Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Receptor activator of NF KB RANK ). Showing records 1 – 30 of 301500 total matches.

[1] [2] [3] [4] [5] … [10050]

Search Limiters

Last 2 Years | English Only

Department

Degrees

Languages

Country

▼ Search Limiters

1. Papoutselis, Menelaos. Ο ρόλος των αδιαφοροποίητων φυσικών κυτταροκτόνων Τ (iNKT) λεμφοκυττάρων στην ομοιοστασία των οστών και την οστική νόσο του πολλαπλού μυελώματος.

Degree: 2017, Democritus University of Thrace (DUTH); Δημοκρίτειο Πανεπιστήμιο Θράκης (ΔΠΘ)

Increased expression of RANKL on osteoblasts and T cells is central for osteoclast activation and development of bone disease in multiple myeloma. We tested the… (more)

Subjects/Keywords: Αδιαφοροποίητα φυσικά κυτταροκτόνα Τ λεμφοκύτταρα; Πολλαπλούν μυέλωμα; Κυτταροκίνη RANKL; Invariant natural killer T lymphocytes; Multiple myeloma; Receptor activator of NF-KB ligand (RANKL)

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Papoutselis, M. (2017). Ο ρόλος των αδιαφοροποίητων φυσικών κυτταροκτόνων Τ (iNKT) λεμφοκυττάρων στην ομοιοστασία των οστών και την οστική νόσο του πολλαπλού μυελώματος. (Thesis). Democritus University of Thrace (DUTH); Δημοκρίτειο Πανεπιστήμιο Θράκης (ΔΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/40696

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Papoutselis, Menelaos. “Ο ρόλος των αδιαφοροποίητων φυσικών κυτταροκτόνων Τ (iNKT) λεμφοκυττάρων στην ομοιοστασία των οστών και την οστική νόσο του πολλαπλού μυελώματος.” 2017. Thesis, Democritus University of Thrace (DUTH); Δημοκρίτειο Πανεπιστήμιο Θράκης (ΔΠΘ). Accessed April 17, 2021. http://hdl.handle.net/10442/hedi/40696.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Papoutselis, Menelaos. “Ο ρόλος των αδιαφοροποίητων φυσικών κυτταροκτόνων Τ (iNKT) λεμφοκυττάρων στην ομοιοστασία των οστών και την οστική νόσο του πολλαπλού μυελώματος.” 2017. Web. 17 Apr 2021.

Vancouver:

Papoutselis M. Ο ρόλος των αδιαφοροποίητων φυσικών κυτταροκτόνων Τ (iNKT) λεμφοκυττάρων στην ομοιοστασία των οστών και την οστική νόσο του πολλαπλού μυελώματος. [Internet] [Thesis]. Democritus University of Thrace (DUTH); Δημοκρίτειο Πανεπιστήμιο Θράκης (ΔΠΘ); 2017. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10442/hedi/40696.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Papoutselis M. Ο ρόλος των αδιαφοροποίητων φυσικών κυτταροκτόνων Τ (iNKT) λεμφοκυττάρων στην ομοιοστασία των οστών και την οστική νόσο του πολλαπλού μυελώματος. [Thesis]. Democritus University of Thrace (DUTH); Δημοκρίτειο Πανεπιστήμιο Θράκης (ΔΠΘ); 2017. Available from: http://hdl.handle.net/10442/hedi/40696

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Τυροβολά, Ιωάννα. Συσχέτιση επιπέδων SRANKLκαι OPG στον ορό του αίματος και στο υγρό της ουλοδοντικής σχισμής και βαθμού ριζικής απορρόφησης κατά την ορθοδοντική θεραπεία.

Degree: 2009, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

The aim of the present study was the determination in a rat model of the levels of OPG and sRANKL in blood and in gingival… (more)

Subjects/Keywords: Οστεοπροτεγερίνη; Ριζική απορρόφηση; Ορθοδοντική θεραπεία; Υγρό ουλοδοντικής σχισμής; Ορός αίματος; Οστική αναδιαμόρφωση; Απόπτωση νεογιλού; Πρωτεΐνες; Osteoprotegerin (OPG); Receptor activator of NF-KB ligand (RANKL); Receptor activator of NF-KB (RANK); Root resorption; Orthodontic treatment; Blood serum; Gingival crevicular fluid (GCF); Proteins

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Τυροβολά, . . (2009). Συσχέτιση επιπέδων SRANKLκαι OPG στον ορό του αίματος και στο υγρό της ουλοδοντικής σχισμής και βαθμού ριζικής απορρόφησης κατά την ορθοδοντική θεραπεία. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/27799

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Τυροβολά, Ιωάννα. “Συσχέτιση επιπέδων SRANKLκαι OPG στον ορό του αίματος και στο υγρό της ουλοδοντικής σχισμής και βαθμού ριζικής απορρόφησης κατά την ορθοδοντική θεραπεία.” 2009. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 17, 2021. http://hdl.handle.net/10442/hedi/27799.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Τυροβολά, Ιωάννα. “Συσχέτιση επιπέδων SRANKLκαι OPG στον ορό του αίματος και στο υγρό της ουλοδοντικής σχισμής και βαθμού ριζικής απορρόφησης κατά την ορθοδοντική θεραπεία.” 2009. Web. 17 Apr 2021.

Vancouver:

Τυροβολά . Συσχέτιση επιπέδων SRANKLκαι OPG στον ορό του αίματος και στο υγρό της ουλοδοντικής σχισμής και βαθμού ριζικής απορρόφησης κατά την ορθοδοντική θεραπεία. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10442/hedi/27799.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Τυροβολά . Συσχέτιση επιπέδων SRANKLκαι OPG στον ορό του αίματος και στο υγρό της ουλοδοντικής σχισμής και βαθμού ριζικής απορρόφησης κατά την ορθοδοντική θεραπεία. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. Available from: http://hdl.handle.net/10442/hedi/27799

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

3. Mohan, Geetha. Mechanisms of osteoarthritis : interrelationships between bone and cartilage.

Degree: 2012, University of Adelaide

 Osteoarthritis (OA) is a progressive joint disease and a common cause of disability. OA is characterised by loss of articular cartilage, subchondral bone sclerosis, cysts,… (more)

Subjects/Keywords: Osteoarthritis; subchondral bone; cartilage; micro-computed tomography; OA animal model; Alendronate; Monosodium iodoacetate; osteoprotegerin; receptor activator of nuclear factor KB ligand

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mohan, G. (2012). Mechanisms of osteoarthritis : interrelationships between bone and cartilage. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/84538

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mohan, Geetha. “Mechanisms of osteoarthritis : interrelationships between bone and cartilage.” 2012. Thesis, University of Adelaide. Accessed April 17, 2021. http://hdl.handle.net/2440/84538.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mohan, Geetha. “Mechanisms of osteoarthritis : interrelationships between bone and cartilage.” 2012. Web. 17 Apr 2021.

Vancouver:

Mohan G. Mechanisms of osteoarthritis : interrelationships between bone and cartilage. [Internet] [Thesis]. University of Adelaide; 2012. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/2440/84538.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mohan G. Mechanisms of osteoarthritis : interrelationships between bone and cartilage. [Thesis]. University of Adelaide; 2012. Available from: http://hdl.handle.net/2440/84538

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. 菊田,純. Jagged1-Notch2 signaling induces root resorption via RANKL and IL-6 : Jagged1-Notch2シグナルはRANKLとIL-6を介して歯根吸収を惹起する.

Degree: 博士(歯学), 2015, Nihon University / 日本大学

Subjects/Keywords: Notchシグナル; Notch signaling; Receptor activator of NF-kB ligand; RANKL; Interleukin-6; IL-6; 矯正力; Orthodontic force; 歯根吸収; Root resorption

Page 1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

菊田,純. (2015). Jagged1-Notch2 signaling induces root resorption via RANKL and IL-6 : Jagged1-Notch2シグナルはRANKLとIL-6を介して歯根吸収を惹起する. (Thesis). Nihon University / 日本大学. Retrieved from http://repository.nihon-u.ac.jp/xmlui/handle/11263/573 ; http://dx.doi.org/10.15006/32665A4967

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

菊田,純. “Jagged1-Notch2 signaling induces root resorption via RANKL and IL-6 : Jagged1-Notch2シグナルはRANKLとIL-6を介して歯根吸収を惹起する.” 2015. Thesis, Nihon University / 日本大学. Accessed April 17, 2021. http://repository.nihon-u.ac.jp/xmlui/handle/11263/573 ; http://dx.doi.org/10.15006/32665A4967.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

菊田,純. “Jagged1-Notch2 signaling induces root resorption via RANKL and IL-6 : Jagged1-Notch2シグナルはRANKLとIL-6を介して歯根吸収を惹起する.” 2015. Web. 17 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

菊田,純. Jagged1-Notch2 signaling induces root resorption via RANKL and IL-6 : Jagged1-Notch2シグナルはRANKLとIL-6を介して歯根吸収を惹起する. [Internet] [Thesis]. Nihon University / 日本大学; 2015. [cited 2021 Apr 17]. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/573 ; http://dx.doi.org/10.15006/32665A4967.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

菊田,純. Jagged1-Notch2 signaling induces root resorption via RANKL and IL-6 : Jagged1-Notch2シグナルはRANKLとIL-6を介して歯根吸収を惹起する. [Thesis]. Nihon University / 日本大学; 2015. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/573 ; http://dx.doi.org/10.15006/32665A4967

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

5. Dolci, Gabriel Schmidt. Efeitos da atorvastatina sobre a ossificação endocondral de fêmures, remodelação óssea e movimentação dentária induzida, estudo em ratos.

Degree: 2016, Brazil

 As estatinas são medicamentos comumente prescritos para a prevenção da hiper-lipidemia. Além da redução do colesterol, tais medicamentos parecem estimular a osteogênese e suprimir a… (more)

Subjects/Keywords: Atorvastatina; Ligante RANK; Ortodontia; Tooth movement; Osteoprotegerin; Receptor activator of nuclear factor-kappa B ligand; Atorvastatin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dolci, G. S. (2016). Efeitos da atorvastatina sobre a ossificação endocondral de fêmures, remodelação óssea e movimentação dentária induzida, estudo em ratos. (Doctoral Dissertation). Brazil. Retrieved from http://hdl.handle.net/10183/142126

Chicago Manual of Style (16th Edition):

Dolci, Gabriel Schmidt. “Efeitos da atorvastatina sobre a ossificação endocondral de fêmures, remodelação óssea e movimentação dentária induzida, estudo em ratos.” 2016. Doctoral Dissertation, Brazil. Accessed April 17, 2021. http://hdl.handle.net/10183/142126.

MLA Handbook (7th Edition):

Dolci, Gabriel Schmidt. “Efeitos da atorvastatina sobre a ossificação endocondral de fêmures, remodelação óssea e movimentação dentária induzida, estudo em ratos.” 2016. Web. 17 Apr 2021.

Vancouver:

Dolci GS. Efeitos da atorvastatina sobre a ossificação endocondral de fêmures, remodelação óssea e movimentação dentária induzida, estudo em ratos. [Internet] [Doctoral dissertation]. Brazil; 2016. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10183/142126.

Council of Science Editors:

Dolci GS. Efeitos da atorvastatina sobre a ossificação endocondral de fêmures, remodelação óssea e movimentação dentária induzida, estudo em ratos. [Doctoral Dissertation]. Brazil; 2016. Available from: http://hdl.handle.net/10183/142126


IUPUI

6. Cogill, Steven B. PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1.

Degree: 2011, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

SIMPL is a transcriptional co-activator that alters the activity of transcription factor, NF-κB. In response to pathogens, cytokines such as… (more)

Subjects/Keywords: SIMPL; Transcriptional Co-Activator; NF-kB; NF-kappa B (DNA-binding protein); Transcription factors; Cytokines; Immunoglobulins; Immunofluorescence

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cogill, S. B. (2011). PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2493

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cogill, Steven B. “PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1.” 2011. Thesis, IUPUI. Accessed April 17, 2021. http://hdl.handle.net/1805/2493.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cogill, Steven B. “PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1.” 2011. Web. 17 Apr 2021.

Vancouver:

Cogill SB. PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1. [Internet] [Thesis]. IUPUI; 2011. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1805/2493.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cogill SB. PURIFICATION OF SIMPL ANTIBODY AND IMMUNOFLUORESCENCE OF SIMPL SUB-CELLULAR LOCALIZATION IN RESPONSE TO TNFα- AND IL-1. [Thesis]. IUPUI; 2011. Available from: http://hdl.handle.net/1805/2493

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

7. PEARSON, JACLYN. Type III effectors of enteropathogenic Escherichia coli.

Degree: 2013, University of Melbourne

 Enteropathogenic Escherichia coli (EPEC) is an extracellular gastrointestinal pathogen that colonises and adheres intimately to the small intestinal mucosa via the formation of attaching and… (more)

Subjects/Keywords: inflammation; apoptosis; death receptor signaling; NF-KB; gastrointestinal pathogens

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

PEARSON, J. (2013). Type III effectors of enteropathogenic Escherichia coli. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38586

Chicago Manual of Style (16th Edition):

PEARSON, JACLYN. “Type III effectors of enteropathogenic Escherichia coli.” 2013. Doctoral Dissertation, University of Melbourne. Accessed April 17, 2021. http://hdl.handle.net/11343/38586.

MLA Handbook (7th Edition):

PEARSON, JACLYN. “Type III effectors of enteropathogenic Escherichia coli.” 2013. Web. 17 Apr 2021.

Vancouver:

PEARSON J. Type III effectors of enteropathogenic Escherichia coli. [Internet] [Doctoral dissertation]. University of Melbourne; 2013. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/11343/38586.

Council of Science Editors:

PEARSON J. Type III effectors of enteropathogenic Escherichia coli. [Doctoral Dissertation]. University of Melbourne; 2013. Available from: http://hdl.handle.net/11343/38586

8. 吉野,智一. TNF-α induces orthodontic root resorption via the expression of RANKL : TNF-αはRANKL存在下で歯科矯正治療中の歯根吸収を増悪させる.

Degree: 博士(歯学), 2014, Nihon University / 日本大学

Subjects/Keywords: Tumor necrosis factor-α; TNF-α; Receptor activator of NF-kB ligand; RANKL; 歯根吸収; Root resorption; 矯正学的歯牙移動; Orthodontic tooth movement

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

吉野,智一. (2014). TNF-α induces orthodontic root resorption via the expression of RANKL : TNF-αはRANKL存在下で歯科矯正治療中の歯根吸収を増悪させる. (Thesis). Nihon University / 日本大学. Retrieved from http://repository.nihon-u.ac.jp/xmlui/handle/11263/246 ; http://dx.doi.org/10.15006/32665A4828

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

吉野,智一. “TNF-α induces orthodontic root resorption via the expression of RANKL : TNF-αはRANKL存在下で歯科矯正治療中の歯根吸収を増悪させる.” 2014. Thesis, Nihon University / 日本大学. Accessed April 17, 2021. http://repository.nihon-u.ac.jp/xmlui/handle/11263/246 ; http://dx.doi.org/10.15006/32665A4828.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

吉野,智一. “TNF-α induces orthodontic root resorption via the expression of RANKL : TNF-αはRANKL存在下で歯科矯正治療中の歯根吸収を増悪させる.” 2014. Web. 17 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

吉野,智一. TNF-α induces orthodontic root resorption via the expression of RANKL : TNF-αはRANKL存在下で歯科矯正治療中の歯根吸収を増悪させる. [Internet] [Thesis]. Nihon University / 日本大学; 2014. [cited 2021 Apr 17]. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/246 ; http://dx.doi.org/10.15006/32665A4828.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

吉野,智一. TNF-α induces orthodontic root resorption via the expression of RANKL : TNF-αはRANKL存在下で歯科矯正治療中の歯根吸収を増悪させる. [Thesis]. Nihon University / 日本大学; 2014. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/246 ; http://dx.doi.org/10.15006/32665A4828

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

9. Masterson, Claire. Understanding the Mechanisms of Action of Hypercapnic Acidosis and the Therapeutic Potential of Human Mesenchymal Stromal Cells in Diminishing Inflammation and Enhancing Repair in Acute Respiratory Distress Syndrome.

Degree: 2013, National University of Ireland – Galway

 Acute respiratory distress syndrome (ARDS) is a term used to describe severe lung injury characterised by uncontrolled inflammatory response and resultant damage to endothelial and… (more)

Subjects/Keywords: ARDS; NF-kB; Hypercapnia; MSCs; Department of Anaesthesia; School of Medicine

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Masterson, C. (2013). Understanding the Mechanisms of Action of Hypercapnic Acidosis and the Therapeutic Potential of Human Mesenchymal Stromal Cells in Diminishing Inflammation and Enhancing Repair in Acute Respiratory Distress Syndrome. (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/4407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Masterson, Claire. “Understanding the Mechanisms of Action of Hypercapnic Acidosis and the Therapeutic Potential of Human Mesenchymal Stromal Cells in Diminishing Inflammation and Enhancing Repair in Acute Respiratory Distress Syndrome. ” 2013. Thesis, National University of Ireland – Galway. Accessed April 17, 2021. http://hdl.handle.net/10379/4407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Masterson, Claire. “Understanding the Mechanisms of Action of Hypercapnic Acidosis and the Therapeutic Potential of Human Mesenchymal Stromal Cells in Diminishing Inflammation and Enhancing Repair in Acute Respiratory Distress Syndrome. ” 2013. Web. 17 Apr 2021.

Vancouver:

Masterson C. Understanding the Mechanisms of Action of Hypercapnic Acidosis and the Therapeutic Potential of Human Mesenchymal Stromal Cells in Diminishing Inflammation and Enhancing Repair in Acute Respiratory Distress Syndrome. [Internet] [Thesis]. National University of Ireland – Galway; 2013. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10379/4407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Masterson C. Understanding the Mechanisms of Action of Hypercapnic Acidosis and the Therapeutic Potential of Human Mesenchymal Stromal Cells in Diminishing Inflammation and Enhancing Repair in Acute Respiratory Distress Syndrome. [Thesis]. National University of Ireland – Galway; 2013. Available from: http://hdl.handle.net/10379/4407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Jules, Joel. In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function.

Degree: PhD, 2010, University of Alabama – Birmingham

The receptor activator of NF-қB (RANK) ligand (RANKL) and its receptor RANK play a critical role in osteoclast biology. RANK has three tumor necrosis factor… (more)

Subjects/Keywords: Amino Acid Motifs<; br>; Cell Differentiation<; br>; Cytoplasm  – metabolism<; br>; Osteoclasts  – cytology<; br>; RANK Ligand  – metabolism<; br>; Receptor Activator of Nuclear Factor-kappa B  – chemistry<; br>; Receptor Activator of Nuclear Factor-kappa B  – metabolism<; br>; Tumor Necrosis Factor-alpha  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jules, J. (2010). In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,917

Chicago Manual of Style (16th Edition):

Jules, Joel. “In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 17, 2021. http://contentdm.mhsl.uab.edu/u?/etd,917.

MLA Handbook (7th Edition):

Jules, Joel. “In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function.” 2010. Web. 17 Apr 2021.

Vancouver:

Jules J. In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2021 Apr 17]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,917.

Council of Science Editors:

Jules J. In vitro elucidation of the role and mechanism of RANKL in TNF- and IL-1-mediated osteoclast formation and function. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,917


University of Alberta

11. Fagan-Garcia, Katharine. A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling.

Degree: MS, Department of Medical Microbiology and Immunology, 2010, University of Alberta

 The classical Nuclear Factor kappa B (NF-κB) signaling pathway is an important regulator of inflammation and innate immune responses. Poxviruses, including vaccinia virus, encode multiple… (more)

Subjects/Keywords: vaccinia; virus; NF-kB; inhibitors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fagan-Garcia, K. (2010). A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/jq085k72q

Chicago Manual of Style (16th Edition):

Fagan-Garcia, Katharine. “A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling.” 2010. Masters Thesis, University of Alberta. Accessed April 17, 2021. https://era.library.ualberta.ca/files/jq085k72q.

MLA Handbook (7th Edition):

Fagan-Garcia, Katharine. “A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling.” 2010. Web. 17 Apr 2021.

Vancouver:

Fagan-Garcia K. A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling. [Internet] [Masters thesis]. University of Alberta; 2010. [cited 2021 Apr 17]. Available from: https://era.library.ualberta.ca/files/jq085k72q.

Council of Science Editors:

Fagan-Garcia K. A large deletion virus reveals the presence of previously uncharacterized vaccinia virus inhibitors of NF-kB signaling. [Masters Thesis]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/jq085k72q

12. Πηλιχού, Αναστασία. Συσχέτιση μεταξύ επιπέδων οστεοπρωτεγερίνης σε άτομα με προχωρημένη οστεοαρθρίτιδα γόνατος και κλινικής βαρύτητας της νόσου.

Degree: 2008, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 Η αναγνώριση και ο χαρακτηρισμός των τριών νέων μορίων (RANKL, RANK, OPG) που ανήκουν στην οικογένεια των παραγόντων νέκρωσης του όγκου (TNF) έχει ανοίξει νέα… (more)

Subjects/Keywords: Γόνατα, Οστεοαρθρίτιδα; Οστεοπρωτεγερίνη; Αρθρικό υγρό; Ορός αίματος; Knee osteoarthritis; Osteoprotegerin; Receptor activator of nuclear factor kb ligand; Synovial fluid; Serum

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Πηλιχού, . . (2008). Συσχέτιση μεταξύ επιπέδων οστεοπρωτεγερίνης σε άτομα με προχωρημένη οστεοαρθρίτιδα γόνατος και κλινικής βαρύτητας της νόσου. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/18463

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Πηλιχού, Αναστασία. “Συσχέτιση μεταξύ επιπέδων οστεοπρωτεγερίνης σε άτομα με προχωρημένη οστεοαρθρίτιδα γόνατος και κλινικής βαρύτητας της νόσου.” 2008. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 17, 2021. http://hdl.handle.net/10442/hedi/18463.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Πηλιχού, Αναστασία. “Συσχέτιση μεταξύ επιπέδων οστεοπρωτεγερίνης σε άτομα με προχωρημένη οστεοαρθρίτιδα γόνατος και κλινικής βαρύτητας της νόσου.” 2008. Web. 17 Apr 2021.

Vancouver:

Πηλιχού . Συσχέτιση μεταξύ επιπέδων οστεοπρωτεγερίνης σε άτομα με προχωρημένη οστεοαρθρίτιδα γόνατος και κλινικής βαρύτητας της νόσου. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2008. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10442/hedi/18463.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Πηλιχού . Συσχέτιση μεταξύ επιπέδων οστεοπρωτεγερίνης σε άτομα με προχωρημένη οστεοαρθρίτιδα γόνατος και κλινικής βαρύτητας της νόσου. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2008. Available from: http://hdl.handle.net/10442/hedi/18463

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Guelph

13. Siracusa, Laura. Mechanisms of Glucocorticoid-Mediated Suppression of Tracheal Antimicrobial Peptide Expression in Bovine Tracheal Epithelial Cells.

Degree: MS, Department of Pathobiology, 2019, University of Guelph

 This research aimed to identify the mechanism by which glucocorticoid downregulates inducible innate immune responses in bovine tracheal epithelial cells (bTEC). It adds knowledge regarding… (more)

Subjects/Keywords: Pathobiology; Molecular Biology; tracheal antimicrobial peptide; dual luciferase; NF-kB; mRNA; IkB; Inflammation; stress; glucocorticoid; cloning; toll-like receptor; TLR 2

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Siracusa, L. (2019). Mechanisms of Glucocorticoid-Mediated Suppression of Tracheal Antimicrobial Peptide Expression in Bovine Tracheal Epithelial Cells. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/14742

Chicago Manual of Style (16th Edition):

Siracusa, Laura. “Mechanisms of Glucocorticoid-Mediated Suppression of Tracheal Antimicrobial Peptide Expression in Bovine Tracheal Epithelial Cells.” 2019. Masters Thesis, University of Guelph. Accessed April 17, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/14742.

MLA Handbook (7th Edition):

Siracusa, Laura. “Mechanisms of Glucocorticoid-Mediated Suppression of Tracheal Antimicrobial Peptide Expression in Bovine Tracheal Epithelial Cells.” 2019. Web. 17 Apr 2021.

Vancouver:

Siracusa L. Mechanisms of Glucocorticoid-Mediated Suppression of Tracheal Antimicrobial Peptide Expression in Bovine Tracheal Epithelial Cells. [Internet] [Masters thesis]. University of Guelph; 2019. [cited 2021 Apr 17]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/14742.

Council of Science Editors:

Siracusa L. Mechanisms of Glucocorticoid-Mediated Suppression of Tracheal Antimicrobial Peptide Expression in Bovine Tracheal Epithelial Cells. [Masters Thesis]. University of Guelph; 2019. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/14742


Vanderbilt University

14. Thu, Yee Mon. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

 Nuclear factor-êB (NF-êB) inducing kinase (NIK) is a MAP3K that regulates activation of NF-êB. NIK is often over-expressed in tumor cells, including melanoma, but the… (more)

Subjects/Keywords: melanoma; cancer; NF-kB inducing kinase; NF-kB

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thu, Y. M. (2011). The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14261

Chicago Manual of Style (16th Edition):

Thu, Yee Mon. “The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed April 17, 2021. http://hdl.handle.net/1803/14261.

MLA Handbook (7th Edition):

Thu, Yee Mon. “The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis.” 2011. Web. 17 Apr 2021.

Vancouver:

Thu YM. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1803/14261.

Council of Science Editors:

Thu YM. The role of NF-kB inducing kinase (NIK) in modulating melanoma tumorigenesis. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/14261

15. Ribeiro Bravo, Isabel. Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells.

Degree: PhD, 2019, University of Edinburgh

 Chronic inflammation has long been considered an enabling characteristic of the cancer microenvironment. The release of growth and survival factors, extracellular matrix-modifying enzymes and other… (more)

Subjects/Keywords: inflammation; NF-kB; zebrafish; inflammatory response; tumour initiation; NF-kB activation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ribeiro Bravo, I. (2019). Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/36166

Chicago Manual of Style (16th Edition):

Ribeiro Bravo, Isabel. “Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells.” 2019. Doctoral Dissertation, University of Edinburgh. Accessed April 17, 2021. http://hdl.handle.net/1842/36166.

MLA Handbook (7th Edition):

Ribeiro Bravo, Isabel. “Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells.” 2019. Web. 17 Apr 2021.

Vancouver:

Ribeiro Bravo I. Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells. [Internet] [Doctoral dissertation]. University of Edinburgh; 2019. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1842/36166.

Council of Science Editors:

Ribeiro Bravo I. Investigating the role of MyD88-NF-kB signalling in regulating the inflammatory response to the emerging pre-neoplastic cells. [Doctoral Dissertation]. University of Edinburgh; 2019. Available from: http://hdl.handle.net/1842/36166

16. Andressa Duarte. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.

Degree: 2013, University of São Paulo

A imunidade inata é responsável pela resposta inicial aos microrganismos, uma vez que impede, controla ou elimina a infecção. Esse sistema consiste em barreiras epiteliais,… (more)

Subjects/Keywords: iNOS; NF-kB; NO; PI3K; iNOS; NF-kB; NO; PI3K

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Duarte, A. (2013). Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/

Chicago Manual of Style (16th Edition):

Duarte, Andressa. “Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.” 2013. Masters Thesis, University of São Paulo. Accessed April 17, 2021. http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/.

MLA Handbook (7th Edition):

Duarte, Andressa. “Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.” 2013. Web. 17 Apr 2021.

Vancouver:

Duarte A. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2021 Apr 17]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/.

Council of Science Editors:

Duarte A. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/


University of Ottawa

17. Phan, Michael. Viral Sensitizers Potentiate the Infection of Cancer Cells Via NF-kB .

Degree: 2020, University of Ottawa

 Genetically engineered oncolytic viruses (OVs) have been proven to be effective anti-cancer agents. However, the heterogeneity of tumours and obligate attenuation of OVs to achieve… (more)

Subjects/Keywords: Cancer; NF-kB; Oncolytic Virus; Cancer Immunotherapy; Mechanism of Action; Interferon; Antiviral Signalling

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Phan, M. (2020). Viral Sensitizers Potentiate the Infection of Cancer Cells Via NF-kB . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/40524

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Phan, Michael. “Viral Sensitizers Potentiate the Infection of Cancer Cells Via NF-kB .” 2020. Thesis, University of Ottawa. Accessed April 17, 2021. http://hdl.handle.net/10393/40524.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Phan, Michael. “Viral Sensitizers Potentiate the Infection of Cancer Cells Via NF-kB .” 2020. Web. 17 Apr 2021.

Vancouver:

Phan M. Viral Sensitizers Potentiate the Infection of Cancer Cells Via NF-kB . [Internet] [Thesis]. University of Ottawa; 2020. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10393/40524.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Phan M. Viral Sensitizers Potentiate the Infection of Cancer Cells Via NF-kB . [Thesis]. University of Ottawa; 2020. Available from: http://hdl.handle.net/10393/40524

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

18. Lejmi Mrad, Rim. Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo .

Degree: 2016, University of Ottawa

 Skeletal muscle atrophy is a debilitating condition caused by pathological conditions including cancer cachexia, disuse and denervation. Disuse atrophy is characterized by reduction in fiber… (more)

Subjects/Keywords: Cellular inhibitor of apoptosis; skeletal muscle atrophy; NF-kB signaling pathway; TWEAK/Fn14 system

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lejmi Mrad, R. (2016). Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/34260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lejmi Mrad, Rim. “Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo .” 2016. Thesis, University of Ottawa. Accessed April 17, 2021. http://hdl.handle.net/10393/34260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lejmi Mrad, Rim. “Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo .” 2016. Web. 17 Apr 2021.

Vancouver:

Lejmi Mrad R. Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo . [Internet] [Thesis]. University of Ottawa; 2016. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10393/34260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lejmi Mrad R. Genetic and Pharmacologic Inhibition of Cellular Inhibitor of Apoptosis 1 (cIAP1) Protein Expression Protects Against Denervation-Induced Skeletal Muscle Atrophy In Vivo . [Thesis]. University of Ottawa; 2016. Available from: http://hdl.handle.net/10393/34260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. 山下, 恭德. RANKL pretreatment plays an important role in the differentiation of pit-forming osteoclasts induced by TNF-α on murine bone marrow macrophages : RANKL 前刺激はマウス骨髄マクロファージにおいてTNF-α 刺激による吸収活性を持つ破骨細胞の分化に重要な役割を持つ.

Degree: 博士(歯学), 2015, Nagasaki University / 長崎大学

 Background Osteoclasts differentiated from bone marrow macrophages (BMMs) induced by TNF-α alone do not have resorbing activity. When BMMs are stimulated with receptor activator of… (more)

Subjects/Keywords: Receptor activator of NF-κB ligand; Tumour necrosis factor-alpha; Osteoclast; Resorbing activity

…x29;:11394–9. 21. Jules J, Shi Z, Liu J, Xu D, Wang S, Feng X. Receptor activator of NF… …activation-induced cytokine/receptor activator of NF-kappa B ligand. J Immunol 2006;177(7)… …osteoclasts formed by TNF-a with or without OPG, which is a decoy receptor of RANKL. This result… …resorption activity in the presence of interleukin (IL)-1.11,12 The signals from RANK… …x28;the receptor of RANKL) or IL-1 receptors were reported to activate the pathway of… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

山下, . (2015). RANKL pretreatment plays an important role in the differentiation of pit-forming osteoclasts induced by TNF-α on murine bone marrow macrophages : RANKL 前刺激はマウス骨髄マクロファージにおいてTNF-α 刺激による吸収活性を持つ破骨細胞の分化に重要な役割を持つ. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/36049

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山下, 恭德. “RANKL pretreatment plays an important role in the differentiation of pit-forming osteoclasts induced by TNF-α on murine bone marrow macrophages : RANKL 前刺激はマウス骨髄マクロファージにおいてTNF-α 刺激による吸収活性を持つ破骨細胞の分化に重要な役割を持つ.” 2015. Thesis, Nagasaki University / 長崎大学. Accessed April 17, 2021. http://hdl.handle.net/10069/36049.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山下, 恭德. “RANKL pretreatment plays an important role in the differentiation of pit-forming osteoclasts induced by TNF-α on murine bone marrow macrophages : RANKL 前刺激はマウス骨髄マクロファージにおいてTNF-α 刺激による吸収活性を持つ破骨細胞の分化に重要な役割を持つ.” 2015. Web. 17 Apr 2021.

Vancouver:

山下 . RANKL pretreatment plays an important role in the differentiation of pit-forming osteoclasts induced by TNF-α on murine bone marrow macrophages : RANKL 前刺激はマウス骨髄マクロファージにおいてTNF-α 刺激による吸収活性を持つ破骨細胞の分化に重要な役割を持つ. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2015. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10069/36049.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山下 . RANKL pretreatment plays an important role in the differentiation of pit-forming osteoclasts induced by TNF-α on murine bone marrow macrophages : RANKL 前刺激はマウス骨髄マクロファージにおいてTNF-α 刺激による吸収活性を持つ破骨細胞の分化に重要な役割を持つ. [Thesis]. Nagasaki University / 長崎大学; 2015. Available from: http://hdl.handle.net/10069/36049

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. LINDA WANG. Development of a novel Toll-like receptor-based two-hybrid assay for detecting protein-protein interactions and its application in the study of CD14 dimerization and FcyRIIA activation.

Degree: 2008, National University of Singapore

Subjects/Keywords: Toll-like receptor; dimerization; CD14; Fcgamma receptor IIA; NF-kB

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

WANG, L. (2008). Development of a novel Toll-like receptor-based two-hybrid assay for detecting protein-protein interactions and its application in the study of CD14 dimerization and FcyRIIA activation. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/13133

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

WANG, LINDA. “Development of a novel Toll-like receptor-based two-hybrid assay for detecting protein-protein interactions and its application in the study of CD14 dimerization and FcyRIIA activation.” 2008. Thesis, National University of Singapore. Accessed April 17, 2021. http://scholarbank.nus.edu.sg/handle/10635/13133.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

WANG, LINDA. “Development of a novel Toll-like receptor-based two-hybrid assay for detecting protein-protein interactions and its application in the study of CD14 dimerization and FcyRIIA activation.” 2008. Web. 17 Apr 2021.

Vancouver:

WANG L. Development of a novel Toll-like receptor-based two-hybrid assay for detecting protein-protein interactions and its application in the study of CD14 dimerization and FcyRIIA activation. [Internet] [Thesis]. National University of Singapore; 2008. [cited 2021 Apr 17]. Available from: http://scholarbank.nus.edu.sg/handle/10635/13133.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

WANG L. Development of a novel Toll-like receptor-based two-hybrid assay for detecting protein-protein interactions and its application in the study of CD14 dimerization and FcyRIIA activation. [Thesis]. National University of Singapore; 2008. Available from: http://scholarbank.nus.edu.sg/handle/10635/13133

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

21. Tong, Yanzheng. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.

Degree: PhD, Medical Sciences, 2014, Texas A&M University

 The NOD-like receptor protein NLRC5 inhibits TLR-induced NF-ҡB and viral infection-mediated type I interferon signaling in vitro. In the first project, we generated NLRC5 knockout… (more)

Subjects/Keywords: innate immunity; NLRC5; inflammasome; NF-kB signaling

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tong, Y. (2014). Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/186991

Chicago Manual of Style (16th Edition):

Tong, Yanzheng. “Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.” 2014. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/186991.

MLA Handbook (7th Edition):

Tong, Yanzheng. “Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses.” 2014. Web. 17 Apr 2021.

Vancouver:

Tong Y. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/186991.

Council of Science Editors:

Tong Y. Functional Characterization of NLRC5 and DDX46 in the Regulation of Innate Immune Responses. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/186991


University of Illinois – Urbana-Champaign

22. Lamb, Acacia. New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB.

Degree: PhD, 0318, 2012, University of Illinois – Urbana-Champaign

 Helicobacter pylori infection is the main cause of chronic gastritis, gastric ulcers and gastric cancer. The gram-negative spirochete, which infects more than half of the… (more)

Subjects/Keywords: Helicobacter pylori; NF-kB; TAK1; ubiquitination

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lamb, A. (2012). New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/31951

Chicago Manual of Style (16th Edition):

Lamb, Acacia. “New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed April 17, 2021. http://hdl.handle.net/2142/31951.

MLA Handbook (7th Edition):

Lamb, Acacia. “New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB.” 2012. Web. 17 Apr 2021.

Vancouver:

Lamb A. New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/2142/31951.

Council of Science Editors:

Lamb A. New insights into a complex host-pathogen interaction: mechanisms of Helicobacter pylori activation of NF-κB. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/31951


University of Southern California

23. Narula, Mansi. Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6.

Degree: MS, Molecular Microbiology and Immunology, 2013, University of Southern California

 Hepatitis C virus (HCV) is a major cause of chronic liver disease and evasion of the host immune system by HCV plays a key role… (more)

Subjects/Keywords: HCV; TRAF6; polyubiquitination; apoptosis; autophagy; NF-kB

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Narula, M. (2013). Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2179

Chicago Manual of Style (16th Edition):

Narula, Mansi. “Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6.” 2013. Masters Thesis, University of Southern California. Accessed April 17, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2179.

MLA Handbook (7th Edition):

Narula, Mansi. “Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6.” 2013. Web. 17 Apr 2021.

Vancouver:

Narula M. Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2021 Apr 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2179.

Council of Science Editors:

Narula M. Effect of hepatitis C virus infection on signal transduction adaptor protein: TRAF6. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/280450/rec/2179


Louisiana State University

24. Verhoeve, Victoria Irene. Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick.

Degree: PhD, Veterinary Pathology and Pathobiology, 2016, Louisiana State University

 Ticks are important worldwide as vectors of bacteria, viruses, and parasites. Pathogenic and non-pathogenic Spotted fever group (SFG) Rickettsia are maintained and transmitted by ticks… (more)

Subjects/Keywords: Rickettsia; immune response; NF-kB; tick

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Verhoeve, V. I. (2016). Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179

Chicago Manual of Style (16th Edition):

Verhoeve, Victoria Irene. “Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick.” 2016. Doctoral Dissertation, Louisiana State University. Accessed April 17, 2021. etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179.

MLA Handbook (7th Edition):

Verhoeve, Victoria Irene. “Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick.” 2016. Web. 17 Apr 2021.

Vancouver:

Verhoeve VI. Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick. [Internet] [Doctoral dissertation]. Louisiana State University; 2016. [cited 2021 Apr 17]. Available from: etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179.

Council of Science Editors:

Verhoeve VI. Identification and Characterization of a Relish-type NF-kB, DvRelish in Dermacentor variabilis, the American Dog Tick. [Doctoral Dissertation]. Louisiana State University; 2016. Available from: etd-04062016-103735 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1179


University of Texas Medical Branch – Galveston

25. [No author]. ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION .

Degree: University of Texas Medical Branch – Galveston

 Ataxia-Telangiectasia Mutated (ATM), a member of the phosphatidylinositol 3 kinase-like kinase family, is a master regulator of the double strand DNA break-repair pathway after genotoxic… (more)

Subjects/Keywords: NF-kB ATM

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

author], [. (n.d.). ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/11201

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION .” Thesis, University of Texas Medical Branch – Galveston. Accessed April 17, 2021. http://hdl.handle.net/2152.3/11201.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION .” Web. 17 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

author] [. ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; [cited 2021 Apr 17]. Available from: http://hdl.handle.net/2152.3/11201.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

author] [. ATM REGULATES THE NF-kB PATHWAY VIA RELA SER 276 PHOSPHORYLATION . [Thesis]. University of Texas Medical Branch – Galveston; Available from: http://hdl.handle.net/2152.3/11201

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Canterbury

26. Tino, Alexandria. An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer.

Degree: MS, Cellular and Molecular Biology, 2014, University of Canterbury

 Ovarian cancer is the deadliest gynaecologic cancer in New Zealand. Its high mortality rate is due to the fact that it is usually diagnosed at… (more)

Subjects/Keywords: ovarian cancer; resveratrol; EGCG; NF-kB

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tino, A. (2014). An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer. (Masters Thesis). University of Canterbury. Retrieved from http://dx.doi.org/10.26021/7096

Chicago Manual of Style (16th Edition):

Tino, Alexandria. “An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer.” 2014. Masters Thesis, University of Canterbury. Accessed April 17, 2021. http://dx.doi.org/10.26021/7096.

MLA Handbook (7th Edition):

Tino, Alexandria. “An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer.” 2014. Web. 17 Apr 2021.

Vancouver:

Tino A. An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer. [Internet] [Masters thesis]. University of Canterbury; 2014. [cited 2021 Apr 17]. Available from: http://dx.doi.org/10.26021/7096.

Council of Science Editors:

Tino A. An investigation into the anti-tumour properties and underlying mechanisms of natural polyphenols against ovarian cancer. [Masters Thesis]. University of Canterbury; 2014. Available from: http://dx.doi.org/10.26021/7096


Université Paris-Sud – Paris XI

27. Poalas, Konstantinos. Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB.

Degree: Docteur es, Biochimie, Biologie Cellulaire et Moléculaire, 2013, Université Paris-Sud – Paris XI

L’activation de la signalisation NF-κB par de nombreux immunorécepteurs met en jeu un large signalosome. Afin de propager cette signalisation en réponse à différents stimuli,… (more)

Subjects/Keywords: NF-kB; TCR; Ubiquitinylation; Déubiquitinylation; USP34; MTDH; NF-kB; TCR; Ubiquitinylation; Deubiquitinylation; USP34; MTDH

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Poalas, K. (2013). Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA11T058

Chicago Manual of Style (16th Edition):

Poalas, Konstantinos. “Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 17, 2021. http://www.theses.fr/2013PA11T058.

MLA Handbook (7th Edition):

Poalas, Konstantinos. “Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB.” 2013. Web. 17 Apr 2021.

Vancouver:

Poalas K. Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2021 Apr 17]. Available from: http://www.theses.fr/2013PA11T058.

Council of Science Editors:

Poalas K. Ubiquitinylation and deubiquitinylation in the regulation of the transcription factor NF-kB activation : Ubiquitinylation et déubiquitinylation dans la régulation de l’activation du facteur de transcription NF-kB. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA11T058

28. Senegas, Anna. Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease.

Degree: Docteur es, Neurosciences - Neurobiologie, 2016, Université Grenoble Alpes (ComUE)

Incontinentia pigmenti (IP, OMIM 308300) est une maladie génétique liée au chromosome X qui est létale chez les garçons. Chez les filles, une dermatose débute… (more)

Subjects/Keywords: Incontinentia pigmenti; Nemo; NF-KB; Neurobiologie; Incontinentia pigmenti; Nemo; NF-KB; Neurobiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Senegas, A. (2016). Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease. (Doctoral Dissertation). Université Grenoble Alpes (ComUE). Retrieved from http://www.theses.fr/2016GREAV075

Chicago Manual of Style (16th Edition):

Senegas, Anna. “Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease.” 2016. Doctoral Dissertation, Université Grenoble Alpes (ComUE). Accessed April 17, 2021. http://www.theses.fr/2016GREAV075.

MLA Handbook (7th Edition):

Senegas, Anna. “Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease.” 2016. Web. 17 Apr 2021.

Vancouver:

Senegas A. Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease. [Internet] [Doctoral dissertation]. Université Grenoble Alpes (ComUE); 2016. [cited 2021 Apr 17]. Available from: http://www.theses.fr/2016GREAV075.

Council of Science Editors:

Senegas A. Caractérisation des défauts cérébraux chez le modèle murin d'Incontinentia Pigmenti, maladie génétique liée à la voie NF-kB : Brain defects characterization of the mouse model of Incontinentia Pigmenti, a NF-kB-related genetic disease. [Doctoral Dissertation]. Université Grenoble Alpes (ComUE); 2016. Available from: http://www.theses.fr/2016GREAV075

29. Ronin, Émilie. Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells.

Degree: Docteur es, Immunologie, 2017, Université Pierre et Marie Curie – Paris VI

Les lymphocytes T CD4+FoxP3+ régulateurs (Tregs) jouent un rôle majeur dans l'homéostasie du système immunitaire et la prévention des maladies auto-immunes en régulant les réponses… (more)

Subjects/Keywords: Tregs; NF- KB; TNF; Auto-immunité; Cancer; Souris; Tregs; NF-KB; Autoimmunity; 616.994

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ronin, E. (2017). Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2017PA066222

Chicago Manual of Style (16th Edition):

Ronin, Émilie. “Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells.” 2017. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed April 17, 2021. http://www.theses.fr/2017PA066222.

MLA Handbook (7th Edition):

Ronin, Émilie. “Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells.” 2017. Web. 17 Apr 2021.

Vancouver:

Ronin E. Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. [cited 2021 Apr 17]. Available from: http://www.theses.fr/2017PA066222.

Council of Science Editors:

Ronin E. Etude du rôle de NF-kB dans les lymphocytes T régulateurs chez la souris : Investigation of the role of NF-kB in mouse regulatory T cells. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. Available from: http://www.theses.fr/2017PA066222

30. Mhaidly, Rana. Effet de la surexpression de la glycéraldéhyde 3-phosphate déshydrogénase (GAPDH) sur le développement et la fonction des lymphocytes T : Impact of GAPH overexpression on T cell development and function.

Degree: Docteur es, Sciences de la vie et de la santé, 2019, Université Côte d'Azur (ComUE)

L’activation des lymphocytes T (LT) et l’acquisition des fonctions effectrices sont des phénomènes récemment décrits pour être régulés par le métabolisme cellulaire, introduisant ainsi la… (more)

Subjects/Keywords: GAPDH; AITL; Lymphocytes T; NF-kB; GAPDH; AITL; T cell Lymphocyte; NF-kB pathway

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mhaidly, R. (2019). Effet de la surexpression de la glycéraldéhyde 3-phosphate déshydrogénase (GAPDH) sur le développement et la fonction des lymphocytes T : Impact of GAPH overexpression on T cell development and function. (Doctoral Dissertation). Université Côte d'Azur (ComUE). Retrieved from http://www.theses.fr/2019AZUR6021

Chicago Manual of Style (16th Edition):

Mhaidly, Rana. “Effet de la surexpression de la glycéraldéhyde 3-phosphate déshydrogénase (GAPDH) sur le développement et la fonction des lymphocytes T : Impact of GAPH overexpression on T cell development and function.” 2019. Doctoral Dissertation, Université Côte d'Azur (ComUE). Accessed April 17, 2021. http://www.theses.fr/2019AZUR6021.

MLA Handbook (7th Edition):

Mhaidly, Rana. “Effet de la surexpression de la glycéraldéhyde 3-phosphate déshydrogénase (GAPDH) sur le développement et la fonction des lymphocytes T : Impact of GAPH overexpression on T cell development and function.” 2019. Web. 17 Apr 2021.

Vancouver:

Mhaidly R. Effet de la surexpression de la glycéraldéhyde 3-phosphate déshydrogénase (GAPDH) sur le développement et la fonction des lymphocytes T : Impact of GAPH overexpression on T cell development and function. [Internet] [Doctoral dissertation]. Université Côte d'Azur (ComUE); 2019. [cited 2021 Apr 17]. Available from: http://www.theses.fr/2019AZUR6021.

Council of Science Editors:

Mhaidly R. Effet de la surexpression de la glycéraldéhyde 3-phosphate déshydrogénase (GAPDH) sur le développement et la fonction des lymphocytes T : Impact of GAPH overexpression on T cell development and function. [Doctoral Dissertation]. Université Côte d'Azur (ComUE); 2019. Available from: http://www.theses.fr/2019AZUR6021

[1] [2] [3] [4] [5] … [10050]

.