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You searched for subject:(Parkin). Showing records 1 – 30 of 67 total matches.

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University of Ottawa

1. Ao, Hei Sio. Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin .

Degree: 2015, University of Ottawa

Parkin gene is associated with the development of autosomal recessive juvenile parkinsonism (Kitada et al., 1998) which is a common form of familial Parkinson’s disease… (more)

Subjects/Keywords: Parkinson's disease; Parkin; Nrf2

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APA (6th Edition):

Ao, H. S. (2015). Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/33368

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ao, Hei Sio. “Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin .” 2015. Thesis, University of Ottawa. Accessed August 05, 2020. http://hdl.handle.net/10393/33368.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ao, Hei Sio. “Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin .” 2015. Web. 05 Aug 2020.

Vancouver:

Ao HS. Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin . [Internet] [Thesis]. University of Ottawa; 2015. [cited 2020 Aug 05]. Available from: http://hdl.handle.net/10393/33368.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ao HS. Investigation of Cis and Trans-acting Transcriptional Regulatory Factors and Signaling Pathways of Parkin . [Thesis]. University of Ottawa; 2015. Available from: http://hdl.handle.net/10393/33368

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

2. Baker, Hanan. Iron and Lactoferrin Levels Remain Unchanged in Parkin Deficient Mouse Brains: Implications for Parkinson’s Disease Etiology.

Degree: Biochemistry & Molecular Biology, 2015, UCLA

 Parkinson’s disease is a devastating neurodegenerative disease in which loss of dopaminergic neurons in the substantia nigra pars compacta leads to a variety of motor… (more)

Subjects/Keywords: Biochemistry; Neurosciences; Lactoferrin; Parkin; Parkinson's Disease

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APA (6th Edition):

Baker, H. (2015). Iron and Lactoferrin Levels Remain Unchanged in Parkin Deficient Mouse Brains: Implications for Parkinson’s Disease Etiology. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/9ss381mk

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baker, Hanan. “Iron and Lactoferrin Levels Remain Unchanged in Parkin Deficient Mouse Brains: Implications for Parkinson’s Disease Etiology.” 2015. Thesis, UCLA. Accessed August 05, 2020. http://www.escholarship.org/uc/item/9ss381mk.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baker, Hanan. “Iron and Lactoferrin Levels Remain Unchanged in Parkin Deficient Mouse Brains: Implications for Parkinson’s Disease Etiology.” 2015. Web. 05 Aug 2020.

Vancouver:

Baker H. Iron and Lactoferrin Levels Remain Unchanged in Parkin Deficient Mouse Brains: Implications for Parkinson’s Disease Etiology. [Internet] [Thesis]. UCLA; 2015. [cited 2020 Aug 05]. Available from: http://www.escholarship.org/uc/item/9ss381mk.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baker H. Iron and Lactoferrin Levels Remain Unchanged in Parkin Deficient Mouse Brains: Implications for Parkinson’s Disease Etiology. [Thesis]. UCLA; 2015. Available from: http://www.escholarship.org/uc/item/9ss381mk

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

3. Lombardo, Portia Sachi. Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis.

Degree: Biology, 2017, University of California – San Diego

 Our laboratory recently identified the autophagy initiating kinase ULK1 as a novel AMPK substrate, which directly connects the highly conserved AMPK-mediated energy sensing pathway to… (more)

Subjects/Keywords: Molecular biology; AMPK; mitophagy; Parkin; ULK1

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APA (6th Edition):

Lombardo, P. S. (2017). Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/515006px

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lombardo, Portia Sachi. “Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis.” 2017. Thesis, University of California – San Diego. Accessed August 05, 2020. http://www.escholarship.org/uc/item/515006px.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lombardo, Portia Sachi. “Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis.” 2017. Web. 05 Aug 2020.

Vancouver:

Lombardo PS. Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2020 Aug 05]. Available from: http://www.escholarship.org/uc/item/515006px.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lombardo PS. Phosphorylation of Parkin by the autophagy initiating kinase ULK1 regulates mitochondrial homeostasis. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/515006px

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Berkeley

4. Killilea, Alison Nisbet. Characterization of Parkin, Tau, and Zinc Interactions with the Microtubule Network.

Degree: Comparative Biochemistry, 2009, University of California – Berkeley

 The microtubule cytoskeleton is essential for regulation of cell morphology, trafficking within the cell and cell cycle progression. These diverse functions involve complex microtubule dynamics… (more)

Subjects/Keywords:

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APA (6th Edition):

Killilea, A. N. (2009). Characterization of Parkin, Tau, and Zinc Interactions with the Microtubule Network. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/6t23f2r1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Killilea, Alison Nisbet. “Characterization of Parkin, Tau, and Zinc Interactions with the Microtubule Network.” 2009. Thesis, University of California – Berkeley. Accessed August 05, 2020. http://www.escholarship.org/uc/item/6t23f2r1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Killilea, Alison Nisbet. “Characterization of Parkin, Tau, and Zinc Interactions with the Microtubule Network.” 2009. Web. 05 Aug 2020.

Vancouver:

Killilea AN. Characterization of Parkin, Tau, and Zinc Interactions with the Microtubule Network. [Internet] [Thesis]. University of California – Berkeley; 2009. [cited 2020 Aug 05]. Available from: http://www.escholarship.org/uc/item/6t23f2r1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Killilea AN. Characterization of Parkin, Tau, and Zinc Interactions with the Microtubule Network. [Thesis]. University of California – Berkeley; 2009. Available from: http://www.escholarship.org/uc/item/6t23f2r1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

5. Feng, Chien-Wei. Establishment of a Parkinsonâs disease model in zebrafish.

Degree: Master, Marine Biotechnology and Resources, 2011, NSYSU

 Recently, the zebrafish has been considered an important animal model that can be used to investigate human diseases and drug development. Parkinsonâs disease (PD), an… (more)

Subjects/Keywords: zebrafish; tyrosine hydroxylase; parkin; Parkinsonâs disease

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APA (6th Edition):

Feng, C. (2011). Establishment of a Parkinsonâs disease model in zebrafish. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0901111-000150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Feng, Chien-Wei. “Establishment of a Parkinsonâs disease model in zebrafish.” 2011. Thesis, NSYSU. Accessed August 05, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0901111-000150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Feng, Chien-Wei. “Establishment of a Parkinsonâs disease model in zebrafish.” 2011. Web. 05 Aug 2020.

Vancouver:

Feng C. Establishment of a Parkinsonâs disease model in zebrafish. [Internet] [Thesis]. NSYSU; 2011. [cited 2020 Aug 05]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0901111-000150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Feng C. Establishment of a Parkinsonâs disease model in zebrafish. [Thesis]. NSYSU; 2011. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0901111-000150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

6. Safarpour, Farzaneh. The Role of Parkinson's Disease Gene PTEN-Induced Putative Kinase 1, PINK1 in Ischemia Induced Neuronal Injury .

Degree: 2016, University of Ottawa

 Stroke results from disturbance in blood flow to an area of the brain, leading to neuronal dysfunction and loss. Mitochondrial dysfunction and oxidative stress are… (more)

Subjects/Keywords: Parkinson's disease; PINK1; Ischemia; Stroke; Parkin

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APA (6th Edition):

Safarpour, F. (2016). The Role of Parkinson's Disease Gene PTEN-Induced Putative Kinase 1, PINK1 in Ischemia Induced Neuronal Injury . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/34475

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Safarpour, Farzaneh. “The Role of Parkinson's Disease Gene PTEN-Induced Putative Kinase 1, PINK1 in Ischemia Induced Neuronal Injury .” 2016. Thesis, University of Ottawa. Accessed August 05, 2020. http://hdl.handle.net/10393/34475.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Safarpour, Farzaneh. “The Role of Parkinson's Disease Gene PTEN-Induced Putative Kinase 1, PINK1 in Ischemia Induced Neuronal Injury .” 2016. Web. 05 Aug 2020.

Vancouver:

Safarpour F. The Role of Parkinson's Disease Gene PTEN-Induced Putative Kinase 1, PINK1 in Ischemia Induced Neuronal Injury . [Internet] [Thesis]. University of Ottawa; 2016. [cited 2020 Aug 05]. Available from: http://hdl.handle.net/10393/34475.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Safarpour F. The Role of Parkinson's Disease Gene PTEN-Induced Putative Kinase 1, PINK1 in Ischemia Induced Neuronal Injury . [Thesis]. University of Ottawa; 2016. Available from: http://hdl.handle.net/10393/34475

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

7. Bernardini, Jonathan. Regulation of the apoptotic machinery by the E3 ubiquitin ligase Parkin.

Degree: 2018, University of Melbourne

 The cyto-protective molecule Parkin is a criticial player in autosomal-recessive Parkinson’s disease, with approximately half of all patients with this condition carrying a mutant allele… (more)

Subjects/Keywords: Ubiquitin; Parkin; BAK; BAX; Apoptosis; Mitochondria; Mitophagy

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APA (6th Edition):

Bernardini, J. (2018). Regulation of the apoptotic machinery by the E3 ubiquitin ligase Parkin. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/220985

Chicago Manual of Style (16th Edition):

Bernardini, Jonathan. “Regulation of the apoptotic machinery by the E3 ubiquitin ligase Parkin.” 2018. Doctoral Dissertation, University of Melbourne. Accessed August 05, 2020. http://hdl.handle.net/11343/220985.

MLA Handbook (7th Edition):

Bernardini, Jonathan. “Regulation of the apoptotic machinery by the E3 ubiquitin ligase Parkin.” 2018. Web. 05 Aug 2020.

Vancouver:

Bernardini J. Regulation of the apoptotic machinery by the E3 ubiquitin ligase Parkin. [Internet] [Doctoral dissertation]. University of Melbourne; 2018. [cited 2020 Aug 05]. Available from: http://hdl.handle.net/11343/220985.

Council of Science Editors:

Bernardini J. Regulation of the apoptotic machinery by the E3 ubiquitin ligase Parkin. [Doctoral Dissertation]. University of Melbourne; 2018. Available from: http://hdl.handle.net/11343/220985


UCLA

8. Zhang, Ting. The role of Mitofusin (Mfn) in the pathogenesis of Parkinson’s disease and myopathy/frontotemporal dementia.

Degree: Molec, Cell, & Integ Physiology, 2018, UCLA

 As life expectancy prolongs, the incidence of neurodegenerative diseases continues to rise. The pathogenesis of neurodegenerative diseases is poorly understood and there is no effective… (more)

Subjects/Keywords: Neurosciences; Genetics; IBMPFD; Mitofusin (Mfn); Parkin; Parkinson's Disease; PINK1; VCP

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APA (6th Edition):

Zhang, T. (2018). The role of Mitofusin (Mfn) in the pathogenesis of Parkinson’s disease and myopathy/frontotemporal dementia. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/15t536ft

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Ting. “The role of Mitofusin (Mfn) in the pathogenesis of Parkinson’s disease and myopathy/frontotemporal dementia.” 2018. Thesis, UCLA. Accessed August 05, 2020. http://www.escholarship.org/uc/item/15t536ft.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Ting. “The role of Mitofusin (Mfn) in the pathogenesis of Parkinson’s disease and myopathy/frontotemporal dementia.” 2018. Web. 05 Aug 2020.

Vancouver:

Zhang T. The role of Mitofusin (Mfn) in the pathogenesis of Parkinson’s disease and myopathy/frontotemporal dementia. [Internet] [Thesis]. UCLA; 2018. [cited 2020 Aug 05]. Available from: http://www.escholarship.org/uc/item/15t536ft.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang T. The role of Mitofusin (Mfn) in the pathogenesis of Parkinson’s disease and myopathy/frontotemporal dementia. [Thesis]. UCLA; 2018. Available from: http://www.escholarship.org/uc/item/15t536ft

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

9. Koehler, Christopher Lee. Metabolic regulation of intestinal stem cell function in Drosophila melanogaster.

Degree: Molecular Biology, 2016, UCLA

 Aging is a degenerative process characterized by the accumulation of cellular damage that results in altered tissue homeostasis, organ function, and, ultimately, death. Tissues with… (more)

Subjects/Keywords: Molecular biology; Cellular biology; Drosophila; Intestinal stem cell; Mitophagy; Parkin; Pink1

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APA (6th Edition):

Koehler, C. L. (2016). Metabolic regulation of intestinal stem cell function in Drosophila melanogaster. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/9cc9p3ck

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Koehler, Christopher Lee. “Metabolic regulation of intestinal stem cell function in Drosophila melanogaster.” 2016. Thesis, UCLA. Accessed August 05, 2020. http://www.escholarship.org/uc/item/9cc9p3ck.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Koehler, Christopher Lee. “Metabolic regulation of intestinal stem cell function in Drosophila melanogaster.” 2016. Web. 05 Aug 2020.

Vancouver:

Koehler CL. Metabolic regulation of intestinal stem cell function in Drosophila melanogaster. [Internet] [Thesis]. UCLA; 2016. [cited 2020 Aug 05]. Available from: http://www.escholarship.org/uc/item/9cc9p3ck.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Koehler CL. Metabolic regulation of intestinal stem cell function in Drosophila melanogaster. [Thesis]. UCLA; 2016. Available from: http://www.escholarship.org/uc/item/9cc9p3ck

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

10. Ha, Ainhi Duy. Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease .

Degree: 2014, University of Sydney

 There is increasing evidence to suggest that mitochondrial dysfunction plays a key role in the patho-physiology of Parkinson’s Disease (PD). The transcriptional coactivator PGC-1alpha is… (more)

Subjects/Keywords: Parkinson’s Disease; PGC-1alpha; mitochondria; PINK1; parkin; ATP13A2; LRRK2

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APA (6th Edition):

Ha, A. D. (2014). Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/12264

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ha, Ainhi Duy. “Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease .” 2014. Thesis, University of Sydney. Accessed August 05, 2020. http://hdl.handle.net/2123/12264.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ha, Ainhi Duy. “Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease .” 2014. Web. 05 Aug 2020.

Vancouver:

Ha AD. Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease . [Internet] [Thesis]. University of Sydney; 2014. [cited 2020 Aug 05]. Available from: http://hdl.handle.net/2123/12264.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ha AD. Mitochondrial Dysfunction and the Role of PGC-1a in Genetic Parkinson’s Disease . [Thesis]. University of Sydney; 2014. Available from: http://hdl.handle.net/2123/12264

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Harvard University

11. Sarraf, Shireen Akhavan. Defining the Landscape of the PARKIN- and PINK1-Dependent Ubiquitin-Modified Proteome in Response to Mitochondrial Dysfunction.

Degree: PhD, Cell Biology, 2013, Harvard University

 Parkinson's disease (PD) is a progressive neurological disorder resulting from loss of dopaminergic neurons of the substantia nigra, in part due to mitochondrial dysfunction. The… (more)

Subjects/Keywords: Cellular biology; Molecular biology; diGly; Parkin; Parkinson's; Pink1; Proteomics; Ubiquitin

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APA (6th Edition):

Sarraf, S. A. (2013). Defining the Landscape of the PARKIN- and PINK1-Dependent Ubiquitin-Modified Proteome in Response to Mitochondrial Dysfunction. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:11110427

Chicago Manual of Style (16th Edition):

Sarraf, Shireen Akhavan. “Defining the Landscape of the PARKIN- and PINK1-Dependent Ubiquitin-Modified Proteome in Response to Mitochondrial Dysfunction.” 2013. Doctoral Dissertation, Harvard University. Accessed August 05, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:11110427.

MLA Handbook (7th Edition):

Sarraf, Shireen Akhavan. “Defining the Landscape of the PARKIN- and PINK1-Dependent Ubiquitin-Modified Proteome in Response to Mitochondrial Dysfunction.” 2013. Web. 05 Aug 2020.

Vancouver:

Sarraf SA. Defining the Landscape of the PARKIN- and PINK1-Dependent Ubiquitin-Modified Proteome in Response to Mitochondrial Dysfunction. [Internet] [Doctoral dissertation]. Harvard University; 2013. [cited 2020 Aug 05]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:11110427.

Council of Science Editors:

Sarraf SA. Defining the Landscape of the PARKIN- and PINK1-Dependent Ubiquitin-Modified Proteome in Response to Mitochondrial Dysfunction. [Doctoral Dissertation]. Harvard University; 2013. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:11110427


Universidade Nova

12. Morais, Sara Peres de. Juvenile Parkinson disease caused by parkin mutations: large deletions and pathogenic mechanisms.

Degree: 2011, Universidade Nova

Dissertação para obtenção do Grau de Mestre em Genética Molecular e Biomedicina

Autosomal recessive juvenile Parkinson disease (AR-JP) is mainly caused by mutations in PARK2.… (more)

Subjects/Keywords: AR-JP; Parkin; Ubiquitin-proteasome system; Protein aggregation; Neuronal death

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APA (6th Edition):

Morais, S. P. d. (2011). Juvenile Parkinson disease caused by parkin mutations: large deletions and pathogenic mechanisms. (Thesis). Universidade Nova. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/6723

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morais, Sara Peres de. “Juvenile Parkinson disease caused by parkin mutations: large deletions and pathogenic mechanisms.” 2011. Thesis, Universidade Nova. Accessed August 05, 2020. http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/6723.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morais, Sara Peres de. “Juvenile Parkinson disease caused by parkin mutations: large deletions and pathogenic mechanisms.” 2011. Web. 05 Aug 2020.

Vancouver:

Morais SPd. Juvenile Parkinson disease caused by parkin mutations: large deletions and pathogenic mechanisms. [Internet] [Thesis]. Universidade Nova; 2011. [cited 2020 Aug 05]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/6723.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morais SPd. Juvenile Parkinson disease caused by parkin mutations: large deletions and pathogenic mechanisms. [Thesis]. Universidade Nova; 2011. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/6723

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

13. Conti, Michael. Small Molecule Probes of Mitochondrial Translocases Elucidate PINK1 Trafficking in PINK1/Parkin Regulated Mitophagy.

Degree: Biochemistry, Molecular and Structural Biology, 2018, UCLA

 Dysfunctional mitochondria result in many human diseases including some cancers and many metabolic diseases. More recently, momentum has been building to implicate defective mitochondria as… (more)

Subjects/Keywords: Biochemistry; Molecular biology; MitoBloCK; mitophagy; Parkin; Parkinson's disease; PINK1; Screen

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APA (6th Edition):

Conti, M. (2018). Small Molecule Probes of Mitochondrial Translocases Elucidate PINK1 Trafficking in PINK1/Parkin Regulated Mitophagy. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/20j5m9bd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Conti, Michael. “Small Molecule Probes of Mitochondrial Translocases Elucidate PINK1 Trafficking in PINK1/Parkin Regulated Mitophagy.” 2018. Thesis, UCLA. Accessed August 05, 2020. http://www.escholarship.org/uc/item/20j5m9bd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Conti, Michael. “Small Molecule Probes of Mitochondrial Translocases Elucidate PINK1 Trafficking in PINK1/Parkin Regulated Mitophagy.” 2018. Web. 05 Aug 2020.

Vancouver:

Conti M. Small Molecule Probes of Mitochondrial Translocases Elucidate PINK1 Trafficking in PINK1/Parkin Regulated Mitophagy. [Internet] [Thesis]. UCLA; 2018. [cited 2020 Aug 05]. Available from: http://www.escholarship.org/uc/item/20j5m9bd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Conti M. Small Molecule Probes of Mitochondrial Translocases Elucidate PINK1 Trafficking in PINK1/Parkin Regulated Mitophagy. [Thesis]. UCLA; 2018. Available from: http://www.escholarship.org/uc/item/20j5m9bd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wayne State University

14. Chauhan, Heli Dineshchandra. The Role Of E3 Ligase Parkin In Trafficking Of Monoamine Storage Vesicles In Rat Model Of Methamphetamine Neurotoxicity.

Degree: MS, Pharmaceutical Sciences, 2015, Wayne State University

  Methamphetamine (METH), a psychostimulant, is a widely used drug of abuse. METH is toxic to dopaminergic (DAergic) and serotonergic (5-HT) nerve terminals in the… (more)

Subjects/Keywords: Methamphetamine; Parkin; VMAT2; Medicinal Chemistry and Pharmaceutics; Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chauhan, H. D. (2015). The Role Of E3 Ligase Parkin In Trafficking Of Monoamine Storage Vesicles In Rat Model Of Methamphetamine Neurotoxicity. (Masters Thesis). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_theses/370

Chicago Manual of Style (16th Edition):

Chauhan, Heli Dineshchandra. “The Role Of E3 Ligase Parkin In Trafficking Of Monoamine Storage Vesicles In Rat Model Of Methamphetamine Neurotoxicity.” 2015. Masters Thesis, Wayne State University. Accessed August 05, 2020. https://digitalcommons.wayne.edu/oa_theses/370.

MLA Handbook (7th Edition):

Chauhan, Heli Dineshchandra. “The Role Of E3 Ligase Parkin In Trafficking Of Monoamine Storage Vesicles In Rat Model Of Methamphetamine Neurotoxicity.” 2015. Web. 05 Aug 2020.

Vancouver:

Chauhan HD. The Role Of E3 Ligase Parkin In Trafficking Of Monoamine Storage Vesicles In Rat Model Of Methamphetamine Neurotoxicity. [Internet] [Masters thesis]. Wayne State University; 2015. [cited 2020 Aug 05]. Available from: https://digitalcommons.wayne.edu/oa_theses/370.

Council of Science Editors:

Chauhan HD. The Role Of E3 Ligase Parkin In Trafficking Of Monoamine Storage Vesicles In Rat Model Of Methamphetamine Neurotoxicity. [Masters Thesis]. Wayne State University; 2015. Available from: https://digitalcommons.wayne.edu/oa_theses/370

15. Williams, Jessica A. R. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2015, University of Kansas

 Acetaminophen (APAP) is the leading cause of acute liver failure in the United States, and alcoholic liver disease (ALD) is a worldwide health problem that… (more)

Subjects/Keywords: Toxicology; Acetaminophen; Alcohol; Autophagy; Liver Injury; Mitophagy; Parkin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Williams, J. A. R. (2015). The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/19470

Chicago Manual of Style (16th Edition):

Williams, Jessica A R. “The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.” 2015. Doctoral Dissertation, University of Kansas. Accessed August 05, 2020. http://hdl.handle.net/1808/19470.

MLA Handbook (7th Edition):

Williams, Jessica A R. “The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries.” 2015. Web. 05 Aug 2020.

Vancouver:

Williams JAR. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. [Internet] [Doctoral dissertation]. University of Kansas; 2015. [cited 2020 Aug 05]. Available from: http://hdl.handle.net/1808/19470.

Council of Science Editors:

Williams JAR. The Role of Parkin and Mitophagy in Acetaminophen and Alcohol-induced Liver Injuries. [Doctoral Dissertation]. University of Kansas; 2015. Available from: http://hdl.handle.net/1808/19470

16. XIAO BIN. THE ROLE OF ROS AND VDAC1 IN PARKIN/PINK1-MEDIATED MITOPHAGY.

Degree: 2014, National University of Singapore

Subjects/Keywords: ROS; VDAC1; Parkin; PINK1; mitophagy

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APA (6th Edition):

BIN, X. (2014). THE ROLE OF ROS AND VDAC1 IN PARKIN/PINK1-MEDIATED MITOPHAGY. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/110955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

BIN, XIAO. “THE ROLE OF ROS AND VDAC1 IN PARKIN/PINK1-MEDIATED MITOPHAGY.” 2014. Thesis, National University of Singapore. Accessed August 05, 2020. http://scholarbank.nus.edu.sg/handle/10635/110955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

BIN, XIAO. “THE ROLE OF ROS AND VDAC1 IN PARKIN/PINK1-MEDIATED MITOPHAGY.” 2014. Web. 05 Aug 2020.

Vancouver:

BIN X. THE ROLE OF ROS AND VDAC1 IN PARKIN/PINK1-MEDIATED MITOPHAGY. [Internet] [Thesis]. National University of Singapore; 2014. [cited 2020 Aug 05]. Available from: http://scholarbank.nus.edu.sg/handle/10635/110955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

BIN X. THE ROLE OF ROS AND VDAC1 IN PARKIN/PINK1-MEDIATED MITOPHAGY. [Thesis]. National University of Singapore; 2014. Available from: http://scholarbank.nus.edu.sg/handle/10635/110955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

17. Bastasic, Joseph. The Impact of Parkinson’s Disease on Mammalian Adult Neurogenesis .

Degree: 2019, University of Ottawa

 Parkinson’s disease (PD) has been reported to negatively affect adult neurogenesis. Mitochondrial dysfunction associated with PD may be involved, given that recent studies have identified… (more)

Subjects/Keywords: Parkinson’s Disease; Adult Neurogenesis; Subventricular Zone; Subgranular Zone; PINK1; Parkin; Mitochondria

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APA (6th Edition):

Bastasic, J. (2019). The Impact of Parkinson’s Disease on Mammalian Adult Neurogenesis . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/39610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bastasic, Joseph. “The Impact of Parkinson’s Disease on Mammalian Adult Neurogenesis .” 2019. Thesis, University of Ottawa. Accessed August 05, 2020. http://hdl.handle.net/10393/39610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bastasic, Joseph. “The Impact of Parkinson’s Disease on Mammalian Adult Neurogenesis .” 2019. Web. 05 Aug 2020.

Vancouver:

Bastasic J. The Impact of Parkinson’s Disease on Mammalian Adult Neurogenesis . [Internet] [Thesis]. University of Ottawa; 2019. [cited 2020 Aug 05]. Available from: http://hdl.handle.net/10393/39610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bastasic J. The Impact of Parkinson’s Disease on Mammalian Adult Neurogenesis . [Thesis]. University of Ottawa; 2019. Available from: http://hdl.handle.net/10393/39610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

18. Ang, Michelle. The Effect of Helicobacter pylori on Innate Immunity.

Degree: 2010, University of Toronto

The innate immune system is important in both acute and chronic infection. In this thesis, I investigated the effect of H. pylori infection on 1)… (more)

Subjects/Keywords: H. pylori; Immunity; STAT3; Dendritic Cell; Autophagy; VacA; ROS; Parkin; 0719

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APA (6th Edition):

Ang, M. (2010). The Effect of Helicobacter pylori on Innate Immunity. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/24529

Chicago Manual of Style (16th Edition):

Ang, Michelle. “The Effect of Helicobacter pylori on Innate Immunity.” 2010. Masters Thesis, University of Toronto. Accessed August 05, 2020. http://hdl.handle.net/1807/24529.

MLA Handbook (7th Edition):

Ang, Michelle. “The Effect of Helicobacter pylori on Innate Immunity.” 2010. Web. 05 Aug 2020.

Vancouver:

Ang M. The Effect of Helicobacter pylori on Innate Immunity. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Aug 05]. Available from: http://hdl.handle.net/1807/24529.

Council of Science Editors:

Ang M. The Effect of Helicobacter pylori on Innate Immunity. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24529


Michigan State University

19. Hawong, Hae-young. Parkin mediated mitochondrial quality control in central dopamine neurons.

Degree: 2014, Michigan State University

Thesis Ph. D. Michigan State University. Biochemistry and Molecular Biology 2014.

Parkinson disease (PD) is the second most common neurodegenerative disease. The hallmark pathology of… (more)

Subjects/Keywords: Dopaminergic neurons; Mitochondria; Parkinson's disease – Pathogenesis – Research; Biochemistry; Neurosciences; Parkin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hawong, H. (2014). Parkin mediated mitochondrial quality control in central dopamine neurons. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:2979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hawong, Hae-young. “Parkin mediated mitochondrial quality control in central dopamine neurons.” 2014. Thesis, Michigan State University. Accessed August 05, 2020. http://etd.lib.msu.edu/islandora/object/etd:2979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hawong, Hae-young. “Parkin mediated mitochondrial quality control in central dopamine neurons.” 2014. Web. 05 Aug 2020.

Vancouver:

Hawong H. Parkin mediated mitochondrial quality control in central dopamine neurons. [Internet] [Thesis]. Michigan State University; 2014. [cited 2020 Aug 05]. Available from: http://etd.lib.msu.edu/islandora/object/etd:2979.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hawong H. Parkin mediated mitochondrial quality control in central dopamine neurons. [Thesis]. Michigan State University; 2014. Available from: http://etd.lib.msu.edu/islandora/object/etd:2979

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

20. George, Susanna. Modulating Parkin E3 Ubiquitin Ligase Activity Using Phospho-Ubiquitin Variants.

Degree: 2016, University of Western Ontario

Parkin is a Parkinson’s disease-linked E3 ubiquitin (Ub) ligase that promotes mitophagy by ubiquitination of mitochondrial outer membrane proteins. Phosphorylation of Ub at Ser65 by… (more)

Subjects/Keywords: Genetic code expansion; parkin; Parkinson’s disease; phosphorylation; tRNA; ubiquitin; Biochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

George, S. (2016). Modulating Parkin E3 Ubiquitin Ligase Activity Using Phospho-Ubiquitin Variants. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4003

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

George, Susanna. “Modulating Parkin E3 Ubiquitin Ligase Activity Using Phospho-Ubiquitin Variants.” 2016. Thesis, University of Western Ontario. Accessed August 05, 2020. https://ir.lib.uwo.ca/etd/4003.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

George, Susanna. “Modulating Parkin E3 Ubiquitin Ligase Activity Using Phospho-Ubiquitin Variants.” 2016. Web. 05 Aug 2020.

Vancouver:

George S. Modulating Parkin E3 Ubiquitin Ligase Activity Using Phospho-Ubiquitin Variants. [Internet] [Thesis]. University of Western Ontario; 2016. [cited 2020 Aug 05]. Available from: https://ir.lib.uwo.ca/etd/4003.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

George S. Modulating Parkin E3 Ubiquitin Ligase Activity Using Phospho-Ubiquitin Variants. [Thesis]. University of Western Ontario; 2016. Available from: https://ir.lib.uwo.ca/etd/4003

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

21. Condos, Tara E. C. Conformational Arrangements of UbcH7-Ubiquitin with OspG and Parkin.

Degree: 2018, University of Western Ontario

 The E2-ubiquitin conjugate is a key regulator of ubiquitination and is therefore an important component of cellular homeostasis. Disruptions to proper E2-ubiquitin functioning have implications… (more)

Subjects/Keywords: Parkinson’s disease; shigellosis; ubiquitination; parkin; OspG; UbcH7-Ubiquitin; Biochemistry

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APA (6th Edition):

Condos, T. E. C. (2018). Conformational Arrangements of UbcH7-Ubiquitin with OspG and Parkin. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/5330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Condos, Tara E C. “Conformational Arrangements of UbcH7-Ubiquitin with OspG and Parkin.” 2018. Thesis, University of Western Ontario. Accessed August 05, 2020. https://ir.lib.uwo.ca/etd/5330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Condos, Tara E C. “Conformational Arrangements of UbcH7-Ubiquitin with OspG and Parkin.” 2018. Web. 05 Aug 2020.

Vancouver:

Condos TEC. Conformational Arrangements of UbcH7-Ubiquitin with OspG and Parkin. [Internet] [Thesis]. University of Western Ontario; 2018. [cited 2020 Aug 05]. Available from: https://ir.lib.uwo.ca/etd/5330.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Condos TEC. Conformational Arrangements of UbcH7-Ubiquitin with OspG and Parkin. [Thesis]. University of Western Ontario; 2018. Available from: https://ir.lib.uwo.ca/etd/5330

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

22. McCarton, Alexander S. Parkin Misfolding, Dysfunction, and Degradation in Parkinson's Disease.

Degree: 2016, University of Western Ontario

 Mutations in the gene encoding parkin (PARK2) result in familial early onset forms of Parkinson’s disease (PD) based on the loss of parkin’s E3 ubiquitin… (more)

Subjects/Keywords: Parkinson’s disease; parkin; oxidative stress; protein misfolding; aggregation; yeast model.; Biochemistry

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APA (6th Edition):

McCarton, A. S. (2016). Parkin Misfolding, Dysfunction, and Degradation in Parkinson's Disease. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/3799

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McCarton, Alexander S. “Parkin Misfolding, Dysfunction, and Degradation in Parkinson's Disease.” 2016. Thesis, University of Western Ontario. Accessed August 05, 2020. https://ir.lib.uwo.ca/etd/3799.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McCarton, Alexander S. “Parkin Misfolding, Dysfunction, and Degradation in Parkinson's Disease.” 2016. Web. 05 Aug 2020.

Vancouver:

McCarton AS. Parkin Misfolding, Dysfunction, and Degradation in Parkinson's Disease. [Internet] [Thesis]. University of Western Ontario; 2016. [cited 2020 Aug 05]. Available from: https://ir.lib.uwo.ca/etd/3799.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McCarton AS. Parkin Misfolding, Dysfunction, and Degradation in Parkinson's Disease. [Thesis]. University of Western Ontario; 2016. Available from: https://ir.lib.uwo.ca/etd/3799

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

23. Bai, Jane J. Characterization of the Interaction between the Parkin Ubiquitin-like domain and Ataxin-3 Ubiquitin Interacting Domains.

Degree: 2012, University of Western Ontario

 The ubiquitin signaling pathway (USP) consists of hundreds of enzymes which are tightly regulated for proper maintenance of intracellular protein level homeostasis. The main goals… (more)

Subjects/Keywords: Parkin; Ataxin-3; Interaction; NMR; HSQC; Multivalent; Biochemistry

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APA (6th Edition):

Bai, J. J. (2012). Characterization of the Interaction between the Parkin Ubiquitin-like domain and Ataxin-3 Ubiquitin Interacting Domains. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/841

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bai, Jane J. “Characterization of the Interaction between the Parkin Ubiquitin-like domain and Ataxin-3 Ubiquitin Interacting Domains.” 2012. Thesis, University of Western Ontario. Accessed August 05, 2020. https://ir.lib.uwo.ca/etd/841.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bai, Jane J. “Characterization of the Interaction between the Parkin Ubiquitin-like domain and Ataxin-3 Ubiquitin Interacting Domains.” 2012. Web. 05 Aug 2020.

Vancouver:

Bai JJ. Characterization of the Interaction between the Parkin Ubiquitin-like domain and Ataxin-3 Ubiquitin Interacting Domains. [Internet] [Thesis]. University of Western Ontario; 2012. [cited 2020 Aug 05]. Available from: https://ir.lib.uwo.ca/etd/841.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bai JJ. Characterization of the Interaction between the Parkin Ubiquitin-like domain and Ataxin-3 Ubiquitin Interacting Domains. [Thesis]. University of Western Ontario; 2012. Available from: https://ir.lib.uwo.ca/etd/841

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

24. Noh, Yeong Ju. Structure of C-terminal Domain of Parkin, IBR-RING2.

Degree: 2013, University of Western Ontario

Parkin is an E3 ubiquitin ligase which degrades misfolded proteins and prevents the formation of abnormal protein aggregates often formed in Parkinson’s disease. The main… (more)

Subjects/Keywords: Parkinson’s Disease; parkin; ARJP; solution structure; nuclear magnetic resonance; ubiquitination; Biochemistry

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APA (6th Edition):

Noh, Y. J. (2013). Structure of C-terminal Domain of Parkin, IBR-RING2. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/1324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Noh, Yeong Ju. “Structure of C-terminal Domain of Parkin, IBR-RING2.” 2013. Thesis, University of Western Ontario. Accessed August 05, 2020. https://ir.lib.uwo.ca/etd/1324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Noh, Yeong Ju. “Structure of C-terminal Domain of Parkin, IBR-RING2.” 2013. Web. 05 Aug 2020.

Vancouver:

Noh YJ. Structure of C-terminal Domain of Parkin, IBR-RING2. [Internet] [Thesis]. University of Western Ontario; 2013. [cited 2020 Aug 05]. Available from: https://ir.lib.uwo.ca/etd/1324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Noh YJ. Structure of C-terminal Domain of Parkin, IBR-RING2. [Thesis]. University of Western Ontario; 2013. Available from: https://ir.lib.uwo.ca/etd/1324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

25. SHI, GUANG. Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control.

Degree: PhD, 2017, University of Toronto

 Mitochondrial quality control (MQC) systems are essential for preserving the functional integrity and the homeostasis of mitochondria. Dysfunction of MQC is emerging as a central… (more)

Subjects/Keywords: mitochondrial quality control; Mitophagy; Parkinson's Diseases; PARL; PDK2; PINK1/PARKIN; 0487

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

SHI, G. (2017). Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/79472

Chicago Manual of Style (16th Edition):

SHI, GUANG. “Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control.” 2017. Doctoral Dissertation, University of Toronto. Accessed August 05, 2020. http://hdl.handle.net/1807/79472.

MLA Handbook (7th Edition):

SHI, GUANG. “Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control.” 2017. Web. 05 Aug 2020.

Vancouver:

SHI G. Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2020 Aug 05]. Available from: http://hdl.handle.net/1807/79472.

Council of Science Editors:

SHI G. Characterizing the Function of the Mitochondrial Protease PARL in Mitophagy and Mitochondrial Quality Control. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/79472


Duke University

26. Kapur, Meghan Danielle. The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease .

Degree: 2015, Duke University

  Advances in modern medicine have helped to prolong human life. These advancements coupled with an ever-increasing population means that diseases associated with aging will… (more)

Subjects/Keywords: Cellular biology; HDAC6; lipid droplets; mitochondria; neurodegeneration; obesity; Parkin

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APA (6th Edition):

Kapur, M. D. (2015). The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kapur, Meghan Danielle. “The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease .” 2015. Thesis, Duke University. Accessed August 05, 2020. http://hdl.handle.net/10161/9852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kapur, Meghan Danielle. “The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease .” 2015. Web. 05 Aug 2020.

Vancouver:

Kapur MD. The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease . [Internet] [Thesis]. Duke University; 2015. [cited 2020 Aug 05]. Available from: http://hdl.handle.net/10161/9852.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kapur MD. The Regulation of Lipid Metabolism and Mitochondrial Quality Control in Health and Disease . [Thesis]. Duke University; 2015. Available from: http://hdl.handle.net/10161/9852

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

27. Yun, Jina. Genetic analysis of Omi/HtrA2 and MUL1 in the PINK1/Parkin pathway.

Degree: Molec & Med Pharmacology, 2013, UCLA

 Parkinson's disease (PD) is the second most common neurodegenerative disorder. Poor understanding of PD pathogenesis has limited the development of effective therapies. Recently, studies on… (more)

Subjects/Keywords: Genetics; Molecular biology; Neurosciences; Mitochondria; Mitofusin; MUL1; omi; Parkinson's disease; PINK1/Parkin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yun, J. (2013). Genetic analysis of Omi/HtrA2 and MUL1 in the PINK1/Parkin pathway. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/5bc5662v

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yun, Jina. “Genetic analysis of Omi/HtrA2 and MUL1 in the PINK1/Parkin pathway.” 2013. Thesis, UCLA. Accessed August 05, 2020. http://www.escholarship.org/uc/item/5bc5662v.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yun, Jina. “Genetic analysis of Omi/HtrA2 and MUL1 in the PINK1/Parkin pathway.” 2013. Web. 05 Aug 2020.

Vancouver:

Yun J. Genetic analysis of Omi/HtrA2 and MUL1 in the PINK1/Parkin pathway. [Internet] [Thesis]. UCLA; 2013. [cited 2020 Aug 05]. Available from: http://www.escholarship.org/uc/item/5bc5662v.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yun J. Genetic analysis of Omi/HtrA2 and MUL1 in the PINK1/Parkin pathway. [Thesis]. UCLA; 2013. Available from: http://www.escholarship.org/uc/item/5bc5662v

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

28. Lekova, Virzhiniya. Role And Impact Of The Gut Microbiota In A Drosophila Model For Parkinsonism.

Degree: 2017, University of Pennsylvania

 Current studies suggest a relationship between microbes in the gut and the brain, including an impact on brain disease development, severity, and progression. However, exploring… (more)

Subjects/Keywords: 16S sequencing; drosophila; dysbiosis; gut microbiome; parkin; parkinsonism; Genetics; Molecular Biology; Nanoscience and Nanotechnology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lekova, V. (2017). Role And Impact Of The Gut Microbiota In A Drosophila Model For Parkinsonism. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lekova, Virzhiniya. “Role And Impact Of The Gut Microbiota In A Drosophila Model For Parkinsonism.” 2017. Thesis, University of Pennsylvania. Accessed August 05, 2020. https://repository.upenn.edu/edissertations/2996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lekova, Virzhiniya. “Role And Impact Of The Gut Microbiota In A Drosophila Model For Parkinsonism.” 2017. Web. 05 Aug 2020.

Vancouver:

Lekova V. Role And Impact Of The Gut Microbiota In A Drosophila Model For Parkinsonism. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2020 Aug 05]. Available from: https://repository.upenn.edu/edissertations/2996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lekova V. Role And Impact Of The Gut Microbiota In A Drosophila Model For Parkinsonism. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/2996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

29. Bae, KyeongMin. Role of Zinc in Parkin RING2 E3 Ubiquitin Ligase Ubiquitination.

Degree: MA, Molecular Biosciences, 2016, University of Kansas

 In common neurodegenerative diseases among older adults, Parkinson’s disease (PD) ranks the second after Alzheimer’s disease. Symptoms of Parkinson’s disease are shaking, slowness of movement,… (more)

Subjects/Keywords: Biochemistry; Biophysics; E3 ubiquitin ligase; Parkin; Parkinson's Disease; ubiquitin; ubiquitination; zinc binding

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bae, K. (2016). Role of Zinc in Parkin RING2 E3 Ubiquitin Ligase Ubiquitination. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/21809

Chicago Manual of Style (16th Edition):

Bae, KyeongMin. “Role of Zinc in Parkin RING2 E3 Ubiquitin Ligase Ubiquitination.” 2016. Masters Thesis, University of Kansas. Accessed August 05, 2020. http://hdl.handle.net/1808/21809.

MLA Handbook (7th Edition):

Bae, KyeongMin. “Role of Zinc in Parkin RING2 E3 Ubiquitin Ligase Ubiquitination.” 2016. Web. 05 Aug 2020.

Vancouver:

Bae K. Role of Zinc in Parkin RING2 E3 Ubiquitin Ligase Ubiquitination. [Internet] [Masters thesis]. University of Kansas; 2016. [cited 2020 Aug 05]. Available from: http://hdl.handle.net/1808/21809.

Council of Science Editors:

Bae K. Role of Zinc in Parkin RING2 E3 Ubiquitin Ligase Ubiquitination. [Masters Thesis]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/21809


Vanderbilt University

30. Chakraborty, Sudipta. Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes.

Degree: PhD, Neuroscience, 2015, Vanderbilt University

 Parkinsonâs disease (PD) is a neurodegenerative, motor disorder that is characterized by selective dopaminergic cell loss in the substantia nigra pars compacta. About 10-20% of… (more)

Subjects/Keywords: Parkinsons disease; manganese; neurotoxicology; C. elegans; parkin; pink1; dj1; ferroportin; DMT1; oxidative stress

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chakraborty, S. (2015). Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03152015-170233/ ;

Chicago Manual of Style (16th Edition):

Chakraborty, Sudipta. “Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed August 05, 2020. http://etd.library.vanderbilt.edu/available/etd-03152015-170233/ ;.

MLA Handbook (7th Edition):

Chakraborty, Sudipta. “Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes.” 2015. Web. 05 Aug 2020.

Vancouver:

Chakraborty S. Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Aug 05]. Available from: http://etd.library.vanderbilt.edu/available/etd-03152015-170233/ ;.

Council of Science Editors:

Chakraborty S. Gene-Environment Interactions Between Manganese Toxicity and Early-Onset Parkinson's Disease Genes. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-03152015-170233/ ;

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