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University: University of Nottingham

You searched for subject:(Qu bec Province ). Showing records 1 – 30 of 62 total matches.

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University of Nottingham

1. Webb, Thomas M. The tumour suppressor protein LIMD1 is a novel regulator of HIF1 and the hypoxic response.

Degree: PhD, 2010, University of Nottingham

 There are three prolyl hydroxylases (PHD1, 2 and 3) that regulate the hypoxia-inducible factors (HIFs), the master transcriptional regulators that respond to changes in intracellular… (more)

Subjects/Keywords: 616.07; QU Biochemistry

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APA (6th Edition):

Webb, T. M. (2010). The tumour suppressor protein LIMD1 is a novel regulator of HIF1 and the hypoxic response. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/11280/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523477

Chicago Manual of Style (16th Edition):

Webb, Thomas M. “The tumour suppressor protein LIMD1 is a novel regulator of HIF1 and the hypoxic response.” 2010. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/11280/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523477.

MLA Handbook (7th Edition):

Webb, Thomas M. “The tumour suppressor protein LIMD1 is a novel regulator of HIF1 and the hypoxic response.” 2010. Web. 28 Mar 2020.

Vancouver:

Webb TM. The tumour suppressor protein LIMD1 is a novel regulator of HIF1 and the hypoxic response. [Internet] [Doctoral dissertation]. University of Nottingham; 2010. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/11280/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523477.

Council of Science Editors:

Webb TM. The tumour suppressor protein LIMD1 is a novel regulator of HIF1 and the hypoxic response. [Doctoral Dissertation]. University of Nottingham; 2010. Available from: http://eprints.nottingham.ac.uk/11280/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523477


University of Nottingham

2. Ahmed, Tarek Mohamed Abdel Moneim Mohamed Elsaba. Role of CD133 in colorectal cancer.

Degree: PhD, 2011, University of Nottingham

 CD133 is a pentaspan transmembrane glycoprotein of ~120 kDa, which was initially used to identify haematopoietic stem cells and, later on, used for the isolation… (more)

Subjects/Keywords: 616.99434707; QU Biochemistry

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APA (6th Edition):

Ahmed, T. M. A. M. M. E. (2011). Role of CD133 in colorectal cancer. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/28630/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555703

Chicago Manual of Style (16th Edition):

Ahmed, Tarek Mohamed Abdel Moneim Mohamed Elsaba. “Role of CD133 in colorectal cancer.” 2011. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/28630/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555703.

MLA Handbook (7th Edition):

Ahmed, Tarek Mohamed Abdel Moneim Mohamed Elsaba. “Role of CD133 in colorectal cancer.” 2011. Web. 28 Mar 2020.

Vancouver:

Ahmed TMAMME. Role of CD133 in colorectal cancer. [Internet] [Doctoral dissertation]. University of Nottingham; 2011. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/28630/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555703.

Council of Science Editors:

Ahmed TMAMME. Role of CD133 in colorectal cancer. [Doctoral Dissertation]. University of Nottingham; 2011. Available from: http://eprints.nottingham.ac.uk/28630/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555703


University of Nottingham

3. Tisdale, P. The regulation of metabolic gene expression in humans.

Degree: PhD, 2011, University of Nottingham

 The regulation of metabolic gene expression is fundamental to maintaining energy balance. Changes in substrate availability can alter metabolic gene expression, in order to modify… (more)

Subjects/Keywords: 591.35; QU Biochemistry

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APA (6th Edition):

Tisdale, P. (2011). The regulation of metabolic gene expression in humans. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/12012/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.541184

Chicago Manual of Style (16th Edition):

Tisdale, P. “The regulation of metabolic gene expression in humans.” 2011. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/12012/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.541184.

MLA Handbook (7th Edition):

Tisdale, P. “The regulation of metabolic gene expression in humans.” 2011. Web. 28 Mar 2020.

Vancouver:

Tisdale P. The regulation of metabolic gene expression in humans. [Internet] [Doctoral dissertation]. University of Nottingham; 2011. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/12012/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.541184.

Council of Science Editors:

Tisdale P. The regulation of metabolic gene expression in humans. [Doctoral Dissertation]. University of Nottingham; 2011. Available from: http://eprints.nottingham.ac.uk/12012/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.541184


University of Nottingham

4. Laithwaite, David. The role of the pyruvate dehydrogenase complex in the regulation of human skeletal muscle fuel metabolism.

Degree: Thesis (M.D.), 2009, University of Nottingham

 The pyruvate dehydrogenase complex (PDC) is the rate limiting step in the entry of glucose derived pyruvate into the tricarboxylic acid (TCA) cycle. As such… (more)

Subjects/Keywords: 616.3; QU Biochemistry

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APA (6th Edition):

Laithwaite, D. (2009). The role of the pyruvate dehydrogenase complex in the regulation of human skeletal muscle fuel metabolism. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/10992/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559512

Chicago Manual of Style (16th Edition):

Laithwaite, David. “The role of the pyruvate dehydrogenase complex in the regulation of human skeletal muscle fuel metabolism.” 2009. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/10992/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559512.

MLA Handbook (7th Edition):

Laithwaite, David. “The role of the pyruvate dehydrogenase complex in the regulation of human skeletal muscle fuel metabolism.” 2009. Web. 28 Mar 2020.

Vancouver:

Laithwaite D. The role of the pyruvate dehydrogenase complex in the regulation of human skeletal muscle fuel metabolism. [Internet] [Doctoral dissertation]. University of Nottingham; 2009. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/10992/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559512.

Council of Science Editors:

Laithwaite D. The role of the pyruvate dehydrogenase complex in the regulation of human skeletal muscle fuel metabolism. [Doctoral Dissertation]. University of Nottingham; 2009. Available from: http://eprints.nottingham.ac.uk/10992/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559512


University of Nottingham

5. Matsa, Elena. Developing a model system to investigate the epigenetic mechanisms underlying pluripotency in human cells.

Degree: PhD, 2010, University of Nottingham

 Pluripotent human embryonic stem cells (hESCs) are a valuable tool for clinical therapies, drug testing and investigation of developmental pathways. Recently, over-expression of four pluripotency-associated… (more)

Subjects/Keywords: 611; QU Biochemistry

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APA (6th Edition):

Matsa, E. (2010). Developing a model system to investigate the epigenetic mechanisms underlying pluripotency in human cells. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/14019/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523599

Chicago Manual of Style (16th Edition):

Matsa, Elena. “Developing a model system to investigate the epigenetic mechanisms underlying pluripotency in human cells.” 2010. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/14019/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523599.

MLA Handbook (7th Edition):

Matsa, Elena. “Developing a model system to investigate the epigenetic mechanisms underlying pluripotency in human cells.” 2010. Web. 28 Mar 2020.

Vancouver:

Matsa E. Developing a model system to investigate the epigenetic mechanisms underlying pluripotency in human cells. [Internet] [Doctoral dissertation]. University of Nottingham; 2010. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/14019/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523599.

Council of Science Editors:

Matsa E. Developing a model system to investigate the epigenetic mechanisms underlying pluripotency in human cells. [Doctoral Dissertation]. University of Nottingham; 2010. Available from: http://eprints.nottingham.ac.uk/14019/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523599


University of Nottingham

6. Foxler, Daniel. Genetic and functional characterisation of the LIMD1 promoter and gene product : from lung cancer to the hypoxic response.

Degree: PhD, 2012, University of Nottingham

 LIM domain containing protein 1 (LIMD1) is a tumour suppressor located at 3p21.3, a region that harbours multiple tumour suppressor genes and is commonly subject(more)

Subjects/Keywords: 616.99424; QU Biochemistry

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APA (6th Edition):

Foxler, D. (2012). Genetic and functional characterisation of the LIMD1 promoter and gene product : from lung cancer to the hypoxic response. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/12604/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559640

Chicago Manual of Style (16th Edition):

Foxler, Daniel. “Genetic and functional characterisation of the LIMD1 promoter and gene product : from lung cancer to the hypoxic response.” 2012. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/12604/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559640.

MLA Handbook (7th Edition):

Foxler, Daniel. “Genetic and functional characterisation of the LIMD1 promoter and gene product : from lung cancer to the hypoxic response.” 2012. Web. 28 Mar 2020.

Vancouver:

Foxler D. Genetic and functional characterisation of the LIMD1 promoter and gene product : from lung cancer to the hypoxic response. [Internet] [Doctoral dissertation]. University of Nottingham; 2012. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/12604/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559640.

Council of Science Editors:

Foxler D. Genetic and functional characterisation of the LIMD1 promoter and gene product : from lung cancer to the hypoxic response. [Doctoral Dissertation]. University of Nottingham; 2012. Available from: http://eprints.nottingham.ac.uk/12604/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559640


University of Nottingham

7. Dionisi, Mauro. Endocannabinoid metabolism and peroxisome proliferator-activated receptor signalling.

Degree: PhD, 2010, University of Nottingham

 The fatty acid amides (FAAs) family includes endocannabinoids, such as anandamide, as well as endocannabinoid-like molecules, such as N-palmitoylethanolamine (PEA) and N-oleoylethanolamine (OEA). Members of… (more)

Subjects/Keywords: 572; QU Biochemistry

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APA (6th Edition):

Dionisi, M. (2010). Endocannabinoid metabolism and peroxisome proliferator-activated receptor signalling. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/11384/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526655

Chicago Manual of Style (16th Edition):

Dionisi, Mauro. “Endocannabinoid metabolism and peroxisome proliferator-activated receptor signalling.” 2010. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/11384/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526655.

MLA Handbook (7th Edition):

Dionisi, Mauro. “Endocannabinoid metabolism and peroxisome proliferator-activated receptor signalling.” 2010. Web. 28 Mar 2020.

Vancouver:

Dionisi M. Endocannabinoid metabolism and peroxisome proliferator-activated receptor signalling. [Internet] [Doctoral dissertation]. University of Nottingham; 2010. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/11384/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526655.

Council of Science Editors:

Dionisi M. Endocannabinoid metabolism and peroxisome proliferator-activated receptor signalling. [Doctoral Dissertation]. University of Nottingham; 2010. Available from: http://eprints.nottingham.ac.uk/11384/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526655


University of Nottingham

8. Wood, Stuart Robert. The repair of DNA breaks in Escherichia coli and analysis of the bacterial recombination protein RecN.

Degree: PhD, 2009, University of Nottingham

 A DNA double-strand break is an exceptionally toxic lesion that threatens the structural and functional integrity of the genome. In this thesis the repair of… (more)

Subjects/Keywords: 572.8; QU Biochemistry

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APA (6th Edition):

Wood, S. R. (2009). The repair of DNA breaks in Escherichia coli and analysis of the bacterial recombination protein RecN. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/10685/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514731

Chicago Manual of Style (16th Edition):

Wood, Stuart Robert. “The repair of DNA breaks in Escherichia coli and analysis of the bacterial recombination protein RecN.” 2009. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/10685/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514731.

MLA Handbook (7th Edition):

Wood, Stuart Robert. “The repair of DNA breaks in Escherichia coli and analysis of the bacterial recombination protein RecN.” 2009. Web. 28 Mar 2020.

Vancouver:

Wood SR. The repair of DNA breaks in Escherichia coli and analysis of the bacterial recombination protein RecN. [Internet] [Doctoral dissertation]. University of Nottingham; 2009. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/10685/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514731.

Council of Science Editors:

Wood SR. The repair of DNA breaks in Escherichia coli and analysis of the bacterial recombination protein RecN. [Doctoral Dissertation]. University of Nottingham; 2009. Available from: http://eprints.nottingham.ac.uk/10685/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514731


University of Nottingham

9. Kim, Kee-Pyo. Defining and manipulating the epigenetic stability of human embryonic stem cells.

Degree: PhD, 2009, University of Nottingham

 This thesis aimed to define and manipulate epigenetic stability of human embryonic stem cells (hESCs). The allele-specific expression of 22 imprinted genes was examined in… (more)

Subjects/Keywords: 572; QU Biochemistry

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APA (6th Edition):

Kim, K. (2009). Defining and manipulating the epigenetic stability of human embryonic stem cells. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/10699/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.517690

Chicago Manual of Style (16th Edition):

Kim, Kee-Pyo. “Defining and manipulating the epigenetic stability of human embryonic stem cells.” 2009. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/10699/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.517690.

MLA Handbook (7th Edition):

Kim, Kee-Pyo. “Defining and manipulating the epigenetic stability of human embryonic stem cells.” 2009. Web. 28 Mar 2020.

Vancouver:

Kim K. Defining and manipulating the epigenetic stability of human embryonic stem cells. [Internet] [Doctoral dissertation]. University of Nottingham; 2009. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/10699/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.517690.

Council of Science Editors:

Kim K. Defining and manipulating the epigenetic stability of human embryonic stem cells. [Doctoral Dissertation]. University of Nottingham; 2009. Available from: http://eprints.nottingham.ac.uk/10699/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.517690


University of Nottingham

10. Upton, Amy Louise. Replication of damaged DNA.

Degree: PhD, 2009, University of Nottingham

 DNA is under constant attack from numerous damaging agents and our cells deal with thousands of lesions every day. With such constant damage it is… (more)

Subjects/Keywords: 572.8; QU Biochemistry

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APA (6th Edition):

Upton, A. L. (2009). Replication of damaged DNA. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/11332/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.515587

Chicago Manual of Style (16th Edition):

Upton, Amy Louise. “Replication of damaged DNA.” 2009. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/11332/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.515587.

MLA Handbook (7th Edition):

Upton, Amy Louise. “Replication of damaged DNA.” 2009. Web. 28 Mar 2020.

Vancouver:

Upton AL. Replication of damaged DNA. [Internet] [Doctoral dissertation]. University of Nottingham; 2009. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/11332/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.515587.

Council of Science Editors:

Upton AL. Replication of damaged DNA. [Doctoral Dissertation]. University of Nottingham; 2009. Available from: http://eprints.nottingham.ac.uk/11332/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.515587


University of Nottingham

11. Burridge, Paul Wesley. Improving the mesodermal differentiation potential of human embryonic stem cells.

Degree: 2008, University of Nottingham

 Human embryonic stem cells (hESCs) are thought to have enormous potential for use in regenerative medicine, whilst simultaneously allowing us insights into human embryonic development,… (more)

Subjects/Keywords: 611.0181; QU Biochemistry

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APA (6th Edition):

Burridge, P. W. (2008). Improving the mesodermal differentiation potential of human embryonic stem cells. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/13169/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.478971

Chicago Manual of Style (16th Edition):

Burridge, Paul Wesley. “Improving the mesodermal differentiation potential of human embryonic stem cells.” 2008. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/13169/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.478971.

MLA Handbook (7th Edition):

Burridge, Paul Wesley. “Improving the mesodermal differentiation potential of human embryonic stem cells.” 2008. Web. 28 Mar 2020.

Vancouver:

Burridge PW. Improving the mesodermal differentiation potential of human embryonic stem cells. [Internet] [Doctoral dissertation]. University of Nottingham; 2008. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/13169/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.478971.

Council of Science Editors:

Burridge PW. Improving the mesodermal differentiation potential of human embryonic stem cells. [Doctoral Dissertation]. University of Nottingham; 2008. Available from: http://eprints.nottingham.ac.uk/13169/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.478971


University of Nottingham

12. Abu Bakar, Suhaili. Generation of diversity at the human beta-defensin copy number.

Degree: 2010, University of Nottingham

 Submicroscopic structural genomic variation includes copy number variation (CNV) that can result changes in DNA dosage, and the impacts can be observed on common disease,… (more)

Subjects/Keywords: 591.35; QU Biochemistry

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APA (6th Edition):

Abu Bakar, S. (2010). Generation of diversity at the human beta-defensin copy number. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/11553/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.527744

Chicago Manual of Style (16th Edition):

Abu Bakar, Suhaili. “Generation of diversity at the human beta-defensin copy number.” 2010. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/11553/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.527744.

MLA Handbook (7th Edition):

Abu Bakar, Suhaili. “Generation of diversity at the human beta-defensin copy number.” 2010. Web. 28 Mar 2020.

Vancouver:

Abu Bakar S. Generation of diversity at the human beta-defensin copy number. [Internet] [Doctoral dissertation]. University of Nottingham; 2010. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/11553/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.527744.

Council of Science Editors:

Abu Bakar S. Generation of diversity at the human beta-defensin copy number. [Doctoral Dissertation]. University of Nottingham; 2010. Available from: http://eprints.nottingham.ac.uk/11553/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.527744


University of Nottingham

13. Bentley, Donna C. Investigation into the ion channels and plasma membrane properties of white adipocytes.

Degree: 2013, University of Nottingham

 Ca2+ is a ubiquitous intracellular signalling molecule that is involved in the regulation of numerous cellular functions. To date Ca2+ influx pathways present in white… (more)

Subjects/Keywords: 611.018276; QU Biochemistry

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APA (6th Edition):

Bentley, D. C. (2013). Investigation into the ion channels and plasma membrane properties of white adipocytes. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/13004/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588320

Chicago Manual of Style (16th Edition):

Bentley, Donna C. “Investigation into the ion channels and plasma membrane properties of white adipocytes.” 2013. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/13004/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588320.

MLA Handbook (7th Edition):

Bentley, Donna C. “Investigation into the ion channels and plasma membrane properties of white adipocytes.” 2013. Web. 28 Mar 2020.

Vancouver:

Bentley DC. Investigation into the ion channels and plasma membrane properties of white adipocytes. [Internet] [Doctoral dissertation]. University of Nottingham; 2013. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/13004/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588320.

Council of Science Editors:

Bentley DC. Investigation into the ion channels and plasma membrane properties of white adipocytes. [Doctoral Dissertation]. University of Nottingham; 2013. Available from: http://eprints.nottingham.ac.uk/13004/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588320


University of Nottingham

14. Pritchard, Kevin. The development of novel antimicrobial peptides with activity against MRSA.

Degree: 2008, University of Nottingham

 MRSA is a significant pathogen, which can cause a range of minor and major infections both in the hospital and community environments. MRSA is developing… (more)

Subjects/Keywords: 615; QU Biochemistry

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APA (6th Edition):

Pritchard, K. (2008). The development of novel antimicrobial peptides with activity against MRSA. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/11609/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.519387

Chicago Manual of Style (16th Edition):

Pritchard, Kevin. “The development of novel antimicrobial peptides with activity against MRSA.” 2008. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/11609/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.519387.

MLA Handbook (7th Edition):

Pritchard, Kevin. “The development of novel antimicrobial peptides with activity against MRSA.” 2008. Web. 28 Mar 2020.

Vancouver:

Pritchard K. The development of novel antimicrobial peptides with activity against MRSA. [Internet] [Doctoral dissertation]. University of Nottingham; 2008. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/11609/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.519387.

Council of Science Editors:

Pritchard K. The development of novel antimicrobial peptides with activity against MRSA. [Doctoral Dissertation]. University of Nottingham; 2008. Available from: http://eprints.nottingham.ac.uk/11609/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.519387


University of Nottingham

15. Awad, Sherif. Metabolic and cellular effects of carbohydrate-based preconditioning drinks.

Degree: 2010, University of Nottingham

 This thesis investigates the metabolic and cellular effects of carbohydrate-based preconditioning drinks in humans. Previous studies have demonstrated that preoperative carbohydrate loading, as opposed to… (more)

Subjects/Keywords: 617; QU Biochemistry

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APA (6th Edition):

Awad, S. (2010). Metabolic and cellular effects of carbohydrate-based preconditioning drinks. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/11572/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.528188

Chicago Manual of Style (16th Edition):

Awad, Sherif. “Metabolic and cellular effects of carbohydrate-based preconditioning drinks.” 2010. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/11572/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.528188.

MLA Handbook (7th Edition):

Awad, Sherif. “Metabolic and cellular effects of carbohydrate-based preconditioning drinks.” 2010. Web. 28 Mar 2020.

Vancouver:

Awad S. Metabolic and cellular effects of carbohydrate-based preconditioning drinks. [Internet] [Doctoral dissertation]. University of Nottingham; 2010. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/11572/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.528188.

Council of Science Editors:

Awad S. Metabolic and cellular effects of carbohydrate-based preconditioning drinks. [Doctoral Dissertation]. University of Nottingham; 2010. Available from: http://eprints.nottingham.ac.uk/11572/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.528188


University of Nottingham

16. McArthur Wilson, Richard John. Agonist stimulus trafficking by human prostanoid CRTH2 (DP2) receptors.

Degree: 2007, University of Nottingham

 Agonists of hormone receptors possess affinity (the ability to bind) & efficacy (the ability to stimulate effect). In this thesis, alternative expressions of efficacy by… (more)

Subjects/Keywords: 615.1; QU Biochemistry

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APA (6th Edition):

McArthur Wilson, R. J. (2007). Agonist stimulus trafficking by human prostanoid CRTH2 (DP2) receptors. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/10308/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514653

Chicago Manual of Style (16th Edition):

McArthur Wilson, Richard John. “Agonist stimulus trafficking by human prostanoid CRTH2 (DP2) receptors.” 2007. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/10308/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514653.

MLA Handbook (7th Edition):

McArthur Wilson, Richard John. “Agonist stimulus trafficking by human prostanoid CRTH2 (DP2) receptors.” 2007. Web. 28 Mar 2020.

Vancouver:

McArthur Wilson RJ. Agonist stimulus trafficking by human prostanoid CRTH2 (DP2) receptors. [Internet] [Doctoral dissertation]. University of Nottingham; 2007. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/10308/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514653.

Council of Science Editors:

McArthur Wilson RJ. Agonist stimulus trafficking by human prostanoid CRTH2 (DP2) receptors. [Doctoral Dissertation]. University of Nottingham; 2007. Available from: http://eprints.nottingham.ac.uk/10308/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514653


University of Nottingham

17. Chokkalingam, Kamalakkannan. Effect of diet, insulin and exercise on the regulation of carbohydrate metabolism in health and type 1 diabetes.

Degree: 2007, University of Nottingham

 The objective of this thesis was to further the understanding of the effect of diet, insulin and exercise on the regulation of carbohydrate metabolism in… (more)

Subjects/Keywords: 612.3; QU Biochemistry

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APA (6th Edition):

Chokkalingam, K. (2007). Effect of diet, insulin and exercise on the regulation of carbohydrate metabolism in health and type 1 diabetes. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/10441/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559501

Chicago Manual of Style (16th Edition):

Chokkalingam, Kamalakkannan. “Effect of diet, insulin and exercise on the regulation of carbohydrate metabolism in health and type 1 diabetes.” 2007. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/10441/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559501.

MLA Handbook (7th Edition):

Chokkalingam, Kamalakkannan. “Effect of diet, insulin and exercise on the regulation of carbohydrate metabolism in health and type 1 diabetes.” 2007. Web. 28 Mar 2020.

Vancouver:

Chokkalingam K. Effect of diet, insulin and exercise on the regulation of carbohydrate metabolism in health and type 1 diabetes. [Internet] [Doctoral dissertation]. University of Nottingham; 2007. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/10441/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559501.

Council of Science Editors:

Chokkalingam K. Effect of diet, insulin and exercise on the regulation of carbohydrate metabolism in health and type 1 diabetes. [Doctoral Dissertation]. University of Nottingham; 2007. Available from: http://eprints.nottingham.ac.uk/10441/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559501


University of Nottingham

18. Norton, Luke. Calpain-10 and insulin resistance in human skeletal muscle.

Degree: 2007, University of Nottingham

 Variation in the calpain-10 gene has been linked to a three-fold increased risk for type 2 diabetes in Pima Indian and some European populations. Furthermore,… (more)

Subjects/Keywords: 616.462042; QU Biochemistry

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APA (6th Edition):

Norton, L. (2007). Calpain-10 and insulin resistance in human skeletal muscle. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/11536/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.441012

Chicago Manual of Style (16th Edition):

Norton, Luke. “Calpain-10 and insulin resistance in human skeletal muscle.” 2007. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/11536/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.441012.

MLA Handbook (7th Edition):

Norton, Luke. “Calpain-10 and insulin resistance in human skeletal muscle.” 2007. Web. 28 Mar 2020.

Vancouver:

Norton L. Calpain-10 and insulin resistance in human skeletal muscle. [Internet] [Doctoral dissertation]. University of Nottingham; 2007. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/11536/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.441012.

Council of Science Editors:

Norton L. Calpain-10 and insulin resistance in human skeletal muscle. [Doctoral Dissertation]. University of Nottingham; 2007. Available from: http://eprints.nottingham.ac.uk/11536/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.441012


University of Nottingham

19. Alshalmani, Salmin Khalid. Comparison of the effects of dietary flavonoids and statins on lipopolysaccharide-induced vascular inflammation.

Degree: 2011, University of Nottingham

 Numerous epidemiological studies indicate that flavonoid intake as part of a balanced diet confers beneficial health effects in man, including improved cardiovascular function, reduced incidence… (more)

Subjects/Keywords: 615.1; QU Biochemistry

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APA (6th Edition):

Alshalmani, S. K. (2011). Comparison of the effects of dietary flavonoids and statins on lipopolysaccharide-induced vascular inflammation. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/12062/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539182

Chicago Manual of Style (16th Edition):

Alshalmani, Salmin Khalid. “Comparison of the effects of dietary flavonoids and statins on lipopolysaccharide-induced vascular inflammation.” 2011. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/12062/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539182.

MLA Handbook (7th Edition):

Alshalmani, Salmin Khalid. “Comparison of the effects of dietary flavonoids and statins on lipopolysaccharide-induced vascular inflammation.” 2011. Web. 28 Mar 2020.

Vancouver:

Alshalmani SK. Comparison of the effects of dietary flavonoids and statins on lipopolysaccharide-induced vascular inflammation. [Internet] [Doctoral dissertation]. University of Nottingham; 2011. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/12062/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539182.

Council of Science Editors:

Alshalmani SK. Comparison of the effects of dietary flavonoids and statins on lipopolysaccharide-induced vascular inflammation. [Doctoral Dissertation]. University of Nottingham; 2011. Available from: http://eprints.nottingham.ac.uk/12062/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.539182


University of Nottingham

20. Shaikh-Omar, Osama. Recombinant expression of the aryl hydrocarbon receptor.

Degree: 2007, University of Nottingham

 Aryl Hydrocarbon Receptor (AhR) mediates drug and toxin action. The AhR proteins have been characterised in several mammalian species, and are soluble proteins found in… (more)

Subjects/Keywords: 541.2242; QU Biochemistry

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APA (6th Edition):

Shaikh-Omar, O. (2007). Recombinant expression of the aryl hydrocarbon receptor. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/10398/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.479342

Chicago Manual of Style (16th Edition):

Shaikh-Omar, Osama. “Recombinant expression of the aryl hydrocarbon receptor.” 2007. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/10398/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.479342.

MLA Handbook (7th Edition):

Shaikh-Omar, Osama. “Recombinant expression of the aryl hydrocarbon receptor.” 2007. Web. 28 Mar 2020.

Vancouver:

Shaikh-Omar O. Recombinant expression of the aryl hydrocarbon receptor. [Internet] [Doctoral dissertation]. University of Nottingham; 2007. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/10398/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.479342.

Council of Science Editors:

Shaikh-Omar O. Recombinant expression of the aryl hydrocarbon receptor. [Doctoral Dissertation]. University of Nottingham; 2007. Available from: http://eprints.nottingham.ac.uk/10398/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.479342


University of Nottingham

21. Vilisaar, Janek. The induction and effects of Substance P and its receptor in human immune cells and neurons : potential relevance in multiple sclerosis.

Degree: 2012, University of Nottingham

 INTRODUCTION: Substance P (SP) has well-established roles in neurogenic inflammation and pain transmission, however recently, a number of SP immunomodulatory effects have been shown. In… (more)

Subjects/Keywords: 616.834; QU Biochemistry

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APA (6th Edition):

Vilisaar, J. (2012). The induction and effects of Substance P and its receptor in human immune cells and neurons : potential relevance in multiple sclerosis. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/12663/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559674

Chicago Manual of Style (16th Edition):

Vilisaar, Janek. “The induction and effects of Substance P and its receptor in human immune cells and neurons : potential relevance in multiple sclerosis.” 2012. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/12663/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559674.

MLA Handbook (7th Edition):

Vilisaar, Janek. “The induction and effects of Substance P and its receptor in human immune cells and neurons : potential relevance in multiple sclerosis.” 2012. Web. 28 Mar 2020.

Vancouver:

Vilisaar J. The induction and effects of Substance P and its receptor in human immune cells and neurons : potential relevance in multiple sclerosis. [Internet] [Doctoral dissertation]. University of Nottingham; 2012. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/12663/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559674.

Council of Science Editors:

Vilisaar J. The induction and effects of Substance P and its receptor in human immune cells and neurons : potential relevance in multiple sclerosis. [Doctoral Dissertation]. University of Nottingham; 2012. Available from: http://eprints.nottingham.ac.uk/12663/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559674


University of Nottingham

22. Hopps, Jamie. Vascular actions of oleamide in health and disease.

Degree: 2013, University of Nottingham

 Oleamide, an endocannabinoid-like mediator, is a fatty acid that shares structural similarities with anandamide. Oleamide induces cannabimimetic responses and is a potent vasodilator of rat… (more)

Subjects/Keywords: 616.12; QU Biochemistry

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APA (6th Edition):

Hopps, J. (2013). Vascular actions of oleamide in health and disease. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/13132/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588333

Chicago Manual of Style (16th Edition):

Hopps, Jamie. “Vascular actions of oleamide in health and disease.” 2013. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/13132/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588333.

MLA Handbook (7th Edition):

Hopps, Jamie. “Vascular actions of oleamide in health and disease.” 2013. Web. 28 Mar 2020.

Vancouver:

Hopps J. Vascular actions of oleamide in health and disease. [Internet] [Doctoral dissertation]. University of Nottingham; 2013. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/13132/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588333.

Council of Science Editors:

Hopps J. Vascular actions of oleamide in health and disease. [Doctoral Dissertation]. University of Nottingham; 2013. Available from: http://eprints.nottingham.ac.uk/13132/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588333


University of Nottingham

23. Styles, Pamela. The evolution of transposable elements in humans and Drosophila.

Degree: 2010, University of Nottingham

 The different genomic environments in which transposable elements reside in the Great Apes and the Drosophila result in substantial differences between the evolution of transposable… (more)

Subjects/Keywords: 591.35; QU Biochemistry

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APA (6th Edition):

Styles, P. (2010). The evolution of transposable elements in humans and Drosophila. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/11241/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523475

Chicago Manual of Style (16th Edition):

Styles, Pamela. “The evolution of transposable elements in humans and Drosophila.” 2010. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/11241/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523475.

MLA Handbook (7th Edition):

Styles, Pamela. “The evolution of transposable elements in humans and Drosophila.” 2010. Web. 28 Mar 2020.

Vancouver:

Styles P. The evolution of transposable elements in humans and Drosophila. [Internet] [Doctoral dissertation]. University of Nottingham; 2010. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/11241/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523475.

Council of Science Editors:

Styles P. The evolution of transposable elements in humans and Drosophila. [Doctoral Dissertation]. University of Nottingham; 2010. Available from: http://eprints.nottingham.ac.uk/11241/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.523475


University of Nottingham

24. Yu, Jing. Structural and genetic analyses of the RdgC protein in Escherichia coli.

Degree: 2009, University of Nottingham

 Previous studies found that RdgC protein plays a role in the DNA repair system in Escherichia coli. In recBC sbcBC strains, loss of rdgC made… (more)

Subjects/Keywords: 579; QU Biochemistry

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APA (6th Edition):

Yu, J. (2009). Structural and genetic analyses of the RdgC protein in Escherichia coli. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/10961/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514872

Chicago Manual of Style (16th Edition):

Yu, Jing. “Structural and genetic analyses of the RdgC protein in Escherichia coli.” 2009. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/10961/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514872.

MLA Handbook (7th Edition):

Yu, Jing. “Structural and genetic analyses of the RdgC protein in Escherichia coli.” 2009. Web. 28 Mar 2020.

Vancouver:

Yu J. Structural and genetic analyses of the RdgC protein in Escherichia coli. [Internet] [Doctoral dissertation]. University of Nottingham; 2009. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/10961/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514872.

Council of Science Editors:

Yu J. Structural and genetic analyses of the RdgC protein in Escherichia coli. [Doctoral Dissertation]. University of Nottingham; 2009. Available from: http://eprints.nottingham.ac.uk/10961/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514872


University of Nottingham

25. McCartney, Karen M. The role of peroxisome proliferator activated receptor alpha (PPARα) in the effect of piroxicam on colon cancer.

Degree: PhD, 2015, University of Nottingham

 Studies with APCMin/+ mice and APCMin/+ PPARα-/- mice were undertaken to investigate whether polyp development in the mouse gut was mediated by PPARα. Additionally, the… (more)

Subjects/Keywords: 616.99; QU Biochemistry

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APA (6th Edition):

McCartney, K. M. (2015). The role of peroxisome proliferator activated receptor alpha (PPARα) in the effect of piroxicam on colon cancer. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/29153/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.668639

Chicago Manual of Style (16th Edition):

McCartney, Karen M. “The role of peroxisome proliferator activated receptor alpha (PPARα) in the effect of piroxicam on colon cancer.” 2015. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/29153/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.668639.

MLA Handbook (7th Edition):

McCartney, Karen M. “The role of peroxisome proliferator activated receptor alpha (PPARα) in the effect of piroxicam on colon cancer.” 2015. Web. 28 Mar 2020.

Vancouver:

McCartney KM. The role of peroxisome proliferator activated receptor alpha (PPARα) in the effect of piroxicam on colon cancer. [Internet] [Doctoral dissertation]. University of Nottingham; 2015. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/29153/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.668639.

Council of Science Editors:

McCartney KM. The role of peroxisome proliferator activated receptor alpha (PPARα) in the effect of piroxicam on colon cancer. [Doctoral Dissertation]. University of Nottingham; 2015. Available from: http://eprints.nottingham.ac.uk/29153/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.668639

26. Saroha, Vivek. Developmental programming of the cell stress response and metabolic inflammation in liver and adipose tissue in an ovine model.

Degree: PhD, 2017, University of Nottingham

 A state of chronic metabolic inflammation and activation of the cell stress response in organs such as liver and adipose tissue are important pathogenic adaptations… (more)

Subjects/Keywords: 616.3; QU Biochemistry

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APA (6th Edition):

Saroha, V. (2017). Developmental programming of the cell stress response and metabolic inflammation in liver and adipose tissue in an ovine model. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/47527/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728600

Chicago Manual of Style (16th Edition):

Saroha, Vivek. “Developmental programming of the cell stress response and metabolic inflammation in liver and adipose tissue in an ovine model.” 2017. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/47527/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728600.

MLA Handbook (7th Edition):

Saroha, Vivek. “Developmental programming of the cell stress response and metabolic inflammation in liver and adipose tissue in an ovine model.” 2017. Web. 28 Mar 2020.

Vancouver:

Saroha V. Developmental programming of the cell stress response and metabolic inflammation in liver and adipose tissue in an ovine model. [Internet] [Doctoral dissertation]. University of Nottingham; 2017. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/47527/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728600.

Council of Science Editors:

Saroha V. Developmental programming of the cell stress response and metabolic inflammation in liver and adipose tissue in an ovine model. [Doctoral Dissertation]. University of Nottingham; 2017. Available from: http://eprints.nottingham.ac.uk/47527/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728600


University of Nottingham

27. Alsaqati, Mouhamed. Characterisation of the P2Y14 receptor in the pancreas : control of vascular tone and insulin secretion.

Degree: PhD, 2014, University of Nottingham

 The P2Y14 receptor is the most recently identified member of the P2Y family of receptors for adenine and uridine nucleotides and nucleotide sugars. It is… (more)

Subjects/Keywords: 612; QP Physiology; QU Biochemistry

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APA (6th Edition):

Alsaqati, M. (2014). Characterisation of the P2Y14 receptor in the pancreas : control of vascular tone and insulin secretion. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/14249/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632481

Chicago Manual of Style (16th Edition):

Alsaqati, Mouhamed. “Characterisation of the P2Y14 receptor in the pancreas : control of vascular tone and insulin secretion.” 2014. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/14249/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632481.

MLA Handbook (7th Edition):

Alsaqati, Mouhamed. “Characterisation of the P2Y14 receptor in the pancreas : control of vascular tone and insulin secretion.” 2014. Web. 28 Mar 2020.

Vancouver:

Alsaqati M. Characterisation of the P2Y14 receptor in the pancreas : control of vascular tone and insulin secretion. [Internet] [Doctoral dissertation]. University of Nottingham; 2014. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/14249/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632481.

Council of Science Editors:

Alsaqati M. Characterisation of the P2Y14 receptor in the pancreas : control of vascular tone and insulin secretion. [Doctoral Dissertation]. University of Nottingham; 2014. Available from: http://eprints.nottingham.ac.uk/14249/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632481


University of Nottingham

28. Alhebshi, Alawiah. The essential iron-sulphur protein Rli1 is a key determinant of oxidative stress resistance in Saccharomyces cerevisiae.

Degree: PhD, 2014, University of Nottingham

 Reactive oxygen species (ROS) are linked to a range of degenerative conditions in humans, and may cause damage to an array of cellular components. However,… (more)

Subjects/Keywords: QP Physiology; QU Biochemistry

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APA (6th Edition):

Alhebshi, A. (2014). The essential iron-sulphur protein Rli1 is a key determinant of oxidative stress resistance in Saccharomyces cerevisiae. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/13974/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632435

Chicago Manual of Style (16th Edition):

Alhebshi, Alawiah. “The essential iron-sulphur protein Rli1 is a key determinant of oxidative stress resistance in Saccharomyces cerevisiae.” 2014. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/13974/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632435.

MLA Handbook (7th Edition):

Alhebshi, Alawiah. “The essential iron-sulphur protein Rli1 is a key determinant of oxidative stress resistance in Saccharomyces cerevisiae.” 2014. Web. 28 Mar 2020.

Vancouver:

Alhebshi A. The essential iron-sulphur protein Rli1 is a key determinant of oxidative stress resistance in Saccharomyces cerevisiae. [Internet] [Doctoral dissertation]. University of Nottingham; 2014. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/13974/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632435.

Council of Science Editors:

Alhebshi A. The essential iron-sulphur protein Rli1 is a key determinant of oxidative stress resistance in Saccharomyces cerevisiae. [Doctoral Dissertation]. University of Nottingham; 2014. Available from: http://eprints.nottingham.ac.uk/13974/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.632435


University of Nottingham

29. Pala, Raquel Rodrigues. Human beta-defensin gene copy number variation and consequences in disease and evolution.

Degree: 2012, University of Nottingham

 Research on human genetic variation has shown that the human genome is not a fixed, invariant framework, but that there can be extensive structural variation.… (more)

Subjects/Keywords: 611.0181663; QH426 Genetics : QU Biochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pala, R. R. (2012). Human beta-defensin gene copy number variation and consequences in disease and evolution. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/14020/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574656

Chicago Manual of Style (16th Edition):

Pala, Raquel Rodrigues. “Human beta-defensin gene copy number variation and consequences in disease and evolution.” 2012. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/14020/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574656.

MLA Handbook (7th Edition):

Pala, Raquel Rodrigues. “Human beta-defensin gene copy number variation and consequences in disease and evolution.” 2012. Web. 28 Mar 2020.

Vancouver:

Pala RR. Human beta-defensin gene copy number variation and consequences in disease and evolution. [Internet] [Doctoral dissertation]. University of Nottingham; 2012. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/14020/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574656.

Council of Science Editors:

Pala RR. Human beta-defensin gene copy number variation and consequences in disease and evolution. [Doctoral Dissertation]. University of Nottingham; 2012. Available from: http://eprints.nottingham.ac.uk/14020/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574656


University of Nottingham

30. Sidique, Idris L. Evaluation of skeletal muscle satellite cell activity in rodent models depicting muscle hypertrophy and atrophy.

Degree: 2013, University of Nottingham

 Satellite cells are muscle-specific progenitor cells involved in the routine maintenance of skeletal muscle homeostasis, growth and regeneration. They are activated by various stimuli (myotrauma,… (more)

Subjects/Keywords: 616.74; QL801 Anatomy; QU Biochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sidique, I. L. (2013). Evaluation of skeletal muscle satellite cell activity in rodent models depicting muscle hypertrophy and atrophy. (Doctoral Dissertation). University of Nottingham. Retrieved from http://eprints.nottingham.ac.uk/13899/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603468

Chicago Manual of Style (16th Edition):

Sidique, Idris L. “Evaluation of skeletal muscle satellite cell activity in rodent models depicting muscle hypertrophy and atrophy.” 2013. Doctoral Dissertation, University of Nottingham. Accessed March 28, 2020. http://eprints.nottingham.ac.uk/13899/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603468.

MLA Handbook (7th Edition):

Sidique, Idris L. “Evaluation of skeletal muscle satellite cell activity in rodent models depicting muscle hypertrophy and atrophy.” 2013. Web. 28 Mar 2020.

Vancouver:

Sidique IL. Evaluation of skeletal muscle satellite cell activity in rodent models depicting muscle hypertrophy and atrophy. [Internet] [Doctoral dissertation]. University of Nottingham; 2013. [cited 2020 Mar 28]. Available from: http://eprints.nottingham.ac.uk/13899/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603468.

Council of Science Editors:

Sidique IL. Evaluation of skeletal muscle satellite cell activity in rodent models depicting muscle hypertrophy and atrophy. [Doctoral Dissertation]. University of Nottingham; 2013. Available from: http://eprints.nottingham.ac.uk/13899/ ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.603468

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