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You searched for subject:(Signal Transducer AND Activator of Transcription STAT3 ). Showing records 1 – 30 of 36 total matches.

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Georgia State University

1. Sharma, Shaligram. EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION.

Degree: MS, Biology, 2017, Georgia State University

  Little is known about the ability of d-δ-tocotrienol to protect against obesity-induced inflammation. These studies were conducted to determine whether d-δ-tocotrienol inhibits highfat diet… (more)

Subjects/Keywords: d-δ-tocotrienol; Anti-inflammatory; Signal Transducer and Activator of Transcription (STAT3)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sharma, S. (2017). EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/79

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sharma, Shaligram. “EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION.” 2017. Thesis, Georgia State University. Accessed October 26, 2020. https://scholarworks.gsu.edu/biology_theses/79.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sharma, Shaligram. “EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION.” 2017. Web. 26 Oct 2020.

Vancouver:

Sharma S. EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION. [Internet] [Thesis]. Georgia State University; 2017. [cited 2020 Oct 26]. Available from: https://scholarworks.gsu.edu/biology_theses/79.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sharma S. EFFECTS OF D-DELTA TOCOTRIENOL ON HIGH-FAT DIET-INDUCED PERIPHERAL INFLAMMATION. [Thesis]. Georgia State University; 2017. Available from: https://scholarworks.gsu.edu/biology_theses/79

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

2. Marggraf, Katharina. The signal transducer and activator of transcription STAT3 and coxsackievirus B3-induced chronic myocarditis.

Degree: 2011, Freie Universität Berlin

 The Signal Transducer and Activator of Transcription 3 (STAT3) plays an important role for both the cytoprotection and hypertrophy of cardiomyocytes. STAT3 is activated via… (more)

Subjects/Keywords: STAT3; signal transducer and activator of transcription; chronic myocarditis; Coxsackievirus B3; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Marggraf, K. (2011). The signal transducer and activator of transcription STAT3 and coxsackievirus B3-induced chronic myocarditis. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-11302

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marggraf, Katharina. “The signal transducer and activator of transcription STAT3 and coxsackievirus B3-induced chronic myocarditis.” 2011. Thesis, Freie Universität Berlin. Accessed October 26, 2020. http://dx.doi.org/10.17169/refubium-11302.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marggraf, Katharina. “The signal transducer and activator of transcription STAT3 and coxsackievirus B3-induced chronic myocarditis.” 2011. Web. 26 Oct 2020.

Vancouver:

Marggraf K. The signal transducer and activator of transcription STAT3 and coxsackievirus B3-induced chronic myocarditis. [Internet] [Thesis]. Freie Universität Berlin; 2011. [cited 2020 Oct 26]. Available from: http://dx.doi.org/10.17169/refubium-11302.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marggraf K. The signal transducer and activator of transcription STAT3 and coxsackievirus B3-induced chronic myocarditis. [Thesis]. Freie Universität Berlin; 2011. Available from: http://dx.doi.org/10.17169/refubium-11302

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of South Florida

3. Haviland, Rachel. Identification of Novel STAT3 Target Genes Associated with Oncogenesis.

Degree: 2011, University of South Florida

 Cytokine and growth factor signaling pathways involving STAT3 are frequently constitutively activated in many human primary tumors, and are known for the transcriptional role they… (more)

Subjects/Keywords: cancer; microarray; necdin; signal transducer & activator of transcription; STAT3; American Studies; Arts and Humanities; Cell Biology; Molecular Biology

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APA (6th Edition):

Haviland, R. (2011). Identification of Novel STAT3 Target Genes Associated with Oncogenesis. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/3729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Haviland, Rachel. “Identification of Novel STAT3 Target Genes Associated with Oncogenesis.” 2011. Thesis, University of South Florida. Accessed October 26, 2020. https://scholarcommons.usf.edu/etd/3729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Haviland, Rachel. “Identification of Novel STAT3 Target Genes Associated with Oncogenesis.” 2011. Web. 26 Oct 2020.

Vancouver:

Haviland R. Identification of Novel STAT3 Target Genes Associated with Oncogenesis. [Internet] [Thesis]. University of South Florida; 2011. [cited 2020 Oct 26]. Available from: https://scholarcommons.usf.edu/etd/3729.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Haviland R. Identification of Novel STAT3 Target Genes Associated with Oncogenesis. [Thesis]. University of South Florida; 2011. Available from: https://scholarcommons.usf.edu/etd/3729

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia State University

4. Dixon, Taylor M. Mechanisms mediating the anti-inflammatory effects of dietary methionine restriction.

Degree: MS, Nutrition, 2016, Georgia State University

  Introduction Obesity is associated with inflammation, which contributes to several obesity-related comorbidities. This inflammation is characterized by increased inflammatory cytokine expression in adipose tissue… (more)

Subjects/Keywords: methionine restriction; inflammation; FGF21; fibroblast growth factor-21; STAT3; signal transducer and activator of transcription 3

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APA (6th Edition):

Dixon, T. M. (2016). Mechanisms mediating the anti-inflammatory effects of dietary methionine restriction. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/nutrition_theses/85

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dixon, Taylor M. “Mechanisms mediating the anti-inflammatory effects of dietary methionine restriction.” 2016. Thesis, Georgia State University. Accessed October 26, 2020. https://scholarworks.gsu.edu/nutrition_theses/85.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dixon, Taylor M. “Mechanisms mediating the anti-inflammatory effects of dietary methionine restriction.” 2016. Web. 26 Oct 2020.

Vancouver:

Dixon TM. Mechanisms mediating the anti-inflammatory effects of dietary methionine restriction. [Internet] [Thesis]. Georgia State University; 2016. [cited 2020 Oct 26]. Available from: https://scholarworks.gsu.edu/nutrition_theses/85.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dixon TM. Mechanisms mediating the anti-inflammatory effects of dietary methionine restriction. [Thesis]. Georgia State University; 2016. Available from: https://scholarworks.gsu.edu/nutrition_theses/85

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pretoria

5. [No author]. The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling .

Degree: 2011, University of Pretoria

 Human cytomegalovirus (HCMV) is a ubiquitous virus of the herpes type that infects a high percentage of some populations. One of the most researched genes… (more)

Subjects/Keywords: Us28; Antibody; Tumour; Cytokine; Interleukin 6 il-6; Signal transducer; Activator of transcription stat3; Oncogene; Ligand; G protein-coupled receptors gpcr; Human cytomegalovirus hcmv; UCTD

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APA (6th Edition):

author], [. (2011). The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling . (Masters Thesis). University of Pretoria. Retrieved from http://upetd.up.ac.za/thesis/available/etd-06222011-170730/

Chicago Manual of Style (16th Edition):

author], [No. “The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling .” 2011. Masters Thesis, University of Pretoria. Accessed October 26, 2020. http://upetd.up.ac.za/thesis/available/etd-06222011-170730/.

MLA Handbook (7th Edition):

author], [No. “The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling .” 2011. Web. 26 Oct 2020.

Vancouver:

author] [. The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling . [Internet] [Masters thesis]. University of Pretoria; 2011. [cited 2020 Oct 26]. Available from: http://upetd.up.ac.za/thesis/available/etd-06222011-170730/.

Council of Science Editors:

author] [. The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling . [Masters Thesis]. University of Pretoria; 2011. Available from: http://upetd.up.ac.za/thesis/available/etd-06222011-170730/


University of Pretoria

6. Subramoney, Preya. The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling.

Degree: Pharmacology, 2011, University of Pretoria

 Human cytomegalovirus (HCMV) is a ubiquitous virus of the herpes type that infects a high percentage of some populations. One of the most researched genes… (more)

Subjects/Keywords: Us28; Antibody; Tumour; Cytokine; Interleukin 6 il-6; Signal transducer; Activator of transcription stat3; Oncogene; Ligand; G protein-coupled receptors gpcr; Human cytomegalovirus hcmv; UCTD

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APA (6th Edition):

Subramoney, P. (2011). The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling. (Masters Thesis). University of Pretoria. Retrieved from http://hdl.handle.net/2263/25753

Chicago Manual of Style (16th Edition):

Subramoney, Preya. “The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling.” 2011. Masters Thesis, University of Pretoria. Accessed October 26, 2020. http://hdl.handle.net/2263/25753.

MLA Handbook (7th Edition):

Subramoney, Preya. “The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling.” 2011. Web. 26 Oct 2020.

Vancouver:

Subramoney P. The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling. [Internet] [Masters thesis]. University of Pretoria; 2011. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/2263/25753.

Council of Science Editors:

Subramoney P. The role of human cytomegalovirus encoded viral G protein-coupled receptors in onco-modulatory signalling. [Masters Thesis]. University of Pretoria; 2011. Available from: http://hdl.handle.net/2263/25753


Harvard University

7. Yuan, Dansu. Identification and Functional Analysis of Novel DOCK8 Mutations.

Degree: Master of Medical Sciences, 2019, Harvard University

 DOCK8, the gene encoding the Dedicator of Cytokinesis 8, was first identified in 2004. DOCK8 is a member of the DOCK family of proteins, found… (more)

Subjects/Keywords: Dedicator of cytokinesis 8; nuclear translocation; signal transducer and activator of transcription 3

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APA (6th Edition):

Yuan, D. (2019). Identification and Functional Analysis of Novel DOCK8 Mutations. (Masters Thesis). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42057398

Chicago Manual of Style (16th Edition):

Yuan, Dansu. “Identification and Functional Analysis of Novel DOCK8 Mutations.” 2019. Masters Thesis, Harvard University. Accessed October 26, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42057398.

MLA Handbook (7th Edition):

Yuan, Dansu. “Identification and Functional Analysis of Novel DOCK8 Mutations.” 2019. Web. 26 Oct 2020.

Vancouver:

Yuan D. Identification and Functional Analysis of Novel DOCK8 Mutations. [Internet] [Masters thesis]. Harvard University; 2019. [cited 2020 Oct 26]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42057398.

Council of Science Editors:

Yuan D. Identification and Functional Analysis of Novel DOCK8 Mutations. [Masters Thesis]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42057398

8. Fujimura, Takanori; Fujimoto, Takashi; Itaya-Hironaka, Asako; Miyaoka, Tomoko; Yoshimoto, Kiyomi; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Kondo, Saori; Takeda, Maiko; Tsujinaka, Hiroki; Azuma, Masayuki; Tanaka, Yasuhito. Interleukin-6/STAT pathway is responsible for the induction of gene expression of REG Iα, a new auto-antigen in Sjögren׳s syndrome patients, in salivary duct epithelial cells. : シェーグレン症候群の新規自己抗原であるREG 蛋白の唾液導管細胞における発現誘導にはIL-6/STAT 経路が重要である.

Degree: 博士(医学), 2015, Nara Medical University / 奈良県立医科大学

The regenerating gene, Reg, was originally isolated from a rat regenerating islet cDNA library, and its human homolog was named REG Iα. Recently, we reported… (more)

Subjects/Keywords: Regenerating gene; Interleukin-6; Sjögren׳s syndrome; Janus kinase; Signal transducer and activator of transcription

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APA (6th Edition):

Fujimura, Takanori; Fujimoto, Takashi; Itaya-Hironaka, Asako; Miyaoka, Tomoko; Yoshimoto, Kiyomi; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Kondo, Saori; Takeda, Maiko; Tsujinaka, Hiroki; Azuma, Masayuki; Tanaka, Y. (2015). Interleukin-6/STAT pathway is responsible for the induction of gene expression of REG Iα, a new auto-antigen in Sjögren׳s syndrome patients, in salivary duct epithelial cells. : シェーグレン症候群の新規自己抗原であるREG 蛋白の唾液導管細胞における発現誘導にはIL-6/STAT 経路が重要である. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/3102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fujimura, Takanori; Fujimoto, Takashi; Itaya-Hironaka, Asako; Miyaoka, Tomoko; Yoshimoto, Kiyomi; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Kondo, Saori; Takeda, Maiko; Tsujinaka, Hiroki; Azuma, Masayuki; Tanaka, Yasuhito. “Interleukin-6/STAT pathway is responsible for the induction of gene expression of REG Iα, a new auto-antigen in Sjögren׳s syndrome patients, in salivary duct epithelial cells. : シェーグレン症候群の新規自己抗原であるREG 蛋白の唾液導管細胞における発現誘導にはIL-6/STAT 経路が重要である.” 2015. Thesis, Nara Medical University / 奈良県立医科大学. Accessed October 26, 2020. http://hdl.handle.net/10564/3102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fujimura, Takanori; Fujimoto, Takashi; Itaya-Hironaka, Asako; Miyaoka, Tomoko; Yoshimoto, Kiyomi; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Kondo, Saori; Takeda, Maiko; Tsujinaka, Hiroki; Azuma, Masayuki; Tanaka, Yasuhito. “Interleukin-6/STAT pathway is responsible for the induction of gene expression of REG Iα, a new auto-antigen in Sjögren׳s syndrome patients, in salivary duct epithelial cells. : シェーグレン症候群の新規自己抗原であるREG 蛋白の唾液導管細胞における発現誘導にはIL-6/STAT 経路が重要である.” 2015. Web. 26 Oct 2020.

Vancouver:

Fujimura, Takanori; Fujimoto, Takashi; Itaya-Hironaka, Asako; Miyaoka, Tomoko; Yoshimoto, Kiyomi; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Kondo, Saori; Takeda, Maiko; Tsujinaka, Hiroki; Azuma, Masayuki; Tanaka Y. Interleukin-6/STAT pathway is responsible for the induction of gene expression of REG Iα, a new auto-antigen in Sjögren׳s syndrome patients, in salivary duct epithelial cells. : シェーグレン症候群の新規自己抗原であるREG 蛋白の唾液導管細胞における発現誘導にはIL-6/STAT 経路が重要である. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2015. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10564/3102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fujimura, Takanori; Fujimoto, Takashi; Itaya-Hironaka, Asako; Miyaoka, Tomoko; Yoshimoto, Kiyomi; Yamauchi, Akiyo; Sakuramoto-Tsuchida, Sumiyo; Kondo, Saori; Takeda, Maiko; Tsujinaka, Hiroki; Azuma, Masayuki; Tanaka Y. Interleukin-6/STAT pathway is responsible for the induction of gene expression of REG Iα, a new auto-antigen in Sjögren׳s syndrome patients, in salivary duct epithelial cells. : シェーグレン症候群の新規自己抗原であるREG 蛋白の唾液導管細胞における発現誘導にはIL-6/STAT 経路が重要である. [Thesis]. Nara Medical University / 奈良県立医科大学; 2015. Available from: http://hdl.handle.net/10564/3102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

9. Zeng, Chang. Regulation of Esophageal Epithelial Function in Eosinophilic Esophagitis.

Degree: PhD, Medicine: Molecular, Cellular and Biochemical Pharmacology, 2018, University of Cincinnati

 Eosinophilic Esophagitis (EoE) is an allergic inflammatory disorder with increasing prevalence in the western world. Patients with EoE demonstrate symptoms including vomiting, dysphagia and food… (more)

Subjects/Keywords: Pharmacology; Eosinophilic Esophagitis; SLC9A3; IL-13; Dilated Intercellular Spaces; signal transducer and activator of transcription

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APA (6th Edition):

Zeng, C. (2018). Regulation of Esophageal Epithelial Function in Eosinophilic Esophagitis. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1530797729542185

Chicago Manual of Style (16th Edition):

Zeng, Chang. “Regulation of Esophageal Epithelial Function in Eosinophilic Esophagitis.” 2018. Doctoral Dissertation, University of Cincinnati. Accessed October 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1530797729542185.

MLA Handbook (7th Edition):

Zeng, Chang. “Regulation of Esophageal Epithelial Function in Eosinophilic Esophagitis.” 2018. Web. 26 Oct 2020.

Vancouver:

Zeng C. Regulation of Esophageal Epithelial Function in Eosinophilic Esophagitis. [Internet] [Doctoral dissertation]. University of Cincinnati; 2018. [cited 2020 Oct 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1530797729542185.

Council of Science Editors:

Zeng C. Regulation of Esophageal Epithelial Function in Eosinophilic Esophagitis. [Doctoral Dissertation]. University of Cincinnati; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1530797729542185

10. Lemadre, Elodie. Nouveaux rôles anti-tumoraux de STAT1 : expression des immunoglobulines et réparation de l'ADN : New anti tumoralroles of STAT1 : immunoglobilin expression and DNA repair.

Degree: Docteur es, Biologie moléculaire et cellulaire, 2014, Paris 13

Le facteur de transcription STAT1 est un effecteur majeur de la réponse à l’interféron exerçant ainsi un rôle clé dans l’immunité innée. Il est également… (more)

Subjects/Keywords: STAT1 (Transducteur de signaux et activateur de transcription 1); STAT1 (Signal Transducer and Activator of Transcription 1)

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APA (6th Edition):

Lemadre, E. (2014). Nouveaux rôles anti-tumoraux de STAT1 : expression des immunoglobulines et réparation de l'ADN : New anti tumoralroles of STAT1 : immunoglobilin expression and DNA repair. (Doctoral Dissertation). Paris 13. Retrieved from http://www.theses.fr/2014PA132020

Chicago Manual of Style (16th Edition):

Lemadre, Elodie. “Nouveaux rôles anti-tumoraux de STAT1 : expression des immunoglobulines et réparation de l'ADN : New anti tumoralroles of STAT1 : immunoglobilin expression and DNA repair.” 2014. Doctoral Dissertation, Paris 13. Accessed October 26, 2020. http://www.theses.fr/2014PA132020.

MLA Handbook (7th Edition):

Lemadre, Elodie. “Nouveaux rôles anti-tumoraux de STAT1 : expression des immunoglobulines et réparation de l'ADN : New anti tumoralroles of STAT1 : immunoglobilin expression and DNA repair.” 2014. Web. 26 Oct 2020.

Vancouver:

Lemadre E. Nouveaux rôles anti-tumoraux de STAT1 : expression des immunoglobulines et réparation de l'ADN : New anti tumoralroles of STAT1 : immunoglobilin expression and DNA repair. [Internet] [Doctoral dissertation]. Paris 13; 2014. [cited 2020 Oct 26]. Available from: http://www.theses.fr/2014PA132020.

Council of Science Editors:

Lemadre E. Nouveaux rôles anti-tumoraux de STAT1 : expression des immunoglobulines et réparation de l'ADN : New anti tumoralroles of STAT1 : immunoglobilin expression and DNA repair. [Doctoral Dissertation]. Paris 13; 2014. Available from: http://www.theses.fr/2014PA132020

11. Johnson, Andrew William. Protection against Angiotensin II-induced endothelial dysfunction and hypertension via small molecule inhibitors of signal transducer and activator of transcription 3.

Degree: PhD, Pharmacology, 2012, University of Iowa

  Angiotensin II (Ang II) promotes vascular disease and hypertension in part by the formation of pro-inflammatory cytokines, oxidative stress and inflammation. Signal transducer and… (more)

Subjects/Keywords: Angiotensin II; Endothelial dysfunction; S3I-201; Signal Transducer and Activator of Transcription 3; STAT3; Pharmacology

…oxidative stress and inflammation. Signal transducer and activator of transcription 3 (STAT3… …oncogene STAT signal transducer and activator of transcription STAT3 signal transducer and… …31 Signal Transducer and Activator of Transcription.................................... 34… …STAT Janus kinase/signal transducer and activator of transcription JH1 Janus homology 1… …activator of transcription 3 STATTIC signal transducer activator of transcription three… 

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APA (6th Edition):

Johnson, A. W. (2012). Protection against Angiotensin II-induced endothelial dysfunction and hypertension via small molecule inhibitors of signal transducer and activator of transcription 3. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/2903

Chicago Manual of Style (16th Edition):

Johnson, Andrew William. “Protection against Angiotensin II-induced endothelial dysfunction and hypertension via small molecule inhibitors of signal transducer and activator of transcription 3.” 2012. Doctoral Dissertation, University of Iowa. Accessed October 26, 2020. https://ir.uiowa.edu/etd/2903.

MLA Handbook (7th Edition):

Johnson, Andrew William. “Protection against Angiotensin II-induced endothelial dysfunction and hypertension via small molecule inhibitors of signal transducer and activator of transcription 3.” 2012. Web. 26 Oct 2020.

Vancouver:

Johnson AW. Protection against Angiotensin II-induced endothelial dysfunction and hypertension via small molecule inhibitors of signal transducer and activator of transcription 3. [Internet] [Doctoral dissertation]. University of Iowa; 2012. [cited 2020 Oct 26]. Available from: https://ir.uiowa.edu/etd/2903.

Council of Science Editors:

Johnson AW. Protection against Angiotensin II-induced endothelial dysfunction and hypertension via small molecule inhibitors of signal transducer and activator of transcription 3. [Doctoral Dissertation]. University of Iowa; 2012. Available from: https://ir.uiowa.edu/etd/2903

12. 田崎, 新資. Docosahexaenoic acid inhibits the phosphorylation of STAT3 and the growth and invasion of renal cancer cells. : ドコサヘキサエン酸はSTAT3 のリン酸化を抑制し、腎癌細胞の増殖と浸潤を抑制する.

Degree: 博士(医学), 2018, Saitama Medical University / 埼玉医科大学

Docosahexaenoic acid (DHA) has a variety of anti-tumor activities. The present study examined the anti-tumor activity of DHA in renal cancer cells and its underlying… (more)

Subjects/Keywords: anti-tumor effect; docosahexaenoic acid; epidermal growth factor receptor; kidney cancer; signal transducer and activator of transcription 3

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APA (6th Edition):

田崎, . (2018). Docosahexaenoic acid inhibits the phosphorylation of STAT3 and the growth and invasion of renal cancer cells. : ドコサヘキサエン酸はSTAT3 のリン酸化を抑制し、腎癌細胞の増殖と浸潤を抑制する. (Thesis). Saitama Medical University / 埼玉医科大学. Retrieved from http://id.nii.ac.jp/1386/00000606/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

田崎, 新資. “Docosahexaenoic acid inhibits the phosphorylation of STAT3 and the growth and invasion of renal cancer cells. : ドコサヘキサエン酸はSTAT3 のリン酸化を抑制し、腎癌細胞の増殖と浸潤を抑制する.” 2018. Thesis, Saitama Medical University / 埼玉医科大学. Accessed October 26, 2020. http://id.nii.ac.jp/1386/00000606/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

田崎, 新資. “Docosahexaenoic acid inhibits the phosphorylation of STAT3 and the growth and invasion of renal cancer cells. : ドコサヘキサエン酸はSTAT3 のリン酸化を抑制し、腎癌細胞の増殖と浸潤を抑制する.” 2018. Web. 26 Oct 2020.

Vancouver:

田崎 . Docosahexaenoic acid inhibits the phosphorylation of STAT3 and the growth and invasion of renal cancer cells. : ドコサヘキサエン酸はSTAT3 のリン酸化を抑制し、腎癌細胞の増殖と浸潤を抑制する. [Internet] [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. [cited 2020 Oct 26]. Available from: http://id.nii.ac.jp/1386/00000606/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

田崎 . Docosahexaenoic acid inhibits the phosphorylation of STAT3 and the growth and invasion of renal cancer cells. : ドコサヘキサエン酸はSTAT3 のリン酸化を抑制し、腎癌細胞の増殖と浸潤を抑制する. [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. Available from: http://id.nii.ac.jp/1386/00000606/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

13. Willson, Tara A. STAT3 Regulation of Mucosal Inflammation in Pediatric Crohn’s Disease and Murine Colitis.

Degree: PhD, Medicine: Cell and Molecular Biology, 2012, University of Cincinnati

 <b>Significance</b>: Signal Transducer and Activator of Transcription 3 (STAT3) is crucial for both innate and adaptive mucosal immune responses in human inflammatory bowel disease (IBD)… (more)

Subjects/Keywords: Cellular Biology; Pediatric Crohn's Disease; Signal Transducer and Activator of Transcription 3; neutrophil recruitment; CXCR2; Dextran Sodium Sulfate (DSS); Interleukin 17A

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APA (6th Edition):

Willson, T. A. (2012). STAT3 Regulation of Mucosal Inflammation in Pediatric Crohn’s Disease and Murine Colitis. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1330024735

Chicago Manual of Style (16th Edition):

Willson, Tara A. “STAT3 Regulation of Mucosal Inflammation in Pediatric Crohn’s Disease and Murine Colitis.” 2012. Doctoral Dissertation, University of Cincinnati. Accessed October 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1330024735.

MLA Handbook (7th Edition):

Willson, Tara A. “STAT3 Regulation of Mucosal Inflammation in Pediatric Crohn’s Disease and Murine Colitis.” 2012. Web. 26 Oct 2020.

Vancouver:

Willson TA. STAT3 Regulation of Mucosal Inflammation in Pediatric Crohn’s Disease and Murine Colitis. [Internet] [Doctoral dissertation]. University of Cincinnati; 2012. [cited 2020 Oct 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1330024735.

Council of Science Editors:

Willson TA. STAT3 Regulation of Mucosal Inflammation in Pediatric Crohn’s Disease and Murine Colitis. [Doctoral Dissertation]. University of Cincinnati; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1330024735


NSYSU

14. Cheng, Shou-Hui. The Role of Activated STAT3 (p-tyr705 and p-ser727-STAT3) in Taiwanese Cervical Cancer.

Degree: Master, Biological Sciences, 2013, NSYSU

 Cervical cancer is one of the most important female deaths caused by cancer in the world. Although the Pap smear has become an effective screening… (more)

Subjects/Keywords: cancer; Cervical cancer; phosphorylated serine residue 727 (p-ser727-STAT3); phosphorylated tyrosine residue 705 (p-tyr705-STAT3); signal transducer and activator

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APA (6th Edition):

Cheng, S. (2013). The Role of Activated STAT3 (p-tyr705 and p-ser727-STAT3) in Taiwanese Cervical Cancer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0813113-071807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cheng, Shou-Hui. “The Role of Activated STAT3 (p-tyr705 and p-ser727-STAT3) in Taiwanese Cervical Cancer.” 2013. Thesis, NSYSU. Accessed October 26, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0813113-071807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cheng, Shou-Hui. “The Role of Activated STAT3 (p-tyr705 and p-ser727-STAT3) in Taiwanese Cervical Cancer.” 2013. Web. 26 Oct 2020.

Vancouver:

Cheng S. The Role of Activated STAT3 (p-tyr705 and p-ser727-STAT3) in Taiwanese Cervical Cancer. [Internet] [Thesis]. NSYSU; 2013. [cited 2020 Oct 26]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0813113-071807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cheng S. The Role of Activated STAT3 (p-tyr705 and p-ser727-STAT3) in Taiwanese Cervical Cancer. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0813113-071807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Paris-Sud – Paris XI

15. Noman, Muhammad zaeem. Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques.

Degree: Docteur es, Cancérologie, 2012, Université Paris-Sud – Paris XI

 L’hypoxie est une caractéristique commune des tumeurs solides et l’une des spécificités du micro environnement tumoral. L’hypoxie tumorale joue un rôle important dans l’angio génèse,… (more)

Subjects/Keywords: Hypoxie; Lymphocytes T cytotoxiques; Autophagie; MicroRNA-210; Hypoxia Inducible Factor 1 alpha; Susceptibilité des cellules tumorale; Signal transducer and activator of transcription 3; Hypoxia; Cytotoxic T lymphocytes; Autophagy; MicroRNA-210; Hypoxia Inducible Factor 1 alpha; Tumor cell susceptibility; Signal transducer and activator of transcription 3

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APA (6th Edition):

Noman, M. z. (2012). Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2012PA11T051

Chicago Manual of Style (16th Edition):

Noman, Muhammad zaeem. “Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques.” 2012. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed October 26, 2020. http://www.theses.fr/2012PA11T051.

MLA Handbook (7th Edition):

Noman, Muhammad zaeem. “Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques.” 2012. Web. 26 Oct 2020.

Vancouver:

Noman Mz. Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2012. [cited 2020 Oct 26]. Available from: http://www.theses.fr/2012PA11T051.

Council of Science Editors:

Noman Mz. Influence of hypoxia on tumour cell susceptibility to cytotoxic T lymphocyte mediated lysis : Influence de l’hypoxie sur la susceptibilité des cellules tumorales à la lyse induite par les lymphocytes T cytotoxiques. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2012. Available from: http://www.theses.fr/2012PA11T051


University of Texas – Austin

16. Gopalan, Archana. Targeting breast cancer with natural forms of vitamin E and simvastatin.

Degree: PhD, Nutritional Sciences, 2012, University of Texas – Austin

 Breast cancer is the second leading cause of death due to cancer in women. A number of effective therapeutic strategies have been implemented in clinics… (more)

Subjects/Keywords: Breast cancer; Vitamin E; Simvastatin; Apoptosis; Stem cells; TIC; Mevalonate pathway; Ceramide synthesis pathways; Death receptor 5 (DR5); c-Jun N-terminal kinase/C/EBP homologous protein (JNK/CHOP); FLICE inhibitory protein (c-FLIP); B-cell lymphoma 2 (Bcl-2); Survivin; Signal transducer and activator of transcription 3 (Stat3); Bcl-xL; Cyclin D1; c-Myc

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APA (6th Edition):

Gopalan, A. (2012). Targeting breast cancer with natural forms of vitamin E and simvastatin. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/ETD-UT-2012-05-5520

Chicago Manual of Style (16th Edition):

Gopalan, Archana. “Targeting breast cancer with natural forms of vitamin E and simvastatin.” 2012. Doctoral Dissertation, University of Texas – Austin. Accessed October 26, 2020. http://hdl.handle.net/2152/ETD-UT-2012-05-5520.

MLA Handbook (7th Edition):

Gopalan, Archana. “Targeting breast cancer with natural forms of vitamin E and simvastatin.” 2012. Web. 26 Oct 2020.

Vancouver:

Gopalan A. Targeting breast cancer with natural forms of vitamin E and simvastatin. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2012. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/2152/ETD-UT-2012-05-5520.

Council of Science Editors:

Gopalan A. Targeting breast cancer with natural forms of vitamin E and simvastatin. [Doctoral Dissertation]. University of Texas – Austin; 2012. Available from: http://hdl.handle.net/2152/ETD-UT-2012-05-5520


Kyoto University / 京都大学

17. Kasai, Yosuke. Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 : ナルディライジンはSTAT3の活性化を介して肝細胞がんの進展に寄与する.

Degree: 博士(医学), 2017, Kyoto University / 京都大学

新制・課程博士

甲第20607号

医博第4256号

Subjects/Keywords: hepatocellular carcinoma; nardilysin; prognostic marker; signal transducer and activator of transcription 3; spheroid

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APA (6th Edition):

Kasai, Y. (2017). Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 : ナルディライジンはSTAT3の活性化を介して肝細胞がんの進展に寄与する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/226761 ; http://dx.doi.org/10.14989/doctor.k20607

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kasai, Yosuke. “Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 : ナルディライジンはSTAT3の活性化を介して肝細胞がんの進展に寄与する.” 2017. Thesis, Kyoto University / 京都大学. Accessed October 26, 2020. http://hdl.handle.net/2433/226761 ; http://dx.doi.org/10.14989/doctor.k20607.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kasai, Yosuke. “Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 : ナルディライジンはSTAT3の活性化を介して肝細胞がんの進展に寄与する.” 2017. Web. 26 Oct 2020.

Vancouver:

Kasai Y. Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 : ナルディライジンはSTAT3の活性化を介して肝細胞がんの進展に寄与する. [Internet] [Thesis]. Kyoto University / 京都大学; 2017. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/2433/226761 ; http://dx.doi.org/10.14989/doctor.k20607.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kasai Y. Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 : ナルディライジンはSTAT3の活性化を介して肝細胞がんの進展に寄与する. [Thesis]. Kyoto University / 京都大学; 2017. Available from: http://hdl.handle.net/2433/226761 ; http://dx.doi.org/10.14989/doctor.k20607

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Kasai, Yosuke. Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 .

Degree: 2017, Kyoto University

Subjects/Keywords: hepatocellular carcinoma; nardilysin; prognostic marker; signal transducer and activator of transcription 3; spheroid

Page 1 Page 2

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APA (6th Edition):

Kasai, Y. (2017). Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/226761

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kasai, Yosuke. “Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 .” 2017. Thesis, Kyoto University. Accessed October 26, 2020. http://hdl.handle.net/2433/226761.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kasai, Yosuke. “Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 .” 2017. Web. 26 Oct 2020.

Vancouver:

Kasai Y. Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 . [Internet] [Thesis]. Kyoto University; 2017. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/2433/226761.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kasai Y. Nardilysin promotes hepatocellular carcinoma through activation of signal transducer and activator of transcription 3 . [Thesis]. Kyoto University; 2017. Available from: http://hdl.handle.net/2433/226761

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

19. Kaucher, Amy V. MECHANISMS OF SPERMATOGONIAL STEM CELL DIFFERENTIATION .

Degree: 2010, Penn State University

 In mammalian testes, the actions of spermatogonial stem cells (SSC) provide a foundation for spermatogenesis during adult life. Like other tissue-specific stem cell populations, SSC… (more)

Subjects/Keywords: SPERMATOGONIAL STEM CELL; SIGNAL TRANSDUCER AND ACTIVATOR OF TRANSCRIPTION 3; NEUROGENIN 3; DIFFERENTIATION

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APA (6th Edition):

Kaucher, A. V. (2010). MECHANISMS OF SPERMATOGONIAL STEM CELL DIFFERENTIATION . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kaucher, Amy V. “MECHANISMS OF SPERMATOGONIAL STEM CELL DIFFERENTIATION .” 2010. Thesis, Penn State University. Accessed October 26, 2020. https://submit-etda.libraries.psu.edu/catalog/11102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kaucher, Amy V. “MECHANISMS OF SPERMATOGONIAL STEM CELL DIFFERENTIATION .” 2010. Web. 26 Oct 2020.

Vancouver:

Kaucher AV. MECHANISMS OF SPERMATOGONIAL STEM CELL DIFFERENTIATION . [Internet] [Thesis]. Penn State University; 2010. [cited 2020 Oct 26]. Available from: https://submit-etda.libraries.psu.edu/catalog/11102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kaucher AV. MECHANISMS OF SPERMATOGONIAL STEM CELL DIFFERENTIATION . [Thesis]. Penn State University; 2010. Available from: https://submit-etda.libraries.psu.edu/catalog/11102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

20. Müller, Ina Maria. Die Rolle des Transkriptionsfaktor STAT3 in intestinalen Epithelzellen während der Infektion mit dem attaching- und effacing- Läsionen auslösenden Bakterium Citrobacter rodentium.

Degree: 2012, Freie Universität Berlin

 Bakteriell ausgelöste Gastroenteritiden sind weltweit mit einer hohen Morbidität und Mortalität assoziiert. Zwei wichtige bakterielle Erreger sind der enteropathogene und enterohämorrhagische Escherichia coli, welche zu… (more)

Subjects/Keywords: signal transducer and activator of transcription (STAT) 3; Citrobacter rodentium; mucosal immunology; infectious colitis; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Müller, I. M. (2012). Die Rolle des Transkriptionsfaktor STAT3 in intestinalen Epithelzellen während der Infektion mit dem attaching- und effacing- Läsionen auslösenden Bakterium Citrobacter rodentium. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/10719

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Müller, Ina Maria. “Die Rolle des Transkriptionsfaktor STAT3 in intestinalen Epithelzellen während der Infektion mit dem attaching- und effacing- Läsionen auslösenden Bakterium Citrobacter rodentium.” 2012. Thesis, Freie Universität Berlin. Accessed October 26, 2020. https://refubium.fu-berlin.de/handle/fub188/10719.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Müller, Ina Maria. “Die Rolle des Transkriptionsfaktor STAT3 in intestinalen Epithelzellen während der Infektion mit dem attaching- und effacing- Läsionen auslösenden Bakterium Citrobacter rodentium.” 2012. Web. 26 Oct 2020.

Vancouver:

Müller IM. Die Rolle des Transkriptionsfaktor STAT3 in intestinalen Epithelzellen während der Infektion mit dem attaching- und effacing- Läsionen auslösenden Bakterium Citrobacter rodentium. [Internet] [Thesis]. Freie Universität Berlin; 2012. [cited 2020 Oct 26]. Available from: https://refubium.fu-berlin.de/handle/fub188/10719.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Müller IM. Die Rolle des Transkriptionsfaktor STAT3 in intestinalen Epithelzellen während der Infektion mit dem attaching- und effacing- Läsionen auslösenden Bakterium Citrobacter rodentium. [Thesis]. Freie Universität Berlin; 2012. Available from: https://refubium.fu-berlin.de/handle/fub188/10719

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

21. Ossa, Juan C. Identifying Mechanisms Used by Adherent-invasive Escherichia coli Associated with Crohn Disease to Evade the Immune System.

Degree: 2012, University of Toronto

Background: Adherent-invasive Escherichia coli (AIEC) is a pathogen isolated from the ileum of patients with CD. IFNγ is a key mediator of immunity, which regulates… (more)

Subjects/Keywords: Crohn Disease (CD); Adherent-Invasive Escherichia coli (AIEC); Interferon gamma (IFNγ); Signal transducer and activator of transcription 1 (STAT1); 0410; 0307; 0982

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APA (6th Edition):

Ossa, J. C. (2012). Identifying Mechanisms Used by Adherent-invasive Escherichia coli Associated with Crohn Disease to Evade the Immune System. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/32615

Chicago Manual of Style (16th Edition):

Ossa, Juan C. “Identifying Mechanisms Used by Adherent-invasive Escherichia coli Associated with Crohn Disease to Evade the Immune System.” 2012. Masters Thesis, University of Toronto. Accessed October 26, 2020. http://hdl.handle.net/1807/32615.

MLA Handbook (7th Edition):

Ossa, Juan C. “Identifying Mechanisms Used by Adherent-invasive Escherichia coli Associated with Crohn Disease to Evade the Immune System.” 2012. Web. 26 Oct 2020.

Vancouver:

Ossa JC. Identifying Mechanisms Used by Adherent-invasive Escherichia coli Associated with Crohn Disease to Evade the Immune System. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1807/32615.

Council of Science Editors:

Ossa JC. Identifying Mechanisms Used by Adherent-invasive Escherichia coli Associated with Crohn Disease to Evade the Immune System. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/32615


Universitat de Valencia

22. Bosch Campos, Ana. Role of the NF-kB pathway and nitric oxide in mammary gland involution after weaning. Implications in breast cancer .

Degree: 2013, Universitat de Valencia

 INTRODUCCIÓN La glándula mamaria es un órgano dinámico que alcanza su máximo desarrollo funcional con la lactancia, momento en el cual, a través de la… (more)

Subjects/Keywords: breast cancer; inflammation; apoptosis; cathepsin D; mammary gland involution; nitric oxide; NOS2-KO; nitrotyrosine; nuclear factor κappa B (NF-κB); weaning; signal transducer and activator of transcription (STAT)

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APA (6th Edition):

Bosch Campos, A. (2013). Role of the NF-kB pathway and nitric oxide in mammary gland involution after weaning. Implications in breast cancer . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/25626

Chicago Manual of Style (16th Edition):

Bosch Campos, Ana. “Role of the NF-kB pathway and nitric oxide in mammary gland involution after weaning. Implications in breast cancer .” 2013. Doctoral Dissertation, Universitat de Valencia. Accessed October 26, 2020. http://hdl.handle.net/10550/25626.

MLA Handbook (7th Edition):

Bosch Campos, Ana. “Role of the NF-kB pathway and nitric oxide in mammary gland involution after weaning. Implications in breast cancer .” 2013. Web. 26 Oct 2020.

Vancouver:

Bosch Campos A. Role of the NF-kB pathway and nitric oxide in mammary gland involution after weaning. Implications in breast cancer . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2013. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10550/25626.

Council of Science Editors:

Bosch Campos A. Role of the NF-kB pathway and nitric oxide in mammary gland involution after weaning. Implications in breast cancer . [Doctoral Dissertation]. Universitat de Valencia; 2013. Available from: http://hdl.handle.net/10550/25626

23. Γεωργαντά, Ειρήνη-Μαρία. Ανάλυση νέων σηματοδοτικών μονοπατιών των οπιοειδών υποδοχέων που οδηγούν σε αλλαγές της συναπτοσωμικής πλαστικότητας.

Degree: 2012, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Opioid receptors are prototypical Gi/o-coupled receptors and participate in mechanisms controlling neural growth, differentiation and synaptic plasticity. Previous work from our laboratory has shown that… (more)

Subjects/Keywords: Οπιοειδείς υποδοχείς; Μεταγραφικός παράγοντας STAT5; Πρωτεΐνες G; Νευριτική ανάπτυξη; Πρωτεϊνικό σύμπλοκο σηματοδότησης; Opioid receptors; Signal transducer and activator of transcription 5; G proteins; Neurite outgrowth; Signaling complex

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Γεωργαντά, . . (2012). Ανάλυση νέων σηματοδοτικών μονοπατιών των οπιοειδών υποδοχέων που οδηγούν σε αλλαγές της συναπτοσωμικής πλαστικότητας. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/35821

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Γεωργαντά, Ειρήνη-Μαρία. “Ανάλυση νέων σηματοδοτικών μονοπατιών των οπιοειδών υποδοχέων που οδηγούν σε αλλαγές της συναπτοσωμικής πλαστικότητας.” 2012. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed October 26, 2020. http://hdl.handle.net/10442/hedi/35821.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Γεωργαντά, Ειρήνη-Μαρία. “Ανάλυση νέων σηματοδοτικών μονοπατιών των οπιοειδών υποδοχέων που οδηγούν σε αλλαγές της συναπτοσωμικής πλαστικότητας.” 2012. Web. 26 Oct 2020.

Vancouver:

Γεωργαντά . Ανάλυση νέων σηματοδοτικών μονοπατιών των οπιοειδών υποδοχέων που οδηγούν σε αλλαγές της συναπτοσωμικής πλαστικότητας. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10442/hedi/35821.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Γεωργαντά . Ανάλυση νέων σηματοδοτικών μονοπατιών των οπιοειδών υποδοχέων που οδηγούν σε αλλαγές της συναπτοσωμικής πλαστικότητας. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. Available from: http://hdl.handle.net/10442/hedi/35821

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loma Linda University

24. Hsu, Heng-Wei. Radiosensitization of Head & Neck Carcinoma Cells by Linifanib, A Receptor Tyrosine Kinase Inhibitor.

Degree: PhD, Basic Sciences, 2013, Loma Linda University

  Tumor angiogenesis is a hallmark of advanced cancers and promotes invasion and metastasis. Over 90% of head and neck squamous cell carcinomas (HNSCC) express… (more)

Subjects/Keywords: Chemical and Pharmacologic Phenomena; Chemicals and Drugs; Enzymes and Coenzymes; Medical Pharmacology; Medicine and Health Sciences; Neoplasms; Oncology; Head and Neck Neoplasms - Pathology; Carcinoma - Squamous Cell; Cell Line - Tumor; Cell Proliferation; Cell Survival - Radiation Effects; Apoptosis Inducing Factor; Focal Adhesion Protein-Tyrosine Kinases - Metabolism; Protein Kinase Inhibitors; Indazoles - Therapeutic Use; Phenylurea Componds - Therapeutic Use; Radiation-Sensitizing Agents - Therapeutic Use; Chemoradiotherapy; Radiation Tolerance; Tumor Angiogenesis; Vascular Endothelial Growth Factor; Antiangiogenic Therapy; Tumor Microenvironment; Signal Transducer and Activator of Transcription 3 (STAT3)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hsu, H. (2013). Radiosensitization of Head & Neck Carcinoma Cells by Linifanib, A Receptor Tyrosine Kinase Inhibitor. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/303

Chicago Manual of Style (16th Edition):

Hsu, Heng-Wei. “Radiosensitization of Head & Neck Carcinoma Cells by Linifanib, A Receptor Tyrosine Kinase Inhibitor.” 2013. Doctoral Dissertation, Loma Linda University. Accessed October 26, 2020. https://scholarsrepository.llu.edu/etd/303.

MLA Handbook (7th Edition):

Hsu, Heng-Wei. “Radiosensitization of Head & Neck Carcinoma Cells by Linifanib, A Receptor Tyrosine Kinase Inhibitor.” 2013. Web. 26 Oct 2020.

Vancouver:

Hsu H. Radiosensitization of Head & Neck Carcinoma Cells by Linifanib, A Receptor Tyrosine Kinase Inhibitor. [Internet] [Doctoral dissertation]. Loma Linda University; 2013. [cited 2020 Oct 26]. Available from: https://scholarsrepository.llu.edu/etd/303.

Council of Science Editors:

Hsu H. Radiosensitization of Head & Neck Carcinoma Cells by Linifanib, A Receptor Tyrosine Kinase Inhibitor. [Doctoral Dissertation]. Loma Linda University; 2013. Available from: https://scholarsrepository.llu.edu/etd/303


Virginia Tech

25. Wang, Ying. Growth Hormone and Nutritional Regulation of Insulin-Like Growth Factor-I Gene Expression.

Degree: PhD, Animal and Poultry Sciences, 2005, Virginia Tech

 The objectives of this research were to characterize insulin-like growth factor-I (IGF-I) gene expression in cattle, to determine how IGF-I gene expression is affected by… (more)

Subjects/Keywords: Signal transducer and activator of transcription 5; Nutrition; Transcriptional regulation; Insulin-like growth factor-I; Growth hormone

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, Y. (2005). Growth Hormone and Nutritional Regulation of Insulin-Like Growth Factor-I Gene Expression. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/30208

Chicago Manual of Style (16th Edition):

Wang, Ying. “Growth Hormone and Nutritional Regulation of Insulin-Like Growth Factor-I Gene Expression.” 2005. Doctoral Dissertation, Virginia Tech. Accessed October 26, 2020. http://hdl.handle.net/10919/30208.

MLA Handbook (7th Edition):

Wang, Ying. “Growth Hormone and Nutritional Regulation of Insulin-Like Growth Factor-I Gene Expression.” 2005. Web. 26 Oct 2020.

Vancouver:

Wang Y. Growth Hormone and Nutritional Regulation of Insulin-Like Growth Factor-I Gene Expression. [Internet] [Doctoral dissertation]. Virginia Tech; 2005. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10919/30208.

Council of Science Editors:

Wang Y. Growth Hormone and Nutritional Regulation of Insulin-Like Growth Factor-I Gene Expression. [Doctoral Dissertation]. Virginia Tech; 2005. Available from: http://hdl.handle.net/10919/30208


Virginia Tech

26. Eleswarapu, Satyanarayana. Mechanisms of Growth Hormone Regulation of Insulin-Like Growth Factor-I Gene Expression in Liver.

Degree: PhD, Animal and Poultry Sciences, 2009, Virginia Tech

 The overall objective of this research was to understand the mechanims by which growth hormone (GH) regulates insulin-like growth factor-I (IGF-I) gene expression in liver.… (more)

Subjects/Keywords: Growth hormone; Signal transducer and activator of transcription 5; Liver; Insulin like growth factor-I; Hepatocyte nuclear factor-3γ

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Eleswarapu, S. (2009). Mechanisms of Growth Hormone Regulation of Insulin-Like Growth Factor-I Gene Expression in Liver. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/37373

Chicago Manual of Style (16th Edition):

Eleswarapu, Satyanarayana. “Mechanisms of Growth Hormone Regulation of Insulin-Like Growth Factor-I Gene Expression in Liver.” 2009. Doctoral Dissertation, Virginia Tech. Accessed October 26, 2020. http://hdl.handle.net/10919/37373.

MLA Handbook (7th Edition):

Eleswarapu, Satyanarayana. “Mechanisms of Growth Hormone Regulation of Insulin-Like Growth Factor-I Gene Expression in Liver.” 2009. Web. 26 Oct 2020.

Vancouver:

Eleswarapu S. Mechanisms of Growth Hormone Regulation of Insulin-Like Growth Factor-I Gene Expression in Liver. [Internet] [Doctoral dissertation]. Virginia Tech; 2009. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/10919/37373.

Council of Science Editors:

Eleswarapu S. Mechanisms of Growth Hormone Regulation of Insulin-Like Growth Factor-I Gene Expression in Liver. [Doctoral Dissertation]. Virginia Tech; 2009. Available from: http://hdl.handle.net/10919/37373


Universiteit Utrecht

27. Buitenhuis, M. (Miranda). Regulation of lineage choices during granulopoiesis.

Degree: 2003, Universiteit Utrecht

 Aberrant regulation of hematopoiesis can lead to severe clinical conditions ranging from myeloproliferative disorder to severe immune deficiencies. It is therefore important to understand the… (more)

Subjects/Keywords: Geneeskunde; hematopoiesis; neutrophils; eosinophils; differentiation; lineage choices; signal transduction; transcription factors; Protein Kinase B; Signal Transducer and Activator of Transcription 5; Id1; p38MAPK

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Buitenhuis, M. (. (2003). Regulation of lineage choices during granulopoiesis. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/510

Chicago Manual of Style (16th Edition):

Buitenhuis, M (Miranda). “Regulation of lineage choices during granulopoiesis.” 2003. Doctoral Dissertation, Universiteit Utrecht. Accessed October 26, 2020. http://dspace.library.uu.nl:8080/handle/1874/510.

MLA Handbook (7th Edition):

Buitenhuis, M (Miranda). “Regulation of lineage choices during granulopoiesis.” 2003. Web. 26 Oct 2020.

Vancouver:

Buitenhuis M(. Regulation of lineage choices during granulopoiesis. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2003. [cited 2020 Oct 26]. Available from: http://dspace.library.uu.nl:8080/handle/1874/510.

Council of Science Editors:

Buitenhuis M(. Regulation of lineage choices during granulopoiesis. [Doctoral Dissertation]. Universiteit Utrecht; 2003. Available from: http://dspace.library.uu.nl:8080/handle/1874/510


IUPUI

28. Zhou, Hongkang. The essential role of Stat3 in bone homeostasis and mechanotransduction.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Signal Transducer and Activator of Transcription 3 (Stat3) is a transcription factor expressed in bone and joint cells that include… (more)

Subjects/Keywords: Signal Transducer and Activator of Transcription 3; Bone formation; Osteoblast; Osteocyte; Mechanotransduction; Bones  – Growth  – Research  – Analysis  – Evaluation; Bones  – Growth  – Molecular aspects; Musculoskeletal system  – Mechanical properties; Human mechanics; Bones  – Physiology; Biomechanics  – Research; Bones  – Metabolism  – Research  – Analysis  – Evaluation; Bones  – Metabolism  – Disorders; Osteocytes  – Research; Osteoblasts  – Research; Osteoclasts  – Research; Bone cells  – Research; Cellular signal transduction  – Research; Transcription factors  – Research; Bone remodeling

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhou, H. (2014). The essential role of Stat3 in bone homeostasis and mechanotransduction. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/6190

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhou, Hongkang. “The essential role of Stat3 in bone homeostasis and mechanotransduction.” 2014. Thesis, IUPUI. Accessed October 26, 2020. http://hdl.handle.net/1805/6190.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhou, Hongkang. “The essential role of Stat3 in bone homeostasis and mechanotransduction.” 2014. Web. 26 Oct 2020.

Vancouver:

Zhou H. The essential role of Stat3 in bone homeostasis and mechanotransduction. [Internet] [Thesis]. IUPUI; 2014. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1805/6190.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhou H. The essential role of Stat3 in bone homeostasis and mechanotransduction. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/6190

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Buitenhuis, M. (Miranda). Regulation of lineage choices during granulopoiesis.

Degree: 2003, University Utrecht

 Aberrant regulation of hematopoiesis can lead to severe clinical conditions ranging from myeloproliferative disorder to severe immune deficiencies. It is therefore important to understand the… (more)

Subjects/Keywords: hematopoiesis; neutrophils; eosinophils; differentiation; lineage choices; signal transduction; transcription factors; Protein Kinase B; Signal Transducer and Activator of Transcription 5; Id1; p38MAPK

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Buitenhuis, M. (. (2003). Regulation of lineage choices during granulopoiesis. (Doctoral Dissertation). University Utrecht. Retrieved from https://dspace.library.uu.nl/handle/1874/510 ; URN:NBN:NL:UI:10-1874-510 ; URN:NBN:NL:UI:10-1874-510 ; https://dspace.library.uu.nl/handle/1874/510

Chicago Manual of Style (16th Edition):

Buitenhuis, M (Miranda). “Regulation of lineage choices during granulopoiesis.” 2003. Doctoral Dissertation, University Utrecht. Accessed October 26, 2020. https://dspace.library.uu.nl/handle/1874/510 ; URN:NBN:NL:UI:10-1874-510 ; URN:NBN:NL:UI:10-1874-510 ; https://dspace.library.uu.nl/handle/1874/510.

MLA Handbook (7th Edition):

Buitenhuis, M (Miranda). “Regulation of lineage choices during granulopoiesis.” 2003. Web. 26 Oct 2020.

Vancouver:

Buitenhuis M(. Regulation of lineage choices during granulopoiesis. [Internet] [Doctoral dissertation]. University Utrecht; 2003. [cited 2020 Oct 26]. Available from: https://dspace.library.uu.nl/handle/1874/510 ; URN:NBN:NL:UI:10-1874-510 ; URN:NBN:NL:UI:10-1874-510 ; https://dspace.library.uu.nl/handle/1874/510.

Council of Science Editors:

Buitenhuis M(. Regulation of lineage choices during granulopoiesis. [Doctoral Dissertation]. University Utrecht; 2003. Available from: https://dspace.library.uu.nl/handle/1874/510 ; URN:NBN:NL:UI:10-1874-510 ; URN:NBN:NL:UI:10-1874-510 ; https://dspace.library.uu.nl/handle/1874/510

30. Buitenhuis, M. (Miranda). Regulation of lineage choices during granulopoiesis.

Degree: 2003, University Utrecht

 Aberrant regulation of hematopoiesis can lead to severe clinical conditions ranging from myeloproliferative disorder to severe immune deficiencies. It is therefore important to understand the… (more)

Subjects/Keywords: hematopoiesis; neutrophils; eosinophils; differentiation; lineage choices; signal transduction; transcription factors; Protein Kinase B; Signal Transducer and Activator of Transcription 5; Id1; p38MAPK

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Buitenhuis, M. (. (2003). Regulation of lineage choices during granulopoiesis. (Doctoral Dissertation). University Utrecht. Retrieved from https://dspace.library.uu.nl/handle/1874/510 ; URN:NBN:NL:UI:10-1874-510 ; URN:NBN:NL:UI:10-1874-510 ; https://dspace.library.uu.nl/handle/1874/510

Chicago Manual of Style (16th Edition):

Buitenhuis, M (Miranda). “Regulation of lineage choices during granulopoiesis.” 2003. Doctoral Dissertation, University Utrecht. Accessed October 26, 2020. https://dspace.library.uu.nl/handle/1874/510 ; URN:NBN:NL:UI:10-1874-510 ; URN:NBN:NL:UI:10-1874-510 ; https://dspace.library.uu.nl/handle/1874/510.

MLA Handbook (7th Edition):

Buitenhuis, M (Miranda). “Regulation of lineage choices during granulopoiesis.” 2003. Web. 26 Oct 2020.

Vancouver:

Buitenhuis M(. Regulation of lineage choices during granulopoiesis. [Internet] [Doctoral dissertation]. University Utrecht; 2003. [cited 2020 Oct 26]. Available from: https://dspace.library.uu.nl/handle/1874/510 ; URN:NBN:NL:UI:10-1874-510 ; URN:NBN:NL:UI:10-1874-510 ; https://dspace.library.uu.nl/handle/1874/510.

Council of Science Editors:

Buitenhuis M(. Regulation of lineage choices during granulopoiesis. [Doctoral Dissertation]. University Utrecht; 2003. Available from: https://dspace.library.uu.nl/handle/1874/510 ; URN:NBN:NL:UI:10-1874-510 ; URN:NBN:NL:UI:10-1874-510 ; https://dspace.library.uu.nl/handle/1874/510

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