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You searched for subject:(Polycystic kidney diseases). Showing records 1 – 7 of 7 total matches.

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1. Nauta, Jeroen. Pathophysiology of Polycystic Kidney Disease: Experimental studies.

Degree: Department of Pediatrics, 2000, Erasmus University Medical Center

Subjects/Keywords: kidney diseases; polycystic kidneys; urology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nauta, J. (2000). Pathophysiology of Polycystic Kidney Disease: Experimental studies. (Doctoral Dissertation). Erasmus University Medical Center. Retrieved from http://hdl.handle.net/1765/21145

Chicago Manual of Style (16th Edition):

Nauta, Jeroen. “Pathophysiology of Polycystic Kidney Disease: Experimental studies.” 2000. Doctoral Dissertation, Erasmus University Medical Center. Accessed December 02, 2020. http://hdl.handle.net/1765/21145.

MLA Handbook (7th Edition):

Nauta, Jeroen. “Pathophysiology of Polycystic Kidney Disease: Experimental studies.” 2000. Web. 02 Dec 2020.

Vancouver:

Nauta J. Pathophysiology of Polycystic Kidney Disease: Experimental studies. [Internet] [Doctoral dissertation]. Erasmus University Medical Center; 2000. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/1765/21145.

Council of Science Editors:

Nauta J. Pathophysiology of Polycystic Kidney Disease: Experimental studies. [Doctoral Dissertation]. Erasmus University Medical Center; 2000. Available from: http://hdl.handle.net/1765/21145


University of Texas Southwestern Medical Center

2. Flowers, Ebony Michelle. Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease.

Degree: 2018, University of Texas Southwestern Medical Center

Polycystic kidney disease is a hereditary disorder characterized by the progressive manifestation of numerous fluid-filled sacs, known as cysts, within the renal epithelia. The enlargement… (more)

Subjects/Keywords: Glutamine; Kidney; Polycystic Kidney Diseases; Protein-Serine-Threonine Kinases; TRPP Cation Channels

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APA (6th Edition):

Flowers, E. M. (2018). Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5750

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Flowers, Ebony Michelle. “Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease.” 2018. Thesis, University of Texas Southwestern Medical Center. Accessed December 02, 2020. http://hdl.handle.net/2152.5/5750.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Flowers, Ebony Michelle. “Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease.” 2018. Web. 02 Dec 2020.

Vancouver:

Flowers EM. Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2018. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/2152.5/5750.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Flowers EM. Targeting Glutamine Metabolism in Kidney Development and Polycystic Kidney Disease. [Thesis]. University of Texas Southwestern Medical Center; 2018. Available from: http://hdl.handle.net/2152.5/5750

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Hovater, Michael (Michael Brian). Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease.

Degree: MS, 2006, University of Alabama – Birmingham

This thesis concerns purinergic signaling in renal epithelial cells of normal and polycystic kidneys. The first section discusses first principles of “purinergic signaling” as they… (more)

Subjects/Keywords: Epithelial Cells  – physiology <; br>; Kidney Tubules  – physiology <; br>; Polycystic Kidney Diseases  – pathology <; br>; Receptors, Purinergic P2  – metabolism <; br>; Signal Transduction  – physiology

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APA (6th Edition):

Hovater, M. (. B. (2006). Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,30

Chicago Manual of Style (16th Edition):

Hovater, Michael (Michael Brian). “Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease.” 2006. Masters Thesis, University of Alabama – Birmingham. Accessed December 02, 2020. http://contentdm.mhsl.uab.edu/u?/etd,30.

MLA Handbook (7th Edition):

Hovater, Michael (Michael Brian). “Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease.” 2006. Web. 02 Dec 2020.

Vancouver:

Hovater M(B. Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2006. [cited 2020 Dec 02]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,30.

Council of Science Editors:

Hovater M(B. Underlying purinergic signaling important for monocilium-dependent signaling in ductal epithelia: implications for polycystic kidney disease. [Masters Thesis]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,30


University of Texas Southwestern Medical Center

4. Choi, Yun-Hee. Understanding HNF-1β through Identification of Interacting Proteins and Target Genes in the Kidney.

Degree: 2010, University of Texas Southwestern Medical Center

 Hepatocyte nuclear factor-1Beta (HNF-1Beta) is a POU/homeodomain-containing transcription factor that regulates tissue-specific gene expression in the liver, kidney, pancreas, and other epithelial organs. During kidney(more)

Subjects/Keywords: Cyclic AMP Receptor Protein; Polycystic Kidney Diseases; Hepatocyte Nuclear Factor 1-beta

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APA (6th Edition):

Choi, Y. (2010). Understanding HNF-1β through Identification of Interacting Proteins and Target Genes in the Kidney. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Choi, Yun-Hee. “Understanding HNF-1β through Identification of Interacting Proteins and Target Genes in the Kidney.” 2010. Thesis, University of Texas Southwestern Medical Center. Accessed December 02, 2020. http://hdl.handle.net/2152.5/803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Choi, Yun-Hee. “Understanding HNF-1β through Identification of Interacting Proteins and Target Genes in the Kidney.” 2010. Web. 02 Dec 2020.

Vancouver:

Choi Y. Understanding HNF-1β through Identification of Interacting Proteins and Target Genes in the Kidney. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2010. [cited 2020 Dec 02]. Available from: http://hdl.handle.net/2152.5/803.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Choi Y. Understanding HNF-1β through Identification of Interacting Proteins and Target Genes in the Kidney. [Thesis]. University of Texas Southwestern Medical Center; 2010. Available from: http://hdl.handle.net/2152.5/803

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Silva, Crysthiane Saveriano Rubião. Apoptose precoce, proliferação celular sincrônica tardia e perfil de expressão de proteínas ao complexo esclerose tuberosa e às doenças renais policísticas durante tubulogênese in vitro.

Degree: Mestrado, Fisiopatologia Experimental, 2013, University of São Paulo

O complexo esclerose tuberosa (CET) e as doenças renais policísticas autossômica dominante (DRPAD) e autossômica recessiva (DRPAR) são doenças monogênicas associadas a cistogênese renal. Os… (more)

Subjects/Keywords: Apoptose; Apoptose; Biologia do desenvolvimento; Cell proliferation; Cells cultured; Células cultivadas; Células epiteliais; Developmental biology; Doenças renais policísticas; Epithelial cells; Esclerose tuberosa; Kidney tubules; Polycystic kidney diseases; Proliferação celular; Tuberous sclerosis; Túbulos renais

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APA (6th Edition):

Silva, C. S. R. (2013). Apoptose precoce, proliferação celular sincrônica tardia e perfil de expressão de proteínas ao complexo esclerose tuberosa e às doenças renais policísticas durante tubulogênese in vitro. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5160/tde-01082013-145925/ ;

Chicago Manual of Style (16th Edition):

Silva, Crysthiane Saveriano Rubião. “Apoptose precoce, proliferação celular sincrônica tardia e perfil de expressão de proteínas ao complexo esclerose tuberosa e às doenças renais policísticas durante tubulogênese in vitro.” 2013. Masters Thesis, University of São Paulo. Accessed December 02, 2020. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-01082013-145925/ ;.

MLA Handbook (7th Edition):

Silva, Crysthiane Saveriano Rubião. “Apoptose precoce, proliferação celular sincrônica tardia e perfil de expressão de proteínas ao complexo esclerose tuberosa e às doenças renais policísticas durante tubulogênese in vitro.” 2013. Web. 02 Dec 2020.

Vancouver:

Silva CSR. Apoptose precoce, proliferação celular sincrônica tardia e perfil de expressão de proteínas ao complexo esclerose tuberosa e às doenças renais policísticas durante tubulogênese in vitro. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2020 Dec 02]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-01082013-145925/ ;.

Council of Science Editors:

Silva CSR. Apoptose precoce, proliferação celular sincrônica tardia e perfil de expressão de proteínas ao complexo esclerose tuberosa e às doenças renais policísticas durante tubulogênese in vitro. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-01082013-145925/ ;

6. Bastos, Ana Paula Almeida. A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos.

Degree: PhD, Nefrologia, 2010, University of São Paulo

A maior parte dos casos de doença renal policística autossômica dominante (DRPAD) é causada por mutações no gene PKD1 (Polycystic Kidney Disease 1). O insulto… (more)

Subjects/Keywords: Autosomal dominant polycystic kidney disease; Cell proliferation; Cyclin-dependent kinase Inhibitor p21; Cystic kidney diseases; Doenças renais císticas; Inibidor de quinase dependente de ciclina p21; Ischemia; Isquemia; Mutação; Mutation; Proliferação de células; Reperfusion injury; Rim policístico autossômico dominante; Traumatismo por reperfusão

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APA (6th Edition):

Bastos, A. P. A. (2010). A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5148/tde-25082010-112042/ ;

Chicago Manual of Style (16th Edition):

Bastos, Ana Paula Almeida. “A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos.” 2010. Doctoral Dissertation, University of São Paulo. Accessed December 02, 2020. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-25082010-112042/ ;.

MLA Handbook (7th Edition):

Bastos, Ana Paula Almeida. “A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos.” 2010. Web. 02 Dec 2020.

Vancouver:

Bastos APA. A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos. [Internet] [Doctoral dissertation]. University of São Paulo; 2010. [cited 2020 Dec 02]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-25082010-112042/ ;.

Council of Science Editors:

Bastos APA. A haploinsuficiência de Pkd1 aumenta a lesão renal e induz formação de microcistos após isquemia/reperfusão em camundongos. [Doctoral Dissertation]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/5/5148/tde-25082010-112042/ ;

7. Fonseca, Jonathan Mackowiak da. O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1.

Degree: Mestrado, Fisiopatologia Experimental, 2012, University of São Paulo

O desenvolvimento de hipertensão arterial (HAS) ocorre dez anos mais cedo em pacientes com doença renal policística autossômica dominante (DRPAD) comparados à população geral, estando… (more)

Subjects/Keywords: Autossomal dominant polycystic kidney disease; Capacidade de concentração renal; Cystic kidney diseases; Doenças renais císticas; Hipertensão; Hypertension; Mutação; Mutation; Nitric oxide; Óxido nítrico; Renal concentration deficit; Renin-angiotensin system; Rim policístico autossômico dominante; Sistema renina-angiotensina

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fonseca, J. M. d. (2012). O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5160/tde-22012013-170944/ ;

Chicago Manual of Style (16th Edition):

Fonseca, Jonathan Mackowiak da. “O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1.” 2012. Masters Thesis, University of São Paulo. Accessed December 02, 2020. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-22012013-170944/ ;.

MLA Handbook (7th Edition):

Fonseca, Jonathan Mackowiak da. “O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1.” 2012. Web. 02 Dec 2020.

Vancouver:

Fonseca JMd. O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2020 Dec 02]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-22012013-170944/ ;.

Council of Science Editors:

Fonseca JMd. O crescimento cístico renal é o principal determinante para o desenvolvimento de hipertensão e déficit de concentração em camundongos com deficiência do gene Pkd1. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-22012013-170944/ ;

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