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You searched for subject:(Phosducin like protein 1). Showing records 1 – 30 of 46134 total matches.

[1] [2] [3] [4] [5] … [1538]

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Brigham Young University

1. Lai, Chun Wan Jeffrey. Mechanism of G Protein Beta-Gamma Assembly Mediated by Phosducin-Like Protein 1.

Degree: PhD, 2011, Brigham Young University

  G-protein coupled receptor signaling (GPCR) is essential for regulating a large variety of hormonal, sensory and neuronal processes in eukaryotic cells. Because the regulation… (more)

Subjects/Keywords: GPCR; Heterotrimeric G proteins; G protein beta-gamma assembly; Phosducin-like protein 1; Biochemistry; Chemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lai, C. W. J. (2011). Mechanism of G Protein Beta-Gamma Assembly Mediated by Phosducin-Like Protein 1. (Doctoral Dissertation). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4189&context=etd

Chicago Manual of Style (16th Edition):

Lai, Chun Wan Jeffrey. “Mechanism of G Protein Beta-Gamma Assembly Mediated by Phosducin-Like Protein 1.” 2011. Doctoral Dissertation, Brigham Young University. Accessed November 15, 2019. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4189&context=etd.

MLA Handbook (7th Edition):

Lai, Chun Wan Jeffrey. “Mechanism of G Protein Beta-Gamma Assembly Mediated by Phosducin-Like Protein 1.” 2011. Web. 15 Nov 2019.

Vancouver:

Lai CWJ. Mechanism of G Protein Beta-Gamma Assembly Mediated by Phosducin-Like Protein 1. [Internet] [Doctoral dissertation]. Brigham Young University; 2011. [cited 2019 Nov 15]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4189&context=etd.

Council of Science Editors:

Lai CWJ. Mechanism of G Protein Beta-Gamma Assembly Mediated by Phosducin-Like Protein 1. [Doctoral Dissertation]. Brigham Young University; 2011. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4189&context=etd


Brigham Young University

2. Howlett, Alyson Cerny. Role of molecular chaperones in G protein B5-Regulator of G protein signaling dimer assembly and G protein By dimer specificity.

Degree: PhD, 2009, Brigham Young University

  In order for G protein signaling to occur, the G protein heterotrimer must be assembled from its nascent polypeptides. The most difficult step in… (more)

Subjects/Keywords: Phosducin-like protein 1; PhLP1; chaperone; chaperonin; CCT; ; 5; ; γ; RGS; Regulator of G protein signaling; G protein assembly; Phosducin-like protein 2; PhLP2; Biochemistry; Chemistry

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APA (6th Edition):

Howlett, A. C. (2009). Role of molecular chaperones in G protein B5-Regulator of G protein signaling dimer assembly and G protein By dimer specificity. (Doctoral Dissertation). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=3064&context=etd

Chicago Manual of Style (16th Edition):

Howlett, Alyson Cerny. “Role of molecular chaperones in G protein B5-Regulator of G protein signaling dimer assembly and G protein By dimer specificity.” 2009. Doctoral Dissertation, Brigham Young University. Accessed November 15, 2019. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=3064&context=etd.

MLA Handbook (7th Edition):

Howlett, Alyson Cerny. “Role of molecular chaperones in G protein B5-Regulator of G protein signaling dimer assembly and G protein By dimer specificity.” 2009. Web. 15 Nov 2019.

Vancouver:

Howlett AC. Role of molecular chaperones in G protein B5-Regulator of G protein signaling dimer assembly and G protein By dimer specificity. [Internet] [Doctoral dissertation]. Brigham Young University; 2009. [cited 2019 Nov 15]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=3064&context=etd.

Council of Science Editors:

Howlett AC. Role of molecular chaperones in G protein B5-Regulator of G protein signaling dimer assembly and G protein By dimer specificity. [Doctoral Dissertation]. Brigham Young University; 2009. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=3064&context=etd


Brigham Young University

3. Gray, Amy Jetaun. Novel Phosducin-Like Protein Binding Partners: Exploring Chaperone and Tumor Suppressor Protein Interactions.

Degree: PhD, 2012, Brigham Young University

 Many proteins cannot fold into their native state without the assistance of one or more molecular chaperones. Chaperonins are an essential class of chaperones that… (more)

Subjects/Keywords: Chaperonin; chaperone; G-protein signaling; phosducin-like protein; PDCD5; Biochemistry; Chemistry

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APA (6th Edition):

Gray, A. J. (2012). Novel Phosducin-Like Protein Binding Partners: Exploring Chaperone and Tumor Suppressor Protein Interactions. (Doctoral Dissertation). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4407&context=etd

Chicago Manual of Style (16th Edition):

Gray, Amy Jetaun. “Novel Phosducin-Like Protein Binding Partners: Exploring Chaperone and Tumor Suppressor Protein Interactions.” 2012. Doctoral Dissertation, Brigham Young University. Accessed November 15, 2019. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4407&context=etd.

MLA Handbook (7th Edition):

Gray, Amy Jetaun. “Novel Phosducin-Like Protein Binding Partners: Exploring Chaperone and Tumor Suppressor Protein Interactions.” 2012. Web. 15 Nov 2019.

Vancouver:

Gray AJ. Novel Phosducin-Like Protein Binding Partners: Exploring Chaperone and Tumor Suppressor Protein Interactions. [Internet] [Doctoral dissertation]. Brigham Young University; 2012. [cited 2019 Nov 15]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4407&context=etd.

Council of Science Editors:

Gray AJ. Novel Phosducin-Like Protein Binding Partners: Exploring Chaperone and Tumor Suppressor Protein Interactions. [Doctoral Dissertation]. Brigham Young University; 2012. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4407&context=etd


Brigham Young University

4. Baker, Christine M. The Mechanism of Assembly of the G-Protein Beta Gamma Subunit Dimer by CK2 Phosphorylated Phosducin-Like Protein and the Chaperonin Containing TCP-1.

Degree: MS, 2006, Brigham Young University

Phosducin-like protein (PhLP) binds G-protein beta gamma subunits and is thought to assist in assembly of the G-protein beta gamma dimer. Phosphorylation of PhLP at… (more)

Subjects/Keywords: G-protein; signal transduction; PhLP; Phosducin-like protein; CCT; chaperonin containing TCP-1; CK2 phosphorylation; protein folding; Biochemistry; Chemistry

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APA (6th Edition):

Baker, C. M. (2006). The Mechanism of Assembly of the G-Protein Beta Gamma Subunit Dimer by CK2 Phosphorylated Phosducin-Like Protein and the Chaperonin Containing TCP-1. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1440&context=etd

Chicago Manual of Style (16th Edition):

Baker, Christine M. “The Mechanism of Assembly of the G-Protein Beta Gamma Subunit Dimer by CK2 Phosphorylated Phosducin-Like Protein and the Chaperonin Containing TCP-1.” 2006. Masters Thesis, Brigham Young University. Accessed November 15, 2019. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1440&context=etd.

MLA Handbook (7th Edition):

Baker, Christine M. “The Mechanism of Assembly of the G-Protein Beta Gamma Subunit Dimer by CK2 Phosphorylated Phosducin-Like Protein and the Chaperonin Containing TCP-1.” 2006. Web. 15 Nov 2019.

Vancouver:

Baker CM. The Mechanism of Assembly of the G-Protein Beta Gamma Subunit Dimer by CK2 Phosphorylated Phosducin-Like Protein and the Chaperonin Containing TCP-1. [Internet] [Masters thesis]. Brigham Young University; 2006. [cited 2019 Nov 15]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1440&context=etd.

Council of Science Editors:

Baker CM. The Mechanism of Assembly of the G-Protein Beta Gamma Subunit Dimer by CK2 Phosphorylated Phosducin-Like Protein and the Chaperonin Containing TCP-1. [Masters Thesis]. Brigham Young University; 2006. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1440&context=etd


University of Debrecen

5. Major, Evelin. A smoothelin-like-1 fehérje szerepének vizsgálata a pajzsmirigybetegségekben .

Degree: DE – Természettudományi és Technológiai Kar – Biológiai és Ökológiai Intézet, 2014, University of Debrecen

A pajzsmirigy alulműködése és túlműködése következtében kialakuló hypothyreosisos és hyperthyreosisos betegek panaszainak hátterében álló molekuláris mechanizmusok felderítésére törekedtünk, különös tekintettel a nemrégiben felfedezett smoothelin-like 1 fehérje szabályozó szerepének bizonyítására ezekben a folyamatokban. Advisors/Committee Members: Lontay, Beáta (advisor).

Subjects/Keywords: pajzsmirigy; smoothelin-like 1 protein

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APA (6th Edition):

Major, E. (2014). A smoothelin-like-1 fehérje szerepének vizsgálata a pajzsmirigybetegségekben . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/190776

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Major, Evelin. “A smoothelin-like-1 fehérje szerepének vizsgálata a pajzsmirigybetegségekben .” 2014. Thesis, University of Debrecen. Accessed November 15, 2019. http://hdl.handle.net/2437/190776.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Major, Evelin. “A smoothelin-like-1 fehérje szerepének vizsgálata a pajzsmirigybetegségekben .” 2014. Web. 15 Nov 2019.

Vancouver:

Major E. A smoothelin-like-1 fehérje szerepének vizsgálata a pajzsmirigybetegségekben . [Internet] [Thesis]. University of Debrecen; 2014. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/2437/190776.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Major E. A smoothelin-like-1 fehérje szerepének vizsgálata a pajzsmirigybetegségekben . [Thesis]. University of Debrecen; 2014. Available from: http://hdl.handle.net/2437/190776

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Brigham Young University

6. Ludtke, Paul Jayson. The Role of Phosducin-like Protein as a Co-chaperone with the Cytosolic Chaperonin Complex in Assembly of the G Protein βγ Subunit Dimer.

Degree: MS, 2007, Brigham Young University

Phosducin-like protein (PhLP) has been shown to interact with the cytosolic chaperonin containing TCP-1 (CCT), and the βγ subunit dimer of heterotrimeric G proteins… (more)

Subjects/Keywords: PhLP; Phosducin-Like Protein; Cytosolic Chaperonin Complex; CCT; G-Protein Signaling; Biochemistry; Chemistry

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APA (6th Edition):

Ludtke, P. J. (2007). The Role of Phosducin-like Protein as a Co-chaperone with the Cytosolic Chaperonin Complex in Assembly of the G Protein βγ Subunit Dimer. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=2313&context=etd

Chicago Manual of Style (16th Edition):

Ludtke, Paul Jayson. “The Role of Phosducin-like Protein as a Co-chaperone with the Cytosolic Chaperonin Complex in Assembly of the G Protein βγ Subunit Dimer.” 2007. Masters Thesis, Brigham Young University. Accessed November 15, 2019. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=2313&context=etd.

MLA Handbook (7th Edition):

Ludtke, Paul Jayson. “The Role of Phosducin-like Protein as a Co-chaperone with the Cytosolic Chaperonin Complex in Assembly of the G Protein βγ Subunit Dimer.” 2007. Web. 15 Nov 2019.

Vancouver:

Ludtke PJ. The Role of Phosducin-like Protein as a Co-chaperone with the Cytosolic Chaperonin Complex in Assembly of the G Protein βγ Subunit Dimer. [Internet] [Masters thesis]. Brigham Young University; 2007. [cited 2019 Nov 15]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=2313&context=etd.

Council of Science Editors:

Ludtke PJ. The Role of Phosducin-like Protein as a Co-chaperone with the Cytosolic Chaperonin Complex in Assembly of the G Protein βγ Subunit Dimer. [Masters Thesis]. Brigham Young University; 2007. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=2313&context=etd


University of Toronto

7. Wei. Identification of Cellular Components Interacting with the Shiga-like Toxin 1 A1 Chain (SLT-1 A1).

Degree: 2012, University of Toronto

Shiga-like toxin 1 (SLT-1) is produced by Escherichia coli strains like the pathogenic strain O157:H7. These bacterial strains are responsible for worldwide cases of food… (more)

Subjects/Keywords: protein toxin; shiga-like toxin 1; 0307

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APA (6th Edition):

Wei. (2012). Identification of Cellular Components Interacting with the Shiga-like Toxin 1 A1 Chain (SLT-1 A1). (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/32501

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Wei. “Identification of Cellular Components Interacting with the Shiga-like Toxin 1 A1 Chain (SLT-1 A1).” 2012. Masters Thesis, University of Toronto. Accessed November 15, 2019. http://hdl.handle.net/1807/32501.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Wei. “Identification of Cellular Components Interacting with the Shiga-like Toxin 1 A1 Chain (SLT-1 A1).” 2012. Web. 15 Nov 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Wei. Identification of Cellular Components Interacting with the Shiga-like Toxin 1 A1 Chain (SLT-1 A1). [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1807/32501.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Wei. Identification of Cellular Components Interacting with the Shiga-like Toxin 1 A1 Chain (SLT-1 A1). [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/32501

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Brigham Young University

8. Hu, Ting. The Role of Phosducin-like Protein and the Cytosolic Chaperonin CCT in G beta gamma dimer Assembly.

Degree: PhD, 2005, Brigham Young University

Phosducin-like protein (PhLP), a G protein beta gamma subunit dimer binder and G protein signaling regulator, was suggested to regulate the activity of cytosolic… (more)

Subjects/Keywords: phosducin-like protein (PhLP); electron microscopy; chaperonin; CK2 phosphorylation; G protein; protein folding; Biochemistry; Chemistry

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APA (6th Edition):

Hu, T. (2005). The Role of Phosducin-like Protein and the Cytosolic Chaperonin CCT in G beta gamma dimer Assembly. (Doctoral Dissertation). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1329&context=etd

Chicago Manual of Style (16th Edition):

Hu, Ting. “The Role of Phosducin-like Protein and the Cytosolic Chaperonin CCT in G beta gamma dimer Assembly.” 2005. Doctoral Dissertation, Brigham Young University. Accessed November 15, 2019. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1329&context=etd.

MLA Handbook (7th Edition):

Hu, Ting. “The Role of Phosducin-like Protein and the Cytosolic Chaperonin CCT in G beta gamma dimer Assembly.” 2005. Web. 15 Nov 2019.

Vancouver:

Hu T. The Role of Phosducin-like Protein and the Cytosolic Chaperonin CCT in G beta gamma dimer Assembly. [Internet] [Doctoral dissertation]. Brigham Young University; 2005. [cited 2019 Nov 15]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1329&context=etd.

Council of Science Editors:

Hu T. The Role of Phosducin-like Protein and the Cytosolic Chaperonin CCT in G beta gamma dimer Assembly. [Doctoral Dissertation]. Brigham Young University; 2005. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=1329&context=etd


UCLA

9. Boback, Bernard Boback. Catabolic Effects of Nell-1 Haploinsufficiency on Articular Cartilage in Murine Knee Joints.

Degree: Oral Biology, 2017, UCLA

 Contemporary therapeutic approaches for cartilage tissue engineering are flawed by significant functional heterogeneity and undesired side effects. A relatively novel growth factor, Nel1-like molecule-1 (Nell-1),… (more)

Subjects/Keywords: Dentistry; Articular cartilage; catabolic effect; NEL-like protein-1; osteoarthritis

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APA (6th Edition):

Boback, B. B. (2017). Catabolic Effects of Nell-1 Haploinsufficiency on Articular Cartilage in Murine Knee Joints. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/7mm7725d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boback, Bernard Boback. “Catabolic Effects of Nell-1 Haploinsufficiency on Articular Cartilage in Murine Knee Joints.” 2017. Thesis, UCLA. Accessed November 15, 2019. http://www.escholarship.org/uc/item/7mm7725d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boback, Bernard Boback. “Catabolic Effects of Nell-1 Haploinsufficiency on Articular Cartilage in Murine Knee Joints.” 2017. Web. 15 Nov 2019.

Vancouver:

Boback BB. Catabolic Effects of Nell-1 Haploinsufficiency on Articular Cartilage in Murine Knee Joints. [Internet] [Thesis]. UCLA; 2017. [cited 2019 Nov 15]. Available from: http://www.escholarship.org/uc/item/7mm7725d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boback BB. Catabolic Effects of Nell-1 Haploinsufficiency on Articular Cartilage in Murine Knee Joints. [Thesis]. UCLA; 2017. Available from: http://www.escholarship.org/uc/item/7mm7725d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

10. Liu, Xiaoyun. Molecular Characterisation of Niemann Pick Type C1 Protein.

Degree: Biotechnology & Biomolecular Sciences, 2014, University of New South Wales

 Niemann-Pick disease type C (NPC) is a fatal neurodegenerative disorder characterised by accumulation of free cholesterol and glycosphingolipids in the late endosome (LE) and lysosome… (more)

Subjects/Keywords: Niemman Pick Type C1; Stomatin-like protein 1

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APA (6th Edition):

Liu, X. (2014). Molecular Characterisation of Niemann Pick Type C1 Protein. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/54413 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:34933/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Liu, Xiaoyun. “Molecular Characterisation of Niemann Pick Type C1 Protein.” 2014. Masters Thesis, University of New South Wales. Accessed November 15, 2019. http://handle.unsw.edu.au/1959.4/54413 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:34933/SOURCE02?view=true.

MLA Handbook (7th Edition):

Liu, Xiaoyun. “Molecular Characterisation of Niemann Pick Type C1 Protein.” 2014. Web. 15 Nov 2019.

Vancouver:

Liu X. Molecular Characterisation of Niemann Pick Type C1 Protein. [Internet] [Masters thesis]. University of New South Wales; 2014. [cited 2019 Nov 15]. Available from: http://handle.unsw.edu.au/1959.4/54413 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:34933/SOURCE02?view=true.

Council of Science Editors:

Liu X. Molecular Characterisation of Niemann Pick Type C1 Protein. [Masters Thesis]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/54413 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:34933/SOURCE02?view=true


UCLA

11. Chen, Eric. Neurexin Family Member Contactin-Associated Protein Like-4 (CNTNAP4) is a specific cell membrane receptor of Neural EGFL Like 1 (NELL1).

Degree: Oral Biology, 2018, UCLA

 Secretory protein neural EGFL like 1 (Nell-1) has been shown to exert potent osteogenic effects in multiple small and large animal models. Here, we identified… (more)

Subjects/Keywords: Molecular biology; Biochemistry; Cellular biology; Contactin-associated protein-like 4 (Cntnap4); Neural EGFL like 1 (Nell-1); Osteogenesis; Receptor

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APA (6th Edition):

Chen, E. (2018). Neurexin Family Member Contactin-Associated Protein Like-4 (CNTNAP4) is a specific cell membrane receptor of Neural EGFL Like 1 (NELL1). (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/67b4g7v5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Eric. “Neurexin Family Member Contactin-Associated Protein Like-4 (CNTNAP4) is a specific cell membrane receptor of Neural EGFL Like 1 (NELL1).” 2018. Thesis, UCLA. Accessed November 15, 2019. http://www.escholarship.org/uc/item/67b4g7v5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Eric. “Neurexin Family Member Contactin-Associated Protein Like-4 (CNTNAP4) is a specific cell membrane receptor of Neural EGFL Like 1 (NELL1).” 2018. Web. 15 Nov 2019.

Vancouver:

Chen E. Neurexin Family Member Contactin-Associated Protein Like-4 (CNTNAP4) is a specific cell membrane receptor of Neural EGFL Like 1 (NELL1). [Internet] [Thesis]. UCLA; 2018. [cited 2019 Nov 15]. Available from: http://www.escholarship.org/uc/item/67b4g7v5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen E. Neurexin Family Member Contactin-Associated Protein Like-4 (CNTNAP4) is a specific cell membrane receptor of Neural EGFL Like 1 (NELL1). [Thesis]. UCLA; 2018. Available from: http://www.escholarship.org/uc/item/67b4g7v5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. 林, 亜由美. Is D-aspartate produced by glutamic-oxaloacetic transaminase-1 like 1(Got1l1), a putative aspartate racemase? : アスパラギン酸ラセマーゼとされる Got1l1 は、本当に D-アスパラギン酸を合成するのか?.

Degree: 博士(医学), 2016, University of Toyama / 富山大学

富山大学・富生命博乙第5号・林亜由美・2015/01/22

Amino Acids. 2015 Jan;47(1):79-86. doi: 10.1007/s00726-014-1847-3.に掲載。出版社版はhttp://link.springer.com/article/10.1007%2Fs00726-014-1847-3(オープンアクセス)

2014

Subjects/Keywords: Glutamic-oxaloacetic transaminase-1 like 1; D-Aspartate; Knockout mice; Testis; Hippocampus; Recombinant protein expression

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APA (6th Edition):

林, . (2016). Is D-aspartate produced by glutamic-oxaloacetic transaminase-1 like 1(Got1l1), a putative aspartate racemase? : アスパラギン酸ラセマーゼとされる Got1l1 は、本当に D-アスパラギン酸を合成するのか?. (Thesis). University of Toyama / 富山大学. Retrieved from http://hdl.handle.net/10110/13665 ; http://dx.doi.org/10.15099/00005039

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

林, 亜由美. “Is D-aspartate produced by glutamic-oxaloacetic transaminase-1 like 1(Got1l1), a putative aspartate racemase? : アスパラギン酸ラセマーゼとされる Got1l1 は、本当に D-アスパラギン酸を合成するのか?.” 2016. Thesis, University of Toyama / 富山大学. Accessed November 15, 2019. http://hdl.handle.net/10110/13665 ; http://dx.doi.org/10.15099/00005039.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

林, 亜由美. “Is D-aspartate produced by glutamic-oxaloacetic transaminase-1 like 1(Got1l1), a putative aspartate racemase? : アスパラギン酸ラセマーゼとされる Got1l1 は、本当に D-アスパラギン酸を合成するのか?.” 2016. Web. 15 Nov 2019.

Vancouver:

林 . Is D-aspartate produced by glutamic-oxaloacetic transaminase-1 like 1(Got1l1), a putative aspartate racemase? : アスパラギン酸ラセマーゼとされる Got1l1 は、本当に D-アスパラギン酸を合成するのか?. [Internet] [Thesis]. University of Toyama / 富山大学; 2016. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/10110/13665 ; http://dx.doi.org/10.15099/00005039.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

林 . Is D-aspartate produced by glutamic-oxaloacetic transaminase-1 like 1(Got1l1), a putative aspartate racemase? : アスパラギン酸ラセマーゼとされる Got1l1 は、本当に D-アスパラギン酸を合成するのか?. [Thesis]. University of Toyama / 富山大学; 2016. Available from: http://hdl.handle.net/10110/13665 ; http://dx.doi.org/10.15099/00005039

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Bowling Green State University

13. Sankey, Megan KH. IGF-1 and IGFBP-3 Levels in Individuals with Varied Kidney Function and the Relation to Dietary Protein Intake.

Degree: MFCS, Family and Consumer Sciences/food and Nutrition, 2009, Bowling Green State University

  PURPOSE: The purpose of the study was to find if IGF-1 and IGFBP-3 levels were altered with varied kidney function in a large sample… (more)

Subjects/Keywords: Nutrition; Insulin-like Growth Factor; IGF-1; IGFBP-3; kidney function; protein intake; NHANES III; animal protein; plant protein; renal

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APA (6th Edition):

Sankey, M. K. (2009). IGF-1 and IGFBP-3 Levels in Individuals with Varied Kidney Function and the Relation to Dietary Protein Intake. (Masters Thesis). Bowling Green State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1237825101

Chicago Manual of Style (16th Edition):

Sankey, Megan KH. “IGF-1 and IGFBP-3 Levels in Individuals with Varied Kidney Function and the Relation to Dietary Protein Intake.” 2009. Masters Thesis, Bowling Green State University. Accessed November 15, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1237825101.

MLA Handbook (7th Edition):

Sankey, Megan KH. “IGF-1 and IGFBP-3 Levels in Individuals with Varied Kidney Function and the Relation to Dietary Protein Intake.” 2009. Web. 15 Nov 2019.

Vancouver:

Sankey MK. IGF-1 and IGFBP-3 Levels in Individuals with Varied Kidney Function and the Relation to Dietary Protein Intake. [Internet] [Masters thesis]. Bowling Green State University; 2009. [cited 2019 Nov 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1237825101.

Council of Science Editors:

Sankey MK. IGF-1 and IGFBP-3 Levels in Individuals with Varied Kidney Function and the Relation to Dietary Protein Intake. [Masters Thesis]. Bowling Green State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=bgsu1237825101

14. Kalisch, Thomas. Caractérisation fonctionnelle et biochimique d'un nouveau partenaire de la poly(ADP-ribose) polymérase I : high-mobility group protein containing 2-like 1 : Biochemical and functionnal characterization of a new partner of poly(ADP-ribose) polymerase I : high-mobility group containing protein 2-like 1.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2013, Université de Strasbourg

La poly(ADP-ribosyl)ation est une modification post-traductionnelle des protéines catalysée par une famille d’enzymes : les poly(ADP-ribose) polymérases. Parmi les plus étudiées, PARP-1 et PARP-2 interviennent… (more)

Subjects/Keywords: Poly(ADP-ribosyl)ation; High-mobility group containing protein 2-like 1; Nucléole; Chaperonne à ARN; Protéine intrinsèquement désordonnée; Biogenèse des ribosomes; Poly(ADP-ribosyl)ation; High-mobility group containing protein 2-like 1; Nucleolus; RNA chaperon protein; Intrisically disordered protein; Biogenesis of ribosomes; 572.8

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APA (6th Edition):

Kalisch, T. (2013). Caractérisation fonctionnelle et biochimique d'un nouveau partenaire de la poly(ADP-ribose) polymérase I : high-mobility group protein containing 2-like 1 : Biochemical and functionnal characterization of a new partner of poly(ADP-ribose) polymerase I : high-mobility group containing protein 2-like 1. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2013STRAJ068

Chicago Manual of Style (16th Edition):

Kalisch, Thomas. “Caractérisation fonctionnelle et biochimique d'un nouveau partenaire de la poly(ADP-ribose) polymérase I : high-mobility group protein containing 2-like 1 : Biochemical and functionnal characterization of a new partner of poly(ADP-ribose) polymerase I : high-mobility group containing protein 2-like 1.” 2013. Doctoral Dissertation, Université de Strasbourg. Accessed November 15, 2019. http://www.theses.fr/2013STRAJ068.

MLA Handbook (7th Edition):

Kalisch, Thomas. “Caractérisation fonctionnelle et biochimique d'un nouveau partenaire de la poly(ADP-ribose) polymérase I : high-mobility group protein containing 2-like 1 : Biochemical and functionnal characterization of a new partner of poly(ADP-ribose) polymerase I : high-mobility group containing protein 2-like 1.” 2013. Web. 15 Nov 2019.

Vancouver:

Kalisch T. Caractérisation fonctionnelle et biochimique d'un nouveau partenaire de la poly(ADP-ribose) polymérase I : high-mobility group protein containing 2-like 1 : Biochemical and functionnal characterization of a new partner of poly(ADP-ribose) polymerase I : high-mobility group containing protein 2-like 1. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2013. [cited 2019 Nov 15]. Available from: http://www.theses.fr/2013STRAJ068.

Council of Science Editors:

Kalisch T. Caractérisation fonctionnelle et biochimique d'un nouveau partenaire de la poly(ADP-ribose) polymérase I : high-mobility group protein containing 2-like 1 : Biochemical and functionnal characterization of a new partner of poly(ADP-ribose) polymerase I : high-mobility group containing protein 2-like 1. [Doctoral Dissertation]. Université de Strasbourg; 2013. Available from: http://www.theses.fr/2013STRAJ068


Purdue University

15. Chen, Meng-Chieh. New Strategies To Reveal Protein Candidates In Protein-Protein Interactome Study.

Degree: MS, Biochemistry, 2014, Purdue University

  Comprehensive protein-protein interaction network analysis can help reveal protein functions in a system-wide manner. A reliable knowledgebase of interaction networks is not only important… (more)

Subjects/Keywords: Pure sciences; Biological sciences; Phosphoproteome; Polo-like kinase 1; Protein arginine methyltransferase 5; Protein-protein interaction; Sindbis virus; Virus-host interaction; Analytical Chemistry; Biochemistry; Molecular Biology

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APA (6th Edition):

Chen, M. (2014). New Strategies To Reveal Protein Candidates In Protein-Protein Interactome Study. (Thesis). Purdue University. Retrieved from http://docs.lib.purdue.edu/open_access_theses/310

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Meng-Chieh. “New Strategies To Reveal Protein Candidates In Protein-Protein Interactome Study.” 2014. Thesis, Purdue University. Accessed November 15, 2019. http://docs.lib.purdue.edu/open_access_theses/310.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Meng-Chieh. “New Strategies To Reveal Protein Candidates In Protein-Protein Interactome Study.” 2014. Web. 15 Nov 2019.

Vancouver:

Chen M. New Strategies To Reveal Protein Candidates In Protein-Protein Interactome Study. [Internet] [Thesis]. Purdue University; 2014. [cited 2019 Nov 15]. Available from: http://docs.lib.purdue.edu/open_access_theses/310.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen M. New Strategies To Reveal Protein Candidates In Protein-Protein Interactome Study. [Thesis]. Purdue University; 2014. Available from: http://docs.lib.purdue.edu/open_access_theses/310

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Yamaguchi, Yuya; Madhyastha, Harishkumar; Madhyastha, Radha; Choijookhuu, Narantsog; Hishikawa, Yoshitaka; Pengjam, Yutthana; Nakajima, Yuichi. Arsenic acid inhibits proliferation of skin fibroblasts, and increases cellular senescence through ROS mediated MST1-FOXO signaling pathway.

Degree: 博士(医学), 2015, University of Miyazaki / 宮崎大学

学位論文の一部を構成しているため、http://hdl.handle.net/10458/5831に本文を掲載。

Arsenic exposure through drinking water is a major public health problem. It causes a number of toxic effects on skin. Arsenic has been reported… (more)

Subjects/Keywords: Arsenic acid; Cell proliferation; Oxidative stress; Mammalian Ste20-like protein kinase 1; Forkhead box O transcription factors; Cellular senescence

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APA (6th Edition):

Yamaguchi, Yuya; Madhyastha, Harishkumar; Madhyastha, Radha; Choijookhuu, Narantsog; Hishikawa, Yoshitaka; Pengjam, Yutthana; Nakajima, Y. (2015). Arsenic acid inhibits proliferation of skin fibroblasts, and increases cellular senescence through ROS mediated MST1-FOXO signaling pathway. (Thesis). University of Miyazaki / 宮崎大学. Retrieved from http://hdl.handle.net/10458/5830

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamaguchi, Yuya; Madhyastha, Harishkumar; Madhyastha, Radha; Choijookhuu, Narantsog; Hishikawa, Yoshitaka; Pengjam, Yutthana; Nakajima, Yuichi. “Arsenic acid inhibits proliferation of skin fibroblasts, and increases cellular senescence through ROS mediated MST1-FOXO signaling pathway.” 2015. Thesis, University of Miyazaki / 宮崎大学. Accessed November 15, 2019. http://hdl.handle.net/10458/5830.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamaguchi, Yuya; Madhyastha, Harishkumar; Madhyastha, Radha; Choijookhuu, Narantsog; Hishikawa, Yoshitaka; Pengjam, Yutthana; Nakajima, Yuichi. “Arsenic acid inhibits proliferation of skin fibroblasts, and increases cellular senescence through ROS mediated MST1-FOXO signaling pathway.” 2015. Web. 15 Nov 2019.

Vancouver:

Yamaguchi, Yuya; Madhyastha, Harishkumar; Madhyastha, Radha; Choijookhuu, Narantsog; Hishikawa, Yoshitaka; Pengjam, Yutthana; Nakajima Y. Arsenic acid inhibits proliferation of skin fibroblasts, and increases cellular senescence through ROS mediated MST1-FOXO signaling pathway. [Internet] [Thesis]. University of Miyazaki / 宮崎大学; 2015. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/10458/5830.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamaguchi, Yuya; Madhyastha, Harishkumar; Madhyastha, Radha; Choijookhuu, Narantsog; Hishikawa, Yoshitaka; Pengjam, Yutthana; Nakajima Y. Arsenic acid inhibits proliferation of skin fibroblasts, and increases cellular senescence through ROS mediated MST1-FOXO signaling pathway. [Thesis]. University of Miyazaki / 宮崎大学; 2015. Available from: http://hdl.handle.net/10458/5830

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Suzuki, Stephanie. Paraxonase-1 Variants Alter Stability and Activity.

Degree: PhD, 2015, University of Washington

 Paraoxonase-1 (PON1) is a high-density lipoprotein-associated enzyme that has been extensively studied due to its ability to hydrolyze and inactivate toxic compounds such as organophosphate… (more)

Subjects/Keywords: Biphenyl hydrolase like protein; Homocysteine thiolactonase; Organophosphates; Paraoxonase 1; Molecular biology; Biochemistry; Toxicology; molecular and cellular biology

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APA (6th Edition):

Suzuki, S. (2015). Paraxonase-1 Variants Alter Stability and Activity. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/27533

Chicago Manual of Style (16th Edition):

Suzuki, Stephanie. “Paraxonase-1 Variants Alter Stability and Activity.” 2015. Doctoral Dissertation, University of Washington. Accessed November 15, 2019. http://hdl.handle.net/1773/27533.

MLA Handbook (7th Edition):

Suzuki, Stephanie. “Paraxonase-1 Variants Alter Stability and Activity.” 2015. Web. 15 Nov 2019.

Vancouver:

Suzuki S. Paraxonase-1 Variants Alter Stability and Activity. [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1773/27533.

Council of Science Editors:

Suzuki S. Paraxonase-1 Variants Alter Stability and Activity. [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/27533


University of Connecticut

18. Apicella, Jenna M. The Effect of Betaine Supplementation on Performance and Muscle Mechanisms.

Degree: MA, Kinesiology, 2011, University of Connecticut

  Background: Recent research has shown that betaine supplementation can increase strength and power performance. To further investigate the ergogenic effects of betaine supplementation a… (more)

Subjects/Keywords: betaine; muscle signaling; mTOR; Akt; AMPK; resistance exercise; resistance training; Growth Hormone; Insulin-like Growth Factor 1; cortisol; protein synthesis

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APA (6th Edition):

Apicella, J. M. (2011). The Effect of Betaine Supplementation on Performance and Muscle Mechanisms. (Masters Thesis). University of Connecticut. Retrieved from https://opencommons.uconn.edu/gs_theses/109

Chicago Manual of Style (16th Edition):

Apicella, Jenna M. “The Effect of Betaine Supplementation on Performance and Muscle Mechanisms.” 2011. Masters Thesis, University of Connecticut. Accessed November 15, 2019. https://opencommons.uconn.edu/gs_theses/109.

MLA Handbook (7th Edition):

Apicella, Jenna M. “The Effect of Betaine Supplementation on Performance and Muscle Mechanisms.” 2011. Web. 15 Nov 2019.

Vancouver:

Apicella JM. The Effect of Betaine Supplementation on Performance and Muscle Mechanisms. [Internet] [Masters thesis]. University of Connecticut; 2011. [cited 2019 Nov 15]. Available from: https://opencommons.uconn.edu/gs_theses/109.

Council of Science Editors:

Apicella JM. The Effect of Betaine Supplementation on Performance and Muscle Mechanisms. [Masters Thesis]. University of Connecticut; 2011. Available from: https://opencommons.uconn.edu/gs_theses/109

19. Cunha, Angela Francisca Trinconi da. Avaliação dos fatores de crescimento insulinóides IGF-1 e IGFBP-3 em mulheres com alto risco para câncer de mama.

Degree: PhD, Obstetrícia e Ginecologia, 2010, University of São Paulo

INTRODUÇÃO: A crescente incidência de câncer de mama, que cada vez mais acomete mulheres jovens, tem despertado muito interesse no diagnóstico precoce e na busca… (more)

Subjects/Keywords: Breast neoplasias; Fator de crescimento insulin-like I; Grupos de risco; Insulin-like growth factor 1; Insulin-like growth factor binding protein 3; Neoplasias da mama; Proteína 3 de ligação a fator de crescimento insulin-like; Risk groups

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APA (6th Edition):

Cunha, A. F. T. d. (2010). Avaliação dos fatores de crescimento insulinóides IGF-1 e IGFBP-3 em mulheres com alto risco para câncer de mama. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5139/tde-04112010-141608/ ;

Chicago Manual of Style (16th Edition):

Cunha, Angela Francisca Trinconi da. “Avaliação dos fatores de crescimento insulinóides IGF-1 e IGFBP-3 em mulheres com alto risco para câncer de mama.” 2010. Doctoral Dissertation, University of São Paulo. Accessed November 15, 2019. http://www.teses.usp.br/teses/disponiveis/5/5139/tde-04112010-141608/ ;.

MLA Handbook (7th Edition):

Cunha, Angela Francisca Trinconi da. “Avaliação dos fatores de crescimento insulinóides IGF-1 e IGFBP-3 em mulheres com alto risco para câncer de mama.” 2010. Web. 15 Nov 2019.

Vancouver:

Cunha AFTd. Avaliação dos fatores de crescimento insulinóides IGF-1 e IGFBP-3 em mulheres com alto risco para câncer de mama. [Internet] [Doctoral dissertation]. University of São Paulo; 2010. [cited 2019 Nov 15]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5139/tde-04112010-141608/ ;.

Council of Science Editors:

Cunha AFTd. Avaliação dos fatores de crescimento insulinóides IGF-1 e IGFBP-3 em mulheres com alto risco para câncer de mama. [Doctoral Dissertation]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/5/5139/tde-04112010-141608/ ;


University of Georgia

20. Mohamed, Islam Nabil Mohamed Badr. High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis.

Degree: PhD, Pharmacy, 2014, University of Georgia

 Diabetic retinopathy (DR) is a leading cause of blindness in US adults. DR starts with an early vaso-regressive stage of non-proliferative diabetic retinopathy (NPDR), which… (more)

Subjects/Keywords: Oxidative stress; inflammation; metabolic syndrome; obesity; high fat diet; hypertension; inflammasome; leukostasis; retinal acellular capillaries; thioredoxin interacting protein; NOD-like receptor protein; caspase-1 & IL-1β.

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APA (6th Edition):

Mohamed, I. N. M. B. (2014). High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/mohamed_islam_n_201408_phd

Chicago Manual of Style (16th Edition):

Mohamed, Islam Nabil Mohamed Badr. “High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis.” 2014. Doctoral Dissertation, University of Georgia. Accessed November 15, 2019. http://purl.galileo.usg.edu/uga_etd/mohamed_islam_n_201408_phd.

MLA Handbook (7th Edition):

Mohamed, Islam Nabil Mohamed Badr. “High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis.” 2014. Web. 15 Nov 2019.

Vancouver:

Mohamed INMB. High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis. [Internet] [Doctoral dissertation]. University of Georgia; 2014. [cited 2019 Nov 15]. Available from: http://purl.galileo.usg.edu/uga_etd/mohamed_islam_n_201408_phd.

Council of Science Editors:

Mohamed INMB. High fat diet induces retinal microvascular inflammation and degeneration: role of TXNIP-NLRP3 inflammasome axis. [Doctoral Dissertation]. University of Georgia; 2014. Available from: http://purl.galileo.usg.edu/uga_etd/mohamed_islam_n_201408_phd


Washington State University

21. [No author]. Understanding the Molecular Link Between FSH and IGF-1 Signaling in Rat Granulosa Cells .

Degree: 2016, Washington State University

 Mouse knockout models define follicle-stimulating hormone (FSH) and insulin-like growth factor-1 (IGF-1) as pivotal co-regulators of ovarian follicle maturation and hence, female fertility. Moreover, numerous… (more)

Subjects/Keywords: Molecular biology; follicle-stimulating hormone (FSH); granulosa cell; insulin-like growth factor-1 (IGF-1); phosphoinositide-3 kinase (PI3K); protein kinase A (PKA)

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APA (6th Edition):

author], [. (2016). Understanding the Molecular Link Between FSH and IGF-1 Signaling in Rat Granulosa Cells . (Thesis). Washington State University. Retrieved from http://hdl.handle.net/2376/12011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Understanding the Molecular Link Between FSH and IGF-1 Signaling in Rat Granulosa Cells .” 2016. Thesis, Washington State University. Accessed November 15, 2019. http://hdl.handle.net/2376/12011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Understanding the Molecular Link Between FSH and IGF-1 Signaling in Rat Granulosa Cells .” 2016. Web. 15 Nov 2019.

Vancouver:

author] [. Understanding the Molecular Link Between FSH and IGF-1 Signaling in Rat Granulosa Cells . [Internet] [Thesis]. Washington State University; 2016. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/2376/12011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Understanding the Molecular Link Between FSH and IGF-1 Signaling in Rat Granulosa Cells . [Thesis]. Washington State University; 2016. Available from: http://hdl.handle.net/2376/12011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

22. Kapakos, Georgia. Modulation of Endothelin-1 and Insulin-like Growth Factor Type 1-induced Signaling by Curcumin in A-10 Vascular Smooth Muscle Cells .

Degree: 2012, Université de Montréal

 Les maladies cardio-vasculaires (MCV), telles que l’hypertension et l’athérosclérose, s’accompagnent de modifications structurales et fonctionnelles au niveau vasculaire. Un fonctionnement aberrant de la migration, l’hypertrophie… (more)

Subjects/Keywords: Curcumine; Cellules musculaires lisses vasculaires (CMLV); ERK1/2; Endothéline-1 (ET-1); IGF-1; IGF-1R; PKB; Egr-1; Curcumin; Endothelin-1 (ET-1); Extracellular signal-regulated kinase 1/2 (ERK1/2); Vascular smooth muscle cells (VSMC); Insulin-like growth factor type 1 (IGF-1); Insulin-like growth factor type 1 receptor (IGF-1R); Protein kinase B (PKB); Early growth response -1 (Egr-1)

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APA (6th Edition):

Kapakos, G. (2012). Modulation of Endothelin-1 and Insulin-like Growth Factor Type 1-induced Signaling by Curcumin in A-10 Vascular Smooth Muscle Cells . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/6223

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kapakos, Georgia. “Modulation of Endothelin-1 and Insulin-like Growth Factor Type 1-induced Signaling by Curcumin in A-10 Vascular Smooth Muscle Cells .” 2012. Thesis, Université de Montréal. Accessed November 15, 2019. http://hdl.handle.net/1866/6223.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kapakos, Georgia. “Modulation of Endothelin-1 and Insulin-like Growth Factor Type 1-induced Signaling by Curcumin in A-10 Vascular Smooth Muscle Cells .” 2012. Web. 15 Nov 2019.

Vancouver:

Kapakos G. Modulation of Endothelin-1 and Insulin-like Growth Factor Type 1-induced Signaling by Curcumin in A-10 Vascular Smooth Muscle Cells . [Internet] [Thesis]. Université de Montréal; 2012. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/1866/6223.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kapakos G. Modulation of Endothelin-1 and Insulin-like Growth Factor Type 1-induced Signaling by Curcumin in A-10 Vascular Smooth Muscle Cells . [Thesis]. Université de Montréal; 2012. Available from: http://hdl.handle.net/1866/6223

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Montenegro, Luciana Ribeiro. Estudo in vitro da sensibilidade ao IGF-1 de fibroblastos de crianças nascidas pequenas para a idade gestacional sem recuperação estatural pós-natal.

Degree: PhD, Endocrinologia, 2009, University of São Paulo

 Introdução: Crianças nascidas pequenas para a idade gestacional (PIG) apresentam maior risco de permanecerem com baixa estatura na vida adulta. Os fatores de crescimento insulino-símile… (more)

Subjects/Keywords: Failure to thrive; Fator de crescimento insulin-like I/genética; Fator de crescimento insulin-like II/genética; Fetal growth retardation; Growth disorders; Insuficiência de crescimento/etiolgia; Insulin-like growth factor binding protein 3; Insulin-like growth factor I; Insulin-like growth factor II; Proteína 3 de ligação a fator de crescimento insulin-like/fisiologia; Receptor IGF tipo 1/genética; Receptor IGF Type 1; Retardo do crescimento fetal/etiologia; Transtornos do crescimento/etiologia

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APA (6th Edition):

Montenegro, L. R. (2009). Estudo in vitro da sensibilidade ao IGF-1 de fibroblastos de crianças nascidas pequenas para a idade gestacional sem recuperação estatural pós-natal. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5135/tde-08092009-132008/ ;

Chicago Manual of Style (16th Edition):

Montenegro, Luciana Ribeiro. “Estudo in vitro da sensibilidade ao IGF-1 de fibroblastos de crianças nascidas pequenas para a idade gestacional sem recuperação estatural pós-natal.” 2009. Doctoral Dissertation, University of São Paulo. Accessed November 15, 2019. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-08092009-132008/ ;.

MLA Handbook (7th Edition):

Montenegro, Luciana Ribeiro. “Estudo in vitro da sensibilidade ao IGF-1 de fibroblastos de crianças nascidas pequenas para a idade gestacional sem recuperação estatural pós-natal.” 2009. Web. 15 Nov 2019.

Vancouver:

Montenegro LR. Estudo in vitro da sensibilidade ao IGF-1 de fibroblastos de crianças nascidas pequenas para a idade gestacional sem recuperação estatural pós-natal. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2019 Nov 15]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-08092009-132008/ ;.

Council of Science Editors:

Montenegro LR. Estudo in vitro da sensibilidade ao IGF-1 de fibroblastos de crianças nascidas pequenas para a idade gestacional sem recuperação estatural pós-natal. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-08092009-132008/ ;

24. Ragusa, Michael J. The Regulation of Protein Phosphatase 1 by Targeting Proteins.

Degree: PhD, Molecular Biology, Cell Biology, and Biochemistry, 2011, Brown University

Protein phosphatase-1 (PP1) is a major serine/threonine phosphatase that dephorphorylates numerous substrates, thereby regulating many cellular events. PP1 contains little substrate specificity on its own,… (more)

Subjects/Keywords: protein phosphatase 1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ragusa, M. J. (2011). The Regulation of Protein Phosphatase 1 by Targeting Proteins. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:11183/

Chicago Manual of Style (16th Edition):

Ragusa, Michael J. “The Regulation of Protein Phosphatase 1 by Targeting Proteins.” 2011. Doctoral Dissertation, Brown University. Accessed November 15, 2019. https://repository.library.brown.edu/studio/item/bdr:11183/.

MLA Handbook (7th Edition):

Ragusa, Michael J. “The Regulation of Protein Phosphatase 1 by Targeting Proteins.” 2011. Web. 15 Nov 2019.

Vancouver:

Ragusa MJ. The Regulation of Protein Phosphatase 1 by Targeting Proteins. [Internet] [Doctoral dissertation]. Brown University; 2011. [cited 2019 Nov 15]. Available from: https://repository.library.brown.edu/studio/item/bdr:11183/.

Council of Science Editors:

Ragusa MJ. The Regulation of Protein Phosphatase 1 by Targeting Proteins. [Doctoral Dissertation]. Brown University; 2011. Available from: https://repository.library.brown.edu/studio/item/bdr:11183/


University of Oulu

25. Kurkinen-Räty, M. (Merja). Preterm birth and preterm infant:a clinical study on certain etiological and diagnostic factors, and the outcome of infants.

Degree: 2000, University of Oulu

 Abstract The aim of the present study was to evaluate whether bacterial vaginosis (BV) diagnosed in early pregnancy and treated with vaginal clindamycin affects pregnancy… (more)

Subjects/Keywords: PPROM; bacterial vaginosis; insulin-like growth factor-binding protein-1; interleukins; ultrasonography

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kurkinen-Räty, M. (. (2000). Preterm birth and preterm infant:a clinical study on certain etiological and diagnostic factors, and the outcome of infants. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9514258266

Chicago Manual of Style (16th Edition):

Kurkinen-Räty, M (Merja). “Preterm birth and preterm infant:a clinical study on certain etiological and diagnostic factors, and the outcome of infants.” 2000. Doctoral Dissertation, University of Oulu. Accessed November 15, 2019. http://urn.fi/urn:isbn:9514258266.

MLA Handbook (7th Edition):

Kurkinen-Räty, M (Merja). “Preterm birth and preterm infant:a clinical study on certain etiological and diagnostic factors, and the outcome of infants.” 2000. Web. 15 Nov 2019.

Vancouver:

Kurkinen-Räty M(. Preterm birth and preterm infant:a clinical study on certain etiological and diagnostic factors, and the outcome of infants. [Internet] [Doctoral dissertation]. University of Oulu; 2000. [cited 2019 Nov 15]. Available from: http://urn.fi/urn:isbn:9514258266.

Council of Science Editors:

Kurkinen-Räty M(. Preterm birth and preterm infant:a clinical study on certain etiological and diagnostic factors, and the outcome of infants. [Doctoral Dissertation]. University of Oulu; 2000. Available from: http://urn.fi/urn:isbn:9514258266


The Ohio State University

26. Shin, Jonghyun. The Role of Chicken Delta-Like Protein 1 Expression in Skeletal Muscle Development and Regeneration.

Degree: PhD, OSU Nutrition, 2009, The Ohio State University

  The role of Delta-like protein 1 (DLK1) has been implicated in the muscle hypertrophy observed in DLK1 transgenic mice, callipyge sheep, mouse paternal uniparental… (more)

Subjects/Keywords: Biology; Chicken Delta-Like Protein 1 (gDLK1); Myogenic Regulatory Factors (MRFs); Muscle hypertrophy; Development; Regeneration; Cell Differentiation; Myofibers; Broilers; Leghorn Layers; Low Score Normal (LSN); Muscular Dystrophy (MD)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shin, J. (2009). The Role of Chicken Delta-Like Protein 1 Expression in Skeletal Muscle Development and Regeneration. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1253332067

Chicago Manual of Style (16th Edition):

Shin, Jonghyun. “The Role of Chicken Delta-Like Protein 1 Expression in Skeletal Muscle Development and Regeneration.” 2009. Doctoral Dissertation, The Ohio State University. Accessed November 15, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1253332067.

MLA Handbook (7th Edition):

Shin, Jonghyun. “The Role of Chicken Delta-Like Protein 1 Expression in Skeletal Muscle Development and Regeneration.” 2009. Web. 15 Nov 2019.

Vancouver:

Shin J. The Role of Chicken Delta-Like Protein 1 Expression in Skeletal Muscle Development and Regeneration. [Internet] [Doctoral dissertation]. The Ohio State University; 2009. [cited 2019 Nov 15]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1253332067.

Council of Science Editors:

Shin J. The Role of Chicken Delta-Like Protein 1 Expression in Skeletal Muscle Development and Regeneration. [Doctoral Dissertation]. The Ohio State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1253332067


University of Western Australia

27. Gebski, Bijanka L. Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy.

Degree: PhD, 2009, University of Western Australia

 [Truncated abstract] The pro-inflammatory cytokine tumour necrosis factor (TNF) has a critical role in skeletal muscle atrophy. The catabolic effect of TNF is partially due… (more)

Subjects/Keywords: JNK mitogen-activated protein kinases; Insulin-like growth factor-binding proteins; Tumor necrosis factor; Duchenne muscular dystrophy; TNF; JNK; IGF-1; Muscle

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APA (6th Edition):

Gebski, B. L. (2009). Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12898&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Gebski, Bijanka L. “Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy.” 2009. Doctoral Dissertation, University of Western Australia. Accessed November 15, 2019. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12898&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Gebski, Bijanka L. “Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy.” 2009. Web. 15 Nov 2019.

Vancouver:

Gebski BL. Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy. [Internet] [Doctoral dissertation]. University of Western Australia; 2009. [cited 2019 Nov 15]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12898&local_base=GEN01-INS01.

Council of Science Editors:

Gebski BL. Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy. [Doctoral Dissertation]. University of Western Australia; 2009. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12898&local_base=GEN01-INS01


University of Illinois – Urbana-Champaign

28. Ramisetty, Sreenivasa Rao. RNA recognition: controlling RNA-protein complexes with small molecules.

Degree: PhD, 0335, 2010, University of Illinois – Urbana-Champaign

 ABSTRACT PART I. Investigation of Small Molecule Binding to an RNA Hairpin Loop Containing a Dangling End PART II. Unraveling the Interaction of Pathogenic RNAs… (more)

Subjects/Keywords: Muscleblind-like protein (MBNL); Zinc finger (ZNF)

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APA (6th Edition):

Ramisetty, S. R. (2010). RNA recognition: controlling RNA-protein complexes with small molecules. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/15581

Chicago Manual of Style (16th Edition):

Ramisetty, Sreenivasa Rao. “RNA recognition: controlling RNA-protein complexes with small molecules.” 2010. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed November 15, 2019. http://hdl.handle.net/2142/15581.

MLA Handbook (7th Edition):

Ramisetty, Sreenivasa Rao. “RNA recognition: controlling RNA-protein complexes with small molecules.” 2010. Web. 15 Nov 2019.

Vancouver:

Ramisetty SR. RNA recognition: controlling RNA-protein complexes with small molecules. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2010. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/2142/15581.

Council of Science Editors:

Ramisetty SR. RNA recognition: controlling RNA-protein complexes with small molecules. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2010. Available from: http://hdl.handle.net/2142/15581


Duke University

29. Amiram, Miriam. Glucagon-Like Peptide-1 Depots for the Treatment of Type-2 Diabetes .

Degree: 2012, Duke University

  Peptide drugs are an exciting class of pharmaceuticals currently in development for the treatment of a variety of diseases; however, their main drawback is… (more)

Subjects/Keywords: Biomedical engineering; Drug Delivery; Elastin Like Polypeptides; Glucagon Like Peptide-1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Amiram, M. (2012). Glucagon-Like Peptide-1 Depots for the Treatment of Type-2 Diabetes . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/5499

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Amiram, Miriam. “Glucagon-Like Peptide-1 Depots for the Treatment of Type-2 Diabetes .” 2012. Thesis, Duke University. Accessed November 15, 2019. http://hdl.handle.net/10161/5499.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Amiram, Miriam. “Glucagon-Like Peptide-1 Depots for the Treatment of Type-2 Diabetes .” 2012. Web. 15 Nov 2019.

Vancouver:

Amiram M. Glucagon-Like Peptide-1 Depots for the Treatment of Type-2 Diabetes . [Internet] [Thesis]. Duke University; 2012. [cited 2019 Nov 15]. Available from: http://hdl.handle.net/10161/5499.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Amiram M. Glucagon-Like Peptide-1 Depots for the Treatment of Type-2 Diabetes . [Thesis]. Duke University; 2012. Available from: http://hdl.handle.net/10161/5499

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Tulane University

30. Pollard, Kevin. Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens.

Degree: 2017, Tulane University

The goal of the following experiments was to determine the cellular mechanisms through which estrogen receptor activity is maintained in hippocampal cells following termination of… (more)

Subjects/Keywords: estrogen; hippocampus; insulin-like growth factor-1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pollard, K. (2017). Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens. (Thesis). Tulane University. Retrieved from https://digitallibrary.tulane.edu/islandora/object/tulane:76927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pollard, Kevin. “Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens.” 2017. Thesis, Tulane University. Accessed November 15, 2019. https://digitallibrary.tulane.edu/islandora/object/tulane:76927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pollard, Kevin. “Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens.” 2017. Web. 15 Nov 2019.

Vancouver:

Pollard K. Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens. [Internet] [Thesis]. Tulane University; 2017. [cited 2019 Nov 15]. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:76927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pollard K. Mechanisms through which nuclear estrogen receptors remain transcriptionally active in the mouse hippocampus in absence of ovarian estrogens. [Thesis]. Tulane University; 2017. Available from: https://digitallibrary.tulane.edu/islandora/object/tulane:76927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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