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You searched for subject:(Sirtuin 1). Showing records 1 – 16 of 16 total matches.

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University of California – Irvine

1. Cinco, Rachel Perlas. Redox State and Ovarian Function.

Degree: Biological Sciences, 2016, University of California – Irvine

 Infertility is recognized worldwide as a public health issue and there is a significant medical need to understand the mechanisms that underlie infertility so that… (more)

Subjects/Keywords: Developmental biology; Biology; Fertility; Metabolism; Ovary; Phasor FLIM; Redox; Sirtuin 1

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APA (6th Edition):

Cinco, R. P. (2016). Redox State and Ovarian Function. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/7503b439

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cinco, Rachel Perlas. “Redox State and Ovarian Function.” 2016. Thesis, University of California – Irvine. Accessed June 05, 2020. http://www.escholarship.org/uc/item/7503b439.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cinco, Rachel Perlas. “Redox State and Ovarian Function.” 2016. Web. 05 Jun 2020.

Vancouver:

Cinco RP. Redox State and Ovarian Function. [Internet] [Thesis]. University of California – Irvine; 2016. [cited 2020 Jun 05]. Available from: http://www.escholarship.org/uc/item/7503b439.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cinco RP. Redox State and Ovarian Function. [Thesis]. University of California – Irvine; 2016. Available from: http://www.escholarship.org/uc/item/7503b439

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. 小山, 淑正. Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.

Degree: 博士(医学), 2016, Oita University / 大分大学

 Reversed feeding uncouples peripheral and master clock gene rhythms and leads to an increased risk of disease development. The aim of this study was to… (more)

Subjects/Keywords: sepsis; sirtuin-1; PGC-1α; pro-inflammatory cytokines; survival rate

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APA (6th Edition):

小山, . (2016). Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15608

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

小山, 淑正. “Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.” 2016. Thesis, Oita University / 大分大学. Accessed June 05, 2020. http://hdl.handle.net/10559/15608.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

小山, 淑正. “Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice.” 2016. Web. 05 Jun 2020.

Vancouver:

小山 . Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. [Internet] [Thesis]. Oita University / 大分大学; 2016. [cited 2020 Jun 05]. Available from: http://hdl.handle.net/10559/15608.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

小山 . Uncoupling of peripheral and master clock gene rhythms by reversed feeding leads to an exacerbated inflammatory response after polymicrobial sepsis in mice. [Thesis]. Oita University / 大分大学; 2016. Available from: http://hdl.handle.net/10559/15608

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

3. Holzhauser, Susanne. Detection and characterization of sirtuin 1-modulating natural products.

Degree: 2013, Freie Universität Berlin

 The progressive increase in life expectancies of our society results in an increasing prevalence of age-related diseases like type-2 diabetes. This causes economic as well… (more)

Subjects/Keywords: sirtuin 1; natural products; MALDI-TOF MS; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie

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APA (6th Edition):

Holzhauser, S. (2013). Detection and characterization of sirtuin 1-modulating natural products. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-4673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Holzhauser, Susanne. “Detection and characterization of sirtuin 1-modulating natural products.” 2013. Thesis, Freie Universität Berlin. Accessed June 05, 2020. http://dx.doi.org/10.17169/refubium-4673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Holzhauser, Susanne. “Detection and characterization of sirtuin 1-modulating natural products.” 2013. Web. 05 Jun 2020.

Vancouver:

Holzhauser S. Detection and characterization of sirtuin 1-modulating natural products. [Internet] [Thesis]. Freie Universität Berlin; 2013. [cited 2020 Jun 05]. Available from: http://dx.doi.org/10.17169/refubium-4673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Holzhauser S. Detection and characterization of sirtuin 1-modulating natural products. [Thesis]. Freie Universität Berlin; 2013. Available from: http://dx.doi.org/10.17169/refubium-4673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

4. Taha, Mohamad. Investigating the Role of Sirtuin 1 in the Pulmonary Vascular Response to Chronic Hypoxia-Induced Pulmonary Hypertension .

Degree: 2018, University of Ottawa

 Background: Pulmonary arterial hypertension (PAH) is a devastating disease characterized by increased pulmonary artery pressure, leading to right ventricle hypertrophy and ultimately heart failure and… (more)

Subjects/Keywords: Sirtuin 1; Pulmonary hypertension; Chronic hypoxia; SIRT1; Transgenic mice; Endothelial cells; Hypoxia inducible factors

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APA (6th Edition):

Taha, M. (2018). Investigating the Role of Sirtuin 1 in the Pulmonary Vascular Response to Chronic Hypoxia-Induced Pulmonary Hypertension . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/37550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Taha, Mohamad. “Investigating the Role of Sirtuin 1 in the Pulmonary Vascular Response to Chronic Hypoxia-Induced Pulmonary Hypertension .” 2018. Thesis, University of Ottawa. Accessed June 05, 2020. http://hdl.handle.net/10393/37550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Taha, Mohamad. “Investigating the Role of Sirtuin 1 in the Pulmonary Vascular Response to Chronic Hypoxia-Induced Pulmonary Hypertension .” 2018. Web. 05 Jun 2020.

Vancouver:

Taha M. Investigating the Role of Sirtuin 1 in the Pulmonary Vascular Response to Chronic Hypoxia-Induced Pulmonary Hypertension . [Internet] [Thesis]. University of Ottawa; 2018. [cited 2020 Jun 05]. Available from: http://hdl.handle.net/10393/37550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Taha M. Investigating the Role of Sirtuin 1 in the Pulmonary Vascular Response to Chronic Hypoxia-Induced Pulmonary Hypertension . [Thesis]. University of Ottawa; 2018. Available from: http://hdl.handle.net/10393/37550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Defour, Aurélia. Fonctions métaboliques de Sirtuine 1 dans le muscle strié squelettique : contribution à l'étude de la régulation de l'expression de SREBP-1c et rôle potentiel lors d'un jeûne chez des myotubes C2C12 : Metabolic functions of sirtuin 1 in skeletal muscle : contribution to the study of regulation of the SREBP-1c expression and potential role during fasting in C2C12 myotubes.

Degree: Docteur es, Biologie et physiologie de l'exercice, 2010, Saint-Etienne

Sirt1 (Sirtuine 1) est une protéine histone déacétylase dépendante de NAD+ qui stimule la néoglucogénèse et inhibe la glycolyse dans le foie, et qui augmente… (more)

Subjects/Keywords: Muscle strié squelettique; Sirtuine 1; Métabolisme du glucose; SREBP-1c; LXR; Jeûne; Masse musculaire; Sirtuin 1

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APA (6th Edition):

Defour, A. (2010). Fonctions métaboliques de Sirtuine 1 dans le muscle strié squelettique : contribution à l'étude de la régulation de l'expression de SREBP-1c et rôle potentiel lors d'un jeûne chez des myotubes C2C12 : Metabolic functions of sirtuin 1 in skeletal muscle : contribution to the study of regulation of the SREBP-1c expression and potential role during fasting in C2C12 myotubes. (Doctoral Dissertation). Saint-Etienne. Retrieved from http://www.theses.fr/2010STET011T

Chicago Manual of Style (16th Edition):

Defour, Aurélia. “Fonctions métaboliques de Sirtuine 1 dans le muscle strié squelettique : contribution à l'étude de la régulation de l'expression de SREBP-1c et rôle potentiel lors d'un jeûne chez des myotubes C2C12 : Metabolic functions of sirtuin 1 in skeletal muscle : contribution to the study of regulation of the SREBP-1c expression and potential role during fasting in C2C12 myotubes.” 2010. Doctoral Dissertation, Saint-Etienne. Accessed June 05, 2020. http://www.theses.fr/2010STET011T.

MLA Handbook (7th Edition):

Defour, Aurélia. “Fonctions métaboliques de Sirtuine 1 dans le muscle strié squelettique : contribution à l'étude de la régulation de l'expression de SREBP-1c et rôle potentiel lors d'un jeûne chez des myotubes C2C12 : Metabolic functions of sirtuin 1 in skeletal muscle : contribution to the study of regulation of the SREBP-1c expression and potential role during fasting in C2C12 myotubes.” 2010. Web. 05 Jun 2020.

Vancouver:

Defour A. Fonctions métaboliques de Sirtuine 1 dans le muscle strié squelettique : contribution à l'étude de la régulation de l'expression de SREBP-1c et rôle potentiel lors d'un jeûne chez des myotubes C2C12 : Metabolic functions of sirtuin 1 in skeletal muscle : contribution to the study of regulation of the SREBP-1c expression and potential role during fasting in C2C12 myotubes. [Internet] [Doctoral dissertation]. Saint-Etienne; 2010. [cited 2020 Jun 05]. Available from: http://www.theses.fr/2010STET011T.

Council of Science Editors:

Defour A. Fonctions métaboliques de Sirtuine 1 dans le muscle strié squelettique : contribution à l'étude de la régulation de l'expression de SREBP-1c et rôle potentiel lors d'un jeûne chez des myotubes C2C12 : Metabolic functions of sirtuin 1 in skeletal muscle : contribution to the study of regulation of the SREBP-1c expression and potential role during fasting in C2C12 myotubes. [Doctoral Dissertation]. Saint-Etienne; 2010. Available from: http://www.theses.fr/2010STET011T


Université Paris-Sud – Paris XI

6. Prola, Alexandre. Rôle de la sirtuine 1 dans la modulation de la réponse des cardiomyocytes au stress RE et à l’apoptose : Role of the sirtuine 1 in the modulation of endoplasmic reticulum stress response and apoptosis in cardiomyocytes.

Degree: Docteur es, Physiopathologie moléculaire et cellulaire, 2014, Université Paris-Sud – Paris XI

Des altérations de fonctions physiologiques du réticulum endoplasmique (RE) induisent un processus appelé stress RE. Dans le domaine cardiovasculaire, plusieurs travaux ont montré que le… (more)

Subjects/Keywords: Sirtuine 1; Stress RE; Pathologies cardiaques; Apoptose; EIF2α; Sirtuin 1; ER stress; Cardiac diseases; Apoptosis; EIF2α

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APA (6th Edition):

Prola, A. (2014). Rôle de la sirtuine 1 dans la modulation de la réponse des cardiomyocytes au stress RE et à l’apoptose : Role of the sirtuine 1 in the modulation of endoplasmic reticulum stress response and apoptosis in cardiomyocytes. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2014PA114815

Chicago Manual of Style (16th Edition):

Prola, Alexandre. “Rôle de la sirtuine 1 dans la modulation de la réponse des cardiomyocytes au stress RE et à l’apoptose : Role of the sirtuine 1 in the modulation of endoplasmic reticulum stress response and apoptosis in cardiomyocytes.” 2014. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed June 05, 2020. http://www.theses.fr/2014PA114815.

MLA Handbook (7th Edition):

Prola, Alexandre. “Rôle de la sirtuine 1 dans la modulation de la réponse des cardiomyocytes au stress RE et à l’apoptose : Role of the sirtuine 1 in the modulation of endoplasmic reticulum stress response and apoptosis in cardiomyocytes.” 2014. Web. 05 Jun 2020.

Vancouver:

Prola A. Rôle de la sirtuine 1 dans la modulation de la réponse des cardiomyocytes au stress RE et à l’apoptose : Role of the sirtuine 1 in the modulation of endoplasmic reticulum stress response and apoptosis in cardiomyocytes. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2014. [cited 2020 Jun 05]. Available from: http://www.theses.fr/2014PA114815.

Council of Science Editors:

Prola A. Rôle de la sirtuine 1 dans la modulation de la réponse des cardiomyocytes au stress RE et à l’apoptose : Role of the sirtuine 1 in the modulation of endoplasmic reticulum stress response and apoptosis in cardiomyocytes. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2014. Available from: http://www.theses.fr/2014PA114815


Université Montpellier II

7. Abdel Khalek, Waed. Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation.

Degree: Docteur es, Biologie Santé, 2013, Université Montpellier II

 Sirt3, une des sept sirtuines chez les mammifères, est une déacétylase mitochondriale NAD+-dépendante qui joue un rôle dans le contrôle des facteurs clés de plusieurs… (more)

Subjects/Keywords: Muscle; Differenciation; Sirtuine 3; PGC-1α; Peroxisome proliferator-activated receptor gamma coactivator 1-alpha; Dérivés réactifs d'oxygeneSirtuine 1; Muscle; Differentiation; Sirtuin 3; PGC-1α; Peroxisome proliferator-activated receptor gamma coactivator 1-alpha; Reactive Oxygen species ROS; Sirtuin 1

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APA (6th Edition):

Abdel Khalek, W. (2013). Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2013MON20022

Chicago Manual of Style (16th Edition):

Abdel Khalek, Waed. “Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation.” 2013. Doctoral Dissertation, Université Montpellier II. Accessed June 05, 2020. http://www.theses.fr/2013MON20022.

MLA Handbook (7th Edition):

Abdel Khalek, Waed. “Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation.” 2013. Web. 05 Jun 2020.

Vancouver:

Abdel Khalek W. Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation. [Internet] [Doctoral dissertation]. Université Montpellier II; 2013. [cited 2020 Jun 05]. Available from: http://www.theses.fr/2013MON20022.

Council of Science Editors:

Abdel Khalek W. Sirt3, une déacetylase mitochondriale NAD+dépendante, est impliquée dans la regulation de la différenciation des myoblastes : SIRT3, a mitochondrial NAD+-dependent deacetylase is involved in the regulation of myoblast differentiation. [Doctoral Dissertation]. Université Montpellier II; 2013. Available from: http://www.theses.fr/2013MON20022


Université de Montréal

8. Mir, Ahmed. Modulation de l'expression de Sirt-1 induite par l'endothéline-1 dans les cellules musculaires lisses vasculaires .

Degree: 2015, Université de Montréal

 Au cours des maladies cardiovasculaires (MCV), il peut se produire divers problèmes de santé, telle que l’insuffisance cardiaque ou encore l’HTA. Ces phénomènes se caractérisent,… (more)

Subjects/Keywords: Maladies cardiovasculaires; Endothéline-1; Cellules vasculaires des muscles lisses; Hypertension artérielle; Histone déacétylase; Sirtuine-1; Cardiovascular diseases; Endothelin-1; Vascular smooth muscle cell; Hypertension; histone deacetylase; Sirtuin-1

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APA (6th Edition):

Mir, A. (2015). Modulation de l'expression de Sirt-1 induite par l'endothéline-1 dans les cellules musculaires lisses vasculaires . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/11549

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mir, Ahmed. “Modulation de l'expression de Sirt-1 induite par l'endothéline-1 dans les cellules musculaires lisses vasculaires .” 2015. Thesis, Université de Montréal. Accessed June 05, 2020. http://hdl.handle.net/1866/11549.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mir, Ahmed. “Modulation de l'expression de Sirt-1 induite par l'endothéline-1 dans les cellules musculaires lisses vasculaires .” 2015. Web. 05 Jun 2020.

Vancouver:

Mir A. Modulation de l'expression de Sirt-1 induite par l'endothéline-1 dans les cellules musculaires lisses vasculaires . [Internet] [Thesis]. Université de Montréal; 2015. [cited 2020 Jun 05]. Available from: http://hdl.handle.net/1866/11549.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mir A. Modulation de l'expression de Sirt-1 induite par l'endothéline-1 dans les cellules musculaires lisses vasculaires . [Thesis]. Université de Montréal; 2015. Available from: http://hdl.handle.net/1866/11549

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

9. Cao, Peter. Age-related structural changes in the liver of old mice with SIRT1 overexpression in conjunction with NMN supplementation .

Degree: 2016, University of Sydney

 Ageing is associated with ultrastructural changes in the liver, including a reduction in hepatocyte mitochondrial number density and porosity of the liver sinusoidal cells. Histologically-verifiable… (more)

Subjects/Keywords: Sirtuin 1 (SIRT1); nicotinamide mononucleotide (NMN); nicotinamide adenine dinucleotide (NAD); liver sinusoidal endothelial cells (LSEC); mitochondria; electron microscopy

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APA (6th Edition):

Cao, P. (2016). Age-related structural changes in the liver of old mice with SIRT1 overexpression in conjunction with NMN supplementation . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/16872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cao, Peter. “Age-related structural changes in the liver of old mice with SIRT1 overexpression in conjunction with NMN supplementation .” 2016. Thesis, University of Sydney. Accessed June 05, 2020. http://hdl.handle.net/2123/16872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cao, Peter. “Age-related structural changes in the liver of old mice with SIRT1 overexpression in conjunction with NMN supplementation .” 2016. Web. 05 Jun 2020.

Vancouver:

Cao P. Age-related structural changes in the liver of old mice with SIRT1 overexpression in conjunction with NMN supplementation . [Internet] [Thesis]. University of Sydney; 2016. [cited 2020 Jun 05]. Available from: http://hdl.handle.net/2123/16872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cao P. Age-related structural changes in the liver of old mice with SIRT1 overexpression in conjunction with NMN supplementation . [Thesis]. University of Sydney; 2016. Available from: http://hdl.handle.net/2123/16872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Gueguen, Cindy. L’activation de la sirtuin 1 : une nouvelle stratégie neuroprotectrice pour le stress oxydant cérébral in vivo ? Implication dans les effets bénéfiques de l’inhibition de la poly(ADP-ribose)polymérase par le 3-aminobenzamide : Sirtuin 1 activation : a neuroprotective strategy for in vivo cerebral oxidative stress ? Involvement of SIRT1 in the beneficial effects of poly(ADP-ribose)polymerase inhibition.

Degree: Docteur es, Pharmacologie, 2013, Université Paris Descartes – Paris V

Le stress oxydant (SO) est un mécanisme commun à l’ischémie cérébrale et au traumatisme crânien qui entraîne notamment l’hyperactivation délétère de la poly(ADP-ribose)polymérase (PARP), une… (more)

Subjects/Keywords: Stress oxydant cérébral; Sirtuin 1 (SIRT1); Poly(ADP-ribose)polymérase (PARP); 3-aminobenzamide (3AB); SRT1720; EX527; NAD+; Déficit neurologique; Lésion cérébrale; Cerebral oxidative stress; Sirtuin 1 (SIRT1); Poly(ADP-ribose)polymerase (PARP); 3-aminobenzamide (3AB); SRT1720; EX527; NAD+; Neurological deficit; Brain lesion; 616.81061

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APA (6th Edition):

Gueguen, C. (2013). L’activation de la sirtuin 1 : une nouvelle stratégie neuroprotectrice pour le stress oxydant cérébral in vivo ? Implication dans les effets bénéfiques de l’inhibition de la poly(ADP-ribose)polymérase par le 3-aminobenzamide : Sirtuin 1 activation : a neuroprotective strategy for in vivo cerebral oxidative stress ? Involvement of SIRT1 in the beneficial effects of poly(ADP-ribose)polymerase inhibition. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2013PA05P610

Chicago Manual of Style (16th Edition):

Gueguen, Cindy. “L’activation de la sirtuin 1 : une nouvelle stratégie neuroprotectrice pour le stress oxydant cérébral in vivo ? Implication dans les effets bénéfiques de l’inhibition de la poly(ADP-ribose)polymérase par le 3-aminobenzamide : Sirtuin 1 activation : a neuroprotective strategy for in vivo cerebral oxidative stress ? Involvement of SIRT1 in the beneficial effects of poly(ADP-ribose)polymerase inhibition.” 2013. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed June 05, 2020. http://www.theses.fr/2013PA05P610.

MLA Handbook (7th Edition):

Gueguen, Cindy. “L’activation de la sirtuin 1 : une nouvelle stratégie neuroprotectrice pour le stress oxydant cérébral in vivo ? Implication dans les effets bénéfiques de l’inhibition de la poly(ADP-ribose)polymérase par le 3-aminobenzamide : Sirtuin 1 activation : a neuroprotective strategy for in vivo cerebral oxidative stress ? Involvement of SIRT1 in the beneficial effects of poly(ADP-ribose)polymerase inhibition.” 2013. Web. 05 Jun 2020.

Vancouver:

Gueguen C. L’activation de la sirtuin 1 : une nouvelle stratégie neuroprotectrice pour le stress oxydant cérébral in vivo ? Implication dans les effets bénéfiques de l’inhibition de la poly(ADP-ribose)polymérase par le 3-aminobenzamide : Sirtuin 1 activation : a neuroprotective strategy for in vivo cerebral oxidative stress ? Involvement of SIRT1 in the beneficial effects of poly(ADP-ribose)polymerase inhibition. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2013. [cited 2020 Jun 05]. Available from: http://www.theses.fr/2013PA05P610.

Council of Science Editors:

Gueguen C. L’activation de la sirtuin 1 : une nouvelle stratégie neuroprotectrice pour le stress oxydant cérébral in vivo ? Implication dans les effets bénéfiques de l’inhibition de la poly(ADP-ribose)polymérase par le 3-aminobenzamide : Sirtuin 1 activation : a neuroprotective strategy for in vivo cerebral oxidative stress ? Involvement of SIRT1 in the beneficial effects of poly(ADP-ribose)polymerase inhibition. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2013. Available from: http://www.theses.fr/2013PA05P610

11. NGUYEN THI KIM OANH. Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses.

Degree: 2018, Ajou University

Mitochondria are dynamic organelles essential for the life and death of the cells. The dynamic nature of mitochondria allows them to organize a network of… (more)

Subjects/Keywords: DNA damage signaling; Mitochondria fission; Nuclear; JNK, Jun N-terminal Kinase; SIRT1, Sirtuin 1; ATM, ataxia-telangiectasia mutated; DNA 손상신호; 미토콘드리아 분열;

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APA (6th Edition):

OANH, N. T. K. (2018). Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

OANH, NGUYEN THI KIM. “Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses.” 2018. Thesis, Ajou University. Accessed June 05, 2020. http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

OANH, NGUYEN THI KIM. “Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses.” 2018. Web. 05 Jun 2020.

Vancouver:

OANH NTK. Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses. [Internet] [Thesis]. Ajou University; 2018. [cited 2020 Jun 05]. Available from: http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

OANH NTK. Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses. [Thesis]. Ajou University; 2018. Available from: http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Diallo, Issa. Nouvelle méthode en protéomique pour améliorer l'identification et la quantification des protéines acétylées : Developement of a new proteomic method to improve identification and quantification of acetylated proteins.

Degree: Docteur es, BIS - Biotechnologie, instrumentation, signal et imagerie pour la biologie, la médecine et l'environnement, 2017, Grenoble Alpes

 L'acétylation des protéines constitue l’une des plus importantes modifications post-traductionnelles (PTMs). Elle intervient dans de multiples processus bologiques et physiopathologiques tels que, l’activité transcriptionnelle, l'apoptose,… (more)

Subjects/Keywords: Spectrometrie de masse; Acétylation; Fractionnement OFFGEL; Sirtuine 1 (SIRT1); Modification post-Traductionnelle (PTM); Di-Méthylation; Mass spectrometry; Acetylation; OFFGEL Fractionation; Sirtuin 1 (SIRT1); Post-Translational modification (PTM); Reductive methylation; 570; 610

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APA (6th Edition):

Diallo, I. (2017). Nouvelle méthode en protéomique pour améliorer l'identification et la quantification des protéines acétylées : Developement of a new proteomic method to improve identification and quantification of acetylated proteins. (Doctoral Dissertation). Grenoble Alpes. Retrieved from http://www.theses.fr/2017GREAS035

Chicago Manual of Style (16th Edition):

Diallo, Issa. “Nouvelle méthode en protéomique pour améliorer l'identification et la quantification des protéines acétylées : Developement of a new proteomic method to improve identification and quantification of acetylated proteins.” 2017. Doctoral Dissertation, Grenoble Alpes. Accessed June 05, 2020. http://www.theses.fr/2017GREAS035.

MLA Handbook (7th Edition):

Diallo, Issa. “Nouvelle méthode en protéomique pour améliorer l'identification et la quantification des protéines acétylées : Developement of a new proteomic method to improve identification and quantification of acetylated proteins.” 2017. Web. 05 Jun 2020.

Vancouver:

Diallo I. Nouvelle méthode en protéomique pour améliorer l'identification et la quantification des protéines acétylées : Developement of a new proteomic method to improve identification and quantification of acetylated proteins. [Internet] [Doctoral dissertation]. Grenoble Alpes; 2017. [cited 2020 Jun 05]. Available from: http://www.theses.fr/2017GREAS035.

Council of Science Editors:

Diallo I. Nouvelle méthode en protéomique pour améliorer l'identification et la quantification des protéines acétylées : Developement of a new proteomic method to improve identification and quantification of acetylated proteins. [Doctoral Dissertation]. Grenoble Alpes; 2017. Available from: http://www.theses.fr/2017GREAS035

13. Pantazi, Eirini. New strategies to reduce liver ischemia – reperfusion injury in fatty and non-fatty livers: a focus on sirtuin 1 implication.

Degree: 2015, Universitat de Barcelona

 La lesión por isquemia-reperfusion (LIR) hepática es causa de una significativa mortalidad en caso de resecciones hepáticas y en el trasplante hepático. La LIR está… (more)

Subjects/Keywords: Malalties del fetge; Enfermedades del higado; Liver diseases; Trasplantament hepàtic; Trasplante hepático; Hepatic transplantation; Lesión por isquemia-reperfusión; Lesió per isquèmia-reperfusió; Ischemia-reperfusion injury; Sirtuina 1; Sirtuin 1; Ciències de la Salut; 577

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APA (6th Edition):

Pantazi, E. (2015). New strategies to reduce liver ischemia – reperfusion injury in fatty and non-fatty livers: a focus on sirtuin 1 implication. (Thesis). Universitat de Barcelona. Retrieved from http://hdl.handle.net/10803/299797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pantazi, Eirini. “New strategies to reduce liver ischemia – reperfusion injury in fatty and non-fatty livers: a focus on sirtuin 1 implication.” 2015. Thesis, Universitat de Barcelona. Accessed June 05, 2020. http://hdl.handle.net/10803/299797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pantazi, Eirini. “New strategies to reduce liver ischemia – reperfusion injury in fatty and non-fatty livers: a focus on sirtuin 1 implication.” 2015. Web. 05 Jun 2020.

Vancouver:

Pantazi E. New strategies to reduce liver ischemia – reperfusion injury in fatty and non-fatty livers: a focus on sirtuin 1 implication. [Internet] [Thesis]. Universitat de Barcelona; 2015. [cited 2020 Jun 05]. Available from: http://hdl.handle.net/10803/299797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pantazi E. New strategies to reduce liver ischemia – reperfusion injury in fatty and non-fatty livers: a focus on sirtuin 1 implication. [Thesis]. Universitat de Barcelona; 2015. Available from: http://hdl.handle.net/10803/299797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Li, Kai. Regulation of WRN Function by Acetylation and SIRT1-Mediated Deacetylation in Response to DNA Damage: A Dissertation.

Degree: Cancer Biology, Cancer Biology, 2010, U of Massachusetts : Med

  Werner syndrome (WS) is an autosomal recessive disorder associated with premature aging and cancer predisposition. WS cells show increased genomic instability and are hypersensitive… (more)

Subjects/Keywords: Exodeoxyribonucleases; RecQ Helicases; Acetylation; Sirtuin 1; DNA Damage; Amino Acids, Peptides, and Proteins; Cancer Biology; Congenital, Hereditary, and Neonatal Diseases and Abnormalities; Enzymes and Coenzymes; Genetic Phenomena; Nutritional and Metabolic Diseases

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APA (6th Edition):

Li, K. (2010). Regulation of WRN Function by Acetylation and SIRT1-Mediated Deacetylation in Response to DNA Damage: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/511

Chicago Manual of Style (16th Edition):

Li, Kai. “Regulation of WRN Function by Acetylation and SIRT1-Mediated Deacetylation in Response to DNA Damage: A Dissertation.” 2010. Doctoral Dissertation, U of Massachusetts : Med. Accessed June 05, 2020. https://escholarship.umassmed.edu/gsbs_diss/511.

MLA Handbook (7th Edition):

Li, Kai. “Regulation of WRN Function by Acetylation and SIRT1-Mediated Deacetylation in Response to DNA Damage: A Dissertation.” 2010. Web. 05 Jun 2020.

Vancouver:

Li K. Regulation of WRN Function by Acetylation and SIRT1-Mediated Deacetylation in Response to DNA Damage: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2010. [cited 2020 Jun 05]. Available from: https://escholarship.umassmed.edu/gsbs_diss/511.

Council of Science Editors:

Li K. Regulation of WRN Function by Acetylation and SIRT1-Mediated Deacetylation in Response to DNA Damage: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2010. Available from: https://escholarship.umassmed.edu/gsbs_diss/511

15. Owczarczyk, Anna. The Role of Sirtuin 1 in the Regulation of Autophagy and Respiratory Syncytial Virus-induced Immune Responses.

Degree: PhD, Molecular & Cellular Path PhD, 2017, University of Michigan

 Respiratory syncytial virus (RSV) is the leading cause of lower respiratory tract disease in children, and a significant source of morbidity and mortality among those… (more)

Subjects/Keywords: SIRT1 regulates DC function and autophagy during RSV-induced immunity; Sirtuin 1; Autophagy; Respiratory Syncytial Virus; Innate Immunity; Microbiology and Immunology; Science

…96 v i L IST OFF IGURES F i gu r e1 .1 : Hum anr e sp i r a t o r ys yn cy t i a lv… …34 F i gu r e3 .1 :S i r t1i sup r e gu l a t edina m ou s em od e lo fR SVin f e c t i… …s en tan t i g ent oTc e l l s . . . . . . . . . . . . . . . 70 v i i F i gu r e4 .1… …i on l e v e l s .. . . . . . . . . . . . . . . . . . . . . . . . . 79 F i gu r e5 .1… …83 v i i i L IST OFTABLES T ab l e1 .1 :P r op e r t i e sandfun c t i on so fm amm… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Owczarczyk, A. (2017). The Role of Sirtuin 1 in the Regulation of Autophagy and Respiratory Syncytial Virus-induced Immune Responses. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/137127

Chicago Manual of Style (16th Edition):

Owczarczyk, Anna. “The Role of Sirtuin 1 in the Regulation of Autophagy and Respiratory Syncytial Virus-induced Immune Responses.” 2017. Doctoral Dissertation, University of Michigan. Accessed June 05, 2020. http://hdl.handle.net/2027.42/137127.

MLA Handbook (7th Edition):

Owczarczyk, Anna. “The Role of Sirtuin 1 in the Regulation of Autophagy and Respiratory Syncytial Virus-induced Immune Responses.” 2017. Web. 05 Jun 2020.

Vancouver:

Owczarczyk A. The Role of Sirtuin 1 in the Regulation of Autophagy and Respiratory Syncytial Virus-induced Immune Responses. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2020 Jun 05]. Available from: http://hdl.handle.net/2027.42/137127.

Council of Science Editors:

Owczarczyk A. The Role of Sirtuin 1 in the Regulation of Autophagy and Respiratory Syncytial Virus-induced Immune Responses. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/137127

16. Atkins, Katelyn Mae. PACS-2 regulates SIRT 1-mediated deacetylation of p53 following DNA damage.

Degree: PhD, 2012, Oregon Health Sciences University

Subjects/Keywords: Membrane proteins; Sirtuins; Carrier proteins; DNA damage; Viral proteins; p53-AIP1 (p53-regulated apoptosis inducing protein 1); PACS-2 (phosphofurin acidic cluster sorting protein-2); Apoptosis Regulatory Proteins; Sirtuin 1; Vesicular Transport Proteins; DNA Damage; Viral Regulatory and Accessory Proteins

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Atkins, K. M. (2012). PACS-2 regulates SIRT 1-mediated deacetylation of p53 following DNA damage. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4MG7MH2 ; http://digitalcommons.ohsu.edu/etd/862

Chicago Manual of Style (16th Edition):

Atkins, Katelyn Mae. “PACS-2 regulates SIRT 1-mediated deacetylation of p53 following DNA damage.” 2012. Doctoral Dissertation, Oregon Health Sciences University. Accessed June 05, 2020. doi:10.6083/M4MG7MH2 ; http://digitalcommons.ohsu.edu/etd/862.

MLA Handbook (7th Edition):

Atkins, Katelyn Mae. “PACS-2 regulates SIRT 1-mediated deacetylation of p53 following DNA damage.” 2012. Web. 05 Jun 2020.

Vancouver:

Atkins KM. PACS-2 regulates SIRT 1-mediated deacetylation of p53 following DNA damage. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2012. [cited 2020 Jun 05]. Available from: doi:10.6083/M4MG7MH2 ; http://digitalcommons.ohsu.edu/etd/862.

Council of Science Editors:

Atkins KM. PACS-2 regulates SIRT 1-mediated deacetylation of p53 following DNA damage. [Doctoral Dissertation]. Oregon Health Sciences University; 2012. Available from: doi:10.6083/M4MG7MH2 ; http://digitalcommons.ohsu.edu/etd/862

.