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You searched for subject:(Placental Transport). Showing records 1 – 5 of 5 total matches.

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University of Toronto

1. Pollex, Erika. Fetal Exposure to Antidiabetic Drugs: The Role of the Placenta.

Degree: 2010, University of Toronto

Gestational diabetes, a common medical complication in pregnancy, may lead to severe fetal consequences if left untreated. A major concern with the use of antidiabetic… (more)

Subjects/Keywords: gestational diabetes; placental transport; insulin glargine; glyburide; 0419; 0383

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APA (6th Edition):

Pollex, E. (2010). Fetal Exposure to Antidiabetic Drugs: The Role of the Placenta. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/24857

Chicago Manual of Style (16th Edition):

Pollex, Erika. “Fetal Exposure to Antidiabetic Drugs: The Role of the Placenta.” 2010. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/24857.

MLA Handbook (7th Edition):

Pollex, Erika. “Fetal Exposure to Antidiabetic Drugs: The Role of the Placenta.” 2010. Web. 15 Apr 2021.

Vancouver:

Pollex E. Fetal Exposure to Antidiabetic Drugs: The Role of the Placenta. [Internet] [Doctoral dissertation]. University of Toronto; 2010. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/24857.

Council of Science Editors:

Pollex E. Fetal Exposure to Antidiabetic Drugs: The Role of the Placenta. [Doctoral Dissertation]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24857


University of Michigan

2. Treinen, Kimberley Anne. Biochemical Toxicology Of Human Placental High Affinity Calcium-stimulated Atpase And Calcium Transport (uptake, Ddt, Dot).

Degree: PhD, Pure Sciences, 1986, University of Michigan

 ATPase activity which is stimulated by submicromolar concentrations of Ca('2+) was identified and characterized in human placental microvillous brush border membranes. The enzyme exhibited an… (more)

Subjects/Keywords: Affinity; Atpase; Biochemical; Calcium; Ddt; Dot; High; Human; Placental; Stimulated; Toxicology; Transport; Uptake

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APA (6th Edition):

Treinen, K. A. (1986). Biochemical Toxicology Of Human Placental High Affinity Calcium-stimulated Atpase And Calcium Transport (uptake, Ddt, Dot). (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/127934

Chicago Manual of Style (16th Edition):

Treinen, Kimberley Anne. “Biochemical Toxicology Of Human Placental High Affinity Calcium-stimulated Atpase And Calcium Transport (uptake, Ddt, Dot).” 1986. Doctoral Dissertation, University of Michigan. Accessed April 15, 2021. http://hdl.handle.net/2027.42/127934.

MLA Handbook (7th Edition):

Treinen, Kimberley Anne. “Biochemical Toxicology Of Human Placental High Affinity Calcium-stimulated Atpase And Calcium Transport (uptake, Ddt, Dot).” 1986. Web. 15 Apr 2021.

Vancouver:

Treinen KA. Biochemical Toxicology Of Human Placental High Affinity Calcium-stimulated Atpase And Calcium Transport (uptake, Ddt, Dot). [Internet] [Doctoral dissertation]. University of Michigan; 1986. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/2027.42/127934.

Council of Science Editors:

Treinen KA. Biochemical Toxicology Of Human Placental High Affinity Calcium-stimulated Atpase And Calcium Transport (uptake, Ddt, Dot). [Doctoral Dissertation]. University of Michigan; 1986. Available from: http://hdl.handle.net/2027.42/127934


University of Washington

3. Dorfman, Elizabeth Howard. Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure.

Degree: PhD, 2015, University of Washington

 This dissertation is principally comprised of three distinct but related projects. The first is an assessment of the quantity and nature of obstetric pharmacology clinical… (more)

Subjects/Keywords: ABCG2; breast cancer resistance protein; fetal drug exposure; glyburide; obstetric pharmacology; placental drug transport; Genetics; Pharmacology; Obstetrics and gynecology; public health genetics

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APA (6th Edition):

Dorfman, E. H. (2015). Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/34161

Chicago Manual of Style (16th Edition):

Dorfman, Elizabeth Howard. “Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure.” 2015. Doctoral Dissertation, University of Washington. Accessed April 15, 2021. http://hdl.handle.net/1773/34161.

MLA Handbook (7th Edition):

Dorfman, Elizabeth Howard. “Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure.” 2015. Web. 15 Apr 2021.

Vancouver:

Dorfman EH. Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure. [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1773/34161.

Council of Science Editors:

Dorfman EH. Translational Research in Obstetric Pharmacology: Historical Trends, Prenatal Pharmacogenomics, and an Opportunistic Study of Placental ABCG2 and Fetal Glyburide Exposure. [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/34161


University of Melbourne

4. Aitken, Elizabeth Helen. Pregnancy-malaria: immunity and a mechanism for low birth weight.

Degree: 2010, University of Melbourne

 Introduction: Pregnant women are more susceptible to Plasmodium falciparum malaria than their non-pregnant peers and the consequences for the mother and child can include maternal… (more)

Subjects/Keywords: malaria; pregnancy; immunity; low birth weight; antibody; IPTp; system-A placental transport

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APA (6th Edition):

Aitken, E. H. (2010). Pregnancy-malaria: immunity and a mechanism for low birth weight. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/36057

Chicago Manual of Style (16th Edition):

Aitken, Elizabeth Helen. “Pregnancy-malaria: immunity and a mechanism for low birth weight.” 2010. Doctoral Dissertation, University of Melbourne. Accessed April 15, 2021. http://hdl.handle.net/11343/36057.

MLA Handbook (7th Edition):

Aitken, Elizabeth Helen. “Pregnancy-malaria: immunity and a mechanism for low birth weight.” 2010. Web. 15 Apr 2021.

Vancouver:

Aitken EH. Pregnancy-malaria: immunity and a mechanism for low birth weight. [Internet] [Doctoral dissertation]. University of Melbourne; 2010. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/11343/36057.

Council of Science Editors:

Aitken EH. Pregnancy-malaria: immunity and a mechanism for low birth weight. [Doctoral Dissertation]. University of Melbourne; 2010. Available from: http://hdl.handle.net/11343/36057


University of Cincinnati

5. SCHMID, KARA E. ENDOGENOUS AND EXOGENOUS SOURCES OF CHOLESTEROL DURING FETAL DEVELOPMENT.

Degree: PhD, Medicine : Pathobiology and Molecular Medicine, 2003, University of Cincinnati

 Cholesterol is essential for proper fetal development. Defects in fetal cholesterol metabolism can cause developmental abnormalities, including mental retardation and stunted growth as demonstrated by… (more)

Subjects/Keywords: Biophysics, Medical; fetal cholesterol metabolism; placental transport; cholesterol synthesis; Smith-Lemli-Optiz

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

SCHMID, K. E. (2003). ENDOGENOUS AND EXOGENOUS SOURCES OF CHOLESTEROL DURING FETAL DEVELOPMENT. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1061304521

Chicago Manual of Style (16th Edition):

SCHMID, KARA E. “ENDOGENOUS AND EXOGENOUS SOURCES OF CHOLESTEROL DURING FETAL DEVELOPMENT.” 2003. Doctoral Dissertation, University of Cincinnati. Accessed April 15, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1061304521.

MLA Handbook (7th Edition):

SCHMID, KARA E. “ENDOGENOUS AND EXOGENOUS SOURCES OF CHOLESTEROL DURING FETAL DEVELOPMENT.” 2003. Web. 15 Apr 2021.

Vancouver:

SCHMID KE. ENDOGENOUS AND EXOGENOUS SOURCES OF CHOLESTEROL DURING FETAL DEVELOPMENT. [Internet] [Doctoral dissertation]. University of Cincinnati; 2003. [cited 2021 Apr 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1061304521.

Council of Science Editors:

SCHMID KE. ENDOGENOUS AND EXOGENOUS SOURCES OF CHOLESTEROL DURING FETAL DEVELOPMENT. [Doctoral Dissertation]. University of Cincinnati; 2003. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1061304521

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