Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Sp1). Showing records 1 – 30 of 74 total matches.

[1] [2] [3]

Search Limiters

Last 2 Years | English Only

▼ Search Limiters


NSYSU

1. Huang, Chia-wen. Correlated expression of TSG101 and Sp1 in cervical intraepithelial neoplasia.

Degree: Master, Biological Sciences, 2008, NSYSU

 Human tumor susceptibility gene 101, TSG101, exhibits a variety of functions including protein sorting, vesicular trafficking, and regulation of transcription, epithelial growth and differentiation. The… (more)

Subjects/Keywords: dysplasia; TSG101; Sp1; CIN

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Huang, C. (2008). Correlated expression of TSG101 and Sp1 in cervical intraepithelial neoplasia. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825108-115834

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Chia-wen. “Correlated expression of TSG101 and Sp1 in cervical intraepithelial neoplasia.” 2008. Thesis, NSYSU. Accessed April 13, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825108-115834.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Chia-wen. “Correlated expression of TSG101 and Sp1 in cervical intraepithelial neoplasia.” 2008. Web. 13 Apr 2021.

Vancouver:

Huang C. Correlated expression of TSG101 and Sp1 in cervical intraepithelial neoplasia. [Internet] [Thesis]. NSYSU; 2008. [cited 2021 Apr 13]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825108-115834.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang C. Correlated expression of TSG101 and Sp1 in cervical intraepithelial neoplasia. [Thesis]. NSYSU; 2008. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0825108-115834

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

2. Chuang, Chun-Wei. Study of the molecular mechanism by which COX-2 regulates CCR7 expression.

Degree: Master, Institute of Biomedical Sciences, 2010, NSYSU

 The metastatic spread of tumor cells is the major lethal aspect of cancer, and lymphatic metastasis is one of the most important routes. Recent studies… (more)

Subjects/Keywords: Sp1; CCR7; AKT; COX-2

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chuang, C. (2010). Study of the molecular mechanism by which COX-2 regulates CCR7 expression. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chuang, Chun-Wei. “Study of the molecular mechanism by which COX-2 regulates CCR7 expression.” 2010. Thesis, NSYSU. Accessed April 13, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chuang, Chun-Wei. “Study of the molecular mechanism by which COX-2 regulates CCR7 expression.” 2010. Web. 13 Apr 2021.

Vancouver:

Chuang C. Study of the molecular mechanism by which COX-2 regulates CCR7 expression. [Internet] [Thesis]. NSYSU; 2010. [cited 2021 Apr 13]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chuang C. Study of the molecular mechanism by which COX-2 regulates CCR7 expression. [Thesis]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

3. Cole, Laura Kathleen. Insights into the Transcriptional Regulation and Physiological Importance of Phosphatidylethanolamine N-Methyltransferase.

Degree: PhD, Department of Biochemistry, 2010, University of Alberta

 Phosphatidylcholine (PC) is made in all nucleated mammalian cells via the CDP-choline pathway. Another major pathway for PC biosynthesis in liver is catalyzed by phosphatidylethanolamine… (more)

Subjects/Keywords: Tamoxifen; Sp1; Phosphatidylcholine; Cardiac Dysfunction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cole, L. K. (2010). Insights into the Transcriptional Regulation and Physiological Importance of Phosphatidylethanolamine N-Methyltransferase. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/m900nt98v

Chicago Manual of Style (16th Edition):

Cole, Laura Kathleen. “Insights into the Transcriptional Regulation and Physiological Importance of Phosphatidylethanolamine N-Methyltransferase.” 2010. Doctoral Dissertation, University of Alberta. Accessed April 13, 2021. https://era.library.ualberta.ca/files/m900nt98v.

MLA Handbook (7th Edition):

Cole, Laura Kathleen. “Insights into the Transcriptional Regulation and Physiological Importance of Phosphatidylethanolamine N-Methyltransferase.” 2010. Web. 13 Apr 2021.

Vancouver:

Cole LK. Insights into the Transcriptional Regulation and Physiological Importance of Phosphatidylethanolamine N-Methyltransferase. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2021 Apr 13]. Available from: https://era.library.ualberta.ca/files/m900nt98v.

Council of Science Editors:

Cole LK. Insights into the Transcriptional Regulation and Physiological Importance of Phosphatidylethanolamine N-Methyltransferase. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/m900nt98v


Addis Ababa University

4. Leta, Hora. The effects of mega flow SP1 superplasticizing admixture on the properties of concrete .

Degree: 2014, Addis Ababa University

 Superplasticizing admixtures interact chemically with the ingredients of concrete and affect the concrete properties in the fresh and hardened state. Now a days, its possible… (more)

Subjects/Keywords: Superplasticizing; mega flow SP1; admixture; concrete

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Leta, H. (2014). The effects of mega flow SP1 superplasticizing admixture on the properties of concrete . (Thesis). Addis Ababa University. Retrieved from http://etd.aau.edu.et/dspace/handle/123456789/5303

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Leta, Hora. “The effects of mega flow SP1 superplasticizing admixture on the properties of concrete .” 2014. Thesis, Addis Ababa University. Accessed April 13, 2021. http://etd.aau.edu.et/dspace/handle/123456789/5303.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Leta, Hora. “The effects of mega flow SP1 superplasticizing admixture on the properties of concrete .” 2014. Web. 13 Apr 2021.

Vancouver:

Leta H. The effects of mega flow SP1 superplasticizing admixture on the properties of concrete . [Internet] [Thesis]. Addis Ababa University; 2014. [cited 2021 Apr 13]. Available from: http://etd.aau.edu.et/dspace/handle/123456789/5303.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Leta H. The effects of mega flow SP1 superplasticizing admixture on the properties of concrete . [Thesis]. Addis Ababa University; 2014. Available from: http://etd.aau.edu.et/dspace/handle/123456789/5303

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

5. Fletcher, Sally C. Regulation of the base excision repair pathway.

Degree: PhD, 2017, University of Oxford

 Maintenance of genomic stability is paramount for survival of an organism; failure to repair DNA damage ultimately leads to the accumulation of genetically unstable cells… (more)

Subjects/Keywords: 572.8; Base excision repair; XRCC1; Sp1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fletcher, S. C. (2017). Regulation of the base excision repair pathway. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:e3db2385-f9aa-4289-9547-3e37cbeddec9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.757778

Chicago Manual of Style (16th Edition):

Fletcher, Sally C. “Regulation of the base excision repair pathway.” 2017. Doctoral Dissertation, University of Oxford. Accessed April 13, 2021. http://ora.ox.ac.uk/objects/uuid:e3db2385-f9aa-4289-9547-3e37cbeddec9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.757778.

MLA Handbook (7th Edition):

Fletcher, Sally C. “Regulation of the base excision repair pathway.” 2017. Web. 13 Apr 2021.

Vancouver:

Fletcher SC. Regulation of the base excision repair pathway. [Internet] [Doctoral dissertation]. University of Oxford; 2017. [cited 2021 Apr 13]. Available from: http://ora.ox.ac.uk/objects/uuid:e3db2385-f9aa-4289-9547-3e37cbeddec9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.757778.

Council of Science Editors:

Fletcher SC. Regulation of the base excision repair pathway. [Doctoral Dissertation]. University of Oxford; 2017. Available from: http://ora.ox.ac.uk/objects/uuid:e3db2385-f9aa-4289-9547-3e37cbeddec9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.757778


University of Melbourne

6. Seo, Myoung Suk. An investigation of the factors that regulate muscarinic receptor expression in schizophrenia.

Degree: 2013, University of Melbourne

 A previous study identified a group of subjects with schizophrenia, who have, on average, a 75% decrease in their cortical cholinergic receptor, muscarinic 1 (CHRM1).… (more)

Subjects/Keywords: schizophrenia; human; cortex; post-mortem; CHRM1; Sp1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Seo, M. S. (2013). An investigation of the factors that regulate muscarinic receptor expression in schizophrenia. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37992

Chicago Manual of Style (16th Edition):

Seo, Myoung Suk. “An investigation of the factors that regulate muscarinic receptor expression in schizophrenia.” 2013. Doctoral Dissertation, University of Melbourne. Accessed April 13, 2021. http://hdl.handle.net/11343/37992.

MLA Handbook (7th Edition):

Seo, Myoung Suk. “An investigation of the factors that regulate muscarinic receptor expression in schizophrenia.” 2013. Web. 13 Apr 2021.

Vancouver:

Seo MS. An investigation of the factors that regulate muscarinic receptor expression in schizophrenia. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/11343/37992.

Council of Science Editors:

Seo MS. An investigation of the factors that regulate muscarinic receptor expression in schizophrenia. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/37992

7. Σιούτης, Δήμος. Διαταραχές πυελικού εδάφους και ακράτεια ούρων στις μετεμμηνοπαυσιακές γυναίκες: συσχέτιση με τα ενδογενή στεροειδή του φύλου.

Degree: 2013, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Urinary incontinence in women (urinary incontinence, UI) is one of the most common situations leading to deterioration of quality of life. Numerous population studies have… (more)

Subjects/Keywords: Ακράτεια ούρων; Στεροειδή φύλου; Πολυμορφισμός SP1; Κολλαγόνο; Urinary incontinence; Hormone steroids; Polymorphism SP1; Collagen

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Σιούτης, . . (2013). Διαταραχές πυελικού εδάφους και ακράτεια ούρων στις μετεμμηνοπαυσιακές γυναίκες: συσχέτιση με τα ενδογενή στεροειδή του φύλου. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/36839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Σιούτης, Δήμος. “Διαταραχές πυελικού εδάφους και ακράτεια ούρων στις μετεμμηνοπαυσιακές γυναίκες: συσχέτιση με τα ενδογενή στεροειδή του φύλου.” 2013. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 13, 2021. http://hdl.handle.net/10442/hedi/36839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Σιούτης, Δήμος. “Διαταραχές πυελικού εδάφους και ακράτεια ούρων στις μετεμμηνοπαυσιακές γυναίκες: συσχέτιση με τα ενδογενή στεροειδή του φύλου.” 2013. Web. 13 Apr 2021.

Vancouver:

Σιούτης . Διαταραχές πυελικού εδάφους και ακράτεια ούρων στις μετεμμηνοπαυσιακές γυναίκες: συσχέτιση με τα ενδογενή στεροειδή του φύλου. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/10442/hedi/36839.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Σιούτης . Διαταραχές πυελικού εδάφους και ακράτεια ούρων στις μετεμμηνοπαυσιακές γυναίκες: συσχέτιση με τα ενδογενή στεροειδή του φύλου. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. Available from: http://hdl.handle.net/10442/hedi/36839

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

8. Hsu, Shih-Fang. Analysis of promoter activity of TSG101 tumor susceptibility gene 101.

Degree: PhD, Biological Sciences, 2013, NSYSU

 Tumor susceptibility gene, TSG101, participates in regulation of cellular functions such as gene transcription, vesicular trafficking, cell growth and differentiation. The steady-state level of TSG101… (more)

Subjects/Keywords: TSG101; Tumor susceptibility gene; transcription factor; Sp1; promoter

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hsu, S. (2013). Analysis of promoter activity of TSG101 tumor susceptibility gene 101. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0431113-134735

Chicago Manual of Style (16th Edition):

Hsu, Shih-Fang. “Analysis of promoter activity of TSG101 tumor susceptibility gene 101.” 2013. Doctoral Dissertation, NSYSU. Accessed April 13, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0431113-134735.

MLA Handbook (7th Edition):

Hsu, Shih-Fang. “Analysis of promoter activity of TSG101 tumor susceptibility gene 101.” 2013. Web. 13 Apr 2021.

Vancouver:

Hsu S. Analysis of promoter activity of TSG101 tumor susceptibility gene 101. [Internet] [Doctoral dissertation]. NSYSU; 2013. [cited 2021 Apr 13]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0431113-134735.

Council of Science Editors:

Hsu S. Analysis of promoter activity of TSG101 tumor susceptibility gene 101. [Doctoral Dissertation]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0431113-134735

9. Oyanagi, Takahito. Suppression of MUC5AC expression in human bronchial epithelial cells by interferon-γ : インターフェロンγによるヒト気道上皮細胞MUC5AC発現抑制.

Degree: 博士(医学), 2016, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第4212号. 学位記番号: 新大院博(医)甲第711号. 学位授与年月日: 平成28年9月20日

Background: Excessive mucin secretion in the airway is an important feature of airway inflammatory diseases. . MUC5AC… (more)

Subjects/Keywords: MUC5AC; mucin; interferon-ɤ; bronchial epithelial cells; Sp1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Oyanagi, T. (2016). Suppression of MUC5AC expression in human bronchial epithelial cells by interferon-γ : インターフェロンγによるヒト気道上皮細胞MUC5AC発現抑制. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/45098

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oyanagi, Takahito. “Suppression of MUC5AC expression in human bronchial epithelial cells by interferon-γ : インターフェロンγによるヒト気道上皮細胞MUC5AC発現抑制.” 2016. Thesis, Niigata University / 新潟大学. Accessed April 13, 2021. http://hdl.handle.net/10191/45098.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oyanagi, Takahito. “Suppression of MUC5AC expression in human bronchial epithelial cells by interferon-γ : インターフェロンγによるヒト気道上皮細胞MUC5AC発現抑制.” 2016. Web. 13 Apr 2021.

Vancouver:

Oyanagi T. Suppression of MUC5AC expression in human bronchial epithelial cells by interferon-γ : インターフェロンγによるヒト気道上皮細胞MUC5AC発現抑制. [Internet] [Thesis]. Niigata University / 新潟大学; 2016. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/10191/45098.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oyanagi T. Suppression of MUC5AC expression in human bronchial epithelial cells by interferon-γ : インターフェロンγによるヒト気道上皮細胞MUC5AC発現抑制. [Thesis]. Niigata University / 新潟大学; 2016. Available from: http://hdl.handle.net/10191/45098

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

10. Kovačević Grujičić, Nataša R., 1970-. Karakterizacija bazalnog promotora humanog SOX3 gena i identifikacija regulatornih elemenata odgovornih za indukciju SOX3 gena retinoičnom kiselinom.

Degree: Biološki fakultet, 2019, Univerzitet u Beogradu

Биологија - молекуларна биологија / Bbiology - molecular biology

SOX3/Sox3 gen je jedan od najranijih neuralnih markera kod kičmenjaka, koji determiniše neuronalni tip ćelijske diferencijacije.… (more)

Subjects/Keywords: minimal promoter; SOX3; Sp1; USF1; NF-Y; CREB; MAZ; ZBP-89

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kovačević Grujičić, Nataša R., 1. (2019). Karakterizacija bazalnog promotora humanog SOX3 gena i identifikacija regulatornih elemenata odgovornih za indukciju SOX3 gena retinoičnom kiselinom. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:19450/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kovačević Grujičić, Nataša R., 1970-. “Karakterizacija bazalnog promotora humanog SOX3 gena i identifikacija regulatornih elemenata odgovornih za indukciju SOX3 gena retinoičnom kiselinom.” 2019. Thesis, Univerzitet u Beogradu. Accessed April 13, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:19450/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kovačević Grujičić, Nataša R., 1970-. “Karakterizacija bazalnog promotora humanog SOX3 gena i identifikacija regulatornih elemenata odgovornih za indukciju SOX3 gena retinoičnom kiselinom.” 2019. Web. 13 Apr 2021.

Vancouver:

Kovačević Grujičić, Nataša R. 1. Karakterizacija bazalnog promotora humanog SOX3 gena i identifikacija regulatornih elemenata odgovornih za indukciju SOX3 gena retinoičnom kiselinom. [Internet] [Thesis]. Univerzitet u Beogradu; 2019. [cited 2021 Apr 13]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:19450/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kovačević Grujičić, Nataša R. 1. Karakterizacija bazalnog promotora humanog SOX3 gena i identifikacija regulatornih elemenata odgovornih za indukciju SOX3 gena retinoičnom kiselinom. [Thesis]. Univerzitet u Beogradu; 2019. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:19450/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Gefroh, Amanda. Epigenetic Patterns: Process-Driven Or Sequence-Driven?.

Degree: MS, Biomedical Sciences, 2013, University of North Dakota

  Some of the most rapidly-evolving fields of study in the medical and research communities presently are those which investigate our genetic code, the elements… (more)

Subjects/Keywords: enhancer; epigenetics; histone modification; promoter; Simian virus 40; Sp1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gefroh, A. (2013). Epigenetic Patterns: Process-Driven Or Sequence-Driven?. (Masters Thesis). University of North Dakota. Retrieved from https://commons.und.edu/theses/1423

Chicago Manual of Style (16th Edition):

Gefroh, Amanda. “Epigenetic Patterns: Process-Driven Or Sequence-Driven?.” 2013. Masters Thesis, University of North Dakota. Accessed April 13, 2021. https://commons.und.edu/theses/1423.

MLA Handbook (7th Edition):

Gefroh, Amanda. “Epigenetic Patterns: Process-Driven Or Sequence-Driven?.” 2013. Web. 13 Apr 2021.

Vancouver:

Gefroh A. Epigenetic Patterns: Process-Driven Or Sequence-Driven?. [Internet] [Masters thesis]. University of North Dakota; 2013. [cited 2021 Apr 13]. Available from: https://commons.und.edu/theses/1423.

Council of Science Editors:

Gefroh A. Epigenetic Patterns: Process-Driven Or Sequence-Driven?. [Masters Thesis]. University of North Dakota; 2013. Available from: https://commons.und.edu/theses/1423


University of Minnesota

12. Dauer, Patricia. Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer.

Degree: PhD, Pharmacology, 2018, University of Minnesota

 Pancreatic ductal adenocarcinoma (PDAC) ranks among the poorest prognoses for cancer patients, with an estimated 5-year survival of just 8%. The stagnant survival rates are… (more)

Subjects/Keywords: Chemoresistance; Endoplasmic Reticulum Stress; GRP78; Pancreatic Cancer; SP1; Tumor microenvironment

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dauer, P. (2018). Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/198397

Chicago Manual of Style (16th Edition):

Dauer, Patricia. “Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer.” 2018. Doctoral Dissertation, University of Minnesota. Accessed April 13, 2021. http://hdl.handle.net/11299/198397.

MLA Handbook (7th Edition):

Dauer, Patricia. “Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer.” 2018. Web. 13 Apr 2021.

Vancouver:

Dauer P. Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer. [Internet] [Doctoral dissertation]. University of Minnesota; 2018. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/11299/198397.

Council of Science Editors:

Dauer P. Endoplasmic Reticulum Stress-Mediated Signaling in Pancreatic Cancer. [Doctoral Dissertation]. University of Minnesota; 2018. Available from: http://hdl.handle.net/11299/198397

13. Kim, KyoungHyun. Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells.

Degree: PhD, Toxicology, 2005, Texas A&M University

 Estrogen Receptor ? (ER?)/Sp1 activation of GC-rich gene promoters in breast cancer cells is dependent, in part, on the activation function 1 (AF1) of ER?.… (more)

Subjects/Keywords: Estrogen receptor; Sp1; antiestrogens

…3.4 ERα/Sp1-mediated transactivation in cells transfected with hERα zinc finger point… …ERE- or ERα/SP1-mediated transactivation..................................... 3.6 Activation… …of hERα/SP1 by E2, 4OHT, and ICI does not require AF2-helix 12-coactivator interactions… …Himge (DEF) region for activation of hERα/Sp1 by estrogen and antiestrogens… …3.9 Screening of putative coactivators of ERα/Sp1 ................... 3.10 Detection of… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kim, K. (2005). Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/2666

Chicago Manual of Style (16th Edition):

Kim, KyoungHyun. “Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells.” 2005. Doctoral Dissertation, Texas A&M University. Accessed April 13, 2021. http://hdl.handle.net/1969.1/2666.

MLA Handbook (7th Edition):

Kim, KyoungHyun. “Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells.” 2005. Web. 13 Apr 2021.

Vancouver:

Kim K. Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells. [Internet] [Doctoral dissertation]. Texas A&M University; 2005. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1969.1/2666.

Council of Science Editors:

Kim K. Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells. [Doctoral Dissertation]. Texas A&M University; 2005. Available from: http://hdl.handle.net/1969.1/2666


University of Gothenburg / Göteborgs Universitet

14. Siaw, Joachim Tetteh. Anaplastic lymphoma kinase activity, a therapeutic target, suppresses neuroblastoma cell differentiation.

Degree: 2020, University of Gothenburg / Göteborgs Universitet

 Neuroblastoma (NB) is the most common extracranial pediatric solid malignancy caused by the failed differentiation of precursor cells of the developing sympathetic nervous system. NB… (more)

Subjects/Keywords: Neuroblastoma; ALK; 11q.; DLG2; SP1; differentiation; crizotinib; ceritinib; brigatinib

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Siaw, J. T. (2020). Anaplastic lymphoma kinase activity, a therapeutic target, suppresses neuroblastoma cell differentiation. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/67088

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Siaw, Joachim Tetteh. “Anaplastic lymphoma kinase activity, a therapeutic target, suppresses neuroblastoma cell differentiation.” 2020. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed April 13, 2021. http://hdl.handle.net/2077/67088.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Siaw, Joachim Tetteh. “Anaplastic lymphoma kinase activity, a therapeutic target, suppresses neuroblastoma cell differentiation.” 2020. Web. 13 Apr 2021.

Vancouver:

Siaw JT. Anaplastic lymphoma kinase activity, a therapeutic target, suppresses neuroblastoma cell differentiation. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2020. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/2077/67088.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Siaw JT. Anaplastic lymphoma kinase activity, a therapeutic target, suppresses neuroblastoma cell differentiation. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2020. Available from: http://hdl.handle.net/2077/67088

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Mehmood, Tahir. Unraveling molecular, cellular and cognitive defects in the mouse model for mental retardation caused by Rsk2 gene mutation : Identification des déficits moléculaires, cellulaires et cognitifs chez le modèle souris du retard mental causé par la mutation du gène Rsk2.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2012, Université de Strasbourg

Le syndrome de Coffin-Lowry (CLS), une déficience intellectuelle liée à l'X, est causée par des mutations du gène RPS6KA3 codant pour la kinase RSK2 régulée… (more)

Subjects/Keywords: Syndrome de Coffin-Lowry; RSK2; Gria2; Récepteur glutamate AMPA; ERK; CREB; PC12; Sp1; Coffin-Lowry Syndrome; RSK2; Gria2; Glutamate receptor; ERK; CREB; PC12; Sp1; 572.8; 572.6

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mehmood, T. (2012). Unraveling molecular, cellular and cognitive defects in the mouse model for mental retardation caused by Rsk2 gene mutation : Identification des déficits moléculaires, cellulaires et cognitifs chez le modèle souris du retard mental causé par la mutation du gène Rsk2. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2012STRAJ112

Chicago Manual of Style (16th Edition):

Mehmood, Tahir. “Unraveling molecular, cellular and cognitive defects in the mouse model for mental retardation caused by Rsk2 gene mutation : Identification des déficits moléculaires, cellulaires et cognitifs chez le modèle souris du retard mental causé par la mutation du gène Rsk2.” 2012. Doctoral Dissertation, Université de Strasbourg. Accessed April 13, 2021. http://www.theses.fr/2012STRAJ112.

MLA Handbook (7th Edition):

Mehmood, Tahir. “Unraveling molecular, cellular and cognitive defects in the mouse model for mental retardation caused by Rsk2 gene mutation : Identification des déficits moléculaires, cellulaires et cognitifs chez le modèle souris du retard mental causé par la mutation du gène Rsk2.” 2012. Web. 13 Apr 2021.

Vancouver:

Mehmood T. Unraveling molecular, cellular and cognitive defects in the mouse model for mental retardation caused by Rsk2 gene mutation : Identification des déficits moléculaires, cellulaires et cognitifs chez le modèle souris du retard mental causé par la mutation du gène Rsk2. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2012. [cited 2021 Apr 13]. Available from: http://www.theses.fr/2012STRAJ112.

Council of Science Editors:

Mehmood T. Unraveling molecular, cellular and cognitive defects in the mouse model for mental retardation caused by Rsk2 gene mutation : Identification des déficits moléculaires, cellulaires et cognitifs chez le modèle souris du retard mental causé par la mutation du gène Rsk2. [Doctoral Dissertation]. Université de Strasbourg; 2012. Available from: http://www.theses.fr/2012STRAJ112


Leiden University

16. Zhang, Q. Total synthesis of alginate and zwitterionic SP1 oligosaccharides.

Degree: 2018, Leiden University

 The work in this thesis has been focused on two subjects. The first is the assembly of alginate oligosaccharides and the generation of building blocks… (more)

Subjects/Keywords: Alginate; Zwitterionic SP1; Oligosaccharides; Synthesis; Vaccine; Alginate; Zwitterionic SP1; Oligosaccharides; Synthesis; Vaccine

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, Q. (2018). Total synthesis of alginate and zwitterionic SP1 oligosaccharides. (Doctoral Dissertation). Leiden University. Retrieved from http://hdl.handle.net/1887/65053

Chicago Manual of Style (16th Edition):

Zhang, Q. “Total synthesis of alginate and zwitterionic SP1 oligosaccharides.” 2018. Doctoral Dissertation, Leiden University. Accessed April 13, 2021. http://hdl.handle.net/1887/65053.

MLA Handbook (7th Edition):

Zhang, Q. “Total synthesis of alginate and zwitterionic SP1 oligosaccharides.” 2018. Web. 13 Apr 2021.

Vancouver:

Zhang Q. Total synthesis of alginate and zwitterionic SP1 oligosaccharides. [Internet] [Doctoral dissertation]. Leiden University; 2018. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1887/65053.

Council of Science Editors:

Zhang Q. Total synthesis of alginate and zwitterionic SP1 oligosaccharides. [Doctoral Dissertation]. Leiden University; 2018. Available from: http://hdl.handle.net/1887/65053

17. 富永, 哲郎. Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整.

Degree: 博士(医学), 2016, Nagasaki University / 長崎大学

 Background: It has been shown that epidermal growth factor receptor (EGFR) mutation status is associated with 5-fluorouracil (5-FU) sensitivity in non-small-cell lung cancer (NSCLC). However,… (more)

Subjects/Keywords: Non-small-cell lung cancer; Sp1; Dihydropyrimidine dehydrogenase; Epidermal growth factor receptor mutation; 5-fluorouracil

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

富永, . (2016). Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/36831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

富永, 哲郎. “Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整.” 2016. Thesis, Nagasaki University / 長崎大学. Accessed April 13, 2021. http://hdl.handle.net/10069/36831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

富永, 哲郎. “Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整.” 2016. Web. 13 Apr 2021.

Vancouver:

富永 . Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2016. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/10069/36831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

富永 . Epidermal growth factor signals regulate dihydropyrimidine dehydrogenase expression in EGFR-mutated non-small-cell lung cancer : EGFR変異型非小細胞肺癌におけるEGFシグナルのDPD発現調整. [Thesis]. Nagasaki University / 長崎大学; 2016. Available from: http://hdl.handle.net/10069/36831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

18. Rodriguez-Malave, Norma Iris. The Role of Long Non-coding RNAs in B-acute Lymphoblastic Leukemia.

Degree: Cellular & Molecular Pathology, 2015, UCLA

 Non-coding RNAs play pivotal roles in a wide variety of molecular processes. The functions of long non-coding RNAs (lncRNAs), in particular, have deep implications for… (more)

Subjects/Keywords: Immunology; Molecular biology; Pathology; B-ALL; B-cell; lncRNAs; MLL; RNA; SP1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rodriguez-Malave, N. I. (2015). The Role of Long Non-coding RNAs in B-acute Lymphoblastic Leukemia. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/7vb1n706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodriguez-Malave, Norma Iris. “The Role of Long Non-coding RNAs in B-acute Lymphoblastic Leukemia.” 2015. Thesis, UCLA. Accessed April 13, 2021. http://www.escholarship.org/uc/item/7vb1n706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodriguez-Malave, Norma Iris. “The Role of Long Non-coding RNAs in B-acute Lymphoblastic Leukemia.” 2015. Web. 13 Apr 2021.

Vancouver:

Rodriguez-Malave NI. The Role of Long Non-coding RNAs in B-acute Lymphoblastic Leukemia. [Internet] [Thesis]. UCLA; 2015. [cited 2021 Apr 13]. Available from: http://www.escholarship.org/uc/item/7vb1n706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodriguez-Malave NI. The Role of Long Non-coding RNAs in B-acute Lymphoblastic Leukemia. [Thesis]. UCLA; 2015. Available from: http://www.escholarship.org/uc/item/7vb1n706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Λογοθέτη, Στυλιανή. Μελέτη του ρόλου των p73 ισομορφών στον καρκίνο του πνεύμονα.

Degree: 2011, University of Crete (UOC); Πανεπιστήμιο Κρήτης

The p73 gene possesses an extrinsic P1 promoter and an intrinsic P2 promoter, resulting in synthesis of the full-length TAp73 and the N-terminal-truncated ΔNp73 isoforms,… (more)

Subjects/Keywords: Μεθυλίωση DNA; Καρκίνος πνεύμονα; ΔΝp73; p73; Sp1; DNA methylation; Lung cancer; TAp73 γ

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Λογοθέτη, . . (2011). Μελέτη του ρόλου των p73 ισομορφών στον καρκίνο του πνεύμονα. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/24877

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Λογοθέτη, Στυλιανή. “Μελέτη του ρόλου των p73 ισομορφών στον καρκίνο του πνεύμονα.” 2011. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed April 13, 2021. http://hdl.handle.net/10442/hedi/24877.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Λογοθέτη, Στυλιανή. “Μελέτη του ρόλου των p73 ισομορφών στον καρκίνο του πνεύμονα.” 2011. Web. 13 Apr 2021.

Vancouver:

Λογοθέτη . Μελέτη του ρόλου των p73 ισομορφών στον καρκίνο του πνεύμονα. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/10442/hedi/24877.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Λογοθέτη . Μελέτη του ρόλου των p73 ισομορφών στον καρκίνο του πνεύμονα. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2011. Available from: http://hdl.handle.net/10442/hedi/24877

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

20. Su, Shengzhong. Transcriptional regulation of the human microsomal epoxide hydrolase gene (EPHX1) driven by a far upstream alternative promoter.

Degree: 2013, Penn State University

 Microsomal epoxide hydrolase (mEH) is an important metabolizing enzyme that plays roles in both detoxification and bioactivation of xenobiotics. Human mEH gene expression is subjected… (more)

Subjects/Keywords: Microsomal epoxide hydrolase; Nrf2; Xenobiotic metabolism; Bioactivation; Lung cancer; Sp1; Sp3; AhR

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Su, S. (2013). Transcriptional regulation of the human microsomal epoxide hydrolase gene (EPHX1) driven by a far upstream alternative promoter. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/18909

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Su, Shengzhong. “Transcriptional regulation of the human microsomal epoxide hydrolase gene (EPHX1) driven by a far upstream alternative promoter.” 2013. Thesis, Penn State University. Accessed April 13, 2021. https://submit-etda.libraries.psu.edu/catalog/18909.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Su, Shengzhong. “Transcriptional regulation of the human microsomal epoxide hydrolase gene (EPHX1) driven by a far upstream alternative promoter.” 2013. Web. 13 Apr 2021.

Vancouver:

Su S. Transcriptional regulation of the human microsomal epoxide hydrolase gene (EPHX1) driven by a far upstream alternative promoter. [Internet] [Thesis]. Penn State University; 2013. [cited 2021 Apr 13]. Available from: https://submit-etda.libraries.psu.edu/catalog/18909.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Su S. Transcriptional regulation of the human microsomal epoxide hydrolase gene (EPHX1) driven by a far upstream alternative promoter. [Thesis]. Penn State University; 2013. Available from: https://submit-etda.libraries.psu.edu/catalog/18909

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ghana

21. Enu-Kwesi, M.A. The Operationalization of the Ecowas Protocol on Democracy and Good Governance (A/Sp1/12/01): The Case of Togo’s Political Crisis (2005-18) .

Degree: 2018, University of Ghana

 The Economic Community of West African States (ECOWAS) was established on the 28th of May, 1975 with the primary objective of promoting economic integration and… (more)

Subjects/Keywords: Ecowas Protocol; Democracy; Good Governance (A/Sp1/12/01); Togo’s Political Crisis (2005-18)

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Enu-Kwesi, M. A. (2018). The Operationalization of the Ecowas Protocol on Democracy and Good Governance (A/Sp1/12/01): The Case of Togo’s Political Crisis (2005-18) . (Masters Thesis). University of Ghana. Retrieved from http://ugspace.ug.edu.gh/handle/123456789/26539

Chicago Manual of Style (16th Edition):

Enu-Kwesi, M A. “The Operationalization of the Ecowas Protocol on Democracy and Good Governance (A/Sp1/12/01): The Case of Togo’s Political Crisis (2005-18) .” 2018. Masters Thesis, University of Ghana. Accessed April 13, 2021. http://ugspace.ug.edu.gh/handle/123456789/26539.

MLA Handbook (7th Edition):

Enu-Kwesi, M A. “The Operationalization of the Ecowas Protocol on Democracy and Good Governance (A/Sp1/12/01): The Case of Togo’s Political Crisis (2005-18) .” 2018. Web. 13 Apr 2021.

Vancouver:

Enu-Kwesi MA. The Operationalization of the Ecowas Protocol on Democracy and Good Governance (A/Sp1/12/01): The Case of Togo’s Political Crisis (2005-18) . [Internet] [Masters thesis]. University of Ghana; 2018. [cited 2021 Apr 13]. Available from: http://ugspace.ug.edu.gh/handle/123456789/26539.

Council of Science Editors:

Enu-Kwesi MA. The Operationalization of the Ecowas Protocol on Democracy and Good Governance (A/Sp1/12/01): The Case of Togo’s Political Crisis (2005-18) . [Masters Thesis]. University of Ghana; 2018. Available from: http://ugspace.ug.edu.gh/handle/123456789/26539


Freie Universität Berlin

22. Hertel, Johannes. Investigation of molecular mechanisms of transcriptional and epigenetic regulation of the human vegfr-3 gene in cell model.

Degree: 2013, Freie Universität Berlin

 Introduction: Angiogenesis, the formation of new blood vessels from endothelial precursors, is a prerequisite for growth and dissemination of solid malignancies. Multiple studies found a… (more)

Subjects/Keywords: vegfr-3; lymphangiogenesis; Sp1; Sp3; epigenetics; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hertel, J. (2013). Investigation of molecular mechanisms of transcriptional and epigenetic regulation of the human vegfr-3 gene in cell model. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/13305

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hertel, Johannes. “Investigation of molecular mechanisms of transcriptional and epigenetic regulation of the human vegfr-3 gene in cell model.” 2013. Thesis, Freie Universität Berlin. Accessed April 13, 2021. https://refubium.fu-berlin.de/handle/fub188/13305.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hertel, Johannes. “Investigation of molecular mechanisms of transcriptional and epigenetic regulation of the human vegfr-3 gene in cell model.” 2013. Web. 13 Apr 2021.

Vancouver:

Hertel J. Investigation of molecular mechanisms of transcriptional and epigenetic regulation of the human vegfr-3 gene in cell model. [Internet] [Thesis]. Freie Universität Berlin; 2013. [cited 2021 Apr 13]. Available from: https://refubium.fu-berlin.de/handle/fub188/13305.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hertel J. Investigation of molecular mechanisms of transcriptional and epigenetic regulation of the human vegfr-3 gene in cell model. [Thesis]. Freie Universität Berlin; 2013. Available from: https://refubium.fu-berlin.de/handle/fub188/13305

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Bat-Erdene, Ariunzaya. Synergistic targeting of Sp1, a critical transcription factor for myeloma cell growth and survival, by panobinostat and proteasome inhibitors : パノビノスタットとプロテアソーム阻害薬は骨髄腫細胞の増殖と生存に必須の転写因子Sp1を相乗的に標的にする.

Degree: 博士(医学), 2016, Tokushima University / 徳島大学

Subjects/Keywords: multiple myeloma; panobinostat; proteasome inhibitors; caspase-8; Sp1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bat-Erdene, A. (2016). Synergistic targeting of Sp1, a critical transcription factor for myeloma cell growth and survival, by panobinostat and proteasome inhibitors : パノビノスタットとプロテアソーム阻害薬は骨髄腫細胞の増殖と生存に必須の転写因子Sp1を相乗的に標的にする. (Thesis). Tokushima University / 徳島大学. Retrieved from http://repo.lib.tokushima-u.ac.jp/109988

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bat-Erdene, Ariunzaya. “Synergistic targeting of Sp1, a critical transcription factor for myeloma cell growth and survival, by panobinostat and proteasome inhibitors : パノビノスタットとプロテアソーム阻害薬は骨髄腫細胞の増殖と生存に必須の転写因子Sp1を相乗的に標的にする.” 2016. Thesis, Tokushima University / 徳島大学. Accessed April 13, 2021. http://repo.lib.tokushima-u.ac.jp/109988.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bat-Erdene, Ariunzaya. “Synergistic targeting of Sp1, a critical transcription factor for myeloma cell growth and survival, by panobinostat and proteasome inhibitors : パノビノスタットとプロテアソーム阻害薬は骨髄腫細胞の増殖と生存に必須の転写因子Sp1を相乗的に標的にする.” 2016. Web. 13 Apr 2021.

Vancouver:

Bat-Erdene A. Synergistic targeting of Sp1, a critical transcription factor for myeloma cell growth and survival, by panobinostat and proteasome inhibitors : パノビノスタットとプロテアソーム阻害薬は骨髄腫細胞の増殖と生存に必須の転写因子Sp1を相乗的に標的にする. [Internet] [Thesis]. Tokushima University / 徳島大学; 2016. [cited 2021 Apr 13]. Available from: http://repo.lib.tokushima-u.ac.jp/109988.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bat-Erdene A. Synergistic targeting of Sp1, a critical transcription factor for myeloma cell growth and survival, by panobinostat and proteasome inhibitors : パノビノスタットとプロテアソーム阻害薬は骨髄腫細胞の増殖と生存に必須の転写因子Sp1を相乗的に標的にする. [Thesis]. Tokushima University / 徳島大学; 2016. Available from: http://repo.lib.tokushima-u.ac.jp/109988

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

24. Trinh, Bon Q. THE ROLE AND MECHANISM OF THE HOMEOBOX GENE DLX4 IN TRANSFORMING GROWTH FACTOR-B RESISTANCE IN CANCER.

Degree: PhD, 2011, Texas Medical Center

  Transforming growth factor-b (TGF-b) is a cytokine that plays essential roles in regulating embryonic development and tissue homeostasis. In normal cells, TGF-b exerts an… (more)

Subjects/Keywords: cell growth; homeobox genes; Smad; Sp1; TGF-b; Biochemistry; Cancer Biology; Cell Biology; Developmental Biology; Molecular Biology; Molecular Genetics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Trinh, B. Q. (2011). THE ROLE AND MECHANISM OF THE HOMEOBOX GENE DLX4 IN TRANSFORMING GROWTH FACTOR-B RESISTANCE IN CANCER. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/119

Chicago Manual of Style (16th Edition):

Trinh, Bon Q. “THE ROLE AND MECHANISM OF THE HOMEOBOX GENE DLX4 IN TRANSFORMING GROWTH FACTOR-B RESISTANCE IN CANCER.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed April 13, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/119.

MLA Handbook (7th Edition):

Trinh, Bon Q. “THE ROLE AND MECHANISM OF THE HOMEOBOX GENE DLX4 IN TRANSFORMING GROWTH FACTOR-B RESISTANCE IN CANCER.” 2011. Web. 13 Apr 2021.

Vancouver:

Trinh BQ. THE ROLE AND MECHANISM OF THE HOMEOBOX GENE DLX4 IN TRANSFORMING GROWTH FACTOR-B RESISTANCE IN CANCER. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2021 Apr 13]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/119.

Council of Science Editors:

Trinh BQ. THE ROLE AND MECHANISM OF THE HOMEOBOX GENE DLX4 IN TRANSFORMING GROWTH FACTOR-B RESISTANCE IN CANCER. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/119


Texas Medical Center

25. Jia, Zhiliang. TARGETED INHIBITION OF SP1 TRANSCRIPTION FACTOR AND ANTI-ANGIOGENESIS OF HUMAN PANCREATIC CANCER.

Degree: PhD, 2010, Texas Medical Center

  Transcription factor Specificity Protein 1(Sp1) is reported to be essential for vascular endothelial growth factor (VEGF) constitutive expression in human pancreatic adenocarcinoma. The definitive… (more)

Subjects/Keywords: Sp1; Pancreatic Cancer; Angiogenesis; Mithramycin; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jia, Z. (2010). TARGETED INHIBITION OF SP1 TRANSCRIPTION FACTOR AND ANTI-ANGIOGENESIS OF HUMAN PANCREATIC CANCER. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/3

Chicago Manual of Style (16th Edition):

Jia, Zhiliang. “TARGETED INHIBITION OF SP1 TRANSCRIPTION FACTOR AND ANTI-ANGIOGENESIS OF HUMAN PANCREATIC CANCER.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed April 13, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/3.

MLA Handbook (7th Edition):

Jia, Zhiliang. “TARGETED INHIBITION OF SP1 TRANSCRIPTION FACTOR AND ANTI-ANGIOGENESIS OF HUMAN PANCREATIC CANCER.” 2010. Web. 13 Apr 2021.

Vancouver:

Jia Z. TARGETED INHIBITION OF SP1 TRANSCRIPTION FACTOR AND ANTI-ANGIOGENESIS OF HUMAN PANCREATIC CANCER. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2021 Apr 13]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/3.

Council of Science Editors:

Jia Z. TARGETED INHIBITION OF SP1 TRANSCRIPTION FACTOR AND ANTI-ANGIOGENESIS OF HUMAN PANCREATIC CANCER. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/3


NSYSU

26. Hsu, Ya-Wen. Epithelial membrane protein 2 inhibits cell proliferation and induces apoptosis via TGFB-SP1 axis and recruitment of P2RX7 in urinary bladder urothelial carcinoma-derived cells.

Degree: Master, Institute of Biomedical Sciences, 2018, NSYSU

 In this study, our objective was to investigate the mechanisms of Epithelial membrane protein 2 (EMP2) inhibits cell proliferation and induces apoptosis in urinary bladder… (more)

Subjects/Keywords: SP1 (Specificity protein-1); TGFB; Epithelial membrane protein 2 (EMP2); Urinary bladder urothelial carcinoma (UBUC); Cell cycle arrest

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hsu, Y. (2018). Epithelial membrane protein 2 inhibits cell proliferation and induces apoptosis via TGFB-SP1 axis and recruitment of P2RX7 in urinary bladder urothelial carcinoma-derived cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0611118-123519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hsu, Ya-Wen. “Epithelial membrane protein 2 inhibits cell proliferation and induces apoptosis via TGFB-SP1 axis and recruitment of P2RX7 in urinary bladder urothelial carcinoma-derived cells.” 2018. Thesis, NSYSU. Accessed April 13, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0611118-123519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hsu, Ya-Wen. “Epithelial membrane protein 2 inhibits cell proliferation and induces apoptosis via TGFB-SP1 axis and recruitment of P2RX7 in urinary bladder urothelial carcinoma-derived cells.” 2018. Web. 13 Apr 2021.

Vancouver:

Hsu Y. Epithelial membrane protein 2 inhibits cell proliferation and induces apoptosis via TGFB-SP1 axis and recruitment of P2RX7 in urinary bladder urothelial carcinoma-derived cells. [Internet] [Thesis]. NSYSU; 2018. [cited 2021 Apr 13]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0611118-123519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hsu Y. Epithelial membrane protein 2 inhibits cell proliferation and induces apoptosis via TGFB-SP1 axis and recruitment of P2RX7 in urinary bladder urothelial carcinoma-derived cells. [Thesis]. NSYSU; 2018. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0611118-123519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

27. Busia, Laura. Die anti-tumorigene Wirkung des Progesteron Rezeptor Antagonist Lonaprisan (ZK230211) in Brustkrebs.

Degree: 2011, Freie Universität Berlin

 Die Entwicklung der Brustdrüse beginnt in der Embryogenese und setzt sich während der Pubertät als Antwort auf die Stimulierung durch Estrogene und Wachstumshormone fort. Eine… (more)

Subjects/Keywords: Progesterone receptor; Lonaprisan; p21; Cell cycle arrest; Sp1; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::572 Biochemie

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Busia, L. (2011). Die anti-tumorigene Wirkung des Progesteron Rezeptor Antagonist Lonaprisan (ZK230211) in Brustkrebs. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-4323

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Busia, Laura. “Die anti-tumorigene Wirkung des Progesteron Rezeptor Antagonist Lonaprisan (ZK230211) in Brustkrebs.” 2011. Thesis, Freie Universität Berlin. Accessed April 13, 2021. http://dx.doi.org/10.17169/refubium-4323.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Busia, Laura. “Die anti-tumorigene Wirkung des Progesteron Rezeptor Antagonist Lonaprisan (ZK230211) in Brustkrebs.” 2011. Web. 13 Apr 2021.

Vancouver:

Busia L. Die anti-tumorigene Wirkung des Progesteron Rezeptor Antagonist Lonaprisan (ZK230211) in Brustkrebs. [Internet] [Thesis]. Freie Universität Berlin; 2011. [cited 2021 Apr 13]. Available from: http://dx.doi.org/10.17169/refubium-4323.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Busia L. Die anti-tumorigene Wirkung des Progesteron Rezeptor Antagonist Lonaprisan (ZK230211) in Brustkrebs. [Thesis]. Freie Universität Berlin; 2011. Available from: http://dx.doi.org/10.17169/refubium-4323

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

28. Pohlabeln, Kerstin. Analysis of the methylation status of the human iodothyronine deiodinase type 1 promotor in thyroid carcinomas.

Degree: 2010, Freie Universität Berlin

 As a key enzyme in thyoid hormone metabolism the human type I iodothyronine deiodinase (5´D1) catalyzes the activation of the prohormone T4 to the biologically… (more)

Subjects/Keywords: Typ 1 Deiodase; methylation; thyroid carcinoma; thyroid response element (DR+12); Sp1; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pohlabeln, K. (2010). Analysis of the methylation status of the human iodothyronine deiodinase type 1 promotor in thyroid carcinomas. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-8975

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pohlabeln, Kerstin. “Analysis of the methylation status of the human iodothyronine deiodinase type 1 promotor in thyroid carcinomas.” 2010. Thesis, Freie Universität Berlin. Accessed April 13, 2021. http://dx.doi.org/10.17169/refubium-8975.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pohlabeln, Kerstin. “Analysis of the methylation status of the human iodothyronine deiodinase type 1 promotor in thyroid carcinomas.” 2010. Web. 13 Apr 2021.

Vancouver:

Pohlabeln K. Analysis of the methylation status of the human iodothyronine deiodinase type 1 promotor in thyroid carcinomas. [Internet] [Thesis]. Freie Universität Berlin; 2010. [cited 2021 Apr 13]. Available from: http://dx.doi.org/10.17169/refubium-8975.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pohlabeln K. Analysis of the methylation status of the human iodothyronine deiodinase type 1 promotor in thyroid carcinomas. [Thesis]. Freie Universität Berlin; 2010. Available from: http://dx.doi.org/10.17169/refubium-8975

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

29. Convissar, Scott M. Role of GATA4, GATA6, and SP1 on Luteal Function and LH Receptor Expression.

Degree: 2018, University of Illinois – Chicago

 Infertility is defined by the inability to achieve pregnancy after 1 year of trying to conceive. According to the most recent report from the Center… (more)

Subjects/Keywords: GATA; corpus luteum; luteal; progesterone; steroidogenesis; LHR; progesterone; SP1; GDF9; BMP15; AMH; granulosa; reproduction; ovary; mRNA

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Convissar, S. M. (2018). Role of GATA4, GATA6, and SP1 on Luteal Function and LH Receptor Expression. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/23039

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Convissar, Scott M. “Role of GATA4, GATA6, and SP1 on Luteal Function and LH Receptor Expression.” 2018. Thesis, University of Illinois – Chicago. Accessed April 13, 2021. http://hdl.handle.net/10027/23039.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Convissar, Scott M. “Role of GATA4, GATA6, and SP1 on Luteal Function and LH Receptor Expression.” 2018. Web. 13 Apr 2021.

Vancouver:

Convissar SM. Role of GATA4, GATA6, and SP1 on Luteal Function and LH Receptor Expression. [Internet] [Thesis]. University of Illinois – Chicago; 2018. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/10027/23039.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Convissar SM. Role of GATA4, GATA6, and SP1 on Luteal Function and LH Receptor Expression. [Thesis]. University of Illinois – Chicago; 2018. Available from: http://hdl.handle.net/10027/23039

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

30. Larsson, Lena. Functional analysis of the -1087 single nucleotide polymorphism in the IL-10 promoter region.

Degree: 2010, University of Gothenburg / Göteborgs Universitet

 Interleukin (IL) 10 is recognized as a pro-inflammatory cytokine that promotes B cell proliferation. The objectives of the present series of studies were to analyze… (more)

Subjects/Keywords: IL-10; B cells; Sp1; transcription factors; gene expression

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Larsson, L. (2010). Functional analysis of the -1087 single nucleotide polymorphism in the IL-10 promoter region. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/22927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Larsson, Lena. “Functional analysis of the -1087 single nucleotide polymorphism in the IL-10 promoter region.” 2010. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed April 13, 2021. http://hdl.handle.net/2077/22927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Larsson, Lena. “Functional analysis of the -1087 single nucleotide polymorphism in the IL-10 promoter region.” 2010. Web. 13 Apr 2021.

Vancouver:

Larsson L. Functional analysis of the -1087 single nucleotide polymorphism in the IL-10 promoter region. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2010. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/2077/22927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Larsson L. Functional analysis of the -1087 single nucleotide polymorphism in the IL-10 promoter region. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2010. Available from: http://hdl.handle.net/2077/22927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3]

.