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You searched for subject:(Smad proteins). Showing records 1 – 15 of 15 total matches.

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University College Cork

1. Hegarty, Shane V. Characterisation of the role of canonical BMP-Smad 1/5/8 signalling in the development of ventral midbrain dopaminergic neurons.

Degree: 2013, University College Cork

 Ventral midbrain (VM) dopaminergic (DA) neurons, which project to the dorsal striatum via the nigrostriatal pathway, are progressively degenerated in Parkinson’s disease (PD). The identification… (more)

Subjects/Keywords: Bone morphogenetic proteins; Growth/differentiation factor 5; Ventral midbrain; Canonical Smad signalling; Nervous system development; Dopaminergic neuron

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APA (6th Edition):

Hegarty, S. V. (2013). Characterisation of the role of canonical BMP-Smad 1/5/8 signalling in the development of ventral midbrain dopaminergic neurons. (Thesis). University College Cork. Retrieved from http://hdl.handle.net/10468/1973

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hegarty, Shane V. “Characterisation of the role of canonical BMP-Smad 1/5/8 signalling in the development of ventral midbrain dopaminergic neurons.” 2013. Thesis, University College Cork. Accessed May 09, 2021. http://hdl.handle.net/10468/1973.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hegarty, Shane V. “Characterisation of the role of canonical BMP-Smad 1/5/8 signalling in the development of ventral midbrain dopaminergic neurons.” 2013. Web. 09 May 2021.

Vancouver:

Hegarty SV. Characterisation of the role of canonical BMP-Smad 1/5/8 signalling in the development of ventral midbrain dopaminergic neurons. [Internet] [Thesis]. University College Cork; 2013. [cited 2021 May 09]. Available from: http://hdl.handle.net/10468/1973.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hegarty SV. Characterisation of the role of canonical BMP-Smad 1/5/8 signalling in the development of ventral midbrain dopaminergic neurons. [Thesis]. University College Cork; 2013. Available from: http://hdl.handle.net/10468/1973

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

2. Polikandriotis, John Anastasios. Elucidating the regulation of vascular smooth muscle alpha-actin gene expression in fibroblasts.

Degree: PhD, Ohio State Biochemistry Program, 2004, The Ohio State University

 Myofibroblasts are contractile cells essential to the wound healing process that contain stress fibers composed of vascular smooth muscle alpha-actin (VSMA) and are thought to… (more)

Subjects/Keywords: VSMA; Pur; proteins; Sp1/3; Smad; Pur protein

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APA (6th Edition):

Polikandriotis, J. A. (2004). Elucidating the regulation of vascular smooth muscle alpha-actin gene expression in fibroblasts. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1078857443

Chicago Manual of Style (16th Edition):

Polikandriotis, John Anastasios. “Elucidating the regulation of vascular smooth muscle alpha-actin gene expression in fibroblasts.” 2004. Doctoral Dissertation, The Ohio State University. Accessed May 09, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1078857443.

MLA Handbook (7th Edition):

Polikandriotis, John Anastasios. “Elucidating the regulation of vascular smooth muscle alpha-actin gene expression in fibroblasts.” 2004. Web. 09 May 2021.

Vancouver:

Polikandriotis JA. Elucidating the regulation of vascular smooth muscle alpha-actin gene expression in fibroblasts. [Internet] [Doctoral dissertation]. The Ohio State University; 2004. [cited 2021 May 09]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1078857443.

Council of Science Editors:

Polikandriotis JA. Elucidating the regulation of vascular smooth muscle alpha-actin gene expression in fibroblasts. [Doctoral Dissertation]. The Ohio State University; 2004. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1078857443

3. Thaís Girão da Silva. Mecanismos moleculares envolvidos no fenótipo endotelial em resposta a estímulos físicos e químicos.

Degree: 2018, University of São Paulo

 O endotélio reveste a parede vascular e possui função essencial na manutenção da homeostase. A célula endotelial é capaz de perceber estímulos extracelulares, como fatores… (more)

Subjects/Keywords: Células endoteliais; Citoesqueleto de actina; Inibidor 1 de ativador de plasminogênio; Proteínas smad; Transdiferenciação celular; Transdução de sinais; Actin cytoskeleton; Cell transdifferentiation; Endothelial cells; Plasminogen activator inhibitor 1; Signal transduction; Smad proteins

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APA (6th Edition):

Silva, T. G. d. (2018). Mecanismos moleculares envolvidos no fenótipo endotelial em resposta a estímulos físicos e químicos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5166/tde-22102018-104726/

Chicago Manual of Style (16th Edition):

Silva, Thaís Girão da. “Mecanismos moleculares envolvidos no fenótipo endotelial em resposta a estímulos físicos e químicos.” 2018. Doctoral Dissertation, University of São Paulo. Accessed May 09, 2021. http://www.teses.usp.br/teses/disponiveis/5/5166/tde-22102018-104726/.

MLA Handbook (7th Edition):

Silva, Thaís Girão da. “Mecanismos moleculares envolvidos no fenótipo endotelial em resposta a estímulos físicos e químicos.” 2018. Web. 09 May 2021.

Vancouver:

Silva TGd. Mecanismos moleculares envolvidos no fenótipo endotelial em resposta a estímulos físicos e químicos. [Internet] [Doctoral dissertation]. University of São Paulo; 2018. [cited 2021 May 09]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5166/tde-22102018-104726/.

Council of Science Editors:

Silva TGd. Mecanismos moleculares envolvidos no fenótipo endotelial em resposta a estímulos físicos e químicos. [Doctoral Dissertation]. University of São Paulo; 2018. Available from: http://www.teses.usp.br/teses/disponiveis/5/5166/tde-22102018-104726/


Universidade Estadual de Campinas

4. Bersaneti, Joseli Assem, 1964-. Eventos moleculares associados ao aumento gengival induzido por ciclosporina A são atenuados pela superexpressão de Smad7.

Degree: Faculdade de Odontologia de Piracicaba; Programa de Pós-Gaduação em Estomatopatologia, 2011, Universidade Estadual de Campinas

Orientador: Ricardo Della Coletta

Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba

Made available in DSpace on 2018-08-18T17:48:45Z (GMT). No. of… (more)

Subjects/Keywords: Proteínas smad; Fator de crescimento transformador beta1; Metaloproteinase 2 da matriz; Smad proteins; Transforming growth factor beta 1; Matrix metalloproteinase 2

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APA (6th Edition):

Bersaneti, Joseli Assem, 1. (2011). Eventos moleculares associados ao aumento gengival induzido por ciclosporina A são atenuados pela superexpressão de Smad7. (Doctoral Dissertation). Universidade Estadual de Campinas. Retrieved from BERSANETI, Joseli Assem. Eventos moleculares associados ao aumento gengival induzido por ciclosporina A são atenuados pela superexpressão de Smad7. 2011. 49 f. Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba, Piracicaba, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/288719>. Acesso em: 18 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/288719

Chicago Manual of Style (16th Edition):

Bersaneti, Joseli Assem, 1964-. “Eventos moleculares associados ao aumento gengival induzido por ciclosporina A são atenuados pela superexpressão de Smad7.” 2011. Doctoral Dissertation, Universidade Estadual de Campinas. Accessed May 09, 2021. BERSANETI, Joseli Assem. Eventos moleculares associados ao aumento gengival induzido por ciclosporina A são atenuados pela superexpressão de Smad7. 2011. 49 f. Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba, Piracicaba, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/288719>. Acesso em: 18 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/288719.

MLA Handbook (7th Edition):

Bersaneti, Joseli Assem, 1964-. “Eventos moleculares associados ao aumento gengival induzido por ciclosporina A são atenuados pela superexpressão de Smad7.” 2011. Web. 09 May 2021.

Vancouver:

Bersaneti, Joseli Assem 1. Eventos moleculares associados ao aumento gengival induzido por ciclosporina A são atenuados pela superexpressão de Smad7. [Internet] [Doctoral dissertation]. Universidade Estadual de Campinas; 2011. [cited 2021 May 09]. Available from: BERSANETI, Joseli Assem. Eventos moleculares associados ao aumento gengival induzido por ciclosporina A são atenuados pela superexpressão de Smad7. 2011. 49 f. Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba, Piracicaba, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/288719>. Acesso em: 18 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/288719.

Council of Science Editors:

Bersaneti, Joseli Assem 1. Eventos moleculares associados ao aumento gengival induzido por ciclosporina A são atenuados pela superexpressão de Smad7. [Doctoral Dissertation]. Universidade Estadual de Campinas; 2011. Available from: BERSANETI, Joseli Assem. Eventos moleculares associados ao aumento gengival induzido por ciclosporina A são atenuados pela superexpressão de Smad7. 2011. 49 f. Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba, Piracicaba, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/288719>. Acesso em: 18 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/288719

5. Chen, Xiaochu. Nuclear Import of Smad: A Dissertation.

Degree: Interdisciplinary Graduate Program, Molecular Medicine, 2011, U of Massachusetts : Med

  Signal transduction by transforming growth factor β (TGF-β) cytokines is mediated by an evolutionarily conserved mechanism that depends on the Smad proteins to transduce… (more)

Subjects/Keywords: Smad Proteins; Receptors; Transforming Growth Factor beta; Active Transport; Cell Nucleus; Karyopherins; Drosophila Proteins; Nuclear Pore Complex Proteins; Amino Acids, Peptides, and Proteins; Animal Experimentation and Research; Biological Factors; Cell Biology; Cells

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APA (6th Edition):

Chen, X. (2011). Nuclear Import of Smad: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/560

Chicago Manual of Style (16th Edition):

Chen, Xiaochu. “Nuclear Import of Smad: A Dissertation.” 2011. Doctoral Dissertation, U of Massachusetts : Med. Accessed May 09, 2021. https://escholarship.umassmed.edu/gsbs_diss/560.

MLA Handbook (7th Edition):

Chen, Xiaochu. “Nuclear Import of Smad: A Dissertation.” 2011. Web. 09 May 2021.

Vancouver:

Chen X. Nuclear Import of Smad: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2011. [cited 2021 May 09]. Available from: https://escholarship.umassmed.edu/gsbs_diss/560.

Council of Science Editors:

Chen X. Nuclear Import of Smad: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2011. Available from: https://escholarship.umassmed.edu/gsbs_diss/560


University of Southern California

6. Pham, Linda Kim. Bimodal effects of bone morphogenetic proteins in prostate cancer.

Degree: PhD, Molecular Microbiology & Immunology, 2011, University of Southern California

 This dissertation describes observations made on the effect of bone morphogenetic protein (BMP) signaling in an aggressive human prostate cancer cell line, C4-2B, two murine… (more)

Subjects/Keywords: Prostate Cancer; Bone Morphogenetic Proteins; BMPs; BMP2; BMP7; apoptosis; Smad; JNK; Survivin; SDF-1; angiogenesis; Noggin; C4-2B; Cancer associated fibroblasts; tumor microenvironment; E8; cE1; Pten prostate cancer mouse model

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APA (6th Edition):

Pham, L. K. (2011). Bimodal effects of bone morphogenetic proteins in prostate cancer. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/441828/rec/1102

Chicago Manual of Style (16th Edition):

Pham, Linda Kim. “Bimodal effects of bone morphogenetic proteins in prostate cancer.” 2011. Doctoral Dissertation, University of Southern California. Accessed May 09, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/441828/rec/1102.

MLA Handbook (7th Edition):

Pham, Linda Kim. “Bimodal effects of bone morphogenetic proteins in prostate cancer.” 2011. Web. 09 May 2021.

Vancouver:

Pham LK. Bimodal effects of bone morphogenetic proteins in prostate cancer. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2021 May 09]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/441828/rec/1102.

Council of Science Editors:

Pham LK. Bimodal effects of bone morphogenetic proteins in prostate cancer. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/441828/rec/1102


Rutgers University

7. Wang, Guannan. The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation.

Degree: PhD, Biochemistry, 2008, Rutgers University

TGF-β regulates cell proliferation, differentiation, apoptosis, and extracellular matrix production. Smad proteins are central mediators of TGF-β signaling. Upon ligand binding, Smad2 and Smad3 are… (more)

Subjects/Keywords: Smad proteins; Transforming growth factors-beta; Cellular signal transduction; Cancer cells – Growth – Regulation

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APA (6th Edition):

Wang, G. (2008). The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17061

Chicago Manual of Style (16th Edition):

Wang, Guannan. “The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation.” 2008. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17061.

MLA Handbook (7th Edition):

Wang, Guannan. “The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation.” 2008. Web. 09 May 2021.

Vancouver:

Wang G. The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation. [Internet] [Doctoral dissertation]. Rutgers University; 2008. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17061.

Council of Science Editors:

Wang G. The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation. [Doctoral Dissertation]. Rutgers University; 2008. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17061

8. Fabiana Aparecida Mayrink de Oliveira. O EFEITO DO LASER DE BAIXA INTENSIDADE NA FIBROSE INTERSTICIAL RENAL.

Degree: 2011, Universidade Federal de Juiz de Fora

Justificativa e Objetivo: Independente da etiologia, a doença renal crônica (DRC) envolve fibrose generalizada e progressiva do tecido, atrofia tubular e a perda da função… (more)

Subjects/Keywords: Terapia de laser de baixa intensidade; obstrução ureteral; fibrose renal; doença renal crônica, fator transformador de crescimento beta; low-level laser therapy; ureteral obstruction; renal fibrosis; chronic kidney; failure; transforming growth factor beta; smad proteins; alfa-smooth muscle actin; SAUDE COLETIVA; proteínas smad; alfa-actina de músculo liso

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APA (6th Edition):

Oliveira, F. A. M. d. (2011). O EFEITO DO LASER DE BAIXA INTENSIDADE NA FIBROSE INTERSTICIAL RENAL. (Thesis). Universidade Federal de Juiz de Fora. Retrieved from http://www.bdtd.ufjf.br/tde_busca/arquivo.php?codArquivo=977

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oliveira, Fabiana Aparecida Mayrink de. “O EFEITO DO LASER DE BAIXA INTENSIDADE NA FIBROSE INTERSTICIAL RENAL.” 2011. Thesis, Universidade Federal de Juiz de Fora. Accessed May 09, 2021. http://www.bdtd.ufjf.br/tde_busca/arquivo.php?codArquivo=977.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oliveira, Fabiana Aparecida Mayrink de. “O EFEITO DO LASER DE BAIXA INTENSIDADE NA FIBROSE INTERSTICIAL RENAL.” 2011. Web. 09 May 2021.

Vancouver:

Oliveira FAMd. O EFEITO DO LASER DE BAIXA INTENSIDADE NA FIBROSE INTERSTICIAL RENAL. [Internet] [Thesis]. Universidade Federal de Juiz de Fora; 2011. [cited 2021 May 09]. Available from: http://www.bdtd.ufjf.br/tde_busca/arquivo.php?codArquivo=977.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oliveira FAMd. O EFEITO DO LASER DE BAIXA INTENSIDADE NA FIBROSE INTERSTICIAL RENAL. [Thesis]. Universidade Federal de Juiz de Fora; 2011. Available from: http://www.bdtd.ufjf.br/tde_busca/arquivo.php?codArquivo=977

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Hoot, Kristina E. The role of keratinocyte-specific Smad2 loss in skin squamous cell carcinomas.

Degree: PhD, 2008, Oregon Health Sciences University

Subjects/Keywords: Transforming Growth Factor beta  – deficiency; Carcinoma, Squamous Cell  – blood supply; Skin Neoplasms  – chemically induced; Smad Proteins

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APA (6th Edition):

Hoot, K. E. (2008). The role of keratinocyte-specific Smad2 loss in skin squamous cell carcinomas. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4028PG3 ; http://digitalcommons.ohsu.edu/etd/418

Chicago Manual of Style (16th Edition):

Hoot, Kristina E. “The role of keratinocyte-specific Smad2 loss in skin squamous cell carcinomas.” 2008. Doctoral Dissertation, Oregon Health Sciences University. Accessed May 09, 2021. doi:10.6083/M4028PG3 ; http://digitalcommons.ohsu.edu/etd/418.

MLA Handbook (7th Edition):

Hoot, Kristina E. “The role of keratinocyte-specific Smad2 loss in skin squamous cell carcinomas.” 2008. Web. 09 May 2021.

Vancouver:

Hoot KE. The role of keratinocyte-specific Smad2 loss in skin squamous cell carcinomas. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2008. [cited 2021 May 09]. Available from: doi:10.6083/M4028PG3 ; http://digitalcommons.ohsu.edu/etd/418.

Council of Science Editors:

Hoot KE. The role of keratinocyte-specific Smad2 loss in skin squamous cell carcinomas. [Doctoral Dissertation]. Oregon Health Sciences University; 2008. Available from: doi:10.6083/M4028PG3 ; http://digitalcommons.ohsu.edu/etd/418

10. Σταυρουλάκη, Μελανθία. Μοριακός μηχανισμός απευαισθητοποίησης μελανοκυττάρων στην κυτταροστατική δράση του αυξητικού παράγοντα μετασχηματισμού β (Transforming Growth Factor β, TGF-β) κατά την καρκινογένεση: στόχος φαρμακολογικής παρέμβασης στη θεραπεία του μελανώματος.

Degree: 2007, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Carcinogenesis is likely to result from the production of autocrine growth factors, as well as from loss of response to polypeptide growth inhibitors. TGF-β inhibits… (more)

Subjects/Keywords: Μελανοκύτταρα; Μελάνωμα; Εξαλλαγή; Μετασχηματίζων αυξητικός παράγοντας β; Πρωτείνες Smad; Πρωτεϊνική κινάση C; Καρκινογόνα; Κυτταρικός πολλαπλασιασμός; Melanocytes; Melanoma; Transformation; Transforming Growth Factor β; Smad proteins; Protein kinase C; Carcinogens; Cell proliferation

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APA (6th Edition):

Σταυρουλάκη, . . (2007). Μοριακός μηχανισμός απευαισθητοποίησης μελανοκυττάρων στην κυτταροστατική δράση του αυξητικού παράγοντα μετασχηματισμού β (Transforming Growth Factor β, TGF-β) κατά την καρκινογένεση: στόχος φαρμακολογικής παρέμβασης στη θεραπεία του μελανώματος. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/16272

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Σταυρουλάκη, Μελανθία. “Μοριακός μηχανισμός απευαισθητοποίησης μελανοκυττάρων στην κυτταροστατική δράση του αυξητικού παράγοντα μετασχηματισμού β (Transforming Growth Factor β, TGF-β) κατά την καρκινογένεση: στόχος φαρμακολογικής παρέμβασης στη θεραπεία του μελανώματος.” 2007. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed May 09, 2021. http://hdl.handle.net/10442/hedi/16272.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Σταυρουλάκη, Μελανθία. “Μοριακός μηχανισμός απευαισθητοποίησης μελανοκυττάρων στην κυτταροστατική δράση του αυξητικού παράγοντα μετασχηματισμού β (Transforming Growth Factor β, TGF-β) κατά την καρκινογένεση: στόχος φαρμακολογικής παρέμβασης στη θεραπεία του μελανώματος.” 2007. Web. 09 May 2021.

Vancouver:

Σταυρουλάκη . Μοριακός μηχανισμός απευαισθητοποίησης μελανοκυττάρων στην κυτταροστατική δράση του αυξητικού παράγοντα μετασχηματισμού β (Transforming Growth Factor β, TGF-β) κατά την καρκινογένεση: στόχος φαρμακολογικής παρέμβασης στη θεραπεία του μελανώματος. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2007. [cited 2021 May 09]. Available from: http://hdl.handle.net/10442/hedi/16272.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Σταυρουλάκη . Μοριακός μηχανισμός απευαισθητοποίησης μελανοκυττάρων στην κυτταροστατική δράση του αυξητικού παράγοντα μετασχηματισμού β (Transforming Growth Factor β, TGF-β) κατά την καρκινογένεση: στόχος φαρμακολογικής παρέμβασης στη θεραπεία του μελανώματος. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2007. Available from: http://hdl.handle.net/10442/hedi/16272

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Πανοπούλου, Αικατερίνη. Αγγειογένεση και κακοήθης μετασχηματισμός: ρόλος και μηχανισμός δράσης της ακτιβίνης Α.

Degree: 2004, University of Ioannina; Πανεπιστήμιο Ιωαννίνων

Subjects/Keywords: Νευροβλαστώματα; Ενίσχυση του N-Myc ογκογονιδίου; Αυξητικός παράγοντας μετασχηματισμού-Β; Αγγειογένεση; Ακτιβίνη Α; Πρωτείνες Smad; Ενδοσώματα; Neuroblastoma; N-Myc amplification; Angiogenesis; Transforming Growth Factor β; Activin A; Smad proteins; SARA; Endosomes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Πανοπούλου, . . (2004). Αγγειογένεση και κακοήθης μετασχηματισμός: ρόλος και μηχανισμός δράσης της ακτιβίνης Α. (Thesis). University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Retrieved from http://hdl.handle.net/10442/hedi/24094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Πανοπούλου, Αικατερίνη. “Αγγειογένεση και κακοήθης μετασχηματισμός: ρόλος και μηχανισμός δράσης της ακτιβίνης Α.” 2004. Thesis, University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Accessed May 09, 2021. http://hdl.handle.net/10442/hedi/24094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Πανοπούλου, Αικατερίνη. “Αγγειογένεση και κακοήθης μετασχηματισμός: ρόλος και μηχανισμός δράσης της ακτιβίνης Α.” 2004. Web. 09 May 2021.

Vancouver:

Πανοπούλου . Αγγειογένεση και κακοήθης μετασχηματισμός: ρόλος και μηχανισμός δράσης της ακτιβίνης Α. [Internet] [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2004. [cited 2021 May 09]. Available from: http://hdl.handle.net/10442/hedi/24094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Πανοπούλου . Αγγειογένεση και κακοήθης μετασχηματισμός: ρόλος και μηχανισμός δράσης της ακτιβίνης Α. [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2004. Available from: http://hdl.handle.net/10442/hedi/24094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Βασιλάκη, Ελευθερία. Μελέτη του μηχανισμού μεταγραφικής ρύθμισης του γονιδίου της μικρής GTPασης RHOB από το μονοπάτι του μετασχηματίζοντα αυξητικού παράγοντα β (TGFβ-1) και ο ρόλος της στις TGFβ-1- επαγώμενες αποκρίσεις του κυττάρου.

Degree: 2009, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Rho proteins are characterized as molecular switches that control many cellular processes including actin and microtubule cytoskeleton organization, cell division, motility, cell adhesion, vesicular trafficking,… (more)

Subjects/Keywords: Μετασχηματίζων αυξητικός παράγοντας β; Μεταγραφική ρύθμιση; Πρωτείνες Smad; Πρωτεΐνες ERK 1/2; Μεταγραφικός παράγοντας NF-Y; Κυτταρική μετακίνηση; Επιθήλιο-μεσεγχυματική μετατροπή των κυττάρων; Κυτταρική ανάπτυξη; RHOB GTPαση; Transforming growth factor β; RHOB GTPase; Transcriptional regulation; Smad proteins; ERK1/2 proteins; Transcription factor NF-Y; Cell migration; Epithelial to mesenchymal transition; Cell growth

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Βασιλάκη, . . (2009). Μελέτη του μηχανισμού μεταγραφικής ρύθμισης του γονιδίου της μικρής GTPασης RHOB από το μονοπάτι του μετασχηματίζοντα αυξητικού παράγοντα β (TGFβ-1) και ο ρόλος της στις TGFβ-1- επαγώμενες αποκρίσεις του κυττάρου. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/18295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Βασιλάκη, Ελευθερία. “Μελέτη του μηχανισμού μεταγραφικής ρύθμισης του γονιδίου της μικρής GTPασης RHOB από το μονοπάτι του μετασχηματίζοντα αυξητικού παράγοντα β (TGFβ-1) και ο ρόλος της στις TGFβ-1- επαγώμενες αποκρίσεις του κυττάρου.” 2009. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed May 09, 2021. http://hdl.handle.net/10442/hedi/18295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Βασιλάκη, Ελευθερία. “Μελέτη του μηχανισμού μεταγραφικής ρύθμισης του γονιδίου της μικρής GTPασης RHOB από το μονοπάτι του μετασχηματίζοντα αυξητικού παράγοντα β (TGFβ-1) και ο ρόλος της στις TGFβ-1- επαγώμενες αποκρίσεις του κυττάρου.” 2009. Web. 09 May 2021.

Vancouver:

Βασιλάκη . Μελέτη του μηχανισμού μεταγραφικής ρύθμισης του γονιδίου της μικρής GTPασης RHOB από το μονοπάτι του μετασχηματίζοντα αυξητικού παράγοντα β (TGFβ-1) και ο ρόλος της στις TGFβ-1- επαγώμενες αποκρίσεις του κυττάρου. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2009. [cited 2021 May 09]. Available from: http://hdl.handle.net/10442/hedi/18295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Βασιλάκη . Μελέτη του μηχανισμού μεταγραφικής ρύθμισης του γονιδίου της μικρής GTPασης RHOB από το μονοπάτι του μετασχηματίζοντα αυξητικού παράγοντα β (TGFβ-1) και ο ρόλος της στις TGFβ-1- επαγώμενες αποκρίσεις του κυττάρου. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2009. Available from: http://hdl.handle.net/10442/hedi/18295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

13. Tannehill-Gregg, Sarah. The development and characterization of animal models of squamous cell carcinoma: the roles of parathyroid hormone-related protein, transforming growth factor-Β, and the osteoclast in disease progression.

Degree: PhD, Veterinary Biosciences, 2005, The Ohio State University

 Squamous cell carcinoma (SCC) is a malignant tumor of keratinocytes. Head and neck SCC (H/N SCC) is a common diagnosis in the cat. SCCs often… (more)

Subjects/Keywords: Animal models; Squamous cell carcinoma; Transforming growth factor-&914; Parathyroid hormone-related protein; Osteoclasts; Bone metastasis; Bioluminescent imaging; Smad proteins; Epithelial carcinogenesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tannehill-Gregg, S. (2005). The development and characterization of animal models of squamous cell carcinoma: the roles of parathyroid hormone-related protein, transforming growth factor-Β, and the osteoclast in disease progression. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1110380593

Chicago Manual of Style (16th Edition):

Tannehill-Gregg, Sarah. “The development and characterization of animal models of squamous cell carcinoma: the roles of parathyroid hormone-related protein, transforming growth factor-Β, and the osteoclast in disease progression.” 2005. Doctoral Dissertation, The Ohio State University. Accessed May 09, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1110380593.

MLA Handbook (7th Edition):

Tannehill-Gregg, Sarah. “The development and characterization of animal models of squamous cell carcinoma: the roles of parathyroid hormone-related protein, transforming growth factor-Β, and the osteoclast in disease progression.” 2005. Web. 09 May 2021.

Vancouver:

Tannehill-Gregg S. The development and characterization of animal models of squamous cell carcinoma: the roles of parathyroid hormone-related protein, transforming growth factor-Β, and the osteoclast in disease progression. [Internet] [Doctoral dissertation]. The Ohio State University; 2005. [cited 2021 May 09]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1110380593.

Council of Science Editors:

Tannehill-Gregg S. The development and characterization of animal models of squamous cell carcinoma: the roles of parathyroid hormone-related protein, transforming growth factor-Β, and the osteoclast in disease progression. [Doctoral Dissertation]. The Ohio State University; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1110380593

14. Χήρα, Παναγιώτα. Μεταγωγή του σήματος από ακτιβίνη Α στα ενδοθηλιακά κύτταρα: διερεύνηση των μηχανισμών και διασύνδεση με την ενδοκυτταρική μεταφορά μεμβρανών.

Degree: 2009, University of Ioannina; Πανεπιστήμιο Ιωαννίνων

 TGFβ superfamily is comprised of a large number of growth factors, which play an important role in a variety of cellular functions. The common SMAD4… (more)

Subjects/Keywords: Παράγοντες ADP - Ριβοζυλίωσης; SMAD αλληλεπιδρώσες πρωτεΐνες; Μεμβρανικη ενδοκυττάρωση - κυκλοφορία; Μεταγωγή του βήματος από ακτιβίνη A / TGFβ; Ενδοκυττάρωση υποδοχέα; ARNO παράγοντας ανταλλαγής του νουκλεουδίου της γονανίνης; ADP - Ribosylation factors ( ARFs); SMAD interacting proteins; Membrane endocytosis - trafficking; Activin A / TGF - β signal transduction; Receptor endocytosis; ARNO GEF (guanine nucleotide exchange factor)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Χήρα, . . (2009). Μεταγωγή του σήματος από ακτιβίνη Α στα ενδοθηλιακά κύτταρα: διερεύνηση των μηχανισμών και διασύνδεση με την ενδοκυτταρική μεταφορά μεμβρανών. (Thesis). University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Retrieved from http://hdl.handle.net/10442/hedi/17442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Χήρα, Παναγιώτα. “Μεταγωγή του σήματος από ακτιβίνη Α στα ενδοθηλιακά κύτταρα: διερεύνηση των μηχανισμών και διασύνδεση με την ενδοκυτταρική μεταφορά μεμβρανών.” 2009. Thesis, University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Accessed May 09, 2021. http://hdl.handle.net/10442/hedi/17442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Χήρα, Παναγιώτα. “Μεταγωγή του σήματος από ακτιβίνη Α στα ενδοθηλιακά κύτταρα: διερεύνηση των μηχανισμών και διασύνδεση με την ενδοκυτταρική μεταφορά μεμβρανών.” 2009. Web. 09 May 2021.

Vancouver:

Χήρα . Μεταγωγή του σήματος από ακτιβίνη Α στα ενδοθηλιακά κύτταρα: διερεύνηση των μηχανισμών και διασύνδεση με την ενδοκυτταρική μεταφορά μεμβρανών. [Internet] [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2009. [cited 2021 May 09]. Available from: http://hdl.handle.net/10442/hedi/17442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Χήρα . Μεταγωγή του σήματος από ακτιβίνη Α στα ενδοθηλιακά κύτταρα: διερεύνηση των μηχανισμών και διασύνδεση με την ενδοκυτταρική μεταφορά μεμβρανών. [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2009. Available from: http://hdl.handle.net/10442/hedi/17442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Oliveira, Fabiana Aparecida Mayrink de. O efeito do laser de baixa intensidade na fibrose intersticial renal.

Degree: 2011, Universidade Federal de Juiz de Fora; Programa de Pós-graduação em Saúde Brasileira; UFJF; Brasil; Faculdade de Medicina

CAPES - Coordenação de Aperfeiçoamento de Pessoal de Nível Superior

FAPEMIG - Fundação de Amparo à Pesquisa do Estado de Minas Gerais

Justificativa e Objetivo:… (more)

Subjects/Keywords: CNPQ::CIENCIAS DA SAUDE; Terapia de laser de baixa intensidade; Obstrução ureteral; Fibrose renal; Doença renal crônica; Fator transformador de crescimento beta; Proteínas smad; Alfa-actina de músculo liso; Low-level laser therapy; Ureteral obstruction; Renal fibrosis; Chronic kidney failure; Transforming growth factor beta; Smad proteins; Alfa-smooth muscle actin

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APA (6th Edition):

Oliveira, F. A. M. d. (2011). O efeito do laser de baixa intensidade na fibrose intersticial renal. (Masters Thesis). Universidade Federal de Juiz de Fora; Programa de Pós-graduação em Saúde Brasileira; UFJF; Brasil; Faculdade de Medicina. Retrieved from https://repositorio.ufjf.br/jspui/handle/ufjf/2129

Chicago Manual of Style (16th Edition):

Oliveira, Fabiana Aparecida Mayrink de. “O efeito do laser de baixa intensidade na fibrose intersticial renal.” 2011. Masters Thesis, Universidade Federal de Juiz de Fora; Programa de Pós-graduação em Saúde Brasileira; UFJF; Brasil; Faculdade de Medicina. Accessed May 09, 2021. https://repositorio.ufjf.br/jspui/handle/ufjf/2129.

MLA Handbook (7th Edition):

Oliveira, Fabiana Aparecida Mayrink de. “O efeito do laser de baixa intensidade na fibrose intersticial renal.” 2011. Web. 09 May 2021.

Vancouver:

Oliveira FAMd. O efeito do laser de baixa intensidade na fibrose intersticial renal. [Internet] [Masters thesis]. Universidade Federal de Juiz de Fora; Programa de Pós-graduação em Saúde Brasileira; UFJF; Brasil; Faculdade de Medicina; 2011. [cited 2021 May 09]. Available from: https://repositorio.ufjf.br/jspui/handle/ufjf/2129.

Council of Science Editors:

Oliveira FAMd. O efeito do laser de baixa intensidade na fibrose intersticial renal. [Masters Thesis]. Universidade Federal de Juiz de Fora; Programa de Pós-graduação em Saúde Brasileira; UFJF; Brasil; Faculdade de Medicina; 2011. Available from: https://repositorio.ufjf.br/jspui/handle/ufjf/2129

.