Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Steatosis). Showing records 1 – 30 of 137 total matches.

[1] [2] [3] [4] [5]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


University of Florida

1. Kim, Min Hyun. Regulation and Function of Zinc and Zinc Transporters during ER Stress.

Degree: PhD, Nutritional Sciences, 2017, University of Florida

 Extensive endoplasmic reticulum (ER) stress damages the liver causing apoptosis and steatosis, despite the activation of the unfolded protein response (UPR). Restriction of zinc from… (more)

Subjects/Keywords: apoptosis  – steatosis  – upr  – zinc  – zip14

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kim, M. H. (2017). Regulation and Function of Zinc and Zinc Transporters during ER Stress. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0051263

Chicago Manual of Style (16th Edition):

Kim, Min Hyun. “Regulation and Function of Zinc and Zinc Transporters during ER Stress.” 2017. Doctoral Dissertation, University of Florida. Accessed July 17, 2019. http://ufdc.ufl.edu/UFE0051263.

MLA Handbook (7th Edition):

Kim, Min Hyun. “Regulation and Function of Zinc and Zinc Transporters during ER Stress.” 2017. Web. 17 Jul 2019.

Vancouver:

Kim MH. Regulation and Function of Zinc and Zinc Transporters during ER Stress. [Internet] [Doctoral dissertation]. University of Florida; 2017. [cited 2019 Jul 17]. Available from: http://ufdc.ufl.edu/UFE0051263.

Council of Science Editors:

Kim MH. Regulation and Function of Zinc and Zinc Transporters during ER Stress. [Doctoral Dissertation]. University of Florida; 2017. Available from: http://ufdc.ufl.edu/UFE0051263


University of Dundee

2. Garbacz, Wojciech G. Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice.

Degree: PhD, 2012, University of Dundee

 Peroxisome proliferator-activated receptor alpha (PPARa) and delta (PPARd) belong to the nuclear receptor superfamily. PPARa is a target of lipid-lowering drugs and PPARd promotes fatty… (more)

Subjects/Keywords: Peroxisome Proliferator Activator Receptor (PPAR); Liver; Steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Garbacz, W. G. (2012). Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice. (Doctoral Dissertation). University of Dundee. Retrieved from http://hdl.handle.net/10588/9b2509f1-3391-45fa-91eb-9b5364a9b6d9

Chicago Manual of Style (16th Edition):

Garbacz, Wojciech G. “Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice.” 2012. Doctoral Dissertation, University of Dundee. Accessed July 17, 2019. http://hdl.handle.net/10588/9b2509f1-3391-45fa-91eb-9b5364a9b6d9.

MLA Handbook (7th Edition):

Garbacz, Wojciech G. “Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice.” 2012. Web. 17 Jul 2019.

Vancouver:

Garbacz WG. Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice. [Internet] [Doctoral dissertation]. University of Dundee; 2012. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/10588/9b2509f1-3391-45fa-91eb-9b5364a9b6d9.

Council of Science Editors:

Garbacz WG. Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice. [Doctoral Dissertation]. University of Dundee; 2012. Available from: http://hdl.handle.net/10588/9b2509f1-3391-45fa-91eb-9b5364a9b6d9


Univerzitet u Beogradu

3. Ivanović, Nevena Đ., 1983-. Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre.

Degree: Farmaceutski fakultet, 2016, Univerzitet u Beogradu

Farmacija - Hemija hrane i dijetetskih proizvoda / Pharmacy - Chemistry of food and dietary products

Prema definiciji Svetske Zdravstvene Organizacije, probiotici su živi mikroorganizmi… (more)

Subjects/Keywords: probiotics; Lactobacillus; high fat diet; NAFLD; steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ivanović, Nevena Đ., 1. (2016). Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:13471/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ivanović, Nevena Đ., 1983-. “Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre.” 2016. Thesis, Univerzitet u Beogradu. Accessed July 17, 2019. https://fedorabg.bg.ac.rs/fedora/get/o:13471/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ivanović, Nevena Đ., 1983-. “Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre.” 2016. Web. 17 Jul 2019.

Vancouver:

Ivanović, Nevena Đ. 1. Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2019 Jul 17]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:13471/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ivanović, Nevena Đ. 1. Uticaj oralne primene Lactobacillus rhamnosus LA68 i Lactobacillus plantarum WCFS1 na imunološke i metaboličke parametre miševa u uslovima eksperimentalno indukovane nealkoholne masne jetre. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:13471/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

4. Kaiser, J. Phillip, 1981-. The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease.

Degree: PhD, 2009, University of Louisville

 Alcoholic liver disease (ALD) is a serious concern for the world's population. It is one of the leading causes of death and is also a… (more)

Subjects/Keywords: Liver disease; Alcoholic liver disease; Steatosis; Fibrosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kaiser, J. Phillip, 1. (2009). The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720

Chicago Manual of Style (16th Edition):

Kaiser, J. Phillip, 1981-. “The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease.” 2009. Doctoral Dissertation, University of Louisville. Accessed July 17, 2019. 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720.

MLA Handbook (7th Edition):

Kaiser, J. Phillip, 1981-. “The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease.” 2009. Web. 17 Jul 2019.

Vancouver:

Kaiser, J. Phillip 1. The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease. [Internet] [Doctoral dissertation]. University of Louisville; 2009. [cited 2019 Jul 17]. Available from: 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720.

Council of Science Editors:

Kaiser, J. Phillip 1. The role of PKC-epsilon in models of alcohol- and toxin-induced liver disease. [Doctoral Dissertation]. University of Louisville; 2009. Available from: 10.18297/etd/720 ; https://ir.library.louisville.edu/etd/720


University of Sydney

5. d'Avigdor, William Mark Henry. Differential gene expression in HCV liver injury .

Degree: 2015, University of Sydney

 Chronic hepatitis C virus (HCV) infection causes liver disease that can lead to cirrhosis and hepatocellular carcinoma (HCC). The predominant HCV genotypes in Australia (G1… (more)

Subjects/Keywords: HCV; genotype; steatosis; HCC; gene expression; miRNA

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

d'Avigdor, W. M. H. (2015). Differential gene expression in HCV liver injury . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/13896

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

d'Avigdor, William Mark Henry. “Differential gene expression in HCV liver injury .” 2015. Thesis, University of Sydney. Accessed July 17, 2019. http://hdl.handle.net/2123/13896.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

d'Avigdor, William Mark Henry. “Differential gene expression in HCV liver injury .” 2015. Web. 17 Jul 2019.

Vancouver:

d'Avigdor WMH. Differential gene expression in HCV liver injury . [Internet] [Thesis]. University of Sydney; 2015. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/2123/13896.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

d'Avigdor WMH. Differential gene expression in HCV liver injury . [Thesis]. University of Sydney; 2015. Available from: http://hdl.handle.net/2123/13896

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

6. Tse, Edmund. Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon.

Degree: 2015, University of Adelaide

 Hepatitis C Virus (HCV) is a significant global health issue that leads to the development of chronic liver inflammation, and subsequent establishment of cirrhosis and… (more)

Subjects/Keywords: HCV; hepatitis C; steatosis; interferon; hepatocyte transcriptome

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tse, E. (2015). Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/100718

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tse, Edmund. “Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon.” 2015. Thesis, University of Adelaide. Accessed July 17, 2019. http://hdl.handle.net/2440/100718.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tse, Edmund. “Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon.” 2015. Web. 17 Jul 2019.

Vancouver:

Tse E. Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon. [Internet] [Thesis]. University of Adelaide; 2015. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/2440/100718.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tse E. Improving treatment predictors of HCV therapy and the impact of steatosis on the hepatocyte transcriptome and anti-HCV action of interferon. [Thesis]. University of Adelaide; 2015. Available from: http://hdl.handle.net/2440/100718

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

7. Bagley, Bradford. The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats.

Degree: PhD, Toxicology, 2017, University of Minnesota

 Perfluorooctane sulfonate (PFOS) is bioaccumulative and prevalent in the human population. PFOS induces hepatic steatosis in male rats at dietary exposures of 100 ppm via… (more)

Subjects/Keywords: Choline; Liver; Perfluorooctane sulfonate; PFOS; Steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bagley, B. (2017). The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/198403

Chicago Manual of Style (16th Edition):

Bagley, Bradford. “The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats.” 2017. Doctoral Dissertation, University of Minnesota. Accessed July 17, 2019. http://hdl.handle.net/11299/198403.

MLA Handbook (7th Edition):

Bagley, Bradford. “The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats.” 2017. Web. 17 Jul 2019.

Vancouver:

Bagley B. The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats. [Internet] [Doctoral dissertation]. University of Minnesota; 2017. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/11299/198403.

Council of Science Editors:

Bagley B. The Effect of Perfluorooctane Sulfonate (PFOS) and Choline Supplementation on Hepatic Steatosis in Sprague Dawley Rats. [Doctoral Dissertation]. University of Minnesota; 2017. Available from: http://hdl.handle.net/11299/198403

8. Ungerleider, Nathan A. Deletions of Fstl3 and/or Fst Isoforms 303 and 315 Results in Hepatic Steatosis.

Degree: MS, Molecular & Cellular Biology, 2010, University of Massachusetts

 TGFβ ligands, activin and myostatin have been shown to stimulate insulin production and secretion. Antagonists, Follistatin (FST) and Follistatin like 3 (FSTL3) were partially and… (more)

Subjects/Keywords: hepatic steatosis; insulin resistance; srebp1; lipid uptake

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ungerleider, N. A. (2010). Deletions of Fstl3 and/or Fst Isoforms 303 and 315 Results in Hepatic Steatosis. (Masters Thesis). University of Massachusetts. Retrieved from https://scholarworks.umass.edu/theses/478

Chicago Manual of Style (16th Edition):

Ungerleider, Nathan A. “Deletions of Fstl3 and/or Fst Isoforms 303 and 315 Results in Hepatic Steatosis.” 2010. Masters Thesis, University of Massachusetts. Accessed July 17, 2019. https://scholarworks.umass.edu/theses/478.

MLA Handbook (7th Edition):

Ungerleider, Nathan A. “Deletions of Fstl3 and/or Fst Isoforms 303 and 315 Results in Hepatic Steatosis.” 2010. Web. 17 Jul 2019.

Vancouver:

Ungerleider NA. Deletions of Fstl3 and/or Fst Isoforms 303 and 315 Results in Hepatic Steatosis. [Internet] [Masters thesis]. University of Massachusetts; 2010. [cited 2019 Jul 17]. Available from: https://scholarworks.umass.edu/theses/478.

Council of Science Editors:

Ungerleider NA. Deletions of Fstl3 and/or Fst Isoforms 303 and 315 Results in Hepatic Steatosis. [Masters Thesis]. University of Massachusetts; 2010. Available from: https://scholarworks.umass.edu/theses/478


Université Paris-Sud – Paris XI

9. Degli Esposti, Davide. Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis.

Degree: Docteur es, Physiopathologie moléculaire et cellulaire, 2011, Université Paris-Sud – Paris XI

 Le foie est un organe essentiel à la vie chez tous les mammifères. C’est un organe central du métabolisme énergétique et de la détoxification des… (more)

Subjects/Keywords: Autophagie; Steatosis; Autophagy; Hepatocellular carcinoma; Cancer; Liver

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Degli Esposti, D. (2011). Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA114809

Chicago Manual of Style (16th Edition):

Degli Esposti, Davide. “Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed July 17, 2019. http://www.theses.fr/2011PA114809.

MLA Handbook (7th Edition):

Degli Esposti, Davide. “Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis.” 2011. Web. 17 Jul 2019.

Vancouver:

Degli Esposti D. Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2019 Jul 17]. Available from: http://www.theses.fr/2011PA114809.

Council of Science Editors:

Degli Esposti D. Les mécanismes de réponse à l'inflammation chronique dans le foie stéatosique et les conséquences sur l'homéostasie cellulaire et la cancérogénèse : Response mechanisms to chronic inflammation in steatotic liver and consequences on cellular homeostasy and carcinogenesis. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA114809

10. Borges, Nádia Juliana Beraldo Goulart. Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar.

Degree: Mestrado, Clínica Médica, 2008, University of São Paulo

A Doença Hepática Gordurosa Não Alcoólica (DHGNA) é uma condição clínicopatológica comum, caracterizada por depósito de lipídeos no hepatócito do parênquima hepático. A esteatose hepática… (more)

Subjects/Keywords: choline; colina; esteatose hepática; fructoologosaccharide; frutooligossacarídeo; liver steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Borges, N. J. B. G. (2008). Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17138/tde-28032012-085320/ ;

Chicago Manual of Style (16th Edition):

Borges, Nádia Juliana Beraldo Goulart. “Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar.” 2008. Masters Thesis, University of São Paulo. Accessed July 17, 2019. http://www.teses.usp.br/teses/disponiveis/17/17138/tde-28032012-085320/ ;.

MLA Handbook (7th Edition):

Borges, Nádia Juliana Beraldo Goulart. “Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar.” 2008. Web. 17 Jul 2019.

Vancouver:

Borges NJBG. Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2019 Jul 17]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17138/tde-28032012-085320/ ;.

Council of Science Editors:

Borges NJBG. Efeitos da suplementação da colina e de frutooligossacarídeos na esteatose hepática em ratos wistar. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/17/17138/tde-28032012-085320/ ;

11. Saviani, Gisele. Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758).

Degree: PhD, Anatomia dos Animais Domésticos e Silvestres, 2009, University of São Paulo

Hoje a criação de avestruz ((Struthio camelus, Linnaeus 1758)) é uma atividade de grande potencial, porém não existem padrões definidos sobre a histologia do seu… (more)

Subjects/Keywords: Avestruzes; Colágeno; Collagen; Esteatose; Fígado; Glicogenio; Glicogênio; Liver; Ostriches; Steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Saviani, G. (2009). Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758). (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/10/10132/tde-20072009-095137/ ;

Chicago Manual of Style (16th Edition):

Saviani, Gisele. “Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758).” 2009. Doctoral Dissertation, University of São Paulo. Accessed July 17, 2019. http://www.teses.usp.br/teses/disponiveis/10/10132/tde-20072009-095137/ ;.

MLA Handbook (7th Edition):

Saviani, Gisele. “Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758).” 2009. Web. 17 Jul 2019.

Vancouver:

Saviani G. Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758). [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2019 Jul 17]. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-20072009-095137/ ;.

Council of Science Editors:

Saviani G. Anatomia das vias sanguíneas e biliares e histologia do fígado de Avestruz (Struthio camelus, Linnaeus, 1758). [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-20072009-095137/ ;

12. chouzouri, vasiliki. Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση.

Degree: 2016, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Over the past five decades the association of essential hypertension with non-alcoholic fatty liver disease is a field of study. It is assumed that essential… (more)

Subjects/Keywords: Αρτηριακή υπέρταση; Στεάτωση ήπατος; Arterial hypertension; Liver steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

chouzouri, v. (2016). Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/36863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

chouzouri, vasiliki. “Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση.” 2016. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed July 17, 2019. http://hdl.handle.net/10442/hedi/36863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

chouzouri, vasiliki. “Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση.” 2016. Web. 17 Jul 2019.

Vancouver:

chouzouri v. Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/10442/hedi/36863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

chouzouri v. Συσχέτιση αρτηριακής πίεσης με μη αλκοολική στεάτωση. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. Available from: http://hdl.handle.net/10442/hedi/36863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

13. Purushotham, Aparna. Role of bioactive compounds in the regulation of insulin sensitivity.

Degree: PhD, Ohio State University Nutrition, 2007, The Ohio State University

 Dietary agents have important implications in the treatment of type 2 diabetes. My first objective was to determine the effectiveness of the dietary fat, conjugated… (more)

Subjects/Keywords: Health Sciences, Nutrition; Insulin resistance; Hepatic steatosis; Conjugated linoleic acid; Naringenin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Purushotham, A. (2007). Role of bioactive compounds in the regulation of insulin sensitivity. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1173108971

Chicago Manual of Style (16th Edition):

Purushotham, Aparna. “Role of bioactive compounds in the regulation of insulin sensitivity.” 2007. Doctoral Dissertation, The Ohio State University. Accessed July 17, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1173108971.

MLA Handbook (7th Edition):

Purushotham, Aparna. “Role of bioactive compounds in the regulation of insulin sensitivity.” 2007. Web. 17 Jul 2019.

Vancouver:

Purushotham A. Role of bioactive compounds in the regulation of insulin sensitivity. [Internet] [Doctoral dissertation]. The Ohio State University; 2007. [cited 2019 Jul 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1173108971.

Council of Science Editors:

Purushotham A. Role of bioactive compounds in the regulation of insulin sensitivity. [Doctoral Dissertation]. The Ohio State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1173108971


University of Cincinnati

14. Chen, Ying. Relationship of Glutathione Deficiency to Oxidative Stress-Related Disease and Aging.

Degree: PhD, Medicine : Toxicology (Environmental Health), 2007, University of Cincinnati

 Glutathione (GSH) is the most abundant cellular thiol antioxidant, disturbance in GSH homeostasis has been associated with several oxidative stress-related diseases and aging. The rate-limiting… (more)

Subjects/Keywords: Health Sciences, Toxicology; glutathione; oxidative stress; knockout; senescence; steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, Y. (2007). Relationship of Glutathione Deficiency to Oxidative Stress-Related Disease and Aging. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1172086646

Chicago Manual of Style (16th Edition):

Chen, Ying. “Relationship of Glutathione Deficiency to Oxidative Stress-Related Disease and Aging.” 2007. Doctoral Dissertation, University of Cincinnati. Accessed July 17, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1172086646.

MLA Handbook (7th Edition):

Chen, Ying. “Relationship of Glutathione Deficiency to Oxidative Stress-Related Disease and Aging.” 2007. Web. 17 Jul 2019.

Vancouver:

Chen Y. Relationship of Glutathione Deficiency to Oxidative Stress-Related Disease and Aging. [Internet] [Doctoral dissertation]. University of Cincinnati; 2007. [cited 2019 Jul 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1172086646.

Council of Science Editors:

Chen Y. Relationship of Glutathione Deficiency to Oxidative Stress-Related Disease and Aging. [Doctoral Dissertation]. University of Cincinnati; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1172086646


Case Western Reserve University

15. Chen, Xiaocong. The Regulation of Adiponectin by Ethanol Feeding and Taurine Supplementation.

Degree: PhD, Nutrition, 2009, Case Western Reserve University

  Chronic ethanol feeding to mice and rats decreases serum adiponectin concentration and adiponectin treatment during chronic ethanol feeding attenuates ethanol-induced liver injury in mice.… (more)

Subjects/Keywords: Nutrition; Adiponectin; ethanol feeding; taurine; oxidative stress; steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, X. (2009). The Regulation of Adiponectin by Ethanol Feeding and Taurine Supplementation. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1221077215

Chicago Manual of Style (16th Edition):

Chen, Xiaocong. “The Regulation of Adiponectin by Ethanol Feeding and Taurine Supplementation.” 2009. Doctoral Dissertation, Case Western Reserve University. Accessed July 17, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1221077215.

MLA Handbook (7th Edition):

Chen, Xiaocong. “The Regulation of Adiponectin by Ethanol Feeding and Taurine Supplementation.” 2009. Web. 17 Jul 2019.

Vancouver:

Chen X. The Regulation of Adiponectin by Ethanol Feeding and Taurine Supplementation. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2009. [cited 2019 Jul 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1221077215.

Council of Science Editors:

Chen X. The Regulation of Adiponectin by Ethanol Feeding and Taurine Supplementation. [Doctoral Dissertation]. Case Western Reserve University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1221077215


University of Saskatchewan

16. Rafiei, Hossein 1981-. Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease.

Degree: 2017, University of Saskatchewan

 Non-alcoholic fatty liver disease (NAFLD) is a public health burden. Steatosis as the “first hit”, and oxidative stress, inflammation, mitochondrial dysfunction, and endoplasmic reticulum stress… (more)

Subjects/Keywords: Non-alcoholic fatty liver disease; Mitochondrial dysfunction; endoplasmic reticulum stress; Steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rafiei, H. 1. (2017). Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/7946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rafiei, Hossein 1981-. “Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease.” 2017. Thesis, University of Saskatchewan. Accessed July 17, 2019. http://hdl.handle.net/10388/7946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rafiei, Hossein 1981-. “Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease.” 2017. Web. 17 Jul 2019.

Vancouver:

Rafiei H1. Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease. [Internet] [Thesis]. University of Saskatchewan; 2017. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/10388/7946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rafiei H1. Molecular mechanisms of protection by dietary polyphenols against free fatty acid-induced mitochondrial dysfunction and endoplasmic reticulum stress in an in vitro model of non-alcoholic fatty liver disease. [Thesis]. University of Saskatchewan; 2017. Available from: http://hdl.handle.net/10388/7946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Saskatchewan

17. Jackel-Cram, Candice Marie. Molecular Mechanisms of Hepatitis C Virus- Associated Steatosis.

Degree: 2009, University of Saskatchewan

 Hepatitis C virus (HCV) infects millions of people worldwide and is one of the leading causes of liver damage. Infection with HCV is strongly correlated… (more)

Subjects/Keywords: Hepatitis C Virus; Cell Signalling; Akt; FAS; SREBP; Lipid Metabolism; Steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jackel-Cram, C. M. (2009). Molecular Mechanisms of Hepatitis C Virus- Associated Steatosis. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/etd-07312009-130703

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jackel-Cram, Candice Marie. “Molecular Mechanisms of Hepatitis C Virus- Associated Steatosis.” 2009. Thesis, University of Saskatchewan. Accessed July 17, 2019. http://hdl.handle.net/10388/etd-07312009-130703.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jackel-Cram, Candice Marie. “Molecular Mechanisms of Hepatitis C Virus- Associated Steatosis.” 2009. Web. 17 Jul 2019.

Vancouver:

Jackel-Cram CM. Molecular Mechanisms of Hepatitis C Virus- Associated Steatosis. [Internet] [Thesis]. University of Saskatchewan; 2009. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/10388/etd-07312009-130703.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jackel-Cram CM. Molecular Mechanisms of Hepatitis C Virus- Associated Steatosis. [Thesis]. University of Saskatchewan; 2009. Available from: http://hdl.handle.net/10388/etd-07312009-130703

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Pais, Raluca. L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies.

Degree: Docteur es, Physiologie et Physiopathologie, 2015, Université Pierre et Marie Curie – Paris VI

L’objectif général de ce travail était de mieux définir à travers des études cliniques le rôle de l’inflammation hépatique dans l’histoire naturelle de la NAFLD.… (more)

Subjects/Keywords: Stéatose; Steatohépatite; Fibrose; Inflammation; Carcinome hépatocellulaire; Athérosclérose; Steatosis; Atherosclerosis; 616.3

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pais, R. (2015). L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066223

Chicago Manual of Style (16th Edition):

Pais, Raluca. “L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed July 17, 2019. http://www.theses.fr/2015PA066223.

MLA Handbook (7th Edition):

Pais, Raluca. “L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies.” 2015. Web. 17 Jul 2019.

Vancouver:

Pais R. L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2019 Jul 17]. Available from: http://www.theses.fr/2015PA066223.

Council of Science Editors:

Pais R. L’inflammation hépatique dans les formes sévères de NAFLD : implications cliniques, médiateurs et stratégies diagnostiques : Role of chronic inflammation in advanced NAFLD : mechanisms, clinical impact and diagnostic strategies. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066223


University of Texas Medical Branch – Galveston

19. [No author]. Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice.

Degree: University of Texas Medical Branch – Galveston

 The aryl hydrocarbon receptor (AhR) is a cytosolic, ligand activated transcription factor commonly known for its role in environmental toxicant metabolism. However, the generation of… (more)

Subjects/Keywords: AhR FGF21 Steatosis Obesity

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

author], [. (n.d.). Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice. (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/11212

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice. ” Thesis, University of Texas Medical Branch – Galveston. Accessed July 17, 2019. http://hdl.handle.net/2152.3/11212.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice. ” Web. 17 Jul 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

author] [. Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice. [Internet] [Thesis]. University of Texas Medical Branch – Galveston; [cited 2019 Jul 17]. Available from: http://hdl.handle.net/2152.3/11212.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

author] [. Glucose and Lipid Dysregulation Following Loss of Hepatic Aryl Hydrocarbon Receptor in Mice. [Thesis]. University of Texas Medical Branch – Galveston; Available from: http://hdl.handle.net/2152.3/11212

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Dundee

20. Garbacz, Wojciech G. Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice.

Degree: PhD, 2012, University of Dundee

 Peroxisome proliferator-activated receptor alpha (PPARa) and delta (PPARd) belong to the nuclear receptor superfamily. PPARa is a target of lipid-lowering drugs and PPARd promotes fatty… (more)

Subjects/Keywords: 616.3; Peroxisome Proliferator Activator Receptor (PPAR); Liver; Steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Garbacz, W. G. (2012). Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/9b2509f1-3391-45fa-91eb-9b5364a9b6d9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578897

Chicago Manual of Style (16th Edition):

Garbacz, Wojciech G. “Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice.” 2012. Doctoral Dissertation, University of Dundee. Accessed July 17, 2019. https://discovery.dundee.ac.uk/en/studentTheses/9b2509f1-3391-45fa-91eb-9b5364a9b6d9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578897.

MLA Handbook (7th Edition):

Garbacz, Wojciech G. “Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice.” 2012. Web. 17 Jul 2019.

Vancouver:

Garbacz WG. Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice. [Internet] [Doctoral dissertation]. University of Dundee; 2012. [cited 2019 Jul 17]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/9b2509f1-3391-45fa-91eb-9b5364a9b6d9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578897.

Council of Science Editors:

Garbacz WG. Cooperation between peroxisome proliferator activated receptor alpha and delta in regulation of body weight and hepatic steatosis in mice. [Doctoral Dissertation]. University of Dundee; 2012. Available from: https://discovery.dundee.ac.uk/en/studentTheses/9b2509f1-3391-45fa-91eb-9b5364a9b6d9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578897

21. GENG WEI. Roles of the microRNAs in fatty liver lipid accumulation.

Degree: 2012, National University of Singapore

Subjects/Keywords: microRNA; NAFLD; lipid; steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

WEI, G. (2012). Roles of the microRNAs in fatty liver lipid accumulation. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/33282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

WEI, GENG. “Roles of the microRNAs in fatty liver lipid accumulation.” 2012. Thesis, National University of Singapore. Accessed July 17, 2019. http://scholarbank.nus.edu.sg/handle/10635/33282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

WEI, GENG. “Roles of the microRNAs in fatty liver lipid accumulation.” 2012. Web. 17 Jul 2019.

Vancouver:

WEI G. Roles of the microRNAs in fatty liver lipid accumulation. [Internet] [Thesis]. National University of Singapore; 2012. [cited 2019 Jul 17]. Available from: http://scholarbank.nus.edu.sg/handle/10635/33282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

WEI G. Roles of the microRNAs in fatty liver lipid accumulation. [Thesis]. National University of Singapore; 2012. Available from: http://scholarbank.nus.edu.sg/handle/10635/33282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

22. Clark, Paul James. Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications.

Degree: Kirby Institute, 2012, University of New South Wales

 Background: The hepatitis C virus (HCV) relies on host lipid pathways for its life cycle, leading to metabolicconsequences including hypocholesterolemia, insulin resistance and hepatic steatosis.… (more)

Subjects/Keywords: Trascriptomics; Hepatitis C; Genomics; Metabolomics; Steatosis; IL28B; PNPLA3; GWAS

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Clark, P. J. (2012). Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52447 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11120/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Clark, Paul James. “Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications.” 2012. Doctoral Dissertation, University of New South Wales. Accessed July 17, 2019. http://handle.unsw.edu.au/1959.4/52447 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11120/SOURCE01?view=true.

MLA Handbook (7th Edition):

Clark, Paul James. “Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications.” 2012. Web. 17 Jul 2019.

Vancouver:

Clark PJ. Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2019 Jul 17]. Available from: http://handle.unsw.edu.au/1959.4/52447 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11120/SOURCE01?view=true.

Council of Science Editors:

Clark PJ. Translational genomics, transcriptomics and metabolomics analyses of the metabolic effects of chronic hepatitis C infection and their clinical implications. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/52447 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11120/SOURCE01?view=true


Virginia Commonwealth University

23. Kwong, Eric K. The Role of Sphingosine Kinase 2 in Alcoholic Liver Disease.

Degree: PhD, Microbiology & Immunology, 2019, Virginia Commonwealth University

  Alcoholic liver disease (ALD) is one of the most common liver diseases worldwide characterized by the accumulation of lipids within the liver, inflammation and… (more)

Subjects/Keywords: sphingolipid; steatosis; lipid signaling; inflammation; bile acids; Biology; Gastroenterology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kwong, E. K. (2019). The Role of Sphingosine Kinase 2 in Alcoholic Liver Disease. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/5808

Chicago Manual of Style (16th Edition):

Kwong, Eric K. “The Role of Sphingosine Kinase 2 in Alcoholic Liver Disease.” 2019. Doctoral Dissertation, Virginia Commonwealth University. Accessed July 17, 2019. https://scholarscompass.vcu.edu/etd/5808.

MLA Handbook (7th Edition):

Kwong, Eric K. “The Role of Sphingosine Kinase 2 in Alcoholic Liver Disease.” 2019. Web. 17 Jul 2019.

Vancouver:

Kwong EK. The Role of Sphingosine Kinase 2 in Alcoholic Liver Disease. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2019. [cited 2019 Jul 17]. Available from: https://scholarscompass.vcu.edu/etd/5808.

Council of Science Editors:

Kwong EK. The Role of Sphingosine Kinase 2 in Alcoholic Liver Disease. [Doctoral Dissertation]. Virginia Commonwealth University; 2019. Available from: https://scholarscompass.vcu.edu/etd/5808


University of Southern California

24. Josephrajan, Ajeetha. Calorie restriction reduces liver steatosis in liver specific Pten deleted mouse model.

Degree: MS, Biochemistry & Molecular Biology, 2010, University of Southern California

 Calorie restriction (CR) is an approach wherein there is moderate restriction of food intake while providing sufficient amount of essential nutrients. CR is beneficial in… (more)

Subjects/Keywords: calorie restriction; fatty acid oxidation; liver; MCAD; Pten; steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Josephrajan, A. (2010). Calorie restriction reduces liver steatosis in liver specific Pten deleted mouse model. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/381914/rec/1213

Chicago Manual of Style (16th Edition):

Josephrajan, Ajeetha. “Calorie restriction reduces liver steatosis in liver specific Pten deleted mouse model.” 2010. Masters Thesis, University of Southern California. Accessed July 17, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/381914/rec/1213.

MLA Handbook (7th Edition):

Josephrajan, Ajeetha. “Calorie restriction reduces liver steatosis in liver specific Pten deleted mouse model.” 2010. Web. 17 Jul 2019.

Vancouver:

Josephrajan A. Calorie restriction reduces liver steatosis in liver specific Pten deleted mouse model. [Internet] [Masters thesis]. University of Southern California; 2010. [cited 2019 Jul 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/381914/rec/1213.

Council of Science Editors:

Josephrajan A. Calorie restriction reduces liver steatosis in liver specific Pten deleted mouse model. [Masters Thesis]. University of Southern California; 2010. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/381914/rec/1213


Boston University

25. Yengul, Sanjay S. Shear wave rheometry with applications in elastography.

Degree: PhD, Mechanical Engineering, 2019, Boston University

 The goal of elastography is to map the mechanical properties of soft tissues associated with health and disease. The mechanical property of interest in this… (more)

Subjects/Keywords: Acoustics; Elastic wave; Liver fibrosis; Medical imaging; NAFLD; Steatosis; Viscoelasticity

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yengul, S. S. (2019). Shear wave rheometry with applications in elastography. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/34923

Chicago Manual of Style (16th Edition):

Yengul, Sanjay S. “Shear wave rheometry with applications in elastography.” 2019. Doctoral Dissertation, Boston University. Accessed July 17, 2019. http://hdl.handle.net/2144/34923.

MLA Handbook (7th Edition):

Yengul, Sanjay S. “Shear wave rheometry with applications in elastography.” 2019. Web. 17 Jul 2019.

Vancouver:

Yengul SS. Shear wave rheometry with applications in elastography. [Internet] [Doctoral dissertation]. Boston University; 2019. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/2144/34923.

Council of Science Editors:

Yengul SS. Shear wave rheometry with applications in elastography. [Doctoral Dissertation]. Boston University; 2019. Available from: http://hdl.handle.net/2144/34923


University of Louisville

26. Donde, Hridgandh. Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury.

Degree: MS, 2013, University of Louisville

  Highly Active Antiretroviral Therapy (HAART) has led to a significant increase in the life expectancy of HIV patients; however, there are significant side effects… (more)

Subjects/Keywords: HAART; Hepatic injury; Ethanol; Steatosis; Pharmacy and Pharmaceutical Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Donde, H. (2013). Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/363 ; https://ir.library.louisville.edu/etd/363

Chicago Manual of Style (16th Edition):

Donde, Hridgandh. “Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury.” 2013. Masters Thesis, University of Louisville. Accessed July 17, 2019. 10.18297/etd/363 ; https://ir.library.louisville.edu/etd/363.

MLA Handbook (7th Edition):

Donde, Hridgandh. “Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury.” 2013. Web. 17 Jul 2019.

Vancouver:

Donde H. Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury. [Internet] [Masters thesis]. University of Louisville; 2013. [cited 2019 Jul 17]. Available from: 10.18297/etd/363 ; https://ir.library.louisville.edu/etd/363.

Council of Science Editors:

Donde H. Role of ethanol as a cofactor in HAART induced hepatic steatosis and injury. [Masters Thesis]. University of Louisville; 2013. Available from: 10.18297/etd/363 ; https://ir.library.louisville.edu/etd/363


University of New South Wales

27. Wainwright, Camilla. Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques.

Degree: Clinical School - St Vincent's Hospital, 2016, University of New South Wales

 The novel technique of 1H MRS at a high-field strength (3 Tesla) was demonstrated to be a reproducible method to assess hepatic lipid content.This technique… (more)

Subjects/Keywords: Advanced imaging techniques; Myocardial and hepatic steatosis; Vivo

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wainwright, C. (2016). Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/60083 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51140/SOURCE2?view=true

Chicago Manual of Style (16th Edition):

Wainwright, Camilla. “Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques.” 2016. Doctoral Dissertation, University of New South Wales. Accessed July 17, 2019. http://handle.unsw.edu.au/1959.4/60083 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51140/SOURCE2?view=true.

MLA Handbook (7th Edition):

Wainwright, Camilla. “Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques.” 2016. Web. 17 Jul 2019.

Vancouver:

Wainwright C. Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques. [Internet] [Doctoral dissertation]. University of New South Wales; 2016. [cited 2019 Jul 17]. Available from: http://handle.unsw.edu.au/1959.4/60083 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51140/SOURCE2?view=true.

Council of Science Editors:

Wainwright C. Assessment of myocardial and hepatic steatosis, fibrosis and viability using ex vivo, in vivo and advanced imaging techniques. [Doctoral Dissertation]. University of New South Wales; 2016. Available from: http://handle.unsw.edu.au/1959.4/60083 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51140/SOURCE2?view=true

28. Castro benitez, Carlos. Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation.

Degree: Docteur es, Toxicologie, 2019, Paris Saclay

La préservation statique à froid (SCS) est l’étalon-or de la préservation des organes après une greffe. En raison de la pénurie d’organes et de l’augmentation… (more)

Subjects/Keywords: Transplantation Hépatique; Solution de préservation; Stéatose; Liver Transplant; Preservation Solution; Steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Castro benitez, C. (2019). Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2019SACLS068

Chicago Manual of Style (16th Edition):

Castro benitez, Carlos. “Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation.” 2019. Doctoral Dissertation, Paris Saclay. Accessed July 17, 2019. http://www.theses.fr/2019SACLS068.

MLA Handbook (7th Edition):

Castro benitez, Carlos. “Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation.” 2019. Web. 17 Jul 2019.

Vancouver:

Castro benitez C. Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation. [Internet] [Doctoral dissertation]. Paris Saclay; 2019. [cited 2019 Jul 17]. Available from: http://www.theses.fr/2019SACLS068.

Council of Science Editors:

Castro benitez C. Innovative strategies to improve liver grafts quality before transplantation : Stratégies innovantes pour l’amélioration des greffons hépatiques avant la transplantation. [Doctoral Dissertation]. Paris Saclay; 2019. Available from: http://www.theses.fr/2019SACLS068


University of Toronto

29. Brandt, Sydney Laura. The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism.

Degree: 2017, University of Toronto

Elevation of the fish oil metabolite 3-carboxy-4-methyl-5-propyl-2-furanpropanoic acid (CMPF) induces metabolic remodeling within the -cell to a preferential use of fatty acids. Although detrimental to… (more)

Subjects/Keywords: ACC Inhibition; CMPF; FGF21; Lipid metabolism; NAFLD; Steatosis; 0719

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brandt, S. L. (2017). The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/88616

Chicago Manual of Style (16th Edition):

Brandt, Sydney Laura. “The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism.” 2017. Masters Thesis, University of Toronto. Accessed July 17, 2019. http://hdl.handle.net/1807/88616.

MLA Handbook (7th Edition):

Brandt, Sydney Laura. “The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism.” 2017. Web. 17 Jul 2019.

Vancouver:

Brandt SL. The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism. [Internet] [Masters thesis]. University of Toronto; 2017. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/1807/88616.

Council of Science Editors:

Brandt SL. The Circulating Furan Fatty Acid Metabolite CMPF Directly Enhances Hepatic FGF21 Secretion and Lipid Metabolism. [Masters Thesis]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/88616


Arizona State University

30. Crawford, Meli'sa Shaunte. Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats.

Degree: Biology, 2019, Arizona State University

 The prevalence of obesity and obesity-related disorders have increased world-wide. In the last decade, the intestinal microbiome has become a major indicator of metabolic and… (more)

Subjects/Keywords: Physiology; High Fat Diet; Inflammation; Metabolic Syndrome; Microbiome; Obesity; Steatosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Crawford, M. S. (2019). Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats. (Doctoral Dissertation). Arizona State University. Retrieved from http://repository.asu.edu/items/53625

Chicago Manual of Style (16th Edition):

Crawford, Meli'sa Shaunte. “Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats.” 2019. Doctoral Dissertation, Arizona State University. Accessed July 17, 2019. http://repository.asu.edu/items/53625.

MLA Handbook (7th Edition):

Crawford, Meli'sa Shaunte. “Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats.” 2019. Web. 17 Jul 2019.

Vancouver:

Crawford MS. Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats. [Internet] [Doctoral dissertation]. Arizona State University; 2019. [cited 2019 Jul 17]. Available from: http://repository.asu.edu/items/53625.

Council of Science Editors:

Crawford MS. Examining the Effects of a High Fat Diet on the Development of Metabolic Syndrome and Gut Leakiness in Male Sprague-Dawley Rats. [Doctoral Dissertation]. Arizona State University; 2019. Available from: http://repository.asu.edu/items/53625

[1] [2] [3] [4] [5]

.