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You searched for subject:(PRKAR1A). Showing records 1 – 9 of 9 total matches.

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University of Guelph

1. Larsen, Dana Meegan. Molecular Determinants of Malignancy in Canine Osteosarcoma.

Degree: Doctor of Veterinary Science, Department of Pathobiology, 2011, University of Guelph

 Cancer is essentially a genetic disease of uncontrolled cell survival and proliferation. Medical therapy has traditionally involved chemotherapy and radiation which inhibit the replication of… (more)

Subjects/Keywords: canine osteosarcoma; IGF2; PRKAR1A

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APA (6th Edition):

Larsen, D. M. (2011). Molecular Determinants of Malignancy in Canine Osteosarcoma. (Doctoral Dissertation). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3186

Chicago Manual of Style (16th Edition):

Larsen, Dana Meegan. “Molecular Determinants of Malignancy in Canine Osteosarcoma.” 2011. Doctoral Dissertation, University of Guelph. Accessed April 17, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3186.

MLA Handbook (7th Edition):

Larsen, Dana Meegan. “Molecular Determinants of Malignancy in Canine Osteosarcoma.” 2011. Web. 17 Apr 2021.

Vancouver:

Larsen DM. Molecular Determinants of Malignancy in Canine Osteosarcoma. [Internet] [Doctoral dissertation]. University of Guelph; 2011. [cited 2021 Apr 17]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3186.

Council of Science Editors:

Larsen DM. Molecular Determinants of Malignancy in Canine Osteosarcoma. [Doctoral Dissertation]. University of Guelph; 2011. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/3186


University of Guelph

2. Wong, Jeremy. Autophagy in siRNA-mediated PRKAR1A knockdown canine osteosarcoma cells.

Degree: MS, Department of Pathobiology, 2016, University of Guelph

 Canine Osteosarcoma (OSA) is a highly malignant form of bone cancer with a mean survival time of only one year even with aggressive treatment. The… (more)

Subjects/Keywords: PRKAR1A; Autophagy; Canine osteosarcoma; Osteosarcoma; Canine cancer; Chemosensitivity; Chemoresistance

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APA (6th Edition):

Wong, J. (2016). Autophagy in siRNA-mediated PRKAR1A knockdown canine osteosarcoma cells. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10047

Chicago Manual of Style (16th Edition):

Wong, Jeremy. “Autophagy in siRNA-mediated PRKAR1A knockdown canine osteosarcoma cells.” 2016. Masters Thesis, University of Guelph. Accessed April 17, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10047.

MLA Handbook (7th Edition):

Wong, Jeremy. “Autophagy in siRNA-mediated PRKAR1A knockdown canine osteosarcoma cells.” 2016. Web. 17 Apr 2021.

Vancouver:

Wong J. Autophagy in siRNA-mediated PRKAR1A knockdown canine osteosarcoma cells. [Internet] [Masters thesis]. University of Guelph; 2016. [cited 2021 Apr 17]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10047.

Council of Science Editors:

Wong J. Autophagy in siRNA-mediated PRKAR1A knockdown canine osteosarcoma cells. [Masters Thesis]. University of Guelph; 2016. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10047

3. Tsigginou, Alexandra. Διαταραχές της επαγωγής σήματος της εξαρτώμενης από το cAMP πρωτεϊνικής κινάσης στον καρκίνο του ενδομητρίου.

Degree: 2013, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Background: The PRKAR1A gene is coding for the type 1α regulatory subunit of the cAMP-dependent protein kinase A (PKA), an enzyme with important role in… (more)

Subjects/Keywords: Πρωτεϊνική κινάση Α; Καρκίνος ενδομητρίου; Διαταραχή σήματος; Σήμα cAMP; Μετάλλαξη PRKAR1A; RIa ρυθμιστική υπομονάδα; Protein kinase A (PKA); Endometrial cancer; Signaling dysregulation; cAMP signaling; PRKAR1A mutation; RIa regulatory subunit

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APA (6th Edition):

Tsigginou, A. (2013). Διαταραχές της επαγωγής σήματος της εξαρτώμενης από το cAMP πρωτεϊνικής κινάσης στον καρκίνο του ενδομητρίου. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/41214

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsigginou, Alexandra. “Διαταραχές της επαγωγής σήματος της εξαρτώμενης από το cAMP πρωτεϊνικής κινάσης στον καρκίνο του ενδομητρίου.” 2013. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 17, 2021. http://hdl.handle.net/10442/hedi/41214.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsigginou, Alexandra. “Διαταραχές της επαγωγής σήματος της εξαρτώμενης από το cAMP πρωτεϊνικής κινάσης στον καρκίνο του ενδομητρίου.” 2013. Web. 17 Apr 2021.

Vancouver:

Tsigginou A. Διαταραχές της επαγωγής σήματος της εξαρτώμενης από το cAMP πρωτεϊνικής κινάσης στον καρκίνο του ενδομητρίου. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10442/hedi/41214.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsigginou A. Διαταραχές της επαγωγής σήματος της εξαρτώμενης από το cAMP πρωτεϊνικής κινάσης στον καρκίνο του ενδομητρίου. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. Available from: http://hdl.handle.net/10442/hedi/41214

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

4. Muhn, Florian. Novel mutations of the PRKAR1A gene in patients with acrodysostosis and brachydactyly type E.

Degree: 2014, Freie Universität Berlin

 Acrodysostosis is characterized by a peripheral dysostosis that is accompanied by short stature, midface hypoplasia, and developmental delay. Recently, it was shown that heterozygous point… (more)

Subjects/Keywords: acrodysostosis; brachydactyly; PRKAR1A; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Muhn, F. (2014). Novel mutations of the PRKAR1A gene in patients with acrodysostosis and brachydactyly type E. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/12230

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Muhn, Florian. “Novel mutations of the PRKAR1A gene in patients with acrodysostosis and brachydactyly type E.” 2014. Thesis, Freie Universität Berlin. Accessed April 17, 2021. https://refubium.fu-berlin.de/handle/fub188/12230.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Muhn, Florian. “Novel mutations of the PRKAR1A gene in patients with acrodysostosis and brachydactyly type E.” 2014. Web. 17 Apr 2021.

Vancouver:

Muhn F. Novel mutations of the PRKAR1A gene in patients with acrodysostosis and brachydactyly type E. [Internet] [Thesis]. Freie Universität Berlin; 2014. [cited 2021 Apr 17]. Available from: https://refubium.fu-berlin.de/handle/fub188/12230.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Muhn F. Novel mutations of the PRKAR1A gene in patients with acrodysostosis and brachydactyly type E. [Thesis]. Freie Universität Berlin; 2014. Available from: https://refubium.fu-berlin.de/handle/fub188/12230

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Motte-Signoret, Emmanuelle. Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene).

Degree: Docteur es, Biologie cellulaire et moléculaire, 2016, Sorbonne Paris Cité

 L’acrodysostose est une chondrodysplasie associant une petite taille et des anomalies des extrémités et de la face. Les mécanismes moléculaires sous-jacents ont récemment été identifiés,… (more)

Subjects/Keywords: Acrodysostose; PDE4D; PRKAR1A; Pseudo-hypoparathyroïdie; GNAS; AMP cyclique; Compartimentalisation cellulaire; Résistance hormonale; Cellular and molecular biology; 576.5

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APA (6th Edition):

Motte-Signoret, E. (2016). Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene). (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2016USPCB098

Chicago Manual of Style (16th Edition):

Motte-Signoret, Emmanuelle. “Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene).” 2016. Doctoral Dissertation, Sorbonne Paris Cité. Accessed April 17, 2021. http://www.theses.fr/2016USPCB098.

MLA Handbook (7th Edition):

Motte-Signoret, Emmanuelle. “Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene).” 2016. Web. 17 Apr 2021.

Vancouver:

Motte-Signoret E. Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene). [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2016. [cited 2021 Apr 17]. Available from: http://www.theses.fr/2016USPCB098.

Council of Science Editors:

Motte-Signoret E. Exploration fonctionnelle in vitro des mécanismes à l'origine de l'acrodysostose sans résistance hormonale par mutation du gène de la PDE4D : comparaison avec l'acrodysostose et résistance plurihormonale (mutations du gène PRKAR1A) et la pseudo-hypoparathyroïdie 1a (mutations du gène GNAS) : In vitro functional exploration of mechanisms causing acrodysostosis without hormonal resistance by mutation of the PDE4D gene : comparison with acrodysostosis and multi-hormonal resistance (mutations of the PRKAR1A gene) and pseudo-hypoparathyroidism 1a (mutations in the GNAS gene). [Doctoral Dissertation]. Sorbonne Paris Cité; 2016. Available from: http://www.theses.fr/2016USPCB098

6. Silvia Ferreira de Sousa. Análise molecular do gene prkar1a na odontogênese e nos tumores odontogênicos mistos.

Degree: 2014, Universidade Federal de Minas Gerais; UFMG

Exportado OPUS

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Os tumores… (more)

Subjects/Keywords: Odontogênese; situ; PRKAR1A; Hibridização in; Patologia molecular; Tumores odontogênicos mistos; Odontogênese; Hibridização in situ; Tumores odontogênicos; Patologia molecular

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APA (6th Edition):

Sousa, S. F. d. (2014). Análise molecular do gene prkar1a na odontogênese e nos tumores odontogênicos mistos. (Doctoral Dissertation). Universidade Federal de Minas Gerais; UFMG. Retrieved from http://hdl.handle.net/1843/BUBD-9ZCN2H

Chicago Manual of Style (16th Edition):

Sousa, Silvia Ferreira de. “Análise molecular do gene prkar1a na odontogênese e nos tumores odontogênicos mistos.” 2014. Doctoral Dissertation, Universidade Federal de Minas Gerais; UFMG. Accessed April 17, 2021. http://hdl.handle.net/1843/BUBD-9ZCN2H.

MLA Handbook (7th Edition):

Sousa, Silvia Ferreira de. “Análise molecular do gene prkar1a na odontogênese e nos tumores odontogênicos mistos.” 2014. Web. 17 Apr 2021.

Vancouver:

Sousa SFd. Análise molecular do gene prkar1a na odontogênese e nos tumores odontogênicos mistos. [Internet] [Doctoral dissertation]. Universidade Federal de Minas Gerais; UFMG; 2014. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1843/BUBD-9ZCN2H.

Council of Science Editors:

Sousa SFd. Análise molecular do gene prkar1a na odontogênese e nos tumores odontogênicos mistos. [Doctoral Dissertation]. Universidade Federal de Minas Gerais; UFMG; 2014. Available from: http://hdl.handle.net/1843/BUBD-9ZCN2H

7. Paolla Freitas Perdigao. Mecanismos moleculares associados a etiopatogênese dos tumores odontogênicos.

Degree: 2005, Universidade Federal de Minas Gerais; UFMG

Exportado OPUS

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As neoplasias… (more)

Subjects/Keywords: PRKAR1A; Tumores odontogênicos; Ameloblastina; Mixoma; Farmacologia; Tumores odontogênicos; Tumores odontogênicos Aspectos moleculares

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APA (6th Edition):

Perdigao, P. F. (2005). Mecanismos moleculares associados a etiopatogênese dos tumores odontogênicos. (Doctoral Dissertation). Universidade Federal de Minas Gerais; UFMG. Retrieved from http://hdl.handle.net/1843/SMOC-7JAJEW

Chicago Manual of Style (16th Edition):

Perdigao, Paolla Freitas. “Mecanismos moleculares associados a etiopatogênese dos tumores odontogênicos.” 2005. Doctoral Dissertation, Universidade Federal de Minas Gerais; UFMG. Accessed April 17, 2021. http://hdl.handle.net/1843/SMOC-7JAJEW.

MLA Handbook (7th Edition):

Perdigao, Paolla Freitas. “Mecanismos moleculares associados a etiopatogênese dos tumores odontogênicos.” 2005. Web. 17 Apr 2021.

Vancouver:

Perdigao PF. Mecanismos moleculares associados a etiopatogênese dos tumores odontogênicos. [Internet] [Doctoral dissertation]. Universidade Federal de Minas Gerais; UFMG; 2005. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1843/SMOC-7JAJEW.

Council of Science Editors:

Perdigao PF. Mecanismos moleculares associados a etiopatogênese dos tumores odontogênicos. [Doctoral Dissertation]. Universidade Federal de Minas Gerais; UFMG; 2005. Available from: http://hdl.handle.net/1843/SMOC-7JAJEW


Universitetet i Tromsø

8. Lamsal, Apsana. Study of human PRKAR1A and its role in autophagy .

Degree: 2018, Universitetet i Tromsø

 The human PRKAR1A gene is a 381- amino acid protein encoding the regulatory subunit (RIα) of the cAMP dependent Protein Kinase A (PKA). The main… (more)

Subjects/Keywords: PRKAR1A/Autophagy; VDP::Medical disciplines: 700; VDP::Medisinske Fag: 700; VDP::Medical disciplines: 700::Basic medical, dental and veterinary science disciplines: 710::Medical molecular biology: 711; VDP::Medisinske Fag: 700::Basale medisinske, odontologiske og veterinærmedisinske fag: 710::Medisinsk molekylærbiologi: 711

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APA (6th Edition):

Lamsal, A. (2018). Study of human PRKAR1A and its role in autophagy . (Masters Thesis). Universitetet i Tromsø. Retrieved from http://hdl.handle.net/10037/18257

Chicago Manual of Style (16th Edition):

Lamsal, Apsana. “Study of human PRKAR1A and its role in autophagy .” 2018. Masters Thesis, Universitetet i Tromsø. Accessed April 17, 2021. http://hdl.handle.net/10037/18257.

MLA Handbook (7th Edition):

Lamsal, Apsana. “Study of human PRKAR1A and its role in autophagy .” 2018. Web. 17 Apr 2021.

Vancouver:

Lamsal A. Study of human PRKAR1A and its role in autophagy . [Internet] [Masters thesis]. Universitetet i Tromsø 2018. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10037/18257.

Council of Science Editors:

Lamsal A. Study of human PRKAR1A and its role in autophagy . [Masters Thesis]. Universitetet i Tromsø 2018. Available from: http://hdl.handle.net/10037/18257


The Ohio State University

9. Jones, Georgette Nicole. Dysregulated PKA Activity Leads to Defective Neural Crest Differentiation and Schwann Cell Tumorigenesis.

Degree: PhD, Molecular, Cellular, and Developmental Biology, 2009, The Ohio State University

  Proper regulation of cAMP-dependent protein kinase (PKA) is essential to normal cell proliferation, but PKA dysregulation leads to tumor formation in a range of… (more)

Subjects/Keywords: Biology; Cellular Biology; Molecular Biology; PKA; Neurofibromatosis; Schwann cell; schwannoma; neural crest; Prkar1a; Carney Complex

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APA (6th Edition):

Jones, G. N. (2009). Dysregulated PKA Activity Leads to Defective Neural Crest Differentiation and Schwann Cell Tumorigenesis. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1262036157

Chicago Manual of Style (16th Edition):

Jones, Georgette Nicole. “Dysregulated PKA Activity Leads to Defective Neural Crest Differentiation and Schwann Cell Tumorigenesis.” 2009. Doctoral Dissertation, The Ohio State University. Accessed April 17, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1262036157.

MLA Handbook (7th Edition):

Jones, Georgette Nicole. “Dysregulated PKA Activity Leads to Defective Neural Crest Differentiation and Schwann Cell Tumorigenesis.” 2009. Web. 17 Apr 2021.

Vancouver:

Jones GN. Dysregulated PKA Activity Leads to Defective Neural Crest Differentiation and Schwann Cell Tumorigenesis. [Internet] [Doctoral dissertation]. The Ohio State University; 2009. [cited 2021 Apr 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1262036157.

Council of Science Editors:

Jones GN. Dysregulated PKA Activity Leads to Defective Neural Crest Differentiation and Schwann Cell Tumorigenesis. [Doctoral Dissertation]. The Ohio State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1262036157

.