Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Phosphatidic acids). Showing records 1 – 4 of 4 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters


Georgia Tech

1. Urs, Aarti N. Reciprocal binding of sphingosine and phosphatidic acid to steroidogenic factor 1 regulates the transcription of CYP17.

Degree: MS, Biology, 2005, Georgia Tech

 Steroidogenic factor (SF1) is an orphan nuclear receptor that is essential for steroid hormone-biosynthesis and endocrine development. Recent studies have demonstrated that phospholipids are ligands… (more)

Subjects/Keywords: CYP17; Sphingosine; Phosphatidic acids; Steroidogenic factor 1; Transcription factors; Steroid hormones Synthesis; Sphingosine; Pregnenolone; Phospholipids; Ligands

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Urs, A. N. (2005). Reciprocal binding of sphingosine and phosphatidic acid to steroidogenic factor 1 regulates the transcription of CYP17. (Masters Thesis). Georgia Tech. Retrieved from http://hdl.handle.net/1853/7638

Chicago Manual of Style (16th Edition):

Urs, Aarti N. “Reciprocal binding of sphingosine and phosphatidic acid to steroidogenic factor 1 regulates the transcription of CYP17.” 2005. Masters Thesis, Georgia Tech. Accessed February 28, 2021. http://hdl.handle.net/1853/7638.

MLA Handbook (7th Edition):

Urs, Aarti N. “Reciprocal binding of sphingosine and phosphatidic acid to steroidogenic factor 1 regulates the transcription of CYP17.” 2005. Web. 28 Feb 2021.

Vancouver:

Urs AN. Reciprocal binding of sphingosine and phosphatidic acid to steroidogenic factor 1 regulates the transcription of CYP17. [Internet] [Masters thesis]. Georgia Tech; 2005. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1853/7638.

Council of Science Editors:

Urs AN. Reciprocal binding of sphingosine and phosphatidic acid to steroidogenic factor 1 regulates the transcription of CYP17. [Masters Thesis]. Georgia Tech; 2005. Available from: http://hdl.handle.net/1853/7638


Michigan State University

2. Lu, Binbin. Functional analysis of a phosphatidic acid transport system in Arabidopsis thaliana.

Degree: PhD, Department of Biochemistry and Molecular Biology, 2009, Michigan State University

Subjects/Keywords: Phosphatidic acids – Physiological transport; Arabidopsis thaliana – Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lu, B. (2009). Functional analysis of a phosphatidic acid transport system in Arabidopsis thaliana. (Doctoral Dissertation). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:17024

Chicago Manual of Style (16th Edition):

Lu, Binbin. “Functional analysis of a phosphatidic acid transport system in Arabidopsis thaliana.” 2009. Doctoral Dissertation, Michigan State University. Accessed February 28, 2021. http://etd.lib.msu.edu/islandora/object/etd:17024.

MLA Handbook (7th Edition):

Lu, Binbin. “Functional analysis of a phosphatidic acid transport system in Arabidopsis thaliana.” 2009. Web. 28 Feb 2021.

Vancouver:

Lu B. Functional analysis of a phosphatidic acid transport system in Arabidopsis thaliana. [Internet] [Doctoral dissertation]. Michigan State University; 2009. [cited 2021 Feb 28]. Available from: http://etd.lib.msu.edu/islandora/object/etd:17024.

Council of Science Editors:

Lu B. Functional analysis of a phosphatidic acid transport system in Arabidopsis thaliana. [Doctoral Dissertation]. Michigan State University; 2009. Available from: http://etd.lib.msu.edu/islandora/object/etd:17024

3. 山本, 梓司. Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.

Degree: 博士(医学), 2018, Saitama Medical University / 埼玉医科大学

Multiple sclerosis is a neuroinflammatory demyelinating and neurodegenerative disease of the central nervous system characterized by recurrent and progressive demyelination/remyelination cycles, neuroinflammation, oligodendrocyte loss, demyelination,… (more)

Subjects/Keywords: Animals; Anti-Inflammatory Agents; Apoptosis; Cell Differentiation; Cell Line, Transformed; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Female; Gene Expression Regulation; Humans; MAP Kinase Signaling System; Male; Mice; Mice, Inbred C57BL; Monoamine Oxidase Inhibitors; Myelin Sheath; NLR Family, Pyrin Domain-Containing 3 Protein; Phosphatidic Acids; Proto-Oncogene Proteins c-bcl-2; p38 Mitogen-Activated Protein Kinases

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

山本, . (2018). Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. (Thesis). Saitama Medical University / 埼玉医科大学. Retrieved from http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Thesis, Saitama Medical University / 埼玉医科大学. Accessed February 28, 2021. http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Web. 28 Feb 2021.

Vancouver:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Internet] [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. [cited 2021 Feb 28]. Available from: http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. Available from: http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Tsukahara, Ryoko. Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents.

Degree: PhD, Biomedical Sciences, 2011, University of Tennessee Health Science Center

  Lysophosphatidic acid (LPA) and its ether analog alkyl glycerophosphate (AGP) elicit arterial wall remodeling when applied intralumenally into the uninjured carotid artery. LPA is… (more)

Subjects/Keywords: lysophosphatidic acid; cyclic phosphatidic acid; phospholipase D; neointima; atherosclerosis; PPAR!; Amino Acids, Peptides, and Proteins; Cardiovascular Diseases; Chemicals and Drugs; Diseases; Lipids; Medical Sciences; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tsukahara, R. (2011). Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/270

Chicago Manual of Style (16th Edition):

Tsukahara, Ryoko. “Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents.” 2011. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed February 28, 2021. https://dc.uthsc.edu/dissertations/270.

MLA Handbook (7th Edition):

Tsukahara, Ryoko. “Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents.” 2011. Web. 28 Feb 2021.

Vancouver:

Tsukahara R. Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2011. [cited 2021 Feb 28]. Available from: https://dc.uthsc.edu/dissertations/270.

Council of Science Editors:

Tsukahara R. Characterization of the Mechanism of PPARγ-Mediated Neointima Formation in Rodents. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2011. Available from: https://dc.uthsc.edu/dissertations/270

.