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You searched for subject:(Protein Kinase C mesh ). Showing records 1 – 30 of 33426 total matches.

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University of Utah

1. Garrone, Nicholas Felix. Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain.

Degree: PhD, Human Genetics, 2010, University of Utah

 A defining feature of NEDD4L protein isoforms is the presence or absence of an amino-terminal C2 domain, a class of subcellular, calcium-dependent targeting domains. We… (more)

Subjects/Keywords: Protein Kinase C

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APA (6th Edition):

Garrone, N. F. (2010). Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1258/rec/518

Chicago Manual of Style (16th Edition):

Garrone, Nicholas Felix. “Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain.” 2010. Doctoral Dissertation, University of Utah. Accessed November 28, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1258/rec/518.

MLA Handbook (7th Edition):

Garrone, Nicholas Felix. “Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain.” 2010. Web. 28 Nov 2020.

Vancouver:

Garrone NF. Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain. [Internet] [Doctoral dissertation]. University of Utah; 2010. [cited 2020 Nov 28]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1258/rec/518.

Council of Science Editors:

Garrone NF. Functional Analysis of Neural Precursor Cell Expressed Developmentally Down Regulated, Gene 4 Isoforms that Contain or Lack a Conserved Region 2 of Protein Kinase C Domain. [Doctoral Dissertation]. University of Utah; 2010. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1258/rec/518

2. Morales-Rivera, Krystal A. The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??.

Degree: 2013, Texas Digital Library

Protein Kinase C (PKC) isoforms function in signaling pathways responsible for controlling cell proliferation, survival and apoptosis. Up or down-regulation of PKCs has been implicated… (more)

Subjects/Keywords: Protein Kinase C

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APA (6th Edition):

Morales-Rivera, K. A. (2013). The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??. (Thesis). Texas Digital Library. Retrieved from http://hdl.handle.net/1969; http://hdl.handle.net/2249.1/66514

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morales-Rivera, Krystal A. “The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??.” 2013. Thesis, Texas Digital Library. Accessed November 28, 2020. http://hdl.handle.net/1969; http://hdl.handle.net/2249.1/66514.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morales-Rivera, Krystal A. “The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??.” 2013. Web. 28 Nov 2020.

Vancouver:

Morales-Rivera KA. The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??. [Internet] [Thesis]. Texas Digital Library; 2013. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969; http://hdl.handle.net/2249.1/66514.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morales-Rivera KA. The Metal-dependent Function of C2??: A Conditional Membrane Domain from Protein Kinase C??. [Thesis]. Texas Digital Library; 2013. Available from: http://hdl.handle.net/1969; http://hdl.handle.net/2249.1/66514

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

3. Moncada de la Rosa,Cesar A. Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling.

Degree: MS, Faculty of Pharmacy and Pharmaceutical Sciences, 2012, University of Alberta

 Blood platelets mediate wound healing by preventing bleeding and by supporting growth of new blood vessels. Blood vessel growth is supported in part by growth… (more)

Subjects/Keywords: Angiogenesis; Protein Kinase C; Platelets

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APA (6th Edition):

A., M. d. l. R. (2012). Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/gx41mh89h

Chicago Manual of Style (16th Edition):

A., Moncada de la Rosa,Cesar. “Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling.” 2012. Masters Thesis, University of Alberta. Accessed November 28, 2020. https://era.library.ualberta.ca/files/gx41mh89h.

MLA Handbook (7th Edition):

A., Moncada de la Rosa,Cesar. “Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling.” 2012. Web. 28 Nov 2020.

Vancouver:

A. MdlR. Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2020 Nov 28]. Available from: https://era.library.ualberta.ca/files/gx41mh89h.

Council of Science Editors:

A. MdlR. Manipulation of the Angiogenic Balance by Pharmacological Inhibition of Platelet PKC Signalling. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/gx41mh89h


Hong Kong University of Science and Technology

4. Wang, Chihao. Biochemical and structural characterization of interactions within par complex.

Degree: 2012, Hong Kong University of Science and Technology

 Cell polarity refers to asymmetric organization of cell structure and differential distribution of cellular materials. It is implicated in a variety of processes including early… (more)

Subjects/Keywords: Protein-protein interactions ; Protein kinase C ; Protein kinases

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APA (6th Edition):

Wang, C. (2012). Biochemical and structural characterization of interactions within par complex. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-7817 ; https://doi.org/10.14711/thesis-b1207876 ; http://repository.ust.hk/ir/bitstream/1783.1-7817/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Chihao. “Biochemical and structural characterization of interactions within par complex.” 2012. Thesis, Hong Kong University of Science and Technology. Accessed November 28, 2020. http://repository.ust.hk/ir/Record/1783.1-7817 ; https://doi.org/10.14711/thesis-b1207876 ; http://repository.ust.hk/ir/bitstream/1783.1-7817/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Chihao. “Biochemical and structural characterization of interactions within par complex.” 2012. Web. 28 Nov 2020.

Vancouver:

Wang C. Biochemical and structural characterization of interactions within par complex. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2012. [cited 2020 Nov 28]. Available from: http://repository.ust.hk/ir/Record/1783.1-7817 ; https://doi.org/10.14711/thesis-b1207876 ; http://repository.ust.hk/ir/bitstream/1783.1-7817/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang C. Biochemical and structural characterization of interactions within par complex. [Thesis]. Hong Kong University of Science and Technology; 2012. Available from: http://repository.ust.hk/ir/Record/1783.1-7817 ; https://doi.org/10.14711/thesis-b1207876 ; http://repository.ust.hk/ir/bitstream/1783.1-7817/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

5. Nunthakungwan, Orathai. Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion.

Degree: MS;, Health Promotion & Education;, 2008, University of Utah

 Previous studies have found oxidative stress and PKC activation with the presence of cardiac hypertrophy in diabetic animal models and diabetic patients. Mice with cardiac… (more)

Subjects/Keywords: Diabetes; Protein kinase C; Heart; Mice

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APA (6th Edition):

Nunthakungwan, O. (2008). Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1734/rec/777

Chicago Manual of Style (16th Edition):

Nunthakungwan, Orathai. “Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion.” 2008. Masters Thesis, University of Utah. Accessed November 28, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1734/rec/777.

MLA Handbook (7th Edition):

Nunthakungwan, Orathai. “Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion.” 2008. Web. 28 Nov 2020.

Vancouver:

Nunthakungwan O. Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion. [Internet] [Masters thesis]. University of Utah; 2008. [cited 2020 Nov 28]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1734/rec/777.

Council of Science Editors:

Nunthakungwan O. Mitochondrial oxidative stress and protein kinase C are not the regulators of cardiac hypertrophy in mice and cardiac specific glucose transporter 4 deletion. [Masters Thesis]. University of Utah; 2008. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1734/rec/777


Boston University

6. Gowda, Asha. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.

Degree: MS, Medical Sciences, 2014, Boston University

 PURPOSE: Uveal melanoma (UM) is the most common intraocular malignancy in adults with an incidence of six per one million individuals each year. Globe conserving… (more)

Subjects/Keywords: Ophthalmology; Beta-catenin; Melanoma; Protein kinase C

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APA (6th Edition):

Gowda, A. (2014). The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/15046

Chicago Manual of Style (16th Edition):

Gowda, Asha. “The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.” 2014. Masters Thesis, Boston University. Accessed November 28, 2020. http://hdl.handle.net/2144/15046.

MLA Handbook (7th Edition):

Gowda, Asha. “The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells.” 2014. Web. 28 Nov 2020.

Vancouver:

Gowda A. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. [Internet] [Masters thesis]. Boston University; 2014. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2144/15046.

Council of Science Editors:

Gowda A. The effect of protein kinase C and Beta-catenin inhibitors on uveal melanoma cells. [Masters Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/15046


Virginia Tech

7. Ojani, Reyhaneh. Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway.

Degree: PhD, Biochemistry, 2016, Virginia Tech

 Juvenile hormone (JH) is an important insect hormone that controls diverse biological processes in postembryonic development and adult reproduction. JH exerts its effects through the… (more)

Subjects/Keywords: Juvenile hormone; Protein kinase C; Gene regulation

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APA (6th Edition):

Ojani, R. (2016). Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/80342

Chicago Manual of Style (16th Edition):

Ojani, Reyhaneh. “Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway.” 2016. Doctoral Dissertation, Virginia Tech. Accessed November 28, 2020. http://hdl.handle.net/10919/80342.

MLA Handbook (7th Edition):

Ojani, Reyhaneh. “Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway.” 2016. Web. 28 Nov 2020.

Vancouver:

Ojani R. Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway. [Internet] [Doctoral dissertation]. Virginia Tech; 2016. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/10919/80342.

Council of Science Editors:

Ojani R. Molecular mechanisms underlying Juvenile hormone (JH) signaling pathway. [Doctoral Dissertation]. Virginia Tech; 2016. Available from: http://hdl.handle.net/10919/80342


University of Florida

8. Martin, Eileen Joan, 1967-. The Role of second messenger systems in the cytoprotective actions of NGF using an in vitro model of neurodegeneration.

Degree: 1994, University of Florida

Subjects/Keywords: Research ( mesh ); Second Messenger Systems ( mesh ); Nerve Growth Factors ( mesh ); Cytoprotection ( mesh ); Models, Biological ( mesh ); Cell Death ( mesh ); Protein Kinase C ( mesh ); Cyclic AMP-Dependent Protein Kinases ( mesh ); Department of Pharmacology and Therapeutics thesis Ph.D ( mesh )

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APA (6th Edition):

Martin, Eileen Joan, 1. (1994). The Role of second messenger systems in the cytoprotective actions of NGF using an in vitro model of neurodegeneration. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00067569

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martin, Eileen Joan, 1967-. “The Role of second messenger systems in the cytoprotective actions of NGF using an in vitro model of neurodegeneration.” 1994. Thesis, University of Florida. Accessed November 28, 2020. https://ufdc.ufl.edu/AA00067569.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martin, Eileen Joan, 1967-. “The Role of second messenger systems in the cytoprotective actions of NGF using an in vitro model of neurodegeneration.” 1994. Web. 28 Nov 2020.

Vancouver:

Martin, Eileen Joan 1. The Role of second messenger systems in the cytoprotective actions of NGF using an in vitro model of neurodegeneration. [Internet] [Thesis]. University of Florida; 1994. [cited 2020 Nov 28]. Available from: https://ufdc.ufl.edu/AA00067569.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martin, Eileen Joan 1. The Role of second messenger systems in the cytoprotective actions of NGF using an in vitro model of neurodegeneration. [Thesis]. University of Florida; 1994. Available from: https://ufdc.ufl.edu/AA00067569

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

9. Noe, Elena Ariane. Charakterisierung der Protein Kinase C Alpha Defizienz im Mausmodell.

Degree: 2016, Freie Universität Berlin

 Die Proteinkinase C (PKC)-Familie wurde erstmals 1977 von Nishizuka und Kollegen beschrieben und bereits vor mehr als 30 Jahren mit verschiedenen Krankheitsbildern assoziiert. Seitdem ist… (more)

Subjects/Keywords: Mice; animal models; protein kinase C (MeSH); pulmonary artery; hypertension; blood pressure; vasoconstriction; 600 Technik, Medizin, angewandte Wissenschaften::630 Landwirtschaft::630 Landwirtschaft und verwandte Bereiche

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APA (6th Edition):

Noe, E. A. (2016). Charakterisierung der Protein Kinase C Alpha Defizienz im Mausmodell. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-9265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Noe, Elena Ariane. “Charakterisierung der Protein Kinase C Alpha Defizienz im Mausmodell.” 2016. Thesis, Freie Universität Berlin. Accessed November 28, 2020. http://dx.doi.org/10.17169/refubium-9265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Noe, Elena Ariane. “Charakterisierung der Protein Kinase C Alpha Defizienz im Mausmodell.” 2016. Web. 28 Nov 2020.

Vancouver:

Noe EA. Charakterisierung der Protein Kinase C Alpha Defizienz im Mausmodell. [Internet] [Thesis]. Freie Universität Berlin; 2016. [cited 2020 Nov 28]. Available from: http://dx.doi.org/10.17169/refubium-9265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Noe EA. Charakterisierung der Protein Kinase C Alpha Defizienz im Mausmodell. [Thesis]. Freie Universität Berlin; 2016. Available from: http://dx.doi.org/10.17169/refubium-9265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Brandeis University

10. Gelles-Watnick, Sara. Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs.

Degree: 2017, Brandeis University

 Tyrosine kinase c-Src is a ubiquitously expressed proto-oncogene implicated in many cancers. The Kern Lab hopes to use NMR to study the regulatory and catalytic… (more)

Subjects/Keywords: src tyrosine kinase; kinase; protein nmr; Sortase; c-Src

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APA (6th Edition):

Gelles-Watnick, S. (2017). Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs. (Thesis). Brandeis University. Retrieved from http://hdl.handle.net/10192/33910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gelles-Watnick, Sara. “Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs.” 2017. Thesis, Brandeis University. Accessed November 28, 2020. http://hdl.handle.net/10192/33910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gelles-Watnick, Sara. “Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs.” 2017. Web. 28 Nov 2020.

Vancouver:

Gelles-Watnick S. Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs. [Internet] [Thesis]. Brandeis University; 2017. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/10192/33910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gelles-Watnick S. Progress towards mechanistic studies of Src tyrosine kinase using ligated protein constructs. [Thesis]. Brandeis University; 2017. Available from: http://hdl.handle.net/10192/33910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Emiliano Ricardo Vasconcelos Rios. Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo.

Degree: PhD, 2014, Universidade Federal do Ceará

Esse trabalho, atà onde se sabe, mostra pela primeira vez o efeito antinociceptivo do acetato de citronelila (CAT) e teve como objetivo caracterizar o perfil… (more)

Subjects/Keywords: FARMACOLOGIA; Nociceptividade; ProteÃna Quinase C; Nociception; Protein Kinase C

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APA (6th Edition):

Rios, E. R. V. (2014). Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo. (Doctoral Dissertation). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12064 ;

Chicago Manual of Style (16th Edition):

Rios, Emiliano Ricardo Vasconcelos. “Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo.” 2014. Doctoral Dissertation, Universidade Federal do Ceará. Accessed November 28, 2020. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12064 ;.

MLA Handbook (7th Edition):

Rios, Emiliano Ricardo Vasconcelos. “Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo.” 2014. Web. 28 Nov 2020.

Vancouver:

Rios ERV. Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo. [Internet] [Doctoral dissertation]. Universidade Federal do Ceará 2014. [cited 2020 Nov 28]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12064 ;.

Council of Science Editors:

Rios ERV. Efeito antinociceptivo do Acetato de Citronelila: estudo dos possÃveis mecanismos de aÃÃo. [Doctoral Dissertation]. Universidade Federal do Ceará 2014. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=12064 ;


University of Florida

12. Mudd, Laura Mary, 1958-. Regulation of insulin effector systems in the brain.

Degree: 1989, University of Florida

Subjects/Keywords: Antibodies; Brain; Cells; Cultured cells; Insulin; Neuroglia; Neurons; Phorbol esters; Rats; Receptors; Brain  – enzymology ( mesh ); Physiology thesis Ph.D ( mesh ); Protein Kinase C ( mesh ); Receptor, Insulin ( mesh )

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APA (6th Edition):

Mudd, Laura Mary, 1. (1989). Regulation of insulin effector systems in the brain. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00011810

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mudd, Laura Mary, 1958-. “Regulation of insulin effector systems in the brain.” 1989. Thesis, University of Florida. Accessed November 28, 2020. https://ufdc.ufl.edu/AA00011810.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mudd, Laura Mary, 1958-. “Regulation of insulin effector systems in the brain.” 1989. Web. 28 Nov 2020.

Vancouver:

Mudd, Laura Mary 1. Regulation of insulin effector systems in the brain. [Internet] [Thesis]. University of Florida; 1989. [cited 2020 Nov 28]. Available from: https://ufdc.ufl.edu/AA00011810.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mudd, Laura Mary 1. Regulation of insulin effector systems in the brain. [Thesis]. University of Florida; 1989. Available from: https://ufdc.ufl.edu/AA00011810

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

13. Zöllinger, Angela Maria. The relevance of the Akt kinase for the viability of tumor cells and as a therapeutic target in multiple myeloma.

Degree: 2008, Freie Universität Berlin

 Summary: The PI3K/Akt pathway has been reported to critically contribute to survival and malignant growth of multiple myeloma (MM). Because most of these data are… (more)

Subjects/Keywords: Myeloma, Multiple Myeloma (MeSH), therapy, protein-kinase, Proto-Oncogene Proteins c-akt (MeSH), Protein Kinase Inhibitors (MeSH), RNA; RNA interference, RNA, Small interfering (MeSH); 600 Technik, Medizin, angewandte Wissenschaften::630 Landwirtschaft

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APA (6th Edition):

Zöllinger, A. M. (2008). The relevance of the Akt kinase for the viability of tumor cells and as a therapeutic target in multiple myeloma. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-6161

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zöllinger, Angela Maria. “The relevance of the Akt kinase for the viability of tumor cells and as a therapeutic target in multiple myeloma.” 2008. Thesis, Freie Universität Berlin. Accessed November 28, 2020. http://dx.doi.org/10.17169/refubium-6161.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zöllinger, Angela Maria. “The relevance of the Akt kinase for the viability of tumor cells and as a therapeutic target in multiple myeloma.” 2008. Web. 28 Nov 2020.

Vancouver:

Zöllinger AM. The relevance of the Akt kinase for the viability of tumor cells and as a therapeutic target in multiple myeloma. [Internet] [Thesis]. Freie Universität Berlin; 2008. [cited 2020 Nov 28]. Available from: http://dx.doi.org/10.17169/refubium-6161.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zöllinger AM. The relevance of the Akt kinase for the viability of tumor cells and as a therapeutic target in multiple myeloma. [Thesis]. Freie Universität Berlin; 2008. Available from: http://dx.doi.org/10.17169/refubium-6161

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

14. Linden, Anne K. PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling.

Degree: Neuroscience, 2017, University of Vermont

  Pituitary adenylate-cyclase activating polypeptide (PACAP) is highly conserved signaling molecule in eukaryotes known to regulate a myriad of metabolic processes within the brain as… (more)

Subjects/Keywords: PACAP; VPAC receptors; Extracellular signal-dependent kinase (ERK); Receptor endocytosis; Protein Kinase A (PKA); Protein Kinase C (PKC)

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APA (6th Edition):

Linden, A. K. (2017). PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/castheses/45

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Linden, Anne K. “PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling.” 2017. Thesis, University of Vermont. Accessed November 28, 2020. https://scholarworks.uvm.edu/castheses/45.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Linden, Anne K. “PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling.” 2017. Web. 28 Nov 2020.

Vancouver:

Linden AK. PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling. [Internet] [Thesis]. University of Vermont; 2017. [cited 2020 Nov 28]. Available from: https://scholarworks.uvm.edu/castheses/45.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Linden AK. PACAP-induced ERK phosphorylation in VPAC1 and VPAC2-expressing HEK cells is mediated by receptor endocytosis and PKC signaling. [Thesis]. University of Vermont; 2017. Available from: https://scholarworks.uvm.edu/castheses/45

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

15. Negro, Christopher Michael. Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers.

Degree: MS, Cell Biology, Neurobiology and Anatomy, 2011, Loyola University Chicago

  Nucleotide excision repair (NER) is the process by which cells identify and repair bulky, helix-distorting DNA lesions such as ultraviolet (UV) radiation-induced cyclobutane pyrimidine… (more)

Subjects/Keywords: Cyclobutane Pyrimidine Dimer; DNA Damage; DNA Repair; Nucleotide Excision Repair; Protein Kinase C; Protein Kinase C Delta; Medical Sciences

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APA (6th Edition):

Negro, C. M. (2011). Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Negro, Christopher Michael. “Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers.” 2011. Thesis, Loyola University Chicago. Accessed November 28, 2020. https://ecommons.luc.edu/luc_theses/550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Negro, Christopher Michael. “Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers.” 2011. Web. 28 Nov 2020.

Vancouver:

Negro CM. Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers. [Internet] [Thesis]. Loyola University Chicago; 2011. [cited 2020 Nov 28]. Available from: https://ecommons.luc.edu/luc_theses/550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Negro CM. Protein Kinase C δ Is Critical for Nucleotide Excision Repair of Cyclobutane Pyrimidine Dimers. [Thesis]. Loyola University Chicago; 2011. Available from: https://ecommons.luc.edu/luc_theses/550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Michigan

16. Swanson, Carter. Protein Kinase C ?: System of Modular Domains.

Degree: PhD, Biophysics, 2016, University of Michigan

 Signaling proteins comprised of modular domains have evolved along with multi-cellularity as a method to facilitate increasing intra-cellular bandwidth. The effects of intra-molecular interactions between… (more)

Subjects/Keywords: protein self-assembly; intramolecular protein interactions; protein kinase C (PKC); Biological Chemistry; Science

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APA (6th Edition):

Swanson, C. (2016). Protein Kinase C ?: System of Modular Domains. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/135936

Chicago Manual of Style (16th Edition):

Swanson, Carter. “Protein Kinase C ?: System of Modular Domains.” 2016. Doctoral Dissertation, University of Michigan. Accessed November 28, 2020. http://hdl.handle.net/2027.42/135936.

MLA Handbook (7th Edition):

Swanson, Carter. “Protein Kinase C ?: System of Modular Domains.” 2016. Web. 28 Nov 2020.

Vancouver:

Swanson C. Protein Kinase C ?: System of Modular Domains. [Internet] [Doctoral dissertation]. University of Michigan; 2016. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/2027.42/135936.

Council of Science Editors:

Swanson C. Protein Kinase C ?: System of Modular Domains. [Doctoral Dissertation]. University of Michigan; 2016. Available from: http://hdl.handle.net/2027.42/135936


University of Florida

17. Paulding, Waltke Reche, 1964-. Regulation of atrial natriuretic peptide receptors in astroglia.

Degree: 1996, University of Florida

Subjects/Keywords: Research ( mesh ); Receptors, Atrial Natriuretic Factor  – physiology ( mesh ); Astrocytes ( mesh ); Cells, Cultured ( mesh ); Protein Kinase C  – physiology ( mesh ); Angiotensin II  – physiology ( mesh ); Rats, Sprague-Dawley ( mesh ); Rats, Inbred SHR ( mesh ); Rats, Inbred WKY ( mesh ); Department of Physiology thesis Ph.D ( mesh )

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APA (6th Edition):

Paulding, Waltke Reche, 1. (1996). Regulation of atrial natriuretic peptide receptors in astroglia. (Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/AA00067432

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Paulding, Waltke Reche, 1964-. “Regulation of atrial natriuretic peptide receptors in astroglia.” 1996. Thesis, University of Florida. Accessed November 28, 2020. https://ufdc.ufl.edu/AA00067432.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Paulding, Waltke Reche, 1964-. “Regulation of atrial natriuretic peptide receptors in astroglia.” 1996. Web. 28 Nov 2020.

Vancouver:

Paulding, Waltke Reche 1. Regulation of atrial natriuretic peptide receptors in astroglia. [Internet] [Thesis]. University of Florida; 1996. [cited 2020 Nov 28]. Available from: https://ufdc.ufl.edu/AA00067432.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Paulding, Waltke Reche 1. Regulation of atrial natriuretic peptide receptors in astroglia. [Thesis]. University of Florida; 1996. Available from: https://ufdc.ufl.edu/AA00067432

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

18. Aroonassala Patten, Shunmoogum. Mechanism underlying the maturation of AMPA receptors in zebrafish.

Degree: PhD, Department of Biological Sciences, 2009, University of Alberta

 Glutamate AMPA receptors (AMPARs) are major excitatory receptors in the vertebrate CNS. In many biological systems there are changes in the properties of AMPARs during… (more)

Subjects/Keywords: Zebrafish; AMPA receptor; Synaptic development; Mauthner neuron; Protein kinase C

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APA (6th Edition):

Aroonassala Patten, S. (2009). Mechanism underlying the maturation of AMPA receptors in zebrafish. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/3t945s03p

Chicago Manual of Style (16th Edition):

Aroonassala Patten, Shunmoogum. “Mechanism underlying the maturation of AMPA receptors in zebrafish.” 2009. Doctoral Dissertation, University of Alberta. Accessed November 28, 2020. https://era.library.ualberta.ca/files/3t945s03p.

MLA Handbook (7th Edition):

Aroonassala Patten, Shunmoogum. “Mechanism underlying the maturation of AMPA receptors in zebrafish.” 2009. Web. 28 Nov 2020.

Vancouver:

Aroonassala Patten S. Mechanism underlying the maturation of AMPA receptors in zebrafish. [Internet] [Doctoral dissertation]. University of Alberta; 2009. [cited 2020 Nov 28]. Available from: https://era.library.ualberta.ca/files/3t945s03p.

Council of Science Editors:

Aroonassala Patten S. Mechanism underlying the maturation of AMPA receptors in zebrafish. [Doctoral Dissertation]. University of Alberta; 2009. Available from: https://era.library.ualberta.ca/files/3t945s03p


University of Illinois – Chicago

19. Molloy, Mary E. Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer.

Degree: 2014, University of Illinois – Chicago

 Breast cancer is the most common female malignancy, affecting 1 in 8 women. Resistance to the antiestrogen tamoxifen (TAM), whether de novo or acquired, is… (more)

Subjects/Keywords: PKC alpha (Protein kinase C); breast cancer; endocrine resistance

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APA (6th Edition):

Molloy, M. E. (2014). Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/18899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Molloy, Mary E. “Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer.” 2014. Thesis, University of Illinois – Chicago. Accessed November 28, 2020. http://hdl.handle.net/10027/18899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Molloy, Mary E. “Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer.” 2014. Web. 28 Nov 2020.

Vancouver:

Molloy ME. Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer. [Internet] [Thesis]. University of Illinois – Chicago; 2014. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/10027/18899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Molloy ME. Selective Estrogen Mimics for the Treatment of Tamoxifen-Resistant, PKCalpha-Overexpressing Breast Cancer. [Thesis]. University of Illinois – Chicago; 2014. Available from: http://hdl.handle.net/10027/18899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. 인, 승민. The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa.

Degree: 2013, Ajou University

The aim of this study was to investigate the effects of a 1.8 GHz continuous electromagnetic fields (EMF) on human nasal mucociliary transport and to… (more)

Subjects/Keywords: Electromagnetic fields; Ciliary motility; Mucociliary transport; Protein kinase C; Mobile phone

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APA (6th Edition):

인, . (2013). The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/8634 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000013577

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

인, 승민. “The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa.” 2013. Thesis, Ajou University. Accessed November 28, 2020. http://repository.ajou.ac.kr/handle/201003/8634 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000013577.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

인, 승민. “The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa.” 2013. Web. 28 Nov 2020.

Vancouver:

인 . The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa. [Internet] [Thesis]. Ajou University; 2013. [cited 2020 Nov 28]. Available from: http://repository.ajou.ac.kr/handle/201003/8634 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000013577.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

인 . The effects of a 1.8 GHz continuous electromagnetic fields on mucociliary transport of human nasal mucosa. [Thesis]. Ajou University; 2013. Available from: http://repository.ajou.ac.kr/handle/201003/8634 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000013577

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

21. Xia, Mengying LIFS. Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis.

Degree: 2017, Hong Kong University of Science and Technology

 Amyotrophic lateral sclerosis (ALS) is the most frequent motor neuron disease, which is characterized by the selective loss of the upper and lower motor neuron… (more)

Subjects/Keywords: Amyotrophic lateral sclerosis ; Superoxide dismutase ; Protein kinase C

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APA (6th Edition):

Xia, M. L. (2017). Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-102116 ; https://doi.org/10.14711/thesis-991012588267103412 ; http://repository.ust.hk/ir/bitstream/1783.1-102116/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xia, Mengying LIFS. “Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis.” 2017. Thesis, Hong Kong University of Science and Technology. Accessed November 28, 2020. http://repository.ust.hk/ir/Record/1783.1-102116 ; https://doi.org/10.14711/thesis-991012588267103412 ; http://repository.ust.hk/ir/bitstream/1783.1-102116/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xia, Mengying LIFS. “Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis.” 2017. Web. 28 Nov 2020.

Vancouver:

Xia ML. Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2017. [cited 2020 Nov 28]. Available from: http://repository.ust.hk/ir/Record/1783.1-102116 ; https://doi.org/10.14711/thesis-991012588267103412 ; http://repository.ust.hk/ir/bitstream/1783.1-102116/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xia ML. Investigating the role of PICK1 in regulating mutant SOD1 in amyotrophic lateral sclerosis. [Thesis]. Hong Kong University of Science and Technology; 2017. Available from: http://repository.ust.hk/ir/Record/1783.1-102116 ; https://doi.org/10.14711/thesis-991012588267103412 ; http://repository.ust.hk/ir/bitstream/1783.1-102116/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Iowa State University

22. Martin, Dustin Paul. Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease.

Degree: 2013, Iowa State University

 Despite several decades of dedicated research by a diverse array of researchers from around the globe, the molecular mechanisms underlying neuronal loss during transmissible spongiform… (more)

Subjects/Keywords: Mahogunin; Manganese; Neurodegeneration; Prion; Protein Kinase C Delta; Neuroscience and Neurobiology

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APA (6th Edition):

Martin, D. P. (2013). Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/13388

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martin, Dustin Paul. “Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease.” 2013. Thesis, Iowa State University. Accessed November 28, 2020. https://lib.dr.iastate.edu/etd/13388.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martin, Dustin Paul. “Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease.” 2013. Web. 28 Nov 2020.

Vancouver:

Martin DP. Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease. [Internet] [Thesis]. Iowa State University; 2013. [cited 2020 Nov 28]. Available from: https://lib.dr.iastate.edu/etd/13388.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martin DP. Roles of manganese and protein kinase signaling in cell culture and animal models of prion disease. [Thesis]. Iowa State University; 2013. Available from: https://lib.dr.iastate.edu/etd/13388

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oregon

23. Bailey, Matthew. Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization.

Degree: PhD, Department of Chemistry and Biochemistry, 2017, University of Oregon

 Animal cells, as distinct as epithelia and migratory cells, have cell polarity that is defined by a common set of molecules. The Par complex polarizes… (more)

Subjects/Keywords: Asymmetric cell division; Atypical Protein Kinase C; Cell polarity; Par complex

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APA (6th Edition):

Bailey, M. (2017). Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization. (Doctoral Dissertation). University of Oregon. Retrieved from http://hdl.handle.net/1794/22782

Chicago Manual of Style (16th Edition):

Bailey, Matthew. “Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization.” 2017. Doctoral Dissertation, University of Oregon. Accessed November 28, 2020. http://hdl.handle.net/1794/22782.

MLA Handbook (7th Edition):

Bailey, Matthew. “Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization.” 2017. Web. 28 Nov 2020.

Vancouver:

Bailey M. Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization. [Internet] [Doctoral dissertation]. University of Oregon; 2017. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1794/22782.

Council of Science Editors:

Bailey M. Deciphering the "Polarity Code": the Mechanism of Par Complex Substrate Polarization. [Doctoral Dissertation]. University of Oregon; 2017. Available from: http://hdl.handle.net/1794/22782


Georgia State University

24. Mathew, Meril. Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina.

Degree: PhD, Biology, 2016, Georgia State University

  Larvae of the marine bryozoan, Bugula neritina, are defended from predation by the bryostatins, polyketides synthesized by its uncultured endosymbiont, “Candidatus Endobugula sertula.” Bryostatins… (more)

Subjects/Keywords: Symbiosis; Bryostatins; Protein kinase C; Host reproduction; Differential gene expression

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APA (6th Edition):

Mathew, M. (2016). Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/182

Chicago Manual of Style (16th Edition):

Mathew, Meril. “Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina.” 2016. Doctoral Dissertation, Georgia State University. Accessed November 28, 2020. https://scholarworks.gsu.edu/biology_diss/182.

MLA Handbook (7th Edition):

Mathew, Meril. “Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina.” 2016. Web. 28 Nov 2020.

Vancouver:

Mathew M. Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina. [Internet] [Doctoral dissertation]. Georgia State University; 2016. [cited 2020 Nov 28]. Available from: https://scholarworks.gsu.edu/biology_diss/182.

Council of Science Editors:

Mathew M. Symbiont Dependent Host Reproduction In The Marine Bryozoan, Bugula neritina. [Doctoral Dissertation]. Georgia State University; 2016. Available from: https://scholarworks.gsu.edu/biology_diss/182


University of New South Wales

25. Diakanastasis, Barbara. The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism.

Degree: Garvan Institute of Medical Research, 2015, University of New South Wales

 Insulin resistance contributes strongly to Type 2 Diabetes, which is a global epidemic. A strong link exists between dietary lipid excess and the development of… (more)

Subjects/Keywords: Lipid oversupply; Ceramide synthase; Protein kinase c epsilon

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APA (6th Edition):

Diakanastasis, B. (2015). The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/54711 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:35619/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Diakanastasis, Barbara. “The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism.” 2015. Masters Thesis, University of New South Wales. Accessed November 28, 2020. http://handle.unsw.edu.au/1959.4/54711 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:35619/SOURCE02?view=true.

MLA Handbook (7th Edition):

Diakanastasis, Barbara. “The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism.” 2015. Web. 28 Nov 2020.

Vancouver:

Diakanastasis B. The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism. [Internet] [Masters thesis]. University of New South Wales; 2015. [cited 2020 Nov 28]. Available from: http://handle.unsw.edu.au/1959.4/54711 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:35619/SOURCE02?view=true.

Council of Science Editors:

Diakanastasis B. The effects of ceramide synthases and protein kinase c epsilon on glucose homeostasis and lipid metabolism. [Masters Thesis]. University of New South Wales; 2015. Available from: http://handle.unsw.edu.au/1959.4/54711 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:35619/SOURCE02?view=true


University of New South Wales

26. Pedersen , David. The role of protein kinase C ε in insulin receptor trafficking and insulin action.

Degree: Clinical School - St Vincent's Hospital, 2012, University of New South Wales

 The development of type 2 diabetes is reaching epidemic proportions and identifying ways to modulate insulin levels in order to maintain euglycaemia is important in… (more)

Subjects/Keywords: Insulin Receptor; Protein Kinase C epsilon; Ceacam1; Trafficking; Signalling

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APA (6th Edition):

Pedersen , D. (2012). The role of protein kinase C ε in insulin receptor trafficking and insulin action. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51788 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10455/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Pedersen , David. “The role of protein kinase C ε in insulin receptor trafficking and insulin action.” 2012. Doctoral Dissertation, University of New South Wales. Accessed November 28, 2020. http://handle.unsw.edu.au/1959.4/51788 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10455/SOURCE02?view=true.

MLA Handbook (7th Edition):

Pedersen , David. “The role of protein kinase C ε in insulin receptor trafficking and insulin action.” 2012. Web. 28 Nov 2020.

Vancouver:

Pedersen D. The role of protein kinase C ε in insulin receptor trafficking and insulin action. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2020 Nov 28]. Available from: http://handle.unsw.edu.au/1959.4/51788 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10455/SOURCE02?view=true.

Council of Science Editors:

Pedersen D. The role of protein kinase C ε in insulin receptor trafficking and insulin action. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/51788 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10455/SOURCE02?view=true


University of Pennsylvania

27. Abera, Mahlet. Pkc Isozymes in Lung Cancer Development and Therapy Resistance.

Degree: 2014, University of Pennsylvania

 ABSTRACT PKC ISOZYMES IN LUNG CANCER DEVELOPMENT AND THERAPY RESISTANCE Mahlet B. Abera Marcelo G. Kazanietz, Ph.D. Non-small cell lung cancer (NSCLC) is one of… (more)

Subjects/Keywords: Drug resistance; Inflammation; Lung Cancer; Protein Kinase C; Tumorigenesis; Oncology; Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Abera, M. (2014). Pkc Isozymes in Lung Cancer Development and Therapy Resistance. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1185

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Abera, Mahlet. “Pkc Isozymes in Lung Cancer Development and Therapy Resistance.” 2014. Thesis, University of Pennsylvania. Accessed November 28, 2020. https://repository.upenn.edu/edissertations/1185.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Abera, Mahlet. “Pkc Isozymes in Lung Cancer Development and Therapy Resistance.” 2014. Web. 28 Nov 2020.

Vancouver:

Abera M. Pkc Isozymes in Lung Cancer Development and Therapy Resistance. [Internet] [Thesis]. University of Pennsylvania; 2014. [cited 2020 Nov 28]. Available from: https://repository.upenn.edu/edissertations/1185.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Abera M. Pkc Isozymes in Lung Cancer Development and Therapy Resistance. [Thesis]. University of Pennsylvania; 2014. Available from: https://repository.upenn.edu/edissertations/1185

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

28. Glasl, Carola. Effect of recombinant human activated protein C on human endothelial cells and endothelial progenitor cells.

Degree: 2010, Freie Universität Berlin

 The endogenous serine protease “activated protein C“ (APC) is a key regulator of blood coagulation and fibrinolysis to maintain hemostasis. In addition to its anticoagulant… (more)

Subjects/Keywords: angiogenesis; sepsis, protein C (MeSH); endothelial cells (MeSH); stem cells (MeSH); vascular endothelial growth gactor C (MeSH); angiopoietin-2 (MeSH); endothelium; man; enzyme activation (MeSH); models; biological (MeSH); umbilical veins (MeSH); 600 Technik, Medizin, angewandte Wissenschaften::630 Landwirtschaft

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Glasl, C. (2010). Effect of recombinant human activated protein C on human endothelial cells and endothelial progenitor cells. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-8012

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Glasl, Carola. “Effect of recombinant human activated protein C on human endothelial cells and endothelial progenitor cells.” 2010. Thesis, Freie Universität Berlin. Accessed November 28, 2020. http://dx.doi.org/10.17169/refubium-8012.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Glasl, Carola. “Effect of recombinant human activated protein C on human endothelial cells and endothelial progenitor cells.” 2010. Web. 28 Nov 2020.

Vancouver:

Glasl C. Effect of recombinant human activated protein C on human endothelial cells and endothelial progenitor cells. [Internet] [Thesis]. Freie Universität Berlin; 2010. [cited 2020 Nov 28]. Available from: http://dx.doi.org/10.17169/refubium-8012.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Glasl C. Effect of recombinant human activated protein C on human endothelial cells and endothelial progenitor cells. [Thesis]. Freie Universität Berlin; 2010. Available from: http://dx.doi.org/10.17169/refubium-8012

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Ferreira, Julio Cesar Batista. Participação da isoforma proteína quinase C βII na insuficiência cardíaca.

Degree: PhD, Biodinâmica do Movimento Humano, 2009, University of São Paulo

A insuficiência cardíaca é uma síndrome clínica de mau prognóstico caracterizada por disfunção cardíaca associada à intolerância aos esforços, retenção de fluído e redução da… (more)

Subjects/Keywords: heart failure; Insuficiência cardíaca; pharmacological therapy; protein kinase C; proteina quinase C; Terapia farmacológica

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ferreira, J. C. B. (2009). Participação da isoforma proteína quinase C βII na insuficiência cardíaca. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/39/39132/tde-08092010-090610/ ;

Chicago Manual of Style (16th Edition):

Ferreira, Julio Cesar Batista. “Participação da isoforma proteína quinase C βII na insuficiência cardíaca.” 2009. Doctoral Dissertation, University of São Paulo. Accessed November 28, 2020. http://www.teses.usp.br/teses/disponiveis/39/39132/tde-08092010-090610/ ;.

MLA Handbook (7th Edition):

Ferreira, Julio Cesar Batista. “Participação da isoforma proteína quinase C βII na insuficiência cardíaca.” 2009. Web. 28 Nov 2020.

Vancouver:

Ferreira JCB. Participação da isoforma proteína quinase C βII na insuficiência cardíaca. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2020 Nov 28]. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-08092010-090610/ ;.

Council of Science Editors:

Ferreira JCB. Participação da isoforma proteína quinase C βII na insuficiência cardíaca. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-08092010-090610/ ;


University of California – San Diego

30. Tobias, Irene. Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 Atypical protein kinase C (aPKC) isozymes are key modulators of insulin signaling, and their dysfunction correlates with insulin-resistant states in both mice and humans. Despite… (more)

Subjects/Keywords: Biochemistry; Pharmacology; insulin signaling; IRS-1; p62; Par6; Protein Kinase C; protein scaffolds

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tobias, I. (2016). Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/5wn6d7qv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tobias, Irene. “Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling.” 2016. Thesis, University of California – San Diego. Accessed November 28, 2020. http://www.escholarship.org/uc/item/5wn6d7qv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tobias, Irene. “Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling.” 2016. Web. 28 Nov 2020.

Vancouver:

Tobias I. Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2020 Nov 28]. Available from: http://www.escholarship.org/uc/item/5wn6d7qv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tobias I. Molecular Mechanisms of Atypical Protein Kinase C Regulation in Insulin Signaling. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/5wn6d7qv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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