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You searched for subject:(Statins Cardiovascular agents ). Showing records 1 – 30 of 10575 total matches.

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University of Aberdeen

1. Jalal, Mohammed Mansour. Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability.

Degree: PhD, 2017, University of Aberdeen

Statins are 3-hydroxy, 3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors, which block the cholesterol biosynthetic pathway to lower total serum levels and LDL-cholesterol. The cholesterol pathway… (more)

Subjects/Keywords: 610; Statins (Cardiovascular agents); Fibrinolytic agents; Blood platelets; Blood

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jalal, M. M. (2017). Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=235575 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.731642

Chicago Manual of Style (16th Edition):

Jalal, Mohammed Mansour. “Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability.” 2017. Doctoral Dissertation, University of Aberdeen. Accessed August 09, 2020. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=235575 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.731642.

MLA Handbook (7th Edition):

Jalal, Mohammed Mansour. “Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability.” 2017. Web. 09 Aug 2020.

Vancouver:

Jalal MM. Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability. [Internet] [Doctoral dissertation]. University of Aberdeen; 2017. [cited 2020 Aug 09]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=235575 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.731642.

Council of Science Editors:

Jalal MM. Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability. [Doctoral Dissertation]. University of Aberdeen; 2017. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=235575 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.731642


University of Hong Kong

2. Tsang, Siu-yee, Patricia. Regulation of cholesterol metabolism in hepatocytes.

Degree: 2000, University of Hong Kong

Subjects/Keywords: Statins (Cardiovascular agents); Hepatoma.; Cholesterol.

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APA (6th Edition):

Tsang, Siu-yee, P. (2000). Regulation of cholesterol metabolism in hepatocytes. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/27610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsang, Siu-yee, Patricia. “Regulation of cholesterol metabolism in hepatocytes.” 2000. Thesis, University of Hong Kong. Accessed August 09, 2020. http://hdl.handle.net/10722/27610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsang, Siu-yee, Patricia. “Regulation of cholesterol metabolism in hepatocytes.” 2000. Web. 09 Aug 2020.

Vancouver:

Tsang, Siu-yee P. Regulation of cholesterol metabolism in hepatocytes. [Internet] [Thesis]. University of Hong Kong; 2000. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/10722/27610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsang, Siu-yee P. Regulation of cholesterol metabolism in hepatocytes. [Thesis]. University of Hong Kong; 2000. Available from: http://hdl.handle.net/10722/27610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

3. Ooi, Kenneth Gek-Jin. The potential immunomodulatory effects of the statins in uveitis.

Degree: 2006, University of Adelaide

 The aim of this study was to delineate the patterns of cytokine expression which occur in various forms of immune-mediated uveitis in order to better… (more)

Subjects/Keywords: uveitis treatment; statins (cardiovascular agents)

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APA (6th Edition):

Ooi, K. G. (2006). The potential immunomodulatory effects of the statins in uveitis. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/69455

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ooi, Kenneth Gek-Jin. “The potential immunomodulatory effects of the statins in uveitis.” 2006. Thesis, University of Adelaide. Accessed August 09, 2020. http://hdl.handle.net/2440/69455.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ooi, Kenneth Gek-Jin. “The potential immunomodulatory effects of the statins in uveitis.” 2006. Web. 09 Aug 2020.

Vancouver:

Ooi KG. The potential immunomodulatory effects of the statins in uveitis. [Internet] [Thesis]. University of Adelaide; 2006. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/2440/69455.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ooi KG. The potential immunomodulatory effects of the statins in uveitis. [Thesis]. University of Adelaide; 2006. Available from: http://hdl.handle.net/2440/69455

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Smelser, Lisa K. Effect of simvastatin pretreatment on immunologic memory and survival in response to secondary Staphylococcus aureus infection.

Degree: Thesis (D. Ed.), 2013, Ball State University

 S. aureus is a leading cause of sepsis in the United States, which is the result of an overly robust inflammatory reaction mounted by the… (more)

Subjects/Keywords: Statins (Cardiovascular agents)  – Immunology; Immunologic memory; Staphylococcus aureus infections  – Immunological aspects

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APA (6th Edition):

Smelser, L. K. (2013). Effect of simvastatin pretreatment on immunologic memory and survival in response to secondary Staphylococcus aureus infection. (Doctoral Dissertation). Ball State University. Retrieved from http://cardinalscholar.bsu.edu/handle/123456789/197138

Chicago Manual of Style (16th Edition):

Smelser, Lisa K. “Effect of simvastatin pretreatment on immunologic memory and survival in response to secondary Staphylococcus aureus infection.” 2013. Doctoral Dissertation, Ball State University. Accessed August 09, 2020. http://cardinalscholar.bsu.edu/handle/123456789/197138.

MLA Handbook (7th Edition):

Smelser, Lisa K. “Effect of simvastatin pretreatment on immunologic memory and survival in response to secondary Staphylococcus aureus infection.” 2013. Web. 09 Aug 2020.

Vancouver:

Smelser LK. Effect of simvastatin pretreatment on immunologic memory and survival in response to secondary Staphylococcus aureus infection. [Internet] [Doctoral dissertation]. Ball State University; 2013. [cited 2020 Aug 09]. Available from: http://cardinalscholar.bsu.edu/handle/123456789/197138.

Council of Science Editors:

Smelser LK. Effect of simvastatin pretreatment on immunologic memory and survival in response to secondary Staphylococcus aureus infection. [Doctoral Dissertation]. Ball State University; 2013. Available from: http://cardinalscholar.bsu.edu/handle/123456789/197138


Rutgers University

5. Casteblanco, Fabian F. Modelling and predicting the free energy and solubility behavior of Lovastatin and Simvastatin using molecular simulations.

Degree: MS, Chemical and Biochemical Engineering, 2013, Rutgers University

 In this master thesis, exploration is done in employing molecular modelling methods to predict drug solubility in liquid solvents, compare the simulation results with experimental… (more)

Subjects/Keywords: Drugs – Solubility – Testing; Molecular dynamics; Statins (Cardiovascular agents)

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APA (6th Edition):

Casteblanco, F. F. (2013). Modelling and predicting the free energy and solubility behavior of Lovastatin and Simvastatin using molecular simulations. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/41739/

Chicago Manual of Style (16th Edition):

Casteblanco, Fabian F. “Modelling and predicting the free energy and solubility behavior of Lovastatin and Simvastatin using molecular simulations.” 2013. Masters Thesis, Rutgers University. Accessed August 09, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/41739/.

MLA Handbook (7th Edition):

Casteblanco, Fabian F. “Modelling and predicting the free energy and solubility behavior of Lovastatin and Simvastatin using molecular simulations.” 2013. Web. 09 Aug 2020.

Vancouver:

Casteblanco FF. Modelling and predicting the free energy and solubility behavior of Lovastatin and Simvastatin using molecular simulations. [Internet] [Masters thesis]. Rutgers University; 2013. [cited 2020 Aug 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/41739/.

Council of Science Editors:

Casteblanco FF. Modelling and predicting the free energy and solubility behavior of Lovastatin and Simvastatin using molecular simulations. [Masters Thesis]. Rutgers University; 2013. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/41739/

6. Glassburn, Jenny E. The effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus infection: Title on signature form: Effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus.

Degree: Thesis (M.S.), 2011, Ball State University

 Sepsis is a systemic inflammatory response that causes, increased heart rate, respirations, fever, and inadequate blood flow to organs. One of the most prevalent causes… (more)

Subjects/Keywords: Statins (Cardiovascular agents)  – Physiological effect.; Staphylococcus aureus infections  – Treatment.; Mice  – Immunology.

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APA (6th Edition):

Glassburn, J. E. (2011). The effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus infection: Title on signature form: Effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus. (Masters Thesis). Ball State University. Retrieved from http://cardinalscholar.bsu.edu/handle/123456789/194745

Chicago Manual of Style (16th Edition):

Glassburn, Jenny E. “The effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus infection: Title on signature form: Effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus.” 2011. Masters Thesis, Ball State University. Accessed August 09, 2020. http://cardinalscholar.bsu.edu/handle/123456789/194745.

MLA Handbook (7th Edition):

Glassburn, Jenny E. “The effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus infection: Title on signature form: Effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus.” 2011. Web. 09 Aug 2020.

Vancouver:

Glassburn JE. The effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus infection: Title on signature form: Effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus. [Internet] [Masters thesis]. Ball State University; 2011. [cited 2020 Aug 09]. Available from: http://cardinalscholar.bsu.edu/handle/123456789/194745.

Council of Science Editors:

Glassburn JE. The effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus infection: Title on signature form: Effects of simvastatin pretreatment on innate immune responses to Staphylococcus aureus. [Masters Thesis]. Ball State University; 2011. Available from: http://cardinalscholar.bsu.edu/handle/123456789/194745


University of Missouri – Columbia

7. Drenkhahn, Sara K., 1985-. Use of Simvastatin to Inhibit Advanced Prostate Cancer Formation in the TRAMP Mouse Model of Prostate Cancer.

Degree: 2013, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT AUTHOR'S REQUEST.] A provocative question currently proposed by the NIH is: do statin medications, which lower serum… (more)

Subjects/Keywords: Statins (Cardiovascular agents)  – Physiological effect  – Animal models.; Prostate  – Cancer  – Prevention  – Animal models.; Oxysterols.

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APA (6th Edition):

Drenkhahn, Sara K., 1. (2013). Use of Simvastatin to Inhibit Advanced Prostate Cancer Formation in the TRAMP Mouse Model of Prostate Cancer. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/43337

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Drenkhahn, Sara K., 1985-. “Use of Simvastatin to Inhibit Advanced Prostate Cancer Formation in the TRAMP Mouse Model of Prostate Cancer.” 2013. Thesis, University of Missouri – Columbia. Accessed August 09, 2020. http://hdl.handle.net/10355/43337.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Drenkhahn, Sara K., 1985-. “Use of Simvastatin to Inhibit Advanced Prostate Cancer Formation in the TRAMP Mouse Model of Prostate Cancer.” 2013. Web. 09 Aug 2020.

Vancouver:

Drenkhahn, Sara K. 1. Use of Simvastatin to Inhibit Advanced Prostate Cancer Formation in the TRAMP Mouse Model of Prostate Cancer. [Internet] [Thesis]. University of Missouri – Columbia; 2013. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/10355/43337.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Drenkhahn, Sara K. 1. Use of Simvastatin to Inhibit Advanced Prostate Cancer Formation in the TRAMP Mouse Model of Prostate Cancer. [Thesis]. University of Missouri – Columbia; 2013. Available from: http://hdl.handle.net/10355/43337

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Caffo, Lindy Marie. Simvastatin and ML 141 inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes: Simvastatin and 4-[3-(4-methoxyphenyl)-5-phenyl-3,4 dihydropyrazol-2-yl]benzenesulfonamide inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes.

Degree: Thesis (M.S.), 2014, Ball State University

 Streptococcus pyogenes is a human pathogen that can manipulate host cell machinery in human umbilical vein endothelial cells (HUVEC), exploiting host cell endocytosis to become… (more)

Subjects/Keywords: Statins (Cardiovascular agents)  – Physiological effect.; Sulfonamides  – Physiological effect.; Endothelial cells.; Guanosine triphosphatase.; Streptococcus pyogenes.

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APA (6th Edition):

Caffo, L. M. (2014). Simvastatin and ML 141 inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes: Simvastatin and 4-[3-(4-methoxyphenyl)-5-phenyl-3,4 dihydropyrazol-2-yl]benzenesulfonamide inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes. (Masters Thesis). Ball State University. Retrieved from http://cardinalscholar.bsu.edu/handle/123456789/198452

Chicago Manual of Style (16th Edition):

Caffo, Lindy Marie. “Simvastatin and ML 141 inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes: Simvastatin and 4-[3-(4-methoxyphenyl)-5-phenyl-3,4 dihydropyrazol-2-yl]benzenesulfonamide inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes.” 2014. Masters Thesis, Ball State University. Accessed August 09, 2020. http://cardinalscholar.bsu.edu/handle/123456789/198452.

MLA Handbook (7th Edition):

Caffo, Lindy Marie. “Simvastatin and ML 141 inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes: Simvastatin and 4-[3-(4-methoxyphenyl)-5-phenyl-3,4 dihydropyrazol-2-yl]benzenesulfonamide inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes.” 2014. Web. 09 Aug 2020.

Vancouver:

Caffo LM. Simvastatin and ML 141 inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes: Simvastatin and 4-[3-(4-methoxyphenyl)-5-phenyl-3,4 dihydropyrazol-2-yl]benzenesulfonamide inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes. [Internet] [Masters thesis]. Ball State University; 2014. [cited 2020 Aug 09]. Available from: http://cardinalscholar.bsu.edu/handle/123456789/198452.

Council of Science Editors:

Caffo LM. Simvastatin and ML 141 inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes: Simvastatin and 4-[3-(4-methoxyphenyl)-5-phenyl-3,4 dihydropyrazol-2-yl]benzenesulfonamide inhibit endothelial host cell processes to limit the invasion of Streptococcus pyogenes. [Masters Thesis]. Ball State University; 2014. Available from: http://cardinalscholar.bsu.edu/handle/123456789/198452


University of Missouri – Columbia

9. Drenkhahn, Sara K., 1985-. Use of simvastatin to inhibit advanced prostate cancer formation in the TRAMP mouse model of prostate cancer.

Degree: 2013, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT AUTHOR'S REQUEST.] A provocative question currently proposed by the NIH is: do statin medications, which lower serum… (more)

Subjects/Keywords: Statins (Cardiovascular agents)  – Physiological effect  – Animal models.; Prostate  – Cancer  – Prevention  – Animal models.; Oxysterols.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Drenkhahn, Sara K., 1. (2013). Use of simvastatin to inhibit advanced prostate cancer formation in the TRAMP mouse model of prostate cancer. (Thesis). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/43337

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Drenkhahn, Sara K., 1985-. “Use of simvastatin to inhibit advanced prostate cancer formation in the TRAMP mouse model of prostate cancer.” 2013. Thesis, University of Missouri – Columbia. Accessed August 09, 2020. https://doi.org/10.32469/10355/43337.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Drenkhahn, Sara K., 1985-. “Use of simvastatin to inhibit advanced prostate cancer formation in the TRAMP mouse model of prostate cancer.” 2013. Web. 09 Aug 2020.

Vancouver:

Drenkhahn, Sara K. 1. Use of simvastatin to inhibit advanced prostate cancer formation in the TRAMP mouse model of prostate cancer. [Internet] [Thesis]. University of Missouri – Columbia; 2013. [cited 2020 Aug 09]. Available from: https://doi.org/10.32469/10355/43337.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Drenkhahn, Sara K. 1. Use of simvastatin to inhibit advanced prostate cancer formation in the TRAMP mouse model of prostate cancer. [Thesis]. University of Missouri – Columbia; 2013. Available from: https://doi.org/10.32469/10355/43337

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

10. Ng, Chun-man. Effect of statins on prevention of cardiovascular diseases in Asian population: a systematic review ofrandomized, controlled trials.

Degree: 2012, University of Hong Kong

 Background Cardiovascular disease (CVD) is the worldwide leading cause of death among non-communicable diseases and results in a huge burden of mortality and morbidity. China,… (more)

Subjects/Keywords: Statins (Cardiovascular agents); Cardiovascular system - Diseases - Prevention. - Asia

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APA (6th Edition):

Ng, C. (2012). Effect of statins on prevention of cardiovascular diseases in Asian population: a systematic review ofrandomized, controlled trials. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/179924

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ng, Chun-man. “Effect of statins on prevention of cardiovascular diseases in Asian population: a systematic review ofrandomized, controlled trials.” 2012. Thesis, University of Hong Kong. Accessed August 09, 2020. http://hdl.handle.net/10722/179924.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ng, Chun-man. “Effect of statins on prevention of cardiovascular diseases in Asian population: a systematic review ofrandomized, controlled trials.” 2012. Web. 09 Aug 2020.

Vancouver:

Ng C. Effect of statins on prevention of cardiovascular diseases in Asian population: a systematic review ofrandomized, controlled trials. [Internet] [Thesis]. University of Hong Kong; 2012. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/10722/179924.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ng C. Effect of statins on prevention of cardiovascular diseases in Asian population: a systematic review ofrandomized, controlled trials. [Thesis]. University of Hong Kong; 2012. Available from: http://hdl.handle.net/10722/179924

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Aberdeen

11. Jalal, Mohammed Mansour. Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability.

Degree: School of Medical Sciences., 2017, University of Aberdeen

Subjects/Keywords: Statins (Cardiovascular agents); Fibrinolytic agents.; Blood platelets.; Blood

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jalal, M. M. (2017). Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=235575 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=235575&custom_att_2=simple_viewer

Chicago Manual of Style (16th Edition):

Jalal, Mohammed Mansour. “Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability.” 2017. Doctoral Dissertation, University of Aberdeen. Accessed August 09, 2020. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=235575 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=235575&custom_att_2=simple_viewer.

MLA Handbook (7th Edition):

Jalal, Mohammed Mansour. “Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability.” 2017. Web. 09 Aug 2020.

Vancouver:

Jalal MM. Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability. [Internet] [Doctoral dissertation]. University of Aberdeen; 2017. [cited 2020 Aug 09]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=235575 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=235575&custom_att_2=simple_viewer.

Council of Science Editors:

Jalal MM. Statins exert antithrombotic action on platelet function and modulate clot formation structure and stability. [Doctoral Dissertation]. University of Aberdeen; 2017. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=235575 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=235575&custom_att_2=simple_viewer


University of Oxford

12. Jayaram, Raja. Effects of peri-operative statin treatment on atrial electrical properties, post-operative atrial fibrillation and in-hospital clinical outcomes in patients undergoing elective cardiac surgery.

Degree: PhD, 2014, University of Oxford

 Surgical myocardial revascularization remains the standard of care for patients with multi-vessel coronary artery disease. A growing body of evidence indicates that systemic inflammation and… (more)

Subjects/Keywords: 617.4; Oxidative stress; Heart – Surgery; Cardiopulmonary bypass; Atrial fibrillation; Statins (Cardiovascular agents); Postoperative complications; Ischaemia reperfusion; Cardiac surgery

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APA (6th Edition):

Jayaram, R. (2014). Effects of peri-operative statin treatment on atrial electrical properties, post-operative atrial fibrillation and in-hospital clinical outcomes in patients undergoing elective cardiac surgery. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:224a03c7-30f5-456b-a996-0679591ea6a8 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711778

Chicago Manual of Style (16th Edition):

Jayaram, Raja. “Effects of peri-operative statin treatment on atrial electrical properties, post-operative atrial fibrillation and in-hospital clinical outcomes in patients undergoing elective cardiac surgery.” 2014. Doctoral Dissertation, University of Oxford. Accessed August 09, 2020. http://ora.ox.ac.uk/objects/uuid:224a03c7-30f5-456b-a996-0679591ea6a8 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711778.

MLA Handbook (7th Edition):

Jayaram, Raja. “Effects of peri-operative statin treatment on atrial electrical properties, post-operative atrial fibrillation and in-hospital clinical outcomes in patients undergoing elective cardiac surgery.” 2014. Web. 09 Aug 2020.

Vancouver:

Jayaram R. Effects of peri-operative statin treatment on atrial electrical properties, post-operative atrial fibrillation and in-hospital clinical outcomes in patients undergoing elective cardiac surgery. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2020 Aug 09]. Available from: http://ora.ox.ac.uk/objects/uuid:224a03c7-30f5-456b-a996-0679591ea6a8 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711778.

Council of Science Editors:

Jayaram R. Effects of peri-operative statin treatment on atrial electrical properties, post-operative atrial fibrillation and in-hospital clinical outcomes in patients undergoing elective cardiac surgery. [Doctoral Dissertation]. University of Oxford; 2014. Available from: http://ora.ox.ac.uk/objects/uuid:224a03c7-30f5-456b-a996-0679591ea6a8 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711778


Michigan State University

13. Rivera, Marta Cristina Abad. The x-ray crystallographic structures of branching enzyme and angiostatin.

Degree: PhD, Department of Chemistry, 2002, Michigan State University

Subjects/Keywords: Statins (Cardiovascular agents) – Structure; Enzymes – Structure; Crystallography

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APA (6th Edition):

Rivera, M. C. A. (2002). The x-ray crystallographic structures of branching enzyme and angiostatin. (Doctoral Dissertation). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:31492

Chicago Manual of Style (16th Edition):

Rivera, Marta Cristina Abad. “The x-ray crystallographic structures of branching enzyme and angiostatin.” 2002. Doctoral Dissertation, Michigan State University. Accessed August 09, 2020. http://etd.lib.msu.edu/islandora/object/etd:31492.

MLA Handbook (7th Edition):

Rivera, Marta Cristina Abad. “The x-ray crystallographic structures of branching enzyme and angiostatin.” 2002. Web. 09 Aug 2020.

Vancouver:

Rivera MCA. The x-ray crystallographic structures of branching enzyme and angiostatin. [Internet] [Doctoral dissertation]. Michigan State University; 2002. [cited 2020 Aug 09]. Available from: http://etd.lib.msu.edu/islandora/object/etd:31492.

Council of Science Editors:

Rivera MCA. The x-ray crystallographic structures of branching enzyme and angiostatin. [Doctoral Dissertation]. Michigan State University; 2002. Available from: http://etd.lib.msu.edu/islandora/object/etd:31492

14. Burns, Erin M. Simvastatin treatment modulates the immune response, increasing the survival of mice infected with Staphylococcus aureus.

Degree: Thesis (M.S.), 2009, Ball State University

 Staphylococcus aureus, the most prevalant etiologic agent causing sepsis (a damaging inflammatory response), is traditionally cleared with antibiotics. Increased numbers of antibiotic-resistant strains mandate additional… (more)

Subjects/Keywords: Statins (Cardiovascular agents)  – Physiological effect.; Staphylococcus aureus infections  – Treatment.

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APA (6th Edition):

Burns, E. M. (2009). Simvastatin treatment modulates the immune response, increasing the survival of mice infected with Staphylococcus aureus. (Masters Thesis). Ball State University. Retrieved from http://cardinalscholar.bsu.edu/handle/123456789/193841

Chicago Manual of Style (16th Edition):

Burns, Erin M. “Simvastatin treatment modulates the immune response, increasing the survival of mice infected with Staphylococcus aureus.” 2009. Masters Thesis, Ball State University. Accessed August 09, 2020. http://cardinalscholar.bsu.edu/handle/123456789/193841.

MLA Handbook (7th Edition):

Burns, Erin M. “Simvastatin treatment modulates the immune response, increasing the survival of mice infected with Staphylococcus aureus.” 2009. Web. 09 Aug 2020.

Vancouver:

Burns EM. Simvastatin treatment modulates the immune response, increasing the survival of mice infected with Staphylococcus aureus. [Internet] [Masters thesis]. Ball State University; 2009. [cited 2020 Aug 09]. Available from: http://cardinalscholar.bsu.edu/handle/123456789/193841.

Council of Science Editors:

Burns EM. Simvastatin treatment modulates the immune response, increasing the survival of mice infected with Staphylococcus aureus. [Masters Thesis]. Ball State University; 2009. Available from: http://cardinalscholar.bsu.edu/handle/123456789/193841


East Carolina University

15. Mayo, William Joseph. The effects of statins on mitochondrial function.

Degree: MS, Exercise and Sports Science, 2013, East Carolina University

Statins, or 3-hydroxy-3-methylglutaryl-coenzyme A (HMG-CoA) reductase inhibitors, are among the most commonly prescribed medications in the United States. They are commonly used to treat hypercholesterolemia,… (more)

Subjects/Keywords: Physiology; Hâ‚‚Oâ‚‚; Mitochondria; Myopathy; Respiratory capacity; Skeletal muscle; Biology, Physiology; Statins (Cardiovascular agents); Mitochondrial pathology

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APA (6th Edition):

Mayo, W. J. (2013). The effects of statins on mitochondrial function. (Masters Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/4201

Chicago Manual of Style (16th Edition):

Mayo, William Joseph. “The effects of statins on mitochondrial function.” 2013. Masters Thesis, East Carolina University. Accessed August 09, 2020. http://hdl.handle.net/10342/4201.

MLA Handbook (7th Edition):

Mayo, William Joseph. “The effects of statins on mitochondrial function.” 2013. Web. 09 Aug 2020.

Vancouver:

Mayo WJ. The effects of statins on mitochondrial function. [Internet] [Masters thesis]. East Carolina University; 2013. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/10342/4201.

Council of Science Editors:

Mayo WJ. The effects of statins on mitochondrial function. [Masters Thesis]. East Carolina University; 2013. Available from: http://hdl.handle.net/10342/4201

16. Kenney, Shelby R. Development of a high throughput small molecule screen using Staphylococcus aureus invasion of cells.

Degree: Thesis (M.S.), 2009, Ball State University

 Staphylococcus aureus is a common and versatile opportunistic pathogen in humans. Increases in the incidence of community acquired and nosocomial infections, coupled with the emergence… (more)

Subjects/Keywords: High throughput screening (Drug development); Staphylococcus aureus.; Statins (Cardiovascular agents)  – Physiological effect.; Endocytosis.

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APA (6th Edition):

Kenney, S. R. (2009). Development of a high throughput small molecule screen using Staphylococcus aureus invasion of cells. (Masters Thesis). Ball State University. Retrieved from http://cardinalscholar.bsu.edu/handle/123456789/193789

Chicago Manual of Style (16th Edition):

Kenney, Shelby R. “Development of a high throughput small molecule screen using Staphylococcus aureus invasion of cells.” 2009. Masters Thesis, Ball State University. Accessed August 09, 2020. http://cardinalscholar.bsu.edu/handle/123456789/193789.

MLA Handbook (7th Edition):

Kenney, Shelby R. “Development of a high throughput small molecule screen using Staphylococcus aureus invasion of cells.” 2009. Web. 09 Aug 2020.

Vancouver:

Kenney SR. Development of a high throughput small molecule screen using Staphylococcus aureus invasion of cells. [Internet] [Masters thesis]. Ball State University; 2009. [cited 2020 Aug 09]. Available from: http://cardinalscholar.bsu.edu/handle/123456789/193789.

Council of Science Editors:

Kenney SR. Development of a high throughput small molecule screen using Staphylococcus aureus invasion of cells. [Masters Thesis]. Ball State University; 2009. Available from: http://cardinalscholar.bsu.edu/handle/123456789/193789


IUPUI

17. Downing, Brandon David. Myeloid cells induce neurofibromatosis type 1 aneurysm formation through inflammation and oxidative stress.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Neurofibromatosis Type 1 (NF1) is a genetic disorder resulting from mutations in the NF1 tumor suppressor gene. Neurofibromin is the… (more)

Subjects/Keywords: Cardiovascular Disease; Neurofibromatosis Type 1; Inflammation; Oxidative Stress; cre/lox; Murine Model; Aneurysm; Neurofibromatosis  – Research  – Evaluation  – Analysis; Neurofibromatosis in children  – Research  – Evaluation  – Analysis; Cardiovascular system  – Diseases; Human molecular genetics; Antioncogenes; Inflammation  – Mediators; Oxidative stress; Aneurysms  – Research; Aortic aneurysms; Statins (Cardiovascular agents); Mice  – Diseases  – Molecular aspects; Animal models in research; Antioxidants  – Therapeutic use; Interleukins

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APA (6th Edition):

Downing, B. D. (2014). Myeloid cells induce neurofibromatosis type 1 aneurysm formation through inflammation and oxidative stress. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/5850

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Downing, Brandon David. “Myeloid cells induce neurofibromatosis type 1 aneurysm formation through inflammation and oxidative stress.” 2014. Thesis, IUPUI. Accessed August 09, 2020. http://hdl.handle.net/1805/5850.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Downing, Brandon David. “Myeloid cells induce neurofibromatosis type 1 aneurysm formation through inflammation and oxidative stress.” 2014. Web. 09 Aug 2020.

Vancouver:

Downing BD. Myeloid cells induce neurofibromatosis type 1 aneurysm formation through inflammation and oxidative stress. [Internet] [Thesis]. IUPUI; 2014. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/1805/5850.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Downing BD. Myeloid cells induce neurofibromatosis type 1 aneurysm formation through inflammation and oxidative stress. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/5850

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

18. Eikenburg, Douglas Charles. Mechanisms for the in vivo cardiovascular actions of 1-alpha-acetylymethadol.

Degree: PhD, Department of Pharmacology & Toxicology, 1979, Michigan State University

Subjects/Keywords: Cardiovascular agents

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APA (6th Edition):

Eikenburg, D. C. (1979). Mechanisms for the in vivo cardiovascular actions of 1-alpha-acetylymethadol. (Doctoral Dissertation). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:42056

Chicago Manual of Style (16th Edition):

Eikenburg, Douglas Charles. “Mechanisms for the in vivo cardiovascular actions of 1-alpha-acetylymethadol.” 1979. Doctoral Dissertation, Michigan State University. Accessed August 09, 2020. http://etd.lib.msu.edu/islandora/object/etd:42056.

MLA Handbook (7th Edition):

Eikenburg, Douglas Charles. “Mechanisms for the in vivo cardiovascular actions of 1-alpha-acetylymethadol.” 1979. Web. 09 Aug 2020.

Vancouver:

Eikenburg DC. Mechanisms for the in vivo cardiovascular actions of 1-alpha-acetylymethadol. [Internet] [Doctoral dissertation]. Michigan State University; 1979. [cited 2020 Aug 09]. Available from: http://etd.lib.msu.edu/islandora/object/etd:42056.

Council of Science Editors:

Eikenburg DC. Mechanisms for the in vivo cardiovascular actions of 1-alpha-acetylymethadol. [Doctoral Dissertation]. Michigan State University; 1979. Available from: http://etd.lib.msu.edu/islandora/object/etd:42056


University of Hong Kong

19. Chen, Zi. Drug testing platform using in vitro electrophysiology and in vivo rodent models for evaluating existing and novel cardiovascular drugs and their potential applications.

Degree: 2015, University of Hong Kong

 Ion channels play crucial roles in normal cardiac function and in maintaining intracellular ion homeostasis in both excitable and non-excitable cells. Cardiovascular diseases and malignancy… (more)

Subjects/Keywords: Cardiovascular agents

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APA (6th Edition):

Chen, Z. (2015). Drug testing platform using in vitro electrophysiology and in vivo rodent models for evaluating existing and novel cardiovascular drugs and their potential applications. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/221170

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Zi. “Drug testing platform using in vitro electrophysiology and in vivo rodent models for evaluating existing and novel cardiovascular drugs and their potential applications.” 2015. Thesis, University of Hong Kong. Accessed August 09, 2020. http://hdl.handle.net/10722/221170.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Zi. “Drug testing platform using in vitro electrophysiology and in vivo rodent models for evaluating existing and novel cardiovascular drugs and their potential applications.” 2015. Web. 09 Aug 2020.

Vancouver:

Chen Z. Drug testing platform using in vitro electrophysiology and in vivo rodent models for evaluating existing and novel cardiovascular drugs and their potential applications. [Internet] [Thesis]. University of Hong Kong; 2015. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/10722/221170.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen Z. Drug testing platform using in vitro electrophysiology and in vivo rodent models for evaluating existing and novel cardiovascular drugs and their potential applications. [Thesis]. University of Hong Kong; 2015. Available from: http://hdl.handle.net/10722/221170

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Otago

20. Crawford, Elisse Brooke. The Tailored Diet Study: A pilot study on the effect of individual tailored dietary advice on blood lipids in Familial Hypercholesterolaemia patients in Otago and Southland .

Degree: 2013, University of Otago

 Background: Cardiovascular disease (CVD) causes 19% of all deaths in New Zealand; the contribution of familial hypercholesterolaemia (FH) to this figure is unknown. FH is… (more)

Subjects/Keywords: Familial hypercholesterolaemia; cardiovascular disease; blood lipids; dietary intervention; statins

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APA (6th Edition):

Crawford, E. B. (2013). The Tailored Diet Study: A pilot study on the effect of individual tailored dietary advice on blood lipids in Familial Hypercholesterolaemia patients in Otago and Southland . (Masters Thesis). University of Otago. Retrieved from http://hdl.handle.net/10523/4194

Chicago Manual of Style (16th Edition):

Crawford, Elisse Brooke. “The Tailored Diet Study: A pilot study on the effect of individual tailored dietary advice on blood lipids in Familial Hypercholesterolaemia patients in Otago and Southland .” 2013. Masters Thesis, University of Otago. Accessed August 09, 2020. http://hdl.handle.net/10523/4194.

MLA Handbook (7th Edition):

Crawford, Elisse Brooke. “The Tailored Diet Study: A pilot study on the effect of individual tailored dietary advice on blood lipids in Familial Hypercholesterolaemia patients in Otago and Southland .” 2013. Web. 09 Aug 2020.

Vancouver:

Crawford EB. The Tailored Diet Study: A pilot study on the effect of individual tailored dietary advice on blood lipids in Familial Hypercholesterolaemia patients in Otago and Southland . [Internet] [Masters thesis]. University of Otago; 2013. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/10523/4194.

Council of Science Editors:

Crawford EB. The Tailored Diet Study: A pilot study on the effect of individual tailored dietary advice on blood lipids in Familial Hypercholesterolaemia patients in Otago and Southland . [Masters Thesis]. University of Otago; 2013. Available from: http://hdl.handle.net/10523/4194


Georgia Tech

21. Salo, Paul David. Protein prenylation inhibitors reveal a novel role for rhoa and rhoc in trafficking of g protein-coupled receptors through recycling endosomes.

Degree: PhD, Chemistry and Biochemistry, 2007, Georgia Tech

 LPA1 lysophosphatidic acid receptors (LPA1Rs) are normally present on the surface of the cell. Our initial findings were that HMG-CoA reductase inhibitors (atorvastatin and mevastatin)… (more)

Subjects/Keywords: G protein-coupled receptors; Lysophosphatidic acid; Statin; GGTI; Statins (Cardiovascular agents); Sequestration (Chemistry); Endocytosis; Cell membranes

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APA (6th Edition):

Salo, P. D. (2007). Protein prenylation inhibitors reveal a novel role for rhoa and rhoc in trafficking of g protein-coupled receptors through recycling endosomes. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/26711

Chicago Manual of Style (16th Edition):

Salo, Paul David. “Protein prenylation inhibitors reveal a novel role for rhoa and rhoc in trafficking of g protein-coupled receptors through recycling endosomes.” 2007. Doctoral Dissertation, Georgia Tech. Accessed August 09, 2020. http://hdl.handle.net/1853/26711.

MLA Handbook (7th Edition):

Salo, Paul David. “Protein prenylation inhibitors reveal a novel role for rhoa and rhoc in trafficking of g protein-coupled receptors through recycling endosomes.” 2007. Web. 09 Aug 2020.

Vancouver:

Salo PD. Protein prenylation inhibitors reveal a novel role for rhoa and rhoc in trafficking of g protein-coupled receptors through recycling endosomes. [Internet] [Doctoral dissertation]. Georgia Tech; 2007. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/1853/26711.

Council of Science Editors:

Salo PD. Protein prenylation inhibitors reveal a novel role for rhoa and rhoc in trafficking of g protein-coupled receptors through recycling endosomes. [Doctoral Dissertation]. Georgia Tech; 2007. Available from: http://hdl.handle.net/1853/26711

22. Millar, Danielle Ann. In vitro assessment of aspalathin-enriched Rooibos (Aspalathus linearis) extract treatment in statin-induced hepatotoxicity.

Degree: MSc, Biomedical Sciences, 2019, Stellenbosch University

ENGLISH ABSTRACT: Rooibos (Aspalathus linearis) has been shown to have various health benefits including antidiabetic, lipid-lowering, and hepatoprotective properties. Although anecdotally Rooibos consumption is regarded… (more)

Subjects/Keywords: Hepatotoxicity; Rooibos tea industry; Atorvastatin; Herb-drug interactions; Statins (Cardiovascular agents); Aspalathus linearis; UCTD

…Co-enzyme Q; ubiquinone CVD: Cardiovascular disease CYP3A4: CYP450 isoform 3A4 CYP450… …23 Figure 2.4.2: Statins interrupt the enzymatic reduction of HMG-CoA to mevalonate… …lowering drugs, statins, act by competitively inhibiting hydroxymethylglutaryl-coenzyme A (… …occurrence, statins are potentially hepatotoxic. The safety of the concurrent use of Rooibos and… …statins thus needs to be elucidated. This study aims to investigate the interaction between the… 

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APA (6th Edition):

Millar, D. A. (2019). In vitro assessment of aspalathin-enriched Rooibos (Aspalathus linearis) extract treatment in statin-induced hepatotoxicity. (Masters Thesis). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/105804

Chicago Manual of Style (16th Edition):

Millar, Danielle Ann. “In vitro assessment of aspalathin-enriched Rooibos (Aspalathus linearis) extract treatment in statin-induced hepatotoxicity.” 2019. Masters Thesis, Stellenbosch University. Accessed August 09, 2020. http://hdl.handle.net/10019.1/105804.

MLA Handbook (7th Edition):

Millar, Danielle Ann. “In vitro assessment of aspalathin-enriched Rooibos (Aspalathus linearis) extract treatment in statin-induced hepatotoxicity.” 2019. Web. 09 Aug 2020.

Vancouver:

Millar DA. In vitro assessment of aspalathin-enriched Rooibos (Aspalathus linearis) extract treatment in statin-induced hepatotoxicity. [Internet] [Masters thesis]. Stellenbosch University; 2019. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/10019.1/105804.

Council of Science Editors:

Millar DA. In vitro assessment of aspalathin-enriched Rooibos (Aspalathus linearis) extract treatment in statin-induced hepatotoxicity. [Masters Thesis]. Stellenbosch University; 2019. Available from: http://hdl.handle.net/10019.1/105804


Boston University

23. Datu Tasik, Grace Marselina. Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1).

Degree: MS, Nutrition and Metabolism, 2019, Boston University

 OBJECTIVE: Cardiovascular disease (CVD) remains the leading cause of death globally. Reducing high blood cholesterol, which is a dominant risk factor for CVD events, is… (more)

Subjects/Keywords: Nutrition; Cardiovascular disease; Cholesterol; Clonal pancreatic beta-cells; Insulin; Statins; Type 2 diabetes

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APA (6th Edition):

Datu Tasik, G. M. (2019). Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1). (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/36546

Chicago Manual of Style (16th Edition):

Datu Tasik, Grace Marselina. “Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1).” 2019. Masters Thesis, Boston University. Accessed August 09, 2020. http://hdl.handle.net/2144/36546.

MLA Handbook (7th Edition):

Datu Tasik, Grace Marselina. “Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1).” 2019. Web. 09 Aug 2020.

Vancouver:

Datu Tasik GM. Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1). [Internet] [Masters thesis]. Boston University; 2019. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/2144/36546.

Council of Science Editors:

Datu Tasik GM. Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1). [Masters Thesis]. Boston University; 2019. Available from: http://hdl.handle.net/2144/36546

24. Sawpheeyah Nima. Responsiveness of cardiac and tracheal B-Adrenoceptors to epinephrine and salbutamol in the absence of presence of B-Adrenoceptor antogonist and their correlation with Both Tissues and plasma cocaine levels of chronic cocaine-Treated Guinea-Pigs .

Degree: Prince of Songkla University Faculty of Science (Pharmacology), 2007, Prince of Songkla University

Subjects/Keywords: Cardiovascular Agents pharmacology; Cardiovascular agents; Cardiovascular agents therapeutic use

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APA (6th Edition):

Nima, S. (2007). Responsiveness of cardiac and tracheal B-Adrenoceptors to epinephrine and salbutamol in the absence of presence of B-Adrenoceptor antogonist and their correlation with Both Tissues and plasma cocaine levels of chronic cocaine-Treated Guinea-Pigs . (Thesis). Prince of Songkla University. Retrieved from http://kb.psu.ac.th/psukb/handle/2553/1344

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nima, Sawpheeyah. “Responsiveness of cardiac and tracheal B-Adrenoceptors to epinephrine and salbutamol in the absence of presence of B-Adrenoceptor antogonist and their correlation with Both Tissues and plasma cocaine levels of chronic cocaine-Treated Guinea-Pigs .” 2007. Thesis, Prince of Songkla University. Accessed August 09, 2020. http://kb.psu.ac.th/psukb/handle/2553/1344.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nima, Sawpheeyah. “Responsiveness of cardiac and tracheal B-Adrenoceptors to epinephrine and salbutamol in the absence of presence of B-Adrenoceptor antogonist and their correlation with Both Tissues and plasma cocaine levels of chronic cocaine-Treated Guinea-Pigs .” 2007. Web. 09 Aug 2020.

Vancouver:

Nima S. Responsiveness of cardiac and tracheal B-Adrenoceptors to epinephrine and salbutamol in the absence of presence of B-Adrenoceptor antogonist and their correlation with Both Tissues and plasma cocaine levels of chronic cocaine-Treated Guinea-Pigs . [Internet] [Thesis]. Prince of Songkla University; 2007. [cited 2020 Aug 09]. Available from: http://kb.psu.ac.th/psukb/handle/2553/1344.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nima S. Responsiveness of cardiac and tracheal B-Adrenoceptors to epinephrine and salbutamol in the absence of presence of B-Adrenoceptor antogonist and their correlation with Both Tissues and plasma cocaine levels of chronic cocaine-Treated Guinea-Pigs . [Thesis]. Prince of Songkla University; 2007. Available from: http://kb.psu.ac.th/psukb/handle/2553/1344

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

25. Bailey, Leslie Edgar. Effect of ouabain on Ca45 kinetics in the myocardium determined by the technique of diffusible indicator-dilution;.

Degree: PhD, Pharmacology & Toxicology;, 1967, University of Utah

 1. The purpose of this research was two-folds a) to investigate the relationship of the positive inotropic effect of ouabain to an action on calcium… (more)

Subjects/Keywords: Cardiovascular Agents; Pharmacokinetics

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APA (6th Edition):

Bailey, L. E. (1967). Effect of ouabain on Ca45 kinetics in the myocardium determined by the technique of diffusible indicator-dilution;. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1161/rec/384

Chicago Manual of Style (16th Edition):

Bailey, Leslie Edgar. “Effect of ouabain on Ca45 kinetics in the myocardium determined by the technique of diffusible indicator-dilution;.” 1967. Doctoral Dissertation, University of Utah. Accessed August 09, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1161/rec/384.

MLA Handbook (7th Edition):

Bailey, Leslie Edgar. “Effect of ouabain on Ca45 kinetics in the myocardium determined by the technique of diffusible indicator-dilution;.” 1967. Web. 09 Aug 2020.

Vancouver:

Bailey LE. Effect of ouabain on Ca45 kinetics in the myocardium determined by the technique of diffusible indicator-dilution;. [Internet] [Doctoral dissertation]. University of Utah; 1967. [cited 2020 Aug 09]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1161/rec/384.

Council of Science Editors:

Bailey LE. Effect of ouabain on Ca45 kinetics in the myocardium determined by the technique of diffusible indicator-dilution;. [Doctoral Dissertation]. University of Utah; 1967. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1161/rec/384


University of Hong Kong

26. 溫啓新. Effects of Salviae miltiorrhizae radix, a herbal medicine, on vascularconstriction and dilatation.

Degree: 2001, University of Hong Kong

Subjects/Keywords: Cardiovascular agents.; Salvia.

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APA (6th Edition):

溫啓新. (2001). Effects of Salviae miltiorrhizae radix, a herbal medicine, on vascularconstriction and dilatation. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/55682

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

溫啓新. “Effects of Salviae miltiorrhizae radix, a herbal medicine, on vascularconstriction and dilatation.” 2001. Thesis, University of Hong Kong. Accessed August 09, 2020. http://hdl.handle.net/10722/55682.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

溫啓新. “Effects of Salviae miltiorrhizae radix, a herbal medicine, on vascularconstriction and dilatation.” 2001. Web. 09 Aug 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

溫啓新. Effects of Salviae miltiorrhizae radix, a herbal medicine, on vascularconstriction and dilatation. [Internet] [Thesis]. University of Hong Kong; 2001. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/10722/55682.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

溫啓新. Effects of Salviae miltiorrhizae radix, a herbal medicine, on vascularconstriction and dilatation. [Thesis]. University of Hong Kong; 2001. Available from: http://hdl.handle.net/10722/55682

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

27. Reynnells, Richard Douglas. Effect of 2,2,9,9-tetramethyl-1,10-decanediol (CI-720) on blood, egg yolk, and excreta cholesterol, blood triglycerides, feed consumption, egg production, and egg component weights.

Degree: MS, Dept. of Poultry Science, 1976, Michigan State University

Subjects/Keywords: Cardiovascular agents; Hyperlipidemia

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APA (6th Edition):

Reynnells, R. D. (1976). Effect of 2,2,9,9-tetramethyl-1,10-decanediol (CI-720) on blood, egg yolk, and excreta cholesterol, blood triglycerides, feed consumption, egg production, and egg component weights. (Masters Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:11695

Chicago Manual of Style (16th Edition):

Reynnells, Richard Douglas. “Effect of 2,2,9,9-tetramethyl-1,10-decanediol (CI-720) on blood, egg yolk, and excreta cholesterol, blood triglycerides, feed consumption, egg production, and egg component weights.” 1976. Masters Thesis, Michigan State University. Accessed August 09, 2020. http://etd.lib.msu.edu/islandora/object/etd:11695.

MLA Handbook (7th Edition):

Reynnells, Richard Douglas. “Effect of 2,2,9,9-tetramethyl-1,10-decanediol (CI-720) on blood, egg yolk, and excreta cholesterol, blood triglycerides, feed consumption, egg production, and egg component weights.” 1976. Web. 09 Aug 2020.

Vancouver:

Reynnells RD. Effect of 2,2,9,9-tetramethyl-1,10-decanediol (CI-720) on blood, egg yolk, and excreta cholesterol, blood triglycerides, feed consumption, egg production, and egg component weights. [Internet] [Masters thesis]. Michigan State University; 1976. [cited 2020 Aug 09]. Available from: http://etd.lib.msu.edu/islandora/object/etd:11695.

Council of Science Editors:

Reynnells RD. Effect of 2,2,9,9-tetramethyl-1,10-decanediol (CI-720) on blood, egg yolk, and excreta cholesterol, blood triglycerides, feed consumption, egg production, and egg component weights. [Masters Thesis]. Michigan State University; 1976. Available from: http://etd.lib.msu.edu/islandora/object/etd:11695


Michigan State University

28. Nyhof, Richard Arlan. Hemodynamic effects of intra-arterial infusion of ouabain in the canine forelimb.

Degree: MS, Dept. of Physiology, 1976, Michigan State University

Subjects/Keywords: Cardiovascular agents; Glycosides

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APA (6th Edition):

Nyhof, R. A. (1976). Hemodynamic effects of intra-arterial infusion of ouabain in the canine forelimb. (Masters Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:12602

Chicago Manual of Style (16th Edition):

Nyhof, Richard Arlan. “Hemodynamic effects of intra-arterial infusion of ouabain in the canine forelimb.” 1976. Masters Thesis, Michigan State University. Accessed August 09, 2020. http://etd.lib.msu.edu/islandora/object/etd:12602.

MLA Handbook (7th Edition):

Nyhof, Richard Arlan. “Hemodynamic effects of intra-arterial infusion of ouabain in the canine forelimb.” 1976. Web. 09 Aug 2020.

Vancouver:

Nyhof RA. Hemodynamic effects of intra-arterial infusion of ouabain in the canine forelimb. [Internet] [Masters thesis]. Michigan State University; 1976. [cited 2020 Aug 09]. Available from: http://etd.lib.msu.edu/islandora/object/etd:12602.

Council of Science Editors:

Nyhof RA. Hemodynamic effects of intra-arterial infusion of ouabain in the canine forelimb. [Masters Thesis]. Michigan State University; 1976. Available from: http://etd.lib.msu.edu/islandora/object/etd:12602


Brock University

29. Boyd, Ryan. Expression and function of endothelin and its receptors in vascular adventitial fibroblasts .

Degree: Applied Health Sciences Program, 2007, Brock University

 Objective: The adventitia has been recognized to play important roles in vascular oxidative stress, remodelling and contraction. We recently demonstrated that adventitial fibroblasts are able… (more)

Subjects/Keywords: Endothelins; Cardiovascular agents.

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APA (6th Edition):

Boyd, R. (2007). Expression and function of endothelin and its receptors in vascular adventitial fibroblasts . (Thesis). Brock University. Retrieved from http://hdl.handle.net/10464/1528

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boyd, Ryan. “Expression and function of endothelin and its receptors in vascular adventitial fibroblasts .” 2007. Thesis, Brock University. Accessed August 09, 2020. http://hdl.handle.net/10464/1528.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boyd, Ryan. “Expression and function of endothelin and its receptors in vascular adventitial fibroblasts .” 2007. Web. 09 Aug 2020.

Vancouver:

Boyd R. Expression and function of endothelin and its receptors in vascular adventitial fibroblasts . [Internet] [Thesis]. Brock University; 2007. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/10464/1528.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boyd R. Expression and function of endothelin and its receptors in vascular adventitial fibroblasts . [Thesis]. Brock University; 2007. Available from: http://hdl.handle.net/10464/1528

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of British Columbia

30. Moldowan, Mervin John. Cardiovascular response to beta-hydroxy thujaplicin and gamma-thujaplicin.

Degree: 1970, University of British Columbia

 An Investigation was undertaken to determine the effects of beta-hydroxy thujaplicin and gamma-thujaplicin, used as the sodium salts, on blood pressure and heart rate in… (more)

Subjects/Keywords: Cardiovascular agents; Cardiovascular pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moldowan, M. J. (1970). Cardiovascular response to beta-hydroxy thujaplicin and gamma-thujaplicin. (Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/34714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Moldowan, Mervin John. “Cardiovascular response to beta-hydroxy thujaplicin and gamma-thujaplicin. ” 1970. Thesis, University of British Columbia. Accessed August 09, 2020. http://hdl.handle.net/2429/34714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Moldowan, Mervin John. “Cardiovascular response to beta-hydroxy thujaplicin and gamma-thujaplicin. ” 1970. Web. 09 Aug 2020.

Vancouver:

Moldowan MJ. Cardiovascular response to beta-hydroxy thujaplicin and gamma-thujaplicin. [Internet] [Thesis]. University of British Columbia; 1970. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/2429/34714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Moldowan MJ. Cardiovascular response to beta-hydroxy thujaplicin and gamma-thujaplicin. [Thesis]. University of British Columbia; 1970. Available from: http://hdl.handle.net/2429/34714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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