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You searched for subject:(Smooth muscle Physiology). Showing records 1 – 30 of 51 total matches.

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Virginia Commonwealth University

1. Al-Shboul, Othman. Differential regulation of MLC20 phosphorylation in tonic and phasic smooth muscles of the stomach.

Degree: PhD, Physiology, 2011, Virginia Commonwealth University

 Gastrointestinal (GI) smooth muscle possesses distinct regional and functional properties that distinguish it from other types of visceral and vascular smooth muscle. On the basis… (more)

Subjects/Keywords: smooth muscle; signaling; phasic; tonic; stomach; Life Sciences; Physiology

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APA (6th Edition):

Al-Shboul, O. (2011). Differential regulation of MLC20 phosphorylation in tonic and phasic smooth muscles of the stomach. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/ATMF-GA72 ; https://scholarscompass.vcu.edu/etd/2362

Chicago Manual of Style (16th Edition):

Al-Shboul, Othman. “Differential regulation of MLC20 phosphorylation in tonic and phasic smooth muscles of the stomach.” 2011. Doctoral Dissertation, Virginia Commonwealth University. Accessed February 17, 2020. https://doi.org/10.25772/ATMF-GA72 ; https://scholarscompass.vcu.edu/etd/2362.

MLA Handbook (7th Edition):

Al-Shboul, Othman. “Differential regulation of MLC20 phosphorylation in tonic and phasic smooth muscles of the stomach.” 2011. Web. 17 Feb 2020.

Vancouver:

Al-Shboul O. Differential regulation of MLC20 phosphorylation in tonic and phasic smooth muscles of the stomach. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2011. [cited 2020 Feb 17]. Available from: https://doi.org/10.25772/ATMF-GA72 ; https://scholarscompass.vcu.edu/etd/2362.

Council of Science Editors:

Al-Shboul O. Differential regulation of MLC20 phosphorylation in tonic and phasic smooth muscles of the stomach. [Doctoral Dissertation]. Virginia Commonwealth University; 2011. Available from: https://doi.org/10.25772/ATMF-GA72 ; https://scholarscompass.vcu.edu/etd/2362


Virginia Commonwealth University

2. Alqudah, Mohammad. Characterization of the neurotrophic factor Brain-Derived Neurotrophic Factor (BDNF) in intestinal smooth muscle cells.

Degree: PhD, Physiology, 2013, Virginia Commonwealth University

 Brain-derived neurotrophic factor (BDNF) belongs to the neurotrophin family of secreted proteins, which include in addition to BDNF, nerve growth factor (NGF) and neurotrophin 3-6… (more)

Subjects/Keywords: BDNF; Longitudinal smooth muscle; Rabbit intestine; Life Sciences; Physiology

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APA (6th Edition):

Alqudah, M. (2013). Characterization of the neurotrophic factor Brain-Derived Neurotrophic Factor (BDNF) in intestinal smooth muscle cells. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/2ZGD-SS06 ; https://scholarscompass.vcu.edu/etd/3095

Chicago Manual of Style (16th Edition):

Alqudah, Mohammad. “Characterization of the neurotrophic factor Brain-Derived Neurotrophic Factor (BDNF) in intestinal smooth muscle cells.” 2013. Doctoral Dissertation, Virginia Commonwealth University. Accessed February 17, 2020. https://doi.org/10.25772/2ZGD-SS06 ; https://scholarscompass.vcu.edu/etd/3095.

MLA Handbook (7th Edition):

Alqudah, Mohammad. “Characterization of the neurotrophic factor Brain-Derived Neurotrophic Factor (BDNF) in intestinal smooth muscle cells.” 2013. Web. 17 Feb 2020.

Vancouver:

Alqudah M. Characterization of the neurotrophic factor Brain-Derived Neurotrophic Factor (BDNF) in intestinal smooth muscle cells. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2013. [cited 2020 Feb 17]. Available from: https://doi.org/10.25772/2ZGD-SS06 ; https://scholarscompass.vcu.edu/etd/3095.

Council of Science Editors:

Alqudah M. Characterization of the neurotrophic factor Brain-Derived Neurotrophic Factor (BDNF) in intestinal smooth muscle cells. [Doctoral Dissertation]. Virginia Commonwealth University; 2013. Available from: https://doi.org/10.25772/2ZGD-SS06 ; https://scholarscompass.vcu.edu/etd/3095


University of Gothenburg / Göteborgs Universitet

3. Jonsson, Olof, 1941-. Extracellular osmolality and vascular smooth muscle activity.

Degree: 1970, University of Gothenburg / Göteborgs Universitet

Subjects/Keywords: Muscle; smooth: physiology Portal vein: physiology

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APA (6th Edition):

Jonsson, Olof, 1. (1970). Extracellular osmolality and vascular smooth muscle activity. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/12078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jonsson, Olof, 1941-. “Extracellular osmolality and vascular smooth muscle activity.” 1970. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed February 17, 2020. http://hdl.handle.net/2077/12078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jonsson, Olof, 1941-. “Extracellular osmolality and vascular smooth muscle activity.” 1970. Web. 17 Feb 2020.

Vancouver:

Jonsson, Olof 1. Extracellular osmolality and vascular smooth muscle activity. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 1970. [cited 2020 Feb 17]. Available from: http://hdl.handle.net/2077/12078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jonsson, Olof 1. Extracellular osmolality and vascular smooth muscle activity. [Thesis]. University of Gothenburg / Göteborgs Universitet; 1970. Available from: http://hdl.handle.net/2077/12078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

4. Sommerville, Laura Jean. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.

Degree: PhD, 2010, Temple University

Molecular and Cellular Physiology

The underlying cause of all vascular proliferative diseases is injury-induced activation of vascular endothelium and vascular smooth muscle cells (VSMC). Activated… (more)

Subjects/Keywords: Biology, Physiology; Allograft Inflammatory Factor-1; Atherosclerosis; Smooth Muscle Cell Activation; Vascular Proliferative Disease

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APA (6th Edition):

Sommerville, L. J. (2010). The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,76756

Chicago Manual of Style (16th Edition):

Sommerville, Laura Jean. “The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.” 2010. Doctoral Dissertation, Temple University. Accessed February 17, 2020. http://digital.library.temple.edu/u?/p245801coll10,76756.

MLA Handbook (7th Edition):

Sommerville, Laura Jean. “The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.” 2010. Web. 17 Feb 2020.

Vancouver:

Sommerville LJ. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Feb 17]. Available from: http://digital.library.temple.edu/u?/p245801coll10,76756.

Council of Science Editors:

Sommerville LJ. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,76756


University of Alberta

5. Ren, Jun. Characterization of the delayed rectifier K channel in rat tail artery vascular smooth muscle cells.

Degree: PhD, Department of Physiology, 1995, University of Alberta

Subjects/Keywords: Rats – Physiology.; Vascular smooth muscle.; Electric current rectifiers.; Ion channels – Physiology.; Muscle cells.

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APA (6th Edition):

Ren, J. (1995). Characterization of the delayed rectifier K channel in rat tail artery vascular smooth muscle cells. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/zw12z767s

Chicago Manual of Style (16th Edition):

Ren, Jun. “Characterization of the delayed rectifier K channel in rat tail artery vascular smooth muscle cells.” 1995. Doctoral Dissertation, University of Alberta. Accessed February 17, 2020. https://era.library.ualberta.ca/files/zw12z767s.

MLA Handbook (7th Edition):

Ren, Jun. “Characterization of the delayed rectifier K channel in rat tail artery vascular smooth muscle cells.” 1995. Web. 17 Feb 2020.

Vancouver:

Ren J. Characterization of the delayed rectifier K channel in rat tail artery vascular smooth muscle cells. [Internet] [Doctoral dissertation]. University of Alberta; 1995. [cited 2020 Feb 17]. Available from: https://era.library.ualberta.ca/files/zw12z767s.

Council of Science Editors:

Ren J. Characterization of the delayed rectifier K channel in rat tail artery vascular smooth muscle cells. [Doctoral Dissertation]. University of Alberta; 1995. Available from: https://era.library.ualberta.ca/files/zw12z767s


University of Hong Kong

6. Keung, Wen-yee, Wendy. Modulation of vascular responses by non-genomic actions of 17{221}-estradiol.

Degree: PhD, 2005, University of Hong Kong

abstract

published_or_final_version

Pharmacology

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Man, RYK.

Subjects/Keywords: Blood-vessels - Pathophysiology; Vascular smooth muscle.; Estradiol.; Rats - Physiology.

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APA (6th Edition):

Keung, Wen-yee, W. (2005). Modulation of vascular responses by non-genomic actions of 17{221}-estradiol. (Doctoral Dissertation). University of Hong Kong. Retrieved from Keung, W. W. [姜韻兒]. (2005). Modulation of vascular responses by non-genomic actions of 17β-estradiol. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3199631 ; http://dx.doi.org/10.5353/th_b3199631 ; http://hdl.handle.net/10722/40936

Chicago Manual of Style (16th Edition):

Keung, Wen-yee, Wendy. “Modulation of vascular responses by non-genomic actions of 17{221}-estradiol.” 2005. Doctoral Dissertation, University of Hong Kong. Accessed February 17, 2020. Keung, W. W. [姜韻兒]. (2005). Modulation of vascular responses by non-genomic actions of 17β-estradiol. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3199631 ; http://dx.doi.org/10.5353/th_b3199631 ; http://hdl.handle.net/10722/40936.

MLA Handbook (7th Edition):

Keung, Wen-yee, Wendy. “Modulation of vascular responses by non-genomic actions of 17{221}-estradiol.” 2005. Web. 17 Feb 2020.

Vancouver:

Keung, Wen-yee W. Modulation of vascular responses by non-genomic actions of 17{221}-estradiol. [Internet] [Doctoral dissertation]. University of Hong Kong; 2005. [cited 2020 Feb 17]. Available from: Keung, W. W. [姜韻兒]. (2005). Modulation of vascular responses by non-genomic actions of 17β-estradiol. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3199631 ; http://dx.doi.org/10.5353/th_b3199631 ; http://hdl.handle.net/10722/40936.

Council of Science Editors:

Keung, Wen-yee W. Modulation of vascular responses by non-genomic actions of 17{221}-estradiol. [Doctoral Dissertation]. University of Hong Kong; 2005. Available from: Keung, W. W. [姜韻兒]. (2005). Modulation of vascular responses by non-genomic actions of 17β-estradiol. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3199631 ; http://dx.doi.org/10.5353/th_b3199631 ; http://hdl.handle.net/10722/40936


University of Florida

7. Chen, Bei. The Impact of Aging and Exercise Training on the Coronary Circulation.

Degree: PhD, Medical Sciences - Physiology and Pharmacology (IDP), 2014, University of Florida

 The risk for diastolic heart failure increases with advancing age. Regular physical exercise prevents and/or ameliorates the progression of agerelated diastolic dysfunction; however, the underlying… (more)

Subjects/Keywords: Arteries; Arterioles; Blood flow; Exercise; Heart; Microvessels; Physiology; Rats; Smooth muscle; Vasodilation; age  – circulation  – coronary  – exercise

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APA (6th Edition):

Chen, B. (2014). The Impact of Aging and Exercise Training on the Coronary Circulation. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0046550

Chicago Manual of Style (16th Edition):

Chen, Bei. “The Impact of Aging and Exercise Training on the Coronary Circulation.” 2014. Doctoral Dissertation, University of Florida. Accessed February 17, 2020. http://ufdc.ufl.edu/UFE0046550.

MLA Handbook (7th Edition):

Chen, Bei. “The Impact of Aging and Exercise Training on the Coronary Circulation.” 2014. Web. 17 Feb 2020.

Vancouver:

Chen B. The Impact of Aging and Exercise Training on the Coronary Circulation. [Internet] [Doctoral dissertation]. University of Florida; 2014. [cited 2020 Feb 17]. Available from: http://ufdc.ufl.edu/UFE0046550.

Council of Science Editors:

Chen B. The Impact of Aging and Exercise Training on the Coronary Circulation. [Doctoral Dissertation]. University of Florida; 2014. Available from: http://ufdc.ufl.edu/UFE0046550


University of Western Ontario

8. Ghoreishi, Sina A. Nicotinamide Riboside and the Aortic Response to Angiotensin II Infusion in Mice.

Degree: 2017, University of Western Ontario

 Damage to vascular cells of the aorta drives vascular dysfunction and disease. Nicotinamide adenine dinucleotide (NAD+) is a cellular metabolite critical to cellular health, but… (more)

Subjects/Keywords: NAD+; nicotinamide riboside; angiotensin II; aorta; endothelial cells; vascular smooth muscle cells; Circulatory and Respiratory Physiology

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APA (6th Edition):

Ghoreishi, S. A. (2017). Nicotinamide Riboside and the Aortic Response to Angiotensin II Infusion in Mice. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4788

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ghoreishi, Sina A. “Nicotinamide Riboside and the Aortic Response to Angiotensin II Infusion in Mice.” 2017. Thesis, University of Western Ontario. Accessed February 17, 2020. https://ir.lib.uwo.ca/etd/4788.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ghoreishi, Sina A. “Nicotinamide Riboside and the Aortic Response to Angiotensin II Infusion in Mice.” 2017. Web. 17 Feb 2020.

Vancouver:

Ghoreishi SA. Nicotinamide Riboside and the Aortic Response to Angiotensin II Infusion in Mice. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2020 Feb 17]. Available from: https://ir.lib.uwo.ca/etd/4788.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ghoreishi SA. Nicotinamide Riboside and the Aortic Response to Angiotensin II Infusion in Mice. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4788

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

9. Xu, Yiwen. Automated Vascular Smooth Muscle Segmentation, Reconstruction, Classification and Simulation on Whole-Slide Histology.

Degree: 2017, University of Western Ontario

 Histology of the microvasculature depicts detailed characteristics relevant to tissue perfusion. One important histologic feature is the smooth muscle component of the microvessel wall, which… (more)

Subjects/Keywords: Microvasculature; Regeneration; Vascular Smooth Muscle; Whole-slide Imaging; Histology; Immunohistochemistry; Reconstruction; Simulation; Segmentation; Machine Learning; Circulatory and Respiratory Physiology; Medical Biophysics

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APA (6th Edition):

Xu, Y. (2017). Automated Vascular Smooth Muscle Segmentation, Reconstruction, Classification and Simulation on Whole-Slide Histology. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4566

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xu, Yiwen. “Automated Vascular Smooth Muscle Segmentation, Reconstruction, Classification and Simulation on Whole-Slide Histology.” 2017. Thesis, University of Western Ontario. Accessed February 17, 2020. https://ir.lib.uwo.ca/etd/4566.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xu, Yiwen. “Automated Vascular Smooth Muscle Segmentation, Reconstruction, Classification and Simulation on Whole-Slide Histology.” 2017. Web. 17 Feb 2020.

Vancouver:

Xu Y. Automated Vascular Smooth Muscle Segmentation, Reconstruction, Classification and Simulation on Whole-Slide Histology. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2020 Feb 17]. Available from: https://ir.lib.uwo.ca/etd/4566.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xu Y. Automated Vascular Smooth Muscle Segmentation, Reconstruction, Classification and Simulation on Whole-Slide Histology. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4566

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

10. Chen, Hong-I. Physiological and pharmacological studies of lower urinary tract smooth muscles.

Degree: PhD, 1990, University of Oxford

 The basic mechanisms regulating the function of the bladder and urethra, under both normal and abnormal conditions, are still incompletely understood, and consequently, many treatments… (more)

Subjects/Keywords: 615.1; Smooth muscle; Physiology; Urinary organs; Diseases

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APA (6th Edition):

Chen, H. (1990). Physiological and pharmacological studies of lower urinary tract smooth muscles. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:e73d80a0-9cfd-4959-bfc7-cd2576fcc1dc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.257735

Chicago Manual of Style (16th Edition):

Chen, Hong-I. “Physiological and pharmacological studies of lower urinary tract smooth muscles.” 1990. Doctoral Dissertation, University of Oxford. Accessed February 17, 2020. http://ora.ox.ac.uk/objects/uuid:e73d80a0-9cfd-4959-bfc7-cd2576fcc1dc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.257735.

MLA Handbook (7th Edition):

Chen, Hong-I. “Physiological and pharmacological studies of lower urinary tract smooth muscles.” 1990. Web. 17 Feb 2020.

Vancouver:

Chen H. Physiological and pharmacological studies of lower urinary tract smooth muscles. [Internet] [Doctoral dissertation]. University of Oxford; 1990. [cited 2020 Feb 17]. Available from: http://ora.ox.ac.uk/objects/uuid:e73d80a0-9cfd-4959-bfc7-cd2576fcc1dc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.257735.

Council of Science Editors:

Chen H. Physiological and pharmacological studies of lower urinary tract smooth muscles. [Doctoral Dissertation]. University of Oxford; 1990. Available from: http://ora.ox.ac.uk/objects/uuid:e73d80a0-9cfd-4959-bfc7-cd2576fcc1dc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.257735


Simon Fraser University

11. Gong, Bang Qiang. Mitogenic factors in coronary vascular smooth muscle proliferation in salmonids.

Degree: 1994, Simon Fraser University

Subjects/Keywords: Salmon.; Mitogens.; Fishes  – Physiology.; Myocardium.; Vascular smooth muscle.

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APA (6th Edition):

Gong, B. Q. (1994). Mitogenic factors in coronary vascular smooth muscle proliferation in salmonids. (Thesis). Simon Fraser University. Retrieved from http://summit.sfu.ca/item/6515

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gong, Bang Qiang. “Mitogenic factors in coronary vascular smooth muscle proliferation in salmonids.” 1994. Thesis, Simon Fraser University. Accessed February 17, 2020. http://summit.sfu.ca/item/6515.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gong, Bang Qiang. “Mitogenic factors in coronary vascular smooth muscle proliferation in salmonids.” 1994. Web. 17 Feb 2020.

Vancouver:

Gong BQ. Mitogenic factors in coronary vascular smooth muscle proliferation in salmonids. [Internet] [Thesis]. Simon Fraser University; 1994. [cited 2020 Feb 17]. Available from: http://summit.sfu.ca/item/6515.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gong BQ. Mitogenic factors in coronary vascular smooth muscle proliferation in salmonids. [Thesis]. Simon Fraser University; 1994. Available from: http://summit.sfu.ca/item/6515

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

12. Hernandez, Jeremy M. ASPECTS OF AIRWAY STRETCH-ACTIVATED CONTRACTIONS ASSESSED IN PERFUSED INTACT BOVINE BRONCHIAL SEGMENTS.

Degree: PhD, 2011, McMaster University

Asthma is a disease characterized by transient airway smooth muscle contraction leading to episodes of reversible airway narrowing. It affects over 300 million people… (more)

Subjects/Keywords: asthma; airway smooth muscle contraction; deep inspiration; bronchoconstriction; airway stretch; airway hyperresponsiveness; Circulatory and Respiratory Physiology; Medical Physiology; Physiological Processes; Respiratory Tract Diseases; Circulatory and Respiratory Physiology

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APA (6th Edition):

Hernandez, J. M. (2011). ASPECTS OF AIRWAY STRETCH-ACTIVATED CONTRACTIONS ASSESSED IN PERFUSED INTACT BOVINE BRONCHIAL SEGMENTS. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/11524

Chicago Manual of Style (16th Edition):

Hernandez, Jeremy M. “ASPECTS OF AIRWAY STRETCH-ACTIVATED CONTRACTIONS ASSESSED IN PERFUSED INTACT BOVINE BRONCHIAL SEGMENTS.” 2011. Doctoral Dissertation, McMaster University. Accessed February 17, 2020. http://hdl.handle.net/11375/11524.

MLA Handbook (7th Edition):

Hernandez, Jeremy M. “ASPECTS OF AIRWAY STRETCH-ACTIVATED CONTRACTIONS ASSESSED IN PERFUSED INTACT BOVINE BRONCHIAL SEGMENTS.” 2011. Web. 17 Feb 2020.

Vancouver:

Hernandez JM. ASPECTS OF AIRWAY STRETCH-ACTIVATED CONTRACTIONS ASSESSED IN PERFUSED INTACT BOVINE BRONCHIAL SEGMENTS. [Internet] [Doctoral dissertation]. McMaster University; 2011. [cited 2020 Feb 17]. Available from: http://hdl.handle.net/11375/11524.

Council of Science Editors:

Hernandez JM. ASPECTS OF AIRWAY STRETCH-ACTIVATED CONTRACTIONS ASSESSED IN PERFUSED INTACT BOVINE BRONCHIAL SEGMENTS. [Doctoral Dissertation]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/11524


IUPUI

13. McKenney, Mikaela Lee. Coronary artery disease progression and calcification in metabolic syndrome.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

For years, the leading killer of Americans has been coronary artery disease (CAD), which has a strong correlation to the… (more)

Subjects/Keywords: coronary artery disease; metabolic syndrome; calcification; coronary smooth muscle; calcium; Ossabaw swine; Coronary heart disease  – Molecular aspects  – Research; Heart  – Diseases  – Research; Metabolic syndrome  – Research; Calcification; Calcium  – Metabolism  – Regulation; Calcium  – Physiological effect; Vascular smooth muscle  – Physiology  – Diseases; Cardiovascular system  – Physiology  – Diseases; Smooth muscle  – Pathophysiology; Swine as laboratory animals  – Pathophysiology; Obesity  – Animal models; Obesity  – United States; Tomography, Emission  – Research

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APA (6th Edition):

McKenney, M. L. (2014). Coronary artery disease progression and calcification in metabolic syndrome. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/6460

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McKenney, Mikaela Lee. “Coronary artery disease progression and calcification in metabolic syndrome.” 2014. Thesis, IUPUI. Accessed February 17, 2020. http://hdl.handle.net/1805/6460.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McKenney, Mikaela Lee. “Coronary artery disease progression and calcification in metabolic syndrome.” 2014. Web. 17 Feb 2020.

Vancouver:

McKenney ML. Coronary artery disease progression and calcification in metabolic syndrome. [Internet] [Thesis]. IUPUI; 2014. [cited 2020 Feb 17]. Available from: http://hdl.handle.net/1805/6460.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McKenney ML. Coronary artery disease progression and calcification in metabolic syndrome. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/6460

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

14. Humber, Brent T. INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING.

Degree: MSc, 2011, McMaster University

Introduction Asthma is a chronic respiratory disease that is becoming more prevalent. Airway hyperresponsivness, a key feature of asthma, involves increased narrowing of the… (more)

Subjects/Keywords: asthma; airway smooth muscle; contraction; airway hyperresponsiveness; src kinase; store-operated calcium entry; Circulatory and Respiratory Physiology; Medical Molecular Biology; Circulatory and Respiratory Physiology

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APA (6th Edition):

Humber, B. T. (2011). INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/11744

Chicago Manual of Style (16th Edition):

Humber, Brent T. “INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING.” 2011. Masters Thesis, McMaster University. Accessed February 17, 2020. http://hdl.handle.net/11375/11744.

MLA Handbook (7th Edition):

Humber, Brent T. “INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING.” 2011. Web. 17 Feb 2020.

Vancouver:

Humber BT. INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING. [Internet] [Masters thesis]. McMaster University; 2011. [cited 2020 Feb 17]. Available from: http://hdl.handle.net/11375/11744.

Council of Science Editors:

Humber BT. INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING. [Masters Thesis]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/11744


University of Hong Kong

15. Shi, Yi. Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes.

Degree: PhD, 2006, University of Hong Kong

published_or_final_version

abstract

Pharmacology

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Vanhoutte, PMGR, Man, RYK.

Subjects/Keywords: Smooth muscle - Physiology.; Endothelium.; Smooth muscle - Effect of drugs on.; Diabetes - Complications - Animal models.

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APA (6th Edition):

Shi, Y. (2006). Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. (Doctoral Dissertation). University of Hong Kong. Retrieved from Shi, Y. [史懿]. (2006). Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3828607 ; http://dx.doi.org/10.5353/th_b3828607 ; http://hdl.handle.net/10722/52322

Chicago Manual of Style (16th Edition):

Shi, Yi. “Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes.” 2006. Doctoral Dissertation, University of Hong Kong. Accessed February 17, 2020. Shi, Y. [史懿]. (2006). Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3828607 ; http://dx.doi.org/10.5353/th_b3828607 ; http://hdl.handle.net/10722/52322.

MLA Handbook (7th Edition):

Shi, Yi. “Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes.” 2006. Web. 17 Feb 2020.

Vancouver:

Shi Y. Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. [Internet] [Doctoral dissertation]. University of Hong Kong; 2006. [cited 2020 Feb 17]. Available from: Shi, Y. [史懿]. (2006). Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3828607 ; http://dx.doi.org/10.5353/th_b3828607 ; http://hdl.handle.net/10722/52322.

Council of Science Editors:

Shi Y. Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. [Doctoral Dissertation]. University of Hong Kong; 2006. Available from: Shi, Y. [史懿]. (2006). Endothelial dysfunction and changes in vascular smooth muscle responsiveness in femoral arteries of rats with type I diabetes. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3828607 ; http://dx.doi.org/10.5353/th_b3828607 ; http://hdl.handle.net/10722/52322


Marshall University

16. Brown-Turner, Dawn Leah. Regulation of Alpha- and Beta-actin Isoforms in the Contracting A7r5 Smooth Muscle Cell.

Degree: 2009, Marshall University

 Two isoforms of actin have been found to be present in A7r5 smooth muscle cells, α- and β-actin. This body of work sets out to… (more)

Subjects/Keywords: <; p>; Smooth muscle - Contraction - Research.<; /p>; <; p>; Smooth muscle.<; /p>;

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APA (6th Edition):

Brown-Turner, D. L. (2009). Regulation of Alpha- and Beta-actin Isoforms in the Contracting A7r5 Smooth Muscle Cell. (Thesis). Marshall University. Retrieved from http://mds.marshall.edu/etd/516

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brown-Turner, Dawn Leah. “Regulation of Alpha- and Beta-actin Isoforms in the Contracting A7r5 Smooth Muscle Cell.” 2009. Thesis, Marshall University. Accessed February 17, 2020. http://mds.marshall.edu/etd/516.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brown-Turner, Dawn Leah. “Regulation of Alpha- and Beta-actin Isoforms in the Contracting A7r5 Smooth Muscle Cell.” 2009. Web. 17 Feb 2020.

Vancouver:

Brown-Turner DL. Regulation of Alpha- and Beta-actin Isoforms in the Contracting A7r5 Smooth Muscle Cell. [Internet] [Thesis]. Marshall University; 2009. [cited 2020 Feb 17]. Available from: http://mds.marshall.edu/etd/516.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brown-Turner DL. Regulation of Alpha- and Beta-actin Isoforms in the Contracting A7r5 Smooth Muscle Cell. [Thesis]. Marshall University; 2009. Available from: http://mds.marshall.edu/etd/516

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

17. Tharp, Darla L., 1980-. Role of the intermediate-conductance Ca²⁺-activated K⁺ channel (K[ca]3.1) in coronary smooth muscle cell phenotypic modulation.

Degree: PhD, 2007, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT AUTHOR'S REQUEST.] Smooth muscle cell phenotypic modulation is characterized by suppression of smooth muscle specific marker gene… (more)

Subjects/Keywords: Calcium-dependent potassium channels  – Physiology; Vascular smooth muscle  – Physiology; Muscle cells  – Physiology; Phenotype; Atherosclerosis  – Prevention; Coronary arteries  – Stenosis  – Relapse  – Prevention

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APA (6th Edition):

Tharp, Darla L., 1. (2007). Role of the intermediate-conductance Ca²⁺-activated K⁺ channel (K[ca]3.1) in coronary smooth muscle cell phenotypic modulation. (Doctoral Dissertation). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/5936

Chicago Manual of Style (16th Edition):

Tharp, Darla L., 1980-. “Role of the intermediate-conductance Ca²⁺-activated K⁺ channel (K[ca]3.1) in coronary smooth muscle cell phenotypic modulation.” 2007. Doctoral Dissertation, University of Missouri – Columbia. Accessed February 17, 2020. https://doi.org/10.32469/10355/5936.

MLA Handbook (7th Edition):

Tharp, Darla L., 1980-. “Role of the intermediate-conductance Ca²⁺-activated K⁺ channel (K[ca]3.1) in coronary smooth muscle cell phenotypic modulation.” 2007. Web. 17 Feb 2020.

Vancouver:

Tharp, Darla L. 1. Role of the intermediate-conductance Ca²⁺-activated K⁺ channel (K[ca]3.1) in coronary smooth muscle cell phenotypic modulation. [Internet] [Doctoral dissertation]. University of Missouri – Columbia; 2007. [cited 2020 Feb 17]. Available from: https://doi.org/10.32469/10355/5936.

Council of Science Editors:

Tharp, Darla L. 1. Role of the intermediate-conductance Ca²⁺-activated K⁺ channel (K[ca]3.1) in coronary smooth muscle cell phenotypic modulation. [Doctoral Dissertation]. University of Missouri – Columbia; 2007. Available from: https://doi.org/10.32469/10355/5936


University of Alberta

18. Muller, Milan J. Adenosine action and cyclic AMP in smooth muscle.

Degree: PhD, Department of Pharmacology, 1985, University of Alberta

Subjects/Keywords: Cyclic adenylic acid.; Adenosine – Physiological effect.; Smooth muscle – Physiology.; Phosphodiesterases.; Adenosine – Receptors.

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APA (6th Edition):

Muller, M. J. (1985). Adenosine action and cyclic AMP in smooth muscle. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/h128nh300

Chicago Manual of Style (16th Edition):

Muller, Milan J. “Adenosine action and cyclic AMP in smooth muscle.” 1985. Doctoral Dissertation, University of Alberta. Accessed February 17, 2020. https://era.library.ualberta.ca/files/h128nh300.

MLA Handbook (7th Edition):

Muller, Milan J. “Adenosine action and cyclic AMP in smooth muscle.” 1985. Web. 17 Feb 2020.

Vancouver:

Muller MJ. Adenosine action and cyclic AMP in smooth muscle. [Internet] [Doctoral dissertation]. University of Alberta; 1985. [cited 2020 Feb 17]. Available from: https://era.library.ualberta.ca/files/h128nh300.

Council of Science Editors:

Muller MJ. Adenosine action and cyclic AMP in smooth muscle. [Doctoral Dissertation]. University of Alberta; 1985. Available from: https://era.library.ualberta.ca/files/h128nh300


East Carolina University

19. Kingsbury, Nathaniel. Fesselin, an intrinsically disordered smooth muscle protein, organizes and stabilizes actin-myosin and myosin.

Degree: 2014, East Carolina University

 Fesselin is an intrinsically disordered protein that is known to bind a large variety of cytoskeletal proteins. The proteins fesselin is known to bind include:… (more)

Subjects/Keywords: Biochemistry; Molecular biology; Actin; Fesselin; Myosin; Rapid kinetics; Synaptopodin-2; Smooth Muscle Myosins – metabolism; Cytoskeletal Proteins – metabolism; Actins – metabolism; Adenosine Triphosphatases – metabolism; Membrane Proteins – physiology; Myosins – metabolism

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APA (6th Edition):

Kingsbury, N. (2014). Fesselin, an intrinsically disordered smooth muscle protein, organizes and stabilizes actin-myosin and myosin. (Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/4663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kingsbury, Nathaniel. “Fesselin, an intrinsically disordered smooth muscle protein, organizes and stabilizes actin-myosin and myosin.” 2014. Thesis, East Carolina University. Accessed February 17, 2020. http://hdl.handle.net/10342/4663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kingsbury, Nathaniel. “Fesselin, an intrinsically disordered smooth muscle protein, organizes and stabilizes actin-myosin and myosin.” 2014. Web. 17 Feb 2020.

Vancouver:

Kingsbury N. Fesselin, an intrinsically disordered smooth muscle protein, organizes and stabilizes actin-myosin and myosin. [Internet] [Thesis]. East Carolina University; 2014. [cited 2020 Feb 17]. Available from: http://hdl.handle.net/10342/4663.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kingsbury N. Fesselin, an intrinsically disordered smooth muscle protein, organizes and stabilizes actin-myosin and myosin. [Thesis]. East Carolina University; 2014. Available from: http://hdl.handle.net/10342/4663

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

20. 葛志東; Ge, Zhidong. Endothelium-dependent hyperpolarization and relaxation of coronary circulationg during cardioplegic arrest of the heart.

Degree: PhD, 2000, University of Hong Kong

published_or_final_version

Surgery

Doctoral

Doctor of Philosophy

Subjects/Keywords: Heart - Surgery.; Vascular endothelium.; Muscle, Smooth, Vascular - Physiology.; Heart Arrest, Induced.

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APA (6th Edition):

葛志東; Ge, Z. (2000). Endothelium-dependent hyperpolarization and relaxation of coronary circulationg during cardioplegic arrest of the heart. (Doctoral Dissertation). University of Hong Kong. Retrieved from Ge, Z. [葛志東]. (2000). Endothelium-dependent hyperpolarization and relaxation of coronary circulationg during cardioplegic arrest of the heart. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124171 ; http://dx.doi.org/10.5353/th_b3124171 ; http://hdl.handle.net/10722/35250

Chicago Manual of Style (16th Edition):

葛志東; Ge, Zhidong. “Endothelium-dependent hyperpolarization and relaxation of coronary circulationg during cardioplegic arrest of the heart.” 2000. Doctoral Dissertation, University of Hong Kong. Accessed February 17, 2020. Ge, Z. [葛志東]. (2000). Endothelium-dependent hyperpolarization and relaxation of coronary circulationg during cardioplegic arrest of the heart. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124171 ; http://dx.doi.org/10.5353/th_b3124171 ; http://hdl.handle.net/10722/35250.

MLA Handbook (7th Edition):

葛志東; Ge, Zhidong. “Endothelium-dependent hyperpolarization and relaxation of coronary circulationg during cardioplegic arrest of the heart.” 2000. Web. 17 Feb 2020.

Vancouver:

葛志東; Ge Z. Endothelium-dependent hyperpolarization and relaxation of coronary circulationg during cardioplegic arrest of the heart. [Internet] [Doctoral dissertation]. University of Hong Kong; 2000. [cited 2020 Feb 17]. Available from: Ge, Z. [葛志東]. (2000). Endothelium-dependent hyperpolarization and relaxation of coronary circulationg during cardioplegic arrest of the heart. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124171 ; http://dx.doi.org/10.5353/th_b3124171 ; http://hdl.handle.net/10722/35250.

Council of Science Editors:

葛志東; Ge Z. Endothelium-dependent hyperpolarization and relaxation of coronary circulationg during cardioplegic arrest of the heart. [Doctoral Dissertation]. University of Hong Kong; 2000. Available from: Ge, Z. [葛志東]. (2000). Endothelium-dependent hyperpolarization and relaxation of coronary circulationg during cardioplegic arrest of the heart. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3124171 ; http://dx.doi.org/10.5353/th_b3124171 ; http://hdl.handle.net/10722/35250


Virginia Commonwealth University

21. May, Alexander T. Identification of Expression and Function of the Glucagon-like Peptide-1 Receptor in Gastrointestinal Smooth Muscle.

Degree: MS, Physiology and Biophysics, 2017, Virginia Commonwealth University

  In response to ingestion of nutrients, enteroendocrine L cells secrete the incretin hormone, glucagon-like peptide-1 (GLP-1), to enhance glucose-dependent insulin release. Therapies related to… (more)

Subjects/Keywords: GLP-1; gastrointestinal smooth muscle; GLP-1R; motility; relaxation; GLP-1 (7-36) amide; Biophysics; Cellular and Molecular Physiology; Digestive System; Endocrinology

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APA (6th Edition):

May, A. T. (2017). Identification of Expression and Function of the Glucagon-like Peptide-1 Receptor in Gastrointestinal Smooth Muscle. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/4886

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

May, Alexander T. “Identification of Expression and Function of the Glucagon-like Peptide-1 Receptor in Gastrointestinal Smooth Muscle.” 2017. Thesis, Virginia Commonwealth University. Accessed February 17, 2020. https://scholarscompass.vcu.edu/etd/4886.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

May, Alexander T. “Identification of Expression and Function of the Glucagon-like Peptide-1 Receptor in Gastrointestinal Smooth Muscle.” 2017. Web. 17 Feb 2020.

Vancouver:

May AT. Identification of Expression and Function of the Glucagon-like Peptide-1 Receptor in Gastrointestinal Smooth Muscle. [Internet] [Thesis]. Virginia Commonwealth University; 2017. [cited 2020 Feb 17]. Available from: https://scholarscompass.vcu.edu/etd/4886.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

May AT. Identification of Expression and Function of the Glucagon-like Peptide-1 Receptor in Gastrointestinal Smooth Muscle. [Thesis]. Virginia Commonwealth University; 2017. Available from: https://scholarscompass.vcu.edu/etd/4886

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

22. Shukla, Juhi. The Expression of Neutrophil Products, Myeloperoxidase and Matrix Metalloproteinase 8, in Systemic Vasculature of Obese and Preeclamptic Women.

Degree: MS, Physiology, 2007, Virginia Commonwealth University

 Evidence shows the activation of neutrophils in the systemic vasculature of obese and preeclamptic women. In this study, I evaluated whether expression of neutrophil products,… (more)

Subjects/Keywords: vascular smooth muscle; hypertension; neutrophil; pregnancy; obesity; preeclampsia; Life Sciences; Physiology

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APA (6th Edition):

Shukla, J. (2007). The Expression of Neutrophil Products, Myeloperoxidase and Matrix Metalloproteinase 8, in Systemic Vasculature of Obese and Preeclamptic Women. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/4QPX-XD68 ; https://scholarscompass.vcu.edu/etd/1261

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shukla, Juhi. “The Expression of Neutrophil Products, Myeloperoxidase and Matrix Metalloproteinase 8, in Systemic Vasculature of Obese and Preeclamptic Women.” 2007. Thesis, Virginia Commonwealth University. Accessed February 17, 2020. https://doi.org/10.25772/4QPX-XD68 ; https://scholarscompass.vcu.edu/etd/1261.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shukla, Juhi. “The Expression of Neutrophil Products, Myeloperoxidase and Matrix Metalloproteinase 8, in Systemic Vasculature of Obese and Preeclamptic Women.” 2007. Web. 17 Feb 2020.

Vancouver:

Shukla J. The Expression of Neutrophil Products, Myeloperoxidase and Matrix Metalloproteinase 8, in Systemic Vasculature of Obese and Preeclamptic Women. [Internet] [Thesis]. Virginia Commonwealth University; 2007. [cited 2020 Feb 17]. Available from: https://doi.org/10.25772/4QPX-XD68 ; https://scholarscompass.vcu.edu/etd/1261.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shukla J. The Expression of Neutrophil Products, Myeloperoxidase and Matrix Metalloproteinase 8, in Systemic Vasculature of Obese and Preeclamptic Women. [Thesis]. Virginia Commonwealth University; 2007. Available from: https://doi.org/10.25772/4QPX-XD68 ; https://scholarscompass.vcu.edu/etd/1261

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

23. Bednarek, Melissa. Adaptation at a Shortened Length in Rabbit Femoral Artery.

Degree: PhD, Physiology, 2009, Virginia Commonwealth University

 It is well known that the overlap between the thick and thin filaments in striated muscle is responsible for the single active length-tension (L-T) curve.… (more)

Subjects/Keywords: length-adaptation; vascular smooth muscle; rabbit femoral artery; Life Sciences; Physiology

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APA (6th Edition):

Bednarek, M. (2009). Adaptation at a Shortened Length in Rabbit Femoral Artery. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/NZWQ-R095 ; https://scholarscompass.vcu.edu/etd/1885

Chicago Manual of Style (16th Edition):

Bednarek, Melissa. “Adaptation at a Shortened Length in Rabbit Femoral Artery.” 2009. Doctoral Dissertation, Virginia Commonwealth University. Accessed February 17, 2020. https://doi.org/10.25772/NZWQ-R095 ; https://scholarscompass.vcu.edu/etd/1885.

MLA Handbook (7th Edition):

Bednarek, Melissa. “Adaptation at a Shortened Length in Rabbit Femoral Artery.” 2009. Web. 17 Feb 2020.

Vancouver:

Bednarek M. Adaptation at a Shortened Length in Rabbit Femoral Artery. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2009. [cited 2020 Feb 17]. Available from: https://doi.org/10.25772/NZWQ-R095 ; https://scholarscompass.vcu.edu/etd/1885.

Council of Science Editors:

Bednarek M. Adaptation at a Shortened Length in Rabbit Femoral Artery. [Doctoral Dissertation]. Virginia Commonwealth University; 2009. Available from: https://doi.org/10.25772/NZWQ-R095 ; https://scholarscompass.vcu.edu/etd/1885


Virginia Commonwealth University

24. Ahmed, Rashad. Signaling Pathways Coupled to Melatonin Receptor MT1 in Gastric Smooth Muscle.

Degree: MS, Physiology, 2010, Virginia Commonwealth University

 The Melatonin, a close derivative of serotonin, is involved in physiological regulation of circadian rhythms. In the gastrointestinal (GI) system, melatonin exhibits endocrine, paracrine and… (more)

Subjects/Keywords: Gastric Motility; GI; Melatonin; Signaling; Smooth Muscle; Life Sciences; Physiology

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APA (6th Edition):

Ahmed, R. (2010). Signaling Pathways Coupled to Melatonin Receptor MT1 in Gastric Smooth Muscle. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/X2MB-6M62 ; https://scholarscompass.vcu.edu/etd/2192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ahmed, Rashad. “Signaling Pathways Coupled to Melatonin Receptor MT1 in Gastric Smooth Muscle.” 2010. Thesis, Virginia Commonwealth University. Accessed February 17, 2020. https://doi.org/10.25772/X2MB-6M62 ; https://scholarscompass.vcu.edu/etd/2192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ahmed, Rashad. “Signaling Pathways Coupled to Melatonin Receptor MT1 in Gastric Smooth Muscle.” 2010. Web. 17 Feb 2020.

Vancouver:

Ahmed R. Signaling Pathways Coupled to Melatonin Receptor MT1 in Gastric Smooth Muscle. [Internet] [Thesis]. Virginia Commonwealth University; 2010. [cited 2020 Feb 17]. Available from: https://doi.org/10.25772/X2MB-6M62 ; https://scholarscompass.vcu.edu/etd/2192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ahmed R. Signaling Pathways Coupled to Melatonin Receptor MT1 in Gastric Smooth Muscle. [Thesis]. Virginia Commonwealth University; 2010. Available from: https://doi.org/10.25772/X2MB-6M62 ; https://scholarscompass.vcu.edu/etd/2192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Belacel Ouari, Milia. Contrôle de la voie de l’AMPc vasculaire par les phosphodiestérases en situation physiopathologique. : Contribution of the phosphodiesterases in the regulation of vascular cAMP in pathophysiological situation.

Degree: Docteur es, Physiologie, physiopathologie, 2016, Université Paris-Saclay (ComUE)

L’AMPc est un second messager exerçant un rôle vasculoprotecteur majeur, par ses effets relaxants et inhibiteurs de la prolifération et de la migration cellulaires. Les… (more)

Subjects/Keywords: Physiologie et physiopathologie vasculaire; Cellule musculaire lisse vasculaire; Signalisation; AMPc; Récepteurs béta-Adrénergiques; Phosphodiestérases; Vascular physiology and pathophysiology; Vascular smooth muscle cell; Signaling; Camp; Beta-Adrenergic receptors; Phosphodiesterases

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APA (6th Edition):

Belacel Ouari, M. (2016). Contrôle de la voie de l’AMPc vasculaire par les phosphodiestérases en situation physiopathologique. : Contribution of the phosphodiesterases in the regulation of vascular cAMP in pathophysiological situation. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2016SACLS504

Chicago Manual of Style (16th Edition):

Belacel Ouari, Milia. “Contrôle de la voie de l’AMPc vasculaire par les phosphodiestérases en situation physiopathologique. : Contribution of the phosphodiesterases in the regulation of vascular cAMP in pathophysiological situation.” 2016. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed February 17, 2020. http://www.theses.fr/2016SACLS504.

MLA Handbook (7th Edition):

Belacel Ouari, Milia. “Contrôle de la voie de l’AMPc vasculaire par les phosphodiestérases en situation physiopathologique. : Contribution of the phosphodiesterases in the regulation of vascular cAMP in pathophysiological situation.” 2016. Web. 17 Feb 2020.

Vancouver:

Belacel Ouari M. Contrôle de la voie de l’AMPc vasculaire par les phosphodiestérases en situation physiopathologique. : Contribution of the phosphodiesterases in the regulation of vascular cAMP in pathophysiological situation. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2016. [cited 2020 Feb 17]. Available from: http://www.theses.fr/2016SACLS504.

Council of Science Editors:

Belacel Ouari M. Contrôle de la voie de l’AMPc vasculaire par les phosphodiestérases en situation physiopathologique. : Contribution of the phosphodiesterases in the regulation of vascular cAMP in pathophysiological situation. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2016. Available from: http://www.theses.fr/2016SACLS504


McGill University

26. Pasternyk, Stephanie Marika, 1983-. Effect of extracellular matrix and mechanical strain on airway smooth muscle.

Degree: MS, Division of Experimental Medicine., 2009, McGill University

 Airway remodeling in asthma includes alterations in extracellular matrix and airway smooth muscle (ASM) mass. For this study, ASM cells were obtained from rats that… (more)

Subjects/Keywords: Asthma  – physiopathology.; Airway Remodeling  – physiology.; Respiratory System  – cytology.; Myocytes, Smooth Muscle  – physiology.; Extracellular Matrix  – physiology.; Stress, Mechanical.

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APA (6th Edition):

Pasternyk, Stephanie Marika, 1. (2009). Effect of extracellular matrix and mechanical strain on airway smooth muscle. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile111560.pdf

Chicago Manual of Style (16th Edition):

Pasternyk, Stephanie Marika, 1983-. “Effect of extracellular matrix and mechanical strain on airway smooth muscle.” 2009. Masters Thesis, McGill University. Accessed February 17, 2020. http://digitool.library.mcgill.ca/thesisfile111560.pdf.

MLA Handbook (7th Edition):

Pasternyk, Stephanie Marika, 1983-. “Effect of extracellular matrix and mechanical strain on airway smooth muscle.” 2009. Web. 17 Feb 2020.

Vancouver:

Pasternyk, Stephanie Marika 1. Effect of extracellular matrix and mechanical strain on airway smooth muscle. [Internet] [Masters thesis]. McGill University; 2009. [cited 2020 Feb 17]. Available from: http://digitool.library.mcgill.ca/thesisfile111560.pdf.

Council of Science Editors:

Pasternyk, Stephanie Marika 1. Effect of extracellular matrix and mechanical strain on airway smooth muscle. [Masters Thesis]. McGill University; 2009. Available from: http://digitool.library.mcgill.ca/thesisfile111560.pdf


University of North Texas

27. Greyner, Henry José. The role of prostaglandins, nitric oxide and oxygen in the ductus arteriosi of the pre-term chicken embryo (Gallus domesticus).

Degree: 2007, University of North Texas

 The chicken ductus arteriosi (DA) are two embryonic blood vessels that shunt blood away from the non-ventilated lungs and towards the body and chorioallantoic membrane.… (more)

Subjects/Keywords: prostaglandins; Ductus artieriosi; chicken embryos; smooth muscle contraction; oxygen; cardiovascular physiology; Ductus arteriosus.; Chickens  – Embryos  – Physiology.; Prostaglandins.; Nitric oxide.; Oxygen.

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APA (6th Edition):

Greyner, H. J. (2007). The role of prostaglandins, nitric oxide and oxygen in the ductus arteriosi of the pre-term chicken embryo (Gallus domesticus). (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc5173/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Greyner, Henry José. “The role of prostaglandins, nitric oxide and oxygen in the ductus arteriosi of the pre-term chicken embryo (Gallus domesticus).” 2007. Thesis, University of North Texas. Accessed February 17, 2020. https://digital.library.unt.edu/ark:/67531/metadc5173/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Greyner, Henry José. “The role of prostaglandins, nitric oxide and oxygen in the ductus arteriosi of the pre-term chicken embryo (Gallus domesticus).” 2007. Web. 17 Feb 2020.

Vancouver:

Greyner HJ. The role of prostaglandins, nitric oxide and oxygen in the ductus arteriosi of the pre-term chicken embryo (Gallus domesticus). [Internet] [Thesis]. University of North Texas; 2007. [cited 2020 Feb 17]. Available from: https://digital.library.unt.edu/ark:/67531/metadc5173/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Greyner HJ. The role of prostaglandins, nitric oxide and oxygen in the ductus arteriosi of the pre-term chicken embryo (Gallus domesticus). [Thesis]. University of North Texas; 2007. Available from: https://digital.library.unt.edu/ark:/67531/metadc5173/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

28. Hernandez, Mark J. (Mark Jerry), 1979-. Caveolin-1 A scaffold for microcompartmental organization of membrane-associated glycolysis.

Degree: PhD, 2007, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT AUTHOR'S REQUEST.] The role of membrane caveolae in the compartmentation of membrane glycolytic metabolism indicates a pivotal… (more)

Subjects/Keywords: Muscle cells; Membrane proteins  – Biosynthesis; Cell membranes; Enzymology; Glycolysis  – Physiology; Caveolin 1  – biosynthesis; Cell Membrane  – enzymology; Glycolysis  – physiology; Myocytes, Smooth Muscle  – enzymology

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APA (6th Edition):

Hernandez, Mark J. (Mark Jerry), 1. (2007). Caveolin-1 A scaffold for microcompartmental organization of membrane-associated glycolysis. (Doctoral Dissertation). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/6007

Chicago Manual of Style (16th Edition):

Hernandez, Mark J. (Mark Jerry), 1979-. “Caveolin-1 A scaffold for microcompartmental organization of membrane-associated glycolysis.” 2007. Doctoral Dissertation, University of Missouri – Columbia. Accessed February 17, 2020. https://doi.org/10.32469/10355/6007.

MLA Handbook (7th Edition):

Hernandez, Mark J. (Mark Jerry), 1979-. “Caveolin-1 A scaffold for microcompartmental organization of membrane-associated glycolysis.” 2007. Web. 17 Feb 2020.

Vancouver:

Hernandez, Mark J. (Mark Jerry) 1. Caveolin-1 A scaffold for microcompartmental organization of membrane-associated glycolysis. [Internet] [Doctoral dissertation]. University of Missouri – Columbia; 2007. [cited 2020 Feb 17]. Available from: https://doi.org/10.32469/10355/6007.

Council of Science Editors:

Hernandez, Mark J. (Mark Jerry) 1. Caveolin-1 A scaffold for microcompartmental organization of membrane-associated glycolysis. [Doctoral Dissertation]. University of Missouri – Columbia; 2007. Available from: https://doi.org/10.32469/10355/6007

29. Hu, Ruoxi. Exploring the role of microRNAs in airway smooth muscle biology and asthma therapy.

Degree: PhD, Biological Sciences in Public Health, 2014, Harvard University

 The pathophysiology of asthma is characterized by airway inflammation, remodeling and hyper-responsiveness. Phenotypic changes in airway smooth muscle cells (ASM) play a pivotal role in… (more)

Subjects/Keywords: Molecular biology; Physiology; Genetics; Airway Smooth Muscle; Asthma; Asthma treatments; Cell proliferation; MicroRNA; Next generation sequencing

…knowledge on airway smooth muscle biology and asthma. I have benefited greatly from the technical… …asthma [1-3]. They bind to β2-adrenergic receptors expressed on airway smooth muscle… …The role of airway smooth muscle (ASM) in airway hyper-responsiveness (AHR… …x29; A link between airway smooth muscle (ASM) and airway hyper-responsiveness… …which we now refer to as airway smooth muscle (ASM). AHR refers to excessive… 

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APA (6th Edition):

Hu, R. (2014). Exploring the role of microRNAs in airway smooth muscle biology and asthma therapy. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274596

Chicago Manual of Style (16th Edition):

Hu, Ruoxi. “Exploring the role of microRNAs in airway smooth muscle biology and asthma therapy.” 2014. Doctoral Dissertation, Harvard University. Accessed February 17, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274596.

MLA Handbook (7th Edition):

Hu, Ruoxi. “Exploring the role of microRNAs in airway smooth muscle biology and asthma therapy.” 2014. Web. 17 Feb 2020.

Vancouver:

Hu R. Exploring the role of microRNAs in airway smooth muscle biology and asthma therapy. [Internet] [Doctoral dissertation]. Harvard University; 2014. [cited 2020 Feb 17]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274596.

Council of Science Editors:

Hu R. Exploring the role of microRNAs in airway smooth muscle biology and asthma therapy. [Doctoral Dissertation]. Harvard University; 2014. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274596

30. Rosner, Sonia Rebecca. Airway Dynamics and the Role of Zyxin.

Degree: PhD, Biological Sciences in Public Health, 2014, Harvard University

 Morbidity and mortality attributable to asthma arise mainly from contraction of airway smooth muscle (ASM) and resulting bronchospasm. Bronchospasm that is induced in the laboratory… (more)

Subjects/Keywords: Biology; Biophysics; Physiology; airway smooth muscle; cryopreservation; deep inspiration; PCLS; stretch; zyxin

…4.3 Airway epithelium and smooth muscle layers are similarly intact in never frozen and… …DI has been attributed to the manner in which stretch fluidizes the airway smooth muscle… …has been associated with increased contractile stimulus of airway smooth muscle (ASM… …in airway smooth muscle, I developed a multi-scale, interdisciplinary approach, collecting… …literature on deep inspiration, airway smooth muscle, and zyxin, with a special focus on the innate… 

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APA (6th Edition):

Rosner, S. R. (2014). Airway Dynamics and the Role of Zyxin. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:12269821

Chicago Manual of Style (16th Edition):

Rosner, Sonia Rebecca. “Airway Dynamics and the Role of Zyxin.” 2014. Doctoral Dissertation, Harvard University. Accessed February 17, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:12269821.

MLA Handbook (7th Edition):

Rosner, Sonia Rebecca. “Airway Dynamics and the Role of Zyxin.” 2014. Web. 17 Feb 2020.

Vancouver:

Rosner SR. Airway Dynamics and the Role of Zyxin. [Internet] [Doctoral dissertation]. Harvard University; 2014. [cited 2020 Feb 17]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12269821.

Council of Science Editors:

Rosner SR. Airway Dynamics and the Role of Zyxin. [Doctoral Dissertation]. Harvard University; 2014. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12269821

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