Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Platelet signaling). Showing records 1 – 11 of 11 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters


University of Rochester

1. Farrar, Christopher S. The influence of extracellular matrix fibronectin on platelet-derived growth factor signaling.

Degree: PhD, 2018, University of Rochester

 Cell signaling in response to platelet-derived growth factor (PDGF) has been found to be either up- or downregulated in a variety of pathological conditions, making… (more)

Subjects/Keywords: Fibronectin; Extracellular matrix; Platelet-derived growth factor; Calcium; Signaling; PI3K

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Farrar, C. S. (2018). The influence of extracellular matrix fibronectin on platelet-derived growth factor signaling. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/33603

Chicago Manual of Style (16th Edition):

Farrar, Christopher S. “The influence of extracellular matrix fibronectin on platelet-derived growth factor signaling.” 2018. Doctoral Dissertation, University of Rochester. Accessed October 21, 2020. http://hdl.handle.net/1802/33603.

MLA Handbook (7th Edition):

Farrar, Christopher S. “The influence of extracellular matrix fibronectin on platelet-derived growth factor signaling.” 2018. Web. 21 Oct 2020.

Vancouver:

Farrar CS. The influence of extracellular matrix fibronectin on platelet-derived growth factor signaling. [Internet] [Doctoral dissertation]. University of Rochester; 2018. [cited 2020 Oct 21]. Available from: http://hdl.handle.net/1802/33603.

Council of Science Editors:

Farrar CS. The influence of extracellular matrix fibronectin on platelet-derived growth factor signaling. [Doctoral Dissertation]. University of Rochester; 2018. Available from: http://hdl.handle.net/1802/33603


University of Illinois – Chicago

2. Xu, Zheng. Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets.

Degree: 2016, University of Illinois – Chicago

 Excessive Reactive Oxygen Species (ROS) is associated with all known cardiovascular risk factors and is believed to play an important role for the onset of… (more)

Subjects/Keywords: integrin; platelet; signaling; Reactive Oxygen Species; NADPH oxidase

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Xu, Z. (2016). Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xu, Zheng. “Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets.” 2016. Thesis, University of Illinois – Chicago. Accessed October 21, 2020. http://hdl.handle.net/10027/21243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xu, Zheng. “Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets.” 2016. Web. 21 Oct 2020.

Vancouver:

Xu Z. Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2020 Oct 21]. Available from: http://hdl.handle.net/10027/21243.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xu Z. Integrin alphaIIbbeta3-dependent Reactive Oxygen Species Production in Platelets. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/21243

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia Tech

3. Chen, Yunfeng. Integrin dynamics and platelet mechanosensing via surface receptors GPIbα and integrin αIIbβ3.

Degree: PhD, Mechanical Engineering, 2016, Georgia Tech

 In hemostasis and thrombosis, platelet adhesion and signaling play key roles. Two platelet receptors, glycoprotein Ibα (GPIbα) and integrin αIIbβ3, mediate the early and mid-stages… (more)

Subjects/Keywords: platelet; molecular mechanotransduction; GPIbα; αIIbβ3; binding kinetics; signaling

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, Y. (2016). Integrin dynamics and platelet mechanosensing via surface receptors GPIbα and integrin αIIbβ3. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/58596

Chicago Manual of Style (16th Edition):

Chen, Yunfeng. “Integrin dynamics and platelet mechanosensing via surface receptors GPIbα and integrin αIIbβ3.” 2016. Doctoral Dissertation, Georgia Tech. Accessed October 21, 2020. http://hdl.handle.net/1853/58596.

MLA Handbook (7th Edition):

Chen, Yunfeng. “Integrin dynamics and platelet mechanosensing via surface receptors GPIbα and integrin αIIbβ3.” 2016. Web. 21 Oct 2020.

Vancouver:

Chen Y. Integrin dynamics and platelet mechanosensing via surface receptors GPIbα and integrin αIIbβ3. [Internet] [Doctoral dissertation]. Georgia Tech; 2016. [cited 2020 Oct 21]. Available from: http://hdl.handle.net/1853/58596.

Council of Science Editors:

Chen Y. Integrin dynamics and platelet mechanosensing via surface receptors GPIbα and integrin αIIbβ3. [Doctoral Dissertation]. Georgia Tech; 2016. Available from: http://hdl.handle.net/1853/58596


Temple University

4. Bynagari, Yamini Saraswathy. Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets.

Degree: PhD, 2010, Temple University

Molecular and Cellular Physiology

Platelets are primary components of hemostasis. However, incongruous activation of platelets lead to thrombosis, which result in multiple cardio-vascular and cerebrovascular… (more)

Subjects/Keywords: Biology, Cell; Biology, Physiology; ADP receptors; Phosphatases; Platelet signaling; Protein Kinase C; thromboxane

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bynagari, Y. S. (2010). Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,103230

Chicago Manual of Style (16th Edition):

Bynagari, Yamini Saraswathy. “Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets.” 2010. Doctoral Dissertation, Temple University. Accessed October 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,103230.

MLA Handbook (7th Edition):

Bynagari, Yamini Saraswathy. “Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets.” 2010. Web. 21 Oct 2020.

Vancouver:

Bynagari YS. Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,103230.

Council of Science Editors:

Bynagari YS. Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,103230


University of Kentucky

5. Fulkerson, Zachary Bennett. LYSOPHOSPHATIDIC ACID PRODUCTION AND SIGNALING IN PLATELETS.

Degree: 2011, University of Kentucky

 Lysophosphatidic acid (LPA) belongs to a class of extracellular lipid signaling molecules. In the vasculature, LPA may regulate platelet activation and modulate endothelial and smooth… (more)

Subjects/Keywords: lysophosphatidic acid; platelet; autotaxin; signaling; integrin; G protein-coupled receptor; Biochemistry; Medical Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fulkerson, Z. B. (2011). LYSOPHOSPHATIDIC ACID PRODUCTION AND SIGNALING IN PLATELETS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/physiology_etds/1

Chicago Manual of Style (16th Edition):

Fulkerson, Zachary Bennett. “LYSOPHOSPHATIDIC ACID PRODUCTION AND SIGNALING IN PLATELETS.” 2011. Doctoral Dissertation, University of Kentucky. Accessed October 21, 2020. https://uknowledge.uky.edu/physiology_etds/1.

MLA Handbook (7th Edition):

Fulkerson, Zachary Bennett. “LYSOPHOSPHATIDIC ACID PRODUCTION AND SIGNALING IN PLATELETS.” 2011. Web. 21 Oct 2020.

Vancouver:

Fulkerson ZB. LYSOPHOSPHATIDIC ACID PRODUCTION AND SIGNALING IN PLATELETS. [Internet] [Doctoral dissertation]. University of Kentucky; 2011. [cited 2020 Oct 21]. Available from: https://uknowledge.uky.edu/physiology_etds/1.

Council of Science Editors:

Fulkerson ZB. LYSOPHOSPHATIDIC ACID PRODUCTION AND SIGNALING IN PLATELETS. [Doctoral Dissertation]. University of Kentucky; 2011. Available from: https://uknowledge.uky.edu/physiology_etds/1

6. 山田, 浩太. Different PDGF receptor dimers drive distinct motility of the mouse skin fibroblast. : 異なった組み合わせの血小板由来増殖因子受容体2量体はマウス皮膚由来線維芽細胞においてそれぞれ異なった運動様式を誘導する.

Degree: 博士(医学), 2016, University of Toyama / 富山大学

富山大学・富医薬博甲第132号・山田浩太・2014/03/21・★論文非公開★

Subjects/Keywords: platelet derived growth factor; receptor; chemotaxis; fibroblasts; polarity; signaling

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

山田, . (2016). Different PDGF receptor dimers drive distinct motility of the mouse skin fibroblast. : 異なった組み合わせの血小板由来増殖因子受容体2量体はマウス皮膚由来線維芽細胞においてそれぞれ異なった運動様式を誘導する. (Thesis). University of Toyama / 富山大学. Retrieved from http://hdl.handle.net/10110/12967 ; http://dx.doi.org/10.15099/00005068

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山田, 浩太. “Different PDGF receptor dimers drive distinct motility of the mouse skin fibroblast. : 異なった組み合わせの血小板由来増殖因子受容体2量体はマウス皮膚由来線維芽細胞においてそれぞれ異なった運動様式を誘導する.” 2016. Thesis, University of Toyama / 富山大学. Accessed October 21, 2020. http://hdl.handle.net/10110/12967 ; http://dx.doi.org/10.15099/00005068.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山田, 浩太. “Different PDGF receptor dimers drive distinct motility of the mouse skin fibroblast. : 異なった組み合わせの血小板由来増殖因子受容体2量体はマウス皮膚由来線維芽細胞においてそれぞれ異なった運動様式を誘導する.” 2016. Web. 21 Oct 2020.

Vancouver:

山田 . Different PDGF receptor dimers drive distinct motility of the mouse skin fibroblast. : 異なった組み合わせの血小板由来増殖因子受容体2量体はマウス皮膚由来線維芽細胞においてそれぞれ異なった運動様式を誘導する. [Internet] [Thesis]. University of Toyama / 富山大学; 2016. [cited 2020 Oct 21]. Available from: http://hdl.handle.net/10110/12967 ; http://dx.doi.org/10.15099/00005068.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山田 . Different PDGF receptor dimers drive distinct motility of the mouse skin fibroblast. : 異なった組み合わせの血小板由来増殖因子受容体2量体はマウス皮膚由来線維芽細胞においてそれぞれ異なった運動様式を誘導する. [Thesis]. University of Toyama / 富山大学; 2016. Available from: http://hdl.handle.net/10110/12967 ; http://dx.doi.org/10.15099/00005068

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Wieschhaus, Adam J. Dematin Regulates Calcium Mobilization and Signaling in Platelets.

Degree: 2013, University of Illinois – Chicago

 Dematin is a broadly expressed membrane cytoskeletal protein that has been well characterized in erythrocytes and to a lesser extent in non-erythroid cells. However, dematin’s… (more)

Subjects/Keywords: dematin; platelet; signaling; calcium; dense tubular system

…mediated signaling events are responsible for platelet shape change… …reorganizations required for platelet signaling (Fox, 1985, Fox et al… …extracellular medium to consolidate and supplement further platelet signaling… …signaling is regulated during platelet activation to provide sufficient… …improve our understanding of platelet signaling pathways and provide… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wieschhaus, A. J. (2013). Dematin Regulates Calcium Mobilization and Signaling in Platelets. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/10301

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wieschhaus, Adam J. “Dematin Regulates Calcium Mobilization and Signaling in Platelets.” 2013. Thesis, University of Illinois – Chicago. Accessed October 21, 2020. http://hdl.handle.net/10027/10301.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wieschhaus, Adam J. “Dematin Regulates Calcium Mobilization and Signaling in Platelets.” 2013. Web. 21 Oct 2020.

Vancouver:

Wieschhaus AJ. Dematin Regulates Calcium Mobilization and Signaling in Platelets. [Internet] [Thesis]. University of Illinois – Chicago; 2013. [cited 2020 Oct 21]. Available from: http://hdl.handle.net/10027/10301.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wieschhaus AJ. Dematin Regulates Calcium Mobilization and Signaling in Platelets. [Thesis]. University of Illinois – Chicago; 2013. Available from: http://hdl.handle.net/10027/10301

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Chen, Kan. Prothrombotic Platelet Signaling By the Scavenger Receptor CD36.

Degree: PhD, Cell Biology, 2009, Case Western Reserve University School of Graduate Studies

Platelet hyperactivity associated with hyperlipidemia may contribute to development of a pro-thrombotic state. We previously showed that oxidized LDL (oxLDL) formed in the setting of… (more)

Subjects/Keywords: Cellular Biology; CD36; JNK; Platelet; Signaling; Thrombosis; Atherosclerosis; OxLDL

…84 Figure 11-1 A schematic overview of platelet CD36 signaling outlined in this study… …101 IX Prothrombotic Platelet Signaling By the Scavenger Receptor CD36 Abstract By KAN… …x29; triggers intracellular signaling events which lead to platelet activation marked by… …signaling pathways activating platelets. Upon platelet activation, α granules are released and P… …autoinhibited state, becoming active Ganosine. 17 1.8 Platelet signaling As described in chapter… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, K. (2009). Prothrombotic Platelet Signaling By the Scavenger Receptor CD36. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1226608419

Chicago Manual of Style (16th Edition):

Chen, Kan. “Prothrombotic Platelet Signaling By the Scavenger Receptor CD36.” 2009. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed October 21, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1226608419.

MLA Handbook (7th Edition):

Chen, Kan. “Prothrombotic Platelet Signaling By the Scavenger Receptor CD36.” 2009. Web. 21 Oct 2020.

Vancouver:

Chen K. Prothrombotic Platelet Signaling By the Scavenger Receptor CD36. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2009. [cited 2020 Oct 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1226608419.

Council of Science Editors:

Chen K. Prothrombotic Platelet Signaling By the Scavenger Receptor CD36. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1226608419


University of Michigan

9. Rui, Liangyou. Identification of SH2-B as a signaling molecule for growth hormone, platelet-derived growth factor and nerve growth factor.

Degree: PhD, Pure Sciences, 1998, University of Michigan

 Polypeptide hormones and growth factors regulate a variety of vital activities via binding and activating their cognate cell surface receptors which in turn recruit intracellular… (more)

Subjects/Keywords: Growth Hormone; Identification; Molecule; Nerve Growth Factor; Platelet-derived Growth Factor; Sh2-b; Signaling

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rui, L. (1998). Identification of SH2-B as a signaling molecule for growth hormone, platelet-derived growth factor and nerve growth factor. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/131319

Chicago Manual of Style (16th Edition):

Rui, Liangyou. “Identification of SH2-B as a signaling molecule for growth hormone, platelet-derived growth factor and nerve growth factor.” 1998. Doctoral Dissertation, University of Michigan. Accessed October 21, 2020. http://hdl.handle.net/2027.42/131319.

MLA Handbook (7th Edition):

Rui, Liangyou. “Identification of SH2-B as a signaling molecule for growth hormone, platelet-derived growth factor and nerve growth factor.” 1998. Web. 21 Oct 2020.

Vancouver:

Rui L. Identification of SH2-B as a signaling molecule for growth hormone, platelet-derived growth factor and nerve growth factor. [Internet] [Doctoral dissertation]. University of Michigan; 1998. [cited 2020 Oct 21]. Available from: http://hdl.handle.net/2027.42/131319.

Council of Science Editors:

Rui L. Identification of SH2-B as a signaling molecule for growth hormone, platelet-derived growth factor and nerve growth factor. [Doctoral Dissertation]. University of Michigan; 1998. Available from: http://hdl.handle.net/2027.42/131319

10. Kruk, Jeffrey Stephen. Transactivation of platelet-derived growth factor receptor type β: Mechanisms and potential relevance in neurobiology.

Degree: 2013, University of Waterloo

 In the absence of ligand, certain growth factor receptors can be activated via G protein-coupled receptor (GPCR) activation in a process termed transactivation. Serotonin (5-HT)… (more)

Subjects/Keywords: serotonin; 5-HT; neurobiology; platelet-derived growth factor; transactivation; TrkB; cellular signaling

…receptor signaling 1.3.1 Platelet-derived growth factors Platelet-derived growth factors (… …43 3.1.5 5-HT signaling to PDGFβ receptors is calcium-dependent… …79 3.2.4 ROS is a signaling mediator in RTK transactivation in this system… …85 Chapter 4 Fluoxetine-induced transactivation of the platelet-derived growth factor type… …13 Figure 1.5. Triple-membrane-passing signaling… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kruk, J. S. (2013). Transactivation of platelet-derived growth factor receptor type β: Mechanisms and potential relevance in neurobiology. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/8104

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kruk, Jeffrey Stephen. “Transactivation of platelet-derived growth factor receptor type β: Mechanisms and potential relevance in neurobiology.” 2013. Thesis, University of Waterloo. Accessed October 21, 2020. http://hdl.handle.net/10012/8104.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kruk, Jeffrey Stephen. “Transactivation of platelet-derived growth factor receptor type β: Mechanisms and potential relevance in neurobiology.” 2013. Web. 21 Oct 2020.

Vancouver:

Kruk JS. Transactivation of platelet-derived growth factor receptor type β: Mechanisms and potential relevance in neurobiology. [Internet] [Thesis]. University of Waterloo; 2013. [cited 2020 Oct 21]. Available from: http://hdl.handle.net/10012/8104.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kruk JS. Transactivation of platelet-derived growth factor receptor type β: Mechanisms and potential relevance in neurobiology. [Thesis]. University of Waterloo; 2013. Available from: http://hdl.handle.net/10012/8104

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Akker, Nynke Margaretha Sophie van den. Endothelial plasticity in cardiovascular development: role of growth factors VEGF and PDGF.

Degree: 2008, Department of Anatomy and Embryology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University

 The central cell type within vascular development is the endothelial cell (EC). It forms during (lymph)vasculogenesis, proliferates during angiogenesis and instructs medial cells during arteriogenesis.… (more)

Subjects/Keywords: Coronary arteriogenesis; Down syndrome; Endothelial cell; Growth factor; Notch-signaling; Nuchal Translucency; Platelet-derived growth factor; Second heart field; Tetralogy of fallot; Turner syndrome; Vascular Endothelial growth factor; Coronary arteriogenesis; Down syndrome; Endothelial cell; Growth factor; Notch-signaling; Nuchal Translucency; Platelet-derived growth factor; Second heart field; Tetralogy of fallot; Turner syndrome; Vascular Endothelial growth factor

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Akker, N. M. S. v. d. (2008). Endothelial plasticity in cardiovascular development: role of growth factors VEGF and PDGF. (Doctoral Dissertation). Department of Anatomy and Embryology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University. Retrieved from http://hdl.handle.net/1887/12700

Chicago Manual of Style (16th Edition):

Akker, Nynke Margaretha Sophie van den. “Endothelial plasticity in cardiovascular development: role of growth factors VEGF and PDGF.” 2008. Doctoral Dissertation, Department of Anatomy and Embryology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University. Accessed October 21, 2020. http://hdl.handle.net/1887/12700.

MLA Handbook (7th Edition):

Akker, Nynke Margaretha Sophie van den. “Endothelial plasticity in cardiovascular development: role of growth factors VEGF and PDGF.” 2008. Web. 21 Oct 2020.

Vancouver:

Akker NMSvd. Endothelial plasticity in cardiovascular development: role of growth factors VEGF and PDGF. [Internet] [Doctoral dissertation]. Department of Anatomy and Embryology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University; 2008. [cited 2020 Oct 21]. Available from: http://hdl.handle.net/1887/12700.

Council of Science Editors:

Akker NMSvd. Endothelial plasticity in cardiovascular development: role of growth factors VEGF and PDGF. [Doctoral Dissertation]. Department of Anatomy and Embryology, Faculty of Medicine / Leiden University Medical Center (LUMC), Leiden University; 2008. Available from: http://hdl.handle.net/1887/12700

.