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You searched for subject:(Rapamycin). Showing records 1 – 30 of 217 total matches.

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Massey University

1. Sciascia, Quentin Leon. Mechanistic target of rapamycin (mTOR) activation during ruminant mammary development and function.

Degree: PhD, Animal Science, 2013, Massey University

 This thesis examines the abundance of total and activated mechanistic target of rapamycin (mTOR) pathway components in the developing and functional ruminant mammary gland. mTOR… (more)

Subjects/Keywords: Rapamycin; Microbiology

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APA (6th Edition):

Sciascia, Q. L. (2013). Mechanistic target of rapamycin (mTOR) activation during ruminant mammary development and function. (Doctoral Dissertation). Massey University. Retrieved from http://hdl.handle.net/10179/14962

Chicago Manual of Style (16th Edition):

Sciascia, Quentin Leon. “Mechanistic target of rapamycin (mTOR) activation during ruminant mammary development and function.” 2013. Doctoral Dissertation, Massey University. Accessed April 14, 2021. http://hdl.handle.net/10179/14962.

MLA Handbook (7th Edition):

Sciascia, Quentin Leon. “Mechanistic target of rapamycin (mTOR) activation during ruminant mammary development and function.” 2013. Web. 14 Apr 2021.

Vancouver:

Sciascia QL. Mechanistic target of rapamycin (mTOR) activation during ruminant mammary development and function. [Internet] [Doctoral dissertation]. Massey University; 2013. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10179/14962.

Council of Science Editors:

Sciascia QL. Mechanistic target of rapamycin (mTOR) activation during ruminant mammary development and function. [Doctoral Dissertation]. Massey University; 2013. Available from: http://hdl.handle.net/10179/14962


University of Kashmir

2. Mushtaq Ahmad Baig. Growth factor dependent regulation of p70S6 kinase Evaluation of domain specific signals sensitive to rapamycin;.

Degree: 2013, University of Kashmir

 newline Phosphorylation dependent regulation of S6K1 has been proposed in accordance with newlinethe strategy of activation adapted by AGC family kinases. Accordingly three newlinephosphorylations at… (more)

Subjects/Keywords: p70S6 kinase; Rapamycin

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APA (6th Edition):

Baig, M. A. (2013). Growth factor dependent regulation of p70S6 kinase Evaluation of domain specific signals sensitive to rapamycin;. (Thesis). University of Kashmir. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/9867

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baig, Mushtaq Ahmad. “Growth factor dependent regulation of p70S6 kinase Evaluation of domain specific signals sensitive to rapamycin;.” 2013. Thesis, University of Kashmir. Accessed April 14, 2021. http://shodhganga.inflibnet.ac.in/handle/10603/9867.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baig, Mushtaq Ahmad. “Growth factor dependent regulation of p70S6 kinase Evaluation of domain specific signals sensitive to rapamycin;.” 2013. Web. 14 Apr 2021.

Vancouver:

Baig MA. Growth factor dependent regulation of p70S6 kinase Evaluation of domain specific signals sensitive to rapamycin;. [Internet] [Thesis]. University of Kashmir; 2013. [cited 2021 Apr 14]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/9867.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baig MA. Growth factor dependent regulation of p70S6 kinase Evaluation of domain specific signals sensitive to rapamycin;. [Thesis]. University of Kashmir; 2013. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/9867

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Central Connecticut State University

3. Marinelli, Alexandra Lucia, 1989-. Identification of Potential Editing Sites in Target of Rapamycin (TOR) in the Bee Apis mellifera.

Degree: Department of Biomolecular Sciences, 2014, Central Connecticut State University

The ability of Target of Rapamycin (TOR) to play a key role in developmental changes that effect caste system determination in Apis mellifera provides a… (more)

Subjects/Keywords: Rapamycin.; Honeybee – Genetics.

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APA (6th Edition):

Marinelli, Alexandra Lucia, 1. (2014). Identification of Potential Editing Sites in Target of Rapamycin (TOR) in the Bee Apis mellifera. (Thesis). Central Connecticut State University. Retrieved from http://content.library.ccsu.edu/u?/ccsutheses,1991

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marinelli, Alexandra Lucia, 1989-. “Identification of Potential Editing Sites in Target of Rapamycin (TOR) in the Bee Apis mellifera.” 2014. Thesis, Central Connecticut State University. Accessed April 14, 2021. http://content.library.ccsu.edu/u?/ccsutheses,1991.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marinelli, Alexandra Lucia, 1989-. “Identification of Potential Editing Sites in Target of Rapamycin (TOR) in the Bee Apis mellifera.” 2014. Web. 14 Apr 2021.

Vancouver:

Marinelli, Alexandra Lucia 1. Identification of Potential Editing Sites in Target of Rapamycin (TOR) in the Bee Apis mellifera. [Internet] [Thesis]. Central Connecticut State University; 2014. [cited 2021 Apr 14]. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1991.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marinelli, Alexandra Lucia 1. Identification of Potential Editing Sites in Target of Rapamycin (TOR) in the Bee Apis mellifera. [Thesis]. Central Connecticut State University; 2014. Available from: http://content.library.ccsu.edu/u?/ccsutheses,1991

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ryerson University

4. Martin, Zechariah. mTor controls lysosome function by modulating the RAB7 GTPASE.

Degree: 2017, Ryerson University

 Mammalian target of rapamycin (mTOR) is a protein kinase that integrates signals including stress and nutrient availability, to modulate the metabolic state of the cell.… (more)

Subjects/Keywords: Lysosomes; Rapamycin; Protein kinases

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APA (6th Edition):

Martin, Z. (2017). mTor controls lysosome function by modulating the RAB7 GTPASE. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A7189

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martin, Zechariah. “mTor controls lysosome function by modulating the RAB7 GTPASE.” 2017. Thesis, Ryerson University. Accessed April 14, 2021. https://digital.library.ryerson.ca/islandora/object/RULA%3A7189.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martin, Zechariah. “mTor controls lysosome function by modulating the RAB7 GTPASE.” 2017. Web. 14 Apr 2021.

Vancouver:

Martin Z. mTor controls lysosome function by modulating the RAB7 GTPASE. [Internet] [Thesis]. Ryerson University; 2017. [cited 2021 Apr 14]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A7189.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martin Z. mTor controls lysosome function by modulating the RAB7 GTPASE. [Thesis]. Ryerson University; 2017. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A7189

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

5. Basalem, Osama Salem. Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line.

Degree: 2016, University of Manchester

Introduction: Congenital Hyperinsulinism (CHI) is a rare neonatal syndrome associated with continuous inappropriate insulin secretion by the pancreatic β-cell in the presence of recurring hypoglycemia.… (more)

Subjects/Keywords: Insulin secretion; Rapamycin; mTOR

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APA (6th Edition):

Basalem, O. S. (2016). Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154

Chicago Manual of Style (16th Edition):

Basalem, Osama Salem. “Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 14, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154.

MLA Handbook (7th Edition):

Basalem, Osama Salem. “Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line.” 2016. Web. 14 Apr 2021.

Vancouver:

Basalem OS. Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 14]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154.

Council of Science Editors:

Basalem OS. Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154


NSYSU

6. WU, HUI-CHUN. Autophagy inducers modulated ATG4B expression in human brain tumor cells.

Degree: Master, Biological Sciences, 2018, NSYSU

 Autophagy is a self-eating mechanism in cells through which damaged proteins and organelles are recruited to autophagosomes and fused with lysosome for their bulk degradation… (more)

Subjects/Keywords: Rapamycin; Chloroquine; Glioma; Autophagy; ATG4B

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APA (6th Edition):

WU, H. (2018). Autophagy inducers modulated ATG4B expression in human brain tumor cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605118-111049

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

WU, HUI-CHUN. “Autophagy inducers modulated ATG4B expression in human brain tumor cells.” 2018. Thesis, NSYSU. Accessed April 14, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605118-111049.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

WU, HUI-CHUN. “Autophagy inducers modulated ATG4B expression in human brain tumor cells.” 2018. Web. 14 Apr 2021.

Vancouver:

WU H. Autophagy inducers modulated ATG4B expression in human brain tumor cells. [Internet] [Thesis]. NSYSU; 2018. [cited 2021 Apr 14]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605118-111049.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

WU H. Autophagy inducers modulated ATG4B expression in human brain tumor cells. [Thesis]. NSYSU; 2018. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605118-111049

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

7. Byles, Vanessa A. The role of mammalian target of rapamycin (mTOR) in macrophage polarization.

Degree: MA, Medicine, 2013, Boston University

 Macrophages are key orchestrators of the innate immune response with a dynamic role in the promotion and resolution of inflammation. Macrophage polarization to a pro-inflammatory… (more)

Subjects/Keywords: Macrophage; Polarization; Rapamycin (mTOR); Inflammation

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APA (6th Edition):

Byles, V. A. (2013). The role of mammalian target of rapamycin (mTOR) in macrophage polarization. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/17122

Chicago Manual of Style (16th Edition):

Byles, Vanessa A. “The role of mammalian target of rapamycin (mTOR) in macrophage polarization.” 2013. Masters Thesis, Boston University. Accessed April 14, 2021. http://hdl.handle.net/2144/17122.

MLA Handbook (7th Edition):

Byles, Vanessa A. “The role of mammalian target of rapamycin (mTOR) in macrophage polarization.” 2013. Web. 14 Apr 2021.

Vancouver:

Byles VA. The role of mammalian target of rapamycin (mTOR) in macrophage polarization. [Internet] [Masters thesis]. Boston University; 2013. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2144/17122.

Council of Science Editors:

Byles VA. The role of mammalian target of rapamycin (mTOR) in macrophage polarization. [Masters Thesis]. Boston University; 2013. Available from: http://hdl.handle.net/2144/17122


University of Illinois – Urbana-Champaign

8. Minton, Dennis Michael. The independent and combined effects of rapamycin and metformin on naturally occurring osteoarthritis.

Degree: MS, Kinesiology, 2020, University of Illinois – Urbana-Champaign

 Osteoarthritis (OA) is among the top 10 diseases limiting human healthspan, and no disease modifying therapies currently exist. OA is a degenerative disease of the… (more)

Subjects/Keywords: Osteoarthritis; Rapamycin; Metformin; mTOR

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APA (6th Edition):

Minton, D. M. (2020). The independent and combined effects of rapamycin and metformin on naturally occurring osteoarthritis. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/107935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Minton, Dennis Michael. “The independent and combined effects of rapamycin and metformin on naturally occurring osteoarthritis.” 2020. Thesis, University of Illinois – Urbana-Champaign. Accessed April 14, 2021. http://hdl.handle.net/2142/107935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Minton, Dennis Michael. “The independent and combined effects of rapamycin and metformin on naturally occurring osteoarthritis.” 2020. Web. 14 Apr 2021.

Vancouver:

Minton DM. The independent and combined effects of rapamycin and metformin on naturally occurring osteoarthritis. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2020. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2142/107935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Minton DM. The independent and combined effects of rapamycin and metformin on naturally occurring osteoarthritis. [Thesis]. University of Illinois – Urbana-Champaign; 2020. Available from: http://hdl.handle.net/2142/107935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

9. Raut, Anuja. Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism.

Degree: MS, Pharmaceutical Sciences, 2014, University of Southern California

Rapamycin is a potent immunosuppressive macrolide which binds to FK506 binding protein (FKBP12), the cytosolic receptor to rapamycin. The FKBP12-Rapamycin complex binds to and allosterically… (more)

Subjects/Keywords: rapamycin; mTOR; immunosuppressant; pharmacology

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APA (6th Edition):

Raut, A. (2014). Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385332/rec/4009

Chicago Manual of Style (16th Edition):

Raut, Anuja. “Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism.” 2014. Masters Thesis, University of Southern California. Accessed April 14, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385332/rec/4009.

MLA Handbook (7th Edition):

Raut, Anuja. “Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism.” 2014. Web. 14 Apr 2021.

Vancouver:

Raut A. Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2021 Apr 14]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385332/rec/4009.

Council of Science Editors:

Raut A. Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385332/rec/4009

10. Suter, Thomas. Enhancer function driving cellular senescence, DNA damage repair, differentiation, and nuclear organization.

Degree: Biology, 2017, University of California – San Diego

 This dissertation, by Thomas Barton Suter, discusses enhancer function driving cellular senescence, DNA damage repair, differentiation, and nuclear organization. Enhancers are a major regulatory feature… (more)

Subjects/Keywords: Molecular biology; Aging; Enhancer; Methylation; Rapamycin; Senescence

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APA (6th Edition):

Suter, T. (2017). Enhancer function driving cellular senescence, DNA damage repair, differentiation, and nuclear organization. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/17t5r581

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Suter, Thomas. “Enhancer function driving cellular senescence, DNA damage repair, differentiation, and nuclear organization.” 2017. Thesis, University of California – San Diego. Accessed April 14, 2021. http://www.escholarship.org/uc/item/17t5r581.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Suter, Thomas. “Enhancer function driving cellular senescence, DNA damage repair, differentiation, and nuclear organization.” 2017. Web. 14 Apr 2021.

Vancouver:

Suter T. Enhancer function driving cellular senescence, DNA damage repair, differentiation, and nuclear organization. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2021 Apr 14]. Available from: http://www.escholarship.org/uc/item/17t5r581.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Suter T. Enhancer function driving cellular senescence, DNA damage repair, differentiation, and nuclear organization. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/17t5r581

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Porto

11. Silva, Mariana Lima da. Impact of rapamycin on chronological life span of filamentous fungi.

Degree: 2011, Universidade do Porto

Dissertação de mestrado integrado conduzida na Universidade de Maryland

Tese de mestrado integrado. Engenharia Química. Universidade do Porto. Faculdade de Engenharia. 2011

Advisors/Committee Members: Marten, Mark R., Universidade do Porto. Faculdade de Engenharia.

Subjects/Keywords: Rapamycin; Autofagia; Via de sinalização da TOR

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APA (6th Edition):

Silva, M. L. d. (2011). Impact of rapamycin on chronological life span of filamentous fungi. (Thesis). Universidade do Porto. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:repositorio-aberto.up.pt:10216/61473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Mariana Lima da. “Impact of rapamycin on chronological life span of filamentous fungi.” 2011. Thesis, Universidade do Porto. Accessed April 14, 2021. http://www.rcaap.pt/detail.jsp?id=oai:repositorio-aberto.up.pt:10216/61473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Mariana Lima da. “Impact of rapamycin on chronological life span of filamentous fungi.” 2011. Web. 14 Apr 2021.

Vancouver:

Silva MLd. Impact of rapamycin on chronological life span of filamentous fungi. [Internet] [Thesis]. Universidade do Porto; 2011. [cited 2021 Apr 14]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio-aberto.up.pt:10216/61473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva MLd. Impact of rapamycin on chronological life span of filamentous fungi. [Thesis]. Universidade do Porto; 2011. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio-aberto.up.pt:10216/61473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. 縣, 信秀. Repetitive Stretching Prevents Muscle Atrophy in Denervated Soleus Muscle via Akt/mTOR/p70S6K Pathways : 周期的伸張刺激はAkt/mTOR/p70S6K 経路を介して除神経によるヒラメ筋萎縮を抑制する.

Degree: 2018, Nagoya University / 名古屋大学

This study was conducted to examine whether stretch-related mechanical loading on skeletal muscle can suppress denervation-induced muscle atrophy, and if so, to depict the underlying… (more)

Subjects/Keywords: Akt; muscle atrophy; p70S6K; rapamycin; repetitive stretch

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APA (6th Edition):

縣, . (2018). Repetitive Stretching Prevents Muscle Atrophy in Denervated Soleus Muscle via Akt/mTOR/p70S6K Pathways : 周期的伸張刺激はAkt/mTOR/p70S6K 経路を介して除神経によるヒラメ筋萎縮を抑制する. (Thesis). Nagoya University / 名古屋大学. Retrieved from http://hdl.handle.net/2237/11470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

縣, 信秀. “Repetitive Stretching Prevents Muscle Atrophy in Denervated Soleus Muscle via Akt/mTOR/p70S6K Pathways : 周期的伸張刺激はAkt/mTOR/p70S6K 経路を介して除神経によるヒラメ筋萎縮を抑制する.” 2018. Thesis, Nagoya University / 名古屋大学. Accessed April 14, 2021. http://hdl.handle.net/2237/11470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

縣, 信秀. “Repetitive Stretching Prevents Muscle Atrophy in Denervated Soleus Muscle via Akt/mTOR/p70S6K Pathways : 周期的伸張刺激はAkt/mTOR/p70S6K 経路を介して除神経によるヒラメ筋萎縮を抑制する.” 2018. Web. 14 Apr 2021.

Vancouver:

縣 . Repetitive Stretching Prevents Muscle Atrophy in Denervated Soleus Muscle via Akt/mTOR/p70S6K Pathways : 周期的伸張刺激はAkt/mTOR/p70S6K 経路を介して除神経によるヒラメ筋萎縮を抑制する. [Internet] [Thesis]. Nagoya University / 名古屋大学; 2018. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2237/11470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

縣 . Repetitive Stretching Prevents Muscle Atrophy in Denervated Soleus Muscle via Akt/mTOR/p70S6K Pathways : 周期的伸張刺激はAkt/mTOR/p70S6K 経路を介して除神経によるヒラメ筋萎縮を抑制する. [Thesis]. Nagoya University / 名古屋大学; 2018. Available from: http://hdl.handle.net/2237/11470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Debrecen

13. Yan, Chen. Rapamycin: Current clinical uses and major adverse effects .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

Rapamycin (Sirolimus) is a mTOR inhibitor. The mTOR pathway is a central regulator of cell growth and metabolism in response to hormonal and environmental stimuli.… (more)

Subjects/Keywords: Rapamycin; mTOR; Sirolimus

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APA (6th Edition):

Yan, C. (n.d.). Rapamycin: Current clinical uses and major adverse effects . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/218723

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yan, Chen. “Rapamycin: Current clinical uses and major adverse effects .” Thesis, University of Debrecen. Accessed April 14, 2021. http://hdl.handle.net/2437/218723.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yan, Chen. “Rapamycin: Current clinical uses and major adverse effects .” Web. 14 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Yan C. Rapamycin: Current clinical uses and major adverse effects . [Internet] [Thesis]. University of Debrecen; [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2437/218723.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Yan C. Rapamycin: Current clinical uses and major adverse effects . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/218723

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Columbia University

14. Siegmund, Stephanie. Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases.

Degree: 2018, Columbia University

 The present work addresses outstanding questions within the field of primary mitochondrial disease biology and treatment, by incorporating methods from structural biology, molecular biology, and… (more)

Subjects/Keywords: Molecular biology; Mitochondrial pathology; Rapamycin; Biology

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APA (6th Edition):

Siegmund, S. (2018). Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8J4050F

Chicago Manual of Style (16th Edition):

Siegmund, Stephanie. “Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases.” 2018. Doctoral Dissertation, Columbia University. Accessed April 14, 2021. https://doi.org/10.7916/D8J4050F.

MLA Handbook (7th Edition):

Siegmund, Stephanie. “Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases.” 2018. Web. 14 Apr 2021.

Vancouver:

Siegmund S. Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases. [Internet] [Doctoral dissertation]. Columbia University; 2018. [cited 2021 Apr 14]. Available from: https://doi.org/10.7916/D8J4050F.

Council of Science Editors:

Siegmund S. Investigations into pathophysiologic mechanisms and treatment of primary mitochondrial diseases. [Doctoral Dissertation]. Columbia University; 2018. Available from: https://doi.org/10.7916/D8J4050F


Hong Kong University of Science and Technology

15. Ding, Yue LIFS. Mammalian target of rapamycin is required for axon growth of adult dorsal root ganglion neurons.

Degree: 2015, Hong Kong University of Science and Technology

 Different from neurons in the central nervous system, adult neurons in the mammalian peripheral nervous system can regenerate axons after injury partially by enhancing the… (more)

Subjects/Keywords: Rapamycin ; Axons ; Central nervous system ; Growth

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APA (6th Edition):

Ding, Y. L. (2015). Mammalian target of rapamycin is required for axon growth of adult dorsal root ganglion neurons. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-97094 ; https://doi.org/10.14711/thesis-b1585232 ; http://repository.ust.hk/ir/bitstream/1783.1-97094/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ding, Yue LIFS. “Mammalian target of rapamycin is required for axon growth of adult dorsal root ganglion neurons.” 2015. Thesis, Hong Kong University of Science and Technology. Accessed April 14, 2021. http://repository.ust.hk/ir/Record/1783.1-97094 ; https://doi.org/10.14711/thesis-b1585232 ; http://repository.ust.hk/ir/bitstream/1783.1-97094/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ding, Yue LIFS. “Mammalian target of rapamycin is required for axon growth of adult dorsal root ganglion neurons.” 2015. Web. 14 Apr 2021.

Vancouver:

Ding YL. Mammalian target of rapamycin is required for axon growth of adult dorsal root ganglion neurons. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2015. [cited 2021 Apr 14]. Available from: http://repository.ust.hk/ir/Record/1783.1-97094 ; https://doi.org/10.14711/thesis-b1585232 ; http://repository.ust.hk/ir/bitstream/1783.1-97094/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ding YL. Mammalian target of rapamycin is required for axon growth of adult dorsal root ganglion neurons. [Thesis]. Hong Kong University of Science and Technology; 2015. Available from: http://repository.ust.hk/ir/Record/1783.1-97094 ; https://doi.org/10.14711/thesis-b1585232 ; http://repository.ust.hk/ir/bitstream/1783.1-97094/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

16. Stead, Sebastian Oliver. Dendritic Cell Targeted Therapy Utilising Porous Silicon Nanoparticles for the Induction of Immunological Tolerance.

Degree: 2018, University of Adelaide

 The work depicted in this thesis, explores the use of drug loaded porous silicon nanoparticles (pSiNP) targeted to dendritic cells both in vitro and in… (more)

Subjects/Keywords: Nanoparticles; rapamycin; T-regs; Immunology; nanomediclne

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APA (6th Edition):

Stead, S. O. (2018). Dendritic Cell Targeted Therapy Utilising Porous Silicon Nanoparticles for the Induction of Immunological Tolerance. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/128635

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stead, Sebastian Oliver. “Dendritic Cell Targeted Therapy Utilising Porous Silicon Nanoparticles for the Induction of Immunological Tolerance.” 2018. Thesis, University of Adelaide. Accessed April 14, 2021. http://hdl.handle.net/2440/128635.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stead, Sebastian Oliver. “Dendritic Cell Targeted Therapy Utilising Porous Silicon Nanoparticles for the Induction of Immunological Tolerance.” 2018. Web. 14 Apr 2021.

Vancouver:

Stead SO. Dendritic Cell Targeted Therapy Utilising Porous Silicon Nanoparticles for the Induction of Immunological Tolerance. [Internet] [Thesis]. University of Adelaide; 2018. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2440/128635.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stead SO. Dendritic Cell Targeted Therapy Utilising Porous Silicon Nanoparticles for the Induction of Immunological Tolerance. [Thesis]. University of Adelaide; 2018. Available from: http://hdl.handle.net/2440/128635

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

17. Braileanu, Anthony Leo. Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer .

Degree: 2019, University of Arizona

 mTOR is a serine-threonine kinase and the central component of the complexes mTORC1 and mTORC2. The protein mTORC1 is most associated with global regulation of… (more)

Subjects/Keywords: breast cancer; cancer; metformin; mTOR; rapamycin; synergy

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APA (6th Edition):

Braileanu, A. L. (2019). Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/636653

Chicago Manual of Style (16th Edition):

Braileanu, Anthony Leo. “Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer .” 2019. Masters Thesis, University of Arizona. Accessed April 14, 2021. http://hdl.handle.net/10150/636653.

MLA Handbook (7th Edition):

Braileanu, Anthony Leo. “Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer .” 2019. Web. 14 Apr 2021.

Vancouver:

Braileanu AL. Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer . [Internet] [Masters thesis]. University of Arizona; 2019. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10150/636653.

Council of Science Editors:

Braileanu AL. Teaching an Old Dog New Tricks: Introducing a Hypothetical Mechanism of Synergy Between Metformin and New Generation mTOR Inhibitors in the Fight Against Cancer . [Masters Thesis]. University of Arizona; 2019. Available from: http://hdl.handle.net/10150/636653


University of Edinburgh

18. Tashkandi, Ghassan Yousuf. Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer.

Degree: PhD, 2017, University of Edinburgh

 The PI3K/Akt/mTOR and MAPK pathways are frequently altered in ovarian cancer cells, making them potential candidates for targeted therapy. A more complete understanding of the… (more)

Subjects/Keywords: ovarian cancer; PI3K; mTOR; cell signalling; rapamycin

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APA (6th Edition):

Tashkandi, G. Y. (2017). Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/28740

Chicago Manual of Style (16th Edition):

Tashkandi, Ghassan Yousuf. “Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed April 14, 2021. http://hdl.handle.net/1842/28740.

MLA Handbook (7th Edition):

Tashkandi, Ghassan Yousuf. “Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer.” 2017. Web. 14 Apr 2021.

Vancouver:

Tashkandi GY. Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1842/28740.

Council of Science Editors:

Tashkandi GY. Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/28740


Massey University

19. Rexin, Daniel. A molecular genetic analysis of the requirement of TOR kinase signalling for plant growth : a thesis presented in partial fulfilment of the requirements for the degree Doctor of Philosophy in Plant Biology at Massey University, Palmerston North, New Zealand .

Degree: 2015, Massey University

 Eukaryotes have developed a highly complex mechanism to incorporate signals from nutrient, energy, stress, developmental, and environmental cues to modulate their growth. To promote this… (more)

Subjects/Keywords: Rapamycin; Protein kinases; Molecular genetics; Arabidopsis thaliana

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rexin, D. (2015). A molecular genetic analysis of the requirement of TOR kinase signalling for plant growth : a thesis presented in partial fulfilment of the requirements for the degree Doctor of Philosophy in Plant Biology at Massey University, Palmerston North, New Zealand . (Thesis). Massey University. Retrieved from http://hdl.handle.net/10179/8347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rexin, Daniel. “A molecular genetic analysis of the requirement of TOR kinase signalling for plant growth : a thesis presented in partial fulfilment of the requirements for the degree Doctor of Philosophy in Plant Biology at Massey University, Palmerston North, New Zealand .” 2015. Thesis, Massey University. Accessed April 14, 2021. http://hdl.handle.net/10179/8347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rexin, Daniel. “A molecular genetic analysis of the requirement of TOR kinase signalling for plant growth : a thesis presented in partial fulfilment of the requirements for the degree Doctor of Philosophy in Plant Biology at Massey University, Palmerston North, New Zealand .” 2015. Web. 14 Apr 2021.

Vancouver:

Rexin D. A molecular genetic analysis of the requirement of TOR kinase signalling for plant growth : a thesis presented in partial fulfilment of the requirements for the degree Doctor of Philosophy in Plant Biology at Massey University, Palmerston North, New Zealand . [Internet] [Thesis]. Massey University; 2015. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10179/8347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rexin D. A molecular genetic analysis of the requirement of TOR kinase signalling for plant growth : a thesis presented in partial fulfilment of the requirements for the degree Doctor of Philosophy in Plant Biology at Massey University, Palmerston North, New Zealand . [Thesis]. Massey University; 2015. Available from: http://hdl.handle.net/10179/8347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

20. Dadehbeigi, Nazanin. Investigation of the role of the mTORC1 signalling pathway in growth and productivity of industrially-relevant GS-CHO cells.

Degree: 2012, University of Manchester

 Understanding the molecular mechanisms that govern productivity and growth of recombinant host cells is essential to devise informed approaches to increase commercial viability and availability… (more)

Subjects/Keywords: CHO; protein synthesis; mammalian target of rapamycin

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APA (6th Edition):

Dadehbeigi, N. (2012). Investigation of the role of the mTORC1 signalling pathway in growth and productivity of industrially-relevant GS-CHO cells. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:181038

Chicago Manual of Style (16th Edition):

Dadehbeigi, Nazanin. “Investigation of the role of the mTORC1 signalling pathway in growth and productivity of industrially-relevant GS-CHO cells.” 2012. Doctoral Dissertation, University of Manchester. Accessed April 14, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:181038.

MLA Handbook (7th Edition):

Dadehbeigi, Nazanin. “Investigation of the role of the mTORC1 signalling pathway in growth and productivity of industrially-relevant GS-CHO cells.” 2012. Web. 14 Apr 2021.

Vancouver:

Dadehbeigi N. Investigation of the role of the mTORC1 signalling pathway in growth and productivity of industrially-relevant GS-CHO cells. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2021 Apr 14]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:181038.

Council of Science Editors:

Dadehbeigi N. Investigation of the role of the mTORC1 signalling pathway in growth and productivity of industrially-relevant GS-CHO cells. [Doctoral Dissertation]. University of Manchester; 2012. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:181038

21. Reid, Justin. Novel insights into protein synthesis rates in the brain following two lifespan-extending treatments.

Degree: MS(M.S.), Health and Exercise Science, 2018, Colorado State University

 The number of individuals 65 years or older is rapidly increasing. Aging is the predominant risk factor for chronic disease and disability. Dramatic increases in… (more)

Subjects/Keywords: healthspan; rapamycin; brain; slowed aging; metformin

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APA (6th Edition):

Reid, J. (2018). Novel insights into protein synthesis rates in the brain following two lifespan-extending treatments. (Masters Thesis). Colorado State University. Retrieved from http://hdl.handle.net/10217/191325

Chicago Manual of Style (16th Edition):

Reid, Justin. “Novel insights into protein synthesis rates in the brain following two lifespan-extending treatments.” 2018. Masters Thesis, Colorado State University. Accessed April 14, 2021. http://hdl.handle.net/10217/191325.

MLA Handbook (7th Edition):

Reid, Justin. “Novel insights into protein synthesis rates in the brain following two lifespan-extending treatments.” 2018. Web. 14 Apr 2021.

Vancouver:

Reid J. Novel insights into protein synthesis rates in the brain following two lifespan-extending treatments. [Internet] [Masters thesis]. Colorado State University; 2018. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10217/191325.

Council of Science Editors:

Reid J. Novel insights into protein synthesis rates in the brain following two lifespan-extending treatments. [Masters Thesis]. Colorado State University; 2018. Available from: http://hdl.handle.net/10217/191325


Rutgers University

22. Tobak, Anne Theresa. Construction of the 3D structure of the mTOR kinase-domain and discovery of novel mTOR inhibitors.

Degree: PhD, Computational Biology and Molecular Biophysics, 2007, Rutgers University

The mammalian target of rapamycin (mTOR) is a serine/threonine kinase involved in the regulation of protein translation and cell proliferation. Based on signals received from… (more)

Subjects/Keywords: Rapamycin; Macrolide antibiotics

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APA (6th Edition):

Tobak, A. T. (2007). Construction of the 3D structure of the mTOR kinase-domain and discovery of novel mTOR inhibitors. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13836

Chicago Manual of Style (16th Edition):

Tobak, Anne Theresa. “Construction of the 3D structure of the mTOR kinase-domain and discovery of novel mTOR inhibitors.” 2007. Doctoral Dissertation, Rutgers University. Accessed April 14, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13836.

MLA Handbook (7th Edition):

Tobak, Anne Theresa. “Construction of the 3D structure of the mTOR kinase-domain and discovery of novel mTOR inhibitors.” 2007. Web. 14 Apr 2021.

Vancouver:

Tobak AT. Construction of the 3D structure of the mTOR kinase-domain and discovery of novel mTOR inhibitors. [Internet] [Doctoral dissertation]. Rutgers University; 2007. [cited 2021 Apr 14]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13836.

Council of Science Editors:

Tobak AT. Construction of the 3D structure of the mTOR kinase-domain and discovery of novel mTOR inhibitors. [Doctoral Dissertation]. Rutgers University; 2007. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13836


Brunel University

23. Rogers-Broadway, Karly-Rai. Exploring mTOR signalling as a targeted approach against ovarian cancer.

Degree: PhD, 2016, Brunel University

 Ovarian cancer is the second most common gynaecological malignancy and was diagnosed in over 7000 women in 2011 in the UK. There are currently no… (more)

Subjects/Keywords: Endometriosis; Rapamycin; Resveratrol; Deptor; Diagnostic testing

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APA (6th Edition):

Rogers-Broadway, K. (2016). Exploring mTOR signalling as a targeted approach against ovarian cancer. (Doctoral Dissertation). Brunel University. Retrieved from http://bura.brunel.ac.uk/handle/2438/12693 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.824242

Chicago Manual of Style (16th Edition):

Rogers-Broadway, Karly-Rai. “Exploring mTOR signalling as a targeted approach against ovarian cancer.” 2016. Doctoral Dissertation, Brunel University. Accessed April 14, 2021. http://bura.brunel.ac.uk/handle/2438/12693 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.824242.

MLA Handbook (7th Edition):

Rogers-Broadway, Karly-Rai. “Exploring mTOR signalling as a targeted approach against ovarian cancer.” 2016. Web. 14 Apr 2021.

Vancouver:

Rogers-Broadway K. Exploring mTOR signalling as a targeted approach against ovarian cancer. [Internet] [Doctoral dissertation]. Brunel University; 2016. [cited 2021 Apr 14]. Available from: http://bura.brunel.ac.uk/handle/2438/12693 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.824242.

Council of Science Editors:

Rogers-Broadway K. Exploring mTOR signalling as a targeted approach against ovarian cancer. [Doctoral Dissertation]. Brunel University; 2016. Available from: http://bura.brunel.ac.uk/handle/2438/12693 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.824242


University of Newcastle

24. Quinn, Rikki Kate. Understanding striatal neuroadaptations in addiction-relapse vulnerability.

Degree: PhD, 2016, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Addiction is a cyclical disorder associated with rigid, habit like behaviour and a high propensity to relapse into… (more)

Subjects/Keywords: mechanistic target of rapamycin (mTOR); rapamycin; nucleus accumbens; GluA1; CAMKIIα; cocaine; striatum; progressive ratio

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APA (6th Edition):

Quinn, R. K. (2016). Understanding striatal neuroadaptations in addiction-relapse vulnerability. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1349956

Chicago Manual of Style (16th Edition):

Quinn, Rikki Kate. “Understanding striatal neuroadaptations in addiction-relapse vulnerability.” 2016. Doctoral Dissertation, University of Newcastle. Accessed April 14, 2021. http://hdl.handle.net/1959.13/1349956.

MLA Handbook (7th Edition):

Quinn, Rikki Kate. “Understanding striatal neuroadaptations in addiction-relapse vulnerability.” 2016. Web. 14 Apr 2021.

Vancouver:

Quinn RK. Understanding striatal neuroadaptations in addiction-relapse vulnerability. [Internet] [Doctoral dissertation]. University of Newcastle; 2016. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1959.13/1349956.

Council of Science Editors:

Quinn RK. Understanding striatal neuroadaptations in addiction-relapse vulnerability. [Doctoral Dissertation]. University of Newcastle; 2016. Available from: http://hdl.handle.net/1959.13/1349956


University of Georgia

25. Foltz, Steven James. Dystroglycanopathy-type muscular dystrophy.

Degree: 2017, University of Georgia

 Muscular dystrophies are a group of genetic disorders characterized by muscle necrosis, atrophy, and progressive weakness. Several of these diseases are caused by defects in… (more)

Subjects/Keywords: skeletal muscle; muscular dystrophy; dystroglycan; fukutin; muscle differentiation; muscle fiber-type; mammalian target of rapamycin (mTOR); rapamycin

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APA (6th Edition):

Foltz, S. J. (2017). Dystroglycanopathy-type muscular dystrophy. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/36738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Foltz, Steven James. “Dystroglycanopathy-type muscular dystrophy.” 2017. Thesis, University of Georgia. Accessed April 14, 2021. http://hdl.handle.net/10724/36738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Foltz, Steven James. “Dystroglycanopathy-type muscular dystrophy.” 2017. Web. 14 Apr 2021.

Vancouver:

Foltz SJ. Dystroglycanopathy-type muscular dystrophy. [Internet] [Thesis]. University of Georgia; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10724/36738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Foltz SJ. Dystroglycanopathy-type muscular dystrophy. [Thesis]. University of Georgia; 2017. Available from: http://hdl.handle.net/10724/36738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

26. Yang, Hsiu-Chen. The Role of ESCRT-1 member Tumor Susceptibility Gene 101 in Cellular Autophagic Pathway.

Degree: Master, Biological Sciences, 2014, NSYSU

 The endocytosis involves in a variety of cellular activities including nutrient intake, and receptor recycling or degradation. The endosomal sorting complexes required for transport (ESCRTs)… (more)

Subjects/Keywords: MAP1LC3-II; Autophagy; amphisome; rapamycin; ESCRT; Tumor Susceptibility Gene 101

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APA (6th Edition):

Yang, H. (2014). The Role of ESCRT-1 member Tumor Susceptibility Gene 101 in Cellular Autophagic Pathway. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721114-182635

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Hsiu-Chen. “The Role of ESCRT-1 member Tumor Susceptibility Gene 101 in Cellular Autophagic Pathway.” 2014. Thesis, NSYSU. Accessed April 14, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721114-182635.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Hsiu-Chen. “The Role of ESCRT-1 member Tumor Susceptibility Gene 101 in Cellular Autophagic Pathway.” 2014. Web. 14 Apr 2021.

Vancouver:

Yang H. The Role of ESCRT-1 member Tumor Susceptibility Gene 101 in Cellular Autophagic Pathway. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Apr 14]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721114-182635.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang H. The Role of ESCRT-1 member Tumor Susceptibility Gene 101 in Cellular Autophagic Pathway. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721114-182635

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

27. Roccio, M. The small GTPase Rheb in insulin and nutrient signaling.

Degree: 2007, Universiteit Utrecht

 The insulin-signaling pathway controls different aspects of cell proliferation, survival, growth and metabolism. The coordinated regulation of cell growth with the availability of nutrients is… (more)

Subjects/Keywords: Geneeskunde; insulin; tuberous sclerosis; protein synthesis; rapamycin; Rheb; mTOR

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APA (6th Edition):

Roccio, M. (2007). The small GTPase Rheb in insulin and nutrient signaling. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/19063

Chicago Manual of Style (16th Edition):

Roccio, M. “The small GTPase Rheb in insulin and nutrient signaling.” 2007. Doctoral Dissertation, Universiteit Utrecht. Accessed April 14, 2021. http://dspace.library.uu.nl:8080/handle/1874/19063.

MLA Handbook (7th Edition):

Roccio, M. “The small GTPase Rheb in insulin and nutrient signaling.” 2007. Web. 14 Apr 2021.

Vancouver:

Roccio M. The small GTPase Rheb in insulin and nutrient signaling. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2007. [cited 2021 Apr 14]. Available from: http://dspace.library.uu.nl:8080/handle/1874/19063.

Council of Science Editors:

Roccio M. The small GTPase Rheb in insulin and nutrient signaling. [Doctoral Dissertation]. Universiteit Utrecht; 2007. Available from: http://dspace.library.uu.nl:8080/handle/1874/19063


Universiteit Utrecht

28. Battaglia, M. Induction of tolerance via T regulatory cells. A clinical reality or just a nice experiment in mice?.

Degree: 2014, Universiteit Utrecht

 T regulatory (Treg) cells are a subset of lymphocytes specifically dedicated to maintain the correct balance between efficient immune responses towards harmful events (such as… (more)

Subjects/Keywords: Geneeskunde; Graft rejection; autoimmunity; tolerance; T regulatory cells; rapamycin; IL-10

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Battaglia, M. (2014). Induction of tolerance via T regulatory cells. A clinical reality or just a nice experiment in mice?. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/300067

Chicago Manual of Style (16th Edition):

Battaglia, M. “Induction of tolerance via T regulatory cells. A clinical reality or just a nice experiment in mice?.” 2014. Doctoral Dissertation, Universiteit Utrecht. Accessed April 14, 2021. http://dspace.library.uu.nl:8080/handle/1874/300067.

MLA Handbook (7th Edition):

Battaglia, M. “Induction of tolerance via T regulatory cells. A clinical reality or just a nice experiment in mice?.” 2014. Web. 14 Apr 2021.

Vancouver:

Battaglia M. Induction of tolerance via T regulatory cells. A clinical reality or just a nice experiment in mice?. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2014. [cited 2021 Apr 14]. Available from: http://dspace.library.uu.nl:8080/handle/1874/300067.

Council of Science Editors:

Battaglia M. Induction of tolerance via T regulatory cells. A clinical reality or just a nice experiment in mice?. [Doctoral Dissertation]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/300067

29. SO, LOMON. The PI3K-mTOR Pathway in Mammals: From Therapeutics to Fundamentals.

Degree: Biological Sciences, 2015, University of California – Irvine

 Cells from multicellular organisms are under the control of extracellular signals to ensure the activation of certain cellular processes only in specific contexts. These extracellular… (more)

Subjects/Keywords: Biology; Cellular biology; Immunology; 4E-BP; eIF4E; Lymphocyte; mTOR; PI3K; Rapamycin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

SO, L. (2015). The PI3K-mTOR Pathway in Mammals: From Therapeutics to Fundamentals. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/10k6n2xd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

SO, LOMON. “The PI3K-mTOR Pathway in Mammals: From Therapeutics to Fundamentals.” 2015. Thesis, University of California – Irvine. Accessed April 14, 2021. http://www.escholarship.org/uc/item/10k6n2xd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

SO, LOMON. “The PI3K-mTOR Pathway in Mammals: From Therapeutics to Fundamentals.” 2015. Web. 14 Apr 2021.

Vancouver:

SO L. The PI3K-mTOR Pathway in Mammals: From Therapeutics to Fundamentals. [Internet] [Thesis]. University of California – Irvine; 2015. [cited 2021 Apr 14]. Available from: http://www.escholarship.org/uc/item/10k6n2xd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SO L. The PI3K-mTOR Pathway in Mammals: From Therapeutics to Fundamentals. [Thesis]. University of California – Irvine; 2015. Available from: http://www.escholarship.org/uc/item/10k6n2xd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

30. Srivastava, Isha Narain. The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy.

Degree: PhD, 2017, Temple University

Biomedical Neuroscience

Background and Purpose –The mammalian target of rapamycin (mTOR) pathway has been implicated in cellular responses to hypoxia and inflammation. Cerebral palsy (CP)… (more)

Subjects/Keywords: Neurosciences;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Srivastava, I. N. (2017). The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,346472

Chicago Manual of Style (16th Edition):

Srivastava, Isha Narain. “The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy.” 2017. Doctoral Dissertation, Temple University. Accessed April 14, 2021. http://digital.library.temple.edu/u?/p245801coll10,346472.

MLA Handbook (7th Edition):

Srivastava, Isha Narain. “The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy.” 2017. Web. 14 Apr 2021.

Vancouver:

Srivastava IN. The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2021 Apr 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,346472.

Council of Science Editors:

Srivastava IN. The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,346472

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