Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(STAT6). Showing records 1 – 22 of 22 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters

1. He, Hongbin. Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum.

Degree: Docteur es, Pathologie humaine, 2010, Aix-Marseille 2

La bilharziose reste un problème de santé majeur. L'équipe du Pr Dessein a montré que les infections élevées étaient déterminées par un locus majeur en… (more)

Subjects/Keywords: Bilharziose; Génétique; Susceptibilité; Fibrose; Ctgf; Stat6; Schistosomiasis; Hepatic fibrosis; Connective tissue growth factor; Stat6 transcription factor

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

He, H. (2010). Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum. (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2010AIX20720

Chicago Manual of Style (16th Edition):

He, Hongbin. “Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum.” 2010. Doctoral Dissertation, Aix-Marseille 2. Accessed April 16, 2021. http://www.theses.fr/2010AIX20720.

MLA Handbook (7th Edition):

He, Hongbin. “Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum.” 2010. Web. 16 Apr 2021.

Vancouver:

He H. Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum. [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2010. [cited 2021 Apr 16]. Available from: http://www.theses.fr/2010AIX20720.

Council of Science Editors:

He H. Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum. [Doctoral Dissertation]. Aix-Marseille 2; 2010. Available from: http://www.theses.fr/2010AIX20720

2. Macagno, Nicolas. Biologie des tumeurs conjonctives de malignité intermédiaire : le modèle des tumeurs fibreuses solitaires méningées : Biology of mesenchymal tumors of intermediate malignancy : the meningeal solitary fibrous tumor model.

Degree: Docteur es, Oncologie, 2019, Aix Marseille Université

Le concept de tumeur conjonctive de malignité intermédiaire a été introduit par les classifications proposées par l’organisation mondiale de la santé (OMS): la tumeur fibreuse… (more)

Subjects/Keywords: Tumeur fibreuse solitaire; Malignité intermédiaire; Méninge; Nab2-Stat6; Tert; Pronostic; Solitary fibrous tumor; Intermediate malignancy; Meninge; Nab2-Stat6; Tert; Prognosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Macagno, N. (2019). Biologie des tumeurs conjonctives de malignité intermédiaire : le modèle des tumeurs fibreuses solitaires méningées : Biology of mesenchymal tumors of intermediate malignancy : the meningeal solitary fibrous tumor model. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2019AIXM0650

Chicago Manual of Style (16th Edition):

Macagno, Nicolas. “Biologie des tumeurs conjonctives de malignité intermédiaire : le modèle des tumeurs fibreuses solitaires méningées : Biology of mesenchymal tumors of intermediate malignancy : the meningeal solitary fibrous tumor model.” 2019. Doctoral Dissertation, Aix Marseille Université. Accessed April 16, 2021. http://www.theses.fr/2019AIXM0650.

MLA Handbook (7th Edition):

Macagno, Nicolas. “Biologie des tumeurs conjonctives de malignité intermédiaire : le modèle des tumeurs fibreuses solitaires méningées : Biology of mesenchymal tumors of intermediate malignancy : the meningeal solitary fibrous tumor model.” 2019. Web. 16 Apr 2021.

Vancouver:

Macagno N. Biologie des tumeurs conjonctives de malignité intermédiaire : le modèle des tumeurs fibreuses solitaires méningées : Biology of mesenchymal tumors of intermediate malignancy : the meningeal solitary fibrous tumor model. [Internet] [Doctoral dissertation]. Aix Marseille Université 2019. [cited 2021 Apr 16]. Available from: http://www.theses.fr/2019AIXM0650.

Council of Science Editors:

Macagno N. Biologie des tumeurs conjonctives de malignité intermédiaire : le modèle des tumeurs fibreuses solitaires méningées : Biology of mesenchymal tumors of intermediate malignancy : the meningeal solitary fibrous tumor model. [Doctoral Dissertation]. Aix Marseille Université 2019. Available from: http://www.theses.fr/2019AIXM0650


Cornell University

3. Gebreselassie, Nebiat. The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis.

Degree: PhD, Immunology, 2012, Cornell University

 Parasitic diseases pose a significant burden on the health of millions of people. Chronic helminth infections are typified by enhanced Th2 responses. Understanding how Th2… (more)

Subjects/Keywords: Trichinella; Nematode; Th2 immune response; stat6; Eosinophils; Nitric oxide; Glucose homeostasis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gebreselassie, N. (2012). The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/31113

Chicago Manual of Style (16th Edition):

Gebreselassie, Nebiat. “The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis.” 2012. Doctoral Dissertation, Cornell University. Accessed April 16, 2021. http://hdl.handle.net/1813/31113.

MLA Handbook (7th Edition):

Gebreselassie, Nebiat. “The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis.” 2012. Web. 16 Apr 2021.

Vancouver:

Gebreselassie N. The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis. [Internet] [Doctoral dissertation]. Cornell University; 2012. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1813/31113.

Council of Science Editors:

Gebreselassie N. The Eosinophil Regulates Immunity To The Parasitic Nematode Trichinella Spiralis. [Doctoral Dissertation]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/31113


Penn State University

4. Stokes, Kindra Nicole. Signaling Pathways in Eosinophils that Regulate Allergic Asthma and Inflammation.

Degree: 2014, Penn State University

 Eosinophils are critical cellular mediators of Th2 responses in allergic asthma and airway inflammation. However, the signals that regulate their functions have not been well… (more)

Subjects/Keywords: Eosinophils; Asthma; Allergy; Inflammation; STAT6; Itk; Btk; Microbiota

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stokes, K. N. (2014). Signaling Pathways in Eosinophils that Regulate Allergic Asthma and Inflammation. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/22898

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stokes, Kindra Nicole. “Signaling Pathways in Eosinophils that Regulate Allergic Asthma and Inflammation.” 2014. Thesis, Penn State University. Accessed April 16, 2021. https://submit-etda.libraries.psu.edu/catalog/22898.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stokes, Kindra Nicole. “Signaling Pathways in Eosinophils that Regulate Allergic Asthma and Inflammation.” 2014. Web. 16 Apr 2021.

Vancouver:

Stokes KN. Signaling Pathways in Eosinophils that Regulate Allergic Asthma and Inflammation. [Internet] [Thesis]. Penn State University; 2014. [cited 2021 Apr 16]. Available from: https://submit-etda.libraries.psu.edu/catalog/22898.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stokes KN. Signaling Pathways in Eosinophils that Regulate Allergic Asthma and Inflammation. [Thesis]. Penn State University; 2014. Available from: https://submit-etda.libraries.psu.edu/catalog/22898

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Debrecen

5. Sánta, Vivien Ibolya. Gyulladásos válaszkészség vizsgálata alternatívan polarizált egér makrofágokban .

Degree: DE – Természettudományi és Technológiai Kar – Biotechnológiai Intézet, University of Debrecen

 A makrofágok a veleszületett immunrendszer funkcionálisan rendkívül heterogén és plasztikus sejtjei, melyek a szöveti homeosztázisban, a kórokozók elleni védekezésben, a szöveti regenerációban és a gyulladásos… (more)

Subjects/Keywords: makrofág; STAT6; alternatív; makrofág; polarizáció

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sánta, V. I. (n.d.). Gyulladásos válaszkészség vizsgálata alternatívan polarizált egér makrofágokban . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/276229

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sánta, Vivien Ibolya. “Gyulladásos válaszkészség vizsgálata alternatívan polarizált egér makrofágokban .” Thesis, University of Debrecen. Accessed April 16, 2021. http://hdl.handle.net/2437/276229.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sánta, Vivien Ibolya. “Gyulladásos válaszkészség vizsgálata alternatívan polarizált egér makrofágokban .” Web. 16 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Sánta VI. Gyulladásos válaszkészség vizsgálata alternatívan polarizált egér makrofágokban . [Internet] [Thesis]. University of Debrecen; [cited 2021 Apr 16]. Available from: http://hdl.handle.net/2437/276229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Sánta VI. Gyulladásos válaszkészség vizsgálata alternatívan polarizált egér makrofágokban . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/276229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

6. Hall, Sara L, M.S. Molecular Mechanisms of Synergy Between IL-13 and IL-17A in Severe Asthma.

Degree: PhD, Medicine: Immunology, 2017, University of Cincinnati

 Increased IL-17A production has been associated with more severe asthma; however, the mechanisms whereby IL-17A can contribute to IL-13-driven pathology in asthmatic patients remain unclear.… (more)

Subjects/Keywords: Surgery; Asthma; IL-13; IL-17A; STAT6; Cytokine; Signal transduction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hall, Sara L, M. S. (2017). Molecular Mechanisms of Synergy Between IL-13 and IL-17A in Severe Asthma. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1505125099399164

Chicago Manual of Style (16th Edition):

Hall, Sara L, M S. “Molecular Mechanisms of Synergy Between IL-13 and IL-17A in Severe Asthma.” 2017. Doctoral Dissertation, University of Cincinnati. Accessed April 16, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1505125099399164.

MLA Handbook (7th Edition):

Hall, Sara L, M S. “Molecular Mechanisms of Synergy Between IL-13 and IL-17A in Severe Asthma.” 2017. Web. 16 Apr 2021.

Vancouver:

Hall, Sara L MS. Molecular Mechanisms of Synergy Between IL-13 and IL-17A in Severe Asthma. [Internet] [Doctoral dissertation]. University of Cincinnati; 2017. [cited 2021 Apr 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1505125099399164.

Council of Science Editors:

Hall, Sara L MS. Molecular Mechanisms of Synergy Between IL-13 and IL-17A in Severe Asthma. [Doctoral Dissertation]. University of Cincinnati; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1505125099399164


Tampere University

7. Pesu, Marko. Regulation of STAT6-mediated transcription in IL-4-induced signal transduction .

Degree: Lääketieteellisen teknologian instituutti - Institute of Medical Technology, 2003, Tampere University

 Sytokiinit ovat liukoisia välittäjäaineita, jotka säätelevät immuunijärjestelmän solujen erilaistumista, kasvua ja toimintaa. Sytokiinin sitoutuminen solukalvolla ilmentyvään reseptoriinsa saa aikaan solun sisäisten signalointireittien aktivaation, ja tämä… (more)

Subjects/Keywords: immunologia ; IL-4 ; STAT6 ; transkriptio ; immunology ; IL-4 ; STAT6 ; transcription

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pesu, M. (2003). Regulation of STAT6-mediated transcription in IL-4-induced signal transduction . (Doctoral Dissertation). Tampere University. Retrieved from https://trepo.tuni.fi/handle/10024/67292

Chicago Manual of Style (16th Edition):

Pesu, Marko. “Regulation of STAT6-mediated transcription in IL-4-induced signal transduction .” 2003. Doctoral Dissertation, Tampere University. Accessed April 16, 2021. https://trepo.tuni.fi/handle/10024/67292.

MLA Handbook (7th Edition):

Pesu, Marko. “Regulation of STAT6-mediated transcription in IL-4-induced signal transduction .” 2003. Web. 16 Apr 2021.

Vancouver:

Pesu M. Regulation of STAT6-mediated transcription in IL-4-induced signal transduction . [Internet] [Doctoral dissertation]. Tampere University; 2003. [cited 2021 Apr 16]. Available from: https://trepo.tuni.fi/handle/10024/67292.

Council of Science Editors:

Pesu M. Regulation of STAT6-mediated transcription in IL-4-induced signal transduction . [Doctoral Dissertation]. Tampere University; 2003. Available from: https://trepo.tuni.fi/handle/10024/67292

8. Gustavo Henrique Oliveira da Rocha. Influência da anexina A1 sobre a fagocitose e a expressão de receptor ativado por proliferador de peroxissomo gama em células da microglia.

Degree: 2017, University of São Paulo

A inflamação é fundamental para a manutenção da homeostasia e para a resposta do organismo à injúria. A resposta inflamatória deve ser adequada aos estímulos… (more)

Subjects/Keywords: Anexina A1; CD36; CREB; Fagocitose; Neurodegeneração; PPARγ;

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rocha, G. H. O. d. (2017). Influência da anexina A1 sobre a fagocitose e a expressão de receptor ativado por proliferador de peroxissomo gama em células da microglia. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/9/9141/tde-16032017-161518/

Chicago Manual of Style (16th Edition):

Rocha, Gustavo Henrique Oliveira da. “Influência da anexina A1 sobre a fagocitose e a expressão de receptor ativado por proliferador de peroxissomo gama em células da microglia.” 2017. Masters Thesis, University of São Paulo. Accessed April 16, 2021. http://www.teses.usp.br/teses/disponiveis/9/9141/tde-16032017-161518/.

MLA Handbook (7th Edition):

Rocha, Gustavo Henrique Oliveira da. “Influência da anexina A1 sobre a fagocitose e a expressão de receptor ativado por proliferador de peroxissomo gama em células da microglia.” 2017. Web. 16 Apr 2021.

Vancouver:

Rocha GHOd. Influência da anexina A1 sobre a fagocitose e a expressão de receptor ativado por proliferador de peroxissomo gama em células da microglia. [Internet] [Masters thesis]. University of São Paulo; 2017. [cited 2021 Apr 16]. Available from: http://www.teses.usp.br/teses/disponiveis/9/9141/tde-16032017-161518/.

Council of Science Editors:

Rocha GHOd. Influência da anexina A1 sobre a fagocitose e a expressão de receptor ativado por proliferador de peroxissomo gama em células da microglia. [Masters Thesis]. University of São Paulo; 2017. Available from: http://www.teses.usp.br/teses/disponiveis/9/9141/tde-16032017-161518/

9. Guiter, Chrystelle. Étude de la voie de signalisation IL-4/IL-13 dans les lymphomes B primitifs du médiastin : The challenges of governance of the Saloum estuary in the perspective of sustainable preservation of the heritage of the Saloum Delta Biosphere Reserve (RBDS in Senegal), wetland area of human-nature interface in degradation, in a context of global warming.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2008, Université Paris-Est

Les lymphomes B primitifs du médiastin (LBPMs) constituent une entité anatomo-clinique particulière au sein des lymphomes diffus à grandes cellules B. Les analyses du transcriptome… (more)

Subjects/Keywords: STAT6; Interleukine 4; Interleukine 13; Lymphome B; Médiastin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guiter, C. (2008). Étude de la voie de signalisation IL-4/IL-13 dans les lymphomes B primitifs du médiastin : The challenges of governance of the Saloum estuary in the perspective of sustainable preservation of the heritage of the Saloum Delta Biosphere Reserve (RBDS in Senegal), wetland area of human-nature interface in degradation, in a context of global warming. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2008PEST0053

Chicago Manual of Style (16th Edition):

Guiter, Chrystelle. “Étude de la voie de signalisation IL-4/IL-13 dans les lymphomes B primitifs du médiastin : The challenges of governance of the Saloum estuary in the perspective of sustainable preservation of the heritage of the Saloum Delta Biosphere Reserve (RBDS in Senegal), wetland area of human-nature interface in degradation, in a context of global warming.” 2008. Doctoral Dissertation, Université Paris-Est. Accessed April 16, 2021. http://www.theses.fr/2008PEST0053.

MLA Handbook (7th Edition):

Guiter, Chrystelle. “Étude de la voie de signalisation IL-4/IL-13 dans les lymphomes B primitifs du médiastin : The challenges of governance of the Saloum estuary in the perspective of sustainable preservation of the heritage of the Saloum Delta Biosphere Reserve (RBDS in Senegal), wetland area of human-nature interface in degradation, in a context of global warming.” 2008. Web. 16 Apr 2021.

Vancouver:

Guiter C. Étude de la voie de signalisation IL-4/IL-13 dans les lymphomes B primitifs du médiastin : The challenges of governance of the Saloum estuary in the perspective of sustainable preservation of the heritage of the Saloum Delta Biosphere Reserve (RBDS in Senegal), wetland area of human-nature interface in degradation, in a context of global warming. [Internet] [Doctoral dissertation]. Université Paris-Est; 2008. [cited 2021 Apr 16]. Available from: http://www.theses.fr/2008PEST0053.

Council of Science Editors:

Guiter C. Étude de la voie de signalisation IL-4/IL-13 dans les lymphomes B primitifs du médiastin : The challenges of governance of the Saloum estuary in the perspective of sustainable preservation of the heritage of the Saloum Delta Biosphere Reserve (RBDS in Senegal), wetland area of human-nature interface in degradation, in a context of global warming. [Doctoral Dissertation]. Université Paris-Est; 2008. Available from: http://www.theses.fr/2008PEST0053

10. Afi Leslie, K. Signal Transducer and Activator of Transcription 6 (STAT6) as a regulator of pancreatic beta cell health.

Degree: PhD, 2019, University of Exeter

 The incidence of type 1 diabetes (T1DM) is increasing annually and the disease pathophysiology remains to be completely understood. The current understanding suggests a complex… (more)

Subjects/Keywords: STAT6; Beta cell; IL-13; Cytokines

…80 1.4.2 The role of IL-13 and Jak/STAT6 in pancreatic beta cell health… …138 Chapter 4.0: Characterisation of the IL-13 Jak/STAT6 Signalling Pathway in beta cells… …145 4.0 Characterisation of the IL-13 Jak/STAT6 Signalling Pathway in beta cells… …153 9 4.3.3 IL-13 and IL-4 both activate STAT6… …160 4.3.4 Jak2 knockdown reduced STAT6 phosphorylation… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Afi Leslie, K. (2019). Signal Transducer and Activator of Transcription 6 (STAT6) as a regulator of pancreatic beta cell health. (Doctoral Dissertation). University of Exeter. Retrieved from http://hdl.handle.net/10871/37361

Chicago Manual of Style (16th Edition):

Afi Leslie, K. “Signal Transducer and Activator of Transcription 6 (STAT6) as a regulator of pancreatic beta cell health.” 2019. Doctoral Dissertation, University of Exeter. Accessed April 16, 2021. http://hdl.handle.net/10871/37361.

MLA Handbook (7th Edition):

Afi Leslie, K. “Signal Transducer and Activator of Transcription 6 (STAT6) as a regulator of pancreatic beta cell health.” 2019. Web. 16 Apr 2021.

Vancouver:

Afi Leslie K. Signal Transducer and Activator of Transcription 6 (STAT6) as a regulator of pancreatic beta cell health. [Internet] [Doctoral dissertation]. University of Exeter; 2019. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/10871/37361.

Council of Science Editors:

Afi Leslie K. Signal Transducer and Activator of Transcription 6 (STAT6) as a regulator of pancreatic beta cell health. [Doctoral Dissertation]. University of Exeter; 2019. Available from: http://hdl.handle.net/10871/37361


Tampere University

11. Välineva, Tuuli. Molecular mechanisms of IL-4 induced transcription: Characterization of coregulatory proteins of STAT6 .

Degree: Lääketieteellisen teknologian instituutti - Institute of Medical Technology, 2007, Tampere University

 Sytokiinit ovat immuunijärjestelmän solujen tuottamia liukoisia välittäjäaineita, jotka säätelevät hematopoeettisten solujen toimintaa. Sytokiinien sitoutuminen spesifisiin reseptoreihinsa kohdesolujen solukalvolla aikaansaa JAK-STAT-signalointireitin aktivoitumisen, ja vaikuttaa useiden geenien… (more)

Subjects/Keywords: STAT6 ; interleukiini-4 ; transkriptio ; interleukin-4 ; transcription

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Välineva, T. (2007). Molecular mechanisms of IL-4 induced transcription: Characterization of coregulatory proteins of STAT6 . (Doctoral Dissertation). Tampere University. Retrieved from https://trepo.tuni.fi/handle/10024/67798

Chicago Manual of Style (16th Edition):

Välineva, Tuuli. “Molecular mechanisms of IL-4 induced transcription: Characterization of coregulatory proteins of STAT6 .” 2007. Doctoral Dissertation, Tampere University. Accessed April 16, 2021. https://trepo.tuni.fi/handle/10024/67798.

MLA Handbook (7th Edition):

Välineva, Tuuli. “Molecular mechanisms of IL-4 induced transcription: Characterization of coregulatory proteins of STAT6 .” 2007. Web. 16 Apr 2021.

Vancouver:

Välineva T. Molecular mechanisms of IL-4 induced transcription: Characterization of coregulatory proteins of STAT6 . [Internet] [Doctoral dissertation]. Tampere University; 2007. [cited 2021 Apr 16]. Available from: https://trepo.tuni.fi/handle/10024/67798.

Council of Science Editors:

Välineva T. Molecular mechanisms of IL-4 induced transcription: Characterization of coregulatory proteins of STAT6 . [Doctoral Dissertation]. Tampere University; 2007. Available from: https://trepo.tuni.fi/handle/10024/67798


North Carolina State University

12. Comfort, Kristen Krupa. Intracellular Signaling Networks in the Immune Response: Pathways Activated by Interleukin-2 and Interleukin-4 Receptors and their Roles in T Cell Proliferation.

Degree: MS, Chemical Engineering, 2006, North Carolina State University

 Cells sense and respond to chemical and physical stimuli through signal transduction pathways, which mediate cell proliferation, differentiation, migration, and survival. The cytokines interleukin-2 (IL-2)… (more)

Subjects/Keywords: Akt; Interleukin-4; STAT6; Interleukin-2; T cell proliferation; signal transduction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Comfort, K. K. (2006). Intracellular Signaling Networks in the Immune Response: Pathways Activated by Interleukin-2 and Interleukin-4 Receptors and their Roles in T Cell Proliferation. (Thesis). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/2057

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Comfort, Kristen Krupa. “Intracellular Signaling Networks in the Immune Response: Pathways Activated by Interleukin-2 and Interleukin-4 Receptors and their Roles in T Cell Proliferation.” 2006. Thesis, North Carolina State University. Accessed April 16, 2021. http://www.lib.ncsu.edu/resolver/1840.16/2057.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Comfort, Kristen Krupa. “Intracellular Signaling Networks in the Immune Response: Pathways Activated by Interleukin-2 and Interleukin-4 Receptors and their Roles in T Cell Proliferation.” 2006. Web. 16 Apr 2021.

Vancouver:

Comfort KK. Intracellular Signaling Networks in the Immune Response: Pathways Activated by Interleukin-2 and Interleukin-4 Receptors and their Roles in T Cell Proliferation. [Internet] [Thesis]. North Carolina State University; 2006. [cited 2021 Apr 16]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/2057.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Comfort KK. Intracellular Signaling Networks in the Immune Response: Pathways Activated by Interleukin-2 and Interleukin-4 Receptors and their Roles in T Cell Proliferation. [Thesis]. North Carolina State University; 2006. Available from: http://www.lib.ncsu.edu/resolver/1840.16/2057

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

13. Knott, Michelle Louise. Host-parasite interactions in primary and secondary infections with Nippostrongylus brasiliensis and Heligmosomoides bakeri.

Degree: 2010, University of Adelaide

 Parasitic helminth infections are a significant problem worldwide. Some helminth species are becoming resistant to current therapies and new forms of treatment and/or vaccines are… (more)

Subjects/Keywords: IL-5; eosinophic parasite; helminth; primary and secondary infections; Nippostrongylus brasiliensis; Heligmosomoides bakeri; STAT6; IL-4Ra; IL-13; eotaxin; FVB/N mouse

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Knott, M. L. (2010). Host-parasite interactions in primary and secondary infections with Nippostrongylus brasiliensis and Heligmosomoides bakeri. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/67240

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Knott, Michelle Louise. “Host-parasite interactions in primary and secondary infections with Nippostrongylus brasiliensis and Heligmosomoides bakeri.” 2010. Thesis, University of Adelaide. Accessed April 16, 2021. http://hdl.handle.net/2440/67240.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Knott, Michelle Louise. “Host-parasite interactions in primary and secondary infections with Nippostrongylus brasiliensis and Heligmosomoides bakeri.” 2010. Web. 16 Apr 2021.

Vancouver:

Knott ML. Host-parasite interactions in primary and secondary infections with Nippostrongylus brasiliensis and Heligmosomoides bakeri. [Internet] [Thesis]. University of Adelaide; 2010. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/2440/67240.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Knott ML. Host-parasite interactions in primary and secondary infections with Nippostrongylus brasiliensis and Heligmosomoides bakeri. [Thesis]. University of Adelaide; 2010. Available from: http://hdl.handle.net/2440/67240

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

14. Jargosch, Manja. The transcription factor IRF8 regulates Th1 and Treg differentiation.

Degree: 2016, Freie Universität Berlin

 Within the past years data integration in terms of integrative network analysis was successfully used to describe new interrelations within biological networks on a molecular… (more)

Subjects/Keywords: T helper cells; Th1; Th2; Treg; differentiation; transcription factors; RNA seq; data integration; STAT4; STAT6; IRF8; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::570 Biowissenschaften; Biologie

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jargosch, M. (2016). The transcription factor IRF8 regulates Th1 and Treg differentiation. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-8215

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jargosch, Manja. “The transcription factor IRF8 regulates Th1 and Treg differentiation.” 2016. Thesis, Freie Universität Berlin. Accessed April 16, 2021. http://dx.doi.org/10.17169/refubium-8215.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jargosch, Manja. “The transcription factor IRF8 regulates Th1 and Treg differentiation.” 2016. Web. 16 Apr 2021.

Vancouver:

Jargosch M. The transcription factor IRF8 regulates Th1 and Treg differentiation. [Internet] [Thesis]. Freie Universität Berlin; 2016. [cited 2021 Apr 16]. Available from: http://dx.doi.org/10.17169/refubium-8215.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jargosch M. The transcription factor IRF8 regulates Th1 and Treg differentiation. [Thesis]. Freie Universität Berlin; 2016. Available from: http://dx.doi.org/10.17169/refubium-8215

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New Mexico

15. De Haro, Sergio A. Immune regulation of the host autophagic response and the elimination of Mycobacterium tuberculosis.

Degree: Biomedical Sciences Graduate Program, 2009, University of New Mexico

 Autophagy is an immune effector response against a variety of intracellular pathogens, including Mycobacterium tuberculosis. Mycobacteria survive in macrophages by blocking maturation of the phagosome.… (more)

Subjects/Keywords: autophagy; tuberculosis; Akt; STAT6; IL-4; IL-13; macrophage; phagosome maturation; IFN (gamma)

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

De Haro, S. A. (2009). Immune regulation of the host autophagic response and the elimination of Mycobacterium tuberculosis. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/2

Chicago Manual of Style (16th Edition):

De Haro, Sergio A. “Immune regulation of the host autophagic response and the elimination of Mycobacterium tuberculosis.” 2009. Doctoral Dissertation, University of New Mexico. Accessed April 16, 2021. https://digitalrepository.unm.edu/biom_etds/2.

MLA Handbook (7th Edition):

De Haro, Sergio A. “Immune regulation of the host autophagic response and the elimination of Mycobacterium tuberculosis.” 2009. Web. 16 Apr 2021.

Vancouver:

De Haro SA. Immune regulation of the host autophagic response and the elimination of Mycobacterium tuberculosis. [Internet] [Doctoral dissertation]. University of New Mexico; 2009. [cited 2021 Apr 16]. Available from: https://digitalrepository.unm.edu/biom_etds/2.

Council of Science Editors:

De Haro SA. Immune regulation of the host autophagic response and the elimination of Mycobacterium tuberculosis. [Doctoral Dissertation]. University of New Mexico; 2009. Available from: https://digitalrepository.unm.edu/biom_etds/2


Kent State University

16. Shivakumar, Vasanth. Regulation of STAT6, STAT3 and STAT1 by the Cytoplasmic Tail of Polycystin-1, the Protein Affected in Polycystic Kidney Disease.

Degree: PhD, College of Arts and Sciences / Department of Biological Sciences, 2007, Kent State University

 Autosomal Dominant Polycystic Kidney Disease a monogenic inherited human disease causing renal epithelial cells to proliferate forming fluid-filled cysts resulting in renal failure. Currently, no… (more)

Subjects/Keywords: POLYCYSTIN; CILIA; P100; STAT6; STAT3; STAT1; IL4; IFN; KIDNEY; POLYCYSTIC KIDNEY DISEASE

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shivakumar, V. (2007). Regulation of STAT6, STAT3 and STAT1 by the Cytoplasmic Tail of Polycystin-1, the Protein Affected in Polycystic Kidney Disease. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1176482324

Chicago Manual of Style (16th Edition):

Shivakumar, Vasanth. “Regulation of STAT6, STAT3 and STAT1 by the Cytoplasmic Tail of Polycystin-1, the Protein Affected in Polycystic Kidney Disease.” 2007. Doctoral Dissertation, Kent State University. Accessed April 16, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=kent1176482324.

MLA Handbook (7th Edition):

Shivakumar, Vasanth. “Regulation of STAT6, STAT3 and STAT1 by the Cytoplasmic Tail of Polycystin-1, the Protein Affected in Polycystic Kidney Disease.” 2007. Web. 16 Apr 2021.

Vancouver:

Shivakumar V. Regulation of STAT6, STAT3 and STAT1 by the Cytoplasmic Tail of Polycystin-1, the Protein Affected in Polycystic Kidney Disease. [Internet] [Doctoral dissertation]. Kent State University; 2007. [cited 2021 Apr 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1176482324.

Council of Science Editors:

Shivakumar V. Regulation of STAT6, STAT3 and STAT1 by the Cytoplasmic Tail of Polycystin-1, the Protein Affected in Polycystic Kidney Disease. [Doctoral Dissertation]. Kent State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1176482324

17. Morin, Florence. Etude des mécanismes fibro-inflammatoires au cours de la sclérodermie systémique : Hedgehog and NF-κB pathways at the heart of cutaneous homeostasis : contribution of the genetic and physiopathological characterization of two X-linked ectodermal dysplasias, Bazex-Dupré-Christol syndrome and Incontinentia Pigmenti.

Degree: Docteur es, Immunologie, 2016, Sorbonne Paris Cité

 La sclérodermie systémique (ScS) est une maladie auto-immune caractérisée par une fibrose cutanée et viscérale ainsi que par des anomalies microcirculatoires. Son origine multifactorielle et… (more)

Subjects/Keywords: Sclérodermie systémique; Maladie du greffon contre l’hôte chronique; Modèle murin; Inflammation; Récepteur à l’EGF; Erlotinib; STAT3; Wnt/β-caténine; AKT; Niclosamide; STAT6; KLF4; Macrophages; Léflunomide; Systemic sclerosis; Graft versus Host Disease; Mouse model; Inflammation; EGF receptor; Erlotinib; STAT3; Wnt/β-catenin; AKT; Niclosamide; STAT6; KLF4; Macrophages; Leflunomide; 616.544

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Morin, F. (2016). Etude des mécanismes fibro-inflammatoires au cours de la sclérodermie systémique : Hedgehog and NF-κB pathways at the heart of cutaneous homeostasis : contribution of the genetic and physiopathological characterization of two X-linked ectodermal dysplasias, Bazex-Dupré-Christol syndrome and Incontinentia Pigmenti. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2016USPCB077

Chicago Manual of Style (16th Edition):

Morin, Florence. “Etude des mécanismes fibro-inflammatoires au cours de la sclérodermie systémique : Hedgehog and NF-κB pathways at the heart of cutaneous homeostasis : contribution of the genetic and physiopathological characterization of two X-linked ectodermal dysplasias, Bazex-Dupré-Christol syndrome and Incontinentia Pigmenti.” 2016. Doctoral Dissertation, Sorbonne Paris Cité. Accessed April 16, 2021. http://www.theses.fr/2016USPCB077.

MLA Handbook (7th Edition):

Morin, Florence. “Etude des mécanismes fibro-inflammatoires au cours de la sclérodermie systémique : Hedgehog and NF-κB pathways at the heart of cutaneous homeostasis : contribution of the genetic and physiopathological characterization of two X-linked ectodermal dysplasias, Bazex-Dupré-Christol syndrome and Incontinentia Pigmenti.” 2016. Web. 16 Apr 2021.

Vancouver:

Morin F. Etude des mécanismes fibro-inflammatoires au cours de la sclérodermie systémique : Hedgehog and NF-κB pathways at the heart of cutaneous homeostasis : contribution of the genetic and physiopathological characterization of two X-linked ectodermal dysplasias, Bazex-Dupré-Christol syndrome and Incontinentia Pigmenti. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2016. [cited 2021 Apr 16]. Available from: http://www.theses.fr/2016USPCB077.

Council of Science Editors:

Morin F. Etude des mécanismes fibro-inflammatoires au cours de la sclérodermie systémique : Hedgehog and NF-κB pathways at the heart of cutaneous homeostasis : contribution of the genetic and physiopathological characterization of two X-linked ectodermal dysplasias, Bazex-Dupré-Christol syndrome and Incontinentia Pigmenti. [Doctoral Dissertation]. Sorbonne Paris Cité; 2016. Available from: http://www.theses.fr/2016USPCB077


Vanderbilt University

18. Bloodworth, Melissa Harintho. Regulation of Immune Responses during Airway Inflammation.

Degree: PhD, Microbiology and Immunology, 2017, Vanderbilt University

 Allergic asthma is refractory to corticosteroid treatment in up to 10% of patients and often leads to hospital admissions caused by respiratory viral and/ or… (more)

Subjects/Keywords: type 2 immunity (Th2); gamma-delta 17 (γδ17) cells; STAT6; Klebsiella pneumoniae; glucagon-like peptide 1 (GLP-1); respiratory syncytial virus (RSV); phenome-wide association study (PheWAS); regulatory T cells (Tregs); prostacyclin (PGI2)

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bloodworth, M. H. (2017). Regulation of Immune Responses during Airway Inflammation. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11246

Chicago Manual of Style (16th Edition):

Bloodworth, Melissa Harintho. “Regulation of Immune Responses during Airway Inflammation.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed April 16, 2021. http://hdl.handle.net/1803/11246.

MLA Handbook (7th Edition):

Bloodworth, Melissa Harintho. “Regulation of Immune Responses during Airway Inflammation.” 2017. Web. 16 Apr 2021.

Vancouver:

Bloodworth MH. Regulation of Immune Responses during Airway Inflammation. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1803/11246.

Council of Science Editors:

Bloodworth MH. Regulation of Immune Responses during Airway Inflammation. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/11246


University of Debrecen

19. Csipkés, Krisztina. STAT6 és EGR2 transzkripciós faktorok szerepe az alternatív makrofág polarizációban .

Degree: DE – Természettudományi és Technológiai Kar – Biotechnológiai Intézet, University of Debrecen

 A makrofágok rendkívül sokrétű és fontos szerepet játszanak az élő szervezetben, fiziológiás és különböző patológiás körülmények közt egyaránt. A molekuláris mikrokörnyezet nagymértékben befolyásolja a makrofágok… (more)

Subjects/Keywords: makrofág; alternatív; immunrendszer; transzkripció; transzkripciós faktor; QPCR; csontvelői eredetű makrofágok; EGR2; STAT6; interleukin-4; mRNS szintű kifejeződés; makrofág; polarizáció

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Csipkés, K. (n.d.). STAT6 és EGR2 transzkripciós faktorok szerepe az alternatív makrofág polarizációban . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/276226

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Csipkés, Krisztina. “STAT6 és EGR2 transzkripciós faktorok szerepe az alternatív makrofág polarizációban .” Thesis, University of Debrecen. Accessed April 16, 2021. http://hdl.handle.net/2437/276226.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Csipkés, Krisztina. “STAT6 és EGR2 transzkripciós faktorok szerepe az alternatív makrofág polarizációban .” Web. 16 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Csipkés K. STAT6 és EGR2 transzkripciós faktorok szerepe az alternatív makrofág polarizációban . [Internet] [Thesis]. University of Debrecen; [cited 2021 Apr 16]. Available from: http://hdl.handle.net/2437/276226.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Csipkés K. STAT6 és EGR2 transzkripciós faktorok szerepe az alternatív makrofág polarizációban . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/276226

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

20. Mandal, Debasmita. TUNING OF INTERLEUKIN-13 SIGNALING IN INTESTINAL EPITHELIAL CELLS VIA IL-13Rα2-INDUCED MAP KINASE ACTIVATION AND INDUCTION OF REACTIVE OXYGEN SPECIES.

Degree: PhD, Pathology, 2010, Case Western Reserve University School of Graduate Studies

 Interleukin-13 (IL-13) is a predominantly T helper 2 (Th2)-derived cytokine that has multiple effects on both haematopoietic and non-haematopoietic cells. Previous findings from our laboratory… (more)

Subjects/Keywords: Oncology; Colitis; colorectal cancer MAPK; STAT6; Reactive Oxygen species; NADPH Oxidase

…induced signaling through STAT6, but not through ERK, JNK, and p38 89 7 2.6 Low to medium… …expression of IL-5ĮIDFLOLWDWHV(5.DFWLYDWLRQ while high expression inhibits STAT6 activation… …phosphorylation of ERK 1/2 and STAT6 3.4 117 IL-13 generates ROS through NADPH oxidase, which… …positively regulates ERK and STAT6 phosphorylation 3.3 95 JAK1 and ERK activation are required… …13 does not activate the signal transducer and activator 6 (STAT6) or mitogen… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mandal, D. (2010). TUNING OF INTERLEUKIN-13 SIGNALING IN INTESTINAL EPITHELIAL CELLS VIA IL-13Rα2-INDUCED MAP KINASE ACTIVATION AND INDUCTION OF REACTIVE OXYGEN SPECIES. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1270237684

Chicago Manual of Style (16th Edition):

Mandal, Debasmita. “TUNING OF INTERLEUKIN-13 SIGNALING IN INTESTINAL EPITHELIAL CELLS VIA IL-13Rα2-INDUCED MAP KINASE ACTIVATION AND INDUCTION OF REACTIVE OXYGEN SPECIES.” 2010. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed April 16, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1270237684.

MLA Handbook (7th Edition):

Mandal, Debasmita. “TUNING OF INTERLEUKIN-13 SIGNALING IN INTESTINAL EPITHELIAL CELLS VIA IL-13Rα2-INDUCED MAP KINASE ACTIVATION AND INDUCTION OF REACTIVE OXYGEN SPECIES.” 2010. Web. 16 Apr 2021.

Vancouver:

Mandal D. TUNING OF INTERLEUKIN-13 SIGNALING IN INTESTINAL EPITHELIAL CELLS VIA IL-13Rα2-INDUCED MAP KINASE ACTIVATION AND INDUCTION OF REACTIVE OXYGEN SPECIES. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2010. [cited 2021 Apr 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1270237684.

Council of Science Editors:

Mandal D. TUNING OF INTERLEUKIN-13 SIGNALING IN INTESTINAL EPITHELIAL CELLS VIA IL-13Rα2-INDUCED MAP KINASE ACTIVATION AND INDUCTION OF REACTIVE OXYGEN SPECIES. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1270237684

21. Penninkhof, T.F.E. Interaction of REPS2 with NF-kappaB in Prostate Cancer Cells.

Degree: 2005, Erasmus University Medical Center

 textabstractLike normal prostate cells, prostate cancer cells are dependent on androgens for growth and survival, and prostate cancer can be treated by androgen ablation therapy.… (more)

Subjects/Keywords: NF-kappaB; REPS2; STAT6; TRAF4; apoptosis; cell survival; mammalian two-hybrid system; prostate cancer; protein-protein interaction; signal transduction; yeast two-hybrid system

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Penninkhof, T. F. E. (2005). Interaction of REPS2 with NF-kappaB in Prostate Cancer Cells. (Doctoral Dissertation). Erasmus University Medical Center. Retrieved from http://hdl.handle.net/1765/6905

Chicago Manual of Style (16th Edition):

Penninkhof, T F E. “Interaction of REPS2 with NF-kappaB in Prostate Cancer Cells.” 2005. Doctoral Dissertation, Erasmus University Medical Center. Accessed April 16, 2021. http://hdl.handle.net/1765/6905.

MLA Handbook (7th Edition):

Penninkhof, T F E. “Interaction of REPS2 with NF-kappaB in Prostate Cancer Cells.” 2005. Web. 16 Apr 2021.

Vancouver:

Penninkhof TFE. Interaction of REPS2 with NF-kappaB in Prostate Cancer Cells. [Internet] [Doctoral dissertation]. Erasmus University Medical Center; 2005. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1765/6905.

Council of Science Editors:

Penninkhof TFE. Interaction of REPS2 with NF-kappaB in Prostate Cancer Cells. [Doctoral Dissertation]. Erasmus University Medical Center; 2005. Available from: http://hdl.handle.net/1765/6905

22. Feldman, Scott. Molecular Studies of Class Switch Recombination in B Lymphocytes: Transcription, DNA Repair, and Looping.

Degree: 2012, University of Illinois – Chicago

 Mature B lymphocytes undergo effector function diversification by means of class switch recombination (CSR), in which there is a cytokine- and/or antigen-dependent switch from the… (more)

Subjects/Keywords: Immunology; B Lymphocytes; Chromatin; Genes, Immunoglobulin; Class Switch Recombination (CSR); Cytidine Deaminase (AID); DNA Double-strand Break (DSB) Repair; 53BP1; NF-KappaB; STAT6; Synaptosome; Chromatin Looping; Germline Transcripts (GLTs); Chromosome Conformation Capture (3C); Chromatin Immunoprecipitation (ChIP); Real-time PCR

…germline transcription are altered by the absence of the 1 promoter or STAT6. A) GLTs were… …of switched B cells in all experiments is shown, along with SEMs. Data for the Stat6 / mice… …after 4d of LPS or LPS+IL4 stimulation in WT and STAT6-deficient B cells in one experiment to… …show the lack of repression of µ ! 3 switching in the absence of STAT6. The total number… …x28;N) of samples used in all experiments can be found in Table VII. . . . 57 STAT6… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Feldman, S. (2012). Molecular Studies of Class Switch Recombination in B Lymphocytes: Transcription, DNA Repair, and Looping. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/9184

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Feldman, Scott. “Molecular Studies of Class Switch Recombination in B Lymphocytes: Transcription, DNA Repair, and Looping.” 2012. Thesis, University of Illinois – Chicago. Accessed April 16, 2021. http://hdl.handle.net/10027/9184.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Feldman, Scott. “Molecular Studies of Class Switch Recombination in B Lymphocytes: Transcription, DNA Repair, and Looping.” 2012. Web. 16 Apr 2021.

Vancouver:

Feldman S. Molecular Studies of Class Switch Recombination in B Lymphocytes: Transcription, DNA Repair, and Looping. [Internet] [Thesis]. University of Illinois – Chicago; 2012. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/10027/9184.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Feldman S. Molecular Studies of Class Switch Recombination in B Lymphocytes: Transcription, DNA Repair, and Looping. [Thesis]. University of Illinois – Chicago; 2012. Available from: http://hdl.handle.net/10027/9184

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.