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You searched for subject:(Reperfusion). Showing records 1 – 30 of 811 total matches.

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1. Olland, Anne. Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation.

Degree: Docteur es, Sciences de la vie et de la santé, 2016, Université de Strasbourg

L’ischémie reperfusion pulmonaire et sa traduction clinique la dysfonction primaire du greffon sont responsables d’une morbi-mortalité importante en transplantation pulmonaire aussi bien à court terme… (more)

Subjects/Keywords: Ischémie reperfusion; Reperfusion ex vivo; Microparticules; Ischemia reperfusion; Ex vivo reperfusion; Microparticles; 571.96; 617.9

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APA (6th Edition):

Olland, A. (2016). Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2016STRAJ035

Chicago Manual of Style (16th Edition):

Olland, Anne. “Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation.” 2016. Doctoral Dissertation, Université de Strasbourg. Accessed March 07, 2021. http://www.theses.fr/2016STRAJ035.

MLA Handbook (7th Edition):

Olland, Anne. “Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation.” 2016. Web. 07 Mar 2021.

Vancouver:

Olland A. Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2016. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2016STRAJ035.

Council of Science Editors:

Olland A. Intérêt des microparticules pour l'étude de l'ischémie reperfusion en tranplantation pulmonaire basé sur un modèle de perfusion ventilation pulmonaire ex vivo chez le rat : Relevance of microparticles for the study of ischemia reperfusion in lung transplantation using an experimental model of ex vivo rat lung reperfusion and ventilation. [Doctoral Dissertation]. Université de Strasbourg; 2016. Available from: http://www.theses.fr/2016STRAJ035


Universiteit Utrecht

2. Soyal, M. Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target.

Degree: 2012, Universiteit Utrecht

 Acute myocardial infarction (AMI) is a major cause of morbidity. AMI is caused by a blockage of one or multiple coronary arteries, leading to an… (more)

Subjects/Keywords: AMI; FOXO; ischemia/reperfusion

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APA (6th Edition):

Soyal, M. (2012). Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/254301

Chicago Manual of Style (16th Edition):

Soyal, M. “Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target.” 2012. Masters Thesis, Universiteit Utrecht. Accessed March 07, 2021. http://dspace.library.uu.nl:8080/handle/1874/254301.

MLA Handbook (7th Edition):

Soyal, M. “Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target.” 2012. Web. 07 Mar 2021.

Vancouver:

Soyal M. Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2021 Mar 07]. Available from: http://dspace.library.uu.nl:8080/handle/1874/254301.

Council of Science Editors:

Soyal M. Forkhead Box O in ischemia/reperfusion injury: a potential therapeutic target. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/254301


University of Cambridge

3. Allen, Fay Marie. Mitochondrial metabolism elucidated by rapid fractionation from tissue.

Degree: PhD, 2020, University of Cambridge

 Mitochondria are metabolic hubs, with many diseases found to have altered metabolism and mitochondrial dysfunction, such as ischaemia-reperfusion injury. A detailed understanding of the metabolic… (more)

Subjects/Keywords: Mitochondria; Metabolism; Ischaemia-Reperfusion Injury

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APA (6th Edition):

Allen, F. M. (2020). Mitochondrial metabolism elucidated by rapid fractionation from tissue. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.49510 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801797

Chicago Manual of Style (16th Edition):

Allen, Fay Marie. “Mitochondrial metabolism elucidated by rapid fractionation from tissue.” 2020. Doctoral Dissertation, University of Cambridge. Accessed March 07, 2021. https://doi.org/10.17863/CAM.49510 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801797.

MLA Handbook (7th Edition):

Allen, Fay Marie. “Mitochondrial metabolism elucidated by rapid fractionation from tissue.” 2020. Web. 07 Mar 2021.

Vancouver:

Allen FM. Mitochondrial metabolism elucidated by rapid fractionation from tissue. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2021 Mar 07]. Available from: https://doi.org/10.17863/CAM.49510 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801797.

Council of Science Editors:

Allen FM. Mitochondrial metabolism elucidated by rapid fractionation from tissue. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://doi.org/10.17863/CAM.49510 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.801797


University of Cambridge

4. Martin, Jack. The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation.

Degree: PhD, 2019, University of Cambridge

 Summary The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation Ischaemia and subsequent reperfusion is inherent to solid organ transplantation and contributes to… (more)

Subjects/Keywords: Organ transplantation; Mitochondria; Ischaemia-Reperfusion

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APA (6th Edition):

Martin, J. (2019). The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/294351

Chicago Manual of Style (16th Edition):

Martin, Jack. “The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation.” 2019. Doctoral Dissertation, University of Cambridge. Accessed March 07, 2021. https://www.repository.cam.ac.uk/handle/1810/294351.

MLA Handbook (7th Edition):

Martin, Jack. “The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation.” 2019. Web. 07 Mar 2021.

Vancouver:

Martin J. The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Mar 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/294351.

Council of Science Editors:

Martin J. The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/294351


University of Cambridge

5. Allen, Fay Marie. Mitochondrial Metabolism Elucidated by Rapid Fractionation from Tissue.

Degree: PhD, 2020, University of Cambridge

 Mitochondria are metabolic hubs, with many diseases found to have altered metabolism and mitochondrial dysfunction, such as ischaemia-reperfusion injury. A detailed understanding of the metabolic… (more)

Subjects/Keywords: Mitochondria; Metabolism; Ischaemia-Reperfusion Injury

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APA (6th Edition):

Allen, F. M. (2020). Mitochondrial Metabolism Elucidated by Rapid Fractionation from Tissue. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/302439

Chicago Manual of Style (16th Edition):

Allen, Fay Marie. “Mitochondrial Metabolism Elucidated by Rapid Fractionation from Tissue.” 2020. Doctoral Dissertation, University of Cambridge. Accessed March 07, 2021. https://www.repository.cam.ac.uk/handle/1810/302439.

MLA Handbook (7th Edition):

Allen, Fay Marie. “Mitochondrial Metabolism Elucidated by Rapid Fractionation from Tissue.” 2020. Web. 07 Mar 2021.

Vancouver:

Allen FM. Mitochondrial Metabolism Elucidated by Rapid Fractionation from Tissue. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2021 Mar 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/302439.

Council of Science Editors:

Allen FM. Mitochondrial Metabolism Elucidated by Rapid Fractionation from Tissue. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/302439


University of Melbourne

6. Thacker, Michelle Anne. Structural damage to the enteric nervous system following ischemia / reperfusion injury.

Degree: 2012, University of Melbourne

 Ischemia reperfusion (I/R) injury can occur in a number of organs, however the intestine is possibly the most susceptible to I/R damage. Intestinal I/R injury… (more)

Subjects/Keywords: enteric glia; ischemia reperfusion injury

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APA (6th Edition):

Thacker, M. A. (2012). Structural damage to the enteric nervous system following ischemia / reperfusion injury. (Masters Thesis). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37853

Chicago Manual of Style (16th Edition):

Thacker, Michelle Anne. “Structural damage to the enteric nervous system following ischemia / reperfusion injury.” 2012. Masters Thesis, University of Melbourne. Accessed March 07, 2021. http://hdl.handle.net/11343/37853.

MLA Handbook (7th Edition):

Thacker, Michelle Anne. “Structural damage to the enteric nervous system following ischemia / reperfusion injury.” 2012. Web. 07 Mar 2021.

Vancouver:

Thacker MA. Structural damage to the enteric nervous system following ischemia / reperfusion injury. [Internet] [Masters thesis]. University of Melbourne; 2012. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/11343/37853.

Council of Science Editors:

Thacker MA. Structural damage to the enteric nervous system following ischemia / reperfusion injury. [Masters Thesis]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37853


University of Cambridge

7. Martin, Jack. The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation.

Degree: PhD, 2019, University of Cambridge

 The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation Ischaemia and subsequent reperfusion is inherent to solid organ transplantation and contributes to tissue… (more)

Subjects/Keywords: Organ transplantation; Mitochondria; Ischaemia-Reperfusion

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APA (6th Edition):

Martin, J. (2019). The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.41449 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782895

Chicago Manual of Style (16th Edition):

Martin, Jack. “The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation.” 2019. Doctoral Dissertation, University of Cambridge. Accessed March 07, 2021. https://doi.org/10.17863/CAM.41449 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782895.

MLA Handbook (7th Edition):

Martin, Jack. “The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation.” 2019. Web. 07 Mar 2021.

Vancouver:

Martin J. The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Mar 07]. Available from: https://doi.org/10.17863/CAM.41449 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782895.

Council of Science Editors:

Martin J. The role of ischaemia-reperfusion injury and mitochondrial dysfunction in organ transplantation. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://doi.org/10.17863/CAM.41449 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782895

8. Hoang, Quoc thang. Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie. Biothérapies, 2016, Sorbonne Paris Cité

Introduction: La lésion d'ischémie - reperfusion (I/R) intestinale est associée à une mortalité élevée.Le rôle de la transmigration des neutrophiles est probablement essentiel dans le… (more)

Subjects/Keywords: Ischémie - reperfusion mésentérique; P8RI; Mesenteric ischemia - reperfusion; P8RI

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APA (6th Edition):

Hoang, Q. t. (2016). Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2016USPCC196

Chicago Manual of Style (16th Edition):

Hoang, Quoc thang. “Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model.” 2016. Doctoral Dissertation, Sorbonne Paris Cité. Accessed March 07, 2021. http://www.theses.fr/2016USPCC196.

MLA Handbook (7th Edition):

Hoang, Quoc thang. “Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model.” 2016. Web. 07 Mar 2021.

Vancouver:

Hoang Qt. Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2016. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2016USPCC196.

Council of Science Editors:

Hoang Qt. Aspects diagnostiques et thérapeutiques du modèle d'ischémie - reperfusion mésentérique : Diagnostic and treatment aspects of mesenteric ischemia - reperfusion model. [Doctoral Dissertation]. Sorbonne Paris Cité; 2016. Available from: http://www.theses.fr/2016USPCC196

9. Sportouch-Dukhan, Catherine. Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation.

Degree: Docteur es, Biochimie et biologie moléculaire, 2012, Université Montpellier I

L'infarctus du myocarde est la première cause de mortalité cardiovasculaire dans les pays occidentaux. La reperfusion la plus précoce possible est actuellement le seul traitement… (more)

Subjects/Keywords: Cardioprotection; Infarctus; Ischémie-reperfusion; Échocardiographie; Cardioprotection; Infarction; Ischemia-reperfusion; Echocardiography

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APA (6th Edition):

Sportouch-Dukhan, C. (2012). Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2012MON1T021

Chicago Manual of Style (16th Edition):

Sportouch-Dukhan, Catherine. “Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation.” 2012. Doctoral Dissertation, Université Montpellier I. Accessed March 07, 2021. http://www.theses.fr/2012MON1T021.

MLA Handbook (7th Edition):

Sportouch-Dukhan, Catherine. “Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation.” 2012. Web. 07 Mar 2021.

Vancouver:

Sportouch-Dukhan C. Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation. [Internet] [Doctoral dissertation]. Université Montpellier I; 2012. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2012MON1T021.

Council of Science Editors:

Sportouch-Dukhan C. Effets cardioprotecteurs du glutamate contre les lésions d'ischémie-reperfusion myocardique chez la souris. Evaluation échocardiographique. : Cardioprotective effects of glutamate against ischemia-reperfusion injury in the mouse heart. Echocardiographic evaluation. [Doctoral Dissertation]. Université Montpellier I; 2012. Available from: http://www.theses.fr/2012MON1T021

10. Chevallier, Stéphane. Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing.

Degree: Docteur es, Pharmacologie, 2011, Reims

La maladie cardiovasculaire la plus courante dans les pays industrialisés est la maladie coronarienne responsable d’une ischémie du tissu cardiaque pouvant conduire à l’infarctus du… (more)

Subjects/Keywords: Ischémie; Reperfusion; Elastine; Lactosylcéramides; Ischemia; Reperfusion; Elastin; Lactosylceramides

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APA (6th Edition):

Chevallier, S. (2011). Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2011REIMM203

Chicago Manual of Style (16th Edition):

Chevallier, Stéphane. “Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing.” 2011. Doctoral Dissertation, Reims. Accessed March 07, 2021. http://www.theses.fr/2011REIMM203.

MLA Handbook (7th Edition):

Chevallier, Stéphane. “Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing.” 2011. Web. 07 Mar 2021.

Vancouver:

Chevallier S. Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing. [Internet] [Doctoral dissertation]. Reims; 2011. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2011REIMM203.

Council of Science Editors:

Chevallier S. Elastokines et Lactosylcéramide : cardioprotection et vieillissement : Elastokines et Lactosylceramide : cardioprotection et Ageing. [Doctoral Dissertation]. Reims; 2011. Available from: http://www.theses.fr/2011REIMM203

11. Paradis, Stephanie. Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol.

Degree: Docteur es, Pharmacologie et Biothérapies, 2012, Université Paris-Est

Dans ce travail, nous avons montré qu'un nouveau ligand du TSPO, le TRO40303, possède des propriétés cardioprotectrices, confirmant que le TSPO joue un rôle important… (more)

Subjects/Keywords: Cholestérol; Ischémie reperfusion; Mitochondrie; Cardioprotection; Cholesterol; Ischemia reperfusion; Mitochondria; Cardioprotection

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APA (6th Edition):

Paradis, S. (2012). Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2012PEST0071

Chicago Manual of Style (16th Edition):

Paradis, Stephanie. “Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol.” 2012. Doctoral Dissertation, Université Paris-Est. Accessed March 07, 2021. http://www.theses.fr/2012PEST0071.

MLA Handbook (7th Edition):

Paradis, Stephanie. “Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol.” 2012. Web. 07 Mar 2021.

Vancouver:

Paradis S. Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol. [Internet] [Doctoral dissertation]. Université Paris-Est; 2012. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2012PEST0071.

Council of Science Editors:

Paradis S. Effet cardioprotecteur des ligands de la protéine translocatrice mitochondriale (TSPO) au cours de l'ischémie-reperfusion myocardique expérimentale : rôle du cholestérol. : Cardioprotective effect of mitochondrial translocator protein (TSPO) ligands during experimental myocardial ischemia-reperfusion : role of cholesterol. [Doctoral Dissertation]. Université Paris-Est; 2012. Available from: http://www.theses.fr/2012PEST0071

12. Noll, Éric. Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring.

Degree: Docteur es, Recherche clinique et innovation technologique, 2016, Université de Strasbourg

Les objectifs de ce travail expérimental étaient : 1. Évaluer la place de la lactatémie capillaire comme dispositif de monitorage de l’ischémie et de la… (more)

Subjects/Keywords: Ischémie-Reperfusion; Lactate; Ischemia-Reperfusion; Lactate; 571.5; 616.13

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APA (6th Edition):

Noll, . (2016). Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2016STRAJ006

Chicago Manual of Style (16th Edition):

Noll, Éric. “Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring.” 2016. Doctoral Dissertation, Université de Strasbourg. Accessed March 07, 2021. http://www.theses.fr/2016STRAJ006.

MLA Handbook (7th Edition):

Noll, Éric. “Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring.” 2016. Web. 07 Mar 2021.

Vancouver:

Noll . Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2016. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2016STRAJ006.

Council of Science Editors:

Noll . Ischémie-reperfusion musculaire squelettique expérimentale : place de la lactatémie capillaire dans le monitorage de la reperfusion et transposition au modèle intestinal : Experimental skeletal muscle ischemia-reperfusion : capillary lactate for reperfusion monitoring. [Doctoral Dissertation]. Université de Strasbourg; 2016. Available from: http://www.theses.fr/2016STRAJ006


University of Hong Kong

13. 毛晓雯. Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury.

Degree: 2013, University of Hong Kong

 Coronary artery disease limits myocardial blood flow and results in myocardial infarction. Reperfusion therapies restore coronary flow, but may also cause myocardial ischemia reperfusion injury… (more)

Subjects/Keywords: Myocardial reperfusion; Reperfusion injury

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APA (6th Edition):

毛晓雯. (2013). Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/193463

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

毛晓雯. “Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury.” 2013. Thesis, University of Hong Kong. Accessed March 07, 2021. http://hdl.handle.net/10722/193463.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

毛晓雯. “Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury.” 2013. Web. 07 Mar 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

毛晓雯. Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. [Internet] [Thesis]. University of Hong Kong; 2013. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10722/193463.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

毛晓雯. Peroxynitrite/Ho-1 interaction in propofol post-conditioning protection against myocardial ischemia reperfusion injury. [Thesis]. University of Hong Kong; 2013. Available from: http://hdl.handle.net/10722/193463

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

14. Feliu, Catherine. Implication des récepteurs P1 et P2 dans la protection des cellules endothéliales au cours de l’hypoxie-reoxygenation : Involvement of P1 and P2 receptors in the protection of endothelial cells during hypoxia-reoxygenation.

Degree: Docteur es, Médecine, 2019, Reims

Au cours de l’ischémie cardiaque, la lésion est initiée au niveau de l’endothélium, et progresse aux cardiomyocytes environnants. La signalisation purinergique joue un rôle important… (more)

Subjects/Keywords: Cardioprotection; Cellules endothéliales; Ischémie/reperfusion; Ischemia/reperfusion; Endothelial cells; Cardioprotection

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Feliu, C. (2019). Implication des récepteurs P1 et P2 dans la protection des cellules endothéliales au cours de l’hypoxie-reoxygenation : Involvement of P1 and P2 receptors in the protection of endothelial cells during hypoxia-reoxygenation. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2019REIMM202

Chicago Manual of Style (16th Edition):

Feliu, Catherine. “Implication des récepteurs P1 et P2 dans la protection des cellules endothéliales au cours de l’hypoxie-reoxygenation : Involvement of P1 and P2 receptors in the protection of endothelial cells during hypoxia-reoxygenation.” 2019. Doctoral Dissertation, Reims. Accessed March 07, 2021. http://www.theses.fr/2019REIMM202.

MLA Handbook (7th Edition):

Feliu, Catherine. “Implication des récepteurs P1 et P2 dans la protection des cellules endothéliales au cours de l’hypoxie-reoxygenation : Involvement of P1 and P2 receptors in the protection of endothelial cells during hypoxia-reoxygenation.” 2019. Web. 07 Mar 2021.

Vancouver:

Feliu C. Implication des récepteurs P1 et P2 dans la protection des cellules endothéliales au cours de l’hypoxie-reoxygenation : Involvement of P1 and P2 receptors in the protection of endothelial cells during hypoxia-reoxygenation. [Internet] [Doctoral dissertation]. Reims; 2019. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2019REIMM202.

Council of Science Editors:

Feliu C. Implication des récepteurs P1 et P2 dans la protection des cellules endothéliales au cours de l’hypoxie-reoxygenation : Involvement of P1 and P2 receptors in the protection of endothelial cells during hypoxia-reoxygenation. [Doctoral Dissertation]. Reims; 2019. Available from: http://www.theses.fr/2019REIMM202


University of Florida

15. Kirkby, Kristin. The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury.

Degree: PhD, Veterinary Medical Sciences - Veterinary Medicine, 2012, University of Florida

 Intestinal ischemia reperfusion injury (IRI) in animals andhumans is associated with mortality rates between 50-80%. Various pharmacologicagents have been studied as potential treatments for IRI;… (more)

Subjects/Keywords: Dosage; Ischemia; Laser therapy; Lasers; Lungs; Oxygen; Physical trauma; Rats; Reperfusion; Reperfusion injury; injury  – intestine  – ischemia  – laser  – lung  – rat  – reperfusion  – therapy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kirkby, K. (2012). The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury. (Doctoral Dissertation). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0044834

Chicago Manual of Style (16th Edition):

Kirkby, Kristin. “The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury.” 2012. Doctoral Dissertation, University of Florida. Accessed March 07, 2021. https://ufdc.ufl.edu/UFE0044834.

MLA Handbook (7th Edition):

Kirkby, Kristin. “The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury.” 2012. Web. 07 Mar 2021.

Vancouver:

Kirkby K. The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury. [Internet] [Doctoral dissertation]. University of Florida; 2012. [cited 2021 Mar 07]. Available from: https://ufdc.ufl.edu/UFE0044834.

Council of Science Editors:

Kirkby K. The Effects of Low Level Laser Therapy on Intestinal Ischemia Reperfusion Injury. [Doctoral Dissertation]. University of Florida; 2012. Available from: https://ufdc.ufl.edu/UFE0044834


Ruhr Universität Bochum

16. Appukuttan, Avinash. Role of type 10 "soluble" adenylyl cyclase in regulation of apoptosis : relevance to cardiovascular diseases.

Degree: 2012, Ruhr Universität Bochum

 In der Arbeit wurde die Rolle der intrazellulär lokalisierten löslichen Adenylylcyclase (LAC) bei der Apoptose- Regulation untersucht. Dazu wurden (1) Behandlung mit einem spezifischen Hemmstoff,… (more)

Subjects/Keywords: Ischämie; Reperfusion; Apoptosis; In vitro; Translokation

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APA (6th Edition):

Appukuttan, A. (2012). Role of type 10 "soluble" adenylyl cyclase in regulation of apoptosis : relevance to cardiovascular diseases. (Thesis). Ruhr Universität Bochum. Retrieved from http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-38004

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Appukuttan, Avinash. “Role of type 10 "soluble" adenylyl cyclase in regulation of apoptosis : relevance to cardiovascular diseases.” 2012. Thesis, Ruhr Universität Bochum. Accessed March 07, 2021. http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-38004.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Appukuttan, Avinash. “Role of type 10 "soluble" adenylyl cyclase in regulation of apoptosis : relevance to cardiovascular diseases.” 2012. Web. 07 Mar 2021.

Vancouver:

Appukuttan A. Role of type 10 "soluble" adenylyl cyclase in regulation of apoptosis : relevance to cardiovascular diseases. [Internet] [Thesis]. Ruhr Universität Bochum; 2012. [cited 2021 Mar 07]. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-38004.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Appukuttan A. Role of type 10 "soluble" adenylyl cyclase in regulation of apoptosis : relevance to cardiovascular diseases. [Thesis]. Ruhr Universität Bochum; 2012. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-38004

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. 下内, 昭人. 網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury.

Degree: 博士(医学), 2016, Asahikawa Medical University / 旭川医科大学

First online: 25 September 2015The final publication is available at Springer via http://dx.doi.org/10.1007/s10384-015-0415-z

PurposeTo determine whether water-dispersible hesperetin (WD-Hpt) can prevent degeneration of ganglion cell… (more)

Subjects/Keywords: Hesperetin; Ischemia–reperfusion; Neuroprotection; Retina; Inflammation

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APA (6th Edition):

下内, . (2016). 網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury. (Thesis). Asahikawa Medical University / 旭川医科大学. Retrieved from http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20160325_K492

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

下内, 昭人. “網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury.” 2016. Thesis, Asahikawa Medical University / 旭川医科大学. Accessed March 07, 2021. http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20160325_K492.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

下内, 昭人. “網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury.” 2016. Web. 07 Mar 2021.

Vancouver:

下内 . 網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury. [Internet] [Thesis]. Asahikawa Medical University / 旭川医科大学; 2016. [cited 2021 Mar 07]. Available from: http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20160325_K492.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

下内 . 網膜虚血再灌流障害における分散ヘスペレチンの神経保護効果 : Neuroprotective effect of water-dispersible hesperetin in retinal ischemia reperfusion injury. [Thesis]. Asahikawa Medical University / 旭川医科大学; 2016. Available from: http://amcor.asahikawa-med.ac.jp/modules/xoonips/detail.php?id=20160325_K492

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. 畑地, 豪. The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する.

Degree: 博士(医学), 2014, Nagasaki University / 長崎大学

 Background: Ischemia-reperfusion (I/R) injury after lung transplantation causes alveolar damage, lung edema, and acute rejection. Poly(adenosine diphosphate-ribose) polymerase (PARP) is a single-stranded DNA repair enzyme… (more)

Subjects/Keywords: Ischemia-reperfusion injury; PARP inhibitor; PJ34; Antioxidants

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APA (6th Edition):

畑地, . (2014). The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/35827

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

畑地, 豪. “The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する.” 2014. Thesis, Nagasaki University / 長崎大学. Accessed March 07, 2021. http://hdl.handle.net/10069/35827.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

畑地, 豪. “The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する.” 2014. Web. 07 Mar 2021.

Vancouver:

畑地 . The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10069/35827.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

畑地 . The Poly(Adenosine Diphosphate-Ribose) Polymerase Inhibitor PJ34 Reduces Pulmonary Ischemia-Reperfusion Injury in Rats : ポリADPリボースポリメレース阻害薬(PJ34)はラットにおける肺虚血再灌流障害を軽減する. [Thesis]. Nagasaki University / 長崎大学; 2014. Available from: http://hdl.handle.net/10069/35827

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Gory, Benjamin. Caractérisation IRM d’un modèle murin d’ischémie-reperfusion cérébrale induit par cathétérisme de l’artère cérébrale moyenne et évaluation du post-conditionnement à la Cyclosporine A : MRI characterization of brain ischemia-reperfusion model induced by middle cerebral artery catheterization in rat and evaluation of Cyclosporine A postconditioning.

Degree: Docteur es, Neurosciences, 2016, Lyon

 La reperfusion complète et précoce est le moyen le plus efficace pour limiter l'extension de l'infarctus cérébral et les séquelles neurologiques. Le traitement de l'infarctus… (more)

Subjects/Keywords: Infarctus cérébral; Thrombectomie; Reperfusion; Recanalisation artérielle; Lésions de reperfusion; Cyclosporine A; Neuroprotection; Post-conditionnement; Ischemic stroke; Thrombectomy; Reperfusion; Arterial recanalization; Reperfusion injuries; Cyclosporine A; Neuroprotection; Postconditioning; 612.8

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APA (6th Edition):

Gory, B. (2016). Caractérisation IRM d’un modèle murin d’ischémie-reperfusion cérébrale induit par cathétérisme de l’artère cérébrale moyenne et évaluation du post-conditionnement à la Cyclosporine A : MRI characterization of brain ischemia-reperfusion model induced by middle cerebral artery catheterization in rat and evaluation of Cyclosporine A postconditioning. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2016LYSE1208

Chicago Manual of Style (16th Edition):

Gory, Benjamin. “Caractérisation IRM d’un modèle murin d’ischémie-reperfusion cérébrale induit par cathétérisme de l’artère cérébrale moyenne et évaluation du post-conditionnement à la Cyclosporine A : MRI characterization of brain ischemia-reperfusion model induced by middle cerebral artery catheterization in rat and evaluation of Cyclosporine A postconditioning.” 2016. Doctoral Dissertation, Lyon. Accessed March 07, 2021. http://www.theses.fr/2016LYSE1208.

MLA Handbook (7th Edition):

Gory, Benjamin. “Caractérisation IRM d’un modèle murin d’ischémie-reperfusion cérébrale induit par cathétérisme de l’artère cérébrale moyenne et évaluation du post-conditionnement à la Cyclosporine A : MRI characterization of brain ischemia-reperfusion model induced by middle cerebral artery catheterization in rat and evaluation of Cyclosporine A postconditioning.” 2016. Web. 07 Mar 2021.

Vancouver:

Gory B. Caractérisation IRM d’un modèle murin d’ischémie-reperfusion cérébrale induit par cathétérisme de l’artère cérébrale moyenne et évaluation du post-conditionnement à la Cyclosporine A : MRI characterization of brain ischemia-reperfusion model induced by middle cerebral artery catheterization in rat and evaluation of Cyclosporine A postconditioning. [Internet] [Doctoral dissertation]. Lyon; 2016. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2016LYSE1208.

Council of Science Editors:

Gory B. Caractérisation IRM d’un modèle murin d’ischémie-reperfusion cérébrale induit par cathétérisme de l’artère cérébrale moyenne et évaluation du post-conditionnement à la Cyclosporine A : MRI characterization of brain ischemia-reperfusion model induced by middle cerebral artery catheterization in rat and evaluation of Cyclosporine A postconditioning. [Doctoral Dissertation]. Lyon; 2016. Available from: http://www.theses.fr/2016LYSE1208


University of Alberta

20. Qadhi, Rawabi. Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury.

Degree: MS, Faculty of Pharmacy and Pharmaceutical Sciences, 2013, University of Alberta

 Cardiovascular disease (CVD) remains one of the leading causes of death worldwide. As such, a vast amount of research has investigated novel therapies for preventing… (more)

Subjects/Keywords: H9c2 cells; Apoptosis; DHA; Ischemia/reperfusion injury

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APA (6th Edition):

Qadhi, R. (2013). Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/fx719m76s

Chicago Manual of Style (16th Edition):

Qadhi, Rawabi. “Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury.” 2013. Masters Thesis, University of Alberta. Accessed March 07, 2021. https://era.library.ualberta.ca/files/fx719m76s.

MLA Handbook (7th Edition):

Qadhi, Rawabi. “Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury.” 2013. Web. 07 Mar 2021.

Vancouver:

Qadhi R. Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury. [Internet] [Masters thesis]. University of Alberta; 2013. [cited 2021 Mar 07]. Available from: https://era.library.ualberta.ca/files/fx719m76s.

Council of Science Editors:

Qadhi R. Docosahexaenoic Acid Induced Apoptosis In H9c2 Cells And Changed Cardiac Function After Ischemia-Reperfusion Injury. [Masters Thesis]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/fx719m76s

21. Güney, Türkan. Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi .

Degree: ESOGÜ, Tıp Fakültesi, Tıbbi Biyokimya, 2015, Eskisehir Osmangazi University

 Aort cerrahisinde abdominal aortun klemplenmesi ile oluşan iskemide birçok dokuda uzak organ hasarı meydana gelmektedir. Bu çalışmada fukoidinin rat aortik iskemi reperfüzyon modelinde böbrek ve… (more)

Subjects/Keywords: İskemi/Reperfüzyon; Fukoidin; Ischemia; Reperfusion; Fucoidin

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APA (6th Edition):

Güney, T. (2015). Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi . (Thesis). Eskisehir Osmangazi University. Retrieved from http://hdl.handle.net/11684/631

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Güney, Türkan. “Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi .” 2015. Thesis, Eskisehir Osmangazi University. Accessed March 07, 2021. http://hdl.handle.net/11684/631.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Güney, Türkan. “Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi .” 2015. Web. 07 Mar 2021.

Vancouver:

Güney T. Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi . [Internet] [Thesis]. Eskisehir Osmangazi University; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/11684/631.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Güney T. Fukoidin’in rat aortik iskemi reperfüzyon modelinde böbrek ve akciğerler üzerine etkisi . [Thesis]. Eskisehir Osmangazi University; 2015. Available from: http://hdl.handle.net/11684/631

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

22. Lafreniere, Gina. mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes .

Degree: Physiology, 2015, Queens University

 Death due to ischemic heart disease (IHD) is the result of cardiac arrhythmias and loss of cardiac pump function. One of the putative underlying mechanisms… (more)

Subjects/Keywords: Ischemia/Reperfusion ; Heart ; Voltage Gated Sodium Channels

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APA (6th Edition):

Lafreniere, G. (2015). mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/12682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lafreniere, Gina. “mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes .” 2015. Thesis, Queens University. Accessed March 07, 2021. http://hdl.handle.net/1974/12682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lafreniere, Gina. “mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes .” 2015. Web. 07 Mar 2021.

Vancouver:

Lafreniere G. mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes . [Internet] [Thesis]. Queens University; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1974/12682.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lafreniere G. mRNA Variants of a Neuronal Sodium Channel in Rat Cardiac Myocytes . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/12682

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Newcastle

23. Lin, Longting. Whole-brain CTP in acute ischemic stroke.

Degree: PhD, 2015, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Perfusion imaging technology not only enables stroke diagnosis by identifying the ischemic lesion earlier, but also helps the… (more)

Subjects/Keywords: CT perfusion; ischemic stroke; penumbra; reperfusion

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APA (6th Edition):

Lin, L. (2015). Whole-brain CTP in acute ischemic stroke. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1310399

Chicago Manual of Style (16th Edition):

Lin, Longting. “Whole-brain CTP in acute ischemic stroke.” 2015. Doctoral Dissertation, University of Newcastle. Accessed March 07, 2021. http://hdl.handle.net/1959.13/1310399.

MLA Handbook (7th Edition):

Lin, Longting. “Whole-brain CTP in acute ischemic stroke.” 2015. Web. 07 Mar 2021.

Vancouver:

Lin L. Whole-brain CTP in acute ischemic stroke. [Internet] [Doctoral dissertation]. University of Newcastle; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1959.13/1310399.

Council of Science Editors:

Lin L. Whole-brain CTP in acute ischemic stroke. [Doctoral Dissertation]. University of Newcastle; 2015. Available from: http://hdl.handle.net/1959.13/1310399


University of Toronto

24. Chaturvedi, Swasti. SLIT2 Prevents Renal Ischemia Reperfusion Injury in Mice.

Degree: 2012, University of Toronto

The Slit family of secreted proteins act as axonal repellents during embryogenesis. Slit2 via its receptor, Roundabout-1, also inhibits chemotaxis of multiple leukocyte subsets. Using… (more)

Subjects/Keywords: acute kidney injury; ischaemia-reperfusion injury; 0379

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chaturvedi, S. (2012). SLIT2 Prevents Renal Ischemia Reperfusion Injury in Mice. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42902

Chicago Manual of Style (16th Edition):

Chaturvedi, Swasti. “SLIT2 Prevents Renal Ischemia Reperfusion Injury in Mice.” 2012. Masters Thesis, University of Toronto. Accessed March 07, 2021. http://hdl.handle.net/1807/42902.

MLA Handbook (7th Edition):

Chaturvedi, Swasti. “SLIT2 Prevents Renal Ischemia Reperfusion Injury in Mice.” 2012. Web. 07 Mar 2021.

Vancouver:

Chaturvedi S. SLIT2 Prevents Renal Ischemia Reperfusion Injury in Mice. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1807/42902.

Council of Science Editors:

Chaturvedi S. SLIT2 Prevents Renal Ischemia Reperfusion Injury in Mice. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/42902


University of Toronto

25. Rohailla, Sagar. A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies.

Degree: 2012, University of Toronto

Remote ischemic preconditioning (rIPC) through transient limb ischemia induces potent cardioprotection against ischemia reperfusion (IR) injury. I examined the delayed phase of protection that appears… (more)

Subjects/Keywords: Ischemia Reperfusion; Myocardial Infarction; Cardiology; Preconditioning; 0719

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APA (6th Edition):

Rohailla, S. (2012). A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33511

Chicago Manual of Style (16th Edition):

Rohailla, Sagar. “A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies.” 2012. Masters Thesis, University of Toronto. Accessed March 07, 2021. http://hdl.handle.net/1807/33511.

MLA Handbook (7th Edition):

Rohailla, Sagar. “A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies.” 2012. Web. 07 Mar 2021.

Vancouver:

Rohailla S. A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1807/33511.

Council of Science Editors:

Rohailla S. A Langendorff-perfused Mouse Heart Model for Delayed Remote Limb Ischemic Preconditioning Studies. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33511


University of Toronto

26. Kwong, Wilson. Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature.

Degree: 2011, University of Toronto

Statins can act as preconditioning agents against ischemia reperfusion (IR)-injury through a mechanism involving cyclooxygenase (COX)-2 and the upregulation of prostaglandin synthesis. The following study… (more)

Subjects/Keywords: statin; ischemia reperfusion injury; preconditioning; cox; 0419

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APA (6th Edition):

Kwong, W. (2011). Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/29586

Chicago Manual of Style (16th Edition):

Kwong, Wilson. “Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature.” 2011. Masters Thesis, University of Toronto. Accessed March 07, 2021. http://hdl.handle.net/1807/29586.

MLA Handbook (7th Edition):

Kwong, Wilson. “Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature.” 2011. Web. 07 Mar 2021.

Vancouver:

Kwong W. Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1807/29586.

Council of Science Editors:

Kwong W. Effect of Selective/Non-selective COX Inhibition on Rosuvastatin-Mediated Protection from Ischemia-reperfusion Induced Endothelial Dysfunction in the Human Forearm Vasculature. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/29586

27. Benhabbouche, Souhila. Protection du cœur ischémique au cours de la reperfusion par le post-conditionnement et la basse pression : Protection of ischemic heart at reperfusion by post conditioning and low pressure reperfusion.

Degree: Docteur es, Physiologie cardiovasculaire, 2011, Université Claude Bernard – Lyon I

Bien qu’il ait prouvé son efficacité dans différentes espèces (lapin, porc, souris,…) ainsi que dans différents organes (rein, foie coeur, poumon,…), le Postconditionnement (PostC) peut… (more)

Subjects/Keywords: Ischémie-reperfusion; Cardioprotection; Basse pression de reperfusion; Postconditionnement; Mitochondrie; PTPm; Cœur isolé de rat; Ischemia-reperfusion; Cardioprotection; Low pressure of reperfusion; Postconditioning; Mitochondria; PTPm; Isolated heart rat; 616.123

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APA (6th Edition):

Benhabbouche, S. (2011). Protection du cœur ischémique au cours de la reperfusion par le post-conditionnement et la basse pression : Protection of ischemic heart at reperfusion by post conditioning and low pressure reperfusion. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2011LYO10263

Chicago Manual of Style (16th Edition):

Benhabbouche, Souhila. “Protection du cœur ischémique au cours de la reperfusion par le post-conditionnement et la basse pression : Protection of ischemic heart at reperfusion by post conditioning and low pressure reperfusion.” 2011. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed March 07, 2021. http://www.theses.fr/2011LYO10263.

MLA Handbook (7th Edition):

Benhabbouche, Souhila. “Protection du cœur ischémique au cours de la reperfusion par le post-conditionnement et la basse pression : Protection of ischemic heart at reperfusion by post conditioning and low pressure reperfusion.” 2011. Web. 07 Mar 2021.

Vancouver:

Benhabbouche S. Protection du cœur ischémique au cours de la reperfusion par le post-conditionnement et la basse pression : Protection of ischemic heart at reperfusion by post conditioning and low pressure reperfusion. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2011. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2011LYO10263.

Council of Science Editors:

Benhabbouche S. Protection du cœur ischémique au cours de la reperfusion par le post-conditionnement et la basse pression : Protection of ischemic heart at reperfusion by post conditioning and low pressure reperfusion. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2011. Available from: http://www.theses.fr/2011LYO10263


Robert Gordon University

28. Hair, Steven C. Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences.

Degree: PhD, 2018, Robert Gordon University

 Obesity has reached worldwide epidemic proportions and with this increased incidence of obesity, comes an increase in incidence of the comorbidities associated with obesity such… (more)

Subjects/Keywords: 610; Myocardial ischaemia; Reperfusion; Lysophosphatidylinositol; Obesity

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APA (6th Edition):

Hair, S. C. (2018). Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences. (Doctoral Dissertation). Robert Gordon University. Retrieved from https://rgu-repository.worktribe.com/output/248960 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758179

Chicago Manual of Style (16th Edition):

Hair, Steven C. “Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences.” 2018. Doctoral Dissertation, Robert Gordon University. Accessed March 07, 2021. https://rgu-repository.worktribe.com/output/248960 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758179.

MLA Handbook (7th Edition):

Hair, Steven C. “Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences.” 2018. Web. 07 Mar 2021.

Vancouver:

Hair SC. Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences. [Internet] [Doctoral dissertation]. Robert Gordon University; 2018. [cited 2021 Mar 07]. Available from: https://rgu-repository.worktribe.com/output/248960 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758179.

Council of Science Editors:

Hair SC. Determining the role of the LPI/GPR55 system in the development of obesity and associated cardiovascular consequences. [Doctoral Dissertation]. Robert Gordon University; 2018. Available from: https://rgu-repository.worktribe.com/output/248960 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.758179


Grand Valley State University

29. Hake, Benjamin Matthew. Changes in Vascular Reactivity of Mesenteric Arteries Following Hyperbaric Oxygen Treatment.

Degree: 2013, Grand Valley State University

 The objective of this study was to evaluate the changes in vascular reactivity of mesenteric arteries resulting from hyperbaric oxygen treatment. It was hypothesized that… (more)

Subjects/Keywords: Hyperbaric; Ischemic Reperfusion; Vascular Reactivity; ATP

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APA (6th Edition):

Hake, B. M. (2013). Changes in Vascular Reactivity of Mesenteric Arteries Following Hyperbaric Oxygen Treatment. (Thesis). Grand Valley State University. Retrieved from https://scholarworks.gvsu.edu/theses/304

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hake, Benjamin Matthew. “Changes in Vascular Reactivity of Mesenteric Arteries Following Hyperbaric Oxygen Treatment.” 2013. Thesis, Grand Valley State University. Accessed March 07, 2021. https://scholarworks.gvsu.edu/theses/304.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hake, Benjamin Matthew. “Changes in Vascular Reactivity of Mesenteric Arteries Following Hyperbaric Oxygen Treatment.” 2013. Web. 07 Mar 2021.

Vancouver:

Hake BM. Changes in Vascular Reactivity of Mesenteric Arteries Following Hyperbaric Oxygen Treatment. [Internet] [Thesis]. Grand Valley State University; 2013. [cited 2021 Mar 07]. Available from: https://scholarworks.gvsu.edu/theses/304.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hake BM. Changes in Vascular Reactivity of Mesenteric Arteries Following Hyperbaric Oxygen Treatment. [Thesis]. Grand Valley State University; 2013. Available from: https://scholarworks.gvsu.edu/theses/304

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

30. Prag, Hiran Ambelal. Developing drugs to attenuate succinate accumulation and oxidation.

Degree: PhD, 2019, University of Cambridge

 Ischaemia-reperfusion (IR) injury is caused by the re-introduction of oxygen to organs, following periods of reduced blood flow (ischaemia). Whilst re-establishing blood flow (reperfusion) to… (more)

Subjects/Keywords: mitochondria; ischaemia-reperfusion injury; succinate dehydrogenase

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APA (6th Edition):

Prag, H. A. (2019). Developing drugs to attenuate succinate accumulation and oxidation. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/289443

Chicago Manual of Style (16th Edition):

Prag, Hiran Ambelal. “Developing drugs to attenuate succinate accumulation and oxidation.” 2019. Doctoral Dissertation, University of Cambridge. Accessed March 07, 2021. https://www.repository.cam.ac.uk/handle/1810/289443.

MLA Handbook (7th Edition):

Prag, Hiran Ambelal. “Developing drugs to attenuate succinate accumulation and oxidation.” 2019. Web. 07 Mar 2021.

Vancouver:

Prag HA. Developing drugs to attenuate succinate accumulation and oxidation. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Mar 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/289443.

Council of Science Editors:

Prag HA. Developing drugs to attenuate succinate accumulation and oxidation. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/289443

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