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You searched for subject:(PKR). Showing records 1 – 30 of 50 total matches.

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Université Catholique de Louvain

1. De Groof, Aurélie. Rôle de PKR dans la physiopathologie du lupus.

Degree: 2018, Université Catholique de Louvain

Systemic lupus erythematosus is a heterogeneous autoimmune disease and understanding the physiopathology is necessary to improve treatments. In this work, we study the role of… (more)

Subjects/Keywords: Lupus; PKR

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APA (6th Edition):

De Groof, A. (2018). Rôle de PKR dans la physiopathologie du lupus. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/199567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

De Groof, Aurélie. “Rôle de PKR dans la physiopathologie du lupus.” 2018. Thesis, Université Catholique de Louvain. Accessed March 29, 2020. http://hdl.handle.net/2078.1/199567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

De Groof, Aurélie. “Rôle de PKR dans la physiopathologie du lupus.” 2018. Web. 29 Mar 2020.

Vancouver:

De Groof A. Rôle de PKR dans la physiopathologie du lupus. [Internet] [Thesis]. Université Catholique de Louvain; 2018. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/2078.1/199567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

De Groof A. Rôle de PKR dans la physiopathologie du lupus. [Thesis]. Université Catholique de Louvain; 2018. Available from: http://hdl.handle.net/2078.1/199567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Lithuanian Academy of Physical Education

2. Nėnienė, Virginija. Kineziterapijos efektyvumas po skirtingų kelio sąnario priekinio kryžminio raiščio rekonstrukcinių operacijų.

Degree: Master, Nursing, 2005, Lithuanian Academy of Physical Education

 SUMMARY The aim of the study was to evaluate the effectiveness of physical therapy after anterior cruciate ligament repair using different grafts. The goals of… (more)

Subjects/Keywords: PKR; ACL

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APA (6th Edition):

Nėnienė, Virginija. (2005). Kineziterapijos efektyvumas po skirtingų kelio sąnario priekinio kryžminio raiščio rekonstrukcinių operacijų. (Masters Thesis). Lithuanian Academy of Physical Education. Retrieved from http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2005~D_20050517_094028-49388 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Nėnienė, Virginija. “Kineziterapijos efektyvumas po skirtingų kelio sąnario priekinio kryžminio raiščio rekonstrukcinių operacijų.” 2005. Masters Thesis, Lithuanian Academy of Physical Education. Accessed March 29, 2020. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2005~D_20050517_094028-49388 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Nėnienė, Virginija. “Kineziterapijos efektyvumas po skirtingų kelio sąnario priekinio kryžminio raiščio rekonstrukcinių operacijų.” 2005. Web. 29 Mar 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Nėnienė, Virginija. Kineziterapijos efektyvumas po skirtingų kelio sąnario priekinio kryžminio raiščio rekonstrukcinių operacijų. [Internet] [Masters thesis]. Lithuanian Academy of Physical Education; 2005. [cited 2020 Mar 29]. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2005~D_20050517_094028-49388 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Nėnienė, Virginija. Kineziterapijos efektyvumas po skirtingų kelio sąnario priekinio kryžminio raiščio rekonstrukcinių operacijų. [Masters Thesis]. Lithuanian Academy of Physical Education; 2005. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2005~D_20050517_094028-49388 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Case Western Reserve University

3. Ogolla, Pauline S. The Protein Kinase Double-stranded RNA-dependent (PKR) Enhances Protection Against Disease Cause by a Non-viral Pathogen.

Degree: MSs, Pathology, 2012, Case Western Reserve University

PKR is well characterized for its function in antiviral immunity. Using Toxoplasma gondii, we examined if PKR promotes resistance to disease caused by a non-viral… (more)

Subjects/Keywords: Immunology; PKR; non-viral pathogen

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APA (6th Edition):

Ogolla, P. S. (2012). The Protein Kinase Double-stranded RNA-dependent (PKR) Enhances Protection Against Disease Cause by a Non-viral Pathogen. (Masters Thesis). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1341592115

Chicago Manual of Style (16th Edition):

Ogolla, Pauline S. “The Protein Kinase Double-stranded RNA-dependent (PKR) Enhances Protection Against Disease Cause by a Non-viral Pathogen.” 2012. Masters Thesis, Case Western Reserve University. Accessed March 29, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1341592115.

MLA Handbook (7th Edition):

Ogolla, Pauline S. “The Protein Kinase Double-stranded RNA-dependent (PKR) Enhances Protection Against Disease Cause by a Non-viral Pathogen.” 2012. Web. 29 Mar 2020.

Vancouver:

Ogolla PS. The Protein Kinase Double-stranded RNA-dependent (PKR) Enhances Protection Against Disease Cause by a Non-viral Pathogen. [Internet] [Masters thesis]. Case Western Reserve University; 2012. [cited 2020 Mar 29]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1341592115.

Council of Science Editors:

Ogolla PS. The Protein Kinase Double-stranded RNA-dependent (PKR) Enhances Protection Against Disease Cause by a Non-viral Pathogen. [Masters Thesis]. Case Western Reserve University; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1341592115


Penn State University

4. Heinicke, Laurie Ann. Regulation of the protein kinase PKR by higher-order RNA secondary and tertiary structures.

Degree: PhD, Chemistry, 2010, Penn State University

 A single strand of RNA can fold into multiple structures, often forming complex secondary and tertiary stuctures. Many factors influence RNA folding, including proteins, small… (more)

Subjects/Keywords: innate immune response; kinase; RNA; PKR

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APA (6th Edition):

Heinicke, L. A. (2010). Regulation of the protein kinase PKR by higher-order RNA secondary and tertiary structures. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/10450

Chicago Manual of Style (16th Edition):

Heinicke, Laurie Ann. “Regulation of the protein kinase PKR by higher-order RNA secondary and tertiary structures.” 2010. Doctoral Dissertation, Penn State University. Accessed March 29, 2020. https://etda.libraries.psu.edu/catalog/10450.

MLA Handbook (7th Edition):

Heinicke, Laurie Ann. “Regulation of the protein kinase PKR by higher-order RNA secondary and tertiary structures.” 2010. Web. 29 Mar 2020.

Vancouver:

Heinicke LA. Regulation of the protein kinase PKR by higher-order RNA secondary and tertiary structures. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2020 Mar 29]. Available from: https://etda.libraries.psu.edu/catalog/10450.

Council of Science Editors:

Heinicke LA. Regulation of the protein kinase PKR by higher-order RNA secondary and tertiary structures. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/10450


Johannes Gutenberg Universität Mainz

5. Herz, Stephanie. Optimization of RNA-based transgene expression by targeting Protein Kinase R.

Degree: 2013, Johannes Gutenberg Universität Mainz

The aim of this thesis was to establish a method for repeated transfection of in vitro transcribed RNA (IVT-RNA) leading to a sustained protein expression… (more)

Subjects/Keywords: IVT-RNS; PKR; Interferon; Proteinexpression; IVT-RNA; PKR; interferon; proteinexpression; Life sciences

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APA (6th Edition):

Herz, S. (2013). Optimization of RNA-based transgene expression by targeting Protein Kinase R. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2015/3994/

Chicago Manual of Style (16th Edition):

Herz, Stephanie. “Optimization of RNA-based transgene expression by targeting Protein Kinase R.” 2013. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed March 29, 2020. http://ubm.opus.hbz-nrw.de/volltexte/2015/3994/.

MLA Handbook (7th Edition):

Herz, Stephanie. “Optimization of RNA-based transgene expression by targeting Protein Kinase R.” 2013. Web. 29 Mar 2020.

Vancouver:

Herz S. Optimization of RNA-based transgene expression by targeting Protein Kinase R. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2013. [cited 2020 Mar 29]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2015/3994/.

Council of Science Editors:

Herz S. Optimization of RNA-based transgene expression by targeting Protein Kinase R. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2013. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2015/3994/

6. Tronel, Claire. Evaluation des effets de molécules à visée neuroprotectrice dans un modèle in vivo de neuroinflammation chez le rat : étude mécanistique et caractérisation du modèle au cours du temps : Evaluation of potential neuroprotective molecules in an in vivo rat neuroinflammatory model : mechanistic study and time characterization.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2013, Université François-Rabelais de Tours

La mise au point de médicaments ciblant la neuroinflammation, une composante importante de la physiopathologie des maladies neurodégénératives, fait l’objet de nombreuses recherches. Dans ce… (more)

Subjects/Keywords: Neuroinflammation; Acide quinolinique; HO-1; PKR; Neuroinflammation; Quinolinic acid; HO-1; PKR

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APA (6th Edition):

Tronel, C. (2013). Evaluation des effets de molécules à visée neuroprotectrice dans un modèle in vivo de neuroinflammation chez le rat : étude mécanistique et caractérisation du modèle au cours du temps : Evaluation of potential neuroprotective molecules in an in vivo rat neuroinflammatory model : mechanistic study and time characterization. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2013TOUR3806

Chicago Manual of Style (16th Edition):

Tronel, Claire. “Evaluation des effets de molécules à visée neuroprotectrice dans un modèle in vivo de neuroinflammation chez le rat : étude mécanistique et caractérisation du modèle au cours du temps : Evaluation of potential neuroprotective molecules in an in vivo rat neuroinflammatory model : mechanistic study and time characterization.” 2013. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed March 29, 2020. http://www.theses.fr/2013TOUR3806.

MLA Handbook (7th Edition):

Tronel, Claire. “Evaluation des effets de molécules à visée neuroprotectrice dans un modèle in vivo de neuroinflammation chez le rat : étude mécanistique et caractérisation du modèle au cours du temps : Evaluation of potential neuroprotective molecules in an in vivo rat neuroinflammatory model : mechanistic study and time characterization.” 2013. Web. 29 Mar 2020.

Vancouver:

Tronel C. Evaluation des effets de molécules à visée neuroprotectrice dans un modèle in vivo de neuroinflammation chez le rat : étude mécanistique et caractérisation du modèle au cours du temps : Evaluation of potential neuroprotective molecules in an in vivo rat neuroinflammatory model : mechanistic study and time characterization. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2013. [cited 2020 Mar 29]. Available from: http://www.theses.fr/2013TOUR3806.

Council of Science Editors:

Tronel C. Evaluation des effets de molécules à visée neuroprotectrice dans un modèle in vivo de neuroinflammation chez le rat : étude mécanistique et caractérisation du modèle au cours du temps : Evaluation of potential neuroprotective molecules in an in vivo rat neuroinflammatory model : mechanistic study and time characterization. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2013. Available from: http://www.theses.fr/2013TOUR3806


Université Paris-Sud – Paris XI

7. Lussignol, Marion. Caractérisation d’une nouvelle fonction de la protéine Us11 dans l’échappement à l’autophagie par le virus Herpès Simplex de type 1 : Characterization of a novel function of Us11 protein in HSV-1 escape from autophagy.

Degree: Docteur es, Microbiologie, 2013, Université Paris-Sud – Paris XI

L’autophagie est un mécanisme vacuolaire de dégradation de matériel cytoplasmique permettant le maintien de l’homéostasie cellulaire, mais elle peut être également activée par de nombreux… (more)

Subjects/Keywords: Autophagie; HSV-1; Us11; PKR; ICP34.5; Beclin 1; Autophagy; HSV-1; Us11; PKR; ICP34.5; Beclin 1

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APA (6th Edition):

Lussignol, M. (2013). Caractérisation d’une nouvelle fonction de la protéine Us11 dans l’échappement à l’autophagie par le virus Herpès Simplex de type 1 : Characterization of a novel function of Us11 protein in HSV-1 escape from autophagy. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA114807

Chicago Manual of Style (16th Edition):

Lussignol, Marion. “Caractérisation d’une nouvelle fonction de la protéine Us11 dans l’échappement à l’autophagie par le virus Herpès Simplex de type 1 : Characterization of a novel function of Us11 protein in HSV-1 escape from autophagy.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed March 29, 2020. http://www.theses.fr/2013PA114807.

MLA Handbook (7th Edition):

Lussignol, Marion. “Caractérisation d’une nouvelle fonction de la protéine Us11 dans l’échappement à l’autophagie par le virus Herpès Simplex de type 1 : Characterization of a novel function of Us11 protein in HSV-1 escape from autophagy.” 2013. Web. 29 Mar 2020.

Vancouver:

Lussignol M. Caractérisation d’une nouvelle fonction de la protéine Us11 dans l’échappement à l’autophagie par le virus Herpès Simplex de type 1 : Characterization of a novel function of Us11 protein in HSV-1 escape from autophagy. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2020 Mar 29]. Available from: http://www.theses.fr/2013PA114807.

Council of Science Editors:

Lussignol M. Caractérisation d’une nouvelle fonction de la protéine Us11 dans l’échappement à l’autophagie par le virus Herpès Simplex de type 1 : Characterization of a novel function of Us11 protein in HSV-1 escape from autophagy. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA114807

8. Salah-Mabed, Imène. Descriptions anatomiques et méthodologiques aux fins d'optimisation de techniques de chirurgie cornéenne à visée réfractive : Anatomical and methodological descriptions leading to optimize corneal refractive surgery procedures.

Degree: Docteur es, Milieux dilués et optique fondamentale, 2018, Université Paris-Saclay (ComUE)

Dans un contexte d’augmentation du nombre d’amétropes dans la population mondiale, et en conséquence, de l’accroissement du recours aux techniques de corrections chirurgicales, la compréhension… (more)

Subjects/Keywords: Chirurgie réfractive; Cornée; Myopie; Epithélium; Pupille; LASIK; PKR; Refractive Surgery; Cornea; Myopia; Epithelium; Pupil; LASIK; PKR

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APA (6th Edition):

Salah-Mabed, I. (2018). Descriptions anatomiques et méthodologiques aux fins d'optimisation de techniques de chirurgie cornéenne à visée réfractive : Anatomical and methodological descriptions leading to optimize corneal refractive surgery procedures. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2018SACLS164

Chicago Manual of Style (16th Edition):

Salah-Mabed, Imène. “Descriptions anatomiques et méthodologiques aux fins d'optimisation de techniques de chirurgie cornéenne à visée réfractive : Anatomical and methodological descriptions leading to optimize corneal refractive surgery procedures.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed March 29, 2020. http://www.theses.fr/2018SACLS164.

MLA Handbook (7th Edition):

Salah-Mabed, Imène. “Descriptions anatomiques et méthodologiques aux fins d'optimisation de techniques de chirurgie cornéenne à visée réfractive : Anatomical and methodological descriptions leading to optimize corneal refractive surgery procedures.” 2018. Web. 29 Mar 2020.

Vancouver:

Salah-Mabed I. Descriptions anatomiques et méthodologiques aux fins d'optimisation de techniques de chirurgie cornéenne à visée réfractive : Anatomical and methodological descriptions leading to optimize corneal refractive surgery procedures. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2018. [cited 2020 Mar 29]. Available from: http://www.theses.fr/2018SACLS164.

Council of Science Editors:

Salah-Mabed I. Descriptions anatomiques et méthodologiques aux fins d'optimisation de techniques de chirurgie cornéenne à visée réfractive : Anatomical and methodological descriptions leading to optimize corneal refractive surgery procedures. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2018. Available from: http://www.theses.fr/2018SACLS164

9. Julian, Adrien. Marqueurs périphériques de l'inflammation et déclin cognitif au cours de la maladie d'Alzheimer : Peripheral inflammatory markers and cognitive decline during Alzheimer's disease.

Degree: Docteur es, Aspects Moléculaires et Cellulaires de la Biologie, 2018, Poitiers

Il existe une composante inflammatoire dans la maladie d’Alzheimer (MA), dont le lien avec le déclin cognitif n’est pas scientifiquement démontré. Dans une cohorte de… (more)

Subjects/Keywords: Maladie d'Alzheimer; Cytokines; Pkr; Plasma; Cellules mononucléées sanguines; Inflammation; Déclin cognitif.; Alzheimer's disease; Cytokines; Pkr; PBMCs; Inflammation; Cognitive decline.; 616.831

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APA (6th Edition):

Julian, A. (2018). Marqueurs périphériques de l'inflammation et déclin cognitif au cours de la maladie d'Alzheimer : Peripheral inflammatory markers and cognitive decline during Alzheimer's disease. (Doctoral Dissertation). Poitiers. Retrieved from http://www.theses.fr/2018POIT1406

Chicago Manual of Style (16th Edition):

Julian, Adrien. “Marqueurs périphériques de l'inflammation et déclin cognitif au cours de la maladie d'Alzheimer : Peripheral inflammatory markers and cognitive decline during Alzheimer's disease.” 2018. Doctoral Dissertation, Poitiers. Accessed March 29, 2020. http://www.theses.fr/2018POIT1406.

MLA Handbook (7th Edition):

Julian, Adrien. “Marqueurs périphériques de l'inflammation et déclin cognitif au cours de la maladie d'Alzheimer : Peripheral inflammatory markers and cognitive decline during Alzheimer's disease.” 2018. Web. 29 Mar 2020.

Vancouver:

Julian A. Marqueurs périphériques de l'inflammation et déclin cognitif au cours de la maladie d'Alzheimer : Peripheral inflammatory markers and cognitive decline during Alzheimer's disease. [Internet] [Doctoral dissertation]. Poitiers; 2018. [cited 2020 Mar 29]. Available from: http://www.theses.fr/2018POIT1406.

Council of Science Editors:

Julian A. Marqueurs périphériques de l'inflammation et déclin cognitif au cours de la maladie d'Alzheimer : Peripheral inflammatory markers and cognitive decline during Alzheimer's disease. [Doctoral Dissertation]. Poitiers; 2018. Available from: http://www.theses.fr/2018POIT1406


University of South Carolina

10. Vaughn, Lauren S. Regulation of Stress Response and Innate Immunity by DS<b>RNA</b>-Binding Proteins PACT and TRBP.

Degree: PhD, Biological Sciences, 2015, University of South Carolina

  An integral aspect of innate immune response to viral infections is the ability to detect non-self molecules to initiate antiviral signaling via pattern recognition… (more)

Subjects/Keywords: Biology; Life Sciences; dystonia; Interferon; PACT; PKR; RIG-I; TRBP

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APA (6th Edition):

Vaughn, L. S. (2015). Regulation of Stress Response and Innate Immunity by DS<b>RNA</b>-Binding Proteins PACT and TRBP. (Doctoral Dissertation). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/3219

Chicago Manual of Style (16th Edition):

Vaughn, Lauren S. “Regulation of Stress Response and Innate Immunity by DS<b>RNA</b>-Binding Proteins PACT and TRBP.” 2015. Doctoral Dissertation, University of South Carolina. Accessed March 29, 2020. https://scholarcommons.sc.edu/etd/3219.

MLA Handbook (7th Edition):

Vaughn, Lauren S. “Regulation of Stress Response and Innate Immunity by DS<b>RNA</b>-Binding Proteins PACT and TRBP.” 2015. Web. 29 Mar 2020.

Vancouver:

Vaughn LS. Regulation of Stress Response and Innate Immunity by DS<b>RNA</b>-Binding Proteins PACT and TRBP. [Internet] [Doctoral dissertation]. University of South Carolina; 2015. [cited 2020 Mar 29]. Available from: https://scholarcommons.sc.edu/etd/3219.

Council of Science Editors:

Vaughn LS. Regulation of Stress Response and Innate Immunity by DS<b>RNA</b>-Binding Proteins PACT and TRBP. [Doctoral Dissertation]. University of South Carolina; 2015. Available from: https://scholarcommons.sc.edu/etd/3219


North Carolina State University

11. Stewart, Michael Jude. PKR, Myocarditis and the Cardiac Response to Reovirus Infection.

Degree: PhD, Microbiology, 2004, North Carolina State University

 Viral myocarditis is an important human disease associated with many viruses. Mechanistically, cardiac damage associated with viral myocarditis can be immune mediated and/or the result… (more)

Subjects/Keywords: reovirus; myocarditis; PKR; interferon

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APA (6th Edition):

Stewart, M. J. (2004). PKR, Myocarditis and the Cardiac Response to Reovirus Infection. (Doctoral Dissertation). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/3338

Chicago Manual of Style (16th Edition):

Stewart, Michael Jude. “PKR, Myocarditis and the Cardiac Response to Reovirus Infection.” 2004. Doctoral Dissertation, North Carolina State University. Accessed March 29, 2020. http://www.lib.ncsu.edu/resolver/1840.16/3338.

MLA Handbook (7th Edition):

Stewart, Michael Jude. “PKR, Myocarditis and the Cardiac Response to Reovirus Infection.” 2004. Web. 29 Mar 2020.

Vancouver:

Stewart MJ. PKR, Myocarditis and the Cardiac Response to Reovirus Infection. [Internet] [Doctoral dissertation]. North Carolina State University; 2004. [cited 2020 Mar 29]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/3338.

Council of Science Editors:

Stewart MJ. PKR, Myocarditis and the Cardiac Response to Reovirus Infection. [Doctoral Dissertation]. North Carolina State University; 2004. Available from: http://www.lib.ncsu.edu/resolver/1840.16/3338


Penn State University

12. Toroney, Rebecca. Investigating Roles of Unconventional RNA Primary, Secondary, and Tertiary Motifs in Regulation of the Protein Kinase PKR.

Degree: PhD, Chemistry, 2010, Penn State University

 Much is known about the importance of protein folding and structure in human health. Like protein, RNA is a biopolymer essential to cellular function that… (more)

Subjects/Keywords: Hepatitis C Virus; RNA-protein interactions; IRES; PKR; innate immunity; triphosphate

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APA (6th Edition):

Toroney, R. (2010). Investigating Roles of Unconventional RNA Primary, Secondary, and Tertiary Motifs in Regulation of the Protein Kinase PKR. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11499

Chicago Manual of Style (16th Edition):

Toroney, Rebecca. “Investigating Roles of Unconventional RNA Primary, Secondary, and Tertiary Motifs in Regulation of the Protein Kinase PKR.” 2010. Doctoral Dissertation, Penn State University. Accessed March 29, 2020. https://etda.libraries.psu.edu/catalog/11499.

MLA Handbook (7th Edition):

Toroney, Rebecca. “Investigating Roles of Unconventional RNA Primary, Secondary, and Tertiary Motifs in Regulation of the Protein Kinase PKR.” 2010. Web. 29 Mar 2020.

Vancouver:

Toroney R. Investigating Roles of Unconventional RNA Primary, Secondary, and Tertiary Motifs in Regulation of the Protein Kinase PKR. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2020 Mar 29]. Available from: https://etda.libraries.psu.edu/catalog/11499.

Council of Science Editors:

Toroney R. Investigating Roles of Unconventional RNA Primary, Secondary, and Tertiary Motifs in Regulation of the Protein Kinase PKR. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11499


University of Illinois – Urbana-Champaign

13. Willis, Kristen L. Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein.

Degree: PhD, 0322, 2011, University of Illinois – Urbana-Champaign

 Vaccinia virus (VV), a member of the poxvirus family of double-stranded DNA viruses, is well-known as a highly effective vaccine against variola virus, the causative… (more)

Subjects/Keywords: vaccinia virus; protein kinase (PKR); NF-kappaB; K1 protein

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APA (6th Edition):

Willis, K. L. (2011). Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/18499

Chicago Manual of Style (16th Edition):

Willis, Kristen L. “Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein.” 2011. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed March 29, 2020. http://hdl.handle.net/2142/18499.

MLA Handbook (7th Edition):

Willis, Kristen L. “Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein.” 2011. Web. 29 Mar 2020.

Vancouver:

Willis KL. Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2011. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/2142/18499.

Council of Science Editors:

Willis KL. Characterization of the anti-viral effects inhibited by the vaccinia virus K1 protein. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2011. Available from: http://hdl.handle.net/2142/18499


Case Western Reserve University

14. Li, Shoudong. Modulations of PACT-PKR Pathway by Cellular Stresses and the NS1 Protein of Influenza A Virus.

Degree: PhD, Molecular Virology, 2005, Case Western Reserve University

 PACT contains three modular domains. While PACT domains 1 and 2 can mediate strong interactions with PKR and dsRNA, the C-terminal domain 3 binds to… (more)

Subjects/Keywords: Biology, Molecular; PKR; PACT; NS1; Activation of PKR; dsRNA; PACT domain; PROTEIN

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APA (6th Edition):

Li, S. (2005). Modulations of PACT-PKR Pathway by Cellular Stresses and the NS1 Protein of Influenza A Virus. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1114788508

Chicago Manual of Style (16th Edition):

Li, Shoudong. “Modulations of PACT-PKR Pathway by Cellular Stresses and the NS1 Protein of Influenza A Virus.” 2005. Doctoral Dissertation, Case Western Reserve University. Accessed March 29, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1114788508.

MLA Handbook (7th Edition):

Li, Shoudong. “Modulations of PACT-PKR Pathway by Cellular Stresses and the NS1 Protein of Influenza A Virus.” 2005. Web. 29 Mar 2020.

Vancouver:

Li S. Modulations of PACT-PKR Pathway by Cellular Stresses and the NS1 Protein of Influenza A Virus. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2005. [cited 2020 Mar 29]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1114788508.

Council of Science Editors:

Li S. Modulations of PACT-PKR Pathway by Cellular Stresses and the NS1 Protein of Influenza A Virus. [Doctoral Dissertation]. Case Western Reserve University; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1114788508


Université de Lorraine

15. Khoury, Georges. Étude des mécanismes moléculaires régulant l'expression de la protéine TAT du virus de l'immunodéficience humaine, au niveau de la production de ses ARN messagers et de leur traduction : Study of molecular mechanisms regulating the expression of tat protein of immunodeficiency human virus at the production of its messenger RNA and their translation.

Degree: Docteur es, Sciences de la vie et de la santé, 2012, Université de Lorraine

La protéine Tat du VIH-1 est essentielle à la multiplication virale. Elle permet la transactivation de la transcription et, par ses propriétés apoptotiques, elle participe… (more)

Subjects/Keywords: VIH-1; Protéine Tat; Épissage; Protéines SR et hnRNP; Traduction; Protéine PKR; HIV-1; Tat protein; Splicing; SR and hnRNP proteins; Translation; PKR; 572.6

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APA (6th Edition):

Khoury, G. (2012). Étude des mécanismes moléculaires régulant l'expression de la protéine TAT du virus de l'immunodéficience humaine, au niveau de la production de ses ARN messagers et de leur traduction : Study of molecular mechanisms regulating the expression of tat protein of immunodeficiency human virus at the production of its messenger RNA and their translation. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2012LORR0251

Chicago Manual of Style (16th Edition):

Khoury, Georges. “Étude des mécanismes moléculaires régulant l'expression de la protéine TAT du virus de l'immunodéficience humaine, au niveau de la production de ses ARN messagers et de leur traduction : Study of molecular mechanisms regulating the expression of tat protein of immunodeficiency human virus at the production of its messenger RNA and their translation.” 2012. Doctoral Dissertation, Université de Lorraine. Accessed March 29, 2020. http://www.theses.fr/2012LORR0251.

MLA Handbook (7th Edition):

Khoury, Georges. “Étude des mécanismes moléculaires régulant l'expression de la protéine TAT du virus de l'immunodéficience humaine, au niveau de la production de ses ARN messagers et de leur traduction : Study of molecular mechanisms regulating the expression of tat protein of immunodeficiency human virus at the production of its messenger RNA and their translation.” 2012. Web. 29 Mar 2020.

Vancouver:

Khoury G. Étude des mécanismes moléculaires régulant l'expression de la protéine TAT du virus de l'immunodéficience humaine, au niveau de la production de ses ARN messagers et de leur traduction : Study of molecular mechanisms regulating the expression of tat protein of immunodeficiency human virus at the production of its messenger RNA and their translation. [Internet] [Doctoral dissertation]. Université de Lorraine; 2012. [cited 2020 Mar 29]. Available from: http://www.theses.fr/2012LORR0251.

Council of Science Editors:

Khoury G. Étude des mécanismes moléculaires régulant l'expression de la protéine TAT du virus de l'immunodéficience humaine, au niveau de la production de ses ARN messagers et de leur traduction : Study of molecular mechanisms regulating the expression of tat protein of immunodeficiency human virus at the production of its messenger RNA and their translation. [Doctoral Dissertation]. Université de Lorraine; 2012. Available from: http://www.theses.fr/2012LORR0251


University of South Carolina

16. Ragin, Monica Yvette. The Potential Role of Insulin (and Stress Response) Pathway Components In Breast Cancer Development and Progression.

Degree: PhD, Biological Sciences, 2012, University of South Carolina

  Breast cancer is characterized into several subtypes, which are further classified by the status of three central receptors: estrogen (ER), progesterone (PR), and human… (more)

Subjects/Keywords: Life Sciences; Medicine and Health Sciences; Physical Sciences and Mathematics; insulin; PACT; PKR; SNPs

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APA (6th Edition):

Ragin, M. Y. (2012). The Potential Role of Insulin (and Stress Response) Pathway Components In Breast Cancer Development and Progression. (Doctoral Dissertation). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/514

Chicago Manual of Style (16th Edition):

Ragin, Monica Yvette. “The Potential Role of Insulin (and Stress Response) Pathway Components In Breast Cancer Development and Progression.” 2012. Doctoral Dissertation, University of South Carolina. Accessed March 29, 2020. https://scholarcommons.sc.edu/etd/514.

MLA Handbook (7th Edition):

Ragin, Monica Yvette. “The Potential Role of Insulin (and Stress Response) Pathway Components In Breast Cancer Development and Progression.” 2012. Web. 29 Mar 2020.

Vancouver:

Ragin MY. The Potential Role of Insulin (and Stress Response) Pathway Components In Breast Cancer Development and Progression. [Internet] [Doctoral dissertation]. University of South Carolina; 2012. [cited 2020 Mar 29]. Available from: https://scholarcommons.sc.edu/etd/514.

Council of Science Editors:

Ragin MY. The Potential Role of Insulin (and Stress Response) Pathway Components In Breast Cancer Development and Progression. [Doctoral Dissertation]. University of South Carolina; 2012. Available from: https://scholarcommons.sc.edu/etd/514


Kent State University

17. Chakrabarti, Arindam. PKR DEPENDENT UPREGULATION OF IMMEDIATE EARLY GENES AND ANTI-INFLAMMATORY CYTOKINE IL-10.

Degree: PhD, College of Biomedical Sciences, 2007, Kent State University

 Viral infection induces expression and activation of several genes involved directly or indirectly in antiviral defense. Protein kinase R (PKR), a Ser/Thr kinase induced by… (more)

Subjects/Keywords: Biology, Molecular; PKR; IL-10; c-jun; egr-1; immediate early genes

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APA (6th Edition):

Chakrabarti, A. (2007). PKR DEPENDENT UPREGULATION OF IMMEDIATE EARLY GENES AND ANTI-INFLAMMATORY CYTOKINE IL-10. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1176136341

Chicago Manual of Style (16th Edition):

Chakrabarti, Arindam. “PKR DEPENDENT UPREGULATION OF IMMEDIATE EARLY GENES AND ANTI-INFLAMMATORY CYTOKINE IL-10.” 2007. Doctoral Dissertation, Kent State University. Accessed March 29, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=kent1176136341.

MLA Handbook (7th Edition):

Chakrabarti, Arindam. “PKR DEPENDENT UPREGULATION OF IMMEDIATE EARLY GENES AND ANTI-INFLAMMATORY CYTOKINE IL-10.” 2007. Web. 29 Mar 2020.

Vancouver:

Chakrabarti A. PKR DEPENDENT UPREGULATION OF IMMEDIATE EARLY GENES AND ANTI-INFLAMMATORY CYTOKINE IL-10. [Internet] [Doctoral dissertation]. Kent State University; 2007. [cited 2020 Mar 29]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1176136341.

Council of Science Editors:

Chakrabarti A. PKR DEPENDENT UPREGULATION OF IMMEDIATE EARLY GENES AND ANTI-INFLAMMATORY CYTOKINE IL-10. [Doctoral Dissertation]. Kent State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1176136341

18. Carret-Rebillat, Anne-Sophie. Contrôle de la neuroinflammation par la kinase PKR dans les processus pathologiques de la maladie d'Alzheimer : Involvement of PKR-mediated inflammation in Alzheimer's disease pathology.

Degree: Docteur es, Neurosciences, 2014, Université Pierre et Marie Curie – Paris VI

La maladie d’Alzheimer (MA) est la pathologie neurodégénérative entraînant une démence la plus fréquente. Elle touche plus de 3% des plus de 65 ans. Les… (more)

Subjects/Keywords: Maladie d'Alzheimer; Inflammation systémique; Neuroinflammation; Endotoxémie; Pkr; Amyloïdogénèse; Alzheimer's disease; Endotoxemia; 616.8

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APA (6th Edition):

Carret-Rebillat, A. (2014). Contrôle de la neuroinflammation par la kinase PKR dans les processus pathologiques de la maladie d'Alzheimer : Involvement of PKR-mediated inflammation in Alzheimer's disease pathology. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2014PA066442

Chicago Manual of Style (16th Edition):

Carret-Rebillat, Anne-Sophie. “Contrôle de la neuroinflammation par la kinase PKR dans les processus pathologiques de la maladie d'Alzheimer : Involvement of PKR-mediated inflammation in Alzheimer's disease pathology.” 2014. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed March 29, 2020. http://www.theses.fr/2014PA066442.

MLA Handbook (7th Edition):

Carret-Rebillat, Anne-Sophie. “Contrôle de la neuroinflammation par la kinase PKR dans les processus pathologiques de la maladie d'Alzheimer : Involvement of PKR-mediated inflammation in Alzheimer's disease pathology.” 2014. Web. 29 Mar 2020.

Vancouver:

Carret-Rebillat A. Contrôle de la neuroinflammation par la kinase PKR dans les processus pathologiques de la maladie d'Alzheimer : Involvement of PKR-mediated inflammation in Alzheimer's disease pathology. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. [cited 2020 Mar 29]. Available from: http://www.theses.fr/2014PA066442.

Council of Science Editors:

Carret-Rebillat A. Contrôle de la neuroinflammation par la kinase PKR dans les processus pathologiques de la maladie d'Alzheimer : Involvement of PKR-mediated inflammation in Alzheimer's disease pathology. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. Available from: http://www.theses.fr/2014PA066442


Freie Universität Berlin

19. Zielecki, Florian. Analysing molecular virulence determinants of the viral NS1 protein of an avian H5N1 influenza A virus.

Degree: 2010, Freie Universität Berlin

 Summary Highly pathogenic avian influenza viruses (HPAIV) of the subtype H5N1 have caused a high mortality rate of about 60% in humans. Due to the… (more)

Subjects/Keywords: Influenza; H5N1; PDZ; PKR; NS1; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie

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APA (6th Edition):

Zielecki, F. (2010). Analysing molecular virulence determinants of the viral NS1 protein of an avian H5N1 influenza A virus. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-6652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zielecki, Florian. “Analysing molecular virulence determinants of the viral NS1 protein of an avian H5N1 influenza A virus.” 2010. Thesis, Freie Universität Berlin. Accessed March 29, 2020. http://dx.doi.org/10.17169/refubium-6652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zielecki, Florian. “Analysing molecular virulence determinants of the viral NS1 protein of an avian H5N1 influenza A virus.” 2010. Web. 29 Mar 2020.

Vancouver:

Zielecki F. Analysing molecular virulence determinants of the viral NS1 protein of an avian H5N1 influenza A virus. [Internet] [Thesis]. Freie Universität Berlin; 2010. [cited 2020 Mar 29]. Available from: http://dx.doi.org/10.17169/refubium-6652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zielecki F. Analysing molecular virulence determinants of the viral NS1 protein of an avian H5N1 influenza A virus. [Thesis]. Freie Universität Berlin; 2010. Available from: http://dx.doi.org/10.17169/refubium-6652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Michigan

20. Goodman, Danielle. The Roles of eIF2 Kinases PKR and GCN2 during Mouse Adenovirus Type 1 Infection.

Degree: PhD, Cellular & Molecular Biology, 2019, University of Michigan

 During viral infection, a major innate host defense mechanism is to reduce global protein synthesis by phosphorylation of eukaryotic translation inhibition factor 2a (eIF2a). eIF2a… (more)

Subjects/Keywords: PKR; GCN2; adenovirus; host-virus interactions; eIF2alpha kinase; proteasome; Microbiology and Immunology; Science

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APA (6th Edition):

Goodman, D. (2019). The Roles of eIF2 Kinases PKR and GCN2 during Mouse Adenovirus Type 1 Infection. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/150028

Chicago Manual of Style (16th Edition):

Goodman, Danielle. “The Roles of eIF2 Kinases PKR and GCN2 during Mouse Adenovirus Type 1 Infection.” 2019. Doctoral Dissertation, University of Michigan. Accessed March 29, 2020. http://hdl.handle.net/2027.42/150028.

MLA Handbook (7th Edition):

Goodman, Danielle. “The Roles of eIF2 Kinases PKR and GCN2 during Mouse Adenovirus Type 1 Infection.” 2019. Web. 29 Mar 2020.

Vancouver:

Goodman D. The Roles of eIF2 Kinases PKR and GCN2 during Mouse Adenovirus Type 1 Infection. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/2027.42/150028.

Council of Science Editors:

Goodman D. The Roles of eIF2 Kinases PKR and GCN2 during Mouse Adenovirus Type 1 Infection. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/150028


Georgia State University

21. Elbahesh, Husni M. Study of Innate Immune Response Components in West Nile Virus Infected Cells.

Degree: PhD, Biology, 2011, Georgia State University

              Two cellular innate responses, the dsRNA protein kinase (PKR) pathway and the 2'-5' oligoadenylate synthetase (OAS)/RNase L pathway, are activated by dsRNAs produced… (more)

Subjects/Keywords: West Nile virus; PKR; dsRNA; 2'-5' oligoadenylate synthetase; Oas1b; EIF2α; Biology

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APA (6th Edition):

Elbahesh, H. M. (2011). Study of Innate Immune Response Components in West Nile Virus Infected Cells. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/94

Chicago Manual of Style (16th Edition):

Elbahesh, Husni M. “Study of Innate Immune Response Components in West Nile Virus Infected Cells.” 2011. Doctoral Dissertation, Georgia State University. Accessed March 29, 2020. https://scholarworks.gsu.edu/biology_diss/94.

MLA Handbook (7th Edition):

Elbahesh, Husni M. “Study of Innate Immune Response Components in West Nile Virus Infected Cells.” 2011. Web. 29 Mar 2020.

Vancouver:

Elbahesh HM. Study of Innate Immune Response Components in West Nile Virus Infected Cells. [Internet] [Doctoral dissertation]. Georgia State University; 2011. [cited 2020 Mar 29]. Available from: https://scholarworks.gsu.edu/biology_diss/94.

Council of Science Editors:

Elbahesh HM. Study of Innate Immune Response Components in West Nile Virus Infected Cells. [Doctoral Dissertation]. Georgia State University; 2011. Available from: https://scholarworks.gsu.edu/biology_diss/94


University of Washington

22. Braggin, Jacquelyn E. Essential Role of Protein Kinase R Antagonism by TRS1 in Human Cytomegalovirus Replication.

Degree: PhD, 2016, University of Washington

 Human cytomegalovirus (HCMV) lacking TRS1 and IRS1 (HCMV[ΔI/ΔT]) cannot replicate in cell culture. Although both proteins can block the protein kinase R (PKR) pathway, they… (more)

Subjects/Keywords: cytomegalovirus; double-stranded RNA; host defense; PKR; translation; TRS1; Virology; Microbiology; Immunology; microbiology

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APA (6th Edition):

Braggin, J. E. (2016). Essential Role of Protein Kinase R Antagonism by TRS1 in Human Cytomegalovirus Replication. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/35260

Chicago Manual of Style (16th Edition):

Braggin, Jacquelyn E. “Essential Role of Protein Kinase R Antagonism by TRS1 in Human Cytomegalovirus Replication.” 2016. Doctoral Dissertation, University of Washington. Accessed March 29, 2020. http://hdl.handle.net/1773/35260.

MLA Handbook (7th Edition):

Braggin, Jacquelyn E. “Essential Role of Protein Kinase R Antagonism by TRS1 in Human Cytomegalovirus Replication.” 2016. Web. 29 Mar 2020.

Vancouver:

Braggin JE. Essential Role of Protein Kinase R Antagonism by TRS1 in Human Cytomegalovirus Replication. [Internet] [Doctoral dissertation]. University of Washington; 2016. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/1773/35260.

Council of Science Editors:

Braggin JE. Essential Role of Protein Kinase R Antagonism by TRS1 in Human Cytomegalovirus Replication. [Doctoral Dissertation]. University of Washington; 2016. Available from: http://hdl.handle.net/1773/35260


University of Toronto

23. Li, John Junyi. Inhibition of eIF2α Kinases by Viral and Small Molecule Inhibitors.

Degree: PhD, 2016, University of Toronto

 Phosphorylation of the alpha subunit of the eukaryotic initiation factor 2 (eIF2α) on Ser51 is a key event in the eukaryotic stress response pathway. Specifically,… (more)

Subjects/Keywords: antimalarial kinase inhibitor; eIF2α kinase; lobe-swap; PK2; PKR; HRI; PK4; viral mimicry; 0487

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APA (6th Edition):

Li, J. J. (2016). Inhibition of eIF2α Kinases by Viral and Small Molecule Inhibitors. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/76440

Chicago Manual of Style (16th Edition):

Li, John Junyi. “Inhibition of eIF2α Kinases by Viral and Small Molecule Inhibitors.” 2016. Doctoral Dissertation, University of Toronto. Accessed March 29, 2020. http://hdl.handle.net/1807/76440.

MLA Handbook (7th Edition):

Li, John Junyi. “Inhibition of eIF2α Kinases by Viral and Small Molecule Inhibitors.” 2016. Web. 29 Mar 2020.

Vancouver:

Li JJ. Inhibition of eIF2α Kinases by Viral and Small Molecule Inhibitors. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/1807/76440.

Council of Science Editors:

Li JJ. Inhibition of eIF2α Kinases by Viral and Small Molecule Inhibitors. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/76440


Kyoto University / 京都大学

24. Yoo, Ji Seung. INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING : PKRによって誘導される抗ウイルスストレス顆粒とRIG-IによるシグナルにおけるDHX36の機能の研究.

Degree: 博士(生命科学), 2014, Kyoto University / 京都大学

新制・課程博士

甲第18485号

生博第314号

Subjects/Keywords: DHX36; RIG-I; PKR; Stress Granule; Innate Immunity

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APA (6th Edition):

Yoo, J. S. (2014). INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING : PKRによって誘導される抗ウイルスストレス顆粒とRIG-IによるシグナルにおけるDHX36の機能の研究. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/189378 ; http://dx.doi.org/10.14989/doctor.k18485

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yoo, Ji Seung. “INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING : PKRによって誘導される抗ウイルスストレス顆粒とRIG-IによるシグナルにおけるDHX36の機能の研究.” 2014. Thesis, Kyoto University / 京都大学. Accessed March 29, 2020. http://hdl.handle.net/2433/189378 ; http://dx.doi.org/10.14989/doctor.k18485.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yoo, Ji Seung. “INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING : PKRによって誘導される抗ウイルスストレス顆粒とRIG-IによるシグナルにおけるDHX36の機能の研究.” 2014. Web. 29 Mar 2020.

Vancouver:

Yoo JS. INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING : PKRによって誘導される抗ウイルスストレス顆粒とRIG-IによるシグナルにおけるDHX36の機能の研究. [Internet] [Thesis]. Kyoto University / 京都大学; 2014. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/2433/189378 ; http://dx.doi.org/10.14989/doctor.k18485.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yoo JS. INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING : PKRによって誘導される抗ウイルスストレス顆粒とRIG-IによるシグナルにおけるDHX36の機能の研究. [Thesis]. Kyoto University / 京都大学; 2014. Available from: http://hdl.handle.net/2433/189378 ; http://dx.doi.org/10.14989/doctor.k18485

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of South Carolina

25. Singh, Madhurima. Regulation of Pkr Activation by Pact and Trbp In Response to Stress Signals.

Degree: PhD, Biological Sciences, 2011, University of South Carolina

PKR (protein kinase, RNA activated) is an interferon (IFN)-induced serine-threonine protein kinase, which plays a crucial role in IFN's antiviral and antiproliferative actions. In… (more)

Subjects/Keywords: Life Sciences; Medicine and Health Sciences; Physical Sciences and Mathematics; apoptosis; eIF2alpha; PACT; PKR; Stress; TRBP

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APA (6th Edition):

Singh, M. (2011). Regulation of Pkr Activation by Pact and Trbp In Response to Stress Signals. (Doctoral Dissertation). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/517

Chicago Manual of Style (16th Edition):

Singh, Madhurima. “Regulation of Pkr Activation by Pact and Trbp In Response to Stress Signals.” 2011. Doctoral Dissertation, University of South Carolina. Accessed March 29, 2020. https://scholarcommons.sc.edu/etd/517.

MLA Handbook (7th Edition):

Singh, Madhurima. “Regulation of Pkr Activation by Pact and Trbp In Response to Stress Signals.” 2011. Web. 29 Mar 2020.

Vancouver:

Singh M. Regulation of Pkr Activation by Pact and Trbp In Response to Stress Signals. [Internet] [Doctoral dissertation]. University of South Carolina; 2011. [cited 2020 Mar 29]. Available from: https://scholarcommons.sc.edu/etd/517.

Council of Science Editors:

Singh M. Regulation of Pkr Activation by Pact and Trbp In Response to Stress Signals. [Doctoral Dissertation]. University of South Carolina; 2011. Available from: https://scholarcommons.sc.edu/etd/517


University of South Carolina

26. Chukwurah, Evelyn E. Modulation Of PKR Activity During HIV Infection And Cellular Stress By PACT And TRBP.

Degree: PhD, Biological Sciences, 2017, University of South Carolina

  A crucial component of the cellular response to stress is the attenuation of protein synthesis to allow the cell to dedicate resources for the… (more)

Subjects/Keywords: Biological Phenomena, Cell Phenomena, and Immunity; Medical Sciences; Medicine and Health Sciences; Modulation; PKR Activity; HIV Infection; Cellular Stress; PACT; TRBP

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chukwurah, E. E. (2017). Modulation Of PKR Activity During HIV Infection And Cellular Stress By PACT And TRBP. (Doctoral Dissertation). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/4497

Chicago Manual of Style (16th Edition):

Chukwurah, Evelyn E. “Modulation Of PKR Activity During HIV Infection And Cellular Stress By PACT And TRBP.” 2017. Doctoral Dissertation, University of South Carolina. Accessed March 29, 2020. https://scholarcommons.sc.edu/etd/4497.

MLA Handbook (7th Edition):

Chukwurah, Evelyn E. “Modulation Of PKR Activity During HIV Infection And Cellular Stress By PACT And TRBP.” 2017. Web. 29 Mar 2020.

Vancouver:

Chukwurah EE. Modulation Of PKR Activity During HIV Infection And Cellular Stress By PACT And TRBP. [Internet] [Doctoral dissertation]. University of South Carolina; 2017. [cited 2020 Mar 29]. Available from: https://scholarcommons.sc.edu/etd/4497.

Council of Science Editors:

Chukwurah EE. Modulation Of PKR Activity During HIV Infection And Cellular Stress By PACT And TRBP. [Doctoral Dissertation]. University of South Carolina; 2017. Available from: https://scholarcommons.sc.edu/etd/4497


University of Pennsylvania

27. Anderson, Bart R. Nucleoside Modifications Suppress RNA Activation of Cytoplasmic RNA Sensors.

Degree: 2010, University of Pennsylvania

 Multiple innate defense pathways exist to recognize and defend against foreign nucleic acids. Unlike innate immune receptors that recognize structures specific for pathogens that are… (more)

Subjects/Keywords: RNA-dependent protein kinase R (PKR); Oligoadenylate synthetase (OAS); Ribonuclease L (RNase L); pseudouridine; innate immunity; nucleofector; Molecular Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Anderson, B. R. (2010). Nucleoside Modifications Suppress RNA Activation of Cytoplasmic RNA Sensors. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anderson, Bart R. “Nucleoside Modifications Suppress RNA Activation of Cytoplasmic RNA Sensors.” 2010. Thesis, University of Pennsylvania. Accessed March 29, 2020. https://repository.upenn.edu/edissertations/1567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anderson, Bart R. “Nucleoside Modifications Suppress RNA Activation of Cytoplasmic RNA Sensors.” 2010. Web. 29 Mar 2020.

Vancouver:

Anderson BR. Nucleoside Modifications Suppress RNA Activation of Cytoplasmic RNA Sensors. [Internet] [Thesis]. University of Pennsylvania; 2010. [cited 2020 Mar 29]. Available from: https://repository.upenn.edu/edissertations/1567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anderson BR. Nucleoside Modifications Suppress RNA Activation of Cytoplasmic RNA Sensors. [Thesis]. University of Pennsylvania; 2010. Available from: https://repository.upenn.edu/edissertations/1567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

28. Barkati, Sapha. Étude de la sensibilité à l'infection de lignées cancéreuses humaines par différents isolats de réovirus .

Degree: 2006, Université de Montréal

Subjects/Keywords: Réovirus; Ras; PKR; Interféron; Cystéines protéases; Décapsidation; Oncolyse; Cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Barkati, S. (2006). Étude de la sensibilité à l'infection de lignées cancéreuses humaines par différents isolats de réovirus . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/15176

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barkati, Sapha. “Étude de la sensibilité à l'infection de lignées cancéreuses humaines par différents isolats de réovirus .” 2006. Thesis, Université de Montréal. Accessed March 29, 2020. http://hdl.handle.net/1866/15176.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barkati, Sapha. “Étude de la sensibilité à l'infection de lignées cancéreuses humaines par différents isolats de réovirus .” 2006. Web. 29 Mar 2020.

Vancouver:

Barkati S. Étude de la sensibilité à l'infection de lignées cancéreuses humaines par différents isolats de réovirus . [Internet] [Thesis]. Université de Montréal; 2006. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/1866/15176.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barkati S. Étude de la sensibilité à l'infection de lignées cancéreuses humaines par différents isolats de réovirus . [Thesis]. Université de Montréal; 2006. Available from: http://hdl.handle.net/1866/15176

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

29. Yoo, Ji Seung. INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING .

Degree: 2014, Kyoto University

Subjects/Keywords: DHX36; RIG-I; PKR; Stress Granule; Innate Immunity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yoo, J. S. (2014). INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/189378

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yoo, Ji Seung. “INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING .” 2014. Thesis, Kyoto University. Accessed March 29, 2020. http://hdl.handle.net/2433/189378.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yoo, Ji Seung. “INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING .” 2014. Web. 29 Mar 2020.

Vancouver:

Yoo JS. INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING . [Internet] [Thesis]. Kyoto University; 2014. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/2433/189378.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yoo JS. INVESTIGATIONS INTO THE ROLES OF PKR-INDUCED ANTIVIRAL STRESS GRANULE AND DHX36 IN RIG-I SIGNALING . [Thesis]. Kyoto University; 2014. Available from: http://hdl.handle.net/2433/189378

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Cooper, Angie. Long-chain fatty acids and endoplasmic reticulum stress in pancreatic beta-cells : the role of Protein Kinase R (PKR).

Degree: PhD, 2013, University of Plymouth

 Type 2 diabetes (T2D) is a growing health-care and economic burden. Obesity is a risk factor for developing T2D, but the underlying molecular mechanisms are… (more)

Subjects/Keywords: 616.4; PKR, ER stress, beta-cells

…apoptotic pathways 1.10 45 PERK: function and role in ER stress 8 47 1.11 PKR: structure and… …function 1.11.1 Proposed models of PKR activation 1.11.2 PKR dsRNA-independent activatory… …mechanisms 1.11.3 PKR as a proapoptotic molecule 49 52 55 55 1.12 Innate immunity and… …with antibodies 2.6 91 94 RNAi 95 2.6.1 PKR inhibition by adenoviral mutant PKR (Ad… …PKR knockdown using shRNA 97 2.6.4 Transformation and plasmid purification using shRNA 98… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cooper, A. (2013). Long-chain fatty acids and endoplasmic reticulum stress in pancreatic beta-cells : the role of Protein Kinase R (PKR). (Doctoral Dissertation). University of Plymouth. Retrieved from http://hdl.handle.net/10026.1/2851

Chicago Manual of Style (16th Edition):

Cooper, Angie. “Long-chain fatty acids and endoplasmic reticulum stress in pancreatic beta-cells : the role of Protein Kinase R (PKR).” 2013. Doctoral Dissertation, University of Plymouth. Accessed March 29, 2020. http://hdl.handle.net/10026.1/2851.

MLA Handbook (7th Edition):

Cooper, Angie. “Long-chain fatty acids and endoplasmic reticulum stress in pancreatic beta-cells : the role of Protein Kinase R (PKR).” 2013. Web. 29 Mar 2020.

Vancouver:

Cooper A. Long-chain fatty acids and endoplasmic reticulum stress in pancreatic beta-cells : the role of Protein Kinase R (PKR). [Internet] [Doctoral dissertation]. University of Plymouth; 2013. [cited 2020 Mar 29]. Available from: http://hdl.handle.net/10026.1/2851.

Council of Science Editors:

Cooper A. Long-chain fatty acids and endoplasmic reticulum stress in pancreatic beta-cells : the role of Protein Kinase R (PKR). [Doctoral Dissertation]. University of Plymouth; 2013. Available from: http://hdl.handle.net/10026.1/2851

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