl-5-Propyl-2-Furanpropanoic Acid (CMPF) in Diabetes. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/75688


University of South Carolina

25. Havighorst, Amanda Rose. Susceptibility to Metabolic Disease and Individual Variations in Unfolded Protein Response in Deer Mice (Peromyscus Maniculatus).

Degree: PhD, College of Pharmacy, 2019, University of South Carolina

  The endoplasmic reticulum is the site where integral membrane and secreted proteins are synthesized and folded, and is also the site of synthesis of… (more)

Subjects/Keywords: Pharmacy and Pharmaceutical Sciences; ER stress; liver; NAFLD; steatosis; UPR; variation

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APA (6th Edition):

Havighorst, A. R. (2019). Susceptibility to Metabolic Disease and Individual Variations in Unfolded Protein Response in Deer Mice (Peromyscus Maniculatus). (Doctoral Dissertation). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/5610

Chicago Manual of Style (16th Edition):

Havighorst, Amanda Rose. “Susceptibility to Metabolic Disease and Individual Variations in Unfolded Protein Response in Deer Mice (Peromyscus Maniculatus).” 2019. Doctoral Dissertation, University of South Carolina. Accessed January 26, 2021. https://scholarcommons.sc.edu/etd/5610.

MLA Handbook (7th Edition):

Havighorst, Amanda Rose. “Susceptibility to Metabolic Disease and Individual Variations in Unfolded Protein Response in Deer Mice (Peromyscus Maniculatus).” 2019. Web. 26 Jan 2021.

Vancouver:

Havighorst AR. Susceptibility to Metabolic Disease and Individual Variations in Unfolded Protein Response in Deer Mice (Peromyscus Maniculatus). [Internet] [Doctoral dissertation]. University of South Carolina; 2019. [cited 2021 Jan 26]. Available from: https://scholarcommons.sc.edu/etd/5610.

Council of Science Editors:

Havighorst AR. Susceptibility to Metabolic Disease and Individual Variations in Unfolded Protein Response in Deer Mice (Peromyscus Maniculatus). [Doctoral Dissertation]. University of South Carolina; 2019. Available from: https://scholarcommons.sc.edu/etd/5610

26. chouzouri, vasiliki. Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση.

Degree: 2016, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Over the past five decades the association of essential hypertension with non-alcoholic fatty liver disease is a field of study. It is assumed that essential… (more)

Subjects/Keywords: Αρτηριακή υπέρταση; Στεάτωση ήπατος; Arterial hypertension; Liver steatosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

chouzouri, v. (2016). Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/36863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

chouzouri, vasiliki. “Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση.” 2016. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed January 26, 2021. http://hdl.handle.net/10442/hedi/36863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

chouzouri, vasiliki. “Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση.” 2016. Web. 26 Jan 2021.

Vancouver:

chouzouri v. Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/10442/hedi/36863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

chouzouri v. Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. Available from: http://hdl.handle.net/10442/hedi/36863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

27. Clark, Paul James. Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications.

Degree: Kirby Institute, 2012, University of New South Wales

 Background: The hepatitis C virus (HCV) relies on host lipid pathways for its life cycle, leading to metabolicconsequences including hypocholesterolemia, insulin resistance and hepatic steatosis.… (more)

Subjects/Keywords: Trascriptomics; Hepatitis C; Genomics; Metabolomics; Steatosis; IL28B; PNPLA3; GWAS

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APA (6th Edition):

Clark, P. J. (2012). Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52447 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11120/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Clark, Paul James. “Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications.” 2012. Doctoral Dissertation, University of New South Wales. Accessed January 26, 2021. http://handle.unsw.edu.au/1959.4/52447 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11120/SOURCE01?view=true.

MLA Handbook (7th Edition):

Clark, Paul James. “Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications.” 2012. Web. 26 Jan 2021.

Vancouver:

Clark PJ. Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2021 Jan 26]. Available from: http://handle.unsw.edu.au/1959.4/52447 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11120/SOURCE01?view=true.

Council of Science Editors:

Clark PJ. Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/52447 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11120/SOURCE01?view=true


University of New South Wales

28. Wainwright, Camilla. Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques.

Degree: Clinical School - St Vincent's Hospital, 2016, University of New South Wales

 The novel technique of 1H MRS at a high-field strength (3 Tesla) was demonstrated to be a reproducible method to assess hepatic lipid content.This technique… (more)

Subjects/Keywords: Advanced imaging techniques; Myocardial and hepatic steatosis; Vivo

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wainwright, C. (2016). Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/60083 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51140/SOURCE2?view=true

Chicago Manual of Style (16th Edition):

Wainwright, Camilla. “Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques.” 2016. Doctoral Dissertation, University of New South Wales. Accessed January 26, 2021. http://handle.unsw.edu.au/1959.4/60083 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51140/SOURCE2?view=true.

MLA Handbook (7th Edition):

Wainwright, Camilla. “Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques.” 2016. Web. 26 Jan 2021.

Vancouver:

Wainwright C. Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques. [Internet] [Doctoral dissertation]. University of New South Wales; 2016. [cited 2021 Jan 26]. Available from: http://handle.unsw.edu.au/1959.4/60083 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51140/SOURCE2?view=true.

Council of Science Editors:

Wainwright C. Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques. [Doctoral Dissertation]. University of New South Wales; 2016. Available from: http://handle.unsw.edu.au/1959.4/60083 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51140/SOURCE2?view=true

29. Pais, Raluca. L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies.

Degree: Docteur es, Physiologie et Physiopathologie, 2015, Université Pierre et Marie Curie – Paris VI

L’objectif général de ce travail était de mieux définir à travers des études cliniques le rôle de l’inflammation hépatique dans l’histoire naturelle de la NAFLD.… (more)

Subjects/Keywords: Stéatose; Steatohépatite; Fibrose; Inflammation; Carcinome hépatocellulaire; Athérosclérose; Steatosis; Atherosclerosis; 616.3

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pais, R. (2015). L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066223

Chicago Manual of Style (16th Edition):

Pais, Raluca. “L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed January 26, 2021. http://www.theses.fr/2015PA066223.

MLA Handbook (7th Edition):

Pais, Raluca. “L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies.” 2015. Web. 26 Jan 2021.

Vancouver:

Pais R. L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2021 Jan 26]. Available from: http://www.theses.fr/2015PA066223.

Council of Science Editors:

Pais R. L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066223

30. Monteiro, Paula Alves [UNESP]. Efeito de diferentes modelos de exercício físico sobre a composição corporal, marcadores metabólicos e inflamatórios em adolescentes obesos.

Degree: 2017, Universidade Estadual Paulista

A prevalência da obesidade na população pediátrica aumenta a um ritmo acelerado em muitos países, e tem se tornado um importante problema de saúde pública.… (more)

Subjects/Keywords: Gordura corporal; Treinamento físico; Obesidade; Esteatose hepática; Body fat; Physical training; Obesity; Hepatic steatosis

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APA (6th Edition):

Monteiro, P. A. [. (2017). Efeito de diferentes modelos de exercício físico sobre a composição corporal, marcadores metabólicos e inflamatórios em adolescentes obesos. (Thesis). Universidade Estadual Paulista. Retrieved from http://hdl.handle.net/11449/150365

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Monteiro, Paula Alves [UNESP]. “Efeito de diferentes modelos de exercício físico sobre a composição corporal, marcadores metabólicos e inflamatórios em adolescentes obesos.” 2017. Thesis, Universidade Estadual Paulista. Accessed January 26, 2021. http://hdl.handle.net/11449/150365.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Monteiro, Paula Alves [UNESP]. “Efeito de diferentes modelos de exercício físico sobre a composição corporal, marcadores metabólicos e inflamatórios em adolescentes obesos.” 2017. Web. 26 Jan 2021.

Vancouver:

Monteiro PA[. Efeito de diferentes modelos de exercício físico sobre a composição corporal, marcadores metabólicos e inflamatórios em adolescentes obesos. [Internet] [Thesis]. Universidade Estadual Paulista; 2017. [cited 2021 Jan 26]. Available from: http://hdl.handle.net/11449/150365.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Monteiro PA[. Efeito de diferentes modelos de exercício físico sobre a composição corporal, marcadores metabólicos e inflamatórios em adolescentes obesos. [Thesis]. Universidade Estadual Paulista; 2017. Available from: http://hdl.handle.net/11449/150365

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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