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You searched for subject:(Podocyte). Showing records 1 – 30 of 65 total matches.

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University of Gothenburg / Göteborgs Universitet

1. Bergwall, Lovisa. A Podocyte view on RhoGTPases and actin cytoskeleton regulation.

Degree: 2020, University of Gothenburg / Göteborgs Universitet

 Proteinuria is a hallmark symptom of chronic kidney disease, that if left to persist constitutes a risk for progression of disease. Symptomatic treatment aiming at… (more)

Subjects/Keywords: Podocyte; RhoGTPases; Actin cytoskeleton regulation

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APA (6th Edition):

Bergwall, L. (2020). A Podocyte view on RhoGTPases and actin cytoskeleton regulation. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/64541

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bergwall, Lovisa. “A Podocyte view on RhoGTPases and actin cytoskeleton regulation.” 2020. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed February 27, 2021. http://hdl.handle.net/2077/64541.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bergwall, Lovisa. “A Podocyte view on RhoGTPases and actin cytoskeleton regulation.” 2020. Web. 27 Feb 2021.

Vancouver:

Bergwall L. A Podocyte view on RhoGTPases and actin cytoskeleton regulation. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2020. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/2077/64541.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bergwall L. A Podocyte view on RhoGTPases and actin cytoskeleton regulation. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2020. Available from: http://hdl.handle.net/2077/64541

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Yamazaki, Mihoko. Possible role for glomerular-derived angiotensinogen in nephrotic syndrome. : ネフローゼ症候群における糸球体由来アンジオテンシノーゲンの役割.

Degree: 博士(医学), 2017, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第4262号. 学位記番号: 新大院博(医)甲第740号. 学位授与年月日: 平成29年3月23日

Journal of the Renin-Angiotensin-Aldosterone System 17(4),2016

Background and objective: Renin-angiotensin system (RAS) inhibitors reduce glomerular injury and… (more)

Subjects/Keywords: Angiotensinogen; podocyte; slit diaphragm; nephrin

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APA (6th Edition):

Yamazaki, M. (2017). Possible role for glomerular-derived angiotensinogen in nephrotic syndrome. : ネフローゼ症候群における糸球体由来アンジオテンシノーゲンの役割. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/47593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamazaki, Mihoko. “Possible role for glomerular-derived angiotensinogen in nephrotic syndrome. : ネフローゼ症候群における糸球体由来アンジオテンシノーゲンの役割.” 2017. Thesis, Niigata University / 新潟大学. Accessed February 27, 2021. http://hdl.handle.net/10191/47593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamazaki, Mihoko. “Possible role for glomerular-derived angiotensinogen in nephrotic syndrome. : ネフローゼ症候群における糸球体由来アンジオテンシノーゲンの役割.” 2017. Web. 27 Feb 2021.

Vancouver:

Yamazaki M. Possible role for glomerular-derived angiotensinogen in nephrotic syndrome. : ネフローゼ症候群における糸球体由来アンジオテンシノーゲンの役割. [Internet] [Thesis]. Niigata University / 新潟大学; 2017. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/10191/47593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamazaki M. Possible role for glomerular-derived angiotensinogen in nephrotic syndrome. : ネフローゼ症候群における糸球体由来アンジオテンシノーゲンの役割. [Thesis]. Niigata University / 新潟大学; 2017. Available from: http://hdl.handle.net/10191/47593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Guelph

3. Mahesaniya, Afreeda. Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway.

Degree: MS, Department of Molecular and Cellular Biology, 2017, University of Guelph

 Chronic kidney disease (CKD) is a debilitating illness which is increasing in frequency in the Canadian population as a consequence of the diabetes epidemic. Physicians… (more)

Subjects/Keywords: Soy; phytoestrogens; Akt; Podocyte; Nephrin

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APA (6th Edition):

Mahesaniya, A. (2017). Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/11603

Chicago Manual of Style (16th Edition):

Mahesaniya, Afreeda. “Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway.” 2017. Masters Thesis, University of Guelph. Accessed February 27, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/11603.

MLA Handbook (7th Edition):

Mahesaniya, Afreeda. “Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway.” 2017. Web. 27 Feb 2021.

Vancouver:

Mahesaniya A. Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway. [Internet] [Masters thesis]. University of Guelph; 2017. [cited 2021 Feb 27]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/11603.

Council of Science Editors:

Mahesaniya A. Investigating the effects of soy phytoestrogens on Nephrin and Akt podocyte survival signaling pathway. [Masters Thesis]. University of Guelph; 2017. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/11603


University of Guelph

4. Dutta, Nikkita. Investigation of Dok1 and Dok2 as novel modulators of focal adhesion dynamics in kidney podocytes.

Degree: MS, Department of Molecular and Cellular Biology, 2019, University of Guelph

Podocyte adhesion to the underlying glomerular basement membrane is required for proper filtration and is mediated by dynamic signalling events. Integrins are a major component… (more)

Subjects/Keywords: Signaling; Podocyte; Adhesion; Dok; Kidney

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APA (6th Edition):

Dutta, N. (2019). Investigation of Dok1 and Dok2 as novel modulators of focal adhesion dynamics in kidney podocytes. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/15994

Chicago Manual of Style (16th Edition):

Dutta, Nikkita. “Investigation of Dok1 and Dok2 as novel modulators of focal adhesion dynamics in kidney podocytes.” 2019. Masters Thesis, University of Guelph. Accessed February 27, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/15994.

MLA Handbook (7th Edition):

Dutta, Nikkita. “Investigation of Dok1 and Dok2 as novel modulators of focal adhesion dynamics in kidney podocytes.” 2019. Web. 27 Feb 2021.

Vancouver:

Dutta N. Investigation of Dok1 and Dok2 as novel modulators of focal adhesion dynamics in kidney podocytes. [Internet] [Masters thesis]. University of Guelph; 2019. [cited 2021 Feb 27]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/15994.

Council of Science Editors:

Dutta N. Investigation of Dok1 and Dok2 as novel modulators of focal adhesion dynamics in kidney podocytes. [Masters Thesis]. University of Guelph; 2019. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/15994


University of Manchester

5. Mccaffrey, James. Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome.

Degree: 2016, University of Manchester

BACKGROUND: Nephrotic syndrome (NS) occurs when the glomerular filtration barrier becomes abnormally permeable, leading to the clinical triad of proteinuria, massive oedema, and hypoalbuminaemia. Historically,… (more)

Subjects/Keywords: Podocyte; Glucocorticoid; Nephrotic Syndrome; Rac1

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APA (6th Edition):

Mccaffrey, J. (2016). Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294549

Chicago Manual of Style (16th Edition):

Mccaffrey, James. “Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome.” 2016. Doctoral Dissertation, University of Manchester. Accessed February 27, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294549.

MLA Handbook (7th Edition):

Mccaffrey, James. “Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome.” 2016. Web. 27 Feb 2021.

Vancouver:

Mccaffrey J. Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Feb 27]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294549.

Council of Science Editors:

Mccaffrey J. Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294549

6. Abdallah, Maya. Développer des hydrogels et étudier les effets des propriétés mécaniques sur les activités biologiques des podocytes : Development of hydrogels and study the effect of their mechanical properties on podocyte behaviors.

Degree: Docteur es, Chimie et physico-chimie des matériaux, 2019, Montpellier

La matrice extracellulaire (MEC) contrôle et maintient les principales activités biologiques telles que la survie, la prolifération et la différenciation cellulaire. Récemment, les hydrogels ont… (more)

Subjects/Keywords: Podocyte; Matrice extracellulaire; Hydrogel; Podocyte; Extracellular Matrix; Hydrogel

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APA (6th Edition):

Abdallah, M. (2019). Développer des hydrogels et étudier les effets des propriétés mécaniques sur les activités biologiques des podocytes : Development of hydrogels and study the effect of their mechanical properties on podocyte behaviors. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2019MONTS065

Chicago Manual of Style (16th Edition):

Abdallah, Maya. “Développer des hydrogels et étudier les effets des propriétés mécaniques sur les activités biologiques des podocytes : Development of hydrogels and study the effect of their mechanical properties on podocyte behaviors.” 2019. Doctoral Dissertation, Montpellier. Accessed February 27, 2021. http://www.theses.fr/2019MONTS065.

MLA Handbook (7th Edition):

Abdallah, Maya. “Développer des hydrogels et étudier les effets des propriétés mécaniques sur les activités biologiques des podocytes : Development of hydrogels and study the effect of their mechanical properties on podocyte behaviors.” 2019. Web. 27 Feb 2021.

Vancouver:

Abdallah M. Développer des hydrogels et étudier les effets des propriétés mécaniques sur les activités biologiques des podocytes : Development of hydrogels and study the effect of their mechanical properties on podocyte behaviors. [Internet] [Doctoral dissertation]. Montpellier; 2019. [cited 2021 Feb 27]. Available from: http://www.theses.fr/2019MONTS065.

Council of Science Editors:

Abdallah M. Développer des hydrogels et étudier les effets des propriétés mécaniques sur les activités biologiques des podocytes : Development of hydrogels and study the effect of their mechanical properties on podocyte behaviors. [Doctoral Dissertation]. Montpellier; 2019. Available from: http://www.theses.fr/2019MONTS065


McMaster University

7. Khalili, Hadiseh. SHROOM3 IN THE KIDNEY.

Degree: MSc, 2015, McMaster University

Chronic kidney disease (CKD), defined as an irreversible reduction in glomerular filtration rate, is a large public health concern. Dissecting the genetic components of CKD… (more)

Subjects/Keywords: Kidney; Glomerulus; Podocyte; Shroom3; CKD; Kidney Development

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APA (6th Edition):

Khalili, H. (2015). SHROOM3 IN THE KIDNEY. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/17413

Chicago Manual of Style (16th Edition):

Khalili, Hadiseh. “SHROOM3 IN THE KIDNEY.” 2015. Masters Thesis, McMaster University. Accessed February 27, 2021. http://hdl.handle.net/11375/17413.

MLA Handbook (7th Edition):

Khalili, Hadiseh. “SHROOM3 IN THE KIDNEY.” 2015. Web. 27 Feb 2021.

Vancouver:

Khalili H. SHROOM3 IN THE KIDNEY. [Internet] [Masters thesis]. McMaster University; 2015. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/11375/17413.

Council of Science Editors:

Khalili H. SHROOM3 IN THE KIDNEY. [Masters Thesis]. McMaster University; 2015. Available from: http://hdl.handle.net/11375/17413


University of Guelph

8. New, Laura Alexandra. Investigation of the Role of Nephrin Phosphorylation and Associated Signaling Pathways in Kidney Podocytes.

Degree: PhD, Department of Molecular and Cellular Biology, 2014, University of Guelph

 The integrity of the glomerulus plays a key role in prevention of kidney damage. The specialized function of the glomerular filter has led to the… (more)

Subjects/Keywords: podocyte; nephrin; Nck; cell signaling; tyrosine phosphorylation

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APA (6th Edition):

New, L. A. (2014). Investigation of the Role of Nephrin Phosphorylation and Associated Signaling Pathways in Kidney Podocytes. (Doctoral Dissertation). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8008

Chicago Manual of Style (16th Edition):

New, Laura Alexandra. “Investigation of the Role of Nephrin Phosphorylation and Associated Signaling Pathways in Kidney Podocytes.” 2014. Doctoral Dissertation, University of Guelph. Accessed February 27, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8008.

MLA Handbook (7th Edition):

New, Laura Alexandra. “Investigation of the Role of Nephrin Phosphorylation and Associated Signaling Pathways in Kidney Podocytes.” 2014. Web. 27 Feb 2021.

Vancouver:

New LA. Investigation of the Role of Nephrin Phosphorylation and Associated Signaling Pathways in Kidney Podocytes. [Internet] [Doctoral dissertation]. University of Guelph; 2014. [cited 2021 Feb 27]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8008.

Council of Science Editors:

New LA. Investigation of the Role of Nephrin Phosphorylation and Associated Signaling Pathways in Kidney Podocytes. [Doctoral Dissertation]. University of Guelph; 2014. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8008


University of Ottawa

9. Shaji, Roya. The Role of Thromboxane A2 Receptors in Diabetic Kidney Disease .

Degree: 2011, University of Ottawa

 Thromboxane receptor (TPr) activity is elevated in diabetes and contributes to complications of diabetic kidney disease (DKD). TPr blockade appears to have therapeutic potential. Several… (more)

Subjects/Keywords: podocyte; diabetic kidney disease; thromboxane A2

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APA (6th Edition):

Shaji, R. (2011). The Role of Thromboxane A2 Receptors in Diabetic Kidney Disease . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/19759

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shaji, Roya. “The Role of Thromboxane A2 Receptors in Diabetic Kidney Disease .” 2011. Thesis, University of Ottawa. Accessed February 27, 2021. http://hdl.handle.net/10393/19759.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shaji, Roya. “The Role of Thromboxane A2 Receptors in Diabetic Kidney Disease .” 2011. Web. 27 Feb 2021.

Vancouver:

Shaji R. The Role of Thromboxane A2 Receptors in Diabetic Kidney Disease . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/10393/19759.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shaji R. The Role of Thromboxane A2 Receptors in Diabetic Kidney Disease . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/19759

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

10. Chen, Junbo. Fine granular deposition of clonal immunoreactivity on podocyte cell bodies: a primary podocytopathy marker and potential clue to disease mechanism.

Degree: MS, Pathology, 2018, Boston University

 Minimal change disease (MCD) and primary (idiopathic) focal segmental glomerulosclerosis (1FSGS), referred to collectively as “primary podocytopathies”, are major causes of nephrotic syndrome in children… (more)

Subjects/Keywords: Pathology; IgG; Minimal change disease; Podocyte

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APA (6th Edition):

Chen, J. (2018). Fine granular deposition of clonal immunoreactivity on podocyte cell bodies: a primary podocytopathy marker and potential clue to disease mechanism. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/31223

Chicago Manual of Style (16th Edition):

Chen, Junbo. “Fine granular deposition of clonal immunoreactivity on podocyte cell bodies: a primary podocytopathy marker and potential clue to disease mechanism.” 2018. Masters Thesis, Boston University. Accessed February 27, 2021. http://hdl.handle.net/2144/31223.

MLA Handbook (7th Edition):

Chen, Junbo. “Fine granular deposition of clonal immunoreactivity on podocyte cell bodies: a primary podocytopathy marker and potential clue to disease mechanism.” 2018. Web. 27 Feb 2021.

Vancouver:

Chen J. Fine granular deposition of clonal immunoreactivity on podocyte cell bodies: a primary podocytopathy marker and potential clue to disease mechanism. [Internet] [Masters thesis]. Boston University; 2018. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/2144/31223.

Council of Science Editors:

Chen J. Fine granular deposition of clonal immunoreactivity on podocyte cell bodies: a primary podocytopathy marker and potential clue to disease mechanism. [Masters Thesis]. Boston University; 2018. Available from: http://hdl.handle.net/2144/31223


University of Manchester

11. Mccaffrey, James. Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome.

Degree: PhD, 2016, University of Manchester

 Background: Nephrotic syndrome (NS) occurs when the glomerular filtration barrier becomes abnormally permeable, leading to the clinical triad of proteinuria, massive oedema, and hypoalbuminaemia. Historically,… (more)

Subjects/Keywords: 618.92; Podocyte; Glucocorticoid; Nephrotic Syndrome; Rac1

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APA (6th Edition):

Mccaffrey, J. (2016). Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/podocytespecific-glucocorticoid-effects-in-childhood-nephrotic-syndrome(559e1d71-6e81-46e5-84ae-7c836e4de042).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680009

Chicago Manual of Style (16th Edition):

Mccaffrey, James. “Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome.” 2016. Doctoral Dissertation, University of Manchester. Accessed February 27, 2021. https://www.research.manchester.ac.uk/portal/en/theses/podocytespecific-glucocorticoid-effects-in-childhood-nephrotic-syndrome(559e1d71-6e81-46e5-84ae-7c836e4de042).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680009.

MLA Handbook (7th Edition):

Mccaffrey, James. “Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome.” 2016. Web. 27 Feb 2021.

Vancouver:

Mccaffrey J. Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Feb 27]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/podocytespecific-glucocorticoid-effects-in-childhood-nephrotic-syndrome(559e1d71-6e81-46e5-84ae-7c836e4de042).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680009.

Council of Science Editors:

Mccaffrey J. Podocyte-specific glucocorticoid effects in childhood nephrotic syndrome. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/podocytespecific-glucocorticoid-effects-in-childhood-nephrotic-syndrome(559e1d71-6e81-46e5-84ae-7c836e4de042).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680009


University of Edinburgh

12. Miller-Hodges, Eve Victoria. WT1 in the adult kidney : podocyte maintenance and the epithelial-mesenchymal balance.

Degree: PhD, 2014, University of Edinburgh

 Glomerular diseases are the leading cause of end stage kidney disease worldwide. Podocyte injury plays a key role in the initiation and development of such… (more)

Subjects/Keywords: 616.6; kidney; podocyte; epithelial-mesenchymal balance

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APA (6th Edition):

Miller-Hodges, E. V. (2014). WT1 in the adult kidney : podocyte maintenance and the epithelial-mesenchymal balance. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/10039

Chicago Manual of Style (16th Edition):

Miller-Hodges, Eve Victoria. “WT1 in the adult kidney : podocyte maintenance and the epithelial-mesenchymal balance.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed February 27, 2021. http://hdl.handle.net/1842/10039.

MLA Handbook (7th Edition):

Miller-Hodges, Eve Victoria. “WT1 in the adult kidney : podocyte maintenance and the epithelial-mesenchymal balance.” 2014. Web. 27 Feb 2021.

Vancouver:

Miller-Hodges EV. WT1 in the adult kidney : podocyte maintenance and the epithelial-mesenchymal balance. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/1842/10039.

Council of Science Editors:

Miller-Hodges EV. WT1 in the adult kidney : podocyte maintenance and the epithelial-mesenchymal balance. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/10039


University of Edinburgh

13. Zhou, Yu Simona. Podocyte repair and recovery in kidney disease.

Degree: PhD, 2011, University of Edinburgh

 Introduction Podocytes are terminally differentiated, highly specialized glomerular cells that form the final barrier to protein loss. Podocyte injury is characterised by proteinuria. Proteinuria is… (more)

Subjects/Keywords: 616.6; Podocyte; kidney disease; glomerulosclerosis; FSGS

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APA (6th Edition):

Zhou, Y. S. (2011). Podocyte repair and recovery in kidney disease. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/5959

Chicago Manual of Style (16th Edition):

Zhou, Yu Simona. “Podocyte repair and recovery in kidney disease.” 2011. Doctoral Dissertation, University of Edinburgh. Accessed February 27, 2021. http://hdl.handle.net/1842/5959.

MLA Handbook (7th Edition):

Zhou, Yu Simona. “Podocyte repair and recovery in kidney disease.” 2011. Web. 27 Feb 2021.

Vancouver:

Zhou YS. Podocyte repair and recovery in kidney disease. [Internet] [Doctoral dissertation]. University of Edinburgh; 2011. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/1842/5959.

Council of Science Editors:

Zhou YS. Podocyte repair and recovery in kidney disease. [Doctoral Dissertation]. University of Edinburgh; 2011. Available from: http://hdl.handle.net/1842/5959

14. N'Gome Sendeyo, Kelhia. Immunopathologie des podocytopathies acquises : rôle de c-mip dans les perturbations immunitaires et podocytaires : Immunopathology of acquired podocytopathy : role of c-mip in alterations of immune system and podocytes.

Degree: Docteur es, Physiopathologie, 2013, Université Paris-Est

Le Syndrome Néphrotique à Lésions Glomérulaires Minimes (SNLGM) et la glomérulonéphrite extra membraneuse (GEM) sont deux podocytopathies primitives d'origine immunitaire associant des altérations immunes et… (more)

Subjects/Keywords: Lymphocytes T; C-Mip; Snlgm; Podocyte; Gem; T lymphocyte; C-Mip; Mcns; Podocyte; Mn; 616.079

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APA (6th Edition):

N'Gome Sendeyo, K. (2013). Immunopathologie des podocytopathies acquises : rôle de c-mip dans les perturbations immunitaires et podocytaires : Immunopathology of acquired podocytopathy : role of c-mip in alterations of immune system and podocytes. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2013PEST0116

Chicago Manual of Style (16th Edition):

N'Gome Sendeyo, Kelhia. “Immunopathologie des podocytopathies acquises : rôle de c-mip dans les perturbations immunitaires et podocytaires : Immunopathology of acquired podocytopathy : role of c-mip in alterations of immune system and podocytes.” 2013. Doctoral Dissertation, Université Paris-Est. Accessed February 27, 2021. http://www.theses.fr/2013PEST0116.

MLA Handbook (7th Edition):

N'Gome Sendeyo, Kelhia. “Immunopathologie des podocytopathies acquises : rôle de c-mip dans les perturbations immunitaires et podocytaires : Immunopathology of acquired podocytopathy : role of c-mip in alterations of immune system and podocytes.” 2013. Web. 27 Feb 2021.

Vancouver:

N'Gome Sendeyo K. Immunopathologie des podocytopathies acquises : rôle de c-mip dans les perturbations immunitaires et podocytaires : Immunopathology of acquired podocytopathy : role of c-mip in alterations of immune system and podocytes. [Internet] [Doctoral dissertation]. Université Paris-Est; 2013. [cited 2021 Feb 27]. Available from: http://www.theses.fr/2013PEST0116.

Council of Science Editors:

N'Gome Sendeyo K. Immunopathologie des podocytopathies acquises : rôle de c-mip dans les perturbations immunitaires et podocytaires : Immunopathology of acquired podocytopathy : role of c-mip in alterations of immune system and podocytes. [Doctoral Dissertation]. Université Paris-Est; 2013. Available from: http://www.theses.fr/2013PEST0116


Universidade do Rio Grande do Sul

15. Rodrigues, Patrícia Garcia. Expressão gênica de moléculas associadas ao podócito em pacientes portadores de glomerulopatias proteinúricas.

Degree: 2012, Universidade do Rio Grande do Sul

Introdução: A injúria ao podócito glomerular tem um papel crítico para o surgimento de proteinúria em diferentes glomerulopatias. Neste estudo avaliamos a expressão gênica das… (more)

Subjects/Keywords: Podocyte; Glomerulonefrite; Glomerulopathies; Podócitos; Expressão gênica; Gene expression; Podocin; Nephrin

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APA (6th Edition):

Rodrigues, P. G. (2012). Expressão gênica de moléculas associadas ao podócito em pacientes portadores de glomerulopatias proteinúricas. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/56680

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodrigues, Patrícia Garcia. “Expressão gênica de moléculas associadas ao podócito em pacientes portadores de glomerulopatias proteinúricas.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed February 27, 2021. http://hdl.handle.net/10183/56680.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodrigues, Patrícia Garcia. “Expressão gênica de moléculas associadas ao podócito em pacientes portadores de glomerulopatias proteinúricas.” 2012. Web. 27 Feb 2021.

Vancouver:

Rodrigues PG. Expressão gênica de moléculas associadas ao podócito em pacientes portadores de glomerulopatias proteinúricas. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/10183/56680.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodrigues PG. Expressão gênica de moléculas associadas ao podócito em pacientes portadores de glomerulopatias proteinúricas. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/56680

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Guelph

16. Stringer, Colin D.M. Development of an inducible and reversible mouse model of podocyte effacement.

Degree: MS, Department of Molecular and Cellular Biology, 2011, University of Guelph

 Podocytes are specialized epithelial cells which wrap glomerular capillaries with numerous interdigitating foot processes (FP). Between adjacent FPs a unique junction, the slit diaphragm (SD),… (more)

Subjects/Keywords: nephropathy; proteinuria; glomerular filtration; podocyte; slit diaphragm; Nck; transgenic mouse model

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APA (6th Edition):

Stringer, C. D. M. (2011). Development of an inducible and reversible mouse model of podocyte effacement. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2935

Chicago Manual of Style (16th Edition):

Stringer, Colin D M. “Development of an inducible and reversible mouse model of podocyte effacement.” 2011. Masters Thesis, University of Guelph. Accessed February 27, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2935.

MLA Handbook (7th Edition):

Stringer, Colin D M. “Development of an inducible and reversible mouse model of podocyte effacement.” 2011. Web. 27 Feb 2021.

Vancouver:

Stringer CDM. Development of an inducible and reversible mouse model of podocyte effacement. [Internet] [Masters thesis]. University of Guelph; 2011. [cited 2021 Feb 27]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2935.

Council of Science Editors:

Stringer CDM. Development of an inducible and reversible mouse model of podocyte effacement. [Masters Thesis]. University of Guelph; 2011. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/2935

17. Martin, Claire. Characterization of signaling pathways regulating nephrin endocytosis in kidney podocytes: novel roles for Nck and ShcA adaptor proteins.

Degree: PhD, Department of Molecular and Cellular Biology, 2017, University of Guelph

 Kidney podocytes maintain blood filtration selectivity through a network of actin-based projections termed foot processes. The slit diaphragm is a molecular barrier that lies laterally… (more)

Subjects/Keywords: kidney; podocyte; nephrin

…1.2.2 The specialized structure of the podocyte and the filtration slits ... ….3 1.2.3… …Podocyte disruption in disease.. . .4 1.3 Nephrin: the core of the slit… …Nephrin is required for podocyte function ...…...6 1.3.4 Nephrin recruits cytosolic… …signaling ….14 viii 1.3.9 Disruption of nephrin tyrosine phosphorylation in podocyte-based… …in podocyte biology 15 1.4.1 Endocytosis in mammalian cells… 

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APA (6th Edition):

Martin, C. (2017). Characterization of signaling pathways regulating nephrin endocytosis in kidney podocytes: novel roles for Nck and ShcA adaptor proteins. (Doctoral Dissertation). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/12112

Chicago Manual of Style (16th Edition):

Martin, Claire. “Characterization of signaling pathways regulating nephrin endocytosis in kidney podocytes: novel roles for Nck and ShcA adaptor proteins.” 2017. Doctoral Dissertation, University of Guelph. Accessed February 27, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/12112.

MLA Handbook (7th Edition):

Martin, Claire. “Characterization of signaling pathways regulating nephrin endocytosis in kidney podocytes: novel roles for Nck and ShcA adaptor proteins.” 2017. Web. 27 Feb 2021.

Vancouver:

Martin C. Characterization of signaling pathways regulating nephrin endocytosis in kidney podocytes: novel roles for Nck and ShcA adaptor proteins. [Internet] [Doctoral dissertation]. University of Guelph; 2017. [cited 2021 Feb 27]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/12112.

Council of Science Editors:

Martin C. Characterization of signaling pathways regulating nephrin endocytosis in kidney podocytes: novel roles for Nck and ShcA adaptor proteins. [Doctoral Dissertation]. University of Guelph; 2017. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/12112


University of Miami

18. Sloan, Alexis Joanna. The Role of Nuclear Factor of Activated T-Cells in Chronic Kidney Disease.

Degree: PhD, Cell Biology and Anatomy (Medicine), 2015, University of Miami

  Aberrant elevation in cytoplasmic calcium levels in podocytes cause changes in podocyte morphology and glomerular permeability. The phosphatase calcineurin responds to elevated calcium by… (more)

Subjects/Keywords: NFAT; FSGS; Minimal Change Disease; Chronic Kidney Disease; podocyte

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APA (6th Edition):

Sloan, A. J. (2015). The Role of Nuclear Factor of Activated T-Cells in Chronic Kidney Disease. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/1443

Chicago Manual of Style (16th Edition):

Sloan, Alexis Joanna. “The Role of Nuclear Factor of Activated T-Cells in Chronic Kidney Disease.” 2015. Doctoral Dissertation, University of Miami. Accessed February 27, 2021. https://scholarlyrepository.miami.edu/oa_dissertations/1443.

MLA Handbook (7th Edition):

Sloan, Alexis Joanna. “The Role of Nuclear Factor of Activated T-Cells in Chronic Kidney Disease.” 2015. Web. 27 Feb 2021.

Vancouver:

Sloan AJ. The Role of Nuclear Factor of Activated T-Cells in Chronic Kidney Disease. [Internet] [Doctoral dissertation]. University of Miami; 2015. [cited 2021 Feb 27]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1443.

Council of Science Editors:

Sloan AJ. The Role of Nuclear Factor of Activated T-Cells in Chronic Kidney Disease. [Doctoral Dissertation]. University of Miami; 2015. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1443


Boston University

19. Belghasem, Mostafa. Pathological and molecular profiling in hypertension-induced glomerular injury.

Degree: PhD, Pathology & Laboratory Medicine, 2015, Boston University

 The increased prevalence of chronic kidney disease (CKD) has become a major global health burden. This increase in CKD burden parallels the increase in hypertension… (more)

Subjects/Keywords: Pathology; Chronic kidney disease; Glomerulosclerosis; Hypertension; Podocyte; Mouse models

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APA (6th Edition):

Belghasem, M. (2015). Pathological and molecular profiling in hypertension-induced glomerular injury. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/13940

Chicago Manual of Style (16th Edition):

Belghasem, Mostafa. “Pathological and molecular profiling in hypertension-induced glomerular injury.” 2015. Doctoral Dissertation, Boston University. Accessed February 27, 2021. http://hdl.handle.net/2144/13940.

MLA Handbook (7th Edition):

Belghasem, Mostafa. “Pathological and molecular profiling in hypertension-induced glomerular injury.” 2015. Web. 27 Feb 2021.

Vancouver:

Belghasem M. Pathological and molecular profiling in hypertension-induced glomerular injury. [Internet] [Doctoral dissertation]. Boston University; 2015. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/2144/13940.

Council of Science Editors:

Belghasem M. Pathological and molecular profiling in hypertension-induced glomerular injury. [Doctoral Dissertation]. Boston University; 2015. Available from: http://hdl.handle.net/2144/13940


Boston University

20. Milo Rasouly, Hila. Discovery and analysis of genes important in kidney development and disease.

Degree: PhD, Genetics & Genomics, 2015, Boston University

 Abnormal kidney development is a relatively prevalent health issue; however, the genetic basis is mostly unknown. The aim of this thesis is to identify genes… (more)

Subjects/Keywords: Developmental biology; Robo2; Zeb2; Glomerulocystic; Kidney; Podocyte; Transgenic

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APA (6th Edition):

Milo Rasouly, H. (2015). Discovery and analysis of genes important in kidney development and disease. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/13984

Chicago Manual of Style (16th Edition):

Milo Rasouly, Hila. “Discovery and analysis of genes important in kidney development and disease.” 2015. Doctoral Dissertation, Boston University. Accessed February 27, 2021. http://hdl.handle.net/2144/13984.

MLA Handbook (7th Edition):

Milo Rasouly, Hila. “Discovery and analysis of genes important in kidney development and disease.” 2015. Web. 27 Feb 2021.

Vancouver:

Milo Rasouly H. Discovery and analysis of genes important in kidney development and disease. [Internet] [Doctoral dissertation]. Boston University; 2015. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/2144/13984.

Council of Science Editors:

Milo Rasouly H. Discovery and analysis of genes important in kidney development and disease. [Doctoral Dissertation]. Boston University; 2015. Available from: http://hdl.handle.net/2144/13984


Harvard University

21. Sidhom, Eriene-Heidi. Unraveling the Molecular and Cellular Complexities of the Kidney in Health and Disease.

Degree: PhD, 2019, Harvard University

Chronic kidney disease (CKD) affects over 500 million people worldwide, and despite this significant disease burden, therapeutic innovation in nephrology has lagged. This trend is… (more)

Subjects/Keywords: Kidney; podocyte; organoid; Coenzyme Q; PDSS2; nephrotic syndrome; polyunsaturated fatty acids

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APA (6th Edition):

Sidhom, E. (2019). Unraveling the Molecular and Cellular Complexities of the Kidney in Health and Disease. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029547

Chicago Manual of Style (16th Edition):

Sidhom, Eriene-Heidi. “Unraveling the Molecular and Cellular Complexities of the Kidney in Health and Disease.” 2019. Doctoral Dissertation, Harvard University. Accessed February 27, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029547.

MLA Handbook (7th Edition):

Sidhom, Eriene-Heidi. “Unraveling the Molecular and Cellular Complexities of the Kidney in Health and Disease.” 2019. Web. 27 Feb 2021.

Vancouver:

Sidhom E. Unraveling the Molecular and Cellular Complexities of the Kidney in Health and Disease. [Internet] [Doctoral dissertation]. Harvard University; 2019. [cited 2021 Feb 27]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029547.

Council of Science Editors:

Sidhom E. Unraveling the Molecular and Cellular Complexities of the Kidney in Health and Disease. [Doctoral Dissertation]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029547


University of Manchester

22. Hosawi, Salman. Global proteomic analysis of insulin action in glomerular podocytes.

Degree: PhD, 2018, University of Manchester

 Introduction & Aim: Insulin signalling contributes to diverse cellular activities including protein synthesis, proliferation and cell survival. Insulin resistance describes the inability of cells to… (more)

Subjects/Keywords: Proteomics; Insulin resistance; Diabetes; Podocyte; Insulin signalling; Free fatty acids

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APA (6th Edition):

Hosawi, S. (2018). Global proteomic analysis of insulin action in glomerular podocytes. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/global-proteomic-analysis-of-insulin-action-in-glomerular-podocytes(8783e6b4-8dc8-4ba0-92f7-6f193eb4c5af).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771402

Chicago Manual of Style (16th Edition):

Hosawi, Salman. “Global proteomic analysis of insulin action in glomerular podocytes.” 2018. Doctoral Dissertation, University of Manchester. Accessed February 27, 2021. https://www.research.manchester.ac.uk/portal/en/theses/global-proteomic-analysis-of-insulin-action-in-glomerular-podocytes(8783e6b4-8dc8-4ba0-92f7-6f193eb4c5af).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771402.

MLA Handbook (7th Edition):

Hosawi, Salman. “Global proteomic analysis of insulin action in glomerular podocytes.” 2018. Web. 27 Feb 2021.

Vancouver:

Hosawi S. Global proteomic analysis of insulin action in glomerular podocytes. [Internet] [Doctoral dissertation]. University of Manchester; 2018. [cited 2021 Feb 27]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/global-proteomic-analysis-of-insulin-action-in-glomerular-podocytes(8783e6b4-8dc8-4ba0-92f7-6f193eb4c5af).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771402.

Council of Science Editors:

Hosawi S. Global proteomic analysis of insulin action in glomerular podocytes. [Doctoral Dissertation]. University of Manchester; 2018. Available from: https://www.research.manchester.ac.uk/portal/en/theses/global-proteomic-analysis-of-insulin-action-in-glomerular-podocytes(8783e6b4-8dc8-4ba0-92f7-6f193eb4c5af).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.771402

23. Lassén, Emelie. Molecular perspectives on glomerular cell physiology in chronic kidney disease.

Degree: 2019, University of Gothenburg / Göteborgs Universitet

 Glomerulonephritis is one of the most common causes of chronic kidney disease (CKD) in the world. Recent establishment of guidelines for classification of CKD in… (more)

Subjects/Keywords: Chronic kidney disease; IgA nephropathy; mesangial cell; podocyte; CKAP4

Page 1 Page 2

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APA (6th Edition):

Lassén, E. (2019). Molecular perspectives on glomerular cell physiology in chronic kidney disease. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/59539

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lassén, Emelie. “Molecular perspectives on glomerular cell physiology in chronic kidney disease.” 2019. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed February 27, 2021. http://hdl.handle.net/2077/59539.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lassén, Emelie. “Molecular perspectives on glomerular cell physiology in chronic kidney disease.” 2019. Web. 27 Feb 2021.

Vancouver:

Lassén E. Molecular perspectives on glomerular cell physiology in chronic kidney disease. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2019. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/2077/59539.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lassén E. Molecular perspectives on glomerular cell physiology in chronic kidney disease. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2019. Available from: http://hdl.handle.net/2077/59539

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Harvard University

24. Clark, Abbe Rose. Single-Cell Transcriptional Landscape of the Kidney Filter in Health and Disease.

Degree: PhD, 2019, Harvard University

Progressive chronic kidney diseases (CKDs) affect more than 700 million people worldwide and are increasing in prevalence. However, the sequence of events resulting in CKD… (more)

Subjects/Keywords: kidney; podocyte; single-cell RNA sequencing; model; ctcf

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APA (6th Edition):

Clark, A. R. (2019). Single-Cell Transcriptional Landscape of the Kidney Filter in Health and Disease. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42106939

Chicago Manual of Style (16th Edition):

Clark, Abbe Rose. “Single-Cell Transcriptional Landscape of the Kidney Filter in Health and Disease.” 2019. Doctoral Dissertation, Harvard University. Accessed February 27, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42106939.

MLA Handbook (7th Edition):

Clark, Abbe Rose. “Single-Cell Transcriptional Landscape of the Kidney Filter in Health and Disease.” 2019. Web. 27 Feb 2021.

Vancouver:

Clark AR. Single-Cell Transcriptional Landscape of the Kidney Filter in Health and Disease. [Internet] [Doctoral dissertation]. Harvard University; 2019. [cited 2021 Feb 27]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42106939.

Council of Science Editors:

Clark AR. Single-Cell Transcriptional Landscape of the Kidney Filter in Health and Disease. [Doctoral Dissertation]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42106939


University of Western Ontario

25. Sull, Alexandra. The Role of Shroom3 in Kidney Development.

Degree: 2015, University of Western Ontario

 Chronic kidney disease (CKD) affects 10% of the population in industrialized nations. Recent genome wide association studies (GWAS) have correlated SHROOM3 with glomerular filtration rate… (more)

Subjects/Keywords: Shroom3; nephrogenesis; chronic kidney disease; actin; podocyte; mouse; Other Physiology

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APA (6th Edition):

Sull, A. (2015). The Role of Shroom3 in Kidney Development. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/2881

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sull, Alexandra. “The Role of Shroom3 in Kidney Development.” 2015. Thesis, University of Western Ontario. Accessed February 27, 2021. https://ir.lib.uwo.ca/etd/2881.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sull, Alexandra. “The Role of Shroom3 in Kidney Development.” 2015. Web. 27 Feb 2021.

Vancouver:

Sull A. The Role of Shroom3 in Kidney Development. [Internet] [Thesis]. University of Western Ontario; 2015. [cited 2021 Feb 27]. Available from: https://ir.lib.uwo.ca/etd/2881.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sull A. The Role of Shroom3 in Kidney Development. [Thesis]. University of Western Ontario; 2015. Available from: https://ir.lib.uwo.ca/etd/2881

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

26. Li, Biao. Role of sFlt1 and Anxa2 in the Glomerular Filtration Barrier Integrity Maintenance.

Degree: PhD, 2016, University of Toronto

Gene targeting studies have confirmed the role of VEGF-VEGFR system in vasculogenesis and angiogenesis. While VEGFR2 is a mediator of major functions of VEGF, the… (more)

Subjects/Keywords: angiogenesis; embryogenesis; podocyte; soluble VEGFR1; Vasculogenesis; VEGF; 0369

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APA (6th Edition):

Li, B. (2016). Role of sFlt1 and Anxa2 in the Glomerular Filtration Barrier Integrity Maintenance. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/89256

Chicago Manual of Style (16th Edition):

Li, Biao. “Role of sFlt1 and Anxa2 in the Glomerular Filtration Barrier Integrity Maintenance.” 2016. Doctoral Dissertation, University of Toronto. Accessed February 27, 2021. http://hdl.handle.net/1807/89256.

MLA Handbook (7th Edition):

Li, Biao. “Role of sFlt1 and Anxa2 in the Glomerular Filtration Barrier Integrity Maintenance.” 2016. Web. 27 Feb 2021.

Vancouver:

Li B. Role of sFlt1 and Anxa2 in the Glomerular Filtration Barrier Integrity Maintenance. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/1807/89256.

Council of Science Editors:

Li B. Role of sFlt1 and Anxa2 in the Glomerular Filtration Barrier Integrity Maintenance. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/89256


Freie Universität Berlin

27. Müller, Tilman. Apelinerges System in der Niere: Implikationen für die diabetische Nierenerkrankung.

Degree: 2020, Freie Universität Berlin

 Für die bioaktiven Peptide des apelinergen Systems und ihres Rezeptors APJ wurden in experimentellen kardiovaskulären Erkrankungen und der diabetischen Nierenkrankheit (DKD) eine protektive Rolle aufgezeigt.… (more)

Subjects/Keywords: Apelin; diabetic kidney disease; Podocyte; APJ; Apelinergic System; ddc:610

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APA (6th Edition):

Müller, T. (2020). Apelinerges System in der Niere: Implikationen für die diabetische Nierenerkrankung. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-27613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Müller, Tilman. “Apelinerges System in der Niere: Implikationen für die diabetische Nierenerkrankung.” 2020. Thesis, Freie Universität Berlin. Accessed February 27, 2021. http://dx.doi.org/10.17169/refubium-27613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Müller, Tilman. “Apelinerges System in der Niere: Implikationen für die diabetische Nierenerkrankung.” 2020. Web. 27 Feb 2021.

Vancouver:

Müller T. Apelinerges System in der Niere: Implikationen für die diabetische Nierenerkrankung. [Internet] [Thesis]. Freie Universität Berlin; 2020. [cited 2021 Feb 27]. Available from: http://dx.doi.org/10.17169/refubium-27613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Müller T. Apelinerges System in der Niere: Implikationen für die diabetische Nierenerkrankung. [Thesis]. Freie Universität Berlin; 2020. Available from: http://dx.doi.org/10.17169/refubium-27613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Koop, Klaas. Proteinuria and function loss in native and transplanted kidneys.

Degree: 2009, Department of Pathology, Faculty of Medicine, Leiden University Medical Center (LUMC), Leiden University

 “Bones can break, muscles can atrophy, glands can loaf, even the brain can go to sleep, without immediately endangering our survival, but when the kidneys… (more)

Subjects/Keywords: Glomerulus; Kidney; Proteinuria; Podocyte; Transplantation; Cyclosporine; Glomerulus; Kidney; Proteinuria; Podocyte; Transplantation; Cyclosporine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Koop, K. (2009). Proteinuria and function loss in native and transplanted kidneys. (Doctoral Dissertation). Department of Pathology, Faculty of Medicine, Leiden University Medical Center (LUMC), Leiden University. Retrieved from http://hdl.handle.net/1887/13951

Chicago Manual of Style (16th Edition):

Koop, Klaas. “Proteinuria and function loss in native and transplanted kidneys.” 2009. Doctoral Dissertation, Department of Pathology, Faculty of Medicine, Leiden University Medical Center (LUMC), Leiden University. Accessed February 27, 2021. http://hdl.handle.net/1887/13951.

MLA Handbook (7th Edition):

Koop, Klaas. “Proteinuria and function loss in native and transplanted kidneys.” 2009. Web. 27 Feb 2021.

Vancouver:

Koop K. Proteinuria and function loss in native and transplanted kidneys. [Internet] [Doctoral dissertation]. Department of Pathology, Faculty of Medicine, Leiden University Medical Center (LUMC), Leiden University; 2009. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/1887/13951.

Council of Science Editors:

Koop K. Proteinuria and function loss in native and transplanted kidneys. [Doctoral Dissertation]. Department of Pathology, Faculty of Medicine, Leiden University Medical Center (LUMC), Leiden University; 2009. Available from: http://hdl.handle.net/1887/13951


Universidade do Rio Grande do Sul

29. Nascimento, Jonathan Fraportti do. Expressão gênica de proteínas do podócito na urina de pacientes diabéticos normo, micro ou macroalbuminúricos e em pré diabeticos.

Degree: 2012, Universidade do Rio Grande do Sul

Introdução: A lesão do podócito exerce um papel crítico na nefropatia diabética (ND) e é um fator preditivo de albuminúria patológica e progressão da doença.… (more)

Subjects/Keywords: Diabetes mellitus; Podocyte; TRPC6; Podócitos; Expressão gênica; Podocin; Nephrin; Urina; Pre-diabetes; Albuminúria; Diabetic nephropathy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nascimento, J. F. d. (2012). Expressão gênica de proteínas do podócito na urina de pacientes diabéticos normo, micro ou macroalbuminúricos e em pré diabeticos. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/142953

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nascimento, Jonathan Fraportti do. “Expressão gênica de proteínas do podócito na urina de pacientes diabéticos normo, micro ou macroalbuminúricos e em pré diabeticos.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed February 27, 2021. http://hdl.handle.net/10183/142953.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nascimento, Jonathan Fraportti do. “Expressão gênica de proteínas do podócito na urina de pacientes diabéticos normo, micro ou macroalbuminúricos e em pré diabeticos.” 2012. Web. 27 Feb 2021.

Vancouver:

Nascimento JFd. Expressão gênica de proteínas do podócito na urina de pacientes diabéticos normo, micro ou macroalbuminúricos e em pré diabeticos. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/10183/142953.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nascimento JFd. Expressão gênica de proteínas do podócito na urina de pacientes diabéticos normo, micro ou macroalbuminúricos e em pré diabeticos. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/142953

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Rezende, Gabriela de Mendonça. Lesão podocitária na nefrite lúpica membranosa pura e proliferativa: mecanismos distintos de proteinúria?.

Degree: PhD, Processos Imunes e Infecciosos, 2015, University of São Paulo

Proteinúria é a principal manifestação da nefrite lúpica (NL) e reflete lesão no podócito. Análise dos biomarcadores do podócito foi realizada com o objetivo de… (more)

Subjects/Keywords: Lúpus eritematoso sistêmico; Lupus nephritis; Nefrite lúpica; Podócito; Podocyte; Proteinuria; Proteinúria; Systemic lupus erythematosus

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rezende, G. d. M. (2015). Lesão podocitária na nefrite lúpica membranosa pura e proliferativa: mecanismos distintos de proteinúria?. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5164/tde-13052015-093352/ ;

Chicago Manual of Style (16th Edition):

Rezende, Gabriela de Mendonça. “Lesão podocitária na nefrite lúpica membranosa pura e proliferativa: mecanismos distintos de proteinúria?.” 2015. Doctoral Dissertation, University of São Paulo. Accessed February 27, 2021. http://www.teses.usp.br/teses/disponiveis/5/5164/tde-13052015-093352/ ;.

MLA Handbook (7th Edition):

Rezende, Gabriela de Mendonça. “Lesão podocitária na nefrite lúpica membranosa pura e proliferativa: mecanismos distintos de proteinúria?.” 2015. Web. 27 Feb 2021.

Vancouver:

Rezende GdM. Lesão podocitária na nefrite lúpica membranosa pura e proliferativa: mecanismos distintos de proteinúria?. [Internet] [Doctoral dissertation]. University of São Paulo; 2015. [cited 2021 Feb 27]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5164/tde-13052015-093352/ ;.

Council of Science Editors:

Rezende GdM. Lesão podocitária na nefrite lúpica membranosa pura e proliferativa: mecanismos distintos de proteinúria?. [Doctoral Dissertation]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/5/5164/tde-13052015-093352/ ;

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