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You searched for subject:(Pancreatic beta cells). Showing records 1 – 30 of 101 total matches.

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University of Toronto

1. Desai, Tejas. Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction.

Degree: 2013, University of Toronto

Within the β-cell, there is evidence that sirtuin 1 (SIRT1), a key regulator of nutrient metabolism, plays a beneficial role on insulin secretion. Excess circulating… (more)

Subjects/Keywords: Lipotoxicity; Pancreatic Beta Cells; Sirtuins; 0719

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APA (6th Edition):

Desai, T. (2013). Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/69993

Chicago Manual of Style (16th Edition):

Desai, Tejas. “Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction.” 2013. Masters Thesis, University of Toronto. Accessed January 19, 2021. http://hdl.handle.net/1807/69993.

MLA Handbook (7th Edition):

Desai, Tejas. “Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction.” 2013. Web. 19 Jan 2021.

Vancouver:

Desai T. Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1807/69993.

Council of Science Editors:

Desai T. Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/69993


Columbia University

2. Gonzalez, Bryan Jose. HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function.

Degree: 2019, Columbia University

 Mutations in HNF1A cause Maturity Onset Diabetes of the Young type 3, the second most frequent form of diabetes caused by single gene mutation. We… (more)

Subjects/Keywords: Cytology; Insulin; Pancreatic beta cells; Biology; Genes

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APA (6th Edition):

Gonzalez, B. J. (2019). HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/d8-d6v3-ca52

Chicago Manual of Style (16th Edition):

Gonzalez, Bryan Jose. “HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function.” 2019. Doctoral Dissertation, Columbia University. Accessed January 19, 2021. https://doi.org/10.7916/d8-d6v3-ca52.

MLA Handbook (7th Edition):

Gonzalez, Bryan Jose. “HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function.” 2019. Web. 19 Jan 2021.

Vancouver:

Gonzalez BJ. HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function. [Internet] [Doctoral dissertation]. Columbia University; 2019. [cited 2021 Jan 19]. Available from: https://doi.org/10.7916/d8-d6v3-ca52.

Council of Science Editors:

Gonzalez BJ. HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function. [Doctoral Dissertation]. Columbia University; 2019. Available from: https://doi.org/10.7916/d8-d6v3-ca52


Columbia University

3. Fan, Jason Chen. Markers and Mechanisms of β-cell Dedifferentiation.

Degree: 2018, Columbia University

 Human and murine diabetes is characterized by pancreatic β-cell dedifferentiation, a process in which β-cells lose expression of markers of maturity and gain those of… (more)

Subjects/Keywords: Medicine; Biology; Cytology; Pancreatic beta cells

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APA (6th Edition):

Fan, J. C. (2018). Markers and Mechanisms of β-cell Dedifferentiation. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8RB8GGD

Chicago Manual of Style (16th Edition):

Fan, Jason Chen. “Markers and Mechanisms of β-cell Dedifferentiation.” 2018. Doctoral Dissertation, Columbia University. Accessed January 19, 2021. https://doi.org/10.7916/D8RB8GGD.

MLA Handbook (7th Edition):

Fan, Jason Chen. “Markers and Mechanisms of β-cell Dedifferentiation.” 2018. Web. 19 Jan 2021.

Vancouver:

Fan JC. Markers and Mechanisms of β-cell Dedifferentiation. [Internet] [Doctoral dissertation]. Columbia University; 2018. [cited 2021 Jan 19]. Available from: https://doi.org/10.7916/D8RB8GGD.

Council of Science Editors:

Fan JC. Markers and Mechanisms of β-cell Dedifferentiation. [Doctoral Dissertation]. Columbia University; 2018. Available from: https://doi.org/10.7916/D8RB8GGD


University of Aberdeen

4. Robertson, Lindsay. Characterisation of the MAL2 proteins and their interaction with TPD52.

Degree: School of Biological Sciences., 2006, University of Aberdeen

Subjects/Keywords: Pancreatic beta cells

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APA (6th Edition):

Robertson, L. (2006). Characterisation of the MAL2 proteins and their interaction with TPD52. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=185669 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=185669&custom_att_2=simple_viewer

Chicago Manual of Style (16th Edition):

Robertson, Lindsay. “Characterisation of the MAL2 proteins and their interaction with TPD52.” 2006. Doctoral Dissertation, University of Aberdeen. Accessed January 19, 2021. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=185669 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=185669&custom_att_2=simple_viewer.

MLA Handbook (7th Edition):

Robertson, Lindsay. “Characterisation of the MAL2 proteins and their interaction with TPD52.” 2006. Web. 19 Jan 2021.

Vancouver:

Robertson L. Characterisation of the MAL2 proteins and their interaction with TPD52. [Internet] [Doctoral dissertation]. University of Aberdeen; 2006. [cited 2021 Jan 19]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=185669 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=185669&custom_att_2=simple_viewer.

Council of Science Editors:

Robertson L. Characterisation of the MAL2 proteins and their interaction with TPD52. [Doctoral Dissertation]. University of Aberdeen; 2006. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=185669 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=185669&custom_att_2=simple_viewer


Louisiana State University

5. Earpina, Srikanth. Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells.

Degree: MS, Life Sciences, 2013, Louisiana State University

 Diabetes is an inflammatory disease associated with hyperglycemia. Chronic exposure of pancreatic â-cells to glucolipotoxicity stimulates a low-grade inflammation associated with the release of pro-inflammatory… (more)

Subjects/Keywords: glucose; palmitic acid; pancreatic beta cells; inflammation

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APA (6th Edition):

Earpina, S. (2013). Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells. (Masters Thesis). Louisiana State University. Retrieved from etd-11102013-230409 ; https://digitalcommons.lsu.edu/gradschool_theses/1770

Chicago Manual of Style (16th Edition):

Earpina, Srikanth. “Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells.” 2013. Masters Thesis, Louisiana State University. Accessed January 19, 2021. etd-11102013-230409 ; https://digitalcommons.lsu.edu/gradschool_theses/1770.

MLA Handbook (7th Edition):

Earpina, Srikanth. “Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells.” 2013. Web. 19 Jan 2021.

Vancouver:

Earpina S. Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells. [Internet] [Masters thesis]. Louisiana State University; 2013. [cited 2021 Jan 19]. Available from: etd-11102013-230409 ; https://digitalcommons.lsu.edu/gradschool_theses/1770.

Council of Science Editors:

Earpina S. Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells. [Masters Thesis]. Louisiana State University; 2013. Available from: etd-11102013-230409 ; https://digitalcommons.lsu.edu/gradschool_theses/1770


University of New South Wales

6. Pearson, Gemma. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.

Degree: Garvan Institute of Medical Research, 2014, University of New South Wales

 Glucose-stimulated insulin secretion (GSIS) from pancreatic β-cells is vital for whole-bodyglucose homeostasis, and loss of β-cell function can result in type 2 diabetes(T2D), a major… (more)

Subjects/Keywords: Lipids; Pancreatic beta cells; Insulin secretion; Lysosomes

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APA (6th Edition):

Pearson, G. (2014). THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Pearson, Gemma. “THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.” 2014. Doctoral Dissertation, University of New South Wales. Accessed January 19, 2021. http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true.

MLA Handbook (7th Edition):

Pearson, Gemma. “THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.” 2014. Web. 19 Jan 2021.

Vancouver:

Pearson G. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. [Internet] [Doctoral dissertation]. University of New South Wales; 2014. [cited 2021 Jan 19]. Available from: http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true.

Council of Science Editors:

Pearson G. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. [Doctoral Dissertation]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

7. Gargani, Sofia. Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας.

Degree: 2013, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

Introduction: Under normal healthy conditions, organisms maintaina dynamic endocrinecell mass throughout life. Pancreatic beta cell massare able to maintain plasma glucose levels increasing insulin secretion… (more)

Subjects/Keywords: Μεταμόσχευση; Ανθρώπινα παγκρεατικά νησίδια; β παγκρεατικά κύτταρα; Παχυσαρκία; Transplantation; Human pancreatic islets; Beta pancreatic cells; Obesity

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APA (6th Edition):

Gargani, S. (2013). Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/38796

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gargani, Sofia. “Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας.” 2013. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed January 19, 2021. http://hdl.handle.net/10442/hedi/38796.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gargani, Sofia. “Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας.” 2013. Web. 19 Jan 2021.

Vancouver:

Gargani S. Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10442/hedi/38796.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gargani S. Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2013. Available from: http://hdl.handle.net/10442/hedi/38796

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Aberdeen

8. Uroić, Daniela Sonja. Differentiation of embryonic stem cells towards pancreatic β-like cells.

Degree: PhD, 2011, University of Aberdeen

 Embryonic stem (ES) cells were used as a model system to understand the signalling events in pancreas development. ES cells were differentiated through known precursor… (more)

Subjects/Keywords: 611; Pancreatic beta cells; Embryonic stem cells; Diabetes

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APA (6th Edition):

Uroić, D. S. (2011). Differentiation of embryonic stem cells towards pancreatic β-like cells. (Doctoral Dissertation). University of Aberdeen. Retrieved from https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152277610005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.542660

Chicago Manual of Style (16th Edition):

Uroić, Daniela Sonja. “Differentiation of embryonic stem cells towards pancreatic β-like cells.” 2011. Doctoral Dissertation, University of Aberdeen. Accessed January 19, 2021. https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152277610005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.542660.

MLA Handbook (7th Edition):

Uroić, Daniela Sonja. “Differentiation of embryonic stem cells towards pancreatic β-like cells.” 2011. Web. 19 Jan 2021.

Vancouver:

Uroić DS. Differentiation of embryonic stem cells towards pancreatic β-like cells. [Internet] [Doctoral dissertation]. University of Aberdeen; 2011. [cited 2021 Jan 19]. Available from: https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152277610005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.542660.

Council of Science Editors:

Uroić DS. Differentiation of embryonic stem cells towards pancreatic β-like cells. [Doctoral Dissertation]. University of Aberdeen; 2011. Available from: https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152277610005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.542660


University of Southern California

9. Bayan, Jennifer-Ann. Pten regulates beta-cell regeneration intrinsically and independently of development.

Degree: PhD, Molecular Pharmacology and Toxicology, 2012, University of Southern California

 The pancreatic β-cells are responsible for producing insulin and maintaining glucose homeostasis. Loss of β-cells or their inability to compensate for insulin resistance is the… (more)

Subjects/Keywords: beta-cells; regeneration; diabetes; Pten; pancreatic stellate cells; extracellular matrix

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APA (6th Edition):

Bayan, J. (2012). Pten regulates beta-cell regeneration intrinsically and independently of development. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/84203/rec/5324

Chicago Manual of Style (16th Edition):

Bayan, Jennifer-Ann. “Pten regulates beta-cell regeneration intrinsically and independently of development.” 2012. Doctoral Dissertation, University of Southern California. Accessed January 19, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/84203/rec/5324.

MLA Handbook (7th Edition):

Bayan, Jennifer-Ann. “Pten regulates beta-cell regeneration intrinsically and independently of development.” 2012. Web. 19 Jan 2021.

Vancouver:

Bayan J. Pten regulates beta-cell regeneration intrinsically and independently of development. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2021 Jan 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/84203/rec/5324.

Council of Science Editors:

Bayan J. Pten regulates beta-cell regeneration intrinsically and independently of development. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/84203/rec/5324


University of Alberta

10. Yang, Kaiyuan. Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats.

Degree: PhD, Department of Agricultural, Food, and Nutritional Science, 2015, University of Alberta

 Proanthocyanidins (PAC) belong to a highly consumed class of flavonoids and their consumption has been linked to beneficial effects on glycemic control in type 2… (more)

Subjects/Keywords: Proanthocyanidins; Bioavailability; CaMKII; Hepatic glucose production; Glucose homeostasis; Pancreatic β-cells

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APA (6th Edition):

Yang, K. (2015). Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/n870zt826

Chicago Manual of Style (16th Edition):

Yang, Kaiyuan. “Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats.” 2015. Doctoral Dissertation, University of Alberta. Accessed January 19, 2021. https://era.library.ualberta.ca/files/n870zt826.

MLA Handbook (7th Edition):

Yang, Kaiyuan. “Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats.” 2015. Web. 19 Jan 2021.

Vancouver:

Yang K. Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats. [Internet] [Doctoral dissertation]. University of Alberta; 2015. [cited 2021 Jan 19]. Available from: https://era.library.ualberta.ca/files/n870zt826.

Council of Science Editors:

Yang K. Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats. [Doctoral Dissertation]. University of Alberta; 2015. Available from: https://era.library.ualberta.ca/files/n870zt826

11. 山岡, 真美. PI3K regulates endocytosis after insulin secretion by mediating signaling crosstalk between Arf6 and Rab27a.

Degree: 博士(医学), 2017, Oita University / 大分大学

 In secretory cells, endocytosis is coupled to exocytosis to enable proper secretion. Although endocytosis is crucial to maintain cellular homeostasis before and after secretion, knowledge… (more)

Subjects/Keywords: Endocytosis; Small GTPase; Insulin; Membrane trafficking; Diabetes; Pancreatic β-cells

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APA (6th Edition):

山岡, . (2017). PI3K regulates endocytosis after insulin secretion by mediating signaling crosstalk between Arf6 and Rab27a. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山岡, 真美. “PI3K regulates endocytosis after insulin secretion by mediating signaling crosstalk between Arf6 and Rab27a.” 2017. Thesis, Oita University / 大分大学. Accessed January 19, 2021. http://hdl.handle.net/10559/15698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山岡, 真美. “PI3K regulates endocytosis after insulin secretion by mediating signaling crosstalk between Arf6 and Rab27a.” 2017. Web. 19 Jan 2021.

Vancouver:

山岡 . PI3K regulates endocytosis after insulin secretion by mediating signaling crosstalk between Arf6 and Rab27a. [Internet] [Thesis]. Oita University / 大分大学; 2017. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10559/15698.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山岡 . PI3K regulates endocytosis after insulin secretion by mediating signaling crosstalk between Arf6 and Rab27a. [Thesis]. Oita University / 大分大学; 2017. Available from: http://hdl.handle.net/10559/15698

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

12. Saunders, Diane Caitlin. Towards Pancreatic β-Cell Regeneration: Modulating Islet Microenvironment and Identifying Markers of β-Cell Maturation.

Degree: PhD, Molecular Physiology and Biophysics, 2018, Vanderbilt University

 Regeneration of endogenous β-cells is a promising therapy to treat diabetes, but there are considerable gaps in our understanding of the microenvironmental signals necessary to… (more)

Subjects/Keywords: β-cell regeneration; pancreatic islet; endothelial cells; microenvironment; macrophage recruitment

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APA (6th Edition):

Saunders, D. C. (2018). Towards Pancreatic β-Cell Regeneration: Modulating Islet Microenvironment and Identifying Markers of β-Cell Maturation. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11179

Chicago Manual of Style (16th Edition):

Saunders, Diane Caitlin. “Towards Pancreatic β-Cell Regeneration: Modulating Islet Microenvironment and Identifying Markers of β-Cell Maturation.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed January 19, 2021. http://hdl.handle.net/1803/11179.

MLA Handbook (7th Edition):

Saunders, Diane Caitlin. “Towards Pancreatic β-Cell Regeneration: Modulating Islet Microenvironment and Identifying Markers of β-Cell Maturation.” 2018. Web. 19 Jan 2021.

Vancouver:

Saunders DC. Towards Pancreatic β-Cell Regeneration: Modulating Islet Microenvironment and Identifying Markers of β-Cell Maturation. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1803/11179.

Council of Science Editors:

Saunders DC. Towards Pancreatic β-Cell Regeneration: Modulating Islet Microenvironment and Identifying Markers of β-Cell Maturation. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/11179


University of Hong Kong

13. 曾少慧. Involvement of Pdzd2 in the regulation of pancreatic beta-cell functions.

Degree: 2007, University of Hong Kong

Subjects/Keywords: Proteins.; Pancreatic beta cells.

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APA (6th Edition):

曾少慧.. (2007). Involvement of Pdzd2 in the regulation of pancreatic beta-cell functions. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/54494

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

曾少慧.. “Involvement of Pdzd2 in the regulation of pancreatic beta-cell functions.” 2007. Thesis, University of Hong Kong. Accessed January 19, 2021. http://hdl.handle.net/10722/54494.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

曾少慧.. “Involvement of Pdzd2 in the regulation of pancreatic beta-cell functions.” 2007. Web. 19 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

曾少慧.. Involvement of Pdzd2 in the regulation of pancreatic beta-cell functions. [Internet] [Thesis]. University of Hong Kong; 2007. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10722/54494.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

曾少慧.. Involvement of Pdzd2 in the regulation of pancreatic beta-cell functions. [Thesis]. University of Hong Kong; 2007. Available from: http://hdl.handle.net/10722/54494

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

14. Costa, Ana. Regulação da actividade da Na,K-ATPase por glucose em célula beta-pancreática na ausência e na presença de um estado de intolerância a esta.

Degree: 2012, Universidade de Évora

 A Na,K-ATPase é responsável pela manutenção dos gradientes transmembranares de iões K+ e Na+. Porém, o papel fisiológico da Na,K-ATPase em célula β-pancreática não está… (more)

Subjects/Keywords: Na,K-ATPase; pancreatic beta-cells; glucose intolerance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Costa, A. (2012). Regulação da actividade da Na,K-ATPase por glucose em célula beta-pancreática na ausência e na presença de um estado de intolerância a esta. (Thesis). Universidade de Évora. Retrieved from https://www.rcaap.pt/detail.jsp?id=oai:dspace.uevora.pt:10174/5771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Costa, Ana. “Regulação da actividade da Na,K-ATPase por glucose em célula beta-pancreática na ausência e na presença de um estado de intolerância a esta.” 2012. Thesis, Universidade de Évora. Accessed January 19, 2021. https://www.rcaap.pt/detail.jsp?id=oai:dspace.uevora.pt:10174/5771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Costa, Ana. “Regulação da actividade da Na,K-ATPase por glucose em célula beta-pancreática na ausência e na presença de um estado de intolerância a esta.” 2012. Web. 19 Jan 2021.

Vancouver:

Costa A. Regulação da actividade da Na,K-ATPase por glucose em célula beta-pancreática na ausência e na presença de um estado de intolerância a esta. [Internet] [Thesis]. Universidade de Évora; 2012. [cited 2021 Jan 19]. Available from: https://www.rcaap.pt/detail.jsp?id=oai:dspace.uevora.pt:10174/5771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Costa A. Regulação da actividade da Na,K-ATPase por glucose em célula beta-pancreática na ausência e na presença de um estado de intolerância a esta. [Thesis]. Universidade de Évora; 2012. Available from: https://www.rcaap.pt/detail.jsp?id=oai:dspace.uevora.pt:10174/5771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

15. Kim, YoungJung. Understanding mechanisms of beta cell susceptibility to type 1 diabetes.

Degree: 2015, Columbia University

 Type 1 diabetes mellitus (T1D) is an autoimmune disease characterized by the inflammation of the insulin-producing pancreatic beta cells, eventually leading to beta cell loss… (more)

Subjects/Keywords: Inflammation; Pancreatic beta cells; Diabetes; Molecular biology; Medicine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kim, Y. (2015). Understanding mechanisms of beta cell susceptibility to type 1 diabetes. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8XW4HJ8

Chicago Manual of Style (16th Edition):

Kim, YoungJung. “Understanding mechanisms of beta cell susceptibility to type 1 diabetes.” 2015. Doctoral Dissertation, Columbia University. Accessed January 19, 2021. https://doi.org/10.7916/D8XW4HJ8.

MLA Handbook (7th Edition):

Kim, YoungJung. “Understanding mechanisms of beta cell susceptibility to type 1 diabetes.” 2015. Web. 19 Jan 2021.

Vancouver:

Kim Y. Understanding mechanisms of beta cell susceptibility to type 1 diabetes. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2021 Jan 19]. Available from: https://doi.org/10.7916/D8XW4HJ8.

Council of Science Editors:

Kim Y. Understanding mechanisms of beta cell susceptibility to type 1 diabetes. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://doi.org/10.7916/D8XW4HJ8


Columbia University

16. Churchill, Angela Josephine. Spatiotemporal and Mechanistic Analysis of Nkx2.2 Function in the Pancreatic Islet.

Degree: 2016, Columbia University

Pancreatic beta cell specification is a complex process, requiring proper function of numerous transcription factors. Nkx2.2 is a transcription factor that is crucial for beta(more)

Subjects/Keywords: Cell differentiation; Pancreatic beta cells; Islands of Langerhans; Endocrinology; Genetics

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APA (6th Edition):

Churchill, A. J. (2016). Spatiotemporal and Mechanistic Analysis of Nkx2.2 Function in the Pancreatic Islet. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D84M94N2

Chicago Manual of Style (16th Edition):

Churchill, Angela Josephine. “Spatiotemporal and Mechanistic Analysis of Nkx2.2 Function in the Pancreatic Islet.” 2016. Doctoral Dissertation, Columbia University. Accessed January 19, 2021. https://doi.org/10.7916/D84M94N2.

MLA Handbook (7th Edition):

Churchill, Angela Josephine. “Spatiotemporal and Mechanistic Analysis of Nkx2.2 Function in the Pancreatic Islet.” 2016. Web. 19 Jan 2021.

Vancouver:

Churchill AJ. Spatiotemporal and Mechanistic Analysis of Nkx2.2 Function in the Pancreatic Islet. [Internet] [Doctoral dissertation]. Columbia University; 2016. [cited 2021 Jan 19]. Available from: https://doi.org/10.7916/D84M94N2.

Council of Science Editors:

Churchill AJ. Spatiotemporal and Mechanistic Analysis of Nkx2.2 Function in the Pancreatic Islet. [Doctoral Dissertation]. Columbia University; 2016. Available from: https://doi.org/10.7916/D84M94N2


Columbia University

17. Dominguez Gutierrez, Giselle. Maintenance of Beta Cell Identity and Function.

Degree: 2016, Columbia University

 The acquisition of beta cell identity and function is a multistage process that involves the sequential regulation of specific factors and signals. The maintenance of… (more)

Subjects/Keywords: Diabetes; Cell physiology; Pancreatic beta cells; Molecular biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dominguez Gutierrez, G. (2016). Maintenance of Beta Cell Identity and Function. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8WW7HZG

Chicago Manual of Style (16th Edition):

Dominguez Gutierrez, Giselle. “Maintenance of Beta Cell Identity and Function.” 2016. Doctoral Dissertation, Columbia University. Accessed January 19, 2021. https://doi.org/10.7916/D8WW7HZG.

MLA Handbook (7th Edition):

Dominguez Gutierrez, Giselle. “Maintenance of Beta Cell Identity and Function.” 2016. Web. 19 Jan 2021.

Vancouver:

Dominguez Gutierrez G. Maintenance of Beta Cell Identity and Function. [Internet] [Doctoral dissertation]. Columbia University; 2016. [cited 2021 Jan 19]. Available from: https://doi.org/10.7916/D8WW7HZG.

Council of Science Editors:

Dominguez Gutierrez G. Maintenance of Beta Cell Identity and Function. [Doctoral Dissertation]. Columbia University; 2016. Available from: https://doi.org/10.7916/D8WW7HZG


Michigan State University

18. Bieber, Brian A. Insulin release and adenine nucleotide changes in INS-1 cells in response to a glucose challenge : creatine loading in small cell bioreactors studied by ³¹P-NMR spectroscopy.

Degree: MS, Physiology, 2005, Michigan State University

Subjects/Keywords: Pancreatic beta cells; Adenine nucleotides

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APA (6th Edition):

Bieber, B. A. (2005). Insulin release and adenine nucleotide changes in INS-1 cells in response to a glucose challenge : creatine loading in small cell bioreactors studied by ³¹P-NMR spectroscopy. (Masters Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:33503

Chicago Manual of Style (16th Edition):

Bieber, Brian A. “Insulin release and adenine nucleotide changes in INS-1 cells in response to a glucose challenge : creatine loading in small cell bioreactors studied by ³¹P-NMR spectroscopy.” 2005. Masters Thesis, Michigan State University. Accessed January 19, 2021. http://etd.lib.msu.edu/islandora/object/etd:33503.

MLA Handbook (7th Edition):

Bieber, Brian A. “Insulin release and adenine nucleotide changes in INS-1 cells in response to a glucose challenge : creatine loading in small cell bioreactors studied by ³¹P-NMR spectroscopy.” 2005. Web. 19 Jan 2021.

Vancouver:

Bieber BA. Insulin release and adenine nucleotide changes in INS-1 cells in response to a glucose challenge : creatine loading in small cell bioreactors studied by ³¹P-NMR spectroscopy. [Internet] [Masters thesis]. Michigan State University; 2005. [cited 2021 Jan 19]. Available from: http://etd.lib.msu.edu/islandora/object/etd:33503.

Council of Science Editors:

Bieber BA. Insulin release and adenine nucleotide changes in INS-1 cells in response to a glucose challenge : creatine loading in small cell bioreactors studied by ³¹P-NMR spectroscopy. [Masters Thesis]. Michigan State University; 2005. Available from: http://etd.lib.msu.edu/islandora/object/etd:33503


University of Aberdeen

19. Manning, Yashka. The role of TPD52 in the pancreatic β cell.

Degree: PhD, 2009, University of Aberdeen

 Tumour protein D52 is hypothesised to be involved in regulated secretion in the pancreatic acinar cell, indicated by rapid phosphorylation in response to secretagogue stimulation.… (more)

Subjects/Keywords: 615.1; Diabetes; Pancreatic beta cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Manning, Y. (2009). The role of TPD52 in the pancreatic β cell. (Doctoral Dissertation). University of Aberdeen. Retrieved from https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152346070005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499641

Chicago Manual of Style (16th Edition):

Manning, Yashka. “The role of TPD52 in the pancreatic β cell.” 2009. Doctoral Dissertation, University of Aberdeen. Accessed January 19, 2021. https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152346070005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499641.

MLA Handbook (7th Edition):

Manning, Yashka. “The role of TPD52 in the pancreatic β cell.” 2009. Web. 19 Jan 2021.

Vancouver:

Manning Y. The role of TPD52 in the pancreatic β cell. [Internet] [Doctoral dissertation]. University of Aberdeen; 2009. [cited 2021 Jan 19]. Available from: https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152346070005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499641.

Council of Science Editors:

Manning Y. The role of TPD52 in the pancreatic β cell. [Doctoral Dissertation]. University of Aberdeen; 2009. Available from: https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12152346070005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499641


University of Aberdeen

20. Robertson, Lindsay. Characterisation of the MAL2 proteins and their interaction with TPD52.

Degree: PhD, 2006, University of Aberdeen

 The human MAL2 protein has been demonstrated to regulate secretion in kidney epithelial cells in a lipid raft-dependent manner.  Further, MAL2 interacts with TPD52, a… (more)

Subjects/Keywords: 572.6; Pancreatic beta cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Robertson, L. (2006). Characterisation of the MAL2 proteins and their interaction with TPD52. (Doctoral Dissertation). University of Aberdeen. Retrieved from https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12153461330005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.439915

Chicago Manual of Style (16th Edition):

Robertson, Lindsay. “Characterisation of the MAL2 proteins and their interaction with TPD52.” 2006. Doctoral Dissertation, University of Aberdeen. Accessed January 19, 2021. https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12153461330005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.439915.

MLA Handbook (7th Edition):

Robertson, Lindsay. “Characterisation of the MAL2 proteins and their interaction with TPD52.” 2006. Web. 19 Jan 2021.

Vancouver:

Robertson L. Characterisation of the MAL2 proteins and their interaction with TPD52. [Internet] [Doctoral dissertation]. University of Aberdeen; 2006. [cited 2021 Jan 19]. Available from: https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12153461330005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.439915.

Council of Science Editors:

Robertson L. Characterisation of the MAL2 proteins and their interaction with TPD52. [Doctoral Dissertation]. University of Aberdeen; 2006. Available from: https://eu03.alma.exlibrisgroup.com/view/delivery/44ABE_INST/12153461330005941 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.439915


University of Aberdeen

21. Manning, Yashka. The role of TPD52 in the pancreatic β cell.

Degree: School of Medical Sciences., 2009, University of Aberdeen

Subjects/Keywords: Diabetes.; Pancreatic beta cells.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Manning, Y. (2009). The role of TPD52 in the pancreatic β cell. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=25933 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=25933&custom_att_2=simple_viewer

Chicago Manual of Style (16th Edition):

Manning, Yashka. “The role of TPD52 in the pancreatic β cell.” 2009. Doctoral Dissertation, University of Aberdeen. Accessed January 19, 2021. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=25933 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=25933&custom_att_2=simple_viewer.

MLA Handbook (7th Edition):

Manning, Yashka. “The role of TPD52 in the pancreatic β cell.” 2009. Web. 19 Jan 2021.

Vancouver:

Manning Y. The role of TPD52 in the pancreatic β cell. [Internet] [Doctoral dissertation]. University of Aberdeen; 2009. [cited 2021 Jan 19]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=25933 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=25933&custom_att_2=simple_viewer.

Council of Science Editors:

Manning Y. The role of TPD52 in the pancreatic β cell. [Doctoral Dissertation]. University of Aberdeen; 2009. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=25933 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=25933&custom_att_2=simple_viewer

22. Condorelli, Angelo Giuseppe. Basi molecolari della differente risposta delle cellule alpha e beta del pancreas di mammifero all apoptosi mediata da citochine: implicazioni patogenetiche nel Diabete Mellito.

Degree: 2015, Università degli Studi di Catania

 L apoptosi è considerata la forma principale di morte delle beta cellule pancreatiche produttrici di insulina nel Diabete mellito di tipo 1 (T1DM) e di… (more)

Subjects/Keywords: Area 05 - Scienze biologiche; mammalian pancreatic alpha cells, mammalian pancreatic beta cells, transcriptome, proteome, interactome, proinflammatory cytokines, apoptosis, Diabetes Mellitus

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APA (6th Edition):

Condorelli, A. G. (2015). Basi molecolari della differente risposta delle cellule alpha e beta del pancreas di mammifero all apoptosi mediata da citochine: implicazioni patogenetiche nel Diabete Mellito. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/1683

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Condorelli, Angelo Giuseppe. “Basi molecolari della differente risposta delle cellule alpha e beta del pancreas di mammifero all apoptosi mediata da citochine: implicazioni patogenetiche nel Diabete Mellito.” 2015. Thesis, Università degli Studi di Catania. Accessed January 19, 2021. http://hdl.handle.net/10761/1683.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Condorelli, Angelo Giuseppe. “Basi molecolari della differente risposta delle cellule alpha e beta del pancreas di mammifero all apoptosi mediata da citochine: implicazioni patogenetiche nel Diabete Mellito.” 2015. Web. 19 Jan 2021.

Vancouver:

Condorelli AG. Basi molecolari della differente risposta delle cellule alpha e beta del pancreas di mammifero all apoptosi mediata da citochine: implicazioni patogenetiche nel Diabete Mellito. [Internet] [Thesis]. Università degli Studi di Catania; 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10761/1683.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Condorelli AG. Basi molecolari della differente risposta delle cellule alpha e beta del pancreas di mammifero all apoptosi mediata da citochine: implicazioni patogenetiche nel Diabete Mellito. [Thesis]. Università degli Studi di Catania; 2015. Available from: http://hdl.handle.net/10761/1683

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

23. 莊子逸. Inducing the progressive differentiation of hESCs into pancreatic progenitor cells.

Degree: 2013, University of Hong Kong

 Diabetes is a chronic disorder of the pancreas, where a decline in the insulin-producing β-cell population disrupts metabolic homeostasis. Pancreatic transplantation has shown to be… (more)

Subjects/Keywords: Pancreatic beta cells; Embryonic stem cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

莊子逸. (2013). Inducing the progressive differentiation of hESCs into pancreatic progenitor cells. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/196433

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

莊子逸. “Inducing the progressive differentiation of hESCs into pancreatic progenitor cells.” 2013. Thesis, University of Hong Kong. Accessed January 19, 2021. http://hdl.handle.net/10722/196433.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

莊子逸. “Inducing the progressive differentiation of hESCs into pancreatic progenitor cells.” 2013. Web. 19 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

莊子逸. Inducing the progressive differentiation of hESCs into pancreatic progenitor cells. [Internet] [Thesis]. University of Hong Kong; 2013. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10722/196433.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

莊子逸. Inducing the progressive differentiation of hESCs into pancreatic progenitor cells. [Thesis]. University of Hong Kong; 2013. Available from: http://hdl.handle.net/10722/196433

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

24. Cui, Ju. Kinesin-1 in pancreatic beta cell and renal epithelial cell.

Degree: 2011, University of Hong Kong

Subjects/Keywords: Pancreatic beta cells; Kinesin; Epithelial cells

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APA (6th Edition):

Cui, J. (2011). Kinesin-1 in pancreatic beta cell and renal epithelial cell. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/197835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cui, Ju. “Kinesin-1 in pancreatic beta cell and renal epithelial cell.” 2011. Thesis, University of Hong Kong. Accessed January 19, 2021. http://hdl.handle.net/10722/197835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cui, Ju. “Kinesin-1 in pancreatic beta cell and renal epithelial cell.” 2011. Web. 19 Jan 2021.

Vancouver:

Cui J. Kinesin-1 in pancreatic beta cell and renal epithelial cell. [Internet] [Thesis]. University of Hong Kong; 2011. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10722/197835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cui J. Kinesin-1 in pancreatic beta cell and renal epithelial cell. [Thesis]. University of Hong Kong; 2011. Available from: http://hdl.handle.net/10722/197835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Kapodistria, Aikaterini. Μελέτη του ρόλου της νεφρίνης και των νεφρινο-συνδεόμενων πρωτεϊνων στη σηματοδότηση και στη φυσιολογία των παγκρεατικών β-κυττάρων.

Degree: 2015, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

In this study, we investigated the potential role of nephrin in the regulation of pancreatic beta cell survival. We utilized endogenous nephrin expression in βTC-6… (more)

Subjects/Keywords: Νεφρίνη; Σακχαρώδης διαβήτης; Παγκρεατικά β κύτταρα; Σηματοδότηση PI3K/Akt; Απόπτωση (προγραμματισμένος κυτταρικός θάνατος); Nephrin; Diabetes mellitus type 2; Pancreatic beta-cells; signaling of PI3K/Akt; Apoptosis (programmed cell death)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kapodistria, A. (2015). Μελέτη του ρόλου της νεφρίνης και των νεφρινο-συνδεόμενων πρωτεϊνων στη σηματοδότηση και στη φυσιολογία των παγκρεατικών β-κυττάρων. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/36709

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kapodistria, Aikaterini. “Μελέτη του ρόλου της νεφρίνης και των νεφρινο-συνδεόμενων πρωτεϊνων στη σηματοδότηση και στη φυσιολογία των παγκρεατικών β-κυττάρων.” 2015. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed January 19, 2021. http://hdl.handle.net/10442/hedi/36709.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kapodistria, Aikaterini. “Μελέτη του ρόλου της νεφρίνης και των νεφρινο-συνδεόμενων πρωτεϊνων στη σηματοδότηση και στη φυσιολογία των παγκρεατικών β-κυττάρων.” 2015. Web. 19 Jan 2021.

Vancouver:

Kapodistria A. Μελέτη του ρόλου της νεφρίνης και των νεφρινο-συνδεόμενων πρωτεϊνων στη σηματοδότηση και στη φυσιολογία των παγκρεατικών β-κυττάρων. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/10442/hedi/36709.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kapodistria A. Μελέτη του ρόλου της νεφρίνης και των νεφρινο-συνδεόμενων πρωτεϊνων στη σηματοδότηση και στη φυσιολογία των παγκρεατικών β-κυττάρων. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. Available from: http://hdl.handle.net/10442/hedi/36709

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Rawal, Sonia. REAGGREGATION OF PANCREATIC ISLET CELLS: IMPLICATIONS FOR ISLET HEALTH, STORAGE AND NEW DRUG DISCOVERY.

Degree: PhD, Physical Therapy & Rehabilitation Sciences, 2016, University of Kansas

 Islets are clusters of cells in the pancreas that monitor and regulate blood glucose levels. In culture, single islet cells reaggregate into clusters, but the… (more)

Subjects/Keywords: Health sciences; Beta cells; Co-culture; Cyopreservation; Diabetes; Insulin secretion; Pancreatic islets

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APA (6th Edition):

Rawal, S. (2016). REAGGREGATION OF PANCREATIC ISLET CELLS: IMPLICATIONS FOR ISLET HEALTH, STORAGE AND NEW DRUG DISCOVERY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/24815

Chicago Manual of Style (16th Edition):

Rawal, Sonia. “REAGGREGATION OF PANCREATIC ISLET CELLS: IMPLICATIONS FOR ISLET HEALTH, STORAGE AND NEW DRUG DISCOVERY.” 2016. Doctoral Dissertation, University of Kansas. Accessed January 19, 2021. http://hdl.handle.net/1808/24815.

MLA Handbook (7th Edition):

Rawal, Sonia. “REAGGREGATION OF PANCREATIC ISLET CELLS: IMPLICATIONS FOR ISLET HEALTH, STORAGE AND NEW DRUG DISCOVERY.” 2016. Web. 19 Jan 2021.

Vancouver:

Rawal S. REAGGREGATION OF PANCREATIC ISLET CELLS: IMPLICATIONS FOR ISLET HEALTH, STORAGE AND NEW DRUG DISCOVERY. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1808/24815.

Council of Science Editors:

Rawal S. REAGGREGATION OF PANCREATIC ISLET CELLS: IMPLICATIONS FOR ISLET HEALTH, STORAGE AND NEW DRUG DISCOVERY. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/24815


Vanderbilt University

27. Caldwell, Brittany Catherine. Dopamine Regulation of Insulin Secretion Investigated by Fluorescence Fluctuation Spectroscopy.

Degree: PhD, Biomedical Engineering, 2016, Vanderbilt University

 Insulin resistance together with insufficient insulin secretion leads to the development of type II diabetes mellitus. Glucose-stimulation of insulin secretion has been extensively studied, but… (more)

Subjects/Keywords: Single Molecule Imaging; Insulin Secretion; Fluorescence Fluctuation Spectroscopy; Fluorescence; Pancreatic Beta Cells

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APA (6th Edition):

Caldwell, B. C. (2016). Dopamine Regulation of Insulin Secretion Investigated by Fluorescence Fluctuation Spectroscopy. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11006

Chicago Manual of Style (16th Edition):

Caldwell, Brittany Catherine. “Dopamine Regulation of Insulin Secretion Investigated by Fluorescence Fluctuation Spectroscopy.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed January 19, 2021. http://hdl.handle.net/1803/11006.

MLA Handbook (7th Edition):

Caldwell, Brittany Catherine. “Dopamine Regulation of Insulin Secretion Investigated by Fluorescence Fluctuation Spectroscopy.” 2016. Web. 19 Jan 2021.

Vancouver:

Caldwell BC. Dopamine Regulation of Insulin Secretion Investigated by Fluorescence Fluctuation Spectroscopy. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1803/11006.

Council of Science Editors:

Caldwell BC. Dopamine Regulation of Insulin Secretion Investigated by Fluorescence Fluctuation Spectroscopy. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/11006


University of Toronto

28. Columbus, Joshua. Examination of Expression and Function of TCF Genes in the Pancreatic Islets.

Degree: 2010, University of Toronto

Specific SNPs in intronic regions of the human TCF7L2 gene are associated with an elevated risk of T2D development and progression. Several investigations have suggested… (more)

Subjects/Keywords: Type 2 Diabetes; TCF7L2; Wnt Signalling Pathway; Insulin; pancreatic beta cells; 0719

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Columbus, J. (2010). Examination of Expression and Function of TCF Genes in the Pancreatic Islets. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25458

Chicago Manual of Style (16th Edition):

Columbus, Joshua. “Examination of Expression and Function of TCF Genes in the Pancreatic Islets.” 2010. Masters Thesis, University of Toronto. Accessed January 19, 2021. http://hdl.handle.net/1807/25458.

MLA Handbook (7th Edition):

Columbus, Joshua. “Examination of Expression and Function of TCF Genes in the Pancreatic Islets.” 2010. Web. 19 Jan 2021.

Vancouver:

Columbus J. Examination of Expression and Function of TCF Genes in the Pancreatic Islets. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/1807/25458.

Council of Science Editors:

Columbus J. Examination of Expression and Function of TCF Genes in the Pancreatic Islets. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25458


Boston University

29. Datu Tasik, Grace Marselina. Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1).

Degree: MS, Nutrition and Metabolism, 2019, Boston University

 OBJECTIVE: Cardiovascular disease (CVD) remains the leading cause of death globally. Reducing high blood cholesterol, which is a dominant risk factor for CVD events, is… (more)

Subjects/Keywords: Nutrition; Cardiovascular disease; Cholesterol; Clonal pancreatic beta-cells; Insulin; Statins; Type 2 diabetes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Datu Tasik, G. M. (2019). Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1). (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/36546

Chicago Manual of Style (16th Edition):

Datu Tasik, Grace Marselina. “Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1).” 2019. Masters Thesis, Boston University. Accessed January 19, 2021. http://hdl.handle.net/2144/36546.

MLA Handbook (7th Edition):

Datu Tasik, Grace Marselina. “Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1).” 2019. Web. 19 Jan 2021.

Vancouver:

Datu Tasik GM. Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1). [Internet] [Masters thesis]. Boston University; 2019. [cited 2021 Jan 19]. Available from: http://hdl.handle.net/2144/36546.

Council of Science Editors:

Datu Tasik GM. Effect of different types of statins: simvastatin, lovastatin and pitavastatin on glucose-stimulated insulin secretion and insulin content from clonal pancreatic beta-cells (INS-1). [Masters Thesis]. Boston University; 2019. Available from: http://hdl.handle.net/2144/36546


University of Aberdeen

30. Kanase, Nilesh. The impact of oxidative stress and potential antioxidant therapy on function and survival of cultured pancreatic β-islet cells.

Degree: School of Medical Sciences., 2010, University of Aberdeen

Subjects/Keywords: Pancreatic beta cells.; Antioxidants; Oxidation, Physiological.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kanase, N. (2010). The impact of oxidative stress and potential antioxidant therapy on function and survival of cultured pancreatic β-islet cells. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=165833 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=165833&custom_att_2=simple_viewer

Chicago Manual of Style (16th Edition):

Kanase, Nilesh. “The impact of oxidative stress and potential antioxidant therapy on function and survival of cultured pancreatic β-islet cells.” 2010. Doctoral Dissertation, University of Aberdeen. Accessed January 19, 2021. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=165833 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=165833&custom_att_2=simple_viewer.

MLA Handbook (7th Edition):

Kanase, Nilesh. “The impact of oxidative stress and potential antioxidant therapy on function and survival of cultured pancreatic β-islet cells.” 2010. Web. 19 Jan 2021.

Vancouver:

Kanase N. The impact of oxidative stress and potential antioxidant therapy on function and survival of cultured pancreatic β-islet cells. [Internet] [Doctoral dissertation]. University of Aberdeen; 2010. [cited 2021 Jan 19]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=165833 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=165833&custom_att_2=simple_viewer.

Council of Science Editors:

Kanase N. The impact of oxidative stress and potential antioxidant therapy on function and survival of cultured pancreatic β-islet cells. [Doctoral Dissertation]. University of Aberdeen; 2010. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=165833 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=165833&custom_att_2=simple_viewer

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