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You searched for subject:(Settore BIO 14 Farmacologia). Showing records 1 – 30 of 10854 total matches.

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1. C. Pavanello. GENETIC LCAT DEFICIENCY AND ATHEROSCLEROSIS: STUDIES ON ENDOTHELIAL CELLS AND MONOCYTES.

Degree: 2018, Università degli Studi di Milano

 Introduzione La lecitin:cholesterol aciltransferasi (LCAT) è l’unico enzima nell’uomo in grado di esterificare il colesterolo nel plasma. Il deficit genetico di LCAT influenza il metabolismo… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Pavanello, C. (2018). GENETIC LCAT DEFICIENCY AND ATHEROSCLEROSIS: STUDIES ON ENDOTHELIAL CELLS AND MONOCYTES. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/597321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pavanello, C.. “GENETIC LCAT DEFICIENCY AND ATHEROSCLEROSIS: STUDIES ON ENDOTHELIAL CELLS AND MONOCYTES.” 2018. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/597321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pavanello, C.. “GENETIC LCAT DEFICIENCY AND ATHEROSCLEROSIS: STUDIES ON ENDOTHELIAL CELLS AND MONOCYTES.” 2018. Web. 19 Jan 2021.

Vancouver:

Pavanello C. GENETIC LCAT DEFICIENCY AND ATHEROSCLEROSIS: STUDIES ON ENDOTHELIAL CELLS AND MONOCYTES. [Internet] [Thesis]. Università degli Studi di Milano; 2018. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/597321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pavanello C. GENETIC LCAT DEFICIENCY AND ATHEROSCLEROSIS: STUDIES ON ENDOTHELIAL CELLS AND MONOCYTES. [Thesis]. Università degli Studi di Milano; 2018. Available from: http://hdl.handle.net/2434/597321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. A. Luoni. BEHAVIORAL, MOLECULAR AND EPIGENETIC CONSEQUENCES OF EARLY LIFE STRESS EXPOSURE AND THEIR IMPACT ON ADULT PSYCHOPATHOLOGY.

Degree: 2014, Università degli Studi di Milano

 There is now consistent evidence that psychiatric diseases may often represent the consequence of exposure to adverse events early in life, which may disrupt the… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Luoni, A. (2014). BEHAVIORAL, MOLECULAR AND EPIGENETIC CONSEQUENCES OF EARLY LIFE STRESS EXPOSURE AND THEIR IMPACT ON ADULT PSYCHOPATHOLOGY. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/229411

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Luoni, A.. “BEHAVIORAL, MOLECULAR AND EPIGENETIC CONSEQUENCES OF EARLY LIFE STRESS EXPOSURE AND THEIR IMPACT ON ADULT PSYCHOPATHOLOGY.” 2014. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/229411.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Luoni, A.. “BEHAVIORAL, MOLECULAR AND EPIGENETIC CONSEQUENCES OF EARLY LIFE STRESS EXPOSURE AND THEIR IMPACT ON ADULT PSYCHOPATHOLOGY.” 2014. Web. 19 Jan 2021.

Vancouver:

Luoni A. BEHAVIORAL, MOLECULAR AND EPIGENETIC CONSEQUENCES OF EARLY LIFE STRESS EXPOSURE AND THEIR IMPACT ON ADULT PSYCHOPATHOLOGY. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/229411.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Luoni A. BEHAVIORAL, MOLECULAR AND EPIGENETIC CONSEQUENCES OF EARLY LIFE STRESS EXPOSURE AND THEIR IMPACT ON ADULT PSYCHOPATHOLOGY. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/229411

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. E. Assi. ROLE OF ACID SPHINGOMYELINASE IN THE TUMOUR MICROENVIRONMENT.

Degree: 2014, Università degli Studi di Milano

 Defective apoptosis represents one of the major causative factors in the development and progression of cancer. The ability of tumour cells to evade engagement of… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Assi, E. (2014). ROLE OF ACID SPHINGOMYELINASE IN THE TUMOUR MICROENVIRONMENT. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/229416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Assi, E.. “ROLE OF ACID SPHINGOMYELINASE IN THE TUMOUR MICROENVIRONMENT.” 2014. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/229416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Assi, E.. “ROLE OF ACID SPHINGOMYELINASE IN THE TUMOUR MICROENVIRONMENT.” 2014. Web. 19 Jan 2021.

Vancouver:

Assi E. ROLE OF ACID SPHINGOMYELINASE IN THE TUMOUR MICROENVIRONMENT. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/229416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Assi E. ROLE OF ACID SPHINGOMYELINASE IN THE TUMOUR MICROENVIRONMENT. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/229416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. P. Amadio. EMERGING ROLE OF NEUROTROPHINS IN CARDIOVASCULAR REGULATION:IMPACT AND CHARACTERIZATION OF GENETIC VARIANT BDNF (VAL66MET) POLYMORPHISM ON THROMBOTIC EVENTS.

Degree: 2014, Università degli Studi di Milano

 Brain-derived neurotrophic factor (BDNF) is a neurotrophin that plays a key role in neuron plasticity (Donovan, et al., 2000), and vascular development (Kermani, et al.,… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Amadio, P. (2014). EMERGING ROLE OF NEUROTROPHINS IN CARDIOVASCULAR REGULATION:IMPACT AND CHARACTERIZATION OF GENETIC VARIANT BDNF (VAL66MET) POLYMORPHISM ON THROMBOTIC EVENTS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/229417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Amadio, P.. “EMERGING ROLE OF NEUROTROPHINS IN CARDIOVASCULAR REGULATION:IMPACT AND CHARACTERIZATION OF GENETIC VARIANT BDNF (VAL66MET) POLYMORPHISM ON THROMBOTIC EVENTS.” 2014. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/229417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Amadio, P.. “EMERGING ROLE OF NEUROTROPHINS IN CARDIOVASCULAR REGULATION:IMPACT AND CHARACTERIZATION OF GENETIC VARIANT BDNF (VAL66MET) POLYMORPHISM ON THROMBOTIC EVENTS.” 2014. Web. 19 Jan 2021.

Vancouver:

Amadio P. EMERGING ROLE OF NEUROTROPHINS IN CARDIOVASCULAR REGULATION:IMPACT AND CHARACTERIZATION OF GENETIC VARIANT BDNF (VAL66MET) POLYMORPHISM ON THROMBOTIC EVENTS. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/229417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Amadio P. EMERGING ROLE OF NEUROTROPHINS IN CARDIOVASCULAR REGULATION:IMPACT AND CHARACTERIZATION OF GENETIC VARIANT BDNF (VAL66MET) POLYMORPHISM ON THROMBOTIC EVENTS. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/229417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. L. Pucci. CARATTERIZZAZIONE FARMACOLOGICA E FUNZIONALE DI NUOVI LIGANDI DEI RECETTORI COLINERGICI NICOTINICI NEURONALI CHE MODULANO IL RILASCIO DI DOPAMINA NELLA VIA MESOSTRIATALE, UNA VIA IMPORTANTE PER GLI EFFETTI COMPORTAMENTALI DELLA NICOTINA.

Degree: 2011, Università degli Studi di Milano

 The α6β2∗ neuronal nicotinic acetylcholine receptor (nAChR) subtype expressed in the dopaminergic mesostriatal pathway mediates many behavioural effects of nicotine, and is selectively blocked by… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Pucci, L. (2011). CARATTERIZZAZIONE FARMACOLOGICA E FUNZIONALE DI NUOVI LIGANDI DEI RECETTORI COLINERGICI NICOTINICI NEURONALI CHE MODULANO IL RILASCIO DI DOPAMINA NELLA VIA MESOSTRIATALE, UNA VIA IMPORTANTE PER GLI EFFETTI COMPORTAMENTALI DELLA NICOTINA. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/150270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pucci, L.. “CARATTERIZZAZIONE FARMACOLOGICA E FUNZIONALE DI NUOVI LIGANDI DEI RECETTORI COLINERGICI NICOTINICI NEURONALI CHE MODULANO IL RILASCIO DI DOPAMINA NELLA VIA MESOSTRIATALE, UNA VIA IMPORTANTE PER GLI EFFETTI COMPORTAMENTALI DELLA NICOTINA.” 2011. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/150270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pucci, L.. “CARATTERIZZAZIONE FARMACOLOGICA E FUNZIONALE DI NUOVI LIGANDI DEI RECETTORI COLINERGICI NICOTINICI NEURONALI CHE MODULANO IL RILASCIO DI DOPAMINA NELLA VIA MESOSTRIATALE, UNA VIA IMPORTANTE PER GLI EFFETTI COMPORTAMENTALI DELLA NICOTINA.” 2011. Web. 19 Jan 2021.

Vancouver:

Pucci L. CARATTERIZZAZIONE FARMACOLOGICA E FUNZIONALE DI NUOVI LIGANDI DEI RECETTORI COLINERGICI NICOTINICI NEURONALI CHE MODULANO IL RILASCIO DI DOPAMINA NELLA VIA MESOSTRIATALE, UNA VIA IMPORTANTE PER GLI EFFETTI COMPORTAMENTALI DELLA NICOTINA. [Internet] [Thesis]. Università degli Studi di Milano; 2011. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/150270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pucci L. CARATTERIZZAZIONE FARMACOLOGICA E FUNZIONALE DI NUOVI LIGANDI DEI RECETTORI COLINERGICI NICOTINICI NEURONALI CHE MODULANO IL RILASCIO DI DOPAMINA NELLA VIA MESOSTRIATALE, UNA VIA IMPORTANTE PER GLI EFFETTI COMPORTAMENTALI DELLA NICOTINA. [Thesis]. Università degli Studi di Milano; 2011. Available from: http://hdl.handle.net/2434/150270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. S. Ancona. STUDIO BIOCHIMICO E FARMACOLOGICO DI CELLULE MUSCOLARI LISCE ISOLATE DA TESSUTI DI PAZIENTI AFFETTI DA TSC2 E LAM. MODIFICAZIONI EPIGENETICHE DEL GENE TSC2 NELLA PATOGENENESI DELLA TSC E DELLA LAM.

Degree: 2011, Università degli Studi di Milano

 Tuberous sclerosis complex (TSC) and lymphangioleiomymatosis (LAM) are rare diseases. TSC is a genetic disease caused by mutation in TSC1 or TSC2 genes. TSC2 cells… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Ancona, S. (2011). STUDIO BIOCHIMICO E FARMACOLOGICO DI CELLULE MUSCOLARI LISCE ISOLATE DA TESSUTI DI PAZIENTI AFFETTI DA TSC2 E LAM. MODIFICAZIONI EPIGENETICHE DEL GENE TSC2 NELLA PATOGENENESI DELLA TSC E DELLA LAM. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/151779

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ancona, S.. “STUDIO BIOCHIMICO E FARMACOLOGICO DI CELLULE MUSCOLARI LISCE ISOLATE DA TESSUTI DI PAZIENTI AFFETTI DA TSC2 E LAM. MODIFICAZIONI EPIGENETICHE DEL GENE TSC2 NELLA PATOGENENESI DELLA TSC E DELLA LAM.” 2011. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/151779.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ancona, S.. “STUDIO BIOCHIMICO E FARMACOLOGICO DI CELLULE MUSCOLARI LISCE ISOLATE DA TESSUTI DI PAZIENTI AFFETTI DA TSC2 E LAM. MODIFICAZIONI EPIGENETICHE DEL GENE TSC2 NELLA PATOGENENESI DELLA TSC E DELLA LAM.” 2011. Web. 19 Jan 2021.

Vancouver:

Ancona S. STUDIO BIOCHIMICO E FARMACOLOGICO DI CELLULE MUSCOLARI LISCE ISOLATE DA TESSUTI DI PAZIENTI AFFETTI DA TSC2 E LAM. MODIFICAZIONI EPIGENETICHE DEL GENE TSC2 NELLA PATOGENENESI DELLA TSC E DELLA LAM. [Internet] [Thesis]. Università degli Studi di Milano; 2011. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/151779.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ancona S. STUDIO BIOCHIMICO E FARMACOLOGICO DI CELLULE MUSCOLARI LISCE ISOLATE DA TESSUTI DI PAZIENTI AFFETTI DA TSC2 E LAM. MODIFICAZIONI EPIGENETICHE DEL GENE TSC2 NELLA PATOGENENESI DELLA TSC E DELLA LAM. [Thesis]. Università degli Studi di Milano; 2011. Available from: http://hdl.handle.net/2434/151779

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. L. Caffino. MODULAZIONE DI MECCANISMI DI PLASTICITA' NEURONALE DOPO SOMMINISTRAZIONE ACUTA O CRONICA DI COCAINA.

Degree: 2010, Università degli Studi di Milano

 The major aim of this thesis was to assess the role of the neurotrophin BDNF and the imemdiate early gene Arc in the mechanism of… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Caffino, L. (2010). MODULAZIONE DI MECCANISMI DI PLASTICITA' NEURONALE DOPO SOMMINISTRAZIONE ACUTA O CRONICA DI COCAINA. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/150094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Caffino, L.. “MODULAZIONE DI MECCANISMI DI PLASTICITA' NEURONALE DOPO SOMMINISTRAZIONE ACUTA O CRONICA DI COCAINA.” 2010. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/150094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Caffino, L.. “MODULAZIONE DI MECCANISMI DI PLASTICITA' NEURONALE DOPO SOMMINISTRAZIONE ACUTA O CRONICA DI COCAINA.” 2010. Web. 19 Jan 2021.

Vancouver:

Caffino L. MODULAZIONE DI MECCANISMI DI PLASTICITA' NEURONALE DOPO SOMMINISTRAZIONE ACUTA O CRONICA DI COCAINA. [Internet] [Thesis]. Università degli Studi di Milano; 2010. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/150094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Caffino L. MODULAZIONE DI MECCANISMI DI PLASTICITA' NEURONALE DOPO SOMMINISTRAZIONE ACUTA O CRONICA DI COCAINA. [Thesis]. Università degli Studi di Milano; 2010. Available from: http://hdl.handle.net/2434/150094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. V. Benedusi. THE ROLE OF PEROXISOME PROLIFERATOR ACTIVATED RECEPTORS IN AMYOTROPHIC LATERAL SCLEROSIS: POTENTIAL MECHANISMS FOR NEUROPROTECTION.

Degree: 2011, Università degli Studi di Milano

 The vast majority of neurodegenerative disorders are adult-onset, incurable diseases. Understanding the pathogenetic mechanisms underlying these disorders and finding molecules apt to correct such processes… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Benedusi, V. (2011). THE ROLE OF PEROXISOME PROLIFERATOR ACTIVATED RECEPTORS IN AMYOTROPHIC LATERAL SCLEROSIS: POTENTIAL MECHANISMS FOR NEUROPROTECTION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/150265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Benedusi, V.. “THE ROLE OF PEROXISOME PROLIFERATOR ACTIVATED RECEPTORS IN AMYOTROPHIC LATERAL SCLEROSIS: POTENTIAL MECHANISMS FOR NEUROPROTECTION.” 2011. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/150265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Benedusi, V.. “THE ROLE OF PEROXISOME PROLIFERATOR ACTIVATED RECEPTORS IN AMYOTROPHIC LATERAL SCLEROSIS: POTENTIAL MECHANISMS FOR NEUROPROTECTION.” 2011. Web. 19 Jan 2021.

Vancouver:

Benedusi V. THE ROLE OF PEROXISOME PROLIFERATOR ACTIVATED RECEPTORS IN AMYOTROPHIC LATERAL SCLEROSIS: POTENTIAL MECHANISMS FOR NEUROPROTECTION. [Internet] [Thesis]. Università degli Studi di Milano; 2011. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/150265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Benedusi V. THE ROLE OF PEROXISOME PROLIFERATOR ACTIVATED RECEPTORS IN AMYOTROPHIC LATERAL SCLEROSIS: POTENTIAL MECHANISMS FOR NEUROPROTECTION. [Thesis]. Università degli Studi di Milano; 2011. Available from: http://hdl.handle.net/2434/150265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. G. Monteleone. FUNCTIONAL ROLE OF ESTROGEN RECEPTORS DURING AGING AND THEIR INVOLVEMENT IN INFLAMMATORY PROCESSESS.

Degree: 2011, Università degli Studi di Milano

 Estrogens are female sex hormones, belonging to a group of steroids, and they are responsible for the sexual characteristics of the female. They also have… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Monteleone, G. (2011). FUNCTIONAL ROLE OF ESTROGEN RECEPTORS DURING AGING AND THEIR INVOLVEMENT IN INFLAMMATORY PROCESSESS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/150266

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Monteleone, G.. “FUNCTIONAL ROLE OF ESTROGEN RECEPTORS DURING AGING AND THEIR INVOLVEMENT IN INFLAMMATORY PROCESSESS.” 2011. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/150266.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Monteleone, G.. “FUNCTIONAL ROLE OF ESTROGEN RECEPTORS DURING AGING AND THEIR INVOLVEMENT IN INFLAMMATORY PROCESSESS.” 2011. Web. 19 Jan 2021.

Vancouver:

Monteleone G. FUNCTIONAL ROLE OF ESTROGEN RECEPTORS DURING AGING AND THEIR INVOLVEMENT IN INFLAMMATORY PROCESSESS. [Internet] [Thesis]. Università degli Studi di Milano; 2011. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/150266.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Monteleone G. FUNCTIONAL ROLE OF ESTROGEN RECEPTORS DURING AGING AND THEIR INVOLVEMENT IN INFLAMMATORY PROCESSESS. [Thesis]. Università degli Studi di Milano; 2011. Available from: http://hdl.handle.net/2434/150266

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. M. Marullo. NEW EVIDENCES OF TWO DIFFERENT KEY PATHOGENIC MECHANISMS IN HUNTINGTON'S DISEASE.

Degree: 2012, Università degli Studi di Milano

 Huntington’s disease (HD) is an adult-onset neurodegenerative disorder characterized by several alterations in critical molecular and cellular pathways. Although genetic and experimental evidence has provided… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Marullo, M. (2012). NEW EVIDENCES OF TWO DIFFERENT KEY PATHOGENIC MECHANISMS IN HUNTINGTON'S DISEASE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/170272

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marullo, M.. “NEW EVIDENCES OF TWO DIFFERENT KEY PATHOGENIC MECHANISMS IN HUNTINGTON'S DISEASE.” 2012. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/170272.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marullo, M.. “NEW EVIDENCES OF TWO DIFFERENT KEY PATHOGENIC MECHANISMS IN HUNTINGTON'S DISEASE.” 2012. Web. 19 Jan 2021.

Vancouver:

Marullo M. NEW EVIDENCES OF TWO DIFFERENT KEY PATHOGENIC MECHANISMS IN HUNTINGTON'S DISEASE. [Internet] [Thesis]. Università degli Studi di Milano; 2012. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/170272.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marullo M. NEW EVIDENCES OF TWO DIFFERENT KEY PATHOGENIC MECHANISMS IN HUNTINGTON'S DISEASE. [Thesis]. Università degli Studi di Milano; 2012. Available from: http://hdl.handle.net/2434/170272

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. C. Bossio. TRANSDIFFERENZIAMENTO DI CELLULE STAMINALI MESENCHIMALI DI RODITORE IN CELLULE DEL SISTEMA NERVOSO CENTRALE.

Degree: 2012, Università degli Studi di Milano

 One of the most important challenges in modern medicine is the identification of cell therapy protocols, enabling identification of personalized treatment strategies. WIthin this context,… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bossio, C. (2012). TRANSDIFFERENZIAMENTO DI CELLULE STAMINALI MESENCHIMALI DI RODITORE IN CELLULE DEL SISTEMA NERVOSO CENTRALE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/171967

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bossio, C.. “TRANSDIFFERENZIAMENTO DI CELLULE STAMINALI MESENCHIMALI DI RODITORE IN CELLULE DEL SISTEMA NERVOSO CENTRALE.” 2012. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/171967.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bossio, C.. “TRANSDIFFERENZIAMENTO DI CELLULE STAMINALI MESENCHIMALI DI RODITORE IN CELLULE DEL SISTEMA NERVOSO CENTRALE.” 2012. Web. 19 Jan 2021.

Vancouver:

Bossio C. TRANSDIFFERENZIAMENTO DI CELLULE STAMINALI MESENCHIMALI DI RODITORE IN CELLULE DEL SISTEMA NERVOSO CENTRALE. [Internet] [Thesis]. Università degli Studi di Milano; 2012. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/171967.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bossio C. TRANSDIFFERENZIAMENTO DI CELLULE STAMINALI MESENCHIMALI DI RODITORE IN CELLULE DEL SISTEMA NERVOSO CENTRALE. [Thesis]. Università degli Studi di Milano; 2012. Available from: http://hdl.handle.net/2434/171967

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. V. Cea. THE INHIBITION OF ILK-1 INCREASES THE EFFICACY OF ANTI-ANGIOGENIC THERAPIES.

Degree: 2013, Università degli Studi di Milano

 The prognosis of the most common glial tumors, the glioblastoma (GBM; World Health Organization grade IV), remains poor with a 2-year survival rate in less… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cea, V. (2013). THE INHIBITION OF ILK-1 INCREASES THE EFFICACY OF ANTI-ANGIOGENIC THERAPIES. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/214972

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cea, V.. “THE INHIBITION OF ILK-1 INCREASES THE EFFICACY OF ANTI-ANGIOGENIC THERAPIES.” 2013. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/214972.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cea, V.. “THE INHIBITION OF ILK-1 INCREASES THE EFFICACY OF ANTI-ANGIOGENIC THERAPIES.” 2013. Web. 19 Jan 2021.

Vancouver:

Cea V. THE INHIBITION OF ILK-1 INCREASES THE EFFICACY OF ANTI-ANGIOGENIC THERAPIES. [Internet] [Thesis]. Università degli Studi di Milano; 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/214972.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cea V. THE INHIBITION OF ILK-1 INCREASES THE EFFICACY OF ANTI-ANGIOGENIC THERAPIES. [Thesis]. Università degli Studi di Milano; 2013. Available from: http://hdl.handle.net/2434/214972

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. A. Raspa. RECUPERO MOTORIO IN MODELLO MURINO DI MORBO DI PARKINSON DOPO TRAPIANTO DI CELLULE STAMINALI POST-MORTEM.

Degree: 2013, Università degli Studi di Milano

Abbiamo studiato una strategia per migliorare i sintomi motori di topi che avevano ricevuto lesioni con una neurotossina specifica, MPTP, un modello murino di morbo… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Raspa, A. (2013). RECUPERO MOTORIO IN MODELLO MURINO DI MORBO DI PARKINSON DOPO TRAPIANTO DI CELLULE STAMINALI POST-MORTEM. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/217712

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Raspa, A.. “RECUPERO MOTORIO IN MODELLO MURINO DI MORBO DI PARKINSON DOPO TRAPIANTO DI CELLULE STAMINALI POST-MORTEM.” 2013. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/217712.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Raspa, A.. “RECUPERO MOTORIO IN MODELLO MURINO DI MORBO DI PARKINSON DOPO TRAPIANTO DI CELLULE STAMINALI POST-MORTEM.” 2013. Web. 19 Jan 2021.

Vancouver:

Raspa A. RECUPERO MOTORIO IN MODELLO MURINO DI MORBO DI PARKINSON DOPO TRAPIANTO DI CELLULE STAMINALI POST-MORTEM. [Internet] [Thesis]. Università degli Studi di Milano; 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/217712.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Raspa A. RECUPERO MOTORIO IN MODELLO MURINO DI MORBO DI PARKINSON DOPO TRAPIANTO DI CELLULE STAMINALI POST-MORTEM. [Thesis]. Università degli Studi di Milano; 2013. Available from: http://hdl.handle.net/2434/217712

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. S. Bolamperti. ESTROGEN- GROWTH HORMONE INTERACTION IN BONE CELLS OF THE OSTEOGENIC LINEAGE: GROWTH HORMONE ANABOLIC ACTIVITY ON HUMAN OSTEOBLASTS AND THEIR MESENCHYMAL PRECURSORS IS MODULATED BY 17ΒETA-ESTRADIOL THROUGH A POST RECEPTOR MECHANISM.

Degree: 2014, Università degli Studi di Milano

 In aging the loss of bone mass goes along with a decline of 17beta-estradiol (E2) and Growth Hormone (GH), which are known to be bone… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Bolamperti, S. (2014). ESTROGEN- GROWTH HORMONE INTERACTION IN BONE CELLS OF THE OSTEOGENIC LINEAGE: GROWTH HORMONE ANABOLIC ACTIVITY ON HUMAN OSTEOBLASTS AND THEIR MESENCHYMAL PRECURSORS IS MODULATED BY 17ΒETA-ESTRADIOL THROUGH A POST RECEPTOR MECHANISM. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/229429

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bolamperti, S.. “ESTROGEN- GROWTH HORMONE INTERACTION IN BONE CELLS OF THE OSTEOGENIC LINEAGE: GROWTH HORMONE ANABOLIC ACTIVITY ON HUMAN OSTEOBLASTS AND THEIR MESENCHYMAL PRECURSORS IS MODULATED BY 17ΒETA-ESTRADIOL THROUGH A POST RECEPTOR MECHANISM.” 2014. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/229429.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bolamperti, S.. “ESTROGEN- GROWTH HORMONE INTERACTION IN BONE CELLS OF THE OSTEOGENIC LINEAGE: GROWTH HORMONE ANABOLIC ACTIVITY ON HUMAN OSTEOBLASTS AND THEIR MESENCHYMAL PRECURSORS IS MODULATED BY 17ΒETA-ESTRADIOL THROUGH A POST RECEPTOR MECHANISM.” 2014. Web. 19 Jan 2021.

Vancouver:

Bolamperti S. ESTROGEN- GROWTH HORMONE INTERACTION IN BONE CELLS OF THE OSTEOGENIC LINEAGE: GROWTH HORMONE ANABOLIC ACTIVITY ON HUMAN OSTEOBLASTS AND THEIR MESENCHYMAL PRECURSORS IS MODULATED BY 17ΒETA-ESTRADIOL THROUGH A POST RECEPTOR MECHANISM. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/229429.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bolamperti S. ESTROGEN- GROWTH HORMONE INTERACTION IN BONE CELLS OF THE OSTEOGENIC LINEAGE: GROWTH HORMONE ANABOLIC ACTIVITY ON HUMAN OSTEOBLASTS AND THEIR MESENCHYMAL PRECURSORS IS MODULATED BY 17ΒETA-ESTRADIOL THROUGH A POST RECEPTOR MECHANISM. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/229429

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. S. Carelli. DEVELOPING A REGENERATIVE MEDICINE APPROACH FOR THE TREATMENT OF PARKINSON'S DISEASE.

Degree: 2014, Università degli Studi di Milano

 Parkinson’s disease (PD) is the second most common neurodegenerative disease, after Alzheimer’s disease, and the most common movement disorder. Drug treatment and deep brain stimulation… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Carelli, S. (2014). DEVELOPING A REGENERATIVE MEDICINE APPROACH FOR THE TREATMENT OF PARKINSON'S DISEASE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/230550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carelli, S.. “DEVELOPING A REGENERATIVE MEDICINE APPROACH FOR THE TREATMENT OF PARKINSON'S DISEASE.” 2014. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/230550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carelli, S.. “DEVELOPING A REGENERATIVE MEDICINE APPROACH FOR THE TREATMENT OF PARKINSON'S DISEASE.” 2014. Web. 19 Jan 2021.

Vancouver:

Carelli S. DEVELOPING A REGENERATIVE MEDICINE APPROACH FOR THE TREATMENT OF PARKINSON'S DISEASE. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/230550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carelli S. DEVELOPING A REGENERATIVE MEDICINE APPROACH FOR THE TREATMENT OF PARKINSON'S DISEASE. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/230550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. V. Galbiati. ASSESSMENT OF THE ALLERGENIC POTENTIAL OF XENOBIOTICS: IN VIVO IN VITRO A BACK-AND-FORTH APPROACH.

Degree: 2014, Università degli Studi di Milano

 Allergic contact dermatitis (ACD) is an important occupational and environmental disease caused by topical exposure to low molecular weight chemical allergens. The development of ACD… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Galbiati, V. (2014). ASSESSMENT OF THE ALLERGENIC POTENTIAL OF XENOBIOTICS: IN VIVO IN VITRO A BACK-AND-FORTH APPROACH. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/246102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Galbiati, V.. “ASSESSMENT OF THE ALLERGENIC POTENTIAL OF XENOBIOTICS: IN VIVO IN VITRO A BACK-AND-FORTH APPROACH.” 2014. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/246102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Galbiati, V.. “ASSESSMENT OF THE ALLERGENIC POTENTIAL OF XENOBIOTICS: IN VIVO IN VITRO A BACK-AND-FORTH APPROACH.” 2014. Web. 19 Jan 2021.

Vancouver:

Galbiati V. ASSESSMENT OF THE ALLERGENIC POTENTIAL OF XENOBIOTICS: IN VIVO IN VITRO A BACK-AND-FORTH APPROACH. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/246102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Galbiati V. ASSESSMENT OF THE ALLERGENIC POTENTIAL OF XENOBIOTICS: IN VIVO IN VITRO A BACK-AND-FORTH APPROACH. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/246102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. S. Pambianco. PERSISTENT INHIBITION OF MITOCHONDRIAL BIOGENESIS IN DYSTROPHIC MICE: IDENTIFICATION OF NITRIC OXIDE-DEPENDENT SALVAGE PATHWAY.

Degree: 2014, Università degli Studi di Milano

 Muscular dystrophies are a group of genetic diseases showing muscle degeneration characterized by progressive skeletal muscle weakness, defects in muscle proteins fiber necrosis, and progressive… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Pambianco, S. (2014). PERSISTENT INHIBITION OF MITOCHONDRIAL BIOGENESIS IN DYSTROPHIC MICE: IDENTIFICATION OF NITRIC OXIDE-DEPENDENT SALVAGE PATHWAY. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/246809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pambianco, S.. “PERSISTENT INHIBITION OF MITOCHONDRIAL BIOGENESIS IN DYSTROPHIC MICE: IDENTIFICATION OF NITRIC OXIDE-DEPENDENT SALVAGE PATHWAY.” 2014. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/246809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pambianco, S.. “PERSISTENT INHIBITION OF MITOCHONDRIAL BIOGENESIS IN DYSTROPHIC MICE: IDENTIFICATION OF NITRIC OXIDE-DEPENDENT SALVAGE PATHWAY.” 2014. Web. 19 Jan 2021.

Vancouver:

Pambianco S. PERSISTENT INHIBITION OF MITOCHONDRIAL BIOGENESIS IN DYSTROPHIC MICE: IDENTIFICATION OF NITRIC OXIDE-DEPENDENT SALVAGE PATHWAY. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/246809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pambianco S. PERSISTENT INHIBITION OF MITOCHONDRIAL BIOGENESIS IN DYSTROPHIC MICE: IDENTIFICATION OF NITRIC OXIDE-DEPENDENT SALVAGE PATHWAY. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/246809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. C. Vicidomini. POSITIVE MODULATION OF MGLU5 REVERSES ASD-LIKE BEHAVIORS FOUND IN SHANK3 KNOCK-OUT MICE.

Degree: 2016, Università degli Studi di Milano

 Shank proteins are the major scaffold proteins that organize the postsynaptic density at the excitatory synapses. Shank1-3 proteins are associated with type I mGluRs via… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Vicidomini, C. (2016). POSITIVE MODULATION OF MGLU5 REVERSES ASD-LIKE BEHAVIORS FOUND IN SHANK3 KNOCK-OUT MICE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/353045

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vicidomini, C.. “POSITIVE MODULATION OF MGLU5 REVERSES ASD-LIKE BEHAVIORS FOUND IN SHANK3 KNOCK-OUT MICE.” 2016. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/353045.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vicidomini, C.. “POSITIVE MODULATION OF MGLU5 REVERSES ASD-LIKE BEHAVIORS FOUND IN SHANK3 KNOCK-OUT MICE.” 2016. Web. 19 Jan 2021.

Vancouver:

Vicidomini C. POSITIVE MODULATION OF MGLU5 REVERSES ASD-LIKE BEHAVIORS FOUND IN SHANK3 KNOCK-OUT MICE. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/353045.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vicidomini C. POSITIVE MODULATION OF MGLU5 REVERSES ASD-LIKE BEHAVIORS FOUND IN SHANK3 KNOCK-OUT MICE. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/353045

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. C.A. Elia. EFFECTS OF MICROVESICLES DERIVED FROM BONE MARROW MESENCHYMAL STEM CELLS IN EXPERIMENTAL MODELS OF ALZHEIMER¿S DISEASE.

Degree: 2016, Università degli Studi di Milano

 Alzheimer's disease (AD) is the most common form of neurodegenerative illness leading to dementia characterized by the accumulation of abnormally folded β-Amyloid (Aβ) and tau… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Elia, C. (2016). EFFECTS OF MICROVESICLES DERIVED FROM BONE MARROW MESENCHYMAL STEM CELLS IN EXPERIMENTAL MODELS OF ALZHEIMER¿S DISEASE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/351930

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Elia, C.A.. “EFFECTS OF MICROVESICLES DERIVED FROM BONE MARROW MESENCHYMAL STEM CELLS IN EXPERIMENTAL MODELS OF ALZHEIMER¿S DISEASE.” 2016. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/351930.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Elia, C.A.. “EFFECTS OF MICROVESICLES DERIVED FROM BONE MARROW MESENCHYMAL STEM CELLS IN EXPERIMENTAL MODELS OF ALZHEIMER¿S DISEASE.” 2016. Web. 19 Jan 2021.

Vancouver:

Elia C. EFFECTS OF MICROVESICLES DERIVED FROM BONE MARROW MESENCHYMAL STEM CELLS IN EXPERIMENTAL MODELS OF ALZHEIMER¿S DISEASE. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/351930.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Elia C. EFFECTS OF MICROVESICLES DERIVED FROM BONE MARROW MESENCHYMAL STEM CELLS IN EXPERIMENTAL MODELS OF ALZHEIMER¿S DISEASE. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/351930

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. M. Tamborini. A NANOTECHNOLOGY APPROACH FOR GLIOBLASTOMA TARGETED THERAPY.

Degree: 2016, Università degli Studi di Milano

 Glioblastoma multiforme (GBM) is an extremely aggressive type of glioma. Life expectancy is around two years after diagnosis, due to recidivism and to the presence… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Tamborini, M. (2016). A NANOTECHNOLOGY APPROACH FOR GLIOBLASTOMA TARGETED THERAPY. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/351946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tamborini, M.. “A NANOTECHNOLOGY APPROACH FOR GLIOBLASTOMA TARGETED THERAPY.” 2016. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/351946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tamborini, M.. “A NANOTECHNOLOGY APPROACH FOR GLIOBLASTOMA TARGETED THERAPY.” 2016. Web. 19 Jan 2021.

Vancouver:

Tamborini M. A NANOTECHNOLOGY APPROACH FOR GLIOBLASTOMA TARGETED THERAPY. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/351946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tamborini M. A NANOTECHNOLOGY APPROACH FOR GLIOBLASTOMA TARGETED THERAPY. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/351946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. E. DI PAOLO. TESTING THE EFFICACY OF BRAIN CHOLESTEROL SUPPLEMENTATION AS A POSSIBLE THERAPEUTIC APPROACH FOR HUNTINGTON'S DISEASE.

Degree: 2017, Università degli Studi di Milano

 Huntington's disease (HD) is a progressive, fatal, adult-onset, neurodegenerative disorder caused by an expanded CAG repeat in the huntingtin gene, which encodes an abnormally long… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

PAOLO, E. D. (2017). TESTING THE EFFICACY OF BRAIN CHOLESTEROL SUPPLEMENTATION AS A POSSIBLE THERAPEUTIC APPROACH FOR HUNTINGTON'S DISEASE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/503284

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

PAOLO, E. DI. “TESTING THE EFFICACY OF BRAIN CHOLESTEROL SUPPLEMENTATION AS A POSSIBLE THERAPEUTIC APPROACH FOR HUNTINGTON'S DISEASE.” 2017. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/503284.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

PAOLO, E. DI. “TESTING THE EFFICACY OF BRAIN CHOLESTEROL SUPPLEMENTATION AS A POSSIBLE THERAPEUTIC APPROACH FOR HUNTINGTON'S DISEASE.” 2017. Web. 19 Jan 2021.

Vancouver:

PAOLO ED. TESTING THE EFFICACY OF BRAIN CHOLESTEROL SUPPLEMENTATION AS A POSSIBLE THERAPEUTIC APPROACH FOR HUNTINGTON'S DISEASE. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/503284.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

PAOLO ED. TESTING THE EFFICACY OF BRAIN CHOLESTEROL SUPPLEMENTATION AS A POSSIBLE THERAPEUTIC APPROACH FOR HUNTINGTON'S DISEASE. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/503284

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. A.C. Rossetti. INFLAMMATION AND VULNERABILITY FOR MAJOR DEPRESSION: IN SEARCH OF COMMON MOLECULAR PATHWAYS.

Degree: 2017, Università degli Studi di Milano

 The comprehension of the molecular mechanisms underpinning Major Depression (MD) is becoming a crucial issue in public health, considering that this psychiatric disorder has been… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Rossetti, A. (2017). INFLAMMATION AND VULNERABILITY FOR MAJOR DEPRESSION: IN SEARCH OF COMMON MOLECULAR PATHWAYS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/468721

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rossetti, A.C.. “INFLAMMATION AND VULNERABILITY FOR MAJOR DEPRESSION: IN SEARCH OF COMMON MOLECULAR PATHWAYS.” 2017. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/468721.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rossetti, A.C.. “INFLAMMATION AND VULNERABILITY FOR MAJOR DEPRESSION: IN SEARCH OF COMMON MOLECULAR PATHWAYS.” 2017. Web. 19 Jan 2021.

Vancouver:

Rossetti A. INFLAMMATION AND VULNERABILITY FOR MAJOR DEPRESSION: IN SEARCH OF COMMON MOLECULAR PATHWAYS. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/468721.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rossetti A. INFLAMMATION AND VULNERABILITY FOR MAJOR DEPRESSION: IN SEARCH OF COMMON MOLECULAR PATHWAYS. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/468721

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. G.L. Pepe. THE PHYSIOLOGICAL RESPONSE OF RESIDENT MACROPHAGES TO THE ESTROGEN SURGE.

Degree: 2017, Università degli Studi di Milano

 I macrofagi sono cellule immunitarie che risiedono in quasi tutti i tessuti dell’organismo e svolgono un ruolo chiave nella difesa dell'organismo contro le infezioni dovute… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Pepe, G. (2017). THE PHYSIOLOGICAL RESPONSE OF RESIDENT MACROPHAGES TO THE ESTROGEN SURGE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/469243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pepe, G.L.. “THE PHYSIOLOGICAL RESPONSE OF RESIDENT MACROPHAGES TO THE ESTROGEN SURGE.” 2017. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/469243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pepe, G.L.. “THE PHYSIOLOGICAL RESPONSE OF RESIDENT MACROPHAGES TO THE ESTROGEN SURGE.” 2017. Web. 19 Jan 2021.

Vancouver:

Pepe G. THE PHYSIOLOGICAL RESPONSE OF RESIDENT MACROPHAGES TO THE ESTROGEN SURGE. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/469243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pepe G. THE PHYSIOLOGICAL RESPONSE OF RESIDENT MACROPHAGES TO THE ESTROGEN SURGE. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/469243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. A. DI MINNO. DIET, OXIDATIVE STRESS STATUS AND INFLAMMATION IN PATIENTS WITH CARDIOVASCULAR DISEASE: IN SEARCH FOR A LINK.

Degree: 2018, Università degli Studi di Milano

 BACKGROUND. Previous studies have provided a rationale for understanding the health benefits of Mediterranean Diet (MD) against a variety of chronic diseases. OBJECTIVES AND EVIDENCE… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

MINNO, A. D. (2018). DIET, OXIDATIVE STRESS STATUS AND INFLAMMATION IN PATIENTS WITH CARDIOVASCULAR DISEASE: IN SEARCH FOR A LINK. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/544432

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

MINNO, A. DI. “DIET, OXIDATIVE STRESS STATUS AND INFLAMMATION IN PATIENTS WITH CARDIOVASCULAR DISEASE: IN SEARCH FOR A LINK.” 2018. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/544432.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

MINNO, A. DI. “DIET, OXIDATIVE STRESS STATUS AND INFLAMMATION IN PATIENTS WITH CARDIOVASCULAR DISEASE: IN SEARCH FOR A LINK.” 2018. Web. 19 Jan 2021.

Vancouver:

MINNO AD. DIET, OXIDATIVE STRESS STATUS AND INFLAMMATION IN PATIENTS WITH CARDIOVASCULAR DISEASE: IN SEARCH FOR A LINK. [Internet] [Thesis]. Università degli Studi di Milano; 2018. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/544432.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

MINNO AD. DIET, OXIDATIVE STRESS STATUS AND INFLAMMATION IN PATIENTS WITH CARDIOVASCULAR DISEASE: IN SEARCH FOR A LINK. [Thesis]. Università degli Studi di Milano; 2018. Available from: http://hdl.handle.net/2434/544432

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. F.M. Fasoli. RECETTORI COLINERGICI NICOTINICI NEURONALI E NON-NEURONALI: ANALISI DEGLI EFFETTI DEL TRATTAMENTO CRONICO CON NICOTINA IN CONDIZIONI FISIOLOGICHE E PATOLOGICHE.

Degree: 2016, Università degli Studi di Milano

I nAChRs sono largamente espressi nel sistema nervoso centrale (SNC) e periferico (SNP), dove funzionano come i classici canali ionici attivati da ligando, facilitando l’ingresso… (more)

Subjects/Keywords: NICOTINE; Settore BIO/14 - Farmacologia

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APA (6th Edition):

Fasoli, F. (2016). RECETTORI COLINERGICI NICOTINICI NEURONALI E NON-NEURONALI: ANALISI DEGLI EFFETTI DEL TRATTAMENTO CRONICO CON NICOTINA IN CONDIZIONI FISIOLOGICHE E PATOLOGICHE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/347546

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fasoli, F.M.. “RECETTORI COLINERGICI NICOTINICI NEURONALI E NON-NEURONALI: ANALISI DEGLI EFFETTI DEL TRATTAMENTO CRONICO CON NICOTINA IN CONDIZIONI FISIOLOGICHE E PATOLOGICHE.” 2016. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/347546.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fasoli, F.M.. “RECETTORI COLINERGICI NICOTINICI NEURONALI E NON-NEURONALI: ANALISI DEGLI EFFETTI DEL TRATTAMENTO CRONICO CON NICOTINA IN CONDIZIONI FISIOLOGICHE E PATOLOGICHE.” 2016. Web. 19 Jan 2021.

Vancouver:

Fasoli F. RECETTORI COLINERGICI NICOTINICI NEURONALI E NON-NEURONALI: ANALISI DEGLI EFFETTI DEL TRATTAMENTO CRONICO CON NICOTINA IN CONDIZIONI FISIOLOGICHE E PATOLOGICHE. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/347546.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fasoli F. RECETTORI COLINERGICI NICOTINICI NEURONALI E NON-NEURONALI: ANALISI DEGLI EFFETTI DEL TRATTAMENTO CRONICO CON NICOTINA IN CONDIZIONI FISIOLOGICHE E PATOLOGICHE. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/347546

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. C. Vitali. EXAMINING THE DIVERSE CONTRIBUTIONS OF EL TO SYSTEMIC LIPID METABOLISM.

Degree: 2017, Università degli Studi di Milano

 La Lipasi Endoteliale (EL) è l’ultimo membro identificato della famiglia delle lipasi plasmatiche dei trigliceridi (plasma triglyceride lipase). EL ha una maggior specificità per i… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Vitali, C. (2017). EXAMINING THE DIVERSE CONTRIBUTIONS OF EL TO SYSTEMIC LIPID METABOLISM. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/468488

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vitali, C.. “EXAMINING THE DIVERSE CONTRIBUTIONS OF EL TO SYSTEMIC LIPID METABOLISM.” 2017. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/468488.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vitali, C.. “EXAMINING THE DIVERSE CONTRIBUTIONS OF EL TO SYSTEMIC LIPID METABOLISM.” 2017. Web. 19 Jan 2021.

Vancouver:

Vitali C. EXAMINING THE DIVERSE CONTRIBUTIONS OF EL TO SYSTEMIC LIPID METABOLISM. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/468488.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vitali C. EXAMINING THE DIVERSE CONTRIBUTIONS OF EL TO SYSTEMIC LIPID METABOLISM. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/468488

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. C. Vanetti. SEX-DEPENDENT PROPERTIES OF HUMAN ENDOTHELIAL CELLS: FOCUS ON ENOS.

Degree: 2017, Università degli Studi di Milano

 Nonostante le malattie cardiovascolari siano percepite come malattie in prevalenza maschili, numerosi dati clinici ed epidemiologici dimostrano che riguardano anche le donne. Quello che differenzia… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Vanetti, C. (2017). SEX-DEPENDENT PROPERTIES OF HUMAN ENDOTHELIAL CELLS: FOCUS ON ENOS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/469139

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vanetti, C.. “SEX-DEPENDENT PROPERTIES OF HUMAN ENDOTHELIAL CELLS: FOCUS ON ENOS.” 2017. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/469139.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vanetti, C.. “SEX-DEPENDENT PROPERTIES OF HUMAN ENDOTHELIAL CELLS: FOCUS ON ENOS.” 2017. Web. 19 Jan 2021.

Vancouver:

Vanetti C. SEX-DEPENDENT PROPERTIES OF HUMAN ENDOTHELIAL CELLS: FOCUS ON ENOS. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/469139.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vanetti C. SEX-DEPENDENT PROPERTIES OF HUMAN ENDOTHELIAL CELLS: FOCUS ON ENOS. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/469139

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. S. Carnevali. THROMBOXANE PROSTANOID RECEPTOR: FUNCTION, ACTIVATION AND POSSIBLE TARGET FOR CARDIOVASCULAR PROTECTION.

Degree: 2017, Università degli Studi di Milano

 Abstract In the last few years cardiovascular diseases are considered one of the major cause of death, and one of the main player is TXA2… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Carnevali, S. (2017). THROMBOXANE PROSTANOID RECEPTOR: FUNCTION, ACTIVATION AND POSSIBLE TARGET FOR CARDIOVASCULAR PROTECTION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/468658

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carnevali, S.. “THROMBOXANE PROSTANOID RECEPTOR: FUNCTION, ACTIVATION AND POSSIBLE TARGET FOR CARDIOVASCULAR PROTECTION.” 2017. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/468658.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carnevali, S.. “THROMBOXANE PROSTANOID RECEPTOR: FUNCTION, ACTIVATION AND POSSIBLE TARGET FOR CARDIOVASCULAR PROTECTION.” 2017. Web. 19 Jan 2021.

Vancouver:

Carnevali S. THROMBOXANE PROSTANOID RECEPTOR: FUNCTION, ACTIVATION AND POSSIBLE TARGET FOR CARDIOVASCULAR PROTECTION. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/468658.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carnevali S. THROMBOXANE PROSTANOID RECEPTOR: FUNCTION, ACTIVATION AND POSSIBLE TARGET FOR CARDIOVASCULAR PROTECTION. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/468658

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. S.C. Pelucchi. INTEGRATION OF DIFFERENT PATHWAYS IN EARLY STAGE OF ALZHEIMER'S DISEASE PATHOGENESIS, FROM ACTIN REMODELLING TO AΒ FORMATION.

Degree: 2017, Università degli Studi di Milano

 Alzheimer disease (AD) pathogenesis is the result of an interplay of crossing pathways, as amyloid cascade and synaptic failure. It has been shown that Aβ… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Pelucchi, S. (2017). INTEGRATION OF DIFFERENT PATHWAYS IN EARLY STAGE OF ALZHEIMER'S DISEASE PATHOGENESIS, FROM ACTIN REMODELLING TO AΒ FORMATION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/469829

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pelucchi, S.C.. “INTEGRATION OF DIFFERENT PATHWAYS IN EARLY STAGE OF ALZHEIMER'S DISEASE PATHOGENESIS, FROM ACTIN REMODELLING TO AΒ FORMATION.” 2017. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/469829.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pelucchi, S.C.. “INTEGRATION OF DIFFERENT PATHWAYS IN EARLY STAGE OF ALZHEIMER'S DISEASE PATHOGENESIS, FROM ACTIN REMODELLING TO AΒ FORMATION.” 2017. Web. 19 Jan 2021.

Vancouver:

Pelucchi S. INTEGRATION OF DIFFERENT PATHWAYS IN EARLY STAGE OF ALZHEIMER'S DISEASE PATHOGENESIS, FROM ACTIN REMODELLING TO AΒ FORMATION. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/469829.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pelucchi S. INTEGRATION OF DIFFERENT PATHWAYS IN EARLY STAGE OF ALZHEIMER'S DISEASE PATHOGENESIS, FROM ACTIN REMODELLING TO AΒ FORMATION. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/469829

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. L. Culotta. DECIPHERING THE ROLE OF SULFOTRANSFERASE 4A1 IN BRAIN DEVELOPMENT AND NEURONAL FUNCTIONING.

Degree: 2018, Università degli Studi di Milano

 Cytosolic sulfotransferases (SULTs) are enzymes that transfer a sulfuryl group from the obligate donor PAPS (3’-phosphoadenosine 5’-phosphosulfate) onto a variety of exogenous and endogenous substrates… (more)

Subjects/Keywords: Settore BIO/14 - Farmacologia

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APA (6th Edition):

Culotta, L. (2018). DECIPHERING THE ROLE OF SULFOTRANSFERASE 4A1 IN BRAIN DEVELOPMENT AND NEURONAL FUNCTIONING. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/545256

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Culotta, L.. “DECIPHERING THE ROLE OF SULFOTRANSFERASE 4A1 IN BRAIN DEVELOPMENT AND NEURONAL FUNCTIONING.” 2018. Thesis, Università degli Studi di Milano. Accessed January 19, 2021. http://hdl.handle.net/2434/545256.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Culotta, L.. “DECIPHERING THE ROLE OF SULFOTRANSFERASE 4A1 IN BRAIN DEVELOPMENT AND NEURONAL FUNCTIONING.” 2018. Web. 19 Jan 2021.

Vancouver:

Culotta L. DECIPHERING THE ROLE OF SULFOTRANSFERASE 4A1 IN BRAIN DEVELOPMENT AND NEURONAL FUNCTIONING. [Internet] [Thesis]. Università degli Studi di Milano; 2018. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2434/545256.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Culotta L. DECIPHERING THE ROLE OF SULFOTRANSFERASE 4A1 IN BRAIN DEVELOPMENT AND NEURONAL FUNCTIONING. [Thesis]. Università degli Studi di Milano; 2018. Available from: http://hdl.handle.net/2434/545256

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [362]

.