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You searched for subject:(Proto Oncogene Proteins c bcl 2). Showing records 1 – 30 of 35394 total matches.

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University of Missouri – Columbia

1. Balkin, Ethan R., 1978-. Targeted 177 Lu antisense radiotherapy of B-cell non-Hodgkin's lymphoma.

Degree: 2011, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT REQUEST OF AUTHOR.] The B-cell lymphoma/leukemia-2 (bcl-2) proto-oncogene in non-Hodgkin's lymphoma (NHL) is a dominant inhibitor of… (more)

Subjects/Keywords: antisense radiotherapy; B-cell lymphoma-2; peptide nucleic acid; cancer therapy; Lymphomas  – Radiotherapy; Antisense nucleic acids  – Therapeutic use; Antisense peptides  – Therapeutic use; Lutetium  – Therapeutic use; Lymphoma, Non-Hodgkin  – radiotherapy; Peptide Nucleic Acid  – pharmacology; Lutetium  – therapeutic use; Molecular Targeted Therapy  – methods; Proto-Oncogene Proteins c-bcl-2  – metabolism; Radiotherapy  – methods

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Balkin, Ethan R., 1. (2011). Targeted 177 Lu antisense radiotherapy of B-cell non-Hodgkin's lymphoma. (Thesis). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/14295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Balkin, Ethan R., 1978-. “Targeted 177 Lu antisense radiotherapy of B-cell non-Hodgkin's lymphoma.” 2011. Thesis, University of Missouri – Columbia. Accessed March 07, 2021. https://doi.org/10.32469/10355/14295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Balkin, Ethan R., 1978-. “Targeted 177 Lu antisense radiotherapy of B-cell non-Hodgkin's lymphoma.” 2011. Web. 07 Mar 2021.

Vancouver:

Balkin, Ethan R. 1. Targeted 177 Lu antisense radiotherapy of B-cell non-Hodgkin's lymphoma. [Internet] [Thesis]. University of Missouri – Columbia; 2011. [cited 2021 Mar 07]. Available from: https://doi.org/10.32469/10355/14295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Balkin, Ethan R. 1. Targeted 177 Lu antisense radiotherapy of B-cell non-Hodgkin's lymphoma. [Thesis]. University of Missouri – Columbia; 2011. Available from: https://doi.org/10.32469/10355/14295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

2. Dospoy, Patrick. Characterizing c-Myc Dependent Lung Cancers.

Degree: 2015, University of Texas Southwestern Medical Center

 MYC is one of the most commonly deregulated oncogenes in human cancer, including breast, colorectal and lung. While mutations in myc are rare, MYC is… (more)

Subjects/Keywords: Lung Neoplasms; Peptide Fragments; Proto-Oncogene Proteins c-myc

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APA (6th Edition):

Dospoy, P. (2015). Characterizing c-Myc Dependent Lung Cancers. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4225

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dospoy, Patrick. “Characterizing c-Myc Dependent Lung Cancers.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/4225.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dospoy, Patrick. “Characterizing c-Myc Dependent Lung Cancers.” 2015. Web. 07 Mar 2021.

Vancouver:

Dospoy P. Characterizing c-Myc Dependent Lung Cancers. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/4225.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dospoy P. Characterizing c-Myc Dependent Lung Cancers. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4225

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

3. Yu, Bingke. Structural and Mechanistic Studies of Two Regulatory Factors in Actin Cytoskeletal Signaling: Vav and VopL.

Degree: 2011, University of Texas Southwestern Medical Center

 Proper control of actin cytoskeletal dynamics is essential for cell survival. The goals of my thesis work have been to characterize the structural and biophysical… (more)

Subjects/Keywords: Proto-Oncogene Proteins c-vav; Models, Molecular; Protein Structure, Tertiary

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APA (6th Edition):

Yu, B. (2011). Structural and Mechanistic Studies of Two Regulatory Factors in Actin Cytoskeletal Signaling: Vav and VopL. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/960

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yu, Bingke. “Structural and Mechanistic Studies of Two Regulatory Factors in Actin Cytoskeletal Signaling: Vav and VopL.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/960.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yu, Bingke. “Structural and Mechanistic Studies of Two Regulatory Factors in Actin Cytoskeletal Signaling: Vav and VopL.” 2011. Web. 07 Mar 2021.

Vancouver:

Yu B. Structural and Mechanistic Studies of Two Regulatory Factors in Actin Cytoskeletal Signaling: Vav and VopL. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/960.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yu B. Structural and Mechanistic Studies of Two Regulatory Factors in Actin Cytoskeletal Signaling: Vav and VopL. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/960

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Estrozi, Bruna. Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos).

Degree: PhD, Patologia, 2015, University of São Paulo

A incidência do melanoma cutâneo em pacientes adultos jovens tem aumentado consideravelmente nos últimos anos. Há, contudo, carência de conhecimentos clinicopatológicos e moleculares sobre os… (more)

Subjects/Keywords: Adulto jovem; Melanoma; Melanoma; Neoplasias cutâneas; Proteínas proto-oncogênicas B-raf; Proteínas proto-oncogênicas c-kit; Proteínas proto-oncogênicas p21(ras); Proto-oncogene proteins B-raf; Proto-oncogene proteins c-kit; Proto-oncogene proteins p21(ras); Skin neoplasms; Younf adult

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APA (6th Edition):

Estrozi, B. (2015). Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos). (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5144/tde-01042015-144721/ ;

Chicago Manual of Style (16th Edition):

Estrozi, Bruna. “Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos).” 2015. Doctoral Dissertation, University of São Paulo. Accessed March 07, 2021. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-01042015-144721/ ;.

MLA Handbook (7th Edition):

Estrozi, Bruna. “Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos).” 2015. Web. 07 Mar 2021.

Vancouver:

Estrozi B. Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos). [Internet] [Doctoral dissertation]. University of São Paulo; 2015. [cited 2021 Mar 07]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-01042015-144721/ ;.

Council of Science Editors:

Estrozi B. Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos). [Doctoral Dissertation]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-01042015-144721/ ;

5. 山本, 梓司. Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.

Degree: 博士(医学), 2018, Saitama Medical University / 埼玉医科大学

Multiple sclerosis is a neuroinflammatory demyelinating and neurodegenerative disease of the central nervous system characterized by recurrent and progressive demyelination/remyelination cycles, neuroinflammation, oligodendrocyte loss, demyelination,… (more)

Subjects/Keywords: Animals; Anti-Inflammatory Agents; Apoptosis; Cell Differentiation; Cell Line, Transformed; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Female; Gene Expression Regulation; Humans; MAP Kinase Signaling System; Male; Mice; Mice, Inbred C57BL; Monoamine Oxidase Inhibitors; Myelin Sheath; NLR Family, Pyrin Domain-Containing 3 Protein; Phosphatidic Acids; Proto-Oncogene Proteins c-bcl-2; p38 Mitogen-Activated Protein Kinases

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APA (6th Edition):

山本, . (2018). Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. (Thesis). Saitama Medical University / 埼玉医科大学. Retrieved from http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Thesis, Saitama Medical University / 埼玉医科大学. Accessed March 07, 2021. http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Web. 07 Mar 2021.

Vancouver:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Internet] [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. [cited 2021 Mar 07]. Available from: http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. Available from: http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oulu

6. Jääskeläinen, M. (Minna). Apoptosis-regulating factors in developing and adult ovaries.

Degree: 2010, University of Oulu

 Abstract Apoptosis plays a crucial part in human ovarian function from fetal development to the end of reproductive potential. Failures in the regulation of ovarian… (more)

Subjects/Keywords: TNF-related apoptosis-inducing ligand; Wnt proteins; apoptosis; caspase 3; estrogens; granulosa cells; oocytes; ovary; proto-oncogen proteins c-Bcl-2; tumor necrosis factor- alpha

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APA (6th Edition):

Jääskeläinen, M. (. (2010). Apoptosis-regulating factors in developing and adult ovaries. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789514263477

Chicago Manual of Style (16th Edition):

Jääskeläinen, M (Minna). “Apoptosis-regulating factors in developing and adult ovaries.” 2010. Doctoral Dissertation, University of Oulu. Accessed March 07, 2021. http://urn.fi/urn:isbn:9789514263477.

MLA Handbook (7th Edition):

Jääskeläinen, M (Minna). “Apoptosis-regulating factors in developing and adult ovaries.” 2010. Web. 07 Mar 2021.

Vancouver:

Jääskeläinen M(. Apoptosis-regulating factors in developing and adult ovaries. [Internet] [Doctoral dissertation]. University of Oulu; 2010. [cited 2021 Mar 07]. Available from: http://urn.fi/urn:isbn:9789514263477.

Council of Science Editors:

Jääskeläinen M(. Apoptosis-regulating factors in developing and adult ovaries. [Doctoral Dissertation]. University of Oulu; 2010. Available from: http://urn.fi/urn:isbn:9789514263477

7. Oliveira Filho, João Bosco de. Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana.

Degree: PhD, Patologia, 2008, University of São Paulo

A subfamília p21 RAS de pequenas GTPases, incluindo KRAS, HRAS e NRAS, participa de muitas redes de sinalização, incluindo proliferação celular, organização do citoesqueleto e… (more)

Subjects/Keywords: Apoptose; Apoptosis; Autoimmunity; Autoimunidade; BCL-2 -like protein 11; BIM; Mitogen activated protein kinase kinases; Proteínas proto-oncogênicas p21 (ras); Proto-oncogene proteins p21 (ras); Quinases de proteína quinase ativadas por mitógeno

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APA (6th Edition):

Oliveira Filho, J. B. d. (2008). Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5144/tde-04112008-174252/ ;

Chicago Manual of Style (16th Edition):

Oliveira Filho, João Bosco de. “Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana.” 2008. Doctoral Dissertation, University of São Paulo. Accessed March 07, 2021. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-04112008-174252/ ;.

MLA Handbook (7th Edition):

Oliveira Filho, João Bosco de. “Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana.” 2008. Web. 07 Mar 2021.

Vancouver:

Oliveira Filho JBd. Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana. [Internet] [Doctoral dissertation]. University of São Paulo; 2008. [cited 2021 Mar 07]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-04112008-174252/ ;.

Council of Science Editors:

Oliveira Filho JBd. Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana. [Doctoral Dissertation]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-04112008-174252/ ;

8. Liphaus, Bernadete de Lourdes. "Expressão das proteínas Fas e Bcl-2 em células mononucleares de crianças e adolescentes com lúpus eritematoso sistêmico".

Degree: PhD, Pediatria, 2005, University of São Paulo

Para verificar a expressão das proteínas Fas e Bcl-2 em linfócitos e suas correlações com a atividade da doença foram avaliados 38 pacientes com lúpus… (more)

Subjects/Keywords: ADOLESCENT; ADOLESCENTE; ANTÍGENOS CD95; ANTIGENS CD95; APOPTOSE; APOPTOSIS; AUTO-IMUNIDADE; AUTOIMMUNITY; CHILD; CITOMETRIA DE FLUXO; CRIANÇA; FLOW CYTOMETRY; ÍNDICE DE GRAVIDADE DE DOENÇA; LÚPUS ERITEMATOSO SISTÊMICO/fisiopatologia; LUPUS ERYTHEMATOSUS SYSTEMIC/physiopathology; PROTEÍNAS PROTO-ONCOGÊNICAS C-BCL-2; PROTO-ONCOGENE PROTEINS c-bcl-2; SEVERITY OF ILLNESS INDEX

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APA (6th Edition):

Liphaus, B. d. L. (2005). "Expressão das proteínas Fas e Bcl-2 em células mononucleares de crianças e adolescentes com lúpus eritematoso sistêmico". (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5141/tde-16022006-162336/ ;

Chicago Manual of Style (16th Edition):

Liphaus, Bernadete de Lourdes. “"Expressão das proteínas Fas e Bcl-2 em células mononucleares de crianças e adolescentes com lúpus eritematoso sistêmico".” 2005. Doctoral Dissertation, University of São Paulo. Accessed March 07, 2021. http://www.teses.usp.br/teses/disponiveis/5/5141/tde-16022006-162336/ ;.

MLA Handbook (7th Edition):

Liphaus, Bernadete de Lourdes. “"Expressão das proteínas Fas e Bcl-2 em células mononucleares de crianças e adolescentes com lúpus eritematoso sistêmico".” 2005. Web. 07 Mar 2021.

Vancouver:

Liphaus BdL. "Expressão das proteínas Fas e Bcl-2 em células mononucleares de crianças e adolescentes com lúpus eritematoso sistêmico". [Internet] [Doctoral dissertation]. University of São Paulo; 2005. [cited 2021 Mar 07]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5141/tde-16022006-162336/ ;.

Council of Science Editors:

Liphaus BdL. "Expressão das proteínas Fas e Bcl-2 em células mononucleares de crianças e adolescentes com lúpus eritematoso sistêmico". [Doctoral Dissertation]. University of São Paulo; 2005. Available from: http://www.teses.usp.br/teses/disponiveis/5/5141/tde-16022006-162336/ ;


University of Texas Southwestern Medical Center

9. Zhou, Huanyu. Molecular Regulation of Direct Cardiac Reprogramming.

Degree: 2017, University of Texas Southwestern Medical Center

 A heart attack (also known as myocardial infarction, MI) happens when the flow of blood to the heart is blocked. A massive heart attack can… (more)

Subjects/Keywords: Cellular Reprogramming; Fibroblasts; Myocytes, Cardiac; Proto-Oncogene Proteins c-akt; Transcription, Genetic

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APA (6th Edition):

Zhou, H. (2017). Molecular Regulation of Direct Cardiac Reprogramming. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7208

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhou, Huanyu. “Molecular Regulation of Direct Cardiac Reprogramming.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/7208.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhou, Huanyu. “Molecular Regulation of Direct Cardiac Reprogramming.” 2017. Web. 07 Mar 2021.

Vancouver:

Zhou H. Molecular Regulation of Direct Cardiac Reprogramming. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/7208.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhou H. Molecular Regulation of Direct Cardiac Reprogramming. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7208

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

10. Ou, Yi-Hung 1977-. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.

Degree: 2013, University of Texas Southwestern Medical Center

 An essential kinase in innate immune signaling, TBK1 couples pathogen surveillance to induction of host defense mechanisms. The pathological activation of TBK1 in cancer can… (more)

Subjects/Keywords: Immunity, Innate; Neoplasms; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt

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APA (6th Edition):

Ou, Y. 1. (2013). Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Web. 07 Mar 2021.

Vancouver:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

11. Bulut, Gamze Betul. Ubiquitination of EpoR and p85 in Ligand Induced EpoR Down-Regulation.

Degree: 2014, University of Texas Southwestern Medical Center

 Erythropoietin (Epo) is the primary cytokine that drives red blood cell production and signals through its receptor, the EpoR, on erythroid progenitor cells. Epo binding… (more)

Subjects/Keywords: Class Ia Phosphatidylinositol 3-Kinase; Endocytosis; Erythropoietin; Proto-Oncogene Proteins c-cbl; Receptors, Erythropoietin

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APA (6th Edition):

Bulut, G. B. (2014). Ubiquitination of EpoR and p85 in Ligand Induced EpoR Down-Regulation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3581

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bulut, Gamze Betul. “Ubiquitination of EpoR and p85 in Ligand Induced EpoR Down-Regulation.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/3581.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bulut, Gamze Betul. “Ubiquitination of EpoR and p85 in Ligand Induced EpoR Down-Regulation.” 2014. Web. 07 Mar 2021.

Vancouver:

Bulut GB. Ubiquitination of EpoR and p85 in Ligand Induced EpoR Down-Regulation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/3581.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bulut GB. Ubiquitination of EpoR and p85 in Ligand Induced EpoR Down-Regulation. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3581

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

12. Espindola, Marilia Bittencourt. Expressão imunoistoquímica da proteína bcl-2 em metástases de melanoma cutâneo e relação com a sobrevida.

Degree: 2007, Universidade do Rio Grande do Sul

 A morte celular programada (apoptose) tem sido implicada no desenvolvimento tumoral e no potencial metastático. O Bcl-2, um proto-oncogene inibidor da apoptose, vem sendo estudado… (more)

Subjects/Keywords: Melanoma; Neoplasias cutâneas; Sobrevida; Metástase neoplásica; Oncogenes; Proteínas proto-oncogênicas c-bcl-2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Espindola, M. B. (2007). Expressão imunoistoquímica da proteína bcl-2 em metástases de melanoma cutâneo e relação com a sobrevida. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/11425

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Espindola, Marilia Bittencourt. “Expressão imunoistoquímica da proteína bcl-2 em metástases de melanoma cutâneo e relação com a sobrevida.” 2007. Thesis, Universidade do Rio Grande do Sul. Accessed March 07, 2021. http://hdl.handle.net/10183/11425.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Espindola, Marilia Bittencourt. “Expressão imunoistoquímica da proteína bcl-2 em metástases de melanoma cutâneo e relação com a sobrevida.” 2007. Web. 07 Mar 2021.

Vancouver:

Espindola MB. Expressão imunoistoquímica da proteína bcl-2 em metástases de melanoma cutâneo e relação com a sobrevida. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2007. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10183/11425.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Espindola MB. Expressão imunoistoquímica da proteína bcl-2 em metástases de melanoma cutâneo e relação com a sobrevida. [Thesis]. Universidade do Rio Grande do Sul; 2007. Available from: http://hdl.handle.net/10183/11425

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

13. Troutman, Ty Dale. B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily.

Degree: 2014, University of Texas Southwestern Medical Center

 Toll-like receptor (TLR)/Interleukin-1 receptor (IL1R) superfamily members share signaling components and (with the exception of TLR3) depend on the adapter myeloid differentiation primary response gene… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing; B-Lymphocytes; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Signal Transduction; Toll-Like Receptors

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APA (6th Edition):

Troutman, T. D. (2014). B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Troutman, Ty Dale. “B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/3328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Troutman, Ty Dale. “B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily.” 2014. Web. 07 Mar 2021.

Vancouver:

Troutman TD. B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/3328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Troutman TD. B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

14. Wu, Nan. Cohesin, the SMC5/6 Complex and SUMO in DNA Repair.

Degree: 2013, University of Texas Southwestern Medical Center

 DNA double-strand breaks (DSBs) fuel cancer-driving chromosome translocations. Two related structural maintenance of chromosomes (Smc) complexes, cohesin and Smc5/6, promote DSB repair through sister-chromatid homologous… (more)

Subjects/Keywords: Chromatids; Proto-Oncogene Proteins; Recombination, Genetic

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APA (6th Edition):

Wu, N. (2013). Cohesin, the SMC5/6 Complex and SUMO in DNA Repair. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1259

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Nan. “Cohesin, the SMC5/6 Complex and SUMO in DNA Repair.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/1259.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Nan. “Cohesin, the SMC5/6 Complex and SUMO in DNA Repair.” 2013. Web. 07 Mar 2021.

Vancouver:

Wu N. Cohesin, the SMC5/6 Complex and SUMO in DNA Repair. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/1259.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu N. Cohesin, the SMC5/6 Complex and SUMO in DNA Repair. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1259

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

15. Guedez, Liliana, 1956-. Differential expression of bcl-2 by hematological tumors anti-apoptosis function and chemotherapy resistance.

Degree: 1995, University of Florida

Subjects/Keywords: Apoptosis; Cell cycle; Cell death; Cell growth; Cell lines; Cells; Cytometry; Free radicals; Mice; Tumors; Apoptosis  – physiology ( mesh ); Bleomycin  – drug effects ( mesh ); Bleomycin  – pharmacology ( mesh ); Cytarabine  – drug effects ( mesh ); Cytarabine  – pharmacology ( mesh ); Drug Resistance, Neoplasm  – physiology ( mesh ); Genes, bcl-2 ( mesh ); Hematologic Neoplasms  – physiopathology ( mesh ); Proto-Oncogene Proteins c-bcl-2 ( mesh )

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APA (6th Edition):

Guedez, Liliana, 1. (1995). Differential expression of bcl-2 by hematological tumors anti-apoptosis function and chemotherapy resistance. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00011162

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guedez, Liliana, 1956-. “Differential expression of bcl-2 by hematological tumors anti-apoptosis function and chemotherapy resistance.” 1995. Thesis, University of Florida. Accessed March 07, 2021. https://ufdc.ufl.edu/AA00011162.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guedez, Liliana, 1956-. “Differential expression of bcl-2 by hematological tumors anti-apoptosis function and chemotherapy resistance.” 1995. Web. 07 Mar 2021.

Vancouver:

Guedez, Liliana 1. Differential expression of bcl-2 by hematological tumors anti-apoptosis function and chemotherapy resistance. [Internet] [Thesis]. University of Florida; 1995. [cited 2021 Mar 07]. Available from: https://ufdc.ufl.edu/AA00011162.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guedez, Liliana 1. Differential expression of bcl-2 by hematological tumors anti-apoptosis function and chemotherapy resistance. [Thesis]. University of Florida; 1995. Available from: https://ufdc.ufl.edu/AA00011162

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Mendonça, Ullyanov Bezerra Toscano de. Análise de mutações do gene KIT em pacientes com melanoma de mucosa de cabeça e pescoço e relação clínica retrospectiva.

Degree: PhD, Clínica Cirúrgica, 2015, University of São Paulo

Introdução: O melanoma mucoso de cabeça e pescoço (MMCP) é mais agressivo do que o melanoma cutâneo, marcadores prognósticos desta patologia não foram completamente esclarecidos… (more)

Subjects/Keywords: Buccal mucosa; KIT; KIT; Melanoma; Melanoma; Mucosa bucal; Mucosa nasal; Mutação; Mutation; Nasal mucosa; Proteínas proto-oncogênicas c-kit; Proto-oncogene proteins c-kit

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APA (6th Edition):

Mendonça, U. B. T. d. (2015). Análise de mutações do gene KIT em pacientes com melanoma de mucosa de cabeça e pescoço e relação clínica retrospectiva. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5132/tde-14122015-114016/ ;

Chicago Manual of Style (16th Edition):

Mendonça, Ullyanov Bezerra Toscano de. “Análise de mutações do gene KIT em pacientes com melanoma de mucosa de cabeça e pescoço e relação clínica retrospectiva.” 2015. Doctoral Dissertation, University of São Paulo. Accessed March 07, 2021. http://www.teses.usp.br/teses/disponiveis/5/5132/tde-14122015-114016/ ;.

MLA Handbook (7th Edition):

Mendonça, Ullyanov Bezerra Toscano de. “Análise de mutações do gene KIT em pacientes com melanoma de mucosa de cabeça e pescoço e relação clínica retrospectiva.” 2015. Web. 07 Mar 2021.

Vancouver:

Mendonça UBTd. Análise de mutações do gene KIT em pacientes com melanoma de mucosa de cabeça e pescoço e relação clínica retrospectiva. [Internet] [Doctoral dissertation]. University of São Paulo; 2015. [cited 2021 Mar 07]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5132/tde-14122015-114016/ ;.

Council of Science Editors:

Mendonça UBTd. Análise de mutações do gene KIT em pacientes com melanoma de mucosa de cabeça e pescoço e relação clínica retrospectiva. [Doctoral Dissertation]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/5/5132/tde-14122015-114016/ ;

17. Escamilla-Powers, Julienne Rebeca. Identification of proteins that regulate c-Myc stability and function.

Degree: PhD, 2008, Oregon Health Sciences University

Subjects/Keywords: Oncogene Proteins  – physiology; Proto-Oncogene Proteins c-myc; Transcriptional Activation; Saccharomyces cerevisiae

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APA (6th Edition):

Escamilla-Powers, J. R. (2008). Identification of proteins that regulate c-Myc stability and function. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4CR5RB9 ; http://digitalcommons.ohsu.edu/etd/500

Chicago Manual of Style (16th Edition):

Escamilla-Powers, Julienne Rebeca. “Identification of proteins that regulate c-Myc stability and function.” 2008. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 07, 2021. doi:10.6083/M4CR5RB9 ; http://digitalcommons.ohsu.edu/etd/500.

MLA Handbook (7th Edition):

Escamilla-Powers, Julienne Rebeca. “Identification of proteins that regulate c-Myc stability and function.” 2008. Web. 07 Mar 2021.

Vancouver:

Escamilla-Powers JR. Identification of proteins that regulate c-Myc stability and function. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2008. [cited 2021 Mar 07]. Available from: doi:10.6083/M4CR5RB9 ; http://digitalcommons.ohsu.edu/etd/500.

Council of Science Editors:

Escamilla-Powers JR. Identification of proteins that regulate c-Myc stability and function. [Doctoral Dissertation]. Oregon Health Sciences University; 2008. Available from: doi:10.6083/M4CR5RB9 ; http://digitalcommons.ohsu.edu/etd/500

18. Hock, Thomas D. Regulation of the human heme oxygenase-1 gene.

Degree: PhD, 2007, University of Alabama – Birmingham

The heme oxygenase-1 (HO-1) gene encodes a microsomal enzyme that catalyzes the conversion of heme to carbon monoxide, Iron, and biliverdin. HO-1 transcription is induced… (more)

Subjects/Keywords: Heme Oxygenase-1  – genetics <; br>; Proto-Oncogene Proteins c-jun  – metabolism <; br>; Transcription Factors  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hock, T. D. (2007). Regulation of the human heme oxygenase-1 gene. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,126

Chicago Manual of Style (16th Edition):

Hock, Thomas D. “Regulation of the human heme oxygenase-1 gene.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,126.

MLA Handbook (7th Edition):

Hock, Thomas D. “Regulation of the human heme oxygenase-1 gene.” 2007. Web. 07 Mar 2021.

Vancouver:

Hock TD. Regulation of the human heme oxygenase-1 gene. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,126.

Council of Science Editors:

Hock TD. Regulation of the human heme oxygenase-1 gene. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,126

19. 竹山, 旭. Expression patterns of CD66a and CD117 in the mouse submandibular gland : CD117 と CD66a による唾液腺上皮細胞の分類.

Degree: 博士(歯学), 2016, Osaka Dental University / 大阪歯科大学

The epithelial tissue of the salivary gland consists of the acinar and ductal parts, the latter of which is further divided into the intercalated, striated… (more)

Subjects/Keywords: Animals; Carcinoembryonic Antigen; Female; Gene Expression Regulation; Mice; Proto-Oncogene Proteins c-kit; Salivary Ducts; Submandibular Gland

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APA (6th Edition):

竹山, . (2016). Expression patterns of CD66a and CD117 in the mouse submandibular gland : CD117 と CD66a による唾液腺上皮細胞の分類. (Thesis). Osaka Dental University / 大阪歯科大学. Retrieved from http://id.nii.ac.jp/1392/00000087/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

竹山, 旭. “Expression patterns of CD66a and CD117 in the mouse submandibular gland : CD117 と CD66a による唾液腺上皮細胞の分類.” 2016. Thesis, Osaka Dental University / 大阪歯科大学. Accessed March 07, 2021. http://id.nii.ac.jp/1392/00000087/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

竹山, 旭. “Expression patterns of CD66a and CD117 in the mouse submandibular gland : CD117 と CD66a による唾液腺上皮細胞の分類.” 2016. Web. 07 Mar 2021.

Vancouver:

竹山 . Expression patterns of CD66a and CD117 in the mouse submandibular gland : CD117 と CD66a による唾液腺上皮細胞の分類. [Internet] [Thesis]. Osaka Dental University / 大阪歯科大学; 2016. [cited 2021 Mar 07]. Available from: http://id.nii.ac.jp/1392/00000087/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

竹山 . Expression patterns of CD66a and CD117 in the mouse submandibular gland : CD117 と CD66a による唾液腺上皮細胞の分類. [Thesis]. Osaka Dental University / 大阪歯科大学; 2016. Available from: http://id.nii.ac.jp/1392/00000087/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Carr, Michael I. The Role of MDM2 Phosphorylation in P53 Responses to DNA Damage and Tumor Suppression: A Dissertation.

Degree: Cell Biology, Radiology, 2016, U of Massachusetts : Med

  The p53 tumor suppressor protein is upregulated in response to DNA damage and other stress signals. The upregulation of p53 involves freeing it from… (more)

Subjects/Keywords: Tumor Suppressor Protein p53; Proto-Oncogene Proteins c-mdm2; DNA Damage; Phosphorylation; Biochemistry; Cancer Biology; Cell Biology

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APA (6th Edition):

Carr, M. I. (2016). The Role of MDM2 Phosphorylation in P53 Responses to DNA Damage and Tumor Suppression: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/847

Chicago Manual of Style (16th Edition):

Carr, Michael I. “The Role of MDM2 Phosphorylation in P53 Responses to DNA Damage and Tumor Suppression: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. http://escholarship.umassmed.edu/gsbs_diss/847.

MLA Handbook (7th Edition):

Carr, Michael I. “The Role of MDM2 Phosphorylation in P53 Responses to DNA Damage and Tumor Suppression: A Dissertation.” 2016. Web. 07 Mar 2021.

Vancouver:

Carr MI. The Role of MDM2 Phosphorylation in P53 Responses to DNA Damage and Tumor Suppression: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2021 Mar 07]. Available from: http://escholarship.umassmed.edu/gsbs_diss/847.

Council of Science Editors:

Carr MI. The Role of MDM2 Phosphorylation in P53 Responses to DNA Damage and Tumor Suppression: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/847


University of Texas Southwestern Medical Center

21. Alibhai, Imran Nizamudin. Regulation of FOSB MRNA Isoforms by Drugs of Abuse.

Degree: 2006, University of Texas Southwestern Medical Center

 ΔFosB, a truncated splice isoform of FosB, is a transcription factor that accumulates within a subset of neurons after chronic administration of drugs of abuse… (more)

Subjects/Keywords: Proto-Oncogene Proteins c-fos; Amphetamine; Drug Abuse

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APA (6th Edition):

Alibhai, I. N. (2006). Regulation of FOSB MRNA Isoforms by Drugs of Abuse. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/586

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alibhai, Imran Nizamudin. “Regulation of FOSB MRNA Isoforms by Drugs of Abuse.” 2006. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/586.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alibhai, Imran Nizamudin. “Regulation of FOSB MRNA Isoforms by Drugs of Abuse.” 2006. Web. 07 Mar 2021.

Vancouver:

Alibhai IN. Regulation of FOSB MRNA Isoforms by Drugs of Abuse. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2006. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/586.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alibhai IN. Regulation of FOSB MRNA Isoforms by Drugs of Abuse. [Thesis]. University of Texas Southwestern Medical Center; 2006. Available from: http://hdl.handle.net/2152.5/586

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

22. Todorova, Pavlina Krasimirova. Mechanistic Analysis of Radiation-Induced Gliomagenesis.

Degree: 2017, University of Texas Southwestern Medical Center

 Glioblastomas (GBM) are devastating brain tumors refractory to any available treatment. Exposure to ionizing radiation (IR) is the only known GBM risk factor. The link… (more)

Subjects/Keywords: Brain Neoplasms; Cyclin-Dependent Kinase Inhibitor p15; Cyclin-Dependent Kinase Inhibitor p16; Glioblastoma; Proto-Oncogene Proteins c-met

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APA (6th Edition):

Todorova, P. K. (2017). Mechanistic Analysis of Radiation-Induced Gliomagenesis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Todorova, Pavlina Krasimirova. “Mechanistic Analysis of Radiation-Induced Gliomagenesis.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/7205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Todorova, Pavlina Krasimirova. “Mechanistic Analysis of Radiation-Induced Gliomagenesis.” 2017. Web. 07 Mar 2021.

Vancouver:

Todorova PK. Mechanistic Analysis of Radiation-Induced Gliomagenesis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/7205.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Todorova PK. Mechanistic Analysis of Radiation-Induced Gliomagenesis. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7205

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

23. Hale, Carly Fenwick. Identification and Characterization of Novel Mechanisms of Functional and Structural Synapse Remodeling: Focus on Vav Guanine Nucleotide Exchange Factors and MEF2 Transcription Factors.

Degree: 2014, University of Texas Southwestern Medical Center

 Proper development of synaptic connectivity is a dynamic process requiring formation, elimination, maintenance, and plasticity of synapses. During early postnatal development, excess synapses are formed… (more)

Subjects/Keywords: Cadherins; Fragile X Mental Retardation Protein; MEF2 Transcription Factors; Neuronal Plasticity; Proto-Oncogene Proteins c-vav; Synapses

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APA (6th Edition):

Hale, C. F. (2014). Identification and Characterization of Novel Mechanisms of Functional and Structural Synapse Remodeling: Focus on Vav Guanine Nucleotide Exchange Factors and MEF2 Transcription Factors. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hale, Carly Fenwick. “Identification and Characterization of Novel Mechanisms of Functional and Structural Synapse Remodeling: Focus on Vav Guanine Nucleotide Exchange Factors and MEF2 Transcription Factors.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/3588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hale, Carly Fenwick. “Identification and Characterization of Novel Mechanisms of Functional and Structural Synapse Remodeling: Focus on Vav Guanine Nucleotide Exchange Factors and MEF2 Transcription Factors.” 2014. Web. 07 Mar 2021.

Vancouver:

Hale CF. Identification and Characterization of Novel Mechanisms of Functional and Structural Synapse Remodeling: Focus on Vav Guanine Nucleotide Exchange Factors and MEF2 Transcription Factors. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/3588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hale CF. Identification and Characterization of Novel Mechanisms of Functional and Structural Synapse Remodeling: Focus on Vav Guanine Nucleotide Exchange Factors and MEF2 Transcription Factors. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

24. Deb, Dhruba. Oncogene-Induced Signaling Heterogeneity in Lung Cancer.

Degree: 2015, University of Texas Southwestern Medical Center

 Lung cancer causes the maximum number of cancer related deaths worldwide. In recent years, the cancer genome atlas (TCGA) initiative has identified 138 frequently occurring… (more)

Subjects/Keywords: Carcinoma, Squamous Cell; Lung Neoplasms; Proto-Oncogene Proteins c-myc; Smad2 Protein; Transforming Growth Factor beta

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Deb, D. (2015). Oncogene-Induced Signaling Heterogeneity in Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4213

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Deb, Dhruba. “Oncogene-Induced Signaling Heterogeneity in Lung Cancer.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/4213.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Deb, Dhruba. “Oncogene-Induced Signaling Heterogeneity in Lung Cancer.” 2015. Web. 07 Mar 2021.

Vancouver:

Deb D. Oncogene-Induced Signaling Heterogeneity in Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/4213.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Deb D. Oncogene-Induced Signaling Heterogeneity in Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4213

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

25. Mohammed Saleem, Yasir Ihsan. MOLECULAR MECHANISM OF ELLAGIC ACID AND UROLITHIN A IN THE SUPPRESSION OF PROSTATE CANCER BY INFLUENCING THE p53-MDM2 PATHWAY.

Degree: PhD, PHD-Pharmacology - Toxicology, 2019, East Carolina University

 Carcinoma of the prostate (CaP) is the most common cancer in men and the second leading cause of cancer-related death in men worldwide. Despite the… (more)

Subjects/Keywords: polyphenols; urolithin a; Prostatic Neoplasms; Tumor Suppressor Protein p53; Ellagic Acid; Heterocyclic Compounds; Proto-Oncogene Proteins c-mdm2; Male

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APA (6th Edition):

Mohammed Saleem, Y. I. (2019). MOLECULAR MECHANISM OF ELLAGIC ACID AND UROLITHIN A IN THE SUPPRESSION OF PROSTATE CANCER BY INFLUENCING THE p53-MDM2 PATHWAY. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/7438

Chicago Manual of Style (16th Edition):

Mohammed Saleem, Yasir Ihsan. “MOLECULAR MECHANISM OF ELLAGIC ACID AND UROLITHIN A IN THE SUPPRESSION OF PROSTATE CANCER BY INFLUENCING THE p53-MDM2 PATHWAY.” 2019. Doctoral Dissertation, East Carolina University. Accessed March 07, 2021. http://hdl.handle.net/10342/7438.

MLA Handbook (7th Edition):

Mohammed Saleem, Yasir Ihsan. “MOLECULAR MECHANISM OF ELLAGIC ACID AND UROLITHIN A IN THE SUPPRESSION OF PROSTATE CANCER BY INFLUENCING THE p53-MDM2 PATHWAY.” 2019. Web. 07 Mar 2021.

Vancouver:

Mohammed Saleem YI. MOLECULAR MECHANISM OF ELLAGIC ACID AND UROLITHIN A IN THE SUPPRESSION OF PROSTATE CANCER BY INFLUENCING THE p53-MDM2 PATHWAY. [Internet] [Doctoral dissertation]. East Carolina University; 2019. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10342/7438.

Council of Science Editors:

Mohammed Saleem YI. MOLECULAR MECHANISM OF ELLAGIC ACID AND UROLITHIN A IN THE SUPPRESSION OF PROSTATE CANCER BY INFLUENCING THE p53-MDM2 PATHWAY. [Doctoral Dissertation]. East Carolina University; 2019. Available from: http://hdl.handle.net/10342/7438

26. Besret, Soizic. Ligations chimiques : synthèse d'inhibiteurs extracellulaires de la signalisation HGF/SF-MET : Chemical Ligation Synthesis of extracellular inhibitors of HGF/SF-MET signalling pathway.

Degree: Docteur es, Sciences du médicament, 2011, Université Lille II – Droit et Santé

Les peptides constituent une famille de biomolécules dont l’utilisation dans différents domaines thérapeutiques (cancer, diabète, sida) s’est fortement développée ces dernières années. Le défi pour… (more)

Subjects/Keywords: Ligations chimiques; Ligations peptidiques; Aza Glypeptide; Inhibiteurs de MET; Proto Oncogene Proteins c met; Peptide Library

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Besret, S. (2011). Ligations chimiques : synthèse d'inhibiteurs extracellulaires de la signalisation HGF/SF-MET : Chemical Ligation Synthesis of extracellular inhibitors of HGF/SF-MET signalling pathway. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2011LIL2S003

Chicago Manual of Style (16th Edition):

Besret, Soizic. “Ligations chimiques : synthèse d'inhibiteurs extracellulaires de la signalisation HGF/SF-MET : Chemical Ligation Synthesis of extracellular inhibitors of HGF/SF-MET signalling pathway.” 2011. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed March 07, 2021. http://www.theses.fr/2011LIL2S003.

MLA Handbook (7th Edition):

Besret, Soizic. “Ligations chimiques : synthèse d'inhibiteurs extracellulaires de la signalisation HGF/SF-MET : Chemical Ligation Synthesis of extracellular inhibitors of HGF/SF-MET signalling pathway.” 2011. Web. 07 Mar 2021.

Vancouver:

Besret S. Ligations chimiques : synthèse d'inhibiteurs extracellulaires de la signalisation HGF/SF-MET : Chemical Ligation Synthesis of extracellular inhibitors of HGF/SF-MET signalling pathway. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2011. [cited 2021 Mar 07]. Available from: http://www.theses.fr/2011LIL2S003.

Council of Science Editors:

Besret S. Ligations chimiques : synthèse d'inhibiteurs extracellulaires de la signalisation HGF/SF-MET : Chemical Ligation Synthesis of extracellular inhibitors of HGF/SF-MET signalling pathway. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2011. Available from: http://www.theses.fr/2011LIL2S003


University of Texas Southwestern Medical Center

27. Ahmed, Kamran. Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury.

Degree: 2016, University of Texas Southwestern Medical Center

The general metadata  – e.g., title, author, abstract, subject headings, etc.  – is publicly available, but access to the submitted files is restricted to UT… (more)

Subjects/Keywords: Heart; Insulin-Like Growth Factor I; Myocardial Reperfusion Injury; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; rho-Associated Kinases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ahmed, K. (2016). Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ahmed, Kamran. “Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/7069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ahmed, Kamran. “Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury.” 2016. Web. 07 Mar 2021.

Vancouver:

Ahmed K. Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/7069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ahmed K. Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/7069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Tortelli Júnior, Tharcisio Citrangulo. Avaliação do papel de proibitina no desenvolvimento de resistência à cisplatina em linhagens de melanoma humano.

Degree: Mestrado, Oncologia, 2008, University of São Paulo

A incidência de melanomas tem crescido mundialmente. Apesar de representar um potencial problema de saúde pública pela sua incidência crescente, melanomas ainda se apresentam como… (more)

Subjects/Keywords: Cell death; Cisplatin; Cisplatina; Melanoma; Melanoma; Morte celular; Proteínas proto-oncogênicas c-akt; Proto-oncogene proteins; RNA interferente pequeno; RNA small interfering

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APA (6th Edition):

Tortelli Júnior, T. C. (2008). Avaliação do papel de proibitina no desenvolvimento de resistência à cisplatina em linhagens de melanoma humano. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5155/tde-12022009-135328/ ;

Chicago Manual of Style (16th Edition):

Tortelli Júnior, Tharcisio Citrangulo. “Avaliação do papel de proibitina no desenvolvimento de resistência à cisplatina em linhagens de melanoma humano.” 2008. Masters Thesis, University of São Paulo. Accessed March 07, 2021. http://www.teses.usp.br/teses/disponiveis/5/5155/tde-12022009-135328/ ;.

MLA Handbook (7th Edition):

Tortelli Júnior, Tharcisio Citrangulo. “Avaliação do papel de proibitina no desenvolvimento de resistência à cisplatina em linhagens de melanoma humano.” 2008. Web. 07 Mar 2021.

Vancouver:

Tortelli Júnior TC. Avaliação do papel de proibitina no desenvolvimento de resistência à cisplatina em linhagens de melanoma humano. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2021 Mar 07]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-12022009-135328/ ;.

Council of Science Editors:

Tortelli Júnior TC. Avaliação do papel de proibitina no desenvolvimento de resistência à cisplatina em linhagens de melanoma humano. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/5/5155/tde-12022009-135328/ ;

29. Willems, Kristen N. Regulation of Humoral Immunity by Pim Kinases: A Dissertation.

Degree: Immunology and Microbiology, Microbiology and Physiological Systems, 2011, U of Massachusetts : Med

  Pim (Provirus Integration site for Moloney murine leukemia virus) kinases are a family of three serine/threonine kinases involved in cell cycle, survival and metabolism.… (more)

Subjects/Keywords: Proto-Oncogene Proteins c-pim-1; Proto-Oncogene Proteins; Protein-Serine-Threonine Kinases; Immunity; Humoral; Amino Acids, Peptides, and Proteins; Enzymes and Coenzymes; Hemic and Immune Systems; Immunology and Infectious Disease; Viruses

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APA (6th Edition):

Willems, K. N. (2011). Regulation of Humoral Immunity by Pim Kinases: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/567

Chicago Manual of Style (16th Edition):

Willems, Kristen N. “Regulation of Humoral Immunity by Pim Kinases: A Dissertation.” 2011. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. https://escholarship.umassmed.edu/gsbs_diss/567.

MLA Handbook (7th Edition):

Willems, Kristen N. “Regulation of Humoral Immunity by Pim Kinases: A Dissertation.” 2011. Web. 07 Mar 2021.

Vancouver:

Willems KN. Regulation of Humoral Immunity by Pim Kinases: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2011. [cited 2021 Mar 07]. Available from: https://escholarship.umassmed.edu/gsbs_diss/567.

Council of Science Editors:

Willems KN. Regulation of Humoral Immunity by Pim Kinases: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2011. Available from: https://escholarship.umassmed.edu/gsbs_diss/567

30. Trabucco, Sally E. The SMURF2-YY1-C-MYC Axis in the Germinal Center Reaction and Diffuse Large B Cell Lymphoma: A Dissertation.

Degree: Cell Biology, Pediatrics, 2016, U of Massachusetts : Med

  Diffuse large B cell lymphoma (DLBCL) is the most common non-Hodgkin’s lymphoma. Patients who fail conventional therapy (~50%) have a poor prognosis and few… (more)

Subjects/Keywords: Diffuse Large B-Cell Lymphoma; YY1 Transcription Factor; Ubiquitin-Protein Ligases; Germinal Center; Proto-Oncogene Proteins c-myc; SMURF2-YY1-C-MYC Axis; Cancer Biology; Cell Biology; Neoplasms

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Trabucco, S. E. (2016). The SMURF2-YY1-C-MYC Axis in the Germinal Center Reaction and Diffuse Large B Cell Lymphoma: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/864

Chicago Manual of Style (16th Edition):

Trabucco, Sally E. “The SMURF2-YY1-C-MYC Axis in the Germinal Center Reaction and Diffuse Large B Cell Lymphoma: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. http://escholarship.umassmed.edu/gsbs_diss/864.

MLA Handbook (7th Edition):

Trabucco, Sally E. “The SMURF2-YY1-C-MYC Axis in the Germinal Center Reaction and Diffuse Large B Cell Lymphoma: A Dissertation.” 2016. Web. 07 Mar 2021.

Vancouver:

Trabucco SE. The SMURF2-YY1-C-MYC Axis in the Germinal Center Reaction and Diffuse Large B Cell Lymphoma: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2021 Mar 07]. Available from: http://escholarship.umassmed.edu/gsbs_diss/864.

Council of Science Editors:

Trabucco SE. The SMURF2-YY1-C-MYC Axis in the Germinal Center Reaction and Diffuse Large B Cell Lymphoma: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/864

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