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You searched for subject:(Protein Kinase C). Showing records 1 – 30 of 264 total matches.

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University of Utah

1. Garrone, Nicholas Felix. Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain.

Degree: PhD, Human Genetics, 2010, University of Utah

 A defining feature of NEDD4L protein isoforms is the presence or absence of an amino-terminal C2 domain, a class of subcellular, calcium-dependent targeting domains. We… (more)

Subjects/Keywords: Protein Kinase C

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APA (6th Edition):

Garrone, N. F. (2010). Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1258/rec/518

Chicago Manual of Style (16th Edition):

Garrone, Nicholas Felix. “Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain.” 2010. Doctoral Dissertation, University of Utah. Accessed September 20, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1258/rec/518.

MLA Handbook (7th Edition):

Garrone, Nicholas Felix. “Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain.” 2010. Web. 20 Sep 2020.

Vancouver:

Garrone NF. Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain. [Internet] [Doctoral dissertation]. University of Utah; 2010. [cited 2020 Sep 20]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1258/rec/518.

Council of Science Editors:

Garrone NF. Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain. [Doctoral Dissertation]. University of Utah; 2010. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1258/rec/518

2. Morales-Rivera, Krystal A. The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??.

Degree: 2013, Texas Digital Library

Protein Kinase C (PKC) isoforms function in signaling pathways responsible for controlling cell proliferation, survival and apoptosis. Up or down-regulation of PKCs has been implicated… (more)

Subjects/Keywords: Protein Kinase C

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APA (6th Edition):

Morales-Rivera, K. A. (2013). The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??. (Thesis). Texas Digital Library. Retrieved from http://hdl.handle.net/1969; http://hdl.handle.net/2249.1/66514

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morales-Rivera, Krystal A. “The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??.” 2013. Thesis, Texas Digital Library. Accessed September 20, 2020. http://hdl.handle.net/1969; http://hdl.handle.net/2249.1/66514.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morales-Rivera, Krystal A. “The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??.” 2013. Web. 20 Sep 2020.

Vancouver:

Morales-Rivera KA. The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??. [Internet] [Thesis]. Texas Digital Library; 2013. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1969; http://hdl.handle.net/2249.1/66514.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morales-Rivera KA. The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??. [Thesis]. Texas Digital Library; 2013. Available from: http://hdl.handle.net/1969; http://hdl.handle.net/2249.1/66514

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

3. Moncada de la Rosa,Cesar A. Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling.

Degree: MS, Faculty of Pharmacy and Pharmaceutical Sciences, 2012, University of Alberta

 Blood platelets mediate wound healing by preventing bleeding and by supporting growth of new blood vessels. Blood vessel growth is supported in part by growth… (more)

Subjects/Keywords: Angiogenesis; Protein Kinase C; Platelets

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APA (6th Edition):

A., M. d. l. R. (2012). Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/gx41mh89h

Chicago Manual of Style (16th Edition):

A., Moncada de la Rosa,Cesar. “Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling.” 2012. Masters Thesis, University of Alberta. Accessed September 20, 2020. https://era.library.ualberta.ca/files/gx41mh89h.

MLA Handbook (7th Edition):

A., Moncada de la Rosa,Cesar. “Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling.” 2012. Web. 20 Sep 2020.

Vancouver:

A. MdlR. Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2020 Sep 20]. Available from: https://era.library.ualberta.ca/files/gx41mh89h.

Council of Science Editors:

A. MdlR. Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/gx41mh89h


Hong Kong University of Science and Technology

4. Wang, Chihao. Biochemical and structural characterization of interactions within par complex.

Degree: 2012, Hong Kong University of Science and Technology

 Cell polarity refers to asymmetric organization of cell structure and differential distribution of cellular materials. It is implicated in a variety of processes including early… (more)

Subjects/Keywords: Protein-protein interactions ; Protein kinase C ; Protein kinases

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APA (6th Edition):

Wang, C. (2012). Biochemical and structural characterization of interactions within par complex. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-7817 ; https://doi.org/10.14711/thesis-b1207876 ; http://repository.ust.hk/ir/bitstream/1783.1-7817/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Chihao. “Biochemical and structural characterization of interactions within par complex.” 2012. Thesis, Hong Kong University of Science and Technology. Accessed September 20, 2020. http://repository.ust.hk/ir/Record/1783.1-7817 ; https://doi.org/10.14711/thesis-b1207876 ; http://repository.ust.hk/ir/bitstream/1783.1-7817/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Chihao. “Biochemical and structural characterization of interactions within par complex.” 2012. Web. 20 Sep 2020.

Vancouver:

Wang C. Biochemical and structural characterization of interactions within par complex. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2012. [cited 2020 Sep 20]. Available from: http://repository.ust.hk/ir/Record/1783.1-7817 ; https://doi.org/10.14711/thesis-b1207876 ; http://repository.ust.hk/ir/bitstream/1783.1-7817/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang C. Biochemical and structural characterization of interactions within par complex. [Thesis]. Hong Kong University of Science and Technology; 2012. Available from: http://repository.ust.hk/ir/Record/1783.1-7817 ; https://doi.org/10.14711/thesis-b1207876 ; http://repository.ust.hk/ir/bitstream/1783.1-7817/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

5. Nunthakungwan, Orathai. Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion.

Degree: MS;, Health Promotion & Education;, 2008, University of Utah

 Previous studies have found oxidative stress and PKC activation with the presence of cardiac hypertrophy in diabetic animal models and diabetic patients. Mice with cardiac… (more)

Subjects/Keywords: Diabetes; Protein kinase C; Heart; Mice

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APA (6th Edition):

Nunthakungwan, O. (2008). Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1734/rec/777

Chicago Manual of Style (16th Edition):

Nunthakungwan, Orathai. “Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion.” 2008. Masters Thesis, University of Utah. Accessed September 20, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1734/rec/777.

MLA Handbook (7th Edition):

Nunthakungwan, Orathai. “Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion.” 2008. Web. 20 Sep 2020.

Vancouver:

Nunthakungwan O. Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion. [Internet] [Masters thesis]. University of Utah; 2008. [cited 2020 Sep 20]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1734/rec/777.

Council of Science Editors:

Nunthakungwan O. Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion. [Masters Thesis]. University of Utah; 2008. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1734/rec/777


Boston University

6. Gowda, Asha. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.

Degree: MS, Medical Sciences, 2014, Boston University

 PURPOSE: Uveal melanoma (UM) is the most common intraocular malignancy in adults with an incidence of six per one million individuals each year. Globe conserving… (more)

Subjects/Keywords: Ophthalmology; Beta-catenin; Melanoma; Protein kinase C

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APA (6th Edition):

Gowda, A. (2014). The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/15046

Chicago Manual of Style (16th Edition):

Gowda, Asha. “The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.” 2014. Masters Thesis, Boston University. Accessed September 20, 2020. http://hdl.handle.net/2144/15046.

MLA Handbook (7th Edition):

Gowda, Asha. “The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.” 2014. Web. 20 Sep 2020.

Vancouver:

Gowda A. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. [Internet] [Masters thesis]. Boston University; 2014. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/2144/15046.

Council of Science Editors:

Gowda A. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. [Masters Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/15046


Virginia Tech

7. Ojani, Reyhaneh. Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway.

Degree: PhD, Biochemistry, 2016, Virginia Tech

 Juvenile hormone (JH) is an important insect hormone that controls diverse biological processes in postembryonic development and adult reproduction. JH exerts its effects through the… (more)

Subjects/Keywords: Juvenile hormone; Protein kinase C; Gene regulation

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APA (6th Edition):

Ojani, R. (2016). Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/80342

Chicago Manual of Style (16th Edition):

Ojani, Reyhaneh. “Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway.” 2016. Doctoral Dissertation, Virginia Tech. Accessed September 20, 2020. http://hdl.handle.net/10919/80342.

MLA Handbook (7th Edition):

Ojani, Reyhaneh. “Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway.” 2016. Web. 20 Sep 2020.

Vancouver:

Ojani R. Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway. [Internet] [Doctoral dissertation]. Virginia Tech; 2016. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/10919/80342.

Council of Science Editors:

Ojani R. Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway. [Doctoral Dissertation]. Virginia Tech; 2016. Available from: http://hdl.handle.net/10919/80342


Brandeis University

8. Gelles-Watnick, Sara. Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs.

Degree: 2017, Brandeis University

 Tyrosine kinase c-Src is a ubiquitously expressed proto-oncogene implicated in many cancers. The Kern Lab hopes to use NMR to study the regulatory and catalytic… (more)

Subjects/Keywords: src tyrosine kinase; kinase; protein nmr; Sortase; c-Src

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APA (6th Edition):

Gelles-Watnick, S. (2017). Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs. (Thesis). Brandeis University. Retrieved from http://hdl.handle.net/10192/33910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gelles-Watnick, Sara. “Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs.” 2017. Thesis, Brandeis University. Accessed September 20, 2020. http://hdl.handle.net/10192/33910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gelles-Watnick, Sara. “Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs.” 2017. Web. 20 Sep 2020.

Vancouver:

Gelles-Watnick S. Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs. [Internet] [Thesis]. Brandeis University; 2017. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/10192/33910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gelles-Watnick S. Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs. [Thesis]. Brandeis University; 2017. Available from: http://hdl.handle.net/10192/33910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Emiliano Ricardo Vasconcelos Rios. Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo.

Degree: PhD, 2014, Universidade Federal do Ceará

Esse trabalho, atà onde se sabe, mostra pela primeira vez o efeito antinociceptivo do acetato de citronelila (CAT) e teve como objetivo caracterizar o perfil… (more)

Subjects/Keywords: FARMACOLOGIA; Nociceptividade; ProteÃna Quinase C; Nociception; Protein Kinase C

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APA (6th Edition):

Rios, E. R. V. (2014). Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo. (Doctoral Dissertation). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12064 ;

Chicago Manual of Style (16th Edition):

Rios, Emiliano Ricardo Vasconcelos. “Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo.” 2014. Doctoral Dissertation, Universidade Federal do Ceará. Accessed September 20, 2020. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12064 ;.

MLA Handbook (7th Edition):

Rios, Emiliano Ricardo Vasconcelos. “Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo.” 2014. Web. 20 Sep 2020.

Vancouver:

Rios ERV. Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo. [Internet] [Doctoral dissertation]. Universidade Federal do Ceará 2014. [cited 2020 Sep 20]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12064 ;.

Council of Science Editors:

Rios ERV. Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo. [Doctoral Dissertation]. Universidade Federal do Ceará 2014. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12064 ;


University of Vermont

10. Linden, Anne K. PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling.

Degree: Neuroscience, 2017, University of Vermont

  Pituitary adenylate-cyclase activating polypeptide (PACAP) is highly conserved signaling molecule in eukaryotes known to regulate a myriad of metabolic processes within the brain as… (more)

Subjects/Keywords: PACAP; VPAC receptors; Extracellular signal-dependent kinase (ERK); Receptor endocytosis; Protein Kinase A (PKA); Protein Kinase C (PKC)

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APA (6th Edition):

Linden, A. K. (2017). PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/castheses/45

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Linden, Anne K. “PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling.” 2017. Thesis, University of Vermont. Accessed September 20, 2020. https://scholarworks.uvm.edu/castheses/45.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Linden, Anne K. “PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling.” 2017. Web. 20 Sep 2020.

Vancouver:

Linden AK. PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling. [Internet] [Thesis]. University of Vermont; 2017. [cited 2020 Sep 20]. Available from: https://scholarworks.uvm.edu/castheses/45.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Linden AK. PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling. [Thesis]. University of Vermont; 2017. Available from: https://scholarworks.uvm.edu/castheses/45

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

11. Negro, Christopher Michael. Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers.

Degree: MS, Cell Biology, Neurobiology and Anatomy, 2011, Loyola University Chicago

  Nucleotide excision repair (NER) is the process by which cells identify and repair bulky, helix-distorting DNA lesions such as ultraviolet (UV) radiation-induced cyclobutane pyrimidine… (more)

Subjects/Keywords: Cyclobutane Pyrimidine Dimer; DNA Damage; DNA Repair; Nucleotide Excision Repair; Protein Kinase C; Protein Kinase C Delta; Medical Sciences

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APA (6th Edition):

Negro, C. M. (2011). Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Negro, Christopher Michael. “Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers.” 2011. Thesis, Loyola University Chicago. Accessed September 20, 2020. https://ecommons.luc.edu/luc_theses/550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Negro, Christopher Michael. “Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers.” 2011. Web. 20 Sep 2020.

Vancouver:

Negro CM. Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers. [Internet] [Thesis]. Loyola University Chicago; 2011. [cited 2020 Sep 20]. Available from: https://ecommons.luc.edu/luc_theses/550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Negro CM. Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers. [Thesis]. Loyola University Chicago; 2011. Available from: https://ecommons.luc.edu/luc_theses/550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Michigan

12. Swanson, Carter. Protein Kinase C ?: System of Modular Domains.

Degree: PhD, Biophysics, 2016, University of Michigan

 Signaling proteins comprised of modular domains have evolved along with multi-cellularity as a method to facilitate increasing intra-cellular bandwidth. The effects of intra-molecular interactions between… (more)

Subjects/Keywords: protein self-assembly; intramolecular protein interactions; protein kinase C (PKC); Biological Chemistry; Science

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APA (6th Edition):

Swanson, C. (2016). Protein Kinase C ?: System of Modular Domains. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/135936

Chicago Manual of Style (16th Edition):

Swanson, Carter. “Protein Kinase C ?: System of Modular Domains.” 2016. Doctoral Dissertation, University of Michigan. Accessed September 20, 2020. http://hdl.handle.net/2027.42/135936.

MLA Handbook (7th Edition):

Swanson, Carter. “Protein Kinase C ?: System of Modular Domains.” 2016. Web. 20 Sep 2020.

Vancouver:

Swanson C. Protein Kinase C ?: System of Modular Domains. [Internet] [Doctoral dissertation]. University of Michigan; 2016. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/2027.42/135936.

Council of Science Editors:

Swanson C. Protein Kinase C ?: System of Modular Domains. [Doctoral Dissertation]. University of Michigan; 2016. Available from: http://hdl.handle.net/2027.42/135936


University of Toledo Health Science Campus

13. Elnakat, Hala. Regulation of Folate Receptor Raft Recycling.

Degree: PhD, College of Graduate Studies, 2007, University of Toledo Health Science Campus

 FR quantitatively recycles, within minutes, between the cell surface and endocytic compartments via a Cdc42-regulated endocytic pathway. In this study, we investigated the molecular mechanism… (more)

Subjects/Keywords: Folate Receptor; Raft; Protein Kinase C; Annexin; Phorbol Ester; Recycling

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APA (6th Edition):

Elnakat, H. (2007). Regulation of Folate Receptor Raft Recycling. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1174569209

Chicago Manual of Style (16th Edition):

Elnakat, Hala. “Regulation of Folate Receptor Raft Recycling.” 2007. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed September 20, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1174569209.

MLA Handbook (7th Edition):

Elnakat, Hala. “Regulation of Folate Receptor Raft Recycling.” 2007. Web. 20 Sep 2020.

Vancouver:

Elnakat H. Regulation of Folate Receptor Raft Recycling. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2007. [cited 2020 Sep 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1174569209.

Council of Science Editors:

Elnakat H. Regulation of Folate Receptor Raft Recycling. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1174569209


University of Alberta

14. Aroonassala Patten, Shunmoogum. Mechanism underlying the maturation of AMPA receptors in zebrafish.

Degree: PhD, Department of Biological Sciences, 2009, University of Alberta

 Glutamate AMPA receptors (AMPARs) are major excitatory receptors in the vertebrate CNS. In many biological systems there are changes in the properties of AMPARs during… (more)

Subjects/Keywords: Zebrafish; AMPA receptor; Synaptic development; Mauthner neuron; Protein kinase C

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APA (6th Edition):

Aroonassala Patten, S. (2009). Mechanism underlying the maturation of AMPA receptors in zebrafish. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/3t945s03p

Chicago Manual of Style (16th Edition):

Aroonassala Patten, Shunmoogum. “Mechanism underlying the maturation of AMPA receptors in zebrafish.” 2009. Doctoral Dissertation, University of Alberta. Accessed September 20, 2020. https://era.library.ualberta.ca/files/3t945s03p.

MLA Handbook (7th Edition):

Aroonassala Patten, Shunmoogum. “Mechanism underlying the maturation of AMPA receptors in zebrafish.” 2009. Web. 20 Sep 2020.

Vancouver:

Aroonassala Patten S. Mechanism underlying the maturation of AMPA receptors in zebrafish. [Internet] [Doctoral dissertation]. University of Alberta; 2009. [cited 2020 Sep 20]. Available from: https://era.library.ualberta.ca/files/3t945s03p.

Council of Science Editors:

Aroonassala Patten S. Mechanism underlying the maturation of AMPA receptors in zebrafish. [Doctoral Dissertation]. University of Alberta; 2009. Available from: https://era.library.ualberta.ca/files/3t945s03p


University of Pennsylvania

15. Abera, Mahlet. Pkc Isozymes in Lung Cancer Development and Therapy Resistance.

Degree: 2014, University of Pennsylvania

 ABSTRACT PKC ISOZYMES IN LUNG CANCER DEVELOPMENT AND THERAPY RESISTANCE Mahlet B. Abera Marcelo G. Kazanietz, Ph.D. Non-small cell lung cancer (NSCLC) is one of… (more)

Subjects/Keywords: Drug resistance; Inflammation; Lung Cancer; Protein Kinase C; Tumorigenesis; Oncology; Pharmacology

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APA (6th Edition):

Abera, M. (2014). Pkc Isozymes in Lung Cancer Development and Therapy Resistance. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1185

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Abera, Mahlet. “Pkc Isozymes in Lung Cancer Development and Therapy Resistance.” 2014. Thesis, University of Pennsylvania. Accessed September 20, 2020. https://repository.upenn.edu/edissertations/1185.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Abera, Mahlet. “Pkc Isozymes in Lung Cancer Development and Therapy Resistance.” 2014. Web. 20 Sep 2020.

Vancouver:

Abera M. Pkc Isozymes in Lung Cancer Development and Therapy Resistance. [Internet] [Thesis]. University of Pennsylvania; 2014. [cited 2020 Sep 20]. Available from: https://repository.upenn.edu/edissertations/1185.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Abera M. Pkc Isozymes in Lung Cancer Development and Therapy Resistance. [Thesis]. University of Pennsylvania; 2014. Available from: https://repository.upenn.edu/edissertations/1185

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

16. Oestreich, Emily A. A Novel phospholipase Ce-dependent mechanism for B-adrenergic receptor signaling in the heart.

Degree: PhD, 2009, University of Rochester

 Agonist regulation of intracellular Ca2+ release and protein kinase C activation through stimulation of phospholipase C (PLC) enzymes modulates a variety of physiological responses in… (more)

Subjects/Keywords: Phospholipase Ce; CAMR11; Epac; Rapl; Protein kinase C; B-adrenergic receptor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Oestreich, E. A. (2009). A Novel phospholipase Ce-dependent mechanism for B-adrenergic receptor signaling in the heart. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/6528

Chicago Manual of Style (16th Edition):

Oestreich, Emily A. “A Novel phospholipase Ce-dependent mechanism for B-adrenergic receptor signaling in the heart.” 2009. Doctoral Dissertation, University of Rochester. Accessed September 20, 2020. http://hdl.handle.net/1802/6528.

MLA Handbook (7th Edition):

Oestreich, Emily A. “A Novel phospholipase Ce-dependent mechanism for B-adrenergic receptor signaling in the heart.” 2009. Web. 20 Sep 2020.

Vancouver:

Oestreich EA. A Novel phospholipase Ce-dependent mechanism for B-adrenergic receptor signaling in the heart. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1802/6528.

Council of Science Editors:

Oestreich EA. A Novel phospholipase Ce-dependent mechanism for B-adrenergic receptor signaling in the heart. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/6528


University of New South Wales

17. Pedersen , David. The role of protein kinase C ε in insulin receptor trafficking and insulin action.

Degree: Clinical School - St Vincent's Hospital, 2012, University of New South Wales

 The development of type 2 diabetes is reaching epidemic proportions and identifying ways to modulate insulin levels in order to maintain euglycaemia is important in… (more)

Subjects/Keywords: Insulin Receptor; Protein Kinase C epsilon; Ceacam1; Trafficking; Signalling

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APA (6th Edition):

Pedersen , D. (2012). The role of protein kinase C ε in insulin receptor trafficking and insulin action. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51788 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10455/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Pedersen , David. “The role of protein kinase C ε in insulin receptor trafficking and insulin action.” 2012. Doctoral Dissertation, University of New South Wales. Accessed September 20, 2020. http://handle.unsw.edu.au/1959.4/51788 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10455/SOURCE02?view=true.

MLA Handbook (7th Edition):

Pedersen , David. “The role of protein kinase C ε in insulin receptor trafficking and insulin action.” 2012. Web. 20 Sep 2020.

Vancouver:

Pedersen D. The role of protein kinase C ε in insulin receptor trafficking and insulin action. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2020 Sep 20]. Available from: http://handle.unsw.edu.au/1959.4/51788 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10455/SOURCE02?view=true.

Council of Science Editors:

Pedersen D. The role of protein kinase C ε in insulin receptor trafficking and insulin action. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/51788 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10455/SOURCE02?view=true


Kent State University

18. Wickley, Peter J. Propofol-Anesthesia, Diabetes and Myocardial Signal Transduction: Role of Protein Kinase C and Nitric Oxide.

Degree: PhD, College of Arts and Sciences / School of Biomedical Sciences, 2008, Kent State University

  Induction and maintenance of anesthesia typically causes myocardial depression in healthy patients presenting for cardiac surgery. Moreover, the diabetic patient presents the anesthesiologist with… (more)

Subjects/Keywords: Biomedical Research; anesthesia; diabetes; cardiac; protein kinase C; nitric oxide

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APA (6th Edition):

Wickley, P. J. (2008). Propofol-Anesthesia, Diabetes and Myocardial Signal Transduction: Role of Protein Kinase C and Nitric Oxide. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1206554412

Chicago Manual of Style (16th Edition):

Wickley, Peter J. “Propofol-Anesthesia, Diabetes and Myocardial Signal Transduction: Role of Protein Kinase C and Nitric Oxide.” 2008. Doctoral Dissertation, Kent State University. Accessed September 20, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=kent1206554412.

MLA Handbook (7th Edition):

Wickley, Peter J. “Propofol-Anesthesia, Diabetes and Myocardial Signal Transduction: Role of Protein Kinase C and Nitric Oxide.” 2008. Web. 20 Sep 2020.

Vancouver:

Wickley PJ. Propofol-Anesthesia, Diabetes and Myocardial Signal Transduction: Role of Protein Kinase C and Nitric Oxide. [Internet] [Doctoral dissertation]. Kent State University; 2008. [cited 2020 Sep 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1206554412.

Council of Science Editors:

Wickley PJ. Propofol-Anesthesia, Diabetes and Myocardial Signal Transduction: Role of Protein Kinase C and Nitric Oxide. [Doctoral Dissertation]. Kent State University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1206554412


University of Illinois – Chicago

19. Molloy, Mary E. Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer.

Degree: 2014, University of Illinois – Chicago

 Breast cancer is the most common female malignancy, affecting 1 in 8 women. Resistance to the antiestrogen tamoxifen (TAM), whether de novo or acquired, is… (more)

Subjects/Keywords: PKC alpha (Protein kinase C); breast cancer; endocrine resistance

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APA (6th Edition):

Molloy, M. E. (2014). Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/18899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Molloy, Mary E. “Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer.” 2014. Thesis, University of Illinois – Chicago. Accessed September 20, 2020. http://hdl.handle.net/10027/18899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Molloy, Mary E. “Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer.” 2014. Web. 20 Sep 2020.

Vancouver:

Molloy ME. Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer. [Internet] [Thesis]. University of Illinois – Chicago; 2014. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/10027/18899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Molloy ME. Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer. [Thesis]. University of Illinois – Chicago; 2014. Available from: http://hdl.handle.net/10027/18899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. 인, 승민. The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa.

Degree: 2013, Ajou University

The aim of this study was to investigate the effects of a 1.8 GHz continuous electromagnetic fields (EMF) on human nasal mucociliary transport and to… (more)

Subjects/Keywords: Electromagnetic fields; Ciliary motility; Mucociliary transport; Protein kinase C; Mobile phone

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

인, . (2013). The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/8634 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000013577

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

인, 승민. “The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa.” 2013. Thesis, Ajou University. Accessed September 20, 2020. http://repository.ajou.ac.kr/handle/201003/8634 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000013577.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

인, 승민. “The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa.” 2013. Web. 20 Sep 2020.

Vancouver:

인 . The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa. [Internet] [Thesis]. Ajou University; 2013. [cited 2020 Sep 20]. Available from: http://repository.ajou.ac.kr/handle/201003/8634 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000013577.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

인 . The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa. [Thesis]. Ajou University; 2013. Available from: http://repository.ajou.ac.kr/handle/201003/8634 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000013577

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

21. Xia, Mengying LIFS. Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis.

Degree: 2017, Hong Kong University of Science and Technology

 Amyotrophic lateral sclerosis (ALS) is the most frequent motor neuron disease, which is characterized by the selective loss of the upper and lower motor neuron… (more)

Subjects/Keywords: Amyotrophic lateral sclerosis ; Superoxide dismutase ; Protein kinase C

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APA (6th Edition):

Xia, M. L. (2017). Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-102116 ; https://doi.org/10.14711/thesis-991012588267103412 ; http://repository.ust.hk/ir/bitstream/1783.1-102116/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xia, Mengying LIFS. “Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis.” 2017. Thesis, Hong Kong University of Science and Technology. Accessed September 20, 2020. http://repository.ust.hk/ir/Record/1783.1-102116 ; https://doi.org/10.14711/thesis-991012588267103412 ; http://repository.ust.hk/ir/bitstream/1783.1-102116/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xia, Mengying LIFS. “Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis.” 2017. Web. 20 Sep 2020.

Vancouver:

Xia ML. Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2017. [cited 2020 Sep 20]. Available from: http://repository.ust.hk/ir/Record/1783.1-102116 ; https://doi.org/10.14711/thesis-991012588267103412 ; http://repository.ust.hk/ir/bitstream/1783.1-102116/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xia ML. Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis. [Thesis]. Hong Kong University of Science and Technology; 2017. Available from: http://repository.ust.hk/ir/Record/1783.1-102116 ; https://doi.org/10.14711/thesis-991012588267103412 ; http://repository.ust.hk/ir/bitstream/1783.1-102116/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

22. Diakanastasis, Barbara. The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism.

Degree: Garvan Institute of Medical Research, 2015, University of New South Wales

 Insulin resistance contributes strongly to Type 2 Diabetes, which is a global epidemic. A strong link exists between dietary lipid excess and the development of… (more)

Subjects/Keywords: Lipid oversupply; Ceramide synthase; Protein kinase c epsilon

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APA (6th Edition):

Diakanastasis, B. (2015). The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/54711 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:35619/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Diakanastasis, Barbara. “The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism.” 2015. Masters Thesis, University of New South Wales. Accessed September 20, 2020. http://handle.unsw.edu.au/1959.4/54711 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:35619/SOURCE02?view=true.

MLA Handbook (7th Edition):

Diakanastasis, Barbara. “The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism.” 2015. Web. 20 Sep 2020.

Vancouver:

Diakanastasis B. The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism. [Internet] [Masters thesis]. University of New South Wales; 2015. [cited 2020 Sep 20]. Available from: http://handle.unsw.edu.au/1959.4/54711 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:35619/SOURCE02?view=true.

Council of Science Editors:

Diakanastasis B. The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism. [Masters Thesis]. University of New South Wales; 2015. Available from: http://handle.unsw.edu.au/1959.4/54711 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:35619/SOURCE02?view=true


Georgia State University

23. Mathew, Meril. Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina.

Degree: PhD, Biology, 2016, Georgia State University

  Larvae of the marine bryozoan, Bugula neritina, are defended from predation by the bryostatins, polyketides synthesized by its uncultured endosymbiont, “Candidatus Endobugula sertula.” Bryostatins… (more)

Subjects/Keywords: Symbiosis; Bryostatins; Protein kinase C; Host reproduction; Differential gene expression

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APA (6th Edition):

Mathew, M. (2016). Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/182

Chicago Manual of Style (16th Edition):

Mathew, Meril. “Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina.” 2016. Doctoral Dissertation, Georgia State University. Accessed September 20, 2020. https://scholarworks.gsu.edu/biology_diss/182.

MLA Handbook (7th Edition):

Mathew, Meril. “Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina.” 2016. Web. 20 Sep 2020.

Vancouver:

Mathew M. Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina. [Internet] [Doctoral dissertation]. Georgia State University; 2016. [cited 2020 Sep 20]. Available from: https://scholarworks.gsu.edu/biology_diss/182.

Council of Science Editors:

Mathew M. Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina. [Doctoral Dissertation]. Georgia State University; 2016. Available from: https://scholarworks.gsu.edu/biology_diss/182


Iowa State University

24. Martin, Dustin Paul. Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease.

Degree: 2013, Iowa State University

 Despite several decades of dedicated research by a diverse array of researchers from around the globe, the molecular mechanisms underlying neuronal loss during transmissible spongiform… (more)

Subjects/Keywords: Mahogunin; Manganese; Neurodegeneration; Prion; Protein Kinase C Delta; Neuroscience and Neurobiology

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APA (6th Edition):

Martin, D. P. (2013). Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/13388

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martin, Dustin Paul. “Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease.” 2013. Thesis, Iowa State University. Accessed September 20, 2020. https://lib.dr.iastate.edu/etd/13388.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martin, Dustin Paul. “Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease.” 2013. Web. 20 Sep 2020.

Vancouver:

Martin DP. Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease. [Internet] [Thesis]. Iowa State University; 2013. [cited 2020 Sep 20]. Available from: https://lib.dr.iastate.edu/etd/13388.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martin DP. Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease. [Thesis]. Iowa State University; 2013. Available from: https://lib.dr.iastate.edu/etd/13388

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oregon

25. Bailey, Matthew. Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization.

Degree: PhD, Department of Chemistry and Biochemistry, 2017, University of Oregon

 Animal cells, as distinct as epithelia and migratory cells, have cell polarity that is defined by a common set of molecules. The Par complex polarizes… (more)

Subjects/Keywords: Asymmetric cell division; Atypical Protein Kinase C; Cell polarity; Par complex

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APA (6th Edition):

Bailey, M. (2017). Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization. (Doctoral Dissertation). University of Oregon. Retrieved from http://hdl.handle.net/1794/22782

Chicago Manual of Style (16th Edition):

Bailey, Matthew. “Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization.” 2017. Doctoral Dissertation, University of Oregon. Accessed September 20, 2020. http://hdl.handle.net/1794/22782.

MLA Handbook (7th Edition):

Bailey, Matthew. “Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization.” 2017. Web. 20 Sep 2020.

Vancouver:

Bailey M. Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization. [Internet] [Doctoral dissertation]. University of Oregon; 2017. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1794/22782.

Council of Science Editors:

Bailey M. Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization. [Doctoral Dissertation]. University of Oregon; 2017. Available from: http://hdl.handle.net/1794/22782

26. Ferreira, Julio Cesar Batista. Participação da isoforma proteína quinase C βII na insuficiência cardíaca.

Degree: PhD, Biodinâmica do Movimento Humano, 2009, University of São Paulo

A insuficiência cardíaca é uma síndrome clínica de mau prognóstico caracterizada por disfunção cardíaca associada à intolerância aos esforços, retenção de fluído e redução da… (more)

Subjects/Keywords: heart failure; Insuficiência cardíaca; pharmacological therapy; protein kinase C; proteina quinase C; Terapia farmacológica

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APA (6th Edition):

Ferreira, J. C. B. (2009). Participação da isoforma proteína quinase C βII na insuficiência cardíaca. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/39/39132/tde-08092010-090610/ ;

Chicago Manual of Style (16th Edition):

Ferreira, Julio Cesar Batista. “Participação da isoforma proteína quinase C βII na insuficiência cardíaca.” 2009. Doctoral Dissertation, University of São Paulo. Accessed September 20, 2020. http://www.teses.usp.br/teses/disponiveis/39/39132/tde-08092010-090610/ ;.

MLA Handbook (7th Edition):

Ferreira, Julio Cesar Batista. “Participação da isoforma proteína quinase C βII na insuficiência cardíaca.” 2009. Web. 20 Sep 2020.

Vancouver:

Ferreira JCB. Participação da isoforma proteína quinase C βII na insuficiência cardíaca. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2020 Sep 20]. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-08092010-090610/ ;.

Council of Science Editors:

Ferreira JCB. Participação da isoforma proteína quinase C βII na insuficiência cardíaca. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-08092010-090610/ ;


University of California – San Diego

27. Tobias, Irene. Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 Atypical protein kinase C (aPKC) isozymes are key modulators of insulin signaling, and their dysfunction correlates with insulin-resistant states in both mice and humans. Despite… (more)

Subjects/Keywords: Biochemistry; Pharmacology; insulin signaling; IRS-1; p62; Par6; Protein Kinase C; protein scaffolds

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APA (6th Edition):

Tobias, I. (2016). Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/5wn6d7qv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tobias, Irene. “Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling.” 2016. Thesis, University of California – San Diego. Accessed September 20, 2020. http://www.escholarship.org/uc/item/5wn6d7qv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tobias, Irene. “Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling.” 2016. Web. 20 Sep 2020.

Vancouver:

Tobias I. Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2020 Sep 20]. Available from: http://www.escholarship.org/uc/item/5wn6d7qv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tobias I. Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/5wn6d7qv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

28. Morales-Rivera, Krystal A. The Metal-dependent Function of C2α: A Conditional Membrane Domain from Protein Kinase.

Degree: PhD, Biochemistry, 2013, Texas A&M University

Protein Kinase C (PKC) isoforms function in signaling pathways responsible for controlling cell proliferation, survival and apoptosis. Up or down-regulation of PKCs has been implicated… (more)

Subjects/Keywords: Protein Kinase C; C2 domain; peripheral membrane protein; Nuclear Magnetic Resonance; Calcium; Lead poisoning

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Morales-Rivera, K. A. (2013). The Metal-dependent Function of C2α: A Conditional Membrane Domain from Protein Kinase Cα. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151621

Chicago Manual of Style (16th Edition):

Morales-Rivera, Krystal A. “The Metal-dependent Function of C2α: A Conditional Membrane Domain from Protein Kinase Cα.” 2013. Doctoral Dissertation, Texas A&M University. Accessed September 20, 2020. http://hdl.handle.net/1969.1/151621.

MLA Handbook (7th Edition):

Morales-Rivera, Krystal A. “The Metal-dependent Function of C2α: A Conditional Membrane Domain from Protein Kinase Cα.” 2013. Web. 20 Sep 2020.

Vancouver:

Morales-Rivera KA. The Metal-dependent Function of C2α: A Conditional Membrane Domain from Protein Kinase Cα. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2020 Sep 20]. Available from: http://hdl.handle.net/1969.1/151621.

Council of Science Editors:

Morales-Rivera KA. The Metal-dependent Function of C2α: A Conditional Membrane Domain from Protein Kinase Cα. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151621


University of Cincinnati

29. Sherrill, Joseph D. Functional Analysis of the Murine Cytomegalovirus G Protein-coupled Receptor M33.

Degree: PhD, Medicine : Molecular Genetics, Biochemistry, and Microbiology, 2008, University of Cincinnati

 Several members of the herpesvirus family encode seven transmembrane-spanning proteins that share significant homology with the cellular G protein-coupled receptors (GPCRs). These viral GPCR homologs… (more)

Subjects/Keywords: Biochemistry; Microbiology; Molecular Biology; Virology; G protein; GPCR; cytomegalovirus; MCMV; M33; CREB; protein kinase C

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sherrill, J. D. (2008). Functional Analysis of the Murine Cytomegalovirus G Protein-coupled Receptor M33. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1225745444

Chicago Manual of Style (16th Edition):

Sherrill, Joseph D. “Functional Analysis of the Murine Cytomegalovirus G Protein-coupled Receptor M33.” 2008. Doctoral Dissertation, University of Cincinnati. Accessed September 20, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1225745444.

MLA Handbook (7th Edition):

Sherrill, Joseph D. “Functional Analysis of the Murine Cytomegalovirus G Protein-coupled Receptor M33.” 2008. Web. 20 Sep 2020.

Vancouver:

Sherrill JD. Functional Analysis of the Murine Cytomegalovirus G Protein-coupled Receptor M33. [Internet] [Doctoral dissertation]. University of Cincinnati; 2008. [cited 2020 Sep 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1225745444.

Council of Science Editors:

Sherrill JD. Functional Analysis of the Murine Cytomegalovirus G Protein-coupled Receptor M33. [Doctoral Dissertation]. University of Cincinnati; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1225745444


Universiteit Utrecht

30. Herczenik, E. The biology of non-native proteins.

Degree: 2007, Universiteit Utrecht

Protein misfolding diseases are linked by common principles of protein aggregation, plaque development and tissue damage. There is no adequate therapy for these highly debilitating… (more)

Subjects/Keywords: Geneeskunde; protein misfolding; platelet activation; platelet receptors; amyloid; aggregation; intravenous immunoglobulins; C-reactive protein; protein kinase inhibitors; cytotoxicity; tumor therapy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Herczenik, E. (2007). The biology of non-native proteins. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/24176

Chicago Manual of Style (16th Edition):

Herczenik, E. “The biology of non-native proteins.” 2007. Doctoral Dissertation, Universiteit Utrecht. Accessed September 20, 2020. http://dspace.library.uu.nl:8080/handle/1874/24176.

MLA Handbook (7th Edition):

Herczenik, E. “The biology of non-native proteins.” 2007. Web. 20 Sep 2020.

Vancouver:

Herczenik E. The biology of non-native proteins. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2007. [cited 2020 Sep 20]. Available from: http://dspace.library.uu.nl:8080/handle/1874/24176.

Council of Science Editors:

Herczenik E. The biology of non-native proteins. [Doctoral Dissertation]. Universiteit Utrecht; 2007. Available from: http://dspace.library.uu.nl:8080/handle/1874/24176

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