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You searched for subject:(Pathophysiology). Showing records 1 – 30 of 458 total matches.

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California State University – East Bay

1. Ames, Bryant. Tetrabrominated Bisphenol A Inhibition Studies on Six Cytochrome P450 Enzymes in Human Liver Microsomes.

Degree: 2013, California State University – East Bay

 In this study, a system was designed, utilizing human microsomes, to determine if a flame retardant, tetrabromobisphenol A (TBBPA), inhibits cytochrome P450 (CYP) enzyme function.… (more)

Subjects/Keywords: Liver  – Pathophysiology

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APA (6th Edition):

Ames, B. (2013). Tetrabrominated Bisphenol A Inhibition Studies on Six Cytochrome P450 Enzymes in Human Liver Microsomes. (Thesis). California State University – East Bay. Retrieved from http://hdl.handle.net/10211.3/4610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ames, Bryant. “Tetrabrominated Bisphenol A Inhibition Studies on Six Cytochrome P450 Enzymes in Human Liver Microsomes.” 2013. Thesis, California State University – East Bay. Accessed October 21, 2019. http://hdl.handle.net/10211.3/4610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ames, Bryant. “Tetrabrominated Bisphenol A Inhibition Studies on Six Cytochrome P450 Enzymes in Human Liver Microsomes.” 2013. Web. 21 Oct 2019.

Vancouver:

Ames B. Tetrabrominated Bisphenol A Inhibition Studies on Six Cytochrome P450 Enzymes in Human Liver Microsomes. [Internet] [Thesis]. California State University – East Bay; 2013. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10211.3/4610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ames B. Tetrabrominated Bisphenol A Inhibition Studies on Six Cytochrome P450 Enzymes in Human Liver Microsomes. [Thesis]. California State University – East Bay; 2013. Available from: http://hdl.handle.net/10211.3/4610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

2. 羅昕; Luo, Xin. The role of CXCL12/CXCR4 axis in central mechanisms of pathological pain.

Degree: PhD, 2015, University of Hong Kong

 Pathological pain is a disease induced by abnormal pain perception under various pathological conditions, and the central mechanisms contribute to the chronification of pain symptom.… (more)

Subjects/Keywords: Pain - Pathophysiology

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APA (6th Edition):

羅昕; Luo, X. (2015). The role of CXCL12/CXCR4 axis in central mechanisms of pathological pain. (Doctoral Dissertation). University of Hong Kong. Retrieved from Luo, X. [羅昕]. (2015). The role of CXCL12/CXCR4 axis in central mechanisms of pathological pain. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736685 ; http://hdl.handle.net/10722/225202

Chicago Manual of Style (16th Edition):

羅昕; Luo, Xin. “The role of CXCL12/CXCR4 axis in central mechanisms of pathological pain.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed October 21, 2019. Luo, X. [羅昕]. (2015). The role of CXCL12/CXCR4 axis in central mechanisms of pathological pain. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736685 ; http://hdl.handle.net/10722/225202.

MLA Handbook (7th Edition):

羅昕; Luo, Xin. “The role of CXCL12/CXCR4 axis in central mechanisms of pathological pain.” 2015. Web. 21 Oct 2019.

Vancouver:

羅昕; Luo X. The role of CXCL12/CXCR4 axis in central mechanisms of pathological pain. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Oct 21]. Available from: Luo, X. [羅昕]. (2015). The role of CXCL12/CXCR4 axis in central mechanisms of pathological pain. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736685 ; http://hdl.handle.net/10722/225202.

Council of Science Editors:

羅昕; Luo X. The role of CXCL12/CXCR4 axis in central mechanisms of pathological pain. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Luo, X. [羅昕]. (2015). The role of CXCL12/CXCR4 axis in central mechanisms of pathological pain. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736685 ; http://hdl.handle.net/10722/225202


University of Hong Kong

3. Tsang, Wing-ting, Andrea. Investigating the potential significance of tau protein in corticosterone-induced depression and neurodegeneration : implication in Alzheimer's disease.

Degree: M. Phil., 2014, University of Hong Kong

Alzheimer’s disease (AD) is a devastating neurodegenerative disease with growing prevalence in our society. Patients suffering from this debilitating disorder also develop neuropsychiatric symptoms. Depression… (more)

Subjects/Keywords: Depression, Mental - Pathophysiology; Alzheimer's disease - Pathophysiology

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APA (6th Edition):

Tsang, Wing-ting, A. (2014). Investigating the potential significance of tau protein in corticosterone-induced depression and neurodegeneration : implication in Alzheimer's disease. (Masters Thesis). University of Hong Kong. Retrieved from Tsang, W. A. [曾詠婷]. (2014). Investigating the potential significance of tau protein in corticosterone-induced depression and neurodegeneration : implication in Alzheimer's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194723 ; http://dx.doi.org/10.5353/th_b5194723 ; http://hdl.handle.net/10722/208418

Chicago Manual of Style (16th Edition):

Tsang, Wing-ting, Andrea. “Investigating the potential significance of tau protein in corticosterone-induced depression and neurodegeneration : implication in Alzheimer's disease.” 2014. Masters Thesis, University of Hong Kong. Accessed October 21, 2019. Tsang, W. A. [曾詠婷]. (2014). Investigating the potential significance of tau protein in corticosterone-induced depression and neurodegeneration : implication in Alzheimer's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194723 ; http://dx.doi.org/10.5353/th_b5194723 ; http://hdl.handle.net/10722/208418.

MLA Handbook (7th Edition):

Tsang, Wing-ting, Andrea. “Investigating the potential significance of tau protein in corticosterone-induced depression and neurodegeneration : implication in Alzheimer's disease.” 2014. Web. 21 Oct 2019.

Vancouver:

Tsang, Wing-ting A. Investigating the potential significance of tau protein in corticosterone-induced depression and neurodegeneration : implication in Alzheimer's disease. [Internet] [Masters thesis]. University of Hong Kong; 2014. [cited 2019 Oct 21]. Available from: Tsang, W. A. [曾詠婷]. (2014). Investigating the potential significance of tau protein in corticosterone-induced depression and neurodegeneration : implication in Alzheimer's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194723 ; http://dx.doi.org/10.5353/th_b5194723 ; http://hdl.handle.net/10722/208418.

Council of Science Editors:

Tsang, Wing-ting A. Investigating the potential significance of tau protein in corticosterone-induced depression and neurodegeneration : implication in Alzheimer's disease. [Masters Thesis]. University of Hong Kong; 2014. Available from: Tsang, W. A. [曾詠婷]. (2014). Investigating the potential significance of tau protein in corticosterone-induced depression and neurodegeneration : implication in Alzheimer's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5194723 ; http://dx.doi.org/10.5353/th_b5194723 ; http://hdl.handle.net/10722/208418


Macquarie University

4. Kamath, Karthik Shantharam. Proteomics of Pseudomonas aeruginosa cystic fibrosis isolates to understand host adaptation.

Degree: 2017, Macquarie University

Theoretical thesis.

Bibliography: pages 173-196.

Chapter-1: Introduction  – Chapter-2: Proteomics of host and pathogens in cystic fibrosis  – Chapter-3: Genetically and phenotypically distinct Pseudomonas aeruginosa… (more)

Subjects/Keywords: Pseudomonas aeruginosa  – Pathophysiology; Proteomics; Cystic fibrosis  – Pathophysiology

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APA (6th Edition):

Kamath, K. S. (2017). Proteomics of Pseudomonas aeruginosa cystic fibrosis isolates to understand host adaptation. (Doctoral Dissertation). Macquarie University. Retrieved from http://hdl.handle.net/1959.14/1265276

Chicago Manual of Style (16th Edition):

Kamath, Karthik Shantharam. “Proteomics of Pseudomonas aeruginosa cystic fibrosis isolates to understand host adaptation.” 2017. Doctoral Dissertation, Macquarie University. Accessed October 21, 2019. http://hdl.handle.net/1959.14/1265276.

MLA Handbook (7th Edition):

Kamath, Karthik Shantharam. “Proteomics of Pseudomonas aeruginosa cystic fibrosis isolates to understand host adaptation.” 2017. Web. 21 Oct 2019.

Vancouver:

Kamath KS. Proteomics of Pseudomonas aeruginosa cystic fibrosis isolates to understand host adaptation. [Internet] [Doctoral dissertation]. Macquarie University; 2017. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1959.14/1265276.

Council of Science Editors:

Kamath KS. Proteomics of Pseudomonas aeruginosa cystic fibrosis isolates to understand host adaptation. [Doctoral Dissertation]. Macquarie University; 2017. Available from: http://hdl.handle.net/1959.14/1265276


University of Utah

5. Umuerri, Oluwatoroti Omowayenwa. Effect of the RNAi pathway on HIV-1 infection and replication;.

Degree: MS;, Pathology;, 2007, University of Utah

 microRNA (miRNA) are 18-25 nucleotides small RNAs that regulate gene expression at the posttranscriptional level. They are expressed in many plants and animal species including… (more)

Subjects/Keywords: Pathogenesis; Pathophysiology; Virus

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APA (6th Edition):

Umuerri, O. O. (2007). Effect of the RNAi pathway on HIV-1 infection and replication;. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/816/rec/394

Chicago Manual of Style (16th Edition):

Umuerri, Oluwatoroti Omowayenwa. “Effect of the RNAi pathway on HIV-1 infection and replication;.” 2007. Masters Thesis, University of Utah. Accessed October 21, 2019. http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/816/rec/394.

MLA Handbook (7th Edition):

Umuerri, Oluwatoroti Omowayenwa. “Effect of the RNAi pathway on HIV-1 infection and replication;.” 2007. Web. 21 Oct 2019.

Vancouver:

Umuerri OO. Effect of the RNAi pathway on HIV-1 infection and replication;. [Internet] [Masters thesis]. University of Utah; 2007. [cited 2019 Oct 21]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/816/rec/394.

Council of Science Editors:

Umuerri OO. Effect of the RNAi pathway on HIV-1 infection and replication;. [Masters Thesis]. University of Utah; 2007. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/816/rec/394


University of Hong Kong

6. Chen, Xinying. Regional disturbances by the [delta]F508 mutation on the cystic fibrosis transmembrane conductance regulator.

Degree: PhD, 2016, University of Hong Kong

 Cystic fibrosis (CF) is a genetic disease caused by dysfunction of the cystic fibrosis transmemberane conductance regulator (CFTR). The ΔF508 mutation, missing the phenylalanine residue… (more)

Subjects/Keywords: Cystic fibrosis - Pathophysiology

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APA (6th Edition):

Chen, X. (2016). Regional disturbances by the [delta]F508 mutation on the cystic fibrosis transmembrane conductance regulator. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/238831

Chicago Manual of Style (16th Edition):

Chen, Xinying. “Regional disturbances by the [delta]F508 mutation on the cystic fibrosis transmembrane conductance regulator.” 2016. Doctoral Dissertation, University of Hong Kong. Accessed October 21, 2019. http://hdl.handle.net/10722/238831.

MLA Handbook (7th Edition):

Chen, Xinying. “Regional disturbances by the [delta]F508 mutation on the cystic fibrosis transmembrane conductance regulator.” 2016. Web. 21 Oct 2019.

Vancouver:

Chen X. Regional disturbances by the [delta]F508 mutation on the cystic fibrosis transmembrane conductance regulator. [Internet] [Doctoral dissertation]. University of Hong Kong; 2016. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10722/238831.

Council of Science Editors:

Chen X. Regional disturbances by the [delta]F508 mutation on the cystic fibrosis transmembrane conductance regulator. [Doctoral Dissertation]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/238831


University of Hong Kong

7. Leung, Yen. Evaluating appropriateness for the use of 6-hydroxydopamine as an experimental model for Parkinson's disease to investigate involvement of tau protein in cognitive dysfunctions of Parkinson's disease.

Degree: M. Phil., 2015, University of Hong Kong

Parkinson’s disease (PD) is a debilitating neurodegenerative disorder which progresses from initial motor impairments to development of cognitive dysfunctions and dementia in advanced stages of… (more)

Subjects/Keywords: Parkinson's disease - Pathophysiology

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APA (6th Edition):

Leung, Y. (2015). Evaluating appropriateness for the use of 6-hydroxydopamine as an experimental model for Parkinson's disease to investigate involvement of tau protein in cognitive dysfunctions of Parkinson's disease. (Masters Thesis). University of Hong Kong. Retrieved from Leung, Y. [梁欣]. (2015). Evaluating appropriateness for the use of 6-hydroxydopamine as an experimental model for Parkinson's disease to investigate involvement of tau protein in cognitive dysfunctions of Parkinson's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435633 ; http://dx.doi.org/10.5353/th_b5435633 ; http://hdl.handle.net/10722/222912

Chicago Manual of Style (16th Edition):

Leung, Yen. “Evaluating appropriateness for the use of 6-hydroxydopamine as an experimental model for Parkinson's disease to investigate involvement of tau protein in cognitive dysfunctions of Parkinson's disease.” 2015. Masters Thesis, University of Hong Kong. Accessed October 21, 2019. Leung, Y. [梁欣]. (2015). Evaluating appropriateness for the use of 6-hydroxydopamine as an experimental model for Parkinson's disease to investigate involvement of tau protein in cognitive dysfunctions of Parkinson's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435633 ; http://dx.doi.org/10.5353/th_b5435633 ; http://hdl.handle.net/10722/222912.

MLA Handbook (7th Edition):

Leung, Yen. “Evaluating appropriateness for the use of 6-hydroxydopamine as an experimental model for Parkinson's disease to investigate involvement of tau protein in cognitive dysfunctions of Parkinson's disease.” 2015. Web. 21 Oct 2019.

Vancouver:

Leung Y. Evaluating appropriateness for the use of 6-hydroxydopamine as an experimental model for Parkinson's disease to investigate involvement of tau protein in cognitive dysfunctions of Parkinson's disease. [Internet] [Masters thesis]. University of Hong Kong; 2015. [cited 2019 Oct 21]. Available from: Leung, Y. [梁欣]. (2015). Evaluating appropriateness for the use of 6-hydroxydopamine as an experimental model for Parkinson's disease to investigate involvement of tau protein in cognitive dysfunctions of Parkinson's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435633 ; http://dx.doi.org/10.5353/th_b5435633 ; http://hdl.handle.net/10722/222912.

Council of Science Editors:

Leung Y. Evaluating appropriateness for the use of 6-hydroxydopamine as an experimental model for Parkinson's disease to investigate involvement of tau protein in cognitive dysfunctions of Parkinson's disease. [Masters Thesis]. University of Hong Kong; 2015. Available from: Leung, Y. [梁欣]. (2015). Evaluating appropriateness for the use of 6-hydroxydopamine as an experimental model for Parkinson's disease to investigate involvement of tau protein in cognitive dysfunctions of Parkinson's disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435633 ; http://dx.doi.org/10.5353/th_b5435633 ; http://hdl.handle.net/10722/222912


Rutgers University

8. Francis, Mary. Mechanisms mediating macrophage accumulation and activation in the lung during the pathogenesis of ozone-induced lung injury.

Degree: PhD, Toxicology, 2017, Rutgers University

 Classically and alternatively activated macrophages and inflammatory mediators they release play a key role in the pathogenesis of ozone-induced lung injury. In these studies, we… (more)

Subjects/Keywords: Macrophages; Lungs – Pathophysiology

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APA (6th Edition):

Francis, M. (2017). Mechanisms mediating macrophage accumulation and activation in the lung during the pathogenesis of ozone-induced lung injury. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/53614/

Chicago Manual of Style (16th Edition):

Francis, Mary. “Mechanisms mediating macrophage accumulation and activation in the lung during the pathogenesis of ozone-induced lung injury.” 2017. Doctoral Dissertation, Rutgers University. Accessed October 21, 2019. https://rucore.libraries.rutgers.edu/rutgers-lib/53614/.

MLA Handbook (7th Edition):

Francis, Mary. “Mechanisms mediating macrophage accumulation and activation in the lung during the pathogenesis of ozone-induced lung injury.” 2017. Web. 21 Oct 2019.

Vancouver:

Francis M. Mechanisms mediating macrophage accumulation and activation in the lung during the pathogenesis of ozone-induced lung injury. [Internet] [Doctoral dissertation]. Rutgers University; 2017. [cited 2019 Oct 21]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/53614/.

Council of Science Editors:

Francis M. Mechanisms mediating macrophage accumulation and activation in the lung during the pathogenesis of ozone-induced lung injury. [Doctoral Dissertation]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/53614/


University of North Carolina – Greensboro

9. Nam, Heesun. Novel mechanisms regulating inflammatory gene expression in adipocytes.

Degree: 2012, University of North Carolina – Greensboro

 Obesity and diabetes are major public health concerns worldwide that contribute to cardiovascular disease, hypertension, and stroke. Research over the last two decades has revealed… (more)

Subjects/Keywords: Adipose tissues – Pathophysiology; Obesity – Physiological aspects; Obesity x Pathophysiology; Gene expression; Inflammation – Pathophysiology

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APA (6th Edition):

Nam, H. (2012). Novel mechanisms regulating inflammatory gene expression in adipocytes. (Doctoral Dissertation). University of North Carolina – Greensboro. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=8657

Chicago Manual of Style (16th Edition):

Nam, Heesun. “Novel mechanisms regulating inflammatory gene expression in adipocytes.” 2012. Doctoral Dissertation, University of North Carolina – Greensboro. Accessed October 21, 2019. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=8657.

MLA Handbook (7th Edition):

Nam, Heesun. “Novel mechanisms regulating inflammatory gene expression in adipocytes.” 2012. Web. 21 Oct 2019.

Vancouver:

Nam H. Novel mechanisms regulating inflammatory gene expression in adipocytes. [Internet] [Doctoral dissertation]. University of North Carolina – Greensboro; 2012. [cited 2019 Oct 21]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=8657.

Council of Science Editors:

Nam H. Novel mechanisms regulating inflammatory gene expression in adipocytes. [Doctoral Dissertation]. University of North Carolina – Greensboro; 2012. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=8657


University of Utah

10. Li, Jun. Binding, uptake, and release of L-glutamate and related compounds in the brain : comparisons between DBA and C57 mice.

Degree: PhD, Physiology;, 1997, University of Utah

 L-Glutamate, the primary excitatory neurotransmitter in the mammalian central nervous system (CNS), is involved in many physiological processes and some neurological disorders including epilepsy. The… (more)

Subjects/Keywords: Etiology; Pathophysiology

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APA (6th Edition):

Li, J. (1997). Binding, uptake, and release of L-glutamate and related compounds in the brain : comparisons between DBA and C57 mice. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1309/rec/153

Chicago Manual of Style (16th Edition):

Li, Jun. “Binding, uptake, and release of L-glutamate and related compounds in the brain : comparisons between DBA and C57 mice.” 1997. Doctoral Dissertation, University of Utah. Accessed October 21, 2019. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1309/rec/153.

MLA Handbook (7th Edition):

Li, Jun. “Binding, uptake, and release of L-glutamate and related compounds in the brain : comparisons between DBA and C57 mice.” 1997. Web. 21 Oct 2019.

Vancouver:

Li J. Binding, uptake, and release of L-glutamate and related compounds in the brain : comparisons between DBA and C57 mice. [Internet] [Doctoral dissertation]. University of Utah; 1997. [cited 2019 Oct 21]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1309/rec/153.

Council of Science Editors:

Li J. Binding, uptake, and release of L-glutamate and related compounds in the brain : comparisons between DBA and C57 mice. [Doctoral Dissertation]. University of Utah; 1997. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1309/rec/153

11. Cherifi, Ibtissem. Impact de la signalisation constitutive du récepteur de la prolactine sur la physiopathologie mammaire : Impact of the constitutive signaling of the prolactin receptor on mammary pathophysiology.

Degree: Docteur es, Physiopathologie, 2014, Université Paris Descartes – Paris V

Le lien entre la prolactine (PRL) et la tumorigenèse mammaire est soutenu par de nombreux arguments expérimentaux, cliniques et épidémiologiques. Cependant, aucune mutation des gènes… (more)

Subjects/Keywords: Physiopathologie; Pathophysiology; 616.994 49

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APA (6th Edition):

Cherifi, I. (2014). Impact de la signalisation constitutive du récepteur de la prolactine sur la physiopathologie mammaire : Impact of the constitutive signaling of the prolactin receptor on mammary pathophysiology. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2014PA05T061

Chicago Manual of Style (16th Edition):

Cherifi, Ibtissem. “Impact de la signalisation constitutive du récepteur de la prolactine sur la physiopathologie mammaire : Impact of the constitutive signaling of the prolactin receptor on mammary pathophysiology.” 2014. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed October 21, 2019. http://www.theses.fr/2014PA05T061.

MLA Handbook (7th Edition):

Cherifi, Ibtissem. “Impact de la signalisation constitutive du récepteur de la prolactine sur la physiopathologie mammaire : Impact of the constitutive signaling of the prolactin receptor on mammary pathophysiology.” 2014. Web. 21 Oct 2019.

Vancouver:

Cherifi I. Impact de la signalisation constitutive du récepteur de la prolactine sur la physiopathologie mammaire : Impact of the constitutive signaling of the prolactin receptor on mammary pathophysiology. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2014. [cited 2019 Oct 21]. Available from: http://www.theses.fr/2014PA05T061.

Council of Science Editors:

Cherifi I. Impact de la signalisation constitutive du récepteur de la prolactine sur la physiopathologie mammaire : Impact of the constitutive signaling of the prolactin receptor on mammary pathophysiology. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2014. Available from: http://www.theses.fr/2014PA05T061


University of Utah

12. Lyons, Bradley W. Development of an assay for modulating anti-acetylcholine autoantibodies using a human rhabdomyosarcoma cell line;.

Degree: MS;, Pathology;, 1998, University of Utah

 Myasthenia gravis (MG) is an autoimmune disease that affects the neuromuscular transmission at the synaptic junction. A polyclonal autoantibody response is elicited against the acetylcholine… (more)

Subjects/Keywords: Pathology; Pathophysiology

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APA (6th Edition):

Lyons, B. W. (1998). Development of an assay for modulating anti-acetylcholine autoantibodies using a human rhabdomyosarcoma cell line;. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/849/rec/310

Chicago Manual of Style (16th Edition):

Lyons, Bradley W. “Development of an assay for modulating anti-acetylcholine autoantibodies using a human rhabdomyosarcoma cell line;.” 1998. Masters Thesis, University of Utah. Accessed October 21, 2019. http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/849/rec/310.

MLA Handbook (7th Edition):

Lyons, Bradley W. “Development of an assay for modulating anti-acetylcholine autoantibodies using a human rhabdomyosarcoma cell line;.” 1998. Web. 21 Oct 2019.

Vancouver:

Lyons BW. Development of an assay for modulating anti-acetylcholine autoantibodies using a human rhabdomyosarcoma cell line;. [Internet] [Masters thesis]. University of Utah; 1998. [cited 2019 Oct 21]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/849/rec/310.

Council of Science Editors:

Lyons BW. Development of an assay for modulating anti-acetylcholine autoantibodies using a human rhabdomyosarcoma cell line;. [Masters Thesis]. University of Utah; 1998. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/849/rec/310


University of Hong Kong

13. 談沛詩.; Tam, Pui-see, Patricia. Developing an in vivo reporter system for the monitoring of therapeutic effects on neuroblastoma.

Degree: Master of Medical Sciences, 2009, University of Hong Kong

published_or_final_version

Surgery

Master

Master of Medical Sciences

Subjects/Keywords: Neuroblastoma - Pathophysiology.

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APA (6th Edition):

談沛詩.; Tam, Pui-see, P. (2009). Developing an in vivo reporter system for the monitoring of therapeutic effects on neuroblastoma. (Masters Thesis). University of Hong Kong. Retrieved from Tam, P. P. [談沛詩]. (2009). Developing an in vivo reporter system for the monitoring of therapeutic effects on neuroblastoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4290523 ; http://dx.doi.org/10.5353/th_b4290523 ; http://hdl.handle.net/10722/56981

Chicago Manual of Style (16th Edition):

談沛詩.; Tam, Pui-see, Patricia. “Developing an in vivo reporter system for the monitoring of therapeutic effects on neuroblastoma.” 2009. Masters Thesis, University of Hong Kong. Accessed October 21, 2019. Tam, P. P. [談沛詩]. (2009). Developing an in vivo reporter system for the monitoring of therapeutic effects on neuroblastoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4290523 ; http://dx.doi.org/10.5353/th_b4290523 ; http://hdl.handle.net/10722/56981.

MLA Handbook (7th Edition):

談沛詩.; Tam, Pui-see, Patricia. “Developing an in vivo reporter system for the monitoring of therapeutic effects on neuroblastoma.” 2009. Web. 21 Oct 2019.

Vancouver:

談沛詩.; Tam, Pui-see P. Developing an in vivo reporter system for the monitoring of therapeutic effects on neuroblastoma. [Internet] [Masters thesis]. University of Hong Kong; 2009. [cited 2019 Oct 21]. Available from: Tam, P. P. [談沛詩]. (2009). Developing an in vivo reporter system for the monitoring of therapeutic effects on neuroblastoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4290523 ; http://dx.doi.org/10.5353/th_b4290523 ; http://hdl.handle.net/10722/56981.

Council of Science Editors:

談沛詩.; Tam, Pui-see P. Developing an in vivo reporter system for the monitoring of therapeutic effects on neuroblastoma. [Masters Thesis]. University of Hong Kong; 2009. Available from: Tam, P. P. [談沛詩]. (2009). Developing an in vivo reporter system for the monitoring of therapeutic effects on neuroblastoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4290523 ; http://dx.doi.org/10.5353/th_b4290523 ; http://hdl.handle.net/10722/56981


University of Hong Kong

14. Dee, Cathleen Michelle Ang. Sublethal iron overload can alter the morphology and function of dendritic cells which may predispose to gram-negative infection in beta-thalassemia.

Degree: PhD, 2014, University of Hong Kong

 Iron overload is the most common complication in transfusion-dependent thalassemias. Iron overloaded patients are frequently susceptible to gram-negative organisms such as Klebsiella pneumonaie. Literature has… (more)

Subjects/Keywords: Thalassemia; Dendritic cells; Iron - Pathophysiology

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APA (6th Edition):

Dee, C. M. A. (2014). Sublethal iron overload can alter the morphology and function of dendritic cells which may predispose to gram-negative infection in beta-thalassemia. (Doctoral Dissertation). University of Hong Kong. Retrieved from Dee, C. M. A. [李明芳]. (2014). Sublethal iron overload can alter the morphology and function of dendritic cells which may predispose to gram-negative infection in beta-thalassemia. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5387950 ; http://dx.doi.org/10.5353/th_b5387950 ; http://hdl.handle.net/10722/221235

Chicago Manual of Style (16th Edition):

Dee, Cathleen Michelle Ang. “Sublethal iron overload can alter the morphology and function of dendritic cells which may predispose to gram-negative infection in beta-thalassemia.” 2014. Doctoral Dissertation, University of Hong Kong. Accessed October 21, 2019. Dee, C. M. A. [李明芳]. (2014). Sublethal iron overload can alter the morphology and function of dendritic cells which may predispose to gram-negative infection in beta-thalassemia. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5387950 ; http://dx.doi.org/10.5353/th_b5387950 ; http://hdl.handle.net/10722/221235.

MLA Handbook (7th Edition):

Dee, Cathleen Michelle Ang. “Sublethal iron overload can alter the morphology and function of dendritic cells which may predispose to gram-negative infection in beta-thalassemia.” 2014. Web. 21 Oct 2019.

Vancouver:

Dee CMA. Sublethal iron overload can alter the morphology and function of dendritic cells which may predispose to gram-negative infection in beta-thalassemia. [Internet] [Doctoral dissertation]. University of Hong Kong; 2014. [cited 2019 Oct 21]. Available from: Dee, C. M. A. [李明芳]. (2014). Sublethal iron overload can alter the morphology and function of dendritic cells which may predispose to gram-negative infection in beta-thalassemia. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5387950 ; http://dx.doi.org/10.5353/th_b5387950 ; http://hdl.handle.net/10722/221235.

Council of Science Editors:

Dee CMA. Sublethal iron overload can alter the morphology and function of dendritic cells which may predispose to gram-negative infection in beta-thalassemia. [Doctoral Dissertation]. University of Hong Kong; 2014. Available from: Dee, C. M. A. [李明芳]. (2014). Sublethal iron overload can alter the morphology and function of dendritic cells which may predispose to gram-negative infection in beta-thalassemia. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5387950 ; http://dx.doi.org/10.5353/th_b5387950 ; http://hdl.handle.net/10722/221235


Ryerson University

15. Jakubovic, Raphael, Y. MRI based physiological parameters are biomarkers of tumor and non tumor tissue response following radiation to brain metastases.

Degree: 2014, Ryerson University

 We sought to determine the utility of early relative blood volume (rCBV), relative blood flow (rCBF) and permeability (K2 trans) measurements as biomarkers of radiation… (more)

Subjects/Keywords: Diagnostic imaging; Cancer  – Pathophysiology

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APA (6th Edition):

Jakubovic, Raphael, Y. (2014). MRI based physiological parameters are biomarkers of tumor and non tumor tissue response following radiation to brain metastases. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A3089

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jakubovic, Raphael, Y. “MRI based physiological parameters are biomarkers of tumor and non tumor tissue response following radiation to brain metastases.” 2014. Thesis, Ryerson University. Accessed October 21, 2019. https://digital.library.ryerson.ca/islandora/object/RULA%3A3089.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jakubovic, Raphael, Y. “MRI based physiological parameters are biomarkers of tumor and non tumor tissue response following radiation to brain metastases.” 2014. Web. 21 Oct 2019.

Vancouver:

Jakubovic, Raphael Y. MRI based physiological parameters are biomarkers of tumor and non tumor tissue response following radiation to brain metastases. [Internet] [Thesis]. Ryerson University; 2014. [cited 2019 Oct 21]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A3089.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jakubovic, Raphael Y. MRI based physiological parameters are biomarkers of tumor and non tumor tissue response following radiation to brain metastases. [Thesis]. Ryerson University; 2014. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A3089

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Victoria

16. Lescheid, David William. Evolution of three neuropeptides isolated from the brain of sturgeon.

Degree: Department of Biology, 2018, University of Victoria

 In vertebrates the brain superimposes control on fundamental processes such as reproduction and growth. Neuropeptides secreted from the brain initiate a cascade of events that… (more)

Subjects/Keywords: Fishes; Physiology; Neuropeptides; Pathophysiology; Neurobiology

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APA (6th Edition):

Lescheid, D. W. (2018). Evolution of three neuropeptides isolated from the brain of sturgeon. (Thesis). University of Victoria. Retrieved from https://dspace.library.uvic.ca//handle/1828/9767

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lescheid, David William. “Evolution of three neuropeptides isolated from the brain of sturgeon.” 2018. Thesis, University of Victoria. Accessed October 21, 2019. https://dspace.library.uvic.ca//handle/1828/9767.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lescheid, David William. “Evolution of three neuropeptides isolated from the brain of sturgeon.” 2018. Web. 21 Oct 2019.

Vancouver:

Lescheid DW. Evolution of three neuropeptides isolated from the brain of sturgeon. [Internet] [Thesis]. University of Victoria; 2018. [cited 2019 Oct 21]. Available from: https://dspace.library.uvic.ca//handle/1828/9767.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lescheid DW. Evolution of three neuropeptides isolated from the brain of sturgeon. [Thesis]. University of Victoria; 2018. Available from: https://dspace.library.uvic.ca//handle/1828/9767

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

17. Yazdi, Fereshteh. The Genetic Basis of Obesity.

Degree: MSc, 2015, McMaster University

This work contains a summary of the current genetic, epigenetic and metagenomic knowledge of obesity, as well as an extensive overview of current genetic approaches… (more)

Subjects/Keywords: Obesity; Genetics; Epigenetics; pathophysiology

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APA (6th Edition):

Yazdi, F. (2015). The Genetic Basis of Obesity. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/18665

Chicago Manual of Style (16th Edition):

Yazdi, Fereshteh. “The Genetic Basis of Obesity.” 2015. Masters Thesis, McMaster University. Accessed October 21, 2019. http://hdl.handle.net/11375/18665.

MLA Handbook (7th Edition):

Yazdi, Fereshteh. “The Genetic Basis of Obesity.” 2015. Web. 21 Oct 2019.

Vancouver:

Yazdi F. The Genetic Basis of Obesity. [Internet] [Masters thesis]. McMaster University; 2015. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/11375/18665.

Council of Science Editors:

Yazdi F. The Genetic Basis of Obesity. [Masters Thesis]. McMaster University; 2015. Available from: http://hdl.handle.net/11375/18665


University of Manitoba

18. Ramjiawan, Angela. Transcriptional regulation of the peroxisome proliferator-activated receptor γ coactivator 1α (PGC-1α) promoter.

Degree: Physiology, 2010, University of Manitoba

 PGC-1α regulates cardiac mitochondrial biogenesis and energy metabolic gene expression, thus transcriptional regulation of PGC-1α gene expression is of great importance in understanding metabolic gene… (more)

Subjects/Keywords: Cardiovascular Physiology; Molecular Pathophysiology

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APA (6th Edition):

Ramjiawan, A. (2010). Transcriptional regulation of the peroxisome proliferator-activated receptor γ coactivator 1α (PGC-1α) promoter. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/4160

Chicago Manual of Style (16th Edition):

Ramjiawan, Angela. “Transcriptional regulation of the peroxisome proliferator-activated receptor γ coactivator 1α (PGC-1α) promoter.” 2010. Masters Thesis, University of Manitoba. Accessed October 21, 2019. http://hdl.handle.net/1993/4160.

MLA Handbook (7th Edition):

Ramjiawan, Angela. “Transcriptional regulation of the peroxisome proliferator-activated receptor γ coactivator 1α (PGC-1α) promoter.” 2010. Web. 21 Oct 2019.

Vancouver:

Ramjiawan A. Transcriptional regulation of the peroxisome proliferator-activated receptor γ coactivator 1α (PGC-1α) promoter. [Internet] [Masters thesis]. University of Manitoba; 2010. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1993/4160.

Council of Science Editors:

Ramjiawan A. Transcriptional regulation of the peroxisome proliferator-activated receptor γ coactivator 1α (PGC-1α) promoter. [Masters Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/4160


University of KwaZulu-Natal

19. Sigauke, Farai Russell. An investigation into the clinical outcomes of women with peripartum cardiomyopathy at Klerksdorp/Tshepong Hospital Complex.

Degree: 2015, University of KwaZulu-Natal

 Introduction: Peripartum Cardiomyopathy (PPCM) is defined on the basis of 4 criteria:  The development of cardiac failure in the last month of pregnancy and… (more)

Subjects/Keywords: Peripartum cardiomyopathy.; Aetiology.; Epidemology.; Pathophysiology.

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APA (6th Edition):

Sigauke, F. R. (2015). An investigation into the clinical outcomes of women with peripartum cardiomyopathy at Klerksdorp/Tshepong Hospital Complex. (Thesis). University of KwaZulu-Natal. Retrieved from http://hdl.handle.net/10413/16052

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sigauke, Farai Russell. “An investigation into the clinical outcomes of women with peripartum cardiomyopathy at Klerksdorp/Tshepong Hospital Complex.” 2015. Thesis, University of KwaZulu-Natal. Accessed October 21, 2019. http://hdl.handle.net/10413/16052.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sigauke, Farai Russell. “An investigation into the clinical outcomes of women with peripartum cardiomyopathy at Klerksdorp/Tshepong Hospital Complex.” 2015. Web. 21 Oct 2019.

Vancouver:

Sigauke FR. An investigation into the clinical outcomes of women with peripartum cardiomyopathy at Klerksdorp/Tshepong Hospital Complex. [Internet] [Thesis]. University of KwaZulu-Natal; 2015. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10413/16052.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sigauke FR. An investigation into the clinical outcomes of women with peripartum cardiomyopathy at Klerksdorp/Tshepong Hospital Complex. [Thesis]. University of KwaZulu-Natal; 2015. Available from: http://hdl.handle.net/10413/16052

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Sadoudi, Sihem. Implication des vésicules extracellulaires d'érythrocytes dans la dysfonction vasculaire drépanocytaire : Capacités de manipulation chez les hominidés : une étude pluridisciplinaire liant comportement, morphologie et modélisation.

Degree: Docteur es, Physiopathologie, 2017, Sorbonne Paris Cité

 L'hémolyse intravasculaire est la destruction prématurée et chronique des globules rouges. Elle est caractérisée par une libération accrue d’hème et de microvésicules (MV) membranaires dans… (more)

Subjects/Keywords: Physiopathologie; Pathophysiology; 616.152 7

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APA (6th Edition):

Sadoudi, S. (2017). Implication des vésicules extracellulaires d'érythrocytes dans la dysfonction vasculaire drépanocytaire : Capacités de manipulation chez les hominidés : une étude pluridisciplinaire liant comportement, morphologie et modélisation. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2017USPCB161

Chicago Manual of Style (16th Edition):

Sadoudi, Sihem. “Implication des vésicules extracellulaires d'érythrocytes dans la dysfonction vasculaire drépanocytaire : Capacités de manipulation chez les hominidés : une étude pluridisciplinaire liant comportement, morphologie et modélisation.” 2017. Doctoral Dissertation, Sorbonne Paris Cité. Accessed October 21, 2019. http://www.theses.fr/2017USPCB161.

MLA Handbook (7th Edition):

Sadoudi, Sihem. “Implication des vésicules extracellulaires d'érythrocytes dans la dysfonction vasculaire drépanocytaire : Capacités de manipulation chez les hominidés : une étude pluridisciplinaire liant comportement, morphologie et modélisation.” 2017. Web. 21 Oct 2019.

Vancouver:

Sadoudi S. Implication des vésicules extracellulaires d'érythrocytes dans la dysfonction vasculaire drépanocytaire : Capacités de manipulation chez les hominidés : une étude pluridisciplinaire liant comportement, morphologie et modélisation. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2017. [cited 2019 Oct 21]. Available from: http://www.theses.fr/2017USPCB161.

Council of Science Editors:

Sadoudi S. Implication des vésicules extracellulaires d'érythrocytes dans la dysfonction vasculaire drépanocytaire : Capacités de manipulation chez les hominidés : une étude pluridisciplinaire liant comportement, morphologie et modélisation. [Doctoral Dissertation]. Sorbonne Paris Cité; 2017. Available from: http://www.theses.fr/2017USPCB161


University of New Mexico

21. Geeda, Anil Reddy. Frequency specific deficits in schizophrenia.

Degree: Electrical and Computer Engineering, 2012, University of New Mexico

 Coherence estimation is one of the methods for understanding functional connectivity deficits and frequency specific deficits in schizophrenia. Coherence between different lobes of the brain… (more)

Subjects/Keywords: Schizophrenia – Pathophysiology; Magnetoencephalography; Delta rhythm.

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APA (6th Edition):

Geeda, A. R. (2012). Frequency specific deficits in schizophrenia. (Masters Thesis). University of New Mexico. Retrieved from http://hdl.handle.net/1928/17428

Chicago Manual of Style (16th Edition):

Geeda, Anil Reddy. “Frequency specific deficits in schizophrenia.” 2012. Masters Thesis, University of New Mexico. Accessed October 21, 2019. http://hdl.handle.net/1928/17428.

MLA Handbook (7th Edition):

Geeda, Anil Reddy. “Frequency specific deficits in schizophrenia.” 2012. Web. 21 Oct 2019.

Vancouver:

Geeda AR. Frequency specific deficits in schizophrenia. [Internet] [Masters thesis]. University of New Mexico; 2012. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1928/17428.

Council of Science Editors:

Geeda AR. Frequency specific deficits in schizophrenia. [Masters Thesis]. University of New Mexico; 2012. Available from: http://hdl.handle.net/1928/17428


UCLA

22. Williams, Drake. The role of host inflammation and the commensal microbiota in the pathogenesis of medication-related osteonecrosis of the jaw.

Degree: Oral Biology, 2019, UCLA

 Bisphosphonates (BPs) and denosumab (Dmab) are anti-resorptive drugs that are clinically used to treat bone disorders like osteoporosis and bone metastases. A detrimental side effect… (more)

Subjects/Keywords: Dentistry; inflammation; microbiota; MRONJ; pathophysiology

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APA (6th Edition):

Williams, D. (2019). The role of host inflammation and the commensal microbiota in the pathogenesis of medication-related osteonecrosis of the jaw. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/32x5r9h4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Drake. “The role of host inflammation and the commensal microbiota in the pathogenesis of medication-related osteonecrosis of the jaw.” 2019. Thesis, UCLA. Accessed October 21, 2019. http://www.escholarship.org/uc/item/32x5r9h4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Drake. “The role of host inflammation and the commensal microbiota in the pathogenesis of medication-related osteonecrosis of the jaw.” 2019. Web. 21 Oct 2019.

Vancouver:

Williams D. The role of host inflammation and the commensal microbiota in the pathogenesis of medication-related osteonecrosis of the jaw. [Internet] [Thesis]. UCLA; 2019. [cited 2019 Oct 21]. Available from: http://www.escholarship.org/uc/item/32x5r9h4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams D. The role of host inflammation and the commensal microbiota in the pathogenesis of medication-related osteonecrosis of the jaw. [Thesis]. UCLA; 2019. Available from: http://www.escholarship.org/uc/item/32x5r9h4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

23. Cunningham, Anna Maria. Immune Mediators of Murine Lyme Arthritis.

Degree: 2014, University of Missouri – Columbia

 Lyme disease is the most common vector-borne disease in both the United States and Europe; however, its pathogenesis is incompletely understood. The studies described in… (more)

Subjects/Keywords: Lyme disease; Eicosanoids  – Pathophysiology; Arthritis

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APA (6th Edition):

Cunningham, A. M. (2014). Immune Mediators of Murine Lyme Arthritis. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/45872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cunningham, Anna Maria. “Immune Mediators of Murine Lyme Arthritis.” 2014. Thesis, University of Missouri – Columbia. Accessed October 21, 2019. http://hdl.handle.net/10355/45872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cunningham, Anna Maria. “Immune Mediators of Murine Lyme Arthritis.” 2014. Web. 21 Oct 2019.

Vancouver:

Cunningham AM. Immune Mediators of Murine Lyme Arthritis. [Internet] [Thesis]. University of Missouri – Columbia; 2014. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10355/45872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cunningham AM. Immune Mediators of Murine Lyme Arthritis. [Thesis]. University of Missouri – Columbia; 2014. Available from: http://hdl.handle.net/10355/45872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

24. Moriarty, Gina M., 1986-. Molecular triggers of disease-related synuclein aggregation in the Parkinson’s environment.

Degree: PhD, Chemistry and Chemical Biology, 2017, Rutgers University

 The works in this thesis explores the challenges involved in recapitulating the environment in which the synuclein family of proteins resides, functions and are active… (more)

Subjects/Keywords: Parkinson's disease – Pathophysiology; Synucleins

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APA (6th Edition):

Moriarty, Gina M., 1. (2017). Molecular triggers of disease-related synuclein aggregation in the Parkinson’s environment. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/55602/

Chicago Manual of Style (16th Edition):

Moriarty, Gina M., 1986-. “Molecular triggers of disease-related synuclein aggregation in the Parkinson’s environment.” 2017. Doctoral Dissertation, Rutgers University. Accessed October 21, 2019. https://rucore.libraries.rutgers.edu/rutgers-lib/55602/.

MLA Handbook (7th Edition):

Moriarty, Gina M., 1986-. “Molecular triggers of disease-related synuclein aggregation in the Parkinson’s environment.” 2017. Web. 21 Oct 2019.

Vancouver:

Moriarty, Gina M. 1. Molecular triggers of disease-related synuclein aggregation in the Parkinson’s environment. [Internet] [Doctoral dissertation]. Rutgers University; 2017. [cited 2019 Oct 21]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55602/.

Council of Science Editors:

Moriarty, Gina M. 1. Molecular triggers of disease-related synuclein aggregation in the Parkinson’s environment. [Doctoral Dissertation]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55602/


Rutgers University

25. Yohn, Christine. Dentate gyrus’s role in the response to antidepressant treatment.

Degree: MS, Psychology, 2019, Rutgers University

 Depression is a complex psychiatric disorder that is a major burden on society as 300 million people are diagnosed worldwide. Furthermore, only 33% of depressed… (more)

Subjects/Keywords: Depression, Mental; Dentate gyrus – Pathophysiology

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APA (6th Edition):

Yohn, C. (2019). Dentate gyrus’s role in the response to antidepressant treatment. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/60108/

Chicago Manual of Style (16th Edition):

Yohn, Christine. “Dentate gyrus’s role in the response to antidepressant treatment.” 2019. Masters Thesis, Rutgers University. Accessed October 21, 2019. https://rucore.libraries.rutgers.edu/rutgers-lib/60108/.

MLA Handbook (7th Edition):

Yohn, Christine. “Dentate gyrus’s role in the response to antidepressant treatment.” 2019. Web. 21 Oct 2019.

Vancouver:

Yohn C. Dentate gyrus’s role in the response to antidepressant treatment. [Internet] [Masters thesis]. Rutgers University; 2019. [cited 2019 Oct 21]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60108/.

Council of Science Editors:

Yohn C. Dentate gyrus’s role in the response to antidepressant treatment. [Masters Thesis]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60108/


Simon Fraser University

26. Crisp, Dianne. Interictal and ictal localization derived from neuroelectric data.

Degree: 1994, Simon Fraser University

Subjects/Keywords: Epilepsy  – Pathophysiology.; Brain  – Pathophysiology.

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APA (6th Edition):

Crisp, D. (1994). Interictal and ictal localization derived from neuroelectric data. (Thesis). Simon Fraser University. Retrieved from http://summit.sfu.ca/item/5108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Crisp, Dianne. “Interictal and ictal localization derived from neuroelectric data.” 1994. Thesis, Simon Fraser University. Accessed October 21, 2019. http://summit.sfu.ca/item/5108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Crisp, Dianne. “Interictal and ictal localization derived from neuroelectric data.” 1994. Web. 21 Oct 2019.

Vancouver:

Crisp D. Interictal and ictal localization derived from neuroelectric data. [Internet] [Thesis]. Simon Fraser University; 1994. [cited 2019 Oct 21]. Available from: http://summit.sfu.ca/item/5108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Crisp D. Interictal and ictal localization derived from neuroelectric data. [Thesis]. Simon Fraser University; 1994. Available from: http://summit.sfu.ca/item/5108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

27. McDevitt, Jane K. N-METHYL-D-ASPARTIC ACID RECEPTOR SUBUNIT NR2A REPEAT.

Degree: PhD, 2013, Temple University

Kinesiology

During a concussion, mechanical forces cause neuron cell strain that initiates dysfunction through the indiscriminate movement of ions through protein channels. Extracellular glutamate binds… (more)

Subjects/Keywords: Kinesiology; Genetics;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McDevitt, J. K. (2013). N-METHYL-D-ASPARTIC ACID RECEPTOR SUBUNIT NR2A REPEAT. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,216597

Chicago Manual of Style (16th Edition):

McDevitt, Jane K. “N-METHYL-D-ASPARTIC ACID RECEPTOR SUBUNIT NR2A REPEAT.” 2013. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,216597.

MLA Handbook (7th Edition):

McDevitt, Jane K. “N-METHYL-D-ASPARTIC ACID RECEPTOR SUBUNIT NR2A REPEAT.” 2013. Web. 21 Oct 2019.

Vancouver:

McDevitt JK. N-METHYL-D-ASPARTIC ACID RECEPTOR SUBUNIT NR2A REPEAT. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,216597.

Council of Science Editors:

McDevitt JK. N-METHYL-D-ASPARTIC ACID RECEPTOR SUBUNIT NR2A REPEAT. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,216597


University of KwaZulu-Natal

28. [No author]. The effect of an anti-inflammatory homeopathic product on cytokine status in venous blood following 90 minutes of downhill running.

Degree: Sports medicine, 2008, University of KwaZulu-Natal

 Background: Downhill running involves eccentric contractions of the gluteal, quadriceps, hamstring and calf muscles and the lengthening of muscle fibres as they contract. Several studies… (more)

Subjects/Keywords: Cytokines – Pathophysiology.; Inflammation – Mediators.; Sports medicine.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

author], [. (2008). The effect of an anti-inflammatory homeopathic product on cytokine status in venous blood following 90 minutes of downhill running. (Thesis). University of KwaZulu-Natal. Retrieved from http://hdl.handle.net/10413/1129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “The effect of an anti-inflammatory homeopathic product on cytokine status in venous blood following 90 minutes of downhill running. ” 2008. Thesis, University of KwaZulu-Natal. Accessed October 21, 2019. http://hdl.handle.net/10413/1129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “The effect of an anti-inflammatory homeopathic product on cytokine status in venous blood following 90 minutes of downhill running. ” 2008. Web. 21 Oct 2019.

Vancouver:

author] [. The effect of an anti-inflammatory homeopathic product on cytokine status in venous blood following 90 minutes of downhill running. [Internet] [Thesis]. University of KwaZulu-Natal; 2008. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10413/1129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. The effect of an anti-inflammatory homeopathic product on cytokine status in venous blood following 90 minutes of downhill running. [Thesis]. University of KwaZulu-Natal; 2008. Available from: http://hdl.handle.net/10413/1129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

29. 謝冰姣; Xie, Bingjiao. Association of arterial stiffness and blood pressure variability with silent brain lesions in healthy hypertensive elderly Chinese.

Degree: PhD, 2015, University of Hong Kong

 Silent brain lesions (SBLs) are frequently detected in elderly subjects with hypertension. Although without apparent stroke symptoms, they are related to high risks of stroke… (more)

Subjects/Keywords: Blood pressure - Physiology; Arteries - Pathophysiology; Neurobehavioral disorders

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

謝冰姣; Xie, B. (2015). Association of arterial stiffness and blood pressure variability with silent brain lesions in healthy hypertensive elderly Chinese. (Doctoral Dissertation). University of Hong Kong. Retrieved from Xie, B. [謝冰姣]. (2015). Association of arterial stiffness and blood pressure variability with silent brain lesions in healthy hypertensive elderly Chinese. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5543987 ; http://dx.doi.org/10.5353/th_b5543987 ; http://hdl.handle.net/10722/225942

Chicago Manual of Style (16th Edition):

謝冰姣; Xie, Bingjiao. “Association of arterial stiffness and blood pressure variability with silent brain lesions in healthy hypertensive elderly Chinese.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed October 21, 2019. Xie, B. [謝冰姣]. (2015). Association of arterial stiffness and blood pressure variability with silent brain lesions in healthy hypertensive elderly Chinese. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5543987 ; http://dx.doi.org/10.5353/th_b5543987 ; http://hdl.handle.net/10722/225942.

MLA Handbook (7th Edition):

謝冰姣; Xie, Bingjiao. “Association of arterial stiffness and blood pressure variability with silent brain lesions in healthy hypertensive elderly Chinese.” 2015. Web. 21 Oct 2019.

Vancouver:

謝冰姣; Xie B. Association of arterial stiffness and blood pressure variability with silent brain lesions in healthy hypertensive elderly Chinese. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Oct 21]. Available from: Xie, B. [謝冰姣]. (2015). Association of arterial stiffness and blood pressure variability with silent brain lesions in healthy hypertensive elderly Chinese. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5543987 ; http://dx.doi.org/10.5353/th_b5543987 ; http://hdl.handle.net/10722/225942.

Council of Science Editors:

謝冰姣; Xie B. Association of arterial stiffness and blood pressure variability with silent brain lesions in healthy hypertensive elderly Chinese. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Xie, B. [謝冰姣]. (2015). Association of arterial stiffness and blood pressure variability with silent brain lesions in healthy hypertensive elderly Chinese. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5543987 ; http://dx.doi.org/10.5353/th_b5543987 ; http://hdl.handle.net/10722/225942


Hong Kong University of Science and Technology

30. Ng, Shuk Han. Role of neuroligin in neuronal secretory pathway.

Degree: 2011, Hong Kong University of Science and Technology

 The neuroligins are a family of postsynaptic transmembrane proteins that associate specifically with their presynaptic partners, beta-neurexins. This trans-synaptic interaction has been extensively studied in… (more)

Subjects/Keywords: Neural transmission; Membrane proteins; Autism; Pathophysiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ng, S. H. (2011). Role of neuroligin in neuronal secretory pathway. (Thesis). Hong Kong University of Science and Technology. Retrieved from https://doi.org/10.14711/thesis-b1155452 ; http://repository.ust.hk/ir/bitstream/1783.1-73406/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ng, Shuk Han. “Role of neuroligin in neuronal secretory pathway.” 2011. Thesis, Hong Kong University of Science and Technology. Accessed October 21, 2019. https://doi.org/10.14711/thesis-b1155452 ; http://repository.ust.hk/ir/bitstream/1783.1-73406/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ng, Shuk Han. “Role of neuroligin in neuronal secretory pathway.” 2011. Web. 21 Oct 2019.

Vancouver:

Ng SH. Role of neuroligin in neuronal secretory pathway. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2011. [cited 2019 Oct 21]. Available from: https://doi.org/10.14711/thesis-b1155452 ; http://repository.ust.hk/ir/bitstream/1783.1-73406/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ng SH. Role of neuroligin in neuronal secretory pathway. [Thesis]. Hong Kong University of Science and Technology; 2011. Available from: https://doi.org/10.14711/thesis-b1155452 ; http://repository.ust.hk/ir/bitstream/1783.1-73406/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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