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You searched for subject:(Paxillin). Showing records 1 – 28 of 28 total matches.

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University of Texas Southwestern Medical Center

1. Young, Melissa Rasar. Paxillin is a Novel Regulator of Xenopus Oocyte Maturation.

Degree: 2010, University of Texas Southwestern Medical Center

 Oocyte maturation is triggered by steroids in a transcription-independent fashion that involves an unusual positive feedback loop whereby MOS (a germ cell specific Raf) activates… (more)

Subjects/Keywords: Oocytes; Paxillin; Gene Expression Regulation

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APA (6th Edition):

Young, M. R. (2010). Paxillin is a Novel Regulator of Xenopus Oocyte Maturation. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/786

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Young, Melissa Rasar. “Paxillin is a Novel Regulator of Xenopus Oocyte Maturation.” 2010. Thesis, University of Texas Southwestern Medical Center. Accessed July 17, 2019. http://hdl.handle.net/2152.5/786.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Young, Melissa Rasar. “Paxillin is a Novel Regulator of Xenopus Oocyte Maturation.” 2010. Web. 17 Jul 2019.

Vancouver:

Young MR. Paxillin is a Novel Regulator of Xenopus Oocyte Maturation. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2010. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/2152.5/786.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Young MR. Paxillin is a Novel Regulator of Xenopus Oocyte Maturation. [Thesis]. University of Texas Southwestern Medical Center; 2010. Available from: http://hdl.handle.net/2152.5/786

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Kratimenos, Panagiotis. Η έκφραση της Focal Adhesion Kinase, Src kinase και Paxillin σε κυτταρολογικό υλικό ασθενών με νευρoβλάστωμα.

Degree: 2015, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 Background: Neuroblastoma (NB) is the most common extracranial tumor in children, arising from the neural crest of the sympathetic ganglia and accounts for 7-10% of… (more)

Subjects/Keywords: Νευροβλάστωμα; Παχιλλίνη; Neuroblastoma; Src kinase; FAK; Paxillin

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APA (6th Edition):

Kratimenos, P. (2015). Η έκφραση της Focal Adhesion Kinase, Src kinase και Paxillin σε κυτταρολογικό υλικό ασθενών με νευρoβλάστωμα. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/43066

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kratimenos, Panagiotis. “Η έκφραση της Focal Adhesion Kinase, Src kinase και Paxillin σε κυτταρολογικό υλικό ασθενών με νευρoβλάστωμα.” 2015. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed July 17, 2019. http://hdl.handle.net/10442/hedi/43066.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kratimenos, Panagiotis. “Η έκφραση της Focal Adhesion Kinase, Src kinase και Paxillin σε κυτταρολογικό υλικό ασθενών με νευρoβλάστωμα.” 2015. Web. 17 Jul 2019.

Vancouver:

Kratimenos P. Η έκφραση της Focal Adhesion Kinase, Src kinase και Paxillin σε κυτταρολογικό υλικό ασθενών με νευρoβλάστωμα. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/10442/hedi/43066.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kratimenos P. Η έκφραση της Focal Adhesion Kinase, Src kinase και Paxillin σε κυτταρολογικό υλικό ασθενών με νευρoβλάστωμα. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. Available from: http://hdl.handle.net/10442/hedi/43066

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

3. St-Pierre, Joëlle. Examination of the Regulation of Phosphorylation Events in Macrophage Adhesion and Response to Zymosan.

Degree: PhD, Department of Medical Microbiology and Immunology, 2012, University of Alberta

 Macrophages play a central role in innate immunity, most notably in tissue repair, phagocytosis of dead or infected cells, secretion of chemokine and cytokines at… (more)

Subjects/Keywords: macrophage; CD45; paxillin; Pyk2; adhesion; calpain

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APA (6th Edition):

St-Pierre, J. (2012). Examination of the Regulation of Phosphorylation Events in Macrophage Adhesion and Response to Zymosan. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/3r074v72p

Chicago Manual of Style (16th Edition):

St-Pierre, Joëlle. “Examination of the Regulation of Phosphorylation Events in Macrophage Adhesion and Response to Zymosan.” 2012. Doctoral Dissertation, University of Alberta. Accessed July 17, 2019. https://era.library.ualberta.ca/files/3r074v72p.

MLA Handbook (7th Edition):

St-Pierre, Joëlle. “Examination of the Regulation of Phosphorylation Events in Macrophage Adhesion and Response to Zymosan.” 2012. Web. 17 Jul 2019.

Vancouver:

St-Pierre J. Examination of the Regulation of Phosphorylation Events in Macrophage Adhesion and Response to Zymosan. [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2019 Jul 17]. Available from: https://era.library.ualberta.ca/files/3r074v72p.

Council of Science Editors:

St-Pierre J. Examination of the Regulation of Phosphorylation Events in Macrophage Adhesion and Response to Zymosan. [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/3r074v72p


Cornell University

4. Yoo, Sungsoo. A Novel Role For The Adaptor Protein And Arf Gtpase-Activating Protein Cat-1/Git-1 In Cellular Transformation .

Degree: 2012, Cornell University

 Cat-1/Git-1 is a multi-functional protein that acts as a GAP (GTPase-activating protein) for Arf GTPases, as well as serves as a scaffold for a number… (more)

Subjects/Keywords: Cat-1; Git1; transformation; paxillin; Arf GTPase

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APA (6th Edition):

Yoo, S. (2012). A Novel Role For The Adaptor Protein And Arf Gtpase-Activating Protein Cat-1/Git-1 In Cellular Transformation . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/31160

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yoo, Sungsoo. “A Novel Role For The Adaptor Protein And Arf Gtpase-Activating Protein Cat-1/Git-1 In Cellular Transformation .” 2012. Thesis, Cornell University. Accessed July 17, 2019. http://hdl.handle.net/1813/31160.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yoo, Sungsoo. “A Novel Role For The Adaptor Protein And Arf Gtpase-Activating Protein Cat-1/Git-1 In Cellular Transformation .” 2012. Web. 17 Jul 2019.

Vancouver:

Yoo S. A Novel Role For The Adaptor Protein And Arf Gtpase-Activating Protein Cat-1/Git-1 In Cellular Transformation . [Internet] [Thesis]. Cornell University; 2012. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/1813/31160.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yoo S. A Novel Role For The Adaptor Protein And Arf Gtpase-Activating Protein Cat-1/Git-1 In Cellular Transformation . [Thesis]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/31160

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

5. Stutchbury, Benjamin. Understanding how focal adhesion proteins sense and respond to mechanical signals.

Degree: PhD, 2016, University of Manchester

 The mechanical properties of the tissue vary widely around the body, from the soft brain to the rigid bone. Tissue cells are able to sense… (more)

Subjects/Keywords: Paxillin; FAK; Vinculin; Talin; Mechanosensing; Mechanotransduction; Focal adhesion

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APA (6th Edition):

Stutchbury, B. (2016). Understanding how focal adhesion proteins sense and respond to mechanical signals. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/understanding-how-focal-adhesion-proteins-sense-and-respond-to-mechanical-signals(f46e2121-1642-41b8-98f6-e0c168d6f01e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764436

Chicago Manual of Style (16th Edition):

Stutchbury, Benjamin. “Understanding how focal adhesion proteins sense and respond to mechanical signals.” 2016. Doctoral Dissertation, University of Manchester. Accessed July 17, 2019. https://www.research.manchester.ac.uk/portal/en/theses/understanding-how-focal-adhesion-proteins-sense-and-respond-to-mechanical-signals(f46e2121-1642-41b8-98f6-e0c168d6f01e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764436.

MLA Handbook (7th Edition):

Stutchbury, Benjamin. “Understanding how focal adhesion proteins sense and respond to mechanical signals.” 2016. Web. 17 Jul 2019.

Vancouver:

Stutchbury B. Understanding how focal adhesion proteins sense and respond to mechanical signals. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2019 Jul 17]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/understanding-how-focal-adhesion-proteins-sense-and-respond-to-mechanical-signals(f46e2121-1642-41b8-98f6-e0c168d6f01e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764436.

Council of Science Editors:

Stutchbury B. Understanding how focal adhesion proteins sense and respond to mechanical signals. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/understanding-how-focal-adhesion-proteins-sense-and-respond-to-mechanical-signals(f46e2121-1642-41b8-98f6-e0c168d6f01e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764436

6. Πανούσης, Δημήτριος. Διερεύνηση του διηθητικού καρκίνου του μαστού με ανοσοκυτταροχημική μέθοδο: συσχέτιση με κλασσικούς και νεώτερους προγνωστικούς δείκτες.

Degree: 2012, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 AIM: The prognostic significance of the evaluation of the immunocytochemical expression of Σopoisomerase II alpha (TOPΟΗΗα), Δnhancer of zeste homologue 2 (EZH2) and Paxillin has… (more)

Subjects/Keywords: Διηθητικός καρκίνος; Ανοσοκυτταροχημεία; Ανοσοαντίδραση; Breast cancer; Immunohistochemistry; EZH2; Paxillin; TOPOIIA

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APA (6th Edition):

Πανούσης, . . (2012). Διερεύνηση του διηθητικού καρκίνου του μαστού με ανοσοκυτταροχημική μέθοδο: συσχέτιση με κλασσικούς και νεώτερους προγνωστικούς δείκτες. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/29711

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Πανούσης, Δημήτριος. “Διερεύνηση του διηθητικού καρκίνου του μαστού με ανοσοκυτταροχημική μέθοδο: συσχέτιση με κλασσικούς και νεώτερους προγνωστικούς δείκτες.” 2012. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed July 17, 2019. http://hdl.handle.net/10442/hedi/29711.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Πανούσης, Δημήτριος. “Διερεύνηση του διηθητικού καρκίνου του μαστού με ανοσοκυτταροχημική μέθοδο: συσχέτιση με κλασσικούς και νεώτερους προγνωστικούς δείκτες.” 2012. Web. 17 Jul 2019.

Vancouver:

Πανούσης . Διερεύνηση του διηθητικού καρκίνου του μαστού με ανοσοκυτταροχημική μέθοδο: συσχέτιση με κλασσικούς και νεώτερους προγνωστικούς δείκτες. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/10442/hedi/29711.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Πανούσης . Διερεύνηση του διηθητικού καρκίνου του μαστού με ανοσοκυτταροχημική μέθοδο: συσχέτιση με κλασσικούς και νεώτερους προγνωστικούς δείκτες. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. Available from: http://hdl.handle.net/10442/hedi/29711

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

7. Stutchbury, Benjamin Peter. Understanding how focal adhesion proteins sense and respond to mechanical signals.

Degree: 2016, University of Manchester

This abstract is for the thesis entitled ‘Understanding how focal adhesion proteins sense and respond to mechanical signals’ by Benjamin Stutchbury. The thesis is submitted… (more)

Subjects/Keywords: Focal adhesion; Mechanotransduction; Mechanosensing; Talin; Vinculin; FAK; Paxillin

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APA (6th Edition):

Stutchbury, B. P. (2016). Understanding how focal adhesion proteins sense and respond to mechanical signals. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305833

Chicago Manual of Style (16th Edition):

Stutchbury, Benjamin Peter. “Understanding how focal adhesion proteins sense and respond to mechanical signals.” 2016. Doctoral Dissertation, University of Manchester. Accessed July 17, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305833.

MLA Handbook (7th Edition):

Stutchbury, Benjamin Peter. “Understanding how focal adhesion proteins sense and respond to mechanical signals.” 2016. Web. 17 Jul 2019.

Vancouver:

Stutchbury BP. Understanding how focal adhesion proteins sense and respond to mechanical signals. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2019 Jul 17]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305833.

Council of Science Editors:

Stutchbury BP. Understanding how focal adhesion proteins sense and respond to mechanical signals. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305833


Vanderbilt University

8. Donato, Dominique Maria. The focal adhesion localization of p130Cas: dynamics, targeting mechanism, and signaling.

Degree: PhD, Cell and Developmental Biology, 2010, Vanderbilt University

 Focal adhesions (FAs) are sites at the interface between the cell and the ECM, linking integrin receptors and the actin cytoskeleton. In addition to serving… (more)

Subjects/Keywords: cell motility; FAK; tyrosine phosphorylation; paxillin; focal adhesion; p130Cas

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APA (6th Edition):

Donato, D. M. (2010). The focal adhesion localization of p130Cas: dynamics, targeting mechanism, and signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-06302010-180728/ ;

Chicago Manual of Style (16th Edition):

Donato, Dominique Maria. “The focal adhesion localization of p130Cas: dynamics, targeting mechanism, and signaling.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed July 17, 2019. http://etd.library.vanderbilt.edu/available/etd-06302010-180728/ ;.

MLA Handbook (7th Edition):

Donato, Dominique Maria. “The focal adhesion localization of p130Cas: dynamics, targeting mechanism, and signaling.” 2010. Web. 17 Jul 2019.

Vancouver:

Donato DM. The focal adhesion localization of p130Cas: dynamics, targeting mechanism, and signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2019 Jul 17]. Available from: http://etd.library.vanderbilt.edu/available/etd-06302010-180728/ ;.

Council of Science Editors:

Donato DM. The focal adhesion localization of p130Cas: dynamics, targeting mechanism, and signaling. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://etd.library.vanderbilt.edu/available/etd-06302010-180728/ ;


University of Toronto

9. Allo, Ghassan. Genomic Characterization of Pleural Solitary Fibrous Tumours.

Degree: 2013, University of Toronto

Pleural solitary fibrous tumours (pSFTs) are uncommon soft tissue tumours of the pleura. that may recur and contribute to the patients’ demise. We analyzed a… (more)

Subjects/Keywords: Solitary fibrous tumours; copy number variations; mutation; paxillin; 0992

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APA (6th Edition):

Allo, G. (2013). Genomic Characterization of Pleural Solitary Fibrous Tumours. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/35578

Chicago Manual of Style (16th Edition):

Allo, Ghassan. “Genomic Characterization of Pleural Solitary Fibrous Tumours.” 2013. Masters Thesis, University of Toronto. Accessed July 17, 2019. http://hdl.handle.net/1807/35578.

MLA Handbook (7th Edition):

Allo, Ghassan. “Genomic Characterization of Pleural Solitary Fibrous Tumours.” 2013. Web. 17 Jul 2019.

Vancouver:

Allo G. Genomic Characterization of Pleural Solitary Fibrous Tumours. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/1807/35578.

Council of Science Editors:

Allo G. Genomic Characterization of Pleural Solitary Fibrous Tumours. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/35578

10. Χαλμούκη, Παναγιώτα. Μελέτη του ρόλου των TNS4/CTEN, ezrin και paxillin στο βασικοκυτταρικό καρκίνωμα του δέρματος.

Degree: 2013, University of Patras

 Το βασικοκυτταρικό καρκίνωμα του δέρματος αποτελεί τον συχνότερο τύπο καρκίνου του δέρματος. Από τους διάφορους ιστολογικούς υποτύπους του βασικοκυτταρικού καρκινώματος, ο διηθητικός ιστολογικός υπότυπος συνοδεύεται… (more)

Subjects/Keywords: Βασικοκυτταρικό καρκίνωμα; Δέρμα; 616.994 77; Basal cell carcinoma; Ezrin; Paxillin; TNS4; CTEN

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APA (6th Edition):

Χαλμούκη, . (2013). Μελέτη του ρόλου των TNS4/CTEN, ezrin και paxillin στο βασικοκυτταρικό καρκίνωμα του δέρματος. (Masters Thesis). University of Patras. Retrieved from http://hdl.handle.net/10889/8622

Chicago Manual of Style (16th Edition):

Χαλμούκη, Παναγιώτα. “Μελέτη του ρόλου των TNS4/CTEN, ezrin και paxillin στο βασικοκυτταρικό καρκίνωμα του δέρματος.” 2013. Masters Thesis, University of Patras. Accessed July 17, 2019. http://hdl.handle.net/10889/8622.

MLA Handbook (7th Edition):

Χαλμούκη, Παναγιώτα. “Μελέτη του ρόλου των TNS4/CTEN, ezrin και paxillin στο βασικοκυτταρικό καρκίνωμα του δέρματος.” 2013. Web. 17 Jul 2019.

Vancouver:

Χαλμούκη . Μελέτη του ρόλου των TNS4/CTEN, ezrin και paxillin στο βασικοκυτταρικό καρκίνωμα του δέρματος. [Internet] [Masters thesis]. University of Patras; 2013. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/10889/8622.

Council of Science Editors:

Χαλμούκη . Μελέτη του ρόλου των TNS4/CTEN, ezrin και paxillin στο βασικοκυτταρικό καρκίνωμα του δέρματος. [Masters Thesis]. University of Patras; 2013. Available from: http://hdl.handle.net/10889/8622

11. Zhang, Chi. Structural-based Investigation of FAK and P130Cas in Focal Adhesion.

Degree: PhD, Biomedical Sciences, 2015, University of Tennessee Health Science Center

  Cell migration is an integrated multistep process accompanying us throughout life, from birth to death. It contributes to a variety of biological activities and… (more)

Subjects/Keywords: FAK; Focal adhesion; Helical constrained peptide; NMR; P130Cas; Paxillin; Medical Cell Biology; Medical Sciences; Medicine and Health Sciences

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APA (6th Edition):

Zhang, C. (2015). Structural-based Investigation of FAK and P130Cas in Focal Adhesion. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/313

Chicago Manual of Style (16th Edition):

Zhang, Chi. “Structural-based Investigation of FAK and P130Cas in Focal Adhesion.” 2015. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed July 17, 2019. https://dc.uthsc.edu/dissertations/313.

MLA Handbook (7th Edition):

Zhang, Chi. “Structural-based Investigation of FAK and P130Cas in Focal Adhesion.” 2015. Web. 17 Jul 2019.

Vancouver:

Zhang C. Structural-based Investigation of FAK and P130Cas in Focal Adhesion. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2015. [cited 2019 Jul 17]. Available from: https://dc.uthsc.edu/dissertations/313.

Council of Science Editors:

Zhang C. Structural-based Investigation of FAK and P130Cas in Focal Adhesion. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2015. Available from: https://dc.uthsc.edu/dissertations/313

12. Toutounchian, Jordan Javad. Discoveries of Targets and Novel Agents for the Treatment of Ischemic Retinopathy and Neovascular Disease.

Degree: PhD, Pharmaceutical Sciences, 2016, University of Tennessee Health Science Center

  Diabetic retinopathy (DR) and age-related macular degeneration (AMD) are among the most common causes of blindness in adults. Vision loss can occur during the… (more)

Subjects/Keywords: Angiogenesis; BXD; Focal Adhesion; Neovascularization; Paxillin; Retinopathy; Medicine and Health Sciences; Pharmaceutics and Drug Design; Pharmacy and Pharmaceutical Sciences

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APA (6th Edition):

Toutounchian, J. J. (2016). Discoveries of Targets and Novel Agents for the Treatment of Ischemic Retinopathy and Neovascular Disease. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/405

Chicago Manual of Style (16th Edition):

Toutounchian, Jordan Javad. “Discoveries of Targets and Novel Agents for the Treatment of Ischemic Retinopathy and Neovascular Disease.” 2016. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed July 17, 2019. https://dc.uthsc.edu/dissertations/405.

MLA Handbook (7th Edition):

Toutounchian, Jordan Javad. “Discoveries of Targets and Novel Agents for the Treatment of Ischemic Retinopathy and Neovascular Disease.” 2016. Web. 17 Jul 2019.

Vancouver:

Toutounchian JJ. Discoveries of Targets and Novel Agents for the Treatment of Ischemic Retinopathy and Neovascular Disease. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2016. [cited 2019 Jul 17]. Available from: https://dc.uthsc.edu/dissertations/405.

Council of Science Editors:

Toutounchian JJ. Discoveries of Targets and Novel Agents for the Treatment of Ischemic Retinopathy and Neovascular Disease. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2016. Available from: https://dc.uthsc.edu/dissertations/405


University of Texas – Austin

13. Alotaibi, Talal Eid. Examination of focal adhesion kinase’s FAT domain structural response to applied mechanical load.

Degree: Mechanical Engineering, 2012, University of Texas – Austin

 Focal adhesion kinase (FAK) is a non-receptor tyrosine kinase. Activated FAK is crucial to many biological processes, such as cell proliferation, migration, and survival, all… (more)

Subjects/Keywords: Focal adhesion kinase; Focal adhesion targeting domain; Paxillin LD motifs; Y925 phosphorylation; Molecular dynamics; Mechanical load; Helix unfolding

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APA (6th Edition):

Alotaibi, T. E. (2012). Examination of focal adhesion kinase’s FAT domain structural response to applied mechanical load. (Thesis). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/ETD-UT-2012-05-5296

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alotaibi, Talal Eid. “Examination of focal adhesion kinase’s FAT domain structural response to applied mechanical load.” 2012. Thesis, University of Texas – Austin. Accessed July 17, 2019. http://hdl.handle.net/2152/ETD-UT-2012-05-5296.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alotaibi, Talal Eid. “Examination of focal adhesion kinase’s FAT domain structural response to applied mechanical load.” 2012. Web. 17 Jul 2019.

Vancouver:

Alotaibi TE. Examination of focal adhesion kinase’s FAT domain structural response to applied mechanical load. [Internet] [Thesis]. University of Texas – Austin; 2012. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/2152/ETD-UT-2012-05-5296.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alotaibi TE. Examination of focal adhesion kinase’s FAT domain structural response to applied mechanical load. [Thesis]. University of Texas – Austin; 2012. Available from: http://hdl.handle.net/2152/ETD-UT-2012-05-5296

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Downey, Christina. Molecular Mechanisms Of Pressure-Stimulated Cancer Cell Signaling.

Degree: PhD, Cancer Biology, 2010, Wayne State University

  ABSTRACT MOLECULAR MECHANISMS OF PRESSURE-STIMULATED CANCER CELL SIGNALING by CHRISTINA DOWNEY June 2010 Advisor: Dr. Marc Basson, MD, PhD Major: Cancer Biology Degree: Doctor… (more)

Subjects/Keywords: Cancer; NF-kB; Paxillin; Pressure; Molecular Biology

…understood. Paxillin may prove to be important in this pathway, as it is involved in focal adhesion… …Paxillin is a 68-kDa focal adhesion protein that has been implicated in diverse cellular events… …osteosarcomas have shown increased paxillin expression relative to their normal counterparts [25… …x5D;. Paxillin acts as a multi-domain adaptor protein that is found at the interface between… …the plasma membrane and the actin cytoskeleton. The carboxyl terminus of paxillin consists… 

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APA (6th Edition):

Downey, C. (2010). Molecular Mechanisms Of Pressure-Stimulated Cancer Cell Signaling. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/11

Chicago Manual of Style (16th Edition):

Downey, Christina. “Molecular Mechanisms Of Pressure-Stimulated Cancer Cell Signaling.” 2010. Doctoral Dissertation, Wayne State University. Accessed July 17, 2019. https://digitalcommons.wayne.edu/oa_dissertations/11.

MLA Handbook (7th Edition):

Downey, Christina. “Molecular Mechanisms Of Pressure-Stimulated Cancer Cell Signaling.” 2010. Web. 17 Jul 2019.

Vancouver:

Downey C. Molecular Mechanisms Of Pressure-Stimulated Cancer Cell Signaling. [Internet] [Doctoral dissertation]. Wayne State University; 2010. [cited 2019 Jul 17]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/11.

Council of Science Editors:

Downey C. Molecular Mechanisms Of Pressure-Stimulated Cancer Cell Signaling. [Doctoral Dissertation]. Wayne State University; 2010. Available from: https://digitalcommons.wayne.edu/oa_dissertations/11

15. ANEESH SATHE. Paxillin?s Nuclear Transport depends upon Focal Adhesion Dynamics and Interactions with the LD4 Binding Region of FAT Domains.

Degree: 2014, National University of Singapore

Subjects/Keywords: paxillin; focal adhesion; nucleus; FAT domain; geometry; FAK

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APA (6th Edition):

SATHE, A. (2014). Paxillin?s Nuclear Transport depends upon Focal Adhesion Dynamics and Interactions with the LD4 Binding Region of FAT Domains. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/119232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

SATHE, ANEESH. “Paxillin?s Nuclear Transport depends upon Focal Adhesion Dynamics and Interactions with the LD4 Binding Region of FAT Domains.” 2014. Thesis, National University of Singapore. Accessed July 17, 2019. http://scholarbank.nus.edu.sg/handle/10635/119232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

SATHE, ANEESH. “Paxillin?s Nuclear Transport depends upon Focal Adhesion Dynamics and Interactions with the LD4 Binding Region of FAT Domains.” 2014. Web. 17 Jul 2019.

Vancouver:

SATHE A. Paxillin?s Nuclear Transport depends upon Focal Adhesion Dynamics and Interactions with the LD4 Binding Region of FAT Domains. [Internet] [Thesis]. National University of Singapore; 2014. [cited 2019 Jul 17]. Available from: http://scholarbank.nus.edu.sg/handle/10635/119232.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SATHE A. Paxillin?s Nuclear Transport depends upon Focal Adhesion Dynamics and Interactions with the LD4 Binding Region of FAT Domains. [Thesis]. National University of Singapore; 2014. Available from: http://scholarbank.nus.edu.sg/handle/10635/119232

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vienna

16. Andreyeva, Natalia. Role of vesicular trafficking in focal adhesion turnover.

Degree: 2008, University of Vienna

Fokalkontakte dienen der Zelle als Verankerungen zwischen der extrazellulären Matrix mit dem Aktin Zytoskelett. Ein dynamisches Gleichgewicht von Abbau im vorderen Teil der Zelle und… (more)

Subjects/Keywords: 42.13 Molekularbiologie; Fokalkontakte / Zytoskelett / Vesikeltransport / Paxillin / siRNA-Microarray; cell migration / vesicular trafficking / paxillin / clathrin / siRNA microarray

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APA (6th Edition):

Andreyeva, N. (2008). Role of vesicular trafficking in focal adhesion turnover. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/1942/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Andreyeva, Natalia. “Role of vesicular trafficking in focal adhesion turnover.” 2008. Thesis, University of Vienna. Accessed July 17, 2019. http://othes.univie.ac.at/1942/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Andreyeva, Natalia. “Role of vesicular trafficking in focal adhesion turnover.” 2008. Web. 17 Jul 2019.

Vancouver:

Andreyeva N. Role of vesicular trafficking in focal adhesion turnover. [Internet] [Thesis]. University of Vienna; 2008. [cited 2019 Jul 17]. Available from: http://othes.univie.ac.at/1942/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Andreyeva N. Role of vesicular trafficking in focal adhesion turnover. [Thesis]. University of Vienna; 2008. Available from: http://othes.univie.ac.at/1942/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vienna

17. Breuss, Martin Werner. The link between membrane trafficking and cell motility.

Degree: 2009, University of Vienna

Um sich fortbewegen zu können, muss eine Zelle mit spezialisierten Adhäsionskomplexen fest an ihrem Substrat haften. Die größten dieser Komplexe, „Fokale Adhäsionen“, sind äußerst stabil… (more)

Subjects/Keywords: 42.15 Zellbiologie; 42.13 Molekularbiologie; Paxillin / Vesikulärer Transport / EpsinR / Fokale Adhäsionen / Zellmigration; Paxillin / vesicular transport / EpsinR / focal adhesions / cell migration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Breuss, M. W. (2009). The link between membrane trafficking and cell motility. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/3569/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Breuss, Martin Werner. “The link between membrane trafficking and cell motility.” 2009. Thesis, University of Vienna. Accessed July 17, 2019. http://othes.univie.ac.at/3569/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Breuss, Martin Werner. “The link between membrane trafficking and cell motility.” 2009. Web. 17 Jul 2019.

Vancouver:

Breuss MW. The link between membrane trafficking and cell motility. [Internet] [Thesis]. University of Vienna; 2009. [cited 2019 Jul 17]. Available from: http://othes.univie.ac.at/3569/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Breuss MW. The link between membrane trafficking and cell motility. [Thesis]. University of Vienna; 2009. Available from: http://othes.univie.ac.at/3569/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Verkoeijen, Sarah. Focal adhesion kinase and paxillin: mediators of breast cancer cell migration.

Degree: 2011, Division of Toxicology, Leiden/Amsterdam Center for Drug Research (LACDR), Faculty of Science, Leiden University

 Despite major advances in breast cancer diagnostics and treatment over the years, the disease is still a leading cause of death in women worldwide. Primary… (more)

Subjects/Keywords: Breast cancer; Cell migration; Chemotherapy; Focal adhesions; Metastasis; Paxillin; Signaling; Breast cancer; Cell migration; Chemotherapy; Focal adhesions; Metastasis; Paxillin; Signaling

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APA (6th Edition):

Verkoeijen, S. (2011). Focal adhesion kinase and paxillin: mediators of breast cancer cell migration. (Doctoral Dissertation). Division of Toxicology, Leiden/Amsterdam Center for Drug Research (LACDR), Faculty of Science, Leiden University. Retrieved from http://hdl.handle.net/1887/16697

Chicago Manual of Style (16th Edition):

Verkoeijen, Sarah. “Focal adhesion kinase and paxillin: mediators of breast cancer cell migration.” 2011. Doctoral Dissertation, Division of Toxicology, Leiden/Amsterdam Center for Drug Research (LACDR), Faculty of Science, Leiden University. Accessed July 17, 2019. http://hdl.handle.net/1887/16697.

MLA Handbook (7th Edition):

Verkoeijen, Sarah. “Focal adhesion kinase and paxillin: mediators of breast cancer cell migration.” 2011. Web. 17 Jul 2019.

Vancouver:

Verkoeijen S. Focal adhesion kinase and paxillin: mediators of breast cancer cell migration. [Internet] [Doctoral dissertation]. Division of Toxicology, Leiden/Amsterdam Center for Drug Research (LACDR), Faculty of Science, Leiden University; 2011. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/1887/16697.

Council of Science Editors:

Verkoeijen S. Focal adhesion kinase and paxillin: mediators of breast cancer cell migration. [Doctoral Dissertation]. Division of Toxicology, Leiden/Amsterdam Center for Drug Research (LACDR), Faculty of Science, Leiden University; 2011. Available from: http://hdl.handle.net/1887/16697

19. Régent, Alexis. Étude du rôle des cellules musculaires lisses vasculaires (CMLV) et des anticorps anti-CMLV dans la pathogénie de l’artérite à cellules géantes (maladie de Horton) : Role of vascular smooth muscle cells (VSMC) and anti-VSMC antibodies in the pathogenesis of giant cell arteritis.

Degree: Docteur es, Pathologie cardio-vasculaire, 2014, Université Paris Descartes – Paris V

Rationnel : L’artérite à cellules géantes (ACG) est une vascularite primitive des gros vaisseaux dont le diagnostic repose sur la mise en évidence d’un infiltrat… (more)

Subjects/Keywords: Artérite à cellules géantes; Cellule musculaire lisse vasculaire; Endothéline 1; Hypertension artérielle pulmonaire; Paxilline 5; Giant cell arteritis; Vascular smooth muscle cells; Endothelin-1; Pulmonary arterial hypertension; Paxillin; 616.1

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APA (6th Edition):

Régent, A. (2014). Étude du rôle des cellules musculaires lisses vasculaires (CMLV) et des anticorps anti-CMLV dans la pathogénie de l’artérite à cellules géantes (maladie de Horton) : Role of vascular smooth muscle cells (VSMC) and anti-VSMC antibodies in the pathogenesis of giant cell arteritis. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2014PA05S021

Chicago Manual of Style (16th Edition):

Régent, Alexis. “Étude du rôle des cellules musculaires lisses vasculaires (CMLV) et des anticorps anti-CMLV dans la pathogénie de l’artérite à cellules géantes (maladie de Horton) : Role of vascular smooth muscle cells (VSMC) and anti-VSMC antibodies in the pathogenesis of giant cell arteritis.” 2014. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed July 17, 2019. http://www.theses.fr/2014PA05S021.

MLA Handbook (7th Edition):

Régent, Alexis. “Étude du rôle des cellules musculaires lisses vasculaires (CMLV) et des anticorps anti-CMLV dans la pathogénie de l’artérite à cellules géantes (maladie de Horton) : Role of vascular smooth muscle cells (VSMC) and anti-VSMC antibodies in the pathogenesis of giant cell arteritis.” 2014. Web. 17 Jul 2019.

Vancouver:

Régent A. Étude du rôle des cellules musculaires lisses vasculaires (CMLV) et des anticorps anti-CMLV dans la pathogénie de l’artérite à cellules géantes (maladie de Horton) : Role of vascular smooth muscle cells (VSMC) and anti-VSMC antibodies in the pathogenesis of giant cell arteritis. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2014. [cited 2019 Jul 17]. Available from: http://www.theses.fr/2014PA05S021.

Council of Science Editors:

Régent A. Étude du rôle des cellules musculaires lisses vasculaires (CMLV) et des anticorps anti-CMLV dans la pathogénie de l’artérite à cellules géantes (maladie de Horton) : Role of vascular smooth muscle cells (VSMC) and anti-VSMC antibodies in the pathogenesis of giant cell arteritis. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2014. Available from: http://www.theses.fr/2014PA05S021

20. Taniguchi, Leandro Utino. Avaliação imunohistoquímica das alterações do citoesqueleto na parede alveolar em modelo experimental de lesão pulmonar induzida pela ventilação mecânica em ratos.

Degree: PhD, Emergências Clínicas, 2009, University of São Paulo

INTRODUÇÃO: A ventilação mecânica é uma terapia importante, mas com possíveis complicações. Uma das mais relevantes é a lesão pulmonar induzida pelo ventilador (VILI do… (more)

Subjects/Keywords: artificial; Citoesqueleto; Cytoskeleton; Focal adhesion kinase 1; Lesão pulmonar induzida por ventilador; Paxilina; Paxillin; Quinase 1 da adesão focal; Ratos; Rats; Respiração artificial; Respiration; Ventilador induced lung injury

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APA (6th Edition):

Taniguchi, L. U. (2009). Avaliação imunohistoquímica das alterações do citoesqueleto na parede alveolar em modelo experimental de lesão pulmonar induzida pela ventilação mecânica em ratos. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5159/tde-07122009-180201/ ;

Chicago Manual of Style (16th Edition):

Taniguchi, Leandro Utino. “Avaliação imunohistoquímica das alterações do citoesqueleto na parede alveolar em modelo experimental de lesão pulmonar induzida pela ventilação mecânica em ratos.” 2009. Doctoral Dissertation, University of São Paulo. Accessed July 17, 2019. http://www.teses.usp.br/teses/disponiveis/5/5159/tde-07122009-180201/ ;.

MLA Handbook (7th Edition):

Taniguchi, Leandro Utino. “Avaliação imunohistoquímica das alterações do citoesqueleto na parede alveolar em modelo experimental de lesão pulmonar induzida pela ventilação mecânica em ratos.” 2009. Web. 17 Jul 2019.

Vancouver:

Taniguchi LU. Avaliação imunohistoquímica das alterações do citoesqueleto na parede alveolar em modelo experimental de lesão pulmonar induzida pela ventilação mecânica em ratos. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2019 Jul 17]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5159/tde-07122009-180201/ ;.

Council of Science Editors:

Taniguchi LU. Avaliação imunohistoquímica das alterações do citoesqueleto na parede alveolar em modelo experimental de lesão pulmonar induzida pela ventilação mecânica em ratos. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/5/5159/tde-07122009-180201/ ;

21. Robu, Carmen Mariana. Study of marrow microenvironment and focal adherences in myelodysplastic syndromes and leukemias : Étude du microenvironnement médullaire et des complexes d’adhérence focale dans le myélodysplasies et leucémies.

Degree: Docteur es, Biologie moléculaire et cellulaire, 2012, Saint-Etienne; Université de Médecine et Pharmacie Grigore T. Popa (Iasi, Roumanie)

Les syndromes myélodysplasiques (SMD) sont considérés comme des maladies clonales des cellules souches hématopoïétiques (CSH). Le microenvironnement joue un rôle important par ses contacts direct… (more)

Subjects/Keywords: Syndromes myélodisplasiques; Cellules stromales mésenchymateuses; Protéines d'adhérence focale; Paxilline; Modèles de croissance; Myelodisplastic syndromes; Refractory aneamia with excess blasts; Mesenchymal stroma cells; Focal adhesion proteins; Paxillin; Growth patterns

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APA (6th Edition):

Robu, C. M. (2012). Study of marrow microenvironment and focal adherences in myelodysplastic syndromes and leukemias : Étude du microenvironnement médullaire et des complexes d’adhérence focale dans le myélodysplasies et leucémies. (Doctoral Dissertation). Saint-Etienne; Université de Médecine et Pharmacie Grigore T. Popa (Iasi, Roumanie). Retrieved from http://www.theses.fr/2012STET001T

Chicago Manual of Style (16th Edition):

Robu, Carmen Mariana. “Study of marrow microenvironment and focal adherences in myelodysplastic syndromes and leukemias : Étude du microenvironnement médullaire et des complexes d’adhérence focale dans le myélodysplasies et leucémies.” 2012. Doctoral Dissertation, Saint-Etienne; Université de Médecine et Pharmacie Grigore T. Popa (Iasi, Roumanie). Accessed July 17, 2019. http://www.theses.fr/2012STET001T.

MLA Handbook (7th Edition):

Robu, Carmen Mariana. “Study of marrow microenvironment and focal adherences in myelodysplastic syndromes and leukemias : Étude du microenvironnement médullaire et des complexes d’adhérence focale dans le myélodysplasies et leucémies.” 2012. Web. 17 Jul 2019.

Vancouver:

Robu CM. Study of marrow microenvironment and focal adherences in myelodysplastic syndromes and leukemias : Étude du microenvironnement médullaire et des complexes d’adhérence focale dans le myélodysplasies et leucémies. [Internet] [Doctoral dissertation]. Saint-Etienne; Université de Médecine et Pharmacie Grigore T. Popa (Iasi, Roumanie); 2012. [cited 2019 Jul 17]. Available from: http://www.theses.fr/2012STET001T.

Council of Science Editors:

Robu CM. Study of marrow microenvironment and focal adherences in myelodysplastic syndromes and leukemias : Étude du microenvironnement médullaire et des complexes d’adhérence focale dans le myélodysplasies et leucémies. [Doctoral Dissertation]. Saint-Etienne; Université de Médecine et Pharmacie Grigore T. Popa (Iasi, Roumanie); 2012. Available from: http://www.theses.fr/2012STET001T

22. Jennifer Leigh, Quizi. SLK-mediated Phosphorylation of Paxillin Is Required for Focal Adhesion Turnover and Cell Migration .

Degree: 2012, University of Ottawa

 The precise mechanism regulating focal adhesion disassembly has yet to be elucidated. Recently, we have implicated the Ste20-like kinase SLK in mediating efficient focal adhesion… (more)

Subjects/Keywords: SLK; Paxillin; Phosphorylation; Focal adhesion turnover; Cell migration

…viii Chapter 4: Paxillin S250 is required for focal adhesion turnover and efficient cell… …migration 89 Paxillin S250 is required for focal adhesion… …turnover and efficient cell migration .90 Paxillin S250 phosphorylation is not required… …for adhesion formation .90 Paxillin S250 phosphorylation is required for cell… …spreading 91 Paxillin S250 phosphorylation is required for efficient cell migration… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jennifer Leigh, Q. (2012). SLK-mediated Phosphorylation of Paxillin Is Required for Focal Adhesion Turnover and Cell Migration . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/20483

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jennifer Leigh, Quizi. “SLK-mediated Phosphorylation of Paxillin Is Required for Focal Adhesion Turnover and Cell Migration .” 2012. Thesis, University of Ottawa. Accessed July 17, 2019. http://hdl.handle.net/10393/20483.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jennifer Leigh, Quizi. “SLK-mediated Phosphorylation of Paxillin Is Required for Focal Adhesion Turnover and Cell Migration .” 2012. Web. 17 Jul 2019.

Vancouver:

Jennifer Leigh Q. SLK-mediated Phosphorylation of Paxillin Is Required for Focal Adhesion Turnover and Cell Migration . [Internet] [Thesis]. University of Ottawa; 2012. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/10393/20483.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jennifer Leigh Q. SLK-mediated Phosphorylation of Paxillin Is Required for Focal Adhesion Turnover and Cell Migration . [Thesis]. University of Ottawa; 2012. Available from: http://hdl.handle.net/10393/20483

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. EVELYN PNG YEN SOO. MOLECULAR MECHANISMS AND BIOLOGY OF TRANSGLUTAMINASE-2 IN CORNEAL EPITHELIUM.

Degree: 2015, National University of Singapore

Subjects/Keywords: transglutaminase-2; corneal epithelium; wound healing; paxillin; hyperosmolarity; focal adhesion

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APA (6th Edition):

SOO, E. P. Y. (2015). MOLECULAR MECHANISMS AND BIOLOGY OF TRANSGLUTAMINASE-2 IN CORNEAL EPITHELIUM. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/136500

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

SOO, EVELYN PNG YEN. “MOLECULAR MECHANISMS AND BIOLOGY OF TRANSGLUTAMINASE-2 IN CORNEAL EPITHELIUM.” 2015. Thesis, National University of Singapore. Accessed July 17, 2019. http://scholarbank.nus.edu.sg/handle/10635/136500.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

SOO, EVELYN PNG YEN. “MOLECULAR MECHANISMS AND BIOLOGY OF TRANSGLUTAMINASE-2 IN CORNEAL EPITHELIUM.” 2015. Web. 17 Jul 2019.

Vancouver:

SOO EPY. MOLECULAR MECHANISMS AND BIOLOGY OF TRANSGLUTAMINASE-2 IN CORNEAL EPITHELIUM. [Internet] [Thesis]. National University of Singapore; 2015. [cited 2019 Jul 17]. Available from: http://scholarbank.nus.edu.sg/handle/10635/136500.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SOO EPY. MOLECULAR MECHANISMS AND BIOLOGY OF TRANSGLUTAMINASE-2 IN CORNEAL EPITHELIUM. [Thesis]. National University of Singapore; 2015. Available from: http://scholarbank.nus.edu.sg/handle/10635/136500

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Κουκουριτάκη, Σεβαστή. Διερεύνηση του μηχανισμού σταθεροποίησης των μικροϊνιδίων στιε νεοπλασίες του ενδομητρίου. Επίδραση στεροειδών ορμονών.

Degree: 2001, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Subjects/Keywords: Κυτταροσκελετός; Μικροϊνίδια; Ακτίνη; Δεξαμεθαζόνη; Ενδομήτριο; Κινάση εστιών πρόσφυσης; Παξιλλίνη; Φωσφορυλίωση; Cytoskeleton; Microfilaments; Actin; Dexamethasone; Endometrium; Focal adhesion kinase; Paxillin; Phosphorylation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Κουκουριτάκη, . . (2001). Διερεύνηση του μηχανισμού σταθεροποίησης των μικροϊνιδίων στιε νεοπλασίες του ενδομητρίου. Επίδραση στεροειδών ορμονών. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/15910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Κουκουριτάκη, Σεβαστή. “Διερεύνηση του μηχανισμού σταθεροποίησης των μικροϊνιδίων στιε νεοπλασίες του ενδομητρίου. Επίδραση στεροειδών ορμονών.” 2001. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed July 17, 2019. http://hdl.handle.net/10442/hedi/15910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Κουκουριτάκη, Σεβαστή. “Διερεύνηση του μηχανισμού σταθεροποίησης των μικροϊνιδίων στιε νεοπλασίες του ενδομητρίου. Επίδραση στεροειδών ορμονών.” 2001. Web. 17 Jul 2019.

Vancouver:

Κουκουριτάκη . Διερεύνηση του μηχανισμού σταθεροποίησης των μικροϊνιδίων στιε νεοπλασίες του ενδομητρίου. Επίδραση στεροειδών ορμονών. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2001. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/10442/hedi/15910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Κουκουριτάκη . Διερεύνηση του μηχανισμού σταθεροποίησης των μικροϊνιδίων στιε νεοπλασίες του ενδομητρίου. Επίδραση στεροειδών ορμονών. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2001. Available from: http://hdl.handle.net/10442/hedi/15910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

25. Easley, Charles, IV. Fibronectin-dependent Activation of CaMK-II Promotes Focal Adhesion Turnover by Inducing Tyrosine Dephosphorylation of FAK and Paxillin.

Degree: PhD, Biochemistry, 2008, Virginia Commonwealth University

  Transient elevations in Ca2+ have previously been shown to promote focal adhesion disassembly and cell motility. Yet the targets of these Ca2+ transients have… (more)

Subjects/Keywords: paxillin; focal adhesion kinase; cell motility; CaMK-II; Biochemistry, Biophysics, and Structural Biology; Life Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Easley, Charles, I. (2008). Fibronectin-dependent Activation of CaMK-II Promotes Focal Adhesion Turnover by Inducing Tyrosine Dephosphorylation of FAK and Paxillin. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/1585

Chicago Manual of Style (16th Edition):

Easley, Charles, IV. “Fibronectin-dependent Activation of CaMK-II Promotes Focal Adhesion Turnover by Inducing Tyrosine Dephosphorylation of FAK and Paxillin.” 2008. Doctoral Dissertation, Virginia Commonwealth University. Accessed July 17, 2019. https://scholarscompass.vcu.edu/etd/1585.

MLA Handbook (7th Edition):

Easley, Charles, IV. “Fibronectin-dependent Activation of CaMK-II Promotes Focal Adhesion Turnover by Inducing Tyrosine Dephosphorylation of FAK and Paxillin.” 2008. Web. 17 Jul 2019.

Vancouver:

Easley, Charles I. Fibronectin-dependent Activation of CaMK-II Promotes Focal Adhesion Turnover by Inducing Tyrosine Dephosphorylation of FAK and Paxillin. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2008. [cited 2019 Jul 17]. Available from: https://scholarscompass.vcu.edu/etd/1585.

Council of Science Editors:

Easley, Charles I. Fibronectin-dependent Activation of CaMK-II Promotes Focal Adhesion Turnover by Inducing Tyrosine Dephosphorylation of FAK and Paxillin. [Doctoral Dissertation]. Virginia Commonwealth University; 2008. Available from: https://scholarscompass.vcu.edu/etd/1585


University of Cincinnati

26. Song, Jaekyung Cecilia. Protein Kinase C-d and Protein Kinase C-e Cooperatively Enhance Epithelial Cell Spreading via Transactivation of Epidermal Growth Factor Receptor and Actin-Dependent Phosphorylation of Focal Adhesion-Associated Proteins.

Degree: PhD, Medicine : Molecular and Cellular Physiology, 2005, University of Cincinnati

 Regulation of epithelial cell motility requires coordinated actin rearrangement, yet the signaling pathways that lead to such modulation are not fully described. Previously we showed… (more)

Subjects/Keywords: Biology, Cell; Protein Kinase C; Cell spreading; Cell migration; Epithelial Cells; Epidermal Growth Factor Receptor; Transactivation; Focal Adhesion; Actin; Focal Adhesion Kinase; Src; Paxillin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Song, J. C. (2005). Protein Kinase C-d and Protein Kinase C-e Cooperatively Enhance Epithelial Cell Spreading via Transactivation of Epidermal Growth Factor Receptor and Actin-Dependent Phosphorylation of Focal Adhesion-Associated Proteins. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1132198567

Chicago Manual of Style (16th Edition):

Song, Jaekyung Cecilia. “Protein Kinase C-d and Protein Kinase C-e Cooperatively Enhance Epithelial Cell Spreading via Transactivation of Epidermal Growth Factor Receptor and Actin-Dependent Phosphorylation of Focal Adhesion-Associated Proteins.” 2005. Doctoral Dissertation, University of Cincinnati. Accessed July 17, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1132198567.

MLA Handbook (7th Edition):

Song, Jaekyung Cecilia. “Protein Kinase C-d and Protein Kinase C-e Cooperatively Enhance Epithelial Cell Spreading via Transactivation of Epidermal Growth Factor Receptor and Actin-Dependent Phosphorylation of Focal Adhesion-Associated Proteins.” 2005. Web. 17 Jul 2019.

Vancouver:

Song JC. Protein Kinase C-d and Protein Kinase C-e Cooperatively Enhance Epithelial Cell Spreading via Transactivation of Epidermal Growth Factor Receptor and Actin-Dependent Phosphorylation of Focal Adhesion-Associated Proteins. [Internet] [Doctoral dissertation]. University of Cincinnati; 2005. [cited 2019 Jul 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1132198567.

Council of Science Editors:

Song JC. Protein Kinase C-d and Protein Kinase C-e Cooperatively Enhance Epithelial Cell Spreading via Transactivation of Epidermal Growth Factor Receptor and Actin-Dependent Phosphorylation of Focal Adhesion-Associated Proteins. [Doctoral Dissertation]. University of Cincinnati; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1132198567

27. Zhang, Ziwei. The Structural and Functional Study of GIT1 Paxillin Binding Domain.

Degree: PhD, Molecular Sciences, 2008, University of Tennessee Health Science Center

  The G protein coupled receptor (GPCR)-kinase (GRK) interacting protein 1 (GIT1) is a multidomain protein that plays an important role in cell adhesion, motility,… (more)

Subjects/Keywords: NMR; structure; GIT; FAK; paxillin; interaction; focal adhesion; signal; regulation; Amino Acids, Peptides, and Proteins; Chemicals and Drugs; Medical Molecular Biology; Medical Sciences; Medicine and Health Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, Z. (2008). The Structural and Functional Study of GIT1 Paxillin Binding Domain. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/318

Chicago Manual of Style (16th Edition):

Zhang, Ziwei. “The Structural and Functional Study of GIT1 Paxillin Binding Domain.” 2008. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed July 17, 2019. https://dc.uthsc.edu/dissertations/318.

MLA Handbook (7th Edition):

Zhang, Ziwei. “The Structural and Functional Study of GIT1 Paxillin Binding Domain.” 2008. Web. 17 Jul 2019.

Vancouver:

Zhang Z. The Structural and Functional Study of GIT1 Paxillin Binding Domain. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2008. [cited 2019 Jul 17]. Available from: https://dc.uthsc.edu/dissertations/318.

Council of Science Editors:

Zhang Z. The Structural and Functional Study of GIT1 Paxillin Binding Domain. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2008. Available from: https://dc.uthsc.edu/dissertations/318

28. Βαρδάκη, Ελευθερία. In vitro μελέτη της επίδρασης του TNFa στον κυτταροσκελετό σπειραματικών επιθηλιακών κυττάρων (ποδοκυττάρων).

Degree: 2002, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Glomerular permeability for macromolecules depends partially on proper attachment of the glomerular epithelial cells (GEC) to the glomerular basement membrane (GBM). The latter requires integrity… (more)

Subjects/Keywords: Κύτταρα, σπειραματικά επιθηλιακά; Ποδοκύτταρα; Παράγων νέκρωσης του όγκου - άλφα (TNF-α); Κυτταροσκελετός ακτίνης; Κυτταρικός όγκος; Κυτταρικός πολλαπλασιασμός; Κυτταρική απόπτωση; Εστιακές προσφύσεις; Κινάση εστιών πρόσφυσης; Παξιλλίνη; Βινκουλίνη; Actin cytoskeleton; Flomerular epithelial cells; Tumor necrosis factor-a (TNF-α); Cell volume; Cell proliferation; Cell apoptosis; Focal adhesion kinase; Paxillin; Vinculin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Βαρδάκη, . . (2002). In vitro μελέτη της επίδρασης του TNFa στον κυτταροσκελετό σπειραματικών επιθηλιακών κυττάρων (ποδοκυττάρων). (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/16065

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Βαρδάκη, Ελευθερία. “In vitro μελέτη της επίδρασης του TNFa στον κυτταροσκελετό σπειραματικών επιθηλιακών κυττάρων (ποδοκυττάρων).” 2002. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed July 17, 2019. http://hdl.handle.net/10442/hedi/16065.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Βαρδάκη, Ελευθερία. “In vitro μελέτη της επίδρασης του TNFa στον κυτταροσκελετό σπειραματικών επιθηλιακών κυττάρων (ποδοκυττάρων).” 2002. Web. 17 Jul 2019.

Vancouver:

Βαρδάκη . In vitro μελέτη της επίδρασης του TNFa στον κυτταροσκελετό σπειραματικών επιθηλιακών κυττάρων (ποδοκυττάρων). [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2002. [cited 2019 Jul 17]. Available from: http://hdl.handle.net/10442/hedi/16065.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Βαρδάκη . In vitro μελέτη της επίδρασης του TNFa στον κυτταροσκελετό σπειραματικών επιθηλιακών κυττάρων (ποδοκυττάρων). [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2002. Available from: http://hdl.handle.net/10442/hedi/16065

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.