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You searched for subject:(Silicon quantum dot solar cell). Showing records 1 – 30 of 73 total matches.

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1. Keifenheim, Daniel L. Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2015, U of Massachusetts : Med

  The coordination between cell growth and division is a highly regulated process that is intimately linked to the cell cycle. Efforts to identify an… (more)

Subjects/Keywords: Cell Size; Cell Cycle; Cell Cycle Checkpoints; Cell Division; Schizosaccharomyces; Cell Biology; Cellular and Molecular Physiology

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APA (6th Edition):

Keifenheim, D. L. (2015). Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/784

Chicago Manual of Style (16th Edition):

Keifenheim, Daniel L. “Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/784.

MLA Handbook (7th Edition):

Keifenheim, Daniel L. “Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.” 2015. Web. 14 Dec 2019.

Vancouver:

Keifenheim DL. Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/784.

Council of Science Editors:

Keifenheim DL. Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: http://escholarship.umassmed.edu/gsbs_diss/784

2. Nelson, Charles J. MicroRNA Regulation of Autophagy during Programmed Cell Death: A Dissertation.

Degree: Interdisciplinary Graduate Program, NeuroNexus Neuroscience Institute, 2015, U of Massachusetts : Med

  Autophagy delivers cytoplasmic material to the lysosome for degradation, and has been implicated in many cellular processes, including stress, infection, survival, and death. Although… (more)

Subjects/Keywords: Autophagy; Cell Death; Cell Survival; MicroRNAs; Cell and Developmental Biology; Cell Biology; Cellular and Molecular Physiology

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APA (6th Edition):

Nelson, C. J. (2015). MicroRNA Regulation of Autophagy during Programmed Cell Death: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/756

Chicago Manual of Style (16th Edition):

Nelson, Charles J. “MicroRNA Regulation of Autophagy during Programmed Cell Death: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/756.

MLA Handbook (7th Edition):

Nelson, Charles J. “MicroRNA Regulation of Autophagy during Programmed Cell Death: A Dissertation.” 2015. Web. 14 Dec 2019.

Vancouver:

Nelson CJ. MicroRNA Regulation of Autophagy during Programmed Cell Death: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/756.

Council of Science Editors:

Nelson CJ. MicroRNA Regulation of Autophagy during Programmed Cell Death: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: http://escholarship.umassmed.edu/gsbs_diss/756

3. Kuo, Tse-Chun. Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2013, U of Massachusetts : Med

  The midbody (MB) is a proteinaceous complex formed between the two daughter cells during cell division and is required for the final cell separation… (more)

Subjects/Keywords: Autophagy; Cell Division; Cell Differentiation; Cell Cycle Proteins; Nuclear Proteins; Cell Biology; Cellular and Molecular Physiology

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APA (6th Edition):

Kuo, T. (2013). Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/670

Chicago Manual of Style (16th Edition):

Kuo, Tse-Chun. “Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/670.

MLA Handbook (7th Edition):

Kuo, Tse-Chun. “Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation.” 2013. Web. 14 Dec 2019.

Vancouver:

Kuo T. Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/670.

Council of Science Editors:

Kuo T. Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/670

4. Hung, Hui-Fang. Roles of the Mother Centriole Appendage Protein Cenexin in Microtubule Organization during Cell Migration and Cell Division: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2016, U of Massachusetts : Med

  Epithelial cells are necessary building blocks of the organs they line. Their apicalbasolateral polarity, characterized by an asymmetric distribution of cell components along their… (more)

Subjects/Keywords: Heat-Shock Proteins; Cell Division; Cell Movement; Cell Polarity; Centrioles; Centrosome; Endosomes; Epithelial Cells; Microtubule-Organizing Center; Microtubules; Cenexin; Cell Biology; Cellular and Molecular Physiology

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APA (6th Edition):

Hung, H. (2016). Roles of the Mother Centriole Appendage Protein Cenexin in Microtubule Organization during Cell Migration and Cell Division: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/842

Chicago Manual of Style (16th Edition):

Hung, Hui-Fang. “Roles of the Mother Centriole Appendage Protein Cenexin in Microtubule Organization during Cell Migration and Cell Division: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/842.

MLA Handbook (7th Edition):

Hung, Hui-Fang. “Roles of the Mother Centriole Appendage Protein Cenexin in Microtubule Organization during Cell Migration and Cell Division: A Dissertation.” 2016. Web. 14 Dec 2019.

Vancouver:

Hung H. Roles of the Mother Centriole Appendage Protein Cenexin in Microtubule Organization during Cell Migration and Cell Division: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/842.

Council of Science Editors:

Hung H. Roles of the Mother Centriole Appendage Protein Cenexin in Microtubule Organization during Cell Migration and Cell Division: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/842

5. Chen, Chun-Ting. Asymmetric Inheritance and Post-mitotic Fate of Midbodies: A Dissertation.

Degree: Interdisciplinary Graduate Program, Molecular Medicine, 2011, U of Massachusetts : Med

  Cytokinesis is the final chapter of cell division and its last page is abscission, the physical separation of two daughter cells. During cytokinesis of… (more)

Subjects/Keywords: Cytokinesis; Cell and Developmental Biology; Cells; Neoplasms

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APA (6th Edition):

Chen, C. (2011). Asymmetric Inheritance and Post-mitotic Fate of Midbodies: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/564

Chicago Manual of Style (16th Edition):

Chen, Chun-Ting. “Asymmetric Inheritance and Post-mitotic Fate of Midbodies: A Dissertation.” 2011. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. https://escholarship.umassmed.edu/gsbs_diss/564.

MLA Handbook (7th Edition):

Chen, Chun-Ting. “Asymmetric Inheritance and Post-mitotic Fate of Midbodies: A Dissertation.” 2011. Web. 14 Dec 2019.

Vancouver:

Chen C. Asymmetric Inheritance and Post-mitotic Fate of Midbodies: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2011. [cited 2019 Dec 14]. Available from: https://escholarship.umassmed.edu/gsbs_diss/564.

Council of Science Editors:

Chen C. Asymmetric Inheritance and Post-mitotic Fate of Midbodies: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2011. Available from: https://escholarship.umassmed.edu/gsbs_diss/564

6. Schmidt, Tracy E. HIV-1 and the Nucleolus: A Role for Nucleophosmin/NPM1 in Viral Replication: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2013, U of Massachusetts : Med

  The nucleolus is a plurifunctional organelle with dynamic protein exchange involved in diverse aspects of cell biology. Additionally, the nucleolus has been shown to… (more)

Subjects/Keywords: Cell Nucleolus; Virus Replication; Nuclear Proteins; HIV-1; Cell Biology; Cellular and Molecular Physiology; Virology

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APA (6th Edition):

Schmidt, T. E. (2013). HIV-1 and the Nucleolus: A Role for Nucleophosmin/NPM1 in Viral Replication: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/690

Chicago Manual of Style (16th Edition):

Schmidt, Tracy E. “HIV-1 and the Nucleolus: A Role for Nucleophosmin/NPM1 in Viral Replication: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/690.

MLA Handbook (7th Edition):

Schmidt, Tracy E. “HIV-1 and the Nucleolus: A Role for Nucleophosmin/NPM1 in Viral Replication: A Dissertation.” 2013. Web. 14 Dec 2019.

Vancouver:

Schmidt TE. HIV-1 and the Nucleolus: A Role for Nucleophosmin/NPM1 in Viral Replication: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/690.

Council of Science Editors:

Schmidt TE. HIV-1 and the Nucleolus: A Role for Nucleophosmin/NPM1 in Viral Replication: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/690

7. Nordberg, Joshua J. The Importance of the Centrosomal Localization Sequence of Cyclin E for Promoting Centrosome Duplication: A Dissertation.

Degree: Interdisciplinary Graduate Program, Radiology, 2011, U of Massachusetts : Med

  This thesis comprises three separate studies that investigate the consequences of supernumary centrosomes, the effect of centrosome loss, and a control mechanism for regulating… (more)

Subjects/Keywords: Centrosome; Cell Cycle; Cyclin-Dependent Kinase 2; Cyclin E; Cell Biology; Cells; Enzymes and Coenzymes

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APA (6th Edition):

Nordberg, J. J. (2011). The Importance of the Centrosomal Localization Sequence of Cyclin E for Promoting Centrosome Duplication: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/547

Chicago Manual of Style (16th Edition):

Nordberg, Joshua J. “The Importance of the Centrosomal Localization Sequence of Cyclin E for Promoting Centrosome Duplication: A Dissertation.” 2011. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. https://escholarship.umassmed.edu/gsbs_diss/547.

MLA Handbook (7th Edition):

Nordberg, Joshua J. “The Importance of the Centrosomal Localization Sequence of Cyclin E for Promoting Centrosome Duplication: A Dissertation.” 2011. Web. 14 Dec 2019.

Vancouver:

Nordberg JJ. The Importance of the Centrosomal Localization Sequence of Cyclin E for Promoting Centrosome Duplication: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2011. [cited 2019 Dec 14]. Available from: https://escholarship.umassmed.edu/gsbs_diss/547.

Council of Science Editors:

Nordberg JJ. The Importance of the Centrosomal Localization Sequence of Cyclin E for Promoting Centrosome Duplication: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2011. Available from: https://escholarship.umassmed.edu/gsbs_diss/547

8. Bhaduri, Samyabrata. Regulation of CDK1 Activity during the G1/S Transition in S. cerevisiae through Specific Cyclin-Substrate Docking: A Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2014, U of Massachusetts : Med

  Several cell cycle events require specific forms of the cyclin-CDK complexes. It has been known for some time that cyclins not only contribute by… (more)

Subjects/Keywords: Cell Cycle; Cell Cycle Proteins; Saccharomyces cerevisiae Proteins; Cyclin B; Cyclin G1; Cyclins; Cyclin-Dependent Kinases; CDC2 Protein; Biochemistry; Cell Biology; Molecular Biology

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APA (6th Edition):

Bhaduri, S. (2014). Regulation of CDK1 Activity during the G1/S Transition in S. cerevisiae through Specific Cyclin-Substrate Docking: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/871

Chicago Manual of Style (16th Edition):

Bhaduri, Samyabrata. “Regulation of CDK1 Activity during the G1/S Transition in S. cerevisiae through Specific Cyclin-Substrate Docking: A Dissertation.” 2014. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/871.

MLA Handbook (7th Edition):

Bhaduri, Samyabrata. “Regulation of CDK1 Activity during the G1/S Transition in S. cerevisiae through Specific Cyclin-Substrate Docking: A Dissertation.” 2014. Web. 14 Dec 2019.

Vancouver:

Bhaduri S. Regulation of CDK1 Activity during the G1/S Transition in S. cerevisiae through Specific Cyclin-Substrate Docking: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2014. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/871.

Council of Science Editors:

Bhaduri S. Regulation of CDK1 Activity during the G1/S Transition in S. cerevisiae through Specific Cyclin-Substrate Docking: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2014. Available from: http://escholarship.umassmed.edu/gsbs_diss/871

9. Gupta, Sneha. Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2013, U of Massachusetts : Med

  The fission yeast Schizosaccharomyces pombe has become a powerful model system for studying cytokinesis, a process of cytoplasmic division by which one cell divides… (more)

Subjects/Keywords: Cytokinesis; Cell Cycle; Cell Cycle Proteins; Cytoskeleton; Cytoskeletal Proteins; Schizosaccharomyces; Schizosaccharomyces pombe Proteins; Cellular and Molecular Physiology

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APA (6th Edition):

Gupta, S. (2013). Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/693

Chicago Manual of Style (16th Edition):

Gupta, Sneha. “Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/693.

MLA Handbook (7th Edition):

Gupta, Sneha. “Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation.” 2013. Web. 14 Dec 2019.

Vancouver:

Gupta S. Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/693.

Council of Science Editors:

Gupta S. Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/693

10. Sun, Xiaoming. Ki-67 Regulates Cell Cycle Progression and Heterochromatin Organization.

Degree: Interdisciplinary Graduate Program, Molecular, Cell and Cancer Biology, 2017, U of Massachusetts : Med

  A subset of eukaryotic heterochromatin is located around the nucleoli, and this localization is correlated with gene silencing. Although there is some evidence for… (more)

Subjects/Keywords: Ki-67; cell cycle; X chromosome inactivation; epigenetics; higher order organization of the genome; gene regulation; Cell Biology; Life Sciences

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APA (6th Edition):

Sun, X. (2017). Ki-67 Regulates Cell Cycle Progression and Heterochromatin Organization. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/920

Chicago Manual of Style (16th Edition):

Sun, Xiaoming. “Ki-67 Regulates Cell Cycle Progression and Heterochromatin Organization.” 2017. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/920.

MLA Handbook (7th Edition):

Sun, Xiaoming. “Ki-67 Regulates Cell Cycle Progression and Heterochromatin Organization.” 2017. Web. 14 Dec 2019.

Vancouver:

Sun X. Ki-67 Regulates Cell Cycle Progression and Heterochromatin Organization. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2017. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/920.

Council of Science Editors:

Sun X. Ki-67 Regulates Cell Cycle Progression and Heterochromatin Organization. [Doctoral Dissertation]. U of Massachusetts : Med; 2017. Available from: http://escholarship.umassmed.edu/gsbs_diss/920

11. Pope, Patricia A. Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2013, U of Massachusetts : Med

  Progression through the cell cycle is tightly controlled, and the decision whether or not to enter a new cell cycle can be influenced by… (more)

Subjects/Keywords: G1 Phase Cell Cycle Checkpoints; Pheromones; Saccharomyces cerevisiae; Cell Biology; Cellular and Molecular Physiology; Molecular Biology

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APA (6th Edition):

Pope, P. A. (2013). Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/680

Chicago Manual of Style (16th Edition):

Pope, Patricia A. “Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/680.

MLA Handbook (7th Edition):

Pope, Patricia A. “Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation.” 2013. Web. 14 Dec 2019.

Vancouver:

Pope PA. Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/680.

Council of Science Editors:

Pope PA. Investigation of Multiple Concerted Mechanisms Underlying Stimulus-induced G1 Arrest in Yeast: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/680

12. Lu, Simin. Calcium Dependent Regulatory Mechanism in Wolfram Syndrome: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2015, U of Massachusetts : Med

  Wolfram syndrome is a genetic disorder characterized by diabetes and neurodegeneration. Two causative genes have been identified so far, WFS1 and WFS2, both encoding… (more)

Subjects/Keywords: Calpain; Cell Death; Endoplasmic Reticulum; Wolfram Syndrome; Cell Biology; Cellular and Molecular Physiology; Congenital, Hereditary, and Neonatal Diseases and Abnormalities

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APA (6th Edition):

Lu, S. (2015). Calcium Dependent Regulatory Mechanism in Wolfram Syndrome: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/733

Chicago Manual of Style (16th Edition):

Lu, Simin. “Calcium Dependent Regulatory Mechanism in Wolfram Syndrome: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/733.

MLA Handbook (7th Edition):

Lu, Simin. “Calcium Dependent Regulatory Mechanism in Wolfram Syndrome: A Dissertation.” 2015. Web. 14 Dec 2019.

Vancouver:

Lu S. Calcium Dependent Regulatory Mechanism in Wolfram Syndrome: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/733.

Council of Science Editors:

Lu S. Calcium Dependent Regulatory Mechanism in Wolfram Syndrome: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: http://escholarship.umassmed.edu/gsbs_diss/733

13. Burke, Samantha L. MicroRNAs Protect the Robustness of Distal Tip Cell Migrations from Temperature Changes in Caenorhabditis elegans: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2015, U of Massachusetts : Med

  MicroRNAs play an important role in protecting biological robustness during development. Biological robustness is the ability to maintain a consistent output despite variation in… (more)

Subjects/Keywords: MicroRNAs; Caenorhabditis elegans; Cell Movement; Temperature; Developmental Biology; Genetics and Genomics

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APA (6th Edition):

Burke, S. L. (2015). MicroRNAs Protect the Robustness of Distal Tip Cell Migrations from Temperature Changes in Caenorhabditis elegans: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/783

Chicago Manual of Style (16th Edition):

Burke, Samantha L. “MicroRNAs Protect the Robustness of Distal Tip Cell Migrations from Temperature Changes in Caenorhabditis elegans: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/783.

MLA Handbook (7th Edition):

Burke, Samantha L. “MicroRNAs Protect the Robustness of Distal Tip Cell Migrations from Temperature Changes in Caenorhabditis elegans: A Dissertation.” 2015. Web. 14 Dec 2019.

Vancouver:

Burke SL. MicroRNAs Protect the Robustness of Distal Tip Cell Migrations from Temperature Changes in Caenorhabditis elegans: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/783.

Council of Science Editors:

Burke SL. MicroRNAs Protect the Robustness of Distal Tip Cell Migrations from Temperature Changes in Caenorhabditis elegans: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: http://escholarship.umassmed.edu/gsbs_diss/783

14. Dutta, Shubham. Role of TRIP6 and Angiomotins in the Regulation of the Hippo Signaling Pathway.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2018, U of Massachusetts : Med

  Mechanical tension is an important regulator of cell proliferation, differentiation, migration and cell death. It is involved in the control of tissue architecture and… (more)

Subjects/Keywords: Hippo signaling pathway; TRIP6; vinculin; LATS; MOB; YAP; stretch; mechanosensing; mechanical tension; adheres junction; cell junction; Biochemistry, Biophysics, and Structural Biology; Biology; Cell and Developmental Biology

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APA (6th Edition):

Dutta, S. (2018). Role of TRIP6 and Angiomotins in the Regulation of the Hippo Signaling Pathway. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/964

Chicago Manual of Style (16th Edition):

Dutta, Shubham. “Role of TRIP6 and Angiomotins in the Regulation of the Hippo Signaling Pathway.” 2018. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. https://escholarship.umassmed.edu/gsbs_diss/964.

MLA Handbook (7th Edition):

Dutta, Shubham. “Role of TRIP6 and Angiomotins in the Regulation of the Hippo Signaling Pathway.” 2018. Web. 14 Dec 2019.

Vancouver:

Dutta S. Role of TRIP6 and Angiomotins in the Regulation of the Hippo Signaling Pathway. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2018. [cited 2019 Dec 14]. Available from: https://escholarship.umassmed.edu/gsbs_diss/964.

Council of Science Editors:

Dutta S. Role of TRIP6 and Angiomotins in the Regulation of the Hippo Signaling Pathway. [Doctoral Dissertation]. U of Massachusetts : Med; 2018. Available from: https://escholarship.umassmed.edu/gsbs_diss/964

15. Min, So Yun. The Origin of Human White, Brown, and Brite/Beige Adipocytes.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2016, U of Massachusetts : Med

  During embryonic development, adipocytes emerge from microvasculature. Lineage-­‐tracing studies in mice have shown that adipocyte progenitors reside in the adipose tissue capillaries. However, the… (more)

Subjects/Keywords: Human adipocyte; White adipocyte; Brown adipocyte; Brite adipocyte; Beige adipocyte; Metabolism; Thermogenic adipose tissue; Insulin resistance; Glucose homeostasis; Adipocyte implantation; Biology; Cell Biology; Medical Cell Biology; Nutritional and Metabolic Diseases

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APA (6th Edition):

Min, S. Y. (2016). The Origin of Human White, Brown, and Brite/Beige Adipocytes. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/878

Chicago Manual of Style (16th Edition):

Min, So Yun. “The Origin of Human White, Brown, and Brite/Beige Adipocytes.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/878.

MLA Handbook (7th Edition):

Min, So Yun. “The Origin of Human White, Brown, and Brite/Beige Adipocytes.” 2016. Web. 14 Dec 2019.

Vancouver:

Min SY. The Origin of Human White, Brown, and Brite/Beige Adipocytes. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/878.

Council of Science Editors:

Min SY. The Origin of Human White, Brown, and Brite/Beige Adipocytes. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/878

16. Elewa, Ahmed M. POS-1 Regulation of Endo-mesoderm Identity in C. elegans: A Dissertation.

Degree: Interdisciplinary Graduate Program, RNA Therapeutics Institute, 2014, U of Massachusetts : Med

  How do embryos develop with such poise from a single zygote to multiple cells with different identities, and yet survive? At the four-cell stage… (more)

Subjects/Keywords: Blastomeres; Caenorhabditis elegans; Carrier Proteins; Cell Differentiation; DNA-Binding Proteins; Ectoderm; Endoderm; Mesoderm; Transcription Factors; Cell Biology; Cellular and Molecular Physiology; Developmental Biology

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APA (6th Edition):

Elewa, A. M. (2014). POS-1 Regulation of Endo-mesoderm Identity in C. elegans: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/711

Chicago Manual of Style (16th Edition):

Elewa, Ahmed M. “POS-1 Regulation of Endo-mesoderm Identity in C. elegans: A Dissertation.” 2014. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/711.

MLA Handbook (7th Edition):

Elewa, Ahmed M. “POS-1 Regulation of Endo-mesoderm Identity in C. elegans: A Dissertation.” 2014. Web. 14 Dec 2019.

Vancouver:

Elewa AM. POS-1 Regulation of Endo-mesoderm Identity in C. elegans: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2014. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/711.

Council of Science Editors:

Elewa AM. POS-1 Regulation of Endo-mesoderm Identity in C. elegans: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2014. Available from: http://escholarship.umassmed.edu/gsbs_diss/711

17. Bright, Alison R. A Role for Intraflagellar Transport Proteins in Mitosis: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2013, U of Massachusetts : Med

  Disruption of cilia proteins results in a range of disorders called ciliopathies. However, the mechanism by which cilia dysfunction contributes to disease is not… (more)

Subjects/Keywords: Carrier Proteins; Cilia; Ciliary Motility Disorders; Mitosis; Cell Biology; Cellular and Molecular Physiology

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APA (6th Edition):

Bright, A. R. (2013). A Role for Intraflagellar Transport Proteins in Mitosis: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/682

Chicago Manual of Style (16th Edition):

Bright, Alison R. “A Role for Intraflagellar Transport Proteins in Mitosis: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/682.

MLA Handbook (7th Edition):

Bright, Alison R. “A Role for Intraflagellar Transport Proteins in Mitosis: A Dissertation.” 2013. Web. 14 Dec 2019.

Vancouver:

Bright AR. A Role for Intraflagellar Transport Proteins in Mitosis: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/682.

Council of Science Editors:

Bright AR. A Role for Intraflagellar Transport Proteins in Mitosis: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/682

18. Barutcu, Seda. Role of JIP1-JNK Signaling in Beta-Cell Function and Autophagy.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2018, U of Massachusetts : Med

  Proper functioning of endocrine cells is crucial for organismal homeostasis. The underlying mechanisms that fine-tune the amount, and the timing of hormone secretion are… (more)

Subjects/Keywords: JIP1 / MAPK8IP1; JNK; Insulin; TSH; Secretion; Autophagy; Biology; Cell Biology; Cellular and Molecular Physiology

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APA (6th Edition):

Barutcu, S. (2018). Role of JIP1-JNK Signaling in Beta-Cell Function and Autophagy. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/954

Chicago Manual of Style (16th Edition):

Barutcu, Seda. “Role of JIP1-JNK Signaling in Beta-Cell Function and Autophagy.” 2018. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. https://escholarship.umassmed.edu/gsbs_diss/954.

MLA Handbook (7th Edition):

Barutcu, Seda. “Role of JIP1-JNK Signaling in Beta-Cell Function and Autophagy.” 2018. Web. 14 Dec 2019.

Vancouver:

Barutcu S. Role of JIP1-JNK Signaling in Beta-Cell Function and Autophagy. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2018. [cited 2019 Dec 14]. Available from: https://escholarship.umassmed.edu/gsbs_diss/954.

Council of Science Editors:

Barutcu S. Role of JIP1-JNK Signaling in Beta-Cell Function and Autophagy. [Doctoral Dissertation]. U of Massachusetts : Med; 2018. Available from: https://escholarship.umassmed.edu/gsbs_diss/954

19. Wang, Mengxi. Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2013, U of Massachusetts : Med

  Skeletal muscle is a complicated and heterogeneous striated muscle tissue that serves critical mechanical and metabolic functions in the organism. The process of generating… (more)

Subjects/Keywords: Cell Differentiation; Cell Line; Developmental Gene Expression Regulation; MAP Kinase Signaling System; Muscle Development; Skeletal Muscle Fibers; Myoblasts; Myogenic Regulatory Factor 5; Protein-Serine-Threonine Kinases; RNA Interference; Small Interfering RNA; Up-Regulation; Cell Biology; Developmental Biology; Molecular Biology; Molecular Genetics

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APA (6th Edition):

Wang, M. (2013). Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/675

Chicago Manual of Style (16th Edition):

Wang, Mengxi. “Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/675.

MLA Handbook (7th Edition):

Wang, Mengxi. “Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation.” 2013. Web. 14 Dec 2019.

Vancouver:

Wang M. Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/675.

Council of Science Editors:

Wang M. Role of Map4k4 in Skeletal Muscle Differentiation: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/675

20. Guo, Chang-An. Genetic Deficiency of CD40 in Mice Exacerbates Metabolic Manifestations of Diet-induced Obesity: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2013, U of Massachusetts : Med

  The past two decades have seen an explosive increase of obesity rates worldwide, with more than one billion adults overweight and 300 million of… (more)

Subjects/Keywords: Adipose tissue inflammation; CD40; CD8+ T cell; hepatic steatosis; insulin resistance; Biochemistry; Cellular and Molecular Physiology; Endocrinology; Immunopathology; Molecular Genetics

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APA (6th Edition):

Guo, C. (2013). Genetic Deficiency of CD40 in Mice Exacerbates Metabolic Manifestations of Diet-induced Obesity: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/678

Chicago Manual of Style (16th Edition):

Guo, Chang-An. “Genetic Deficiency of CD40 in Mice Exacerbates Metabolic Manifestations of Diet-induced Obesity: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/678.

MLA Handbook (7th Edition):

Guo, Chang-An. “Genetic Deficiency of CD40 in Mice Exacerbates Metabolic Manifestations of Diet-induced Obesity: A Dissertation.” 2013. Web. 14 Dec 2019.

Vancouver:

Guo C. Genetic Deficiency of CD40 in Mice Exacerbates Metabolic Manifestations of Diet-induced Obesity: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/678.

Council of Science Editors:

Guo C. Genetic Deficiency of CD40 in Mice Exacerbates Metabolic Manifestations of Diet-induced Obesity: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/678

21. DiStefano, Marina T. A Role for the Lipid Droplet Protein HIG2 in Promoting Lipid Deposition in Liver and Adipose Tissue: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2016, U of Massachusetts : Med

  Chronic exposure of humans or rodents to high calorie diets leads to hypertriglyceridemia and ectopic lipid deposition throughout the body, resulting in metabolic disease.… (more)

Subjects/Keywords: Brown Adipocytes; Brown Adipose Tissue; Glucose Intolerance; Lipid Droplets; Hepatocytes; Cell Biology; Cellular and Molecular Physiology; Endocrinology

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APA (6th Edition):

DiStefano, M. T. (2016). A Role for the Lipid Droplet Protein HIG2 in Promoting Lipid Deposition in Liver and Adipose Tissue: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/830

Chicago Manual of Style (16th Edition):

DiStefano, Marina T. “A Role for the Lipid Droplet Protein HIG2 in Promoting Lipid Deposition in Liver and Adipose Tissue: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/830.

MLA Handbook (7th Edition):

DiStefano, Marina T. “A Role for the Lipid Droplet Protein HIG2 in Promoting Lipid Deposition in Liver and Adipose Tissue: A Dissertation.” 2016. Web. 14 Dec 2019.

Vancouver:

DiStefano MT. A Role for the Lipid Droplet Protein HIG2 in Promoting Lipid Deposition in Liver and Adipose Tissue: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/830.

Council of Science Editors:

DiStefano MT. A Role for the Lipid Droplet Protein HIG2 in Promoting Lipid Deposition in Liver and Adipose Tissue: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/830

22. Oruganty, Aparna. Role of the Cytoplasmic Polyadenylation Element Binding Proteins in Neuron: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2013, U of Massachusetts : Med

  Genome regulation is an extremely complex phenomenon. There are various mechanisms in place to ensure smooth performance of the organism. Post-transcriptional regulation of gene… (more)

Subjects/Keywords: Polyadenylation; RNA-Binding Proteins; Transcription Factors; mRNA Cleavage and Polyadenylation Factors; Neurons; Cell and Developmental Biology

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APA (6th Edition):

Oruganty, A. (2013). Role of the Cytoplasmic Polyadenylation Element Binding Proteins in Neuron: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/648

Chicago Manual of Style (16th Edition):

Oruganty, Aparna. “Role of the Cytoplasmic Polyadenylation Element Binding Proteins in Neuron: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/648.

MLA Handbook (7th Edition):

Oruganty, Aparna. “Role of the Cytoplasmic Polyadenylation Element Binding Proteins in Neuron: A Dissertation.” 2013. Web. 14 Dec 2019.

Vancouver:

Oruganty A. Role of the Cytoplasmic Polyadenylation Element Binding Proteins in Neuron: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/648.

Council of Science Editors:

Oruganty A. Role of the Cytoplasmic Polyadenylation Element Binding Proteins in Neuron: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/648

23. Malinkevich, Anna. MIRAGE DNA Transposon Silencing by C. elegans Condensin II Subunit HCP-6: A Masters Thesis.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2014, U of Massachusetts : Med

  Mobile genetic elements represent a large portion of the genome in many species. Posing a danger to the integrity of genetic information, silencing and… (more)

Subjects/Keywords: Caenorhabditis elegans; DNA Transposable Elements; Gene Silencing; Genome; Cell Cycle Proteins; Biochemistry; Genetics and Genomics; Genomics

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APA (6th Edition):

Malinkevich, A. (2014). MIRAGE DNA Transposon Silencing by C. elegans Condensin II Subunit HCP-6: A Masters Thesis. (Masters Thesis). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/754

Chicago Manual of Style (16th Edition):

Malinkevich, Anna. “MIRAGE DNA Transposon Silencing by C. elegans Condensin II Subunit HCP-6: A Masters Thesis.” 2014. Masters Thesis, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/754.

MLA Handbook (7th Edition):

Malinkevich, Anna. “MIRAGE DNA Transposon Silencing by C. elegans Condensin II Subunit HCP-6: A Masters Thesis.” 2014. Web. 14 Dec 2019.

Vancouver:

Malinkevich A. MIRAGE DNA Transposon Silencing by C. elegans Condensin II Subunit HCP-6: A Masters Thesis. [Internet] [Masters thesis]. U of Massachusetts : Med; 2014. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/754.

Council of Science Editors:

Malinkevich A. MIRAGE DNA Transposon Silencing by C. elegans Condensin II Subunit HCP-6: A Masters Thesis. [Masters Thesis]. U of Massachusetts : Med; 2014. Available from: http://escholarship.umassmed.edu/gsbs_diss/754

24. Senol-Cosar, Ozlem. A Role for TNMD in Adipocyte Differentiation and Adipose Tissue Function: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2016, U of Massachusetts : Med

  Adipose tissue is one of the most dynamic tissues in the body and is vital for metabolic homeostasis. In the case of excess nutrient… (more)

Subjects/Keywords: Adipocytes; Adipogenesis; Brown Adipose Tissue; Adiposity; Insulin; Insulin Resistance; Membrane Proteins; Tissue Expansion; Cell Biology; Cellular and Molecular Physiology; Developmental Biology

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APA (6th Edition):

Senol-Cosar, O. (2016). A Role for TNMD in Adipocyte Differentiation and Adipose Tissue Function: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/837

Chicago Manual of Style (16th Edition):

Senol-Cosar, Ozlem. “A Role for TNMD in Adipocyte Differentiation and Adipose Tissue Function: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/837.

MLA Handbook (7th Edition):

Senol-Cosar, Ozlem. “A Role for TNMD in Adipocyte Differentiation and Adipose Tissue Function: A Dissertation.” 2016. Web. 14 Dec 2019.

Vancouver:

Senol-Cosar O. A Role for TNMD in Adipocyte Differentiation and Adipose Tissue Function: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/837.

Council of Science Editors:

Senol-Cosar O. A Role for TNMD in Adipocyte Differentiation and Adipose Tissue Function: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/837

25. Smee, Kevin M. Role of Hdac3 in Murine Coronary Vessel Development: A Master's Thesis.

Degree: Interdisciplinary Graduate Program, Medicine, 2014, U of Massachusetts : Med

  Coronary vessel development is a crucial part of heart development requiring the interplay of the epicardial, myocardial and endocardial layers of the heart for… (more)

Subjects/Keywords: Coronary Vessels; Histone Deacetylases; Developmental Gene Expression Regulation; Cell and Developmental Biology; Developmental Biology; Genetics and Genomics

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APA (6th Edition):

Smee, K. M. (2014). Role of Hdac3 in Murine Coronary Vessel Development: A Master's Thesis. (Masters Thesis). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/766

Chicago Manual of Style (16th Edition):

Smee, Kevin M. “Role of Hdac3 in Murine Coronary Vessel Development: A Master's Thesis.” 2014. Masters Thesis, U of Massachusetts : Med. Accessed December 14, 2019. http://escholarship.umassmed.edu/gsbs_diss/766.

MLA Handbook (7th Edition):

Smee, Kevin M. “Role of Hdac3 in Murine Coronary Vessel Development: A Master's Thesis.” 2014. Web. 14 Dec 2019.

Vancouver:

Smee KM. Role of Hdac3 in Murine Coronary Vessel Development: A Master's Thesis. [Internet] [Masters thesis]. U of Massachusetts : Med; 2014. [cited 2019 Dec 14]. Available from: http://escholarship.umassmed.edu/gsbs_diss/766.

Council of Science Editors:

Smee KM. Role of Hdac3 in Murine Coronary Vessel Development: A Master's Thesis. [Masters Thesis]. U of Massachusetts : Med; 2014. Available from: http://escholarship.umassmed.edu/gsbs_diss/766

26. McCauley, Sean Matthew. Innate Detection of HIV-1 in Myeloid Dendritic Cells.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2018, U of Massachusetts : Med

  Protective antiviral immune responses require priming of naïve T cells by dendritic cells (DCs) that have matured sufficiently to produce co-stimulatory cell surface molecules… (more)

Subjects/Keywords: Innate immunity; dendritic cell; HIV-1; RNA export; pathogen recognition; nucleic acid detection; Immunology and Infectious Disease; Virology

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APA (6th Edition):

McCauley, S. M. (2018). Innate Detection of HIV-1 in Myeloid Dendritic Cells. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/993

Chicago Manual of Style (16th Edition):

McCauley, Sean Matthew. “Innate Detection of HIV-1 in Myeloid Dendritic Cells.” 2018. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. https://escholarship.umassmed.edu/gsbs_diss/993.

MLA Handbook (7th Edition):

McCauley, Sean Matthew. “Innate Detection of HIV-1 in Myeloid Dendritic Cells.” 2018. Web. 14 Dec 2019.

Vancouver:

McCauley SM. Innate Detection of HIV-1 in Myeloid Dendritic Cells. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2018. [cited 2019 Dec 14]. Available from: https://escholarship.umassmed.edu/gsbs_diss/993.

Council of Science Editors:

McCauley SM. Innate Detection of HIV-1 in Myeloid Dendritic Cells. [Doctoral Dissertation]. U of Massachusetts : Med; 2018. Available from: https://escholarship.umassmed.edu/gsbs_diss/993

27. Ray, Samriddha. Elucidation of the Multi-Faceted Roles of the SIN (Septation Initiation Network); Understanding How the SIN Promotes Cytokinesis and Inhibits Interphase Growth in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2010, U of Massachusetts : Med

  Cytokinesis is the cytoplasmic division of one cell into two independent daughter cells. Precise regulation of cytokinesis during cell cycle is essential for healthy… (more)

Subjects/Keywords: Cytokinesis; Schizosaccharomyces pombe Proteins; Signal Transduction; Amino Acids, Peptides, and Proteins; Cell and Developmental Biology; Cells; Fungi; Genetic Phenomena

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APA (6th Edition):

Ray, S. (2010). Elucidation of the Multi-Faceted Roles of the SIN (Septation Initiation Network); Understanding How the SIN Promotes Cytokinesis and Inhibits Interphase Growth in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/482

Chicago Manual of Style (16th Edition):

Ray, Samriddha. “Elucidation of the Multi-Faceted Roles of the SIN (Septation Initiation Network); Understanding How the SIN Promotes Cytokinesis and Inhibits Interphase Growth in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.” 2010. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. https://escholarship.umassmed.edu/gsbs_diss/482.

MLA Handbook (7th Edition):

Ray, Samriddha. “Elucidation of the Multi-Faceted Roles of the SIN (Septation Initiation Network); Understanding How the SIN Promotes Cytokinesis and Inhibits Interphase Growth in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.” 2010. Web. 14 Dec 2019.

Vancouver:

Ray S. Elucidation of the Multi-Faceted Roles of the SIN (Septation Initiation Network); Understanding How the SIN Promotes Cytokinesis and Inhibits Interphase Growth in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2010. [cited 2019 Dec 14]. Available from: https://escholarship.umassmed.edu/gsbs_diss/482.

Council of Science Editors:

Ray S. Elucidation of the Multi-Faceted Roles of the SIN (Septation Initiation Network); Understanding How the SIN Promotes Cytokinesis and Inhibits Interphase Growth in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2010. Available from: https://escholarship.umassmed.edu/gsbs_diss/482

28. Yildirim, Ozlem. Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2012, U of Massachusetts : Med

  Different cell types in multi-cellular organisms heritably maintain different gene expression patterns despite carrying the same genome; a phenomenon termed epigenetics. It is widely… (more)

Subjects/Keywords: Chromatin; Embryonic Stem Cells; Epigenesis; Genetic; Amino Acids, Peptides, and Proteins; Cell and Developmental Biology; Cells; Genetic Phenomena; Genetics and Genomics

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APA (6th Edition):

Yildirim, O. (2012). Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/623

Chicago Manual of Style (16th Edition):

Yildirim, Ozlem. “Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation.” 2012. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. https://escholarship.umassmed.edu/gsbs_diss/623.

MLA Handbook (7th Edition):

Yildirim, Ozlem. “Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation.” 2012. Web. 14 Dec 2019.

Vancouver:

Yildirim O. Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2012. [cited 2019 Dec 14]. Available from: https://escholarship.umassmed.edu/gsbs_diss/623.

Council of Science Editors:

Yildirim O. Chromatin Dynamics in Pluripotency and Differentiation: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2012. Available from: https://escholarship.umassmed.edu/gsbs_diss/623

29. Chen, Poshen B. Function and Regulation of the Tip60-p400 Complex in Embryonic Stem Cells: A Dissertation.

Degree: Interdisciplinary Graduate Program, Molecular, Cell and Cancer Biology Department, 2015, U of Massachusetts : Med

  The following work examines the mechanisms by which Tip60-p400 chromatin remodeling complex regulates gene expression in embryonic stem cells (ESCs). Tip60-p400 complex has distinct… (more)

Subjects/Keywords: Chromatin; Embryonic Stem Cells; Developmental Gene Expression Regulation; Histone Acetyltransferases; Cell Biology; Developmental Biology; Genetics and Genomics

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APA (6th Edition):

Chen, P. B. (2015). Function and Regulation of the Tip60-p400 Complex in Embryonic Stem Cells: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/785

Chicago Manual of Style (16th Edition):

Chen, Poshen B. “Function and Regulation of the Tip60-p400 Complex in Embryonic Stem Cells: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. https://escholarship.umassmed.edu/gsbs_diss/785.

MLA Handbook (7th Edition):

Chen, Poshen B. “Function and Regulation of the Tip60-p400 Complex in Embryonic Stem Cells: A Dissertation.” 2015. Web. 14 Dec 2019.

Vancouver:

Chen PB. Function and Regulation of the Tip60-p400 Complex in Embryonic Stem Cells: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2019 Dec 14]. Available from: https://escholarship.umassmed.edu/gsbs_diss/785.

Council of Science Editors:

Chen PB. Function and Regulation of the Tip60-p400 Complex in Embryonic Stem Cells: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: https://escholarship.umassmed.edu/gsbs_diss/785

30. Bing, Xin Y. Mechanistic Basis for Control of Early Embryonic Development by a 5’ tRNA Fragment.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2019, U of Massachusetts : Med

  Ancestral environmental conditions can instruct offspring development, although the mechanism(s) underlying such transgenerational epigenetic inheritance is unclear. In murine models focused on paternal dietary… (more)

Subjects/Keywords: Histones; small RNA; tRNA fragment; MERVL; ERV; non-coding RNA; U7; chromatin; Cell and Developmental Biology; Genomics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bing, X. Y. (2019). Mechanistic Basis for Control of Early Embryonic Development by a 5’ tRNA Fragment. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/1035

Chicago Manual of Style (16th Edition):

Bing, Xin Y. “Mechanistic Basis for Control of Early Embryonic Development by a 5’ tRNA Fragment.” 2019. Doctoral Dissertation, U of Massachusetts : Med. Accessed December 14, 2019. https://escholarship.umassmed.edu/gsbs_diss/1035.

MLA Handbook (7th Edition):

Bing, Xin Y. “Mechanistic Basis for Control of Early Embryonic Development by a 5’ tRNA Fragment.” 2019. Web. 14 Dec 2019.

Vancouver:

Bing XY. Mechanistic Basis for Control of Early Embryonic Development by a 5’ tRNA Fragment. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2019. [cited 2019 Dec 14]. Available from: https://escholarship.umassmed.edu/gsbs_diss/1035.

Council of Science Editors:

Bing XY. Mechanistic Basis for Control of Early Embryonic Development by a 5’ tRNA Fragment. [Doctoral Dissertation]. U of Massachusetts : Med; 2019. Available from: https://escholarship.umassmed.edu/gsbs_diss/1035

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