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You searched for subject:(Smooth Muscle Cell). Showing records 1 – 30 of 162 total matches.

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University of Cambridge

1. Harman, Jennifer. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression.

Degree: PhD, 2019, University of Cambridge

 Widespread changes in gene expression accompany vascular smooth muscle cell (VSMC) phenotypic switching, a hallmark of vascular disease. Upon insult, VSMCs downregulate contractile proteins and… (more)

Subjects/Keywords: Vascular smooth muscle cell; H3K9me2; Vascular smooth muscle cell phenotypic switch

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APA (6th Edition):

Harman, J. (2019). Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/289975

Chicago Manual of Style (16th Edition):

Harman, Jennifer. “Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression.” 2019. Doctoral Dissertation, University of Cambridge. Accessed August 08, 2020. https://www.repository.cam.ac.uk/handle/1810/289975.

MLA Handbook (7th Edition):

Harman, Jennifer. “Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression.” 2019. Web. 08 Aug 2020.

Vancouver:

Harman J. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Aug 08]. Available from: https://www.repository.cam.ac.uk/handle/1810/289975.

Council of Science Editors:

Harman J. Investigating the role of histone H3 lysine 9 dimethylation in regulating disease-associated vascular smooth muscle cell gene expression. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/289975

2. Watanabe, Kenichi. Soluble LR11 is a novel biomarker for vascular lesions late after Kawasaki disease : 川崎病遠隔期における血管障害の新たなバイオマーカー:可溶型LR11.

Degree: 博士(医学), 2016, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第4211号. 学位記番号: 新大院博(医)甲第710号. 学位授与年月日: 平成28年9月20日

Objective : Coronary artery lesions (CALs)and a risk for early onset of atherosclerosis are major concerns following… (more)

Subjects/Keywords: LR11; smooth muscle cell; Kawasaki disease; biomarker

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APA (6th Edition):

Watanabe, K. (2016). Soluble LR11 is a novel biomarker for vascular lesions late after Kawasaki disease : 川崎病遠隔期における血管障害の新たなバイオマーカー:可溶型LR11. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/45097

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Watanabe, Kenichi. “Soluble LR11 is a novel biomarker for vascular lesions late after Kawasaki disease : 川崎病遠隔期における血管障害の新たなバイオマーカー:可溶型LR11.” 2016. Thesis, Niigata University / 新潟大学. Accessed August 08, 2020. http://hdl.handle.net/10191/45097.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Watanabe, Kenichi. “Soluble LR11 is a novel biomarker for vascular lesions late after Kawasaki disease : 川崎病遠隔期における血管障害の新たなバイオマーカー:可溶型LR11.” 2016. Web. 08 Aug 2020.

Vancouver:

Watanabe K. Soluble LR11 is a novel biomarker for vascular lesions late after Kawasaki disease : 川崎病遠隔期における血管障害の新たなバイオマーカー:可溶型LR11. [Internet] [Thesis]. Niigata University / 新潟大学; 2016. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/10191/45097.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Watanabe K. Soluble LR11 is a novel biomarker for vascular lesions late after Kawasaki disease : 川崎病遠隔期における血管障害の新たなバイオマーカー:可溶型LR11. [Thesis]. Niigata University / 新潟大学; 2016. Available from: http://hdl.handle.net/10191/45097

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Miami

3. Song, Lei. Role of c-Kit in Atherosclerosis: A Mechanistic Study.

Degree: PhD, Molecular and Cellular Pharmacology (Medicine), 2017, University of Miami

 Rationale: c-Kit, as a receptor tyrosine kinase (RTK), is present in multiple cell types including vascular smooth muscle cells (SMCs). However, so far, limited progress… (more)

Subjects/Keywords: c-Kit; Smooth Muscle Cell; Atherosclerosis

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APA (6th Edition):

Song, L. (2017). Role of c-Kit in Atherosclerosis: A Mechanistic Study. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/1972

Chicago Manual of Style (16th Edition):

Song, Lei. “Role of c-Kit in Atherosclerosis: A Mechanistic Study.” 2017. Doctoral Dissertation, University of Miami. Accessed August 08, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/1972.

MLA Handbook (7th Edition):

Song, Lei. “Role of c-Kit in Atherosclerosis: A Mechanistic Study.” 2017. Web. 08 Aug 2020.

Vancouver:

Song L. Role of c-Kit in Atherosclerosis: A Mechanistic Study. [Internet] [Doctoral dissertation]. University of Miami; 2017. [cited 2020 Aug 08]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1972.

Council of Science Editors:

Song L. Role of c-Kit in Atherosclerosis: A Mechanistic Study. [Doctoral Dissertation]. University of Miami; 2017. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1972


Queen Mary, University of London

4. Zhao, Hanqing. MicroRNA-22 regulates smooth muscle cell differentiation from stem cells by targeting methyl CpG binding protein 2.

Degree: PhD, 2015, Queen Mary, University of London

 In recent years, microRNAs have emerged as important regulators in various biological processes, as a new class of biomarkers, and as novel therapeutic drugs for… (more)

Subjects/Keywords: Medicine; microRNAs; smooth muscle cell differentiation

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APA (6th Edition):

Zhao, H. (2015). MicroRNA-22 regulates smooth muscle cell differentiation from stem cells by targeting methyl CpG binding protein 2. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/12967 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775178

Chicago Manual of Style (16th Edition):

Zhao, Hanqing. “MicroRNA-22 regulates smooth muscle cell differentiation from stem cells by targeting methyl CpG binding protein 2.” 2015. Doctoral Dissertation, Queen Mary, University of London. Accessed August 08, 2020. http://qmro.qmul.ac.uk/xmlui/handle/123456789/12967 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775178.

MLA Handbook (7th Edition):

Zhao, Hanqing. “MicroRNA-22 regulates smooth muscle cell differentiation from stem cells by targeting methyl CpG binding protein 2.” 2015. Web. 08 Aug 2020.

Vancouver:

Zhao H. MicroRNA-22 regulates smooth muscle cell differentiation from stem cells by targeting methyl CpG binding protein 2. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2015. [cited 2020 Aug 08]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/12967 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775178.

Council of Science Editors:

Zhao H. MicroRNA-22 regulates smooth muscle cell differentiation from stem cells by targeting methyl CpG binding protein 2. [Doctoral Dissertation]. Queen Mary, University of London; 2015. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/12967 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775178


Boston University

5. Pessolano, Lawrence. Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation.

Degree: PhD, Biochemistry, 2016, Boston University

 Vascular smooth muscle cells (SMCs) regulate vessel contraction but during diseases including atherosclerosis, SMCs undergo functional changes that contribute to pathology. Chronic inflammation in the… (more)

Subjects/Keywords: Molecular biology; Atherosclerosis; Cholesterol trafficking; Smooth muscle cell; Smooth muscle cell differentiation; Vascular biology

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APA (6th Edition):

Pessolano, L. (2016). Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/14599

Chicago Manual of Style (16th Edition):

Pessolano, Lawrence. “Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation.” 2016. Doctoral Dissertation, Boston University. Accessed August 08, 2020. http://hdl.handle.net/2144/14599.

MLA Handbook (7th Edition):

Pessolano, Lawrence. “Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation.” 2016. Web. 08 Aug 2020.

Vancouver:

Pessolano L. Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation. [Internet] [Doctoral dissertation]. Boston University; 2016. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/2144/14599.

Council of Science Editors:

Pessolano L. Serum amyloid A and toll-like receptor 2 regulate vascular smooth muscle cell cholesterol trafficking and differentiation. [Doctoral Dissertation]. Boston University; 2016. Available from: http://hdl.handle.net/2144/14599


University of Toronto

6. Lipsett, Kathryn Richelle. Proteomic Analysis of Endothelial, Smooth Muscle, Myocytes and Fibroblasts Derived from Human Cardiac Tissue Identified Cell Type Specific Exprsession Signatures.

Degree: 2014, University of Toronto

The heart is composed of multiple constituent cell types; the primary cells being fibroblasts, cardiomyocytes, endothelial and smooth muscle cells. Here, I employed membrane isolation… (more)

Subjects/Keywords: Cardiomyocyte; Endothelial Cell; Fibroblast; Heart; Mass Spectrometry; Smooth Muscle Cell; 0719

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APA (6th Edition):

Lipsett, K. R. (2014). Proteomic Analysis of Endothelial, Smooth Muscle, Myocytes and Fibroblasts Derived from Human Cardiac Tissue Identified Cell Type Specific Exprsession Signatures. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/82618

Chicago Manual of Style (16th Edition):

Lipsett, Kathryn Richelle. “Proteomic Analysis of Endothelial, Smooth Muscle, Myocytes and Fibroblasts Derived from Human Cardiac Tissue Identified Cell Type Specific Exprsession Signatures.” 2014. Masters Thesis, University of Toronto. Accessed August 08, 2020. http://hdl.handle.net/1807/82618.

MLA Handbook (7th Edition):

Lipsett, Kathryn Richelle. “Proteomic Analysis of Endothelial, Smooth Muscle, Myocytes and Fibroblasts Derived from Human Cardiac Tissue Identified Cell Type Specific Exprsession Signatures.” 2014. Web. 08 Aug 2020.

Vancouver:

Lipsett KR. Proteomic Analysis of Endothelial, Smooth Muscle, Myocytes and Fibroblasts Derived from Human Cardiac Tissue Identified Cell Type Specific Exprsession Signatures. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1807/82618.

Council of Science Editors:

Lipsett KR. Proteomic Analysis of Endothelial, Smooth Muscle, Myocytes and Fibroblasts Derived from Human Cardiac Tissue Identified Cell Type Specific Exprsession Signatures. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/82618


University of Toronto

7. Hui, Sonya. Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy.

Degree: 2010, University of Toronto

Increased intracellular calcium (Ca2+) is required for vascular smooth muscle cell (VSMC) proliferation through mechanisms that are not well-known. Preventing calmodulin (CaM)-cyclin E interaction with… (more)

Subjects/Keywords: Calcium; Cell Cycle; Vascular smooth muscle cell; 0719

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APA (6th Edition):

Hui, S. (2010). Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25625

Chicago Manual of Style (16th Edition):

Hui, Sonya. “Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy.” 2010. Masters Thesis, University of Toronto. Accessed August 08, 2020. http://hdl.handle.net/1807/25625.

MLA Handbook (7th Edition):

Hui, Sonya. “Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy.” 2010. Web. 08 Aug 2020.

Vancouver:

Hui S. Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1807/25625.

Council of Science Editors:

Hui S. Calcium-sensitive Mmechanisms in Vascular Smooth Muscle Cell Cycle Progression as Targets for Therapy. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25625


Queens University

8. Truong, Tammy. Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes .

Degree: Pathology and Molecular Medicine, 2014, Queens University

 During the development of atherosclerosis, contractile vascular smooth muscle cells (VSMCs) change to cells capable of migrating and proliferating to mediate repair, where the responses… (more)

Subjects/Keywords: cell migration ; vascular smooth muscle cell ; cAMP ; compartmentalization ; phosphodiesterase 4D

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APA (6th Edition):

Truong, T. (2014). Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8658

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Truong, Tammy. “Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes .” 2014. Thesis, Queens University. Accessed August 08, 2020. http://hdl.handle.net/1974/8658.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Truong, Tammy. “Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes .” 2014. Web. 08 Aug 2020.

Vancouver:

Truong T. Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes . [Internet] [Thesis]. Queens University; 2014. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1974/8658.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Truong T. Compartmentalized phosphodiesterase 4D isoforms expression, targeting and localization in vascular myocytes . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/8658

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

9. Wang, Li. Role of Muscle-Derived Tissue Factor in Arterial Thrombosis, Intimal Hyperplasis, and Cardiac Fibrosis.

Degree: PhD, 2009, University of Rochester

 Tissue factor (TF) initiates coagulation, regulates hemostasis and plays a critical role in mediating thrombosis. TF also plays a key role in mediating intimal hyperplasia… (more)

Subjects/Keywords: Smooth Muscle Cell; Heart Homeostasis; Hemastasis; Cell Proliferation and Migration; Cell Survival

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APA (6th Edition):

Wang, L. (2009). Role of Muscle-Derived Tissue Factor in Arterial Thrombosis, Intimal Hyperplasis, and Cardiac Fibrosis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/8299

Chicago Manual of Style (16th Edition):

Wang, Li. “Role of Muscle-Derived Tissue Factor in Arterial Thrombosis, Intimal Hyperplasis, and Cardiac Fibrosis.” 2009. Doctoral Dissertation, University of Rochester. Accessed August 08, 2020. http://hdl.handle.net/1802/8299.

MLA Handbook (7th Edition):

Wang, Li. “Role of Muscle-Derived Tissue Factor in Arterial Thrombosis, Intimal Hyperplasis, and Cardiac Fibrosis.” 2009. Web. 08 Aug 2020.

Vancouver:

Wang L. Role of Muscle-Derived Tissue Factor in Arterial Thrombosis, Intimal Hyperplasis, and Cardiac Fibrosis. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1802/8299.

Council of Science Editors:

Wang L. Role of Muscle-Derived Tissue Factor in Arterial Thrombosis, Intimal Hyperplasis, and Cardiac Fibrosis. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/8299


Dalhousie University

10. Walker, Matthew. The Development of a 3D Piezoelectric Active Microtissue Model for Airway Smooth Muscle.

Degree: Master of Applied Science, Department of Biomedical Engineering, 2013, Dalhousie University

 Although asthma is primarily thought to be an inflammatory disease of the airways, it has recently been hypothesized that the altered mechanical environment of an… (more)

Subjects/Keywords: Microtisse; Piezoelectric; 3D Cell Culture; Airway Smooth Muscle; Stretch; Asthma; Mechanobiology

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APA (6th Edition):

Walker, M. (2013). The Development of a 3D Piezoelectric Active Microtissue Model for Airway Smooth Muscle. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/21862

Chicago Manual of Style (16th Edition):

Walker, Matthew. “The Development of a 3D Piezoelectric Active Microtissue Model for Airway Smooth Muscle.” 2013. Masters Thesis, Dalhousie University. Accessed August 08, 2020. http://hdl.handle.net/10222/21862.

MLA Handbook (7th Edition):

Walker, Matthew. “The Development of a 3D Piezoelectric Active Microtissue Model for Airway Smooth Muscle.” 2013. Web. 08 Aug 2020.

Vancouver:

Walker M. The Development of a 3D Piezoelectric Active Microtissue Model for Airway Smooth Muscle. [Internet] [Masters thesis]. Dalhousie University; 2013. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/10222/21862.

Council of Science Editors:

Walker M. The Development of a 3D Piezoelectric Active Microtissue Model for Airway Smooth Muscle. [Masters Thesis]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/21862


Texas A&M University

11. Richardson, William. Vascular Smooth Muscle Precursor Cell Behavior in Non-Uniform Stretch Environments.

Degree: 2012, Texas A&M University

 Cells in the body respond to mechanical loads in ways that are crucial to normal and disease physiology. Understanding these processes is difficult due to… (more)

Subjects/Keywords: phenotype modulation; smooth muscle cell; stretch gradient; mechanobiology

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APA (6th Edition):

Richardson, W. (2012). Vascular Smooth Muscle Precursor Cell Behavior in Non-Uniform Stretch Environments. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/148094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Richardson, William. “Vascular Smooth Muscle Precursor Cell Behavior in Non-Uniform Stretch Environments.” 2012. Thesis, Texas A&M University. Accessed August 08, 2020. http://hdl.handle.net/1969.1/148094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Richardson, William. “Vascular Smooth Muscle Precursor Cell Behavior in Non-Uniform Stretch Environments.” 2012. Web. 08 Aug 2020.

Vancouver:

Richardson W. Vascular Smooth Muscle Precursor Cell Behavior in Non-Uniform Stretch Environments. [Internet] [Thesis]. Texas A&M University; 2012. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1969.1/148094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Richardson W. Vascular Smooth Muscle Precursor Cell Behavior in Non-Uniform Stretch Environments. [Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/148094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

12. Arshad, Haroon. Mathematical modelling of PulmonaryArterial Smooth Muscle Cell Subtypes.

Degree: 2016, University of Manchester

 Alteration in the tone of pulmonary arteries may lead to disease such as pulmonary hypertension often associated with major cardiac complications. This dysfunction is partly… (more)

Subjects/Keywords: mathematical; modelling; pulmonary; single-cell; computational; model; smooth muscle; PASMC

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APA (6th Edition):

Arshad, H. (2016). Mathematical modelling of PulmonaryArterial Smooth Muscle Cell Subtypes. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294455

Chicago Manual of Style (16th Edition):

Arshad, Haroon. “Mathematical modelling of PulmonaryArterial Smooth Muscle Cell Subtypes.” 2016. Doctoral Dissertation, University of Manchester. Accessed August 08, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294455.

MLA Handbook (7th Edition):

Arshad, Haroon. “Mathematical modelling of PulmonaryArterial Smooth Muscle Cell Subtypes.” 2016. Web. 08 Aug 2020.

Vancouver:

Arshad H. Mathematical modelling of PulmonaryArterial Smooth Muscle Cell Subtypes. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Aug 08]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294455.

Council of Science Editors:

Arshad H. Mathematical modelling of PulmonaryArterial Smooth Muscle Cell Subtypes. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294455


Virginia Tech

13. Miller, Zachary Dalton. Tensile Properties of Single Vaginal Smooth Muscle Cells.

Degree: MS, Engineering Science and Mechanics, 2018, Virginia Tech

 Improving treatment and prevention of pelvic organ prolapse, a disorder affecting up to half of parous women, requires thorough mechanical analysis of the vagina and… (more)

Subjects/Keywords: Biomechanics; Cell Mechanics; Smooth Muscle; Vagina; Pelvic Organ Prolapse

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APA (6th Edition):

Miller, Z. D. (2018). Tensile Properties of Single Vaginal Smooth Muscle Cells. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/83567

Chicago Manual of Style (16th Edition):

Miller, Zachary Dalton. “Tensile Properties of Single Vaginal Smooth Muscle Cells.” 2018. Masters Thesis, Virginia Tech. Accessed August 08, 2020. http://hdl.handle.net/10919/83567.

MLA Handbook (7th Edition):

Miller, Zachary Dalton. “Tensile Properties of Single Vaginal Smooth Muscle Cells.” 2018. Web. 08 Aug 2020.

Vancouver:

Miller ZD. Tensile Properties of Single Vaginal Smooth Muscle Cells. [Internet] [Masters thesis]. Virginia Tech; 2018. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/10919/83567.

Council of Science Editors:

Miller ZD. Tensile Properties of Single Vaginal Smooth Muscle Cells. [Masters Thesis]. Virginia Tech; 2018. Available from: http://hdl.handle.net/10919/83567


University of Toronto

14. Mantella, Laura-Eve. Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms.

Degree: 2016, University of Toronto

Long noncoding RNAs (lncRNAs) are a new class of noncoding RNA and emerging evidence suggests that they represent a cellular hub for coordination of various… (more)

Subjects/Keywords: aortic aneurysm; hypertension; mechanical stretch; vascular smooth muscle cell; 0379

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APA (6th Edition):

Mantella, L. (2016). Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/75343

Chicago Manual of Style (16th Edition):

Mantella, Laura-Eve. “Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms.” 2016. Masters Thesis, University of Toronto. Accessed August 08, 2020. http://hdl.handle.net/1807/75343.

MLA Handbook (7th Edition):

Mantella, Laura-Eve. “Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms.” 2016. Web. 08 Aug 2020.

Vancouver:

Mantella L. Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1807/75343.

Council of Science Editors:

Mantella L. Long Non-coding RNA Fingerprint Regulated by Cyclic Mechanical Stress in Vascular Smooth Muscle Cells: Implications for Hypertension and Aortic Aneurysms. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/75343


University of Manchester

15. Arshad, Haroon. Mathematical modelling of pulmonary arterial smooth muscle cell subtypes.

Degree: PhD, 2016, University of Manchester

 Alteration in the tone of pulmonary arteries may lead to disease such as pulmonary hypertension often associated with major cardiac complications. This dysfunction is partly… (more)

Subjects/Keywords: 612.1; mathematical; modelling; pulmonary; single-cell; computational; model; smooth muscle; PASMC

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APA (6th Edition):

Arshad, H. (2016). Mathematical modelling of pulmonary arterial smooth muscle cell subtypes. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/mathematical-modelling-of-pulmonaryarterial-smooth-muscle-cell-subtypes(c1110807-d94d-487c-90b8-8714d5e42d16).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679992

Chicago Manual of Style (16th Edition):

Arshad, Haroon. “Mathematical modelling of pulmonary arterial smooth muscle cell subtypes.” 2016. Doctoral Dissertation, University of Manchester. Accessed August 08, 2020. https://www.research.manchester.ac.uk/portal/en/theses/mathematical-modelling-of-pulmonaryarterial-smooth-muscle-cell-subtypes(c1110807-d94d-487c-90b8-8714d5e42d16).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679992.

MLA Handbook (7th Edition):

Arshad, Haroon. “Mathematical modelling of pulmonary arterial smooth muscle cell subtypes.” 2016. Web. 08 Aug 2020.

Vancouver:

Arshad H. Mathematical modelling of pulmonary arterial smooth muscle cell subtypes. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Aug 08]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/mathematical-modelling-of-pulmonaryarterial-smooth-muscle-cell-subtypes(c1110807-d94d-487c-90b8-8714d5e42d16).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679992.

Council of Science Editors:

Arshad H. Mathematical modelling of pulmonary arterial smooth muscle cell subtypes. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/mathematical-modelling-of-pulmonaryarterial-smooth-muscle-cell-subtypes(c1110807-d94d-487c-90b8-8714d5e42d16).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.679992


University of Aberdeen

16. McKean, Jenny Susan. The role of the cAMP mediator Epac in vascular smooth muscle cell migration.

Degree: PhD, 2015, University of Aberdeen

 Surgical intervention can result in endothelial denudation, driving growth factor-stimulated vascular smooth muscle cell (VSMC) migration towards the intima, leading to luminal narrowing and restenosis.… (more)

Subjects/Keywords: 610; Coronary arteries; Vascular smooth muscle; Cell migration; Cyclic adenylic acid

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APA (6th Edition):

McKean, J. S. (2015). The role of the cAMP mediator Epac in vascular smooth muscle cell migration. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=227109 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.659069

Chicago Manual of Style (16th Edition):

McKean, Jenny Susan. “The role of the cAMP mediator Epac in vascular smooth muscle cell migration.” 2015. Doctoral Dissertation, University of Aberdeen. Accessed August 08, 2020. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=227109 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.659069.

MLA Handbook (7th Edition):

McKean, Jenny Susan. “The role of the cAMP mediator Epac in vascular smooth muscle cell migration.” 2015. Web. 08 Aug 2020.

Vancouver:

McKean JS. The role of the cAMP mediator Epac in vascular smooth muscle cell migration. [Internet] [Doctoral dissertation]. University of Aberdeen; 2015. [cited 2020 Aug 08]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=227109 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.659069.

Council of Science Editors:

McKean JS. The role of the cAMP mediator Epac in vascular smooth muscle cell migration. [Doctoral Dissertation]. University of Aberdeen; 2015. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=227109 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.659069


University of Cambridge

17. Dobnikar, Lina. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.

Degree: PhD, 2020, University of Cambridge

 Vascular smooth muscle cells (VSMCs) possess a remarkable capacity to change phenotype in response to injury or inflammation. In healthy arteries, VSMCs exist in a… (more)

Subjects/Keywords: single-cell RNA-seq; vascular smooth muscle cells; cardiovascular

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APA (6th Edition):

Dobnikar, L. (2020). A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/301990

Chicago Manual of Style (16th Edition):

Dobnikar, Lina. “A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.” 2020. Doctoral Dissertation, University of Cambridge. Accessed August 08, 2020. https://www.repository.cam.ac.uk/handle/1810/301990.

MLA Handbook (7th Edition):

Dobnikar, Lina. “A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.” 2020. Web. 08 Aug 2020.

Vancouver:

Dobnikar L. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Aug 08]. Available from: https://www.repository.cam.ac.uk/handle/1810/301990.

Council of Science Editors:

Dobnikar L. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/301990


University of Western Ontario

18. Leung, Sharon Z. The Role of Sirtuin 6 in Maintaining Vascular Integrity.

Degree: 2016, University of Western Ontario

 Oxidative stress is an underlying cause for vascular pathologies including inflammation, hypertension, and atherosclerosis. Sirtuins (SIRTs) are a family of NAD+ dependent deacetylases with pronounced… (more)

Subjects/Keywords: SIRT6; vascular smooth muscle cell; angiotensin II; inflammation; Biochemistry

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APA (6th Edition):

Leung, S. Z. (2016). The Role of Sirtuin 6 in Maintaining Vascular Integrity. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/3676

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Leung, Sharon Z. “The Role of Sirtuin 6 in Maintaining Vascular Integrity.” 2016. Thesis, University of Western Ontario. Accessed August 08, 2020. https://ir.lib.uwo.ca/etd/3676.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Leung, Sharon Z. “The Role of Sirtuin 6 in Maintaining Vascular Integrity.” 2016. Web. 08 Aug 2020.

Vancouver:

Leung SZ. The Role of Sirtuin 6 in Maintaining Vascular Integrity. [Internet] [Thesis]. University of Western Ontario; 2016. [cited 2020 Aug 08]. Available from: https://ir.lib.uwo.ca/etd/3676.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Leung SZ. The Role of Sirtuin 6 in Maintaining Vascular Integrity. [Thesis]. University of Western Ontario; 2016. Available from: https://ir.lib.uwo.ca/etd/3676

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

19. Chanakira, Alice. Differential modulation of arterial vs venous smooth muscle cell proliferation and migration by hypoxia and hypoxia inducible endothelial cell growth factors.

Degree: PhD, Pharmacology, 2011, University of Minnesota

 Despite intensive research studies, theories have yet to focus on the contribution of hypoxia to patency differences observed clinically between arterial vs. venous grafts. This… (more)

Subjects/Keywords: Hypoxia; Migration; PDGF-BB; Proliferation; Smooth muscle cell; VEGF-A

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APA (6th Edition):

Chanakira, A. (2011). Differential modulation of arterial vs venous smooth muscle cell proliferation and migration by hypoxia and hypoxia inducible endothelial cell growth factors. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/117320

Chicago Manual of Style (16th Edition):

Chanakira, Alice. “Differential modulation of arterial vs venous smooth muscle cell proliferation and migration by hypoxia and hypoxia inducible endothelial cell growth factors.” 2011. Doctoral Dissertation, University of Minnesota. Accessed August 08, 2020. http://purl.umn.edu/117320.

MLA Handbook (7th Edition):

Chanakira, Alice. “Differential modulation of arterial vs venous smooth muscle cell proliferation and migration by hypoxia and hypoxia inducible endothelial cell growth factors.” 2011. Web. 08 Aug 2020.

Vancouver:

Chanakira A. Differential modulation of arterial vs venous smooth muscle cell proliferation and migration by hypoxia and hypoxia inducible endothelial cell growth factors. [Internet] [Doctoral dissertation]. University of Minnesota; 2011. [cited 2020 Aug 08]. Available from: http://purl.umn.edu/117320.

Council of Science Editors:

Chanakira A. Differential modulation of arterial vs venous smooth muscle cell proliferation and migration by hypoxia and hypoxia inducible endothelial cell growth factors. [Doctoral Dissertation]. University of Minnesota; 2011. Available from: http://purl.umn.edu/117320


Clemson University

20. Bradley, Suzanne Nicole. Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces.

Degree: MS, Bioengineering, 2018, Clemson University

 Vascular smooth muscle cells regulate blood flow by contracting and relaxing blood vessels. Vascular smooth muscle cells display one of two distinct phenotypes: contractile or… (more)

Subjects/Keywords: Cell Stretching; Micropatterning; Microstamping; Vascular Smooth Muscle Cells

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APA (6th Edition):

Bradley, S. N. (2018). Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces. (Masters Thesis). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_theses/2957

Chicago Manual of Style (16th Edition):

Bradley, Suzanne Nicole. “Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces.” 2018. Masters Thesis, Clemson University. Accessed August 08, 2020. https://tigerprints.clemson.edu/all_theses/2957.

MLA Handbook (7th Edition):

Bradley, Suzanne Nicole. “Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces.” 2018. Web. 08 Aug 2020.

Vancouver:

Bradley SN. Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces. [Internet] [Masters thesis]. Clemson University; 2018. [cited 2020 Aug 08]. Available from: https://tigerprints.clemson.edu/all_theses/2957.

Council of Science Editors:

Bradley SN. Stretching Vascular Smooth Muscle Cells on Micropatterned Surfaces. [Masters Thesis]. Clemson University; 2018. Available from: https://tigerprints.clemson.edu/all_theses/2957


University of Cambridge

21. Dobnikar, Lina. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.

Degree: PhD, 2020, University of Cambridge

 Vascular smooth muscle cells (VSMCs) possess a remarkable capacity to change phenotype in response to injury or inflammation. In healthy arteries, VSMCs exist in a… (more)

Subjects/Keywords: single-cell RNA-seq; vascular smooth muscle cells; cardiovascular

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APA (6th Edition):

Dobnikar, L. (2020). A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/301990 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801760

Chicago Manual of Style (16th Edition):

Dobnikar, Lina. “A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.” 2020. Doctoral Dissertation, University of Cambridge. Accessed August 08, 2020. https://www.repository.cam.ac.uk/handle/1810/301990 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801760.

MLA Handbook (7th Edition):

Dobnikar, Lina. “A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity.” 2020. Web. 08 Aug 2020.

Vancouver:

Dobnikar L. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Aug 08]. Available from: https://www.repository.cam.ac.uk/handle/1810/301990 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801760.

Council of Science Editors:

Dobnikar L. A genome-wide, single-cell analysis of vascular smooth muscle cell plasticity. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/301990 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801760


Queens University

22. Wudwud, Alexandra. Investigating the role of PDE4D7 in vascular smooth muscle cell migration .

Degree: Pathology and Molecular Medicine, 2015, Queens University

 Inhibiting the maladaptive migration of vascular smooth muscle cells (VSMCs) in injured blood vessels can help to reduce the development of intimal lesions in vascular… (more)

Subjects/Keywords: phosphodiesterase ; PDE4D7 ; smooth muscle cell migration ; RhoA ROCK

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APA (6th Edition):

Wudwud, A. (2015). Investigating the role of PDE4D7 in vascular smooth muscle cell migration . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/13548

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wudwud, Alexandra. “Investigating the role of PDE4D7 in vascular smooth muscle cell migration .” 2015. Thesis, Queens University. Accessed August 08, 2020. http://hdl.handle.net/1974/13548.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wudwud, Alexandra. “Investigating the role of PDE4D7 in vascular smooth muscle cell migration .” 2015. Web. 08 Aug 2020.

Vancouver:

Wudwud A. Investigating the role of PDE4D7 in vascular smooth muscle cell migration . [Internet] [Thesis]. Queens University; 2015. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1974/13548.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wudwud A. Investigating the role of PDE4D7 in vascular smooth muscle cell migration . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/13548

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

23. Chen, Yao-Chang. The inhibitory effects of marine-derived compound on smooth muscle cells proliferation.

Degree: PhD, Marine Biotechnology and Resources, 2015, NSYSU

 Arterial reconstruction procedures, including balloon angioplasty, stenting and coronary artery bypass, are used to restore blood flow in atherosclerotic arteries. Restenosis of these arteries is… (more)

Subjects/Keywords: vascular smooth muscle cell; dihydroaustrasulfone alcohol; proliferation; migration; restenosis; phenotypic modulation; cell cycle

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APA (6th Edition):

Chen, Y. (2015). The inhibitory effects of marine-derived compound on smooth muscle cells proliferation. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0616115-205046

Chicago Manual of Style (16th Edition):

Chen, Yao-Chang. “The inhibitory effects of marine-derived compound on smooth muscle cells proliferation.” 2015. Doctoral Dissertation, NSYSU. Accessed August 08, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0616115-205046.

MLA Handbook (7th Edition):

Chen, Yao-Chang. “The inhibitory effects of marine-derived compound on smooth muscle cells proliferation.” 2015. Web. 08 Aug 2020.

Vancouver:

Chen Y. The inhibitory effects of marine-derived compound on smooth muscle cells proliferation. [Internet] [Doctoral dissertation]. NSYSU; 2015. [cited 2020 Aug 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0616115-205046.

Council of Science Editors:

Chen Y. The inhibitory effects of marine-derived compound on smooth muscle cells proliferation. [Doctoral Dissertation]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0616115-205046


University of Toronto

24. Xu, Songyi. N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases.

Degree: PhD, 2020, University of Toronto

 N-cadherin mediates cell-cell contacts in vascular smooth muscle cells (VSMCs) and regulates VSMC behaviours. Discoidin domain receptor 1 (DDR1) is a collagen binding receptor also… (more)

Subjects/Keywords: Cell-cell interaction; DDR1; N-cadherin; Rho GTPases; Vascular smooth muscle cells; 0307

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APA (6th Edition):

Xu, S. (2020). N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/101328

Chicago Manual of Style (16th Edition):

Xu, Songyi. “N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases.” 2020. Doctoral Dissertation, University of Toronto. Accessed August 08, 2020. http://hdl.handle.net/1807/101328.

MLA Handbook (7th Edition):

Xu, Songyi. “N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases.” 2020. Web. 08 Aug 2020.

Vancouver:

Xu S. N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases. [Internet] [Doctoral dissertation]. University of Toronto; 2020. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/1807/101328.

Council of Science Editors:

Xu S. N-cadherin Regulation of Vascular Smooth Muscle Cells; the Role of DDR1 and Rho GTPases. [Doctoral Dissertation]. University of Toronto; 2020. Available from: http://hdl.handle.net/1807/101328

25. Lin, Lin. Engineering poly (ethylene glycol) hydrogels to regulate smooth muscle cell migration and proliferation.

Degree: PhD, Biomedical Engineering, 2014, Case Western Reserve University School of Graduate Studies

 The key role of smooth muscle cell (SMC) migration and proliferation in vascular physiological and pathological remodeling necessitates the exploration of mechanisms underlying these functions.… (more)

Subjects/Keywords: Biomedical Engineering; Polymers; Smooth muscle cells; PEG hydrogels; 3D cell migration; 3D cell proliferation; biomimetic

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APA (6th Edition):

Lin, L. (2014). Engineering poly (ethylene glycol) hydrogels to regulate smooth muscle cell migration and proliferation. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1401711613

Chicago Manual of Style (16th Edition):

Lin, Lin. “Engineering poly (ethylene glycol) hydrogels to regulate smooth muscle cell migration and proliferation.” 2014. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed August 08, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1401711613.

MLA Handbook (7th Edition):

Lin, Lin. “Engineering poly (ethylene glycol) hydrogels to regulate smooth muscle cell migration and proliferation.” 2014. Web. 08 Aug 2020.

Vancouver:

Lin L. Engineering poly (ethylene glycol) hydrogels to regulate smooth muscle cell migration and proliferation. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2014. [cited 2020 Aug 08]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1401711613.

Council of Science Editors:

Lin L. Engineering poly (ethylene glycol) hydrogels to regulate smooth muscle cell migration and proliferation. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1401711613


East Carolina University

26. Holt, Andrew W. Vasodilator-stimulated phosphoprotein regulates arterial smooth muscle cell migration.

Degree: PhD, PHD-Physiology, 2016, East Carolina University

 Cardiovascular disease (CVD) is a multi-faceted pathology that remains the number one killer of all Americans and people worldwide. Vascular inflammation, endothelial dysfunction, extracellular matrix… (more)

Subjects/Keywords: PKG; VASP; Cardiovascular Diseases; Cell Adhesion Molecules; Cell Movement; Microfilament Proteins; Myocytes, Smooth Muscle; Phosphoproteins

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APA (6th Edition):

Holt, A. W. (2016). Vasodilator-stimulated phosphoprotein regulates arterial smooth muscle cell migration. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/5999

Chicago Manual of Style (16th Edition):

Holt, Andrew W. “Vasodilator-stimulated phosphoprotein regulates arterial smooth muscle cell migration.” 2016. Doctoral Dissertation, East Carolina University. Accessed August 08, 2020. http://hdl.handle.net/10342/5999.

MLA Handbook (7th Edition):

Holt, Andrew W. “Vasodilator-stimulated phosphoprotein regulates arterial smooth muscle cell migration.” 2016. Web. 08 Aug 2020.

Vancouver:

Holt AW. Vasodilator-stimulated phosphoprotein regulates arterial smooth muscle cell migration. [Internet] [Doctoral dissertation]. East Carolina University; 2016. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/10342/5999.

Council of Science Editors:

Holt AW. Vasodilator-stimulated phosphoprotein regulates arterial smooth muscle cell migration. [Doctoral Dissertation]. East Carolina University; 2016. Available from: http://hdl.handle.net/10342/5999

27. Reidinger, Amanda Zoe. Changes in Passive and Dynamic Mechanical Environments Promote Differentiation to a Contractile Phenotype in Vascular Smooth Muscle Cells.

Degree: PhD, 2015, Worcester Polytechnic Institute

 Every year, 400,000 coronary artery bypasses (CABG) are performed in the United States. However, one third of all patients who need a CABG cannot undergo… (more)

Subjects/Keywords: 3D culture; phenotype; smooth muscle cell; vascular tissue engineering; cell-derived matrix

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APA (6th Edition):

Reidinger, A. Z. (2015). Changes in Passive and Dynamic Mechanical Environments Promote Differentiation to a Contractile Phenotype in Vascular Smooth Muscle Cells. (Doctoral Dissertation). Worcester Polytechnic Institute. Retrieved from etd-042915-160154 ; https://digitalcommons.wpi.edu/etd-dissertations/230

Chicago Manual of Style (16th Edition):

Reidinger, Amanda Zoe. “Changes in Passive and Dynamic Mechanical Environments Promote Differentiation to a Contractile Phenotype in Vascular Smooth Muscle Cells.” 2015. Doctoral Dissertation, Worcester Polytechnic Institute. Accessed August 08, 2020. etd-042915-160154 ; https://digitalcommons.wpi.edu/etd-dissertations/230.

MLA Handbook (7th Edition):

Reidinger, Amanda Zoe. “Changes in Passive and Dynamic Mechanical Environments Promote Differentiation to a Contractile Phenotype in Vascular Smooth Muscle Cells.” 2015. Web. 08 Aug 2020.

Vancouver:

Reidinger AZ. Changes in Passive and Dynamic Mechanical Environments Promote Differentiation to a Contractile Phenotype in Vascular Smooth Muscle Cells. [Internet] [Doctoral dissertation]. Worcester Polytechnic Institute; 2015. [cited 2020 Aug 08]. Available from: etd-042915-160154 ; https://digitalcommons.wpi.edu/etd-dissertations/230.

Council of Science Editors:

Reidinger AZ. Changes in Passive and Dynamic Mechanical Environments Promote Differentiation to a Contractile Phenotype in Vascular Smooth Muscle Cells. [Doctoral Dissertation]. Worcester Polytechnic Institute; 2015. Available from: etd-042915-160154 ; https://digitalcommons.wpi.edu/etd-dissertations/230


University of Arizona

28. Robb, Tiffany Marie. Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia .

Degree: 2012, University of Arizona

 Intimal hyperplasia (IH) is characteristic of a cell population increase within the innermost layer of the arterial wall. It is hypothesized that extracellular matrix vascular… (more)

Subjects/Keywords: Vascular endothleial cell; Vascular smooth muscle cell; Pharmacology & Toxicology; Intimal hyperplasia; Simvastatin

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APA (6th Edition):

Robb, T. M. (2012). Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/228464

Chicago Manual of Style (16th Edition):

Robb, Tiffany Marie. “Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia .” 2012. Masters Thesis, University of Arizona. Accessed August 08, 2020. http://hdl.handle.net/10150/228464.

MLA Handbook (7th Edition):

Robb, Tiffany Marie. “Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia .” 2012. Web. 08 Aug 2020.

Vancouver:

Robb TM. Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia . [Internet] [Masters thesis]. University of Arizona; 2012. [cited 2020 Aug 08]. Available from: http://hdl.handle.net/10150/228464.

Council of Science Editors:

Robb TM. Immune Mechanisms of Extracellular Matrix Remodeling in the Common Carotid: A Model of Intimal Hyperplasia . [Masters Thesis]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/228464


Case Western Reserve University

29. Beamish, Jeffrey Alan. Engineering Poly(Ethylene Glycol) Hydrogel Scaffolds to Modulate Smooth Muscle Cell Phenotype.

Degree: PhD, Biomedical Engineering, 2009, Case Western Reserve University

 This work investigated the hypothesis that smooth muscle cell (SMC) phenotype can be modulated by a cell-instructive hydrogel scaffold. By modulating SMCs toward a quiescent… (more)

Subjects/Keywords: Biomedical Research; Engineering; smooth muscle cell; hydrogel; tissue engineering; poly(ethylene glycol)

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APA (6th Edition):

Beamish, J. A. (2009). Engineering Poly(Ethylene Glycol) Hydrogel Scaffolds to Modulate Smooth Muscle Cell Phenotype. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1246649015

Chicago Manual of Style (16th Edition):

Beamish, Jeffrey Alan. “Engineering Poly(Ethylene Glycol) Hydrogel Scaffolds to Modulate Smooth Muscle Cell Phenotype.” 2009. Doctoral Dissertation, Case Western Reserve University. Accessed August 08, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1246649015.

MLA Handbook (7th Edition):

Beamish, Jeffrey Alan. “Engineering Poly(Ethylene Glycol) Hydrogel Scaffolds to Modulate Smooth Muscle Cell Phenotype.” 2009. Web. 08 Aug 2020.

Vancouver:

Beamish JA. Engineering Poly(Ethylene Glycol) Hydrogel Scaffolds to Modulate Smooth Muscle Cell Phenotype. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2009. [cited 2020 Aug 08]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1246649015.

Council of Science Editors:

Beamish JA. Engineering Poly(Ethylene Glycol) Hydrogel Scaffolds to Modulate Smooth Muscle Cell Phenotype. [Doctoral Dissertation]. Case Western Reserve University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1246649015


UCLA

30. Kobayashi, Masae. Maintenance of Interstitial Cells of Cajal for Intestinal Smooth Muscle Engineering.

Degree: Bioengineering, 2016, UCLA

 Interstitial cells of Cajal (ICC) are the pacemakers that enable the oriented intestinal smooth muscle layers to contract rhythmically and autonomously. However, disruption of ICC… (more)

Subjects/Keywords: Biomedical engineering; cellular alingnment; Interstitial Cells of Cajal; Motility; Scaffold; Smooth muscle cell; Tissue engineering

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kobayashi, M. (2016). Maintenance of Interstitial Cells of Cajal for Intestinal Smooth Muscle Engineering. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/54q8x8xq

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kobayashi, Masae. “Maintenance of Interstitial Cells of Cajal for Intestinal Smooth Muscle Engineering.” 2016. Thesis, UCLA. Accessed August 08, 2020. http://www.escholarship.org/uc/item/54q8x8xq.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kobayashi, Masae. “Maintenance of Interstitial Cells of Cajal for Intestinal Smooth Muscle Engineering.” 2016. Web. 08 Aug 2020.

Vancouver:

Kobayashi M. Maintenance of Interstitial Cells of Cajal for Intestinal Smooth Muscle Engineering. [Internet] [Thesis]. UCLA; 2016. [cited 2020 Aug 08]. Available from: http://www.escholarship.org/uc/item/54q8x8xq.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kobayashi M. Maintenance of Interstitial Cells of Cajal for Intestinal Smooth Muscle Engineering. [Thesis]. UCLA; 2016. Available from: http://www.escholarship.org/uc/item/54q8x8xq

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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