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You searched for subject:(Receptor Insulin physiology mesh ). Showing records 1 – 30 of 29295 total matches.

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University of Florida

1. Boyd, Frederick Tilghman, 1959-. Characterization and physiological significance of brain insulin receptors.

Degree: 1985, University of Florida

Subjects/Keywords: Astrocytes; Brain; Cells; Cultured cells; Insulin; Neuroglia; Neurons; Norepinephrine; Rats; Receptors; Binding Sites ( mesh ); Brain Chemistry ( mesh ); Physiology thesis Ph.D ( mesh ); Receptor, Insulin  – physiology ( mesh )

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APA (6th Edition):

Boyd, Frederick Tilghman, 1. (1985). Characterization and physiological significance of brain insulin receptors. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00053670

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boyd, Frederick Tilghman, 1959-. “Characterization and physiological significance of brain insulin receptors.” 1985. Thesis, University of Florida. Accessed January 16, 2021. https://ufdc.ufl.edu/AA00053670.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boyd, Frederick Tilghman, 1959-. “Characterization and physiological significance of brain insulin receptors.” 1985. Web. 16 Jan 2021.

Vancouver:

Boyd, Frederick Tilghman 1. Characterization and physiological significance of brain insulin receptors. [Internet] [Thesis]. University of Florida; 1985. [cited 2021 Jan 16]. Available from: https://ufdc.ufl.edu/AA00053670.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boyd, Frederick Tilghman 1. Characterization and physiological significance of brain insulin receptors. [Thesis]. University of Florida; 1985. Available from: https://ufdc.ufl.edu/AA00053670

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Canterbury

2. Subramanian, Kannan. Kinetics of insulin - insulin receptor interaction using a surface plasmon resonance (SPR).

Degree: PhD, Chemical Engineering, 2014, University of Canterbury

 Type 2 diabetes or adult onset diabetes, has been a global epidemic for the past two decades, and the number of new cases accelerates every… (more)

Subjects/Keywords: insulin; insulin-receptor; kinetics; surface plasmon resonance

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APA (6th Edition):

Subramanian, K. (2014). Kinetics of insulin - insulin receptor interaction using a surface plasmon resonance (SPR). (Doctoral Dissertation). University of Canterbury. Retrieved from http://dx.doi.org/10.26021/2927

Chicago Manual of Style (16th Edition):

Subramanian, Kannan. “Kinetics of insulin - insulin receptor interaction using a surface plasmon resonance (SPR).” 2014. Doctoral Dissertation, University of Canterbury. Accessed January 16, 2021. http://dx.doi.org/10.26021/2927.

MLA Handbook (7th Edition):

Subramanian, Kannan. “Kinetics of insulin - insulin receptor interaction using a surface plasmon resonance (SPR).” 2014. Web. 16 Jan 2021.

Vancouver:

Subramanian K. Kinetics of insulin - insulin receptor interaction using a surface plasmon resonance (SPR). [Internet] [Doctoral dissertation]. University of Canterbury; 2014. [cited 2021 Jan 16]. Available from: http://dx.doi.org/10.26021/2927.

Council of Science Editors:

Subramanian K. Kinetics of insulin - insulin receptor interaction using a surface plasmon resonance (SPR). [Doctoral Dissertation]. University of Canterbury; 2014. Available from: http://dx.doi.org/10.26021/2927


University of Toronto

3. Lino, Marsel. The rRole of Intestinal Scavenger Receptor Class B Type I in Chylomicron Production in Normal and Insulin Resistant States.

Degree: 2012, University of Toronto

In recent years, studies have revealed a central role for the intestine in regulation of lipid homeostasis and development of insulin resistance and type-2 diabetes.… (more)

Subjects/Keywords: Lipoprotein Metabolism; Insulin Resistance; Intestinal Physiology; Scavenger Receptor Class B Type I; 0719; 0307; 0433; 0571

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APA (6th Edition):

Lino, M. (2012). The rRole of Intestinal Scavenger Receptor Class B Type I in Chylomicron Production in Normal and Insulin Resistant States. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42399

Chicago Manual of Style (16th Edition):

Lino, Marsel. “The rRole of Intestinal Scavenger Receptor Class B Type I in Chylomicron Production in Normal and Insulin Resistant States.” 2012. Masters Thesis, University of Toronto. Accessed January 16, 2021. http://hdl.handle.net/1807/42399.

MLA Handbook (7th Edition):

Lino, Marsel. “The rRole of Intestinal Scavenger Receptor Class B Type I in Chylomicron Production in Normal and Insulin Resistant States.” 2012. Web. 16 Jan 2021.

Vancouver:

Lino M. The rRole of Intestinal Scavenger Receptor Class B Type I in Chylomicron Production in Normal and Insulin Resistant States. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1807/42399.

Council of Science Editors:

Lino M. The rRole of Intestinal Scavenger Receptor Class B Type I in Chylomicron Production in Normal and Insulin Resistant States. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/42399


University of Adelaide

4. Bonython, Eric Richard. Investigation of insulin-like receptor systems.

Degree: 2005, University of Adelaide

 The insulin and insulin-like growth factor receptor (IR and IGF-lR respectively) networks are ancient and fundamental systems that control growth and metabolism in multicellular organisms.… (more)

Subjects/Keywords: insulin; receptor

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APA (6th Edition):

Bonython, E. R. (2005). Investigation of insulin-like receptor systems. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/59217

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bonython, Eric Richard. “Investigation of insulin-like receptor systems.” 2005. Thesis, University of Adelaide. Accessed January 16, 2021. http://hdl.handle.net/2440/59217.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bonython, Eric Richard. “Investigation of insulin-like receptor systems.” 2005. Web. 16 Jan 2021.

Vancouver:

Bonython ER. Investigation of insulin-like receptor systems. [Internet] [Thesis]. University of Adelaide; 2005. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2440/59217.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bonython ER. Investigation of insulin-like receptor systems. [Thesis]. University of Adelaide; 2005. Available from: http://hdl.handle.net/2440/59217

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Fulzele, Keertik. Insulin signaling and function in osteoblasts.

Degree: PhD, 2009, University of Alabama – Birmingham

Insulin and insulin-like growth factor-1 (IGF-1) are evolutionarily conserved hormonal signaling pathways with structurally similar ligands and receptors. Recent studies suggest that that insulin and… (more)

Subjects/Keywords: Insulin  – metabolism<; br>; Osteoblasts  – metabolism<; br>; Osteogenesis  – physiology<; br>; Receptor, IGF Type 1  – metabolism<; br>; Receptor, Insulin  – metabolism<; br>; Signal Transduction  – physiology<; br>; Skull  – metabolism

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APA (6th Edition):

Fulzele, K. (2009). Insulin signaling and function in osteoblasts. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,999

Chicago Manual of Style (16th Edition):

Fulzele, Keertik. “Insulin signaling and function in osteoblasts.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 16, 2021. http://contentdm.mhsl.uab.edu/u?/etd,999.

MLA Handbook (7th Edition):

Fulzele, Keertik. “Insulin signaling and function in osteoblasts.” 2009. Web. 16 Jan 2021.

Vancouver:

Fulzele K. Insulin signaling and function in osteoblasts. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Jan 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,999.

Council of Science Editors:

Fulzele K. Insulin signaling and function in osteoblasts. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,999


University of Florida

6. Masters, Brian Andrew, 1962-. Insulin-like growth factors in cell differentiation and development in the nervous system.

Degree: 1990, University of Florida

Subjects/Keywords: Astrocytes; Brain; Cultured cells; Insulin; Neurons; Oligodendroglia; Progenitor cells; Rats; Receptors; Somatomedins; Department of Physiology thesis Ph.D ( mesh ); Insulin-Like Growth Factor I ( mesh ); Nervous System  – growth &; development ( mesh ); Oligodendroglia ( mesh ); Receptors, Somatomedin ( mesh ); Research ( mesh )

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APA (6th Edition):

Masters, Brian Andrew, 1. (1990). Insulin-like growth factors in cell differentiation and development in the nervous system. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00056906

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Masters, Brian Andrew, 1962-. “Insulin-like growth factors in cell differentiation and development in the nervous system.” 1990. Thesis, University of Florida. Accessed January 16, 2021. https://ufdc.ufl.edu/AA00056906.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Masters, Brian Andrew, 1962-. “Insulin-like growth factors in cell differentiation and development in the nervous system.” 1990. Web. 16 Jan 2021.

Vancouver:

Masters, Brian Andrew 1. Insulin-like growth factors in cell differentiation and development in the nervous system. [Internet] [Thesis]. University of Florida; 1990. [cited 2021 Jan 16]. Available from: https://ufdc.ufl.edu/AA00056906.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Masters, Brian Andrew 1. Insulin-like growth factors in cell differentiation and development in the nervous system. [Thesis]. University of Florida; 1990. Available from: https://ufdc.ufl.edu/AA00056906

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

7. Richards-Sumners, Elaine Mary, 1957-. Catecholamine and angiotensin II interactions in primary cultures of brain cells.

Degree: 1988, University of Florida

Subjects/Keywords: Brain; Catecholamines; Cultured cells; Incubation; Insulin; Neuroglia; Neurons; Norepinephrine; Rats; Receptors; Angiotensin II  – physiology ( mesh ); Brain Chemistry ( mesh ); Catecholamines  – physiology ( mesh ); Cells, Cultured ( mesh ); Physiology thesis Ph.D ( mesh )

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APA (6th Edition):

Richards-Sumners, Elaine Mary, 1. (1988). Catecholamine and angiotensin II interactions in primary cultures of brain cells. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00054447

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Richards-Sumners, Elaine Mary, 1957-. “Catecholamine and angiotensin II interactions in primary cultures of brain cells.” 1988. Thesis, University of Florida. Accessed January 16, 2021. https://ufdc.ufl.edu/AA00054447.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Richards-Sumners, Elaine Mary, 1957-. “Catecholamine and angiotensin II interactions in primary cultures of brain cells.” 1988. Web. 16 Jan 2021.

Vancouver:

Richards-Sumners, Elaine Mary 1. Catecholamine and angiotensin II interactions in primary cultures of brain cells. [Internet] [Thesis]. University of Florida; 1988. [cited 2021 Jan 16]. Available from: https://ufdc.ufl.edu/AA00054447.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Richards-Sumners, Elaine Mary 1. Catecholamine and angiotensin II interactions in primary cultures of brain cells. [Thesis]. University of Florida; 1988. Available from: https://ufdc.ufl.edu/AA00054447

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

8. Bowman, Mark Andrew. Environmental agents and pathogenic mechanisms of insulin dependent diabetes.

Degree: 1995, University of Florida

Subjects/Keywords: Animal models; Antigens; Beta cells; Diabetes; Diabetes complications; Diabetes mellitus; Diseases; Insulin; Mice; Type 1 diabetes mellitus; Department of Pathology and Laboratory Medicine thesis Ph.D ( mesh ); Diabetes Mellitus, Type I  – etiology ( mesh ); Diabetes Mellitus, Type I  – physiopathology ( mesh ); Glutamate Decarboxylase  – physiology ( mesh ); Insulin  – physiology ( mesh ); Insulin  – therapeutic use ( mesh ); Mice, Inbred NOD ( mesh ); Nitric Oxide  – physiology ( mesh ); Nitric-Oxide Synthase  – physiology ( mesh ); Research ( mesh ); Serum Albumin, Bovine  – pharmacology ( mesh )

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APA (6th Edition):

Bowman, M. A. (1995). Environmental agents and pathogenic mechanisms of insulin dependent diabetes. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00053824

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bowman, Mark Andrew. “Environmental agents and pathogenic mechanisms of insulin dependent diabetes.” 1995. Thesis, University of Florida. Accessed January 16, 2021. https://ufdc.ufl.edu/AA00053824.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bowman, Mark Andrew. “Environmental agents and pathogenic mechanisms of insulin dependent diabetes.” 1995. Web. 16 Jan 2021.

Vancouver:

Bowman MA. Environmental agents and pathogenic mechanisms of insulin dependent diabetes. [Internet] [Thesis]. University of Florida; 1995. [cited 2021 Jan 16]. Available from: https://ufdc.ufl.edu/AA00053824.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bowman MA. Environmental agents and pathogenic mechanisms of insulin dependent diabetes. [Thesis]. University of Florida; 1995. Available from: https://ufdc.ufl.edu/AA00053824

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

9. Fagan, Dedra Hannah. Examination of molecular changes in acquired tamoxifen resistance and subsequent response to anti-IGF1R therapy.

Degree: PhD, 2012, University of Minnesota

 The type-I insulin like growth factor (IGF1R) contributes to the proliferation, survival, and metastasis of breast cancer cells. Disruption of IGF1R signaling alone or in… (more)

Subjects/Keywords: Endocrine resistance; IGF1R; Insulin receptor; Pharmacology

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APA (6th Edition):

Fagan, D. H. (2012). Examination of molecular changes in acquired tamoxifen resistance and subsequent response to anti-IGF1R therapy. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/131512

Chicago Manual of Style (16th Edition):

Fagan, Dedra Hannah. “Examination of molecular changes in acquired tamoxifen resistance and subsequent response to anti-IGF1R therapy.” 2012. Doctoral Dissertation, University of Minnesota. Accessed January 16, 2021. http://purl.umn.edu/131512.

MLA Handbook (7th Edition):

Fagan, Dedra Hannah. “Examination of molecular changes in acquired tamoxifen resistance and subsequent response to anti-IGF1R therapy.” 2012. Web. 16 Jan 2021.

Vancouver:

Fagan DH. Examination of molecular changes in acquired tamoxifen resistance and subsequent response to anti-IGF1R therapy. [Internet] [Doctoral dissertation]. University of Minnesota; 2012. [cited 2021 Jan 16]. Available from: http://purl.umn.edu/131512.

Council of Science Editors:

Fagan DH. Examination of molecular changes in acquired tamoxifen resistance and subsequent response to anti-IGF1R therapy. [Doctoral Dissertation]. University of Minnesota; 2012. Available from: http://purl.umn.edu/131512


University of Iowa

10. Starks, Rachel Diaz. Molecular basis of insulin resistance in Bardet Biedl syndrome.

Degree: PhD, Molecular and Cell Biology, 2015, University of Iowa

  Bardet Biedl Syndrome (BBS) displays heterogeneity in the genes involved and clinical features. Mutations in 19 genes have been associated with BBS. Eight BBS… (more)

Subjects/Keywords: BBSome; BBS proteins; glucose homeostasis; insulin receptor; insulin resistance; Cell Biology

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APA (6th Edition):

Starks, R. D. (2015). Molecular basis of insulin resistance in Bardet Biedl syndrome. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5641

Chicago Manual of Style (16th Edition):

Starks, Rachel Diaz. “Molecular basis of insulin resistance in Bardet Biedl syndrome.” 2015. Doctoral Dissertation, University of Iowa. Accessed January 16, 2021. https://ir.uiowa.edu/etd/5641.

MLA Handbook (7th Edition):

Starks, Rachel Diaz. “Molecular basis of insulin resistance in Bardet Biedl syndrome.” 2015. Web. 16 Jan 2021.

Vancouver:

Starks RD. Molecular basis of insulin resistance in Bardet Biedl syndrome. [Internet] [Doctoral dissertation]. University of Iowa; 2015. [cited 2021 Jan 16]. Available from: https://ir.uiowa.edu/etd/5641.

Council of Science Editors:

Starks RD. Molecular basis of insulin resistance in Bardet Biedl syndrome. [Doctoral Dissertation]. University of Iowa; 2015. Available from: https://ir.uiowa.edu/etd/5641


Cleveland State University

11. Liu, Danting. RNASE L MEDIATES GLUCOSE HOMEOSTASIS THROUGH REGULATING THE INSULIN SIGNALING PATHWAY.

Degree: PhD, College of Sciences and Health Professions, 2018, Cleveland State University

 Diabetes is characterized by hyperglycemia mainly due to defect in insulin secretion and/or action. Regulation of glucose transport and use by insulin is central to… (more)

Subjects/Keywords: Biochemistry; Molecular Biology; RNASE L; Insulin Resistance; Insulin Receptor

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APA (6th Edition):

Liu, D. (2018). RNASE L MEDIATES GLUCOSE HOMEOSTASIS THROUGH REGULATING THE INSULIN SIGNALING PATHWAY. (Doctoral Dissertation). Cleveland State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=csu1544627440336052

Chicago Manual of Style (16th Edition):

Liu, Danting. “RNASE L MEDIATES GLUCOSE HOMEOSTASIS THROUGH REGULATING THE INSULIN SIGNALING PATHWAY.” 2018. Doctoral Dissertation, Cleveland State University. Accessed January 16, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=csu1544627440336052.

MLA Handbook (7th Edition):

Liu, Danting. “RNASE L MEDIATES GLUCOSE HOMEOSTASIS THROUGH REGULATING THE INSULIN SIGNALING PATHWAY.” 2018. Web. 16 Jan 2021.

Vancouver:

Liu D. RNASE L MEDIATES GLUCOSE HOMEOSTASIS THROUGH REGULATING THE INSULIN SIGNALING PATHWAY. [Internet] [Doctoral dissertation]. Cleveland State University; 2018. [cited 2021 Jan 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1544627440336052.

Council of Science Editors:

Liu D. RNASE L MEDIATES GLUCOSE HOMEOSTASIS THROUGH REGULATING THE INSULIN SIGNALING PATHWAY. [Doctoral Dissertation]. Cleveland State University; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1544627440336052


University of Florida

12. Olson, John Ackerman, 1964-. Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain.

Degree: 1990, University of Florida

Subjects/Keywords: Astrocytes; Brain; Cell growth; Cells; Cultural studies; Cultured cells; Neurons; Rats; Receptors; Secretion; Angiotensin II  – physiology ( mesh ); Astrocytes  – growth &; development ( mesh ); Astrocytes  – secretion ( mesh ); Brain Chemistry ( mesh ); Department of Pharmacology and Therapeutics thesis Ph.D ( mesh ); Insulin-Like Growth Factor-Binding Protein 2 ( mesh ); Proteins  – secretion ( mesh ); Rats ( mesh ); Research ( mesh )

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APA (6th Edition):

Olson, John Ackerman, 1. (1990). Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00022809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Olson, John Ackerman, 1964-. “Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain.” 1990. Thesis, University of Florida. Accessed January 16, 2021. https://ufdc.ufl.edu/AA00022809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Olson, John Ackerman, 1964-. “Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain.” 1990. Web. 16 Jan 2021.

Vancouver:

Olson, John Ackerman 1. Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain. [Internet] [Thesis]. University of Florida; 1990. [cited 2021 Jan 16]. Available from: https://ufdc.ufl.edu/AA00022809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Olson, John Ackerman 1. Developmental regulation of constitutive and angiotensin II-induced protein secretion by astrocytes of the brain. [Thesis]. University of Florida; 1990. Available from: https://ufdc.ufl.edu/AA00022809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

13. Cervantes, David. Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation.

Degree: PhD, 0325, 2012, University of Illinois – Urbana-Champaign

 Heart disease and diabetes mellitus are growing epidemics, consistently ranking within the top ten causes of death in the United States. Both diseases are associated… (more)

Subjects/Keywords: adrenergic receptor; insulin receptor; extracellular signal-regulated kinase (ERK); heart; arrestin

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APA (6th Edition):

Cervantes, D. (2012). Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/29837

Chicago Manual of Style (16th Edition):

Cervantes, David. “Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed January 16, 2021. http://hdl.handle.net/2142/29837.

MLA Handbook (7th Edition):

Cervantes, David. “Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation.” 2012. Web. 16 Jan 2021.

Vancouver:

Cervantes D. Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2142/29837.

Council of Science Editors:

Cervantes D. Modulation of mitogenic signaling and growth by sympathetic adrenergic regulation. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/29837


Texas A&M University

14. Lu, Hsiao Ling. Vitellogenin Receptor and Neuropeptide Receptors Involved in Reproduction of the Red Imported Fire Ant (Solenopsis invicta Buren).

Degree: PhD, Entomology, 2012, Texas A&M University

 Social insects have complex forms of social organization. Molecular mechanisms involved in the regulation of their reproduction are not fully understood. This dissertation investigated the… (more)

Subjects/Keywords: Fire ants; vitellogenin receptor; short neuropeptide F receptor; insulin receptor; queen reproduction

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APA (6th Edition):

Lu, H. L. (2012). Vitellogenin Receptor and Neuropeptide Receptors Involved in Reproduction of the Red Imported Fire Ant (Solenopsis invicta Buren). (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2011-12-10218

Chicago Manual of Style (16th Edition):

Lu, Hsiao Ling. “Vitellogenin Receptor and Neuropeptide Receptors Involved in Reproduction of the Red Imported Fire Ant (Solenopsis invicta Buren).” 2012. Doctoral Dissertation, Texas A&M University. Accessed January 16, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-2011-12-10218.

MLA Handbook (7th Edition):

Lu, Hsiao Ling. “Vitellogenin Receptor and Neuropeptide Receptors Involved in Reproduction of the Red Imported Fire Ant (Solenopsis invicta Buren).” 2012. Web. 16 Jan 2021.

Vancouver:

Lu HL. Vitellogenin Receptor and Neuropeptide Receptors Involved in Reproduction of the Red Imported Fire Ant (Solenopsis invicta Buren). [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-12-10218.

Council of Science Editors:

Lu HL. Vitellogenin Receptor and Neuropeptide Receptors Involved in Reproduction of the Red Imported Fire Ant (Solenopsis invicta Buren). [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-12-10218


University of Toledo Health Science Campus

15. Wang, Mengjie. Brain Insulin-Like Growth Factor 1 Receptor and Insulin Receptor in Metabolism and Reproduction.

Degree: PhD, Biomedical Sciences (Molecular Medicine), 2019, University of Toledo Health Science Campus

Insulin-like growth factor 1 (IGF-1) and insulin exert biological effects through highly homologous tyrosine kinase receptors, which are ubiquitously expressed in rodents. During the last… (more)

Subjects/Keywords: Biomedical Research; Insulin-like growth factor 1 receptor; insulin receptor; leptin receptor-expressing neuron; kisspeptin neuron; metabolism; reproduction; gut micriobiota

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APA (6th Edition):

Wang, M. (2019). Brain Insulin-Like Growth Factor 1 Receptor and Insulin Receptor in Metabolism and Reproduction. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1564676824418256

Chicago Manual of Style (16th Edition):

Wang, Mengjie. “Brain Insulin-Like Growth Factor 1 Receptor and Insulin Receptor in Metabolism and Reproduction.” 2019. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed January 16, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=mco1564676824418256.

MLA Handbook (7th Edition):

Wang, Mengjie. “Brain Insulin-Like Growth Factor 1 Receptor and Insulin Receptor in Metabolism and Reproduction.” 2019. Web. 16 Jan 2021.

Vancouver:

Wang M. Brain Insulin-Like Growth Factor 1 Receptor and Insulin Receptor in Metabolism and Reproduction. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2019. [cited 2021 Jan 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1564676824418256.

Council of Science Editors:

Wang M. Brain Insulin-Like Growth Factor 1 Receptor and Insulin Receptor in Metabolism and Reproduction. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2019. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1564676824418256

16. Walton, R. Grace. Role of NR4A3 nuclear receptor in physiological regulation of metabolism.

Degree: PhD, 2011, University of Alabama – Birmingham

Nuclear receptor NR4A3 (NOR-1, MINOR) mediates transcriptional responses to {esc}gb{esc}s-adrenergic signaling, is increased in insulin sensitive mice and humans, and enhances insulin-stimulated glucose transport in… (more)

Subjects/Keywords: Diabetes Mellitus, Type 2<; br>; Energy Metabolism – physiology<; br>; Insulin Resistance<; br>; Muscle Proteins – metabolism.<; br>; Muscle, Skeletal – physiology.<; br>; Nuclear Receptor Subfamily 4, Group A, Member 3.<; br>; Oxygen Consumption – physiology<; br>; Rest – physiology<; br>; Signal Transduction – physiology

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APA (6th Edition):

Walton, R. G. (2011). Role of NR4A3 nuclear receptor in physiological regulation of metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1817

Chicago Manual of Style (16th Edition):

Walton, R Grace. “Role of NR4A3 nuclear receptor in physiological regulation of metabolism.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 16, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1817.

MLA Handbook (7th Edition):

Walton, R Grace. “Role of NR4A3 nuclear receptor in physiological regulation of metabolism.” 2011. Web. 16 Jan 2021.

Vancouver:

Walton RG. Role of NR4A3 nuclear receptor in physiological regulation of metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2021 Jan 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1817.

Council of Science Editors:

Walton RG. Role of NR4A3 nuclear receptor in physiological regulation of metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1817


University of Michigan

17. Uhm, Maeran. Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport.

Degree: PhD, Molecular and Integrative Physiology, 2015, University of Michigan

Insulin stimulates glucose uptake in muscle and fat by promoting translocation of the facilitative transporter GLUT4 from intracellular compartments to the plasma membrane. While the… (more)

Subjects/Keywords: Insulin-stimulated glucose transport; Physiology; Health Sciences

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APA (6th Edition):

Uhm, M. (2015). Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/113634

Chicago Manual of Style (16th Edition):

Uhm, Maeran. “Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport.” 2015. Doctoral Dissertation, University of Michigan. Accessed January 16, 2021. http://hdl.handle.net/2027.42/113634.

MLA Handbook (7th Edition):

Uhm, Maeran. “Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport.” 2015. Web. 16 Jan 2021.

Vancouver:

Uhm M. Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport. [Internet] [Doctoral dissertation]. University of Michigan; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2027.42/113634.

Council of Science Editors:

Uhm M. Role of the Protein Kinase TBK1 in Insulin-Stimulated Glucose Transport. [Doctoral Dissertation]. University of Michigan; 2015. Available from: http://hdl.handle.net/2027.42/113634


Montana State University

18. Ackerman, Kimberly Lynn. The statistical variance of blood glucose levels of medicial intensive care unit patients while on an insulin infusion protocol.

Degree: M Nursing, College of Nursing, 2006, Montana State University

 Hyperglycemia has been shown to have many negative consequences in the critically ill patient. Many physicians and nurses have been searching for ways to provide… (more)

Subjects/Keywords: Insulin.; Physiology.; Hyperglycemia.

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APA (6th Edition):

Ackerman, K. L. (2006). The statistical variance of blood glucose levels of medicial intensive care unit patients while on an insulin infusion protocol. (Thesis). Montana State University. Retrieved from https://scholarworks.montana.edu/xmlui/handle/1/803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ackerman, Kimberly Lynn. “The statistical variance of blood glucose levels of medicial intensive care unit patients while on an insulin infusion protocol.” 2006. Thesis, Montana State University. Accessed January 16, 2021. https://scholarworks.montana.edu/xmlui/handle/1/803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ackerman, Kimberly Lynn. “The statistical variance of blood glucose levels of medicial intensive care unit patients while on an insulin infusion protocol.” 2006. Web. 16 Jan 2021.

Vancouver:

Ackerman KL. The statistical variance of blood glucose levels of medicial intensive care unit patients while on an insulin infusion protocol. [Internet] [Thesis]. Montana State University; 2006. [cited 2021 Jan 16]. Available from: https://scholarworks.montana.edu/xmlui/handle/1/803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ackerman KL. The statistical variance of blood glucose levels of medicial intensive care unit patients while on an insulin infusion protocol. [Thesis]. Montana State University; 2006. Available from: https://scholarworks.montana.edu/xmlui/handle/1/803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

19. Prodanović, Radiša, 1981-. Insulinska rezistencija kod krava Holštajn rase tokom perioda zasušenja i rane laktacije.

Degree: Fakultet veterinarske medicine, 2015, Univerzitet u Beogradu

Veterinarska medicina - Bolesti papkara / Veterinary Medicine - Ruminants and Swine Diseases

Cilj istraživanja u okviru ove disertacije je bio da se ispita da… (more)

Subjects/Keywords: high-yielding dairy cows; body condition; insulin resistance; glucose tolerance test; insulin receptor; GLUT 4

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APA (6th Edition):

Prodanović, Radiša, 1. (2015). Insulinska rezistencija kod krava Holštajn rase tokom perioda zasušenja i rane laktacije. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:8045/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Prodanović, Radiša, 1981-. “Insulinska rezistencija kod krava Holštajn rase tokom perioda zasušenja i rane laktacije.” 2015. Thesis, Univerzitet u Beogradu. Accessed January 16, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:8045/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Prodanović, Radiša, 1981-. “Insulinska rezistencija kod krava Holštajn rase tokom perioda zasušenja i rane laktacije.” 2015. Web. 16 Jan 2021.

Vancouver:

Prodanović, Radiša 1. Insulinska rezistencija kod krava Holštajn rase tokom perioda zasušenja i rane laktacije. [Internet] [Thesis]. Univerzitet u Beogradu; 2015. [cited 2021 Jan 16]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:8045/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Prodanović, Radiša 1. Insulinska rezistencija kod krava Holštajn rase tokom perioda zasušenja i rane laktacije. [Thesis]. Univerzitet u Beogradu; 2015. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:8045/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

20. Oakie, Amanda. The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion.

Degree: 2019, University of Western Ontario

 The receptor tyrosine kinases (RTKs) c-Kit and insulin receptor (IR) initiate similar intracellular signalling pathways in pancreatic beta cells to regulate beta cell proliferation, survival,… (more)

Subjects/Keywords: c-Kit; insulin receptor; diabetes mellitus; insulin secretion; SNARE protein; Cre recombinase; Endocrinology

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APA (6th Edition):

Oakie, A. (2019). The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/6232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oakie, Amanda. “The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion.” 2019. Thesis, University of Western Ontario. Accessed January 16, 2021. https://ir.lib.uwo.ca/etd/6232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oakie, Amanda. “The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion.” 2019. Web. 16 Jan 2021.

Vancouver:

Oakie A. The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion. [Internet] [Thesis]. University of Western Ontario; 2019. [cited 2021 Jan 16]. Available from: https://ir.lib.uwo.ca/etd/6232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oakie A. The role of c-Kit and insulin receptor tyrosine kinases in beta cell function and insulin secretion. [Thesis]. University of Western Ontario; 2019. Available from: https://ir.lib.uwo.ca/etd/6232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

21. Ananthakrishnan, Kameswari. Improved β-Cell Targeting and Therapeutics Using Multivalent Glucagon-Like Peptide-1 (GLP-1) Linked to the α2AR Antagonist Yohimbine (YHB): Evaluating the Binding, Selectivity and Signaling .

Degree: 2016, University of Arizona

 Diabetes Mellitus (DM) is a metabolic disorder in which the body fails to achieve glucose homeostasis, due to either insulin resistance or reduced insulin secretion… (more)

Subjects/Keywords: GLP-1; Insulin secretion; Multivalency; Receptor targeting; β-cell imaging; Adrenergic receptor

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APA (6th Edition):

Ananthakrishnan, K. (2016). Improved β-Cell Targeting and Therapeutics Using Multivalent Glucagon-Like Peptide-1 (GLP-1) Linked to the α2AR Antagonist Yohimbine (YHB): Evaluating the Binding, Selectivity and Signaling . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/623004

Chicago Manual of Style (16th Edition):

Ananthakrishnan, Kameswari. “Improved β-Cell Targeting and Therapeutics Using Multivalent Glucagon-Like Peptide-1 (GLP-1) Linked to the α2AR Antagonist Yohimbine (YHB): Evaluating the Binding, Selectivity and Signaling .” 2016. Doctoral Dissertation, University of Arizona. Accessed January 16, 2021. http://hdl.handle.net/10150/623004.

MLA Handbook (7th Edition):

Ananthakrishnan, Kameswari. “Improved β-Cell Targeting and Therapeutics Using Multivalent Glucagon-Like Peptide-1 (GLP-1) Linked to the α2AR Antagonist Yohimbine (YHB): Evaluating the Binding, Selectivity and Signaling .” 2016. Web. 16 Jan 2021.

Vancouver:

Ananthakrishnan K. Improved β-Cell Targeting and Therapeutics Using Multivalent Glucagon-Like Peptide-1 (GLP-1) Linked to the α2AR Antagonist Yohimbine (YHB): Evaluating the Binding, Selectivity and Signaling . [Internet] [Doctoral dissertation]. University of Arizona; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10150/623004.

Council of Science Editors:

Ananthakrishnan K. Improved β-Cell Targeting and Therapeutics Using Multivalent Glucagon-Like Peptide-1 (GLP-1) Linked to the α2AR Antagonist Yohimbine (YHB): Evaluating the Binding, Selectivity and Signaling . [Doctoral Dissertation]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/623004


Ryerson University

22. Bone, Leslie N. The acyltransferase lycat controls specific phosphoinositides and related membrane traffic and hormone receptor signaling.

Degree: 2017, Ryerson University

 Phosphoinositdes (PIPs) are a group of signaling phospholipids involved in regulating many cellular processes, including organelle dynamics, nutrient uptake, autophagy and apoptosis. Through the action… (more)

Subjects/Keywords: Phosphoinositides; Phospholipids; Acyltransferases; Hormone receptor; Cell physiology

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APA (6th Edition):

Bone, L. N. (2017). The acyltransferase lycat controls specific phosphoinositides and related membrane traffic and hormone receptor signaling. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A7219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bone, Leslie N. “The acyltransferase lycat controls specific phosphoinositides and related membrane traffic and hormone receptor signaling.” 2017. Thesis, Ryerson University. Accessed January 16, 2021. https://digital.library.ryerson.ca/islandora/object/RULA%3A7219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bone, Leslie N. “The acyltransferase lycat controls specific phosphoinositides and related membrane traffic and hormone receptor signaling.” 2017. Web. 16 Jan 2021.

Vancouver:

Bone LN. The acyltransferase lycat controls specific phosphoinositides and related membrane traffic and hormone receptor signaling. [Internet] [Thesis]. Ryerson University; 2017. [cited 2021 Jan 16]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A7219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bone LN. The acyltransferase lycat controls specific phosphoinositides and related membrane traffic and hormone receptor signaling. [Thesis]. Ryerson University; 2017. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A7219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Queensland

23. Tan, Hwee Yim Angeline. Investigating the role of melanocortin system in regulating linear growth and growth hormone secretion.

Degree: School of Biomedical Sciences, 2014, University of Queensland

Subjects/Keywords: Growth hormone; Melanocortin 4 receptor; Insulin; Hyperphagia; Obesity; Rapid pubertal linear growth; 1109 Neurosciences; 1116 Medical Physiology

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APA (6th Edition):

Tan, H. Y. A. (2014). Investigating the role of melanocortin system in regulating linear growth and growth hormone secretion. (Thesis). University of Queensland. Retrieved from http://espace.library.uq.edu.au/view/UQ:345754

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tan, Hwee Yim Angeline. “Investigating the role of melanocortin system in regulating linear growth and growth hormone secretion.” 2014. Thesis, University of Queensland. Accessed January 16, 2021. http://espace.library.uq.edu.au/view/UQ:345754.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tan, Hwee Yim Angeline. “Investigating the role of melanocortin system in regulating linear growth and growth hormone secretion.” 2014. Web. 16 Jan 2021.

Vancouver:

Tan HYA. Investigating the role of melanocortin system in regulating linear growth and growth hormone secretion. [Internet] [Thesis]. University of Queensland; 2014. [cited 2021 Jan 16]. Available from: http://espace.library.uq.edu.au/view/UQ:345754.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tan HYA. Investigating the role of melanocortin system in regulating linear growth and growth hormone secretion. [Thesis]. University of Queensland; 2014. Available from: http://espace.library.uq.edu.au/view/UQ:345754

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pretoria

24. Hughes, Stephen Bernard. Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue .

Degree: 2012, University of Pretoria

 has been significant progress in the development of new technologies and methodologies to characterize gene expression. The fluorescent-based real-time reverse transcription (RT) polymerase chain reaction… (more)

Subjects/Keywords: Insulin receptor; UCTD; Equine tissue; Transcription polymerase; Tyrosine protein kinases

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APA (6th Edition):

Hughes, S. B. (2012). Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue . (Masters Thesis). University of Pretoria. Retrieved from http://upetd.up.ac.za/thesis/available/etd-08082012-144801/

Chicago Manual of Style (16th Edition):

Hughes, Stephen Bernard. “Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue .” 2012. Masters Thesis, University of Pretoria. Accessed January 16, 2021. http://upetd.up.ac.za/thesis/available/etd-08082012-144801/.

MLA Handbook (7th Edition):

Hughes, Stephen Bernard. “Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue .” 2012. Web. 16 Jan 2021.

Vancouver:

Hughes SB. Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue . [Internet] [Masters thesis]. University of Pretoria; 2012. [cited 2021 Jan 16]. Available from: http://upetd.up.ac.za/thesis/available/etd-08082012-144801/.

Council of Science Editors:

Hughes SB. Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue . [Masters Thesis]. University of Pretoria; 2012. Available from: http://upetd.up.ac.za/thesis/available/etd-08082012-144801/


University of Utah

25. Komanetsky, Susan M. Characterization of retinol binding protein receptor 2, a putative retinol transporter and serum retinol binding protein receptor.

Degree: MS, Nutrition, 2013, University of Utah

 min A (retinol) is essential for life; however, little is known about how it istransported into cells. Ninety-five percent of retinol found in blood is… (more)

Subjects/Keywords: Insulin resistance; RBP4; RBP4 receptor; Retinol; Retinol homeostasis; Retinol transport

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APA (6th Edition):

Komanetsky, S. M. (2013). Characterization of retinol binding protein receptor 2, a putative retinol transporter and serum retinol binding protein receptor. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2302/rec/432

Chicago Manual of Style (16th Edition):

Komanetsky, Susan M. “Characterization of retinol binding protein receptor 2, a putative retinol transporter and serum retinol binding protein receptor.” 2013. Masters Thesis, University of Utah. Accessed January 16, 2021. http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2302/rec/432.

MLA Handbook (7th Edition):

Komanetsky, Susan M. “Characterization of retinol binding protein receptor 2, a putative retinol transporter and serum retinol binding protein receptor.” 2013. Web. 16 Jan 2021.

Vancouver:

Komanetsky SM. Characterization of retinol binding protein receptor 2, a putative retinol transporter and serum retinol binding protein receptor. [Internet] [Masters thesis]. University of Utah; 2013. [cited 2021 Jan 16]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2302/rec/432.

Council of Science Editors:

Komanetsky SM. Characterization of retinol binding protein receptor 2, a putative retinol transporter and serum retinol binding protein receptor. [Masters Thesis]. University of Utah; 2013. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/2302/rec/432

26. Landis, Justine M. Mechanistic Analysis of Differential Signal Transduction Mediated by the Insulin Receptor Substrate Proteins IRS-1 and IRS-2: A Dissertation.

Degree: Cancer Biology, Molecular, Cell and Cancer Biology Department, 2014, U of Massachusetts : Med

  The Insulin Receptor Substrate (IRS) proteins IRS-1 and IRS-2 are cytoplasmic adaptor proteins that organize and propagate intracellular signaling downstream of specific growth factor… (more)

Subjects/Keywords: Signal Transduction; Insulin Receptor Substrate Proteins; Biochemistry; Cancer Biology

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APA (6th Edition):

Landis, J. M. (2014). Mechanistic Analysis of Differential Signal Transduction Mediated by the Insulin Receptor Substrate Proteins IRS-1 and IRS-2: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/735

Chicago Manual of Style (16th Edition):

Landis, Justine M. “Mechanistic Analysis of Differential Signal Transduction Mediated by the Insulin Receptor Substrate Proteins IRS-1 and IRS-2: A Dissertation.” 2014. Doctoral Dissertation, U of Massachusetts : Med. Accessed January 16, 2021. http://escholarship.umassmed.edu/gsbs_diss/735.

MLA Handbook (7th Edition):

Landis, Justine M. “Mechanistic Analysis of Differential Signal Transduction Mediated by the Insulin Receptor Substrate Proteins IRS-1 and IRS-2: A Dissertation.” 2014. Web. 16 Jan 2021.

Vancouver:

Landis JM. Mechanistic Analysis of Differential Signal Transduction Mediated by the Insulin Receptor Substrate Proteins IRS-1 and IRS-2: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2014. [cited 2021 Jan 16]. Available from: http://escholarship.umassmed.edu/gsbs_diss/735.

Council of Science Editors:

Landis JM. Mechanistic Analysis of Differential Signal Transduction Mediated by the Insulin Receptor Substrate Proteins IRS-1 and IRS-2: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2014. Available from: http://escholarship.umassmed.edu/gsbs_diss/735


Vanderbilt University

27. Ustione, Alessandro. Dopaminergic regulation of insulin secretion from the pancreatic islet.

Degree: PhD, Molecular Physiology and Biophysics, 2012, Vanderbilt University

Insulin secretion is the natural response to hyperglycemia, and it is crucial to maintain glucose homeostasis in healthy individuals. Impairment in this regulation eventually results… (more)

Subjects/Keywords: pancreatic islet; dopamine; dopamine receptor; dopamine transporter; diabetes; insulin secretion

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APA (6th Edition):

Ustione, A. (2012). Dopaminergic regulation of insulin secretion from the pancreatic islet. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14923

Chicago Manual of Style (16th Edition):

Ustione, Alessandro. “Dopaminergic regulation of insulin secretion from the pancreatic islet.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed January 16, 2021. http://hdl.handle.net/1803/14923.

MLA Handbook (7th Edition):

Ustione, Alessandro. “Dopaminergic regulation of insulin secretion from the pancreatic islet.” 2012. Web. 16 Jan 2021.

Vancouver:

Ustione A. Dopaminergic regulation of insulin secretion from the pancreatic islet. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1803/14923.

Council of Science Editors:

Ustione A. Dopaminergic regulation of insulin secretion from the pancreatic islet. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/14923


University of Pretoria

28. Hughes, Stephen Bernard. Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue.

Degree: Production Animal Studies, 2011, University of Pretoria

 has been significant progress in the development of new technologies and methodologies to characterize gene expression. The fluorescent-based real-time reverse transcription (RT) polymerase chain reaction… (more)

Subjects/Keywords: Insulin receptor; UCTD; Equine tissue; Transcription polymerase; Tyrosine protein kinases

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APA (6th Edition):

Hughes, S. B. (2011). Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue. (Masters Thesis). University of Pretoria. Retrieved from http://hdl.handle.net/2263/31135

Chicago Manual of Style (16th Edition):

Hughes, Stephen Bernard. “Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue.” 2011. Masters Thesis, University of Pretoria. Accessed January 16, 2021. http://hdl.handle.net/2263/31135.

MLA Handbook (7th Edition):

Hughes, Stephen Bernard. “Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue.” 2011. Web. 16 Jan 2021.

Vancouver:

Hughes SB. Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue. [Internet] [Masters thesis]. University of Pretoria; 2011. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2263/31135.

Council of Science Editors:

Hughes SB. Development of real-time reverse transcription polymerase chain reaction assays to quantify insulin-like growth factor-1 receptor and insulin receptor expression in equine tissue. [Masters Thesis]. University of Pretoria; 2011. Available from: http://hdl.handle.net/2263/31135


Harvard University

29. Green, Delbert Andre. Developmental and Genetic Mechanisms of Ovariole Number Evolution in Drosophila.

Degree: PhD, Biology, Molecular and Cellular, 2014, Harvard University

 The goal of the "Quantitative Trait Gene" (QTG) program is to identify genes and mutations that underlie natural phenotypic variation. My goal with this work… (more)

Subjects/Keywords: Biology; convergent evolution; Drosophila; Insulin Receptor; ovariole; plasticity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Green, D. A. (2014). Developmental and Genetic Mechanisms of Ovariole Number Evolution in Drosophila. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274190

Chicago Manual of Style (16th Edition):

Green, Delbert Andre. “Developmental and Genetic Mechanisms of Ovariole Number Evolution in Drosophila.” 2014. Doctoral Dissertation, Harvard University. Accessed January 16, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274190.

MLA Handbook (7th Edition):

Green, Delbert Andre. “Developmental and Genetic Mechanisms of Ovariole Number Evolution in Drosophila.” 2014. Web. 16 Jan 2021.

Vancouver:

Green DA. Developmental and Genetic Mechanisms of Ovariole Number Evolution in Drosophila. [Internet] [Doctoral dissertation]. Harvard University; 2014. [cited 2021 Jan 16]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274190.

Council of Science Editors:

Green DA. Developmental and Genetic Mechanisms of Ovariole Number Evolution in Drosophila. [Doctoral Dissertation]. Harvard University; 2014. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274190


University of Toronto

30. Bansal, Pritpal. Insulin-induced Suppression of A-type GABA Receptor Signaling in the INS-1 Pancreatic β-cell Line.

Degree: 2010, University of Toronto

GABA and GABA type A receptor (GABAAR) are expressed in pancreatic β-cells and comprise an autocrine signaling system. How the GABA-GABAAR system is regulated is… (more)

Subjects/Keywords: GABA; Insulin; Beta-cell; Autocrine; GABA receptor; Islet; Electrophysiology; 0719

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bansal, P. (2010). Insulin-induced Suppression of A-type GABA Receptor Signaling in the INS-1 Pancreatic β-cell Line. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25419

Chicago Manual of Style (16th Edition):

Bansal, Pritpal. “Insulin-induced Suppression of A-type GABA Receptor Signaling in the INS-1 Pancreatic β-cell Line.” 2010. Masters Thesis, University of Toronto. Accessed January 16, 2021. http://hdl.handle.net/1807/25419.

MLA Handbook (7th Edition):

Bansal, Pritpal. “Insulin-induced Suppression of A-type GABA Receptor Signaling in the INS-1 Pancreatic β-cell Line.” 2010. Web. 16 Jan 2021.

Vancouver:

Bansal P. Insulin-induced Suppression of A-type GABA Receptor Signaling in the INS-1 Pancreatic β-cell Line. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1807/25419.

Council of Science Editors:

Bansal P. Insulin-induced Suppression of A-type GABA Receptor Signaling in the INS-1 Pancreatic β-cell Line. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25419

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