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You searched for subject:(Phosphatidylinositol 3 Kinases). Showing records 1 – 12 of 12 total matches.

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1. Mujalli, Abdulrahman. Phosphoinositides in blood platelet : mapping of molecular species and evidence for a new localization and role of PI3P : Phosphoinositides plaquettaires : cartographie d'espèces moléculaires et mise en évidence d'une nouvelle localisation et d'un nouvau rôle du PI3P.

Degree: Docteur es, Physiopathologie, 2018, Université Toulouse III – Paul Sabatier

 Les phosphoinositides (PIs) sont des phospholipides membranaires qui jouent un rôle crucial dans le contrôle de l'organisation spatio-temporelle de nombreuses voies de signalisation intracellulaire, du… (more)

Subjects/Keywords: Phosphoinositides; PI-kinases; Espèces moléculaires; Phosphatidylinositol 3 monophosphate

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APA (6th Edition):

Mujalli, A. (2018). Phosphoinositides in blood platelet : mapping of molecular species and evidence for a new localization and role of PI3P : Phosphoinositides plaquettaires : cartographie d'espèces moléculaires et mise en évidence d'une nouvelle localisation et d'un nouvau rôle du PI3P. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2018TOU30110

Chicago Manual of Style (16th Edition):

Mujalli, Abdulrahman. “Phosphoinositides in blood platelet : mapping of molecular species and evidence for a new localization and role of PI3P : Phosphoinositides plaquettaires : cartographie d'espèces moléculaires et mise en évidence d'une nouvelle localisation et d'un nouvau rôle du PI3P.” 2018. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed August 09, 2020. http://www.theses.fr/2018TOU30110.

MLA Handbook (7th Edition):

Mujalli, Abdulrahman. “Phosphoinositides in blood platelet : mapping of molecular species and evidence for a new localization and role of PI3P : Phosphoinositides plaquettaires : cartographie d'espèces moléculaires et mise en évidence d'une nouvelle localisation et d'un nouvau rôle du PI3P.” 2018. Web. 09 Aug 2020.

Vancouver:

Mujalli A. Phosphoinositides in blood platelet : mapping of molecular species and evidence for a new localization and role of PI3P : Phosphoinositides plaquettaires : cartographie d'espèces moléculaires et mise en évidence d'une nouvelle localisation et d'un nouvau rôle du PI3P. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2018. [cited 2020 Aug 09]. Available from: http://www.theses.fr/2018TOU30110.

Council of Science Editors:

Mujalli A. Phosphoinositides in blood platelet : mapping of molecular species and evidence for a new localization and role of PI3P : Phosphoinositides plaquettaires : cartographie d'espèces moléculaires et mise en évidence d'une nouvelle localisation et d'un nouvau rôle du PI3P. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2018. Available from: http://www.theses.fr/2018TOU30110


University of Texas Southwestern Medical Center

2. DeSevo, Christopher Gerard. MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas.

Degree: 2013, University of Texas Southwestern Medical Center

Phosphatidylinositol 3-kinases (PI3Ks) are enzymes involved in diverse cellular functions including cell growth, proliferation, differentiation, motility, survival and apoptosis. Many of these functions relate to… (more)

Subjects/Keywords: Carcinoma, Non-Small-Cell Lung; MicroRNAs; Phosphatidylinositol 3-Kinases

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APA (6th Edition):

DeSevo, C. G. (2013). MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/2717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DeSevo, Christopher Gerard. “MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed August 09, 2020. http://hdl.handle.net/2152.5/2717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DeSevo, Christopher Gerard. “MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas.” 2013. Web. 09 Aug 2020.

Vancouver:

DeSevo CG. MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/2152.5/2717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DeSevo CG. MiR-10a Regulation of Drug Response and Cancer Stem Cell Populations in Non-Small Cell Lung Carcinomas. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/2717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

3. Ahmed, Kamran. Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury.

Degree: 2016, University of Texas Southwestern Medical Center

Note: The general metadata  – e.g., title, author, abstract, subject headings, etc.  – is publicly available, but access to the submitted files is restricted to… (more)

Subjects/Keywords: Heart; Insulin-Like Growth Factor I; Myocardial Reperfusion Injury; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; rho-Associated Kinases

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APA (6th Edition):

Ahmed, K. (2016). Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ahmed, Kamran. “Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed August 09, 2020. http://hdl.handle.net/2152.5/7069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ahmed, Kamran. “Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury.” 2016. Web. 09 Aug 2020.

Vancouver:

Ahmed K. Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/2152.5/7069.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ahmed K. Proteomic Discovery of Functionally Important Pathways in Myocardial Ischemia-Reperfusion Injury. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/7069

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Rajurkar, Mihir S. GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation.

Degree: Cancer Biology, Molecular, Cell and Cancer Biology Department, 2014, U of Massachusetts : Med

  Pancreatic ductal adenocarcinoma (PDAC), one of the most aggressive human malignancies, is thought to be initiated by KRAS activation. Here, we find that transcriptional… (more)

Subjects/Keywords: Pancreatic Ductal Carcinoma; I-kappa B Kinase; Pancreatic Neoplasms; Phosphatidylinositol 3-Kinases; Transcription Factors; Carcinogenesis; Transcriptional Activation; Cancer Biology; Neoplasms

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APA (6th Edition):

Rajurkar, M. S. (2014). GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/753

Chicago Manual of Style (16th Edition):

Rajurkar, Mihir S. “GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation.” 2014. Doctoral Dissertation, U of Massachusetts : Med. Accessed August 09, 2020. http://escholarship.umassmed.edu/gsbs_diss/753.

MLA Handbook (7th Edition):

Rajurkar, Mihir S. “GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation.” 2014. Web. 09 Aug 2020.

Vancouver:

Rajurkar MS. GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2014. [cited 2020 Aug 09]. Available from: http://escholarship.umassmed.edu/gsbs_diss/753.

Council of Science Editors:

Rajurkar MS. GLI-IKBKE Requirement In KRAS-Induced Pancreatic Tumorigenesis: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2014. Available from: http://escholarship.umassmed.edu/gsbs_diss/753


University of Texas Southwestern Medical Center

5. Troutman, Ty Dale. B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily.

Degree: 2014, University of Texas Southwestern Medical Center

 Toll-like receptor (TLR)/Interleukin-1 receptor (IL1R) superfamily members share signaling components and (with the exception of TLR3) depend on the adapter myeloid differentiation primary response gene… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing; B-Lymphocytes; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Signal Transduction; Toll-Like Receptors

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APA (6th Edition):

Troutman, T. D. (2014). B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Troutman, Ty Dale. “B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed August 09, 2020. http://hdl.handle.net/2152.5/3328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Troutman, Ty Dale. “B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily.” 2014. Web. 09 Aug 2020.

Vancouver:

Troutman TD. B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/2152.5/3328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Troutman TD. B-Cell Adapter for Phosphoinositide 3-Kinase Is a Signaling Adapter in the Toll-Like Receptor/Interleukin-1 Receptor Superfamily. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

6. Zhang, Wei. Study of the Mechanisms Underlying Hippocampal Neuron Synaptogenesis: The Roles of Neurotrophin Signaling and MicroRNAs.

Degree: 2010, University of Texas Southwestern Medical Center

 Synapse formation requires contacts between dendrites and axons. Although this process is often viewed as axon mediated, dendritic filopodia may be actively involved in mediating… (more)

Subjects/Keywords: Hippocampus; Phosphatidylinositol 3-Kinases; Synaptic Transmission; Gene Expression

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APA (6th Edition):

Zhang, W. (2010). Study of the Mechanisms Underlying Hippocampal Neuron Synaptogenesis: The Roles of Neurotrophin Signaling and MicroRNAs. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/824

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Wei. “Study of the Mechanisms Underlying Hippocampal Neuron Synaptogenesis: The Roles of Neurotrophin Signaling and MicroRNAs.” 2010. Thesis, University of Texas Southwestern Medical Center. Accessed August 09, 2020. http://hdl.handle.net/2152.5/824.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Wei. “Study of the Mechanisms Underlying Hippocampal Neuron Synaptogenesis: The Roles of Neurotrophin Signaling and MicroRNAs.” 2010. Web. 09 Aug 2020.

Vancouver:

Zhang W. Study of the Mechanisms Underlying Hippocampal Neuron Synaptogenesis: The Roles of Neurotrophin Signaling and MicroRNAs. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2010. [cited 2020 Aug 09]. Available from: http://hdl.handle.net/2152.5/824.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang W. Study of the Mechanisms Underlying Hippocampal Neuron Synaptogenesis: The Roles of Neurotrophin Signaling and MicroRNAs. [Thesis]. University of Texas Southwestern Medical Center; 2010. Available from: http://hdl.handle.net/2152.5/824

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Gavin, Cristin. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.

Degree: PhD, 2012, University of Alabama – Birmingham

Dynamic changes to the actin cytoskeleton are required for synaptic plasticity and long-term memory formation. However, the molecular mechanisms that mediate filamentous actin (F-actin) dynamics… (more)

Subjects/Keywords: Bone Neoplasms – secondary<; br>; Breast Neoplasms – pathology<; br>; Cell Communication<; br>; Cyclic AMP-Dependent Protein Kinases – metabolism.<; br>; Gap Junctions – metabolism.<; br>; Osteoblasts – cytology.<; br>; Phosphatidylinositol 3-Kinases – metabolism.

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APA (6th Edition):

Gavin, C. (2012). Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1396

Chicago Manual of Style (16th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1396.

MLA Handbook (7th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Web. 09 Aug 2020.

Vancouver:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Aug 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396.

Council of Science Editors:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396

8. Hobson, Susan A. The role of the calcium-sensing receptor in calcium-sensitive proliferation and intracellular signaling.

Degree: PhD, 1999, Oregon Health Sciences University

Subjects/Keywords: Receptors, Cell Surface; Calcium  – physiology; Signal Transduction; src-Family Kinases; Cell Differentiation; Cell Division; Phosphatidylinositol 3-Kinases

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APA (6th Edition):

Hobson, S. A. (1999). The role of the calcium-sensing receptor in calcium-sensitive proliferation and intracellular signaling. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4X065CG ; http://digitalcommons.ohsu.edu/etd/3384

Chicago Manual of Style (16th Edition):

Hobson, Susan A. “The role of the calcium-sensing receptor in calcium-sensitive proliferation and intracellular signaling.” 1999. Doctoral Dissertation, Oregon Health Sciences University. Accessed August 09, 2020. doi:10.6083/M4X065CG ; http://digitalcommons.ohsu.edu/etd/3384.

MLA Handbook (7th Edition):

Hobson, Susan A. “The role of the calcium-sensing receptor in calcium-sensitive proliferation and intracellular signaling.” 1999. Web. 09 Aug 2020.

Vancouver:

Hobson SA. The role of the calcium-sensing receptor in calcium-sensitive proliferation and intracellular signaling. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 1999. [cited 2020 Aug 09]. Available from: doi:10.6083/M4X065CG ; http://digitalcommons.ohsu.edu/etd/3384.

Council of Science Editors:

Hobson SA. The role of the calcium-sensing receptor in calcium-sensitive proliferation and intracellular signaling. [Doctoral Dissertation]. Oregon Health Sciences University; 1999. Available from: doi:10.6083/M4X065CG ; http://digitalcommons.ohsu.edu/etd/3384

9. Sundgren, Nathan Cole. Extracellular signal-regulated kinase and growth factor regulation of the fetal ovine cardiac myocyte.

Degree: PhD, 2003, Oregon Health Sciences University

Subjects/Keywords: Myocytes, Cardiac; Insulin-Like Growth Factor I; Angiotensin II; Mitogen-Activated Protein Kinases; Phosphatidylinositol 3-Kinases

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APA (6th Edition):

Sundgren, N. C. (2003). Extracellular signal-regulated kinase and growth factor regulation of the fetal ovine cardiac myocyte. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M4Z60MBH ; http://digitalcommons.ohsu.edu/etd/3135

Chicago Manual of Style (16th Edition):

Sundgren, Nathan Cole. “Extracellular signal-regulated kinase and growth factor regulation of the fetal ovine cardiac myocyte.” 2003. Doctoral Dissertation, Oregon Health Sciences University. Accessed August 09, 2020. doi:10.6083/M4Z60MBH ; http://digitalcommons.ohsu.edu/etd/3135.

MLA Handbook (7th Edition):

Sundgren, Nathan Cole. “Extracellular signal-regulated kinase and growth factor regulation of the fetal ovine cardiac myocyte.” 2003. Web. 09 Aug 2020.

Vancouver:

Sundgren NC. Extracellular signal-regulated kinase and growth factor regulation of the fetal ovine cardiac myocyte. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2003. [cited 2020 Aug 09]. Available from: doi:10.6083/M4Z60MBH ; http://digitalcommons.ohsu.edu/etd/3135.

Council of Science Editors:

Sundgren NC. Extracellular signal-regulated kinase and growth factor regulation of the fetal ovine cardiac myocyte. [Doctoral Dissertation]. Oregon Health Sciences University; 2003. Available from: doi:10.6083/M4Z60MBH ; http://digitalcommons.ohsu.edu/etd/3135


Royal Holloway, University of London

10. Kelly, Elizabeth. Identifying the mechanism of action of valproic acid on phosphoinositide signalling.

Degree: PhD, 2018, Royal Holloway, University of London

 A third of epilepsy patients are resistant to currently available medication, highlighting a need to identify new antiepileptic drugs. To identify new drugs, researchers often… (more)

Subjects/Keywords: Dictyostelium discoideum; Epilepsy; Bipolar Disorder; Valproic acid; Lithium; Valpromide; decanoic acid; Octanoic acid; 4-ethyloctanoic acid; 2-methylhexanoic acid; Phosphoinositol salvage pathway; Diacylglycerol; Diacylglycerol kinase; Cytidine-diphosphate-diacylglycerol-inositol-3-phosphatidyltransferase; Cytidine diphosphate-diacylglycerol synthase; Cytidine diphosphate-diacylglycerol; Phosphatidic acid; PHOSPHOLIPASE-C; phospholipids; phosphatidylinositol 4,5-bisphosphate; Phosphatidylinositol 3-Kinases; phosphatidylinositol 3,4,5-trisphosphate; phosphatase and tensin homolog; Phosphatidate phosphatase LPIN2; Elizabeth Kelly; Royal Holloway

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APA (6th Edition):

Kelly, E. (2018). Identifying the mechanism of action of valproic acid on phosphoinositide signalling. (Doctoral Dissertation). Royal Holloway, University of London. Retrieved from https://pure.royalholloway.ac.uk/portal/en/publications/identifying-the-mechanism-of-action-of-valproic-acid-on-phosphoinositide-signalling(0c887dd0-8a51-4b0d-a783-42b77e241199).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.792868

Chicago Manual of Style (16th Edition):

Kelly, Elizabeth. “Identifying the mechanism of action of valproic acid on phosphoinositide signalling.” 2018. Doctoral Dissertation, Royal Holloway, University of London. Accessed August 09, 2020. https://pure.royalholloway.ac.uk/portal/en/publications/identifying-the-mechanism-of-action-of-valproic-acid-on-phosphoinositide-signalling(0c887dd0-8a51-4b0d-a783-42b77e241199).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.792868.

MLA Handbook (7th Edition):

Kelly, Elizabeth. “Identifying the mechanism of action of valproic acid on phosphoinositide signalling.” 2018. Web. 09 Aug 2020.

Vancouver:

Kelly E. Identifying the mechanism of action of valproic acid on phosphoinositide signalling. [Internet] [Doctoral dissertation]. Royal Holloway, University of London; 2018. [cited 2020 Aug 09]. Available from: https://pure.royalholloway.ac.uk/portal/en/publications/identifying-the-mechanism-of-action-of-valproic-acid-on-phosphoinositide-signalling(0c887dd0-8a51-4b0d-a783-42b77e241199).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.792868.

Council of Science Editors:

Kelly E. Identifying the mechanism of action of valproic acid on phosphoinositide signalling. [Doctoral Dissertation]. Royal Holloway, University of London; 2018. Available from: https://pure.royalholloway.ac.uk/portal/en/publications/identifying-the-mechanism-of-action-of-valproic-acid-on-phosphoinositide-signalling(0c887dd0-8a51-4b0d-a783-42b77e241199).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.792868

11. Malyala, Anna. Hypothalamic gene expression and signal transduction effects of estrogen.

Degree: PhD, 2007, Oregon Health Sciences University

Subjects/Keywords: Hypothalamus  – physiology; Gene Expression Regulation; Receptors, Estrogen  – genetics; Phosphatidylinositol 3-Kinases; Estrogens  – physiology; Signal Transduction

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APA (6th Edition):

Malyala, A. (2007). Hypothalamic gene expression and signal transduction effects of estrogen. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M46M34TF ; http://digitalcommons.ohsu.edu/etd/811

Chicago Manual of Style (16th Edition):

Malyala, Anna. “Hypothalamic gene expression and signal transduction effects of estrogen.” 2007. Doctoral Dissertation, Oregon Health Sciences University. Accessed August 09, 2020. doi:10.6083/M46M34TF ; http://digitalcommons.ohsu.edu/etd/811.

MLA Handbook (7th Edition):

Malyala, Anna. “Hypothalamic gene expression and signal transduction effects of estrogen.” 2007. Web. 09 Aug 2020.

Vancouver:

Malyala A. Hypothalamic gene expression and signal transduction effects of estrogen. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2007. [cited 2020 Aug 09]. Available from: doi:10.6083/M46M34TF ; http://digitalcommons.ohsu.edu/etd/811.

Council of Science Editors:

Malyala A. Hypothalamic gene expression and signal transduction effects of estrogen. [Doctoral Dissertation]. Oregon Health Sciences University; 2007. Available from: doi:10.6083/M46M34TF ; http://digitalcommons.ohsu.edu/etd/811

12. Perez, Jessica. Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor.

Degree: PhD, 2010, University of Alabama – Birmingham

Growth factors are important inducers of vascular cell growth whose regulation is altered during the pathogenesis of atherosclerosis. An increase in growth factor and cytokine… (more)

Subjects/Keywords: Cell Proliferation  – drug effects<; br>; Glucose  – metabolism<; br>; Muscle, Smooth, Vascular  – cytology<; br>; Myocytes, Smooth Muscle  – cytology<; br>; Myocytes, Smooth Muscle  – drug effects<; br>; Phosphatidylinositol 3-Kinases  – antagonists & inhibitors<; br>; Platelet-Derived Growth Factor  – pharmacology<; br>

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APA (6th Edition):

Perez, J. (2010). Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,928

Chicago Manual of Style (16th Edition):

Perez, Jessica. “Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed August 09, 2020. http://contentdm.mhsl.uab.edu/u?/etd,928.

MLA Handbook (7th Edition):

Perez, Jessica. “Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor.” 2010. Web. 09 Aug 2020.

Vancouver:

Perez J. Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Aug 09]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,928.

Council of Science Editors:

Perez J. Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,928

.