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You searched for subject:(Pancreatic cancer). Showing records 1 – 30 of 482 total matches.

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University of Georgia

1. Hung, Sau Wai. Investigation of approaches for overcoming chemoresistance in pancreatic cancer.

Degree: PhD, Pharmacy, 2014, University of Georgia

Pancreatic cancer continues to be one of the most devastating malignances with a 5-year patient survival rate of less than 6%. One of the main… (more)

Subjects/Keywords: Pancreatic cancer

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APA (6th Edition):

Hung, S. W. (2014). Investigation of approaches for overcoming chemoresistance in pancreatic cancer. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/hung_sau-wai_201408_phd

Chicago Manual of Style (16th Edition):

Hung, Sau Wai. “Investigation of approaches for overcoming chemoresistance in pancreatic cancer.” 2014. Doctoral Dissertation, University of Georgia. Accessed April 07, 2020. http://purl.galileo.usg.edu/uga_etd/hung_sau-wai_201408_phd.

MLA Handbook (7th Edition):

Hung, Sau Wai. “Investigation of approaches for overcoming chemoresistance in pancreatic cancer.” 2014. Web. 07 Apr 2020.

Vancouver:

Hung SW. Investigation of approaches for overcoming chemoresistance in pancreatic cancer. [Internet] [Doctoral dissertation]. University of Georgia; 2014. [cited 2020 Apr 07]. Available from: http://purl.galileo.usg.edu/uga_etd/hung_sau-wai_201408_phd.

Council of Science Editors:

Hung SW. Investigation of approaches for overcoming chemoresistance in pancreatic cancer. [Doctoral Dissertation]. University of Georgia; 2014. Available from: http://purl.galileo.usg.edu/uga_etd/hung_sau-wai_201408_phd


Harvard University

2. Liu, Hao. Angiotensin System Inhibition in Pancreatic Ductal Adenocarcinoma.

Degree: PhD, 2017, Harvard University

Pancreatic ductal adenocarcinoma (PDAC) is a deadly disease that is characterized by its rich stroma. Targeting the PDAC stroma holds great potential as a novel… (more)

Subjects/Keywords: Angiotensin; Pancreatic Cancer

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APA (6th Edition):

Liu, H. (2017). Angiotensin System Inhibition in Pancreatic Ductal Adenocarcinoma. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:41141509

Chicago Manual of Style (16th Edition):

Liu, Hao. “Angiotensin System Inhibition in Pancreatic Ductal Adenocarcinoma.” 2017. Doctoral Dissertation, Harvard University. Accessed April 07, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:41141509.

MLA Handbook (7th Edition):

Liu, Hao. “Angiotensin System Inhibition in Pancreatic Ductal Adenocarcinoma.” 2017. Web. 07 Apr 2020.

Vancouver:

Liu H. Angiotensin System Inhibition in Pancreatic Ductal Adenocarcinoma. [Internet] [Doctoral dissertation]. Harvard University; 2017. [cited 2020 Apr 07]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:41141509.

Council of Science Editors:

Liu H. Angiotensin System Inhibition in Pancreatic Ductal Adenocarcinoma. [Doctoral Dissertation]. Harvard University; 2017. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:41141509


Queen Mary, University of London

3. Pantelidou, Constantia. E1B19K-deleted oncolytic adenoviruses enhancee the cytotoxicity of DNA-damaging drugs in pancreatic cancer through deregulation of cell-cycle mechanisms.

Degree: PhD, 2014, Queen Mary, University of London

Pancreatic cancer is an aggressive disease with poor prognosis and a high fatality rate. Gemcitabine, the standard first-line chemotherapy for advanced disease, has negligible effects,… (more)

Subjects/Keywords: 616.99; Cancer; Pancreatic cancer

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APA (6th Edition):

Pantelidou, C. (2014). E1B19K-deleted oncolytic adenoviruses enhancee the cytotoxicity of DNA-damaging drugs in pancreatic cancer through deregulation of cell-cycle mechanisms. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/8819 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667291

Chicago Manual of Style (16th Edition):

Pantelidou, Constantia. “E1B19K-deleted oncolytic adenoviruses enhancee the cytotoxicity of DNA-damaging drugs in pancreatic cancer through deregulation of cell-cycle mechanisms.” 2014. Doctoral Dissertation, Queen Mary, University of London. Accessed April 07, 2020. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8819 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667291.

MLA Handbook (7th Edition):

Pantelidou, Constantia. “E1B19K-deleted oncolytic adenoviruses enhancee the cytotoxicity of DNA-damaging drugs in pancreatic cancer through deregulation of cell-cycle mechanisms.” 2014. Web. 07 Apr 2020.

Vancouver:

Pantelidou C. E1B19K-deleted oncolytic adenoviruses enhancee the cytotoxicity of DNA-damaging drugs in pancreatic cancer through deregulation of cell-cycle mechanisms. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2014. [cited 2020 Apr 07]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8819 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667291.

Council of Science Editors:

Pantelidou C. E1B19K-deleted oncolytic adenoviruses enhancee the cytotoxicity of DNA-damaging drugs in pancreatic cancer through deregulation of cell-cycle mechanisms. [Doctoral Dissertation]. Queen Mary, University of London; 2014. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8819 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667291


Queen Mary, University of London

4. Vallath, Sabarinath S. Studying the role of integrin αVβ6 in pancreatic cancer.

Degree: PhD, 2013, Queen Mary, University of London

Pancreatic cancer is often referred to as the “silent killer“ due to the asymptomatic nature of the disease in the early stages and the extremely… (more)

Subjects/Keywords: 616.99; Medicine; Cancer; Pancreatic cancer

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APA (6th Edition):

Vallath, S. S. (2013). Studying the role of integrin αVβ6 in pancreatic cancer. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/8663 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667175

Chicago Manual of Style (16th Edition):

Vallath, Sabarinath S. “Studying the role of integrin αVβ6 in pancreatic cancer.” 2013. Doctoral Dissertation, Queen Mary, University of London. Accessed April 07, 2020. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8663 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667175.

MLA Handbook (7th Edition):

Vallath, Sabarinath S. “Studying the role of integrin αVβ6 in pancreatic cancer.” 2013. Web. 07 Apr 2020.

Vancouver:

Vallath SS. Studying the role of integrin αVβ6 in pancreatic cancer. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2013. [cited 2020 Apr 07]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8663 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667175.

Council of Science Editors:

Vallath SS. Studying the role of integrin αVβ6 in pancreatic cancer. [Doctoral Dissertation]. Queen Mary, University of London; 2013. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8663 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667175


Penn State University

5. Sharzehi, Setareh. IS ALCOHOL FREE ABLATION EFFECTIVE AND SAFE FOR TREATMENT OF PRE-CANCEROUS PANCREATIC CYSTS?.

Degree: 2018, Penn State University

 Background: Approximately 15% of precancerous pancreatic cysts will develop into pancreatic cancer. Endoscopic Ultrasound (EUS)-fine needle infusion is a new technique to deliver drugs and… (more)

Subjects/Keywords: Pancreatic Cancer Prevention; Chemotherapy; Mucinous Pancreatic Cysts

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APA (6th Edition):

Sharzehi, S. (2018). IS ALCOHOL FREE ABLATION EFFECTIVE AND SAFE FOR TREATMENT OF PRE-CANCEROUS PANCREATIC CYSTS?. (Thesis). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/15436sus60

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sharzehi, Setareh. “IS ALCOHOL FREE ABLATION EFFECTIVE AND SAFE FOR TREATMENT OF PRE-CANCEROUS PANCREATIC CYSTS?.” 2018. Thesis, Penn State University. Accessed April 07, 2020. https://etda.libraries.psu.edu/catalog/15436sus60.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sharzehi, Setareh. “IS ALCOHOL FREE ABLATION EFFECTIVE AND SAFE FOR TREATMENT OF PRE-CANCEROUS PANCREATIC CYSTS?.” 2018. Web. 07 Apr 2020.

Vancouver:

Sharzehi S. IS ALCOHOL FREE ABLATION EFFECTIVE AND SAFE FOR TREATMENT OF PRE-CANCEROUS PANCREATIC CYSTS?. [Internet] [Thesis]. Penn State University; 2018. [cited 2020 Apr 07]. Available from: https://etda.libraries.psu.edu/catalog/15436sus60.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sharzehi S. IS ALCOHOL FREE ABLATION EFFECTIVE AND SAFE FOR TREATMENT OF PRE-CANCEROUS PANCREATIC CYSTS?. [Thesis]. Penn State University; 2018. Available from: https://etda.libraries.psu.edu/catalog/15436sus60

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

6. Akerman, Anouschka. Metabolically rewiring pancreatic stromal cells in pancreatic cancer.

Degree: Prince of Wales Clinical School, 2019, University of New South Wales

Pancreatic cancer (PC) is a lethal malignancy characterised by chemoresistance and metastatic spread. This is largely due to the presence of dense stromal fibrosis which… (more)

Subjects/Keywords: Pancreatic cancer; Metabolic rewiring; Pancreatic stromal cells

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APA (6th Edition):

Akerman, A. (2019). Metabolically rewiring pancreatic stromal cells in pancreatic cancer. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/64923

Chicago Manual of Style (16th Edition):

Akerman, Anouschka. “Metabolically rewiring pancreatic stromal cells in pancreatic cancer.” 2019. Doctoral Dissertation, University of New South Wales. Accessed April 07, 2020. http://handle.unsw.edu.au/1959.4/64923.

MLA Handbook (7th Edition):

Akerman, Anouschka. “Metabolically rewiring pancreatic stromal cells in pancreatic cancer.” 2019. Web. 07 Apr 2020.

Vancouver:

Akerman A. Metabolically rewiring pancreatic stromal cells in pancreatic cancer. [Internet] [Doctoral dissertation]. University of New South Wales; 2019. [cited 2020 Apr 07]. Available from: http://handle.unsw.edu.au/1959.4/64923.

Council of Science Editors:

Akerman A. Metabolically rewiring pancreatic stromal cells in pancreatic cancer. [Doctoral Dissertation]. University of New South Wales; 2019. Available from: http://handle.unsw.edu.au/1959.4/64923

7. Tsiaousidou, Anastasia. Ανοσοϊστοχημικοί δείκτες ως προγνωστικοί παράγοντες στον καρκίνο του παγκρέατος.

Degree: 2015, Democritus University of Thrace (DUTH); Δημοκρίτειο Πανεπιστήμιο Θράκης (ΔΠΘ)

Pancreatic cancer (PC) is one of the most lethal tumors of the gastrointestinal tract. The ability to predict which patients would benet most from surgical… (more)

Subjects/Keywords: Καρκίνος παγκρέατος; Pancreatic cancer

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APA (6th Edition):

Tsiaousidou, A. (2015). Ανοσοϊστοχημικοί δείκτες ως προγνωστικοί παράγοντες στον καρκίνο του παγκρέατος. (Thesis). Democritus University of Thrace (DUTH); Δημοκρίτειο Πανεπιστήμιο Θράκης (ΔΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/35321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsiaousidou, Anastasia. “Ανοσοϊστοχημικοί δείκτες ως προγνωστικοί παράγοντες στον καρκίνο του παγκρέατος.” 2015. Thesis, Democritus University of Thrace (DUTH); Δημοκρίτειο Πανεπιστήμιο Θράκης (ΔΠΘ). Accessed April 07, 2020. http://hdl.handle.net/10442/hedi/35321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsiaousidou, Anastasia. “Ανοσοϊστοχημικοί δείκτες ως προγνωστικοί παράγοντες στον καρκίνο του παγκρέατος.” 2015. Web. 07 Apr 2020.

Vancouver:

Tsiaousidou A. Ανοσοϊστοχημικοί δείκτες ως προγνωστικοί παράγοντες στον καρκίνο του παγκρέατος. [Internet] [Thesis]. Democritus University of Thrace (DUTH); Δημοκρίτειο Πανεπιστήμιο Θράκης (ΔΠΘ); 2015. [cited 2020 Apr 07]. Available from: http://hdl.handle.net/10442/hedi/35321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsiaousidou A. Ανοσοϊστοχημικοί δείκτες ως προγνωστικοί παράγοντες στον καρκίνο του παγκρέατος. [Thesis]. Democritus University of Thrace (DUTH); Δημοκρίτειο Πανεπιστήμιο Θράκης (ΔΠΘ); 2015. Available from: http://hdl.handle.net/10442/hedi/35321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

8. Collins, Laura. The Mechanism of Action of a New Class of Nucleoside Analogs Targeting Gastrointestinal Tumours .

Degree: 2019, University of Ottawa

 Gastrointestinal malignancies such as liver and pancreatic cancers are the deadliest due to late detection and drug resistance. Nucleoside analogues, like Gemcitabine, are the conventional… (more)

Subjects/Keywords: Nucleoside Analogs; Pancreatic Cancer

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APA (6th Edition):

Collins, L. (2019). The Mechanism of Action of a New Class of Nucleoside Analogs Targeting Gastrointestinal Tumours . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/38845

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Collins, Laura. “The Mechanism of Action of a New Class of Nucleoside Analogs Targeting Gastrointestinal Tumours .” 2019. Thesis, University of Ottawa. Accessed April 07, 2020. http://hdl.handle.net/10393/38845.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Collins, Laura. “The Mechanism of Action of a New Class of Nucleoside Analogs Targeting Gastrointestinal Tumours .” 2019. Web. 07 Apr 2020.

Vancouver:

Collins L. The Mechanism of Action of a New Class of Nucleoside Analogs Targeting Gastrointestinal Tumours . [Internet] [Thesis]. University of Ottawa; 2019. [cited 2020 Apr 07]. Available from: http://hdl.handle.net/10393/38845.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Collins L. The Mechanism of Action of a New Class of Nucleoside Analogs Targeting Gastrointestinal Tumours . [Thesis]. University of Ottawa; 2019. Available from: http://hdl.handle.net/10393/38845

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

9. Nikolić, Aleksandra, 1975-. Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka.

Degree: Biološki fakultet, 2014, Univerzitet u Beogradu

Molekularna biologija / Molecular biology

Karcinom pankreasa spada u najinvazivnije i najprogresivnije humane maligne bolesti. Genetske promene karakteristične za ovu bolest su aktivacija onkogena KRAS… (more)

Subjects/Keywords: pancreatic cancer; SMAD4; KRAS

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APA (6th Edition):

Nikolić, Aleksandra, 1. (2014). Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nikolić, Aleksandra, 1975-. “Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka.” 2014. Thesis, Univerzitet u Beogradu. Accessed April 07, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nikolić, Aleksandra, 1975-. “Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka.” 2014. Web. 07 Apr 2020.

Vancouver:

Nikolić, Aleksandra 1. Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka. [Internet] [Thesis]. Univerzitet u Beogradu; 2014. [cited 2020 Apr 07]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nikolić, Aleksandra 1. Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka. [Thesis]. Univerzitet u Beogradu; 2014. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

10. Hua, Alexander Mach. PPAR-alpha: a novel target in pancreatic cancer.

Degree: MS, Medical Sciences, 2015, Boston University

 Background: Current targeted therapies in pancreatic cancer have been ineffective. The tumor stroma, including intra- and peri-tumoral inflammation and fibrosis, is increasingly implicated in pancreatic(more)

Subjects/Keywords: Biology; PPAR; Pancreatic cancer

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APA (6th Edition):

Hua, A. M. (2015). PPAR-alpha: a novel target in pancreatic cancer. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/13985

Chicago Manual of Style (16th Edition):

Hua, Alexander Mach. “PPAR-alpha: a novel target in pancreatic cancer.” 2015. Masters Thesis, Boston University. Accessed April 07, 2020. http://hdl.handle.net/2144/13985.

MLA Handbook (7th Edition):

Hua, Alexander Mach. “PPAR-alpha: a novel target in pancreatic cancer.” 2015. Web. 07 Apr 2020.

Vancouver:

Hua AM. PPAR-alpha: a novel target in pancreatic cancer. [Internet] [Masters thesis]. Boston University; 2015. [cited 2020 Apr 07]. Available from: http://hdl.handle.net/2144/13985.

Council of Science Editors:

Hua AM. PPAR-alpha: a novel target in pancreatic cancer. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/13985


University of Cambridge

11. Mills, Graham David. Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer.

Degree: PhD, 2019, University of Cambridge

Pancreatic ductal adenocarcinoma (PDAC) remains one of the most difficult to treat major cancers, with a 5-year survival of less than 5%, and little to… (more)

Subjects/Keywords: pancreatic cancer; chemotherapy; tumour microenvironment

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APA (6th Edition):

Mills, G. D. (2019). Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/294215

Chicago Manual of Style (16th Edition):

Mills, Graham David. “Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer.” 2019. Doctoral Dissertation, University of Cambridge. Accessed April 07, 2020. https://www.repository.cam.ac.uk/handle/1810/294215.

MLA Handbook (7th Edition):

Mills, Graham David. “Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer.” 2019. Web. 07 Apr 2020.

Vancouver:

Mills GD. Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Apr 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/294215.

Council of Science Editors:

Mills GD. Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/294215


University of Cambridge

12. Mills, Graham David. Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer.

Degree: PhD, 2019, University of Cambridge

Pancreatic ductal adenocarcinoma (PDAC) remains one of the most difficult to treat major cancers, with a 5-year survival of less than 5%, and little to… (more)

Subjects/Keywords: pancreatic cancer; chemotherapy; tumour microenvironment

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APA (6th Edition):

Mills, G. D. (2019). Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/294215 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782882

Chicago Manual of Style (16th Edition):

Mills, Graham David. “Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer.” 2019. Doctoral Dissertation, University of Cambridge. Accessed April 07, 2020. https://www.repository.cam.ac.uk/handle/1810/294215 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782882.

MLA Handbook (7th Edition):

Mills, Graham David. “Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer.” 2019. Web. 07 Apr 2020.

Vancouver:

Mills GD. Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Apr 07]. Available from: https://www.repository.cam.ac.uk/handle/1810/294215 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782882.

Council of Science Editors:

Mills GD. Identification of mechanisms of gemcitabine resistance using a whole genome shRNA screen in a novel in vitro coculture model of pancreatic cancer. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/294215 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782882

13. Marzijarani, Tayebeh Anajafi. Targeted Drug Delivery in Pancreatic Cancer.

Degree: School of Pharmacy, 2017, North Dakota State University

Tayebeh Anajafi Marzijarani, 2017 North Dakota State University Three Minute Thesis (3MT) competition champion, talks about her research on targeted drug delivery for pancreatic cancer.

Subjects/Keywords: Drug delivery systems.; Pancreatic cancer.

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APA (6th Edition):

Marzijarani, T. A. (2017). Targeted Drug Delivery in Pancreatic Cancer. (Thesis). North Dakota State University. Retrieved from http://hdl.handle.net/10365/27554

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marzijarani, Tayebeh Anajafi. “Targeted Drug Delivery in Pancreatic Cancer.” 2017. Thesis, North Dakota State University. Accessed April 07, 2020. http://hdl.handle.net/10365/27554.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marzijarani, Tayebeh Anajafi. “Targeted Drug Delivery in Pancreatic Cancer.” 2017. Web. 07 Apr 2020.

Vancouver:

Marzijarani TA. Targeted Drug Delivery in Pancreatic Cancer. [Internet] [Thesis]. North Dakota State University; 2017. [cited 2020 Apr 07]. Available from: http://hdl.handle.net/10365/27554.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marzijarani TA. Targeted Drug Delivery in Pancreatic Cancer. [Thesis]. North Dakota State University; 2017. Available from: http://hdl.handle.net/10365/27554

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Erdmann, Joris. Classification and Treatment of Pancreatic and Non-Pancreatic Periampullary Cancers.

Degree: 2015, Erasmus University Medical Center

Subjects/Keywords: Pancreatic cancer

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APA (6th Edition):

Erdmann, J. (2015). Classification and Treatment of Pancreatic and Non-Pancreatic Periampullary Cancers. (Doctoral Dissertation). Erasmus University Medical Center. Retrieved from http://hdl.handle.net/1765/78962

Chicago Manual of Style (16th Edition):

Erdmann, Joris. “Classification and Treatment of Pancreatic and Non-Pancreatic Periampullary Cancers.” 2015. Doctoral Dissertation, Erasmus University Medical Center. Accessed April 07, 2020. http://hdl.handle.net/1765/78962.

MLA Handbook (7th Edition):

Erdmann, Joris. “Classification and Treatment of Pancreatic and Non-Pancreatic Periampullary Cancers.” 2015. Web. 07 Apr 2020.

Vancouver:

Erdmann J. Classification and Treatment of Pancreatic and Non-Pancreatic Periampullary Cancers. [Internet] [Doctoral dissertation]. Erasmus University Medical Center; 2015. [cited 2020 Apr 07]. Available from: http://hdl.handle.net/1765/78962.

Council of Science Editors:

Erdmann J. Classification and Treatment of Pancreatic and Non-Pancreatic Periampullary Cancers. [Doctoral Dissertation]. Erasmus University Medical Center; 2015. Available from: http://hdl.handle.net/1765/78962


University of Toronto

15. Matkar, Pratiek. Novel Oncogenic Roles of Neuropilin-1 in Pancreatic Adenocarcinoma and its Relevance as a Therapeutic Target.

Degree: PhD, 2017, University of Toronto

Pancreatic ductal adenocarcinoma (PDAC), the most common type of pancreatic cancer, is a prominent cause of cancer mortality in Canada, with the lowest five-year survival… (more)

Subjects/Keywords: Chemoresistance; Neuropilin; Pancreatic cancer; 0379

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APA (6th Edition):

Matkar, P. (2017). Novel Oncogenic Roles of Neuropilin-1 in Pancreatic Adenocarcinoma and its Relevance as a Therapeutic Target. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/80824

Chicago Manual of Style (16th Edition):

Matkar, Pratiek. “Novel Oncogenic Roles of Neuropilin-1 in Pancreatic Adenocarcinoma and its Relevance as a Therapeutic Target.” 2017. Doctoral Dissertation, University of Toronto. Accessed April 07, 2020. http://hdl.handle.net/1807/80824.

MLA Handbook (7th Edition):

Matkar, Pratiek. “Novel Oncogenic Roles of Neuropilin-1 in Pancreatic Adenocarcinoma and its Relevance as a Therapeutic Target.” 2017. Web. 07 Apr 2020.

Vancouver:

Matkar P. Novel Oncogenic Roles of Neuropilin-1 in Pancreatic Adenocarcinoma and its Relevance as a Therapeutic Target. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2020 Apr 07]. Available from: http://hdl.handle.net/1807/80824.

Council of Science Editors:

Matkar P. Novel Oncogenic Roles of Neuropilin-1 in Pancreatic Adenocarcinoma and its Relevance as a Therapeutic Target. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/80824


Queen Mary, University of London

16. Kadaba, Raghunandan. Desmoplastic stromal cells modulate tumour cell behaviour in pancreatic cancer.

Degree: Thesis (M.D.), 2013, Queen Mary, University of London

Pancreatic ductal adenocarcinoma (PDAC) is characterised by an intense desmoplastic stromal response that can comprise 60 to 80% of tumour volume and has been implicated… (more)

Subjects/Keywords: 616.99; Medicine; Cancer; Pancreatic cancer; Pancreatic ductal adenocarcinoma

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APA (6th Edition):

Kadaba, R. (2013). Desmoplastic stromal cells modulate tumour cell behaviour in pancreatic cancer. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/8825 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667231

Chicago Manual of Style (16th Edition):

Kadaba, Raghunandan. “Desmoplastic stromal cells modulate tumour cell behaviour in pancreatic cancer.” 2013. Doctoral Dissertation, Queen Mary, University of London. Accessed April 07, 2020. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8825 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667231.

MLA Handbook (7th Edition):

Kadaba, Raghunandan. “Desmoplastic stromal cells modulate tumour cell behaviour in pancreatic cancer.” 2013. Web. 07 Apr 2020.

Vancouver:

Kadaba R. Desmoplastic stromal cells modulate tumour cell behaviour in pancreatic cancer. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2013. [cited 2020 Apr 07]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8825 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667231.

Council of Science Editors:

Kadaba R. Desmoplastic stromal cells modulate tumour cell behaviour in pancreatic cancer. [Doctoral Dissertation]. Queen Mary, University of London; 2013. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/8825 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667231


Queen Mary, University of London

17. Li Causi, Eleanor. The role of CX3CR1 in pancreatic cancer.

Degree: PhD, 2018, Queen Mary, University of London

Pancreatic adenocarcinoma (PDAC) is the fourth leading cause of cancer death in Western countries. The PDAC tumour microenvironment (TME) is characterized by a dense stromal… (more)

Subjects/Keywords: Cancer and Inflammation; pancreatic cancer; Pancreatic adenocarcinoma; CX3CR1

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APA (6th Edition):

Li Causi, E. (2018). The role of CX3CR1 in pancreatic cancer. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/36210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766116

Chicago Manual of Style (16th Edition):

Li Causi, Eleanor. “The role of CX3CR1 in pancreatic cancer.” 2018. Doctoral Dissertation, Queen Mary, University of London. Accessed April 07, 2020. http://qmro.qmul.ac.uk/xmlui/handle/123456789/36210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766116.

MLA Handbook (7th Edition):

Li Causi, Eleanor. “The role of CX3CR1 in pancreatic cancer.” 2018. Web. 07 Apr 2020.

Vancouver:

Li Causi E. The role of CX3CR1 in pancreatic cancer. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2018. [cited 2020 Apr 07]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/36210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766116.

Council of Science Editors:

Li Causi E. The role of CX3CR1 in pancreatic cancer. [Doctoral Dissertation]. Queen Mary, University of London; 2018. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/36210 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766116


Texas Medical Center

18. xia, qianghua. THE ROLE OF TAK1 IN PANCREATIC CANCER DEVELOPMENT.

Degree: PhD, 2011, Texas Medical Center

Pancreatic cancer is the fourth leading cause of cancer-related mortality in the United States and the fifth leading cause of cancer-related mortality worldwide. Pancreatic(more)

Subjects/Keywords: pancreatic cancer; TAK1; TAB1; acetylation; Cancer Biology

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APA (6th Edition):

xia, q. (2011). THE ROLE OF TAK1 IN PANCREATIC CANCER DEVELOPMENT. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/201

Chicago Manual of Style (16th Edition):

xia, qianghua. “THE ROLE OF TAK1 IN PANCREATIC CANCER DEVELOPMENT.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed April 07, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/201.

MLA Handbook (7th Edition):

xia, qianghua. “THE ROLE OF TAK1 IN PANCREATIC CANCER DEVELOPMENT.” 2011. Web. 07 Apr 2020.

Vancouver:

xia q. THE ROLE OF TAK1 IN PANCREATIC CANCER DEVELOPMENT. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2020 Apr 07]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/201.

Council of Science Editors:

xia q. THE ROLE OF TAK1 IN PANCREATIC CANCER DEVELOPMENT. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/201


University of Alberta

19. Abualhassan, Nasser. Improving Techniques for Cell Implantation and their Impact on Managing Diseases of the Pancreas.

Degree: MS, Department of Surgery, 2016, University of Alberta

 The use of experimental animals early in the twentieth century has revolutionized medicine and significantly improved our understanding of disease processes. The ability to reproduce… (more)

Subjects/Keywords: Pancreatic cancer; Islet transplantation; Cancer xenograft

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APA (6th Edition):

Abualhassan, N. (2016). Improving Techniques for Cell Implantation and their Impact on Managing Diseases of the Pancreas. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/c7s75dc68b

Chicago Manual of Style (16th Edition):

Abualhassan, Nasser. “Improving Techniques for Cell Implantation and their Impact on Managing Diseases of the Pancreas.” 2016. Masters Thesis, University of Alberta. Accessed April 07, 2020. https://era.library.ualberta.ca/files/c7s75dc68b.

MLA Handbook (7th Edition):

Abualhassan, Nasser. “Improving Techniques for Cell Implantation and their Impact on Managing Diseases of the Pancreas.” 2016. Web. 07 Apr 2020.

Vancouver:

Abualhassan N. Improving Techniques for Cell Implantation and their Impact on Managing Diseases of the Pancreas. [Internet] [Masters thesis]. University of Alberta; 2016. [cited 2020 Apr 07]. Available from: https://era.library.ualberta.ca/files/c7s75dc68b.

Council of Science Editors:

Abualhassan N. Improving Techniques for Cell Implantation and their Impact on Managing Diseases of the Pancreas. [Masters Thesis]. University of Alberta; 2016. Available from: https://era.library.ualberta.ca/files/c7s75dc68b

20. Coe, Peter. Validation and early qualification of pancreatic fat deposition as an imaging biomarker of pancreatic cancer risk.

Degree: 2016, University of Manchester

 Introduction Pancreatic cancer is the 10th most common cause of cancer in the United Kingdom (UK) yet the 5th most common cause of cancer related… (more)

Subjects/Keywords: Intra-organ fat; Obesity; Cancer; Pancreatic cancer

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APA (6th Edition):

Coe, P. (2016). Validation and early qualification of pancreatic fat deposition as an imaging biomarker of pancreatic cancer risk. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:303410

Chicago Manual of Style (16th Edition):

Coe, Peter. “Validation and early qualification of pancreatic fat deposition as an imaging biomarker of pancreatic cancer risk.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 07, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:303410.

MLA Handbook (7th Edition):

Coe, Peter. “Validation and early qualification of pancreatic fat deposition as an imaging biomarker of pancreatic cancer risk.” 2016. Web. 07 Apr 2020.

Vancouver:

Coe P. Validation and early qualification of pancreatic fat deposition as an imaging biomarker of pancreatic cancer risk. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Apr 07]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:303410.

Council of Science Editors:

Coe P. Validation and early qualification of pancreatic fat deposition as an imaging biomarker of pancreatic cancer risk. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:303410


University of New South Wales

21. Susanto, Johana. Investigating the use of retinoids and epigenetic modification agents as new therapeutic strategies for the treatment of pancreatic cancer.

Degree: Clinical School - St Vincent's Hospital, 2017, University of New South Wales

 New therapeutic strategies are needed to improve the outcome and the quality of life for pancreatic cancer (PC) patients. This study aimed to investigate new… (more)

Subjects/Keywords: Epigenetics; Pancreatic cancer; Retinoids; Cancer treatment

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APA (6th Edition):

Susanto, J. (2017). Investigating the use of retinoids and epigenetic modification agents as new therapeutic strategies for the treatment of pancreatic cancer. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/58819 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:47664/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Susanto, Johana. “Investigating the use of retinoids and epigenetic modification agents as new therapeutic strategies for the treatment of pancreatic cancer.” 2017. Doctoral Dissertation, University of New South Wales. Accessed April 07, 2020. http://handle.unsw.edu.au/1959.4/58819 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:47664/SOURCE02?view=true.

MLA Handbook (7th Edition):

Susanto, Johana. “Investigating the use of retinoids and epigenetic modification agents as new therapeutic strategies for the treatment of pancreatic cancer.” 2017. Web. 07 Apr 2020.

Vancouver:

Susanto J. Investigating the use of retinoids and epigenetic modification agents as new therapeutic strategies for the treatment of pancreatic cancer. [Internet] [Doctoral dissertation]. University of New South Wales; 2017. [cited 2020 Apr 07]. Available from: http://handle.unsw.edu.au/1959.4/58819 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:47664/SOURCE02?view=true.

Council of Science Editors:

Susanto J. Investigating the use of retinoids and epigenetic modification agents as new therapeutic strategies for the treatment of pancreatic cancer. [Doctoral Dissertation]. University of New South Wales; 2017. Available from: http://handle.unsw.edu.au/1959.4/58819 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:47664/SOURCE02?view=true


University of Houston

22. -8379-9505. Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers.

Degree: PhD, Cell and Molecular Biology, 2015, University of Houston

 Alcohol Regulates Genes that are Associated with Response to Endocrine Therapy and Attenuates the Actions of Tamoxifen in Breast Cancer Cells. Pancreatic ductal adenocarcinoma (PDAC)… (more)

Subjects/Keywords: LXR; Pancreatic cancer; ER; Breast cancer; Alcohol

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APA (6th Edition):

-8379-9505. (2015). Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/5367

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-8379-9505. “Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers.” 2015. Doctoral Dissertation, University of Houston. Accessed April 07, 2020. http://hdl.handle.net/10657/5367.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-8379-9505. “Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers.” 2015. Web. 07 Apr 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-8379-9505. Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2020 Apr 07]. Available from: http://hdl.handle.net/10657/5367.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-8379-9505. Modulation of Nuclear Receptor Functions in Breast and Pancreatic Cancers. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/5367

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Southern California

23. Aletomeh, Yasaman. Pancreatic cancer: a review on biology, genetics and therapeutics.

Degree: MS, Pharmaceutical Sciences, 2014, University of Southern California

Pancreatic cancer is the fourth leading cause of cancer death in the United States and remains a major unsolved health problem in the 21st century.… (more)

Subjects/Keywords: pancreatic cancer; cancer biology; genetics; therapeutics

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APA (6th Edition):

Aletomeh, Y. (2014). Pancreatic cancer: a review on biology, genetics and therapeutics. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/516607/rec/4906

Chicago Manual of Style (16th Edition):

Aletomeh, Yasaman. “Pancreatic cancer: a review on biology, genetics and therapeutics.” 2014. Masters Thesis, University of Southern California. Accessed April 07, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/516607/rec/4906.

MLA Handbook (7th Edition):

Aletomeh, Yasaman. “Pancreatic cancer: a review on biology, genetics and therapeutics.” 2014. Web. 07 Apr 2020.

Vancouver:

Aletomeh Y. Pancreatic cancer: a review on biology, genetics and therapeutics. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Apr 07]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/516607/rec/4906.

Council of Science Editors:

Aletomeh Y. Pancreatic cancer: a review on biology, genetics and therapeutics. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/516607/rec/4906


Penn State University

24. Mirkin, Katelin. Minimally Invasive Surgical Approaches Offer Earlier Time to Adjuvant Chemotherapy but Not Improved Survival in Resected Pancreatic Cancer.

Degree: 2017, Penn State University

 Background: Pancreatic surgery encompasses complex operations with significant potential morbidity. Greater experience in minimally invasive surgery (MIS) has allowed resections to be performed laparoscopically and… (more)

Subjects/Keywords: MIS; Minimally Invasive Surgery; Adjuvant Chemotherapy; Pancreatic Cancer; Pancreatic Adenocarcinoma

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APA (6th Edition):

Mirkin, K. (2017). Minimally Invasive Surgical Approaches Offer Earlier Time to Adjuvant Chemotherapy but Not Improved Survival in Resected Pancreatic Cancer. (Thesis). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/13789kam79

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mirkin, Katelin. “Minimally Invasive Surgical Approaches Offer Earlier Time to Adjuvant Chemotherapy but Not Improved Survival in Resected Pancreatic Cancer.” 2017. Thesis, Penn State University. Accessed April 07, 2020. https://etda.libraries.psu.edu/catalog/13789kam79.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mirkin, Katelin. “Minimally Invasive Surgical Approaches Offer Earlier Time to Adjuvant Chemotherapy but Not Improved Survival in Resected Pancreatic Cancer.” 2017. Web. 07 Apr 2020.

Vancouver:

Mirkin K. Minimally Invasive Surgical Approaches Offer Earlier Time to Adjuvant Chemotherapy but Not Improved Survival in Resected Pancreatic Cancer. [Internet] [Thesis]. Penn State University; 2017. [cited 2020 Apr 07]. Available from: https://etda.libraries.psu.edu/catalog/13789kam79.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mirkin K. Minimally Invasive Surgical Approaches Offer Earlier Time to Adjuvant Chemotherapy but Not Improved Survival in Resected Pancreatic Cancer. [Thesis]. Penn State University; 2017. Available from: https://etda.libraries.psu.edu/catalog/13789kam79

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

25. Chan, Alison Hui-Wai. Validation of Candidate Biomarkers for the Development of a Multi-Parametric Panel for Early Detection of Pancreatic Ductal Adenocarcinoma (PDAC).

Degree: 2013, University of Toronto

High-throughput mass spectrometry has discovered a plethora of candidates in the biomarker field, however, subsequent verification and validation studies are urgently needed to assess the… (more)

Subjects/Keywords: pancreatic cancer; early diagnosis; pancreatic ductal adenocarcinoma; biomarker; validation; 0992; 0571

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APA (6th Edition):

Chan, A. H. (2013). Validation of Candidate Biomarkers for the Development of a Multi-Parametric Panel for Early Detection of Pancreatic Ductal Adenocarcinoma (PDAC). (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42707

Chicago Manual of Style (16th Edition):

Chan, Alison Hui-Wai. “Validation of Candidate Biomarkers for the Development of a Multi-Parametric Panel for Early Detection of Pancreatic Ductal Adenocarcinoma (PDAC).” 2013. Masters Thesis, University of Toronto. Accessed April 07, 2020. http://hdl.handle.net/1807/42707.

MLA Handbook (7th Edition):

Chan, Alison Hui-Wai. “Validation of Candidate Biomarkers for the Development of a Multi-Parametric Panel for Early Detection of Pancreatic Ductal Adenocarcinoma (PDAC).” 2013. Web. 07 Apr 2020.

Vancouver:

Chan AH. Validation of Candidate Biomarkers for the Development of a Multi-Parametric Panel for Early Detection of Pancreatic Ductal Adenocarcinoma (PDAC). [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2020 Apr 07]. Available from: http://hdl.handle.net/1807/42707.

Council of Science Editors:

Chan AH. Validation of Candidate Biomarkers for the Development of a Multi-Parametric Panel for Early Detection of Pancreatic Ductal Adenocarcinoma (PDAC). [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42707


Louisiana State University

26. Karki, Namrata. In Vitro Investigation on Therapeutic Potential of Juglone, a Naphthoquinone from Walnuts against Pancreatic Cancer.

Degree: PhD, Life Sciences, 2016, Louisiana State University

 Juglone, a naphthoquinone found in Juglandaceae family, which includes black walnut, European walnut, and butter nut possess various biological activities. The anti-cancer properties of juglone… (more)

Subjects/Keywords: pancreatic cancer; quinones; juglone; naphthoquinone; black walnut

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APA (6th Edition):

Karki, N. (2016). In Vitro Investigation on Therapeutic Potential of Juglone, a Naphthoquinone from Walnuts against Pancreatic Cancer. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-03252016-224603 ; https://digitalcommons.lsu.edu/gradschool_dissertations/320

Chicago Manual of Style (16th Edition):

Karki, Namrata. “In Vitro Investigation on Therapeutic Potential of Juglone, a Naphthoquinone from Walnuts against Pancreatic Cancer.” 2016. Doctoral Dissertation, Louisiana State University. Accessed April 07, 2020. etd-03252016-224603 ; https://digitalcommons.lsu.edu/gradschool_dissertations/320.

MLA Handbook (7th Edition):

Karki, Namrata. “In Vitro Investigation on Therapeutic Potential of Juglone, a Naphthoquinone from Walnuts against Pancreatic Cancer.” 2016. Web. 07 Apr 2020.

Vancouver:

Karki N. In Vitro Investigation on Therapeutic Potential of Juglone, a Naphthoquinone from Walnuts against Pancreatic Cancer. [Internet] [Doctoral dissertation]. Louisiana State University; 2016. [cited 2020 Apr 07]. Available from: etd-03252016-224603 ; https://digitalcommons.lsu.edu/gradschool_dissertations/320.

Council of Science Editors:

Karki N. In Vitro Investigation on Therapeutic Potential of Juglone, a Naphthoquinone from Walnuts against Pancreatic Cancer. [Doctoral Dissertation]. Louisiana State University; 2016. Available from: etd-03252016-224603 ; https://digitalcommons.lsu.edu/gradschool_dissertations/320

27. 趙, 茗. Significance of combination therapy of zoledronic acid and gemcitabine on pancreatic cancer : 膵癌におけるゾレドロン酸及びゲムシタビン併用療法の意義.

Degree: 博士(医学), 2013, Kyushu University / 九州大学

 In this study, we examined the cytotoxic effects of combination therapy with zoledronic acid (ZOL) and gemcitabine (GEM) on pancreatic cancer cells in vitro and… (more)

Subjects/Keywords: combination therapy; zoledronic acid; gemcitabine; pancreatic cancer

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APA (6th Edition):

趙, . (2013). Significance of combination therapy of zoledronic acid and gemcitabine on pancreatic cancer : 膵癌におけるゾレドロン酸及びゲムシタビン併用療法の意義. (Thesis). Kyushu University / 九州大学. Retrieved from http://hdl.handle.net/2324/26344 ; http://dx.doi.org/10.15017/26344

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

趙, 茗. “Significance of combination therapy of zoledronic acid and gemcitabine on pancreatic cancer : 膵癌におけるゾレドロン酸及びゲムシタビン併用療法の意義.” 2013. Thesis, Kyushu University / 九州大学. Accessed April 07, 2020. http://hdl.handle.net/2324/26344 ; http://dx.doi.org/10.15017/26344.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

趙, 茗. “Significance of combination therapy of zoledronic acid and gemcitabine on pancreatic cancer : 膵癌におけるゾレドロン酸及びゲムシタビン併用療法の意義.” 2013. Web. 07 Apr 2020.

Vancouver:

趙 . Significance of combination therapy of zoledronic acid and gemcitabine on pancreatic cancer : 膵癌におけるゾレドロン酸及びゲムシタビン併用療法の意義. [Internet] [Thesis]. Kyushu University / 九州大学; 2013. [cited 2020 Apr 07]. Available from: http://hdl.handle.net/2324/26344 ; http://dx.doi.org/10.15017/26344.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

趙 . Significance of combination therapy of zoledronic acid and gemcitabine on pancreatic cancer : 膵癌におけるゾレドロン酸及びゲムシタビン併用療法の意義. [Thesis]. Kyushu University / 九州大学; 2013. Available from: http://hdl.handle.net/2324/26344 ; http://dx.doi.org/10.15017/26344

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

28. Howell, Justin Lee. Biomodeling of pancreatic tumor mass.

Degree: M. Eng., 2013, University of Louisville

Pancreatic adenocarcinoma is the fourth leading cause of cancer death in the United States. It is identified by its rapid, invasive progression with a profound… (more)

Subjects/Keywords: Pancreatic cancer; Mathematical model; Tumor modeling

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Howell, J. L. (2013). Biomodeling of pancreatic tumor mass. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/643 ; https://ir.library.louisville.edu/etd/643

Chicago Manual of Style (16th Edition):

Howell, Justin Lee. “Biomodeling of pancreatic tumor mass.” 2013. Masters Thesis, University of Louisville. Accessed April 07, 2020. 10.18297/etd/643 ; https://ir.library.louisville.edu/etd/643.

MLA Handbook (7th Edition):

Howell, Justin Lee. “Biomodeling of pancreatic tumor mass.” 2013. Web. 07 Apr 2020.

Vancouver:

Howell JL. Biomodeling of pancreatic tumor mass. [Internet] [Masters thesis]. University of Louisville; 2013. [cited 2020 Apr 07]. Available from: 10.18297/etd/643 ; https://ir.library.louisville.edu/etd/643.

Council of Science Editors:

Howell JL. Biomodeling of pancreatic tumor mass. [Masters Thesis]. University of Louisville; 2013. Available from: 10.18297/etd/643 ; https://ir.library.louisville.edu/etd/643


University of Rochester

29. Balakrishnan, Vijaya. Regulation of Autophagy and Lysosome Integrity by Plac8 in Cancer.

Degree: PhD, 2015, University of Rochester

 The upregulation of stress control pathways such as autophagy is an emerging hallmark of cancer. Autophagy is a cellular recycling process whereby damaged organelles are… (more)

Subjects/Keywords: Autophagy; Lysosome; Cancer; Plac8; Pancreatic; Onzin; Exosomes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Balakrishnan, V. (2015). Regulation of Autophagy and Lysosome Integrity by Plac8 in Cancer. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/29352

Chicago Manual of Style (16th Edition):

Balakrishnan, Vijaya. “Regulation of Autophagy and Lysosome Integrity by Plac8 in Cancer.” 2015. Doctoral Dissertation, University of Rochester. Accessed April 07, 2020. http://hdl.handle.net/1802/29352.

MLA Handbook (7th Edition):

Balakrishnan, Vijaya. “Regulation of Autophagy and Lysosome Integrity by Plac8 in Cancer.” 2015. Web. 07 Apr 2020.

Vancouver:

Balakrishnan V. Regulation of Autophagy and Lysosome Integrity by Plac8 in Cancer. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2020 Apr 07]. Available from: http://hdl.handle.net/1802/29352.

Council of Science Editors:

Balakrishnan V. Regulation of Autophagy and Lysosome Integrity by Plac8 in Cancer. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/29352


NSYSU

30. Hsu, Wen-Ti. Study of pancreatic cancer antigen detection using AlGaN/GaN high electron mobility transistor biosensor with different aluminum content.

Degree: Master, Physics, 2013, NSYSU

 Compared to classical lab instrumentation, the biosensors are being increasingly exploited as promising diagnostic devices for their advantages, such as label-free, ultrasensitive, highly selective, high… (more)

Subjects/Keywords: Nitride; biosensor; high electron mobility; pancreatic cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hsu, W. (2013). Study of pancreatic cancer antigen detection using AlGaN/GaN high electron mobility transistor biosensor with different aluminum content. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0711113-222628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hsu, Wen-Ti. “Study of pancreatic cancer antigen detection using AlGaN/GaN high electron mobility transistor biosensor with different aluminum content.” 2013. Thesis, NSYSU. Accessed April 07, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0711113-222628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hsu, Wen-Ti. “Study of pancreatic cancer antigen detection using AlGaN/GaN high electron mobility transistor biosensor with different aluminum content.” 2013. Web. 07 Apr 2020.

Vancouver:

Hsu W. Study of pancreatic cancer antigen detection using AlGaN/GaN high electron mobility transistor biosensor with different aluminum content. [Internet] [Thesis]. NSYSU; 2013. [cited 2020 Apr 07]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0711113-222628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hsu W. Study of pancreatic cancer antigen detection using AlGaN/GaN high electron mobility transistor biosensor with different aluminum content. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0711113-222628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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