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You searched for subject:(PPARgamma). Showing records 1 – 30 of 35 total matches.

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Case Western Reserve University

1. Mandrekar-Colucci, Shweta Dilip. PPAR¿ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease.

Degree: PhD, Neurosciences, 2011, Case Western Reserve University

  Alzheimer’s disease (AD) is a chronic neurodegenerative disease characterized by the progressive loss of cognition and memory. The pathological hallmarks of AD include extracellular… (more)

Subjects/Keywords: Neurosciences; Alzheimer's Disease; Microglia; PPARgamma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mandrekar-Colucci, S. D. (2011). PPAR¿ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1307730357

Chicago Manual of Style (16th Edition):

Mandrekar-Colucci, Shweta Dilip. “PPAR¿ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease.” 2011. Doctoral Dissertation, Case Western Reserve University. Accessed April 02, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1307730357.

MLA Handbook (7th Edition):

Mandrekar-Colucci, Shweta Dilip. “PPAR¿ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease.” 2011. Web. 02 Apr 2020.

Vancouver:

Mandrekar-Colucci SD. PPAR¿ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2011. [cited 2020 Apr 02]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1307730357.

Council of Science Editors:

Mandrekar-Colucci SD. PPAR¿ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease. [Doctoral Dissertation]. Case Western Reserve University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1307730357


Wayne State University

2. Kadam, Leena. The Role Of Pparγ In Placental Development And Disease.

Degree: PhD, Physiology, 2017, Wayne State University

  The placenta in mammals forms the maternal fetal interface serving as the source of nutrition for the fetus throughout gestation. It comprises of two… (more)

Subjects/Keywords: Inflammation; Placenta; PPARgamma; Trophoblast; Physiology

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APA (6th Edition):

Kadam, L. (2017). The Role Of Pparγ In Placental Development And Disease. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1818

Chicago Manual of Style (16th Edition):

Kadam, Leena. “The Role Of Pparγ In Placental Development And Disease.” 2017. Doctoral Dissertation, Wayne State University. Accessed April 02, 2020. https://digitalcommons.wayne.edu/oa_dissertations/1818.

MLA Handbook (7th Edition):

Kadam, Leena. “The Role Of Pparγ In Placental Development And Disease.” 2017. Web. 02 Apr 2020.

Vancouver:

Kadam L. The Role Of Pparγ In Placental Development And Disease. [Internet] [Doctoral dissertation]. Wayne State University; 2017. [cited 2020 Apr 02]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1818.

Council of Science Editors:

Kadam L. The Role Of Pparγ In Placental Development And Disease. [Doctoral Dissertation]. Wayne State University; 2017. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1818


Temple University

3. Evans, Kyle William. PPAR gamma AND eNOS CONTRIBUTE TO THE RESOLUTION OF CHRONIC INFLAMMATION.

Degree: PhD, 2011, Temple University

Microbiology and Immunology

Chronic inflammation follows defined phases of induction, inflammation, and resolution. The resolution phase requires cycloxygenase-2 (COX-2) activity. This study aims to address… (more)

Subjects/Keywords: Biology; eNOS; inflammation; PPARgamma; resolution; rheumatoid arthrits

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APA (6th Edition):

Evans, K. W. (2011). PPAR gamma AND eNOS CONTRIBUTE TO THE RESOLUTION OF CHRONIC INFLAMMATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,197871

Chicago Manual of Style (16th Edition):

Evans, Kyle William. “PPAR gamma AND eNOS CONTRIBUTE TO THE RESOLUTION OF CHRONIC INFLAMMATION.” 2011. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,197871.

MLA Handbook (7th Edition):

Evans, Kyle William. “PPAR gamma AND eNOS CONTRIBUTE TO THE RESOLUTION OF CHRONIC INFLAMMATION.” 2011. Web. 02 Apr 2020.

Vancouver:

Evans KW. PPAR gamma AND eNOS CONTRIBUTE TO THE RESOLUTION OF CHRONIC INFLAMMATION. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,197871.

Council of Science Editors:

Evans KW. PPAR gamma AND eNOS CONTRIBUTE TO THE RESOLUTION OF CHRONIC INFLAMMATION. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,197871


Boston University

4. Su, Shi. The role of retinaldehyde and PPARgamma signaling in systemic lupus erythematosus.

Degree: 2014, Boston University

 Systemic Lupus Erythematosus (SLE) is an autoimmune disease with chronic inflammation affecting multiple organ systems, as well as accelerated atherosclerosis as a major complication. Prior… (more)

Subjects/Keywords: Biology; PPARgamma; Retinaldehyde; Systemic Lupus Erythematosus

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APA (6th Edition):

Su, S. (2014). The role of retinaldehyde and PPARgamma signaling in systemic lupus erythematosus. (Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/15078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Su, Shi. “The role of retinaldehyde and PPARgamma signaling in systemic lupus erythematosus.” 2014. Thesis, Boston University. Accessed April 02, 2020. http://hdl.handle.net/2144/15078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Su, Shi. “The role of retinaldehyde and PPARgamma signaling in systemic lupus erythematosus.” 2014. Web. 02 Apr 2020.

Vancouver:

Su S. The role of retinaldehyde and PPARgamma signaling in systemic lupus erythematosus. [Internet] [Thesis]. Boston University; 2014. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/2144/15078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Su S. The role of retinaldehyde and PPARgamma signaling in systemic lupus erythematosus. [Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/15078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

5. Skelhorne-Gross, Graham. The Stromal Role of Peroxisome Proliferator-Activated Receptor Gamma in Breast Tumourigenesis .

Degree: Pathology and Molecular Medicine, 2014, Queens University

 Breast cancer is the most commonly diagnosed female cancer, and kills 1 in 30 Canadian women, mostly from metastatic disease. Previous studies showed PPARγ decreases… (more)

Subjects/Keywords: Breast Cancer; DMBA; High-Fat Diet; PPARgamma

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APA (6th Edition):

Skelhorne-Gross, G. (2014). The Stromal Role of Peroxisome Proliferator-Activated Receptor Gamma in Breast Tumourigenesis . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/12348

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Skelhorne-Gross, Graham. “The Stromal Role of Peroxisome Proliferator-Activated Receptor Gamma in Breast Tumourigenesis .” 2014. Thesis, Queens University. Accessed April 02, 2020. http://hdl.handle.net/1974/12348.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Skelhorne-Gross, Graham. “The Stromal Role of Peroxisome Proliferator-Activated Receptor Gamma in Breast Tumourigenesis .” 2014. Web. 02 Apr 2020.

Vancouver:

Skelhorne-Gross G. The Stromal Role of Peroxisome Proliferator-Activated Receptor Gamma in Breast Tumourigenesis . [Internet] [Thesis]. Queens University; 2014. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/1974/12348.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Skelhorne-Gross G. The Stromal Role of Peroxisome Proliferator-Activated Receptor Gamma in Breast Tumourigenesis . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/12348

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Lambert, Juliette. Potentiel thérapeutique de l'activation du récepteur nucléaire PPARgamma dans la myélofibrose : Therapeutic Potential of Activation of the Nuclear Receptor PPARgamma Pathway in Myelofibrosis.

Degree: Docteur es, Sciences de la vie et de la santé, 2019, Université Paris-Saclay (ComUE)

La myélofibrose primitive (MFP) est un néoplasme myéloprolifératif (NMP) classique BCR-ABL négatif associé à une forte altération de la qualité de vie et à une… (more)

Subjects/Keywords: Myélofibrose; PPARgamma; Néoplasmes myéloprolifératifs; TGFbeta; P300; Pioglitazone; Myelofibrosis; PPARgamma; Myeloproliferative neoplasms; TGFbeta; P300; Pioglitazone

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APA (6th Edition):

Lambert, J. (2019). Potentiel thérapeutique de l'activation du récepteur nucléaire PPARgamma dans la myélofibrose : Therapeutic Potential of Activation of the Nuclear Receptor PPARgamma Pathway in Myelofibrosis. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLS536

Chicago Manual of Style (16th Edition):

Lambert, Juliette. “Potentiel thérapeutique de l'activation du récepteur nucléaire PPARgamma dans la myélofibrose : Therapeutic Potential of Activation of the Nuclear Receptor PPARgamma Pathway in Myelofibrosis.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed April 02, 2020. http://www.theses.fr/2019SACLS536.

MLA Handbook (7th Edition):

Lambert, Juliette. “Potentiel thérapeutique de l'activation du récepteur nucléaire PPARgamma dans la myélofibrose : Therapeutic Potential of Activation of the Nuclear Receptor PPARgamma Pathway in Myelofibrosis.” 2019. Web. 02 Apr 2020.

Vancouver:

Lambert J. Potentiel thérapeutique de l'activation du récepteur nucléaire PPARgamma dans la myélofibrose : Therapeutic Potential of Activation of the Nuclear Receptor PPARgamma Pathway in Myelofibrosis. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2020 Apr 02]. Available from: http://www.theses.fr/2019SACLS536.

Council of Science Editors:

Lambert J. Potentiel thérapeutique de l'activation du récepteur nucléaire PPARgamma dans la myélofibrose : Therapeutic Potential of Activation of the Nuclear Receptor PPARgamma Pathway in Myelofibrosis. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLS536

7. Gaillard, François. Étude mécanistique de la fibrose interstitielle rénale et essai de preuve de concept thérapeutique : Renal interstitial fibrosis pathways and proof of concept for therapy.

Degree: Docteur es, Physiopathologie, 2018, Sorbonne Paris Cité

 Les mécanismes responsables des lésions rénales sont multiples, initialement propres à chaque pathologie. En revanche, les processus de cicatrisation qui suivent cette agression initiale convergent… (more)

Subjects/Keywords: Rein; Fibrose; Stat3; Ppargamma; Hipk2; Ubiquitine; Kidney; Fibrosis; Stat3; Ppargamma; Hipk2; Ubiquitin; 616.6

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APA (6th Edition):

Gaillard, F. (2018). Étude mécanistique de la fibrose interstitielle rénale et essai de preuve de concept thérapeutique : Renal interstitial fibrosis pathways and proof of concept for therapy. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2018USPCB096

Chicago Manual of Style (16th Edition):

Gaillard, François. “Étude mécanistique de la fibrose interstitielle rénale et essai de preuve de concept thérapeutique : Renal interstitial fibrosis pathways and proof of concept for therapy.” 2018. Doctoral Dissertation, Sorbonne Paris Cité. Accessed April 02, 2020. http://www.theses.fr/2018USPCB096.

MLA Handbook (7th Edition):

Gaillard, François. “Étude mécanistique de la fibrose interstitielle rénale et essai de preuve de concept thérapeutique : Renal interstitial fibrosis pathways and proof of concept for therapy.” 2018. Web. 02 Apr 2020.

Vancouver:

Gaillard F. Étude mécanistique de la fibrose interstitielle rénale et essai de preuve de concept thérapeutique : Renal interstitial fibrosis pathways and proof of concept for therapy. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2018. [cited 2020 Apr 02]. Available from: http://www.theses.fr/2018USPCB096.

Council of Science Editors:

Gaillard F. Étude mécanistique de la fibrose interstitielle rénale et essai de preuve de concept thérapeutique : Renal interstitial fibrosis pathways and proof of concept for therapy. [Doctoral Dissertation]. Sorbonne Paris Cité; 2018. Available from: http://www.theses.fr/2018USPCB096


Université de Grenoble

8. Garnier, Vanessa. Rôle du facteur pro-angiogène EG-VEGF dans le développement placentaire au cours de premier trimestre de grossesse : Role of EG-VEGF in placental developpment in first trimester of pregnancy.

Degree: Docteur es, Biologie cellulaire, 2014, Université de Grenoble

Le développement placentaire est un processus finement contrôlé dans le temps et dans l'espace. Il est caractérisé par une invasion précoce et profonde, de l'endomètre… (more)

Subjects/Keywords: EG-VEGF; Pré-éclamspsie; Placenta; PPARgamma; Invasion trophoblastique; Hématopoïése; EG-VEGF; Pre-eclamspsia; Placenta; PPARgamma; Trophoblastic invasion; Hematopoiesis; 570

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APA (6th Edition):

Garnier, V. (2014). Rôle du facteur pro-angiogène EG-VEGF dans le développement placentaire au cours de premier trimestre de grossesse : Role of EG-VEGF in placental developpment in first trimester of pregnancy. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2014GRENV021

Chicago Manual of Style (16th Edition):

Garnier, Vanessa. “Rôle du facteur pro-angiogène EG-VEGF dans le développement placentaire au cours de premier trimestre de grossesse : Role of EG-VEGF in placental developpment in first trimester of pregnancy.” 2014. Doctoral Dissertation, Université de Grenoble. Accessed April 02, 2020. http://www.theses.fr/2014GRENV021.

MLA Handbook (7th Edition):

Garnier, Vanessa. “Rôle du facteur pro-angiogène EG-VEGF dans le développement placentaire au cours de premier trimestre de grossesse : Role of EG-VEGF in placental developpment in first trimester of pregnancy.” 2014. Web. 02 Apr 2020.

Vancouver:

Garnier V. Rôle du facteur pro-angiogène EG-VEGF dans le développement placentaire au cours de premier trimestre de grossesse : Role of EG-VEGF in placental developpment in first trimester of pregnancy. [Internet] [Doctoral dissertation]. Université de Grenoble; 2014. [cited 2020 Apr 02]. Available from: http://www.theses.fr/2014GRENV021.

Council of Science Editors:

Garnier V. Rôle du facteur pro-angiogène EG-VEGF dans le développement placentaire au cours de premier trimestre de grossesse : Role of EG-VEGF in placental developpment in first trimester of pregnancy. [Doctoral Dissertation]. Université de Grenoble; 2014. Available from: http://www.theses.fr/2014GRENV021


Université de Lorraine

9. Colin-Cassin, Christelle. Activité PPARgamma-indépendante des ligands de PPARgamma : une piste pour le traitement des cancers du sein ? : PPARgamma-independente activity of PPARgamma ligands : a new perspective for the treatment of breast cancers ?.

Degree: Docteur es, Sciences de la vie et de la santé, 2013, Université de Lorraine

L'un des enjeux majeurs de la recherche anti-cancéreuse est de développer de nouvelles thérapies en direction des tumeurs réfractaires aux traitements conventionnels. Dans ce contexte,… (more)

Subjects/Keywords: Cancer mammaire; PPARgamma-indépendants; Thiazolidinediones; Apoptose; Stress du réticulum endoplasmique; Breast cancer; PPARgamma-independent; Thiazolidinedione; Apoptosis; Endoplasmic reticulum stress; 572.8; 615.7

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APA (6th Edition):

Colin-Cassin, C. (2013). Activité PPARgamma-indépendante des ligands de PPARgamma : une piste pour le traitement des cancers du sein ? : PPARgamma-independente activity of PPARgamma ligands : a new perspective for the treatment of breast cancers ?. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2013LORR0232

Chicago Manual of Style (16th Edition):

Colin-Cassin, Christelle. “Activité PPARgamma-indépendante des ligands de PPARgamma : une piste pour le traitement des cancers du sein ? : PPARgamma-independente activity of PPARgamma ligands : a new perspective for the treatment of breast cancers ?.” 2013. Doctoral Dissertation, Université de Lorraine. Accessed April 02, 2020. http://www.theses.fr/2013LORR0232.

MLA Handbook (7th Edition):

Colin-Cassin, Christelle. “Activité PPARgamma-indépendante des ligands de PPARgamma : une piste pour le traitement des cancers du sein ? : PPARgamma-independente activity of PPARgamma ligands : a new perspective for the treatment of breast cancers ?.” 2013. Web. 02 Apr 2020.

Vancouver:

Colin-Cassin C. Activité PPARgamma-indépendante des ligands de PPARgamma : une piste pour le traitement des cancers du sein ? : PPARgamma-independente activity of PPARgamma ligands : a new perspective for the treatment of breast cancers ?. [Internet] [Doctoral dissertation]. Université de Lorraine; 2013. [cited 2020 Apr 02]. Available from: http://www.theses.fr/2013LORR0232.

Council of Science Editors:

Colin-Cassin C. Activité PPARgamma-indépendante des ligands de PPARgamma : une piste pour le traitement des cancers du sein ? : PPARgamma-independente activity of PPARgamma ligands : a new perspective for the treatment of breast cancers ?. [Doctoral Dissertation]. Université de Lorraine; 2013. Available from: http://www.theses.fr/2013LORR0232


University of Rochester

10. Jin, Youngnam. Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation.

Degree: PhD, 2011, University of Rochester

 Huntington’s disease (HD) is an inherited neurodegenerative disease resulting from an abnormal expansion of polyglutamine in huntingtin (Htt). Many studies have shown that energetic deficits… (more)

Subjects/Keywords: Huntington Disease; Transcription; Metabolism; PGC-lalpha; PPARgamma; Superoxide; Mitochondria

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APA (6th Edition):

Jin, Y. (2011). Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/14596

Chicago Manual of Style (16th Edition):

Jin, Youngnam. “Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation.” 2011. Doctoral Dissertation, University of Rochester. Accessed April 02, 2020. http://hdl.handle.net/1802/14596.

MLA Handbook (7th Edition):

Jin, Youngnam. “Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation.” 2011. Web. 02 Apr 2020.

Vancouver:

Jin Y. Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/1802/14596.

Council of Science Editors:

Jin Y. Transcriptional Dysregulation and Metabolic Defects in Huntington Disease: The Beneficial Effects of PPARgamma Activation. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/14596


Penn State University

11. Nelson, Shakira Melissa. The Essential Role of Selenoproteins on Macrophage phenotype switching in Helminth clearance.

Degree: PhD, Pathobiology, 2013, Penn State University

 Selenium (Se) is an essential micronutrient with anti-inflammatory properties that are fundamental to human health. A vital component of many metabolic pathways, an inverse causal… (more)

Subjects/Keywords: Selenium; macrophages; helminths; PPARgamma; NSAIDS; small intestines; Nippostrongylus brasiliensis

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APA (6th Edition):

Nelson, S. M. (2013). The Essential Role of Selenoproteins on Macrophage phenotype switching in Helminth clearance. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/17549

Chicago Manual of Style (16th Edition):

Nelson, Shakira Melissa. “The Essential Role of Selenoproteins on Macrophage phenotype switching in Helminth clearance.” 2013. Doctoral Dissertation, Penn State University. Accessed April 02, 2020. https://etda.libraries.psu.edu/catalog/17549.

MLA Handbook (7th Edition):

Nelson, Shakira Melissa. “The Essential Role of Selenoproteins on Macrophage phenotype switching in Helminth clearance.” 2013. Web. 02 Apr 2020.

Vancouver:

Nelson SM. The Essential Role of Selenoproteins on Macrophage phenotype switching in Helminth clearance. [Internet] [Doctoral dissertation]. Penn State University; 2013. [cited 2020 Apr 02]. Available from: https://etda.libraries.psu.edu/catalog/17549.

Council of Science Editors:

Nelson SM. The Essential Role of Selenoproteins on Macrophage phenotype switching in Helminth clearance. [Doctoral Dissertation]. Penn State University; 2013. Available from: https://etda.libraries.psu.edu/catalog/17549


Universitat Rovira i Virgili

12. Saumoy Linares, Maria. Lipodistròfia en pacients infectats pel vih tractats amb fàrmacs antiretrovirals: determinants genètics i moleculars.

Degree: Departament de Medicina i Cirurgia, 2010, Universitat Rovira i Virgili

 Lipodystrophy is a body fat redistribution condition. It is a common long term adverse effect in HIV-infected patients associated with antiretroviral drugs, HIV itself and… (more)

Subjects/Keywords: VIH; lipodistròfia; interleuquina-6; lipina; PPARgamma; determinants genètics; 61

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APA (6th Edition):

Saumoy Linares, M. (2010). Lipodistròfia en pacients infectats pel vih tractats amb fàrmacs antiretrovirals: determinants genètics i moleculars. (Thesis). Universitat Rovira i Virgili. Retrieved from http://hdl.handle.net/10803/285938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Saumoy Linares, Maria. “Lipodistròfia en pacients infectats pel vih tractats amb fàrmacs antiretrovirals: determinants genètics i moleculars.” 2010. Thesis, Universitat Rovira i Virgili. Accessed April 02, 2020. http://hdl.handle.net/10803/285938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Saumoy Linares, Maria. “Lipodistròfia en pacients infectats pel vih tractats amb fàrmacs antiretrovirals: determinants genètics i moleculars.” 2010. Web. 02 Apr 2020.

Vancouver:

Saumoy Linares M. Lipodistròfia en pacients infectats pel vih tractats amb fàrmacs antiretrovirals: determinants genètics i moleculars. [Internet] [Thesis]. Universitat Rovira i Virgili; 2010. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/10803/285938.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Saumoy Linares M. Lipodistròfia en pacients infectats pel vih tractats amb fàrmacs antiretrovirals: determinants genètics i moleculars. [Thesis]. Universitat Rovira i Virgili; 2010. Available from: http://hdl.handle.net/10803/285938

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Marques, Milano Felipe dos Santos Ferreira. Caracterização de genes e proteínas plasmáticas relacionadas ao diabetes melito do tipo 2 em indivíduos tratados com pioglitazona.

Degree: Mestrado, Análises Clínicas, 2008, University of São Paulo

O diabete melito é um grupo de doenças metabólicas caracterizadas por hiperglicemia, resultado de deficiências na secreção de insulina, em sua acção ou ambos. A… (more)

Subjects/Keywords: ATP dependent potassium channels; Canal de Potássio dependente de ATP; Diabetes Melito; Diabetes mellitus; Expressão gênica; Gene expression; Pioglitazona; Pioglitazone; PPARgamma; PPARgamma

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APA (6th Edition):

Marques, M. F. d. S. F. (2008). Caracterização de genes e proteínas plasmáticas relacionadas ao diabetes melito do tipo 2 em indivíduos tratados com pioglitazona. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/9/9136/tde-16092008-151426/ ;

Chicago Manual of Style (16th Edition):

Marques, Milano Felipe dos Santos Ferreira. “Caracterização de genes e proteínas plasmáticas relacionadas ao diabetes melito do tipo 2 em indivíduos tratados com pioglitazona.” 2008. Masters Thesis, University of São Paulo. Accessed April 02, 2020. http://www.teses.usp.br/teses/disponiveis/9/9136/tde-16092008-151426/ ;.

MLA Handbook (7th Edition):

Marques, Milano Felipe dos Santos Ferreira. “Caracterização de genes e proteínas plasmáticas relacionadas ao diabetes melito do tipo 2 em indivíduos tratados com pioglitazona.” 2008. Web. 02 Apr 2020.

Vancouver:

Marques MFdSF. Caracterização de genes e proteínas plasmáticas relacionadas ao diabetes melito do tipo 2 em indivíduos tratados com pioglitazona. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2020 Apr 02]. Available from: http://www.teses.usp.br/teses/disponiveis/9/9136/tde-16092008-151426/ ;.

Council of Science Editors:

Marques MFdSF. Caracterização de genes e proteínas plasmáticas relacionadas ao diabetes melito do tipo 2 em indivíduos tratados com pioglitazona. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/9/9136/tde-16092008-151426/ ;

14. Mlih, Mohamed. Implication de LRP1 et ShcA dans deux pathologies cardiovasculaires : l'arthérosclérose et l'insuffisance cardiaque : Implication of LRP1 and ShcA in two cardiovascular diseases : atherosclerosis and heart failure.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2012, Université de Strasbourg

Les maladies cardiovasculaires sont la première cause de mortalité dans le monde. Une meilleure compréhension des mécanismes physiopathologiques est nécessaire. Dans ce travail de thèse… (more)

Subjects/Keywords: ShcA; LRP1; PPARgamma; Wnt5; Sarcomère; Costamère; Insuffisance cardiaque; Coeur; Athérosclérose; Calcification; Chondrogenèse; ShcA; LRP1; PPARgamma; Wnt5a; Sarcomer; Costamer; Heart failure; Heart; Atherosclerosis; Calcification; Chondrogenesis; 572.8; 616.1

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APA (6th Edition):

Mlih, M. (2012). Implication de LRP1 et ShcA dans deux pathologies cardiovasculaires : l'arthérosclérose et l'insuffisance cardiaque : Implication of LRP1 and ShcA in two cardiovascular diseases : atherosclerosis and heart failure. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2012STRAJ086

Chicago Manual of Style (16th Edition):

Mlih, Mohamed. “Implication de LRP1 et ShcA dans deux pathologies cardiovasculaires : l'arthérosclérose et l'insuffisance cardiaque : Implication of LRP1 and ShcA in two cardiovascular diseases : atherosclerosis and heart failure.” 2012. Doctoral Dissertation, Université de Strasbourg. Accessed April 02, 2020. http://www.theses.fr/2012STRAJ086.

MLA Handbook (7th Edition):

Mlih, Mohamed. “Implication de LRP1 et ShcA dans deux pathologies cardiovasculaires : l'arthérosclérose et l'insuffisance cardiaque : Implication of LRP1 and ShcA in two cardiovascular diseases : atherosclerosis and heart failure.” 2012. Web. 02 Apr 2020.

Vancouver:

Mlih M. Implication de LRP1 et ShcA dans deux pathologies cardiovasculaires : l'arthérosclérose et l'insuffisance cardiaque : Implication of LRP1 and ShcA in two cardiovascular diseases : atherosclerosis and heart failure. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2012. [cited 2020 Apr 02]. Available from: http://www.theses.fr/2012STRAJ086.

Council of Science Editors:

Mlih M. Implication de LRP1 et ShcA dans deux pathologies cardiovasculaires : l'arthérosclérose et l'insuffisance cardiaque : Implication of LRP1 and ShcA in two cardiovascular diseases : atherosclerosis and heart failure. [Doctoral Dissertation]. Université de Strasbourg; 2012. Available from: http://www.theses.fr/2012STRAJ086


Louisiana State University

15. Hogan, Jessica C. Modulation of adipocyte genes by signal transducers and activators of transcription.

Degree: PhD, 2004, Louisiana State University

 Members of the STAT transcription factor family are expressed in adipocytes, including STATs 1, 3, 5A, 5B, and 6. Although STATs 1 and 5 proteins… (more)

Subjects/Keywords: stats; cytokine; adipocyte; fas; lpl; ppargamma

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APA (6th Edition):

Hogan, J. C. (2004). Modulation of adipocyte genes by signal transducers and activators of transcription. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-01092005-181433 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1670

Chicago Manual of Style (16th Edition):

Hogan, Jessica C. “Modulation of adipocyte genes by signal transducers and activators of transcription.” 2004. Doctoral Dissertation, Louisiana State University. Accessed April 02, 2020. etd-01092005-181433 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1670.

MLA Handbook (7th Edition):

Hogan, Jessica C. “Modulation of adipocyte genes by signal transducers and activators of transcription.” 2004. Web. 02 Apr 2020.

Vancouver:

Hogan JC. Modulation of adipocyte genes by signal transducers and activators of transcription. [Internet] [Doctoral dissertation]. Louisiana State University; 2004. [cited 2020 Apr 02]. Available from: etd-01092005-181433 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1670.

Council of Science Editors:

Hogan JC. Modulation of adipocyte genes by signal transducers and activators of transcription. [Doctoral Dissertation]. Louisiana State University; 2004. Available from: etd-01092005-181433 ; https://digitalcommons.lsu.edu/gradschool_dissertations/1670

16. Ala Eddine, Mohamad. Impact de l'IL-13 dans l'acquisition des fonctions tumoricides des macrophages : rôle des récepteurs lectine de type-C et implication dans la progression d'un lymphome T : Role of IL-13 in the acquisition of antitumor activities of macrophages : involvement of C type lectin receptors and implication in T-cell lymphoma development.

Degree: Docteur es, Physiopathologie, 2016, Université Toulouse III – Paul Sabatier

 Les macrophages associés aux tumeurs (TAMs) proviennent des monocytes circulants attirés par l'inflammation chronique due à la tumeur. Ces monocytes vont se différencier en une… (more)

Subjects/Keywords: Macrophages; STAT-6; Polarisation; PPARgamma; Interleukine-13; Lymphome-T; Récepteurs lectine de type C; Immunothérapie

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APA (6th Edition):

Ala Eddine, M. (2016). Impact de l'IL-13 dans l'acquisition des fonctions tumoricides des macrophages : rôle des récepteurs lectine de type-C et implication dans la progression d'un lymphome T : Role of IL-13 in the acquisition of antitumor activities of macrophages : involvement of C type lectin receptors and implication in T-cell lymphoma development. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2016TOU30065

Chicago Manual of Style (16th Edition):

Ala Eddine, Mohamad. “Impact de l'IL-13 dans l'acquisition des fonctions tumoricides des macrophages : rôle des récepteurs lectine de type-C et implication dans la progression d'un lymphome T : Role of IL-13 in the acquisition of antitumor activities of macrophages : involvement of C type lectin receptors and implication in T-cell lymphoma development.” 2016. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed April 02, 2020. http://www.theses.fr/2016TOU30065.

MLA Handbook (7th Edition):

Ala Eddine, Mohamad. “Impact de l'IL-13 dans l'acquisition des fonctions tumoricides des macrophages : rôle des récepteurs lectine de type-C et implication dans la progression d'un lymphome T : Role of IL-13 in the acquisition of antitumor activities of macrophages : involvement of C type lectin receptors and implication in T-cell lymphoma development.” 2016. Web. 02 Apr 2020.

Vancouver:

Ala Eddine M. Impact de l'IL-13 dans l'acquisition des fonctions tumoricides des macrophages : rôle des récepteurs lectine de type-C et implication dans la progression d'un lymphome T : Role of IL-13 in the acquisition of antitumor activities of macrophages : involvement of C type lectin receptors and implication in T-cell lymphoma development. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2016. [cited 2020 Apr 02]. Available from: http://www.theses.fr/2016TOU30065.

Council of Science Editors:

Ala Eddine M. Impact de l'IL-13 dans l'acquisition des fonctions tumoricides des macrophages : rôle des récepteurs lectine de type-C et implication dans la progression d'un lymphome T : Role of IL-13 in the acquisition of antitumor activities of macrophages : involvement of C type lectin receptors and implication in T-cell lymphoma development. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2016. Available from: http://www.theses.fr/2016TOU30065


University of California – San Francisco

17. Mera, Linet. GTF2IRD1 is a PRDM16-interacting transcription factor that represses TGF-beta-mediated inhibition of beige fat differentiation.

Degree: Biochemistry and Molecular Biology, 2014, University of California – San Francisco

 The identification of beige fat within the last decade, its ability to burn energy in adult humans, and its great potential for therapeutic applications has… (more)

Subjects/Keywords: Molecular biology; Biogeochemistry; Cellular biology; adipose; beige; GTF2IRD1; PPARgamma; PRDM16; TGF-beta

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APA (6th Edition):

Mera, L. (2014). GTF2IRD1 is a PRDM16-interacting transcription factor that represses TGF-beta-mediated inhibition of beige fat differentiation. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/7m14812z

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mera, Linet. “GTF2IRD1 is a PRDM16-interacting transcription factor that represses TGF-beta-mediated inhibition of beige fat differentiation.” 2014. Thesis, University of California – San Francisco. Accessed April 02, 2020. http://www.escholarship.org/uc/item/7m14812z.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mera, Linet. “GTF2IRD1 is a PRDM16-interacting transcription factor that represses TGF-beta-mediated inhibition of beige fat differentiation.” 2014. Web. 02 Apr 2020.

Vancouver:

Mera L. GTF2IRD1 is a PRDM16-interacting transcription factor that represses TGF-beta-mediated inhibition of beige fat differentiation. [Internet] [Thesis]. University of California – San Francisco; 2014. [cited 2020 Apr 02]. Available from: http://www.escholarship.org/uc/item/7m14812z.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mera L. GTF2IRD1 is a PRDM16-interacting transcription factor that represses TGF-beta-mediated inhibition of beige fat differentiation. [Thesis]. University of California – San Francisco; 2014. Available from: http://www.escholarship.org/uc/item/7m14812z

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. MANAV. A study into the different mechanisms of anti-cancer effect of docosahexanoic acid in colon cancer cells.

Degree: 2005, National University of Singapore

Subjects/Keywords: DHA; colon; JNK; ROS; PPARgamma and COX-2

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APA (6th Edition):

MANAV. (2005). A study into the different mechanisms of anti-cancer effect of docosahexanoic acid in colon cancer cells. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/14829

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

MANAV. “A study into the different mechanisms of anti-cancer effect of docosahexanoic acid in colon cancer cells.” 2005. Thesis, National University of Singapore. Accessed April 02, 2020. http://scholarbank.nus.edu.sg/handle/10635/14829.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

MANAV. “A study into the different mechanisms of anti-cancer effect of docosahexanoic acid in colon cancer cells.” 2005. Web. 02 Apr 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

MANAV. A study into the different mechanisms of anti-cancer effect of docosahexanoic acid in colon cancer cells. [Internet] [Thesis]. National University of Singapore; 2005. [cited 2020 Apr 02]. Available from: http://scholarbank.nus.edu.sg/handle/10635/14829.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

MANAV. A study into the different mechanisms of anti-cancer effect of docosahexanoic acid in colon cancer cells. [Thesis]. National University of Singapore; 2005. Available from: http://scholarbank.nus.edu.sg/handle/10635/14829

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

19. NG CHUN CHI. IDENTIFICATION OF A NOVEL SMALL-MOLECULE PPARGAMMA ACTIVATOR THAT INDUCES AUTOPHAGIC CELL DEATH IN CANCER CELLS.

Degree: 2011, National University of Singapore

Subjects/Keywords: PPARgamma; activator; autophagy; cell death; cancer cells

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APA (6th Edition):

CHI, N. C. (2011). IDENTIFICATION OF A NOVEL SMALL-MOLECULE PPARGAMMA ACTIVATOR THAT INDUCES AUTOPHAGIC CELL DEATH IN CANCER CELLS. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/32863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

CHI, NG CHUN. “IDENTIFICATION OF A NOVEL SMALL-MOLECULE PPARGAMMA ACTIVATOR THAT INDUCES AUTOPHAGIC CELL DEATH IN CANCER CELLS.” 2011. Thesis, National University of Singapore. Accessed April 02, 2020. http://scholarbank.nus.edu.sg/handle/10635/32863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

CHI, NG CHUN. “IDENTIFICATION OF A NOVEL SMALL-MOLECULE PPARGAMMA ACTIVATOR THAT INDUCES AUTOPHAGIC CELL DEATH IN CANCER CELLS.” 2011. Web. 02 Apr 2020.

Vancouver:

CHI NC. IDENTIFICATION OF A NOVEL SMALL-MOLECULE PPARGAMMA ACTIVATOR THAT INDUCES AUTOPHAGIC CELL DEATH IN CANCER CELLS. [Internet] [Thesis]. National University of Singapore; 2011. [cited 2020 Apr 02]. Available from: http://scholarbank.nus.edu.sg/handle/10635/32863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

CHI NC. IDENTIFICATION OF A NOVEL SMALL-MOLECULE PPARGAMMA ACTIVATOR THAT INDUCES AUTOPHAGIC CELL DEATH IN CANCER CELLS. [Thesis]. National University of Singapore; 2011. Available from: http://scholarbank.nus.edu.sg/handle/10635/32863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. ZHOU TING. THE MECHANISM OF PPARN3-MEDIATED DOWN-REGULATION OF SODIUM HYDROGEN EXCHANGER 1 (NHE1) GENE EPXRESSION AND ITS INHIBITION BY ESTROGEN RECEPTOR N1.

Degree: 2012, National University of Singapore

Subjects/Keywords: PPARgamma; Estrogen Receptor; NHE1; breast cancer; estrogen; transcriptional regulation

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APA (6th Edition):

TING, Z. (2012). THE MECHANISM OF PPARN3-MEDIATED DOWN-REGULATION OF SODIUM HYDROGEN EXCHANGER 1 (NHE1) GENE EPXRESSION AND ITS INHIBITION BY ESTROGEN RECEPTOR N1. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/35524

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

TING, ZHOU. “THE MECHANISM OF PPARN3-MEDIATED DOWN-REGULATION OF SODIUM HYDROGEN EXCHANGER 1 (NHE1) GENE EPXRESSION AND ITS INHIBITION BY ESTROGEN RECEPTOR N1.” 2012. Thesis, National University of Singapore. Accessed April 02, 2020. http://scholarbank.nus.edu.sg/handle/10635/35524.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

TING, ZHOU. “THE MECHANISM OF PPARN3-MEDIATED DOWN-REGULATION OF SODIUM HYDROGEN EXCHANGER 1 (NHE1) GENE EPXRESSION AND ITS INHIBITION BY ESTROGEN RECEPTOR N1.” 2012. Web. 02 Apr 2020.

Vancouver:

TING Z. THE MECHANISM OF PPARN3-MEDIATED DOWN-REGULATION OF SODIUM HYDROGEN EXCHANGER 1 (NHE1) GENE EPXRESSION AND ITS INHIBITION BY ESTROGEN RECEPTOR N1. [Internet] [Thesis]. National University of Singapore; 2012. [cited 2020 Apr 02]. Available from: http://scholarbank.nus.edu.sg/handle/10635/35524.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

TING Z. THE MECHANISM OF PPARN3-MEDIATED DOWN-REGULATION OF SODIUM HYDROGEN EXCHANGER 1 (NHE1) GENE EPXRESSION AND ITS INHIBITION BY ESTROGEN RECEPTOR N1. [Thesis]. National University of Singapore; 2012. Available from: http://scholarbank.nus.edu.sg/handle/10635/35524

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Ambrozewicz, Marta A. Mechanisms of Regulation of Proximal Tubule Sodium Transporters in Obesity-Induced Hypertension.

Degree: PhD, 2009, Old Dominion University

  Hypertension is one of the common complications of obesity. Using a rat model of diet induced obesity and hypertension we investigated some of the… (more)

Subjects/Keywords: Hypertension; Obesity; PPARgamma; Proximal tubules; Sodium transporters; Diseases; Physiology

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APA (6th Edition):

Ambrozewicz, M. A. (2009). Mechanisms of Regulation of Proximal Tubule Sodium Transporters in Obesity-Induced Hypertension. (Doctoral Dissertation). Old Dominion University. Retrieved from 9781109705966 ; https://digitalcommons.odu.edu/biomedicalsciences_etds/2

Chicago Manual of Style (16th Edition):

Ambrozewicz, Marta A. “Mechanisms of Regulation of Proximal Tubule Sodium Transporters in Obesity-Induced Hypertension.” 2009. Doctoral Dissertation, Old Dominion University. Accessed April 02, 2020. 9781109705966 ; https://digitalcommons.odu.edu/biomedicalsciences_etds/2.

MLA Handbook (7th Edition):

Ambrozewicz, Marta A. “Mechanisms of Regulation of Proximal Tubule Sodium Transporters in Obesity-Induced Hypertension.” 2009. Web. 02 Apr 2020.

Vancouver:

Ambrozewicz MA. Mechanisms of Regulation of Proximal Tubule Sodium Transporters in Obesity-Induced Hypertension. [Internet] [Doctoral dissertation]. Old Dominion University; 2009. [cited 2020 Apr 02]. Available from: 9781109705966 ; https://digitalcommons.odu.edu/biomedicalsciences_etds/2.

Council of Science Editors:

Ambrozewicz MA. Mechanisms of Regulation of Proximal Tubule Sodium Transporters in Obesity-Induced Hypertension. [Doctoral Dissertation]. Old Dominion University; 2009. Available from: 9781109705966 ; https://digitalcommons.odu.edu/biomedicalsciences_etds/2


Freie Universität Berlin

22. Foryst-Ludwig, Anna. die Rolle von Estrogenen, Estrogenrezeptoren und PPARgamma.

Degree: 2014, Freie Universität Berlin

 Die Prävalenz von Adipositas und Adipositas-bedingten metabolischen und kardiovaskulären (kardiometabolischen) Erkrankungen nimmt immer weiter zu. Hieraus folgt ein massiver Anstieg der kardiovaskulären Mortalität. Derzeit sind… (more)

Subjects/Keywords: obesity-related cardiovascular diseases; sex-specific differences; PPARgamma; cardiac hypertrophy; lipolysis; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Foryst-Ludwig, A. (2014). die Rolle von Estrogenen, Estrogenrezeptoren und PPARgamma. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/10694

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Foryst-Ludwig, Anna. “die Rolle von Estrogenen, Estrogenrezeptoren und PPARgamma.” 2014. Thesis, Freie Universität Berlin. Accessed April 02, 2020. https://refubium.fu-berlin.de/handle/fub188/10694.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Foryst-Ludwig, Anna. “die Rolle von Estrogenen, Estrogenrezeptoren und PPARgamma.” 2014. Web. 02 Apr 2020.

Vancouver:

Foryst-Ludwig A. die Rolle von Estrogenen, Estrogenrezeptoren und PPARgamma. [Internet] [Thesis]. Freie Universität Berlin; 2014. [cited 2020 Apr 02]. Available from: https://refubium.fu-berlin.de/handle/fub188/10694.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Foryst-Ludwig A. die Rolle von Estrogenen, Estrogenrezeptoren und PPARgamma. [Thesis]. Freie Universität Berlin; 2014. Available from: https://refubium.fu-berlin.de/handle/fub188/10694

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

23. Monemdjou, Roxana. The role of PPARgamma in cartilage growth and development using cartilage-specific PPARgamma knockout mice .

Degree: 2012, Université de Montréal

 Le cartilage est un tissu conjonctif composé d’une seule sorte de cellule nommée chondrocytes. Ce tissu offre une fondation pour la formation des os. Les… (more)

Subjects/Keywords: PPARgamma; Osteoarthritis; Knockout mice; Cartilage growth and development; Aging; Chondrogenesis; Endochondral ossification; Arthrose; Souris ayant une délétion au PPARgamma; Croissance et le développement du cartilage; Ossification endochondral; Formation du cartilage; Vieillissement

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APA (6th Edition):

Monemdjou, R. (2012). The role of PPARgamma in cartilage growth and development using cartilage-specific PPARgamma knockout mice . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/6908

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Monemdjou, Roxana. “The role of PPARgamma in cartilage growth and development using cartilage-specific PPARgamma knockout mice .” 2012. Thesis, Université de Montréal. Accessed April 02, 2020. http://hdl.handle.net/1866/6908.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Monemdjou, Roxana. “The role of PPARgamma in cartilage growth and development using cartilage-specific PPARgamma knockout mice .” 2012. Web. 02 Apr 2020.

Vancouver:

Monemdjou R. The role of PPARgamma in cartilage growth and development using cartilage-specific PPARgamma knockout mice . [Internet] [Thesis]. Université de Montréal; 2012. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/1866/6908.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Monemdjou R. The role of PPARgamma in cartilage growth and development using cartilage-specific PPARgamma knockout mice . [Thesis]. Université de Montréal; 2012. Available from: http://hdl.handle.net/1866/6908

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

24. Lamontagne, Julien. Effets directs et aigus de médicaments insulinosensibilisateurs sur la cellule bêta des îlots pancréatiques : de l’outil de recherche à l’identification de la décélération métabolique comme mode d’action .

Degree: 2013, Université de Montréal

 Le diabète de type 2 (DT2) apparaît lorsque la sécrétion d’insuline par les cellules β des îlots du pancréas ne parvient plus à compenser la… (more)

Subjects/Keywords: Diabète de type 2; Cellule bêta pancréatique; Sécrétion d'insuline; Métabolisme; Thiazolidinedione; Pioglitazone; Metformine; AMPK; PPARgamma; Décélération métabolique; Type 2 diabetes; Pancreatic beta-cell; Insulin secretion; Metabolism; Thiazolidinedione; Pioglitazone; Metformin; AMPK; PPARgamma; Metabolic deceleration

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APA (6th Edition):

Lamontagne, J. (2013). Effets directs et aigus de médicaments insulinosensibilisateurs sur la cellule bêta des îlots pancréatiques : de l’outil de recherche à l’identification de la décélération métabolique comme mode d’action . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/9004

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lamontagne, Julien. “Effets directs et aigus de médicaments insulinosensibilisateurs sur la cellule bêta des îlots pancréatiques : de l’outil de recherche à l’identification de la décélération métabolique comme mode d’action .” 2013. Thesis, Université de Montréal. Accessed April 02, 2020. http://hdl.handle.net/1866/9004.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lamontagne, Julien. “Effets directs et aigus de médicaments insulinosensibilisateurs sur la cellule bêta des îlots pancréatiques : de l’outil de recherche à l’identification de la décélération métabolique comme mode d’action .” 2013. Web. 02 Apr 2020.

Vancouver:

Lamontagne J. Effets directs et aigus de médicaments insulinosensibilisateurs sur la cellule bêta des îlots pancréatiques : de l’outil de recherche à l’identification de la décélération métabolique comme mode d’action . [Internet] [Thesis]. Université de Montréal; 2013. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/1866/9004.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lamontagne J. Effets directs et aigus de médicaments insulinosensibilisateurs sur la cellule bêta des îlots pancréatiques : de l’outil de recherche à l’identification de la décélération métabolique comme mode d’action . [Thesis]. Université de Montréal; 2013. Available from: http://hdl.handle.net/1866/9004

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

25. Rodrigue-Way, Amélie. Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36 .

Degree: 2011, Université de Montréal

 Les sécrétines de l’hormone de croissance (GHRPs) sont de petits peptides synthétiques capables de stimuler la sécrétion de l’hormone de croissance à partir de l’hypophyse… (more)

Subjects/Keywords: CD36; Hexarelin; PPARgamma; PGC-1alpha; Mitochondrial biogenesis; UCP-1; Fatty acid oxidation; AMPK; HMG-CoA Reductase; Insig-2; Hexaréline; Biogenèse mitochondriale; Oxydation des acides gras; Erk; Adipocytes; Hépatocytes; LKB1

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APA (6th Edition):

Rodrigue-Way, A. (2011). Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36 . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/5998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rodrigue-Way, Amélie. “Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36 .” 2011. Thesis, Université de Montréal. Accessed April 02, 2020. http://hdl.handle.net/1866/5998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rodrigue-Way, Amélie. “Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36 .” 2011. Web. 02 Apr 2020.

Vancouver:

Rodrigue-Way A. Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36 . [Internet] [Thesis]. Université de Montréal; 2011. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/1866/5998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rodrigue-Way A. Regulation of lipid metabolism in adipocytes and hepatocytes by hexarelin through scavenger receptor CD36 . [Thesis]. Université de Montréal; 2011. Available from: http://hdl.handle.net/1866/5998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

26. Demers, Annie. Régulation de l'activité transcriptionnelle de PPARgamma via l'activation des récepteurs CD36 et GHS-R1a : potentiel anti-athérosclérotique .

Degree: 2010, Université de Montréal

 Les sécrétines peptidiques de l’hormone de croissance (GHRPs) constituent une classe de peptides synthétiques capables de stimuler la sécrétion de l’hormone de croissance (GH). Cette… (more)

Subjects/Keywords: Athérosclérose; Atherosclerosis; Fyn kinase; GHS-R1a; Hexaréline; Hexarelin; LXRalpha; PPARgamma; MAPK/ERK; Akt; Transporteurs de type ABC; ABC transporter; Ghréline; Ghrelin; CD36

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APA (6th Edition):

Demers, A. (2010). Régulation de l'activité transcriptionnelle de PPARgamma via l'activation des récepteurs CD36 et GHS-R1a : potentiel anti-athérosclérotique . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/3477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Demers, Annie. “Régulation de l'activité transcriptionnelle de PPARgamma via l'activation des récepteurs CD36 et GHS-R1a : potentiel anti-athérosclérotique .” 2010. Thesis, Université de Montréal. Accessed April 02, 2020. http://hdl.handle.net/1866/3477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Demers, Annie. “Régulation de l'activité transcriptionnelle de PPARgamma via l'activation des récepteurs CD36 et GHS-R1a : potentiel anti-athérosclérotique .” 2010. Web. 02 Apr 2020.

Vancouver:

Demers A. Régulation de l'activité transcriptionnelle de PPARgamma via l'activation des récepteurs CD36 et GHS-R1a : potentiel anti-athérosclérotique . [Internet] [Thesis]. Université de Montréal; 2010. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/1866/3477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Demers A. Régulation de l'activité transcriptionnelle de PPARgamma via l'activation des récepteurs CD36 et GHS-R1a : potentiel anti-athérosclérotique . [Thesis]. Université de Montréal; 2010. Available from: http://hdl.handle.net/1866/3477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Tennessee State University

27. Yang, Lin. The Signaling Pathway of Oxysterol Induced Apoptosis in Chinese Hamster Ovary (CHO)-K1 Cells.

Degree: PhD, Biomedical Sciences, 2002, East Tennessee State University

  Apoptosis, a form of genetically programmed cell death, plays a key role in regulation of cellularity of the arterial wall. During atherogenesis, improper apoptosis… (more)

Subjects/Keywords: caspase; cPLA2; PPAR; PPARgamma; oxysterol; Apoptosis; Medical Sciences; Medicine and Health Sciences

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APA (6th Edition):

Yang, L. (2002). The Signaling Pathway of Oxysterol Induced Apoptosis in Chinese Hamster Ovary (CHO)-K1 Cells. (Doctoral Dissertation). East Tennessee State University. Retrieved from https://dc.etsu.edu/etd/678

Chicago Manual of Style (16th Edition):

Yang, Lin. “The Signaling Pathway of Oxysterol Induced Apoptosis in Chinese Hamster Ovary (CHO)-K1 Cells.” 2002. Doctoral Dissertation, East Tennessee State University. Accessed April 02, 2020. https://dc.etsu.edu/etd/678.

MLA Handbook (7th Edition):

Yang, Lin. “The Signaling Pathway of Oxysterol Induced Apoptosis in Chinese Hamster Ovary (CHO)-K1 Cells.” 2002. Web. 02 Apr 2020.

Vancouver:

Yang L. The Signaling Pathway of Oxysterol Induced Apoptosis in Chinese Hamster Ovary (CHO)-K1 Cells. [Internet] [Doctoral dissertation]. East Tennessee State University; 2002. [cited 2020 Apr 02]. Available from: https://dc.etsu.edu/etd/678.

Council of Science Editors:

Yang L. The Signaling Pathway of Oxysterol Induced Apoptosis in Chinese Hamster Ovary (CHO)-K1 Cells. [Doctoral Dissertation]. East Tennessee State University; 2002. Available from: https://dc.etsu.edu/etd/678

28. Guasch Pàmies, Laura. Identification of natural products as antidiabetic agents using computer-aided drug design methods.

Degree: Departament de Bioquímica i Biotecnologia, 2011, Universitat Rovira i Virgili

 Natural products derived from medicinal plants are an abundant source of biologically active compounds, many of which have formed the basis for development of nutraceuticals… (more)

Subjects/Keywords: antidiabetics; PPARgamma; DPP-IV; drug design; chemoinformatics; Natural products; 577

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APA (6th Edition):

Guasch Pàmies, L. (2011). Identification of natural products as antidiabetic agents using computer-aided drug design methods. (Thesis). Universitat Rovira i Virgili. Retrieved from http://hdl.handle.net/10803/111094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guasch Pàmies, Laura. “Identification of natural products as antidiabetic agents using computer-aided drug design methods.” 2011. Thesis, Universitat Rovira i Virgili. Accessed April 02, 2020. http://hdl.handle.net/10803/111094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guasch Pàmies, Laura. “Identification of natural products as antidiabetic agents using computer-aided drug design methods.” 2011. Web. 02 Apr 2020.

Vancouver:

Guasch Pàmies L. Identification of natural products as antidiabetic agents using computer-aided drug design methods. [Internet] [Thesis]. Universitat Rovira i Virgili; 2011. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/10803/111094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guasch Pàmies L. Identification of natural products as antidiabetic agents using computer-aided drug design methods. [Thesis]. Universitat Rovira i Virgili; 2011. Available from: http://hdl.handle.net/10803/111094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

29. Hansmann, Georg. Die Protektive Rolle von PPARgamma bei Pulmonalarterieller Hypertonie.

Degree: 2010, Freie Universität Berlin

 Nachweis eines neuen antiproliferativen Signalwegs downstream von BMP-2/BMP- RII, der die Aktivierung des Transkriptionsfaktors PPARgamma und seines Zielgens Apolipoprotein E (APOE) erfordert: Aktivierung des BMP-2/BMP-RII… (more)

Subjects/Keywords: pulmonary vascular disease; PPARgamma; apolipoprotein E; adiponectin; gowth factor; proliferation; remodeling; smooth muscle cell; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Hansmann, G. (2010). Die Protektive Rolle von PPARgamma bei Pulmonalarterieller Hypertonie. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-9494

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hansmann, Georg. “Die Protektive Rolle von PPARgamma bei Pulmonalarterieller Hypertonie.” 2010. Thesis, Freie Universität Berlin. Accessed April 02, 2020. http://dx.doi.org/10.17169/refubium-9494.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hansmann, Georg. “Die Protektive Rolle von PPARgamma bei Pulmonalarterieller Hypertonie.” 2010. Web. 02 Apr 2020.

Vancouver:

Hansmann G. Die Protektive Rolle von PPARgamma bei Pulmonalarterieller Hypertonie. [Internet] [Thesis]. Freie Universität Berlin; 2010. [cited 2020 Apr 02]. Available from: http://dx.doi.org/10.17169/refubium-9494.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hansmann G. Die Protektive Rolle von PPARgamma bei Pulmonalarterieller Hypertonie. [Thesis]. Freie Universität Berlin; 2010. Available from: http://dx.doi.org/10.17169/refubium-9494

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

30. Gustafson, Birgit 1951-. Effects of cytokines on preadipocyte differentiation and Wnt signalling.

Degree: 2006, University of Gothenburg / Göteborgs Universitet

 The relationship between amount of adipose tissue, the Metabolic Syndrome and Type 2 diabetes has been recognized for several years. However, the mechanisms for this… (more)

Subjects/Keywords: adipocyte differentiation; adiponectin; beta-Catenin; C/EBPalpha; IL-6; PPARgamma; TNFalpha; Wnt signalling

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APA (6th Edition):

Gustafson, B. 1. (2006). Effects of cytokines on preadipocyte differentiation and Wnt signalling. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/16788

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gustafson, Birgit 1951-. “Effects of cytokines on preadipocyte differentiation and Wnt signalling.” 2006. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed April 02, 2020. http://hdl.handle.net/2077/16788.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gustafson, Birgit 1951-. “Effects of cytokines on preadipocyte differentiation and Wnt signalling.” 2006. Web. 02 Apr 2020.

Vancouver:

Gustafson B1. Effects of cytokines on preadipocyte differentiation and Wnt signalling. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2006. [cited 2020 Apr 02]. Available from: http://hdl.handle.net/2077/16788.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gustafson B1. Effects of cytokines on preadipocyte differentiation and Wnt signalling. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2006. Available from: http://hdl.handle.net/2077/16788

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

.