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You searched for subject:(Pharmacology). Showing records 1 – 30 of 4741 total matches.

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Wake Forest University

1. Silberman, Yuval. Ethanol Enhancement of Two Distinct Inhibitory Pathways in the Rat Basolateral Amygdala: Implications for Anxiety and Alcoholism.

Degree: 2009, Wake Forest University

 The basolateral amygdala (BLA) acts as a key interface between the interpretation of external stimuli and the production of emotional responses, such as fear and… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Silberman, Y. (2009). Ethanol Enhancement of Two Distinct Inhibitory Pathways in the Rat Basolateral Amygdala: Implications for Anxiety and Alcoholism. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/14768

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silberman, Yuval. “Ethanol Enhancement of Two Distinct Inhibitory Pathways in the Rat Basolateral Amygdala: Implications for Anxiety and Alcoholism.” 2009. Thesis, Wake Forest University. Accessed October 21, 2017. http://hdl.handle.net/10339/14768.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silberman, Yuval. “Ethanol Enhancement of Two Distinct Inhibitory Pathways in the Rat Basolateral Amygdala: Implications for Anxiety and Alcoholism.” 2009. Web. 21 Oct 2017.

Vancouver:

Silberman Y. Ethanol Enhancement of Two Distinct Inhibitory Pathways in the Rat Basolateral Amygdala: Implications for Anxiety and Alcoholism. [Internet] [Thesis]. Wake Forest University; 2009. [cited 2017 Oct 21]. Available from: http://hdl.handle.net/10339/14768.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silberman Y. Ethanol Enhancement of Two Distinct Inhibitory Pathways in the Rat Basolateral Amygdala: Implications for Anxiety and Alcoholism. [Thesis]. Wake Forest University; 2009. Available from: http://hdl.handle.net/10339/14768

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

2. Fang, Pu. HYPERHOMOCYSTEINEMIA ACCELERATES ATHEROSCLEROSIS BY INDUCING INFLAMMATORY MONOCYTE DIFFERENTIATION IN A HYPERGLYCEMIC MOUSE MODEL.

Degree: PhD, 2012, Temple University

Pharmacology

Homocysteine (Hcy) is a thiol amino acid formed upon methionine de - methylation. A number of studies have revealed an association between hyperhomocysteinemia (HHcy),… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Fang, P. (2012). HYPERHOMOCYSTEINEMIA ACCELERATES ATHEROSCLEROSIS BY INDUCING INFLAMMATORY MONOCYTE DIFFERENTIATION IN A HYPERGLYCEMIC MOUSE MODEL. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,223888

Chicago Manual of Style (16th Edition):

Fang, Pu. “HYPERHOMOCYSTEINEMIA ACCELERATES ATHEROSCLEROSIS BY INDUCING INFLAMMATORY MONOCYTE DIFFERENTIATION IN A HYPERGLYCEMIC MOUSE MODEL.” 2012. Doctoral Dissertation, Temple University. Accessed October 21, 2017. http://digital.library.temple.edu/u?/p245801coll10,223888.

MLA Handbook (7th Edition):

Fang, Pu. “HYPERHOMOCYSTEINEMIA ACCELERATES ATHEROSCLEROSIS BY INDUCING INFLAMMATORY MONOCYTE DIFFERENTIATION IN A HYPERGLYCEMIC MOUSE MODEL.” 2012. Web. 21 Oct 2017.

Vancouver:

Fang P. HYPERHOMOCYSTEINEMIA ACCELERATES ATHEROSCLEROSIS BY INDUCING INFLAMMATORY MONOCYTE DIFFERENTIATION IN A HYPERGLYCEMIC MOUSE MODEL. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2017 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,223888.

Council of Science Editors:

Fang P. HYPERHOMOCYSTEINEMIA ACCELERATES ATHEROSCLEROSIS BY INDUCING INFLAMMATORY MONOCYTE DIFFERENTIATION IN A HYPERGLYCEMIC MOUSE MODEL. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,223888


Temple University

3. Pansuria, Meghanaben. EFFECT AND MECHANISM OF HYPERHOMOCYSTEINEMIA ON ENDOTHELIAL INSULIN SIGNALING.

Degree: PhD, 2013, Temple University

Pharmacology

Hyperhomocysteinemia (HHcy) is an independent risk factor for cardiovascular disease (CVD). Both HHcy and insulin resistance (IR) are associated with atherosclerotic CVD. Recent studies… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Pansuria, M. (2013). EFFECT AND MECHANISM OF HYPERHOMOCYSTEINEMIA ON ENDOTHELIAL INSULIN SIGNALING. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,228447

Chicago Manual of Style (16th Edition):

Pansuria, Meghanaben. “EFFECT AND MECHANISM OF HYPERHOMOCYSTEINEMIA ON ENDOTHELIAL INSULIN SIGNALING.” 2013. Doctoral Dissertation, Temple University. Accessed October 21, 2017. http://digital.library.temple.edu/u?/p245801coll10,228447.

MLA Handbook (7th Edition):

Pansuria, Meghanaben. “EFFECT AND MECHANISM OF HYPERHOMOCYSTEINEMIA ON ENDOTHELIAL INSULIN SIGNALING.” 2013. Web. 21 Oct 2017.

Vancouver:

Pansuria M. EFFECT AND MECHANISM OF HYPERHOMOCYSTEINEMIA ON ENDOTHELIAL INSULIN SIGNALING. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2017 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,228447.

Council of Science Editors:

Pansuria M. EFFECT AND MECHANISM OF HYPERHOMOCYSTEINEMIA ON ENDOTHELIAL INSULIN SIGNALING. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,228447


The Ohio State University

4. Mazumder, Sarmistha. Effect of omega-3 fatty acids on ventricular action potentials in a canine model of sudden cardiac death.

Degree: MS, Pharmacy, 2010, The Ohio State University

  Background: Sudden cardiac death (SCD) is the most common cause of death in the United States and in most developed countries, accounting for ~… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Mazumder, S. (2010). Effect of omega-3 fatty acids on ventricular action potentials in a canine model of sudden cardiac death. (Masters Thesis). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1292203189

Chicago Manual of Style (16th Edition):

Mazumder, Sarmistha. “Effect of omega-3 fatty acids on ventricular action potentials in a canine model of sudden cardiac death.” 2010. Masters Thesis, The Ohio State University. Accessed October 21, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1292203189.

MLA Handbook (7th Edition):

Mazumder, Sarmistha. “Effect of omega-3 fatty acids on ventricular action potentials in a canine model of sudden cardiac death.” 2010. Web. 21 Oct 2017.

Vancouver:

Mazumder S. Effect of omega-3 fatty acids on ventricular action potentials in a canine model of sudden cardiac death. [Internet] [Masters thesis]. The Ohio State University; 2010. [cited 2017 Oct 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1292203189.

Council of Science Editors:

Mazumder S. Effect of omega-3 fatty acids on ventricular action potentials in a canine model of sudden cardiac death. [Masters Thesis]. The Ohio State University; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1292203189


The Ohio State University

5. Ma, Yihui. DEVELOPMENT OF ANTICANCER AGENTS BY MODIFICATION OF A NOVEL IMMUNOSUPPRESSANT FTY720 AND PDK1 INHIBITOR OSU-03012.

Degree: PhD, Pharmacy, 2011, The Ohio State University

  FTY720 is a novel immunomodulator that has been approved by FDA for the treatment of relapsing Multiple sclerosis (MS). It features a unique mechanism… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Ma, Y. (2011). DEVELOPMENT OF ANTICANCER AGENTS BY MODIFICATION OF A NOVEL IMMUNOSUPPRESSANT FTY720 AND PDK1 INHIBITOR OSU-03012. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1315497619

Chicago Manual of Style (16th Edition):

Ma, Yihui. “DEVELOPMENT OF ANTICANCER AGENTS BY MODIFICATION OF A NOVEL IMMUNOSUPPRESSANT FTY720 AND PDK1 INHIBITOR OSU-03012.” 2011. Doctoral Dissertation, The Ohio State University. Accessed October 21, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1315497619.

MLA Handbook (7th Edition):

Ma, Yihui. “DEVELOPMENT OF ANTICANCER AGENTS BY MODIFICATION OF A NOVEL IMMUNOSUPPRESSANT FTY720 AND PDK1 INHIBITOR OSU-03012.” 2011. Web. 21 Oct 2017.

Vancouver:

Ma Y. DEVELOPMENT OF ANTICANCER AGENTS BY MODIFICATION OF A NOVEL IMMUNOSUPPRESSANT FTY720 AND PDK1 INHIBITOR OSU-03012. [Internet] [Doctoral dissertation]. The Ohio State University; 2011. [cited 2017 Oct 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1315497619.

Council of Science Editors:

Ma Y. DEVELOPMENT OF ANTICANCER AGENTS BY MODIFICATION OF A NOVEL IMMUNOSUPPRESSANT FTY720 AND PDK1 INHIBITOR OSU-03012. [Doctoral Dissertation]. The Ohio State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1315497619


University of Cape Town

6. Gabriels, Gary Anthony. The investigation and assessment of Nutritional and Traditional Supplement products for content validity, contamination and adulteration.

Degree: Ph D, Division of Clinical Pharmacology, 2013, University of Cape Town

Includes abstract.

Nutritional supplements are used by competitive and recreational athletes of all ages. As a consequence the supplement industry has grown to meet the… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Gabriels, G. A. (2013). The investigation and assessment of Nutritional and Traditional Supplement products for content validity, contamination and adulteration. (Doctoral Dissertation). University of Cape Town. Retrieved from http://hdl.handle.net/11427/3281

Chicago Manual of Style (16th Edition):

Gabriels, Gary Anthony. “The investigation and assessment of Nutritional and Traditional Supplement products for content validity, contamination and adulteration.” 2013. Doctoral Dissertation, University of Cape Town. Accessed October 21, 2017. http://hdl.handle.net/11427/3281.

MLA Handbook (7th Edition):

Gabriels, Gary Anthony. “The investigation and assessment of Nutritional and Traditional Supplement products for content validity, contamination and adulteration.” 2013. Web. 21 Oct 2017.

Vancouver:

Gabriels GA. The investigation and assessment of Nutritional and Traditional Supplement products for content validity, contamination and adulteration. [Internet] [Doctoral dissertation]. University of Cape Town; 2013. [cited 2017 Oct 21]. Available from: http://hdl.handle.net/11427/3281.

Council of Science Editors:

Gabriels GA. The investigation and assessment of Nutritional and Traditional Supplement products for content validity, contamination and adulteration. [Doctoral Dissertation]. University of Cape Town; 2013. Available from: http://hdl.handle.net/11427/3281


University of Cape Town

7. Meredith, Sandra Allison. The characterization of adaptor protein homologues in Plasmodium falciparum.

Degree: PhD, Division of Clinical Pharmacology, 2009, University of Cape Town

Includes abstract.

Plasmodium falciparum is becoming increasingly more resistant to regular antimalarial drugs, making it necessary to identify novel drug candidates and drug targets. Components… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Meredith, S. A. (2009). The characterization of adaptor protein homologues in Plasmodium falciparum. (Doctoral Dissertation). University of Cape Town. Retrieved from http://hdl.handle.net/11427/3291

Chicago Manual of Style (16th Edition):

Meredith, Sandra Allison. “The characterization of adaptor protein homologues in Plasmodium falciparum.” 2009. Doctoral Dissertation, University of Cape Town. Accessed October 21, 2017. http://hdl.handle.net/11427/3291.

MLA Handbook (7th Edition):

Meredith, Sandra Allison. “The characterization of adaptor protein homologues in Plasmodium falciparum.” 2009. Web. 21 Oct 2017.

Vancouver:

Meredith SA. The characterization of adaptor protein homologues in Plasmodium falciparum. [Internet] [Doctoral dissertation]. University of Cape Town; 2009. [cited 2017 Oct 21]. Available from: http://hdl.handle.net/11427/3291.

Council of Science Editors:

Meredith SA. The characterization of adaptor protein homologues in Plasmodium falciparum. [Doctoral Dissertation]. University of Cape Town; 2009. Available from: http://hdl.handle.net/11427/3291


University of Cape Town

8. Amlabu, Veronica Asibi. Isoniazid and acetylisoniazid urine concentrations as a maker of adherence to isoniazid preventative therapy in children.

Degree: MMed, Division of Clinical Pharmacology, 2013, University of Cape Town

Includes abstract.

The World Health Organization recommends the use of isoniazid preventive therapy to reduce the incidence of tuberculosis disease in populations at risk for… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Amlabu, V. A. (2013). Isoniazid and acetylisoniazid urine concentrations as a maker of adherence to isoniazid preventative therapy in children. (Masters Thesis). University of Cape Town. Retrieved from http://hdl.handle.net/11427/3307

Chicago Manual of Style (16th Edition):

Amlabu, Veronica Asibi. “Isoniazid and acetylisoniazid urine concentrations as a maker of adherence to isoniazid preventative therapy in children.” 2013. Masters Thesis, University of Cape Town. Accessed October 21, 2017. http://hdl.handle.net/11427/3307.

MLA Handbook (7th Edition):

Amlabu, Veronica Asibi. “Isoniazid and acetylisoniazid urine concentrations as a maker of adherence to isoniazid preventative therapy in children.” 2013. Web. 21 Oct 2017.

Vancouver:

Amlabu VA. Isoniazid and acetylisoniazid urine concentrations as a maker of adherence to isoniazid preventative therapy in children. [Internet] [Masters thesis]. University of Cape Town; 2013. [cited 2017 Oct 21]. Available from: http://hdl.handle.net/11427/3307.

Council of Science Editors:

Amlabu VA. Isoniazid and acetylisoniazid urine concentrations as a maker of adherence to isoniazid preventative therapy in children. [Masters Thesis]. University of Cape Town; 2013. Available from: http://hdl.handle.net/11427/3307


University of Cape Town

9. Melariri, Paula E. The therapeutic effectiveness of some local Nigerian plants used in the treatment of malaria.

Degree: PhD, Division of Clinical Pharmacology, 2010, University of Cape Town

Includes abstract.

In Nigeria most of the populace relies heavily on medicinal plants for the treatment of malaria. This thesis describes the investigation of the… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Melariri, P. E. (2010). The therapeutic effectiveness of some local Nigerian plants used in the treatment of malaria. (Doctoral Dissertation). University of Cape Town. Retrieved from http://hdl.handle.net/11427/11552

Chicago Manual of Style (16th Edition):

Melariri, Paula E. “The therapeutic effectiveness of some local Nigerian plants used in the treatment of malaria.” 2010. Doctoral Dissertation, University of Cape Town. Accessed October 21, 2017. http://hdl.handle.net/11427/11552.

MLA Handbook (7th Edition):

Melariri, Paula E. “The therapeutic effectiveness of some local Nigerian plants used in the treatment of malaria.” 2010. Web. 21 Oct 2017.

Vancouver:

Melariri PE. The therapeutic effectiveness of some local Nigerian plants used in the treatment of malaria. [Internet] [Doctoral dissertation]. University of Cape Town; 2010. [cited 2017 Oct 21]. Available from: http://hdl.handle.net/11427/11552.

Council of Science Editors:

Melariri PE. The therapeutic effectiveness of some local Nigerian plants used in the treatment of malaria. [Doctoral Dissertation]. University of Cape Town; 2010. Available from: http://hdl.handle.net/11427/11552


Duke University

10. Li, Ran. Abl Kinases Modulate Epithelial Architecture by Regulating Beta1 Integrin and c-Met Signals.

Degree: 2011, Duke University

  Normal development and homeostasis require dynamic and tight regulation of epithelial architecture. Abnormal epithelial physiology is associated with various pathological conditions including cancers, and… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Li, R. (2011). Abl Kinases Modulate Epithelial Architecture by Regulating Beta1 Integrin and c-Met Signals. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/5025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Ran. “Abl Kinases Modulate Epithelial Architecture by Regulating Beta1 Integrin and c-Met Signals.” 2011. Thesis, Duke University. Accessed October 21, 2017. http://hdl.handle.net/10161/5025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Ran. “Abl Kinases Modulate Epithelial Architecture by Regulating Beta1 Integrin and c-Met Signals.” 2011. Web. 21 Oct 2017.

Vancouver:

Li R. Abl Kinases Modulate Epithelial Architecture by Regulating Beta1 Integrin and c-Met Signals. [Internet] [Thesis]. Duke University; 2011. [cited 2017 Oct 21]. Available from: http://hdl.handle.net/10161/5025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li R. Abl Kinases Modulate Epithelial Architecture by Regulating Beta1 Integrin and c-Met Signals. [Thesis]. Duke University; 2011. Available from: http://hdl.handle.net/10161/5025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

11. Pham, Trang Thuy. A Role for Inositol Hexakisphosphate in N-­terminal Acetylation and Mitochondrial Distribution.

Degree: 2012, Duke University

  Inositol phosphates (IPs) are versatile metabolites that play important roles in multiple cellular processes. They have been considered signaling messengers that relay extracellular signals… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Pham, T. T. (2012). A Role for Inositol Hexakisphosphate in N-­terminal Acetylation and Mitochondrial Distribution. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/6147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pham, Trang Thuy. “A Role for Inositol Hexakisphosphate in N-­terminal Acetylation and Mitochondrial Distribution.” 2012. Thesis, Duke University. Accessed October 21, 2017. http://hdl.handle.net/10161/6147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pham, Trang Thuy. “A Role for Inositol Hexakisphosphate in N-­terminal Acetylation and Mitochondrial Distribution.” 2012. Web. 21 Oct 2017.

Vancouver:

Pham TT. A Role for Inositol Hexakisphosphate in N-­terminal Acetylation and Mitochondrial Distribution. [Internet] [Thesis]. Duke University; 2012. [cited 2017 Oct 21]. Available from: http://hdl.handle.net/10161/6147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pham TT. A Role for Inositol Hexakisphosphate in N-­terminal Acetylation and Mitochondrial Distribution. [Thesis]. Duke University; 2012. Available from: http://hdl.handle.net/10161/6147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

12. Yano, Hideaki. Deconstructing G Protein-Coupled Receptor Dimer Pharmacology: Case Studies in Dopamine D1 and D2 Receptors.

Degree: PhD, Pharmacology and Molecular SignalingPsychiatryPharmacology, 2012, Columbia University

 Dopamine receptors mediate various important neurophysiological functions. At a molecular level, G protein coupling is considered the main activation mechanism for most of the receptor-mediated… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Yano, H. (2012). Deconstructing G Protein-Coupled Receptor Dimer Pharmacology: Case Studies in Dopamine D1 and D2 Receptors. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:148832 %%% handle:10022/AC

Chicago Manual of Style (16th Edition):

Yano, Hideaki. “Deconstructing G Protein-Coupled Receptor Dimer Pharmacology: Case Studies in Dopamine D1 and D2 Receptors.” 2012. Doctoral Dissertation, Columbia University. Accessed October 21, 2017. https://academiccommons.columbia.edu/catalog/ac:148832 %%% handle:10022/AC.

MLA Handbook (7th Edition):

Yano, Hideaki. “Deconstructing G Protein-Coupled Receptor Dimer Pharmacology: Case Studies in Dopamine D1 and D2 Receptors.” 2012. Web. 21 Oct 2017.

Vancouver:

Yano H. Deconstructing G Protein-Coupled Receptor Dimer Pharmacology: Case Studies in Dopamine D1 and D2 Receptors. [Internet] [Doctoral dissertation]. Columbia University; 2012. [cited 2017 Oct 21]. Available from: https://academiccommons.columbia.edu/catalog/ac:148832 %%% handle:10022/AC.

Council of Science Editors:

Yano H. Deconstructing G Protein-Coupled Receptor Dimer Pharmacology: Case Studies in Dopamine D1 and D2 Receptors. [Doctoral Dissertation]. Columbia University; 2012. Available from: https://academiccommons.columbia.edu/catalog/ac:148832 %%% handle:10022/AC


Columbia University

13. Donthamsetti, Prashant Chandra. Dissecting Dopamine D2 Receptor Signaling.

Degree: PhD, Pharmacology and Molecular Signaling, 2015, Columbia University

 Dopamine D2 receptor (D2R) is a G protein-coupled receptor (GPCR) that activates G protein and arrestin signaling molecules. D2R antagonism has been a hallmark of… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Donthamsetti, P. C. (2015). Dissecting Dopamine D2 Receptor Signaling. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:207955 %%% doi:10.7916/D8QN660V

Chicago Manual of Style (16th Edition):

Donthamsetti, Prashant Chandra. “Dissecting Dopamine D2 Receptor Signaling.” 2015. Doctoral Dissertation, Columbia University. Accessed October 21, 2017. https://academiccommons.columbia.edu/catalog/ac:207955 %%% doi:10.7916/D8QN660V.

MLA Handbook (7th Edition):

Donthamsetti, Prashant Chandra. “Dissecting Dopamine D2 Receptor Signaling.” 2015. Web. 21 Oct 2017.

Vancouver:

Donthamsetti PC. Dissecting Dopamine D2 Receptor Signaling. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2017 Oct 21]. Available from: https://academiccommons.columbia.edu/catalog/ac:207955 %%% doi:10.7916/D8QN660V.

Council of Science Editors:

Donthamsetti PC. Dissecting Dopamine D2 Receptor Signaling. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://academiccommons.columbia.edu/catalog/ac:207955 %%% doi:10.7916/D8QN660V


Columbia University

14. Chan, Priscilla Jay. Functional and Biochemical Characterization of KCNQ1/KCNE1 Subunit Interactions in the Cardiac IKs Potassium Channel.

Degree: PhD, Pharmacology and Molecular SignalingPharmacology, 2011, Columbia University

 The IKs potassium channel, critical to control of heart electrical activity, requires assembly of pore-forming alpha subunits (KCNQ1) and accessory beta (KCNE1) subunits. IKs is… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Chan, P. J. (2011). Functional and Biochemical Characterization of KCNQ1/KCNE1 Subunit Interactions in the Cardiac IKs Potassium Channel. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:141922 %%% handle:10022/AC

Chicago Manual of Style (16th Edition):

Chan, Priscilla Jay. “Functional and Biochemical Characterization of KCNQ1/KCNE1 Subunit Interactions in the Cardiac IKs Potassium Channel.” 2011. Doctoral Dissertation, Columbia University. Accessed October 21, 2017. https://academiccommons.columbia.edu/catalog/ac:141922 %%% handle:10022/AC.

MLA Handbook (7th Edition):

Chan, Priscilla Jay. “Functional and Biochemical Characterization of KCNQ1/KCNE1 Subunit Interactions in the Cardiac IKs Potassium Channel.” 2011. Web. 21 Oct 2017.

Vancouver:

Chan PJ. Functional and Biochemical Characterization of KCNQ1/KCNE1 Subunit Interactions in the Cardiac IKs Potassium Channel. [Internet] [Doctoral dissertation]. Columbia University; 2011. [cited 2017 Oct 21]. Available from: https://academiccommons.columbia.edu/catalog/ac:141922 %%% handle:10022/AC.

Council of Science Editors:

Chan PJ. Functional and Biochemical Characterization of KCNQ1/KCNE1 Subunit Interactions in the Cardiac IKs Potassium Channel. [Doctoral Dissertation]. Columbia University; 2011. Available from: https://academiccommons.columbia.edu/catalog/ac:141922 %%% handle:10022/AC


Columbia University

15. Harleton, Erin Rachel. Phosphorylation of TASK-1 and its role in atrial arrhythmias.

Degree: PhD, Pharmacology and Molecular SignalingPharmacology, 2011, Columbia University

 Atrial fibrillation (AF) is the most common sustained arrhythmia in human patients, and is associated with an increased risk of stroke and other morbidity. Acute-onset… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Harleton, E. R. (2011). Phosphorylation of TASK-1 and its role in atrial arrhythmias. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:168846

Chicago Manual of Style (16th Edition):

Harleton, Erin Rachel. “Phosphorylation of TASK-1 and its role in atrial arrhythmias.” 2011. Doctoral Dissertation, Columbia University. Accessed October 21, 2017. https://academiccommons.columbia.edu/catalog/ac:168846.

MLA Handbook (7th Edition):

Harleton, Erin Rachel. “Phosphorylation of TASK-1 and its role in atrial arrhythmias.” 2011. Web. 21 Oct 2017.

Vancouver:

Harleton ER. Phosphorylation of TASK-1 and its role in atrial arrhythmias. [Internet] [Doctoral dissertation]. Columbia University; 2011. [cited 2017 Oct 21]. Available from: https://academiccommons.columbia.edu/catalog/ac:168846.

Council of Science Editors:

Harleton ER. Phosphorylation of TASK-1 and its role in atrial arrhythmias. [Doctoral Dissertation]. Columbia University; 2011. Available from: https://academiccommons.columbia.edu/catalog/ac:168846


Columbia University

16. Puckerin, Akil Anthony. Inhibition of Voltage-dependent Calcium Channels by Small G-proteins: Determinants, Mechanisms and Applications.

Degree: PhD, Pharmacology and Molecular SignalingPhysiology and Cellular Biophysics, 2015, Columbia University

 In excitable cells, high-voltage activated calcium channels (CaV1/CaV2) are essential for translating electrical signals into biological responses. These channels are multimeric subunit protein complexes composed… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Puckerin, A. A. (2015). Inhibition of Voltage-dependent Calcium Channels by Small G-proteins: Determinants, Mechanisms and Applications. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:187998 %%% doi:10.7916/D8542MQK

Chicago Manual of Style (16th Edition):

Puckerin, Akil Anthony. “Inhibition of Voltage-dependent Calcium Channels by Small G-proteins: Determinants, Mechanisms and Applications.” 2015. Doctoral Dissertation, Columbia University. Accessed October 21, 2017. https://academiccommons.columbia.edu/catalog/ac:187998 %%% doi:10.7916/D8542MQK.

MLA Handbook (7th Edition):

Puckerin, Akil Anthony. “Inhibition of Voltage-dependent Calcium Channels by Small G-proteins: Determinants, Mechanisms and Applications.” 2015. Web. 21 Oct 2017.

Vancouver:

Puckerin AA. Inhibition of Voltage-dependent Calcium Channels by Small G-proteins: Determinants, Mechanisms and Applications. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2017 Oct 21]. Available from: https://academiccommons.columbia.edu/catalog/ac:187998 %%% doi:10.7916/D8542MQK.

Council of Science Editors:

Puckerin AA. Inhibition of Voltage-dependent Calcium Channels by Small G-proteins: Determinants, Mechanisms and Applications. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://academiccommons.columbia.edu/catalog/ac:187998 %%% doi:10.7916/D8542MQK


Columbia University

17. Wang, Mi. The Role of GM-CSF/IL-3/IL-5 Receptor Common β Subunit (CBS) in Hematopoietic Stem and Progenitor Cell (HSPC) Expansion, Monocytosis and Atherosclerosis.

Degree: PhD, Pharmacology and Molecular SignalingMolecular Medicine, 2013, Columbia University

 Atherosclerosis is caused by the recruitment of monocytes to the subendothelial space where they develop into macrophage foam cells and give rise to atherosclerotic plaques.… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Wang, M. (2013). The Role of GM-CSF/IL-3/IL-5 Receptor Common β Subunit (CBS) in Hematopoietic Stem and Progenitor Cell (HSPC) Expansion, Monocytosis and Atherosclerosis. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:188978 %%% doi:10.7916/D8D21X2W

Chicago Manual of Style (16th Edition):

Wang, Mi. “The Role of GM-CSF/IL-3/IL-5 Receptor Common β Subunit (CBS) in Hematopoietic Stem and Progenitor Cell (HSPC) Expansion, Monocytosis and Atherosclerosis.” 2013. Doctoral Dissertation, Columbia University. Accessed October 21, 2017. https://academiccommons.columbia.edu/catalog/ac:188978 %%% doi:10.7916/D8D21X2W.

MLA Handbook (7th Edition):

Wang, Mi. “The Role of GM-CSF/IL-3/IL-5 Receptor Common β Subunit (CBS) in Hematopoietic Stem and Progenitor Cell (HSPC) Expansion, Monocytosis and Atherosclerosis.” 2013. Web. 21 Oct 2017.

Vancouver:

Wang M. The Role of GM-CSF/IL-3/IL-5 Receptor Common β Subunit (CBS) in Hematopoietic Stem and Progenitor Cell (HSPC) Expansion, Monocytosis and Atherosclerosis. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2017 Oct 21]. Available from: https://academiccommons.columbia.edu/catalog/ac:188978 %%% doi:10.7916/D8D21X2W.

Council of Science Editors:

Wang M. The Role of GM-CSF/IL-3/IL-5 Receptor Common β Subunit (CBS) in Hematopoietic Stem and Progenitor Cell (HSPC) Expansion, Monocytosis and Atherosclerosis. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://academiccommons.columbia.edu/catalog/ac:188978 %%% doi:10.7916/D8D21X2W


University of Cape Town

18. Combrinck, Jill Michelle. The role of haem in the mechanism of action of antimalarials in Plasmodium falciparum.

Degree: PhD, Division of Clinical Pharmacology, 2016, University of Cape Town

 The malaria parasite detoxifies host red blood cell derived haem by conversion into the inert biocrystal haemozoin. Inhibiting this critical pathway is proposed to be… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Combrinck, J. M. (2016). The role of haem in the mechanism of action of antimalarials in Plasmodium falciparum. (Doctoral Dissertation). University of Cape Town. Retrieved from http://hdl.handle.net/11427/22760

Chicago Manual of Style (16th Edition):

Combrinck, Jill Michelle. “The role of haem in the mechanism of action of antimalarials in Plasmodium falciparum.” 2016. Doctoral Dissertation, University of Cape Town. Accessed October 21, 2017. http://hdl.handle.net/11427/22760.

MLA Handbook (7th Edition):

Combrinck, Jill Michelle. “The role of haem in the mechanism of action of antimalarials in Plasmodium falciparum.” 2016. Web. 21 Oct 2017.

Vancouver:

Combrinck JM. The role of haem in the mechanism of action of antimalarials in Plasmodium falciparum. [Internet] [Doctoral dissertation]. University of Cape Town; 2016. [cited 2017 Oct 21]. Available from: http://hdl.handle.net/11427/22760.

Council of Science Editors:

Combrinck JM. The role of haem in the mechanism of action of antimalarials in Plasmodium falciparum. [Doctoral Dissertation]. University of Cape Town; 2016. Available from: http://hdl.handle.net/11427/22760


Wayne State University

19. Shen, Min. Overcoming Tumor Drug Resistance By Activating Amp-Activated Protein Kinase And Destabilizing Oncoproteins.

Degree: PhD, Pharmacology, 2013, Wayne State University

  Although considerable progress has been achieved in the field of cancer therapeutics, primary or acquired drug resistance remains a fundamental cause of therapeutic failure… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Shen, M. (2013). Overcoming Tumor Drug Resistance By Activating Amp-Activated Protein Kinase And Destabilizing Oncoproteins. (Doctoral Dissertation). Wayne State University. Retrieved from http://digitalcommons.wayne.edu/oa_dissertations/796

Chicago Manual of Style (16th Edition):

Shen, Min. “Overcoming Tumor Drug Resistance By Activating Amp-Activated Protein Kinase And Destabilizing Oncoproteins.” 2013. Doctoral Dissertation, Wayne State University. Accessed October 21, 2017. http://digitalcommons.wayne.edu/oa_dissertations/796.

MLA Handbook (7th Edition):

Shen, Min. “Overcoming Tumor Drug Resistance By Activating Amp-Activated Protein Kinase And Destabilizing Oncoproteins.” 2013. Web. 21 Oct 2017.

Vancouver:

Shen M. Overcoming Tumor Drug Resistance By Activating Amp-Activated Protein Kinase And Destabilizing Oncoproteins. [Internet] [Doctoral dissertation]. Wayne State University; 2013. [cited 2017 Oct 21]. Available from: http://digitalcommons.wayne.edu/oa_dissertations/796.

Council of Science Editors:

Shen M. Overcoming Tumor Drug Resistance By Activating Amp-Activated Protein Kinase And Destabilizing Oncoproteins. [Doctoral Dissertation]. Wayne State University; 2013. Available from: http://digitalcommons.wayne.edu/oa_dissertations/796


University of Toledo Health Science Campus

20. Saternos, Hannah C. Newly Discovered Muscarinic Acetylcholine Receptors in the Primary Cilia.

Degree: MSP, Pharmaceutical Sciences (Pharmacology/Toxicology), 2017, University of Toledo Health Science Campus

 Primary endothelial cilia are mechanosensory organelles that are projected into the lumen of blood vessels. Defects in cilia assembly or function can lead to multiple… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Saternos, H. C. (2017). Newly Discovered Muscarinic Acetylcholine Receptors in the Primary Cilia. (Masters Thesis). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1468922731

Chicago Manual of Style (16th Edition):

Saternos, Hannah C. “Newly Discovered Muscarinic Acetylcholine Receptors in the Primary Cilia.” 2017. Masters Thesis, University of Toledo Health Science Campus. Accessed October 21, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=mco1468922731.

MLA Handbook (7th Edition):

Saternos, Hannah C. “Newly Discovered Muscarinic Acetylcholine Receptors in the Primary Cilia.” 2017. Web. 21 Oct 2017.

Vancouver:

Saternos HC. Newly Discovered Muscarinic Acetylcholine Receptors in the Primary Cilia. [Internet] [Masters thesis]. University of Toledo Health Science Campus; 2017. [cited 2017 Oct 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1468922731.

Council of Science Editors:

Saternos HC. Newly Discovered Muscarinic Acetylcholine Receptors in the Primary Cilia. [Masters Thesis]. University of Toledo Health Science Campus; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1468922731


UCLA

21. Johnson, Tremylla. Porphyrin production and regulation in different Propionibacterium acnes lineages contribute to acne vulgaris pathogenesis.

Degree: Molecular and Medical Pharmacology, 2017, UCLA

 Propionibacterium acnes is a dominant human skin commensal. It has been implicated in acne pathogenesis, but its role remains unclear. Recent metagenomic studies have revealed… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Johnson, T. (2017). Porphyrin production and regulation in different Propionibacterium acnes lineages contribute to acne vulgaris pathogenesis. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/9g02d15m

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Johnson, Tremylla. “Porphyrin production and regulation in different Propionibacterium acnes lineages contribute to acne vulgaris pathogenesis.” 2017. Thesis, UCLA. Accessed October 21, 2017. http://www.escholarship.org/uc/item/9g02d15m.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Johnson, Tremylla. “Porphyrin production and regulation in different Propionibacterium acnes lineages contribute to acne vulgaris pathogenesis.” 2017. Web. 21 Oct 2017.

Vancouver:

Johnson T. Porphyrin production and regulation in different Propionibacterium acnes lineages contribute to acne vulgaris pathogenesis. [Internet] [Thesis]. UCLA; 2017. [cited 2017 Oct 21]. Available from: http://www.escholarship.org/uc/item/9g02d15m.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Johnson T. Porphyrin production and regulation in different Propionibacterium acnes lineages contribute to acne vulgaris pathogenesis. [Thesis]. UCLA; 2017. Available from: http://www.escholarship.org/uc/item/9g02d15m

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

22. Rymut, Sharon Marie. Microtubule Regulation in Cystic Fibrosis Pathophysiology.

Degree: PhD, Pharmacology, 2015, Case Western Reserve University

 Cystic fibrosis (CF)-related inflammation remains an obstacle in CF treatment and a leading cause for lung disease as the mechanism underlying inflammation is not well… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Rymut, S. M. (2015). Microtubule Regulation in Cystic Fibrosis Pathophysiology. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1432730616

Chicago Manual of Style (16th Edition):

Rymut, Sharon Marie. “Microtubule Regulation in Cystic Fibrosis Pathophysiology.” 2015. Doctoral Dissertation, Case Western Reserve University. Accessed October 21, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=case1432730616.

MLA Handbook (7th Edition):

Rymut, Sharon Marie. “Microtubule Regulation in Cystic Fibrosis Pathophysiology.” 2015. Web. 21 Oct 2017.

Vancouver:

Rymut SM. Microtubule Regulation in Cystic Fibrosis Pathophysiology. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2015. [cited 2017 Oct 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1432730616.

Council of Science Editors:

Rymut SM. Microtubule Regulation in Cystic Fibrosis Pathophysiology. [Doctoral Dissertation]. Case Western Reserve University; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1432730616


University of Toledo

23. Al Omran , Alzahra J. The Effect of Ethanol on Three Types of Ependymal Cilia in The Brain Lateral Ventricle.

Degree: MS, Pharmaceutical Science, 2015, University of Toledo

 Ependymal cells are multiciliated cells that line the central canal of the brain ventricles and the spinal cord, contributing to cerebral spinal fluid (CSF) circulation.… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Al Omran , A. J. (2015). The Effect of Ethanol on Three Types of Ependymal Cilia in The Brain Lateral Ventricle. (Masters Thesis). University of Toledo. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=toledo1434979511

Chicago Manual of Style (16th Edition):

Al Omran , Alzahra J. “The Effect of Ethanol on Three Types of Ependymal Cilia in The Brain Lateral Ventricle.” 2015. Masters Thesis, University of Toledo. Accessed October 21, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1434979511.

MLA Handbook (7th Edition):

Al Omran , Alzahra J. “The Effect of Ethanol on Three Types of Ependymal Cilia in The Brain Lateral Ventricle.” 2015. Web. 21 Oct 2017.

Vancouver:

Al Omran AJ. The Effect of Ethanol on Three Types of Ependymal Cilia in The Brain Lateral Ventricle. [Internet] [Masters thesis]. University of Toledo; 2015. [cited 2017 Oct 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1434979511.

Council of Science Editors:

Al Omran AJ. The Effect of Ethanol on Three Types of Ependymal Cilia in The Brain Lateral Ventricle. [Masters Thesis]. University of Toledo; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1434979511


The Ohio State University

24. Kang, Chen. ION CHANNELS, PROTEIN KINASE C AND CAVEOLAE IN CARDIOPROTECTION.

Degree: PhD, Pharmacy, 2015, The Ohio State University

 Ischemic heart disease is one of the leading causes for death in the whole world. It has been the top one killer. In general, ischemic… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Kang, C. (2015). ION CHANNELS, PROTEIN KINASE C AND CAVEOLAE IN CARDIOPROTECTION. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1449158171

Chicago Manual of Style (16th Edition):

Kang, Chen. “ION CHANNELS, PROTEIN KINASE C AND CAVEOLAE IN CARDIOPROTECTION.” 2015. Doctoral Dissertation, The Ohio State University. Accessed October 21, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1449158171.

MLA Handbook (7th Edition):

Kang, Chen. “ION CHANNELS, PROTEIN KINASE C AND CAVEOLAE IN CARDIOPROTECTION.” 2015. Web. 21 Oct 2017.

Vancouver:

Kang C. ION CHANNELS, PROTEIN KINASE C AND CAVEOLAE IN CARDIOPROTECTION. [Internet] [Doctoral dissertation]. The Ohio State University; 2015. [cited 2017 Oct 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1449158171.

Council of Science Editors:

Kang C. ION CHANNELS, PROTEIN KINASE C AND CAVEOLAE IN CARDIOPROTECTION. [Doctoral Dissertation]. The Ohio State University; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1449158171


UCLA

25. Thosani, Niyati Mehta. ApoE-/- Mice Lacking Hemopexin Develop Increased Atherosclerosis via Mechanisms That Include Oxidative Stress and Altered Macrophage Function.

Degree: Molecular and Medical Pharmacology, 2015, UCLA

 Objective: We previously reported that Hemopexin (Hx), an acute phase protein and a heme scavenger, is significantly increased and associated with proinflammatory HDL under atherogenic… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Thosani, N. M. (2015). ApoE-/- Mice Lacking Hemopexin Develop Increased Atherosclerosis via Mechanisms That Include Oxidative Stress and Altered Macrophage Function. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/8kd0v8cm

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thosani, Niyati Mehta. “ApoE-/- Mice Lacking Hemopexin Develop Increased Atherosclerosis via Mechanisms That Include Oxidative Stress and Altered Macrophage Function.” 2015. Thesis, UCLA. Accessed October 21, 2017. http://www.escholarship.org/uc/item/8kd0v8cm.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thosani, Niyati Mehta. “ApoE-/- Mice Lacking Hemopexin Develop Increased Atherosclerosis via Mechanisms That Include Oxidative Stress and Altered Macrophage Function.” 2015. Web. 21 Oct 2017.

Vancouver:

Thosani NM. ApoE-/- Mice Lacking Hemopexin Develop Increased Atherosclerosis via Mechanisms That Include Oxidative Stress and Altered Macrophage Function. [Internet] [Thesis]. UCLA; 2015. [cited 2017 Oct 21]. Available from: http://www.escholarship.org/uc/item/8kd0v8cm.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thosani NM. ApoE-/- Mice Lacking Hemopexin Develop Increased Atherosclerosis via Mechanisms That Include Oxidative Stress and Altered Macrophage Function. [Thesis]. UCLA; 2015. Available from: http://www.escholarship.org/uc/item/8kd0v8cm

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

26. Zhang, Jin. The subproteome of mitoBKCa channels from cardiomyocytes reveals novel insights into its mitochondrial import mechanism and function.

Degree: Pharmacology, 2016, UCLA

 BKCa channels are widely expressed ion channels and characterized by their large conductance to potassium and sensitivity to calcium and voltage. It is typically observed… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Zhang, J. (2016). The subproteome of mitoBKCa channels from cardiomyocytes reveals novel insights into its mitochondrial import mechanism and function. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/5qr271m5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Jin. “The subproteome of mitoBKCa channels from cardiomyocytes reveals novel insights into its mitochondrial import mechanism and function.” 2016. Thesis, UCLA. Accessed October 21, 2017. http://www.escholarship.org/uc/item/5qr271m5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Jin. “The subproteome of mitoBKCa channels from cardiomyocytes reveals novel insights into its mitochondrial import mechanism and function.” 2016. Web. 21 Oct 2017.

Vancouver:

Zhang J. The subproteome of mitoBKCa channels from cardiomyocytes reveals novel insights into its mitochondrial import mechanism and function. [Internet] [Thesis]. UCLA; 2016. [cited 2017 Oct 21]. Available from: http://www.escholarship.org/uc/item/5qr271m5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang J. The subproteome of mitoBKCa channels from cardiomyocytes reveals novel insights into its mitochondrial import mechanism and function. [Thesis]. UCLA; 2016. Available from: http://www.escholarship.org/uc/item/5qr271m5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

27. Park, John. Mechanisms of Thrombospondin-4 in Pain Modulation.

Degree: Pharmacology and Toxicology, 2014, University of California – Irvine

 Upregulation of the thrombospondin-4 (TSP4) or calcium channel alpha-2-delta-1 subunit (Cava2d1) in the dorsal spinal cord and dorsal root ganglia plays a causal role in… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Park, J. (2014). Mechanisms of Thrombospondin-4 in Pain Modulation. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/2496030f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Park, John. “Mechanisms of Thrombospondin-4 in Pain Modulation.” 2014. Thesis, University of California – Irvine. Accessed October 21, 2017. http://www.escholarship.org/uc/item/2496030f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Park, John. “Mechanisms of Thrombospondin-4 in Pain Modulation.” 2014. Web. 21 Oct 2017.

Vancouver:

Park J. Mechanisms of Thrombospondin-4 in Pain Modulation. [Internet] [Thesis]. University of California – Irvine; 2014. [cited 2017 Oct 21]. Available from: http://www.escholarship.org/uc/item/2496030f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Park J. Mechanisms of Thrombospondin-4 in Pain Modulation. [Thesis]. University of California – Irvine; 2014. Available from: http://www.escholarship.org/uc/item/2496030f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

28. Cohen, Alexa Nichelle. Glucose Metabolism and CD44 in Small Cell Neuroendocrine Carcinoma of the Prostate.

Degree: Molecular and Medical Pharmacology, 2015, UCLA

 Prostatic adenocarcinomas can recur with aggressive and lethal small cell neuroendocrine carcinoma (SCNC). Since glycolysis is a feature of malignancy and the degree generally correlates… (more)

Subjects/Keywords: Pharmacology

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APA (6th Edition):

Cohen, A. N. (2015). Glucose Metabolism and CD44 in Small Cell Neuroendocrine Carcinoma of the Prostate. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/37p2f58x

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cohen, Alexa Nichelle. “Glucose Metabolism and CD44 in Small Cell Neuroendocrine Carcinoma of the Prostate.” 2015. Thesis, UCLA. Accessed October 21, 2017. http://www.escholarship.org/uc/item/37p2f58x.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cohen, Alexa Nichelle. “Glucose Metabolism and CD44 in Small Cell Neuroendocrine Carcinoma of the Prostate.” 2015. Web. 21 Oct 2017.

Vancouver:

Cohen AN. Glucose Metabolism and CD44 in Small Cell Neuroendocrine Carcinoma of the Prostate. [Internet] [Thesis]. UCLA; 2015. [cited 2017 Oct 21]. Available from: http://www.escholarship.org/uc/item/37p2f58x.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cohen AN. Glucose Metabolism and CD44 in Small Cell Neuroendocrine Carcinoma of the Prostate. [Thesis]. UCLA; 2015. Available from: http://www.escholarship.org/uc/item/37p2f58x

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Stellenbosch University

29. Fasinu, Pius Sedowhe. In vitro assessment of some traditional medications used in South Africa for pharmacokinetics drug interaction potential.

Degree: PhD, Medicine, 2013, Stellenbosch University

 ENGLISH ABSTRACT: Introduction Earlier studies have shown the popularity of herbal products among people as traditional, complementary or alternative medication. One of the major clinical… (more)

Subjects/Keywords: Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fasinu, P. S. (2013). In vitro assessment of some traditional medications used in South Africa for pharmacokinetics drug interaction potential. (Doctoral Dissertation). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/85850

Chicago Manual of Style (16th Edition):

Fasinu, Pius Sedowhe. “In vitro assessment of some traditional medications used in South Africa for pharmacokinetics drug interaction potential.” 2013. Doctoral Dissertation, Stellenbosch University. Accessed October 21, 2017. http://hdl.handle.net/10019.1/85850.

MLA Handbook (7th Edition):

Fasinu, Pius Sedowhe. “In vitro assessment of some traditional medications used in South Africa for pharmacokinetics drug interaction potential.” 2013. Web. 21 Oct 2017.

Vancouver:

Fasinu PS. In vitro assessment of some traditional medications used in South Africa for pharmacokinetics drug interaction potential. [Internet] [Doctoral dissertation]. Stellenbosch University; 2013. [cited 2017 Oct 21]. Available from: http://hdl.handle.net/10019.1/85850.

Council of Science Editors:

Fasinu PS. In vitro assessment of some traditional medications used in South Africa for pharmacokinetics drug interaction potential. [Doctoral Dissertation]. Stellenbosch University; 2013. Available from: http://hdl.handle.net/10019.1/85850


Stellenbosch University

30. De Kock, Lizanne. Pharmacokinetics of twice-daily versus once-daily dosing with granular slow-release para-aminosalicylic acid in adults on second-line anti-tuberculosis and antiretroviral treatment.

Degree: MSc in Medical Science, Medicine, 2013, Stellenbosch University

 ENGLISH ABSTRACT: Background: Para-aminosalicylic acid (PAS) is one of the first effective anti-tuberculosis agents and has become one of the principal second-line drugs to treat… (more)

Subjects/Keywords: Pharmacology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

De Kock, L. (2013). Pharmacokinetics of twice-daily versus once-daily dosing with granular slow-release para-aminosalicylic acid in adults on second-line anti-tuberculosis and antiretroviral treatment. (Masters Thesis). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/85726

Chicago Manual of Style (16th Edition):

De Kock, Lizanne. “Pharmacokinetics of twice-daily versus once-daily dosing with granular slow-release para-aminosalicylic acid in adults on second-line anti-tuberculosis and antiretroviral treatment.” 2013. Masters Thesis, Stellenbosch University. Accessed October 21, 2017. http://hdl.handle.net/10019.1/85726.

MLA Handbook (7th Edition):

De Kock, Lizanne. “Pharmacokinetics of twice-daily versus once-daily dosing with granular slow-release para-aminosalicylic acid in adults on second-line anti-tuberculosis and antiretroviral treatment.” 2013. Web. 21 Oct 2017.

Vancouver:

De Kock L. Pharmacokinetics of twice-daily versus once-daily dosing with granular slow-release para-aminosalicylic acid in adults on second-line anti-tuberculosis and antiretroviral treatment. [Internet] [Masters thesis]. Stellenbosch University; 2013. [cited 2017 Oct 21]. Available from: http://hdl.handle.net/10019.1/85726.

Council of Science Editors:

De Kock L. Pharmacokinetics of twice-daily versus once-daily dosing with granular slow-release para-aminosalicylic acid in adults on second-line anti-tuberculosis and antiretroviral treatment. [Masters Thesis]. Stellenbosch University; 2013. Available from: http://hdl.handle.net/10019.1/85726

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