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You searched for subject:(Pharmacology Cellular AND Molecular). Showing records 1 – 30 of 2900 total matches.

[1] [2] [3] [4] [5] … [97]

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Rutgers University

1. Lou, Liping, 1982-. Investigation of the function and regulation of the TRPM7 ion channel in the renal proximal tubule.

Degree: PhD, TRPM7, 2019, Rutgers University

 The TRPM7 (Transient Receptor Potential Melastatin 7) ion channel is a unique member of the TRP channel family, possessing its own functional kinase domain at… (more)

Subjects/Keywords: Pharmacology, Cellular and Molecular; TRP channels

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APA (6th Edition):

Lou, Liping, 1. (2019). Investigation of the function and regulation of the TRPM7 ion channel in the renal proximal tubule. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/60852/

Chicago Manual of Style (16th Edition):

Lou, Liping, 1982-. “Investigation of the function and regulation of the TRPM7 ion channel in the renal proximal tubule.” 2019. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/60852/.

MLA Handbook (7th Edition):

Lou, Liping, 1982-. “Investigation of the function and regulation of the TRPM7 ion channel in the renal proximal tubule.” 2019. Web. 12 Apr 2021.

Vancouver:

Lou, Liping 1. Investigation of the function and regulation of the TRPM7 ion channel in the renal proximal tubule. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60852/.

Council of Science Editors:

Lou, Liping 1. Investigation of the function and regulation of the TRPM7 ion channel in the renal proximal tubule. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60852/


Wright State University

2. Saqar, Wedad Ali. Characterization of Small Conductance Calcium-Activated Potassium Channels in a Human Lens Epithelium Cell Line (B3).

Degree: MS, Pharmacology and Toxicology, 2014, Wright State University

 Small conductance calcium-activated potassium (SK) channels belong to the family of K+ channels. They play a significant role in cell volume regulation and in lens… (more)

Subjects/Keywords: Pharmacology; Cellular Biology; Biophysics; Molecular Biology; pharmacology; cellular biology; biophysics; molecular biology

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APA (6th Edition):

Saqar, W. A. (2014). Characterization of Small Conductance Calcium-Activated Potassium Channels in a Human Lens Epithelium Cell Line (B3). (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1401279427

Chicago Manual of Style (16th Edition):

Saqar, Wedad Ali. “Characterization of Small Conductance Calcium-Activated Potassium Channels in a Human Lens Epithelium Cell Line (B3).” 2014. Masters Thesis, Wright State University. Accessed April 12, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=wright1401279427.

MLA Handbook (7th Edition):

Saqar, Wedad Ali. “Characterization of Small Conductance Calcium-Activated Potassium Channels in a Human Lens Epithelium Cell Line (B3).” 2014. Web. 12 Apr 2021.

Vancouver:

Saqar WA. Characterization of Small Conductance Calcium-Activated Potassium Channels in a Human Lens Epithelium Cell Line (B3). [Internet] [Masters thesis]. Wright State University; 2014. [cited 2021 Apr 12]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1401279427.

Council of Science Editors:

Saqar WA. Characterization of Small Conductance Calcium-Activated Potassium Channels in a Human Lens Epithelium Cell Line (B3). [Masters Thesis]. Wright State University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1401279427


University of Washington

3. Aggarwal, Stacey. Depletion of dAKAP1 signaling complexes accompanies pathological changes in mitochondrial dynamics during breast cancer progression.

Degree: PhD, 2019, University of Washington

 The dual-specificity A-kinase anchoring protein 1 (dAKAP1) is a mitochondrial anchoring protein responsible for recruiting protein kinase A (PKA), along with other signaling molecules, to… (more)

Subjects/Keywords: breast cancer; cancer biology; cellular migration; mesenchymal; metastasis; mitochondria; Cellular biology; Molecular biology; Pharmacology

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APA (6th Edition):

Aggarwal, S. (2019). Depletion of dAKAP1 signaling complexes accompanies pathological changes in mitochondrial dynamics during breast cancer progression. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/43440

Chicago Manual of Style (16th Edition):

Aggarwal, Stacey. “Depletion of dAKAP1 signaling complexes accompanies pathological changes in mitochondrial dynamics during breast cancer progression.” 2019. Doctoral Dissertation, University of Washington. Accessed April 12, 2021. http://hdl.handle.net/1773/43440.

MLA Handbook (7th Edition):

Aggarwal, Stacey. “Depletion of dAKAP1 signaling complexes accompanies pathological changes in mitochondrial dynamics during breast cancer progression.” 2019. Web. 12 Apr 2021.

Vancouver:

Aggarwal S. Depletion of dAKAP1 signaling complexes accompanies pathological changes in mitochondrial dynamics during breast cancer progression. [Internet] [Doctoral dissertation]. University of Washington; 2019. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1773/43440.

Council of Science Editors:

Aggarwal S. Depletion of dAKAP1 signaling complexes accompanies pathological changes in mitochondrial dynamics during breast cancer progression. [Doctoral Dissertation]. University of Washington; 2019. Available from: http://hdl.handle.net/1773/43440


University of California – San Diego

4. Smith, Thomas Horace. Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 G protein-coupled receptors (GPCRs) are transmembrane proteins that allow cells to respond to extracellular stimuli. GPCR activation occurs when a ligand binds to the extracellular… (more)

Subjects/Keywords: Pharmacology; Molecular biology; Cellular biology; dimerization; endocytosis; GPCR; thrombin

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APA (6th Edition):

Smith, T. H. (2016). Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/93g0t82v

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Thomas Horace. “Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling.” 2016. Thesis, University of California – San Diego. Accessed April 12, 2021. http://www.escholarship.org/uc/item/93g0t82v.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Thomas Horace. “Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling.” 2016. Web. 12 Apr 2021.

Vancouver:

Smith TH. Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2021 Apr 12]. Available from: http://www.escholarship.org/uc/item/93g0t82v.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith TH. Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/93g0t82v

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

5. Bone, Derek B J. Purine Transport and Metabolism in Microvascular Endothelial Cells.

Degree: 2011, University of Western Ontario

 The microvascular endothelium serves as the barrier between the blood and perfused tissues. Proper function of the endothelium is dependent on the ability of the… (more)

Subjects/Keywords: adenosine; hypoxanthine; microvascular endothelium; transporters; Cellular and Molecular Physiology; Pharmacology

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APA (6th Edition):

Bone, D. B. J. (2011). Purine Transport and Metabolism in Microvascular Endothelial Cells. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/241

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bone, Derek B J. “Purine Transport and Metabolism in Microvascular Endothelial Cells.” 2011. Thesis, University of Western Ontario. Accessed April 12, 2021. https://ir.lib.uwo.ca/etd/241.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bone, Derek B J. “Purine Transport and Metabolism in Microvascular Endothelial Cells.” 2011. Web. 12 Apr 2021.

Vancouver:

Bone DBJ. Purine Transport and Metabolism in Microvascular Endothelial Cells. [Internet] [Thesis]. University of Western Ontario; 2011. [cited 2021 Apr 12]. Available from: https://ir.lib.uwo.ca/etd/241.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bone DBJ. Purine Transport and Metabolism in Microvascular Endothelial Cells. [Thesis]. University of Western Ontario; 2011. Available from: https://ir.lib.uwo.ca/etd/241

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

6. McDonnell, Eoin. The Role of Acetylation in the Metabolic Reprogramming of Cancer Cells .

Degree: 2016, Duke University

  Identifying metabolic vulnerabilities of cancer cells remains a subject of investigation for the identification of potential metabolically based therapies for cancer. It is well… (more)

Subjects/Keywords: Molecular biology; Pharmacology; Cellular biology; Acetylation; Cancer; Epigenetics; Histone; Metabolism

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APA (6th Edition):

McDonnell, E. (2016). The Role of Acetylation in the Metabolic Reprogramming of Cancer Cells . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/13387

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McDonnell, Eoin. “The Role of Acetylation in the Metabolic Reprogramming of Cancer Cells .” 2016. Thesis, Duke University. Accessed April 12, 2021. http://hdl.handle.net/10161/13387.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McDonnell, Eoin. “The Role of Acetylation in the Metabolic Reprogramming of Cancer Cells .” 2016. Web. 12 Apr 2021.

Vancouver:

McDonnell E. The Role of Acetylation in the Metabolic Reprogramming of Cancer Cells . [Internet] [Thesis]. Duke University; 2016. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/10161/13387.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McDonnell E. The Role of Acetylation in the Metabolic Reprogramming of Cancer Cells . [Thesis]. Duke University; 2016. Available from: http://hdl.handle.net/10161/13387

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arkansas

7. Kurtz, Samantha Leigh. Development and Characterization of an Autologous Whole Cell Breast Cancer Vaccine.

Degree: MSBME, 2014, University of Arkansas

  Approximately 40,000 women will die from breast cancer in the United States in 2014. About 90% of these deaths will be due to metastases,… (more)

Subjects/Keywords: Immunity; Molecular, Cellular, and Tissue Engineering; Oncology; Pharmacology

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APA (6th Edition):

Kurtz, S. L. (2014). Development and Characterization of an Autologous Whole Cell Breast Cancer Vaccine. (Masters Thesis). University of Arkansas. Retrieved from https://scholarworks.uark.edu/etd/2127

Chicago Manual of Style (16th Edition):

Kurtz, Samantha Leigh. “Development and Characterization of an Autologous Whole Cell Breast Cancer Vaccine.” 2014. Masters Thesis, University of Arkansas. Accessed April 12, 2021. https://scholarworks.uark.edu/etd/2127.

MLA Handbook (7th Edition):

Kurtz, Samantha Leigh. “Development and Characterization of an Autologous Whole Cell Breast Cancer Vaccine.” 2014. Web. 12 Apr 2021.

Vancouver:

Kurtz SL. Development and Characterization of an Autologous Whole Cell Breast Cancer Vaccine. [Internet] [Masters thesis]. University of Arkansas; 2014. [cited 2021 Apr 12]. Available from: https://scholarworks.uark.edu/etd/2127.

Council of Science Editors:

Kurtz SL. Development and Characterization of an Autologous Whole Cell Breast Cancer Vaccine. [Masters Thesis]. University of Arkansas; 2014. Available from: https://scholarworks.uark.edu/etd/2127


University of Toledo Health Science Campus

8. Hossain Saad, Md Zubayer. Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease.

Degree: MSP, Pharmaceutical Sciences (Pharmacology/Toxicology), 2016, University of Toledo Health Science Campus

 Autosomal Dominant Polycystic Kidney Disease (ADPKD) is the most common life-threatening hereditary genetic disease. Early cardiovascular complications, including hypertension, have been witnessed in the majority… (more)

Subjects/Keywords: Pharmacology; Cellular Biology; Molecular Biology; cilia, Polycystic kidney disease, hypertension, endothelium

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APA (6th Edition):

Hossain Saad, M. Z. (2016). Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease. (Masters Thesis). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1461947903

Chicago Manual of Style (16th Edition):

Hossain Saad, Md Zubayer. “Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease.” 2016. Masters Thesis, University of Toledo Health Science Campus. Accessed April 12, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=mco1461947903.

MLA Handbook (7th Edition):

Hossain Saad, Md Zubayer. “Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease.” 2016. Web. 12 Apr 2021.

Vancouver:

Hossain Saad MZ. Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease. [Internet] [Masters thesis]. University of Toledo Health Science Campus; 2016. [cited 2021 Apr 12]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1461947903.

Council of Science Editors:

Hossain Saad MZ. Mechanosensory Role of Vascular Endothelial Primary Cilia in the Development of Hypertension in Polycystic Kidney Disease. [Masters Thesis]. University of Toledo Health Science Campus; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1461947903


Virginia Commonwealth University

9. Shin, Jong M. Role of C121A in mGluR2 homodimeric expression and function.

Degree: MS, Physiology and Biophysics, 2018, Virginia Commonwealth University

  The group II metabotropic glutamate receptors are known for their involvement in various psychiatric disorders. The mGluR2 in particular is linked with etiology of… (more)

Subjects/Keywords: LY341495; GTPyS; Head Twitch Response; LY379268; heteromer; 5-HT2A; Cellular and Molecular Physiology; Molecular and Cellular Neuroscience; Pharmacology

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APA (6th Edition):

Shin, J. M. (2018). Role of C121A in mGluR2 homodimeric expression and function. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/GBP9-YD08 ; https://scholarscompass.vcu.edu/etd/5576

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shin, Jong M. “Role of C121A in mGluR2 homodimeric expression and function.” 2018. Thesis, Virginia Commonwealth University. Accessed April 12, 2021. https://doi.org/10.25772/GBP9-YD08 ; https://scholarscompass.vcu.edu/etd/5576.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shin, Jong M. “Role of C121A in mGluR2 homodimeric expression and function.” 2018. Web. 12 Apr 2021.

Vancouver:

Shin JM. Role of C121A in mGluR2 homodimeric expression and function. [Internet] [Thesis]. Virginia Commonwealth University; 2018. [cited 2021 Apr 12]. Available from: https://doi.org/10.25772/GBP9-YD08 ; https://scholarscompass.vcu.edu/etd/5576.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shin JM. Role of C121A in mGluR2 homodimeric expression and function. [Thesis]. Virginia Commonwealth University; 2018. Available from: https://doi.org/10.25772/GBP9-YD08 ; https://scholarscompass.vcu.edu/etd/5576

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

10. Narla, V V Chakravarthi. Corticotropin releasing factor receptor type 1 signaling in epilepsy and traumatic brain injury.

Degree: 2016, University of Western Ontario

 Stress increases the frequency by which epileptic seizures occur. Corticotropin-releasing factor (CRF) coordinates neuroendocrine, autonomic and behavioral response to stress. This thesis sought to study… (more)

Subjects/Keywords: Epilepsy; Stress; Anxiety; Traumatic brain injury; Corticotropin releasing factor; Piriform Cortex; Cellular and Molecular Physiology; Molecular and Cellular Neuroscience; Pharmacology

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APA (6th Edition):

Narla, V. V. C. (2016). Corticotropin releasing factor receptor type 1 signaling in epilepsy and traumatic brain injury. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4218

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Narla, V V Chakravarthi. “Corticotropin releasing factor receptor type 1 signaling in epilepsy and traumatic brain injury.” 2016. Thesis, University of Western Ontario. Accessed April 12, 2021. https://ir.lib.uwo.ca/etd/4218.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Narla, V V Chakravarthi. “Corticotropin releasing factor receptor type 1 signaling in epilepsy and traumatic brain injury.” 2016. Web. 12 Apr 2021.

Vancouver:

Narla VVC. Corticotropin releasing factor receptor type 1 signaling in epilepsy and traumatic brain injury. [Internet] [Thesis]. University of Western Ontario; 2016. [cited 2021 Apr 12]. Available from: https://ir.lib.uwo.ca/etd/4218.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Narla VVC. Corticotropin releasing factor receptor type 1 signaling in epilepsy and traumatic brain injury. [Thesis]. University of Western Ontario; 2016. Available from: https://ir.lib.uwo.ca/etd/4218

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan Technological University

11. Chapp, Andrew. ACETATE AS AN ACTIVE METABOLITE OF ETHANOL: NEURAL AND CARDIOVASCULAR IMPLICATIONS.

Degree: PhD, Department of Biological Sciences, 2017, Michigan Technological University

  Alcohol use disorders (AUD) and alcohol associated central nervous system (CNS) pathologies, including hypertension, excitotoxicity and dependence remain a large component of excessive ethanol… (more)

Subjects/Keywords: Acetate; Apoptosis; Ethanol; NMDAR; Sympathoexcitation; Cellular and Molecular Physiology; Molecular and Cellular Neuroscience; Pharmacology; Systems and Integrative Physiology; Systems Neuroscience; Toxicology

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APA (6th Edition):

Chapp, A. (2017). ACETATE AS AN ACTIVE METABOLITE OF ETHANOL: NEURAL AND CARDIOVASCULAR IMPLICATIONS. (Doctoral Dissertation). Michigan Technological University. Retrieved from https://digitalcommons.mtu.edu/etdr/490

Chicago Manual of Style (16th Edition):

Chapp, Andrew. “ACETATE AS AN ACTIVE METABOLITE OF ETHANOL: NEURAL AND CARDIOVASCULAR IMPLICATIONS.” 2017. Doctoral Dissertation, Michigan Technological University. Accessed April 12, 2021. https://digitalcommons.mtu.edu/etdr/490.

MLA Handbook (7th Edition):

Chapp, Andrew. “ACETATE AS AN ACTIVE METABOLITE OF ETHANOL: NEURAL AND CARDIOVASCULAR IMPLICATIONS.” 2017. Web. 12 Apr 2021.

Vancouver:

Chapp A. ACETATE AS AN ACTIVE METABOLITE OF ETHANOL: NEURAL AND CARDIOVASCULAR IMPLICATIONS. [Internet] [Doctoral dissertation]. Michigan Technological University; 2017. [cited 2021 Apr 12]. Available from: https://digitalcommons.mtu.edu/etdr/490.

Council of Science Editors:

Chapp A. ACETATE AS AN ACTIVE METABOLITE OF ETHANOL: NEURAL AND CARDIOVASCULAR IMPLICATIONS. [Doctoral Dissertation]. Michigan Technological University; 2017. Available from: https://digitalcommons.mtu.edu/etdr/490


University of Washington

12. Turnham, Rigney Elizabeth. Uncovering pathogenesis of the DNAJ-PKAc fusion in fibrolamellar carcinoma.

Degree: PhD, 2019, University of Washington

 Fibrolamellar carcinoma (FLC) is a rare liver cancer that occurs in healthy adolescents and young adults. FLCs uniquely produce DNAJ-PKAc, a de novo chimeric enzyme… (more)

Subjects/Keywords: DNAJ-PKAc; fibrolamellar carcinoma; in vitro model; Molecular biology; Biochemistry; Cellular biology; Pharmacology

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APA (6th Edition):

Turnham, R. E. (2019). Uncovering pathogenesis of the DNAJ-PKAc fusion in fibrolamellar carcinoma. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/43439

Chicago Manual of Style (16th Edition):

Turnham, Rigney Elizabeth. “Uncovering pathogenesis of the DNAJ-PKAc fusion in fibrolamellar carcinoma.” 2019. Doctoral Dissertation, University of Washington. Accessed April 12, 2021. http://hdl.handle.net/1773/43439.

MLA Handbook (7th Edition):

Turnham, Rigney Elizabeth. “Uncovering pathogenesis of the DNAJ-PKAc fusion in fibrolamellar carcinoma.” 2019. Web. 12 Apr 2021.

Vancouver:

Turnham RE. Uncovering pathogenesis of the DNAJ-PKAc fusion in fibrolamellar carcinoma. [Internet] [Doctoral dissertation]. University of Washington; 2019. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1773/43439.

Council of Science Editors:

Turnham RE. Uncovering pathogenesis of the DNAJ-PKAc fusion in fibrolamellar carcinoma. [Doctoral Dissertation]. University of Washington; 2019. Available from: http://hdl.handle.net/1773/43439


University of Western Ontario

13. Dakroub, Mouhamed. Cardioprotective Role of the Cholinergic System.

Degree: 2015, University of Western Ontario

 The process of aging is an irreversible continuum experienced by all individuals. A large number of physiological transformations occur to the cardiovascular system as one… (more)

Subjects/Keywords: Aging; Heart failure; Cholinergic; Cellular and Molecular Physiology; Medicinal Chemistry and Pharmaceutics; Other Physiology; Pharmacology

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APA (6th Edition):

Dakroub, M. (2015). Cardioprotective Role of the Cholinergic System. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/3425

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dakroub, Mouhamed. “Cardioprotective Role of the Cholinergic System.” 2015. Thesis, University of Western Ontario. Accessed April 12, 2021. https://ir.lib.uwo.ca/etd/3425.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dakroub, Mouhamed. “Cardioprotective Role of the Cholinergic System.” 2015. Web. 12 Apr 2021.

Vancouver:

Dakroub M. Cardioprotective Role of the Cholinergic System. [Internet] [Thesis]. University of Western Ontario; 2015. [cited 2021 Apr 12]. Available from: https://ir.lib.uwo.ca/etd/3425.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dakroub M. Cardioprotective Role of the Cholinergic System. [Thesis]. University of Western Ontario; 2015. Available from: https://ir.lib.uwo.ca/etd/3425

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

14. Hoj, Jacob Peter. The Characterization of Tyrosine Kinase-Dependent Signaling Networks Required for Lung Cancer Brain Metastasis .

Degree: 2020, Duke University

  Brain metastases are a devastating consequence of lung cancer resulting in significantly increased mortality. Currently, no effective therapies exist to treat brain metastases due… (more)

Subjects/Keywords: Pharmacology; Cellular biology; Molecular biology; ABL kinases; AXL; Brain metastasis; HSF1; L1CAM; Lung adenocarcinoma

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APA (6th Edition):

Hoj, J. P. (2020). The Characterization of Tyrosine Kinase-Dependent Signaling Networks Required for Lung Cancer Brain Metastasis . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/20910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoj, Jacob Peter. “The Characterization of Tyrosine Kinase-Dependent Signaling Networks Required for Lung Cancer Brain Metastasis .” 2020. Thesis, Duke University. Accessed April 12, 2021. http://hdl.handle.net/10161/20910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoj, Jacob Peter. “The Characterization of Tyrosine Kinase-Dependent Signaling Networks Required for Lung Cancer Brain Metastasis .” 2020. Web. 12 Apr 2021.

Vancouver:

Hoj JP. The Characterization of Tyrosine Kinase-Dependent Signaling Networks Required for Lung Cancer Brain Metastasis . [Internet] [Thesis]. Duke University; 2020. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/10161/20910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoj JP. The Characterization of Tyrosine Kinase-Dependent Signaling Networks Required for Lung Cancer Brain Metastasis . [Thesis]. Duke University; 2020. Available from: http://hdl.handle.net/10161/20910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

15. Beltejar, Michael Claude. Analyses of PDE-regulated phosphoproteomes reveal unique and specific cAMP signaling modules in T cells.

Degree: PhD, 2017, University of Washington

 Specific functions for different cyclic nucleotide phosphodiesterases (PDEs) have not yet been identified in most cell types. Conventional approaches to study PDE function typically rely… (more)

Subjects/Keywords: Cyclic AMP; Mass Spectrometry; Phosphodiesterase; Phosphoproteomics; Protein Kinase A; Cellular biology; Pharmacology; Molecular and cellular biology

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APA (6th Edition):

Beltejar, M. C. (2017). Analyses of PDE-regulated phosphoproteomes reveal unique and specific cAMP signaling modules in T cells. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/40262

Chicago Manual of Style (16th Edition):

Beltejar, Michael Claude. “Analyses of PDE-regulated phosphoproteomes reveal unique and specific cAMP signaling modules in T cells.” 2017. Doctoral Dissertation, University of Washington. Accessed April 12, 2021. http://hdl.handle.net/1773/40262.

MLA Handbook (7th Edition):

Beltejar, Michael Claude. “Analyses of PDE-regulated phosphoproteomes reveal unique and specific cAMP signaling modules in T cells.” 2017. Web. 12 Apr 2021.

Vancouver:

Beltejar MC. Analyses of PDE-regulated phosphoproteomes reveal unique and specific cAMP signaling modules in T cells. [Internet] [Doctoral dissertation]. University of Washington; 2017. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1773/40262.

Council of Science Editors:

Beltejar MC. Analyses of PDE-regulated phosphoproteomes reveal unique and specific cAMP signaling modules in T cells. [Doctoral Dissertation]. University of Washington; 2017. Available from: http://hdl.handle.net/1773/40262


Wright State University

16. Ravilla, Nagendra Babu. K-Cl Cotransport: Role of KCC3 in cellular Potassium (K) homeostasis in KCC3- transfected HEK-293 cells.

Degree: MS, Pharmacology and Toxicology, 2013, Wright State University

 K-Cl cotransport (KCC) mediated by four protein isoforms, KCC1 to KCC4, plays a significant role in cell volume regulation, and in K and Cl homeostasis.… (more)

Subjects/Keywords: Biophysics; Cellular Biology; Molecular Biology; Pharmacology; Physiology; K-Cl cotransport, KCC3, KCC3 mutations, cellular K homeostasis, cell volume regulation

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APA (6th Edition):

Ravilla, N. B. (2013). K-Cl Cotransport: Role of KCC3 in cellular Potassium (K) homeostasis in KCC3- transfected HEK-293 cells. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1377519708

Chicago Manual of Style (16th Edition):

Ravilla, Nagendra Babu. “K-Cl Cotransport: Role of KCC3 in cellular Potassium (K) homeostasis in KCC3- transfected HEK-293 cells.” 2013. Masters Thesis, Wright State University. Accessed April 12, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=wright1377519708.

MLA Handbook (7th Edition):

Ravilla, Nagendra Babu. “K-Cl Cotransport: Role of KCC3 in cellular Potassium (K) homeostasis in KCC3- transfected HEK-293 cells.” 2013. Web. 12 Apr 2021.

Vancouver:

Ravilla NB. K-Cl Cotransport: Role of KCC3 in cellular Potassium (K) homeostasis in KCC3- transfected HEK-293 cells. [Internet] [Masters thesis]. Wright State University; 2013. [cited 2021 Apr 12]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1377519708.

Council of Science Editors:

Ravilla NB. K-Cl Cotransport: Role of KCC3 in cellular Potassium (K) homeostasis in KCC3- transfected HEK-293 cells. [Masters Thesis]. Wright State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1377519708


University of Washington

17. Bucko, Paula Jolanta. Decoding protein kinase signaling during mitosis: Exploiting molecular scaffolds and developing drug-targeting tools for studying local kinase biology.

Degree: PhD, 2020, University of Washington

 Fundamental cellular processes such as cell division, migration, differentiation, and growth require environmental signals to be converted into chemical responses that generate biological outputs. Accordingly,… (more)

Subjects/Keywords: Aurora A; drug-targeting; Gravin; local kinase biology; molecular scaffolds; polo-like kinase 1; Cellular biology; Molecular biology; Biochemistry; Pharmacology

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APA (6th Edition):

Bucko, P. J. (2020). Decoding protein kinase signaling during mitosis: Exploiting molecular scaffolds and developing drug-targeting tools for studying local kinase biology. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/46541

Chicago Manual of Style (16th Edition):

Bucko, Paula Jolanta. “Decoding protein kinase signaling during mitosis: Exploiting molecular scaffolds and developing drug-targeting tools for studying local kinase biology.” 2020. Doctoral Dissertation, University of Washington. Accessed April 12, 2021. http://hdl.handle.net/1773/46541.

MLA Handbook (7th Edition):

Bucko, Paula Jolanta. “Decoding protein kinase signaling during mitosis: Exploiting molecular scaffolds and developing drug-targeting tools for studying local kinase biology.” 2020. Web. 12 Apr 2021.

Vancouver:

Bucko PJ. Decoding protein kinase signaling during mitosis: Exploiting molecular scaffolds and developing drug-targeting tools for studying local kinase biology. [Internet] [Doctoral dissertation]. University of Washington; 2020. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1773/46541.

Council of Science Editors:

Bucko PJ. Decoding protein kinase signaling during mitosis: Exploiting molecular scaffolds and developing drug-targeting tools for studying local kinase biology. [Doctoral Dissertation]. University of Washington; 2020. Available from: http://hdl.handle.net/1773/46541


Texas Medical Center

18. Howe, Matthew D. The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy.

Degree: PhD, 2018, Texas Medical Center

  In healthy brain tissue, toxic amyloid-β (Aβ) proteins are transported by the pulsatile flow of cerebrospinal fluid (CSF) along perivascular drainage pathways. Ischemic stroke… (more)

Subjects/Keywords: Stroke; Fibronectin; Integrin; Amyloid; CAA; Glymphatic; CSF; TGF-beta; Astrocytes; Neurodegeneration; Biology; Cellular and Molecular Physiology; Medicine and Health Sciences; Molecular and Cellular Neuroscience; Pharmacology; Translational Medical Research

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APA (6th Edition):

Howe, M. D. (2018). The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/875

Chicago Manual of Style (16th Edition):

Howe, Matthew D. “The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy.” 2018. Doctoral Dissertation, Texas Medical Center. Accessed April 12, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/875.

MLA Handbook (7th Edition):

Howe, Matthew D. “The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy.” 2018. Web. 12 Apr 2021.

Vancouver:

Howe MD. The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy. [Internet] [Doctoral dissertation]. Texas Medical Center; 2018. [cited 2021 Apr 12]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/875.

Council of Science Editors:

Howe MD. The Role of Perivascular FIbrosis in Post-Stroke Glymphatic Impairment and Cerebral Amyloid Angiopathy. [Doctoral Dissertation]. Texas Medical Center; 2018. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/875

19. Kosenko, Anastasia. KCNQ2 Channels: Dynamic Molecular Interactions and Functional Role in Learning and Memory.

Degree: Pharmacology and Toxicology, 2015, University of California – Irvine

 Voltage-gated ion channels encoded by the members of KCNQ gene family (KCNQ2-5) conduct the M-type potassium current. Several neurotransmitters and signaling events have been shown… (more)

Subjects/Keywords: Pharmacology; Neurosciences; Molecular biology; cellular signaling pathway; ion channel; learning and memory behavior; neuronal excitability; protein-protein interaction

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APA (6th Edition):

Kosenko, A. (2015). KCNQ2 Channels: Dynamic Molecular Interactions and Functional Role in Learning and Memory. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/1sj0n3md

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kosenko, Anastasia. “KCNQ2 Channels: Dynamic Molecular Interactions and Functional Role in Learning and Memory.” 2015. Thesis, University of California – Irvine. Accessed April 12, 2021. http://www.escholarship.org/uc/item/1sj0n3md.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kosenko, Anastasia. “KCNQ2 Channels: Dynamic Molecular Interactions and Functional Role in Learning and Memory.” 2015. Web. 12 Apr 2021.

Vancouver:

Kosenko A. KCNQ2 Channels: Dynamic Molecular Interactions and Functional Role in Learning and Memory. [Internet] [Thesis]. University of California – Irvine; 2015. [cited 2021 Apr 12]. Available from: http://www.escholarship.org/uc/item/1sj0n3md.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kosenko A. KCNQ2 Channels: Dynamic Molecular Interactions and Functional Role in Learning and Memory. [Thesis]. University of California – Irvine; 2015. Available from: http://www.escholarship.org/uc/item/1sj0n3md

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Kaissarian, Nayiri. Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease.

Degree: PhD, Pharmacology, 2017, University of Michigan

 Fabry disease is a rare, X-linked lysosomal storage disease arising from deficiency of the lysosomal hydrolase, α-galactosidase A (GLA). Reduced GLA activity disrupts glycosphingolipid (GSL)… (more)

Subjects/Keywords: Fabry disease; Endothelial dysfunction; Biological Chemistry; Molecular, Cellular and Developmental Biology; Pharmacy and Pharmacology; Physiology; Science (General); Health Sciences; Science

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APA (6th Edition):

Kaissarian, N. (2017). Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/138731

Chicago Manual of Style (16th Edition):

Kaissarian, Nayiri. “Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease.” 2017. Doctoral Dissertation, University of Michigan. Accessed April 12, 2021. http://hdl.handle.net/2027.42/138731.

MLA Handbook (7th Edition):

Kaissarian, Nayiri. “Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease.” 2017. Web. 12 Apr 2021.

Vancouver:

Kaissarian N. Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/2027.42/138731.

Council of Science Editors:

Kaissarian N. Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/138731


West Virginia University

21. Zhang, Zhuo. Vanadate-induced cell cycle regulation and its signal transduction pathway.

Degree: PhD, Microbiology, Immunology, and Cell Biology, 2002, West Virginia University

 Vanadate is potent toxic and carcinogenic agent. The mechanisms of its toxic and carcinogenic actions are still under investigation. Cell cycle arrest is an important… (more)

Subjects/Keywords: Molecular biology; Pharmacology; Oncology; Toxicology; Cellular biology

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APA (6th Edition):

Zhang, Z. (2002). Vanadate-induced cell cycle regulation and its signal transduction pathway. (Doctoral Dissertation). West Virginia University. Retrieved from https://doi.org/10.33915/etd.1655 ; https://researchrepository.wvu.edu/etd/1655

Chicago Manual of Style (16th Edition):

Zhang, Zhuo. “Vanadate-induced cell cycle regulation and its signal transduction pathway.” 2002. Doctoral Dissertation, West Virginia University. Accessed April 12, 2021. https://doi.org/10.33915/etd.1655 ; https://researchrepository.wvu.edu/etd/1655.

MLA Handbook (7th Edition):

Zhang, Zhuo. “Vanadate-induced cell cycle regulation and its signal transduction pathway.” 2002. Web. 12 Apr 2021.

Vancouver:

Zhang Z. Vanadate-induced cell cycle regulation and its signal transduction pathway. [Internet] [Doctoral dissertation]. West Virginia University; 2002. [cited 2021 Apr 12]. Available from: https://doi.org/10.33915/etd.1655 ; https://researchrepository.wvu.edu/etd/1655.

Council of Science Editors:

Zhang Z. Vanadate-induced cell cycle regulation and its signal transduction pathway. [Doctoral Dissertation]. West Virginia University; 2002. Available from: https://doi.org/10.33915/etd.1655 ; https://researchrepository.wvu.edu/etd/1655


Virginia Commonwealth University

22. Marks, William D. The effects of the HIV-1 Tat protein and morphine on the structure and function of the hippocampal CA1 subfield.

Degree: PhD, Neuroscience, 2017, Virginia Commonwealth University

  HIV is capable of causing a set of neurological diseases collectively termed the HIV Associated Neurocognitive Disorders (HAND). Worsening pathology is observed in HIV+… (more)

Subjects/Keywords: HIV; Tat; interneuron; morphine; hippocampus; CA1; memory; microcircuit; drug abuse; pathology; Molecular and Cellular Neuroscience; Pharmacology; Systems Neuroscience; Toxicology; Virology

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APA (6th Edition):

Marks, W. D. (2017). The effects of the HIV-1 Tat protein and morphine on the structure and function of the hippocampal CA1 subfield. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/D6TZ-AW62 ; https://scholarscompass.vcu.edu/etd/5168

Chicago Manual of Style (16th Edition):

Marks, William D. “The effects of the HIV-1 Tat protein and morphine on the structure and function of the hippocampal CA1 subfield.” 2017. Doctoral Dissertation, Virginia Commonwealth University. Accessed April 12, 2021. https://doi.org/10.25772/D6TZ-AW62 ; https://scholarscompass.vcu.edu/etd/5168.

MLA Handbook (7th Edition):

Marks, William D. “The effects of the HIV-1 Tat protein and morphine on the structure and function of the hippocampal CA1 subfield.” 2017. Web. 12 Apr 2021.

Vancouver:

Marks WD. The effects of the HIV-1 Tat protein and morphine on the structure and function of the hippocampal CA1 subfield. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2017. [cited 2021 Apr 12]. Available from: https://doi.org/10.25772/D6TZ-AW62 ; https://scholarscompass.vcu.edu/etd/5168.

Council of Science Editors:

Marks WD. The effects of the HIV-1 Tat protein and morphine on the structure and function of the hippocampal CA1 subfield. [Doctoral Dissertation]. Virginia Commonwealth University; 2017. Available from: https://doi.org/10.25772/D6TZ-AW62 ; https://scholarscompass.vcu.edu/etd/5168

23. Prachanronarong, Kristina L. Understanding Drug Resistance and Antibody Neutralization Escape in Antivirals: A Dissertation.

Degree: PhD, Department of Biochemistry and Molecular Pharmacology, 2016, U of Massachusetts : Med

  Antiviral drug resistance is a major problem in the treatment of viral infections, including influenza and hepatitis C virus (HCV). Influenza neuraminidase (NA) is… (more)

Subjects/Keywords: drug resistance; antibody neutralization; antivirals; influenza; Biochemistry; Biophysics; Cellular and Molecular Physiology; Immunoprophylaxis and Therapy; Pharmacology; Structural Biology; Virology; Virus Diseases

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APA (6th Edition):

Prachanronarong, K. L. (2016). Understanding Drug Resistance and Antibody Neutralization Escape in Antivirals: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/840

Chicago Manual of Style (16th Edition):

Prachanronarong, Kristina L. “Understanding Drug Resistance and Antibody Neutralization Escape in Antivirals: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed April 12, 2021. https://escholarship.umassmed.edu/gsbs_diss/840.

MLA Handbook (7th Edition):

Prachanronarong, Kristina L. “Understanding Drug Resistance and Antibody Neutralization Escape in Antivirals: A Dissertation.” 2016. Web. 12 Apr 2021.

Vancouver:

Prachanronarong KL. Understanding Drug Resistance and Antibody Neutralization Escape in Antivirals: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2021 Apr 12]. Available from: https://escholarship.umassmed.edu/gsbs_diss/840.

Council of Science Editors:

Prachanronarong KL. Understanding Drug Resistance and Antibody Neutralization Escape in Antivirals: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: https://escholarship.umassmed.edu/gsbs_diss/840


University of Washington

24. Whiting, Jennifer Lee. The AKAP220 signaling complex regulates renal aquaporin-2 localization.

Degree: PhD, 2015, University of Washington

 A kinase anchoring proteins (AKAPs) localize signaling molecules such as kinases and phosphatases in close proximity to their substrates to control the scope and duration… (more)

Subjects/Keywords: A kinase anchoring protein; aquaporin-2; diabetes insipidus; protein kinase A; signaling scaffold; signal transduction; Molecular biology; Cellular biology; Biochemistry; pharmacology

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APA (6th Edition):

Whiting, J. L. (2015). The AKAP220 signaling complex regulates renal aquaporin-2 localization. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/34119

Chicago Manual of Style (16th Edition):

Whiting, Jennifer Lee. “The AKAP220 signaling complex regulates renal aquaporin-2 localization.” 2015. Doctoral Dissertation, University of Washington. Accessed April 12, 2021. http://hdl.handle.net/1773/34119.

MLA Handbook (7th Edition):

Whiting, Jennifer Lee. “The AKAP220 signaling complex regulates renal aquaporin-2 localization.” 2015. Web. 12 Apr 2021.

Vancouver:

Whiting JL. The AKAP220 signaling complex regulates renal aquaporin-2 localization. [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1773/34119.

Council of Science Editors:

Whiting JL. The AKAP220 signaling complex regulates renal aquaporin-2 localization. [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/34119


University of Western Ontario

25. Engineer, Anish. Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies.

Degree: 2019, University of Western Ontario

 Congenital heart defects (CHDs) arise from perturbations in complex molecular and cellular processes underlying normal embryonic heart development. CHDs are the most common congenital malformation,… (more)

Subjects/Keywords: Pregestational Diabetes; Congenital Heart Defects; Heart Development; Sapropterin; miR-122; coronary arteries; Cardiovascular Diseases; Cellular and Molecular Physiology; Developmental Biology; Pharmacology

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APA (6th Edition):

Engineer, A. (2019). Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/6328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Engineer, Anish. “Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies.” 2019. Thesis, University of Western Ontario. Accessed April 12, 2021. https://ir.lib.uwo.ca/etd/6328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Engineer, Anish. “Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies.” 2019. Web. 12 Apr 2021.

Vancouver:

Engineer A. Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies. [Internet] [Thesis]. University of Western Ontario; 2019. [cited 2021 Apr 12]. Available from: https://ir.lib.uwo.ca/etd/6328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Engineer A. Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies. [Thesis]. University of Western Ontario; 2019. Available from: https://ir.lib.uwo.ca/etd/6328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Coons, Laurel Aubrie. Elucidation of the Molecular Mechanisms Underlying Estrogen-Mediated Estrogen Receptor Activation .

Degree: 2017, Duke University

  Every cell in our body contains the same genetic material, but what differentiates one cell type from another is the way in which that… (more)

Subjects/Keywords: Pharmacology; Molecular biology; Cellular biology; Functional genomics; Gene regulation; High-throughput biology; Microfluidics; Nuclear receptors; Sequence constraints

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APA (6th Edition):

Coons, L. A. (2017). Elucidation of the Molecular Mechanisms Underlying Estrogen-Mediated Estrogen Receptor Activation . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/16283

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Coons, Laurel Aubrie. “Elucidation of the Molecular Mechanisms Underlying Estrogen-Mediated Estrogen Receptor Activation .” 2017. Thesis, Duke University. Accessed April 12, 2021. http://hdl.handle.net/10161/16283.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Coons, Laurel Aubrie. “Elucidation of the Molecular Mechanisms Underlying Estrogen-Mediated Estrogen Receptor Activation .” 2017. Web. 12 Apr 2021.

Vancouver:

Coons LA. Elucidation of the Molecular Mechanisms Underlying Estrogen-Mediated Estrogen Receptor Activation . [Internet] [Thesis]. Duke University; 2017. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/10161/16283.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Coons LA. Elucidation of the Molecular Mechanisms Underlying Estrogen-Mediated Estrogen Receptor Activation . [Thesis]. Duke University; 2017. Available from: http://hdl.handle.net/10161/16283

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kentucky

27. Frazier, Hilaree N. Exploring the Role of Insulin Receptor Signaling in Hippocampal Learning and Memory, Neuronal Calcium Dysregulation, and Glucose Metabolism.

Degree: 2019, University of Kentucky

 In the late 90’s, emerging evidence revealed that the brain is insulin-sensitive, highlighted by broad expression of brain-specific insulin receptors and reports of circulating brain… (more)

Subjects/Keywords: Insulin Receptor; Hippocampus; Aging; Calcium Dysregulation; Glucose Metabolism; Brain Insulin Resistance; Behavioral Neurobiology; Cognitive Neuroscience; Molecular and Cellular Neuroscience; Pharmacology

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APA (6th Edition):

Frazier, H. N. (2019). Exploring the Role of Insulin Receptor Signaling in Hippocampal Learning and Memory, Neuronal Calcium Dysregulation, and Glucose Metabolism. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/pharmacol_etds/32

Chicago Manual of Style (16th Edition):

Frazier, Hilaree N. “Exploring the Role of Insulin Receptor Signaling in Hippocampal Learning and Memory, Neuronal Calcium Dysregulation, and Glucose Metabolism.” 2019. Doctoral Dissertation, University of Kentucky. Accessed April 12, 2021. https://uknowledge.uky.edu/pharmacol_etds/32.

MLA Handbook (7th Edition):

Frazier, Hilaree N. “Exploring the Role of Insulin Receptor Signaling in Hippocampal Learning and Memory, Neuronal Calcium Dysregulation, and Glucose Metabolism.” 2019. Web. 12 Apr 2021.

Vancouver:

Frazier HN. Exploring the Role of Insulin Receptor Signaling in Hippocampal Learning and Memory, Neuronal Calcium Dysregulation, and Glucose Metabolism. [Internet] [Doctoral dissertation]. University of Kentucky; 2019. [cited 2021 Apr 12]. Available from: https://uknowledge.uky.edu/pharmacol_etds/32.

Council of Science Editors:

Frazier HN. Exploring the Role of Insulin Receptor Signaling in Hippocampal Learning and Memory, Neuronal Calcium Dysregulation, and Glucose Metabolism. [Doctoral Dissertation]. University of Kentucky; 2019. Available from: https://uknowledge.uky.edu/pharmacol_etds/32


University of Montana

28. Lyda, Jennene. An Environmental Toxic Model of Parkinson's Disease: The Fruit Fly.

Degree: MS, 2014, University of Montana

  Parkinson’s disease (PD) is the second most common neurodegenerative disorder with no known specific cause; although genetic risk factors and/or environmental exposure are thought… (more)

Subjects/Keywords: Environmental Toxic Model; Parkinson's Disease; Fruit Fly; Molecular and Cellular Neuroscience; Other Pharmacology, Toxicology and Environmental Health; Toxicology

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APA (6th Edition):

Lyda, J. (2014). An Environmental Toxic Model of Parkinson's Disease: The Fruit Fly. (Masters Thesis). University of Montana. Retrieved from https://scholarworks.umt.edu/etd/4380

Chicago Manual of Style (16th Edition):

Lyda, Jennene. “An Environmental Toxic Model of Parkinson's Disease: The Fruit Fly.” 2014. Masters Thesis, University of Montana. Accessed April 12, 2021. https://scholarworks.umt.edu/etd/4380.

MLA Handbook (7th Edition):

Lyda, Jennene. “An Environmental Toxic Model of Parkinson's Disease: The Fruit Fly.” 2014. Web. 12 Apr 2021.

Vancouver:

Lyda J. An Environmental Toxic Model of Parkinson's Disease: The Fruit Fly. [Internet] [Masters thesis]. University of Montana; 2014. [cited 2021 Apr 12]. Available from: https://scholarworks.umt.edu/etd/4380.

Council of Science Editors:

Lyda J. An Environmental Toxic Model of Parkinson's Disease: The Fruit Fly. [Masters Thesis]. University of Montana; 2014. Available from: https://scholarworks.umt.edu/etd/4380


The Ohio State University

29. Henderson, Sally E. Translating the Anti-Tumor/Anti-Cachectic Activity of AR-42, a Novel HDAC Inhibitor, into Pancreatic Cancer Therapy.

Degree: PhD, Comparative and Veterinary Medicine, 2017, The Ohio State University

 Pancreatic cancer is the 3rd leading cause of cancer death in the United States and has a 5-year survival of less than 9% for all… (more)

Subjects/Keywords: Molecular Biology; Pharmacology; Cellular Biology; Pancreatic cancer; muscle wasting; cachexia; mouse models of pancreatic cancer-induced cachexia; AR-42; HDAC inhibitor

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APA (6th Edition):

Henderson, S. E. (2017). Translating the Anti-Tumor/Anti-Cachectic Activity of AR-42, a Novel HDAC Inhibitor, into Pancreatic Cancer Therapy. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1495801301660324

Chicago Manual of Style (16th Edition):

Henderson, Sally E. “Translating the Anti-Tumor/Anti-Cachectic Activity of AR-42, a Novel HDAC Inhibitor, into Pancreatic Cancer Therapy.” 2017. Doctoral Dissertation, The Ohio State University. Accessed April 12, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1495801301660324.

MLA Handbook (7th Edition):

Henderson, Sally E. “Translating the Anti-Tumor/Anti-Cachectic Activity of AR-42, a Novel HDAC Inhibitor, into Pancreatic Cancer Therapy.” 2017. Web. 12 Apr 2021.

Vancouver:

Henderson SE. Translating the Anti-Tumor/Anti-Cachectic Activity of AR-42, a Novel HDAC Inhibitor, into Pancreatic Cancer Therapy. [Internet] [Doctoral dissertation]. The Ohio State University; 2017. [cited 2021 Apr 12]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1495801301660324.

Council of Science Editors:

Henderson SE. Translating the Anti-Tumor/Anti-Cachectic Activity of AR-42, a Novel HDAC Inhibitor, into Pancreatic Cancer Therapy. [Doctoral Dissertation]. The Ohio State University; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1495801301660324

30. Farabaugh, Kenneth Thomas, kt. Insights into a Novel Signaling Pathway that Determines Cell Fate in Response to Hyperosmotic Stress.

Degree: PhD, Pharmacology, 2019, Case Western Reserve University School of Graduate Studies

 Hyperosmotic stress is relevant in both physiological and pathological conditions. The response to hyperosmotic stress includes a vast body of research that with each passing… (more)

Subjects/Keywords: Pharmacology; Biomedical Research; Cellular Biology; Genetics; Molecular Biology; PKR; PACT; NF-kB; p65; c-Rel; hyperosmotic stress; osmoadaptation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Farabaugh, Kenneth Thomas, k. (2019). Insights into a Novel Signaling Pathway that Determines Cell Fate in Response to Hyperosmotic Stress. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1571692276973131

Chicago Manual of Style (16th Edition):

Farabaugh, Kenneth Thomas, kt. “Insights into a Novel Signaling Pathway that Determines Cell Fate in Response to Hyperosmotic Stress.” 2019. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed April 12, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1571692276973131.

MLA Handbook (7th Edition):

Farabaugh, Kenneth Thomas, kt. “Insights into a Novel Signaling Pathway that Determines Cell Fate in Response to Hyperosmotic Stress.” 2019. Web. 12 Apr 2021.

Vancouver:

Farabaugh, Kenneth Thomas k. Insights into a Novel Signaling Pathway that Determines Cell Fate in Response to Hyperosmotic Stress. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2019. [cited 2021 Apr 12]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1571692276973131.

Council of Science Editors:

Farabaugh, Kenneth Thomas k. Insights into a Novel Signaling Pathway that Determines Cell Fate in Response to Hyperosmotic Stress. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2019. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1571692276973131

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