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You searched for subject:(PTEN). Showing records 1 – 30 of 234 total matches.

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1. Bolduc, David Michael. PHOSPHORYLATION-INDUCED CONFORMATIONAL CHANGES OF PTEN REVEALED BY PROTEIN SEMISYNTHESIS.

Degree: 2013, Johns Hopkins University

PTEN (phosphatase and tensin homolog deleted on chromosome 10) is a tumor suppressing lipid phosphatase that negatively regulates the PI3K/PTEN/AKT signaling pathway by dephosphorylating the… (more)

Subjects/Keywords: PTEN; Phosphorylation

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APA (6th Edition):

Bolduc, D. M. (2013). PHOSPHORYLATION-INDUCED CONFORMATIONAL CHANGES OF PTEN REVEALED BY PROTEIN SEMISYNTHESIS. (Thesis). Johns Hopkins University. Retrieved from http://jhir.library.jhu.edu/handle/1774.2/37060

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bolduc, David Michael. “PHOSPHORYLATION-INDUCED CONFORMATIONAL CHANGES OF PTEN REVEALED BY PROTEIN SEMISYNTHESIS.” 2013. Thesis, Johns Hopkins University. Accessed October 29, 2020. http://jhir.library.jhu.edu/handle/1774.2/37060.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bolduc, David Michael. “PHOSPHORYLATION-INDUCED CONFORMATIONAL CHANGES OF PTEN REVEALED BY PROTEIN SEMISYNTHESIS.” 2013. Web. 29 Oct 2020.

Vancouver:

Bolduc DM. PHOSPHORYLATION-INDUCED CONFORMATIONAL CHANGES OF PTEN REVEALED BY PROTEIN SEMISYNTHESIS. [Internet] [Thesis]. Johns Hopkins University; 2013. [cited 2020 Oct 29]. Available from: http://jhir.library.jhu.edu/handle/1774.2/37060.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bolduc DM. PHOSPHORYLATION-INDUCED CONFORMATIONAL CHANGES OF PTEN REVEALED BY PROTEIN SEMISYNTHESIS. [Thesis]. Johns Hopkins University; 2013. Available from: http://jhir.library.jhu.edu/handle/1774.2/37060

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

2. CHIA, YEONG-CHIT. Investigation of the mechanism of catalysis and regulation of PTEN by C-terminal tail phosphorylation.

Degree: 2015, University of Melbourne

 The tumour suppressor PTEN is a phosphatase possessing both phospholipid- and phosphoprotein-phosphatase activities. It exerts its tumour suppressor function primarily by antagonising the PI3K-Akt growth… (more)

Subjects/Keywords: PTEN tumour suppressor

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APA (6th Edition):

CHIA, Y. (2015). Investigation of the mechanism of catalysis and regulation of PTEN by C-terminal tail phosphorylation. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/56505

Chicago Manual of Style (16th Edition):

CHIA, YEONG-CHIT. “Investigation of the mechanism of catalysis and regulation of PTEN by C-terminal tail phosphorylation.” 2015. Doctoral Dissertation, University of Melbourne. Accessed October 29, 2020. http://hdl.handle.net/11343/56505.

MLA Handbook (7th Edition):

CHIA, YEONG-CHIT. “Investigation of the mechanism of catalysis and regulation of PTEN by C-terminal tail phosphorylation.” 2015. Web. 29 Oct 2020.

Vancouver:

CHIA Y. Investigation of the mechanism of catalysis and regulation of PTEN by C-terminal tail phosphorylation. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/11343/56505.

Council of Science Editors:

CHIA Y. Investigation of the mechanism of catalysis and regulation of PTEN by C-terminal tail phosphorylation. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/56505


Universiteit Utrecht

3. Spijker, H.M. van 't. The role of PTEN in the formation of thalamocortical axonal tracts.

Degree: 2014, Universiteit Utrecht

 Autism spectrum disorder (ASD) is a heritable disorder which affect ̴1% of the population. Unfortunately, no targeted therapy is available. It is widely established that… (more)

Subjects/Keywords: PTEN; tahalmocortical axon tract; ASD

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APA (6th Edition):

Spijker, H. M. v. '. (2014). The role of PTEN in the formation of thalamocortical axonal tracts. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/295450

Chicago Manual of Style (16th Edition):

Spijker, H M van 't. “The role of PTEN in the formation of thalamocortical axonal tracts.” 2014. Masters Thesis, Universiteit Utrecht. Accessed October 29, 2020. http://dspace.library.uu.nl:8080/handle/1874/295450.

MLA Handbook (7th Edition):

Spijker, H M van 't. “The role of PTEN in the formation of thalamocortical axonal tracts.” 2014. Web. 29 Oct 2020.

Vancouver:

Spijker HMv'. The role of PTEN in the formation of thalamocortical axonal tracts. [Internet] [Masters thesis]. Universiteit Utrecht; 2014. [cited 2020 Oct 29]. Available from: http://dspace.library.uu.nl:8080/handle/1874/295450.

Council of Science Editors:

Spijker HMv'. The role of PTEN in the formation of thalamocortical axonal tracts. [Masters Thesis]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/295450


Universiteit Utrecht

4. Stumpf, Miriam. Interplay of PTEN subcellular localization and catalytic activities in vivo.

Degree: 2016, Universiteit Utrecht

 This thesis describes the use of mammalian cells, S. cerevisiae and D. rerio to unravel the complex interplay of PTEN subcellular localization and catalytic activities.… (more)

Subjects/Keywords: PTEN; Zebrafish; Cancer; Angiogenesis; Importins

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APA (6th Edition):

Stumpf, M. (2016). Interplay of PTEN subcellular localization and catalytic activities in vivo. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/334160

Chicago Manual of Style (16th Edition):

Stumpf, Miriam. “Interplay of PTEN subcellular localization and catalytic activities in vivo.” 2016. Doctoral Dissertation, Universiteit Utrecht. Accessed October 29, 2020. http://dspace.library.uu.nl:8080/handle/1874/334160.

MLA Handbook (7th Edition):

Stumpf, Miriam. “Interplay of PTEN subcellular localization and catalytic activities in vivo.” 2016. Web. 29 Oct 2020.

Vancouver:

Stumpf M. Interplay of PTEN subcellular localization and catalytic activities in vivo. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2016. [cited 2020 Oct 29]. Available from: http://dspace.library.uu.nl:8080/handle/1874/334160.

Council of Science Editors:

Stumpf M. Interplay of PTEN subcellular localization and catalytic activities in vivo. [Doctoral Dissertation]. Universiteit Utrecht; 2016. Available from: http://dspace.library.uu.nl:8080/handle/1874/334160


University of Texas Southwestern Medical Center

5. McEllin, Brian Matthew. Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems.

Degree: 2012, University of Texas Southwestern Medical Center

 Glioblastoma multiforme (GBM) are deadly brain tumors that are refractory to radiation and chemotherapy. Despite decades of work, little progress has been made in improving… (more)

Subjects/Keywords: PTEN Phosphohydrolase; Brain Neoplasms; Glioblastoma

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APA (6th Edition):

McEllin, B. M. (2012). Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1000

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McEllin, Brian Matthew. “Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed October 29, 2020. http://hdl.handle.net/2152.5/1000.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McEllin, Brian Matthew. “Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems.” 2012. Web. 29 Oct 2020.

Vancouver:

McEllin BM. Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/2152.5/1000.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McEllin BM. Dissecting Molecular Mechanisms of Radioresistance Using in Vitro and in Vivo Brain Tumor Model Systems. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1000

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston College

6. Wei, Yang. Mechanistic studies of two phosphatase enzymes involved in inostiol metabolism.

Degree: PhD, Chemistry, 2013, Boston College

 Inositol-containing molecules and inositol phosphatases have diverse roles in cells. One of the inositol phospholipids phosphatases, PTEN (Phosphatase and Tensin Homolog deleted on Chromosome Ten),… (more)

Subjects/Keywords: inositol; inositol monophosphatase; PTEN

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APA (6th Edition):

Wei, Y. (2013). Mechanistic studies of two phosphatase enzymes involved in inostiol metabolism. (Doctoral Dissertation). Boston College. Retrieved from http://dlib.bc.edu/islandora/object/bc-ir:101374

Chicago Manual of Style (16th Edition):

Wei, Yang. “Mechanistic studies of two phosphatase enzymes involved in inostiol metabolism.” 2013. Doctoral Dissertation, Boston College. Accessed October 29, 2020. http://dlib.bc.edu/islandora/object/bc-ir:101374.

MLA Handbook (7th Edition):

Wei, Yang. “Mechanistic studies of two phosphatase enzymes involved in inostiol metabolism.” 2013. Web. 29 Oct 2020.

Vancouver:

Wei Y. Mechanistic studies of two phosphatase enzymes involved in inostiol metabolism. [Internet] [Doctoral dissertation]. Boston College; 2013. [cited 2020 Oct 29]. Available from: http://dlib.bc.edu/islandora/object/bc-ir:101374.

Council of Science Editors:

Wei Y. Mechanistic studies of two phosphatase enzymes involved in inostiol metabolism. [Doctoral Dissertation]. Boston College; 2013. Available from: http://dlib.bc.edu/islandora/object/bc-ir:101374


University of Notre Dame

7. Hui Yu. Function of Lipid Binding Ability of Nedd4 on Its Regulation of PTEN and Other Substrates</h1>.

Degree: Chemistry and Biochemistry, 2014, University of Notre Dame

  Nedd4 (neural precursor cell expressed, developmentally down-regulated 4) proteins are a family of E3 ubiquitin ligases. These enzymes play important roles in various cellular… (more)

Subjects/Keywords: Nedd4; Ebola; VP40; PTEN; Cancer

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APA (6th Edition):

Yu, H. (2014). Function of Lipid Binding Ability of Nedd4 on Its Regulation of PTEN and Other Substrates</h1>. (Thesis). University of Notre Dame. Retrieved from https://curate.nd.edu/show/ns064457n0m

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yu, Hui. “Function of Lipid Binding Ability of Nedd4 on Its Regulation of PTEN and Other Substrates</h1>.” 2014. Thesis, University of Notre Dame. Accessed October 29, 2020. https://curate.nd.edu/show/ns064457n0m.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yu, Hui. “Function of Lipid Binding Ability of Nedd4 on Its Regulation of PTEN and Other Substrates</h1>.” 2014. Web. 29 Oct 2020.

Vancouver:

Yu H. Function of Lipid Binding Ability of Nedd4 on Its Regulation of PTEN and Other Substrates</h1>. [Internet] [Thesis]. University of Notre Dame; 2014. [cited 2020 Oct 29]. Available from: https://curate.nd.edu/show/ns064457n0m.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yu H. Function of Lipid Binding Ability of Nedd4 on Its Regulation of PTEN and Other Substrates</h1>. [Thesis]. University of Notre Dame; 2014. Available from: https://curate.nd.edu/show/ns064457n0m

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

8. Low, Ley Hian. The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners.

Degree: 2012, University of Melbourne

 The Nedd4 family interacting protein 1 (Ndfip1) is a neuroprotective protein, highly up-regulated in the neuron following brain injury. Many fundamental questions regarding the functions… (more)

Subjects/Keywords: Ndfip1; Nedd4; ubiquitination; PTEN

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APA (6th Edition):

Low, L. H. (2012). The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37781

Chicago Manual of Style (16th Edition):

Low, Ley Hian. “The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners.” 2012. Doctoral Dissertation, University of Melbourne. Accessed October 29, 2020. http://hdl.handle.net/11343/37781.

MLA Handbook (7th Edition):

Low, Ley Hian. “The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners.” 2012. Web. 29 Oct 2020.

Vancouver:

Low LH. The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/11343/37781.

Council of Science Editors:

Low LH. The control of Nedd4 family interacting protein 1 (Ndfip1) expression and its binding partners. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37781


University of Southern California

9. Yang, Kai-Ting. Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression.

Degree: MS, Biochemistry and Molecular Biology, 2013, University of Southern California

 β-cells, which produce and release insulin play a major role in maintaining glucose homeostasis. PTEN is a lipid phosphatase that antagonizes PI3K/AKT signaling pathway and… (more)

Subjects/Keywords: Pten; beta-cell; cell proliferation

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APA (6th Edition):

Yang, K. (2013). Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316863/rec/5321

Chicago Manual of Style (16th Edition):

Yang, Kai-Ting. “Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression.” 2013. Masters Thesis, University of Southern California. Accessed October 29, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316863/rec/5321.

MLA Handbook (7th Edition):

Yang, Kai-Ting. “Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression.” 2013. Web. 29 Oct 2020.

Vancouver:

Yang K. Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2020 Oct 29]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316863/rec/5321.

Council of Science Editors:

Yang K. Pten deletion in adult pancreatic beta-cells induces cell proliferation and G1/S cell cycle progression. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316863/rec/5321


University of Southern California

10. Wey, Shiuan. The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2013, University of Southern California

 The endoplasmic reticulum (ER) is an intracellular organelle for protein folding, lipid synthesis and Ca²⁺ storage. It is also responsible for the transportation for most… (more)

Subjects/Keywords: GRP78; AKT; PTEN; leukemia; hematopoiesis

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APA (6th Edition):

Wey, S. (2013). The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/629905/rec/7218

Chicago Manual of Style (16th Edition):

Wey, Shiuan. “The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis.” 2013. Doctoral Dissertation, University of Southern California. Accessed October 29, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/629905/rec/7218.

MLA Handbook (7th Edition):

Wey, Shiuan. “The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis.” 2013. Web. 29 Oct 2020.

Vancouver:

Wey S. The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2020 Oct 29]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/629905/rec/7218.

Council of Science Editors:

Wey S. The role of endoplasmic reticulum protein GRP78 in normal hematopoeises and PTEN-null leukemogenesis. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/629905/rec/7218


University of Southern California

11. Patil, Ishan Y. Neuronal and glial metabolic alterations in the liver-specific PTEN knockout mouse model.

Degree: MS, Molecular Pharmacology and Toxicology, 2014, University of Southern California

 Phosphatase and Tensin Homologue (PTEN) is a negative regulator of the phosphatidylinositol 3‐kinase/AKT pathway. Liver‐specific deletion of PTEN results in increased fatty acid synthesis, accompanied… (more)

Subjects/Keywords: PTEN; NMR; FDG-PET; metabolism

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APA (6th Edition):

Patil, I. Y. (2014). Neuronal and glial metabolic alterations in the liver-specific PTEN knockout mouse model. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/449019/rec/4385

Chicago Manual of Style (16th Edition):

Patil, Ishan Y. “Neuronal and glial metabolic alterations in the liver-specific PTEN knockout mouse model.” 2014. Masters Thesis, University of Southern California. Accessed October 29, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/449019/rec/4385.

MLA Handbook (7th Edition):

Patil, Ishan Y. “Neuronal and glial metabolic alterations in the liver-specific PTEN knockout mouse model.” 2014. Web. 29 Oct 2020.

Vancouver:

Patil IY. Neuronal and glial metabolic alterations in the liver-specific PTEN knockout mouse model. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Oct 29]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/449019/rec/4385.

Council of Science Editors:

Patil IY. Neuronal and glial metabolic alterations in the liver-specific PTEN knockout mouse model. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/449019/rec/4385


Queens University

12. Liu, Jun. A Role for Insulin Signaling in Regulating the PTEN Tumour Suppressor in Caenorhabditis Elegans .

Degree: Biology, 2013, Queens University

 Many obese individuals and type 2 diabetes mellitus (T2DM) patients have elevated levels of insulin. Hyperinsulinemia is a major cancer risk factor in T2DM individuals… (more)

Subjects/Keywords: C. Elegans ; PTEN ; Insulin Signaling

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APA (6th Edition):

Liu, J. (2013). A Role for Insulin Signaling in Regulating the PTEN Tumour Suppressor in Caenorhabditis Elegans . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Jun. “A Role for Insulin Signaling in Regulating the PTEN Tumour Suppressor in Caenorhabditis Elegans .” 2013. Thesis, Queens University. Accessed October 29, 2020. http://hdl.handle.net/1974/7809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Jun. “A Role for Insulin Signaling in Regulating the PTEN Tumour Suppressor in Caenorhabditis Elegans .” 2013. Web. 29 Oct 2020.

Vancouver:

Liu J. A Role for Insulin Signaling in Regulating the PTEN Tumour Suppressor in Caenorhabditis Elegans . [Internet] [Thesis]. Queens University; 2013. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1974/7809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu J. A Role for Insulin Signaling in Regulating the PTEN Tumour Suppressor in Caenorhabditis Elegans . [Thesis]. Queens University; 2013. Available from: http://hdl.handle.net/1974/7809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

13. Kechagioglou, Petros. Γενωμική μελέτη και συσχέτιση με την καρκινογένεση, της ομόλογης με την τενσίνη, φωσφατάσης: χρήση νανοϋλικών και μελέτη της ογκοκατασταλτικής της δράσης.

Degree: 2014, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

Phosphatase and tensin homolog protein (PTEN), usually observed with loss of heterozygosity on chromosome 10, is one of the most frequently mutated human tumor suppressor… (more)

Subjects/Keywords: Ομόλογη της τενσίνης; Φωσφατάση; PTEN

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APA (6th Edition):

Kechagioglou, P. (2014). Γενωμική μελέτη και συσχέτιση με την καρκινογένεση, της ομόλογης με την τενσίνη, φωσφατάσης: χρήση νανοϋλικών και μελέτη της ογκοκατασταλτικής της δράσης. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/35040

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kechagioglou, Petros. “Γενωμική μελέτη και συσχέτιση με την καρκινογένεση, της ομόλογης με την τενσίνη, φωσφατάσης: χρήση νανοϋλικών και μελέτη της ογκοκατασταλτικής της δράσης.” 2014. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed October 29, 2020. http://hdl.handle.net/10442/hedi/35040.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kechagioglou, Petros. “Γενωμική μελέτη και συσχέτιση με την καρκινογένεση, της ομόλογης με την τενσίνη, φωσφατάσης: χρήση νανοϋλικών και μελέτη της ογκοκατασταλτικής της δράσης.” 2014. Web. 29 Oct 2020.

Vancouver:

Kechagioglou P. Γενωμική μελέτη και συσχέτιση με την καρκινογένεση, της ομόλογης με την τενσίνη, φωσφατάσης: χρήση νανοϋλικών και μελέτη της ογκοκατασταλτικής της δράσης. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2014. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/10442/hedi/35040.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kechagioglou P. Γενωμική μελέτη και συσχέτιση με την καρκινογένεση, της ομόλογης με την τενσίνη, φωσφατάσης: χρήση νανοϋλικών και μελέτη της ογκοκατασταλτικής της δράσης. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2014. Available from: http://hdl.handle.net/10442/hedi/35040

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Prieto-Oliveira, Paula. Análise histológica e imuno-histoquímica de leiomiomas e leiomiossarcomas de boca.

Degree: Mestrado, Patologia Bucal, 2012, University of São Paulo

O objetivo deste estudo foi comparar leiomiomas e leiomiossarcomas de boca por meio de análise histológica e imunoistoquímica, utilizando os marcadores Ki67, p53 e PTEN.… (more)

Subjects/Keywords: ki67; ki67; Leiomioma; Leiomiossarcoma; Leiomyoma; Leiomyosarcoma; p53; p53; PTEN; PTEN

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APA (6th Edition):

Prieto-Oliveira, P. (2012). Análise histológica e imuno-histoquímica de leiomiomas e leiomiossarcomas de boca. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13042013-114813/ ;

Chicago Manual of Style (16th Edition):

Prieto-Oliveira, Paula. “Análise histológica e imuno-histoquímica de leiomiomas e leiomiossarcomas de boca.” 2012. Masters Thesis, University of São Paulo. Accessed October 29, 2020. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13042013-114813/ ;.

MLA Handbook (7th Edition):

Prieto-Oliveira, Paula. “Análise histológica e imuno-histoquímica de leiomiomas e leiomiossarcomas de boca.” 2012. Web. 29 Oct 2020.

Vancouver:

Prieto-Oliveira P. Análise histológica e imuno-histoquímica de leiomiomas e leiomiossarcomas de boca. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2020 Oct 29]. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13042013-114813/ ;.

Council of Science Editors:

Prieto-Oliveira P. Análise histológica e imuno-histoquímica de leiomiomas e leiomiossarcomas de boca. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13042013-114813/ ;

15. Bonnet, Mélanie. Dérégulation de MYC dans les Leucémies Aiguës Lymphoblastiques T : A posteriori estimation method and domain decomposition.

Degree: Docteur es, Biologie, Spécialité Immunologie, 2011, Aix-Marseille 2

La leucémie aiguë lymphoblastique (LAL-T) est une hémopathie maligne qui représente 10 à 15% des LAL pédiatriques et 25% des LAL de l’adulte. Bien que… (more)

Subjects/Keywords: Lal-t; Myc; Pten; Notch1; T-all; Myc; Pten; Notch1

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APA (6th Edition):

Bonnet, M. (2011). Dérégulation de MYC dans les Leucémies Aiguës Lymphoblastiques T : A posteriori estimation method and domain decomposition. (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2011AIX22090

Chicago Manual of Style (16th Edition):

Bonnet, Mélanie. “Dérégulation de MYC dans les Leucémies Aiguës Lymphoblastiques T : A posteriori estimation method and domain decomposition.” 2011. Doctoral Dissertation, Aix-Marseille 2. Accessed October 29, 2020. http://www.theses.fr/2011AIX22090.

MLA Handbook (7th Edition):

Bonnet, Mélanie. “Dérégulation de MYC dans les Leucémies Aiguës Lymphoblastiques T : A posteriori estimation method and domain decomposition.” 2011. Web. 29 Oct 2020.

Vancouver:

Bonnet M. Dérégulation de MYC dans les Leucémies Aiguës Lymphoblastiques T : A posteriori estimation method and domain decomposition. [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2011. [cited 2020 Oct 29]. Available from: http://www.theses.fr/2011AIX22090.

Council of Science Editors:

Bonnet M. Dérégulation de MYC dans les Leucémies Aiguës Lymphoblastiques T : A posteriori estimation method and domain decomposition. [Doctoral Dissertation]. Aix-Marseille 2; 2011. Available from: http://www.theses.fr/2011AIX22090

16. Dufour, Julie. Rôle des récepteurs des oxystérols LXRs (Liver X Réceptors) dans le processus de carcinogenèse prostatique chez la souris : Role of LXRs (Liver X Receptors) oxysterol receptors in the prostate carcinogenesis process in mice.

Degree: Docteur es, Physiologie et Génétique Moléculaire, 2013, Université Blaise-Pascale, Clermont-Ferrand II

De nombreuses études épidémiologiques associent le cholestérol avec l’incidence et le développement du cancer de la prostate. Parmi les acteurs impliqués dans le métabolisme du… (more)

Subjects/Keywords: Cancer; Prostate; LXRs; Cholestérol; PTEN; Cancer; Prostate; LXRs; Cholesterol; PTEN

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APA (6th Edition):

Dufour, J. (2013). Rôle des récepteurs des oxystérols LXRs (Liver X Réceptors) dans le processus de carcinogenèse prostatique chez la souris : Role of LXRs (Liver X Receptors) oxysterol receptors in the prostate carcinogenesis process in mice. (Doctoral Dissertation). Université Blaise-Pascale, Clermont-Ferrand II. Retrieved from http://www.theses.fr/2013CLF22342

Chicago Manual of Style (16th Edition):

Dufour, Julie. “Rôle des récepteurs des oxystérols LXRs (Liver X Réceptors) dans le processus de carcinogenèse prostatique chez la souris : Role of LXRs (Liver X Receptors) oxysterol receptors in the prostate carcinogenesis process in mice.” 2013. Doctoral Dissertation, Université Blaise-Pascale, Clermont-Ferrand II. Accessed October 29, 2020. http://www.theses.fr/2013CLF22342.

MLA Handbook (7th Edition):

Dufour, Julie. “Rôle des récepteurs des oxystérols LXRs (Liver X Réceptors) dans le processus de carcinogenèse prostatique chez la souris : Role of LXRs (Liver X Receptors) oxysterol receptors in the prostate carcinogenesis process in mice.” 2013. Web. 29 Oct 2020.

Vancouver:

Dufour J. Rôle des récepteurs des oxystérols LXRs (Liver X Réceptors) dans le processus de carcinogenèse prostatique chez la souris : Role of LXRs (Liver X Receptors) oxysterol receptors in the prostate carcinogenesis process in mice. [Internet] [Doctoral dissertation]. Université Blaise-Pascale, Clermont-Ferrand II; 2013. [cited 2020 Oct 29]. Available from: http://www.theses.fr/2013CLF22342.

Council of Science Editors:

Dufour J. Rôle des récepteurs des oxystérols LXRs (Liver X Réceptors) dans le processus de carcinogenèse prostatique chez la souris : Role of LXRs (Liver X Receptors) oxysterol receptors in the prostate carcinogenesis process in mice. [Doctoral Dissertation]. Université Blaise-Pascale, Clermont-Ferrand II; 2013. Available from: http://www.theses.fr/2013CLF22342

17. Boudra, Rafik. Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology.

Degree: Docteur es, Physiologie et Génétique Moléculaires, 2015, Université Blaise-Pascale, Clermont-Ferrand II

La Nucléophosmine (NPM1/B23) est une petite chaperonne moléculaire impliquée dans de nombreux processus cellulaires, tels que la régulation de l’expression génique ou le contrôle du… (more)

Subjects/Keywords: Prostate; Cancer; Nucléophosmine; NPM1; PTEN; MTOR; Nucleophosmin; NPM1; PTEN; MTOR

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APA (6th Edition):

Boudra, R. (2015). Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology. (Doctoral Dissertation). Université Blaise-Pascale, Clermont-Ferrand II. Retrieved from http://www.theses.fr/2015CLF22598

Chicago Manual of Style (16th Edition):

Boudra, Rafik. “Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology.” 2015. Doctoral Dissertation, Université Blaise-Pascale, Clermont-Ferrand II. Accessed October 29, 2020. http://www.theses.fr/2015CLF22598.

MLA Handbook (7th Edition):

Boudra, Rafik. “Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology.” 2015. Web. 29 Oct 2020.

Vancouver:

Boudra R. Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology. [Internet] [Doctoral dissertation]. Université Blaise-Pascale, Clermont-Ferrand II; 2015. [cited 2020 Oct 29]. Available from: http://www.theses.fr/2015CLF22598.

Council of Science Editors:

Boudra R. Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology. [Doctoral Dissertation]. Université Blaise-Pascale, Clermont-Ferrand II; 2015. Available from: http://www.theses.fr/2015CLF22598

18. Longvert, Christine. Rôle de NRAS et PTEN au cours de la mélanomagenèse : NRAS and role during the PTEN melanomagenese.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2012, Université Paris Descartes – Paris V

La mélanomagenèse est un processus complexe sous-tendu par des mécanismes cellulaires et moléculaires variés. L’ensemble de ces mécanismes moléculaires est impliqué dans les réseaux moléculaires… (more)

Subjects/Keywords: NRAS; PTEN; Mélanome; Inititation; Sénescence; NRAS; PTEN; Melanoma; Initiation; Senescence

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APA (6th Edition):

Longvert, C. (2012). Rôle de NRAS et PTEN au cours de la mélanomagenèse : NRAS and role during the PTEN melanomagenese. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2012PA05T047

Chicago Manual of Style (16th Edition):

Longvert, Christine. “Rôle de NRAS et PTEN au cours de la mélanomagenèse : NRAS and role during the PTEN melanomagenese.” 2012. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed October 29, 2020. http://www.theses.fr/2012PA05T047.

MLA Handbook (7th Edition):

Longvert, Christine. “Rôle de NRAS et PTEN au cours de la mélanomagenèse : NRAS and role during the PTEN melanomagenese.” 2012. Web. 29 Oct 2020.

Vancouver:

Longvert C. Rôle de NRAS et PTEN au cours de la mélanomagenèse : NRAS and role during the PTEN melanomagenese. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2012. [cited 2020 Oct 29]. Available from: http://www.theses.fr/2012PA05T047.

Council of Science Editors:

Longvert C. Rôle de NRAS et PTEN au cours de la mélanomagenèse : NRAS and role during the PTEN melanomagenese. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2012. Available from: http://www.theses.fr/2012PA05T047


NSYSU

19. HSIN, HUNG. Genetically Engineered Mouse Model Recapitulating LKB1 and PTEN Loss In Gastric Cancer.

Degree: Master, Institute of Biomedical Sciences, 2016, NSYSU

 Gastric cancer (GC) is an aggressive disease with the highest rate of mortality among cancers and is the second leading cause of cancer death worldwide.… (more)

Subjects/Keywords: mouse model; PTEN; LKB1; Gastric cancer; adenocarcinoma

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APA (6th Edition):

HSIN, H. (2016). Genetically Engineered Mouse Model Recapitulating LKB1 and PTEN Loss In Gastric Cancer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-104605

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

HSIN, HUNG. “Genetically Engineered Mouse Model Recapitulating LKB1 and PTEN Loss In Gastric Cancer.” 2016. Thesis, NSYSU. Accessed October 29, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-104605.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

HSIN, HUNG. “Genetically Engineered Mouse Model Recapitulating LKB1 and PTEN Loss In Gastric Cancer.” 2016. Web. 29 Oct 2020.

Vancouver:

HSIN H. Genetically Engineered Mouse Model Recapitulating LKB1 and PTEN Loss In Gastric Cancer. [Internet] [Thesis]. NSYSU; 2016. [cited 2020 Oct 29]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-104605.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

HSIN H. Genetically Engineered Mouse Model Recapitulating LKB1 and PTEN Loss In Gastric Cancer. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-104605

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. 和田, 英雄. Expression of Somatostatin Receptor Type 2A and PTEN in Neuroendocrine Neoplasms Is Associated with Tumor Grade but Not with Site of Origin : 神経内分泌腫瘍のソマトスタチン受容体2A型とPTENの発現は異型度と関連するが発生部位とは関連しない.

Degree: 博士(医学), 2016, Nagasaki University / 長崎大学

 Neuroendocrine neoplasms (NENs) are derived from endocrine cells in various organs and share common morphological features. This study aimed to clarify whether NENs of different… (more)

Subjects/Keywords: Neuroendocrine neoplasm; PTEN; SSTR2A; p53; FISH; ddPCR

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APA (6th Edition):

和田, . (2016). Expression of Somatostatin Receptor Type 2A and PTEN in Neuroendocrine Neoplasms Is Associated with Tumor Grade but Not with Site of Origin : 神経内分泌腫瘍のソマトスタチン受容体2A型とPTENの発現は異型度と関連するが発生部位とは関連しない. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/37822

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

和田, 英雄. “Expression of Somatostatin Receptor Type 2A and PTEN in Neuroendocrine Neoplasms Is Associated with Tumor Grade but Not with Site of Origin : 神経内分泌腫瘍のソマトスタチン受容体2A型とPTENの発現は異型度と関連するが発生部位とは関連しない.” 2016. Thesis, Nagasaki University / 長崎大学. Accessed October 29, 2020. http://hdl.handle.net/10069/37822.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

和田, 英雄. “Expression of Somatostatin Receptor Type 2A and PTEN in Neuroendocrine Neoplasms Is Associated with Tumor Grade but Not with Site of Origin : 神経内分泌腫瘍のソマトスタチン受容体2A型とPTENの発現は異型度と関連するが発生部位とは関連しない.” 2016. Web. 29 Oct 2020.

Vancouver:

和田 . Expression of Somatostatin Receptor Type 2A and PTEN in Neuroendocrine Neoplasms Is Associated with Tumor Grade but Not with Site of Origin : 神経内分泌腫瘍のソマトスタチン受容体2A型とPTENの発現は異型度と関連するが発生部位とは関連しない. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2016. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/10069/37822.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

和田 . Expression of Somatostatin Receptor Type 2A and PTEN in Neuroendocrine Neoplasms Is Associated with Tumor Grade but Not with Site of Origin : 神経内分泌腫瘍のソマトスタチン受容体2A型とPTENの発現は異型度と関連するが発生部位とは関連しない. [Thesis]. Nagasaki University / 長崎大学; 2016. Available from: http://hdl.handle.net/10069/37822

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Saskatchewan

21. Wu, Qi. MOLECULAR PATHOGENESIS OF HEPATITIS B VIRUS AND HEPATITIS C VIRUS INFECTIONS.

Degree: 2017, University of Saskatchewan

 Hepatitis B virus (HBV) and hepatitis C virus (HCV) infections cause a wide range of liver diseases including hepatocellular carcinoma (HCC) worldwide. Because these two… (more)

Subjects/Keywords: HBV; HCV; HBx; HCV core; PTEN; SREBP

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APA (6th Edition):

Wu, Q. (2017). MOLECULAR PATHOGENESIS OF HEPATITIS B VIRUS AND HEPATITIS C VIRUS INFECTIONS. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/7984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Qi. “MOLECULAR PATHOGENESIS OF HEPATITIS B VIRUS AND HEPATITIS C VIRUS INFECTIONS.” 2017. Thesis, University of Saskatchewan. Accessed October 29, 2020. http://hdl.handle.net/10388/7984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Qi. “MOLECULAR PATHOGENESIS OF HEPATITIS B VIRUS AND HEPATITIS C VIRUS INFECTIONS.” 2017. Web. 29 Oct 2020.

Vancouver:

Wu Q. MOLECULAR PATHOGENESIS OF HEPATITIS B VIRUS AND HEPATITIS C VIRUS INFECTIONS. [Internet] [Thesis]. University of Saskatchewan; 2017. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/10388/7984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu Q. MOLECULAR PATHOGENESIS OF HEPATITIS B VIRUS AND HEPATITIS C VIRUS INFECTIONS. [Thesis]. University of Saskatchewan; 2017. Available from: http://hdl.handle.net/10388/7984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

22. Spinella, Anthony J. In vivo characterization of hippocampal electrophysiological processes in the heterozygous Pten knockout model of autism.

Degree: Neuroscience, 2018, University of Vermont

  While cognitive deficits have been described in the heterozygous Pten (+/-) KO mouse model of autism, little work has been done to demonstrate how… (more)

Subjects/Keywords: Pten; Autism; CA1; Place Cells; in vivo

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APA (6th Edition):

Spinella, A. J. (2018). In vivo characterization of hippocampal electrophysiological processes in the heterozygous Pten knockout model of autism. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/258

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Spinella, Anthony J. “In vivo characterization of hippocampal electrophysiological processes in the heterozygous Pten knockout model of autism.” 2018. Thesis, University of Vermont. Accessed October 29, 2020. https://scholarworks.uvm.edu/hcoltheses/258.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Spinella, Anthony J. “In vivo characterization of hippocampal electrophysiological processes in the heterozygous Pten knockout model of autism.” 2018. Web. 29 Oct 2020.

Vancouver:

Spinella AJ. In vivo characterization of hippocampal electrophysiological processes in the heterozygous Pten knockout model of autism. [Internet] [Thesis]. University of Vermont; 2018. [cited 2020 Oct 29]. Available from: https://scholarworks.uvm.edu/hcoltheses/258.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Spinella AJ. In vivo characterization of hippocampal electrophysiological processes in the heterozygous Pten knockout model of autism. [Thesis]. University of Vermont; 2018. Available from: https://scholarworks.uvm.edu/hcoltheses/258

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Newcastle

23. Guo, Su Tang. The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer.

Degree: PhD, 2018, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Colon cancer is one of the most common and deadly malignancies. Despite recent advances in early diagnosis and… (more)

Subjects/Keywords: INPP4B; colon cancer; pten; transgenic mice

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APA (6th Edition):

Guo, S. T. (2018). The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1386333

Chicago Manual of Style (16th Edition):

Guo, Su Tang. “The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer.” 2018. Doctoral Dissertation, University of Newcastle. Accessed October 29, 2020. http://hdl.handle.net/1959.13/1386333.

MLA Handbook (7th Edition):

Guo, Su Tang. “The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer.” 2018. Web. 29 Oct 2020.

Vancouver:

Guo ST. The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer. [Internet] [Doctoral dissertation]. University of Newcastle; 2018. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1959.13/1386333.

Council of Science Editors:

Guo ST. The role of inositol polyphosphate 4-phosphatase II (INPP4B) in the pathogeneis of colon ccancer. [Doctoral Dissertation]. University of Newcastle; 2018. Available from: http://hdl.handle.net/1959.13/1386333


University of Toronto

24. Yin-Liao, May. Characterization of ATM-mediated Phosphorylation of PTEN.

Degree: 2019, University of Toronto

Phosphatase and tensin homolog (PTEN) is a tumour suppressor gene that is commonly lost in many human cancers, such as glioblastoma, prostate, and breast. PTEN’s… (more)

Subjects/Keywords: ATM; phosphorylation; PTEN; radiation; subcellular localization; 0487

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APA (6th Edition):

Yin-Liao, M. (2019). Characterization of ATM-mediated Phosphorylation of PTEN. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/98439

Chicago Manual of Style (16th Edition):

Yin-Liao, May. “Characterization of ATM-mediated Phosphorylation of PTEN.” 2019. Masters Thesis, University of Toronto. Accessed October 29, 2020. http://hdl.handle.net/1807/98439.

MLA Handbook (7th Edition):

Yin-Liao, May. “Characterization of ATM-mediated Phosphorylation of PTEN.” 2019. Web. 29 Oct 2020.

Vancouver:

Yin-Liao M. Characterization of ATM-mediated Phosphorylation of PTEN. [Internet] [Masters thesis]. University of Toronto; 2019. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1807/98439.

Council of Science Editors:

Yin-Liao M. Characterization of ATM-mediated Phosphorylation of PTEN. [Masters Thesis]. University of Toronto; 2019. Available from: http://hdl.handle.net/1807/98439


Univerzitet u Beogradu

25. Ursulović, Tamara, 1969-. Povezanost ekspresije PTEN proteina i prognoze bolesti kod žena obolelih od ranog karcinoma dojke lečenih ovarijalnom ablacijom.

Degree: Medicinski fakultet, 2019, Univerzitet u Beogradu

medicina - onkologija / medicine - oncology

Sve pacijentkinje sa detektabilnom ekspresijom hormonskih receptora (HR) za karcinom dojke (KD) trebalo bi da se leče endokrinom… (more)

Subjects/Keywords: early breast cancer; hormone receptors; PTEN

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APA (6th Edition):

Ursulović, Tamara, 1. (2019). Povezanost ekspresije PTEN proteina i prognoze bolesti kod žena obolelih od ranog karcinoma dojke lečenih ovarijalnom ablacijom. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:19402/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ursulović, Tamara, 1969-. “Povezanost ekspresije PTEN proteina i prognoze bolesti kod žena obolelih od ranog karcinoma dojke lečenih ovarijalnom ablacijom.” 2019. Thesis, Univerzitet u Beogradu. Accessed October 29, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:19402/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ursulović, Tamara, 1969-. “Povezanost ekspresije PTEN proteina i prognoze bolesti kod žena obolelih od ranog karcinoma dojke lečenih ovarijalnom ablacijom.” 2019. Web. 29 Oct 2020.

Vancouver:

Ursulović, Tamara 1. Povezanost ekspresije PTEN proteina i prognoze bolesti kod žena obolelih od ranog karcinoma dojke lečenih ovarijalnom ablacijom. [Internet] [Thesis]. Univerzitet u Beogradu; 2019. [cited 2020 Oct 29]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:19402/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ursulović, Tamara 1. Povezanost ekspresije PTEN proteina i prognoze bolesti kod žena obolelih od ranog karcinoma dojke lečenih ovarijalnom ablacijom. [Thesis]. Univerzitet u Beogradu; 2019. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:19402/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

26. Wozniak, Darren J. Molecular Regulation of Protein Tyrosine Kinase 6 in Prostate Cancer.

Degree: 2017, University of Illinois – Chicago

 Protein tyrosine kinase 6 (PTK6) is an intracellular tyrosine kinase that is oncogenic when activated at the plasma membrane. Activation of PTK6 at the plasma… (more)

Subjects/Keywords: Prostate cancer; tyrosine kinase; phosphatase; PTK6; PTEN

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wozniak, D. J. (2017). Molecular Regulation of Protein Tyrosine Kinase 6 in Prostate Cancer. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21969

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wozniak, Darren J. “Molecular Regulation of Protein Tyrosine Kinase 6 in Prostate Cancer.” 2017. Thesis, University of Illinois – Chicago. Accessed October 29, 2020. http://hdl.handle.net/10027/21969.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wozniak, Darren J. “Molecular Regulation of Protein Tyrosine Kinase 6 in Prostate Cancer.” 2017. Web. 29 Oct 2020.

Vancouver:

Wozniak DJ. Molecular Regulation of Protein Tyrosine Kinase 6 in Prostate Cancer. [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/10027/21969.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wozniak DJ. Molecular Regulation of Protein Tyrosine Kinase 6 in Prostate Cancer. [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/21969

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

27. Wang, Sharon. Cooperating Oncogenic Alterations in Aggressive Pten-Deficient Breast Cancer.

Degree: PhD, 2017, University of Toronto

PTEN is one of the most frequently inactivated tumor suppressors in human malignancies including breast cancer, and its mutation or loss is often implicated in… (more)

Subjects/Keywords: Breast; Mouse model; p53; PTEN; 0992

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, S. (2017). Cooperating Oncogenic Alterations in Aggressive Pten-Deficient Breast Cancer. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/91193

Chicago Manual of Style (16th Edition):

Wang, Sharon. “Cooperating Oncogenic Alterations in Aggressive Pten-Deficient Breast Cancer.” 2017. Doctoral Dissertation, University of Toronto. Accessed October 29, 2020. http://hdl.handle.net/1807/91193.

MLA Handbook (7th Edition):

Wang, Sharon. “Cooperating Oncogenic Alterations in Aggressive Pten-Deficient Breast Cancer.” 2017. Web. 29 Oct 2020.

Vancouver:

Wang S. Cooperating Oncogenic Alterations in Aggressive Pten-Deficient Breast Cancer. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1807/91193.

Council of Science Editors:

Wang S. Cooperating Oncogenic Alterations in Aggressive Pten-Deficient Breast Cancer. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/91193


University of Toronto

28. Bassi, Christian. Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress.

Degree: PhD, 2015, University of Toronto

 Loss of function of the phosphatase and tensin homolog (PTEN) tumor suppressor is frequently found in many human malignancies. PTEN antagonizes the Phosphatidylinositide 3-kinase (PI3K)… (more)

Subjects/Keywords: Cancer; DNA damage; PTEN; tumor suppressor; 0307

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bassi, C. (2015). Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/69214

Chicago Manual of Style (16th Edition):

Bassi, Christian. “Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress.” 2015. Doctoral Dissertation, University of Toronto. Accessed October 29, 2020. http://hdl.handle.net/1807/69214.

MLA Handbook (7th Edition):

Bassi, Christian. “Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress.” 2015. Web. 29 Oct 2020.

Vancouver:

Bassi C. Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2020 Oct 29]. Available from: http://hdl.handle.net/1807/69214.

Council of Science Editors:

Bassi C. Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/69214


University of Southern California

29. Galicia Medina, Vivian A. PTEN deletion induced tumor initiating cells: Strategies to accelerate the disease progression of liver cancer.

Degree: PhD, Systems Biology & Disease, 2011, University of Southern California

 Progenitor or tumor initiating cells (TICs) are “altered” stem cells with the capacity to form solid tumors. Tumor suppressor PTEN (phosphatase and tensin homologue deleted… (more)

Subjects/Keywords: Pten; tumor initiating cells; liver cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Galicia Medina, V. A. (2011). PTEN deletion induced tumor initiating cells: Strategies to accelerate the disease progression of liver cancer. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/470211/rec/5322

Chicago Manual of Style (16th Edition):

Galicia Medina, Vivian A. “PTEN deletion induced tumor initiating cells: Strategies to accelerate the disease progression of liver cancer.” 2011. Doctoral Dissertation, University of Southern California. Accessed October 29, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/470211/rec/5322.

MLA Handbook (7th Edition):

Galicia Medina, Vivian A. “PTEN deletion induced tumor initiating cells: Strategies to accelerate the disease progression of liver cancer.” 2011. Web. 29 Oct 2020.

Vancouver:

Galicia Medina VA. PTEN deletion induced tumor initiating cells: Strategies to accelerate the disease progression of liver cancer. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2020 Oct 29]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/470211/rec/5322.

Council of Science Editors:

Galicia Medina VA. PTEN deletion induced tumor initiating cells: Strategies to accelerate the disease progression of liver cancer. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/470211/rec/5322


University of Southern California

30. Li, Yang. Regulation of mitochondrial bioenergetics via PTEN (phosphatase and tensin homolog deleted on chromosome 10)/estrogen-related receptor alpha (ERRα) signaling.

Degree: PhD, Molecular Pharmacology and Toxicology, 2014, University of Southern California

 Mitochondrial abnormalities are associated with cancer development, yet how oncogenic signals affect mitochondrial function has not been fully understood. The purpose of the current study… (more)

Subjects/Keywords: mitochondria; PTEN; ERR&alpha;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, Y. (2014). Regulation of mitochondrial bioenergetics via PTEN (phosphatase and tensin homolog deleted on chromosome 10)/estrogen-related receptor alpha (ERRα) signaling. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/508341/rec/5499

Chicago Manual of Style (16th Edition):

Li, Yang. “Regulation of mitochondrial bioenergetics via PTEN (phosphatase and tensin homolog deleted on chromosome 10)/estrogen-related receptor alpha (ERRα) signaling.” 2014. Doctoral Dissertation, University of Southern California. Accessed October 29, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/508341/rec/5499.

MLA Handbook (7th Edition):

Li, Yang. “Regulation of mitochondrial bioenergetics via PTEN (phosphatase and tensin homolog deleted on chromosome 10)/estrogen-related receptor alpha (ERRα) signaling.” 2014. Web. 29 Oct 2020.

Vancouver:

Li Y. Regulation of mitochondrial bioenergetics via PTEN (phosphatase and tensin homolog deleted on chromosome 10)/estrogen-related receptor alpha (ERRα) signaling. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2020 Oct 29]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/508341/rec/5499.

Council of Science Editors:

Li Y. Regulation of mitochondrial bioenergetics via PTEN (phosphatase and tensin homolog deleted on chromosome 10)/estrogen-related receptor alpha (ERRα) signaling. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/508341/rec/5499

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