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You searched for subject:(Protein Serine Threonine Kinases). Showing records 1 – 30 of 19099 total matches.

[1] [2] [3] [4] [5] … [637]

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University of Texas Southwestern Medical Center

1. Zaman, Aubhishek. Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection.

Degree: 2017, University of Texas Southwestern Medical Center

 Monomeric RALGTPases, via direct binding to the exocyst (a.k.a Sec6/8 complex), help mount productive cell autonomous responses to trophic and immunogenic signals. However, RAL-exocyst downstream… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Signal Transduction; TOR Serine-Threonine Kinases

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APA (6th Edition):

Zaman, A. (2017). Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zaman, Aubhishek. “Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/7737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zaman, Aubhishek. “Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection.” 2017. Web. 09 Jul 2020.

Vancouver:

Zaman A. Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/7737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zaman A. Dynamic Modulation of Exocyst Interaction Networks Integrates Hippo and mTOR Pathway Response to Pathogen Detection. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

2. Shang, Libin. Mechanistic Studies of Autophagy Initiation in Mammalian Cells.

Degree: PhD, 2011, University of Texas Southwestern Medical Center

 Macroautophagy (herein referred to as autophagy) is an evolutionarily conserved self-digestive process cells use to adapt to starvation and other stresses. During autophagy, portions of… (more)

Subjects/Keywords: TOR-Serine-Threonine Kinases; Protein-Serine-Threonine Kinases; Autophagy

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APA (6th Edition):

Shang, L. (2011). Mechanistic Studies of Autophagy Initiation in Mammalian Cells. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/886

Chicago Manual of Style (16th Edition):

Shang, Libin. “Mechanistic Studies of Autophagy Initiation in Mammalian Cells.” 2011. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/886.

MLA Handbook (7th Edition):

Shang, Libin. “Mechanistic Studies of Autophagy Initiation in Mammalian Cells.” 2011. Web. 09 Jul 2020.

Vancouver:

Shang L. Mechanistic Studies of Autophagy Initiation in Mammalian Cells. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/886.

Council of Science Editors:

Shang L. Mechanistic Studies of Autophagy Initiation in Mammalian Cells. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/886


University of Texas Southwestern Medical Center

3. Wong, Sze. Regulation of Human Telomerase Alternative Splicing.

Degree: 2014, University of Texas Southwestern Medical Center

 Telomerase adds TTAGGG repeats onto chromosome ends (telomeres). Since telomerase is expressed in ~90% of all human cancer cells while being absent in most somatic… (more)

Subjects/Keywords: Alternative Splicing; Protein-Serine-Threonine Kinases; Telomerase

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APA (6th Edition):

Wong, S. (2014). Regulation of Human Telomerase Alternative Splicing. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3574

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wong, Sze. “Regulation of Human Telomerase Alternative Splicing.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/3574.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wong, Sze. “Regulation of Human Telomerase Alternative Splicing.” 2014. Web. 09 Jul 2020.

Vancouver:

Wong S. Regulation of Human Telomerase Alternative Splicing. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/3574.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wong S. Regulation of Human Telomerase Alternative Splicing. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3574

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Estrada, Armando, III 1980-. Regulation of Cell Migration by WNK1.

Degree: 2012, University of Texas Southwestern Medical Center

 Cell motility is an immensely complex process that involves reorganization of the cytoskeleton, and consequent membrane deformation, triggered by a variety of motogenic stimuli, including… (more)

Subjects/Keywords: Cell Movement; Cytoskeleton; Protein-Serine-Threonine Kinases

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APA (6th Edition):

Estrada, Armando, I. 1. (2012). Regulation of Cell Migration by WNK1. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Estrada, Armando, III 1980-. “Regulation of Cell Migration by WNK1.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Estrada, Armando, III 1980-. “Regulation of Cell Migration by WNK1.” 2012. Web. 09 Jul 2020.

Vancouver:

Estrada, Armando I1. Regulation of Cell Migration by WNK1. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Estrada, Armando I1. Regulation of Cell Migration by WNK1. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2012-12-65

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

5. Wedin, Kyle Edward. With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases.

Degree: PhD, 2011, University of Texas Southwestern Medical Center

 With no lysine (WNK) kinases are a family of protein kinases characterized by unusual kinase domain architecture. These large proteins, divergent outside of a kinase… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Hypertension; Transcription Factors

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APA (6th Edition):

Wedin, K. E. (2011). With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/892

Chicago Manual of Style (16th Edition):

Wedin, Kyle Edward. “With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases.” 2011. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/892.

MLA Handbook (7th Edition):

Wedin, Kyle Edward. “With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases.” 2011. Web. 09 Jul 2020.

Vancouver:

Wedin KE. With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/892.

Council of Science Editors:

Wedin KE. With No Lysine (WNK) Family Proteins and Their Interaction with Downstream Kinases. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/892


University of Texas Southwestern Medical Center

6. Moon, Thomas Matthew. Autoinhibition and Chloride Sensing in the WNK1 Kinase.

Degree: PhD, Molecular Biophysics, 2012, University of Texas Southwestern Medical Center

Protein kinases control diverse cellular pathways and have are the subject of intensive study regarding how they maintain specificity toward sub- strates. The research presented… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Crystallography, X-Ray; Protein Kinases Inhibitors

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APA (6th Edition):

Moon, T. M. (2012). Autoinhibition and Chloride Sensing in the WNK1 Kinase. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1001

Chicago Manual of Style (16th Edition):

Moon, Thomas Matthew. “Autoinhibition and Chloride Sensing in the WNK1 Kinase.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1001.

MLA Handbook (7th Edition):

Moon, Thomas Matthew. “Autoinhibition and Chloride Sensing in the WNK1 Kinase.” 2012. Web. 09 Jul 2020.

Vancouver:

Moon TM. Autoinhibition and Chloride Sensing in the WNK1 Kinase. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1001.

Council of Science Editors:

Moon TM. Autoinhibition and Chloride Sensing in the WNK1 Kinase. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1001


University of Texas Southwestern Medical Center

7. Taylor, Clinton A., IV. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.

Degree: 2016, University of Texas Southwestern Medical Center

Protein-protein interactions are essential for nearly every cellular process. Within signaling pathways, such interactions carry out numerous functions such as defining substrate specificity, inhibition of… (more)

Subjects/Keywords: Mitogen-Activated Protein Kinase Kinases; Protein-Serine-Threonine Kinases; Signal Transduction; Transcription Factors

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APA (6th Edition):

Taylor, Clinton A., I. (2016). Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Taylor, Clinton A., IV. “Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/6152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Taylor, Clinton A., IV. “Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.” 2016. Web. 09 Jul 2020.

Vancouver:

Taylor, Clinton A. I. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/6152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Taylor, Clinton A. I. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

8. Tu, Szu-Wei. Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking.

Degree: 2013, University of Texas Southwestern Medical Center

The file named "TU-DISSERTATION-2013.pdf" is the primary dissertation file. Twelve (12) supplemental videos are also provided (in Audio Video Interleaved format).

With No Lysine (WNK)… (more)

Subjects/Keywords: Cytokinesis; Mitosis; Protein-Serine-Threonine Kinases; Spindle Apparatus

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APA (6th Edition):

Tu, S. (2013). Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1742

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tu, Szu-Wei. “Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1742.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tu, Szu-Wei. “Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking.” 2013. Web. 09 Jul 2020.

Vancouver:

Tu S. Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1742.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tu S. Investigating the Biological Functions of the Protein Kinase WNK1 in the Regulation of Cytoskeletal Structures and Membrane Trafficking. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1742

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

9. Yoon, Jimok. Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling.

Degree: 2013, University of Texas Southwestern Medical Center

 The mechanisms that regulate the cellular behaviors including morphology, motility, navigation, and connectivity that are critical for normal human health are still incompletely understood. These… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Transcription Factors; DNA-Binding Proteins

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APA (6th Edition):

Yoon, J. (2013). Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1744

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yoon, Jimok. “Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1744.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yoon, Jimok. “Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling.” 2013. Web. 09 Jul 2020.

Vancouver:

Yoon J. Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1744.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yoon J. Characterizing New Molecules and Mechanisms of Semaphorin/Plexin/MICAL Signaling. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1744

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

10. Shi, Qing. Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway.

Degree: PhD, Genetics and Development, 2012, University of Texas Southwestern Medical Center

 Hedgehog (Hh) signaling is essential for both embryonic development and adult tissue homeostasis. Malfunction of Hh signaling pathway causes many human disorders including birth defects… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Drosophila Proteins; Hedgehog Proteins

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APA (6th Edition):

Shi, Q. (2012). Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1037

Chicago Manual of Style (16th Edition):

Shi, Qing. “Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1037.

MLA Handbook (7th Edition):

Shi, Qing. “Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway.” 2012. Web. 09 Jul 2020.

Vancouver:

Shi Q. Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1037.

Council of Science Editors:

Shi Q. Dissection of Mechanisms Regulating the Drosophila Hedgehog Pathway. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1037


University of Texas Southwestern Medical Center

11. Jacob, Leni Susan. Functional Genomics Based Interrogation of Cell-Fate Determination Pathways.

Degree: PhD, 2011, University of Texas Southwestern Medical Center

 The Hedgehog (Hh) and Wnt signal transduction pathways are master regulators of embryogenesis and tissue renewal and represent anticancer therapeutic targets. Using genome-wide RNA interference… (more)

Subjects/Keywords: Signal Transduction; Protein-Serine-Threonine Kinases; Wnt Proteins

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APA (6th Edition):

Jacob, L. S. (2011). Functional Genomics Based Interrogation of Cell-Fate Determination Pathways. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/884

Chicago Manual of Style (16th Edition):

Jacob, Leni Susan. “Functional Genomics Based Interrogation of Cell-Fate Determination Pathways.” 2011. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/884.

MLA Handbook (7th Edition):

Jacob, Leni Susan. “Functional Genomics Based Interrogation of Cell-Fate Determination Pathways.” 2011. Web. 09 Jul 2020.

Vancouver:

Jacob LS. Functional Genomics Based Interrogation of Cell-Fate Determination Pathways. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/884.

Council of Science Editors:

Jacob LS. Functional Genomics Based Interrogation of Cell-Fate Determination Pathways. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/884


University of Texas Southwestern Medical Center

12. Yue, Tao. Studies of the Hippo Signaling Pathway.

Degree: PhD, Genetics and Development, 2012, University of Texas Southwestern Medical Center

 How multicellular organisms control their growth to reach proper organ size during development is a fascinating question. Recent studies, initially from Drosophila, have identified the… (more)

Subjects/Keywords: Tumor Suppressor Proteins; Protein-Serine-Threonine Kinases; Drosophila melanogaster

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yue, T. (2012). Studies of the Hippo Signaling Pathway. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1104

Chicago Manual of Style (16th Edition):

Yue, Tao. “Studies of the Hippo Signaling Pathway.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1104.

MLA Handbook (7th Edition):

Yue, Tao. “Studies of the Hippo Signaling Pathway.” 2012. Web. 09 Jul 2020.

Vancouver:

Yue T. Studies of the Hippo Signaling Pathway. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1104.

Council of Science Editors:

Yue T. Studies of the Hippo Signaling Pathway. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1104


University of Texas Southwestern Medical Center

13. Cheng, Chih-Jen. Understanding Potassium Homeostasis Using Human and Mouse Genetic Models.

Degree: PhD, Integrative Biology, 2013, University of Texas Southwestern Medical Center

 Potassium homeostasis is one of the most sophisticated processes involving multiple organs in mammals. Many physiological functions, such as excitability of muscles and neurons, rely… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Potassium Channels, Inwardly Rectifying; Kidney

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APA (6th Edition):

Cheng, C. (2013). Understanding Potassium Homeostasis Using Human and Mouse Genetic Models. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1234

Chicago Manual of Style (16th Edition):

Cheng, Chih-Jen. “Understanding Potassium Homeostasis Using Human and Mouse Genetic Models.” 2013. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1234.

MLA Handbook (7th Edition):

Cheng, Chih-Jen. “Understanding Potassium Homeostasis Using Human and Mouse Genetic Models.” 2013. Web. 09 Jul 2020.

Vancouver:

Cheng C. Understanding Potassium Homeostasis Using Human and Mouse Genetic Models. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1234.

Council of Science Editors:

Cheng C. Understanding Potassium Homeostasis Using Human and Mouse Genetic Models. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1234


University of Texas Southwestern Medical Center

14. Ou, Yi-Hung 1977-. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.

Degree: 2013, University of Texas Southwestern Medical Center

 An essential kinase in innate immune signaling, TBK1 couples pathogen surveillance to induction of host defense mechanisms. The pathological activation of TBK1 in cancer can… (more)

Subjects/Keywords: Immunity, Innate; Neoplasms; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt

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APA (6th Edition):

Ou, Y. 1. (2013). Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ou, Yi-Hung 1977-. “Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation.” 2013. Web. 09 Jul 2020.

Vancouver:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ou Y1. Molecular Mechanisms Underlying Innate Immune Kinase TBK1-Driven Oncogenic Transformation. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/ETD-UTSWMED-2013-05-125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

15. Peña, Christopher George. LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression.

Degree: 2015, University of Texas Southwestern Medical Center

 Cancer of the uterus is a common malignancy in women with no adequate treatments for tumors that have progressed beyond the uterus. The serine-threonine kinase… (more)

Subjects/Keywords: Adenocarcinoma; Chemokine CCL2; Endometrial Neoplasms; Macrophages; Neoplasm Proteins; Protein-Serine-Threonine Kinases

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APA (6th Edition):

Peña, C. G. (2015). LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Peña, Christopher George. “LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Peña, Christopher George. “LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression.” 2015. Web. 09 Jul 2020.

Vancouver:

Peña CG. LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4126.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Peña CG. LKB1, CCL2, and Macrophages: A New Axis of Endometrial Cancer Progression. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4126

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

16. Flowers, Ebony Michelle. Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease.

Degree: 2018, University of Texas Southwestern Medical Center

 Polycystic kidney disease is a hereditary disorder characterized by the progressive manifestation of numerous fluid-filled sacs, known as cysts, within the renal epithelia. The enlargement… (more)

Subjects/Keywords: Glutamine; Kidney; Polycystic Kidney Diseases; Protein-Serine-Threonine Kinases; TRPP Cation Channels

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APA (6th Edition):

Flowers, E. M. (2018). Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5750

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Flowers, Ebony Michelle. “Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease.” 2018. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/5750.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Flowers, Ebony Michelle. “Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease.” 2018. Web. 09 Jul 2020.

Vancouver:

Flowers EM. Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2018. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/5750.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Flowers EM. Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease. [Thesis]. University of Texas Southwestern Medical Center; 2018. Available from: http://hdl.handle.net/2152.5/5750

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

17. Ghosh, Anwesha. Studies on Cellular Nutrient Responses and Protein Degradation.

Degree: 2015, University of Texas Southwestern Medical Center

 I have worked on two projects. The first project investigates mechanisms involved in cellular responses to amino acids. Amino-acid abundance promotes protein synthesis and cell… (more)

Subjects/Keywords: Autophagy; Intercellular Signaling Peptides and Proteins; TOR Serine-Threonine Kinases; Ubiquitin-Protein Ligases

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APA (6th Edition):

Ghosh, A. (2015). Studies on Cellular Nutrient Responses and Protein Degradation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4204

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ghosh, Anwesha. “Studies on Cellular Nutrient Responses and Protein Degradation.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4204.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ghosh, Anwesha. “Studies on Cellular Nutrient Responses and Protein Degradation.” 2015. Web. 09 Jul 2020.

Vancouver:

Ghosh A. Studies on Cellular Nutrient Responses and Protein Degradation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4204.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ghosh A. Studies on Cellular Nutrient Responses and Protein Degradation. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4204

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

18. Öncel, Dilhan. Analysis of Aurora B Regulation and Signaling.

Degree: 2006, University of Texas Southwestern Medical Center

 Aurora B is a serine/threonine kinase that functions in a complex with two other chromosomal passenger proteins called INCENP and Survivin. Its function is implicated… (more)

Subjects/Keywords: Mitosis; Protein-Serine-Threonine Kinases; Cytokinesis

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APA (6th Edition):

Öncel, D. (2006). Analysis of Aurora B Regulation and Signaling. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/591

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Öncel, Dilhan. “Analysis of Aurora B Regulation and Signaling.” 2006. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/591.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Öncel, Dilhan. “Analysis of Aurora B Regulation and Signaling.” 2006. Web. 09 Jul 2020.

Vancouver:

Öncel D. Analysis of Aurora B Regulation and Signaling. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2006. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/591.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Öncel D. Analysis of Aurora B Regulation and Signaling. [Thesis]. University of Texas Southwestern Medical Center; 2006. Available from: http://hdl.handle.net/2152.5/591

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

19. Ren, Fangfang. The Hippo Signaling Pathway in Organ Size Control and Regeneraton.

Degree: PhD, Genetics and Development, 2012, University of Texas Southwestern Medical Center

 The Hippo (Hpo) signaling pathway controls cell growth, proliferation and apoptosis in both Drosophila and vertebrates. Our lab has previously demonstrated that Hpo signaling regulates… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; 14-3-3 Proteins; Intracellular Signaling Peptides and Proteins

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APA (6th Edition):

Ren, F. (2012). The Hippo Signaling Pathway in Organ Size Control and Regeneraton. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1005

Chicago Manual of Style (16th Edition):

Ren, Fangfang. “The Hippo Signaling Pathway in Organ Size Control and Regeneraton.” 2012. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1005.

MLA Handbook (7th Edition):

Ren, Fangfang. “The Hippo Signaling Pathway in Organ Size Control and Regeneraton.” 2012. Web. 09 Jul 2020.

Vancouver:

Ren F. The Hippo Signaling Pathway in Organ Size Control and Regeneraton. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1005.

Council of Science Editors:

Ren F. The Hippo Signaling Pathway in Organ Size Control and Regeneraton. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1005


University of Texas Southwestern Medical Center

20. Lee, Byung-Hoon. Identification of Substrates and Pathways Regulated by WNK1.

Degree: PhD, 2004, University of Texas Southwestern Medical Center

 WNK (With No lysine (K)), a serine/threonine protein kinase, is a unique molecule not belonging to any other canonical protein kinase family including mitogen-activated protein(more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Mutagenesis; Hypertension

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APA (6th Edition):

Lee, B. (2004). Identification of Substrates and Pathways Regulated by WNK1. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/599

Chicago Manual of Style (16th Edition):

Lee, Byung-Hoon. “Identification of Substrates and Pathways Regulated by WNK1.” 2004. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/599.

MLA Handbook (7th Edition):

Lee, Byung-Hoon. “Identification of Substrates and Pathways Regulated by WNK1.” 2004. Web. 09 Jul 2020.

Vancouver:

Lee B. Identification of Substrates and Pathways Regulated by WNK1. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2004. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/599.

Council of Science Editors:

Lee B. Identification of Substrates and Pathways Regulated by WNK1. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2004. Available from: http://hdl.handle.net/2152.5/599


University of Texas Southwestern Medical Center

21. Sengupta, Samarpita. A WNK and a Nudge towards Kinase Biology.

Degree: PhD, Cell Regulation, 2013, University of Texas Southwestern Medical Center

 A family of four atypical protein kinases; WNKs are characterized by a non-canonical position of the catalytic lysine. The significance of WNKs was first realized… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Sodium-Potassium-Chloride Symporters; Intracellular Signaling Peptides and Proteins

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APA (6th Edition):

Sengupta, S. (2013). A WNK and a Nudge towards Kinase Biology. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1256

Chicago Manual of Style (16th Edition):

Sengupta, Samarpita. “A WNK and a Nudge towards Kinase Biology.” 2013. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/1256.

MLA Handbook (7th Edition):

Sengupta, Samarpita. “A WNK and a Nudge towards Kinase Biology.” 2013. Web. 09 Jul 2020.

Vancouver:

Sengupta S. A WNK and a Nudge towards Kinase Biology. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/1256.

Council of Science Editors:

Sengupta S. A WNK and a Nudge towards Kinase Biology. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1256


University of Texas Southwestern Medical Center

22. Cooper, Jonathan Mark. Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1).

Degree: 2016, University of Texas Southwestern Medical Center

 Oncogenic mutation of Ras or Ras effector signaling characterizes roughly thirty percent of all cancers. Persistent obstacles to the treatment of these diseases by direct… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; Lung Neoplasms; Mesoderm; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins c-akt; Small Molecule Libraries; TOR Serine-Threonine Kinases

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APA (6th Edition):

Cooper, J. M. (2016). Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1). (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6140

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cooper, Jonathan Mark. “Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1).” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/6140.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cooper, Jonathan Mark. “Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1).” 2016. Web. 09 Jul 2020.

Vancouver:

Cooper JM. Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1). [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/6140.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cooper JM. Context-Selective Support of the AKT/mTOR Regulatory Axis by Tank-Binding Kinase 1 (TBK1). [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6140

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

23. Self, Jon Tate. Interaction Mapping of the Atypical Protein Kinase WNK3.

Degree: PhD, Cell Regulation, 2009, University of Texas Southwestern Medical Center

 The story of the protein kinase "with no lysine 3" (WNK3) represents a unique chapter in the larger story of protein kinases, the so-called 'molecular… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Protein Interaction Mapping; Phosphorylation

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APA (6th Edition):

Self, J. T. (2009). Interaction Mapping of the Atypical Protein Kinase WNK3. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/400

Chicago Manual of Style (16th Edition):

Self, Jon Tate. “Interaction Mapping of the Atypical Protein Kinase WNK3.” 2009. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/400.

MLA Handbook (7th Edition):

Self, Jon Tate. “Interaction Mapping of the Atypical Protein Kinase WNK3.” 2009. Web. 09 Jul 2020.

Vancouver:

Self JT. Interaction Mapping of the Atypical Protein Kinase WNK3. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2009. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/400.

Council of Science Editors:

Self JT. Interaction Mapping of the Atypical Protein Kinase WNK3. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2009. Available from: http://hdl.handle.net/2152.5/400

24. Luis Antonio de Oliveira Alves. ExpressÃo e regulaÃÃo da quinase celular intestinal (ICK) em modelo de desnutriÃÃo in vivo e in vitro .

Degree: PhD, 2014, Universidade Federal do Ceará

 A desnutriÃÃo pode afetar a arquitetura intestinal, causando atrofia da mucosa e comprometendo o turnover epitelial. Embora o intestino seja capaz de responder compensatoriamente Ã… (more)

Subjects/Keywords: BIOQUIMICA DA NUTRICAO; DesnutriÃÃo; ProliferaÃÃo de CÃlulas; Apoptose; ProteÃnas Serina-Treonina Quinases; Malnutrition ; Cell Proliferation; Apoptosis; Protein-Serine-Threonine Kinases

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APA (6th Edition):

Alves, L. A. d. O. (2014). ExpressÃo e regulaÃÃo da quinase celular intestinal (ICK) em modelo de desnutriÃÃo in vivo e in vitro . (Doctoral Dissertation). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12743 ;

Chicago Manual of Style (16th Edition):

Alves, Luis Antonio de Oliveira. “ExpressÃo e regulaÃÃo da quinase celular intestinal (ICK) em modelo de desnutriÃÃo in vivo e in vitro .” 2014. Doctoral Dissertation, Universidade Federal do Ceará. Accessed July 09, 2020. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12743 ;.

MLA Handbook (7th Edition):

Alves, Luis Antonio de Oliveira. “ExpressÃo e regulaÃÃo da quinase celular intestinal (ICK) em modelo de desnutriÃÃo in vivo e in vitro .” 2014. Web. 09 Jul 2020.

Vancouver:

Alves LAdO. ExpressÃo e regulaÃÃo da quinase celular intestinal (ICK) em modelo de desnutriÃÃo in vivo e in vitro . [Internet] [Doctoral dissertation]. Universidade Federal do Ceará 2014. [cited 2020 Jul 09]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12743 ;.

Council of Science Editors:

Alves LAdO. ExpressÃo e regulaÃÃo da quinase celular intestinal (ICK) em modelo de desnutriÃÃo in vivo e in vitro . [Doctoral Dissertation]. Universidade Federal do Ceará 2014. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12743 ;


University of Texas Southwestern Medical Center

25. Jia, Luying. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.

Degree: 2015, University of Texas Southwestern Medical Center

 The spindle checkpoint is an essential mechanism to ensure accurate chromosome segregation during mitosis. The checkpoint signal originates from the kinetochore, which is a huge… (more)

Subjects/Keywords: Anaphase-Promoting Complex-Cyclosome; Cdc20 Proteins; Cell Cycle Proteins; M Phase Cell Cycle Checkpoints; Protein-Serine-Threonine Kinases; Proto-Oncogene Proteins

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APA (6th Edition):

Jia, L. (2015). The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jia, Luying. “The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/4456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jia, Luying. “The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint.” 2015. Web. 09 Jul 2020.

Vancouver:

Jia L. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/4456.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jia L. The Multifunctional Kinase Bub1 Acts as a Signaling Hub for the Spindle Checkpoint. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4456

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Carolina University

26. DeVaul, Nicole. PPP1R42 is a positive regulator of PP1 in centrosome duplication and separation and cilia dynamics.

Degree: PhD, Anatomy and Cell Biology, 2014, East Carolina University

 The centrosome is a dynamic structure that undergoes structural and functional transitions, which are coordinated with the cell cycle. It functions as the microtubule organizing… (more)

Subjects/Keywords: Cellular biology; Aurora A; Centrosome; Cilia; Nek2; PP1; PPP1R42; Neoplasms; Phosphoprotein Phosphatases – physiology; Protein-Serine-Threonine Kinases – physiology; Centrosome – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

DeVaul, N. (2014). PPP1R42 is a positive regulator of PP1 in centrosome duplication and separation and cilia dynamics. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/4670

Chicago Manual of Style (16th Edition):

DeVaul, Nicole. “PPP1R42 is a positive regulator of PP1 in centrosome duplication and separation and cilia dynamics.” 2014. Doctoral Dissertation, East Carolina University. Accessed July 09, 2020. http://hdl.handle.net/10342/4670.

MLA Handbook (7th Edition):

DeVaul, Nicole. “PPP1R42 is a positive regulator of PP1 in centrosome duplication and separation and cilia dynamics.” 2014. Web. 09 Jul 2020.

Vancouver:

DeVaul N. PPP1R42 is a positive regulator of PP1 in centrosome duplication and separation and cilia dynamics. [Internet] [Doctoral dissertation]. East Carolina University; 2014. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/10342/4670.

Council of Science Editors:

DeVaul N. PPP1R42 is a positive regulator of PP1 in centrosome duplication and separation and cilia dynamics. [Doctoral Dissertation]. East Carolina University; 2014. Available from: http://hdl.handle.net/10342/4670


University of Texas Southwestern Medical Center

27. Probst, Brandon Linn. Identification of Substrates and Pathways Regulated by PAS Kinase.

Degree: PhD, Biological Chemistry, 2005, University of Texas Southwestern Medical Center

 PAS kinase, a serine/threonine protein kinase, is unique in that it comprises the only mammalian protein kinase regulated by a PAS domain. The interest of… (more)

Subjects/Keywords: Protein-Serine-Threonine Kinases; Yeast, Baker; Aminosalicylic Acids

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APA (6th Edition):

Probst, B. L. (2005). Identification of Substrates and Pathways Regulated by PAS Kinase. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/296

Chicago Manual of Style (16th Edition):

Probst, Brandon Linn. “Identification of Substrates and Pathways Regulated by PAS Kinase.” 2005. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/296.

MLA Handbook (7th Edition):

Probst, Brandon Linn. “Identification of Substrates and Pathways Regulated by PAS Kinase.” 2005. Web. 09 Jul 2020.

Vancouver:

Probst BL. Identification of Substrates and Pathways Regulated by PAS Kinase. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2005. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/296.

Council of Science Editors:

Probst BL. Identification of Substrates and Pathways Regulated by PAS Kinase. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2005. Available from: http://hdl.handle.net/2152.5/296


University of Texas Southwestern Medical Center

28. Lenertz, Lisa Yvonne. With No Lysine 1 (WNK1): A Potential Regulator of The Lysosomal Degradation Pathway.

Degree: PhD, Cell Regulation, 2007, University of Texas Southwestern Medical Center

 With no Lysine (K) 1 (WNK1) is an atypical serine/threonine protein kinase that has its catalytic lysine positioned in a unique location. This kinase, along… (more)

Subjects/Keywords: Epithelial Cells; Hypertension; Protein-Serine-Threonine Kinases

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APA (6th Edition):

Lenertz, L. Y. (2007). With No Lysine 1 (WNK1): A Potential Regulator of The Lysosomal Degradation Pathway. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/381

Chicago Manual of Style (16th Edition):

Lenertz, Lisa Yvonne. “With No Lysine 1 (WNK1): A Potential Regulator of The Lysosomal Degradation Pathway.” 2007. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/381.

MLA Handbook (7th Edition):

Lenertz, Lisa Yvonne. “With No Lysine 1 (WNK1): A Potential Regulator of The Lysosomal Degradation Pathway.” 2007. Web. 09 Jul 2020.

Vancouver:

Lenertz LY. With No Lysine 1 (WNK1): A Potential Regulator of The Lysosomal Degradation Pathway. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2007. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/381.

Council of Science Editors:

Lenertz LY. With No Lysine 1 (WNK1): A Potential Regulator of The Lysosomal Degradation Pathway. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2007. Available from: http://hdl.handle.net/2152.5/381


University of Texas Southwestern Medical Center

29. Qi, Wei. Regulation and Mechanism of Bub1-Mediated Spindle Checkpoint Signaling.

Degree: PhD, Cell Regulation, 2006, University of Texas Southwestern Medical Center

 The spindle checkpoint is a surveillance mechanism that ensures the fidelity of chromosome segregation during mitosis and meiosis. Bub1 is a highly conserved protein serine/threonine(more)

Subjects/Keywords: Kinetochores; Cell Cycle Proteins; Protein-Serine-Threonine Kinases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Qi, W. (2006). Regulation and Mechanism of Bub1-Mediated Spindle Checkpoint Signaling. (Doctoral Dissertation). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/350

Chicago Manual of Style (16th Edition):

Qi, Wei. “Regulation and Mechanism of Bub1-Mediated Spindle Checkpoint Signaling.” 2006. Doctoral Dissertation, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/350.

MLA Handbook (7th Edition):

Qi, Wei. “Regulation and Mechanism of Bub1-Mediated Spindle Checkpoint Signaling.” 2006. Web. 09 Jul 2020.

Vancouver:

Qi W. Regulation and Mechanism of Bub1-Mediated Spindle Checkpoint Signaling. [Internet] [Doctoral dissertation]. University of Texas Southwestern Medical Center; 2006. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/350.

Council of Science Editors:

Qi W. Regulation and Mechanism of Bub1-Mediated Spindle Checkpoint Signaling. [Doctoral Dissertation]. University of Texas Southwestern Medical Center; 2006. Available from: http://hdl.handle.net/2152.5/350


University of Texas Southwestern Medical Center

30. Ji, Zhejian. Safeguard of Mitosis: The Spindle Checkpoint.

Degree: 2016, University of Texas Southwestern Medical Center

 In mitosis, the kinetochore-microtubule attachment is under surveillance by the spindle checkpoint to ensure the fidelity of chromosome segregation. Defects in the checkpoint could lead… (more)

Subjects/Keywords: Cell Cycle Checkpoints; Cell Cycle Proteins; Microtubule-Associated Proteins; Nuclear Proteins; Protein Processing, Post-Translational; Protein-Serine-Threonine Kinases; Protein-Tyrosine Kinases; Signal Transduction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ji, Z. (2016). Safeguard of Mitosis: The Spindle Checkpoint. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6144

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ji, Zhejian. “Safeguard of Mitosis: The Spindle Checkpoint.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed July 09, 2020. http://hdl.handle.net/2152.5/6144.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ji, Zhejian. “Safeguard of Mitosis: The Spindle Checkpoint.” 2016. Web. 09 Jul 2020.

Vancouver:

Ji Z. Safeguard of Mitosis: The Spindle Checkpoint. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jul 09]. Available from: http://hdl.handle.net/2152.5/6144.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ji Z. Safeguard of Mitosis: The Spindle Checkpoint. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6144

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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