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You searched for subject:(SWI SNF). Showing records 1 – 30 of 52 total matches.

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1. 仲里, 秀次. Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発.

Degree: 博士(医学), 2017, University of the Ryukyus / 琉球大学

 The SWI/SNF chromatin remodeling complex is frequently inactivated by somaticmutations of its various components in various types of cancers, and also by aberrant DNA methylation.… (more)

Subjects/Keywords: Epigenetics; SWI/SNF; mutation; ESCC

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

仲里, . (2017). Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発. (Thesis). University of the Ryukyus / 琉球大学. Retrieved from http://hdl.handle.net/20.500.12000/36588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

仲里, 秀次. “Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発.” 2017. Thesis, University of the Ryukyus / 琉球大学. Accessed July 11, 2020. http://hdl.handle.net/20.500.12000/36588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

仲里, 秀次. “Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発.” 2017. Web. 11 Jul 2020.

Vancouver:

仲里 . Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発. [Internet] [Thesis]. University of the Ryukyus / 琉球大学; 2017. [cited 2020 Jul 11]. Available from: http://hdl.handle.net/20.500.12000/36588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

仲里 . Early-Stage Induction of SWI/SNF Mutations during Esophageal Squamous Cell Carcinogenesis : SIW/SNF遺伝子異常の食道扁平上皮がん発がんの早期における誘発. [Thesis]. University of the Ryukyus / 琉球大学; 2017. Available from: http://hdl.handle.net/20.500.12000/36588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. AL-HUSSAIN, MOHAMED MUBARAK. Investigating chromatin remodelling by the Swi-Snf and Tup1-Cyc8 complexes.

Degree: School of Genetics & Microbiology. Discipline of Microbiology, 2020, Trinity College Dublin

Swi-Snf is an ATP-dependent chromatin remodelling complex which generally acts as a co-activator of gene transcription via its removal of promoter nucleosomes. Conversely, Tup1-Cyc8 (Ssn6)… (more)

Subjects/Keywords: Chromatin; Swi-Snf; Tup1-Cyc8

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APA (6th Edition):

AL-HUSSAIN, M. M. (2020). Investigating chromatin remodelling by the Swi-Snf and Tup1-Cyc8 complexes. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/91851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

AL-HUSSAIN, MOHAMED MUBARAK. “Investigating chromatin remodelling by the Swi-Snf and Tup1-Cyc8 complexes.” 2020. Thesis, Trinity College Dublin. Accessed July 11, 2020. http://hdl.handle.net/2262/91851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

AL-HUSSAIN, MOHAMED MUBARAK. “Investigating chromatin remodelling by the Swi-Snf and Tup1-Cyc8 complexes.” 2020. Web. 11 Jul 2020.

Vancouver:

AL-HUSSAIN MM. Investigating chromatin remodelling by the Swi-Snf and Tup1-Cyc8 complexes. [Internet] [Thesis]. Trinity College Dublin; 2020. [cited 2020 Jul 11]. Available from: http://hdl.handle.net/2262/91851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

AL-HUSSAIN MM. Investigating chromatin remodelling by the Swi-Snf and Tup1-Cyc8 complexes. [Thesis]. Trinity College Dublin; 2020. Available from: http://hdl.handle.net/2262/91851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Masliah-Planchon, Julien. Complexe SWI/SNF et cancer _ Altérations génétiques et anomalies métaboliques : SWI/SNF Complexe in Oncogenesis _ Genetic Alterations and Metbolic Anomalies.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2018, Université Paris-Saclay (ComUE)

Il y a presque 20 ans, la mise en évidence de mutations bi-alléliques inactivatrices du gène SMARCB1 dans les tumeurs rhabdoïdes établissait la première démonstration… (more)

Subjects/Keywords: Complexe SWI/SNF; Cancer; Métabolisme; SWI/SNF complex; Cancer; Metabolism

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APA (6th Edition):

Masliah-Planchon, J. (2018). Complexe SWI/SNF et cancer _ Altérations génétiques et anomalies métaboliques : SWI/SNF Complexe in Oncogenesis _ Genetic Alterations and Metbolic Anomalies. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2018SACLS112

Chicago Manual of Style (16th Edition):

Masliah-Planchon, Julien. “Complexe SWI/SNF et cancer _ Altérations génétiques et anomalies métaboliques : SWI/SNF Complexe in Oncogenesis _ Genetic Alterations and Metbolic Anomalies.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed July 11, 2020. http://www.theses.fr/2018SACLS112.

MLA Handbook (7th Edition):

Masliah-Planchon, Julien. “Complexe SWI/SNF et cancer _ Altérations génétiques et anomalies métaboliques : SWI/SNF Complexe in Oncogenesis _ Genetic Alterations and Metbolic Anomalies.” 2018. Web. 11 Jul 2020.

Vancouver:

Masliah-Planchon J. Complexe SWI/SNF et cancer _ Altérations génétiques et anomalies métaboliques : SWI/SNF Complexe in Oncogenesis _ Genetic Alterations and Metbolic Anomalies. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2018. [cited 2020 Jul 11]. Available from: http://www.theses.fr/2018SACLS112.

Council of Science Editors:

Masliah-Planchon J. Complexe SWI/SNF et cancer _ Altérations génétiques et anomalies métaboliques : SWI/SNF Complexe in Oncogenesis _ Genetic Alterations and Metbolic Anomalies. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2018. Available from: http://www.theses.fr/2018SACLS112


University of Toledo Health Science Campus

4. Keenen, Bridget. The Role of SWI/SNF Chromatin Remodeling Complex in Melanoma.

Degree: PhD, College of Medicine, 2010, University of Toledo Health Science Campus

 The microphthalmia-associated transcription factor (MITF) promotes melanocyte differentiation and cell cycle arrest. Paradoxically, MITF also promotes melanoma survival and proliferation, acting like a lineage survival… (more)

Subjects/Keywords: Molecular Biology; SWI/SNF; BRG1; Melanoma; BRM

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APA (6th Edition):

Keenen, B. (2010). The Role of SWI/SNF Chromatin Remodeling Complex in Melanoma. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1271819328

Chicago Manual of Style (16th Edition):

Keenen, Bridget. “The Role of SWI/SNF Chromatin Remodeling Complex in Melanoma.” 2010. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed July 11, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1271819328.

MLA Handbook (7th Edition):

Keenen, Bridget. “The Role of SWI/SNF Chromatin Remodeling Complex in Melanoma.” 2010. Web. 11 Jul 2020.

Vancouver:

Keenen B. The Role of SWI/SNF Chromatin Remodeling Complex in Melanoma. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2010. [cited 2020 Jul 11]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1271819328.

Council of Science Editors:

Keenen B. The Role of SWI/SNF Chromatin Remodeling Complex in Melanoma. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1271819328


University of Sydney

5. Farrell, Andrew William. The Role of Brm in Non-­‐Melanoma Skin Cancer Progression .

Degree: 2016, University of Sydney

 Background: Australia has the highest incidence of non-melanoma skin cancer (NMSC) in the world. These cancers are predominantly caused by exposure to ultraviolet radiation (UVR)… (more)

Subjects/Keywords: Skin cancer; chromation remodelling; Brm; SWI/SNF

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APA (6th Edition):

Farrell, A. W. (2016). The Role of Brm in Non-­‐Melanoma Skin Cancer Progression . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/16438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Farrell, Andrew William. “The Role of Brm in Non-­‐Melanoma Skin Cancer Progression .” 2016. Thesis, University of Sydney. Accessed July 11, 2020. http://hdl.handle.net/2123/16438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Farrell, Andrew William. “The Role of Brm in Non-­‐Melanoma Skin Cancer Progression .” 2016. Web. 11 Jul 2020.

Vancouver:

Farrell AW. The Role of Brm in Non-­‐Melanoma Skin Cancer Progression . [Internet] [Thesis]. University of Sydney; 2016. [cited 2020 Jul 11]. Available from: http://hdl.handle.net/2123/16438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Farrell AW. The Role of Brm in Non-­‐Melanoma Skin Cancer Progression . [Thesis]. University of Sydney; 2016. Available from: http://hdl.handle.net/2123/16438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Κοταντάκη, Πανωραία. Δημιουργία ζώων μοντέλων για τη διερεύνηση του in vivo ρόλου της geminin : αδρανοποίηση του γονιδίου σε μύες με τη χρήση ολοδύναμων εμβρυονικών κυττάρων (knockout) και ιστοειδική υπερέκφραση σε διαγονιδιακούς μύες.

Degree: 2010, University of Patras

 Η ανάπτυξη του ανοσοποιητικού συστήματος ξεκινά από πολυδύναμα αρχέγονα αιμοποιητικά κύτταρα, τα οποία διαδοχικά δίνουν γένεση σε ενδιάμεσους πληθυσμούς προγονικών κυττάρων οι οποίοι σταδιακά χάνουν… (more)

Subjects/Keywords: Μύες; Αδρανοποίηση; Υπερέκφραση; Ανοσοποιητικό σύστημα; SWI/SNF σύμπλοκο; 616.079; Geminin; Cre-loxP; Knockout; Mice; Inactivation; Overexpression; Immune system; SWI/SNF complex

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APA (6th Edition):

Κοταντάκη, . (2010). Δημιουργία ζώων μοντέλων για τη διερεύνηση του in vivo ρόλου της geminin : αδρανοποίηση του γονιδίου σε μύες με τη χρήση ολοδύναμων εμβρυονικών κυττάρων (knockout) και ιστοειδική υπερέκφραση σε διαγονιδιακούς μύες. (Doctoral Dissertation). University of Patras. Retrieved from http://nemertes.lis.upatras.gr/jspui/handle/10889/4356

Chicago Manual of Style (16th Edition):

Κοταντάκη, Πανωραία. “Δημιουργία ζώων μοντέλων για τη διερεύνηση του in vivo ρόλου της geminin : αδρανοποίηση του γονιδίου σε μύες με τη χρήση ολοδύναμων εμβρυονικών κυττάρων (knockout) και ιστοειδική υπερέκφραση σε διαγονιδιακούς μύες.” 2010. Doctoral Dissertation, University of Patras. Accessed July 11, 2020. http://nemertes.lis.upatras.gr/jspui/handle/10889/4356.

MLA Handbook (7th Edition):

Κοταντάκη, Πανωραία. “Δημιουργία ζώων μοντέλων για τη διερεύνηση του in vivo ρόλου της geminin : αδρανοποίηση του γονιδίου σε μύες με τη χρήση ολοδύναμων εμβρυονικών κυττάρων (knockout) και ιστοειδική υπερέκφραση σε διαγονιδιακούς μύες.” 2010. Web. 11 Jul 2020.

Vancouver:

Κοταντάκη . Δημιουργία ζώων μοντέλων για τη διερεύνηση του in vivo ρόλου της geminin : αδρανοποίηση του γονιδίου σε μύες με τη χρήση ολοδύναμων εμβρυονικών κυττάρων (knockout) και ιστοειδική υπερέκφραση σε διαγονιδιακούς μύες. [Internet] [Doctoral dissertation]. University of Patras; 2010. [cited 2020 Jul 11]. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/4356.

Council of Science Editors:

Κοταντάκη . Δημιουργία ζώων μοντέλων για τη διερεύνηση του in vivo ρόλου της geminin : αδρανοποίηση του γονιδίου σε μύες με τη χρήση ολοδύναμων εμβρυονικών κυττάρων (knockout) και ιστοειδική υπερέκφραση σε διαγονιδιακούς μύες. [Doctoral Dissertation]. University of Patras; 2010. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/4356

7. Ertl, Iris Elisabeth. Functional interplay of two SWI/SNF chromatin-remodeling accessory subunits during C. elegans development.

Degree: Departament de Ciències Experimentals i de la Salut, 2015, Universitat Pompeu Fabra

SWI/SNF complexes are ATP-dependent chromatin remodelers highly conserved through evolution. By altering the chromatin state, these complexes can regulate the accessibility of a given genomic… (more)

Subjects/Keywords: Chromatin remodeler; SWI/SNF complexes; C. elegans; ham-3; swsn-2.2; Remodeladors de cromatina; Complexos SWI/SNF; 575

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APA (6th Edition):

Ertl, I. E. (2015). Functional interplay of two SWI/SNF chromatin-remodeling accessory subunits during C. elegans development. (Thesis). Universitat Pompeu Fabra. Retrieved from http://hdl.handle.net/10803/286067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ertl, Iris Elisabeth. “Functional interplay of two SWI/SNF chromatin-remodeling accessory subunits during C. elegans development.” 2015. Thesis, Universitat Pompeu Fabra. Accessed July 11, 2020. http://hdl.handle.net/10803/286067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ertl, Iris Elisabeth. “Functional interplay of two SWI/SNF chromatin-remodeling accessory subunits during C. elegans development.” 2015. Web. 11 Jul 2020.

Vancouver:

Ertl IE. Functional interplay of two SWI/SNF chromatin-remodeling accessory subunits during C. elegans development. [Internet] [Thesis]. Universitat Pompeu Fabra; 2015. [cited 2020 Jul 11]. Available from: http://hdl.handle.net/10803/286067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ertl IE. Functional interplay of two SWI/SNF chromatin-remodeling accessory subunits during C. elegans development. [Thesis]. Universitat Pompeu Fabra; 2015. Available from: http://hdl.handle.net/10803/286067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

8. Minard, Laura. Chromatin regulation by histone chaperone Asf1.

Degree: PhD, Department of Biochemistry, 2010, University of Alberta

 Asf1 is a conserved H3/H4 histone chaperone with multiple functions in chromatin modulation. Using budding yeast as a model, we identify new pathways of Asf1… (more)

Subjects/Keywords: Asf1; SWI/SNF; histone chaperone; hydroxyurea; chromatin; DNA damage response

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APA (6th Edition):

Minard, L. (2010). Chromatin regulation by histone chaperone Asf1. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/44558d82z

Chicago Manual of Style (16th Edition):

Minard, Laura. “Chromatin regulation by histone chaperone Asf1.” 2010. Doctoral Dissertation, University of Alberta. Accessed July 11, 2020. https://era.library.ualberta.ca/files/44558d82z.

MLA Handbook (7th Edition):

Minard, Laura. “Chromatin regulation by histone chaperone Asf1.” 2010. Web. 11 Jul 2020.

Vancouver:

Minard L. Chromatin regulation by histone chaperone Asf1. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2020 Jul 11]. Available from: https://era.library.ualberta.ca/files/44558d82z.

Council of Science Editors:

Minard L. Chromatin regulation by histone chaperone Asf1. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/44558d82z


University of Vermont

9. Taber, Thomas Howland. Thyroid Hormone Receptor SS (trß) Regulation Of Runt-Related Transcription Factor 2 (runx2) In Thyroid Tumorigenesis: Determination Of The Trß Nuclear Protein Complexes That Associate With The Runx2 Gene.

Degree: MS, Pharmacology, 2017, University of Vermont

  Thyroid Tumorigenesis is typically a well understood process, with well delineated oncogenic factors. Follicular and papillary thyroid cancers are typically survivable, with 5-year survival… (more)

Subjects/Keywords: Cancer; RUNX2; SWI/SNF; THRB; Thyroid; Thyroid Cancer; Cell Biology; Oncology

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APA (6th Edition):

Taber, T. H. (2017). Thyroid Hormone Receptor SS (trß) Regulation Of Runt-Related Transcription Factor 2 (runx2) In Thyroid Tumorigenesis: Determination Of The Trß Nuclear Protein Complexes That Associate With The Runx2 Gene. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/820

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Taber, Thomas Howland. “Thyroid Hormone Receptor SS (trß) Regulation Of Runt-Related Transcription Factor 2 (runx2) In Thyroid Tumorigenesis: Determination Of The Trß Nuclear Protein Complexes That Associate With The Runx2 Gene.” 2017. Thesis, University of Vermont. Accessed July 11, 2020. https://scholarworks.uvm.edu/graddis/820.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Taber, Thomas Howland. “Thyroid Hormone Receptor SS (trß) Regulation Of Runt-Related Transcription Factor 2 (runx2) In Thyroid Tumorigenesis: Determination Of The Trß Nuclear Protein Complexes That Associate With The Runx2 Gene.” 2017. Web. 11 Jul 2020.

Vancouver:

Taber TH. Thyroid Hormone Receptor SS (trß) Regulation Of Runt-Related Transcription Factor 2 (runx2) In Thyroid Tumorigenesis: Determination Of The Trß Nuclear Protein Complexes That Associate With The Runx2 Gene. [Internet] [Thesis]. University of Vermont; 2017. [cited 2020 Jul 11]. Available from: https://scholarworks.uvm.edu/graddis/820.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Taber TH. Thyroid Hormone Receptor SS (trß) Regulation Of Runt-Related Transcription Factor 2 (runx2) In Thyroid Tumorigenesis: Determination Of The Trß Nuclear Protein Complexes That Associate With The Runx2 Gene. [Thesis]. University of Vermont; 2017. Available from: https://scholarworks.uvm.edu/graddis/820

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

10. Nguyen, Thinh. CDX2 Regulates Gene Expression Through Recruitment of BRG1-Associated SWI/SNF Chromatin Remodeling Activity .

Degree: 2016, University of Ottawa

 The packaging of genomic DNA into nucleosomes creates a barrier to transcription which can be relieved through ATP-dependent chromatin remodeling via complexes such as the… (more)

Subjects/Keywords: Cdx2; transcription; Brg1; SWI/SNF; Chromatin Remodeling; development

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APA (6th Edition):

Nguyen, T. (2016). CDX2 Regulates Gene Expression Through Recruitment of BRG1-Associated SWI/SNF Chromatin Remodeling Activity . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/35377

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nguyen, Thinh. “CDX2 Regulates Gene Expression Through Recruitment of BRG1-Associated SWI/SNF Chromatin Remodeling Activity .” 2016. Thesis, University of Ottawa. Accessed July 11, 2020. http://hdl.handle.net/10393/35377.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nguyen, Thinh. “CDX2 Regulates Gene Expression Through Recruitment of BRG1-Associated SWI/SNF Chromatin Remodeling Activity .” 2016. Web. 11 Jul 2020.

Vancouver:

Nguyen T. CDX2 Regulates Gene Expression Through Recruitment of BRG1-Associated SWI/SNF Chromatin Remodeling Activity . [Internet] [Thesis]. University of Ottawa; 2016. [cited 2020 Jul 11]. Available from: http://hdl.handle.net/10393/35377.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nguyen T. CDX2 Regulates Gene Expression Through Recruitment of BRG1-Associated SWI/SNF Chromatin Remodeling Activity . [Thesis]. University of Ottawa; 2016. Available from: http://hdl.handle.net/10393/35377

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

11. Tu, William Bo Chen. Chromatin Regulators of MYC Oncoprotein Activity in Cancer.

Degree: PhD, 2017, University of Toronto

The MYC oncoprotein is a key contributor to the development of virtually all cancer types. MYC is known as a global regulator of transcription that… (more)

Subjects/Keywords: Cancer; Epigenetics; G9a; MYC; SWI/SNF; Transcription; 0307

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APA (6th Edition):

Tu, W. B. C. (2017). Chromatin Regulators of MYC Oncoprotein Activity in Cancer. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/88986

Chicago Manual of Style (16th Edition):

Tu, William Bo Chen. “Chromatin Regulators of MYC Oncoprotein Activity in Cancer.” 2017. Doctoral Dissertation, University of Toronto. Accessed July 11, 2020. http://hdl.handle.net/1807/88986.

MLA Handbook (7th Edition):

Tu, William Bo Chen. “Chromatin Regulators of MYC Oncoprotein Activity in Cancer.” 2017. Web. 11 Jul 2020.

Vancouver:

Tu WBC. Chromatin Regulators of MYC Oncoprotein Activity in Cancer. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2020 Jul 11]. Available from: http://hdl.handle.net/1807/88986.

Council of Science Editors:

Tu WBC. Chromatin Regulators of MYC Oncoprotein Activity in Cancer. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/88986


Boston University

12. Buckshaw II, Robert S. Cloning and nextPBM analysis of the mediator and BRG1 associated factor complexes.

Degree: MS, Medical Sciences, 2020, Boston University

 Coordination of gene expression within the cell requires the integrated actions of various multi-protein, gene regulatory complexes. The Mediator and BRG1 Associate Factor (BAF) complexes… (more)

Subjects/Keywords: Systematic biology; BAF; Epigenetic regulation; Immunology; Mediator; PBM; SWI/SNF

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APA (6th Edition):

Buckshaw II, R. S. (2020). Cloning and nextPBM analysis of the mediator and BRG1 associated factor complexes. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/41179

Chicago Manual of Style (16th Edition):

Buckshaw II, Robert S. “Cloning and nextPBM analysis of the mediator and BRG1 associated factor complexes.” 2020. Masters Thesis, Boston University. Accessed July 11, 2020. http://hdl.handle.net/2144/41179.

MLA Handbook (7th Edition):

Buckshaw II, Robert S. “Cloning and nextPBM analysis of the mediator and BRG1 associated factor complexes.” 2020. Web. 11 Jul 2020.

Vancouver:

Buckshaw II RS. Cloning and nextPBM analysis of the mediator and BRG1 associated factor complexes. [Internet] [Masters thesis]. Boston University; 2020. [cited 2020 Jul 11]. Available from: http://hdl.handle.net/2144/41179.

Council of Science Editors:

Buckshaw II RS. Cloning and nextPBM analysis of the mediator and BRG1 associated factor complexes. [Masters Thesis]. Boston University; 2020. Available from: http://hdl.handle.net/2144/41179


Université Paris-Sud – Paris XI

13. Jégu, Teddy. Rôle du facteur de remodelage de la chromatine BAF60 au cours de la progression du cycle cellulaire et du développement chez Arabidopsis thaliana : Role of BAF60, a chromatin-remodeling factor, during cell cycle progression and development of Arabidopsis thaliana.

Degree: Docteur es, Biologie, 2015, Université Paris-Sud – Paris XI

Bien que l’ADN contenu dans les cellules eucaryotes permette le stockage de l’information génétique, c’est l’empaquetage de l’ADN en chromatine qui permet d’organiser finement cette… (more)

Subjects/Keywords: Epigénétique; Chromatine; Complexe de remodelage de la chromatine dépendants de l’ATP de type SWI/SNF; Cycle cellulaire; Développement; Epigenetic; Chromatin; SWI/SNF; Cell cycle; Development

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APA (6th Edition):

Jégu, T. (2015). Rôle du facteur de remodelage de la chromatine BAF60 au cours de la progression du cycle cellulaire et du développement chez Arabidopsis thaliana : Role of BAF60, a chromatin-remodeling factor, during cell cycle progression and development of Arabidopsis thaliana. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2015PA112088

Chicago Manual of Style (16th Edition):

Jégu, Teddy. “Rôle du facteur de remodelage de la chromatine BAF60 au cours de la progression du cycle cellulaire et du développement chez Arabidopsis thaliana : Role of BAF60, a chromatin-remodeling factor, during cell cycle progression and development of Arabidopsis thaliana.” 2015. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed July 11, 2020. http://www.theses.fr/2015PA112088.

MLA Handbook (7th Edition):

Jégu, Teddy. “Rôle du facteur de remodelage de la chromatine BAF60 au cours de la progression du cycle cellulaire et du développement chez Arabidopsis thaliana : Role of BAF60, a chromatin-remodeling factor, during cell cycle progression and development of Arabidopsis thaliana.” 2015. Web. 11 Jul 2020.

Vancouver:

Jégu T. Rôle du facteur de remodelage de la chromatine BAF60 au cours de la progression du cycle cellulaire et du développement chez Arabidopsis thaliana : Role of BAF60, a chromatin-remodeling factor, during cell cycle progression and development of Arabidopsis thaliana. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2015. [cited 2020 Jul 11]. Available from: http://www.theses.fr/2015PA112088.

Council of Science Editors:

Jégu T. Rôle du facteur de remodelage de la chromatine BAF60 au cours de la progression du cycle cellulaire et du développement chez Arabidopsis thaliana : Role of BAF60, a chromatin-remodeling factor, during cell cycle progression and development of Arabidopsis thaliana. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2015. Available from: http://www.theses.fr/2015PA112088

14. Lesbats, Paul. Architecture fonctionnelle du complexe d’integration du vih-1 : Functional architecture of the hiv-1 integration complex.

Degree: Docteur es, Sciences, technologie, santé. Microbiologie, 2011, Université de Bordeaux Segalen

L’intégrase (IN) du VIH-1 est une enzyme clé catalysant l’insertion de l’ADN proviral dans le génome cellulaire au sein d’un complexe nucléoprotéique d’intégration.Les nombreux efforts… (more)

Subjects/Keywords: Rétrovirus; VIH; Intégrase; Complexe d’intégration; Structure-fonction; Oligomérisation; Chromatine; SWI/SNF; Retrovirus; HIV; Integrase; Integration complex; Structure-function; Oligomerization; Chromatin; SWI/SNF

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APA (6th Edition):

Lesbats, P. (2011). Architecture fonctionnelle du complexe d’integration du vih-1 : Functional architecture of the hiv-1 integration complex. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2011BOR21879

Chicago Manual of Style (16th Edition):

Lesbats, Paul. “Architecture fonctionnelle du complexe d’integration du vih-1 : Functional architecture of the hiv-1 integration complex.” 2011. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed July 11, 2020. http://www.theses.fr/2011BOR21879.

MLA Handbook (7th Edition):

Lesbats, Paul. “Architecture fonctionnelle du complexe d’integration du vih-1 : Functional architecture of the hiv-1 integration complex.” 2011. Web. 11 Jul 2020.

Vancouver:

Lesbats P. Architecture fonctionnelle du complexe d’integration du vih-1 : Functional architecture of the hiv-1 integration complex. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2011. [cited 2020 Jul 11]. Available from: http://www.theses.fr/2011BOR21879.

Council of Science Editors:

Lesbats P. Architecture fonctionnelle du complexe d’integration du vih-1 : Functional architecture of the hiv-1 integration complex. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2011. Available from: http://www.theses.fr/2011BOR21879

15. Leruste, Amaury. Immune context of malignant rhabdoid tumors : description and identification of new therapeutic targets : Contexte immunitaire des tumeurs rhabdoïdes : description et identification de nouvelles cibles thérapeutiques.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2019, Université Paris-Saclay (ComUE)

Les tumeurs rhabdoïdes (TR) constituent un rare cancer indifférencié du jeune enfant et du nourrisson, avec un âge médian au diagnostic de 20 mois. Ces… (more)

Subjects/Keywords: Microenvironnement tumoral; Immunothérapie; Récepteur T; Swi/snf; Tumeurs rhabdoïdes; Rétrovirus endogènes; Tumor microenvironment; Immunotherapy; T cell receptor; Swi/snf; Rhabdoid tumors; Endogenous retroviruses

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APA (6th Edition):

Leruste, A. (2019). Immune context of malignant rhabdoid tumors : description and identification of new therapeutic targets : Contexte immunitaire des tumeurs rhabdoïdes : description et identification de nouvelles cibles thérapeutiques. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLS050

Chicago Manual of Style (16th Edition):

Leruste, Amaury. “Immune context of malignant rhabdoid tumors : description and identification of new therapeutic targets : Contexte immunitaire des tumeurs rhabdoïdes : description et identification de nouvelles cibles thérapeutiques.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed July 11, 2020. http://www.theses.fr/2019SACLS050.

MLA Handbook (7th Edition):

Leruste, Amaury. “Immune context of malignant rhabdoid tumors : description and identification of new therapeutic targets : Contexte immunitaire des tumeurs rhabdoïdes : description et identification de nouvelles cibles thérapeutiques.” 2019. Web. 11 Jul 2020.

Vancouver:

Leruste A. Immune context of malignant rhabdoid tumors : description and identification of new therapeutic targets : Contexte immunitaire des tumeurs rhabdoïdes : description et identification de nouvelles cibles thérapeutiques. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2020 Jul 11]. Available from: http://www.theses.fr/2019SACLS050.

Council of Science Editors:

Leruste A. Immune context of malignant rhabdoid tumors : description and identification of new therapeutic targets : Contexte immunitaire des tumeurs rhabdoïdes : description et identification de nouvelles cibles thérapeutiques. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLS050


University of Toledo Health Science Campus

16. Saladi, SrinivasVinod. SWI/SNF Chromatin Remodeling Enzymes: Epigenetic Modulators in Melanoma Invasiveness and Survival.

Degree: PhD, College of Medicine, 2011, University of Toledo Health Science Campus

SWI/SNF enzymes are multi subunit complexes consisting of an ATPase subunit, either BRG1 or BRM. They act as epigenetic modulators and remodel the chromatin in… (more)

Subjects/Keywords: Biomedical Research; Cellular Biology; Molecular Biology; SWI/SNF; Chromatin remodeling; DNA repair; Invasion; Apoptosis

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APA (6th Edition):

Saladi, S. (2011). SWI/SNF Chromatin Remodeling Enzymes: Epigenetic Modulators in Melanoma Invasiveness and Survival. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1310065995

Chicago Manual of Style (16th Edition):

Saladi, SrinivasVinod. “SWI/SNF Chromatin Remodeling Enzymes: Epigenetic Modulators in Melanoma Invasiveness and Survival.” 2011. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed July 11, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1310065995.

MLA Handbook (7th Edition):

Saladi, SrinivasVinod. “SWI/SNF Chromatin Remodeling Enzymes: Epigenetic Modulators in Melanoma Invasiveness and Survival.” 2011. Web. 11 Jul 2020.

Vancouver:

Saladi S. SWI/SNF Chromatin Remodeling Enzymes: Epigenetic Modulators in Melanoma Invasiveness and Survival. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2011. [cited 2020 Jul 11]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1310065995.

Council of Science Editors:

Saladi S. SWI/SNF Chromatin Remodeling Enzymes: Epigenetic Modulators in Melanoma Invasiveness and Survival. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1310065995


Penn State University

17. Chandy, Mark. HISTONE MODIFICATIONS INFLUENCE CHROMATIN MODIFYING AND.

Degree: PhD, Biochemistry and Molecular Biology, 2006, Penn State University

 Chromatin condenses DNA and packages it into the nucleus. The fundamental unit of chromatin is the nucleosome, which compacts and forms higher order structures. Chromatin… (more)

Subjects/Keywords: SAGA; SWI/SNF; chromatin remodeling; bromodomain

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APA (6th Edition):

Chandy, M. (2006). HISTONE MODIFICATIONS INFLUENCE CHROMATIN MODIFYING AND. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/6926

Chicago Manual of Style (16th Edition):

Chandy, Mark. “HISTONE MODIFICATIONS INFLUENCE CHROMATIN MODIFYING AND.” 2006. Doctoral Dissertation, Penn State University. Accessed July 11, 2020. https://etda.libraries.psu.edu/catalog/6926.

MLA Handbook (7th Edition):

Chandy, Mark. “HISTONE MODIFICATIONS INFLUENCE CHROMATIN MODIFYING AND.” 2006. Web. 11 Jul 2020.

Vancouver:

Chandy M. HISTONE MODIFICATIONS INFLUENCE CHROMATIN MODIFYING AND. [Internet] [Doctoral dissertation]. Penn State University; 2006. [cited 2020 Jul 11]. Available from: https://etda.libraries.psu.edu/catalog/6926.

Council of Science Editors:

Chandy M. HISTONE MODIFICATIONS INFLUENCE CHROMATIN MODIFYING AND. [Doctoral Dissertation]. Penn State University; 2006. Available from: https://etda.libraries.psu.edu/catalog/6926


Texas Medical Center

18. Kwan, Suet Yan. REGULATION OF THE OXIDATIVE STRESS RESPONSE BY ARID1A.

Degree: PhD, 2015, Texas Medical Center

SWI/SNF is mutated in about 20% of all human cancers; in particular ARID1A is the most frequently mutated SWI/SNF subunit. ARID1A is a tumor… (more)

Subjects/Keywords: ARID1A; SWI/SNF; oxidative stress; ROS; NRF2; elesclomol; Medicine and Health Sciences; Oncology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kwan, S. Y. (2015). REGULATION OF THE OXIDATIVE STRESS RESPONSE BY ARID1A. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/608

Chicago Manual of Style (16th Edition):

Kwan, Suet Yan. “REGULATION OF THE OXIDATIVE STRESS RESPONSE BY ARID1A.” 2015. Doctoral Dissertation, Texas Medical Center. Accessed July 11, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/608.

MLA Handbook (7th Edition):

Kwan, Suet Yan. “REGULATION OF THE OXIDATIVE STRESS RESPONSE BY ARID1A.” 2015. Web. 11 Jul 2020.

Vancouver:

Kwan SY. REGULATION OF THE OXIDATIVE STRESS RESPONSE BY ARID1A. [Internet] [Doctoral dissertation]. Texas Medical Center; 2015. [cited 2020 Jul 11]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/608.

Council of Science Editors:

Kwan SY. REGULATION OF THE OXIDATIVE STRESS RESPONSE BY ARID1A. [Doctoral Dissertation]. Texas Medical Center; 2015. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/608

19. Faralli, Hervé. Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis.

Degree: Docteur es, Biologie des eucaryotes, 2010, Aix-Marseille 2

L’unité cellulaire du muscle squelettique est la myofibre, un syncytium hautement spécialisé générant la contraction musculaire. Au cours de la croissance et de la régénération… (more)

Subjects/Keywords: Muscle squelettique; Cellules satellites; Développement; Régénération; Tshz3; MyoD; Myog; Baf57; Swi/snf

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APA (6th Edition):

Faralli, H. (2010). Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis. (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2010AIX22045

Chicago Manual of Style (16th Edition):

Faralli, Hervé. “Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis.” 2010. Doctoral Dissertation, Aix-Marseille 2. Accessed July 11, 2020. http://www.theses.fr/2010AIX22045.

MLA Handbook (7th Edition):

Faralli, Hervé. “Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis.” 2010. Web. 11 Jul 2020.

Vancouver:

Faralli H. Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis. [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2010. [cited 2020 Jul 11]. Available from: http://www.theses.fr/2010AIX22045.

Council of Science Editors:

Faralli H. Tshz3 un marqueur des cellules satellites : une étude de sa fonction dans la régulation de la myogenèse chez la souris : Tshz3 a marker of Satellite cells : study of his role in the regulation of mouse myogenesis. [Doctoral Dissertation]. Aix-Marseille 2; 2010. Available from: http://www.theses.fr/2010AIX22045


Cornell University

20. Hah, Nasun. Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses .

Degree: 2011, Cornell University

 Estrogens play crucial roles in regulating gene expression in physiological and disease states. Estrogens acts through estrogen receptors (ERs) and their binding sites in genomic… (more)

Subjects/Keywords: estrogen; estrogen receptor; GRO-seq; swi/snf; baf57; baf180; silac; proteomic; enhancer; edc; estrogen signaling

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APA (6th Edition):

Hah, N. (2011). Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/33589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hah, Nasun. “Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses .” 2011. Thesis, Cornell University. Accessed July 11, 2020. http://hdl.handle.net/1813/33589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hah, Nasun. “Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses .” 2011. Web. 11 Jul 2020.

Vancouver:

Hah N. Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses . [Internet] [Thesis]. Cornell University; 2011. [cited 2020 Jul 11]. Available from: http://hdl.handle.net/1813/33589.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hah N. Signal Regulated Gene Expression: Defining The Effects Of Estrogen Signaling Through Genomic And Proteomic Analyses . [Thesis]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/33589

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

21. Stewart, Kathleen Marie. The Role of SWI/SNF Chromatin Remodeling in Breast Tumorigenesis.

Degree: Biomedical Sciences, 2010, University of California – San Francisco

 Mammary epithelial cell (MEC)-extracellular matrix (ECM) interactions are critical for normal breast tissue development, differentiation and homeostasis by engaging a repertoire of ECM adhesion receptors… (more)

Subjects/Keywords: Biology, Cell; Biology, Molecular; Breast; Breast Cancer; Chromatin remodeling; Integrin; SWI/SNF

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APA (6th Edition):

Stewart, K. M. (2010). The Role of SWI/SNF Chromatin Remodeling in Breast Tumorigenesis. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/0645d0zw

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stewart, Kathleen Marie. “The Role of SWI/SNF Chromatin Remodeling in Breast Tumorigenesis.” 2010. Thesis, University of California – San Francisco. Accessed July 11, 2020. http://www.escholarship.org/uc/item/0645d0zw.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stewart, Kathleen Marie. “The Role of SWI/SNF Chromatin Remodeling in Breast Tumorigenesis.” 2010. Web. 11 Jul 2020.

Vancouver:

Stewart KM. The Role of SWI/SNF Chromatin Remodeling in Breast Tumorigenesis. [Internet] [Thesis]. University of California – San Francisco; 2010. [cited 2020 Jul 11]. Available from: http://www.escholarship.org/uc/item/0645d0zw.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stewart KM. The Role of SWI/SNF Chromatin Remodeling in Breast Tumorigenesis. [Thesis]. University of California – San Francisco; 2010. Available from: http://www.escholarship.org/uc/item/0645d0zw

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

22. Engel, Karin Buser. Participation of Regulatory Factors and the Coregulator Brm in the Combinatorial Control of Transcription.

Degree: Chemistry and Chemical Biology, 2011, University of California – San Francisco

 Metazoan transcriptional regulation is governed by combinatorial control, in which distinct multifactor regulatory complexes composed of various combinations of factors assemble at different genomic sites.… (more)

Subjects/Keywords: Molecular Biology; chromatin remodeling; combinatorial control; glucocorticoid receptor; NFkappaB; Swi/Snf; transcriptional regulation

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APA (6th Edition):

Engel, K. B. (2011). Participation of Regulatory Factors and the Coregulator Brm in the Combinatorial Control of Transcription. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/1c55146w

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Engel, Karin Buser. “Participation of Regulatory Factors and the Coregulator Brm in the Combinatorial Control of Transcription.” 2011. Thesis, University of California – San Francisco. Accessed July 11, 2020. http://www.escholarship.org/uc/item/1c55146w.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Engel, Karin Buser. “Participation of Regulatory Factors and the Coregulator Brm in the Combinatorial Control of Transcription.” 2011. Web. 11 Jul 2020.

Vancouver:

Engel KB. Participation of Regulatory Factors and the Coregulator Brm in the Combinatorial Control of Transcription. [Internet] [Thesis]. University of California – San Francisco; 2011. [cited 2020 Jul 11]. Available from: http://www.escholarship.org/uc/item/1c55146w.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Engel KB. Participation of Regulatory Factors and the Coregulator Brm in the Combinatorial Control of Transcription. [Thesis]. University of California – San Francisco; 2011. Available from: http://www.escholarship.org/uc/item/1c55146w

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

23. Chubak, Melissa C. Role of the SWI/SNF Chromatin Remodelling Complex in the Axon Development of the Drosophila Mushroom Body.

Degree: 2017, University of Western Ontario

 The SWI/SNF complex is an evolutionarily conserved ATP-dependent chromatin remodelling complex that has been implicated in the aetiology of intellectual disability (ID). Among the dominant… (more)

Subjects/Keywords: Drosophila melanogaster; SWI/SNF complex; mushroom body; epigenetics; chromatin remodelling; axon development; Developmental Neuroscience

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APA (6th Edition):

Chubak, M. C. (2017). Role of the SWI/SNF Chromatin Remodelling Complex in the Axon Development of the Drosophila Mushroom Body. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4911

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chubak, Melissa C. “Role of the SWI/SNF Chromatin Remodelling Complex in the Axon Development of the Drosophila Mushroom Body.” 2017. Thesis, University of Western Ontario. Accessed July 11, 2020. https://ir.lib.uwo.ca/etd/4911.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chubak, Melissa C. “Role of the SWI/SNF Chromatin Remodelling Complex in the Axon Development of the Drosophila Mushroom Body.” 2017. Web. 11 Jul 2020.

Vancouver:

Chubak MC. Role of the SWI/SNF Chromatin Remodelling Complex in the Axon Development of the Drosophila Mushroom Body. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2020 Jul 11]. Available from: https://ir.lib.uwo.ca/etd/4911.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chubak MC. Role of the SWI/SNF Chromatin Remodelling Complex in the Axon Development of the Drosophila Mushroom Body. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4911

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New Mexico

24. Wiest, Nathaniel E. MAINTENANCE OF GENETIC AND EPIGENETIC STABILITY DURING DNA DOUBLE-STRAND BREAK REPAIR AND DNA REPLICATION.

Degree: Biomedical Sciences Graduate Program, 2019, University of New Mexico

  Eukaryotic genomes are assembled into a complex of DNA and proteins known as chromatin. The packaging of DNA into chromatin is the foundational strategy… (more)

Subjects/Keywords: Chromatin; SWI/SNF; Double-Strand Break Repair; DNA Replication; DNA Ligase; Genome Stability; Molecular Genetics

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APA (6th Edition):

Wiest, N. E. (2019). MAINTENANCE OF GENETIC AND EPIGENETIC STABILITY DURING DNA DOUBLE-STRAND BREAK REPAIR AND DNA REPLICATION. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/191

Chicago Manual of Style (16th Edition):

Wiest, Nathaniel E. “MAINTENANCE OF GENETIC AND EPIGENETIC STABILITY DURING DNA DOUBLE-STRAND BREAK REPAIR AND DNA REPLICATION.” 2019. Doctoral Dissertation, University of New Mexico. Accessed July 11, 2020. https://digitalrepository.unm.edu/biom_etds/191.

MLA Handbook (7th Edition):

Wiest, Nathaniel E. “MAINTENANCE OF GENETIC AND EPIGENETIC STABILITY DURING DNA DOUBLE-STRAND BREAK REPAIR AND DNA REPLICATION.” 2019. Web. 11 Jul 2020.

Vancouver:

Wiest NE. MAINTENANCE OF GENETIC AND EPIGENETIC STABILITY DURING DNA DOUBLE-STRAND BREAK REPAIR AND DNA REPLICATION. [Internet] [Doctoral dissertation]. University of New Mexico; 2019. [cited 2020 Jul 11]. Available from: https://digitalrepository.unm.edu/biom_etds/191.

Council of Science Editors:

Wiest NE. MAINTENANCE OF GENETIC AND EPIGENETIC STABILITY DURING DNA DOUBLE-STRAND BREAK REPAIR AND DNA REPLICATION. [Doctoral Dissertation]. University of New Mexico; 2019. Available from: https://digitalrepository.unm.edu/biom_etds/191


University of Cincinnati

25. STROBECK, MATTHEW WILLIAM. THE ROLE OF CHROMATIN REMODELING IN RB-MEDIATED CELL CYCLE ARREST.

Degree: PhD, Medicine : Cell and Molecular Biology, 2002, University of Cincinnati

 The anti-proliferative action of the retinoblastoma tumor suppressor protein, RB, is disrupted in the majority of human cancers by multiple mechanisms including: i) viral oncoprotein… (more)

Subjects/Keywords: retinoblastoma; cell cycle; Cyclin A; BRG-1; SWI/SNF

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

STROBECK, M. W. (2002). THE ROLE OF CHROMATIN REMODELING IN RB-MEDIATED CELL CYCLE ARREST. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1014728057

Chicago Manual of Style (16th Edition):

STROBECK, MATTHEW WILLIAM. “THE ROLE OF CHROMATIN REMODELING IN RB-MEDIATED CELL CYCLE ARREST.” 2002. Doctoral Dissertation, University of Cincinnati. Accessed July 11, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1014728057.

MLA Handbook (7th Edition):

STROBECK, MATTHEW WILLIAM. “THE ROLE OF CHROMATIN REMODELING IN RB-MEDIATED CELL CYCLE ARREST.” 2002. Web. 11 Jul 2020.

Vancouver:

STROBECK MW. THE ROLE OF CHROMATIN REMODELING IN RB-MEDIATED CELL CYCLE ARREST. [Internet] [Doctoral dissertation]. University of Cincinnati; 2002. [cited 2020 Jul 11]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1014728057.

Council of Science Editors:

STROBECK MW. THE ROLE OF CHROMATIN REMODELING IN RB-MEDIATED CELL CYCLE ARREST. [Doctoral Dissertation]. University of Cincinnati; 2002. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1014728057

26. McKenna, Brian Douglas. Investigations into islet pdx1 activity: contribution to mature β-cell identity and the identification and characterization of Pdx1 coregulators.

Degree: PhD, Molecular Physiology and Biophysics, 2015, Vanderbilt University

 Pdx1 is a transcription factor of fundamental importance to pancreas formation and adult islet β-cell function. However, little is known regarding its role in mature… (more)

Subjects/Keywords: swi/snf; diabetes; islet; Pdx1; coregulators

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APA (6th Edition):

McKenna, B. D. (2015). Investigations into islet pdx1 activity: contribution to mature β-cell identity and the identification and characterization of Pdx1 coregulators. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-05272015-163131/ ;

Chicago Manual of Style (16th Edition):

McKenna, Brian Douglas. “Investigations into islet pdx1 activity: contribution to mature β-cell identity and the identification and characterization of Pdx1 coregulators.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed July 11, 2020. http://etd.library.vanderbilt.edu/available/etd-05272015-163131/ ;.

MLA Handbook (7th Edition):

McKenna, Brian Douglas. “Investigations into islet pdx1 activity: contribution to mature β-cell identity and the identification and characterization of Pdx1 coregulators.” 2015. Web. 11 Jul 2020.

Vancouver:

McKenna BD. Investigations into islet pdx1 activity: contribution to mature β-cell identity and the identification and characterization of Pdx1 coregulators. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Jul 11]. Available from: http://etd.library.vanderbilt.edu/available/etd-05272015-163131/ ;.

Council of Science Editors:

McKenna BD. Investigations into islet pdx1 activity: contribution to mature β-cell identity and the identification and characterization of Pdx1 coregulators. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-05272015-163131/ ;

27. LEE PEI. THE STUDY OF THE EFFECTS OF A CHANGE IN THE EXPRESSION OF MIXED LINEAGE LEUKEMIA 5 ON TRANSCRIPTION REGULATION.

Degree: 2012, National University of Singapore

Subjects/Keywords: MLL5; SC35; RNAPII; transcription; splicing; SWI/SNF

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APA (6th Edition):

PEI, L. (2012). THE STUDY OF THE EFFECTS OF A CHANGE IN THE EXPRESSION OF MIXED LINEAGE LEUKEMIA 5 ON TRANSCRIPTION REGULATION. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/32363

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

PEI, LEE. “THE STUDY OF THE EFFECTS OF A CHANGE IN THE EXPRESSION OF MIXED LINEAGE LEUKEMIA 5 ON TRANSCRIPTION REGULATION.” 2012. Thesis, National University of Singapore. Accessed July 11, 2020. http://scholarbank.nus.edu.sg/handle/10635/32363.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

PEI, LEE. “THE STUDY OF THE EFFECTS OF A CHANGE IN THE EXPRESSION OF MIXED LINEAGE LEUKEMIA 5 ON TRANSCRIPTION REGULATION.” 2012. Web. 11 Jul 2020.

Vancouver:

PEI L. THE STUDY OF THE EFFECTS OF A CHANGE IN THE EXPRESSION OF MIXED LINEAGE LEUKEMIA 5 ON TRANSCRIPTION REGULATION. [Internet] [Thesis]. National University of Singapore; 2012. [cited 2020 Jul 11]. Available from: http://scholarbank.nus.edu.sg/handle/10635/32363.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

PEI L. THE STUDY OF THE EFFECTS OF A CHANGE IN THE EXPRESSION OF MIXED LINEAGE LEUKEMIA 5 ON TRANSCRIPTION REGULATION. [Thesis]. National University of Singapore; 2012. Available from: http://scholarbank.nus.edu.sg/handle/10635/32363

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. FONG HEI TUNG. LDB-1 AND SWI/SNF CHROMATIN REMODELING COMPLEX PARTICIPATE IN THE TRANSCRIPTIONAL ACTIVATION BY C. ELEGANS BLIMP1/PRDM1.

Degree: 2019, National University of Singapore

Subjects/Keywords: LDB-1; SWI/SNF; BLIMP1; PRDM1; C. elegans; transcription

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APA (6th Edition):

TUNG, F. H. (2019). LDB-1 AND SWI/SNF CHROMATIN REMODELING COMPLEX PARTICIPATE IN THE TRANSCRIPTIONAL ACTIVATION BY C. ELEGANS BLIMP1/PRDM1. (Thesis). National University of Singapore. Retrieved from https://scholarbank.nus.edu.sg/handle/10635/154990

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

TUNG, FONG HEI. “LDB-1 AND SWI/SNF CHROMATIN REMODELING COMPLEX PARTICIPATE IN THE TRANSCRIPTIONAL ACTIVATION BY C. ELEGANS BLIMP1/PRDM1.” 2019. Thesis, National University of Singapore. Accessed July 11, 2020. https://scholarbank.nus.edu.sg/handle/10635/154990.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

TUNG, FONG HEI. “LDB-1 AND SWI/SNF CHROMATIN REMODELING COMPLEX PARTICIPATE IN THE TRANSCRIPTIONAL ACTIVATION BY C. ELEGANS BLIMP1/PRDM1.” 2019. Web. 11 Jul 2020.

Vancouver:

TUNG FH. LDB-1 AND SWI/SNF CHROMATIN REMODELING COMPLEX PARTICIPATE IN THE TRANSCRIPTIONAL ACTIVATION BY C. ELEGANS BLIMP1/PRDM1. [Internet] [Thesis]. National University of Singapore; 2019. [cited 2020 Jul 11]. Available from: https://scholarbank.nus.edu.sg/handle/10635/154990.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

TUNG FH. LDB-1 AND SWI/SNF CHROMATIN REMODELING COMPLEX PARTICIPATE IN THE TRANSCRIPTIONAL ACTIVATION BY C. ELEGANS BLIMP1/PRDM1. [Thesis]. National University of Singapore; 2019. Available from: https://scholarbank.nus.edu.sg/handle/10635/154990

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

29. Fawcett, Emily Marie. A beneficial toxin: The response to hydrogen sulfide improves survival in a changing environment.

Degree: PhD, 2015, University of Washington

 Fluctuations in environmental conditions can be deadly. The ability to rapidly and appropriately respond to stressful conditions can mean the difference between life and death.… (more)

Subjects/Keywords: bookmark; C. elegans; epigenetics; H3K4me; hydrogen sulfide; SWI/SNF; Cellular biology; Biochemistry; Molecular biology; molecular and cellular biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fawcett, E. M. (2015). A beneficial toxin: The response to hydrogen sulfide improves survival in a changing environment. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/34061

Chicago Manual of Style (16th Edition):

Fawcett, Emily Marie. “A beneficial toxin: The response to hydrogen sulfide improves survival in a changing environment.” 2015. Doctoral Dissertation, University of Washington. Accessed July 11, 2020. http://hdl.handle.net/1773/34061.

MLA Handbook (7th Edition):

Fawcett, Emily Marie. “A beneficial toxin: The response to hydrogen sulfide improves survival in a changing environment.” 2015. Web. 11 Jul 2020.

Vancouver:

Fawcett EM. A beneficial toxin: The response to hydrogen sulfide improves survival in a changing environment. [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2020 Jul 11]. Available from: http://hdl.handle.net/1773/34061.

Council of Science Editors:

Fawcett EM. A beneficial toxin: The response to hydrogen sulfide improves survival in a changing environment. [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/34061


Kyoto University

30. Tsuda, Motoyuki. The BRG1/SOX9 axis is critical for acinar cell-derived pancreatic tumorigenesis .

Degree: 2019, Kyoto University

Subjects/Keywords: BRG1; SWI/SNF; pancreatic cancer; PanIN; SOX9

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APA (6th Edition):

Tsuda, M. (2019). The BRG1/SOX9 axis is critical for acinar cell-derived pancreatic tumorigenesis . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/242378

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsuda, Motoyuki. “The BRG1/SOX9 axis is critical for acinar cell-derived pancreatic tumorigenesis .” 2019. Thesis, Kyoto University. Accessed July 11, 2020. http://hdl.handle.net/2433/242378.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsuda, Motoyuki. “The BRG1/SOX9 axis is critical for acinar cell-derived pancreatic tumorigenesis .” 2019. Web. 11 Jul 2020.

Vancouver:

Tsuda M. The BRG1/SOX9 axis is critical for acinar cell-derived pancreatic tumorigenesis . [Internet] [Thesis]. Kyoto University; 2019. [cited 2020 Jul 11]. Available from: http://hdl.handle.net/2433/242378.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsuda M. The BRG1/SOX9 axis is critical for acinar cell-derived pancreatic tumorigenesis . [Thesis]. Kyoto University; 2019. Available from: http://hdl.handle.net/2433/242378

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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