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You searched for subject:(Rasmussen s encephalitis IL 1beta IL 1beta receptor Caspase 1 epilepsy epileptic seizure neuroinflammation microglia). Showing records 1 – 30 of 48360 total matches.

[1] [2] [3] [4] [5] … [1612]

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University of Vienna

1. Quemada Garrido, Lucia. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.

Degree: 2017, University of Vienna

Rasmussen Enzephalitis (RE) ist eine seltene progressive Form der Epilepsie mit unbekannter Ätiologie. Die Hauptcharakteristika sind unilaterale hemisphärische Entzündung, arzneimittelresistente fokale Epilepsie und progressive neurologischer… (more)

Subjects/Keywords: 44.45 Immunologie; 42.13 Molekularbiologie; 44.90 Neurologie; Rasmussen Enzephalitis / IL-1beta / IL-1beta Rezeptor / Caspase-1 / Epilepsie / Krampfanfall / Neuroinflammation / Mikroglia; Rasmussen’s encephalitis / IL-1beta / IL-1beta receptor / Caspase-1 / epilepsy / epileptic seizure / neuroinflammation / microglia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Quemada Garrido, L. (2017). Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/46783/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Quemada Garrido, Lucia. “Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.” 2017. Thesis, University of Vienna. Accessed October 22, 2019. http://othes.univie.ac.at/46783/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Quemada Garrido, Lucia. “Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.” 2017. Web. 22 Oct 2019.

Vancouver:

Quemada Garrido L. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. [Internet] [Thesis]. University of Vienna; 2017. [cited 2019 Oct 22]. Available from: http://othes.univie.ac.at/46783/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Quemada Garrido L. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. [Thesis]. University of Vienna; 2017. Available from: http://othes.univie.ac.at/46783/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

2. Simmatis, Leif. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .

Degree: Neuroscience Studies, 2015, Queens University

 The pannexin-1 (panx1) hemichannel is associated with the Nod-Like Receptor Protein-1 (NLRP1) inflammasome following MCAO in a mouse model of focal ischaemic stroke. We investigated… (more)

Subjects/Keywords: Hemichannel; IL-1beta; neuroinflammation; NLRP1; stroke; ischemia; pannexin-1; interleukin-1beta

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APA (6th Edition):

Simmatis, L. (2015). The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/13868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Simmatis, Leif. “The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .” 2015. Thesis, Queens University. Accessed October 22, 2019. http://hdl.handle.net/1974/13868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Simmatis, Leif. “The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .” 2015. Web. 22 Oct 2019.

Vancouver:

Simmatis L. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . [Internet] [Thesis]. Queens University; 2015. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1974/13868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Simmatis L. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/13868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

3. Kuiper, J.J.W. Reactive Oxygen Species in Inflammasome Activation.

Degree: 2009, Universiteit Utrecht

 The fundamental role of the innate immune system is to form the first barrier in bacterial and viral defence. It is based upon the recognition… (more)

Subjects/Keywords: Geneeskunde; NOD-like receptor, P2x7-receptor, NLRP3, Reactive Oxygen Species, ROS, Caspase-1, IL-1beta, IL-18, Inflammasome, ERK1/2, Cryopyrinopathies, CAPS

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APA (6th Edition):

Kuiper, J. J. W. (2009). Reactive Oxygen Species in Inflammasome Activation. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/35211

Chicago Manual of Style (16th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Masters Thesis, Universiteit Utrecht. Accessed October 22, 2019. http://dspace.library.uu.nl:8080/handle/1874/35211.

MLA Handbook (7th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Web. 22 Oct 2019.

Vancouver:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Internet] [Masters thesis]. Universiteit Utrecht; 2009. [cited 2019 Oct 22]. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211.

Council of Science Editors:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Masters Thesis]. Universiteit Utrecht; 2009. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211


University of Manchester

4. Nguyen, Loan. The role of interleukin-1 receptors in brain cell signalling.

Degree: PhD, 2010, University of Manchester

IL-1α and IL-1β are two IL-1 agonists which signals at the same receptor complex composed of IL-1R1/IL-1RAcP. However, IL-1α and IL-1β exert differential actions. A… (more)

Subjects/Keywords: 573.8; IL-1 receptors, IL-1RAcPb, IL-1alpha, IL-1beta, cytokines, inflammation, IL-6, MAP kinase, Src kinase

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APA (6th Edition):

Nguyen, L. (2010). The role of interleukin-1 receptors in brain cell signalling. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239

Chicago Manual of Style (16th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Doctoral Dissertation, University of Manchester. Accessed October 22, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239.

MLA Handbook (7th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Web. 22 Oct 2019.

Vancouver:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2010. [cited 2019 Oct 22]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239.

Council of Science Editors:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Doctoral Dissertation]. University of Manchester; 2010. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239


University of Manchester

5. Nguyen, Loan. The role of interleukin-1 receptors in brain cell signalling.

Degree: 2010, University of Manchester

IL-1α and IL-1β are two IL-1 agonists which signals at the same receptor complex composed of IL-1R1/IL-1RAcP. However, IL-1α and IL-1β exert differential actions. A… (more)

Subjects/Keywords: IL-1 receptors; IL-1RAcPb; IL-1alpha; IL-1beta; cytokines; inflammation; IL-6; MAP kinase; Src kinase

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nguyen, L. (2010). The role of interleukin-1 receptors in brain cell signalling. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038

Chicago Manual of Style (16th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Doctoral Dissertation, University of Manchester. Accessed October 22, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038.

MLA Handbook (7th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Web. 22 Oct 2019.

Vancouver:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2010. [cited 2019 Oct 22]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038.

Council of Science Editors:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Doctoral Dissertation]. University of Manchester; 2010. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038


Georgia State University

6. Chesnokov, Anton P, Mr. Modulation of NALP3 Inflammasome Genes by Estrogen.

Degree: MS, Biology, 2011, Georgia State University

  Immunity is known to be sexually dimorphic. This dimorphism may be attributed to the action of different hormones. Aluminum is a component of several… (more)

Subjects/Keywords: Nalp3 inflammasome; Estrogen; IL-1beta; Aluminum hydroxide

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APA (6th Edition):

Chesnokov, Anton P, M. (2011). Modulation of NALP3 Inflammasome Genes by Estrogen. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Thesis, Georgia State University. Accessed October 22, 2019. https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Web. 22 Oct 2019.

Vancouver:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Internet] [Thesis]. Georgia State University; 2011. [cited 2019 Oct 22]. Available from: https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Thesis]. Georgia State University; 2011. Available from: https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

7. Kahlenberg, Joanne Michelle. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.

Degree: PhD, Pathology, 2004, Case Western Reserve University

 Recent advances in understanding of the biology underlying hereditary inflammatory diseases have focused attention on the importance of regulating levels of the proinflammatory cytokine IL-1beta(more)

Subjects/Keywords: Caspase-1; IL-1beta; Inflammasome; Macrophage; Monocyte; Potassium; Toll-Like Receptors

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APA (6th Edition):

Kahlenberg, J. M. (2004). Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614

Chicago Manual of Style (16th Edition):

Kahlenberg, Joanne Michelle. “Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.” 2004. Doctoral Dissertation, Case Western Reserve University. Accessed October 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614.

MLA Handbook (7th Edition):

Kahlenberg, Joanne Michelle. “Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands.” 2004. Web. 22 Oct 2019.

Vancouver:

Kahlenberg JM. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2004. [cited 2019 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614.

Council of Science Editors:

Kahlenberg JM. Activation of Caspase-1 Signaling Complexes by the P2X7 Receptor Requires Intracellular K + Efflux and Protein Synthesis Induced by Priming with Toll-Like Receptor Ligands. [Doctoral Dissertation]. Case Western Reserve University; 2004. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1088450614


Loyola University Chicago

8. Zack, Stephanie. Lidocaine Attenuates an Induced Inflammatory Response.

Degree: MS, Microbiology and Immunology, 2018, Loyola University Chicago

  Inflammation is common to the pathogenesis of a variety of diseases. Therapeutics which can regulate and reduce damaging inflammation are therefore valuable in the… (more)

Subjects/Keywords: IL-1beta; inflammation; lidocaine; NLRP3 inflammasome; toll-like receptor 4; Immunology and Infectious Disease

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APA (6th Edition):

Zack, S. (2018). Lidocaine Attenuates an Induced Inflammatory Response. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/3714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zack, Stephanie. “Lidocaine Attenuates an Induced Inflammatory Response.” 2018. Thesis, Loyola University Chicago. Accessed October 22, 2019. https://ecommons.luc.edu/luc_theses/3714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zack, Stephanie. “Lidocaine Attenuates an Induced Inflammatory Response.” 2018. Web. 22 Oct 2019.

Vancouver:

Zack S. Lidocaine Attenuates an Induced Inflammatory Response. [Internet] [Thesis]. Loyola University Chicago; 2018. [cited 2019 Oct 22]. Available from: https://ecommons.luc.edu/luc_theses/3714.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zack S. Lidocaine Attenuates an Induced Inflammatory Response. [Thesis]. Loyola University Chicago; 2018. Available from: https://ecommons.luc.edu/luc_theses/3714

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Gómez Ochoa, Pablo. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.

Degree: 2011, Universidad de Zaragoza

 El diseño de este trabajo intenta establecer las condiciones de cultivo y la metodología para la evaluación del grupo de las Stem Cells adultas y… (more)

Subjects/Keywords: Campos electromagnéticos Pulsados; Stem Cells; TNF-alfa; IL-1beta; IL-10; PEMF; Stem Cells; TNF-alfa; IL-1beta; IL-10

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APA (6th Edition):

Gómez Ochoa, P. (2011). Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. (Thesis). Universidad de Zaragoza. Retrieved from http://zaguan.unizar.es/record/4497

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gómez Ochoa, Pablo. “Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.” 2011. Thesis, Universidad de Zaragoza. Accessed October 22, 2019. http://zaguan.unizar.es/record/4497.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gómez Ochoa, Pablo. “Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.” 2011. Web. 22 Oct 2019.

Vancouver:

Gómez Ochoa P. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. [Internet] [Thesis]. Universidad de Zaragoza; 2011. [cited 2019 Oct 22]. Available from: http://zaguan.unizar.es/record/4497.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gómez Ochoa P. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. [Thesis]. Universidad de Zaragoza; 2011. Available from: http://zaguan.unizar.es/record/4497

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

10. Zhang, Ning. Transcriptional regulation of PDGF receptor alpha in fibroblasts.

Degree: Centre for Vascular Research, 2011, University of New South Wales

 Wound healing is a process of stimulating cell growth and proliferation to achieve the result of repair. This process is controlled by cytokines and growth… (more)

Subjects/Keywords: PDGF receptor alpha; fibroblasts; interleukin 1 beta (IL-1beta); tumour necrosis factor alpha (TNF-alpha); receptor alpha (PDGF-R-alpha); wound healing

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APA (6th Edition):

Zhang, N. (2011). Transcriptional regulation of PDGF receptor alpha in fibroblasts. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Zhang, Ning. “Transcriptional regulation of PDGF receptor alpha in fibroblasts.” 2011. Doctoral Dissertation, University of New South Wales. Accessed October 22, 2019. http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true.

MLA Handbook (7th Edition):

Zhang, Ning. “Transcriptional regulation of PDGF receptor alpha in fibroblasts.” 2011. Web. 22 Oct 2019.

Vancouver:

Zhang N. Transcriptional regulation of PDGF receptor alpha in fibroblasts. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2019 Oct 22]. Available from: http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true.

Council of Science Editors:

Zhang N. Transcriptional regulation of PDGF receptor alpha in fibroblasts. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true


Freie Universität Berlin

11. Kalb, Alexander Christof. Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model.

Degree: 2014, Freie Universität Berlin

 Exogenous stress like tissue damage and pathogen invasion during surgical trauma could lead to a peripheral inflammatory response and induce neuroinflammation, which can result in… (more)

Subjects/Keywords: neuroinflammation; POCD; acetylcholinesterase-inhibitor; rat; il-1beta; neurodegeneration; physostigmine; neostigmine; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Kalb, A. C. (2014). Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/11511

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kalb, Alexander Christof. “Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model.” 2014. Thesis, Freie Universität Berlin. Accessed October 22, 2019. https://refubium.fu-berlin.de/handle/fub188/11511.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kalb, Alexander Christof. “Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model.” 2014. Web. 22 Oct 2019.

Vancouver:

Kalb AC. Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model. [Internet] [Thesis]. Freie Universität Berlin; 2014. [cited 2019 Oct 22]. Available from: https://refubium.fu-berlin.de/handle/fub188/11511.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kalb AC. Acetylcholinesterase inhibitors reduce neuroinflammation and -degeneration in the cortex and hippocampus of a surgery stress rat model. [Thesis]. Freie Universität Berlin; 2014. Available from: https://refubium.fu-berlin.de/handle/fub188/11511

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Dumont, Adélie. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.

Degree: Docteur es, Physiologie, 2018, Bourgogne Franche-Comté

Selon une étude précédente, une limitation à l'efficacité anticancéreuse du 5-Fluorouracile (5-FU) repose sur la sécrétion d'IL-1β par des cellules myéloïdes immunosuppressives (MDSC). La libération… (more)

Subjects/Keywords: Mdsc; 5 fluorouracil; Dha; IL-1 beta; Jnk; Mdsc; 5-Fluorouracil; Dha; IL-1beta; Jnk; 612

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APA (6th Edition):

Dumont, A. (2018). L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. (Doctoral Dissertation). Bourgogne Franche-Comté. Retrieved from http://www.theses.fr/2018UBFCI013

Chicago Manual of Style (16th Edition):

Dumont, Adélie. “L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.” 2018. Doctoral Dissertation, Bourgogne Franche-Comté. Accessed October 22, 2019. http://www.theses.fr/2018UBFCI013.

MLA Handbook (7th Edition):

Dumont, Adélie. “L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.” 2018. Web. 22 Oct 2019.

Vancouver:

Dumont A. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. [Internet] [Doctoral dissertation]. Bourgogne Franche-Comté; 2018. [cited 2019 Oct 22]. Available from: http://www.theses.fr/2018UBFCI013.

Council of Science Editors:

Dumont A. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. [Doctoral Dissertation]. Bourgogne Franche-Comté; 2018. Available from: http://www.theses.fr/2018UBFCI013

13. Shamaa, Obada. Intracellular and extracellular regulation of the inflammatory protease caspase-1.

Degree: PhD, Biomedical Sciences, 2014, The Ohio State University

Caspase-1 is an inflammatory cysteine protease which cleaves the pro-inflammatory cytokines, interleukin-1beta (IL-1beta) and IL-18, into their mature, bioactive forms. In addition, caspase-1 plays a… (more)

Subjects/Keywords: Immunology; Medicine; Biochemistry; Biomedical Research; caspase-1; inflammasome; IL-1beta; IL-18; mitochondria; calcium; monocyte; THP1

…antagonist interleukin-1beta inhibitory CARD IL-1 receptor associated kinase lymphocyte activating… …8 Figure 1.2 Structure and caspase-1 cleavage site for IL-1β and IL-18… …response syndrome transforming growth factor beta Toll/IL-1 receptor like protein Toll-like… …thus the mammalian homologue of Toll was called Toll/IL-1 receptor-like protein (TIL… …intracellular Toll/IL-1 Receptor (TIR) domain for signaling. Nod-like Receptors (NLR… 

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APA (6th Edition):

Shamaa, O. (2014). Intracellular and extracellular regulation of the inflammatory protease caspase-1. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292

Chicago Manual of Style (16th Edition):

Shamaa, Obada. “Intracellular and extracellular regulation of the inflammatory protease caspase-1.” 2014. Doctoral Dissertation, The Ohio State University. Accessed October 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292.

MLA Handbook (7th Edition):

Shamaa, Obada. “Intracellular and extracellular regulation of the inflammatory protease caspase-1.” 2014. Web. 22 Oct 2019.

Vancouver:

Shamaa O. Intracellular and extracellular regulation of the inflammatory protease caspase-1. [Internet] [Doctoral dissertation]. The Ohio State University; 2014. [cited 2019 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292.

Council of Science Editors:

Shamaa O. Intracellular and extracellular regulation of the inflammatory protease caspase-1. [Doctoral Dissertation]. The Ohio State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1399024292


Case Western Reserve University

14. Shive, Carey Lynn. The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease.

Degree: PhD, Pathology, 2014, Case Western Reserve University

 HIV disease is characterized by the disruption of immune homeostasis and persistent immune activation. The use of anti-retroviral therapy dramatically reduces mortality caused by HIV,… (more)

Subjects/Keywords: Biomedical Research; Immunology; HIV; immune failure; IL-6; IL-1beta; IL-7; inflammation

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APA (6th Edition):

Shive, C. L. (2014). The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515

Chicago Manual of Style (16th Edition):

Shive, Carey Lynn. “The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease.” 2014. Doctoral Dissertation, Case Western Reserve University. Accessed October 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515.

MLA Handbook (7th Edition):

Shive, Carey Lynn. “The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease.” 2014. Web. 22 Oct 2019.

Vancouver:

Shive CL. The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2014. [cited 2019 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515.

Council of Science Editors:

Shive CL. The impact of inflammatory cytokines, il-6 and il-1beta, on the pathogenesis of immune failure in HIV disease. [Doctoral Dissertation]. Case Western Reserve University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1396627515


Vanderbilt University

15. Li, Chunsheng. MyD88: central relay station of Interleukin 1 signaling pathway.

Degree: PhD, Microbiology and Immunology, 2005, Vanderbilt University

 As inflammation is a major mechanism of disease, we investigated the signal transduction processes induced by the key inflammatory cytokine Interleukin (IL) 1 beta as… (more)

Subjects/Keywords: IL-1beta signaling pathway; MyD88; gene expression profiling; inflammation; nuclear import; IL-1 receptor accessary protein; Interleukin-1; Cellular signal transduction

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APA (6th Edition):

Li, C. (2005). MyD88: central relay station of Interleukin 1 signaling pathway. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;

Chicago Manual of Style (16th Edition):

Li, Chunsheng. “MyD88: central relay station of Interleukin 1 signaling pathway.” 2005. Doctoral Dissertation, Vanderbilt University. Accessed October 22, 2019. http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;.

MLA Handbook (7th Edition):

Li, Chunsheng. “MyD88: central relay station of Interleukin 1 signaling pathway.” 2005. Web. 22 Oct 2019.

Vancouver:

Li C. MyD88: central relay station of Interleukin 1 signaling pathway. [Internet] [Doctoral dissertation]. Vanderbilt University; 2005. [cited 2019 Oct 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;.

Council of Science Editors:

Li C. MyD88: central relay station of Interleukin 1 signaling pathway. [Doctoral Dissertation]. Vanderbilt University; 2005. Available from: http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;


Kyoto University

16. Kanamori, Yohei. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .

Degree: 2018, Kyoto University

Subjects/Keywords: hepcidin; AP-1; activin B; IL-1beta; inflammation

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APA (6th Edition):

Kanamori, Y. (2018). Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/232336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kanamori, Yohei. “Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .” 2018. Thesis, Kyoto University. Accessed October 22, 2019. http://hdl.handle.net/2433/232336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kanamori, Yohei. “Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .” 2018. Web. 22 Oct 2019.

Vancouver:

Kanamori Y. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . [Internet] [Thesis]. Kyoto University; 2018. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/2433/232336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kanamori Y. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . [Thesis]. Kyoto University; 2018. Available from: http://hdl.handle.net/2433/232336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

17. Englezou, Pavlos. P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages.

Degree: PhD, 2013, University of Manchester

 The P2X7R is a functionally distinct member of the P2X non-selective cation channels and has been implicated in the initiation of immune responses. One of… (more)

Subjects/Keywords: 616.07; P2X7R; Dendritic cells; Macrophages; IL-1beta; IL-1alpha; P2X7R splice variants

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APA (6th Edition):

Englezou, P. (2013). P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121

Chicago Manual of Style (16th Edition):

Englezou, Pavlos. “P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages.” 2013. Doctoral Dissertation, University of Manchester. Accessed October 22, 2019. https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121.

MLA Handbook (7th Edition):

Englezou, Pavlos. “P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages.” 2013. Web. 22 Oct 2019.

Vancouver:

Englezou P. P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2019 Oct 22]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121.

Council of Science Editors:

Englezou P. P2X7R-driven IL-1 responses in differentiated murine dendritic cells : comparison with macrophages. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/p2x7rdriven-il1-responses-in-differentiated-murine-dendritic-cells-comparison-with-macrophages(81ddc8a9-6d43-4fe6-b9ec-e2b1b9a156a4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603121


The Ohio State University

18. Norman, Greg. Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß.

Degree: MA, Psychology, 2009, The Ohio State University

  The physiological link between neuropathic pain and depression remains unknown despite a high co-morbidity between these two disorders. A mouse model of spared nerve… (more)

Subjects/Keywords: Psychobiology; Pain; oxytocin; social; IL-1beta; depression; frontal-cortex; inflammation

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APA (6th Edition):

Norman, G. (2009). Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß. (Masters Thesis). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029

Chicago Manual of Style (16th Edition):

Norman, Greg. “Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß.” 2009. Masters Thesis, The Ohio State University. Accessed October 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029.

MLA Handbook (7th Edition):

Norman, Greg. “Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß.” 2009. Web. 22 Oct 2019.

Vancouver:

Norman G. Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß. [Internet] [Masters thesis]. The Ohio State University; 2009. [cited 2019 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029.

Council of Science Editors:

Norman G. Social Influences on Depressive-Like Behaivor Following Neuropathic Injury: A Role for Oxytocin and IL-1ß. [Masters Thesis]. The Ohio State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1258626029

19. Farooq, Rai Khalid. Implication fonctionnelle du récepteur P2X7 dans les mécanismes neuroinflammatoires associés à la dépression : étude préclinique : Functional implication of PLX7 receptors in neuroinflammatory phenomena associates with depression : a preclinical study.

Degree: Docteur es, Sciences de la vie et de la santé, spécialité Neurosciences, 2012, Université François-Rabelais de Tours

Le projet de cette thèse s'est attaché à caractériser le rôle de l'IL-1 beta et les récepteurs P2X7 dans la dépression. Les résultats suggèrent que… (more)

Subjects/Keywords: Neuroinflammation; Axe HPA; Neuroinflammation; Major depression; UCMS; P2X7 receptor; Neurogenesis; HPA-axis; Antidepressant resistance; IL-1β; Microglia

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APA (6th Edition):

Farooq, R. K. (2012). Implication fonctionnelle du récepteur P2X7 dans les mécanismes neuroinflammatoires associés à la dépression : étude préclinique : Functional implication of PLX7 receptors in neuroinflammatory phenomena associates with depression : a preclinical study. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2012TOUR4022

Chicago Manual of Style (16th Edition):

Farooq, Rai Khalid. “Implication fonctionnelle du récepteur P2X7 dans les mécanismes neuroinflammatoires associés à la dépression : étude préclinique : Functional implication of PLX7 receptors in neuroinflammatory phenomena associates with depression : a preclinical study.” 2012. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed October 22, 2019. http://www.theses.fr/2012TOUR4022.

MLA Handbook (7th Edition):

Farooq, Rai Khalid. “Implication fonctionnelle du récepteur P2X7 dans les mécanismes neuroinflammatoires associés à la dépression : étude préclinique : Functional implication of PLX7 receptors in neuroinflammatory phenomena associates with depression : a preclinical study.” 2012. Web. 22 Oct 2019.

Vancouver:

Farooq RK. Implication fonctionnelle du récepteur P2X7 dans les mécanismes neuroinflammatoires associés à la dépression : étude préclinique : Functional implication of PLX7 receptors in neuroinflammatory phenomena associates with depression : a preclinical study. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2012. [cited 2019 Oct 22]. Available from: http://www.theses.fr/2012TOUR4022.

Council of Science Editors:

Farooq RK. Implication fonctionnelle du récepteur P2X7 dans les mécanismes neuroinflammatoires associés à la dépression : étude préclinique : Functional implication of PLX7 receptors in neuroinflammatory phenomena associates with depression : a preclinical study. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2012. Available from: http://www.theses.fr/2012TOUR4022


Universidade de Lisboa

20. Rodrigues, Ana Rita Cascão, 1983-. Role of IL-1β in rheumatoid arthritis.

Degree: 2012, Universidade de Lisboa

 Rheumatoid arthritis is a chronic inflammatory and immuno-mediated disease with a significant involvement of the synovial membrane of multiple small joints and progressive destruction of… (more)

Subjects/Keywords: Interleucina-1beta; Artrite reumatóide; Caspase 1; Alergia e imunologia

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APA (6th Edition):

Rodrigues, Ana Rita Cascão, 1. (2012). Role of IL-1β in rheumatoid arthritis. (Thesis). Universidade de Lisboa. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7058

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodrigues, Ana Rita Cascão, 1983-. “Role of IL-1β in rheumatoid arthritis.” 2012. Thesis, Universidade de Lisboa. Accessed October 22, 2019. http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7058.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodrigues, Ana Rita Cascão, 1983-. “Role of IL-1β in rheumatoid arthritis.” 2012. Web. 22 Oct 2019.

Vancouver:

Rodrigues, Ana Rita Cascão 1. Role of IL-1β in rheumatoid arthritis. [Internet] [Thesis]. Universidade de Lisboa; 2012. [cited 2019 Oct 22]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7058.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodrigues, Ana Rita Cascão 1. Role of IL-1β in rheumatoid arthritis. [Thesis]. Universidade de Lisboa; 2012. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/7058

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Bernard, Marine. Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors.

Degree: Docteur es, Immunologie, 2014, Université Claude Bernard – Lyon I

La dermatite atopique (DA) est une dermatose inflammatoire dont la prévalence élevée est en constante augmentation dans les pays industrialisés. La physiopathologie de la DA… (more)

Subjects/Keywords: Dermatite atopique; Épiderme reconstruit humain; Apoptose; UV; IL-1beta; TSLP; Dermatophagoïdes farinae; Atopic dermatitis; Reconstructed human epidermis; Apoptosis; UV; IL-1beta; TSLP; Dermatophagoïdes farinae; 571.96

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APA (6th Edition):

Bernard, M. (2014). Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2014LYO10361

Chicago Manual of Style (16th Edition):

Bernard, Marine. “Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors.” 2014. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed October 22, 2019. http://www.theses.fr/2014LYO10361.

MLA Handbook (7th Edition):

Bernard, Marine. “Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors.” 2014. Web. 22 Oct 2019.

Vancouver:

Bernard M. Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2014. [cited 2019 Oct 22]. Available from: http://www.theses.fr/2014LYO10361.

Council of Science Editors:

Bernard M. Rôle de l'épiderme dans la physiopathologie de la dermatite atopique : étude reposant sur un modèle d'épiderme reconstruit issu de sujets sains ou atopiques : Epidermis role in atopic dermatitis physiopathology : study based on human reconstruted epidermis from healthy or atopic donors. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2014. Available from: http://www.theses.fr/2014LYO10361

22. Williams, Clarence David. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2012, University of Kansas

 Acetaminophen (APAP) overdose is the leading cause of acute liver failure in the US. APAP is metabolized to a reactive metabolite that causes hepatotoxicity in… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Il-1beta; Neutrophil

…of APAP hepatotoxicity based on the observation that IL-1 receptor-deficient (IL-1R1… …ASC-/- and caspase-1-/- mice, it was concluded that processing of pro-IL-1β and pro-IL18 to… …2009). The IL-1 receptor 1 (IL-1R1) and TLRs share a common signaling domain… …called the Toll-like/IL-1 Receptor (TIR) domain on the cytoplasmic side of the cell… …IL-1 receptor-1 (IL-1R1) (Sims and Smith, 2010). Both IL-1α and IL-1β… 

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APA (6th Edition):

Williams, C. D. (2012). Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/10290

Chicago Manual of Style (16th Edition):

Williams, Clarence David. “Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.” 2012. Doctoral Dissertation, University of Kansas. Accessed October 22, 2019. http://hdl.handle.net/1808/10290.

MLA Handbook (7th Edition):

Williams, Clarence David. “Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose.” 2012. Web. 22 Oct 2019.

Vancouver:

Williams CD. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1808/10290.

Council of Science Editors:

Williams CD. Role of Interleukin-1beta and neutrophil activation during acetaminophen overdose. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/10290


Universiteit Utrecht

23. Kleibeuker, W. GRK2 deficiency is a pain.

Degree: 2007, Universiteit Utrecht

 G protein-coupled receptor (GPCR) kinase (GRK)2 regulates the responsiveness of many GPCRS, e.g. CCR1, CCR2, CCR5, neurokinin-1 receptor, and metabotropic glutamate receptors, which are involved… (more)

Subjects/Keywords: Geneeskunde; GRK2; GPCR; hyperalgesia; allodynia; IL-1beta; signaling; inflammatory pain; spinal cord; nerve damage; astrocytes

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APA (6th Edition):

Kleibeuker, W. (2007). GRK2 deficiency is a pain. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/25012

Chicago Manual of Style (16th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Doctoral Dissertation, Universiteit Utrecht. Accessed October 22, 2019. http://dspace.library.uu.nl:8080/handle/1874/25012.

MLA Handbook (7th Edition):

Kleibeuker, W. “GRK2 deficiency is a pain.” 2007. Web. 22 Oct 2019.

Vancouver:

Kleibeuker W. GRK2 deficiency is a pain. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2007. [cited 2019 Oct 22]. Available from: http://dspace.library.uu.nl:8080/handle/1874/25012.

Council of Science Editors:

Kleibeuker W. GRK2 deficiency is a pain. [Doctoral Dissertation]. Universiteit Utrecht; 2007. Available from: http://dspace.library.uu.nl:8080/handle/1874/25012


Cleveland State University

24. Narayanan, Padmini. Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion.

Degree: PhD, College of Sciences and Health Professions, 2014, Cleveland State University

 Anucleate platelets alter their transcriptome through stimulated splicing of intron and exon containing hnRNA e.g. Interleukin-1ß (IL-1ß), BCL-3. Platelet TLR4 activation upon Lipopolysaccharide (LPS) binding… (more)

Subjects/Keywords: Biology; Cellular Biology; Immunology; Molecular Biology; Platelets; Interleukin1; IL-1R1; IL-1beta; microparticles; exosomes; inflammatory response; sterile inflammation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Narayanan, P. (2014). Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion. (Doctoral Dissertation). Cleveland State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007

Chicago Manual of Style (16th Edition):

Narayanan, Padmini. “Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion.” 2014. Doctoral Dissertation, Cleveland State University. Accessed October 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007.

MLA Handbook (7th Edition):

Narayanan, Padmini. “Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion.” 2014. Web. 22 Oct 2019.

Vancouver:

Narayanan P. Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion. [Internet] [Doctoral dissertation]. Cleveland State University; 2014. [cited 2019 Oct 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007.

Council of Science Editors:

Narayanan P. Interleukin 1 Receptor1 signaling in Platelet Inflammatory responses Interleukin-1ß processing and secretion. [Doctoral Dissertation]. Cleveland State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=csu1425911007


Universidade Estadual de Campinas

25. Castro, Lívia Furquim de, 1990-. Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis .

Degree: 2015, Universidade Estadual de Campinas

 Resumo: Diversos estudos demonstram que a resposta inflamatória é de extrema importância para o controle da Paracoccidioidomicose (PCM). Essa resposta inflamatória é iniciada pelo reconhecimento… (more)

Subjects/Keywords: Células dendríticas; Interleucina-1beta; Receptor NOD-like P3; Inflamassomos; Dectina 1

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APA (6th Edition):

Castro, Lívia Furquim de, 1. (2015). Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis . (Thesis). Universidade Estadual de Campinas. Retrieved from http://repositorio.unicamp.br/jspui/handle/REPOSIP/313033

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Castro, Lívia Furquim de, 1990-. “Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis .” 2015. Thesis, Universidade Estadual de Campinas. Accessed October 22, 2019. http://repositorio.unicamp.br/jspui/handle/REPOSIP/313033.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Castro, Lívia Furquim de, 1990-. “Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis .” 2015. Web. 22 Oct 2019.

Vancouver:

Castro, Lívia Furquim de 1. Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis . [Internet] [Thesis]. Universidade Estadual de Campinas; 2015. [cited 2019 Oct 22]. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/313033.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Castro, Lívia Furquim de 1. Estudo da participação do inflamassoma NLRP3 na resposta inflamatória induzida pelo fungo dimórfico Paracoccidioides brasiliensis . [Thesis]. Universidade Estadual de Campinas; 2015. Available from: http://repositorio.unicamp.br/jspui/handle/REPOSIP/313033

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

26. DRAGON, Stephane. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.

Degree: Immunology, 2010, University of Manitoba

 Immunopathological disorders are no longer defined by dysregulated T-helper (Th) type 1/ Th2 responses but account for modulatory cell types such as regulatory and Th17… (more)

Subjects/Keywords: Human; Inflammation; Cytokines; IL-17; IL-1beta; IL-22; CXCL-8; airway smooth muscle cells; neutrophils; IL-17R; signal transduction; gene expression; apoptosis

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APA (6th Edition):

DRAGON, S. (2010). Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/3945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DRAGON, Stephane. “Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.” 2010. Thesis, University of Manitoba. Accessed October 22, 2019. http://hdl.handle.net/1993/3945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DRAGON, Stephane. “Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils.” 2010. Web. 22 Oct 2019.

Vancouver:

DRAGON S. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. [Internet] [Thesis]. University of Manitoba; 2010. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/1993/3945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DRAGON S. Molecular Signaling Mechanisms and Effector Functions of the Interleukin-17 Receptor in Human Airway Smooth Muscle Cells and Polymorphonuclear Neutrophils. [Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/3945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

27. O'SULLIVAN-GREENE, ELMA. Reevaluating electroencephalography for epileptic seizure prediction.

Degree: 2011, University of Melbourne

 Prediction of epileptic seizures would greatly reduce the burden of epilepsy for over 50 million people worldwide. Seizure events are typically characterised by a loss… (more)

Subjects/Keywords: epilepsy; epileptic seizure prediction; observability

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APA (6th Edition):

O'SULLIVAN-GREENE, E. (2011). Reevaluating electroencephalography for epileptic seizure prediction. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/36630

Chicago Manual of Style (16th Edition):

O'SULLIVAN-GREENE, ELMA. “Reevaluating electroencephalography for epileptic seizure prediction.” 2011. Doctoral Dissertation, University of Melbourne. Accessed October 22, 2019. http://hdl.handle.net/11343/36630.

MLA Handbook (7th Edition):

O'SULLIVAN-GREENE, ELMA. “Reevaluating electroencephalography for epileptic seizure prediction.” 2011. Web. 22 Oct 2019.

Vancouver:

O'SULLIVAN-GREENE E. Reevaluating electroencephalography for epileptic seizure prediction. [Internet] [Doctoral dissertation]. University of Melbourne; 2011. [cited 2019 Oct 22]. Available from: http://hdl.handle.net/11343/36630.

Council of Science Editors:

O'SULLIVAN-GREENE E. Reevaluating electroencephalography for epileptic seizure prediction. [Doctoral Dissertation]. University of Melbourne; 2011. Available from: http://hdl.handle.net/11343/36630

28. Preira, Pascal. Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity.

Degree: Docteur es, Biophysique, 2012, Aix Marseille Université

Le Syndrome de Détresse Respiratoire Aiguë (SDRA) est une maladie inflammatoire courante en service de réanimation qui touche environ 10% des patients. Une augmentation pathologique… (more)

Subjects/Keywords: Syndrome de Détresse Respiratoire Aiguë (SDRA); Lésion Aigue Pulmonaire ALI; Microfluidique; Rigidité cellulaire; Cytokine; IL-1beta,IL-8; TNF-alpha; ARDS(acute respiratory,distress syndrome; Acute Lung Injyry; Microfluidique; Cell Stifness; Cytokine; IL-1beta,IL-8; TNF-alpha

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Preira, P. (2012). Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2012AIXM4020

Chicago Manual of Style (16th Edition):

Preira, Pascal. “Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity.” 2012. Doctoral Dissertation, Aix Marseille Université. Accessed October 22, 2019. http://www.theses.fr/2012AIXM4020.

MLA Handbook (7th Edition):

Preira, Pascal. “Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity.” 2012. Web. 22 Oct 2019.

Vancouver:

Preira P. Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity. [Internet] [Doctoral dissertation]. Aix Marseille Université 2012. [cited 2019 Oct 22]. Available from: http://www.theses.fr/2012AIXM4020.

Council of Science Editors:

Preira P. Etude microfluidique de la rigidité leucocytaire liée au syndrome de détresse respiratoire aigue (SDRA) : Study of the regulation of MAVS, a mitochondrial protein involves in antiviral innate immunity. [Doctoral Dissertation]. Aix Marseille Université 2012. Available from: http://www.theses.fr/2012AIXM4020


University of Vienna

29. Gessl, Denise. Microglial nodules and their potential role in brain inflammation.

Degree: 2018, University of Vienna

Rasmussen Enzephalitis (RE) ist eine seltene, progressive neurologische Erkrankung, die durch unilaterale medikamentenresistente epileptische Anfälle, Hemiparese und kognitiven Verfall charakterisiert ist. Zu den wichtigsten histopathologischen… (more)

Subjects/Keywords: 42.13 Molekularbiologie; 44.35 Histologie; 44.90 Neurologie; 44.47 Pathologie; 44.45 Immunologie; Neurobiologie / Rasmussen Enzephalitis / Gehirnentzündungen / Mikroglia / Epilepsie / Interleukine; Neurobiology / Rasmussen encephalitis / brain inflammation / microglia / epilepsy / Interleukins

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gessl, D. (2018). Microglial nodules and their potential role in brain inflammation. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/50858/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gessl, Denise. “Microglial nodules and their potential role in brain inflammation.” 2018. Thesis, University of Vienna. Accessed October 22, 2019. http://othes.univie.ac.at/50858/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gessl, Denise. “Microglial nodules and their potential role in brain inflammation.” 2018. Web. 22 Oct 2019.

Vancouver:

Gessl D. Microglial nodules and their potential role in brain inflammation. [Internet] [Thesis]. University of Vienna; 2018. [cited 2019 Oct 22]. Available from: http://othes.univie.ac.at/50858/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gessl D. Microglial nodules and their potential role in brain inflammation. [Thesis]. University of Vienna; 2018. Available from: http://othes.univie.ac.at/50858/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Symington, Jane. Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis.

Degree: PhD, Biology & Biomedical Sciences (Molecular Microbiology & Microbial Pathogenesis), 2016, Washington University in St. Louis

  Urinary tract infections (UTIs) are among the most common infectious diseases and are primarily caused by uropathogenic E. coli (UPEC). Given the greater incidence… (more)

Subjects/Keywords: ATG16L1, autophagy, bladder, IL-1beta, macrophage, UTI; Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Microbiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Symington, J. (2016). Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/art_sci_etds/764

Chicago Manual of Style (16th Edition):

Symington, Jane. “Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis.” 2016. Doctoral Dissertation, Washington University in St. Louis. Accessed October 22, 2019. https://openscholarship.wustl.edu/art_sci_etds/764.

MLA Handbook (7th Edition):

Symington, Jane. “Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis.” 2016. Web. 22 Oct 2019.

Vancouver:

Symington J. Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2016. [cited 2019 Oct 22]. Available from: https://openscholarship.wustl.edu/art_sci_etds/764.

Council of Science Editors:

Symington J. Role of ATG16L1 in Uropathogenic E. Coli Pathogenesis. [Doctoral Dissertation]. Washington University in St. Louis; 2016. Available from: https://openscholarship.wustl.edu/art_sci_etds/764

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