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You searched for subject:(Protein Transport 60). Showing records 1 – 8 of 8 total matches.

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1. Marsh, Amie. Localization of proteins involved in trafficking in frog and mouse retina.

Degree: MS, 2012, University of Alabama – Birmingham

The connecting cilium serves as the major route for protein transport from the inner to outer segment of photoreceptor cells. The hypothesis that all Congenital… (more)

Subjects/Keywords: Cilia<; br>; Night Blindness<; br>; Protein Transport  – physiology<; br>; Rod Cell Outer Segment  – ultrastructure<; br>; Rhodopsin  – metabolism<; br>; Xenopus laevis

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APA (6th Edition):

Marsh, A. (2012). Localization of proteins involved in trafficking in frog and mouse retina. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1186

Chicago Manual of Style (16th Edition):

Marsh, Amie. “Localization of proteins involved in trafficking in frog and mouse retina.” 2012. Masters Thesis, University of Alabama – Birmingham. Accessed December 10, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1186.

MLA Handbook (7th Edition):

Marsh, Amie. “Localization of proteins involved in trafficking in frog and mouse retina.” 2012. Web. 10 Dec 2019.

Vancouver:

Marsh A. Localization of proteins involved in trafficking in frog and mouse retina. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2012. [cited 2019 Dec 10]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1186.

Council of Science Editors:

Marsh A. Localization of proteins involved in trafficking in frog and mouse retina. [Masters Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1186

2. Grabski, Robert. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.

Degree: PhD, 2008, University of Alabama – Birmingham

Metazoan cells are characterized with elaborate network of intracellular membranous compartments. These membranes allow the cell to spatially separate antagonistic processes and environments, and maintain… (more)

Subjects/Keywords: Carrier Proteins  – metabolism <; br>; Golgi Apparatus  – metabolism <; br>; Membrane Proteins  – metabolism <; br>; Protein Transport <; br>; RNA Interference

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APA (6th Edition):

Grabski, R. (2008). Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,328

Chicago Manual of Style (16th Edition):

Grabski, Robert. “Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 10, 2019. http://contentdm.mhsl.uab.edu/u?/etd,328.

MLA Handbook (7th Edition):

Grabski, Robert. “Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic.” 2008. Web. 10 Dec 2019.

Vancouver:

Grabski R. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Dec 10]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,328.

Council of Science Editors:

Grabski R. Using RNA interference to study the function of the tethering protein p115 in ER-Golgi traffic. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,328

3. Williams, Corey L. Analysis of cystic kidney disease-related genes in Caenorhabditis elegans.

Degree: PhD, 2009, University of Alabama – Birmingham

Cilia are evolutionarily conserved, membrane-bound, microtubule-based organelles found on a diverse array of cell types in eukaryotic organisms. Inherited diseases of cilia protein dysfunction include… (more)

Subjects/Keywords: Caenorhabditis elegans  – cytology<; br>; Caenorhabditis elegans  – metabolism<; br>; Caenorhabditis elegans Proteins  – metabolism<; br>; Cilia  – metabolism<; br>; Protein Transport

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APA (6th Edition):

Williams, C. L. (2009). Analysis of cystic kidney disease-related genes in Caenorhabditis elegans. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,407

Chicago Manual of Style (16th Edition):

Williams, Corey L. “Analysis of cystic kidney disease-related genes in Caenorhabditis elegans.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 10, 2019. http://contentdm.mhsl.uab.edu/u?/etd,407.

MLA Handbook (7th Edition):

Williams, Corey L. “Analysis of cystic kidney disease-related genes in Caenorhabditis elegans.” 2009. Web. 10 Dec 2019.

Vancouver:

Williams CL. Analysis of cystic kidney disease-related genes in Caenorhabditis elegans. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 10]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,407.

Council of Science Editors:

Williams CL. Analysis of cystic kidney disease-related genes in Caenorhabditis elegans. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,407

4. Cui, Wenjun. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.

Degree: PhD, 2010, University of Alabama – Birmingham

The unfolded protein response is one mechanism utilized by endoplasmic reticulum (ER) to maintain the homeostasis between ER protein folding machinery and ER proteins. UPR… (more)

Subjects/Keywords: Mitochondrial Membrane Transport Proteins  – chemistry<; br>; Mitochondrial Membrane Transport Proteins  – metabolism<; br>; Mitochondrial Membranes  – metabolism<; br>; Saccharomyces cerevisiae  – metabolism<; br>; Saccharomyces cerevisiae Proteins  – chemistry<; br>; Saccharomyces cerevisiae Proteins  – metabolism<; br>; Unfolded Protein Response

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APA (6th Edition):

Cui, W. (2010). Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1088

Chicago Manual of Style (16th Edition):

Cui, Wenjun. “Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 10, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1088.

MLA Handbook (7th Edition):

Cui, Wenjun. “Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.” 2010. Web. 10 Dec 2019.

Vancouver:

Cui W. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Dec 10]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1088.

Council of Science Editors:

Cui W. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1088

5. Pyle, Louise Clare. Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis.

Degree: PhD, 2009, University of Alabama – Birmingham

Cystic fibrosis (CF) is a lethal genetic disorder leading to pulmonary decline and premature death. The gene responsible for CF, the cystic fibrosis transmembrane conductance… (more)

Subjects/Keywords: Biological Markers  – metabolism<; br>; Cystic Fibrosis  – metabolism<; br>; Cystic Fibrosis Transmembrane Conductance Regulator<; br>; High-Throughput Screening Assays<; br>; Ion Channel Gating  – drug effects<; br>; Membrane Transport Modulators  – pharmacology<; br>; Mutant Proteins  – metabolism<; br>; Protein Structure, Tertiary  – genetics<; br>; Quercetin  – pharmacology

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APA (6th Edition):

Pyle, L. C. (2009). Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1106

Chicago Manual of Style (16th Edition):

Pyle, Louise Clare. “Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 10, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1106.

MLA Handbook (7th Edition):

Pyle, Louise Clare. “Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis.” 2009. Web. 10 Dec 2019.

Vancouver:

Pyle LC. Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Dec 10]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1106.

Council of Science Editors:

Pyle LC. Cystic fibrosis transmembrane conductance regulator (CFTR) modulators and their mechanistic basis: relevance to emerging therapies for cystic fibrosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1106

6. Cuddapah, Vishnu Anand. Regulation Of Clc-3 In Human Malignant Glioma.

Degree: PhD, 2012, University of Alabama – Birmingham

Malignant gliomas are the most common and deadly form of primary brain cancer afflicting adults. Current treatment regimens, including surgical debulking, radiotherapy, and chemotherapy, have… (more)

Subjects/Keywords: Brain Neoplasms – metabolism<; br>; Calcium-Calmodulin-Dependent Protein Kinase Type 2 – metabolism.<; br>; Cell Movement – physiology<; br>; Chloride Channels – metabolism.<; br>; Gene Expression Regulation<; br>; Gene Expression Regulation, Enzymologic<; br>; Gene Expression Regulation, Neoplastic<; br>; Glioma – metabolism<; br>; Ion Channels – metabolism<; br>; Membrane Transport Proteins – metabolism.<; br>; Mitosis<; br>; Neoplasms – metabolism<; br>; Neoplasms – pathology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cuddapah, V. A. (2012). Regulation Of Clc-3 In Human Malignant Glioma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1394

Chicago Manual of Style (16th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 10, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1394.

MLA Handbook (7th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Web. 10 Dec 2019.

Vancouver:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Dec 10]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394.

Council of Science Editors:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394

7. Brantley, Emily Claire. Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression.

Degree: PhD, 2007, University of Alabama – Birmingham

Glioblastoma multiforme (GBM) is the most common form of cancer in the central nervous system in adults. Because of the infiltrative and aggressive nature of… (more)

Subjects/Keywords: Brain Neoplasms  – metabolism <; br>; Brain Neoplasms  – physiopathology <; br>; Glioblastoma  – metabolism <; br>; Glioblastoma  – physiopathology <; br>; Protein Transport  – genetics <; br>; STAT3 Transcription Factor

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APA (6th Edition):

Brantley, E. C. (2007). Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,193

Chicago Manual of Style (16th Edition):

Brantley, Emily Claire. “Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 10, 2019. http://contentdm.mhsl.uab.edu/u?/etd,193.

MLA Handbook (7th Edition):

Brantley, Emily Claire. “Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression.” 2007. Web. 10 Dec 2019.

Vancouver:

Brantley EC. Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Dec 10]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,193.

Council of Science Editors:

Brantley EC. Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,193

8. Winkelbauer, Marlene Elizabeth. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.

Degree: PhD, 2007, University of Alabama – Birmingham

Numerous disorders are characterized by the presence of cystic lesions within the kidney. The proteins associated with these disorders often localize to cilia, and improper… (more)

Subjects/Keywords: Caenorhabditis elegans  – genetics<; br>; Caenorhabditis elegans Proteins  – genetics<; br>; Cilia  – metabolism<; br>; Neurons, Afferent  – physiology<; br>; Protein Transport<; br>; Transcription Factors  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Winkelbauer, M. E. (2007). Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,619

Chicago Manual of Style (16th Edition):

Winkelbauer, Marlene Elizabeth. “Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed December 10, 2019. http://contentdm.mhsl.uab.edu/u?/etd,619.

MLA Handbook (7th Edition):

Winkelbauer, Marlene Elizabeth. “Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model.” 2007. Web. 10 Dec 2019.

Vancouver:

Winkelbauer ME. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Dec 10]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,619.

Council of Science Editors:

Winkelbauer ME. Elucidating the role of nephronophthisis proteins utilizing Caenorhabditis elegans as a model. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,619

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