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You searched for subject:(PI3K). Showing records 1 – 30 of 322 total matches.

[1] [2] [3] [4] [5] … [11]

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University of Southern California

1. Ramanathan, Anita. Defining function of an autism pathway in human neuronal cells.

Degree: MS, Biochemistry and Molecular Biology, 2011, University of Southern California

 Autism is a neurodevelopmental disorder that affects 1% of the population and causes deficits in social interaction, communication and behavioral flexibility. Autism is highly heritable,… (more)

Subjects/Keywords: autism; luminex; PI3K signaling; PI3K Akt pathway

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APA (6th Edition):

Ramanathan, A. (2011). Defining function of an autism pathway in human neuronal cells. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1816

Chicago Manual of Style (16th Edition):

Ramanathan, Anita. “Defining function of an autism pathway in human neuronal cells.” 2011. Masters Thesis, University of Southern California. Accessed February 25, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1816.

MLA Handbook (7th Edition):

Ramanathan, Anita. “Defining function of an autism pathway in human neuronal cells.” 2011. Web. 25 Feb 2020.

Vancouver:

Ramanathan A. Defining function of an autism pathway in human neuronal cells. [Internet] [Masters thesis]. University of Southern California; 2011. [cited 2020 Feb 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1816.

Council of Science Editors:

Ramanathan A. Defining function of an autism pathway in human neuronal cells. [Masters Thesis]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/627881/rec/1816


University of California – San Francisco

2. Williams, Olusegun. Discovery and development of dual PI3Kdelta/gamma inhibitors for use as novel anti-inflammatory agents.

Degree: Chemistry and Chemical Biology, 2011, University of California – San Francisco

 PI3Kdelta and PI3Kgamma regulate immune cell signaling, while the related PI3Kalpha; and PI3Kbeta; regulate cell survival and metabolism. Selective dual inhibitors of PI3Kdelta and PI3Kgamma;… (more)

Subjects/Keywords: Chemistry, Pharmaceutical; anti-inflammatory; glucocorticoid; Intellikine; PI3K; PI3K delta; PI3K gamma

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APA (6th Edition):

Williams, O. (2011). Discovery and development of dual PI3Kdelta/gamma inhibitors for use as novel anti-inflammatory agents. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/0vr1d8sv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Olusegun. “Discovery and development of dual PI3Kdelta/gamma inhibitors for use as novel anti-inflammatory agents.” 2011. Thesis, University of California – San Francisco. Accessed February 25, 2020. http://www.escholarship.org/uc/item/0vr1d8sv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Olusegun. “Discovery and development of dual PI3Kdelta/gamma inhibitors for use as novel anti-inflammatory agents.” 2011. Web. 25 Feb 2020.

Vancouver:

Williams O. Discovery and development of dual PI3Kdelta/gamma inhibitors for use as novel anti-inflammatory agents. [Internet] [Thesis]. University of California – San Francisco; 2011. [cited 2020 Feb 25]. Available from: http://www.escholarship.org/uc/item/0vr1d8sv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams O. Discovery and development of dual PI3Kdelta/gamma inhibitors for use as novel anti-inflammatory agents. [Thesis]. University of California – San Francisco; 2011. Available from: http://www.escholarship.org/uc/item/0vr1d8sv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Levade, Marie. Mécanismes moléculaires de la production et des fonctions plaquettaires : rôle de Vps34 et impact des inhibiteurs ciblés de kinases : Molecular mechanisms of platelet production and functions : role of Vps34 and impact of kinase inhibitors.

Degree: Docteur es, Physiopathologie, 2017, Université Toulouse III – Paul Sabatier

 Les plaquettes sanguines jouent un rôle essentiel dans le maintien de l'intégrité des vaisseaux sanguins. En cas de brèche vasculaire, elles conduisent à la formation… (more)

Subjects/Keywords: Plaquettes; PI3K; Thrombus; Thérapies ciblées

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APA (6th Edition):

Levade, M. (2017). Mécanismes moléculaires de la production et des fonctions plaquettaires : rôle de Vps34 et impact des inhibiteurs ciblés de kinases : Molecular mechanisms of platelet production and functions : role of Vps34 and impact of kinase inhibitors. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2017TOU30036

Chicago Manual of Style (16th Edition):

Levade, Marie. “Mécanismes moléculaires de la production et des fonctions plaquettaires : rôle de Vps34 et impact des inhibiteurs ciblés de kinases : Molecular mechanisms of platelet production and functions : role of Vps34 and impact of kinase inhibitors.” 2017. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed February 25, 2020. http://www.theses.fr/2017TOU30036.

MLA Handbook (7th Edition):

Levade, Marie. “Mécanismes moléculaires de la production et des fonctions plaquettaires : rôle de Vps34 et impact des inhibiteurs ciblés de kinases : Molecular mechanisms of platelet production and functions : role of Vps34 and impact of kinase inhibitors.” 2017. Web. 25 Feb 2020.

Vancouver:

Levade M. Mécanismes moléculaires de la production et des fonctions plaquettaires : rôle de Vps34 et impact des inhibiteurs ciblés de kinases : Molecular mechanisms of platelet production and functions : role of Vps34 and impact of kinase inhibitors. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2017. [cited 2020 Feb 25]. Available from: http://www.theses.fr/2017TOU30036.

Council of Science Editors:

Levade M. Mécanismes moléculaires de la production et des fonctions plaquettaires : rôle de Vps34 et impact des inhibiteurs ciblés de kinases : Molecular mechanisms of platelet production and functions : role of Vps34 and impact of kinase inhibitors. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2017. Available from: http://www.theses.fr/2017TOU30036


University of Alberta

4. Farhan, Maikel AA. Investigating the endothelial PI3 kinase signalling pathway in vascular repair.

Degree: PhD, Department of Medicine, 2016, University of Alberta

 Thrombotic microangiopathy (TMA) is a broad term for a range of diseases that usually manifest with rapid failure of the affected organ. Although different in… (more)

Subjects/Keywords: Angiogenesis; Endothelial cells; PI3K; FGD5

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APA (6th Edition):

Farhan, M. A. (2016). Investigating the endothelial PI3 kinase signalling pathway in vascular repair. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cmp48sd05t

Chicago Manual of Style (16th Edition):

Farhan, Maikel AA. “Investigating the endothelial PI3 kinase signalling pathway in vascular repair.” 2016. Doctoral Dissertation, University of Alberta. Accessed February 25, 2020. https://era.library.ualberta.ca/files/cmp48sd05t.

MLA Handbook (7th Edition):

Farhan, Maikel AA. “Investigating the endothelial PI3 kinase signalling pathway in vascular repair.” 2016. Web. 25 Feb 2020.

Vancouver:

Farhan MA. Investigating the endothelial PI3 kinase signalling pathway in vascular repair. [Internet] [Doctoral dissertation]. University of Alberta; 2016. [cited 2020 Feb 25]. Available from: https://era.library.ualberta.ca/files/cmp48sd05t.

Council of Science Editors:

Farhan MA. Investigating the endothelial PI3 kinase signalling pathway in vascular repair. [Doctoral Dissertation]. University of Alberta; 2016. Available from: https://era.library.ualberta.ca/files/cmp48sd05t


University of Minnesota

5. Lepley, Michael Alan. Identification of arterial phenotype in endothelial cells derived from human pluripotent stem cells.

Degree: MS, Stem Cell Biology, 2013, University of Minnesota

 The use of human pluripotent stem cells (HPSC) to generate mature endothelial cells (EC) has been described previously, but is an inefficient process. We adopted… (more)

Subjects/Keywords: Arterial; CXCR4; Endothelial; PI3K

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APA (6th Edition):

Lepley, M. A. (2013). Identification of arterial phenotype in endothelial cells derived from human pluripotent stem cells. (Masters Thesis). University of Minnesota. Retrieved from http://hdl.handle.net/11299/162844

Chicago Manual of Style (16th Edition):

Lepley, Michael Alan. “Identification of arterial phenotype in endothelial cells derived from human pluripotent stem cells.” 2013. Masters Thesis, University of Minnesota. Accessed February 25, 2020. http://hdl.handle.net/11299/162844.

MLA Handbook (7th Edition):

Lepley, Michael Alan. “Identification of arterial phenotype in endothelial cells derived from human pluripotent stem cells.” 2013. Web. 25 Feb 2020.

Vancouver:

Lepley MA. Identification of arterial phenotype in endothelial cells derived from human pluripotent stem cells. [Internet] [Masters thesis]. University of Minnesota; 2013. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/11299/162844.

Council of Science Editors:

Lepley MA. Identification of arterial phenotype in endothelial cells derived from human pluripotent stem cells. [Masters Thesis]. University of Minnesota; 2013. Available from: http://hdl.handle.net/11299/162844


NSYSU

6. Tsai, Ching-yi. Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death.

Degree: PhD, Biological Sciences, 2009, NSYSU

 As the origin of a âlife-and-deathâ signal that reflects central cardiovascular regulatory failure during brain stem death, the rostral ventrolateral medulla (RVLM) is a suitable… (more)

Subjects/Keywords: PKA; Akt; PI3K; mevinphos

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APA (6th Edition):

Tsai, C. (2009). Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714109-192732

Chicago Manual of Style (16th Edition):

Tsai, Ching-yi. “Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death.” 2009. Doctoral Dissertation, NSYSU. Accessed February 25, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714109-192732.

MLA Handbook (7th Edition):

Tsai, Ching-yi. “Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death.” 2009. Web. 25 Feb 2020.

Vancouver:

Tsai C. Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death. [Internet] [Doctoral dissertation]. NSYSU; 2009. [cited 2020 Feb 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714109-192732.

Council of Science Editors:

Tsai C. Roles of PI3K, Akt and PKA at Rostral Ventrolateral Medulla in a Mevinphos Intoxication Model of Brain Stem Death. [Doctoral Dissertation]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0714109-192732

7. Laurent, Pierre-Alexandre. Rôles des phosphoinositides 3-kinases (PI3Ks) α et β de classe IA dans les processus de l'activation plaquettaire et de la thrombose : Roles of class IA PI3Ks alpha and beta in platelet activation and thrombosis.

Degree: Docteur es, Physiopathologie, 2015, Université Toulouse III – Paul Sabatier

 Les plaquettes jouent un rôle majeur dans l'hémostase mais également dans les maladies cardiovasculaires qui représentent une des principales causes de mortalité dans les pays… (more)

Subjects/Keywords: Plaquettes; Hémostase; PI3K; Phosphoinositides; Thrombus

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APA (6th Edition):

Laurent, P. (2015). Rôles des phosphoinositides 3-kinases (PI3Ks) α et β de classe IA dans les processus de l'activation plaquettaire et de la thrombose : Roles of class IA PI3Ks alpha and beta in platelet activation and thrombosis. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2015TOU30296

Chicago Manual of Style (16th Edition):

Laurent, Pierre-Alexandre. “Rôles des phosphoinositides 3-kinases (PI3Ks) α et β de classe IA dans les processus de l'activation plaquettaire et de la thrombose : Roles of class IA PI3Ks alpha and beta in platelet activation and thrombosis.” 2015. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed February 25, 2020. http://www.theses.fr/2015TOU30296.

MLA Handbook (7th Edition):

Laurent, Pierre-Alexandre. “Rôles des phosphoinositides 3-kinases (PI3Ks) α et β de classe IA dans les processus de l'activation plaquettaire et de la thrombose : Roles of class IA PI3Ks alpha and beta in platelet activation and thrombosis.” 2015. Web. 25 Feb 2020.

Vancouver:

Laurent P. Rôles des phosphoinositides 3-kinases (PI3Ks) α et β de classe IA dans les processus de l'activation plaquettaire et de la thrombose : Roles of class IA PI3Ks alpha and beta in platelet activation and thrombosis. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2015. [cited 2020 Feb 25]. Available from: http://www.theses.fr/2015TOU30296.

Council of Science Editors:

Laurent P. Rôles des phosphoinositides 3-kinases (PI3Ks) α et β de classe IA dans les processus de l'activation plaquettaire et de la thrombose : Roles of class IA PI3Ks alpha and beta in platelet activation and thrombosis. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2015. Available from: http://www.theses.fr/2015TOU30296


Universitat de Valencia

8. Oliver Guillén, María Dolores. Caracterización funcional de mutaciones oncogénicas de PI3K .

Degree: 2015, Universitat de Valencia

 El cáncer es una enfermedad compleja originada por alteraciones genéticas en proto-oncogenes y/o supresores tumorales. El proto-oncogén PI3K se encuentra alterado en numerosos cánceres. La… (more)

Subjects/Keywords: cáncer; PI3K; p85; p65; tumor

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APA (6th Edition):

Oliver Guillén, M. D. (2015). Caracterización funcional de mutaciones oncogénicas de PI3K . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/48103

Chicago Manual of Style (16th Edition):

Oliver Guillén, María Dolores. “Caracterización funcional de mutaciones oncogénicas de PI3K .” 2015. Doctoral Dissertation, Universitat de Valencia. Accessed February 25, 2020. http://hdl.handle.net/10550/48103.

MLA Handbook (7th Edition):

Oliver Guillén, María Dolores. “Caracterización funcional de mutaciones oncogénicas de PI3K .” 2015. Web. 25 Feb 2020.

Vancouver:

Oliver Guillén MD. Caracterización funcional de mutaciones oncogénicas de PI3K . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2015. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/10550/48103.

Council of Science Editors:

Oliver Guillén MD. Caracterización funcional de mutaciones oncogénicas de PI3K . [Doctoral Dissertation]. Universitat de Valencia; 2015. Available from: http://hdl.handle.net/10550/48103


University of Cambridge

9. Alam, Rafeah. T cell phenotyping of a mouse model of Activated PI3Kdelta syndrome.

Degree: PhD, 2019, University of Cambridge

 Activated PI3Kdelta Syndrome (APDS) is immunodeficiency caused by a heterozygous gain-of-function mutation (E1021K) in the PIK3CD gene, encoding for the p110delta catalytic subunit of phosphoinositide… (more)

Subjects/Keywords: APDS; PI3K; T cells

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APA (6th Edition):

Alam, R. (2019). T cell phenotyping of a mouse model of Activated PI3Kdelta syndrome. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/293595

Chicago Manual of Style (16th Edition):

Alam, Rafeah. “T cell phenotyping of a mouse model of Activated PI3Kdelta syndrome.” 2019. Doctoral Dissertation, University of Cambridge. Accessed February 25, 2020. https://www.repository.cam.ac.uk/handle/1810/293595.

MLA Handbook (7th Edition):

Alam, Rafeah. “T cell phenotyping of a mouse model of Activated PI3Kdelta syndrome.” 2019. Web. 25 Feb 2020.

Vancouver:

Alam R. T cell phenotyping of a mouse model of Activated PI3Kdelta syndrome. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Feb 25]. Available from: https://www.repository.cam.ac.uk/handle/1810/293595.

Council of Science Editors:

Alam R. T cell phenotyping of a mouse model of Activated PI3Kdelta syndrome. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/293595


University of Melbourne

10. Palmieri, Michelle. Understanding PI3K signalling in colorectal cancer: from function to therapy.

Degree: 2016, University of Melbourne

 Aberrant phosphoinositide-3 kinase (PI3K) signalling is associated with the development and progression of several types of cancer including colorectal cancer (CRC) and accordingly a target… (more)

Subjects/Keywords: colon cancer; PI3K; cell signalling

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APA (6th Edition):

Palmieri, M. (2016). Understanding PI3K signalling in colorectal cancer: from function to therapy. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/112365

Chicago Manual of Style (16th Edition):

Palmieri, Michelle. “Understanding PI3K signalling in colorectal cancer: from function to therapy.” 2016. Doctoral Dissertation, University of Melbourne. Accessed February 25, 2020. http://hdl.handle.net/11343/112365.

MLA Handbook (7th Edition):

Palmieri, Michelle. “Understanding PI3K signalling in colorectal cancer: from function to therapy.” 2016. Web. 25 Feb 2020.

Vancouver:

Palmieri M. Understanding PI3K signalling in colorectal cancer: from function to therapy. [Internet] [Doctoral dissertation]. University of Melbourne; 2016. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/11343/112365.

Council of Science Editors:

Palmieri M. Understanding PI3K signalling in colorectal cancer: from function to therapy. [Doctoral Dissertation]. University of Melbourne; 2016. Available from: http://hdl.handle.net/11343/112365


University of Manitoba

11. Zhang, Ting-ting. Regulatory roles of PI3Ks and PH domain-containing adaptor protein Bam32 in humoral immune responses.

Degree: Immunology, 2010, University of Manitoba

 PI3Ks (phosphoinositide 3-kinases), a family of enzymes expressed in immune cells, are activated in response to a wide variety of stimuli by generating second lipid… (more)

Subjects/Keywords: PI3K; Bam32; humoral immune responses

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APA (6th Edition):

Zhang, T. (2010). Regulatory roles of PI3Ks and PH domain-containing adaptor protein Bam32 in humoral immune responses. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/3980

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Ting-ting. “Regulatory roles of PI3Ks and PH domain-containing adaptor protein Bam32 in humoral immune responses.” 2010. Thesis, University of Manitoba. Accessed February 25, 2020. http://hdl.handle.net/1993/3980.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Ting-ting. “Regulatory roles of PI3Ks and PH domain-containing adaptor protein Bam32 in humoral immune responses.” 2010. Web. 25 Feb 2020.

Vancouver:

Zhang T. Regulatory roles of PI3Ks and PH domain-containing adaptor protein Bam32 in humoral immune responses. [Internet] [Thesis]. University of Manitoba; 2010. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/1993/3980.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang T. Regulatory roles of PI3Ks and PH domain-containing adaptor protein Bam32 in humoral immune responses. [Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/3980

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Bath

12. Proctor, Victoria Kate. Signalling pathways linking interleukin 13 receptor activation to lung epithelial cell function.

Degree: PhD, 2013, University of Bath

 The passage of fluid, ions and macromolecules across the epithelium is controlled primarily by epithelial tight junctions. Altered epithelial permeability is associated with lung disease,… (more)

Subjects/Keywords: 611.0181; PI3K; tight junctions

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APA (6th Edition):

Proctor, V. K. (2013). Signalling pathways linking interleukin 13 receptor activation to lung epithelial cell function. (Doctoral Dissertation). University of Bath. Retrieved from https://researchportal.bath.ac.uk/en/studentthesis/signalling-pathways-linking-interleukin-13-receptor-activation-to-lung-epithelial-cell-function(54e639e8-31a0-4fd4-835a-407a28c84eb2).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589658

Chicago Manual of Style (16th Edition):

Proctor, Victoria Kate. “Signalling pathways linking interleukin 13 receptor activation to lung epithelial cell function.” 2013. Doctoral Dissertation, University of Bath. Accessed February 25, 2020. https://researchportal.bath.ac.uk/en/studentthesis/signalling-pathways-linking-interleukin-13-receptor-activation-to-lung-epithelial-cell-function(54e639e8-31a0-4fd4-835a-407a28c84eb2).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589658.

MLA Handbook (7th Edition):

Proctor, Victoria Kate. “Signalling pathways linking interleukin 13 receptor activation to lung epithelial cell function.” 2013. Web. 25 Feb 2020.

Vancouver:

Proctor VK. Signalling pathways linking interleukin 13 receptor activation to lung epithelial cell function. [Internet] [Doctoral dissertation]. University of Bath; 2013. [cited 2020 Feb 25]. Available from: https://researchportal.bath.ac.uk/en/studentthesis/signalling-pathways-linking-interleukin-13-receptor-activation-to-lung-epithelial-cell-function(54e639e8-31a0-4fd4-835a-407a28c84eb2).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589658.

Council of Science Editors:

Proctor VK. Signalling pathways linking interleukin 13 receptor activation to lung epithelial cell function. [Doctoral Dissertation]. University of Bath; 2013. Available from: https://researchportal.bath.ac.uk/en/studentthesis/signalling-pathways-linking-interleukin-13-receptor-activation-to-lung-epithelial-cell-function(54e639e8-31a0-4fd4-835a-407a28c84eb2).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589658


Queens University

13. Mcdonald, Gail. Inhibition of Phosphatidylinositol 3-Kinase (PI3K) Signalling Leads to Resistance to Chemotherapeutic Agents in Human Cancer Cells .

Degree: Anatomy and Cell Biology, 2008, Queens University

 One of the major challenges associated with cancer therapy is the acquisition of chemoresistance by tumour cells. Many novel therapeutic approaches to overcome chemoresistance have… (more)

Subjects/Keywords: PI3K; Chemoresistance

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APA (6th Edition):

Mcdonald, G. (2008). Inhibition of Phosphatidylinositol 3-Kinase (PI3K) Signalling Leads to Resistance to Chemotherapeutic Agents in Human Cancer Cells . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/1475

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mcdonald, Gail. “Inhibition of Phosphatidylinositol 3-Kinase (PI3K) Signalling Leads to Resistance to Chemotherapeutic Agents in Human Cancer Cells .” 2008. Thesis, Queens University. Accessed February 25, 2020. http://hdl.handle.net/1974/1475.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mcdonald, Gail. “Inhibition of Phosphatidylinositol 3-Kinase (PI3K) Signalling Leads to Resistance to Chemotherapeutic Agents in Human Cancer Cells .” 2008. Web. 25 Feb 2020.

Vancouver:

Mcdonald G. Inhibition of Phosphatidylinositol 3-Kinase (PI3K) Signalling Leads to Resistance to Chemotherapeutic Agents in Human Cancer Cells . [Internet] [Thesis]. Queens University; 2008. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/1974/1475.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mcdonald G. Inhibition of Phosphatidylinositol 3-Kinase (PI3K) Signalling Leads to Resistance to Chemotherapeutic Agents in Human Cancer Cells . [Thesis]. Queens University; 2008. Available from: http://hdl.handle.net/1974/1475

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Duarte, Andressa. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.

Degree: Mestrado, Imunologia Básica e Aplicada, 2013, University of São Paulo

A imunidade inata é responsável pela resposta inicial aos microrganismos, uma vez que impede, controla ou elimina a infecção. Esse sistema consiste em barreiras epiteliais,… (more)

Subjects/Keywords: iNOS; iNOS; NF-kB; NF-kB; NO; NO; PI3K; PI3K

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APA (6th Edition):

Duarte, A. (2013). Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;

Chicago Manual of Style (16th Edition):

Duarte, Andressa. “Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.” 2013. Masters Thesis, University of São Paulo. Accessed February 25, 2020. http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;.

MLA Handbook (7th Edition):

Duarte, Andressa. “Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais.” 2013. Web. 25 Feb 2020.

Vancouver:

Duarte A. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2020 Feb 25]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;.

Council of Science Editors:

Duarte A. Participação da via PI3K/AKT na produção de óxido nítrico por macrófagos peritoneais. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21102013-112320/ ;

15. Baliou, Evangelia. Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού.

Degree: 2018, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

PI3K/Akt pathway is one of the most important signal transduction pathways activated by many stimuli, such as hormones and growth factors. It’s role in modulating… (more)

Subjects/Keywords: Μονοπάτι PI3K/Akt; Μαστός; Pathway PI3K/Akt; Breast

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APA (6th Edition):

Baliou, E. (2018). Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/44625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baliou, Evangelia. “Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού.” 2018. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed February 25, 2020. http://hdl.handle.net/10442/hedi/44625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baliou, Evangelia. “Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού.” 2018. Web. 25 Feb 2020.

Vancouver:

Baliou E. Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2018. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/10442/hedi/44625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baliou E. Ανοσοφαινοτυπική μελέτη μοριακών δεικτών εμπλεκόμενων στην παθογενετική οδό ΡΙ3Κ/ΑΚΤ σε διηθητικά και μη καρκινώματα του μαστού. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2018. Available from: http://hdl.handle.net/10442/hedi/44625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

16. Chu, Ying Ying Julia. Apoptosis is promoted by unconventional FcγR-PI3KCdc42-Pak-Mek-Erk signalling in the human neutrophil.

Degree: PhD, 2017, University of Edinburgh

 Neutrophils form a first line of defence against infections. These short-lived, terminally differentiated cells perform many important functions, including chemotaxis, degranulation, reactive oxygen species (ROS)… (more)

Subjects/Keywords: neutrophils; PI3K signalling; signalling pathways; PI3K-Cdc42-Pak-Mek-Erk

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APA (6th Edition):

Chu, Y. Y. J. (2017). Apoptosis is promoted by unconventional FcγR-PI3KCdc42-Pak-Mek-Erk signalling in the human neutrophil. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/28813

Chicago Manual of Style (16th Edition):

Chu, Ying Ying Julia. “Apoptosis is promoted by unconventional FcγR-PI3KCdc42-Pak-Mek-Erk signalling in the human neutrophil.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed February 25, 2020. http://hdl.handle.net/1842/28813.

MLA Handbook (7th Edition):

Chu, Ying Ying Julia. “Apoptosis is promoted by unconventional FcγR-PI3KCdc42-Pak-Mek-Erk signalling in the human neutrophil.” 2017. Web. 25 Feb 2020.

Vancouver:

Chu YYJ. Apoptosis is promoted by unconventional FcγR-PI3KCdc42-Pak-Mek-Erk signalling in the human neutrophil. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/1842/28813.

Council of Science Editors:

Chu YYJ. Apoptosis is promoted by unconventional FcγR-PI3KCdc42-Pak-Mek-Erk signalling in the human neutrophil. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/28813

17. Pizon, Mathieu. L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis.

Degree: Docteur es, Sciences, technologie, santé. Microbiologie, 2010, Université de Bordeaux Segalen

Le CD95 appartient à la famille du TNF-R. Il est capable de déclencher un signal apoptotique et joue un rôle prépondérant dans le maintien de… (more)

Subjects/Keywords: Apoptose; Cd95; Pi3k; Akt; Microdomaines; Apoptosis; Microdomains

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APA (6th Edition):

Pizon, M. (2010). L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2010BOR21710

Chicago Manual of Style (16th Edition):

Pizon, Mathieu. “L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis.” 2010. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed February 25, 2020. http://www.theses.fr/2010BOR21710.

MLA Handbook (7th Edition):

Pizon, Mathieu. “L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis.” 2010. Web. 25 Feb 2020.

Vancouver:

Pizon M. L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2010. [cited 2020 Feb 25]. Available from: http://www.theses.fr/2010BOR21710.

Council of Science Editors:

Pizon M. L'inhibition de la voie Phosphoinositide-3 kinase (PI3K)/AKT induit un signal apoptotique via la redistribution du récepteur de mort CD95 dans les radeaux lipidiques : Effects of Met tyrosine kinase receptor cleavage by calpains on cell motility and cell necrosis. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2010. Available from: http://www.theses.fr/2010BOR21710


Temple University

18. Brennan, Tracy A. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.

Degree: PhD, 2011, Temple University

Cell Biology

Cbl is a multivalent protein that interacts with a number of signaling molecules that affect cell proliferation, migration and apoptosis. Although it is… (more)

Subjects/Keywords: Cellular Biology; Biology; Bone; Cbl; Osteoblasts; PI3K

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APA (6th Edition):

Brennan, T. A. (2011). Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,107475

Chicago Manual of Style (16th Edition):

Brennan, Tracy A. “Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.” 2011. Doctoral Dissertation, Temple University. Accessed February 25, 2020. http://digital.library.temple.edu/u?/p245801coll10,107475.

MLA Handbook (7th Edition):

Brennan, Tracy A. “Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.” 2011. Web. 25 Feb 2020.

Vancouver:

Brennan TA. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Feb 25]. Available from: http://digital.library.temple.edu/u?/p245801coll10,107475.

Council of Science Editors:

Brennan TA. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,107475


University of Alberta

19. Urbanowski, Matthew D. Characterization of the anti-apoptotic properties of flavivirus capsid proteins.

Degree: PhD, Department of Cell Biology, 2014, University of Alberta

 The introduction of WNV into North America in 1999 was followed by rapid spread throughout the continent. Today, WNV is an endemic pathogen in the… (more)

Subjects/Keywords: apoptosis; virus; capsid; PI3K; flavivirus; akt

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APA (6th Edition):

Urbanowski, M. D. (2014). Characterization of the anti-apoptotic properties of flavivirus capsid proteins. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/sb3978635

Chicago Manual of Style (16th Edition):

Urbanowski, Matthew D. “Characterization of the anti-apoptotic properties of flavivirus capsid proteins.” 2014. Doctoral Dissertation, University of Alberta. Accessed February 25, 2020. https://era.library.ualberta.ca/files/sb3978635.

MLA Handbook (7th Edition):

Urbanowski, Matthew D. “Characterization of the anti-apoptotic properties of flavivirus capsid proteins.” 2014. Web. 25 Feb 2020.

Vancouver:

Urbanowski MD. Characterization of the anti-apoptotic properties of flavivirus capsid proteins. [Internet] [Doctoral dissertation]. University of Alberta; 2014. [cited 2020 Feb 25]. Available from: https://era.library.ualberta.ca/files/sb3978635.

Council of Science Editors:

Urbanowski MD. Characterization of the anti-apoptotic properties of flavivirus capsid proteins. [Doctoral Dissertation]. University of Alberta; 2014. Available from: https://era.library.ualberta.ca/files/sb3978635


Duquesne University

20. Monlish, Darlene A. Age-Related Effects on the Mitogen-Activated Protein Kinase and Phosphatidylinositol 3-Kinase Pathways in Breast Cancer and the Characterization of Novel MEK5 Inhibitors.

Degree: PhD, Pharmacology-Toxicology, 2013, Duquesne University

 Age remains the most common demographic risk factor in breast cancer. The underlying cellular signaling mechanisms responsible for the age-associated rise in disease incidence have… (more)

Subjects/Keywords: Age; Breast Cancer; ERK5; Inhibitors; MAPK; PI3K

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APA (6th Edition):

Monlish, D. A. (2013). Age-Related Effects on the Mitogen-Activated Protein Kinase and Phosphatidylinositol 3-Kinase Pathways in Breast Cancer and the Characterization of Novel MEK5 Inhibitors. (Doctoral Dissertation). Duquesne University. Retrieved from https://dsc.duq.edu/etd/941

Chicago Manual of Style (16th Edition):

Monlish, Darlene A. “Age-Related Effects on the Mitogen-Activated Protein Kinase and Phosphatidylinositol 3-Kinase Pathways in Breast Cancer and the Characterization of Novel MEK5 Inhibitors.” 2013. Doctoral Dissertation, Duquesne University. Accessed February 25, 2020. https://dsc.duq.edu/etd/941.

MLA Handbook (7th Edition):

Monlish, Darlene A. “Age-Related Effects on the Mitogen-Activated Protein Kinase and Phosphatidylinositol 3-Kinase Pathways in Breast Cancer and the Characterization of Novel MEK5 Inhibitors.” 2013. Web. 25 Feb 2020.

Vancouver:

Monlish DA. Age-Related Effects on the Mitogen-Activated Protein Kinase and Phosphatidylinositol 3-Kinase Pathways in Breast Cancer and the Characterization of Novel MEK5 Inhibitors. [Internet] [Doctoral dissertation]. Duquesne University; 2013. [cited 2020 Feb 25]. Available from: https://dsc.duq.edu/etd/941.

Council of Science Editors:

Monlish DA. Age-Related Effects on the Mitogen-Activated Protein Kinase and Phosphatidylinositol 3-Kinase Pathways in Breast Cancer and the Characterization of Novel MEK5 Inhibitors. [Doctoral Dissertation]. Duquesne University; 2013. Available from: https://dsc.duq.edu/etd/941


North Carolina State University

21. Melvin, Adam Thomas. Relating phosphoinositide 3-kinase (PI3K) signaling and cell motility dynamics during PDGF-stimulated chemotaxis.

Degree: PhD, Chemical Engineering, 2010, North Carolina State University

 Cell migration is essential for wound healing, the immune response, embryogenesis, and cancer metastasis. Chemotaxis, or cell migration directed by an external gradient of chemoattractant,… (more)

Subjects/Keywords: PI3K; Cell migration; PDGF; Chemotaxis; Microfluidics; Fibroblasts

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APA (6th Edition):

Melvin, A. T. (2010). Relating phosphoinositide 3-kinase (PI3K) signaling and cell motility dynamics during PDGF-stimulated chemotaxis. (Doctoral Dissertation). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/6177

Chicago Manual of Style (16th Edition):

Melvin, Adam Thomas. “Relating phosphoinositide 3-kinase (PI3K) signaling and cell motility dynamics during PDGF-stimulated chemotaxis.” 2010. Doctoral Dissertation, North Carolina State University. Accessed February 25, 2020. http://www.lib.ncsu.edu/resolver/1840.16/6177.

MLA Handbook (7th Edition):

Melvin, Adam Thomas. “Relating phosphoinositide 3-kinase (PI3K) signaling and cell motility dynamics during PDGF-stimulated chemotaxis.” 2010. Web. 25 Feb 2020.

Vancouver:

Melvin AT. Relating phosphoinositide 3-kinase (PI3K) signaling and cell motility dynamics during PDGF-stimulated chemotaxis. [Internet] [Doctoral dissertation]. North Carolina State University; 2010. [cited 2020 Feb 25]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/6177.

Council of Science Editors:

Melvin AT. Relating phosphoinositide 3-kinase (PI3K) signaling and cell motility dynamics during PDGF-stimulated chemotaxis. [Doctoral Dissertation]. North Carolina State University; 2010. Available from: http://www.lib.ncsu.edu/resolver/1840.16/6177


Kent State University

22. Redfern, Roberta E. Characterization of Binding of PTEN and its Disease Related Mutants to Phospholipid Model Membranes.

Degree: PhD, College of Arts and Sciences / Department of Chemistry, 2008, Kent State University

 PTEN, phosphatase and tensin homologue deleted on chromosome 10, is a tumor suppressor that is commonly lost or mutated in many different diseases, including cancer,… (more)

Subjects/Keywords: Biochemistry; PTEN; phosphoinositides; PI3K pathway; cholesterol

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APA (6th Edition):

Redfern, R. E. (2008). Characterization of Binding of PTEN and its Disease Related Mutants to Phospholipid Model Membranes. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1216671104

Chicago Manual of Style (16th Edition):

Redfern, Roberta E. “Characterization of Binding of PTEN and its Disease Related Mutants to Phospholipid Model Membranes.” 2008. Doctoral Dissertation, Kent State University. Accessed February 25, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=kent1216671104.

MLA Handbook (7th Edition):

Redfern, Roberta E. “Characterization of Binding of PTEN and its Disease Related Mutants to Phospholipid Model Membranes.” 2008. Web. 25 Feb 2020.

Vancouver:

Redfern RE. Characterization of Binding of PTEN and its Disease Related Mutants to Phospholipid Model Membranes. [Internet] [Doctoral dissertation]. Kent State University; 2008. [cited 2020 Feb 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1216671104.

Council of Science Editors:

Redfern RE. Characterization of Binding of PTEN and its Disease Related Mutants to Phospholipid Model Membranes. [Doctoral Dissertation]. Kent State University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1216671104


Penn State University

23. Lee, Yunkyoung. ANTI-INFLAMMATORY AND ANTI-ATHEROGENIC EFFECTS OF 9E,11E-CONJUGATED LINOLEIC ACID.

Degree: PhD, Integrative Biosciences, 2008, Penn State University

 Conjugated linoleic acid (CLA) is a mixture of dietary fatty acids that has various beneficial effects including a reduction of cancer, atherosclerosis and inflammation in… (more)

Subjects/Keywords: mTOR; anti-inflammatory; anti-atherogenic; CLA; PI3K

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APA (6th Edition):

Lee, Y. (2008). ANTI-INFLAMMATORY AND ANTI-ATHEROGENIC EFFECTS OF 9E,11E-CONJUGATED LINOLEIC ACID. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/8198

Chicago Manual of Style (16th Edition):

Lee, Yunkyoung. “ANTI-INFLAMMATORY AND ANTI-ATHEROGENIC EFFECTS OF 9E,11E-CONJUGATED LINOLEIC ACID.” 2008. Doctoral Dissertation, Penn State University. Accessed February 25, 2020. https://etda.libraries.psu.edu/catalog/8198.

MLA Handbook (7th Edition):

Lee, Yunkyoung. “ANTI-INFLAMMATORY AND ANTI-ATHEROGENIC EFFECTS OF 9E,11E-CONJUGATED LINOLEIC ACID.” 2008. Web. 25 Feb 2020.

Vancouver:

Lee Y. ANTI-INFLAMMATORY AND ANTI-ATHEROGENIC EFFECTS OF 9E,11E-CONJUGATED LINOLEIC ACID. [Internet] [Doctoral dissertation]. Penn State University; 2008. [cited 2020 Feb 25]. Available from: https://etda.libraries.psu.edu/catalog/8198.

Council of Science Editors:

Lee Y. ANTI-INFLAMMATORY AND ANTI-ATHEROGENIC EFFECTS OF 9E,11E-CONJUGATED LINOLEIC ACID. [Doctoral Dissertation]. Penn State University; 2008. Available from: https://etda.libraries.psu.edu/catalog/8198


University of Manchester

24. Rowling, Emily. Pre-clinical evaluation of novel anti-metastatic targets.

Degree: PhD, 2014, University of Manchester

 Background: Radiotherapy is used in the treatment of over 50% of cancer patients and bar surgery, is the most effective cancer intervention. However, in the… (more)

Subjects/Keywords: 615.8; Src; metastasis; Radiotherapy; PI3K; Thyroid

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APA (6th Edition):

Rowling, E. (2014). Pre-clinical evaluation of novel anti-metastatic targets. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/preclinical-evaluation-of-novel-antimetastatic-targets(caa9ab41-c054-4559-b575-3fd8974005a7).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.634898

Chicago Manual of Style (16th Edition):

Rowling, Emily. “Pre-clinical evaluation of novel anti-metastatic targets.” 2014. Doctoral Dissertation, University of Manchester. Accessed February 25, 2020. https://www.research.manchester.ac.uk/portal/en/theses/preclinical-evaluation-of-novel-antimetastatic-targets(caa9ab41-c054-4559-b575-3fd8974005a7).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.634898.

MLA Handbook (7th Edition):

Rowling, Emily. “Pre-clinical evaluation of novel anti-metastatic targets.” 2014. Web. 25 Feb 2020.

Vancouver:

Rowling E. Pre-clinical evaluation of novel anti-metastatic targets. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2020 Feb 25]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/preclinical-evaluation-of-novel-antimetastatic-targets(caa9ab41-c054-4559-b575-3fd8974005a7).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.634898.

Council of Science Editors:

Rowling E. Pre-clinical evaluation of novel anti-metastatic targets. [Doctoral Dissertation]. University of Manchester; 2014. Available from: https://www.research.manchester.ac.uk/portal/en/theses/preclinical-evaluation-of-novel-antimetastatic-targets(caa9ab41-c054-4559-b575-3fd8974005a7).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.634898


University of Saskatchewan

25. Marshall, Jeremy Davin Seiberling. STRUCTURAL ANALYSIS OF THE P85 BH DOMAIN IN COMPLEX WITH BINDING PARTNERS AND EFFECT OF MUTATIONS.

Degree: 2017, University of Saskatchewan

 The phosphatidylinositol 3-kinase (PI3K)/PTEN (phosphatase and tensin homologue deleted on chromosome 10) pathway is activated upon stimulation of receptor tyrosine kinases (RTKs) and regulates downstream… (more)

Subjects/Keywords: p85; crystallography; structural study; cancer; PI3K

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APA (6th Edition):

Marshall, J. D. S. (2017). STRUCTURAL ANALYSIS OF THE P85 BH DOMAIN IN COMPLEX WITH BINDING PARTNERS AND EFFECT OF MUTATIONS. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/8321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marshall, Jeremy Davin Seiberling. “STRUCTURAL ANALYSIS OF THE P85 BH DOMAIN IN COMPLEX WITH BINDING PARTNERS AND EFFECT OF MUTATIONS.” 2017. Thesis, University of Saskatchewan. Accessed February 25, 2020. http://hdl.handle.net/10388/8321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marshall, Jeremy Davin Seiberling. “STRUCTURAL ANALYSIS OF THE P85 BH DOMAIN IN COMPLEX WITH BINDING PARTNERS AND EFFECT OF MUTATIONS.” 2017. Web. 25 Feb 2020.

Vancouver:

Marshall JDS. STRUCTURAL ANALYSIS OF THE P85 BH DOMAIN IN COMPLEX WITH BINDING PARTNERS AND EFFECT OF MUTATIONS. [Internet] [Thesis]. University of Saskatchewan; 2017. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/10388/8321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marshall JDS. STRUCTURAL ANALYSIS OF THE P85 BH DOMAIN IN COMPLEX WITH BINDING PARTNERS AND EFFECT OF MUTATIONS. [Thesis]. University of Saskatchewan; 2017. Available from: http://hdl.handle.net/10388/8321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Sherbrooke

26. Fortin, Carl. Régulation de la production de chimiokines induite par des stimuli inflammatoires chez les neutrophiles humains : rôle des phosphatidylinositol 3 kinases (PI3Ks), des MAP kinase interacting kinases (MNKs), et de l'interleukine (IL)-18 .

Degree: 2010, Université de Sherbrooke

 Les neutrophiles produisent plusieurs médiateurs peptidiques contribuant à l'inflammation lors de la réponse immunitaire contre les agents infectieux. Ces médiateurs sont encodés par des gènes… (more)

Subjects/Keywords: Chimiokines; IL-18; MNK; PI3K; Neutrophiles humains

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fortin, C. (2010). Régulation de la production de chimiokines induite par des stimuli inflammatoires chez les neutrophiles humains : rôle des phosphatidylinositol 3 kinases (PI3Ks), des MAP kinase interacting kinases (MNKs), et de l'interleukine (IL)-18 . (Doctoral Dissertation). Université de Sherbrooke. Retrieved from http://savoirs.usherbrooke.ca/handle/11143/4316

Chicago Manual of Style (16th Edition):

Fortin, Carl. “Régulation de la production de chimiokines induite par des stimuli inflammatoires chez les neutrophiles humains : rôle des phosphatidylinositol 3 kinases (PI3Ks), des MAP kinase interacting kinases (MNKs), et de l'interleukine (IL)-18 .” 2010. Doctoral Dissertation, Université de Sherbrooke. Accessed February 25, 2020. http://savoirs.usherbrooke.ca/handle/11143/4316.

MLA Handbook (7th Edition):

Fortin, Carl. “Régulation de la production de chimiokines induite par des stimuli inflammatoires chez les neutrophiles humains : rôle des phosphatidylinositol 3 kinases (PI3Ks), des MAP kinase interacting kinases (MNKs), et de l'interleukine (IL)-18 .” 2010. Web. 25 Feb 2020.

Vancouver:

Fortin C. Régulation de la production de chimiokines induite par des stimuli inflammatoires chez les neutrophiles humains : rôle des phosphatidylinositol 3 kinases (PI3Ks), des MAP kinase interacting kinases (MNKs), et de l'interleukine (IL)-18 . [Internet] [Doctoral dissertation]. Université de Sherbrooke; 2010. [cited 2020 Feb 25]. Available from: http://savoirs.usherbrooke.ca/handle/11143/4316.

Council of Science Editors:

Fortin C. Régulation de la production de chimiokines induite par des stimuli inflammatoires chez les neutrophiles humains : rôle des phosphatidylinositol 3 kinases (PI3Ks), des MAP kinase interacting kinases (MNKs), et de l'interleukine (IL)-18 . [Doctoral Dissertation]. Université de Sherbrooke; 2010. Available from: http://savoirs.usherbrooke.ca/handle/11143/4316


University of British Columbia

27. Ma, Kewei. Investigation of the phosphatidylinositol 3-kinase pathway in B cells .

Degree: 2009, University of British Columbia

 There is hardly a cellular process that is not regulated in some way by phosphoinositides, which makes biochemical and physiological studies of these lipids extremely… (more)

Subjects/Keywords: Cellular signaling; PI3K

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ma, K. (2009). Investigation of the phosphatidylinositol 3-kinase pathway in B cells . (Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/3818

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ma, Kewei. “Investigation of the phosphatidylinositol 3-kinase pathway in B cells .” 2009. Thesis, University of British Columbia. Accessed February 25, 2020. http://hdl.handle.net/2429/3818.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ma, Kewei. “Investigation of the phosphatidylinositol 3-kinase pathway in B cells .” 2009. Web. 25 Feb 2020.

Vancouver:

Ma K. Investigation of the phosphatidylinositol 3-kinase pathway in B cells . [Internet] [Thesis]. University of British Columbia; 2009. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/2429/3818.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ma K. Investigation of the phosphatidylinositol 3-kinase pathway in B cells . [Thesis]. University of British Columbia; 2009. Available from: http://hdl.handle.net/2429/3818

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Kachrilas, Stefanos. Γενετικές και επιγενετικές αλλαγές στο μονοπάτι σηματοδότησης p13k / akt στον καρκίνο της ουροδόχου κύστεως.

Degree: 2018, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

The purpose of this PhD thesis was to examine the involvement of specific components of the PI3K/AKT pathway in urinary bladder cancer. Samples from sixty-five… (more)

Subjects/Keywords: PI3K / Akt signaling pathway; Cancer; Bladder cancer

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APA (6th Edition):

Kachrilas, S. (2018). Γενετικές και επιγενετικές αλλαγές στο μονοπάτι σηματοδότησης p13k / akt στον καρκίνο της ουροδόχου κύστεως. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/43242

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kachrilas, Stefanos. “Γενετικές και επιγενετικές αλλαγές στο μονοπάτι σηματοδότησης p13k / akt στον καρκίνο της ουροδόχου κύστεως.” 2018. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed February 25, 2020. http://hdl.handle.net/10442/hedi/43242.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kachrilas, Stefanos. “Γενετικές και επιγενετικές αλλαγές στο μονοπάτι σηματοδότησης p13k / akt στον καρκίνο της ουροδόχου κύστεως.” 2018. Web. 25 Feb 2020.

Vancouver:

Kachrilas S. Γενετικές και επιγενετικές αλλαγές στο μονοπάτι σηματοδότησης p13k / akt στον καρκίνο της ουροδόχου κύστεως. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2018. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/10442/hedi/43242.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kachrilas S. Γενετικές και επιγενετικές αλλαγές στο μονοπάτι σηματοδότησης p13k / akt στον καρκίνο της ουροδόχου κύστεως. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2018. Available from: http://hdl.handle.net/10442/hedi/43242

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

29. Tashkandi, Ghassan Yousuf. Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer.

Degree: PhD, 2017, University of Edinburgh

 The PI3K/Akt/mTOR and MAPK pathways are frequently altered in ovarian cancer cells, making them potential candidates for targeted therapy. A more complete understanding of the… (more)

Subjects/Keywords: ovarian cancer; PI3K; mTOR; cell signalling; rapamycin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tashkandi, G. Y. (2017). Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/28740

Chicago Manual of Style (16th Edition):

Tashkandi, Ghassan Yousuf. “Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed February 25, 2020. http://hdl.handle.net/1842/28740.

MLA Handbook (7th Edition):

Tashkandi, Ghassan Yousuf. “Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer.” 2017. Web. 25 Feb 2020.

Vancouver:

Tashkandi GY. Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2020 Feb 25]. Available from: http://hdl.handle.net/1842/28740.

Council of Science Editors:

Tashkandi GY. Phosphoproteomic profiling and targeting of the PI3K/Akt/mTOR and MAPK pathways in ovarian cancer. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/28740


University of California – San Francisco

30. Salt, Megan. Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival.

Degree: Biomedical Sciences, 2014, University of California – San Francisco

 Tumors showing evidence of epithelial to mesenchymal transition (EMT) have been associated with metastasis, drug resistance, and poor prognosis. Heterogeneity along the EMT spectrum is… (more)

Subjects/Keywords: Cellular biology; Biology; EMT; ERBB3; PI3K

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Salt, M. (2014). Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/52s838hv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Salt, Megan. “Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival.” 2014. Thesis, University of California – San Francisco. Accessed February 25, 2020. http://www.escholarship.org/uc/item/52s838hv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Salt, Megan. “Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival.” 2014. Web. 25 Feb 2020.

Vancouver:

Salt M. Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival. [Internet] [Thesis]. University of California – San Francisco; 2014. [cited 2020 Feb 25]. Available from: http://www.escholarship.org/uc/item/52s838hv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Salt M. Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival. [Thesis]. University of California – San Francisco; 2014. Available from: http://www.escholarship.org/uc/item/52s838hv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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