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You searched for subject:(Skeletal Muscle). Showing records 1 – 30 of 1009 total matches.

[1] [2] [3] [4] [5] … [34]

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University of Georgia

1. Foltz, Steven James. Dystroglycanopathy-type muscular dystrophy: evaluation of pathophysiology and therapeutic targeting in a mouse model of fukutin deficiency.

Degree: PhD, Pharmacy, 2016, University of Georgia

 Muscular dystrophies are a group of genetic disorders characterized by muscle necrosis, atrophy, and progressive weakness. Several of these diseases are caused by defects in… (more)

Subjects/Keywords: skeletal muscle

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Foltz, S. J. (2016). Dystroglycanopathy-type muscular dystrophy: evaluation of pathophysiology and therapeutic targeting in a mouse model of fukutin deficiency. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/foltz_steven_j_201608_phd

Chicago Manual of Style (16th Edition):

Foltz, Steven James. “Dystroglycanopathy-type muscular dystrophy: evaluation of pathophysiology and therapeutic targeting in a mouse model of fukutin deficiency.” 2016. Doctoral Dissertation, University of Georgia. Accessed May 22, 2019. http://purl.galileo.usg.edu/uga_etd/foltz_steven_j_201608_phd.

MLA Handbook (7th Edition):

Foltz, Steven James. “Dystroglycanopathy-type muscular dystrophy: evaluation of pathophysiology and therapeutic targeting in a mouse model of fukutin deficiency.” 2016. Web. 22 May 2019.

Vancouver:

Foltz SJ. Dystroglycanopathy-type muscular dystrophy: evaluation of pathophysiology and therapeutic targeting in a mouse model of fukutin deficiency. [Internet] [Doctoral dissertation]. University of Georgia; 2016. [cited 2019 May 22]. Available from: http://purl.galileo.usg.edu/uga_etd/foltz_steven_j_201608_phd.

Council of Science Editors:

Foltz SJ. Dystroglycanopathy-type muscular dystrophy: evaluation of pathophysiology and therapeutic targeting in a mouse model of fukutin deficiency. [Doctoral Dissertation]. University of Georgia; 2016. Available from: http://purl.galileo.usg.edu/uga_etd/foltz_steven_j_201608_phd

2. Lee, Peter H.U. Skeletal Muscle Atrophy in Bioengineered Skeletal Muscle: A New Model System.

Degree: PhD, Pathobiology, 2015, Brown University

Skeletal muscle atrophy has been well characterized in various animal models, and while certain pathways that lead to disuse atrophy and its associated functional deficits… (more)

Subjects/Keywords: skeletal muscle

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APA (6th Edition):

Lee, P. H. U. (2015). Skeletal Muscle Atrophy in Bioengineered Skeletal Muscle: A New Model System. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:419515/

Chicago Manual of Style (16th Edition):

Lee, Peter H U. “Skeletal Muscle Atrophy in Bioengineered Skeletal Muscle: A New Model System.” 2015. Doctoral Dissertation, Brown University. Accessed May 22, 2019. https://repository.library.brown.edu/studio/item/bdr:419515/.

MLA Handbook (7th Edition):

Lee, Peter H U. “Skeletal Muscle Atrophy in Bioengineered Skeletal Muscle: A New Model System.” 2015. Web. 22 May 2019.

Vancouver:

Lee PHU. Skeletal Muscle Atrophy in Bioengineered Skeletal Muscle: A New Model System. [Internet] [Doctoral dissertation]. Brown University; 2015. [cited 2019 May 22]. Available from: https://repository.library.brown.edu/studio/item/bdr:419515/.

Council of Science Editors:

Lee PHU. Skeletal Muscle Atrophy in Bioengineered Skeletal Muscle: A New Model System. [Doctoral Dissertation]. Brown University; 2015. Available from: https://repository.library.brown.edu/studio/item/bdr:419515/


University of Illinois – Chicago

3. Chaves, Alec B. Effect of Acute Aerobic Exercise on the Thioredoxin System in Lean, Healthy Subjects.

Degree: 2017, University of Illinois – Chicago

 Acute aerobic exercise was able to significantly reduce TXNIP and REDD1 protein expression. However, the change in TXNIP protein expression could not be explained by… (more)

Subjects/Keywords: Thioredoxin; skeletal muscle

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APA (6th Edition):

Chaves, A. B. (2017). Effect of Acute Aerobic Exercise on the Thioredoxin System in Lean, Healthy Subjects. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21911

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chaves, Alec B. “Effect of Acute Aerobic Exercise on the Thioredoxin System in Lean, Healthy Subjects.” 2017. Thesis, University of Illinois – Chicago. Accessed May 22, 2019. http://hdl.handle.net/10027/21911.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chaves, Alec B. “Effect of Acute Aerobic Exercise on the Thioredoxin System in Lean, Healthy Subjects.” 2017. Web. 22 May 2019.

Vancouver:

Chaves AB. Effect of Acute Aerobic Exercise on the Thioredoxin System in Lean, Healthy Subjects. [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2019 May 22]. Available from: http://hdl.handle.net/10027/21911.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chaves AB. Effect of Acute Aerobic Exercise on the Thioredoxin System in Lean, Healthy Subjects. [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/21911

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Waterloo

4. Mitchell, Andrew. Morphological and Apoptotic Alterations in Skeletal Muscle of Mice Deficient in Apoptosis Repressor with Caspase Recruitment Domain.

Degree: 2012, University of Waterloo

 Altered apoptotic signaling in skeletal muscle has been observed in a number of disease states associated with skeletal muscle atrophy. Therefore, understanding the mechanisms that… (more)

Subjects/Keywords: Skeletal Muscle; Apoptosis

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APA (6th Edition):

Mitchell, A. (2012). Morphological and Apoptotic Alterations in Skeletal Muscle of Mice Deficient in Apoptosis Repressor with Caspase Recruitment Domain. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/6434

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mitchell, Andrew. “Morphological and Apoptotic Alterations in Skeletal Muscle of Mice Deficient in Apoptosis Repressor with Caspase Recruitment Domain.” 2012. Thesis, University of Waterloo. Accessed May 22, 2019. http://hdl.handle.net/10012/6434.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mitchell, Andrew. “Morphological and Apoptotic Alterations in Skeletal Muscle of Mice Deficient in Apoptosis Repressor with Caspase Recruitment Domain.” 2012. Web. 22 May 2019.

Vancouver:

Mitchell A. Morphological and Apoptotic Alterations in Skeletal Muscle of Mice Deficient in Apoptosis Repressor with Caspase Recruitment Domain. [Internet] [Thesis]. University of Waterloo; 2012. [cited 2019 May 22]. Available from: http://hdl.handle.net/10012/6434.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mitchell A. Morphological and Apoptotic Alterations in Skeletal Muscle of Mice Deficient in Apoptosis Repressor with Caspase Recruitment Domain. [Thesis]. University of Waterloo; 2012. Available from: http://hdl.handle.net/10012/6434

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

5. Coriati, Adèle. Skeletal Muscle Specific IRES Activity of Utrophin A Is Enhanced by Eef1a2 .

Degree: 2011, University of Ottawa

 Understanding the regulatory mechanisms controlling utrophin A expression at the sarcolemma of dystrophic muscles will facilitate the development of therapeutic strategies to ameliorate the pathophysiological… (more)

Subjects/Keywords: skeletal muscle; utrophin

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APA (6th Edition):

Coriati, A. (2011). Skeletal Muscle Specific IRES Activity of Utrophin A Is Enhanced by Eef1a2 . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/19866

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Coriati, Adèle. “Skeletal Muscle Specific IRES Activity of Utrophin A Is Enhanced by Eef1a2 .” 2011. Thesis, University of Ottawa. Accessed May 22, 2019. http://hdl.handle.net/10393/19866.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Coriati, Adèle. “Skeletal Muscle Specific IRES Activity of Utrophin A Is Enhanced by Eef1a2 .” 2011. Web. 22 May 2019.

Vancouver:

Coriati A. Skeletal Muscle Specific IRES Activity of Utrophin A Is Enhanced by Eef1a2 . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2019 May 22]. Available from: http://hdl.handle.net/10393/19866.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Coriati A. Skeletal Muscle Specific IRES Activity of Utrophin A Is Enhanced by Eef1a2 . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/19866

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Deakin University

6. Sepulveda, Patricio. Development of novel interventions to enhance muscle function.

Degree: School of Exercise and Nutrition Sciences, 2012, Deakin University

 The research explored the effects of novel potential therapeutics interventions (made of genetically modified viral vectors) to improve skeletal muscle force, increase resistance to fatigue… (more)

Subjects/Keywords: skeletal muscle physiology

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APA (6th Edition):

Sepulveda, P. (2012). Development of novel interventions to enhance muscle function. (Thesis). Deakin University. Retrieved from http://hdl.handle.net/10536/DRO/DU:30056802

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sepulveda, Patricio. “Development of novel interventions to enhance muscle function.” 2012. Thesis, Deakin University. Accessed May 22, 2019. http://hdl.handle.net/10536/DRO/DU:30056802.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sepulveda, Patricio. “Development of novel interventions to enhance muscle function.” 2012. Web. 22 May 2019.

Vancouver:

Sepulveda P. Development of novel interventions to enhance muscle function. [Internet] [Thesis]. Deakin University; 2012. [cited 2019 May 22]. Available from: http://hdl.handle.net/10536/DRO/DU:30056802.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sepulveda P. Development of novel interventions to enhance muscle function. [Thesis]. Deakin University; 2012. Available from: http://hdl.handle.net/10536/DRO/DU:30056802

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Wang, Lin. Minimally Invasive Approach to the Repair of InjuredSkeletal Muscle with Biodegradable Scaffolds.

Degree: PhD, Artificial Organs, Biomaterials, and Cell Technology, 2011, Brown University

 The repair of damaged skeletal muscle may be enhanced by the injection of muscle stem cells and/or recombinant growth factors, but is currently limited by… (more)

Subjects/Keywords: injured skeletal muscle

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APA (6th Edition):

Wang, L. (2011). Minimally Invasive Approach to the Repair of InjuredSkeletal Muscle with Biodegradable Scaffolds. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:11244/

Chicago Manual of Style (16th Edition):

Wang, Lin. “Minimally Invasive Approach to the Repair of InjuredSkeletal Muscle with Biodegradable Scaffolds.” 2011. Doctoral Dissertation, Brown University. Accessed May 22, 2019. https://repository.library.brown.edu/studio/item/bdr:11244/.

MLA Handbook (7th Edition):

Wang, Lin. “Minimally Invasive Approach to the Repair of InjuredSkeletal Muscle with Biodegradable Scaffolds.” 2011. Web. 22 May 2019.

Vancouver:

Wang L. Minimally Invasive Approach to the Repair of InjuredSkeletal Muscle with Biodegradable Scaffolds. [Internet] [Doctoral dissertation]. Brown University; 2011. [cited 2019 May 22]. Available from: https://repository.library.brown.edu/studio/item/bdr:11244/.

Council of Science Editors:

Wang L. Minimally Invasive Approach to the Repair of InjuredSkeletal Muscle with Biodegradable Scaffolds. [Doctoral Dissertation]. Brown University; 2011. Available from: https://repository.library.brown.edu/studio/item/bdr:11244/


University of Alberta

8. Stretch, Cynthia. Study of molecular and metabolic changes in skeletal muscle in response to cancer.

Degree: PhD, Department of Oncology, 2013, University of Alberta

 Cancer cachexia is a multifactorial syndrome characterized by involuntary weight loss, wasting of skeletal muscle driven by reduced food intake and abnormal metabolism. Cachexia has… (more)

Subjects/Keywords: cachexia; microarray; skeletal muscle; metabolomics

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APA (6th Edition):

Stretch, C. (2013). Study of molecular and metabolic changes in skeletal muscle in response to cancer. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/f4752h156

Chicago Manual of Style (16th Edition):

Stretch, Cynthia. “Study of molecular and metabolic changes in skeletal muscle in response to cancer.” 2013. Doctoral Dissertation, University of Alberta. Accessed May 22, 2019. https://era.library.ualberta.ca/files/f4752h156.

MLA Handbook (7th Edition):

Stretch, Cynthia. “Study of molecular and metabolic changes in skeletal muscle in response to cancer.” 2013. Web. 22 May 2019.

Vancouver:

Stretch C. Study of molecular and metabolic changes in skeletal muscle in response to cancer. [Internet] [Doctoral dissertation]. University of Alberta; 2013. [cited 2019 May 22]. Available from: https://era.library.ualberta.ca/files/f4752h156.

Council of Science Editors:

Stretch C. Study of molecular and metabolic changes in skeletal muscle in response to cancer. [Doctoral Dissertation]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/f4752h156


University of Guelph

9. Leung, Matthew. Skeletal Muscle Regulatory Volume Response by Monocarboxylate Transporters to Increased Extracellular Lactate .

Degree: 2011, University of Guelph

 The purpose of this thesis was to investigate the role of lactate in the regulatory volume response of mammalian skeletal muscle to hypertonic challenge-induced cell… (more)

Subjects/Keywords: skeletal muscle; volume regulation; lactate

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APA (6th Edition):

Leung, M. (2011). Skeletal Muscle Regulatory Volume Response by Monocarboxylate Transporters to Increased Extracellular Lactate . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3175

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Leung, Matthew. “Skeletal Muscle Regulatory Volume Response by Monocarboxylate Transporters to Increased Extracellular Lactate .” 2011. Thesis, University of Guelph. Accessed May 22, 2019. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3175.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Leung, Matthew. “Skeletal Muscle Regulatory Volume Response by Monocarboxylate Transporters to Increased Extracellular Lactate .” 2011. Web. 22 May 2019.

Vancouver:

Leung M. Skeletal Muscle Regulatory Volume Response by Monocarboxylate Transporters to Increased Extracellular Lactate . [Internet] [Thesis]. University of Guelph; 2011. [cited 2019 May 22]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3175.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Leung M. Skeletal Muscle Regulatory Volume Response by Monocarboxylate Transporters to Increased Extracellular Lactate . [Thesis]. University of Guelph; 2011. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3175

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Guelph

10. Lally, James. The Implications of CD36 Alteration on Rodent Skeletal Muscle Lipid Metabolism .

Degree: 2012, University of Guelph

 Fatty acid transport across the plasma membrane is an important site of regulation in skeletal muscle lipid metabolism, and is governed by a number of… (more)

Subjects/Keywords: skeletal muscle fatty acid metabolism

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APA (6th Edition):

Lally, J. (2012). The Implications of CD36 Alteration on Rodent Skeletal Muscle Lipid Metabolism . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lally, James. “The Implications of CD36 Alteration on Rodent Skeletal Muscle Lipid Metabolism .” 2012. Thesis, University of Guelph. Accessed May 22, 2019. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lally, James. “The Implications of CD36 Alteration on Rodent Skeletal Muscle Lipid Metabolism .” 2012. Web. 22 May 2019.

Vancouver:

Lally J. The Implications of CD36 Alteration on Rodent Skeletal Muscle Lipid Metabolism . [Internet] [Thesis]. University of Guelph; 2012. [cited 2019 May 22]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lally J. The Implications of CD36 Alteration on Rodent Skeletal Muscle Lipid Metabolism . [Thesis]. University of Guelph; 2012. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Catholique de Louvain

11. Gilson, Hélène. Muscle hypertrophy induced by myostatin inhibition : a new therapeutic approach of muscle atrophy.

Degree: 2009, Université Catholique de Louvain

Increasing size and strength of skeletal muscle represents a promising therapeutic strategy for muscular disorders. One possible new tool is Myostatin (Mstn) because it plays… (more)

Subjects/Keywords: Myostatin; Follistatin; Skeletal muscle

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APA (6th Edition):

Gilson, H. (2009). Muscle hypertrophy induced by myostatin inhibition : a new therapeutic approach of muscle atrophy. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/24943

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gilson, Hélène. “Muscle hypertrophy induced by myostatin inhibition : a new therapeutic approach of muscle atrophy.” 2009. Thesis, Université Catholique de Louvain. Accessed May 22, 2019. http://hdl.handle.net/2078.1/24943.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gilson, Hélène. “Muscle hypertrophy induced by myostatin inhibition : a new therapeutic approach of muscle atrophy.” 2009. Web. 22 May 2019.

Vancouver:

Gilson H. Muscle hypertrophy induced by myostatin inhibition : a new therapeutic approach of muscle atrophy. [Internet] [Thesis]. Université Catholique de Louvain; 2009. [cited 2019 May 22]. Available from: http://hdl.handle.net/2078.1/24943.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gilson H. Muscle hypertrophy induced by myostatin inhibition : a new therapeutic approach of muscle atrophy. [Thesis]. Université Catholique de Louvain; 2009. Available from: http://hdl.handle.net/2078.1/24943

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Texas

12. Luk, Hui Ying. Effect of the Resistance Exercise-Induced Hormonal Changes on Satellite Cell Myogenic State.

Degree: 2018, University of North Texas

Skeletal muscle satellite cells are important for muscle repairing and muscle mass growth. For a successful muscle regenerative process, satellite cells have to sequentially undergoing… (more)

Subjects/Keywords: Skeletal Muscle Stem Cells; Hormones

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APA (6th Edition):

Luk, H. Y. (2018). Effect of the Resistance Exercise-Induced Hormonal Changes on Satellite Cell Myogenic State. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc1157528/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Luk, Hui Ying. “Effect of the Resistance Exercise-Induced Hormonal Changes on Satellite Cell Myogenic State.” 2018. Thesis, University of North Texas. Accessed May 22, 2019. https://digital.library.unt.edu/ark:/67531/metadc1157528/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Luk, Hui Ying. “Effect of the Resistance Exercise-Induced Hormonal Changes on Satellite Cell Myogenic State.” 2018. Web. 22 May 2019.

Vancouver:

Luk HY. Effect of the Resistance Exercise-Induced Hormonal Changes on Satellite Cell Myogenic State. [Internet] [Thesis]. University of North Texas; 2018. [cited 2019 May 22]. Available from: https://digital.library.unt.edu/ark:/67531/metadc1157528/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Luk HY. Effect of the Resistance Exercise-Induced Hormonal Changes on Satellite Cell Myogenic State. [Thesis]. University of North Texas; 2018. Available from: https://digital.library.unt.edu/ark:/67531/metadc1157528/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

13. Dieli-Conwright, Christina M. Influence of hormone replacement therapy with maximal eccentric exercise on estrogen receptor coregulators and skeletal muscle damage in postmenopausal women.

Degree: PhD, Biokinesiology & Physical Therapy, 2009, University of Southern California

 The use of hormone replacement therapy (HRT) is a potential treatment to relieve symptoms of menopause and prevent the onset of disease such as osteoporosis… (more)

Subjects/Keywords: skeletal muscle; resistance exercise; estrogen

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APA (6th Edition):

Dieli-Conwright, C. M. (2009). Influence of hormone replacement therapy with maximal eccentric exercise on estrogen receptor coregulators and skeletal muscle damage in postmenopausal women. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/218682/rec/3480

Chicago Manual of Style (16th Edition):

Dieli-Conwright, Christina M. “Influence of hormone replacement therapy with maximal eccentric exercise on estrogen receptor coregulators and skeletal muscle damage in postmenopausal women.” 2009. Doctoral Dissertation, University of Southern California. Accessed May 22, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/218682/rec/3480.

MLA Handbook (7th Edition):

Dieli-Conwright, Christina M. “Influence of hormone replacement therapy with maximal eccentric exercise on estrogen receptor coregulators and skeletal muscle damage in postmenopausal women.” 2009. Web. 22 May 2019.

Vancouver:

Dieli-Conwright CM. Influence of hormone replacement therapy with maximal eccentric exercise on estrogen receptor coregulators and skeletal muscle damage in postmenopausal women. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2019 May 22]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/218682/rec/3480.

Council of Science Editors:

Dieli-Conwright CM. Influence of hormone replacement therapy with maximal eccentric exercise on estrogen receptor coregulators and skeletal muscle damage in postmenopausal women. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/218682/rec/3480


University of Illinois – Chicago

14. Miranda Jr, Edwin R. Effects of Acute Aerobic Exercise Intensity on Plasma sRAGE in Lean Healthy Individuals.

Degree: 2016, University of Illinois – Chicago

Skeletal muscle inflammation is implicated in the pathogenesis of many health disparities including diabetes. The Receptor for Advanced Glycation End Products (RAGE) is a major… (more)

Subjects/Keywords: Exercise; Skeletal muscle; RAGE; sRAGE

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APA (6th Edition):

Miranda Jr, E. R. (2016). Effects of Acute Aerobic Exercise Intensity on Plasma sRAGE in Lean Healthy Individuals. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21285

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Miranda Jr, Edwin R. “Effects of Acute Aerobic Exercise Intensity on Plasma sRAGE in Lean Healthy Individuals.” 2016. Thesis, University of Illinois – Chicago. Accessed May 22, 2019. http://hdl.handle.net/10027/21285.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Miranda Jr, Edwin R. “Effects of Acute Aerobic Exercise Intensity on Plasma sRAGE in Lean Healthy Individuals.” 2016. Web. 22 May 2019.

Vancouver:

Miranda Jr ER. Effects of Acute Aerobic Exercise Intensity on Plasma sRAGE in Lean Healthy Individuals. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2019 May 22]. Available from: http://hdl.handle.net/10027/21285.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Miranda Jr ER. Effects of Acute Aerobic Exercise Intensity on Plasma sRAGE in Lean Healthy Individuals. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/21285

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

15. Al-Sajee, Dhuha. INVESTIGATING THE ROLES OF XIN IN SKELETAL MUSCLE AND ITS SATELLITE CELL POPULATION.

Degree: PhD, 2017, McMaster University

Skeletal muscle disease (myopathy) carries an enormous psychological, social and economic impact on the lives of the patients and their caregivers. There is also an… (more)

Subjects/Keywords: Xin; skeletal muscle; satellite cell

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APA (6th Edition):

Al-Sajee, D. (2017). INVESTIGATING THE ROLES OF XIN IN SKELETAL MUSCLE AND ITS SATELLITE CELL POPULATION. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/21246

Chicago Manual of Style (16th Edition):

Al-Sajee, Dhuha. “INVESTIGATING THE ROLES OF XIN IN SKELETAL MUSCLE AND ITS SATELLITE CELL POPULATION.” 2017. Doctoral Dissertation, McMaster University. Accessed May 22, 2019. http://hdl.handle.net/11375/21246.

MLA Handbook (7th Edition):

Al-Sajee, Dhuha. “INVESTIGATING THE ROLES OF XIN IN SKELETAL MUSCLE AND ITS SATELLITE CELL POPULATION.” 2017. Web. 22 May 2019.

Vancouver:

Al-Sajee D. INVESTIGATING THE ROLES OF XIN IN SKELETAL MUSCLE AND ITS SATELLITE CELL POPULATION. [Internet] [Doctoral dissertation]. McMaster University; 2017. [cited 2019 May 22]. Available from: http://hdl.handle.net/11375/21246.

Council of Science Editors:

Al-Sajee D. INVESTIGATING THE ROLES OF XIN IN SKELETAL MUSCLE AND ITS SATELLITE CELL POPULATION. [Doctoral Dissertation]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/21246


McMaster University

16. Hector, Amy. STRATEGIES TO IMPROVE SKELETAL MUSCLE PROTEIN TURNOVER DURING DIETARY ENERGY RESTRICTION.

Degree: PhD, 2017, McMaster University

Weight loss through dietary energy restriction (ER) is an effective method to promote fat mass loss. However, a negative consequence of ER is the loss… (more)

Subjects/Keywords: Energy Restriction; Skeletal Muscle

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APA (6th Edition):

Hector, A. (2017). STRATEGIES TO IMPROVE SKELETAL MUSCLE PROTEIN TURNOVER DURING DIETARY ENERGY RESTRICTION. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/22015

Chicago Manual of Style (16th Edition):

Hector, Amy. “STRATEGIES TO IMPROVE SKELETAL MUSCLE PROTEIN TURNOVER DURING DIETARY ENERGY RESTRICTION.” 2017. Doctoral Dissertation, McMaster University. Accessed May 22, 2019. http://hdl.handle.net/11375/22015.

MLA Handbook (7th Edition):

Hector, Amy. “STRATEGIES TO IMPROVE SKELETAL MUSCLE PROTEIN TURNOVER DURING DIETARY ENERGY RESTRICTION.” 2017. Web. 22 May 2019.

Vancouver:

Hector A. STRATEGIES TO IMPROVE SKELETAL MUSCLE PROTEIN TURNOVER DURING DIETARY ENERGY RESTRICTION. [Internet] [Doctoral dissertation]. McMaster University; 2017. [cited 2019 May 22]. Available from: http://hdl.handle.net/11375/22015.

Council of Science Editors:

Hector A. STRATEGIES TO IMPROVE SKELETAL MUSCLE PROTEIN TURNOVER DURING DIETARY ENERGY RESTRICTION. [Doctoral Dissertation]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/22015


Université Catholique de Louvain

17. Zbinden Foncea, Hermann. Activation of Toll Like receptors 2 and 4 by fatty acids in skeletal muscle.

Degree: 2013, Université Catholique de Louvain

Toll-like receptors 2 (TLR2) and 4 (TLR4) are present in the plasma membrane of skeletal muscles, where their functions are only partially understood. A variety… (more)

Subjects/Keywords: Fatty acids; Exercise; Skeletal muscle

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APA (6th Edition):

Zbinden Foncea, H. (2013). Activation of Toll Like receptors 2 and 4 by fatty acids in skeletal muscle. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/128993

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zbinden Foncea, Hermann. “Activation of Toll Like receptors 2 and 4 by fatty acids in skeletal muscle.” 2013. Thesis, Université Catholique de Louvain. Accessed May 22, 2019. http://hdl.handle.net/2078.1/128993.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zbinden Foncea, Hermann. “Activation of Toll Like receptors 2 and 4 by fatty acids in skeletal muscle.” 2013. Web. 22 May 2019.

Vancouver:

Zbinden Foncea H. Activation of Toll Like receptors 2 and 4 by fatty acids in skeletal muscle. [Internet] [Thesis]. Université Catholique de Louvain; 2013. [cited 2019 May 22]. Available from: http://hdl.handle.net/2078.1/128993.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zbinden Foncea H. Activation of Toll Like receptors 2 and 4 by fatty acids in skeletal muscle. [Thesis]. Université Catholique de Louvain; 2013. Available from: http://hdl.handle.net/2078.1/128993

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

18. Denko, Laura Michelle. Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin.

Degree: MS, Human Nutrition, Foods, and Exercise, 2012, Virginia Tech

 Obesity-related metabolic derangements have been linked to toll-like receptor 4 (TLR4), an innate immune system receptor, due to its role in proinflammatory pathways. Lipopolysaccharide (LPS),… (more)

Subjects/Keywords: TLR4; endotoxin; LPS; skeletal muscle

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APA (6th Edition):

Denko, L. M. (2012). Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/76808

Chicago Manual of Style (16th Edition):

Denko, Laura Michelle. “Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin.” 2012. Masters Thesis, Virginia Tech. Accessed May 22, 2019. http://hdl.handle.net/10919/76808.

MLA Handbook (7th Edition):

Denko, Laura Michelle. “Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin.” 2012. Web. 22 May 2019.

Vancouver:

Denko LM. Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin. [Internet] [Masters thesis]. Virginia Tech; 2012. [cited 2019 May 22]. Available from: http://hdl.handle.net/10919/76808.

Council of Science Editors:

Denko LM. Cellular Reprogramming in Skeletal Muscle after Repeated Exposures to Endotoxin. [Masters Thesis]. Virginia Tech; 2012. Available from: http://hdl.handle.net/10919/76808


Cornell University

19. Leineweber, Matthew. Applying Ultrasound Elastography To Detecting Skeletal Muscle Injury .

Degree: 2014, Cornell University

Skeletal muscle injuries are frequent in sports and exercise, accounting for between 2055% of all sports injuries, with the vast majority of these injuries being… (more)

Subjects/Keywords: ultrasound; elastography; skeletal muscle

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APA (6th Edition):

Leineweber, M. (2014). Applying Ultrasound Elastography To Detecting Skeletal Muscle Injury . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/38881

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Leineweber, Matthew. “Applying Ultrasound Elastography To Detecting Skeletal Muscle Injury .” 2014. Thesis, Cornell University. Accessed May 22, 2019. http://hdl.handle.net/1813/38881.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Leineweber, Matthew. “Applying Ultrasound Elastography To Detecting Skeletal Muscle Injury .” 2014. Web. 22 May 2019.

Vancouver:

Leineweber M. Applying Ultrasound Elastography To Detecting Skeletal Muscle Injury . [Internet] [Thesis]. Cornell University; 2014. [cited 2019 May 22]. Available from: http://hdl.handle.net/1813/38881.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Leineweber M. Applying Ultrasound Elastography To Detecting Skeletal Muscle Injury . [Thesis]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/38881

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

20. Iyer, Shama. Chitosan, Skeletal Muscle Regeneration And Fibrosis Inhibition .

Degree: 2015, Cornell University

 Treatment of ventral hernias frequently employs use of synthetic and biological meshes, which often results in scar tissue formation and incomplete muscle regeneration, leading to… (more)

Subjects/Keywords: skeletal muscle regeneration; fibrosis; chitosan

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APA (6th Edition):

Iyer, S. (2015). Chitosan, Skeletal Muscle Regeneration And Fibrosis Inhibition . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/41025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Iyer, Shama. “Chitosan, Skeletal Muscle Regeneration And Fibrosis Inhibition .” 2015. Thesis, Cornell University. Accessed May 22, 2019. http://hdl.handle.net/1813/41025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Iyer, Shama. “Chitosan, Skeletal Muscle Regeneration And Fibrosis Inhibition .” 2015. Web. 22 May 2019.

Vancouver:

Iyer S. Chitosan, Skeletal Muscle Regeneration And Fibrosis Inhibition . [Internet] [Thesis]. Cornell University; 2015. [cited 2019 May 22]. Available from: http://hdl.handle.net/1813/41025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Iyer S. Chitosan, Skeletal Muscle Regeneration And Fibrosis Inhibition . [Thesis]. Cornell University; 2015. Available from: http://hdl.handle.net/1813/41025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Waterloo

21. Gamu, Daniel. Examination of Voluntary Wheel Running and Skeletal Muscle Metabolism in the Sarcolipin Knock-Out Mouse.

Degree: 2012, University of Waterloo

 Sarcolipin (SLN) is a small sarcoplasmic reticulum (SR) integral membrane protein that regulates the SR Ca2+-ATPase (SERCA). Previous studies indicate that the functional interaction between… (more)

Subjects/Keywords: Sarcolipin; Obesity; Skeletal Muscle; Metabolism

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APA (6th Edition):

Gamu, D. (2012). Examination of Voluntary Wheel Running and Skeletal Muscle Metabolism in the Sarcolipin Knock-Out Mouse. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/6468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gamu, Daniel. “Examination of Voluntary Wheel Running and Skeletal Muscle Metabolism in the Sarcolipin Knock-Out Mouse.” 2012. Thesis, University of Waterloo. Accessed May 22, 2019. http://hdl.handle.net/10012/6468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gamu, Daniel. “Examination of Voluntary Wheel Running and Skeletal Muscle Metabolism in the Sarcolipin Knock-Out Mouse.” 2012. Web. 22 May 2019.

Vancouver:

Gamu D. Examination of Voluntary Wheel Running and Skeletal Muscle Metabolism in the Sarcolipin Knock-Out Mouse. [Internet] [Thesis]. University of Waterloo; 2012. [cited 2019 May 22]. Available from: http://hdl.handle.net/10012/6468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gamu D. Examination of Voluntary Wheel Running and Skeletal Muscle Metabolism in the Sarcolipin Knock-Out Mouse. [Thesis]. University of Waterloo; 2012. Available from: http://hdl.handle.net/10012/6468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Waterloo

22. Paré, Marie-France. Apoptotic signalling and structural alterations in autophagy-deficient murine skeletal muscle.

Degree: 2014, University of Waterloo

 Autophagy is a catabolic process by which the cell targets and degrades cytoplasmic materials, such as proteins and organelles. Autophagy is required for the control… (more)

Subjects/Keywords: autophagy; apoptosis; skeletal muscle

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APA (6th Edition):

Paré, M. (2014). Apoptotic signalling and structural alterations in autophagy-deficient murine skeletal muscle. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/8900

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Paré, Marie-France. “Apoptotic signalling and structural alterations in autophagy-deficient murine skeletal muscle.” 2014. Thesis, University of Waterloo. Accessed May 22, 2019. http://hdl.handle.net/10012/8900.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Paré, Marie-France. “Apoptotic signalling and structural alterations in autophagy-deficient murine skeletal muscle.” 2014. Web. 22 May 2019.

Vancouver:

Paré M. Apoptotic signalling and structural alterations in autophagy-deficient murine skeletal muscle. [Internet] [Thesis]. University of Waterloo; 2014. [cited 2019 May 22]. Available from: http://hdl.handle.net/10012/8900.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Paré M. Apoptotic signalling and structural alterations in autophagy-deficient murine skeletal muscle. [Thesis]. University of Waterloo; 2014. Available from: http://hdl.handle.net/10012/8900

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Waterloo

23. Vorobej, Kira. The Role of Apoptosis Repressor with Caspase Recruitment Domain in Aging Mouse Skeletal Muscle Morphology and Apoptotic Signaling.

Degree: 2015, University of Waterloo

 Abstract Aging is associated with a loss of skeletal muscle mass, known as sarcopenia, which results in numerous degenerative alterations and decreased strength. One of… (more)

Subjects/Keywords: Apoptosis; Aging; Skeletal Muscle

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APA (6th Edition):

Vorobej, K. (2015). The Role of Apoptosis Repressor with Caspase Recruitment Domain in Aging Mouse Skeletal Muscle Morphology and Apoptotic Signaling. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/9143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vorobej, Kira. “The Role of Apoptosis Repressor with Caspase Recruitment Domain in Aging Mouse Skeletal Muscle Morphology and Apoptotic Signaling.” 2015. Thesis, University of Waterloo. Accessed May 22, 2019. http://hdl.handle.net/10012/9143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vorobej, Kira. “The Role of Apoptosis Repressor with Caspase Recruitment Domain in Aging Mouse Skeletal Muscle Morphology and Apoptotic Signaling.” 2015. Web. 22 May 2019.

Vancouver:

Vorobej K. The Role of Apoptosis Repressor with Caspase Recruitment Domain in Aging Mouse Skeletal Muscle Morphology and Apoptotic Signaling. [Internet] [Thesis]. University of Waterloo; 2015. [cited 2019 May 22]. Available from: http://hdl.handle.net/10012/9143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vorobej K. The Role of Apoptosis Repressor with Caspase Recruitment Domain in Aging Mouse Skeletal Muscle Morphology and Apoptotic Signaling. [Thesis]. University of Waterloo; 2015. Available from: http://hdl.handle.net/10012/9143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Waterloo

24. Campbell, Troy. Examining the role of autophagy in modulating apoptotic signaling and skeletal muscle recovery after cardiotoxin injury in tamoxifen-inducible, skeletal muscle-specific Atg7 knockdown mice.

Degree: 2015, University of Waterloo

Skeletal muscle is a unique tissue composed of multinucleated, post-mitotic fibers. Despite its post-mitotic nature, skeletal muscle has an astounding ability to regenerate after injury… (more)

Subjects/Keywords: autophagy; apoptosis; skeletal muscle

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APA (6th Edition):

Campbell, T. (2015). Examining the role of autophagy in modulating apoptotic signaling and skeletal muscle recovery after cardiotoxin injury in tamoxifen-inducible, skeletal muscle-specific Atg7 knockdown mice. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/9807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Campbell, Troy. “Examining the role of autophagy in modulating apoptotic signaling and skeletal muscle recovery after cardiotoxin injury in tamoxifen-inducible, skeletal muscle-specific Atg7 knockdown mice.” 2015. Thesis, University of Waterloo. Accessed May 22, 2019. http://hdl.handle.net/10012/9807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Campbell, Troy. “Examining the role of autophagy in modulating apoptotic signaling and skeletal muscle recovery after cardiotoxin injury in tamoxifen-inducible, skeletal muscle-specific Atg7 knockdown mice.” 2015. Web. 22 May 2019.

Vancouver:

Campbell T. Examining the role of autophagy in modulating apoptotic signaling and skeletal muscle recovery after cardiotoxin injury in tamoxifen-inducible, skeletal muscle-specific Atg7 knockdown mice. [Internet] [Thesis]. University of Waterloo; 2015. [cited 2019 May 22]. Available from: http://hdl.handle.net/10012/9807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Campbell T. Examining the role of autophagy in modulating apoptotic signaling and skeletal muscle recovery after cardiotoxin injury in tamoxifen-inducible, skeletal muscle-specific Atg7 knockdown mice. [Thesis]. University of Waterloo; 2015. Available from: http://hdl.handle.net/10012/9807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

25. Williams, Andrew H. Regulation of Skeletal Muscle Innervation and ALS Pathogenesis by MicroRNA 206.

Degree: 2010, University of Texas Southwestern Medical Center

 Motor neurons and the skeletal muscle fibers they innervate maintain an intimate relationship that requires bidirectional signaling for the establishment and maintenance of neuromuscular synapses… (more)

Subjects/Keywords: MicroRNAs; Muscle, Skeletal; Neuromuscular Junction

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APA (6th Edition):

Williams, A. H. (2010). Regulation of Skeletal Muscle Innervation and ALS Pathogenesis by MicroRNA 206. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/917

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Andrew H. “Regulation of Skeletal Muscle Innervation and ALS Pathogenesis by MicroRNA 206.” 2010. Thesis, University of Texas Southwestern Medical Center. Accessed May 22, 2019. http://hdl.handle.net/2152.5/917.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Andrew H. “Regulation of Skeletal Muscle Innervation and ALS Pathogenesis by MicroRNA 206.” 2010. Web. 22 May 2019.

Vancouver:

Williams AH. Regulation of Skeletal Muscle Innervation and ALS Pathogenesis by MicroRNA 206. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2010. [cited 2019 May 22]. Available from: http://hdl.handle.net/2152.5/917.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams AH. Regulation of Skeletal Muscle Innervation and ALS Pathogenesis by MicroRNA 206. [Thesis]. University of Texas Southwestern Medical Center; 2010. Available from: http://hdl.handle.net/2152.5/917

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

26. Pryce, Benjamin. Role of the Ste20 Like Kinase in Muscle Development and Muscular Dystrophy .

Degree: 2019, University of Ottawa

 Duchenne Muscular Dystrophy (DMD) is a fatal X-linked disorder affecting 1 out of every 3500 male births. The underlying cause of DMD is mutations within… (more)

Subjects/Keywords: SLK; Skeletal Muscle; Muscular Dystrophy

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APA (6th Edition):

Pryce, B. (2019). Role of the Ste20 Like Kinase in Muscle Development and Muscular Dystrophy . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/38710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pryce, Benjamin. “Role of the Ste20 Like Kinase in Muscle Development and Muscular Dystrophy .” 2019. Thesis, University of Ottawa. Accessed May 22, 2019. http://hdl.handle.net/10393/38710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pryce, Benjamin. “Role of the Ste20 Like Kinase in Muscle Development and Muscular Dystrophy .” 2019. Web. 22 May 2019.

Vancouver:

Pryce B. Role of the Ste20 Like Kinase in Muscle Development and Muscular Dystrophy . [Internet] [Thesis]. University of Ottawa; 2019. [cited 2019 May 22]. Available from: http://hdl.handle.net/10393/38710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pryce B. Role of the Ste20 Like Kinase in Muscle Development and Muscular Dystrophy . [Thesis]. University of Ottawa; 2019. Available from: http://hdl.handle.net/10393/38710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Miami

27. Aydogdu, Tufan. STAT3 Regulation of Skeletal Muscle Wasting in Cancer Cachexia.

Degree: PhD, Molecular Cell and Developmental Biology (Medicine), 2010, University of Miami

 Cachexia is a highly complex syndrome identified by metabolic, hormonal and cytokine-related abnormalities, but can be shortly characterized as accelerated skeletal muscle and adipose tissue… (more)

Subjects/Keywords: Cachexia; Skeletal Muscle; APR; STAT3

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APA (6th Edition):

Aydogdu, T. (2010). STAT3 Regulation of Skeletal Muscle Wasting in Cancer Cachexia. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/652

Chicago Manual of Style (16th Edition):

Aydogdu, Tufan. “STAT3 Regulation of Skeletal Muscle Wasting in Cancer Cachexia.” 2010. Doctoral Dissertation, University of Miami. Accessed May 22, 2019. https://scholarlyrepository.miami.edu/oa_dissertations/652.

MLA Handbook (7th Edition):

Aydogdu, Tufan. “STAT3 Regulation of Skeletal Muscle Wasting in Cancer Cachexia.” 2010. Web. 22 May 2019.

Vancouver:

Aydogdu T. STAT3 Regulation of Skeletal Muscle Wasting in Cancer Cachexia. [Internet] [Doctoral dissertation]. University of Miami; 2010. [cited 2019 May 22]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/652.

Council of Science Editors:

Aydogdu T. STAT3 Regulation of Skeletal Muscle Wasting in Cancer Cachexia. [Doctoral Dissertation]. University of Miami; 2010. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/652


University of New South Wales

28. Hoy, Andrew James. The mechanisms involved in the development of nutrient oversupply-induced insulin resistance in skeletal muscle.

Degree: Garvan Institute of Medical Research, 2009, University of New South Wales

 Insulin resistance is a major metabolic defect associated with obesity and type 2 diabetes. The incidences of both are increasing at an alarming rate. Excessive… (more)

Subjects/Keywords: Insulin resistance; Skeletal muscle; Metabolism

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APA (6th Edition):

Hoy, A. J. (2009). The mechanisms involved in the development of nutrient oversupply-induced insulin resistance in skeletal muscle. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/43635 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:5079/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Hoy, Andrew James. “The mechanisms involved in the development of nutrient oversupply-induced insulin resistance in skeletal muscle.” 2009. Doctoral Dissertation, University of New South Wales. Accessed May 22, 2019. http://handle.unsw.edu.au/1959.4/43635 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:5079/SOURCE02?view=true.

MLA Handbook (7th Edition):

Hoy, Andrew James. “The mechanisms involved in the development of nutrient oversupply-induced insulin resistance in skeletal muscle.” 2009. Web. 22 May 2019.

Vancouver:

Hoy AJ. The mechanisms involved in the development of nutrient oversupply-induced insulin resistance in skeletal muscle. [Internet] [Doctoral dissertation]. University of New South Wales; 2009. [cited 2019 May 22]. Available from: http://handle.unsw.edu.au/1959.4/43635 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:5079/SOURCE02?view=true.

Council of Science Editors:

Hoy AJ. The mechanisms involved in the development of nutrient oversupply-induced insulin resistance in skeletal muscle. [Doctoral Dissertation]. University of New South Wales; 2009. Available from: http://handle.unsw.edu.au/1959.4/43635 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:5079/SOURCE02?view=true


University of Illinois – Urbana-Champaign

29. Waldemer-Streyer, Rachel Jean. Muscle-derived cytokines: novel regulators of skeletal myogenesis.

Degree: PhD, Cell and Developmental Biology, 2016, University of Illinois – Urbana-Champaign

 Mature skeletal muscle has a uniquely robust capacity for regeneration. This ability is due in large part to the resident population of muscle stem cells… (more)

Subjects/Keywords: skeletal muscle; cytokine; differentiation; regeneration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Waldemer-Streyer, R. J. (2016). Muscle-derived cytokines: novel regulators of skeletal myogenesis. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/92918

Chicago Manual of Style (16th Edition):

Waldemer-Streyer, Rachel Jean. “Muscle-derived cytokines: novel regulators of skeletal myogenesis.” 2016. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed May 22, 2019. http://hdl.handle.net/2142/92918.

MLA Handbook (7th Edition):

Waldemer-Streyer, Rachel Jean. “Muscle-derived cytokines: novel regulators of skeletal myogenesis.” 2016. Web. 22 May 2019.

Vancouver:

Waldemer-Streyer RJ. Muscle-derived cytokines: novel regulators of skeletal myogenesis. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2016. [cited 2019 May 22]. Available from: http://hdl.handle.net/2142/92918.

Council of Science Editors:

Waldemer-Streyer RJ. Muscle-derived cytokines: novel regulators of skeletal myogenesis. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2016. Available from: http://hdl.handle.net/2142/92918


University of Illinois – Urbana-Champaign

30. Cvetkovic, Caroline. The development of a skeletal muscle bio-actuator using 3-D stereolithography.

Degree: MS, 0408, 2013, University of Illinois – Urbana-Champaign

 Over the past decade, a new scientific discipline has emerged, integrating mechanics with biology to create complex engineered living systems. The building blocks – different… (more)

Subjects/Keywords: skeletal muscle; bio-actuator; stereolithography

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cvetkovic, C. (2013). The development of a skeletal muscle bio-actuator using 3-D stereolithography. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/44448

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cvetkovic, Caroline. “The development of a skeletal muscle bio-actuator using 3-D stereolithography.” 2013. Thesis, University of Illinois – Urbana-Champaign. Accessed May 22, 2019. http://hdl.handle.net/2142/44448.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cvetkovic, Caroline. “The development of a skeletal muscle bio-actuator using 3-D stereolithography.” 2013. Web. 22 May 2019.

Vancouver:

Cvetkovic C. The development of a skeletal muscle bio-actuator using 3-D stereolithography. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2013. [cited 2019 May 22]. Available from: http://hdl.handle.net/2142/44448.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cvetkovic C. The development of a skeletal muscle bio-actuator using 3-D stereolithography. [Thesis]. University of Illinois – Urbana-Champaign; 2013. Available from: http://hdl.handle.net/2142/44448

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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